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You searched for subject:(Dystrophin). Showing records 1 – 30 of 91 total matches.

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University of Minnesota

1. Horn, Benjamin. Microscopic Properties Determine Macroscopic Characteristics of Proteins.

Degree: MS, Chemistry, 2016, University of Minnesota

 Macroscopic characteristics of a peptide or protein are dependent on microscopic properties. One microscopic property that has an effect on the macroscopic characteristics is the… (more)

Subjects/Keywords: Dystrophin; Oxidation; Synaptotagmin

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APA (6th Edition):

Horn, B. (2016). Microscopic Properties Determine Macroscopic Characteristics of Proteins. (Masters Thesis). University of Minnesota. Retrieved from http://hdl.handle.net/11299/185556

Chicago Manual of Style (16th Edition):

Horn, Benjamin. “Microscopic Properties Determine Macroscopic Characteristics of Proteins.” 2016. Masters Thesis, University of Minnesota. Accessed September 19, 2020. http://hdl.handle.net/11299/185556.

MLA Handbook (7th Edition):

Horn, Benjamin. “Microscopic Properties Determine Macroscopic Characteristics of Proteins.” 2016. Web. 19 Sep 2020.

Vancouver:

Horn B. Microscopic Properties Determine Macroscopic Characteristics of Proteins. [Internet] [Masters thesis]. University of Minnesota; 2016. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11299/185556.

Council of Science Editors:

Horn B. Microscopic Properties Determine Macroscopic Characteristics of Proteins. [Masters Thesis]. University of Minnesota; 2016. Available from: http://hdl.handle.net/11299/185556


University of Minnesota

2. Fealey, Michael E. Structural And Intrinsic Disorder In The Regulation Of Protein-Protein Interactions.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2019, University of Minnesota

 This thesis applied spectroscopy and molecular dynamics simulation to study the structural biology of actin-binding domains (ABDs) from the spectrin superfamily of proteins as well… (more)

Subjects/Keywords: dystrophin; spectrin; synaptotagmin

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APA (6th Edition):

Fealey, M. E. (2019). Structural And Intrinsic Disorder In The Regulation Of Protein-Protein Interactions. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/206287

Chicago Manual of Style (16th Edition):

Fealey, Michael E. “Structural And Intrinsic Disorder In The Regulation Of Protein-Protein Interactions.” 2019. Doctoral Dissertation, University of Minnesota. Accessed September 19, 2020. http://hdl.handle.net/11299/206287.

MLA Handbook (7th Edition):

Fealey, Michael E. “Structural And Intrinsic Disorder In The Regulation Of Protein-Protein Interactions.” 2019. Web. 19 Sep 2020.

Vancouver:

Fealey ME. Structural And Intrinsic Disorder In The Regulation Of Protein-Protein Interactions. [Internet] [Doctoral dissertation]. University of Minnesota; 2019. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11299/206287.

Council of Science Editors:

Fealey ME. Structural And Intrinsic Disorder In The Regulation Of Protein-Protein Interactions. [Doctoral Dissertation]. University of Minnesota; 2019. Available from: http://hdl.handle.net/11299/206287


University of Michigan

3. Barnabei, Matthew Scott. The Functional Role of Dystrophin in the Heart: Implications for Inherited and Non-Inherited Heart Disease.

Degree: PhD, Molecular and Integrative Physiology, 2012, University of Michigan

 The central focus of this thesis is the pathogenesis of cardiomyopathy caused by loss of dystrophin, and how this may be a unifying mechanism for… (more)

Subjects/Keywords: Dystrophin; Compliance; Enterovirus; Physiology; Science

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APA (6th Edition):

Barnabei, M. S. (2012). The Functional Role of Dystrophin in the Heart: Implications for Inherited and Non-Inherited Heart Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/91611

Chicago Manual of Style (16th Edition):

Barnabei, Matthew Scott. “The Functional Role of Dystrophin in the Heart: Implications for Inherited and Non-Inherited Heart Disease.” 2012. Doctoral Dissertation, University of Michigan. Accessed September 19, 2020. http://hdl.handle.net/2027.42/91611.

MLA Handbook (7th Edition):

Barnabei, Matthew Scott. “The Functional Role of Dystrophin in the Heart: Implications for Inherited and Non-Inherited Heart Disease.” 2012. Web. 19 Sep 2020.

Vancouver:

Barnabei MS. The Functional Role of Dystrophin in the Heart: Implications for Inherited and Non-Inherited Heart Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2012. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/2027.42/91611.

Council of Science Editors:

Barnabei MS. The Functional Role of Dystrophin in the Heart: Implications for Inherited and Non-Inherited Heart Disease. [Doctoral Dissertation]. University of Michigan; 2012. Available from: http://hdl.handle.net/2027.42/91611


University of Minnesota

4. Coffman, Christian. Calorimetric Determination of Dystrophin ABD1 Unfolding Energetics.

Degree: MS, Chemistry, 2018, University of Minnesota

 Muscular Dystrophy (MD) is a disease that effects the structural integrity of muscle cells. Studies have linked the Dystrophin protein to MD as the most… (more)

Subjects/Keywords: Calorimetry; Dystrophin; Muscular Dystrophy; Thermodynamics

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APA (6th Edition):

Coffman, C. (2018). Calorimetric Determination of Dystrophin ABD1 Unfolding Energetics. (Masters Thesis). University of Minnesota. Retrieved from http://hdl.handle.net/11299/202099

Chicago Manual of Style (16th Edition):

Coffman, Christian. “Calorimetric Determination of Dystrophin ABD1 Unfolding Energetics.” 2018. Masters Thesis, University of Minnesota. Accessed September 19, 2020. http://hdl.handle.net/11299/202099.

MLA Handbook (7th Edition):

Coffman, Christian. “Calorimetric Determination of Dystrophin ABD1 Unfolding Energetics.” 2018. Web. 19 Sep 2020.

Vancouver:

Coffman C. Calorimetric Determination of Dystrophin ABD1 Unfolding Energetics. [Internet] [Masters thesis]. University of Minnesota; 2018. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11299/202099.

Council of Science Editors:

Coffman C. Calorimetric Determination of Dystrophin ABD1 Unfolding Energetics. [Masters Thesis]. University of Minnesota; 2018. Available from: http://hdl.handle.net/11299/202099


The Ohio State University

5. Acharyya, Swarnali. Elucidating molecular mechanisms of muscle wasting in chronic diseases.

Degree: PhD, Integrated Biomedical Science, 2007, The Ohio State University

 Skeletal muscle represents one of the most abundant tissues in our body that primarily acts as a protein reservoir and maintains the structural framework. Skeletal… (more)

Subjects/Keywords: Biology, Genetics; skeletal muscle; dystrophin; regeneration; dystrophin; cachexia

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APA (6th Edition):

Acharyya, S. (2007). Elucidating molecular mechanisms of muscle wasting in chronic diseases. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1180096565

Chicago Manual of Style (16th Edition):

Acharyya, Swarnali. “Elucidating molecular mechanisms of muscle wasting in chronic diseases.” 2007. Doctoral Dissertation, The Ohio State University. Accessed September 19, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1180096565.

MLA Handbook (7th Edition):

Acharyya, Swarnali. “Elucidating molecular mechanisms of muscle wasting in chronic diseases.” 2007. Web. 19 Sep 2020.

Vancouver:

Acharyya S. Elucidating molecular mechanisms of muscle wasting in chronic diseases. [Internet] [Doctoral dissertation]. The Ohio State University; 2007. [cited 2020 Sep 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1180096565.

Council of Science Editors:

Acharyya S. Elucidating molecular mechanisms of muscle wasting in chronic diseases. [Doctoral Dissertation]. The Ohio State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1180096565


University of Ottawa

6. Al-Rewashdy, Hasanen. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .

Degree: 2014, University of Ottawa

 Duchenne Muscular Dystrophy (DMD) results from the absence of a functional dystrophin protein. Among its possible therapeutic options is the upregulation of dystrophin’s autosomal analogue,… (more)

Subjects/Keywords: Utrophin; Duchenne Muscular Dystrophy; Dystrophin; AMPK; AICAR

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APA (6th Edition):

Al-Rewashdy, H. (2014). Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/31470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al-Rewashdy, Hasanen. “Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .” 2014. Thesis, University of Ottawa. Accessed September 19, 2020. http://hdl.handle.net/10393/31470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al-Rewashdy, Hasanen. “Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation .” 2014. Web. 19 Sep 2020.

Vancouver:

Al-Rewashdy H. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . [Internet] [Thesis]. University of Ottawa; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10393/31470.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al-Rewashdy H. Determining the Contribution of Utrophin A Versus Other Components of the Slow, Oxidative Phenotype in the Beneficial Adaptations of Dystrophic Muscle Fibers Following AMPK Activation . [Thesis]. University of Ottawa; 2014. Available from: http://hdl.handle.net/10393/31470

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Royal Holloway, University of London

7. Parolaro, Nathalie. The molecular composition and localization of Dystrophin-Associated Protein Complexes and ε-sarcoglycan in mammalian brain.

Degree: PhD, 2012, Royal Holloway, University of London

 Duchenne muscular dystrophies (DMD) arise from mutations in the DMD gene. In the brain, the DMD gene product, dystrophin, localizes at the post-synaptic density (PSD)… (more)

Subjects/Keywords: Dystrophin-Associated Protein Complex; epsilon sarcoglycan

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APA (6th Edition):

Parolaro, N. (2012). The molecular composition and localization of Dystrophin-Associated Protein Complexes and ε-sarcoglycan in mammalian brain. (Doctoral Dissertation). Royal Holloway, University of London. Retrieved from https://pure.royalholloway.ac.uk/portal/en/publications/the-molecular-composition-and-localization-of-dystrophinassociated-protein-complexes-and-sarcoglycan-in-mammalian-brain(1e88612e-ba63-4d02-a9d9-c615660db639).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792125

Chicago Manual of Style (16th Edition):

Parolaro, Nathalie. “The molecular composition and localization of Dystrophin-Associated Protein Complexes and ε-sarcoglycan in mammalian brain.” 2012. Doctoral Dissertation, Royal Holloway, University of London. Accessed September 19, 2020. https://pure.royalholloway.ac.uk/portal/en/publications/the-molecular-composition-and-localization-of-dystrophinassociated-protein-complexes-and-sarcoglycan-in-mammalian-brain(1e88612e-ba63-4d02-a9d9-c615660db639).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792125.

MLA Handbook (7th Edition):

Parolaro, Nathalie. “The molecular composition and localization of Dystrophin-Associated Protein Complexes and ε-sarcoglycan in mammalian brain.” 2012. Web. 19 Sep 2020.

Vancouver:

Parolaro N. The molecular composition and localization of Dystrophin-Associated Protein Complexes and ε-sarcoglycan in mammalian brain. [Internet] [Doctoral dissertation]. Royal Holloway, University of London; 2012. [cited 2020 Sep 19]. Available from: https://pure.royalholloway.ac.uk/portal/en/publications/the-molecular-composition-and-localization-of-dystrophinassociated-protein-complexes-and-sarcoglycan-in-mammalian-brain(1e88612e-ba63-4d02-a9d9-c615660db639).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792125.

Council of Science Editors:

Parolaro N. The molecular composition and localization of Dystrophin-Associated Protein Complexes and ε-sarcoglycan in mammalian brain. [Doctoral Dissertation]. Royal Holloway, University of London; 2012. Available from: https://pure.royalholloway.ac.uk/portal/en/publications/the-molecular-composition-and-localization-of-dystrophinassociated-protein-complexes-and-sarcoglycan-in-mammalian-brain(1e88612e-ba63-4d02-a9d9-c615660db639).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.792125


Massey University

8. Muthu, Muralidharan. Structural and biochemical characterisation of utrophin and dystrophin spectrin repeat domains : submitted in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry, Massey University, Palmerston North, NZ .

Degree: 2012, Massey University

 Duchenne and Becker muscular dystrophies are muscle-wasting disorders caused by mutations in the X-linked dystrophin gene. Dystrophin is a large cytoskeletal protein belonging to the… (more)

Subjects/Keywords: Duchenne muscular dystrophy; Genetic aspects; Dystrophin; Spectrin

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APA (6th Edition):

Muthu, M. (2012). Structural and biochemical characterisation of utrophin and dystrophin spectrin repeat domains : submitted in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry, Massey University, Palmerston North, NZ . (Thesis). Massey University. Retrieved from http://hdl.handle.net/10179/4949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muthu, Muralidharan. “Structural and biochemical characterisation of utrophin and dystrophin spectrin repeat domains : submitted in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry, Massey University, Palmerston North, NZ .” 2012. Thesis, Massey University. Accessed September 19, 2020. http://hdl.handle.net/10179/4949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muthu, Muralidharan. “Structural and biochemical characterisation of utrophin and dystrophin spectrin repeat domains : submitted in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry, Massey University, Palmerston North, NZ .” 2012. Web. 19 Sep 2020.

Vancouver:

Muthu M. Structural and biochemical characterisation of utrophin and dystrophin spectrin repeat domains : submitted in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry, Massey University, Palmerston North, NZ . [Internet] [Thesis]. Massey University; 2012. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10179/4949.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muthu M. Structural and biochemical characterisation of utrophin and dystrophin spectrin repeat domains : submitted in partial fulfilment of the requirements for the degree of Doctor of Philosophy in Biochemistry, Massey University, Palmerston North, NZ . [Thesis]. Massey University; 2012. Available from: http://hdl.handle.net/10179/4949

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Campos, Érica Carolina. Isoproterenol induz a perda primária de distrofina: correlação com a injúria miocárdica.

Degree: Mestrado, Patologia, 2008, University of São Paulo

 Este estudo teve como objetivo avaliar as alterações do complexo de glicoproteínas associadas à distrofina que conferem estabilidade estrutural aos cardiomiócitos na isquemia miocárdica induzida… (more)

Subjects/Keywords: complexo de glicoproteínas; distrofina; dystrophin dystrophin-glycoprotein complex; ischemia; isoproterenol; isoproterenol; isquemia; miocitólise; myocytolysis

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APA (6th Edition):

Campos, . C. (2008). Isoproterenol induz a perda primária de distrofina: correlação com a injúria miocárdica. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17143/tde-18082008-123133/ ;

Chicago Manual of Style (16th Edition):

Campos, Érica Carolina. “Isoproterenol induz a perda primária de distrofina: correlação com a injúria miocárdica.” 2008. Masters Thesis, University of São Paulo. Accessed September 19, 2020. http://www.teses.usp.br/teses/disponiveis/17/17143/tde-18082008-123133/ ;.

MLA Handbook (7th Edition):

Campos, Érica Carolina. “Isoproterenol induz a perda primária de distrofina: correlação com a injúria miocárdica.” 2008. Web. 19 Sep 2020.

Vancouver:

Campos C. Isoproterenol induz a perda primária de distrofina: correlação com a injúria miocárdica. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2020 Sep 19]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-18082008-123133/ ;.

Council of Science Editors:

Campos C. Isoproterenol induz a perda primária de distrofina: correlação com a injúria miocárdica. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/17/17143/tde-18082008-123133/ ;


University of Lund

10. Murugesan, Vignesh. Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis.

Degree: 2017, University of Lund

 Atherosclerosis is the underlying cause for myocardal infarction, stroke and peripheral arterial disease, collectively referred to as cardiovascular diseases. These represent the major cause of… (more)

Subjects/Keywords: Cardiac and Cardiovascular Systems; Dystrophin glycoprotein complex, beta-sarcoglycan, dystrophin, Plaque, vascular smooth muscle cells, atherosclerosis, restenosis

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APA (6th Edition):

Murugesan, V. (2017). Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; https://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf

Chicago Manual of Style (16th Edition):

Murugesan, Vignesh. “Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis.” 2017. Doctoral Dissertation, University of Lund. Accessed September 19, 2020. https://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; https://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf.

MLA Handbook (7th Edition):

Murugesan, Vignesh. “Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis.” 2017. Web. 19 Sep 2020.

Vancouver:

Murugesan V. Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis. [Internet] [Doctoral dissertation]. University of Lund; 2017. [cited 2020 Sep 19]. Available from: https://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; https://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf.

Council of Science Editors:

Murugesan V. Challenging the mystique Connection. Deciphering cell-matrix interactions role in atherosclerosis and restenosis. [Doctoral Dissertation]. University of Lund; 2017. Available from: https://lup.lub.lu.se/record/e0975189-b9f3-4aa7-8b56-2b7a08b15e91 ; https://portal.research.lu.se/ws/files/29633052/Challenging_the_mystique_connection_Vignesh_Murugesan.pdf

11. Araújo, Karla Patrícia Cardoso. Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular.

Degree: PhD, Anatomia dos Animais Domésticos e Silvestres, 2009, University of São Paulo

A distrofia muscular progressiva no cão Golden Retriever (GRMD) é uma miopatia genética homóloga à Distrofia Muscular de Duchenne (DMD) que acomete humanos. A utilização… (more)

Subjects/Keywords: Bortezomib; Bortezomibe; Distrofina; Dystrophin; GRMD; GRMD model; Proteasome; Proteassoma

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APA (6th Edition):

Araújo, K. P. C. (2009). Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;

Chicago Manual of Style (16th Edition):

Araújo, Karla Patrícia Cardoso. “Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular.” 2009. Doctoral Dissertation, University of São Paulo. Accessed September 19, 2020. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;.

MLA Handbook (7th Edition):

Araújo, Karla Patrícia Cardoso. “Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular.” 2009. Web. 19 Sep 2020.

Vancouver:

Araújo KPC. Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2020 Sep 19]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;.

Council of Science Editors:

Araújo KPC. Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;

12. Esper, Greyson Vitor Zanatta. Terapia celular sob aquapuntura em modelos murinos para distrofia muscular de Duchenne.

Degree: PhD, Anatomia dos Animais Domésticos e Silvestres, 2012, University of São Paulo

 O camundongo mdx é um modelo animal muito utilizado para estudar a distrofia muscular degenerativa de Duchenne (DMD) e apesar de o modelo apresentar uma… (more)

Subjects/Keywords: acupuncture; acupuntura; células-tronco; distrofina; dystrophin; mdx; mdx; stem cells

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APA (6th Edition):

Esper, G. V. Z. (2012). Terapia celular sob aquapuntura em modelos murinos para distrofia muscular de Duchenne. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10132/tde-12062013-114220/ ;

Chicago Manual of Style (16th Edition):

Esper, Greyson Vitor Zanatta. “Terapia celular sob aquapuntura em modelos murinos para distrofia muscular de Duchenne.” 2012. Doctoral Dissertation, University of São Paulo. Accessed September 19, 2020. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-12062013-114220/ ;.

MLA Handbook (7th Edition):

Esper, Greyson Vitor Zanatta. “Terapia celular sob aquapuntura em modelos murinos para distrofia muscular de Duchenne.” 2012. Web. 19 Sep 2020.

Vancouver:

Esper GVZ. Terapia celular sob aquapuntura em modelos murinos para distrofia muscular de Duchenne. [Internet] [Doctoral dissertation]. University of São Paulo; 2012. [cited 2020 Sep 19]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-12062013-114220/ ;.

Council of Science Editors:

Esper GVZ. Terapia celular sob aquapuntura em modelos murinos para distrofia muscular de Duchenne. [Doctoral Dissertation]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-12062013-114220/ ;


Duke University

13. Robinson-Hamm, Jacqueline. Gene Editing for Duchenne Muscular Dystrophy .

Degree: 2018, Duke University

  Duchenne muscular dystrophy (DMD) is a muscle wasting disease that results from a lack of dystrophin protein, which is an essential musculoskeletal protein. Patients… (more)

Subjects/Keywords: Biomedical engineering; CRISPR-Cas9; Duchenne muscular dystrophy; Dystrophin; Gene editing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Robinson-Hamm, J. (2018). Gene Editing for Duchenne Muscular Dystrophy . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/17490

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robinson-Hamm, Jacqueline. “Gene Editing for Duchenne Muscular Dystrophy .” 2018. Thesis, Duke University. Accessed September 19, 2020. http://hdl.handle.net/10161/17490.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robinson-Hamm, Jacqueline. “Gene Editing for Duchenne Muscular Dystrophy .” 2018. Web. 19 Sep 2020.

Vancouver:

Robinson-Hamm J. Gene Editing for Duchenne Muscular Dystrophy . [Internet] [Thesis]. Duke University; 2018. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10161/17490.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robinson-Hamm J. Gene Editing for Duchenne Muscular Dystrophy . [Thesis]. Duke University; 2018. Available from: http://hdl.handle.net/10161/17490

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

14. Long, Chengzu. Prevention of Muscular Dystrophy in Mice by Gene Editing.

Degree: 2014, University of Texas Southwestern Medical Center

 Duchenne muscular dystrophy (DMD) is an inherited X-linked disease caused by mutations in the gene encoding dystrophin, a protein required for muscle fiber integrity. DMD… (more)

Subjects/Keywords: CRISPR-Cas Systems; Dystrophin; Gene Targeting; Muscular Dystrophy, Duchenne

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APA (6th Edition):

Long, C. (2014). Prevention of Muscular Dystrophy in Mice by Gene Editing. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/3953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Long, Chengzu. “Prevention of Muscular Dystrophy in Mice by Gene Editing.” 2014. Thesis, University of Texas Southwestern Medical Center. Accessed September 19, 2020. http://hdl.handle.net/2152.5/3953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Long, Chengzu. “Prevention of Muscular Dystrophy in Mice by Gene Editing.” 2014. Web. 19 Sep 2020.

Vancouver:

Long C. Prevention of Muscular Dystrophy in Mice by Gene Editing. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/2152.5/3953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Long C. Prevention of Muscular Dystrophy in Mice by Gene Editing. [Thesis]. University of Texas Southwestern Medical Center; 2014. Available from: http://hdl.handle.net/2152.5/3953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. LIM PEI PING, DOROTHY. Molecular characterization of point mutations in dystrophin gene and drug-induced readthroughs of stop codons.

Degree: 2007, National University of Singapore

Subjects/Keywords: dystrophin; point mutations; readthroughs

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APA (6th Edition):

LIM PEI PING, D. (2007). Molecular characterization of point mutations in dystrophin gene and drug-induced readthroughs of stop codons. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/23160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

LIM PEI PING, DOROTHY. “Molecular characterization of point mutations in dystrophin gene and drug-induced readthroughs of stop codons.” 2007. Thesis, National University of Singapore. Accessed September 19, 2020. http://scholarbank.nus.edu.sg/handle/10635/23160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

LIM PEI PING, DOROTHY. “Molecular characterization of point mutations in dystrophin gene and drug-induced readthroughs of stop codons.” 2007. Web. 19 Sep 2020.

Vancouver:

LIM PEI PING D. Molecular characterization of point mutations in dystrophin gene and drug-induced readthroughs of stop codons. [Internet] [Thesis]. National University of Singapore; 2007. [cited 2020 Sep 19]. Available from: http://scholarbank.nus.edu.sg/handle/10635/23160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

LIM PEI PING D. Molecular characterization of point mutations in dystrophin gene and drug-induced readthroughs of stop codons. [Thesis]. National University of Singapore; 2007. Available from: http://scholarbank.nus.edu.sg/handle/10635/23160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

16. Kueh, Sindy. CNS function in dystrophinopathies: an electrophysiological and molecular study in the cerebellum.

Degree: Medical Sciences, 2012, University of New South Wales

 DMD is caused by mutations in the dystrophin gene, which lead to the absence of the protein dystrophin or expression of a non-functional truncated protein… (more)

Subjects/Keywords: Mdx mouse; Dystrophin; Duchenne muscular dystrophy; Purkinje cell; Cerebellum

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APA (6th Edition):

Kueh, S. (2012). CNS function in dystrophinopathies: an electrophysiological and molecular study in the cerebellum. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/52279 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10951/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Kueh, Sindy. “CNS function in dystrophinopathies: an electrophysiological and molecular study in the cerebellum.” 2012. Doctoral Dissertation, University of New South Wales. Accessed September 19, 2020. http://handle.unsw.edu.au/1959.4/52279 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10951/SOURCE01?view=true.

MLA Handbook (7th Edition):

Kueh, Sindy. “CNS function in dystrophinopathies: an electrophysiological and molecular study in the cerebellum.” 2012. Web. 19 Sep 2020.

Vancouver:

Kueh S. CNS function in dystrophinopathies: an electrophysiological and molecular study in the cerebellum. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2020 Sep 19]. Available from: http://handle.unsw.edu.au/1959.4/52279 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10951/SOURCE01?view=true.

Council of Science Editors:

Kueh S. CNS function in dystrophinopathies: an electrophysiological and molecular study in the cerebellum. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/52279 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10951/SOURCE01?view=true


Brigham Young University

17. Benson, Brenda. Downhill Treadmill Running Does Not Induce Muscle Damage in FVB Mice.

Degree: MS, 2014, Brigham Young University

  Downhill treadmill running is a commonly used method to cause exercise-induced muscle damage, especially in rodents. Previous studies have evaluated which muscles in rats… (more)

Subjects/Keywords: downhill running; mice; muscle damage; Evans Blue dye; dystrophin; Exercise Science

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APA (6th Edition):

Benson, B. (2014). Downhill Treadmill Running Does Not Induce Muscle Damage in FVB Mice. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5258&context=etd

Chicago Manual of Style (16th Edition):

Benson, Brenda. “Downhill Treadmill Running Does Not Induce Muscle Damage in FVB Mice.” 2014. Masters Thesis, Brigham Young University. Accessed September 19, 2020. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5258&context=etd.

MLA Handbook (7th Edition):

Benson, Brenda. “Downhill Treadmill Running Does Not Induce Muscle Damage in FVB Mice.” 2014. Web. 19 Sep 2020.

Vancouver:

Benson B. Downhill Treadmill Running Does Not Induce Muscle Damage in FVB Mice. [Internet] [Masters thesis]. Brigham Young University; 2014. [cited 2020 Sep 19]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5258&context=etd.

Council of Science Editors:

Benson B. Downhill Treadmill Running Does Not Induce Muscle Damage in FVB Mice. [Masters Thesis]. Brigham Young University; 2014. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=5258&context=etd


Iowa State University

18. Spaulding, Hannah. Stimulation of PGC-1α to attenuate Duchenne muscular dystrophy disease pathology and activate autophagy.

Degree: 2019, Iowa State University

 Duchenne muscular dystrophy (DMD) is a fatal muscle wasting disease caused by the absence of functional dystrophin protein. Dystrophin-deficiency results in numerous cellular dysfunctions and… (more)

Subjects/Keywords: D2-mdx; DMD; dystrophin; mdx; quercetin; TFEB; Molecular Biology

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APA (6th Edition):

Spaulding, H. (2019). Stimulation of PGC-1α to attenuate Duchenne muscular dystrophy disease pathology and activate autophagy. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/etd/17326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Spaulding, Hannah. “Stimulation of PGC-1α to attenuate Duchenne muscular dystrophy disease pathology and activate autophagy.” 2019. Thesis, Iowa State University. Accessed September 19, 2020. https://lib.dr.iastate.edu/etd/17326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Spaulding, Hannah. “Stimulation of PGC-1α to attenuate Duchenne muscular dystrophy disease pathology and activate autophagy.” 2019. Web. 19 Sep 2020.

Vancouver:

Spaulding H. Stimulation of PGC-1α to attenuate Duchenne muscular dystrophy disease pathology and activate autophagy. [Internet] [Thesis]. Iowa State University; 2019. [cited 2020 Sep 19]. Available from: https://lib.dr.iastate.edu/etd/17326.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Spaulding H. Stimulation of PGC-1α to attenuate Duchenne muscular dystrophy disease pathology and activate autophagy. [Thesis]. Iowa State University; 2019. Available from: https://lib.dr.iastate.edu/etd/17326

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

19. Crain, Jonathan. Conformational changes in actinin-type actin binding domains: probing actin-induced structural dynamics in dystrophin and utrophin using EPR spectroscopy.

Degree: MS, Biochemistry, Molecular Bio, and Biophysics, 2014, University of Minnesota

 The underlying cause of Duchenne and Becker muscular dystrophies is a lack of functional dystrophin, a large multidomain protein. Dystrophin is normally expressed in muscle,… (more)

Subjects/Keywords: Deer; Dystrophin; Utrophin; Biochemistry, molecular bio, and biophysics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Crain, J. (2014). Conformational changes in actinin-type actin binding domains: probing actin-induced structural dynamics in dystrophin and utrophin using EPR spectroscopy. (Masters Thesis). University of Minnesota. Retrieved from http://hdl.handle.net/11299/170672

Chicago Manual of Style (16th Edition):

Crain, Jonathan. “Conformational changes in actinin-type actin binding domains: probing actin-induced structural dynamics in dystrophin and utrophin using EPR spectroscopy.” 2014. Masters Thesis, University of Minnesota. Accessed September 19, 2020. http://hdl.handle.net/11299/170672.

MLA Handbook (7th Edition):

Crain, Jonathan. “Conformational changes in actinin-type actin binding domains: probing actin-induced structural dynamics in dystrophin and utrophin using EPR spectroscopy.” 2014. Web. 19 Sep 2020.

Vancouver:

Crain J. Conformational changes in actinin-type actin binding domains: probing actin-induced structural dynamics in dystrophin and utrophin using EPR spectroscopy. [Internet] [Masters thesis]. University of Minnesota; 2014. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11299/170672.

Council of Science Editors:

Crain J. Conformational changes in actinin-type actin binding domains: probing actin-induced structural dynamics in dystrophin and utrophin using EPR spectroscopy. [Masters Thesis]. University of Minnesota; 2014. Available from: http://hdl.handle.net/11299/170672


University of Minnesota

20. McCourt, Jackie. Biophysical and functional consequences of sequence changes on dystrophin and utrophin.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2017, University of Minnesota

 Mutations in the DMD gene result in Duchenne (DMD) and Becker (BMD) muscular dystrophies. The DMD gene encodes the protein, dystrophin that is predominantly expressed… (more)

Subjects/Keywords: Atomic Force Microscopy; Dystrophin; Missense mutations; Muscular Dystrophy; Utrophin

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APA (6th Edition):

McCourt, J. (2017). Biophysical and functional consequences of sequence changes on dystrophin and utrophin. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/198377

Chicago Manual of Style (16th Edition):

McCourt, Jackie. “Biophysical and functional consequences of sequence changes on dystrophin and utrophin.” 2017. Doctoral Dissertation, University of Minnesota. Accessed September 19, 2020. http://hdl.handle.net/11299/198377.

MLA Handbook (7th Edition):

McCourt, Jackie. “Biophysical and functional consequences of sequence changes on dystrophin and utrophin.” 2017. Web. 19 Sep 2020.

Vancouver:

McCourt J. Biophysical and functional consequences of sequence changes on dystrophin and utrophin. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11299/198377.

Council of Science Editors:

McCourt J. Biophysical and functional consequences of sequence changes on dystrophin and utrophin. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/198377


University of Lund

21. Shami, Annelie. Fibromodulin and Dystrophin in Atherosclerosis: Novel roles for extracellular matrix in plaque development.

Degree: 2014, University of Lund

 Cardiovascular disease represents nearly half the cases of noncommunicable diseases worldwide and is the leading global cause of death. The main underlying cause is atherosclerosis,… (more)

Subjects/Keywords: Basic Medicine; Atherosclerosis; fibromodulin; dystrophin; carotid artery; lipid accumulation; restenosis; plaque

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APA (6th Edition):

Shami, A. (2014). Fibromodulin and Dystrophin in Atherosclerosis: Novel roles for extracellular matrix in plaque development. (Doctoral Dissertation). University of Lund. Retrieved from https://lup.lub.lu.se/record/4286040 ; https://portal.research.lu.se/ws/files/3720249/4286054.pdf

Chicago Manual of Style (16th Edition):

Shami, Annelie. “Fibromodulin and Dystrophin in Atherosclerosis: Novel roles for extracellular matrix in plaque development.” 2014. Doctoral Dissertation, University of Lund. Accessed September 19, 2020. https://lup.lub.lu.se/record/4286040 ; https://portal.research.lu.se/ws/files/3720249/4286054.pdf.

MLA Handbook (7th Edition):

Shami, Annelie. “Fibromodulin and Dystrophin in Atherosclerosis: Novel roles for extracellular matrix in plaque development.” 2014. Web. 19 Sep 2020.

Vancouver:

Shami A. Fibromodulin and Dystrophin in Atherosclerosis: Novel roles for extracellular matrix in plaque development. [Internet] [Doctoral dissertation]. University of Lund; 2014. [cited 2020 Sep 19]. Available from: https://lup.lub.lu.se/record/4286040 ; https://portal.research.lu.se/ws/files/3720249/4286054.pdf.

Council of Science Editors:

Shami A. Fibromodulin and Dystrophin in Atherosclerosis: Novel roles for extracellular matrix in plaque development. [Doctoral Dissertation]. University of Lund; 2014. Available from: https://lup.lub.lu.se/record/4286040 ; https://portal.research.lu.se/ws/files/3720249/4286054.pdf

22. Thorley, Matthew. Analysis of the dystrophin interactome : Analyse de l'interactome dystrophine.

Degree: Docteur es, Complexité du Vivant, 2016, Université Pierre et Marie Curie – Paris VI

Le but de ce projet était d'identifier de manière méthodique et standardisée les partenaires interagissant avec la protéine dystrophine dans les cellules musculaires squelettiques humaines… (more)

Subjects/Keywords: Interactome; Dystrophine; Transcriptome; Immortalisation; Protéomique; Spectrométrie de masse; QUICK; SILAC; Interactome; Dystrophin; Proteomics; 571.6

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APA (6th Edition):

Thorley, M. (2016). Analysis of the dystrophin interactome : Analyse de l'interactome dystrophine. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066619

Chicago Manual of Style (16th Edition):

Thorley, Matthew. “Analysis of the dystrophin interactome : Analyse de l'interactome dystrophine.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed September 19, 2020. http://www.theses.fr/2016PA066619.

MLA Handbook (7th Edition):

Thorley, Matthew. “Analysis of the dystrophin interactome : Analyse de l'interactome dystrophine.” 2016. Web. 19 Sep 2020.

Vancouver:

Thorley M. Analysis of the dystrophin interactome : Analyse de l'interactome dystrophine. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2016PA066619.

Council of Science Editors:

Thorley M. Analysis of the dystrophin interactome : Analyse de l'interactome dystrophine. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066619

23. Dos Santos Morais, Raphael. Interaction dystrophine-membrane : structure 3D de fragments de la dystrophine en présence de phospholipides : Dystrophin-membrane interaction : 3D structure of dystrophin fragments in the presence of phospholipids.

Degree: Docteur es, Biologie et sciences de la santé, 2017, Rennes 1

La dystrophine est une grande protéine membranaire périphérique qui assure un rôle de soutien du sarcolemme permettant aux cellules musculaires de résister aux stress mécaniques… (more)

Subjects/Keywords: Dystrophine; Interaction protéine/lipide; Bicelle; Sans/saxs; Dystrophin; Protein/lipid interaction; Bicelle; Sans/saxs

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APA (6th Edition):

Dos Santos Morais, R. (2017). Interaction dystrophine-membrane : structure 3D de fragments de la dystrophine en présence de phospholipides : Dystrophin-membrane interaction : 3D structure of dystrophin fragments in the presence of phospholipids. (Doctoral Dissertation). Rennes 1. Retrieved from http://www.theses.fr/2017REN1B062

Chicago Manual of Style (16th Edition):

Dos Santos Morais, Raphael. “Interaction dystrophine-membrane : structure 3D de fragments de la dystrophine en présence de phospholipides : Dystrophin-membrane interaction : 3D structure of dystrophin fragments in the presence of phospholipids.” 2017. Doctoral Dissertation, Rennes 1. Accessed September 19, 2020. http://www.theses.fr/2017REN1B062.

MLA Handbook (7th Edition):

Dos Santos Morais, Raphael. “Interaction dystrophine-membrane : structure 3D de fragments de la dystrophine en présence de phospholipides : Dystrophin-membrane interaction : 3D structure of dystrophin fragments in the presence of phospholipids.” 2017. Web. 19 Sep 2020.

Vancouver:

Dos Santos Morais R. Interaction dystrophine-membrane : structure 3D de fragments de la dystrophine en présence de phospholipides : Dystrophin-membrane interaction : 3D structure of dystrophin fragments in the presence of phospholipids. [Internet] [Doctoral dissertation]. Rennes 1; 2017. [cited 2020 Sep 19]. Available from: http://www.theses.fr/2017REN1B062.

Council of Science Editors:

Dos Santos Morais R. Interaction dystrophine-membrane : structure 3D de fragments de la dystrophine en présence de phospholipides : Dystrophin-membrane interaction : 3D structure of dystrophin fragments in the presence of phospholipids. [Doctoral Dissertation]. Rennes 1; 2017. Available from: http://www.theses.fr/2017REN1B062

24. Vogiatzakis, Nikolaos. Εφαρμογή νέων τεχνικών για την ανίχνευση μοριακών διαταραχών σε ασθενείς με δυστροφινοπάθειες χωρίς έλλειψη στο γονίδιο της δυστροφίνης.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

IntroductionMutation detection in the DMD gene has historically been challenging, duemainly to the large size of the gene and the fact that over 90% of… (more)

Subjects/Keywords: Δυστροφίνη; Νέες τεχνολογίες; Διάγνωση; Dystrophin; ECMA; MLPA; New methodologies; Diagnosis; DMD; BMD

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APA (6th Edition):

Vogiatzakis, N. (2013). Εφαρμογή νέων τεχνικών για την ανίχνευση μοριακών διαταραχών σε ασθενείς με δυστροφινοπάθειες χωρίς έλλειψη στο γονίδιο της δυστροφίνης. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/41094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vogiatzakis, Nikolaos. “Εφαρμογή νέων τεχνικών για την ανίχνευση μοριακών διαταραχών σε ασθενείς με δυστροφινοπάθειες χωρίς έλλειψη στο γονίδιο της δυστροφίνης.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed September 19, 2020. http://hdl.handle.net/10442/hedi/41094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vogiatzakis, Nikolaos. “Εφαρμογή νέων τεχνικών για την ανίχνευση μοριακών διαταραχών σε ασθενείς με δυστροφινοπάθειες χωρίς έλλειψη στο γονίδιο της δυστροφίνης.” 2013. Web. 19 Sep 2020.

Vancouver:

Vogiatzakis N. Εφαρμογή νέων τεχνικών για την ανίχνευση μοριακών διαταραχών σε ασθενείς με δυστροφινοπάθειες χωρίς έλλειψη στο γονίδιο της δυστροφίνης. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/10442/hedi/41094.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vogiatzakis N. Εφαρμογή νέων τεχνικών για την ανίχνευση μοριακών διαταραχών σε ασθενείς με δυστροφινοπάθειες χωρίς έλλειψη στο γονίδιο της δυστροφίνης. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/41094

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Jawaharlal Nehru University

25. Handa, Vaishali. Genetic and immunochemical study of dystrophin in human muscular dystrophy; -.

Degree: Life Sciences, 2000, Jawaharlal Nehru University

None

Bibliography p.121-152

Advisors/Committee Members: Upadhyaya, K C.

Subjects/Keywords: Life Science; Genetic; dystrophin; dystrophy; immunochemical

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APA (6th Edition):

Handa, V. (2000). Genetic and immunochemical study of dystrophin in human muscular dystrophy; -. (Thesis). Jawaharlal Nehru University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/18102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Handa, Vaishali. “Genetic and immunochemical study of dystrophin in human muscular dystrophy; -.” 2000. Thesis, Jawaharlal Nehru University. Accessed September 19, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/18102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Handa, Vaishali. “Genetic and immunochemical study of dystrophin in human muscular dystrophy; -.” 2000. Web. 19 Sep 2020.

Vancouver:

Handa V. Genetic and immunochemical study of dystrophin in human muscular dystrophy; -. [Internet] [Thesis]. Jawaharlal Nehru University; 2000. [cited 2020 Sep 19]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/18102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Handa V. Genetic and immunochemical study of dystrophin in human muscular dystrophy; -. [Thesis]. Jawaharlal Nehru University; 2000. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/18102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

26. Lekan, Jaimy Marie. Exercise-induced mechanisms of muscle adaptation in mdx mice.

Degree: PhD, Educational Studies: Hums, Science, Tech and Voc, 2004, The Ohio State University

 Duchenne Muscular Dystrophy (DMD) is a genetic disease that results from absence of the dystrophin protein, affects 1 in 3,500 male births, and leads to… (more)

Subjects/Keywords: dystrophin; swim training; Duchenne muscular dystrophy

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APA (6th Edition):

Lekan, J. M. (2004). Exercise-induced mechanisms of muscle adaptation in mdx mice. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1095372379

Chicago Manual of Style (16th Edition):

Lekan, Jaimy Marie. “Exercise-induced mechanisms of muscle adaptation in mdx mice.” 2004. Doctoral Dissertation, The Ohio State University. Accessed September 19, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=osu1095372379.

MLA Handbook (7th Edition):

Lekan, Jaimy Marie. “Exercise-induced mechanisms of muscle adaptation in mdx mice.” 2004. Web. 19 Sep 2020.

Vancouver:

Lekan JM. Exercise-induced mechanisms of muscle adaptation in mdx mice. [Internet] [Doctoral dissertation]. The Ohio State University; 2004. [cited 2020 Sep 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1095372379.

Council of Science Editors:

Lekan JM. Exercise-induced mechanisms of muscle adaptation in mdx mice. [Doctoral Dissertation]. The Ohio State University; 2004. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1095372379


University of New South Wales

27. Anderson, Jennifer Louise. Cerebellar synaptic plasticity in two animal models of muscular dystrophy.

Degree: Medical Sciences, 2008, University of New South Wales

 Duchenne muscular dystrophy (DMD) and congenital muscular dystrophy 1A (MDC1A) are the two most common forms of muscular dystrophy in humans, caused by mutations in… (more)

Subjects/Keywords: laminin alpha2; muscular dystrophy; dystrophin; cerebellum

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APA (6th Edition):

Anderson, J. L. (2008). Cerebellar synaptic plasticity in two animal models of muscular dystrophy. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Anderson, Jennifer Louise. “Cerebellar synaptic plasticity in two animal models of muscular dystrophy.” 2008. Doctoral Dissertation, University of New South Wales. Accessed September 19, 2020. http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true.

MLA Handbook (7th Edition):

Anderson, Jennifer Louise. “Cerebellar synaptic plasticity in two animal models of muscular dystrophy.” 2008. Web. 19 Sep 2020.

Vancouver:

Anderson JL. Cerebellar synaptic plasticity in two animal models of muscular dystrophy. [Internet] [Doctoral dissertation]. University of New South Wales; 2008. [cited 2020 Sep 19]. Available from: http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true.

Council of Science Editors:

Anderson JL. Cerebellar synaptic plasticity in two animal models of muscular dystrophy. [Doctoral Dissertation]. University of New South Wales; 2008. Available from: http://handle.unsw.edu.au/1959.4/43524 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:4998/SOURCE02?view=true


University of Washington

28. Ramos, Julian Nathan. Evaluating Properties of Dystrophin and Delivery Methods of rAAV Gene Therapy for Duchenne Muscular Dystrophy.

Degree: PhD, 2015, University of Washington

 Duchenne muscular dystrophy (DMD) is a recessive muscle wasting disease caused by a deleterious mutation in the gene encoding the dystrophin protein. Dystrophin is an… (more)

Subjects/Keywords: AAV; Duchenne; dystrophin; dystrophy; mdx; muscle; Molecular biology; Cellular biology; molecular and cellular biology

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APA (6th Edition):

Ramos, J. N. (2015). Evaluating Properties of Dystrophin and Delivery Methods of rAAV Gene Therapy for Duchenne Muscular Dystrophy. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/27534

Chicago Manual of Style (16th Edition):

Ramos, Julian Nathan. “Evaluating Properties of Dystrophin and Delivery Methods of rAAV Gene Therapy for Duchenne Muscular Dystrophy.” 2015. Doctoral Dissertation, University of Washington. Accessed September 19, 2020. http://hdl.handle.net/1773/27534.

MLA Handbook (7th Edition):

Ramos, Julian Nathan. “Evaluating Properties of Dystrophin and Delivery Methods of rAAV Gene Therapy for Duchenne Muscular Dystrophy.” 2015. Web. 19 Sep 2020.

Vancouver:

Ramos JN. Evaluating Properties of Dystrophin and Delivery Methods of rAAV Gene Therapy for Duchenne Muscular Dystrophy. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/1773/27534.

Council of Science Editors:

Ramos JN. Evaluating Properties of Dystrophin and Delivery Methods of rAAV Gene Therapy for Duchenne Muscular Dystrophy. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/27534


University of Minnesota

29. Henderson, Davin Michael. A biochemical and biophysical study of dystrophin.

Degree: PhD, Biochemistry, Molecular Bio, and Biophysics, 2011, University of Minnesota

 The primary role of a muscle cell is to contract and produce force that moves an organism. A vast majority of a muscle is made… (more)

Subjects/Keywords: Actin binding protein; Dystrophin; Muscular Dystrophy; Protein aggregation; Thermal stability; Biochemistry, Molecular Bio, and Biophysics

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APA (6th Edition):

Henderson, D. M. (2011). A biochemical and biophysical study of dystrophin. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/120598

Chicago Manual of Style (16th Edition):

Henderson, Davin Michael. “A biochemical and biophysical study of dystrophin.” 2011. Doctoral Dissertation, University of Minnesota. Accessed September 19, 2020. http://purl.umn.edu/120598.

MLA Handbook (7th Edition):

Henderson, Davin Michael. “A biochemical and biophysical study of dystrophin.” 2011. Web. 19 Sep 2020.

Vancouver:

Henderson DM. A biochemical and biophysical study of dystrophin. [Internet] [Doctoral dissertation]. University of Minnesota; 2011. [cited 2020 Sep 19]. Available from: http://purl.umn.edu/120598.

Council of Science Editors:

Henderson DM. A biochemical and biophysical study of dystrophin. [Doctoral Dissertation]. University of Minnesota; 2011. Available from: http://purl.umn.edu/120598


University of Minnesota

30. Belanto, Joseph John. A biochemical and molecular analysis of functional differences between dystrophin and utrophin.

Degree: PhD, Molecular, Cellular, Developmental Biology and Genetics, 2013, University of Minnesota

 The DMD gene encodes the protein dystrophin, a 427kD cytoplasmic protein responsible for linking the actin cytoskeleton to the extracellular matrix via the dystrophin-glycoprotein complex.… (more)

Subjects/Keywords: DMD; Duchenne muscular dystrophy; Dystrophin; mdx; Microtubules; Utrophin; Molecular, cellular, developmental biology and genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Belanto, J. J. (2013). A biochemical and molecular analysis of functional differences between dystrophin and utrophin. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/170101

Chicago Manual of Style (16th Edition):

Belanto, Joseph John. “A biochemical and molecular analysis of functional differences between dystrophin and utrophin.” 2013. Doctoral Dissertation, University of Minnesota. Accessed September 19, 2020. http://hdl.handle.net/11299/170101.

MLA Handbook (7th Edition):

Belanto, Joseph John. “A biochemical and molecular analysis of functional differences between dystrophin and utrophin.” 2013. Web. 19 Sep 2020.

Vancouver:

Belanto JJ. A biochemical and molecular analysis of functional differences between dystrophin and utrophin. [Internet] [Doctoral dissertation]. University of Minnesota; 2013. [cited 2020 Sep 19]. Available from: http://hdl.handle.net/11299/170101.

Council of Science Editors:

Belanto JJ. A biochemical and molecular analysis of functional differences between dystrophin and utrophin. [Doctoral Dissertation]. University of Minnesota; 2013. Available from: http://hdl.handle.net/11299/170101

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