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You searched for subject:(Desipramine). Showing records 1 – 8 of 8 total matches.

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University of Illinois – Urbana-Champaign

1. Brooks, Alexandra Kelly. Expression and regulation of the rate-limiting enzymes of the kynurenine pathway in the mouse brain.

Degree: PhD, Neuroscience, 2017, University of Illinois – Urbana-Champaign

 During the mid-1900’s, early anti-depressants were developed to increase levels of catecholamines within the brain, leading to the theory that depression symptomology was a result… (more)

Subjects/Keywords: Indoleamine; Kynurenine; Desipramine; Depression; Inflammation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brooks, A. K. (2017). Expression and regulation of the rate-limiting enzymes of the kynurenine pathway in the mouse brain. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/97581

Chicago Manual of Style (16th Edition):

Brooks, Alexandra Kelly. “Expression and regulation of the rate-limiting enzymes of the kynurenine pathway in the mouse brain.” 2017. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed October 23, 2019. http://hdl.handle.net/2142/97581.

MLA Handbook (7th Edition):

Brooks, Alexandra Kelly. “Expression and regulation of the rate-limiting enzymes of the kynurenine pathway in the mouse brain.” 2017. Web. 23 Oct 2019.

Vancouver:

Brooks AK. Expression and regulation of the rate-limiting enzymes of the kynurenine pathway in the mouse brain. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2017. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/2142/97581.

Council of Science Editors:

Brooks AK. Expression and regulation of the rate-limiting enzymes of the kynurenine pathway in the mouse brain. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2017. Available from: http://hdl.handle.net/2142/97581


Harvard University

2. Chang, Celene Hyunju. The Rat Ventromedial Prefrontal Cortex in the Neural Circuitries of Depression and Sleep.

Degree: PhD, Neuroscience, 2013, Harvard University

 Major depressive disorder (MDD) is a debilitating disorder affecting hundreds of millions of people worldwide. The etiology of the disease is unknown, and how antidepressant… (more)

Subjects/Keywords: Neurosciences; depression; desipramine; sleep; ventromedial prefrontal cortex

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APA (6th Edition):

Chang, C. H. (2013). The Rat Ventromedial Prefrontal Cortex in the Neural Circuitries of Depression and Sleep. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110430

Chicago Manual of Style (16th Edition):

Chang, Celene Hyunju. “The Rat Ventromedial Prefrontal Cortex in the Neural Circuitries of Depression and Sleep.” 2013. Doctoral Dissertation, Harvard University. Accessed October 23, 2019. http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110430.

MLA Handbook (7th Edition):

Chang, Celene Hyunju. “The Rat Ventromedial Prefrontal Cortex in the Neural Circuitries of Depression and Sleep.” 2013. Web. 23 Oct 2019.

Vancouver:

Chang CH. The Rat Ventromedial Prefrontal Cortex in the Neural Circuitries of Depression and Sleep. [Internet] [Doctoral dissertation]. Harvard University; 2013. [cited 2019 Oct 23]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110430.

Council of Science Editors:

Chang CH. The Rat Ventromedial Prefrontal Cortex in the Neural Circuitries of Depression and Sleep. [Doctoral Dissertation]. Harvard University; 2013. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:11110430


University of Rochester

3. Szpunar, Mercedes J.; Brown, Edward B. Evidence for Sympathetic Nervous System Regulation of Breast Cancer Progression.

Degree: PhD, 2012, University of Rochester

 Breast cancer patients experience chronic emotional stress. A major pathway underlying the stress response is sympathetic nervous system release of norepinephrine (NE) and stimulation of… (more)

Subjects/Keywords: Norepinephrine; Angiogenesis; VEGF; IL-6; Desipramine; Dexmedetomidine

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APA (6th Edition):

Szpunar, Mercedes J.; Brown, E. B. (2012). Evidence for Sympathetic Nervous System Regulation of Breast Cancer Progression. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25535

Chicago Manual of Style (16th Edition):

Szpunar, Mercedes J.; Brown, Edward B. “Evidence for Sympathetic Nervous System Regulation of Breast Cancer Progression.” 2012. Doctoral Dissertation, University of Rochester. Accessed October 23, 2019. http://hdl.handle.net/1802/25535.

MLA Handbook (7th Edition):

Szpunar, Mercedes J.; Brown, Edward B. “Evidence for Sympathetic Nervous System Regulation of Breast Cancer Progression.” 2012. Web. 23 Oct 2019.

Vancouver:

Szpunar, Mercedes J.; Brown EB. Evidence for Sympathetic Nervous System Regulation of Breast Cancer Progression. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/1802/25535.

Council of Science Editors:

Szpunar, Mercedes J.; Brown EB. Evidence for Sympathetic Nervous System Regulation of Breast Cancer Progression. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25535


Wright State University

4. Robinson, Sergei Alexander. AQUAPORIN 4 EXPRESSION AND DISTRIBUTION DURING OSMOTIC BRAIN EDEMA AND FOLLOWING CHRONIC TREATMENT OF DESIPRAMINE.

Degree: MS, Anatomy, 2011, Wright State University

 Osmotic brain edema or chronic treatment with desipramine alters brain water permeability. In this study we investigated aquaporin 4 expression and distribution in these two… (more)

Subjects/Keywords: Neurosciences; AQP4; aquaporin 4; BBB; blood-brain barrier; TCA; desipramine; astrocytes; water permeability

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APA (6th Edition):

Robinson, S. A. (2011). AQUAPORIN 4 EXPRESSION AND DISTRIBUTION DURING OSMOTIC BRAIN EDEMA AND FOLLOWING CHRONIC TREATMENT OF DESIPRAMINE. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1313150441

Chicago Manual of Style (16th Edition):

Robinson, Sergei Alexander. “AQUAPORIN 4 EXPRESSION AND DISTRIBUTION DURING OSMOTIC BRAIN EDEMA AND FOLLOWING CHRONIC TREATMENT OF DESIPRAMINE.” 2011. Masters Thesis, Wright State University. Accessed October 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=wright1313150441.

MLA Handbook (7th Edition):

Robinson, Sergei Alexander. “AQUAPORIN 4 EXPRESSION AND DISTRIBUTION DURING OSMOTIC BRAIN EDEMA AND FOLLOWING CHRONIC TREATMENT OF DESIPRAMINE.” 2011. Web. 23 Oct 2019.

Vancouver:

Robinson SA. AQUAPORIN 4 EXPRESSION AND DISTRIBUTION DURING OSMOTIC BRAIN EDEMA AND FOLLOWING CHRONIC TREATMENT OF DESIPRAMINE. [Internet] [Masters thesis]. Wright State University; 2011. [cited 2019 Oct 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1313150441.

Council of Science Editors:

Robinson SA. AQUAPORIN 4 EXPRESSION AND DISTRIBUTION DURING OSMOTIC BRAIN EDEMA AND FOLLOWING CHRONIC TREATMENT OF DESIPRAMINE. [Masters Thesis]. Wright State University; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1313150441

5. 千葉, 雅人. 三環系抗うつ薬イミプラミンとその活性代謝物デシプラミンの非線形体内動態 : Nonlinear pharmacokinetics of imipramine and its active metabolite, desipramine.

Degree: Chiba University / 千葉大学

研究科: 千葉大学大学院薬学研究院

学位:千大院薬博乙第80号

Advisors/Committee Members: 千葉大学大学院薬学研究院.

Subjects/Keywords: imipramine; desipramine; nonlinear; イミプラミン; デジプラミン; 非線形

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APA (6th Edition):

千葉, . (n.d.). 三環系抗うつ薬イミプラミンとその活性代謝物デシプラミンの非線形体内動態 : Nonlinear pharmacokinetics of imipramine and its active metabolite, desipramine. (Thesis). Chiba University / 千葉大学. Retrieved from http://opac.ll.chiba-u.jp/da/curator/900022688/

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

千葉, 雅人. “三環系抗うつ薬イミプラミンとその活性代謝物デシプラミンの非線形体内動態 : Nonlinear pharmacokinetics of imipramine and its active metabolite, desipramine.” Thesis, Chiba University / 千葉大学. Accessed October 23, 2019. http://opac.ll.chiba-u.jp/da/curator/900022688/.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

千葉, 雅人. “三環系抗うつ薬イミプラミンとその活性代謝物デシプラミンの非線形体内動態 : Nonlinear pharmacokinetics of imipramine and its active metabolite, desipramine.” Web. 23 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

千葉 . 三環系抗うつ薬イミプラミンとその活性代謝物デシプラミンの非線形体内動態 : Nonlinear pharmacokinetics of imipramine and its active metabolite, desipramine. [Internet] [Thesis]. Chiba University / 千葉大学; [cited 2019 Oct 23]. Available from: http://opac.ll.chiba-u.jp/da/curator/900022688/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

千葉 . 三環系抗うつ薬イミプラミンとその活性代謝物デシプラミンの非線形体内動態 : Nonlinear pharmacokinetics of imipramine and its active metabolite, desipramine. [Thesis]. Chiba University / 千葉大学; Available from: http://opac.ll.chiba-u.jp/da/curator/900022688/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Houston

6. Saha, Kaustuv. Changes in GRK3 and Norepinephrine Responsiveness in Locus Coeruleus Neurons are Associated with Learned Helplessness After Repeated Forced Swim Stress.

Degree: Pharmacological and Pharmaceutical Sciences, Department of, 2011, University of Houston

 In brain, locus coeruleus plays an important role in mediating the stress responses. Two important neurotransmitter/hormones released in locus coeruleus (LC) during exposure to stress… (more)

Subjects/Keywords: GKR3; LH; α2A-AR; CRF1-R; FORCED SWIM; DESIPRAMINE; ELECTROPHYSIOLOGY; CONCENTRATION RESPONSE CURVE; STRESS; BRAIN; NOREPINEPHRINE

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APA (6th Edition):

Saha, K. (2011). Changes in GRK3 and Norepinephrine Responsiveness in Locus Coeruleus Neurons are Associated with Learned Helplessness After Repeated Forced Swim Stress. (Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/242

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saha, Kaustuv. “Changes in GRK3 and Norepinephrine Responsiveness in Locus Coeruleus Neurons are Associated with Learned Helplessness After Repeated Forced Swim Stress.” 2011. Thesis, University of Houston. Accessed October 23, 2019. http://hdl.handle.net/10657/242.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saha, Kaustuv. “Changes in GRK3 and Norepinephrine Responsiveness in Locus Coeruleus Neurons are Associated with Learned Helplessness After Repeated Forced Swim Stress.” 2011. Web. 23 Oct 2019.

Vancouver:

Saha K. Changes in GRK3 and Norepinephrine Responsiveness in Locus Coeruleus Neurons are Associated with Learned Helplessness After Repeated Forced Swim Stress. [Internet] [Thesis]. University of Houston; 2011. [cited 2019 Oct 23]. Available from: http://hdl.handle.net/10657/242.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saha K. Changes in GRK3 and Norepinephrine Responsiveness in Locus Coeruleus Neurons are Associated with Learned Helplessness After Repeated Forced Swim Stress. [Thesis]. University of Houston; 2011. Available from: http://hdl.handle.net/10657/242

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

7. Kinne, Anita. Studies for thyroid hormone transport mediated by the membrane transporters of the MCT and LAT families.

Degree: 2011, Freie Universität Berlin

 The Allan-Herndon-Dudley syndrome (AHDS) is a severe X-linked disease characterized by movement disorders and mental retardation with speech problems. The disease is associated with mutations… (more)

Subjects/Keywords: AHDS; MCT8; Oatp14; Lat2; desipramine; thyroid hormone transport; thyroid hormone; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

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APA (6th Edition):

Kinne, A. (2011). Studies for thyroid hormone transport mediated by the membrane transporters of the MCT and LAT families. (Thesis). Freie Universität Berlin. Retrieved from https://refubium.fu-berlin.de/handle/fub188/11575

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kinne, Anita. “Studies for thyroid hormone transport mediated by the membrane transporters of the MCT and LAT families.” 2011. Thesis, Freie Universität Berlin. Accessed October 23, 2019. https://refubium.fu-berlin.de/handle/fub188/11575.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kinne, Anita. “Studies for thyroid hormone transport mediated by the membrane transporters of the MCT and LAT families.” 2011. Web. 23 Oct 2019.

Vancouver:

Kinne A. Studies for thyroid hormone transport mediated by the membrane transporters of the MCT and LAT families. [Internet] [Thesis]. Freie Universität Berlin; 2011. [cited 2019 Oct 23]. Available from: https://refubium.fu-berlin.de/handle/fub188/11575.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kinne A. Studies for thyroid hormone transport mediated by the membrane transporters of the MCT and LAT families. [Thesis]. Freie Universität Berlin; 2011. Available from: https://refubium.fu-berlin.de/handle/fub188/11575

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Cheetham, Chad Christopher. Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models.

Degree: PhD, 2011, University of Alabama – Birmingham

DYT1 dystonia is an inherited movement disorder caused by a trinucleotide deletion (DeltaGAG) in the DYT1 (TOR1A) gene, which codes for the torsinA protein. Dr.… (more)

Subjects/Keywords: Adrenergic Neurons – physiology.<; br>; Adrenergic Uptake Inhibitors – pharmacology.<; br>; Antidepressive Agents – pharmacology<; br>; Antidepressive Agents, Tricyclic – pharmacology.<; br>; Arrestins – physiology.<; br>; Desipramine – pharmacology.<; br>; Microfilament Proteins – physiology.<; br>; Mitogen-Activated Protein Kinase 1 – biosynthesis.<; br>; Mitogen-Activated Protein Kinase 3 – biosynthesis.<; br>; Nerve Tissue Proteins – physiology<; br>; Receptors, Adrenergic, alpha-2 – physiology.

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APA (6th Edition):

Cheetham, C. C. (2011). Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1433

Chicago Manual of Style (16th Edition):

Cheetham, Chad Christopher. “Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 23, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1433.

MLA Handbook (7th Edition):

Cheetham, Chad Christopher. “Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models.” 2011. Web. 23 Oct 2019.

Vancouver:

Cheetham CC. Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 23]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1433.

Council of Science Editors:

Cheetham CC. Pathophysiology Of Dyt1 Dystonia: Targeted Mouse Models. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1433

.