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You searched for subject:(Disease Models Animal). Showing records 1 – 30 of 197 total matches.

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1. Iarocci, Andrew Lee. ErbB2 Expression Increases in the Spinal Cord of FVB/nJ Mice Following Infection with Theiler's Murine Encephalomyelitis Virus.

Degree: M.S. in Medical Microbiology and Immunology, Medical Microbiology and Immunology (graduate program), 2012, Creighton University

 The neuregulins are a family of epidermal growth factors necessary for nervous system development. Neuregulins signal via the epidermal growth factor family of tyrosine kinase… (more)

Subjects/Keywords: Neuregulins; Theilovirus; Disease Models, Animal

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APA (6th Edition):

Iarocci, A. L. (2012). ErbB2 Expression Increases in the Spinal Cord of FVB/nJ Mice Following Infection with Theiler's Murine Encephalomyelitis Virus. (Masters Thesis). Creighton University. Retrieved from http://hdl.handle.net/10504/31220

Chicago Manual of Style (16th Edition):

Iarocci, Andrew Lee. “ErbB2 Expression Increases in the Spinal Cord of FVB/nJ Mice Following Infection with Theiler's Murine Encephalomyelitis Virus.” 2012. Masters Thesis, Creighton University. Accessed March 07, 2021. http://hdl.handle.net/10504/31220.

MLA Handbook (7th Edition):

Iarocci, Andrew Lee. “ErbB2 Expression Increases in the Spinal Cord of FVB/nJ Mice Following Infection with Theiler's Murine Encephalomyelitis Virus.” 2012. Web. 07 Mar 2021.

Vancouver:

Iarocci AL. ErbB2 Expression Increases in the Spinal Cord of FVB/nJ Mice Following Infection with Theiler's Murine Encephalomyelitis Virus. [Internet] [Masters thesis]. Creighton University; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10504/31220.

Council of Science Editors:

Iarocci AL. ErbB2 Expression Increases in the Spinal Cord of FVB/nJ Mice Following Infection with Theiler's Murine Encephalomyelitis Virus. [Masters Thesis]. Creighton University; 2012. Available from: http://hdl.handle.net/10504/31220


University of Texas Southwestern Medical Center

2. Lancaster, Karen. Mechanisms Controlling Virulence Thresholds of Mixed Viral Populations and Identification of Novel Host Barriers to Poliovirus Neuropathogenesis.

Degree: 2012, University of Texas Southwestern Medical Center

 Neurotropic viruses comprise some of the worlds most widespread and deadly pathogens, including West Nile virus, rabies virus, and poliovirus. Poliovirus, as a model neurotropic… (more)

Subjects/Keywords: RNA Viruses; Poliomyelitis; Antibodies; Disease Models, Animal

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APA (6th Edition):

Lancaster, K. (2012). Mechanisms Controlling Virulence Thresholds of Mixed Viral Populations and Identification of Novel Host Barriers to Poliovirus Neuropathogenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1030

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lancaster, Karen. “Mechanisms Controlling Virulence Thresholds of Mixed Viral Populations and Identification of Novel Host Barriers to Poliovirus Neuropathogenesis.” 2012. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/1030.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lancaster, Karen. “Mechanisms Controlling Virulence Thresholds of Mixed Viral Populations and Identification of Novel Host Barriers to Poliovirus Neuropathogenesis.” 2012. Web. 07 Mar 2021.

Vancouver:

Lancaster K. Mechanisms Controlling Virulence Thresholds of Mixed Viral Populations and Identification of Novel Host Barriers to Poliovirus Neuropathogenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/1030.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lancaster K. Mechanisms Controlling Virulence Thresholds of Mixed Viral Populations and Identification of Novel Host Barriers to Poliovirus Neuropathogenesis. [Thesis]. University of Texas Southwestern Medical Center; 2012. Available from: http://hdl.handle.net/2152.5/1030

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

3. Shi, Xiaolei. Metabolomic Investigation of Melanoma Metastasis in a Patient-Derived Xenograft Mouse Model.

Degree: 2017, University of Texas Southwestern Medical Center

 Metabolic reprogramming is considered a major factor in cellular transformation and tumor initiation, but whether or how metabolism supports tumor metastasis remains an open question.… (more)

Subjects/Keywords: Disease Models, Animal; Melanoma; Metabolomics; Neoplasm Metastasis

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APA (6th Edition):

Shi, X. (2017). Metabolomic Investigation of Melanoma Metastasis in a Patient-Derived Xenograft Mouse Model. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/7193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shi, Xiaolei. “Metabolomic Investigation of Melanoma Metastasis in a Patient-Derived Xenograft Mouse Model.” 2017. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/7193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shi, Xiaolei. “Metabolomic Investigation of Melanoma Metastasis in a Patient-Derived Xenograft Mouse Model.” 2017. Web. 07 Mar 2021.

Vancouver:

Shi X. Metabolomic Investigation of Melanoma Metastasis in a Patient-Derived Xenograft Mouse Model. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/7193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shi X. Metabolomic Investigation of Melanoma Metastasis in a Patient-Derived Xenograft Mouse Model. [Thesis]. University of Texas Southwestern Medical Center; 2017. Available from: http://hdl.handle.net/2152.5/7193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

4. Seamans, Katherine Webster. Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins.

Degree: 2011, University of Texas Southwestern Medical Center

 Parkinson’s disease is a progressive neurodegenerative disease characterized by a loss of dopaminergic neurons in the substantia nigra. The cause of Parkinson’s disease remains uncertain,… (more)

Subjects/Keywords: Receptors, Dopamine; Parkinson Disease; Models, Animal

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APA (6th Edition):

Seamans, K. W. (2011). Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Seamans, Katherine Webster. “Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Seamans, Katherine Webster. “Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins.” 2011. Web. 07 Mar 2021.

Vancouver:

Seamans KW. Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Seamans KW. Behavioral, Neurochemical, and Histological Characterization of Mice Deficient for Parkin, DJ-1, and Antioxidant Proteins. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

5. Enns, Lea R. Individual Level Models of Infectious Disease Transmission For Animal Experiments.

Degree: MS, Department of Mathematics and Statistics, 2015, University of Guelph

 The control of infectious disease transmission among animals is crucial in minimizing risk to public health. Typically, such diseases are controlled with the help of… (more)

Subjects/Keywords: individual level models; infectious disease; animal experiments

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APA (6th Edition):

Enns, L. R. (2015). Individual Level Models of Infectious Disease Transmission For Animal Experiments. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9398

Chicago Manual of Style (16th Edition):

Enns, Lea R. “Individual Level Models of Infectious Disease Transmission For Animal Experiments.” 2015. Masters Thesis, University of Guelph. Accessed March 07, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9398.

MLA Handbook (7th Edition):

Enns, Lea R. “Individual Level Models of Infectious Disease Transmission For Animal Experiments.” 2015. Web. 07 Mar 2021.

Vancouver:

Enns LR. Individual Level Models of Infectious Disease Transmission For Animal Experiments. [Internet] [Masters thesis]. University of Guelph; 2015. [cited 2021 Mar 07]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9398.

Council of Science Editors:

Enns LR. Individual Level Models of Infectious Disease Transmission For Animal Experiments. [Masters Thesis]. University of Guelph; 2015. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/9398


Massey University

6. Russ, Anna-Lynne Elizabeth. Effects of dietary sheep, cow and goat milk solids on colitis in the interleukin-10 gene deficient mouse model of inflammatory bowel disease.

Degree: PhD, Physiology, 2013, Massey University

 Inflammatory Bowel Disease (IBD) is a group of chronic, immunologically-mediated gastrointestinal disorders resulting from interactions between environmental influences, host genetic susceptibility, and the intestinal microbiota.… (more)

Subjects/Keywords: Inflammatory bowel disease (IBD); Animal models

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APA (6th Edition):

Russ, A. E. (2013). Effects of dietary sheep, cow and goat milk solids on colitis in the interleukin-10 gene deficient mouse model of inflammatory bowel disease. (Doctoral Dissertation). Massey University. Retrieved from http://hdl.handle.net/10179/4326

Chicago Manual of Style (16th Edition):

Russ, Anna-Lynne Elizabeth. “Effects of dietary sheep, cow and goat milk solids on colitis in the interleukin-10 gene deficient mouse model of inflammatory bowel disease.” 2013. Doctoral Dissertation, Massey University. Accessed March 07, 2021. http://hdl.handle.net/10179/4326.

MLA Handbook (7th Edition):

Russ, Anna-Lynne Elizabeth. “Effects of dietary sheep, cow and goat milk solids on colitis in the interleukin-10 gene deficient mouse model of inflammatory bowel disease.” 2013. Web. 07 Mar 2021.

Vancouver:

Russ AE. Effects of dietary sheep, cow and goat milk solids on colitis in the interleukin-10 gene deficient mouse model of inflammatory bowel disease. [Internet] [Doctoral dissertation]. Massey University; 2013. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10179/4326.

Council of Science Editors:

Russ AE. Effects of dietary sheep, cow and goat milk solids on colitis in the interleukin-10 gene deficient mouse model of inflammatory bowel disease. [Doctoral Dissertation]. Massey University; 2013. Available from: http://hdl.handle.net/10179/4326


University of Sydney

7. El Massri, Nabil. The impact of photobiomodulation in animal models of Parkinson's disease and ageing .

Degree: 2018, University of Sydney

 The aim of this thesis was to explore whether photobiomodulation improved behaviour and offered neuroprotection in animal models of Parkinson's disease and in ageing. The… (more)

Subjects/Keywords: photobiomodulation; animal models; Parkinson's disease; ageing

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APA (6th Edition):

El Massri, N. (2018). The impact of photobiomodulation in animal models of Parkinson's disease and ageing . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/19702

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

El Massri, Nabil. “The impact of photobiomodulation in animal models of Parkinson's disease and ageing .” 2018. Thesis, University of Sydney. Accessed March 07, 2021. http://hdl.handle.net/2123/19702.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

El Massri, Nabil. “The impact of photobiomodulation in animal models of Parkinson's disease and ageing .” 2018. Web. 07 Mar 2021.

Vancouver:

El Massri N. The impact of photobiomodulation in animal models of Parkinson's disease and ageing . [Internet] [Thesis]. University of Sydney; 2018. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2123/19702.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

El Massri N. The impact of photobiomodulation in animal models of Parkinson's disease and ageing . [Thesis]. University of Sydney; 2018. Available from: http://hdl.handle.net/2123/19702

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

8. Matin, Nusrat. Soluble epoxide hydrolase inhibition improves dilation in parenchymal arterioles and prevents memory impairment inhypertensive rats with chronic cerebral hypoperfusion.

Degree: 2016, Michigan State University

Thesis Ph. D. Michigan State University. Pharmacology and Toxicology 2016

Parenchymal arterioles (PAs) control blood flow to the neurovascular unit that consists of the neurons,… (more)

Subjects/Keywords: Vascular dementia – Animal models; Cerebrovascular disease – Animal models; Hydrolases; Cerebrovascular disease – Treatment; Pharmacology

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APA (6th Edition):

Matin, N. (2016). Soluble epoxide hydrolase inhibition improves dilation in parenchymal arterioles and prevents memory impairment inhypertensive rats with chronic cerebral hypoperfusion. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:4208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Matin, Nusrat. “Soluble epoxide hydrolase inhibition improves dilation in parenchymal arterioles and prevents memory impairment inhypertensive rats with chronic cerebral hypoperfusion.” 2016. Thesis, Michigan State University. Accessed March 07, 2021. http://etd.lib.msu.edu/islandora/object/etd:4208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Matin, Nusrat. “Soluble epoxide hydrolase inhibition improves dilation in parenchymal arterioles and prevents memory impairment inhypertensive rats with chronic cerebral hypoperfusion.” 2016. Web. 07 Mar 2021.

Vancouver:

Matin N. Soluble epoxide hydrolase inhibition improves dilation in parenchymal arterioles and prevents memory impairment inhypertensive rats with chronic cerebral hypoperfusion. [Internet] [Thesis]. Michigan State University; 2016. [cited 2021 Mar 07]. Available from: http://etd.lib.msu.edu/islandora/object/etd:4208.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Matin N. Soluble epoxide hydrolase inhibition improves dilation in parenchymal arterioles and prevents memory impairment inhypertensive rats with chronic cerebral hypoperfusion. [Thesis]. Michigan State University; 2016. Available from: http://etd.lib.msu.edu/islandora/object/etd:4208

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

9. Marshall, Courtney Anne. D3 Receptor Agonism Attenuates Deficits Observed in Rodents with 6-OHDA-Induced Medial Forebrain Bundle Lesions.

Degree: 2018, Drexel University

Parkinson’s Disease (PD) is a neurodegenerative disorder associated with the progressive loss of dopaminergic neurons in the substantia nigra. This pathology impairs the dopaminergic nigrostriatal… (more)

Subjects/Keywords: Neurosciences; Dopamine – Receptors; Dopamine – Agonists; Parkinson's disease; Parkinson's disease – Animal models

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APA (6th Edition):

Marshall, C. A. (2018). D3 Receptor Agonism Attenuates Deficits Observed in Rodents with 6-OHDA-Induced Medial Forebrain Bundle Lesions. (Thesis). Drexel University. Retrieved from https://idea.library.drexel.edu/islandora/object/idea%3A8257

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marshall, Courtney Anne. “D3 Receptor Agonism Attenuates Deficits Observed in Rodents with 6-OHDA-Induced Medial Forebrain Bundle Lesions.” 2018. Thesis, Drexel University. Accessed March 07, 2021. https://idea.library.drexel.edu/islandora/object/idea%3A8257.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marshall, Courtney Anne. “D3 Receptor Agonism Attenuates Deficits Observed in Rodents with 6-OHDA-Induced Medial Forebrain Bundle Lesions.” 2018. Web. 07 Mar 2021.

Vancouver:

Marshall CA. D3 Receptor Agonism Attenuates Deficits Observed in Rodents with 6-OHDA-Induced Medial Forebrain Bundle Lesions. [Internet] [Thesis]. Drexel University; 2018. [cited 2021 Mar 07]. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A8257.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marshall CA. D3 Receptor Agonism Attenuates Deficits Observed in Rodents with 6-OHDA-Induced Medial Forebrain Bundle Lesions. [Thesis]. Drexel University; 2018. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A8257

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Macquarie University

10. Hunt, Louise Ruby. The effect of complex environmental enrichment on locomotor, memory and affective function following dopamine receptor antagonism in adult rats with an investigation into a model of early-stage Parkinson's disease in aged rats.

Degree: 2012, Macquarie University

1. Introduction  – 2. The effect of complex environmental enrichment on locomotor, memory and affective function following dopamine receptor antagonism in rats  – 3. Motor… (more)

Subjects/Keywords: Parkinson's disease; Parkinson's disease  – Research; Parkinson's disease  – Animal models; Cognition disorders; Movement disorders; environmental enrichment; cognition; rats; dopamine; Parkinson's disease

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APA (6th Edition):

Hunt, L. R. (2012). The effect of complex environmental enrichment on locomotor, memory and affective function following dopamine receptor antagonism in adult rats with an investigation into a model of early-stage Parkinson's disease in aged rats. (Thesis). Macquarie University. Retrieved from http://hdl.handle.net/1959.14/264953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hunt, Louise Ruby. “The effect of complex environmental enrichment on locomotor, memory and affective function following dopamine receptor antagonism in adult rats with an investigation into a model of early-stage Parkinson's disease in aged rats.” 2012. Thesis, Macquarie University. Accessed March 07, 2021. http://hdl.handle.net/1959.14/264953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hunt, Louise Ruby. “The effect of complex environmental enrichment on locomotor, memory and affective function following dopamine receptor antagonism in adult rats with an investigation into a model of early-stage Parkinson's disease in aged rats.” 2012. Web. 07 Mar 2021.

Vancouver:

Hunt LR. The effect of complex environmental enrichment on locomotor, memory and affective function following dopamine receptor antagonism in adult rats with an investigation into a model of early-stage Parkinson's disease in aged rats. [Internet] [Thesis]. Macquarie University; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1959.14/264953.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hunt LR. The effect of complex environmental enrichment on locomotor, memory and affective function following dopamine receptor antagonism in adult rats with an investigation into a model of early-stage Parkinson's disease in aged rats. [Thesis]. Macquarie University; 2012. Available from: http://hdl.handle.net/1959.14/264953

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Luitel, Krishna. Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis.

Degree: 2019, University of Texas Southwestern Medical Center

 Lung cancer accounts for more cancer-related deaths than any other cancer type among both men and women. The overall increase in radiation risk for human… (more)

Subjects/Keywords: Disease Models, Animal; Inflammation; Lung Neoplasms; Neoplasms, Radiation-Induced; Protons

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APA (6th Edition):

Luitel, K. (2019). Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/6625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luitel, Krishna. “Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis.” 2019. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/6625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luitel, Krishna. “Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis.” 2019. Web. 07 Mar 2021.

Vancouver:

Luitel K. Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2019. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/6625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luitel K. Investigating the Effects of Particle Radiation Exposure on Lung Carcinogenesis. [Thesis]. University of Texas Southwestern Medical Center; 2019. Available from: http://hdl.handle.net/2152.5/6625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

12. Krishnan, Vaishnav. Vulnerability and Resilience to Social Defeat: The Role of Neuroplasticity Within the Mesolimbic Dopamine Circuit.

Degree: 2010, University of Texas Southwestern Medical Center

 The pathophysiology of major depression and post-traumatic stress disorder are poorly understood. In particular, while stressful life events are an important cause of psychopathology, most… (more)

Subjects/Keywords: Psychiatry; Disease Models, Animal; Research

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APA (6th Edition):

Krishnan, V. (2010). Vulnerability and Resilience to Social Defeat: The Role of Neuroplasticity Within the Mesolimbic Dopamine Circuit. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Krishnan, Vaishnav. “Vulnerability and Resilience to Social Defeat: The Role of Neuroplasticity Within the Mesolimbic Dopamine Circuit.” 2010. Thesis, University of Texas Southwestern Medical Center. Accessed March 07, 2021. http://hdl.handle.net/2152.5/708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Krishnan, Vaishnav. “Vulnerability and Resilience to Social Defeat: The Role of Neuroplasticity Within the Mesolimbic Dopamine Circuit.” 2010. Web. 07 Mar 2021.

Vancouver:

Krishnan V. Vulnerability and Resilience to Social Defeat: The Role of Neuroplasticity Within the Mesolimbic Dopamine Circuit. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2010. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2152.5/708.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Krishnan V. Vulnerability and Resilience to Social Defeat: The Role of Neuroplasticity Within the Mesolimbic Dopamine Circuit. [Thesis]. University of Texas Southwestern Medical Center; 2010. Available from: http://hdl.handle.net/2152.5/708

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade Nova

13. Afonso, Ricardo Alexandre da Silva Santos. Sensibilidade à insulina pós-prandial: mecanismos fisiológicos e de activação e fisiopatologia na obesidade.

Degree: 2008, Universidade Nova

 RESUMO A acção hipoglicemiante da insulina é máxima no estado pós-prandial e depende da substância hepática sensibilizadora da insulina (HISS). Esta dissertação visa o estudo… (more)

Subjects/Keywords: Diabetes Mellitus - physiophatology; Obesity-complications ; Disease Models, Animal ; Insulin Resistance; Fisiologia

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APA (6th Edition):

Afonso, R. A. d. S. S. (2008). Sensibilidade à insulina pós-prandial: mecanismos fisiológicos e de activação e fisiopatologia na obesidade. (Thesis). Universidade Nova. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/5102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Afonso, Ricardo Alexandre da Silva Santos. “Sensibilidade à insulina pós-prandial: mecanismos fisiológicos e de activação e fisiopatologia na obesidade.” 2008. Thesis, Universidade Nova. Accessed March 07, 2021. http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/5102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Afonso, Ricardo Alexandre da Silva Santos. “Sensibilidade à insulina pós-prandial: mecanismos fisiológicos e de activação e fisiopatologia na obesidade.” 2008. Web. 07 Mar 2021.

Vancouver:

Afonso RAdSS. Sensibilidade à insulina pós-prandial: mecanismos fisiológicos e de activação e fisiopatologia na obesidade. [Internet] [Thesis]. Universidade Nova; 2008. [cited 2021 Mar 07]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/5102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Afonso RAdSS. Sensibilidade à insulina pós-prandial: mecanismos fisiológicos e de activação e fisiopatologia na obesidade. [Thesis]. Universidade Nova; 2008. Available from: http://www.rcaap.pt/detail.jsp?id=oai:run.unl.pt:10362/5102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

14. Bien-Ly, Nga. INVESTIGATING THE EFFECT OF C-TERMINAL TRUNCATED APOLIPOPROTEIN E4 ON AMYLOID-BETA-INDUCED NEURONAL AND BEHAVIORAL DEFICITS IN MICE.

Degree: Biomedical Sciences, 2011, University of California – San Francisco

 Among the common apolipoprotein (apo) E isoforms (apoE2, apoE3, and apoE4), apoE4 is the major known genetic risk factor for Alzheimer's disease (AD). Previous in… (more)

Subjects/Keywords: Neurosciences; Morphology; Behavioral Sciences; Alzheimer's disease; animal models; apoE; neurodegeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bien-Ly, N. (2011). INVESTIGATING THE EFFECT OF C-TERMINAL TRUNCATED APOLIPOPROTEIN E4 ON AMYLOID-BETA-INDUCED NEURONAL AND BEHAVIORAL DEFICITS IN MICE. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/5nn9k8p1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bien-Ly, Nga. “INVESTIGATING THE EFFECT OF C-TERMINAL TRUNCATED APOLIPOPROTEIN E4 ON AMYLOID-BETA-INDUCED NEURONAL AND BEHAVIORAL DEFICITS IN MICE.” 2011. Thesis, University of California – San Francisco. Accessed March 07, 2021. http://www.escholarship.org/uc/item/5nn9k8p1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bien-Ly, Nga. “INVESTIGATING THE EFFECT OF C-TERMINAL TRUNCATED APOLIPOPROTEIN E4 ON AMYLOID-BETA-INDUCED NEURONAL AND BEHAVIORAL DEFICITS IN MICE.” 2011. Web. 07 Mar 2021.

Vancouver:

Bien-Ly N. INVESTIGATING THE EFFECT OF C-TERMINAL TRUNCATED APOLIPOPROTEIN E4 ON AMYLOID-BETA-INDUCED NEURONAL AND BEHAVIORAL DEFICITS IN MICE. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2021 Mar 07]. Available from: http://www.escholarship.org/uc/item/5nn9k8p1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bien-Ly N. INVESTIGATING THE EFFECT OF C-TERMINAL TRUNCATED APOLIPOPROTEIN E4 ON AMYLOID-BETA-INDUCED NEURONAL AND BEHAVIORAL DEFICITS IN MICE. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/5nn9k8p1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

15. Naughton, Carol. Development and characterisation of novel gene-environment interaction rat models of Parkinson's disease .

Degree: 2016, National University of Ireland – Galway

 Despite its discovery almost 60 years ago, levodopa remains the most effective therapy for the treatment of the motor symptoms associated with Parkinson’s disease. However,… (more)

Subjects/Keywords: Parkinson's Disease; Gene-environment interactions; Animal models; Pharmacology and Therapeutics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Naughton, C. (2016). Development and characterisation of novel gene-environment interaction rat models of Parkinson's disease . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Naughton, Carol. “Development and characterisation of novel gene-environment interaction rat models of Parkinson's disease .” 2016. Thesis, National University of Ireland – Galway. Accessed March 07, 2021. http://hdl.handle.net/10379/5984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Naughton, Carol. “Development and characterisation of novel gene-environment interaction rat models of Parkinson's disease .” 2016. Web. 07 Mar 2021.

Vancouver:

Naughton C. Development and characterisation of novel gene-environment interaction rat models of Parkinson's disease . [Internet] [Thesis]. National University of Ireland – Galway; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10379/5984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Naughton C. Development and characterisation of novel gene-environment interaction rat models of Parkinson's disease . [Thesis]. National University of Ireland – Galway; 2016. Available from: http://hdl.handle.net/10379/5984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Staunton, Lisa. Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease.

Degree: 2011, RIAN

 Skeletal muscle provides an organism with a means of reacting to its environments. It is a complex and versatile tissue that is capable of change… (more)

Subjects/Keywords: Biology; Proteomic Profiling; Animal Models; Myotonia; Motor Neuron Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Staunton, L. (2011). Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease. (Thesis). RIAN. Retrieved from http://eprints.maynoothuniversity.ie/2862/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Staunton, Lisa. “Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease.” 2011. Thesis, RIAN. Accessed March 07, 2021. http://eprints.maynoothuniversity.ie/2862/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Staunton, Lisa. “Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease.” 2011. Web. 07 Mar 2021.

Vancouver:

Staunton L. Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease. [Internet] [Thesis]. RIAN; 2011. [cited 2021 Mar 07]. Available from: http://eprints.maynoothuniversity.ie/2862/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Staunton L. Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease. [Thesis]. RIAN; 2011. Available from: http://eprints.maynoothuniversity.ie/2862/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

17. Aloi, Macarena Sofia. miR-155 expression modulates microglia functions in vitro and in the APP/PS1 mouse model of Alzheimer’s disease.

Degree: PhD, 2019, University of Washington

 Alzheimer’s Disease (AD) is characterized by the accumulation of extracellular Amyloid-β (Aβ) as well as both CNS and systemic inflammation. Microglia, myeloid cells resident to… (more)

Subjects/Keywords: Alzheimer's Disease; Animal models; Microglia; microRNA; Seizures; Neurosciences; Pathology; Pathology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aloi, M. S. (2019). miR-155 expression modulates microglia functions in vitro and in the APP/PS1 mouse model of Alzheimer’s disease. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/44877

Chicago Manual of Style (16th Edition):

Aloi, Macarena Sofia. “miR-155 expression modulates microglia functions in vitro and in the APP/PS1 mouse model of Alzheimer’s disease.” 2019. Doctoral Dissertation, University of Washington. Accessed March 07, 2021. http://hdl.handle.net/1773/44877.

MLA Handbook (7th Edition):

Aloi, Macarena Sofia. “miR-155 expression modulates microglia functions in vitro and in the APP/PS1 mouse model of Alzheimer’s disease.” 2019. Web. 07 Mar 2021.

Vancouver:

Aloi MS. miR-155 expression modulates microglia functions in vitro and in the APP/PS1 mouse model of Alzheimer’s disease. [Internet] [Doctoral dissertation]. University of Washington; 2019. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1773/44877.

Council of Science Editors:

Aloi MS. miR-155 expression modulates microglia functions in vitro and in the APP/PS1 mouse model of Alzheimer’s disease. [Doctoral Dissertation]. University of Washington; 2019. Available from: http://hdl.handle.net/1773/44877


University of Washington

18. Cao, Jessica. Therapeutic potential of the endocannabinoid system in a novel mouse model of Huntington’s Disease.

Degree: PhD, 2018, University of Washington

 Huntington’s Disease is a fatal, inherited neurodegenerative disease characterized by profound disturbances of the basal ganglia and gradual deterioration of motor and cognitive functions with… (more)

Subjects/Keywords: ABHD6; animal models; endocannbinoids; Huntington's Disease; polyglutamine; Pharmacology; Neurosciences; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cao, J. (2018). Therapeutic potential of the endocannabinoid system in a novel mouse model of Huntington’s Disease. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/41839

Chicago Manual of Style (16th Edition):

Cao, Jessica. “Therapeutic potential of the endocannabinoid system in a novel mouse model of Huntington’s Disease.” 2018. Doctoral Dissertation, University of Washington. Accessed March 07, 2021. http://hdl.handle.net/1773/41839.

MLA Handbook (7th Edition):

Cao, Jessica. “Therapeutic potential of the endocannabinoid system in a novel mouse model of Huntington’s Disease.” 2018. Web. 07 Mar 2021.

Vancouver:

Cao J. Therapeutic potential of the endocannabinoid system in a novel mouse model of Huntington’s Disease. [Internet] [Doctoral dissertation]. University of Washington; 2018. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1773/41839.

Council of Science Editors:

Cao J. Therapeutic potential of the endocannabinoid system in a novel mouse model of Huntington’s Disease. [Doctoral Dissertation]. University of Washington; 2018. Available from: http://hdl.handle.net/1773/41839

19. Staunton, Lisa. Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease.

Degree: 2011, RIAN

 Skeletal muscle provides an organism with a means of reacting to its environments. It is a complex and versatile tissue that is capable of change… (more)

Subjects/Keywords: Biology; Proteomic Profiling; Animal Models; Myotonia; Motor Neuron Disease

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Staunton, L. (2011). Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease. (Thesis). RIAN. Retrieved from http://mural.maynoothuniversity.ie/2862/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Staunton, Lisa. “Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease.” 2011. Thesis, RIAN. Accessed March 07, 2021. http://mural.maynoothuniversity.ie/2862/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Staunton, Lisa. “Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease.” 2011. Web. 07 Mar 2021.

Vancouver:

Staunton L. Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease. [Internet] [Thesis]. RIAN; 2011. [cited 2021 Mar 07]. Available from: http://mural.maynoothuniversity.ie/2862/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Staunton L. Proteomic Profiling of Animal Models of Myotonia and Motor Neuron Disease. [Thesis]. RIAN; 2011. Available from: http://mural.maynoothuniversity.ie/2862/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Holland, Ashling. Proteomic Profiling of Animal Models of Motor Neuron Disease and Muscular Dystrophy.

Degree: 2015, RIAN

 Proteomic profiling plays a decisive role in the identification of novel biomarkers of neuromuscular disorders and the elucidation of new pathobiochemical mechanisms that underlie these… (more)

Subjects/Keywords: Proteomic Profiling; Animal Models; Motor Neuron Disease; Muscular Dystrophy

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Holland, A. (2015). Proteomic Profiling of Animal Models of Motor Neuron Disease and Muscular Dystrophy. (Thesis). RIAN. Retrieved from http://mural.maynoothuniversity.ie/6767/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Holland, Ashling. “Proteomic Profiling of Animal Models of Motor Neuron Disease and Muscular Dystrophy.” 2015. Thesis, RIAN. Accessed March 07, 2021. http://mural.maynoothuniversity.ie/6767/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Holland, Ashling. “Proteomic Profiling of Animal Models of Motor Neuron Disease and Muscular Dystrophy.” 2015. Web. 07 Mar 2021.

Vancouver:

Holland A. Proteomic Profiling of Animal Models of Motor Neuron Disease and Muscular Dystrophy. [Internet] [Thesis]. RIAN; 2015. [cited 2021 Mar 07]. Available from: http://mural.maynoothuniversity.ie/6767/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Holland A. Proteomic Profiling of Animal Models of Motor Neuron Disease and Muscular Dystrophy. [Thesis]. RIAN; 2015. Available from: http://mural.maynoothuniversity.ie/6767/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Maranis, Sotirios. Μελέτη της ανάπτυξης φαρμακοεπαγώμενων υπερκινησιών σε πειραματικά μοντέλα νόσου Πάρκισον.

Degree: 2017, University of Ioannina; Πανεπιστήμιο Ιωαννίνων

Long-term levodopa replacement therapy in Parkinson’s disease is confounded by abnormal involuntary movements, known as Levodopa Induced Dyskinesia (LID). Dysfunctional glutamatergic neurotransmission has been implicated… (more)

Subjects/Keywords: Νόσος Πάρκινσον; Υπερκινησία; Πειραματικά μοντέλα; Parkinson's disease; Dyskinesia; Animal models

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APA (6th Edition):

Maranis, S. (2017). Μελέτη της ανάπτυξης φαρμακοεπαγώμενων υπερκινησιών σε πειραματικά μοντέλα νόσου Πάρκισον. (Thesis). University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Retrieved from http://hdl.handle.net/10442/hedi/45014

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maranis, Sotirios. “Μελέτη της ανάπτυξης φαρμακοεπαγώμενων υπερκινησιών σε πειραματικά μοντέλα νόσου Πάρκισον.” 2017. Thesis, University of Ioannina; Πανεπιστήμιο Ιωαννίνων. Accessed March 07, 2021. http://hdl.handle.net/10442/hedi/45014.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maranis, Sotirios. “Μελέτη της ανάπτυξης φαρμακοεπαγώμενων υπερκινησιών σε πειραματικά μοντέλα νόσου Πάρκισον.” 2017. Web. 07 Mar 2021.

Vancouver:

Maranis S. Μελέτη της ανάπτυξης φαρμακοεπαγώμενων υπερκινησιών σε πειραματικά μοντέλα νόσου Πάρκισον. [Internet] [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10442/hedi/45014.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maranis S. Μελέτη της ανάπτυξης φαρμακοεπαγώμενων υπερκινησιών σε πειραματικά μοντέλα νόσου Πάρκισον. [Thesis]. University of Ioannina; Πανεπιστήμιο Ιωαννίνων; 2017. Available from: http://hdl.handle.net/10442/hedi/45014

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Missouri – Columbia

22. Villalón Landeros, Eric, 1986-. Peripheral nervous system alterations in disease and injury.

Degree: 2016, University of Missouri – Columbia

 Charcot-Marie-Tooth (CMT) is the most common inherited neuropathy of the PNS affecting approximately 2.8 million people. CMT type 2E, an axonal form of CMT, has… (more)

Subjects/Keywords: Charcot-Marie-Tooth disease; Diseases  – Animal models; Nerves, Peripheral; Neuropathy

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APA (6th Edition):

Villalón Landeros, Eric, 1. (2016). Peripheral nervous system alterations in disease and injury. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/59789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Villalón Landeros, Eric, 1986-. “Peripheral nervous system alterations in disease and injury.” 2016. Thesis, University of Missouri – Columbia. Accessed March 07, 2021. http://hdl.handle.net/10355/59789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Villalón Landeros, Eric, 1986-. “Peripheral nervous system alterations in disease and injury.” 2016. Web. 07 Mar 2021.

Vancouver:

Villalón Landeros, Eric 1. Peripheral nervous system alterations in disease and injury. [Internet] [Thesis]. University of Missouri – Columbia; 2016. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10355/59789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Villalón Landeros, Eric 1. Peripheral nervous system alterations in disease and injury. [Thesis]. University of Missouri – Columbia; 2016. Available from: http://hdl.handle.net/10355/59789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Western Washington University

23. Minnig, Shawn. Behavioral Characterization of a Knock-in Mouse Model of Huntington's Disease.

Degree: MS, Psychology, 2016, Western Washington University

  Huntington’s disease (HD) is a progressive, fatal neurodegenerative disease caused by an inherited CAG expansion on the Huntingtin (HTT) gene resulting in cognitive, affective,… (more)

Subjects/Keywords: Psychology; Huntington's disease – Animal models; Huntington's disease – Research; Mice as laboratory animals – Behavior; masters theses

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APA (6th Edition):

Minnig, S. (2016). Behavioral Characterization of a Knock-in Mouse Model of Huntington's Disease. (Masters Thesis). Western Washington University. Retrieved from https://doi.org/10.25710/f5yg-9v24 ; https://cedar.wwu.edu/wwuet/479

Chicago Manual of Style (16th Edition):

Minnig, Shawn. “Behavioral Characterization of a Knock-in Mouse Model of Huntington's Disease.” 2016. Masters Thesis, Western Washington University. Accessed March 07, 2021. https://doi.org/10.25710/f5yg-9v24 ; https://cedar.wwu.edu/wwuet/479.

MLA Handbook (7th Edition):

Minnig, Shawn. “Behavioral Characterization of a Knock-in Mouse Model of Huntington's Disease.” 2016. Web. 07 Mar 2021.

Vancouver:

Minnig S. Behavioral Characterization of a Knock-in Mouse Model of Huntington's Disease. [Internet] [Masters thesis]. Western Washington University; 2016. [cited 2021 Mar 07]. Available from: https://doi.org/10.25710/f5yg-9v24 ; https://cedar.wwu.edu/wwuet/479.

Council of Science Editors:

Minnig S. Behavioral Characterization of a Knock-in Mouse Model of Huntington's Disease. [Masters Thesis]. Western Washington University; 2016. Available from: https://doi.org/10.25710/f5yg-9v24 ; https://cedar.wwu.edu/wwuet/479

24. Francisco Edson Ximenes Gomes Pereira. Development and a form of endometriose validation subcutaneous under study for possible mechanisms of rats phatophysiological and drug effect.

Degree: Master, 2013, Universidade Federal do Ceará

Endometriosis is defined as the presence of endometrial tissue (gland and stroma) outside the uterus. The objective of this study was to design a model… (more)

Subjects/Keywords: CIRURGIA; Endometriose; Modelos Animais; Modelos Animais de DoenÃas; Ratos; Endometriosis; Models, Animal; Disease Models, Animal; Rats

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APA (6th Edition):

Pereira, F. E. X. G. (2013). Development and a form of endometriose validation subcutaneous under study for possible mechanisms of rats phatophysiological and drug effect. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=14411 ;

Chicago Manual of Style (16th Edition):

Pereira, Francisco Edson Ximenes Gomes. “Development and a form of endometriose validation subcutaneous under study for possible mechanisms of rats phatophysiological and drug effect.” 2013. Masters Thesis, Universidade Federal do Ceará. Accessed March 07, 2021. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=14411 ;.

MLA Handbook (7th Edition):

Pereira, Francisco Edson Ximenes Gomes. “Development and a form of endometriose validation subcutaneous under study for possible mechanisms of rats phatophysiological and drug effect.” 2013. Web. 07 Mar 2021.

Vancouver:

Pereira FEXG. Development and a form of endometriose validation subcutaneous under study for possible mechanisms of rats phatophysiological and drug effect. [Internet] [Masters thesis]. Universidade Federal do Ceará 2013. [cited 2021 Mar 07]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=14411 ;.

Council of Science Editors:

Pereira FEXG. Development and a form of endometriose validation subcutaneous under study for possible mechanisms of rats phatophysiological and drug effect. [Masters Thesis]. Universidade Federal do Ceará 2013. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=14411 ;


Northeastern University

25. Barchet, Thomas Matthew. Novel MRI approaches to the detection and evaluation of neurodegenerative disease.

Degree: PhD, School of Pharmacy, 2015, Northeastern University

 This dissertation describes a novel approach to noninvasively evaluating animal models of neurodegeneration using magnetic resonance imaging. This work includes studies of three separate models(more)

Subjects/Keywords: biomarkers; MRI; neurodegeneration; Nervous system; Degeneration; Animal models; Brain; Magnetic resonance imaging; Brain; Wounds and injuries; Animal models; Alzheimer's disease; Animal models; Biochemical markers; Diffusion tensor imaging

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barchet, T. M. (2015). Novel MRI approaches to the detection and evaluation of neurodegenerative disease. (Doctoral Dissertation). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20196171

Chicago Manual of Style (16th Edition):

Barchet, Thomas Matthew. “Novel MRI approaches to the detection and evaluation of neurodegenerative disease.” 2015. Doctoral Dissertation, Northeastern University. Accessed March 07, 2021. http://hdl.handle.net/2047/D20196171.

MLA Handbook (7th Edition):

Barchet, Thomas Matthew. “Novel MRI approaches to the detection and evaluation of neurodegenerative disease.” 2015. Web. 07 Mar 2021.

Vancouver:

Barchet TM. Novel MRI approaches to the detection and evaluation of neurodegenerative disease. [Internet] [Doctoral dissertation]. Northeastern University; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2047/D20196171.

Council of Science Editors:

Barchet TM. Novel MRI approaches to the detection and evaluation of neurodegenerative disease. [Doctoral Dissertation]. Northeastern University; 2015. Available from: http://hdl.handle.net/2047/D20196171


University of Zambia

26. Muyenga, Tumelo. The effect of Kigelia Africana fruit extract on blood glucose in diabetes induced mice .

Degree: 2015, University of Zambia

 Objective: To determine the effect Kigelia africana fruit extract has on blood glucose levels of diabetes mice and its phytochemical profile Specific objectives: 1. To… (more)

Subjects/Keywords: Diabetes Mellitus; Diabetes-Animal Model; Mice-Effect of drugs on; Disease models, Animals; Mice

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APA (6th Edition):

Muyenga, T. (2015). The effect of Kigelia Africana fruit extract on blood glucose in diabetes induced mice . (Thesis). University of Zambia. Retrieved from http://dspace.unza.zm:8080/xmlui/handle/123456789/4401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muyenga, Tumelo. “The effect of Kigelia Africana fruit extract on blood glucose in diabetes induced mice .” 2015. Thesis, University of Zambia. Accessed March 07, 2021. http://dspace.unza.zm:8080/xmlui/handle/123456789/4401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muyenga, Tumelo. “The effect of Kigelia Africana fruit extract on blood glucose in diabetes induced mice .” 2015. Web. 07 Mar 2021.

Vancouver:

Muyenga T. The effect of Kigelia Africana fruit extract on blood glucose in diabetes induced mice . [Internet] [Thesis]. University of Zambia; 2015. [cited 2021 Mar 07]. Available from: http://dspace.unza.zm:8080/xmlui/handle/123456789/4401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muyenga T. The effect of Kigelia Africana fruit extract on blood glucose in diabetes induced mice . [Thesis]. University of Zambia; 2015. Available from: http://dspace.unza.zm:8080/xmlui/handle/123456789/4401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Ioshimoto, Gabriela Lourençon. Estudo da eletrorretinografia do camundongo modelo de alzheimer (3xTg-AD).

Degree: Mestrado, Neurociências e Comportamento, 2011, University of São Paulo

Objetivo: Avaliar eletrofisiologicamente a função da retina do camundongo modelo de Alzheimer (3xTg-AD) comparando com seu controle (b6;129-PS1) em um estudo longitudinal com seis idades… (more)

Subjects/Keywords: Alzheimer disease; Animal models; Camundongos; Doença de Alzheimer; Electroretinography; Eletrorretinografia; Mice; Modelos animais

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APA (6th Edition):

Ioshimoto, G. L. (2011). Estudo da eletrorretinografia do camundongo modelo de alzheimer (3xTg-AD). (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/47/47135/tde-28042011-155725/ ;

Chicago Manual of Style (16th Edition):

Ioshimoto, Gabriela Lourençon. “Estudo da eletrorretinografia do camundongo modelo de alzheimer (3xTg-AD).” 2011. Masters Thesis, University of São Paulo. Accessed March 07, 2021. http://www.teses.usp.br/teses/disponiveis/47/47135/tde-28042011-155725/ ;.

MLA Handbook (7th Edition):

Ioshimoto, Gabriela Lourençon. “Estudo da eletrorretinografia do camundongo modelo de alzheimer (3xTg-AD).” 2011. Web. 07 Mar 2021.

Vancouver:

Ioshimoto GL. Estudo da eletrorretinografia do camundongo modelo de alzheimer (3xTg-AD). [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2021 Mar 07]. Available from: http://www.teses.usp.br/teses/disponiveis/47/47135/tde-28042011-155725/ ;.

Council of Science Editors:

Ioshimoto GL. Estudo da eletrorretinografia do camundongo modelo de alzheimer (3xTg-AD). [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/47/47135/tde-28042011-155725/ ;


Edith Cowan University

28. Avdesh, Avdesh. Pharmacological modulation of learning and memory in mice and zebrafish: Implications for Alzheimer’s disease animal models.

Degree: 2013, Edith Cowan University

 Learning and memory impairments are the clinical manifestations of several neurodegenerative disorders including Alzheimer’s disease (AD), the most common form of dementia affecting millions of… (more)

Subjects/Keywords: Learning and memory; mice; zebrafish; Alzheimer's disease; animal models; pharmacological modulation; Medical Sciences

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APA (6th Edition):

Avdesh, A. (2013). Pharmacological modulation of learning and memory in mice and zebrafish: Implications for Alzheimer’s disease animal models. (Thesis). Edith Cowan University. Retrieved from https://ro.ecu.edu.au/theses/570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Avdesh, Avdesh. “Pharmacological modulation of learning and memory in mice and zebrafish: Implications for Alzheimer’s disease animal models.” 2013. Thesis, Edith Cowan University. Accessed March 07, 2021. https://ro.ecu.edu.au/theses/570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Avdesh, Avdesh. “Pharmacological modulation of learning and memory in mice and zebrafish: Implications for Alzheimer’s disease animal models.” 2013. Web. 07 Mar 2021.

Vancouver:

Avdesh A. Pharmacological modulation of learning and memory in mice and zebrafish: Implications for Alzheimer’s disease animal models. [Internet] [Thesis]. Edith Cowan University; 2013. [cited 2021 Mar 07]. Available from: https://ro.ecu.edu.au/theses/570.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Avdesh A. Pharmacological modulation of learning and memory in mice and zebrafish: Implications for Alzheimer’s disease animal models. [Thesis]. Edith Cowan University; 2013. Available from: https://ro.ecu.edu.au/theses/570

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

29. Lai-Zhao, Yue. The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis.

Degree: 2020, University of Western Ontario

 In osteoarthritis (OA), synovitis is associated with symptom severity. As synovium secretes both catabolic and anabolic factors into the joint, the impact of synovitis in… (more)

Subjects/Keywords: Animal models of disease; chondrocyte; inflammation; liposomal drug delivery; STAT inhibitors; tissue culture.; Musculoskeletal Diseases

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APA (6th Edition):

Lai-Zhao, Y. (2020). The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/7075

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lai-Zhao, Yue. “The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis.” 2020. Thesis, University of Western Ontario. Accessed March 07, 2021. https://ir.lib.uwo.ca/etd/7075.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lai-Zhao, Yue. “The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis.” 2020. Web. 07 Mar 2021.

Vancouver:

Lai-Zhao Y. The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis. [Internet] [Thesis]. University of Western Ontario; 2020. [cited 2021 Mar 07]. Available from: https://ir.lib.uwo.ca/etd/7075.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lai-Zhao Y. The Role of Synovium and Synovial Macrophages in Experimental Post-Traumatic Knee Osteoarthritis. [Thesis]. University of Western Ontario; 2020. Available from: https://ir.lib.uwo.ca/etd/7075

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

30. McPhail, Mike Grant. Histopathology in Skeletal Muscle of Transgenic Mice Suggests Dual-Locus of Onset (DLO) in Kennedy's Disease.

Degree: 2014, University of Toronto

Kennedy's Disease or Spinal-Bulbar Muscular Atrophy (KD/SBMA) is a neuromuscular disease associated with a CAG tri-nucleotide repeat expansion within the androgen receptor (AR) gene. Mouse… (more)

Subjects/Keywords: Animal Models; Histopathology; Kennedy's Disease; Neuromuscular; polyQ Disorders; Spinal-Bulbar Muscular Atrophy; 0306

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APA (6th Edition):

McPhail, M. G. (2014). Histopathology in Skeletal Muscle of Transgenic Mice Suggests Dual-Locus of Onset (DLO) in Kennedy's Disease. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/68596

Chicago Manual of Style (16th Edition):

McPhail, Mike Grant. “Histopathology in Skeletal Muscle of Transgenic Mice Suggests Dual-Locus of Onset (DLO) in Kennedy's Disease.” 2014. Masters Thesis, University of Toronto. Accessed March 07, 2021. http://hdl.handle.net/1807/68596.

MLA Handbook (7th Edition):

McPhail, Mike Grant. “Histopathology in Skeletal Muscle of Transgenic Mice Suggests Dual-Locus of Onset (DLO) in Kennedy's Disease.” 2014. Web. 07 Mar 2021.

Vancouver:

McPhail MG. Histopathology in Skeletal Muscle of Transgenic Mice Suggests Dual-Locus of Onset (DLO) in Kennedy's Disease. [Internet] [Masters thesis]. University of Toronto; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1807/68596.

Council of Science Editors:

McPhail MG. Histopathology in Skeletal Muscle of Transgenic Mice Suggests Dual-Locus of Onset (DLO) in Kennedy's Disease. [Masters Thesis]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/68596

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