Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(DNMT). Showing records 1 – 22 of 22 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters

1. Preethi, Devanand. Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers.

Degree: 2018, Ajou University

B cell translocation gene 2 (BTG2/TIS21/PC3) belongs to the family of antiproliferative (APRO) genes and reported as a tumor suppressor by our group and others.… (more)

Subjects/Keywords: BTG2; 침윤성 암; DNMT; H. pylori; TNFα

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Preethi, D. (2018). Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Preethi, Devanand. “Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers.” 2018. Thesis, Ajou University. Accessed January 16, 2021. http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Preethi, Devanand. “Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers.” 2018. Web. 16 Jan 2021.

Vancouver:

Preethi D. Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers. [Internet] [Thesis]. Ajou University; 2018. [cited 2021 Jan 16]. Available from: http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Preethi D. Epigenetic regulation of BTG2/TIS21/PC3 and its tumor suppressive role in invasive human cancers. [Thesis]. Ajou University; 2018. Available from: http://repository.ajou.ac.kr/handle/201003/16536 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027461

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Dahlet, Thomas. Méthylation de l'ADN : fonctions et ciblage au cours du développement chez la souris : DNA methylation : functions and recruitment during mouse development.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2019, Université de Strasbourg

La méthylation des cytosines est une modification épigénétique catalysée par la famille des ADN méthyltransférases (DNMTs). C’est une marque répressive lorsqu’elle est adressée sur les… (more)

Subjects/Keywords: Épigénitique; Développement; DNMT; Méthylation de l’ADN; Epigenetics; Development; DNMT; DNA methylation; 572.8

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dahlet, T. (2019). Méthylation de l'ADN : fonctions et ciblage au cours du développement chez la souris : DNA methylation : functions and recruitment during mouse development. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2019STRAJ075

Chicago Manual of Style (16th Edition):

Dahlet, Thomas. “Méthylation de l'ADN : fonctions et ciblage au cours du développement chez la souris : DNA methylation : functions and recruitment during mouse development.” 2019. Doctoral Dissertation, Université de Strasbourg. Accessed January 16, 2021. http://www.theses.fr/2019STRAJ075.

MLA Handbook (7th Edition):

Dahlet, Thomas. “Méthylation de l'ADN : fonctions et ciblage au cours du développement chez la souris : DNA methylation : functions and recruitment during mouse development.” 2019. Web. 16 Jan 2021.

Vancouver:

Dahlet T. Méthylation de l'ADN : fonctions et ciblage au cours du développement chez la souris : DNA methylation : functions and recruitment during mouse development. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2019. [cited 2021 Jan 16]. Available from: http://www.theses.fr/2019STRAJ075.

Council of Science Editors:

Dahlet T. Méthylation de l'ADN : fonctions et ciblage au cours du développement chez la souris : DNA methylation : functions and recruitment during mouse development. [Doctoral Dissertation]. Université de Strasbourg; 2019. Available from: http://www.theses.fr/2019STRAJ075

3. Menon, Yoann. Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation.

Degree: Docteur es, Pharmacologie, 2016, Université Toulouse III – Paul Sabatier

Les modifications épigénétiques participent au contrôle de l'expression génique. Il a été montré que la méthylation des désoxycytidines (dC) de l'ADN joue un rôle clé… (more)

Subjects/Keywords: Epigénétique; Méthylation de l'ADN; Inhibiteurs de DNMT; Flavanones; Epigenetic; DNA methylation; DNMT inhibitors; Flavanones

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Menon, Y. (2016). Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2016TOU30004

Chicago Manual of Style (16th Edition):

Menon, Yoann. “Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation.” 2016. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 16, 2021. http://www.theses.fr/2016TOU30004.

MLA Handbook (7th Edition):

Menon, Yoann. “Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation.” 2016. Web. 16 Jan 2021.

Vancouver:

Menon Y. Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2016. [cited 2021 Jan 16]. Available from: http://www.theses.fr/2016TOU30004.

Council of Science Editors:

Menon Y. Etude des effets pharmacologiques d'inhibiteurs non nucléosidiques de la méthylation de l'ADN : Study of the pharmacological effects of non-nucleoside inhibitors of DNA methylation. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2016. Available from: http://www.theses.fr/2016TOU30004


New Jersey Institute of Technology

4. Rana, Jagruti. Synthetic approaches to the understanding of DNA nucleobase methylation.

Degree: PhD, Chemistry and Environmental Science, 2015, New Jersey Institute of Technology

  DNA methylation is a major source of genetic variation and cancer. Methylation occurs when nucleophilic DNA bases react with methylating agent methyl methanesulfonate (MMS),… (more)

Subjects/Keywords: DNA methylation; Synthesis of 7Me-Deaza-deoxyguanosine; DNMT; DNMT inhibitors; Oligonucleotides; Polymerase bypass; Chemistry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rana, J. (2015). Synthetic approaches to the understanding of DNA nucleobase methylation. (Doctoral Dissertation). New Jersey Institute of Technology. Retrieved from https://digitalcommons.njit.edu/dissertations/107

Chicago Manual of Style (16th Edition):

Rana, Jagruti. “Synthetic approaches to the understanding of DNA nucleobase methylation.” 2015. Doctoral Dissertation, New Jersey Institute of Technology. Accessed January 16, 2021. https://digitalcommons.njit.edu/dissertations/107.

MLA Handbook (7th Edition):

Rana, Jagruti. “Synthetic approaches to the understanding of DNA nucleobase methylation.” 2015. Web. 16 Jan 2021.

Vancouver:

Rana J. Synthetic approaches to the understanding of DNA nucleobase methylation. [Internet] [Doctoral dissertation]. New Jersey Institute of Technology; 2015. [cited 2021 Jan 16]. Available from: https://digitalcommons.njit.edu/dissertations/107.

Council of Science Editors:

Rana J. Synthetic approaches to the understanding of DNA nucleobase methylation. [Doctoral Dissertation]. New Jersey Institute of Technology; 2015. Available from: https://digitalcommons.njit.edu/dissertations/107

5. Viziteu, Elena. RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple.

Degree: Docteur es, Biologie Santé, 2015, Montpellier

 Le myélome multiple (MM) est une néoplasie B caractérisée par l’accumulation d’un clone plasmocytaire dans la moelle osseuse. Des études ont démontré que les modifications… (more)

Subjects/Keywords: Myélome multiple; Dnmt; Hélicase RECQ1; Genes; Pronostic; Traitement; Multiple Myeloma; Dnmt; Recq1; Genes; Prognosis; Treatment

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Viziteu, E. (2015). RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2015MONTT008

Chicago Manual of Style (16th Edition):

Viziteu, Elena. “RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple.” 2015. Doctoral Dissertation, Montpellier. Accessed January 16, 2021. http://www.theses.fr/2015MONTT008.

MLA Handbook (7th Edition):

Viziteu, Elena. “RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple.” 2015. Web. 16 Jan 2021.

Vancouver:

Viziteu E. RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple. [Internet] [Doctoral dissertation]. Montpellier; 2015. [cited 2021 Jan 16]. Available from: http://www.theses.fr/2015MONTT008.

Council of Science Editors:

Viziteu E. RECQ1 Helicase Involvement in the Resistance to Replication Stress and Chemotherapy in Multiple Myeloma Myélome Multiple : Implication de l'hélicase RECQ1 dans la resistance au stress réplicatif et à la chimiothérapie dans le myélome multiple. [Doctoral Dissertation]. Montpellier; 2015. Available from: http://www.theses.fr/2015MONTT008

6. Bagacean, Cristina. Epigenetics in leukemia : Epigénétique dans les leucémies.

Degree: Docteur es, Immunologie, 2018, Brest

Les dérivés de la cytosine sont d’importantes modifications épigénétiques dont le rôle dans l’évolution de la leucémie lymphoïde chronique (LLC) n’est pas totalement exploré. Dans… (more)

Subjects/Keywords: Leucémie lymphoïde chronique; Méthylation de l'ADN; Hydroxyméthylation de l'ADN; TET; DNMT; Chronic lymphocytic leukemia; DNA methylation; DNA hydroxymethylation; TET; DNMT

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bagacean, C. (2018). Epigenetics in leukemia : Epigénétique dans les leucémies. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2018BRES0012

Chicago Manual of Style (16th Edition):

Bagacean, Cristina. “Epigenetics in leukemia : Epigénétique dans les leucémies.” 2018. Doctoral Dissertation, Brest. Accessed January 16, 2021. http://www.theses.fr/2018BRES0012.

MLA Handbook (7th Edition):

Bagacean, Cristina. “Epigenetics in leukemia : Epigénétique dans les leucémies.” 2018. Web. 16 Jan 2021.

Vancouver:

Bagacean C. Epigenetics in leukemia : Epigénétique dans les leucémies. [Internet] [Doctoral dissertation]. Brest; 2018. [cited 2021 Jan 16]. Available from: http://www.theses.fr/2018BRES0012.

Council of Science Editors:

Bagacean C. Epigenetics in leukemia : Epigénétique dans les leucémies. [Doctoral Dissertation]. Brest; 2018. Available from: http://www.theses.fr/2018BRES0012

7. Sabou, Alina Marcela. Identification d'une protéine parasitaire interagissant avec le facteur de transcription UHRF1 dans les cellules infectées par Toxoplasma gondii : Toxoplasma gondii ROP16 kinase silences the cyclin B1 gene promoter by hijacking host cell UHRF1-dependent epigenetic pathways.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2018, Université de Strasbourg

La toxoplasmose, déterminée par le parasite Toxoplasma gondii, est l'une des infections les plus répandues au monde, en raison de la persistance à vie sous… (more)

Subjects/Keywords: Toxoplasma gondii; UHFR1; Cycline B1; DNMT; Régulation épigenetique; ROP16; Toxoplasma gondii; UHFR1; ROP16; Cyclin B1; DNMT; Epigenetic regulation; 572.6; 572.8

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sabou, A. M. (2018). Identification d'une protéine parasitaire interagissant avec le facteur de transcription UHRF1 dans les cellules infectées par Toxoplasma gondii : Toxoplasma gondii ROP16 kinase silences the cyclin B1 gene promoter by hijacking host cell UHRF1-dependent epigenetic pathways. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2018STRAJ050

Chicago Manual of Style (16th Edition):

Sabou, Alina Marcela. “Identification d'une protéine parasitaire interagissant avec le facteur de transcription UHRF1 dans les cellules infectées par Toxoplasma gondii : Toxoplasma gondii ROP16 kinase silences the cyclin B1 gene promoter by hijacking host cell UHRF1-dependent epigenetic pathways.” 2018. Doctoral Dissertation, Université de Strasbourg. Accessed January 16, 2021. http://www.theses.fr/2018STRAJ050.

MLA Handbook (7th Edition):

Sabou, Alina Marcela. “Identification d'une protéine parasitaire interagissant avec le facteur de transcription UHRF1 dans les cellules infectées par Toxoplasma gondii : Toxoplasma gondii ROP16 kinase silences the cyclin B1 gene promoter by hijacking host cell UHRF1-dependent epigenetic pathways.” 2018. Web. 16 Jan 2021.

Vancouver:

Sabou AM. Identification d'une protéine parasitaire interagissant avec le facteur de transcription UHRF1 dans les cellules infectées par Toxoplasma gondii : Toxoplasma gondii ROP16 kinase silences the cyclin B1 gene promoter by hijacking host cell UHRF1-dependent epigenetic pathways. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2018. [cited 2021 Jan 16]. Available from: http://www.theses.fr/2018STRAJ050.

Council of Science Editors:

Sabou AM. Identification d'une protéine parasitaire interagissant avec le facteur de transcription UHRF1 dans les cellules infectées par Toxoplasma gondii : Toxoplasma gondii ROP16 kinase silences the cyclin B1 gene promoter by hijacking host cell UHRF1-dependent epigenetic pathways. [Doctoral Dissertation]. Université de Strasbourg; 2018. Available from: http://www.theses.fr/2018STRAJ050


University of Debrecen

8. Alsubhi, Abdulrahman. Epigenetic Therapy in Cancer .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 The epigenetic modifications can change the state of selective part in the DNA by different enzymes such DNA methyltransferases and the histone deacetylases. Dysregulation of… (more)

Subjects/Keywords: Epigenetic DNMT HDAC Therapy Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alsubhi, A. (n.d.). Epigenetic Therapy in Cancer . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/233528

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alsubhi, Abdulrahman. “Epigenetic Therapy in Cancer .” Thesis, University of Debrecen. Accessed January 16, 2021. http://hdl.handle.net/2437/233528.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alsubhi, Abdulrahman. “Epigenetic Therapy in Cancer .” Web. 16 Jan 2021.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Alsubhi A. Epigenetic Therapy in Cancer . [Internet] [Thesis]. University of Debrecen; [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2437/233528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Alsubhi A. Epigenetic Therapy in Cancer . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/233528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

9. Azadi, Sahar. Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer.

Degree: The Institute of Technology, 2012, Linköping UniversityLinköping University

  During the past decade it has been shown that alternative splicing is a important mechanism for the proteome diversity. AS is a mechanism that… (more)

Subjects/Keywords: Alternativ splicing; BRCA1; BRCA2; Breast Cancer; Splicing; DNMT; PARP

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Azadi, S. (2012). Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer. (Thesis). Linköping UniversityLinköping University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Azadi, Sahar. “Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer.” 2012. Thesis, Linköping UniversityLinköping University. Accessed January 16, 2021. http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Azadi, Sahar. “Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer.” 2012. Web. 16 Jan 2021.

Vancouver:

Azadi S. Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer. [Internet] [Thesis]. Linköping UniversityLinköping University; 2012. [cited 2021 Jan 16]. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Azadi S. Alternativ splicing : Mutationer i BRCA1 och BRCA2 orsakar bröstcancer. [Thesis]. Linköping UniversityLinköping University; 2012. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:liu:diva-106377

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

10. Li, Yihong. Sulforaphane protects against ethanol-induced apoptosis and teratogenesis through epigenetic modulation of anti-apoptotic genes.

Degree: PhD, 2020, University of Louisville

  Background. Ethanol-induced excessive apoptosis in neural crest cells (NCCs), a multipotent progenitor cell population, is one of the major mechanisms underlying the pathogenesis of… (more)

Subjects/Keywords: FASD; epigenetics; apoptosis; DNMT; HDAC; KDM; Other Pharmacy and Pharmaceutical Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, Y. (2020). Sulforaphane protects against ethanol-induced apoptosis and teratogenesis through epigenetic modulation of anti-apoptotic genes. (Doctoral Dissertation). University of Louisville. Retrieved from https://ir.library.louisville.edu/etd/3417

Chicago Manual of Style (16th Edition):

Li, Yihong. “Sulforaphane protects against ethanol-induced apoptosis and teratogenesis through epigenetic modulation of anti-apoptotic genes.” 2020. Doctoral Dissertation, University of Louisville. Accessed January 16, 2021. https://ir.library.louisville.edu/etd/3417.

MLA Handbook (7th Edition):

Li, Yihong. “Sulforaphane protects against ethanol-induced apoptosis and teratogenesis through epigenetic modulation of anti-apoptotic genes.” 2020. Web. 16 Jan 2021.

Vancouver:

Li Y. Sulforaphane protects against ethanol-induced apoptosis and teratogenesis through epigenetic modulation of anti-apoptotic genes. [Internet] [Doctoral dissertation]. University of Louisville; 2020. [cited 2021 Jan 16]. Available from: https://ir.library.louisville.edu/etd/3417.

Council of Science Editors:

Li Y. Sulforaphane protects against ethanol-induced apoptosis and teratogenesis through epigenetic modulation of anti-apoptotic genes. [Doctoral Dissertation]. University of Louisville; 2020. Available from: https://ir.library.louisville.edu/etd/3417


The Ohio State University

11. Shaw, Yeng-Jeng. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.

Degree: PhD, Pharmacy, 2005, The Ohio State University

 Prostate cancer is the most common form of cancer in men and the second leading cause of cancer-related deaths in the United States. So far,… (more)

Subjects/Keywords: Akt; Bcl-xL; DNMT

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shaw, Y. (2005). Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982

Chicago Manual of Style (16th Edition):

Shaw, Yeng-Jeng. “Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.” 2005. Doctoral Dissertation, The Ohio State University. Accessed January 16, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982.

MLA Handbook (7th Edition):

Shaw, Yeng-Jeng. “Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.” 2005. Web. 16 Jan 2021.

Vancouver:

Shaw Y. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2021 Jan 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982.

Council of Science Editors:

Shaw Y. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982

12. Erdmann, Alexandre. Conception, synthèse et caractérisation de nouveaux inhibiteurs de méthyltranférases d'ADN à visée anticancéreuse : Conception, sy,thesis and characterization of new DNA methyltransferase inhibitors as anticancer drug.

Degree: Docteur es, Chimie médicinale, 2015, Université Toulouse III – Paul Sabatier

Le domaine de l'épigénétique couvre l'ensemble des phénomènes héritables et transmissibles qui interviennent dans l'expression du génome sans modifier la séquence nucléotidique. L'information épigénétique est… (more)

Subjects/Keywords: Méthylation de l’ADN; Méthyltransférases d’ADN; Inhibiteur de DNMT; Drug design; Iaminopyrimidines; Inhibiteurs sélectifs; Inhibiteurs covalents; Cancer; DNA methylation; DNA methyltransferases; DNMT inhibitors; Drug design; Diaminopyrimidines; Selective inhibitor; Covalent inhibitor; Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Erdmann, A. (2015). Conception, synthèse et caractérisation de nouveaux inhibiteurs de méthyltranférases d'ADN à visée anticancéreuse : Conception, sy,thesis and characterization of new DNA methyltransferase inhibitors as anticancer drug. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2015TOU30270

Chicago Manual of Style (16th Edition):

Erdmann, Alexandre. “Conception, synthèse et caractérisation de nouveaux inhibiteurs de méthyltranférases d'ADN à visée anticancéreuse : Conception, sy,thesis and characterization of new DNA methyltransferase inhibitors as anticancer drug.” 2015. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 16, 2021. http://www.theses.fr/2015TOU30270.

MLA Handbook (7th Edition):

Erdmann, Alexandre. “Conception, synthèse et caractérisation de nouveaux inhibiteurs de méthyltranférases d'ADN à visée anticancéreuse : Conception, sy,thesis and characterization of new DNA methyltransferase inhibitors as anticancer drug.” 2015. Web. 16 Jan 2021.

Vancouver:

Erdmann A. Conception, synthèse et caractérisation de nouveaux inhibiteurs de méthyltranférases d'ADN à visée anticancéreuse : Conception, sy,thesis and characterization of new DNA methyltransferase inhibitors as anticancer drug. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2015. [cited 2021 Jan 16]. Available from: http://www.theses.fr/2015TOU30270.

Council of Science Editors:

Erdmann A. Conception, synthèse et caractérisation de nouveaux inhibiteurs de méthyltranférases d'ADN à visée anticancéreuse : Conception, sy,thesis and characterization of new DNA methyltransferase inhibitors as anticancer drug. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2015. Available from: http://www.theses.fr/2015TOU30270

13. Chappuit, Lucrezia. Design, synthèse et évaluation biochimique d’inhibiteurs des ADN Méthyltransférases : Design, synthesis and biochemical evaluation of DNA Methyltransferases inhibitors.

Degree: Docteur es, Chimie moléculaire, 2018, Sorbonne université

Ce manuscrit présente le développement de nouveaux inhibiteurs de la DNMT1 (DNA Methyl- transferase), enzyme impliquée dans les modifications épigénétiques de l’ADN et dont la… (more)

Subjects/Keywords: Cancer épigénétique; DNMT; Méthylation ADN; Modélisation moléculaire; Prolino-homo-tryptophanes, pyrazoles; Test d’inhibition; Pyrazoles; Epigenetic cancer; DNMT; DNA methylation; Molecular modeling; Prolino-homo-tryptophan; Inhibition test; Pyrazole; 572; 614.5999

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chappuit, L. (2018). Design, synthèse et évaluation biochimique d’inhibiteurs des ADN Méthyltransférases : Design, synthesis and biochemical evaluation of DNA Methyltransferases inhibitors. (Doctoral Dissertation). Sorbonne université. Retrieved from http://www.theses.fr/2018SORUS372

Chicago Manual of Style (16th Edition):

Chappuit, Lucrezia. “Design, synthèse et évaluation biochimique d’inhibiteurs des ADN Méthyltransférases : Design, synthesis and biochemical evaluation of DNA Methyltransferases inhibitors.” 2018. Doctoral Dissertation, Sorbonne université. Accessed January 16, 2021. http://www.theses.fr/2018SORUS372.

MLA Handbook (7th Edition):

Chappuit, Lucrezia. “Design, synthèse et évaluation biochimique d’inhibiteurs des ADN Méthyltransférases : Design, synthesis and biochemical evaluation of DNA Methyltransferases inhibitors.” 2018. Web. 16 Jan 2021.

Vancouver:

Chappuit L. Design, synthèse et évaluation biochimique d’inhibiteurs des ADN Méthyltransférases : Design, synthesis and biochemical evaluation of DNA Methyltransferases inhibitors. [Internet] [Doctoral dissertation]. Sorbonne université; 2018. [cited 2021 Jan 16]. Available from: http://www.theses.fr/2018SORUS372.

Council of Science Editors:

Chappuit L. Design, synthèse et évaluation biochimique d’inhibiteurs des ADN Méthyltransférases : Design, synthesis and biochemical evaluation of DNA Methyltransferases inhibitors. [Doctoral Dissertation]. Sorbonne université; 2018. Available from: http://www.theses.fr/2018SORUS372


University of Guelph

14. Mitchnick, Krista. The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats.

Degree: PhD, Department of Psychology, 2018, University of Guelph

 This thesis investigated the involvement of several epigenetic proteins in hippocampus (HPC)- and perirhinal cortex (PRh)-mediated object recognition. Previous literature has demonstrated the requirement for… (more)

Subjects/Keywords: DNA methylation; DNA demethylation; histone acetylation; DNMT; GADD45; PCAF; CBP; p300; estrogen receptor alpha

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mitchnick, K. (2018). The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats. (Doctoral Dissertation). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096

Chicago Manual of Style (16th Edition):

Mitchnick, Krista. “The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats.” 2018. Doctoral Dissertation, University of Guelph. Accessed January 16, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096.

MLA Handbook (7th Edition):

Mitchnick, Krista. “The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats.” 2018. Web. 16 Jan 2021.

Vancouver:

Mitchnick K. The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats. [Internet] [Doctoral dissertation]. University of Guelph; 2018. [cited 2021 Jan 16]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096.

Council of Science Editors:

Mitchnick K. The Dissociable Involvement of Epigenetic Mechanisms in Hippocampus- and Perirhinal Cortex-mediated Object Memory in Male Rats. [Doctoral Dissertation]. University of Guelph; 2018. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/13096


Freie Universität Berlin

15. Höche, Alexander. Expression analysis of the DNA methyltransferases DNMT1 and DNMT3b in childhood acute leukemias.

Degree: 2012, Freie Universität Berlin

 INTRODUCTION AND AIM OF STUDY: The DNA methyltransferases DNMT1, DNMT3a, and DNMT3b catalyze DNA methylation. This leads to compression and inactivation of the methylated DNA… (more)

Subjects/Keywords: DNMT expression; pediatric AML; pediatric ALL; DNA methylation; Real-Time-PCR; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Höche, A. (2012). Expression analysis of the DNA methyltransferases DNMT1 and DNMT3b in childhood acute leukemias. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-6415

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Höche, Alexander. “Expression analysis of the DNA methyltransferases DNMT1 and DNMT3b in childhood acute leukemias.” 2012. Thesis, Freie Universität Berlin. Accessed January 16, 2021. http://dx.doi.org/10.17169/refubium-6415.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Höche, Alexander. “Expression analysis of the DNA methyltransferases DNMT1 and DNMT3b in childhood acute leukemias.” 2012. Web. 16 Jan 2021.

Vancouver:

Höche A. Expression analysis of the DNA methyltransferases DNMT1 and DNMT3b in childhood acute leukemias. [Internet] [Thesis]. Freie Universität Berlin; 2012. [cited 2021 Jan 16]. Available from: http://dx.doi.org/10.17169/refubium-6415.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Höche A. Expression analysis of the DNA methyltransferases DNMT1 and DNMT3b in childhood acute leukemias. [Thesis]. Freie Universität Berlin; 2012. Available from: http://dx.doi.org/10.17169/refubium-6415

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loughborough University

16. Hunter, David. The impact of exercise interventions and omega-3 polyunsaturated fatty acid supplementation on DNA methylation and gene expression.

Degree: PhD, 2019, Loughborough University

 Epigenetics is a rapidly developing field of study which investigates chemical modifications to the genome, independent of the DNA sequence, which regulate gene expression profiles.… (more)

Subjects/Keywords: Medical and Health Sciences not elsewhere classified; DNA methylation; Epigenetics; Epigenetic; PPARGC1A; IL6; TNF; n-3 PUFA; Exercise; DNMT

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hunter, D. (2019). The impact of exercise interventions and omega-3 polyunsaturated fatty acid supplementation on DNA methylation and gene expression. (Doctoral Dissertation). Loughborough University. Retrieved from https://doi.org/10.26174/thesis.lboro.8268551.v1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785284

Chicago Manual of Style (16th Edition):

Hunter, David. “The impact of exercise interventions and omega-3 polyunsaturated fatty acid supplementation on DNA methylation and gene expression.” 2019. Doctoral Dissertation, Loughborough University. Accessed January 16, 2021. https://doi.org/10.26174/thesis.lboro.8268551.v1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785284.

MLA Handbook (7th Edition):

Hunter, David. “The impact of exercise interventions and omega-3 polyunsaturated fatty acid supplementation on DNA methylation and gene expression.” 2019. Web. 16 Jan 2021.

Vancouver:

Hunter D. The impact of exercise interventions and omega-3 polyunsaturated fatty acid supplementation on DNA methylation and gene expression. [Internet] [Doctoral dissertation]. Loughborough University; 2019. [cited 2021 Jan 16]. Available from: https://doi.org/10.26174/thesis.lboro.8268551.v1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785284.

Council of Science Editors:

Hunter D. The impact of exercise interventions and omega-3 polyunsaturated fatty acid supplementation on DNA methylation and gene expression. [Doctoral Dissertation]. Loughborough University; 2019. Available from: https://doi.org/10.26174/thesis.lboro.8268551.v1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.785284


Johannes Gutenberg Universität Mainz

17. Weyrich, Alexandra. DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila.

Degree: 2007, Johannes Gutenberg Universität Mainz

Drosophila melanogaster enthält eine geringe Menge an 5-methyl-Cytosin. Die von mir untersuchte männliche Keimbahn von Drosophila weist jedoch keine nachweisbaren Mengen an DNA-Methylierung auf. Eine… (more)

Subjects/Keywords: Drosophila, Spermatogenese, Epigenetik, DNA-Methylierung, Histon-Modifikationen, Dnmt, E2F, Trankriptionsfaktor; Drosophila, Spermatogenesis, Epigenetic, DNA methylation, histone modifications, Dnmt, E2F, transcriptionfactor; Life sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Weyrich, A. (2007). DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/

Chicago Manual of Style (16th Edition):

Weyrich, Alexandra. “DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila.” 2007. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed January 16, 2021. http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/.

MLA Handbook (7th Edition):

Weyrich, Alexandra. “DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila.” 2007. Web. 16 Jan 2021.

Vancouver:

Weyrich A. DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2007. [cited 2021 Jan 16]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/.

Council of Science Editors:

Weyrich A. DNA methylation, histone modification and the transcription factor dE2F1 in Drosophila. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2007. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2007/1317/


University of Vienna

18. Habenschuss, Thomas. Epigenetics.

Degree: 2009, University of Vienna

Eine der am meisten erforschten epigenetischen Mechanismen ist die Methylierung von DNA. Diese Arbeit beschränkt sich auf die Cytosin-Methylierung, von der eine Vielfalt an Organismen… (more)

Subjects/Keywords: 42.20 Genetik; 42.13 Molekularbiologie; Epigenetik / DNA-Methylierung / Cytosin-Methylierung / DNMT / Methylgruppen-Donatoren / genomisches imprinting / metastabile Epiallele; epigenetics / DNA methylation / cytosine methylation / DNMT / methyl group donators / genomic imprinting / metastable epialleles

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Habenschuss, T. (2009). Epigenetics. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/4003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Habenschuss, Thomas. “Epigenetics.” 2009. Thesis, University of Vienna. Accessed January 16, 2021. http://othes.univie.ac.at/4003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Habenschuss, Thomas. “Epigenetics.” 2009. Web. 16 Jan 2021.

Vancouver:

Habenschuss T. Epigenetics. [Internet] [Thesis]. University of Vienna; 2009. [cited 2021 Jan 16]. Available from: http://othes.univie.ac.at/4003/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Habenschuss T. Epigenetics. [Thesis]. University of Vienna; 2009. Available from: http://othes.univie.ac.at/4003/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Farias, Nathan. The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro.

Degree: MS, Department of Biomedical Sciences, 2014, University of Guelph

 Folic acid is a B vitamin involved in DNA CpG methylation. Mandated dietary fortification has led to a subsequent increase in blood folate concentration which… (more)

Subjects/Keywords: Folate; Folic Acid; DNMT; Cancer Stem Cell; Colorectal Cancer; DNA Methylation; HCT116; SW480

…54 Figure 15. DNMT protein expression in 10 day old SW480 and HCT116 colonospheres ....55… …viii LIST OF ABBREVIATIONS APC APS BMP BSA CIMP CpG CRC CSC DAPI DHFR DMEM DNA DNMT EGF FBS… …reestablished in the 18 embryo by a group of de novo DNA methyltransferases (DNMT)… …deoxycytidine disrupted DNMT protein interactions and sensitized cancer cells to 22… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Farias, N. (2014). The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro. (Masters Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734

Chicago Manual of Style (16th Edition):

Farias, Nathan. “The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro.” 2014. Masters Thesis, University of Guelph. Accessed January 16, 2021. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734.

MLA Handbook (7th Edition):

Farias, Nathan. “The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro.” 2014. Web. 16 Jan 2021.

Vancouver:

Farias N. The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro. [Internet] [Masters thesis]. University of Guelph; 2014. [cited 2021 Jan 16]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734.

Council of Science Editors:

Farias N. The role of folic acid in maintaining colorectal cancer cell DNA methylation patterns, and cancer stem cell phenotype in vitro. [Masters Thesis]. University of Guelph; 2014. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/7734


Freie Universität Berlin

20. Schäffer, Susanne. Investigations about the function of the cGMP-dependent kinase I alpha during the development of the nervous system.

Degree: 2006, Freie Universität Berlin

 To broaden our knowledge about the role of cGKIa in the nervous system, especially in sensory neuron axonal growth, this thesis focussed on the following… (more)

Subjects/Keywords: cGKIalpha; cGMP; VASP; Dnmt; nervous system; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schäffer, S. (2006). Investigations about the function of the cGMP-dependent kinase I alpha during the development of the nervous system. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-13474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schäffer, Susanne. “Investigations about the function of the cGMP-dependent kinase I alpha during the development of the nervous system.” 2006. Thesis, Freie Universität Berlin. Accessed January 16, 2021. http://dx.doi.org/10.17169/refubium-13474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schäffer, Susanne. “Investigations about the function of the cGMP-dependent kinase I alpha during the development of the nervous system.” 2006. Web. 16 Jan 2021.

Vancouver:

Schäffer S. Investigations about the function of the cGMP-dependent kinase I alpha during the development of the nervous system. [Internet] [Thesis]. Freie Universität Berlin; 2006. [cited 2021 Jan 16]. Available from: http://dx.doi.org/10.17169/refubium-13474.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schäffer S. Investigations about the function of the cGMP-dependent kinase I alpha during the development of the nervous system. [Thesis]. Freie Universität Berlin; 2006. Available from: http://dx.doi.org/10.17169/refubium-13474

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Dunn, Jessilyn. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.

Degree: PhD, Biomedical Engineering (Joint GT/Emory Department), 2015, Georgia Tech

 Atherosclerosis is an inflammatory disease of the arterial walls and is the major cause of heart attack and stroke. Atherosclerosis is localized to curves or… (more)

Subjects/Keywords: DNMT; Transcriptome; Shear stress; Disturbed flow; Atherosclerosis; HoxA5; Klf3; Endothelial cells; DNA methylome; Gene expression

…Global DNA Methylation Status In Vitro 30! DNMT Activity Assay 31! Monocyte adhesion assay… …siRNA Inhibits D-flow-induced Inflammation In Vitro 38! DNMT Inhibition by 5-Aza-2… …Figure 3.12: DNMT inhibition blocks OS-induced endothelial inflammation. 40! Figure 3.13… …Treatment with the DNMT inhibitor 5Aza inhibits atherosclerosis. 42! Figure 3.14: 5Aza treatment… …Disturbed flow DMR Differentially methylated region DNMT DNA (cytosine-5-)… 

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dunn, J. (2015). Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/53536

Chicago Manual of Style (16th Edition):

Dunn, Jessilyn. “Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.” 2015. Doctoral Dissertation, Georgia Tech. Accessed January 16, 2021. http://hdl.handle.net/1853/53536.

MLA Handbook (7th Edition):

Dunn, Jessilyn. “Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis.” 2015. Web. 16 Jan 2021.

Vancouver:

Dunn J. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. [Internet] [Doctoral dissertation]. Georgia Tech; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1853/53536.

Council of Science Editors:

Dunn J. Genome-scale DNA methylation changes in endothelial cells by disturbed flow and its role in atherosclerosis. [Doctoral Dissertation]. Georgia Tech; 2015. Available from: http://hdl.handle.net/1853/53536

22. Skiles, William Mark. Environmentally Induced Epimutations, Their Persistence, and Potential Causality in the Development of Disease in the Offspring of Exposed Individuals.

Degree: PhD, Biomedical Sciences, 2017, Texas A&M University

 In recent years, there has been increased interest into better understanding how environmental In recent years, there has been increased interest into better understanding how… (more)

Subjects/Keywords: oxidative stress; assisted reproductive technologies; genomic imprinting; histone demethylase; TET; DNMT; DNA methylation; epigenetics; developmental programming; DOHAD; birth defect; epigenetics; preconception; sperm

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Skiles, W. M. (2017). Environmentally Induced Epimutations, Their Persistence, and Potential Causality in the Development of Disease in the Offspring of Exposed Individuals. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/173200

Chicago Manual of Style (16th Edition):

Skiles, William Mark. “Environmentally Induced Epimutations, Their Persistence, and Potential Causality in the Development of Disease in the Offspring of Exposed Individuals.” 2017. Doctoral Dissertation, Texas A&M University. Accessed January 16, 2021. http://hdl.handle.net/1969.1/173200.

MLA Handbook (7th Edition):

Skiles, William Mark. “Environmentally Induced Epimutations, Their Persistence, and Potential Causality in the Development of Disease in the Offspring of Exposed Individuals.” 2017. Web. 16 Jan 2021.

Vancouver:

Skiles WM. Environmentally Induced Epimutations, Their Persistence, and Potential Causality in the Development of Disease in the Offspring of Exposed Individuals. [Internet] [Doctoral dissertation]. Texas A&M University; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/1969.1/173200.

Council of Science Editors:

Skiles WM. Environmentally Induced Epimutations, Their Persistence, and Potential Causality in the Development of Disease in the Offspring of Exposed Individuals. [Doctoral Dissertation]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/173200

.