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You searched for subject:(Developmental pharmacology). Showing records 1 – 30 of 42 total matches.

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1. Schafler, Eric D. ART-27 Regulates Mammalian Spermatogonial Stem Cell Survival and Differentiation.

Degree: 2016, New York University

  Male mammals must simultaneously produce prodigious numbers of sperm and maintain an adequate reserve of stem cells to ensure continuous production of gametes throughout… (more)

Subjects/Keywords: Genetics; Pharmacology; Developmental biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schafler, E. D. (2016). ART-27 Regulates Mammalian Spermatogonial Stem Cell Survival and Differentiation. (Thesis). New York University. Retrieved from http://pqdtopen.proquest.com/#viewpdf?dispub=10192355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schafler, Eric D. “ART-27 Regulates Mammalian Spermatogonial Stem Cell Survival and Differentiation.” 2016. Thesis, New York University. Accessed January 16, 2021. http://pqdtopen.proquest.com/#viewpdf?dispub=10192355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schafler, Eric D. “ART-27 Regulates Mammalian Spermatogonial Stem Cell Survival and Differentiation.” 2016. Web. 16 Jan 2021.

Vancouver:

Schafler ED. ART-27 Regulates Mammalian Spermatogonial Stem Cell Survival and Differentiation. [Internet] [Thesis]. New York University; 2016. [cited 2021 Jan 16]. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10192355.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schafler ED. ART-27 Regulates Mammalian Spermatogonial Stem Cell Survival and Differentiation. [Thesis]. New York University; 2016. Available from: http://pqdtopen.proquest.com/#viewpdf?dispub=10192355

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

2. Bisen-Hersh, Emily Beth. Effects of Early Chemotherapeutic Treatment on Learning, Novelty, and Drug Reward in Adolescent Mice.

Degree: PhD, 2012, Temple University

Psychology

Among children diagnosed with acute lymphoblastic leukemia (ALL) and given chemotherapy-only treatment, 40-70% of survivors experience neurocognitive impairment. Psychostimulants such as methylphenidate are becoming… (more)

Subjects/Keywords: Psychology; Pharmacology; Neurosciences; Behavioral pharmacology; Chemotherapy; Developmental neurotoxicity; Drug Reward; Learning

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APA (6th Edition):

Bisen-Hersh, E. B. (2012). Effects of Early Chemotherapeutic Treatment on Learning, Novelty, and Drug Reward in Adolescent Mice. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,190153

Chicago Manual of Style (16th Edition):

Bisen-Hersh, Emily Beth. “Effects of Early Chemotherapeutic Treatment on Learning, Novelty, and Drug Reward in Adolescent Mice.” 2012. Doctoral Dissertation, Temple University. Accessed January 16, 2021. http://digital.library.temple.edu/u?/p245801coll10,190153.

MLA Handbook (7th Edition):

Bisen-Hersh, Emily Beth. “Effects of Early Chemotherapeutic Treatment on Learning, Novelty, and Drug Reward in Adolescent Mice.” 2012. Web. 16 Jan 2021.

Vancouver:

Bisen-Hersh EB. Effects of Early Chemotherapeutic Treatment on Learning, Novelty, and Drug Reward in Adolescent Mice. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2021 Jan 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,190153.

Council of Science Editors:

Bisen-Hersh EB. Effects of Early Chemotherapeutic Treatment on Learning, Novelty, and Drug Reward in Adolescent Mice. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,190153


University of North Carolina

3. Bargiacchi, Francesca. Molecular Dissection of Breast Luminal Transcription Networks.

Degree: Pharmacology, 2015, University of North Carolina

 During mammary development, cellular differentiation and lineage commitment to various epithelial and mesenchymal cell types are driven by hormonal and paracrine signaling mechanisms. Understanding mechanisms… (more)

Subjects/Keywords: Developmental biology; Oncology; School of Medicine; Department of Pharmacology

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APA (6th Edition):

Bargiacchi, F. (2015). Molecular Dissection of Breast Luminal Transcription Networks. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7fc72b4c-2217-4bad-9e19-c18aab09a1d4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bargiacchi, Francesca. “Molecular Dissection of Breast Luminal Transcription Networks.” 2015. Thesis, University of North Carolina. Accessed January 16, 2021. https://cdr.lib.unc.edu/record/uuid:7fc72b4c-2217-4bad-9e19-c18aab09a1d4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bargiacchi, Francesca. “Molecular Dissection of Breast Luminal Transcription Networks.” 2015. Web. 16 Jan 2021.

Vancouver:

Bargiacchi F. Molecular Dissection of Breast Luminal Transcription Networks. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2021 Jan 16]. Available from: https://cdr.lib.unc.edu/record/uuid:7fc72b4c-2217-4bad-9e19-c18aab09a1d4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bargiacchi F. Molecular Dissection of Breast Luminal Transcription Networks. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:7fc72b4c-2217-4bad-9e19-c18aab09a1d4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Uppsala University

4. Brolin, Elisabeth. Does prenatal hypoxia lead to permanent cardiovascular change in the offspring?.

Degree: Pharmaceutical Biosciences, 2015, Uppsala University

  Chronic prenatal hypoxia is associated with intrauterine growth retardation and there is now some evidence that it also induces programmed hypertension in offspring. However… (more)

Subjects/Keywords: Developmental programming; hypoxia; dofetilide; telemetry; Pharmacology and Toxicology; Farmakologi och toxikologi

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APA (6th Edition):

Brolin, E. (2015). Does prenatal hypoxia lead to permanent cardiovascular change in the offspring?. (Thesis). Uppsala University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-242903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brolin, Elisabeth. “Does prenatal hypoxia lead to permanent cardiovascular change in the offspring?.” 2015. Thesis, Uppsala University. Accessed January 16, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-242903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brolin, Elisabeth. “Does prenatal hypoxia lead to permanent cardiovascular change in the offspring?.” 2015. Web. 16 Jan 2021.

Vancouver:

Brolin E. Does prenatal hypoxia lead to permanent cardiovascular change in the offspring?. [Internet] [Thesis]. Uppsala University; 2015. [cited 2021 Jan 16]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-242903.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brolin E. Does prenatal hypoxia lead to permanent cardiovascular change in the offspring?. [Thesis]. Uppsala University; 2015. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:uu:diva-242903

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

5. Mussar, Kristin Elizabeth. Regenerative Cues of Myeloid Cells on Pancreatic Epithelium.

Degree: PhD, 2016, University of Washington

 Tissue regeneration is an attractive approach to treating many degenerative diseases including diabetes mellitus, a devastating disease caused by the destruction or dysfunction of insulin-producing… (more)

Subjects/Keywords: Diabetes; Myeloid; Pancreas; Regeneration; Repair; Biology; Cellular biology; Developmental biology; pharmacology

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APA (6th Edition):

Mussar, K. E. (2016). Regenerative Cues of Myeloid Cells on Pancreatic Epithelium. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/37214

Chicago Manual of Style (16th Edition):

Mussar, Kristin Elizabeth. “Regenerative Cues of Myeloid Cells on Pancreatic Epithelium.” 2016. Doctoral Dissertation, University of Washington. Accessed January 16, 2021. http://hdl.handle.net/1773/37214.

MLA Handbook (7th Edition):

Mussar, Kristin Elizabeth. “Regenerative Cues of Myeloid Cells on Pancreatic Epithelium.” 2016. Web. 16 Jan 2021.

Vancouver:

Mussar KE. Regenerative Cues of Myeloid Cells on Pancreatic Epithelium. [Internet] [Doctoral dissertation]. University of Washington; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1773/37214.

Council of Science Editors:

Mussar KE. Regenerative Cues of Myeloid Cells on Pancreatic Epithelium. [Doctoral Dissertation]. University of Washington; 2016. Available from: http://hdl.handle.net/1773/37214


University of Louisville

6. Wahlang, Banrida. Evaluating the effects of aroclor 1260 in non-alcoholic fatty liver disease : the role of xenobiotic receptors.

Degree: PhD, 2014, University of Louisville

  Polychlorinated biphenyls (PCBs) are persistent environmental toxicants, present in 100% of US adults and dose-dependently associated with nonalcoholic fatty liver disease (NAFLD) in epidemiologic… (more)

Subjects/Keywords: Cell and Developmental Biology; Pharmacology, Toxicology and Environmental Health

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APA (6th Edition):

Wahlang, B. (2014). Evaluating the effects of aroclor 1260 in non-alcoholic fatty liver disease : the role of xenobiotic receptors. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1733 ; https://ir.library.louisville.edu/etd/1733

Chicago Manual of Style (16th Edition):

Wahlang, Banrida. “Evaluating the effects of aroclor 1260 in non-alcoholic fatty liver disease : the role of xenobiotic receptors.” 2014. Doctoral Dissertation, University of Louisville. Accessed January 16, 2021. 10.18297/etd/1733 ; https://ir.library.louisville.edu/etd/1733.

MLA Handbook (7th Edition):

Wahlang, Banrida. “Evaluating the effects of aroclor 1260 in non-alcoholic fatty liver disease : the role of xenobiotic receptors.” 2014. Web. 16 Jan 2021.

Vancouver:

Wahlang B. Evaluating the effects of aroclor 1260 in non-alcoholic fatty liver disease : the role of xenobiotic receptors. [Internet] [Doctoral dissertation]. University of Louisville; 2014. [cited 2021 Jan 16]. Available from: 10.18297/etd/1733 ; https://ir.library.louisville.edu/etd/1733.

Council of Science Editors:

Wahlang B. Evaluating the effects of aroclor 1260 in non-alcoholic fatty liver disease : the role of xenobiotic receptors. [Doctoral Dissertation]. University of Louisville; 2014. Available from: 10.18297/etd/1733 ; https://ir.library.louisville.edu/etd/1733


Columbia University

7. Tannenholz, Lindsay Elsa. The Impact of Modulating the Activity of Adult-born Hippocampal Neurons on Neurogenesis and Behavior.

Degree: 2016, Columbia University

 Adult hippocampal neurogenesis—a unique form of plasticity in the dentate gyrus (DG)—is regulated by experience, and when manipulated can have specific effects on behavior. Different… (more)

Subjects/Keywords: Hippocampus (Brain) – Physiology; Developmental neurobiology; Dentate gyrus; Neuroplasticity; Neural circuitry; Hippocampus (Brain); Neurosciences; Pharmacology

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APA (6th Edition):

Tannenholz, L. E. (2016). The Impact of Modulating the Activity of Adult-born Hippocampal Neurons on Neurogenesis and Behavior. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8CN73Q9

Chicago Manual of Style (16th Edition):

Tannenholz, Lindsay Elsa. “The Impact of Modulating the Activity of Adult-born Hippocampal Neurons on Neurogenesis and Behavior.” 2016. Doctoral Dissertation, Columbia University. Accessed January 16, 2021. https://doi.org/10.7916/D8CN73Q9.

MLA Handbook (7th Edition):

Tannenholz, Lindsay Elsa. “The Impact of Modulating the Activity of Adult-born Hippocampal Neurons on Neurogenesis and Behavior.” 2016. Web. 16 Jan 2021.

Vancouver:

Tannenholz LE. The Impact of Modulating the Activity of Adult-born Hippocampal Neurons on Neurogenesis and Behavior. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2021 Jan 16]. Available from: https://doi.org/10.7916/D8CN73Q9.

Council of Science Editors:

Tannenholz LE. The Impact of Modulating the Activity of Adult-born Hippocampal Neurons on Neurogenesis and Behavior. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8CN73Q9


University of Alabama

8. Di Bona, Kristin Rogers. Pharmaceutical & toxicological investigations of metals: investigations of supplemental chromium(iii) and iron oxide nanoparticles in rodent models.

Degree: 2014, University of Alabama

 Trivalent chromium (Cr3+) has widely been accepted as a nutritional element necessary for proper carbohydrate and lipid metabolism in mammals. Upon closer examination, beneficial effects… (more)

Subjects/Keywords: Electronic Thesis or Dissertation;  – thesis; Biochemistry; Developmental biology; Pharmacology; Chromium; Diabetes; Insulin; Iron Oxide; Nanoparticles; Toxicology

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APA (6th Edition):

Di Bona, K. R. (2014). Pharmaceutical & toxicological investigations of metals: investigations of supplemental chromium(iii) and iron oxide nanoparticles in rodent models. (Thesis). University of Alabama. Retrieved from http://purl.lib.ua.edu/120468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Di Bona, Kristin Rogers. “Pharmaceutical & toxicological investigations of metals: investigations of supplemental chromium(iii) and iron oxide nanoparticles in rodent models.” 2014. Thesis, University of Alabama. Accessed January 16, 2021. http://purl.lib.ua.edu/120468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Di Bona, Kristin Rogers. “Pharmaceutical & toxicological investigations of metals: investigations of supplemental chromium(iii) and iron oxide nanoparticles in rodent models.” 2014. Web. 16 Jan 2021.

Vancouver:

Di Bona KR. Pharmaceutical & toxicological investigations of metals: investigations of supplemental chromium(iii) and iron oxide nanoparticles in rodent models. [Internet] [Thesis]. University of Alabama; 2014. [cited 2021 Jan 16]. Available from: http://purl.lib.ua.edu/120468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Di Bona KR. Pharmaceutical & toxicological investigations of metals: investigations of supplemental chromium(iii) and iron oxide nanoparticles in rodent models. [Thesis]. University of Alabama; 2014. Available from: http://purl.lib.ua.edu/120468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Kaissarian, Nayiri. Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease.

Degree: PhD, Pharmacology, 2017, University of Michigan

 Fabry disease is a rare, X-linked lysosomal storage disease arising from deficiency of the lysosomal hydrolase, α-galactosidase A (GLA). Reduced GLA activity disrupts glycosphingolipid (GSL)… (more)

Subjects/Keywords: Fabry disease; Endothelial dysfunction; Biological Chemistry; Molecular, Cellular and Developmental Biology; Pharmacy and Pharmacology; Physiology; Science (General); Health Sciences; Science

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APA (6th Edition):

Kaissarian, N. (2017). Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/138731

Chicago Manual of Style (16th Edition):

Kaissarian, Nayiri. “Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease.” 2017. Doctoral Dissertation, University of Michigan. Accessed January 16, 2021. http://hdl.handle.net/2027.42/138731.

MLA Handbook (7th Edition):

Kaissarian, Nayiri. “Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease.” 2017. Web. 16 Jan 2021.

Vancouver:

Kaissarian N. Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease. [Internet] [Doctoral dissertation]. University of Michigan; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2027.42/138731.

Council of Science Editors:

Kaissarian N. Investigating Endothelial Dysfunction in a CRISPR/Cas9 Model of Fabry Disease. [Doctoral Dissertation]. University of Michigan; 2017. Available from: http://hdl.handle.net/2027.42/138731


University of Western Ontario

10. Engineer, Anish. Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies.

Degree: 2019, University of Western Ontario

 Congenital heart defects (CHDs) arise from perturbations in complex molecular and cellular processes underlying normal embryonic heart development. CHDs are the most common congenital malformation,… (more)

Subjects/Keywords: Pregestational Diabetes; Congenital Heart Defects; Heart Development; Sapropterin; miR-122; coronary arteries; Cardiovascular Diseases; Cellular and Molecular Physiology; Developmental Biology; Pharmacology

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APA (6th Edition):

Engineer, A. (2019). Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Engineer, Anish. “Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies.” 2019. Thesis, University of Western Ontario. Accessed January 16, 2021. https://ir.lib.uwo.ca/etd/6328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Engineer, Anish. “Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies.” 2019. Web. 16 Jan 2021.

Vancouver:

Engineer A. Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2021 Jan 16]. Available from: https://ir.lib.uwo.ca/etd/6328.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Engineer A. Pregestational Diabetes Induced Congenital Heart Defects and Coronary Artery Malformations; Mechanisms and Preventative Therapies. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6328

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


East Tennessee State University

11. Whitted, Crystal L. Pharmacokinetics, Tissue Distribution, Synergistic Activity, and Antitumor Activity of Two Isomeric Flavones.

Degree: PhD, Biomedical Sciences, 2016, East Tennessee State University

  Flavonoids are polyphenolic secondary metabolites found in plants that have bioactive properties including antiviral, antioxidant, and anticancer. Two isomeric flavone were extracted from Gnaphalium… (more)

Subjects/Keywords: flavone A; flavone B; pharmacokinetics; colon cancer; anticancer; Biology; Cancer Biology; Cell and Developmental Biology; Molecular Biology; Pharmacology

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APA (6th Edition):

Whitted, C. L. (2016). Pharmacokinetics, Tissue Distribution, Synergistic Activity, and Antitumor Activity of Two Isomeric Flavones. (Doctoral Dissertation). East Tennessee State University. Retrieved from https://dc.etsu.edu/etd/3167

Chicago Manual of Style (16th Edition):

Whitted, Crystal L. “Pharmacokinetics, Tissue Distribution, Synergistic Activity, and Antitumor Activity of Two Isomeric Flavones.” 2016. Doctoral Dissertation, East Tennessee State University. Accessed January 16, 2021. https://dc.etsu.edu/etd/3167.

MLA Handbook (7th Edition):

Whitted, Crystal L. “Pharmacokinetics, Tissue Distribution, Synergistic Activity, and Antitumor Activity of Two Isomeric Flavones.” 2016. Web. 16 Jan 2021.

Vancouver:

Whitted CL. Pharmacokinetics, Tissue Distribution, Synergistic Activity, and Antitumor Activity of Two Isomeric Flavones. [Internet] [Doctoral dissertation]. East Tennessee State University; 2016. [cited 2021 Jan 16]. Available from: https://dc.etsu.edu/etd/3167.

Council of Science Editors:

Whitted CL. Pharmacokinetics, Tissue Distribution, Synergistic Activity, and Antitumor Activity of Two Isomeric Flavones. [Doctoral Dissertation]. East Tennessee State University; 2016. Available from: https://dc.etsu.edu/etd/3167

12. Bernardo, Gina M. Discerning the Role of FOXA1 in Mammary Gland Development and Breast Cancer.

Degree: PhD, Pharmacology, 2011, Case Western Reserve University School of Graduate Studies

  Breast cancer is a heterogeneous disease with distinct subtypes that are predictive of patient prognosis. Luminal subtype tumors confer the most favorable prognosis due… (more)

Subjects/Keywords: Biomedical Research; Developmental Biology; Oncology; Pharmacology; FOXA1; ER; GATA3; breast cancer; mammary gland development; luminal; basal

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APA (6th Edition):

Bernardo, G. M. (2011). Discerning the Role of FOXA1 in Mammary Gland Development and Breast Cancer. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1315607905

Chicago Manual of Style (16th Edition):

Bernardo, Gina M. “Discerning the Role of FOXA1 in Mammary Gland Development and Breast Cancer.” 2011. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed January 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=case1315607905.

MLA Handbook (7th Edition):

Bernardo, Gina M. “Discerning the Role of FOXA1 in Mammary Gland Development and Breast Cancer.” 2011. Web. 16 Jan 2021.

Vancouver:

Bernardo GM. Discerning the Role of FOXA1 in Mammary Gland Development and Breast Cancer. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2011. [cited 2021 Jan 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1315607905.

Council of Science Editors:

Bernardo GM. Discerning the Role of FOXA1 in Mammary Gland Development and Breast Cancer. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1315607905


University of Pennsylvania

13. Shinde, Mansi. Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3.

Degree: 2016, University of Pennsylvania

 Glycogen Synthase Kinase-3 (GSK-3) is a constitutively active, ubiquitously expressed kinase that acts as a critical regulator of many signaling pathways. These pathways, when dysregulated,… (more)

Subjects/Keywords: Alternative Splicing; Glycogen Synthase Kinase-3; GSK-3; Murine embryonic stem cells; Phosphoproteome; Cell Biology; Developmental Biology; Pharmacology

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APA (6th Edition):

Shinde, M. (2016). Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shinde, Mansi. “Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3.” 2016. Thesis, University of Pennsylvania. Accessed January 16, 2021. https://repository.upenn.edu/edissertations/2583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shinde, Mansi. “Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3.” 2016. Web. 16 Jan 2021.

Vancouver:

Shinde M. Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3. [Internet] [Thesis]. University of Pennsylvania; 2016. [cited 2021 Jan 16]. Available from: https://repository.upenn.edu/edissertations/2583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shinde M. Phosphoproteomic Characterization Of Glycogen Synthase Kinase-3. [Thesis]. University of Pennsylvania; 2016. Available from: https://repository.upenn.edu/edissertations/2583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

14. Hutch, Chelsea Reed. Cannabinoid action in the mouse olfactory epithelium.

Degree: 2014, Michigan State University

Thesis Ph. D. Michigan State University. Neuroscience - Environmental Toxicology 2014.

ABSTRACTCANNABINOID ACTION IN THE MOUSE OLFACTORY EPITHELIUMByChelsea Reed Hutch Mammalian adult neurogenesis, i.e., the… (more)

Subjects/Keywords: Cannabinoids – Receptors; Epithelium; Mice – Sense organs; Olfactory mucosa; Homeostasis; Developmental neurobiology; Neurosciences; Toxicology; Pharmacology; Olfactory epithelium

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APA (6th Edition):

Hutch, C. R. (2014). Cannabinoid action in the mouse olfactory epithelium. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2774

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hutch, Chelsea Reed. “Cannabinoid action in the mouse olfactory epithelium.” 2014. Thesis, Michigan State University. Accessed January 16, 2021. http://etd.lib.msu.edu/islandora/object/etd:2774.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hutch, Chelsea Reed. “Cannabinoid action in the mouse olfactory epithelium.” 2014. Web. 16 Jan 2021.

Vancouver:

Hutch CR. Cannabinoid action in the mouse olfactory epithelium. [Internet] [Thesis]. Michigan State University; 2014. [cited 2021 Jan 16]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2774.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hutch CR. Cannabinoid action in the mouse olfactory epithelium. [Thesis]. Michigan State University; 2014. Available from: http://etd.lib.msu.edu/islandora/object/etd:2774

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo

15. Al Shaban, Amani. Developmental Toxicity of Ambroxol in Zebrafish Embryos/Larvae: Relevance of SULT-mediated Sulfation of Ambroxol.

Degree: MSin Pharmaceutical Sciences, Pharmaceutical Science, 2010, University of Toledo

 Ambroxol is an active metabolite of bromexine that has been proven to possess a great bronchosecretolytic effect and has been used to treat infants from… (more)

Subjects/Keywords: Pharmacology; ambroxol; cytosolic sulfotranseferases; sulfation; zebrafish developmental toxicity; developmental toxicity

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APA (6th Edition):

Al Shaban, A. (2010). Developmental Toxicity of Ambroxol in Zebrafish Embryos/Larvae: Relevance of SULT-mediated Sulfation of Ambroxol. (Masters Thesis). University of Toledo. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=toledo1271349511

Chicago Manual of Style (16th Edition):

Al Shaban, Amani. “Developmental Toxicity of Ambroxol in Zebrafish Embryos/Larvae: Relevance of SULT-mediated Sulfation of Ambroxol.” 2010. Masters Thesis, University of Toledo. Accessed January 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=toledo1271349511.

MLA Handbook (7th Edition):

Al Shaban, Amani. “Developmental Toxicity of Ambroxol in Zebrafish Embryos/Larvae: Relevance of SULT-mediated Sulfation of Ambroxol.” 2010. Web. 16 Jan 2021.

Vancouver:

Al Shaban A. Developmental Toxicity of Ambroxol in Zebrafish Embryos/Larvae: Relevance of SULT-mediated Sulfation of Ambroxol. [Internet] [Masters thesis]. University of Toledo; 2010. [cited 2021 Jan 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1271349511.

Council of Science Editors:

Al Shaban A. Developmental Toxicity of Ambroxol in Zebrafish Embryos/Larvae: Relevance of SULT-mediated Sulfation of Ambroxol. [Masters Thesis]. University of Toledo; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=toledo1271349511


University of Michigan

16. Kruger, Larisa Christine. Voltage-gated Na+ Channels: Not Just for Conduction.

Degree: PhD, Pharmacology, 2016, University of Michigan

 Voltage-gated sodium channels (VGSCs), composed of a pore-forming alpha subunit and up to two associated Beta subunits, are critical for the initiation of the action… (more)

Subjects/Keywords: Voltage-gated sodium channel beta1 subunit; pediatric epilepsy mouse model; SCN1B; GEFS+; Molecular, Cellular and Developmental Biology; Neurosciences; Pharmacy and Pharmacology; Physiology; Health Sciences; Science

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APA (6th Edition):

Kruger, L. C. (2016). Voltage-gated Na+ Channels: Not Just for Conduction. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/120810

Chicago Manual of Style (16th Edition):

Kruger, Larisa Christine. “Voltage-gated Na+ Channels: Not Just for Conduction.” 2016. Doctoral Dissertation, University of Michigan. Accessed January 16, 2021. http://hdl.handle.net/2027.42/120810.

MLA Handbook (7th Edition):

Kruger, Larisa Christine. “Voltage-gated Na+ Channels: Not Just for Conduction.” 2016. Web. 16 Jan 2021.

Vancouver:

Kruger LC. Voltage-gated Na+ Channels: Not Just for Conduction. [Internet] [Doctoral dissertation]. University of Michigan; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2027.42/120810.

Council of Science Editors:

Kruger LC. Voltage-gated Na+ Channels: Not Just for Conduction. [Doctoral Dissertation]. University of Michigan; 2016. Available from: http://hdl.handle.net/2027.42/120810


Texas Medical Center

17. Hendon, Laura G. Physician perceptions of risk regarding mood disorders and pharmacological management during pregnancy: What is current practice?.

Degree: MS, 2011, Texas Medical Center

  Mood disorders are the most common form of mental illness and one of the leading causes of morbidity worldwide.  Major depressive disorder and bipolar… (more)

Subjects/Keywords: mood disorders; teratogen counseling; risk perception; risk communication; genetic counseling; managing pregnant women with mood disorders; Cognition and Perception; Developmental Biology; Health Communication; Pharmacology

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APA (6th Edition):

Hendon, L. G. (2011). Physician perceptions of risk regarding mood disorders and pharmacological management during pregnancy: What is current practice?. (Thesis). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hendon, Laura G. “Physician perceptions of risk regarding mood disorders and pharmacological management during pregnancy: What is current practice?.” 2011. Thesis, Texas Medical Center. Accessed January 16, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hendon, Laura G. “Physician perceptions of risk regarding mood disorders and pharmacological management during pregnancy: What is current practice?.” 2011. Web. 16 Jan 2021.

Vancouver:

Hendon LG. Physician perceptions of risk regarding mood disorders and pharmacological management during pregnancy: What is current practice?. [Internet] [Thesis]. Texas Medical Center; 2011. [cited 2021 Jan 16]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/125.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hendon LG. Physician perceptions of risk regarding mood disorders and pharmacological management during pregnancy: What is current practice?. [Thesis]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/125

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Western Michigan University

18. Lentz, Susan J. Cellular Proliferation, Death and Histology in Gambusia Affinis Livers after Exposure to 2-Aminofluorene and Benzidine.

Degree: PhD, Biological Sciences, 2003, Western Michigan University

  A fundamental need exists for aquatic animals in the biomonitoring of aquatic ecosystems for environmental carcinogens. G. affinis, a small fish species, is selected… (more)

Subjects/Keywords: Biology; Cell and Developmental Biology; Pharmacology, Toxicology and Environmental Health

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APA (6th Edition):

Lentz, S. J. (2003). Cellular Proliferation, Death and Histology in Gambusia Affinis Livers after Exposure to 2-Aminofluorene and Benzidine. (Doctoral Dissertation). Western Michigan University. Retrieved from https://scholarworks.wmich.edu/dissertations/1248

Chicago Manual of Style (16th Edition):

Lentz, Susan J. “Cellular Proliferation, Death and Histology in Gambusia Affinis Livers after Exposure to 2-Aminofluorene and Benzidine.” 2003. Doctoral Dissertation, Western Michigan University. Accessed January 16, 2021. https://scholarworks.wmich.edu/dissertations/1248.

MLA Handbook (7th Edition):

Lentz, Susan J. “Cellular Proliferation, Death and Histology in Gambusia Affinis Livers after Exposure to 2-Aminofluorene and Benzidine.” 2003. Web. 16 Jan 2021.

Vancouver:

Lentz SJ. Cellular Proliferation, Death and Histology in Gambusia Affinis Livers after Exposure to 2-Aminofluorene and Benzidine. [Internet] [Doctoral dissertation]. Western Michigan University; 2003. [cited 2021 Jan 16]. Available from: https://scholarworks.wmich.edu/dissertations/1248.

Council of Science Editors:

Lentz SJ. Cellular Proliferation, Death and Histology in Gambusia Affinis Livers after Exposure to 2-Aminofluorene and Benzidine. [Doctoral Dissertation]. Western Michigan University; 2003. Available from: https://scholarworks.wmich.edu/dissertations/1248


Iowa State University

19. Boury, Nancy A. Maroushek. Cloning, expression and bioactivity of porcine soluble TNF receptor 1 and the comparison of PK(15) and WEHI 164(13)-based TNF bioassays.

Degree: 1997, Iowa State University

 Tumor necrosis factor (TNF) is a pro-inflammatory cytokine that activates both leukocytes and endothelium and facilitates the movement of inflammatory cells from circulation to sites… (more)

Subjects/Keywords: Veterinary physiology and pharmacology; Molecular; cellular; and developmental biology; Molecular Biology; Veterinary Medicine

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APA (6th Edition):

Boury, N. A. M. (1997). Cloning, expression and bioactivity of porcine soluble TNF receptor 1 and the comparison of PK(15) and WEHI 164(13)-based TNF bioassays. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/rtd/11443

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boury, Nancy A Maroushek. “Cloning, expression and bioactivity of porcine soluble TNF receptor 1 and the comparison of PK(15) and WEHI 164(13)-based TNF bioassays.” 1997. Thesis, Iowa State University. Accessed January 16, 2021. https://lib.dr.iastate.edu/rtd/11443.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boury, Nancy A Maroushek. “Cloning, expression and bioactivity of porcine soluble TNF receptor 1 and the comparison of PK(15) and WEHI 164(13)-based TNF bioassays.” 1997. Web. 16 Jan 2021.

Vancouver:

Boury NAM. Cloning, expression and bioactivity of porcine soluble TNF receptor 1 and the comparison of PK(15) and WEHI 164(13)-based TNF bioassays. [Internet] [Thesis]. Iowa State University; 1997. [cited 2021 Jan 16]. Available from: https://lib.dr.iastate.edu/rtd/11443.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boury NAM. Cloning, expression and bioactivity of porcine soluble TNF receptor 1 and the comparison of PK(15) and WEHI 164(13)-based TNF bioassays. [Thesis]. Iowa State University; 1997. Available from: https://lib.dr.iastate.edu/rtd/11443

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Iowa State University

20. Vistica, David Thomas. The development of lead encephalopathy in the suckling rat: effects of lead on: I. Capillary basement membrane, II. Collagen metabolism.

Degree: 1975, Iowa State University

Subjects/Keywords: Veterinary anatomy; pharmacology and physiology; Molecular; cellular; and developmental biology; Biology

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APA (6th Edition):

Vistica, D. T. (1975). The development of lead encephalopathy in the suckling rat: effects of lead on: I. Capillary basement membrane, II. Collagen metabolism. (Thesis). Iowa State University. Retrieved from https://lib.dr.iastate.edu/rtd/5766

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vistica, David Thomas. “The development of lead encephalopathy in the suckling rat: effects of lead on: I. Capillary basement membrane, II. Collagen metabolism.” 1975. Thesis, Iowa State University. Accessed January 16, 2021. https://lib.dr.iastate.edu/rtd/5766.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vistica, David Thomas. “The development of lead encephalopathy in the suckling rat: effects of lead on: I. Capillary basement membrane, II. Collagen metabolism.” 1975. Web. 16 Jan 2021.

Vancouver:

Vistica DT. The development of lead encephalopathy in the suckling rat: effects of lead on: I. Capillary basement membrane, II. Collagen metabolism. [Internet] [Thesis]. Iowa State University; 1975. [cited 2021 Jan 16]. Available from: https://lib.dr.iastate.edu/rtd/5766.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vistica DT. The development of lead encephalopathy in the suckling rat: effects of lead on: I. Capillary basement membrane, II. Collagen metabolism. [Thesis]. Iowa State University; 1975. Available from: https://lib.dr.iastate.edu/rtd/5766

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

21. Smith, Bradley Harrison, 1961-. Reliability, validity and unique contributions of self-reports by adolescents being treated for attention deficit hyperactivity disorder .

Degree: 1997, University of Arizona

 The current study assessed critical psychometric properties of self-reports by 46 adolescents enrolled in an eight week-long Summer Treatment Program (STP). Self-report instruments included the… (more)

Subjects/Keywords: Health Sciences, Mental Health.; Health Sciences, Pharmacology.; Psychology, Developmental.; Psychology, Clinical.; Psychology, Psychometrics.

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APA (6th Edition):

Smith, Bradley Harrison, 1. (1997). Reliability, validity and unique contributions of self-reports by adolescents being treated for attention deficit hyperactivity disorder . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/282354

Chicago Manual of Style (16th Edition):

Smith, Bradley Harrison, 1961-. “Reliability, validity and unique contributions of self-reports by adolescents being treated for attention deficit hyperactivity disorder .” 1997. Doctoral Dissertation, University of Arizona. Accessed January 16, 2021. http://hdl.handle.net/10150/282354.

MLA Handbook (7th Edition):

Smith, Bradley Harrison, 1961-. “Reliability, validity and unique contributions of self-reports by adolescents being treated for attention deficit hyperactivity disorder .” 1997. Web. 16 Jan 2021.

Vancouver:

Smith, Bradley Harrison 1. Reliability, validity and unique contributions of self-reports by adolescents being treated for attention deficit hyperactivity disorder . [Internet] [Doctoral dissertation]. University of Arizona; 1997. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/10150/282354.

Council of Science Editors:

Smith, Bradley Harrison 1. Reliability, validity and unique contributions of self-reports by adolescents being treated for attention deficit hyperactivity disorder . [Doctoral Dissertation]. University of Arizona; 1997. Available from: http://hdl.handle.net/10150/282354


University of South Carolina

22. Balapanage, Sumith Jayasinghe. G-Protein-Coupled Estrogen Receptor-1 (GPER): A Potential Target For Xenoestrogens During Vertebrate Embryogenesis.

Degree: PhD, Environmental Health Sciences, 2012, University of South Carolina

  G-protein-coupled estrogen receptor 1 (GPER) is a G-protein-coupled receptor that induces non-genomic signaling in response to some nuclear estrogen receptor ligands. To date, the… (more)

Subjects/Keywords: Environmental Health; Environmental Sciences; Life Sciences; Pharmacology, Toxicology and Environmental Health; Physical Sciences and Mathematics; cardiovascular impairments; Developmental toxicology; endocrine distruption; GPER/ GPR30; ligand-induced activation; Zebrafish embryogenesis

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APA (6th Edition):

Balapanage, S. J. (2012). G-Protein-Coupled Estrogen Receptor-1 (GPER): A Potential Target For Xenoestrogens During Vertebrate Embryogenesis. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/1114

Chicago Manual of Style (16th Edition):

Balapanage, Sumith Jayasinghe. “G-Protein-Coupled Estrogen Receptor-1 (GPER): A Potential Target For Xenoestrogens During Vertebrate Embryogenesis.” 2012. Doctoral Dissertation, University of South Carolina. Accessed January 16, 2021. https://scholarcommons.sc.edu/etd/1114.

MLA Handbook (7th Edition):

Balapanage, Sumith Jayasinghe. “G-Protein-Coupled Estrogen Receptor-1 (GPER): A Potential Target For Xenoestrogens During Vertebrate Embryogenesis.” 2012. Web. 16 Jan 2021.

Vancouver:

Balapanage SJ. G-Protein-Coupled Estrogen Receptor-1 (GPER): A Potential Target For Xenoestrogens During Vertebrate Embryogenesis. [Internet] [Doctoral dissertation]. University of South Carolina; 2012. [cited 2021 Jan 16]. Available from: https://scholarcommons.sc.edu/etd/1114.

Council of Science Editors:

Balapanage SJ. G-Protein-Coupled Estrogen Receptor-1 (GPER): A Potential Target For Xenoestrogens During Vertebrate Embryogenesis. [Doctoral Dissertation]. University of South Carolina; 2012. Available from: https://scholarcommons.sc.edu/etd/1114


University of Michigan

23. Su, Anthony. Investigation of Apoptosis and Metabolism as Mechanisms of Trichloroethylene Toxicity During Pregnancy.

Degree: PhD, Toxicology, 2020, University of Michigan

 Trichloroethylene (TCE), an organic solvent with multiple industrial uses, is associated with adverse pregnancy outcomes in epidemiological studies. In timed-pregnant Wistar rats, TCE exposure reduces… (more)

Subjects/Keywords: Toxicology; Trichloroethylene; Metabolism; Apoptosis; Reactive oxygen species; Pregnancy; Biological Chemistry; Molecular, Cellular and Developmental Biology; Pharmacy and Pharmacology; Public Health; Health Sciences

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APA (6th Edition):

Su, A. (2020). Investigation of Apoptosis and Metabolism as Mechanisms of Trichloroethylene Toxicity During Pregnancy. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/163113

Chicago Manual of Style (16th Edition):

Su, Anthony. “Investigation of Apoptosis and Metabolism as Mechanisms of Trichloroethylene Toxicity During Pregnancy.” 2020. Doctoral Dissertation, University of Michigan. Accessed January 16, 2021. http://hdl.handle.net/2027.42/163113.

MLA Handbook (7th Edition):

Su, Anthony. “Investigation of Apoptosis and Metabolism as Mechanisms of Trichloroethylene Toxicity During Pregnancy.” 2020. Web. 16 Jan 2021.

Vancouver:

Su A. Investigation of Apoptosis and Metabolism as Mechanisms of Trichloroethylene Toxicity During Pregnancy. [Internet] [Doctoral dissertation]. University of Michigan; 2020. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2027.42/163113.

Council of Science Editors:

Su A. Investigation of Apoptosis and Metabolism as Mechanisms of Trichloroethylene Toxicity During Pregnancy. [Doctoral Dissertation]. University of Michigan; 2020. Available from: http://hdl.handle.net/2027.42/163113


Wright State University

24. Sammohi, Shamili. Effect of progesterone, terbutaline and leptin on the function of alveolar type II cells.

Degree: MS, Pharmacology and Toxicology, 2015, Wright State University

 The “surfactant” produced by type II pneumonocytes is deficient in term and preterm infants born and diagnosed with Respiratory Distress Syndrome (RDS). Corticosteroids such as… (more)

Subjects/Keywords: Developmental Biology; Obstetrics; Pharmacology; Surfactant production, betamethasone, terbutaline, leptin, progesterone, mRNA, surfactant-B protein, SP-B, type II pneumonocytes, respiratory distress syndrome, RDS

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APA (6th Edition):

Sammohi, S. (2015). Effect of progesterone, terbutaline and leptin on the function of alveolar type II cells. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1441040764

Chicago Manual of Style (16th Edition):

Sammohi, Shamili. “Effect of progesterone, terbutaline and leptin on the function of alveolar type II cells.” 2015. Masters Thesis, Wright State University. Accessed January 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1441040764.

MLA Handbook (7th Edition):

Sammohi, Shamili. “Effect of progesterone, terbutaline and leptin on the function of alveolar type II cells.” 2015. Web. 16 Jan 2021.

Vancouver:

Sammohi S. Effect of progesterone, terbutaline and leptin on the function of alveolar type II cells. [Internet] [Masters thesis]. Wright State University; 2015. [cited 2021 Jan 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1441040764.

Council of Science Editors:

Sammohi S. Effect of progesterone, terbutaline and leptin on the function of alveolar type II cells. [Masters Thesis]. Wright State University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1441040764


University of Western Ontario

25. Greco, Elizabeth. Maternal nicotine exposure induces congenital heart defects in the offspring of mice.

Degree: 2019, University of Western Ontario

 Congenital heart defects are the most prevalent birth defect, and maternal cigarette smoking is a known risk factor. Nicotine replacement therapies are recommended to pregnant… (more)

Subjects/Keywords: Congenital heart defects; nicotine; maternal nicotine exposure; heart development; coronary artery development; reactive oxygen species; Animal Experimentation and Research; Cardiology; Cardiovascular Diseases; Cellular and Molecular Physiology; Congenital, Hereditary, and Neonatal Diseases and Abnormalities; Developmental Biology; Organic Chemicals; Other Cell and Developmental Biology; Other Physiology; Pharmacology

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APA (6th Edition):

Greco, E. (2019). Maternal nicotine exposure induces congenital heart defects in the offspring of mice. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/6306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Greco, Elizabeth. “Maternal nicotine exposure induces congenital heart defects in the offspring of mice.” 2019. Thesis, University of Western Ontario. Accessed January 16, 2021. https://ir.lib.uwo.ca/etd/6306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Greco, Elizabeth. “Maternal nicotine exposure induces congenital heart defects in the offspring of mice.” 2019. Web. 16 Jan 2021.

Vancouver:

Greco E. Maternal nicotine exposure induces congenital heart defects in the offspring of mice. [Internet] [Thesis]. University of Western Ontario; 2019. [cited 2021 Jan 16]. Available from: https://ir.lib.uwo.ca/etd/6306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Greco E. Maternal nicotine exposure induces congenital heart defects in the offspring of mice. [Thesis]. University of Western Ontario; 2019. Available from: https://ir.lib.uwo.ca/etd/6306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

26. Greathouse, Kristen L. XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT.

Degree: PhD, 2010, Texas Medical Center

 Environmental exposures during sensitive windows of development can reprogram normal physiological responses and alter disease susceptibility later in life in a process known as developmental(more)

Subjects/Keywords: Xenoestrogens; developmental reprogramming; uterine leiomyoma; epigenetics; non-genomic signaling; estrogen receptor; Cancer Biology; Developmental Biology; Molecular Biology; Other Pharmacology, Toxicology and Environmental Health

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APA (6th Edition):

Greathouse, K. L. (2010). XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/51

Chicago Manual of Style (16th Edition):

Greathouse, Kristen L. “XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT.” 2010. Doctoral Dissertation, Texas Medical Center. Accessed January 16, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/51.

MLA Handbook (7th Edition):

Greathouse, Kristen L. “XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT.” 2010. Web. 16 Jan 2021.

Vancouver:

Greathouse KL. XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT. [Internet] [Doctoral dissertation]. Texas Medical Center; 2010. [cited 2021 Jan 16]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/51.

Council of Science Editors:

Greathouse KL. XENOESTROGEN-SPECIFIC MECHANISMS OF DEVELOPMENTAL REPROGRAMMING CORRELATE WITH GENE EXPRESSION AND TUMOR DEVELOPMENT. [Doctoral Dissertation]. Texas Medical Center; 2010. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/51

27. Roarty, Kevin Patrick. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.

Degree: PhD, 2008, University of Alabama – Birmingham

Breast cancer is the second most common cancer worldwide behind lung cancer, affecting women of all ages, races, ethnicities, and socioeconomic strata. Concerted efforts have… (more)

Subjects/Keywords: Gene Expression Regulation, Developmental <; br>; Mammary Glands, Animal  – growth & development <; br>; Mammary Glands, Animal  – metabolism <; br>; Transforming Growth Factor beta1  – pharmacology <; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Roarty, K. P. (2008). The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,337

Chicago Manual of Style (16th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 16, 2021. http://contentdm.mhsl.uab.edu/u?/etd,337.

MLA Handbook (7th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Web. 16 Jan 2021.

Vancouver:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Jan 16]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337.

Council of Science Editors:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337

28. Vallaster, Markus Parzival. Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2017, U of Massachusetts : Med

  Environmental conditions imposed onto organisms during certain phases of their life cycles such as embryogenesis or puberty can not only impact the organisms’ own… (more)

Subjects/Keywords: chromosomes; epigenetics; genes; mouse; paternal effects; substance abuse; neuroscience; Behavioral Neurobiology; Biochemical Phenomena, Metabolism, and Nutrition; Cell Biology; Cellular and Molecular Physiology; Developmental Neuroscience; Genetics; Molecular Genetics; Other Pharmacology, Toxicology and Environmental Health

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APA (6th Edition):

Vallaster, M. P. (2017). Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/913

Chicago Manual of Style (16th Edition):

Vallaster, Markus Parzival. “Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure.” 2017. Doctoral Dissertation, U of Massachusetts : Med. Accessed January 16, 2021. http://escholarship.umassmed.edu/gsbs_diss/913.

MLA Handbook (7th Edition):

Vallaster, Markus Parzival. “Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure.” 2017. Web. 16 Jan 2021.

Vancouver:

Vallaster MP. Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2017. [cited 2021 Jan 16]. Available from: http://escholarship.umassmed.edu/gsbs_diss/913.

Council of Science Editors:

Vallaster MP. Intergenerational Effects of Nicotine in an Animal Model of Paternal Nicotine Exposure. [Doctoral Dissertation]. U of Massachusetts : Med; 2017. Available from: http://escholarship.umassmed.edu/gsbs_diss/913

29. Patel, Isha B. Medication use and Healthcare Outcomes in Developmentally Disabled Medicaid Adults with Type 2 Dibetes: a Quantitative Race Based Analysis.

Degree: PhD, Social and Administrative Sciences, 2014, University of Michigan

 Developmentally disabled (DD) individuals (Cerebral palsy, Autism, Down’s syndrome and Cognitive disabilities) experience delays in detection of chronic comorbidities such as diabetes, obesity, high blood… (more)

Subjects/Keywords: Developmental Disabilities, Medication Adherence, Medication Persistence, Healthcare Utilization, Healthcare Costs; Pharmacy and Pharmacology; Health Sciences

…42 2.2 Developmental Disabilities .. 45 2.2.1 Introduction… …utilization 50 2.2.7 Chronic disease management in patients with developmental disabilities… …63 2.2.8 Racial health disparities in patients with developmental disabilities …65… …2.2.9 Quality of care in diabetic adults with developmental disabilities …... 66 2.3… …Medicaid Enrollees with Developmental Disabilities and type 2 diabetes Abstract… 

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APA (6th Edition):

Patel, I. B. (2014). Medication use and Healthcare Outcomes in Developmentally Disabled Medicaid Adults with Type 2 Dibetes: a Quantitative Race Based Analysis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/107206

Chicago Manual of Style (16th Edition):

Patel, Isha B. “Medication use and Healthcare Outcomes in Developmentally Disabled Medicaid Adults with Type 2 Dibetes: a Quantitative Race Based Analysis.” 2014. Doctoral Dissertation, University of Michigan. Accessed January 16, 2021. http://hdl.handle.net/2027.42/107206.

MLA Handbook (7th Edition):

Patel, Isha B. “Medication use and Healthcare Outcomes in Developmentally Disabled Medicaid Adults with Type 2 Dibetes: a Quantitative Race Based Analysis.” 2014. Web. 16 Jan 2021.

Vancouver:

Patel IB. Medication use and Healthcare Outcomes in Developmentally Disabled Medicaid Adults with Type 2 Dibetes: a Quantitative Race Based Analysis. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2027.42/107206.

Council of Science Editors:

Patel IB. Medication use and Healthcare Outcomes in Developmentally Disabled Medicaid Adults with Type 2 Dibetes: a Quantitative Race Based Analysis. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/107206


Texas Medical Center

30. Hall, Mandy A. Increased geranylgeranylated K-Ras contributes to antineoplastic effects of farnesyltransferase inhibitors.

Degree: PhD, 2012, Texas Medical Center

  The Ras family of small GTPases (N-, H-, and K-Ras) is a group of important signaling mediators. Ras is frequently activated in some cancers,… (more)

Subjects/Keywords: Ras; Farnesyltransferase; Farnesyltransferase inhibitor; Geranylgeranylated; ERK MAPK; p38 MAPK; Cancer; Biochemistry, Biophysics, and Structural Biology; Biology; Cancer Biology; Cell and Developmental Biology; Diseases; Laboratory and Basic Science Research; Life Sciences; Lipids; Medicine and Health Sciences; Other Chemicals and Drugs; Pharmaceutical Preparations; Pharmacology, Toxicology and Environmental Health

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hall, M. A. (2012). Increased geranylgeranylated K-Ras contributes to antineoplastic effects of farnesyltransferase inhibitors. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/237

Chicago Manual of Style (16th Edition):

Hall, Mandy A. “Increased geranylgeranylated K-Ras contributes to antineoplastic effects of farnesyltransferase inhibitors.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed January 16, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/237.

MLA Handbook (7th Edition):

Hall, Mandy A. “Increased geranylgeranylated K-Ras contributes to antineoplastic effects of farnesyltransferase inhibitors.” 2012. Web. 16 Jan 2021.

Vancouver:

Hall MA. Increased geranylgeranylated K-Ras contributes to antineoplastic effects of farnesyltransferase inhibitors. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2021 Jan 16]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/237.

Council of Science Editors:

Hall MA. Increased geranylgeranylated K-Ras contributes to antineoplastic effects of farnesyltransferase inhibitors. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/237

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