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You searched for subject:(Dendritic cell migration). Showing records 1 – 16 of 16 total matches.

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Penn State University

1. Fischer, Matthew Anthony. The Immune Response to Vaccinia Virus Infection: Inflammatory Monocytes, Leukotrienes, and CD8+ T Cell Immunodominance.

Degree: PhD, Microbiology and Immunology, 2009, Penn State University

 Although Vaccinia virus (VACV) has been used extensively as a vaccine to convey protection against other poxviruses and, through the expression of recombinant antigens, other… (more)

Subjects/Keywords: immunodominance; leukotrienes; inflammatory monocytes; Vaccinia virus; dendritic cell migration

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APA (6th Edition):

Fischer, M. A. (2009). The Immune Response to Vaccinia Virus Infection: Inflammatory Monocytes, Leukotrienes, and CD8+ T Cell Immunodominance. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/10147

Chicago Manual of Style (16th Edition):

Fischer, Matthew Anthony. “The Immune Response to Vaccinia Virus Infection: Inflammatory Monocytes, Leukotrienes, and CD8+ T Cell Immunodominance.” 2009. Doctoral Dissertation, Penn State University. Accessed October 21, 2019. https://etda.libraries.psu.edu/catalog/10147.

MLA Handbook (7th Edition):

Fischer, Matthew Anthony. “The Immune Response to Vaccinia Virus Infection: Inflammatory Monocytes, Leukotrienes, and CD8+ T Cell Immunodominance.” 2009. Web. 21 Oct 2019.

Vancouver:

Fischer MA. The Immune Response to Vaccinia Virus Infection: Inflammatory Monocytes, Leukotrienes, and CD8+ T Cell Immunodominance. [Internet] [Doctoral dissertation]. Penn State University; 2009. [cited 2019 Oct 21]. Available from: https://etda.libraries.psu.edu/catalog/10147.

Council of Science Editors:

Fischer MA. The Immune Response to Vaccinia Virus Infection: Inflammatory Monocytes, Leukotrienes, and CD8+ T Cell Immunodominance. [Doctoral Dissertation]. Penn State University; 2009. Available from: https://etda.libraries.psu.edu/catalog/10147

2. Batich, Kristen Anne. Enhancing Dendritic Cell Migration to Drive Antitumor Responses .

Degree: 2017, Duke University

  The histologic subtypes of malignant glial neoplasms range from anaplastic astrocytoma to the most deadly World Health Organization (WHO) Grade IV glioblastoma (GBM), the… (more)

Subjects/Keywords: Immunology; Biology; dendritic cell; glioblastoma; malignant glioma; migration; tumor immunology

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APA (6th Edition):

Batich, K. A. (2017). Enhancing Dendritic Cell Migration to Drive Antitumor Responses . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/10453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Batich, Kristen Anne. “Enhancing Dendritic Cell Migration to Drive Antitumor Responses .” 2017. Thesis, Duke University. Accessed October 21, 2019. http://hdl.handle.net/10161/10453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Batich, Kristen Anne. “Enhancing Dendritic Cell Migration to Drive Antitumor Responses .” 2017. Web. 21 Oct 2019.

Vancouver:

Batich KA. Enhancing Dendritic Cell Migration to Drive Antitumor Responses . [Internet] [Thesis]. Duke University; 2017. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/10161/10453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Batich KA. Enhancing Dendritic Cell Migration to Drive Antitumor Responses . [Thesis]. Duke University; 2017. Available from: http://hdl.handle.net/10161/10453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Evans, Joseph Corey. Regulation of Dendritic Spine Development and Cell Migration through Asef2-mediated Signaling.

Degree: PhD, Biological Sciences, 2014, Vanderbilt University

 Rho GTPases are molecular switches that mediate the formation of dendritic spines â actin-enriched protrusions that make excitatory synapses with nearby neurons. Once activated by… (more)

Subjects/Keywords: dendritic spine; Guanine nucleotide exchange factor; neuron; Rho GTPases; cell migration

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APA (6th Edition):

Evans, J. C. (2014). Regulation of Dendritic Spine Development and Cell Migration through Asef2-mediated Signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07232014-132105/ ;

Chicago Manual of Style (16th Edition):

Evans, Joseph Corey. “Regulation of Dendritic Spine Development and Cell Migration through Asef2-mediated Signaling.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed October 21, 2019. http://etd.library.vanderbilt.edu/available/etd-07232014-132105/ ;.

MLA Handbook (7th Edition):

Evans, Joseph Corey. “Regulation of Dendritic Spine Development and Cell Migration through Asef2-mediated Signaling.” 2014. Web. 21 Oct 2019.

Vancouver:

Evans JC. Regulation of Dendritic Spine Development and Cell Migration through Asef2-mediated Signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2019 Oct 21]. Available from: http://etd.library.vanderbilt.edu/available/etd-07232014-132105/ ;.

Council of Science Editors:

Evans JC. Regulation of Dendritic Spine Development and Cell Migration through Asef2-mediated Signaling. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu/available/etd-07232014-132105/ ;


University of Toronto

4. Rajkumar, Andrew. The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells.

Degree: 2018, University of Toronto

Intimal dendritic-like cells (DCs) reside in regions of the aorta predisposed to atherosclerosis. LPS and interleukin-1 beta (IL-1β) injections into C57BL/6 mice result in decreased… (more)

Subjects/Keywords: Dendritic Cell; Interleukin-1 Beta; Intima; Lipopolysaccharide; Reverse Transendothelial Migration; 0982

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APA (6th Edition):

Rajkumar, A. (2018). The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/91394

Chicago Manual of Style (16th Edition):

Rajkumar, Andrew. “The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells.” 2018. Masters Thesis, University of Toronto. Accessed October 21, 2019. http://hdl.handle.net/1807/91394.

MLA Handbook (7th Edition):

Rajkumar, Andrew. “The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells.” 2018. Web. 21 Oct 2019.

Vancouver:

Rajkumar A. The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells. [Internet] [Masters thesis]. University of Toronto; 2018. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1807/91394.

Council of Science Editors:

Rajkumar A. The Role of Interleukin-1 Beta in Mediating LPS-Induced Reverse Transendothelial Migration of Intimal Dendritic Cells. [Masters Thesis]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91394


Université de Montréal

5. Raymond, Marianne. Rôle des cellules dendritiques SIRPα+ dans l’asthme expérimental .

Degree: 2011, Université de Montréal

 L’asthme est une maladie multifactorielle hétérogène qui engendre une inflammation pulmonaire associée à une variété de manifestations cliniques, dont des difficultés respiratoires graves. Globalement, l’asthme… (more)

Subjects/Keywords: Asthme; Inflammation pulmonaire; Cellules Dendritiques; CD47; Sirp; Th2; Th17; Migration; Asthma; Airway Inflammation; Dendritic Cell; CD47; Sirp; Th2; Th17; Migration

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APA (6th Edition):

Raymond, M. (2011). Rôle des cellules dendritiques SIRPα+ dans l’asthme expérimental . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/4719

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Raymond, Marianne. “Rôle des cellules dendritiques SIRPα+ dans l’asthme expérimental .” 2011. Thesis, Université de Montréal. Accessed October 21, 2019. http://hdl.handle.net/1866/4719.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Raymond, Marianne. “Rôle des cellules dendritiques SIRPα+ dans l’asthme expérimental .” 2011. Web. 21 Oct 2019.

Vancouver:

Raymond M. Rôle des cellules dendritiques SIRPα+ dans l’asthme expérimental . [Internet] [Thesis]. Université de Montréal; 2011. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/1866/4719.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Raymond M. Rôle des cellules dendritiques SIRPα+ dans l’asthme expérimental . [Thesis]. Université de Montréal; 2011. Available from: http://hdl.handle.net/1866/4719

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

6. Thacker, Robert I. Modulation of Human Dendritic Cell Activity by Adsorbed Fibrin(ogen).

Degree: PhD, Medicine : Pathobiology and Molecular Medicine, 2008, University of Cincinnati

 Fibrinogen, a plasma protein central to clot formation, has long been considered to play a role in inflammation and immunity. Fibrin(ogen) interactions with various immune… (more)

Subjects/Keywords: Immunology; Pathology; fibrinogen; surfaces; inflammation; CD18; vaccine adjuvants; oil emulsion; dendritic cell; extracellular translocation; cancer metastasis; dendritic cell migration; olive oil; integrins; cytokine; chemokines

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APA (6th Edition):

Thacker, R. I. (2008). Modulation of Human Dendritic Cell Activity by Adsorbed Fibrin(ogen). (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1218553202

Chicago Manual of Style (16th Edition):

Thacker, Robert I. “Modulation of Human Dendritic Cell Activity by Adsorbed Fibrin(ogen).” 2008. Doctoral Dissertation, University of Cincinnati. Accessed October 21, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1218553202.

MLA Handbook (7th Edition):

Thacker, Robert I. “Modulation of Human Dendritic Cell Activity by Adsorbed Fibrin(ogen).” 2008. Web. 21 Oct 2019.

Vancouver:

Thacker RI. Modulation of Human Dendritic Cell Activity by Adsorbed Fibrin(ogen). [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2019 Oct 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1218553202.

Council of Science Editors:

Thacker RI. Modulation of Human Dendritic Cell Activity by Adsorbed Fibrin(ogen). [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1218553202

7. Kimura, Telma Fátima Emídio. Análise da capacidade migratória de células dendríticas na cromoblastomicose experimental.

Degree: Mestrado, Análises Clínicas, 2012, University of São Paulo

A cromoblastomicose é uma micose subcutânea, com alto índice de morbidade, sendo Fonsecaea pedrosoi (F. pedrosoi) considerado o maior agente etiológico dessa micose, caracterizando uma… (more)

Subjects/Keywords: Cell migration; Células dendríticas; Chromoblastomycosis (Medicine); Cromoblastomicose (Medicina); Dendritic cells; Immunology; Imunologia; Medical micology; Micologia médica; Migração celular

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APA (6th Edition):

Kimura, T. F. E. (2012). Análise da capacidade migratória de células dendríticas na cromoblastomicose experimental. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/9/9136/tde-01032013-092230/ ;

Chicago Manual of Style (16th Edition):

Kimura, Telma Fátima Emídio. “Análise da capacidade migratória de células dendríticas na cromoblastomicose experimental.” 2012. Masters Thesis, University of São Paulo. Accessed October 21, 2019. http://www.teses.usp.br/teses/disponiveis/9/9136/tde-01032013-092230/ ;.

MLA Handbook (7th Edition):

Kimura, Telma Fátima Emídio. “Análise da capacidade migratória de células dendríticas na cromoblastomicose experimental.” 2012. Web. 21 Oct 2019.

Vancouver:

Kimura TFE. Análise da capacidade migratória de células dendríticas na cromoblastomicose experimental. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2019 Oct 21]. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-01032013-092230/ ;.

Council of Science Editors:

Kimura TFE. Análise da capacidade migratória de células dendríticas na cromoblastomicose experimental. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/9/9136/tde-01032013-092230/ ;


Temple University

8. Adhikary, Sabina. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.

Degree: PhD, 2013, Temple University

Physiology

Several studies have reported that administration of cannabinoid receptor agonists in inflammatory/autoimmune and CNS injury models resulted in significant attenuation of clinical disease. The… (more)

Subjects/Keywords: Immunology; Cannabinoid Receptor 2; Chemokines; Dendritic cell migration; Matrix Metalloproteinase 9; Multiple sclerosis; Spinal cord injury

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APA (6th Edition):

Adhikary, S. (2013). CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214799

Chicago Manual of Style (16th Edition):

Adhikary, Sabina. “CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.” 2013. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,214799.

MLA Handbook (7th Edition):

Adhikary, Sabina. “CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.” 2013. Web. 21 Oct 2019.

Vancouver:

Adhikary S. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214799.

Council of Science Editors:

Adhikary S. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214799

9. Solanes, Paola. IP3 Receptor 3 controls migration persistency and environment patrolling by immature dendritic cells : Le récepteur IP3R-3 contrôle la persistance migratoire des cellules dendritiques immatures et leur capacité à explorer l’environnement.

Degree: Docteur es, Immunologie, 2013, Université Paris Descartes – Paris V

Le succès de la réponse immunitaire repose en grande partie sur la capacité des leucocytes à se déplacer et à accomplir leur fonction au sein… (more)

Subjects/Keywords: Migration cellulaire; Canaux calciques; Myosine II; Capture et présentation d’antigène; Cellules dendritiques; Cell migration; Calcium channels; Myosin II; Antigen capture and presentation; Dendritic cells; 616.079

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APA (6th Edition):

Solanes, P. (2013). IP3 Receptor 3 controls migration persistency and environment patrolling by immature dendritic cells : Le récepteur IP3R-3 contrôle la persistance migratoire des cellules dendritiques immatures et leur capacité à explorer l’environnement. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2013PA05T020

Chicago Manual of Style (16th Edition):

Solanes, Paola. “IP3 Receptor 3 controls migration persistency and environment patrolling by immature dendritic cells : Le récepteur IP3R-3 contrôle la persistance migratoire des cellules dendritiques immatures et leur capacité à explorer l’environnement.” 2013. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed October 21, 2019. http://www.theses.fr/2013PA05T020.

MLA Handbook (7th Edition):

Solanes, Paola. “IP3 Receptor 3 controls migration persistency and environment patrolling by immature dendritic cells : Le récepteur IP3R-3 contrôle la persistance migratoire des cellules dendritiques immatures et leur capacité à explorer l’environnement.” 2013. Web. 21 Oct 2019.

Vancouver:

Solanes P. IP3 Receptor 3 controls migration persistency and environment patrolling by immature dendritic cells : Le récepteur IP3R-3 contrôle la persistance migratoire des cellules dendritiques immatures et leur capacité à explorer l’environnement. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2013. [cited 2019 Oct 21]. Available from: http://www.theses.fr/2013PA05T020.

Council of Science Editors:

Solanes P. IP3 Receptor 3 controls migration persistency and environment patrolling by immature dendritic cells : Le récepteur IP3R-3 contrôle la persistance migratoire des cellules dendritiques immatures et leur capacité à explorer l’environnement. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2013. Available from: http://www.theses.fr/2013PA05T020

10. Chabaud, Mélanie. Cell migration and antigen uptake are two antagonistic functions that are coupled by Myosin II in dendritic cells : La migration cellulaire et la capture d'antigènes sont des fonctions antagonistes couplées par la Myosine II dans les cellules dendritiques.

Degree: Docteur es, Biologie cellulaire, 2014, Université Paris Descartes – Paris V

Les cellules dendritiques (DCs) patrouillent les tissus périphériques à la recherche de dangers potentiels en se déplaçant à travers les tissus et en incorporant de… (more)

Subjects/Keywords: Migration cellulaire; Macropinocytose; Myosine II; Chaine Invariante (CD74); Trafic endocytique; Capture d'antigènes; Cellules dendritiques; Cell Migration; Macropinocytosis; Myosin II; Invariant Chain (CD74); Endocytic trafficking; Antigen capture; Dendritic cells; 571.964 5

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APA (6th Edition):

Chabaud, M. (2014). Cell migration and antigen uptake are two antagonistic functions that are coupled by Myosin II in dendritic cells : La migration cellulaire et la capture d'antigènes sont des fonctions antagonistes couplées par la Myosine II dans les cellules dendritiques. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05T021

Chicago Manual of Style (16th Edition):

Chabaud, Mélanie. “Cell migration and antigen uptake are two antagonistic functions that are coupled by Myosin II in dendritic cells : La migration cellulaire et la capture d'antigènes sont des fonctions antagonistes couplées par la Myosine II dans les cellules dendritiques.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed October 21, 2019. http://www.theses.fr/2014PA05T021.

MLA Handbook (7th Edition):

Chabaud, Mélanie. “Cell migration and antigen uptake are two antagonistic functions that are coupled by Myosin II in dendritic cells : La migration cellulaire et la capture d'antigènes sont des fonctions antagonistes couplées par la Myosine II dans les cellules dendritiques.” 2014. Web. 21 Oct 2019.

Vancouver:

Chabaud M. Cell migration and antigen uptake are two antagonistic functions that are coupled by Myosin II in dendritic cells : La migration cellulaire et la capture d'antigènes sont des fonctions antagonistes couplées par la Myosine II dans les cellules dendritiques. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2019 Oct 21]. Available from: http://www.theses.fr/2014PA05T021.

Council of Science Editors:

Chabaud M. Cell migration and antigen uptake are two antagonistic functions that are coupled by Myosin II in dendritic cells : La migration cellulaire et la capture d'antigènes sont des fonctions antagonistes couplées par la Myosine II dans les cellules dendritiques. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05T021

11. Otten, Marielle Anna. Antibody therapeutic approaches for cancer.

Degree: 2006, University Utrecht

 FcalphaRI as target for antibody therapy: Anti-tumor antibodies are promising therapeutics for cancer. Currently, all FDA-approved therapeutic antibodies are of the IgG class, which interact… (more)

Subjects/Keywords: Fc receptor; antibody; FcalphaRI; FcgammaR; immunotherapy; neutrophil; dendritic cell; migration; tumor; liver metastases

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APA (6th Edition):

Otten, M. A. (2006). Antibody therapeutic approaches for cancer. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465

Chicago Manual of Style (16th Edition):

Otten, Marielle Anna. “Antibody therapeutic approaches for cancer.” 2006. Doctoral Dissertation, University Utrecht. Accessed October 21, 2019. http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465.

MLA Handbook (7th Edition):

Otten, Marielle Anna. “Antibody therapeutic approaches for cancer.” 2006. Web. 21 Oct 2019.

Vancouver:

Otten MA. Antibody therapeutic approaches for cancer. [Internet] [Doctoral dissertation]. University Utrecht; 2006. [cited 2019 Oct 21]. Available from: http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465.

Council of Science Editors:

Otten MA. Antibody therapeutic approaches for cancer. [Doctoral Dissertation]. University Utrecht; 2006. Available from: http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465

12. Otten, Marielle Anna. Antibody therapeutic approaches for cancer.

Degree: 2006, University Utrecht

 FcalphaRI as target for antibody therapy: Anti-tumor antibodies are promising therapeutics for cancer. Currently, all FDA-approved therapeutic antibodies are of the IgG class, which interact… (more)

Subjects/Keywords: Fc receptor; antibody; FcalphaRI; FcgammaR; immunotherapy; neutrophil; dendritic cell; migration; tumor; liver metastases

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APA (6th Edition):

Otten, M. A. (2006). Antibody therapeutic approaches for cancer. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465

Chicago Manual of Style (16th Edition):

Otten, Marielle Anna. “Antibody therapeutic approaches for cancer.” 2006. Doctoral Dissertation, University Utrecht. Accessed October 21, 2019. http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465.

MLA Handbook (7th Edition):

Otten, Marielle Anna. “Antibody therapeutic approaches for cancer.” 2006. Web. 21 Oct 2019.

Vancouver:

Otten MA. Antibody therapeutic approaches for cancer. [Internet] [Doctoral dissertation]. University Utrecht; 2006. [cited 2019 Oct 21]. Available from: http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465.

Council of Science Editors:

Otten MA. Antibody therapeutic approaches for cancer. [Doctoral Dissertation]. University Utrecht; 2006. Available from: http://dspace.library.uu.nl/handle/1874/9465 ; URN:NBN:NL:UI:10-1874-9465 ; urn:isbn:90-8559-171-6 ; URN:NBN:NL:UI:10-1874-9465 ; http://dspace.library.uu.nl/handle/1874/9465


University of Oxford

13. Giblin, Sean. Investigating cell lineage specific biosynthesis of tenascin-C during inflammation.

Degree: PhD, 2018, University of Oxford

 The extracellular matrix (ECM) is a complex network of molecules secreted by cells, which is essential for providing structural support and facilitating cell processes including… (more)

Subjects/Keywords: Cell biology; Alternative splicing; Extracellular matrix biology; Immunology; Protein biochemistry; FnIII; Cell migration; Fibroblasts; Alternative RNA splicing; Dendritic cells; Tenascin-C; Fibronectin type 3-like domains; Extracellular Matrix; Macrophages; Lymphocytes; standard curve qPCR; Cell adhesion; Primary human immune cells; Cytokine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Giblin, S. (2018). Investigating cell lineage specific biosynthesis of tenascin-C during inflammation. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:8c7306d8-53cf-4131-a134-f74885e37cc9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748981

Chicago Manual of Style (16th Edition):

Giblin, Sean. “Investigating cell lineage specific biosynthesis of tenascin-C during inflammation.” 2018. Doctoral Dissertation, University of Oxford. Accessed October 21, 2019. http://ora.ox.ac.uk/objects/uuid:8c7306d8-53cf-4131-a134-f74885e37cc9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748981.

MLA Handbook (7th Edition):

Giblin, Sean. “Investigating cell lineage specific biosynthesis of tenascin-C during inflammation.” 2018. Web. 21 Oct 2019.

Vancouver:

Giblin S. Investigating cell lineage specific biosynthesis of tenascin-C during inflammation. [Internet] [Doctoral dissertation]. University of Oxford; 2018. [cited 2019 Oct 21]. Available from: http://ora.ox.ac.uk/objects/uuid:8c7306d8-53cf-4131-a134-f74885e37cc9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748981.

Council of Science Editors:

Giblin S. Investigating cell lineage specific biosynthesis of tenascin-C during inflammation. [Doctoral Dissertation]. University of Oxford; 2018. Available from: http://ora.ox.ac.uk/objects/uuid:8c7306d8-53cf-4131-a134-f74885e37cc9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748981


Universiteit Utrecht

14. Otten, Marielle Anna. Antibody therapeutic approaches for cancer.

Degree: 2006, Universiteit Utrecht

 FcalphaRI as target for antibody therapy: Anti-tumor antibodies are promising therapeutics for cancer. Currently, all FDA-approved therapeutic antibodies are of the IgG class, which interact… (more)

Subjects/Keywords: Geneeskunde; Fc receptor; antibody; FcalphaRI; FcgammaR; immunotherapy; neutrophil; dendritic cell; migration; tumor; liver metastases

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Otten, M. A. (2006). Antibody therapeutic approaches for cancer. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/9465

Chicago Manual of Style (16th Edition):

Otten, Marielle Anna. “Antibody therapeutic approaches for cancer.” 2006. Doctoral Dissertation, Universiteit Utrecht. Accessed October 21, 2019. http://dspace.library.uu.nl:8080/handle/1874/9465.

MLA Handbook (7th Edition):

Otten, Marielle Anna. “Antibody therapeutic approaches for cancer.” 2006. Web. 21 Oct 2019.

Vancouver:

Otten MA. Antibody therapeutic approaches for cancer. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2006. [cited 2019 Oct 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/9465.

Council of Science Editors:

Otten MA. Antibody therapeutic approaches for cancer. [Doctoral Dissertation]. Universiteit Utrecht; 2006. Available from: http://dspace.library.uu.nl:8080/handle/1874/9465

15. GE QING. Interplay of innate and adaptive systems in influenza A infection and Pulmonary Inflammation: Role of Natural Killer cells.

Degree: 2014, National University of Singapore

Subjects/Keywords: Natural killer cells; Dendritic cells migration; IFN-g; SP-D; CTL; T cell recruitment

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APA (6th Edition):

QING, G. (2014). Interplay of innate and adaptive systems in influenza A infection and Pulmonary Inflammation: Role of Natural Killer cells. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/80182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

QING, GE. “Interplay of innate and adaptive systems in influenza A infection and Pulmonary Inflammation: Role of Natural Killer cells.” 2014. Thesis, National University of Singapore. Accessed October 21, 2019. http://scholarbank.nus.edu.sg/handle/10635/80182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

QING, GE. “Interplay of innate and adaptive systems in influenza A infection and Pulmonary Inflammation: Role of Natural Killer cells.” 2014. Web. 21 Oct 2019.

Vancouver:

QING G. Interplay of innate and adaptive systems in influenza A infection and Pulmonary Inflammation: Role of Natural Killer cells. [Internet] [Thesis]. National University of Singapore; 2014. [cited 2019 Oct 21]. Available from: http://scholarbank.nus.edu.sg/handle/10635/80182.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

QING G. Interplay of innate and adaptive systems in influenza A infection and Pulmonary Inflammation: Role of Natural Killer cells. [Thesis]. National University of Singapore; 2014. Available from: http://scholarbank.nus.edu.sg/handle/10635/80182

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


ETH Zürich

16. Russo, Erica. Interactions between Lymphatic Vessels and Leukocytes: Elucidating their impact on lymphatic function and immunity.

Degree: 2016, ETH Zürich

Subjects/Keywords: LYMPHE + LYMPHSYSTEM + RETIKULO-ENDOTHELIALES SYSTEM (ANATOMIE UND PHYSIOLOGIE); GEFÄSSWAND-ENDOTHEL (ANATOMIE, HISTOLOGIE, PHYSIOLOGIE); LEUKOZYTEN + AMÖBOIDE BLUTZELLEN (BLUTZELLEN); DENDRITISCHE ZELLEN (IMMUNOLOGIE); ZELLWANDERUNG (CYTOLOGISCHE HISTOLOGIE); IMMUNREAKTION + IMMUNANTWORT (IMMUNOLOGIE); LYMPH + LYMPHATIC SYSTEM + RETICULO-ENDOTHELIAL SYSTEM (ANATOMY AND PHYSIOLOGY); VASCULAR ENDOTHELIUM (ANATOMY, HISTOLOGY, PHYSIOLOGY); LEUKOCYTES + AMOEBOID BLOOD CELLS (BLOOD CELLS); DENDRITIC CELLS (IMMUNOLOGY); CELL MIGRATION (CYTOLOGICAL HISTOLOGY); IMMUNE REACTION + IMMUNE RESPONSE (IMMUNOLOGY); info:eu-repo/classification/ddc/610; Medical sciences, medicine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Russo, E. (2016). Interactions between Lymphatic Vessels and Leukocytes: Elucidating their impact on lymphatic function and immunity. (Doctoral Dissertation). ETH Zürich. Retrieved from http://hdl.handle.net/20.500.11850/155752

Chicago Manual of Style (16th Edition):

Russo, Erica. “Interactions between Lymphatic Vessels and Leukocytes: Elucidating their impact on lymphatic function and immunity.” 2016. Doctoral Dissertation, ETH Zürich. Accessed October 21, 2019. http://hdl.handle.net/20.500.11850/155752.

MLA Handbook (7th Edition):

Russo, Erica. “Interactions between Lymphatic Vessels and Leukocytes: Elucidating their impact on lymphatic function and immunity.” 2016. Web. 21 Oct 2019.

Vancouver:

Russo E. Interactions between Lymphatic Vessels and Leukocytes: Elucidating their impact on lymphatic function and immunity. [Internet] [Doctoral dissertation]. ETH Zürich; 2016. [cited 2019 Oct 21]. Available from: http://hdl.handle.net/20.500.11850/155752.

Council of Science Editors:

Russo E. Interactions between Lymphatic Vessels and Leukocytes: Elucidating their impact on lymphatic function and immunity. [Doctoral Dissertation]. ETH Zürich; 2016. Available from: http://hdl.handle.net/20.500.11850/155752

.