Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Department of Cell Biology AND Physiology). Showing records 1 – 30 of 3471 total matches.

[1] [2] [3] [4] [5] … [116]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of North Carolina

1. Wetzel-Strong, Sarah. Effect of adrenomedullin over-expression in the cardiovascular system during development and disease.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 Since the discovery of adrenomedullin (Adm - gene; AM - protein) in 1993, many roles for this widely expressed peptide have been described in the… (more)

Subjects/Keywords: Physiology; School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wetzel-Strong, S. (2015). Effect of adrenomedullin over-expression in the cardiovascular system during development and disease. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wetzel-Strong, Sarah. “Effect of adrenomedullin over-expression in the cardiovascular system during development and disease.” 2015. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wetzel-Strong, Sarah. “Effect of adrenomedullin over-expression in the cardiovascular system during development and disease.” 2015. Web. 24 Sep 2020.

Vancouver:

Wetzel-Strong S. Effect of adrenomedullin over-expression in the cardiovascular system during development and disease. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wetzel-Strong S. Effect of adrenomedullin over-expression in the cardiovascular system during development and disease. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:a0e5c581-09be-45c2-9181-f3dd435e726d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Klein, Klara. Adrenomedullin, Chemokine Receptors, and Receptor Activity Modifying Proteins in Lymphangiogenesis.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 Over the past decade, we have begun to appreciate that the lymphatic vascular system does more than simply return plasma back into the circulatory system… (more)

Subjects/Keywords: Physiology; Biology; Molecular biology; School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Klein, K. (2015). Adrenomedullin, Chemokine Receptors, and Receptor Activity Modifying Proteins in Lymphangiogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ef2c8a24-597a-4c0d-9642-2850b8f8557e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Klein, Klara. “Adrenomedullin, Chemokine Receptors, and Receptor Activity Modifying Proteins in Lymphangiogenesis.” 2015. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:ef2c8a24-597a-4c0d-9642-2850b8f8557e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Klein, Klara. “Adrenomedullin, Chemokine Receptors, and Receptor Activity Modifying Proteins in Lymphangiogenesis.” 2015. Web. 24 Sep 2020.

Vancouver:

Klein K. Adrenomedullin, Chemokine Receptors, and Receptor Activity Modifying Proteins in Lymphangiogenesis. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:ef2c8a24-597a-4c0d-9642-2850b8f8557e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Klein K. Adrenomedullin, Chemokine Receptors, and Receptor Activity Modifying Proteins in Lymphangiogenesis. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:ef2c8a24-597a-4c0d-9642-2850b8f8557e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Harrell, Jessica R. How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 Understanding morphogenesis, the spatial and temporal distribution of cells during development of an organism, is a key goal in studies in developmental biology. Throughout diverse… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harrell, J. R. (2010). How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Harrell, Jessica R. “How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme.” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Harrell, Jessica R. “How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme.” 2010. Web. 24 Sep 2020.

Vancouver:

Harrell JR. How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Harrell JR. How diverse cells position themselves in an embryo: variations on a common cytoskeletal theme. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:e5f2ee99-86c7-47b1-ac7f-60201771c102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. Newman, Martin A. The regulation of let-7 microRNA biogenesis in early embryogenesis.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 microRNAs (miRNAs) are small non-protein-coding RNAs that silence gene expression post-transcriptionally and have critical functions in development, tissue homeostasis and in the pathogenesis of many… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Newman, M. A. (2010). The regulation of let-7 microRNA biogenesis in early embryogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:5ea25c60-14e6-453a-a6fd-2ba6174d8375

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Newman, Martin A. “The regulation of let-7 microRNA biogenesis in early embryogenesis.” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:5ea25c60-14e6-453a-a6fd-2ba6174d8375.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Newman, Martin A. “The regulation of let-7 microRNA biogenesis in early embryogenesis.” 2010. Web. 24 Sep 2020.

Vancouver:

Newman MA. The regulation of let-7 microRNA biogenesis in early embryogenesis. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:5ea25c60-14e6-453a-a6fd-2ba6174d8375.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Newman MA. The regulation of let-7 microRNA biogenesis in early embryogenesis. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:5ea25c60-14e6-453a-a6fd-2ba6174d8375

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Hamilton, Kathryn Elizabeth. Role of suppressor of cytokine signaling 3 in colorectal cancer.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 Patients with inflammatory bowel diseases (IBD) have an increased lifetime risk of developing colorectal cancer (CRC). Suppressors of cytokine signaling (SOCS) are intracellular proteins that… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hamilton, K. E. (2010). Role of suppressor of cytokine signaling 3 in colorectal cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hamilton, Kathryn Elizabeth. “Role of suppressor of cytokine signaling 3 in colorectal cancer.” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hamilton, Kathryn Elizabeth. “Role of suppressor of cytokine signaling 3 in colorectal cancer.” 2010. Web. 24 Sep 2020.

Vancouver:

Hamilton KE. Role of suppressor of cytokine signaling 3 in colorectal cancer. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hamilton KE. Role of suppressor of cytokine signaling 3 in colorectal cancer. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:095fdc60-260b-46ee-81c2-299f00899349

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Fellner, Robert C. Prouroguanylin’s role in the homeostatic response to dietary sodium: hormone precursor or active signaling agent?.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 High blood pressure affects more than 70 million Americans, putting them at a much greater risk for heart disease or a stroke. Sodium (Na+) is… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fellner, R. C. (2010). Prouroguanylin’s role in the homeostatic response to dietary sodium: hormone precursor or active signaling agent?. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:58233f79-43ae-4eb6-81c7-d73394886510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fellner, Robert C. “Prouroguanylin’s role in the homeostatic response to dietary sodium: hormone precursor or active signaling agent?.” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:58233f79-43ae-4eb6-81c7-d73394886510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fellner, Robert C. “Prouroguanylin’s role in the homeostatic response to dietary sodium: hormone precursor or active signaling agent?.” 2010. Web. 24 Sep 2020.

Vancouver:

Fellner RC. Prouroguanylin’s role in the homeostatic response to dietary sodium: hormone precursor or active signaling agent?. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:58233f79-43ae-4eb6-81c7-d73394886510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fellner RC. Prouroguanylin’s role in the homeostatic response to dietary sodium: hormone precursor or active signaling agent?. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:58233f79-43ae-4eb6-81c7-d73394886510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Rao, Deepti. Neuromodulation of intrinsic and synaptic plasticity in auditory cortex.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 The study of auditory system development and plasticity is important. Large populations of people suffer from hearing loss throughout their lifetime. To optimize treatment for… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rao, D. (2011). Neuromodulation of intrinsic and synaptic plasticity in auditory cortex. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:73690b27-6c99-44fe-8131-af4ab785fe99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rao, Deepti. “Neuromodulation of intrinsic and synaptic plasticity in auditory cortex.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:73690b27-6c99-44fe-8131-af4ab785fe99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rao, Deepti. “Neuromodulation of intrinsic and synaptic plasticity in auditory cortex.” 2011. Web. 24 Sep 2020.

Vancouver:

Rao D. Neuromodulation of intrinsic and synaptic plasticity in auditory cortex. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:73690b27-6c99-44fe-8131-af4ab785fe99.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rao D. Neuromodulation of intrinsic and synaptic plasticity in auditory cortex. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:73690b27-6c99-44fe-8131-af4ab785fe99

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Kadmiel, Mahita. Role of receptor activity modifying protein-2 (RAMP2) in endocrine physiology of female mice.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Receptor activity modifying proteins (RAMPs 1, 2, and 3) are single-pass transmembrane proteins that can regulate the trafficking, ligand-binding, and signaling of several G protein-coupled… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kadmiel, M. (2011). Role of receptor activity modifying protein-2 (RAMP2) in endocrine physiology of female mice. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9d6c630f-a00d-433f-ab02-d2db0c6d5cef

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kadmiel, Mahita. “Role of receptor activity modifying protein-2 (RAMP2) in endocrine physiology of female mice.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:9d6c630f-a00d-433f-ab02-d2db0c6d5cef.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kadmiel, Mahita. “Role of receptor activity modifying protein-2 (RAMP2) in endocrine physiology of female mice.” 2011. Web. 24 Sep 2020.

Vancouver:

Kadmiel M. Role of receptor activity modifying protein-2 (RAMP2) in endocrine physiology of female mice. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:9d6c630f-a00d-433f-ab02-d2db0c6d5cef.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kadmiel M. Role of receptor activity modifying protein-2 (RAMP2) in endocrine physiology of female mice. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:9d6c630f-a00d-433f-ab02-d2db0c6d5cef

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Haddock, Brookelyn J. The role of MARCKS in mast cell regulated exocytosis.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 MARCKS is implicated as a critical regulator of regulated exocytosis. We studied the role of MARCKS in mast cell regulated exocytosis by comparing the secretion… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haddock, B. J. (2010). The role of MARCKS in mast cell regulated exocytosis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:2c1b4bbe-9b9a-429b-9a34-b74807cec8a3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haddock, Brookelyn J. “The role of MARCKS in mast cell regulated exocytosis.” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:2c1b4bbe-9b9a-429b-9a34-b74807cec8a3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haddock, Brookelyn J. “The role of MARCKS in mast cell regulated exocytosis.” 2010. Web. 24 Sep 2020.

Vancouver:

Haddock BJ. The role of MARCKS in mast cell regulated exocytosis. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:2c1b4bbe-9b9a-429b-9a34-b74807cec8a3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haddock BJ. The role of MARCKS in mast cell regulated exocytosis. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:2c1b4bbe-9b9a-429b-9a34-b74807cec8a3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Newton, Victoria Ann. Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer.

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 The insulin-like growth factor (IGF-I) pathway is associated with increased risk and progression of colorectal cancer. Understanding mechanisms that inhibit or promote this key pathway… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Newton, V. A. (2010). Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Newton, Victoria Ann. “Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer.” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Newton, Victoria Ann. “Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer.” 2010. Web. 24 Sep 2020.

Vancouver:

Newton VA. Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Newton VA. Suppressor of cytokine signaling-2 (SOCS2) and epidermal growth factor (EGF) modulate insulin-like growth factor-I receptor (IGF-IR) signaling in intestinal cancer. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:6fc8c0f8-378d-46a4-b570-a706ccfd67ef

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Gracz, Adam David. The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Stem cell biology, though a well-established concept in the scientific zeitgeist, is only beginning to emerge as an independent field of study. An understanding of… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gracz, A. D. (2011). The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gracz, Adam David. “The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gracz, Adam David. “The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract.” 2011. Web. 24 Sep 2020.

Vancouver:

Gracz AD. The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gracz AD. The role of Sry-Box (Sox) transcription factors in epithelial stem cell biology of the gastrointestinal tract. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:b263fe64-d6f7-4056-90b7-51d43225a651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Gee, Stephen Timothy. The role of Yes-associated protein (YAP) in vertebrate development.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Yes-associated protein 65 (YAP) contains multiple protein-protein interaction domains and functions as both a transcriptional co-activator and as a scaffolding protein within the cytoplasm or… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gee, S. T. (2011). The role of Yes-associated protein (YAP) in vertebrate development. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b8b3739c-65c2-408d-9596-a11dd7dd6af4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gee, Stephen Timothy. “The role of Yes-associated protein (YAP) in vertebrate development.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:b8b3739c-65c2-408d-9596-a11dd7dd6af4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gee, Stephen Timothy. “The role of Yes-associated protein (YAP) in vertebrate development.” 2011. Web. 24 Sep 2020.

Vancouver:

Gee ST. The role of Yes-associated protein (YAP) in vertebrate development. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:b8b3739c-65c2-408d-9596-a11dd7dd6af4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gee ST. The role of Yes-associated protein (YAP) in vertebrate development. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:b8b3739c-65c2-408d-9596-a11dd7dd6af4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Lendrum, Stephanie G. Drosophila melanogaster Myosin-18 Represents a Highly Divergent Molecular Motor with Actin Tethering Properties.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Myosins are actin-based molecular motors that use energy from ATP hydrolysis to perform work within the cell. On a cellular scale their influence is vast,… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lendrum, S. G. (2011). Drosophila melanogaster Myosin-18 Represents a Highly Divergent Molecular Motor with Actin Tethering Properties. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a06964e9-b458-4dac-8aba-2c244ed690c4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lendrum, Stephanie G. “Drosophila melanogaster Myosin-18 Represents a Highly Divergent Molecular Motor with Actin Tethering Properties.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:a06964e9-b458-4dac-8aba-2c244ed690c4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lendrum, Stephanie G. “Drosophila melanogaster Myosin-18 Represents a Highly Divergent Molecular Motor with Actin Tethering Properties.” 2011. Web. 24 Sep 2020.

Vancouver:

Lendrum SG. Drosophila melanogaster Myosin-18 Represents a Highly Divergent Molecular Motor with Actin Tethering Properties. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:a06964e9-b458-4dac-8aba-2c244ed690c4.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lendrum SG. Drosophila melanogaster Myosin-18 Represents a Highly Divergent Molecular Motor with Actin Tethering Properties. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:a06964e9-b458-4dac-8aba-2c244ed690c4

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Aghajanian, Amir. A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 The endothelial lining of the vasculature plays a critical role in regulating the passage of fluid, macromolecules, and cells between the blood and surrounding tissues.… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aghajanian, A. (2011). A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aghajanian, Amir. “A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aghajanian, Amir. “A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration.” 2011. Web. 24 Sep 2020.

Vancouver:

Aghajanian A. A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aghajanian A. A Novel Role for Reactive Oxygen Species in the Regulation of RhoA: Implications for Endothelial Permeability and Leukocyte Transmigration. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:e4986982-9dec-4e6f-99f4-fc1035e50693

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Tribble, Emily K. On the role of the phosphatidylinositol transfer protein alpha in the nuclear import of phosphatidylinositol.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Lipid metabolism within the nuclear matrix of mammalian cells is robust. Numerous lipid-signaling pathway components are found within purified nuclear fractions, depleted of nuclear envelope… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tribble, E. K. (2011). On the role of the phosphatidylinositol transfer protein alpha in the nuclear import of phosphatidylinositol. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:689ebf04-478a-42f7-a946-ac62b546ce3e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tribble, Emily K. “On the role of the phosphatidylinositol transfer protein alpha in the nuclear import of phosphatidylinositol.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:689ebf04-478a-42f7-a946-ac62b546ce3e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tribble, Emily K. “On the role of the phosphatidylinositol transfer protein alpha in the nuclear import of phosphatidylinositol.” 2011. Web. 24 Sep 2020.

Vancouver:

Tribble EK. On the role of the phosphatidylinositol transfer protein alpha in the nuclear import of phosphatidylinositol. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:689ebf04-478a-42f7-a946-ac62b546ce3e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tribble EK. On the role of the phosphatidylinositol transfer protein alpha in the nuclear import of phosphatidylinositol. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:689ebf04-478a-42f7-a946-ac62b546ce3e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

16. Luk, Flora C. Role of post translational modifications: glycosylation and palmitoylation in Toxoplasma gondii.

Degree: Cell Biology and Physiology, 2011, University of North Carolina

 Many eukaryotic proteins are post-translationally modified to regulate protein-protein interactions, protein stability, folding, localization, and proteolysis. Modified proteins play essential roles in cellular processes such… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Luk, F. C. (2011). Role of post translational modifications: glycosylation and palmitoylation in Toxoplasma gondii. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7dd2d8ff-1920-4af2-9344-54825bc3efa8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luk, Flora C. “Role of post translational modifications: glycosylation and palmitoylation in Toxoplasma gondii.” 2011. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:7dd2d8ff-1920-4af2-9344-54825bc3efa8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luk, Flora C. “Role of post translational modifications: glycosylation and palmitoylation in Toxoplasma gondii.” 2011. Web. 24 Sep 2020.

Vancouver:

Luk FC. Role of post translational modifications: glycosylation and palmitoylation in Toxoplasma gondii. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:7dd2d8ff-1920-4af2-9344-54825bc3efa8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luk FC. Role of post translational modifications: glycosylation and palmitoylation in Toxoplasma gondii. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:7dd2d8ff-1920-4af2-9344-54825bc3efa8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

17. Hast, Bridgid Elizabeth. Mechanisms controlling the KEAP1-NRF2 signaling pathway in lung cancer.

Degree: Cell Biology and Physiology, 2013, University of North Carolina

 An ability to effectively regulate intracellular reactive oxygen species is imperative to prevent conditions of oxidative stress, and ultimately aberrant cell death. The primary means… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hast, B. E. (2013). Mechanisms controlling the KEAP1-NRF2 signaling pathway in lung cancer. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:bf8cdb47-b0c3-4f0d-aabc-5c47bd650dcf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hast, Bridgid Elizabeth. “Mechanisms controlling the KEAP1-NRF2 signaling pathway in lung cancer.” 2013. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:bf8cdb47-b0c3-4f0d-aabc-5c47bd650dcf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hast, Bridgid Elizabeth. “Mechanisms controlling the KEAP1-NRF2 signaling pathway in lung cancer.” 2013. Web. 24 Sep 2020.

Vancouver:

Hast BE. Mechanisms controlling the KEAP1-NRF2 signaling pathway in lung cancer. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:bf8cdb47-b0c3-4f0d-aabc-5c47bd650dcf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hast BE. Mechanisms controlling the KEAP1-NRF2 signaling pathway in lung cancer. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:bf8cdb47-b0c3-4f0d-aabc-5c47bd650dcf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

18. Lenhart, Patricia Marie. Adrenomedullin and Receptor Activity Modifying Protein 3 in Women's Health.

Degree: Cell Biology and Physiology, 2013, University of North Carolina

 Adrenomedullin (AM) is a 52-amino acid peptide vasodilator that is elevated in the serum during normal human pregnancies but is often blunted in pregnancy complications… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lenhart, P. M. (2013). Adrenomedullin and Receptor Activity Modifying Protein 3 in Women's Health. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ca2b0e44-03cd-4c3a-9821-dd5663b9ef36

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lenhart, Patricia Marie. “Adrenomedullin and Receptor Activity Modifying Protein 3 in Women's Health.” 2013. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:ca2b0e44-03cd-4c3a-9821-dd5663b9ef36.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lenhart, Patricia Marie. “Adrenomedullin and Receptor Activity Modifying Protein 3 in Women's Health.” 2013. Web. 24 Sep 2020.

Vancouver:

Lenhart PM. Adrenomedullin and Receptor Activity Modifying Protein 3 in Women's Health. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:ca2b0e44-03cd-4c3a-9821-dd5663b9ef36.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lenhart PM. Adrenomedullin and Receptor Activity Modifying Protein 3 in Women's Health. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:ca2b0e44-03cd-4c3a-9821-dd5663b9ef36

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

19. Nile, Aaron Hugh. Chemical Inhibitors of Phosphatidylinositol Transfer Proteins Enable Highly Selective Interference With Specific Pathways of Phosphoinositide Signaling in Cells.

Degree: Cell Biology and Physiology, 2014, University of North Carolina

 Phosphatidylinositol phosphates (PIP) are phosphorylated derivatives of phosphatidylinositol (PtdIns) that signal to and regulate diverse cellular functions including membrane trafficking, cytokinesis, cell cycle regulation and… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nile, A. H. (2014). Chemical Inhibitors of Phosphatidylinositol Transfer Proteins Enable Highly Selective Interference With Specific Pathways of Phosphoinositide Signaling in Cells. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:49923399-32a8-4874-b9f4-2bd1220d9fe2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nile, Aaron Hugh. “Chemical Inhibitors of Phosphatidylinositol Transfer Proteins Enable Highly Selective Interference With Specific Pathways of Phosphoinositide Signaling in Cells.” 2014. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:49923399-32a8-4874-b9f4-2bd1220d9fe2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nile, Aaron Hugh. “Chemical Inhibitors of Phosphatidylinositol Transfer Proteins Enable Highly Selective Interference With Specific Pathways of Phosphoinositide Signaling in Cells.” 2014. Web. 24 Sep 2020.

Vancouver:

Nile AH. Chemical Inhibitors of Phosphatidylinositol Transfer Proteins Enable Highly Selective Interference With Specific Pathways of Phosphoinositide Signaling in Cells. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:49923399-32a8-4874-b9f4-2bd1220d9fe2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nile AH. Chemical Inhibitors of Phosphatidylinositol Transfer Proteins Enable Highly Selective Interference With Specific Pathways of Phosphoinositide Signaling in Cells. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:49923399-32a8-4874-b9f4-2bd1220d9fe2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

20. Tolbert, Caitlin Elizabeth. Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function.

Degree: Cell Biology and Physiology, 2013, University of North Carolina

 Vinculin is an essential adhesion protein involved in controlling motility and force transduction, in part by coupling the actin cytoskeleton to the extracellular matrix. Vinculin… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tolbert, C. E. (2013). Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tolbert, Caitlin Elizabeth. “Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function.” 2013. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tolbert, Caitlin Elizabeth. “Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function.” 2013. Web. 24 Sep 2020.

Vancouver:

Tolbert CE. Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tolbert CE. Characterizing the Role of Actin Binding, Bundling, and Tyrosine Phosphorylation in Modulating Vinculin Function. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:6e6836c7-afdf-4aa9-9fe0-d93e30cbe2b5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

21. Collins, Caitlin. Understanding PECAM-1-mediated mechanotransduction: from the protein to the vessel.

Degree: Cell Biology and Physiology, 2013, University of North Carolina

 Hemodynamic forces are critical for endothelial cell (EC) function and vessel health. Platelet endothelial cell adhesion-1 (PECAM-1) has been identified as a critical endothelial mechanosensor… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Collins, C. (2013). Understanding PECAM-1-mediated mechanotransduction: from the protein to the vessel. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:2e2df269-4bec-47f4-bcab-355e713fa432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Collins, Caitlin. “Understanding PECAM-1-mediated mechanotransduction: from the protein to the vessel.” 2013. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:2e2df269-4bec-47f4-bcab-355e713fa432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Collins, Caitlin. “Understanding PECAM-1-mediated mechanotransduction: from the protein to the vessel.” 2013. Web. 24 Sep 2020.

Vancouver:

Collins C. Understanding PECAM-1-mediated mechanotransduction: from the protein to the vessel. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:2e2df269-4bec-47f4-bcab-355e713fa432.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Collins C. Understanding PECAM-1-mediated mechanotransduction: from the protein to the vessel. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:2e2df269-4bec-47f4-bcab-355e713fa432

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

22. Gracz, Adam D. Intrinsic and extrinsic regulation of potency in the intestinal stem cell niche.

Degree: Cell Biology and Physiology, 2013, University of North Carolina

 The intestinal epithelium is one of the most proliferative tissues in the adult body, undergoing near total renewal every 5-7 days. This remarkable turnover is… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gracz, A. D. (2013). Intrinsic and extrinsic regulation of potency in the intestinal stem cell niche. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:220a46ad-110a-48fb-8504-e4f3e32ec959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gracz, Adam D. “Intrinsic and extrinsic regulation of potency in the intestinal stem cell niche.” 2013. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:220a46ad-110a-48fb-8504-e4f3e32ec959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gracz, Adam D. “Intrinsic and extrinsic regulation of potency in the intestinal stem cell niche.” 2013. Web. 24 Sep 2020.

Vancouver:

Gracz AD. Intrinsic and extrinsic regulation of potency in the intestinal stem cell niche. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:220a46ad-110a-48fb-8504-e4f3e32ec959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gracz AD. Intrinsic and extrinsic regulation of potency in the intestinal stem cell niche. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:220a46ad-110a-48fb-8504-e4f3e32ec959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

23. Larsen, Rylan Scott. Subtype-specific roles for presynaptic NMDA receptors in experience-dependent plasticity and visual cortical development.

Degree: Cell Biology and Physiology, 2013, University of North Carolina

 A defining property of the brain is its ability to modify neuronal circuits in response to sensory stimuli to allow for adaptive responses to the… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Larsen, R. S. (2013). Subtype-specific roles for presynaptic NMDA receptors in experience-dependent plasticity and visual cortical development. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:7e255891-7582-411b-935e-eda43f3b3158

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Larsen, Rylan Scott. “Subtype-specific roles for presynaptic NMDA receptors in experience-dependent plasticity and visual cortical development.” 2013. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:7e255891-7582-411b-935e-eda43f3b3158.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Larsen, Rylan Scott. “Subtype-specific roles for presynaptic NMDA receptors in experience-dependent plasticity and visual cortical development.” 2013. Web. 24 Sep 2020.

Vancouver:

Larsen RS. Subtype-specific roles for presynaptic NMDA receptors in experience-dependent plasticity and visual cortical development. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:7e255891-7582-411b-935e-eda43f3b3158.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Larsen RS. Subtype-specific roles for presynaptic NMDA receptors in experience-dependent plasticity and visual cortical development. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:7e255891-7582-411b-935e-eda43f3b3158

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

24. Haynes, Elizabeth. GMFβ Controls Branched Actin Content and Lamellipodial Retraction in Fibroblasts.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 The lamellipodium is an important structure for cell migration containing branched actin nucleated via the Arp2/3 complex. The formation of branched actin is relatively well… (more)

Subjects/Keywords: Cytology; School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haynes, E. (2015). GMFβ Controls Branched Actin Content and Lamellipodial Retraction in Fibroblasts. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e5600e13-7990-465c-b3d2-981e18a7c4e0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Haynes, Elizabeth. “GMFβ Controls Branched Actin Content and Lamellipodial Retraction in Fibroblasts.” 2015. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:e5600e13-7990-465c-b3d2-981e18a7c4e0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Haynes, Elizabeth. “GMFβ Controls Branched Actin Content and Lamellipodial Retraction in Fibroblasts.” 2015. Web. 24 Sep 2020.

Vancouver:

Haynes E. GMFβ Controls Branched Actin Content and Lamellipodial Retraction in Fibroblasts. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:e5600e13-7990-465c-b3d2-981e18a7c4e0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Haynes E. GMFβ Controls Branched Actin Content and Lamellipodial Retraction in Fibroblasts. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:e5600e13-7990-465c-b3d2-981e18a7c4e0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

25. Jones, Stephen. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.

Degree: Cell Biology and Physiology, 2015, University of North Carolina

 Extended circulation of nanoparticles in blood is essential for most clinical applications. Nanoparticles are rapidly cleared by cells of the mononuclear phagocyte system (MPS). Approaches… (more)

Subjects/Keywords: Cytology; School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jones, S. (2015). Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jones, Stephen. “Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.” 2015. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jones, Stephen. “Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background.” 2015. Web. 24 Sep 2020.

Vancouver:

Jones S. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jones S. Nanoparticle Clearance is Governed by Th1/Th2 Immunity and Strain Background. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:5180f990-8a54-4ac9-aec1-e00cd03b9ab3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

26. Rollins, Brett. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).

Degree: Cell Biology and Physiology, 2010, University of North Carolina

 The airways rely on mucociliary clearance (MCC) to remove inhaled particulates. Initiating events in chronic airway diseases, such as cystic fibrosis (CF), have been traced… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rollins, B. (2010). Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rollins, Brett. “Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).” 2010. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rollins, Brett. “Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1).” 2010. Web. 24 Sep 2020.

Vancouver:

Rollins B. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). [Internet] [Thesis]. University of North Carolina; 2010. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rollins B. Regulation of the Epithelial Sodium Channel (ENaC) by the Short Palate, Lung, and Nasal Epithelial Clone (SPLUNC1). [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:23a7af78-feaa-4a84-8f8f-be2a38b9c449

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

27. Xiao, Lin. REGULATION OF ENDOTHELIAL PLASTICITY BY TGFβ IN TUMORS.

Degree: Cell Biology and Physiology, 2016, University of North Carolina

 Tumor endothelial cells (TEC) are abnormal in morphology, structure, and function. They exhibit high cellular plasticity and may acquire stem-cell features in response to the… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Xiao, L. (2016). REGULATION OF ENDOTHELIAL PLASTICITY BY TGFβ IN TUMORS. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9ff020fe-d670-42f7-8d69-93ad16b5709d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xiao, Lin. “REGULATION OF ENDOTHELIAL PLASTICITY BY TGFβ IN TUMORS.” 2016. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:9ff020fe-d670-42f7-8d69-93ad16b5709d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xiao, Lin. “REGULATION OF ENDOTHELIAL PLASTICITY BY TGFβ IN TUMORS.” 2016. Web. 24 Sep 2020.

Vancouver:

Xiao L. REGULATION OF ENDOTHELIAL PLASTICITY BY TGFβ IN TUMORS. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:9ff020fe-d670-42f7-8d69-93ad16b5709d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xiao L. REGULATION OF ENDOTHELIAL PLASTICITY BY TGFβ IN TUMORS. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:9ff020fe-d670-42f7-8d69-93ad16b5709d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

28. Dunleavey, James. PECAM-1-DEPENDENT VASCULAR MIMICRY IN MELANOMA: CONTRIBUTIONS TO ANTI-ANGIOGENIC RESISTANCE.

Degree: Cell Biology and Physiology, 2016, University of North Carolina

 The development of a perfused blood vasculature is a requirement both in development and for many human pathologies. Critically, the formation of new blood vessels… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dunleavey, J. (2016). PECAM-1-DEPENDENT VASCULAR MIMICRY IN MELANOMA: CONTRIBUTIONS TO ANTI-ANGIOGENIC RESISTANCE. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:cab5d7bc-9082-401f-95d7-c632f7cd1027

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dunleavey, James. “PECAM-1-DEPENDENT VASCULAR MIMICRY IN MELANOMA: CONTRIBUTIONS TO ANTI-ANGIOGENIC RESISTANCE.” 2016. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:cab5d7bc-9082-401f-95d7-c632f7cd1027.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dunleavey, James. “PECAM-1-DEPENDENT VASCULAR MIMICRY IN MELANOMA: CONTRIBUTIONS TO ANTI-ANGIOGENIC RESISTANCE.” 2016. Web. 24 Sep 2020.

Vancouver:

Dunleavey J. PECAM-1-DEPENDENT VASCULAR MIMICRY IN MELANOMA: CONTRIBUTIONS TO ANTI-ANGIOGENIC RESISTANCE. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:cab5d7bc-9082-401f-95d7-c632f7cd1027.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dunleavey J. PECAM-1-DEPENDENT VASCULAR MIMICRY IN MELANOMA: CONTRIBUTIONS TO ANTI-ANGIOGENIC RESISTANCE. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:cab5d7bc-9082-401f-95d7-c632f7cd1027

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

29. Lang, Patrick. ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development.

Degree: Cell Biology and Physiology, 2017, University of North Carolina

 Microcephaly and brain tumors can both arise as a consequence of dysregulated expansion of neural progenitor cells. Inadequate progenitor proliferation, premature cell cycle exit, and… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lang, P. (2017). ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lang, Patrick. “ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development.” 2017. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lang, Patrick. “ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development.” 2017. Web. 24 Sep 2020.

Vancouver:

Lang P. ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lang P. ATR is a Novel Therapeutic Target for Medulloblastoma Identified by its Role in Cerebellar Development. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:08fa1633-1467-4166-81f0-2aabbd239f0f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

30. Nwachukwu, Kala. THE EFFECTS OF IONS ON MUCIN RHEOLOGY AND ORGANIZATION.

Degree: Cell Biology and Physiology, 2016, University of North Carolina

 Increased mucus thickness is a common symptom of pulmonary diseases, such as cystic fibrosis (CF) and also occurs in chronic cigarette smokers. The thickening of… (more)

Subjects/Keywords: School of Medicine; Department of Cell Biology and Physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nwachukwu, K. (2016). THE EFFECTS OF IONS ON MUCIN RHEOLOGY AND ORGANIZATION. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d9e560a1-8e85-4e18-9651-cb8d998f022e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nwachukwu, Kala. “THE EFFECTS OF IONS ON MUCIN RHEOLOGY AND ORGANIZATION.” 2016. Thesis, University of North Carolina. Accessed September 24, 2020. https://cdr.lib.unc.edu/record/uuid:d9e560a1-8e85-4e18-9651-cb8d998f022e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nwachukwu, Kala. “THE EFFECTS OF IONS ON MUCIN RHEOLOGY AND ORGANIZATION.” 2016. Web. 24 Sep 2020.

Vancouver:

Nwachukwu K. THE EFFECTS OF IONS ON MUCIN RHEOLOGY AND ORGANIZATION. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2020 Sep 24]. Available from: https://cdr.lib.unc.edu/record/uuid:d9e560a1-8e85-4e18-9651-cb8d998f022e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nwachukwu K. THE EFFECTS OF IONS ON MUCIN RHEOLOGY AND ORGANIZATION. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:d9e560a1-8e85-4e18-9651-cb8d998f022e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [116]

.