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You searched for subject:(DNMT). Showing records 1 – 12 of 12 total matches.

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1. Preethi Devanand. Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers.

Degree: 2018, Ajou University

B cell translocation gene 2 (BTG2/TIS21/PC3) belongs to the family of antiproliferative (APRO) genes and reported as a tumor suppressor by our group and others.… (more)

Subjects/Keywords: BTG2; 침윤성 암; DNMT; H. pylori; TNFα

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Devanand, P. (2018). Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Devanand, Preethi. “Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers.” 2018. Thesis, Ajou University. Accessed January 23, 2019. http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Devanand, Preethi. “Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers.” 2018. Web. 23 Jan 2019.

Vancouver:

Devanand P. Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers. [Internet] [Thesis]. Ajou University; 2018. [cited 2019 Jan 23]. Available from: http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Devanand P. Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers. [Thesis]. Ajou University; 2018. Available from: http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Menon, Yoann. Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation.

Degree: Docteur es, Pharmacologie, 2016, Université Toulouse III – Paul Sabatier

Les modifications épigénétiques participent au contrôle de l'expression génique. Il a été montré que la méthylation des désoxycytidines (dC) de l'ADN joue un rôle clé… (more)

Subjects/Keywords: Epigénétique; Méthylation de l'ADN; Inhibiteurs de DNMT; Flavanones; Epigenetic; DNA methylation; DNMT inhibitors; Flavanones

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APA (6th Edition):

Menon, Y. (2016). Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2016TOU30004

Chicago Manual of Style (16th Edition):

Menon, Yoann. “Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation.” 2016. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 23, 2019. http://www.theses.fr/2016TOU30004.

MLA Handbook (7th Edition):

Menon, Yoann. “Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation.” 2016. Web. 23 Jan 2019.

Vancouver:

Menon Y. Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2016. [cited 2019 Jan 23]. Available from: http://www.theses.fr/2016TOU30004.

Council of Science Editors:

Menon Y. Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2016. Available from: http://www.theses.fr/2016TOU30004

3. Viziteu, Elena. RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple.

Degree: Docteur es, Biologie Santé, 2015, Montpellier

 Le myélome multiple (MM) est une néoplasie B caractérisée par l’accumulation d’un clone plasmocytaire dans la moelle osseuse. Des études ont démontré que les modifications… (more)

Subjects/Keywords: Myélome Multiple; Dnmt; Hélicase RECQ1; Genes; Pronostic; Traitement; Multiple Myeloma; Dnmt; Recq1; Genes; Prognosis; Treatment

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APA (6th Edition):

Viziteu, E. (2015). RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2015MONTT008

Chicago Manual of Style (16th Edition):

Viziteu, Elena. “RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple.” 2015. Doctoral Dissertation, Montpellier. Accessed January 23, 2019. http://www.theses.fr/2015MONTT008.

MLA Handbook (7th Edition):

Viziteu, Elena. “RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple.” 2015. Web. 23 Jan 2019.

Vancouver:

Viziteu E. RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple. [Internet] [Doctoral dissertation]. Montpellier; 2015. [cited 2019 Jan 23]. Available from: http://www.theses.fr/2015MONTT008.

Council of Science Editors:

Viziteu E. RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple. [Doctoral Dissertation]. Montpellier; 2015. Available from: http://www.theses.fr/2015MONTT008

4. Bagacean, Cristina. Epigenetics in leukemia : Epigénétique dans les leucémies.

Degree: Docteur es, Immunologie, 2018, Brest

Les dérivés de la cytosine sont d’importantes modifications épigénétiques dont le rôle dans l’évolution de la leucémie lymphoïde chronique (LLC) n’est pas totalement exploré. Dans… (more)

Subjects/Keywords: Leucémie lymphoïde chronique; Méthylation de l'ADN; Hydroxyméthylation de l'ADN; TET; DNMT; Chronic lymphocytic leukemia; DNA methylation; DNA hydroxymethylation; TET; DNMT

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APA (6th Edition):

Bagacean, C. (2018). Epigenetics in leukemia : Epigénétique dans les leucémies. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2018BRES0012

Chicago Manual of Style (16th Edition):

Bagacean, Cristina. “Epigenetics in leukemia : Epigénétique dans les leucémies.” 2018. Doctoral Dissertation, Brest. Accessed January 23, 2019. http://www.theses.fr/2018BRES0012.

MLA Handbook (7th Edition):

Bagacean, Cristina. “Epigenetics in leukemia : Epigénétique dans les leucémies.” 2018. Web. 23 Jan 2019.

Vancouver:

Bagacean C. Epigenetics in leukemia : Epigénétique dans les leucémies. [Internet] [Doctoral dissertation]. Brest; 2018. [cited 2019 Jan 23]. Available from: http://www.theses.fr/2018BRES0012.

Council of Science Editors:

Bagacean C. Epigenetics in leukemia : Epigénétique dans les leucémies. [Doctoral Dissertation]. Brest; 2018. Available from: http://www.theses.fr/2018BRES0012


The Ohio State University

5. Shaw, Yeng-Jeng. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.

Degree: PhD, Pharmacy, 2005, The Ohio State University

 Prostate cancer is the most common form of cancer in men and the second leading cause of cancer-related deaths in the United States. So far,… (more)

Subjects/Keywords: Akt; Bcl-xL; DNMT

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APA (6th Edition):

Shaw, Y. (2005). Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982

Chicago Manual of Style (16th Edition):

Shaw, Yeng-Jeng. “Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.” 2005. Doctoral Dissertation, The Ohio State University. Accessed January 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982.

MLA Handbook (7th Edition):

Shaw, Yeng-Jeng. “Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.” 2005. Web. 23 Jan 2019.

Vancouver:

Shaw Y. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2019 Jan 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982.

Council of Science Editors:

Shaw Y. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982


University of Debrecen

6. Alsubhi, Abdulrahman. Epigenetic Therapy in Cancer .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 The epigenetic modifications can change the state of selective part in the DNA by different enzymes such DNA methyltransferases and the histone deacetylases. Dysregulation of… (more)

Subjects/Keywords: Epigenetic DNMT HDAC Therapy Cancer

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APA (6th Edition):

Alsubhi, A. (n.d.). Epigenetic Therapy in Cancer . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/233528

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alsubhi, Abdulrahman. “Epigenetic Therapy in Cancer .” Thesis, University of Debrecen. Accessed January 23, 2019. http://hdl.handle.net/2437/233528.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alsubhi, Abdulrahman. “Epigenetic Therapy in Cancer .” Web. 23 Jan 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Alsubhi A. Epigenetic Therapy in Cancer . [Internet] [Thesis]. University of Debrecen; [cited 2019 Jan 23]. Available from: http://hdl.handle.net/2437/233528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Alsubhi A. Epigenetic Therapy in Cancer . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/233528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

7. Azadi, Sahar. Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer.

Degree: The Institute of Technology, 2012, Linköping UniversityLinköping University

  During the past decade it has been shown that alternative splicing is a important mechanism for the proteome diversity. AS is a mechanism that… (more)

Subjects/Keywords: Alternativ splicing; BRCA1; BRCA2; Breast Cancer; Splicing; DNMT; PARP

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APA (6th Edition):

Azadi, S. (2012). Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer. (Thesis). Linköping UniversityLinköping University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Azadi, Sahar. “Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer.” 2012. Thesis, Linköping UniversityLinköping University. Accessed January 23, 2019. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Azadi, Sahar. “Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer.” 2012. Web. 23 Jan 2019.

Vancouver:

Azadi S. Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer. [Internet] [Thesis]. Linköping UniversityLinköping University; 2012. [cited 2019 Jan 23]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Azadi S. Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer. [Thesis]. Linköping UniversityLinköping University; 2012. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

8. Habenschuss, Thomas. Epigenetics.

Degree: 2009, University of Vienna

Eine der am meisten erforschten epigenetischen Mechanismen ist die Methylierung von DNA. Diese Arbeit beschränkt sich auf die Cytosin-Methylierung, von der eine Vielfalt an Organismen… (more)

Subjects/Keywords: 42.20 Genetik; 42.13 Molekularbiologie; Epigenetik / DNA-Methylierung / Cytosin-Methylierung / DNMT / Methylgruppen-Donatoren / genomisches imprinting / metastabile Epiallele; epigenetics / DNA methylation / cytosine methylation / DNMT / methyl group donators / genomic imprinting / metastable epialleles

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APA (6th Edition):

Habenschuss, T. (2009). Epigenetics. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/4003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Habenschuss, Thomas. “Epigenetics.” 2009. Thesis, University of Vienna. Accessed January 23, 2019. http://othes.univie.ac.at/4003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Habenschuss, Thomas. “Epigenetics.” 2009. Web. 23 Jan 2019.

Vancouver:

Habenschuss T. Epigenetics. [Internet] [Thesis]. University of Vienna; 2009. [cited 2019 Jan 23]. Available from: http://othes.univie.ac.at/4003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Habenschuss T. Epigenetics. [Thesis]. University of Vienna; 2009. Available from: http://othes.univie.ac.at/4003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vienna

9. Klisaroska, Aleksandra. Effects of DNA Methyltransferase- and Histone Deacetylase Inhibitors on ALK Positive Anaplastic Large Cell Lymphoma.

Degree: 2011, University of Vienna

Epigenetische Modifikationen sind vererbbar und können Genexpressionmuster bei verschiedenen Krebszelllinien verändern. Diese Veränderungen beinhalten Prozesse wie DNA Methylierung, Histonmodifikationen oder RNA-abhängige Mechanismen. Die Behandlung von… (more)

Subjects/Keywords: 42.13 Molekularbiologie; ALCL / DNMT inhibitoren / 5-Aza-2'-deoxycytidine / HDAC inhibitoren / MS-275 / DNMT1 / NPM-ALK; ALCL / DNMT inhibitors / 5-Aza-2'-deoxycytidine / HDAC inhibitors / MS-275 / DNMT1 / NPM-ALK

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APA (6th Edition):

Klisaroska, A. (2011). Effects of DNA Methyltransferase- and Histone Deacetylase Inhibitors on ALK Positive Anaplastic Large Cell Lymphoma. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/15090/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Klisaroska, Aleksandra. “Effects of DNA Methyltransferase- and Histone Deacetylase Inhibitors on ALK Positive Anaplastic Large Cell Lymphoma.” 2011. Thesis, University of Vienna. Accessed January 23, 2019. http://othes.univie.ac.at/15090/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Klisaroska, Aleksandra. “Effects of DNA Methyltransferase- and Histone Deacetylase Inhibitors on ALK Positive Anaplastic Large Cell Lymphoma.” 2011. Web. 23 Jan 2019.

Vancouver:

Klisaroska A. Effects of DNA Methyltransferase- and Histone Deacetylase Inhibitors on ALK Positive Anaplastic Large Cell Lymphoma. [Internet] [Thesis]. University of Vienna; 2011. [cited 2019 Jan 23]. Available from: http://othes.univie.ac.at/15090/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Klisaroska A. Effects of DNA Methyltransferase- and Histone Deacetylase Inhibitors on ALK Positive Anaplastic Large Cell Lymphoma. [Thesis]. University of Vienna; 2011. Available from: http://othes.univie.ac.at/15090/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Johannes Gutenberg Universität Mainz

10. Weyrich, Alexandra. DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila.

Degree: 2007, Johannes Gutenberg Universität Mainz

Drosophila melanogaster enthält eine geringe Menge an 5-methyl-Cytosin. Die von mir untersuchte männliche Keimbahn von Drosophila weist jedoch keine nachweisbaren Mengen an DNA-Methylierung auf. Eine… (more)

Subjects/Keywords: Drosophila, Spermatogenese, Epigenetik, DNA-Methylierung, Histon-Modifikationen, Dnmt, E2F, Trankriptionsfaktor; Drosophila, Spermatogenesis, Epigenetic, DNA methylation, histone modifications, Dnmt, E2F, transcriptionfactor; Life sciences

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APA (6th Edition):

Weyrich, A. (2007). DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/

Chicago Manual of Style (16th Edition):

Weyrich, Alexandra. “DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila.” 2007. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed January 23, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/.

MLA Handbook (7th Edition):

Weyrich, Alexandra. “DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila.” 2007. Web. 23 Jan 2019.

Vancouver:

Weyrich A. DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2007. [cited 2019 Jan 23]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/.

Council of Science Editors:

Weyrich A. DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2007. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/

11. Farias, Nathan. The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro .

Degree: 2014, University of Guelph

 Folic acid is a B vitamin involved in DNA CpG methylation. Mandated dietary fortification has led to a subsequent increase in blood folate concentration which… (more)

Subjects/Keywords: Folate; Folic Acid; DNMT; Cancer Stem Cell; Colorectal Cancer; DNA Methylation; HCT116; SW480

…54 Figure 15. DNMT protein expression in 10 day old SW480 and HCT116 colonospheres ....55… …viii LIST OF ABBREVIATIONS APC APS BMP BSA CIMP CpG CRC CSC DAPI DHFR DMEM DNA DNMT EGF FBS… …reestablished in the 18 embryo by a group of de novo DNA methyltransferases (DNMT)… …deoxycytidine disrupted DNMT protein interactions and sensitized cancer cells to 22… 

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APA (6th Edition):

Farias, N. (2014). The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farias, Nathan. “The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro .” 2014. Thesis, University of Guelph. Accessed January 23, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farias, Nathan. “The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro .” 2014. Web. 23 Jan 2019.

Vancouver:

Farias N. The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro . [Internet] [Thesis]. University of Guelph; 2014. [cited 2019 Jan 23]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farias N. The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro . [Thesis]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Dunn, Jessilyn. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2015, Georgia Tech

 Atherosclerosis is an inflammatory disease of the arterial walls and is the major cause of heart attack and stroke. Atherosclerosis is localized to curves or… (more)

Subjects/Keywords: DNMT; Transcriptome; Shear stress; Disturbed flow; Atherosclerosis; HoxA5; Klf3; Endothelial cells; DNA methylome; Gene expression

…Global DNA Methylation Status In Vitro 30! DNMT Activity Assay 31! Monocyte adhesion assay… …siRNA Inhibits D-flow-induced Inflammation In Vitro 38! DNMT Inhibition by 5-Aza-2… …Figure 3.12: DNMT inhibition blocks OS-induced endothelial inflammation. 40! Figure 3.13… …Treatment with the DNMT inhibitor 5Aza inhibits atherosclerosis. 42! Figure 3.14: 5Aza treatment… …Disturbed flow DMR Differentially methylated region DNMT DNA (cytosine-5-)… 

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APA (6th Edition):

Dunn, J. (2015). Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/53536

Chicago Manual of Style (16th Edition):

Dunn, Jessilyn. “Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.” 2015. Doctoral Dissertation, Georgia Tech. Accessed January 23, 2019. http://hdl.handle.net/1853/53536.

MLA Handbook (7th Edition):

Dunn, Jessilyn. “Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.” 2015. Web. 23 Jan 2019.

Vancouver:

Dunn J. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. [Internet] [Doctoral dissertation]. Georgia Tech; 2015. [cited 2019 Jan 23]. Available from: http://hdl.handle.net/1853/53536.

Council of Science Editors:

Dunn J. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. [Doctoral Dissertation]. Georgia Tech; 2015. Available from: http://hdl.handle.net/1853/53536

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