Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(DNA repair). Showing records 1 – 30 of 1142 total matches.

[1] [2] [3] [4] [5] … [39]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Hong Kong

1. 陳杰; Chen, Jie. Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair.

Degree: PhD, 2015, University of Hong Kong

Ubiquitylation at DNA double-strand breaks (DSBs) plays a critical role in the orchestration of DNA damage signaling and repair. However, it remains obscure how ubiquitylation… (more)

Subjects/Keywords: DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

陳杰; Chen, J. (2015). Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. (Doctoral Dissertation). University of Hong Kong. Retrieved from Chen, J. [陳杰]. (2015). Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760913. ; http://dx.doi.org/10.5353/th_b5760913 ; http://hdl.handle.net/10722/239633

Chicago Manual of Style (16th Edition):

陳杰; Chen, Jie. “Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed February 24, 2020. Chen, J. [陳杰]. (2015). Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760913. ; http://dx.doi.org/10.5353/th_b5760913 ; http://hdl.handle.net/10722/239633.

MLA Handbook (7th Edition):

陳杰; Chen, Jie. “Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair.” 2015. Web. 24 Feb 2020.

Vancouver:

陳杰; Chen J. Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2020 Feb 24]. Available from: Chen, J. [陳杰]. (2015). Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760913. ; http://dx.doi.org/10.5353/th_b5760913 ; http://hdl.handle.net/10722/239633.

Council of Science Editors:

陳杰; Chen J. Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Chen, J. [陳杰]. (2015). Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5760913. ; http://dx.doi.org/10.5353/th_b5760913 ; http://hdl.handle.net/10722/239633


Rutgers University

2. Rodriguez-Colon, Lizahira, 1987-. Role of G9a methyltransferase in the dna damage response signal.

Degree: PhD, Pharmacology, Cellular and Molecular, 2018, Rutgers University

DNA damage induces a choreographed set of local changes in histone modifications which leads to efficient recruitment of DNA repair factors. The regulation of these… (more)

Subjects/Keywords: DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rodriguez-Colon, Lizahira, 1. (2018). Role of G9a methyltransferase in the dna damage response signal. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/57687/

Chicago Manual of Style (16th Edition):

Rodriguez-Colon, Lizahira, 1987-. “Role of G9a methyltransferase in the dna damage response signal.” 2018. Doctoral Dissertation, Rutgers University. Accessed February 24, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/57687/.

MLA Handbook (7th Edition):

Rodriguez-Colon, Lizahira, 1987-. “Role of G9a methyltransferase in the dna damage response signal.” 2018. Web. 24 Feb 2020.

Vancouver:

Rodriguez-Colon, Lizahira 1. Role of G9a methyltransferase in the dna damage response signal. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2020 Feb 24]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57687/.

Council of Science Editors:

Rodriguez-Colon, Lizahira 1. Role of G9a methyltransferase in the dna damage response signal. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57687/


Cornell University

3. Hartford, Suzanne. The Role For Dna Replication And Repair Genes In Germ-Line Maintenance And Tumor Suppression .

Degree: 2012, Cornell University

 : Faithful DNA replication and repair of DNA damage is important for prevention of disease and birth defects. My thesis work utilized reverse and forward… (more)

Subjects/Keywords: DNA Replication; DNA Repair; mcm9

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hartford, S. (2012). The Role For Dna Replication And Repair Genes In Germ-Line Maintenance And Tumor Suppression . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/29477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hartford, Suzanne. “The Role For Dna Replication And Repair Genes In Germ-Line Maintenance And Tumor Suppression .” 2012. Thesis, Cornell University. Accessed February 24, 2020. http://hdl.handle.net/1813/29477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hartford, Suzanne. “The Role For Dna Replication And Repair Genes In Germ-Line Maintenance And Tumor Suppression .” 2012. Web. 24 Feb 2020.

Vancouver:

Hartford S. The Role For Dna Replication And Repair Genes In Germ-Line Maintenance And Tumor Suppression . [Internet] [Thesis]. Cornell University; 2012. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1813/29477.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hartford S. The Role For Dna Replication And Repair Genes In Germ-Line Maintenance And Tumor Suppression . [Thesis]. Cornell University; 2012. Available from: http://hdl.handle.net/1813/29477

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

4. Cardona, Cristina Ochoa, 1979-. Homologous recombination sites during drosophila female meiosis and the role of the drosophila ino80 complex in meiotic recombination.

Degree: PhD, Cell and Developmental Biology, 2014, Rutgers University

DNA Double Strand breaks (DSBs) are one of the most lethal types of genomic damage. Irregularities in the repairs of DSBs can lead to chromosomal… (more)

Subjects/Keywords: DNA repair; DNA recombination

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cardona, Cristina Ochoa, 1. (2014). Homologous recombination sites during drosophila female meiosis and the role of the drosophila ino80 complex in meiotic recombination. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/45217/

Chicago Manual of Style (16th Edition):

Cardona, Cristina Ochoa, 1979-. “Homologous recombination sites during drosophila female meiosis and the role of the drosophila ino80 complex in meiotic recombination.” 2014. Doctoral Dissertation, Rutgers University. Accessed February 24, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/45217/.

MLA Handbook (7th Edition):

Cardona, Cristina Ochoa, 1979-. “Homologous recombination sites during drosophila female meiosis and the role of the drosophila ino80 complex in meiotic recombination.” 2014. Web. 24 Feb 2020.

Vancouver:

Cardona, Cristina Ochoa 1. Homologous recombination sites during drosophila female meiosis and the role of the drosophila ino80 complex in meiotic recombination. [Internet] [Doctoral dissertation]. Rutgers University; 2014. [cited 2020 Feb 24]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/45217/.

Council of Science Editors:

Cardona, Cristina Ochoa 1. Homologous recombination sites during drosophila female meiosis and the role of the drosophila ino80 complex in meiotic recombination. [Doctoral Dissertation]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/45217/


Rutgers University

5. Misenko, Sarah Marie, 1990-. 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism.

Degree: PhD, Biochemistry, 2017, Rutgers University

DNA end resection is believed to be a step that influences the choice between the two major DNA double-strand break (DSB) repair pathways: homologous recombination… (more)

Subjects/Keywords: DNA damage; DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Misenko, Sarah Marie, 1. (2017). 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/55597/

Chicago Manual of Style (16th Edition):

Misenko, Sarah Marie, 1990-. “53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism.” 2017. Doctoral Dissertation, Rutgers University. Accessed February 24, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/55597/.

MLA Handbook (7th Edition):

Misenko, Sarah Marie, 1990-. “53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism.” 2017. Web. 24 Feb 2020.

Vancouver:

Misenko, Sarah Marie 1. 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2020 Feb 24]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55597/.

Council of Science Editors:

Misenko, Sarah Marie 1. 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55597/


University of Utah

6. Richards, Jody Lyn. Recognition and repair of DNA damage by bacterial adenine glycosylases.

Degree: MS;, Chemistry;, 2008, University of Utah

 E. coli MutY is a base excision repair (BER) glycosylase that excises adenine mispaired opposite 7,8-dihydro-8-oxo-2'-deoxyguanine. MutY has helix-hairpin-helix and Gly/Pro-Asp structural motifs characteristic of… (more)

Subjects/Keywords: DNA repair; Adenine

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Richards, J. L. (2008). Recognition and repair of DNA damage by bacterial adenine glycosylases. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/550/rec/959

Chicago Manual of Style (16th Edition):

Richards, Jody Lyn. “Recognition and repair of DNA damage by bacterial adenine glycosylases.” 2008. Masters Thesis, University of Utah. Accessed February 24, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/550/rec/959.

MLA Handbook (7th Edition):

Richards, Jody Lyn. “Recognition and repair of DNA damage by bacterial adenine glycosylases.” 2008. Web. 24 Feb 2020.

Vancouver:

Richards JL. Recognition and repair of DNA damage by bacterial adenine glycosylases. [Internet] [Masters thesis]. University of Utah; 2008. [cited 2020 Feb 24]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/550/rec/959.

Council of Science Editors:

Richards JL. Recognition and repair of DNA damage by bacterial adenine glycosylases. [Masters Thesis]. University of Utah; 2008. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/550/rec/959


Brock University

7. Page, Melissa Maire. Intracellular antioxidant and DNA repair enzymes as correlates of stress resistance and longevity in vertebrates .

Degree: Department of Biological Sciences, 2011, Brock University

 In animals, both stress resistance and longevity appear to be influenced by the insulin/insulin-like growth factor-l signaling (lIS) pathway, the basic organization of which is… (more)

Subjects/Keywords: DNA repair; Longevity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Page, M. M. (2011). Intracellular antioxidant and DNA repair enzymes as correlates of stress resistance and longevity in vertebrates . (Thesis). Brock University. Retrieved from http://hdl.handle.net/10464/3425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Page, Melissa Maire. “Intracellular antioxidant and DNA repair enzymes as correlates of stress resistance and longevity in vertebrates .” 2011. Thesis, Brock University. Accessed February 24, 2020. http://hdl.handle.net/10464/3425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Page, Melissa Maire. “Intracellular antioxidant and DNA repair enzymes as correlates of stress resistance and longevity in vertebrates .” 2011. Web. 24 Feb 2020.

Vancouver:

Page MM. Intracellular antioxidant and DNA repair enzymes as correlates of stress resistance and longevity in vertebrates . [Internet] [Thesis]. Brock University; 2011. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/10464/3425.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Page MM. Intracellular antioxidant and DNA repair enzymes as correlates of stress resistance and longevity in vertebrates . [Thesis]. Brock University; 2011. Available from: http://hdl.handle.net/10464/3425

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

8. Baddock, Hannah. Understanding the role of the SNM1B and EXD2 in DNA damage repair.

Degree: PhD, 2017, University of Oxford

 Unrepaired, or misrepaired, DNA damage can be carcinogenic or mutagenic; thus functional DNA damage repair pathways are essential for the safeguarding of the genome. SNM1B… (more)

Subjects/Keywords: DNA damage repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baddock, H. (2017). Understanding the role of the SNM1B and EXD2 in DNA damage repair. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895

Chicago Manual of Style (16th Edition):

Baddock, Hannah. “Understanding the role of the SNM1B and EXD2 in DNA damage repair.” 2017. Doctoral Dissertation, University of Oxford. Accessed February 24, 2020. http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895.

MLA Handbook (7th Edition):

Baddock, Hannah. “Understanding the role of the SNM1B and EXD2 in DNA damage repair.” 2017. Web. 24 Feb 2020.

Vancouver:

Baddock H. Understanding the role of the SNM1B and EXD2 in DNA damage repair. [Internet] [Doctoral dissertation]. University of Oxford; 2017. [cited 2020 Feb 24]. Available from: http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895.

Council of Science Editors:

Baddock H. Understanding the role of the SNM1B and EXD2 in DNA damage repair. [Doctoral Dissertation]. University of Oxford; 2017. Available from: http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895


Queens University

9. Sierant, Megan. Novel Aspects to the Role of Rad9A During the DNA Damage Response .

Degree: Biochemistry, 2010, Queens University

 The human Rad9A checkpoint protein is required for genomic stability and proper execution of the DNA damage checkpoint. Previous work has shown Rad9A to be… (more)

Subjects/Keywords: Checkpoint; DNA Repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sierant, M. (2010). Novel Aspects to the Role of Rad9A During the DNA Damage Response . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/6138

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sierant, Megan. “Novel Aspects to the Role of Rad9A During the DNA Damage Response .” 2010. Thesis, Queens University. Accessed February 24, 2020. http://hdl.handle.net/1974/6138.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sierant, Megan. “Novel Aspects to the Role of Rad9A During the DNA Damage Response .” 2010. Web. 24 Feb 2020.

Vancouver:

Sierant M. Novel Aspects to the Role of Rad9A During the DNA Damage Response . [Internet] [Thesis]. Queens University; 2010. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1974/6138.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sierant M. Novel Aspects to the Role of Rad9A During the DNA Damage Response . [Thesis]. Queens University; 2010. Available from: http://hdl.handle.net/1974/6138

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas – Austin

10. Fu, Qiong, Ph. D. Regulation of the activity of a budding yeast DNA damage repair enzyme Sae2.

Degree: PhD, Microbiology, 2014, University of Texas – Austin

 In response to DNA damage, many repair and signaling molecules mobilize rapidly to the sites of DNA double-strand breaks (DSBs). This network of immediate responses… (more)

Subjects/Keywords: DNA repair; Sae2

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fu, Qiong, P. D. (2014). Regulation of the activity of a budding yeast DNA damage repair enzyme Sae2. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/46516

Chicago Manual of Style (16th Edition):

Fu, Qiong, Ph D. “Regulation of the activity of a budding yeast DNA damage repair enzyme Sae2.” 2014. Doctoral Dissertation, University of Texas – Austin. Accessed February 24, 2020. http://hdl.handle.net/2152/46516.

MLA Handbook (7th Edition):

Fu, Qiong, Ph D. “Regulation of the activity of a budding yeast DNA damage repair enzyme Sae2.” 2014. Web. 24 Feb 2020.

Vancouver:

Fu, Qiong PD. Regulation of the activity of a budding yeast DNA damage repair enzyme Sae2. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2014. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/2152/46516.

Council of Science Editors:

Fu, Qiong PD. Regulation of the activity of a budding yeast DNA damage repair enzyme Sae2. [Doctoral Dissertation]. University of Texas – Austin; 2014. Available from: http://hdl.handle.net/2152/46516

11. Jatsenko, Tatjana. Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads .

Degree: 2018, Tartu University

 Kahjustused DNA-s, mis tekivad kas rakkude normaalse elutegevuse käigus või erinevate keskonnategurite mõjul (näiteks UV-kiirgus, DNA-d kahjustavad kemikaalid), pärsivad genoomi replikatsiooni, takistades replikatiivse DNA polümeraasi… (more)

Subjects/Keywords: mutations; DNA damage; DNA polymerase; DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jatsenko, T. (2018). Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads . (Thesis). Tartu University. Retrieved from http://hdl.handle.net/10062/61278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jatsenko, Tatjana. “Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads .” 2018. Thesis, Tartu University. Accessed February 24, 2020. http://hdl.handle.net/10062/61278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jatsenko, Tatjana. “Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads .” 2018. Web. 24 Feb 2020.

Vancouver:

Jatsenko T. Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads . [Internet] [Thesis]. Tartu University; 2018. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/10062/61278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jatsenko T. Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads . [Thesis]. Tartu University; 2018. Available from: http://hdl.handle.net/10062/61278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

12. Li, Mingyang. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.

Degree: PhD, Laboratory and Basic Science Research, 2017, Louisiana State University

  In eukaryotes, DNA repair mechanisms detect and repair damaged DNA. DNA damage is primarily caused by a variety of exogenous and endogenous sources. Several… (more)

Subjects/Keywords: DNA repair; DNA damage; Base excision repair; Double-strand break repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, M. (2017). DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. (Doctoral Dissertation). Louisiana State University. Retrieved from https://digitalcommons.lsu.edu/gradschool_dissertations/4186

Chicago Manual of Style (16th Edition):

Li, Mingyang. “DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.” 2017. Doctoral Dissertation, Louisiana State University. Accessed February 24, 2020. https://digitalcommons.lsu.edu/gradschool_dissertations/4186.

MLA Handbook (7th Edition):

Li, Mingyang. “DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.” 2017. Web. 24 Feb 2020.

Vancouver:

Li M. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. [Internet] [Doctoral dissertation]. Louisiana State University; 2017. [cited 2020 Feb 24]. Available from: https://digitalcommons.lsu.edu/gradschool_dissertations/4186.

Council of Science Editors:

Li M. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. [Doctoral Dissertation]. Louisiana State University; 2017. Available from: https://digitalcommons.lsu.edu/gradschool_dissertations/4186


Drexel University

13. Atkins, Andrew James. Predicting the effects of intracellular protein variation on base excision repair capacity in human cells.

Degree: 2012, Drexel University

Base excision repair (BER) is one of the primary means by which cells cope with genotoxic stress and DNA damage. BER is carried out by… (more)

Subjects/Keywords: Biomedical engineering; DNA repair; Base excision repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Atkins, A. J. (2012). Predicting the effects of intracellular protein variation on base excision repair capacity in human cells. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/3765

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Atkins, Andrew James. “Predicting the effects of intracellular protein variation on base excision repair capacity in human cells.” 2012. Thesis, Drexel University. Accessed February 24, 2020. http://hdl.handle.net/1860/3765.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Atkins, Andrew James. “Predicting the effects of intracellular protein variation on base excision repair capacity in human cells.” 2012. Web. 24 Feb 2020.

Vancouver:

Atkins AJ. Predicting the effects of intracellular protein variation on base excision repair capacity in human cells. [Internet] [Thesis]. Drexel University; 2012. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1860/3765.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Atkins AJ. Predicting the effects of intracellular protein variation on base excision repair capacity in human cells. [Thesis]. Drexel University; 2012. Available from: http://hdl.handle.net/1860/3765

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

14. Pawelczak, Katherine S. Determining molecular mechanisms of DNA Non-Homologous End Joining proteins.

Degree: 2011, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

DNA double strand breaks (DSB), particularly those induced by ionizing radiation (IR) are complex lesions and if not repaired, these… (more)

Subjects/Keywords: DNA repair, non-homologous end joining; DNA-protein interactions; DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pawelczak, K. S. (2011). Determining molecular mechanisms of DNA Non-Homologous End Joining proteins. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pawelczak, Katherine S. “Determining molecular mechanisms of DNA Non-Homologous End Joining proteins.” 2011. Thesis, IUPUI. Accessed February 24, 2020. http://hdl.handle.net/1805/2517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pawelczak, Katherine S. “Determining molecular mechanisms of DNA Non-Homologous End Joining proteins.” 2011. Web. 24 Feb 2020.

Vancouver:

Pawelczak KS. Determining molecular mechanisms of DNA Non-Homologous End Joining proteins. [Internet] [Thesis]. IUPUI; 2011. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1805/2517.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pawelczak KS. Determining molecular mechanisms of DNA Non-Homologous End Joining proteins. [Thesis]. IUPUI; 2011. Available from: http://hdl.handle.net/1805/2517

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

15. Leung, Charles. Molecular basis of TopBP1 BRCT domain interactions in the DNA damage response.

Degree: PhD, Department of Biochemistry, 2011, University of Alberta

 Topoisomerase II-beta binding protein 1 (TopBP1) is a critical regulatory protein that integrates diverse signals in the DNA damage response. In response to DNA replication… (more)

Subjects/Keywords: DNA repair; X-ray crystallography

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Leung, C. (2011). Molecular basis of TopBP1 BRCT domain interactions in the DNA damage response. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/jw827c22z

Chicago Manual of Style (16th Edition):

Leung, Charles. “Molecular basis of TopBP1 BRCT domain interactions in the DNA damage response.” 2011. Doctoral Dissertation, University of Alberta. Accessed February 24, 2020. https://era.library.ualberta.ca/files/jw827c22z.

MLA Handbook (7th Edition):

Leung, Charles. “Molecular basis of TopBP1 BRCT domain interactions in the DNA damage response.” 2011. Web. 24 Feb 2020.

Vancouver:

Leung C. Molecular basis of TopBP1 BRCT domain interactions in the DNA damage response. [Internet] [Doctoral dissertation]. University of Alberta; 2011. [cited 2020 Feb 24]. Available from: https://era.library.ualberta.ca/files/jw827c22z.

Council of Science Editors:

Leung C. Molecular basis of TopBP1 BRCT domain interactions in the DNA damage response. [Doctoral Dissertation]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/jw827c22z


Oregon State University

16. Smith-Roe, Stephanie L. DNA mismatch repair-dependent responses to the food-borne carcinogen 2-Amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the mouse.

Degree: PhD, Toxicology, 2006, Oregon State University

Subjects/Keywords: DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Smith-Roe, S. L. (2006). DNA mismatch repair-dependent responses to the food-borne carcinogen 2-Amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the mouse. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/29017

Chicago Manual of Style (16th Edition):

Smith-Roe, Stephanie L. “DNA mismatch repair-dependent responses to the food-borne carcinogen 2-Amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the mouse.” 2006. Doctoral Dissertation, Oregon State University. Accessed February 24, 2020. http://hdl.handle.net/1957/29017.

MLA Handbook (7th Edition):

Smith-Roe, Stephanie L. “DNA mismatch repair-dependent responses to the food-borne carcinogen 2-Amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the mouse.” 2006. Web. 24 Feb 2020.

Vancouver:

Smith-Roe SL. DNA mismatch repair-dependent responses to the food-borne carcinogen 2-Amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the mouse. [Internet] [Doctoral dissertation]. Oregon State University; 2006. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1957/29017.

Council of Science Editors:

Smith-Roe SL. DNA mismatch repair-dependent responses to the food-borne carcinogen 2-Amino-1-methyl-6-phenylimidazo[4,5-b]pyridine (PhIP) in the mouse. [Doctoral Dissertation]. Oregon State University; 2006. Available from: http://hdl.handle.net/1957/29017


University of Hong Kong

17. Chen, Jie. Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair.

Degree: PhD, 2015, University of Hong Kong

abstract

Biomedical Sciences

Doctoral

Doctor of Philosophy

Subjects/Keywords: DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, J. (2015). Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/226731

Chicago Manual of Style (16th Edition):

Chen, Jie. “Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed February 24, 2020. http://hdl.handle.net/10722/226731.

MLA Handbook (7th Edition):

Chen, Jie. “Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair.” 2015. Web. 24 Feb 2020.

Vancouver:

Chen J. Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/10722/226731.

Council of Science Editors:

Chen J. Functional characterization of the RNF169-DYRK1A complex in DNA double-strand break signaling and repair. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/226731


University of Rochester

18. DeRan, Michael. The Role of NPAT in S-Phase-Dependent Histone Gene Transcription and DNA Double-Strand Break Repair.

Degree: PhD, 2011, University of Rochester

 In eukaryotic cells, DNA is packaged into chromatin by the histone proteins. The bulk of histone synthesis occurs in S-phase in order to package the… (more)

Subjects/Keywords: Histone; Transcription; DNA Repair; NPAT

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

DeRan, M. (2011). The Role of NPAT in S-Phase-Dependent Histone Gene Transcription and DNA Double-Strand Break Repair. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/14593

Chicago Manual of Style (16th Edition):

DeRan, Michael. “The Role of NPAT in S-Phase-Dependent Histone Gene Transcription and DNA Double-Strand Break Repair.” 2011. Doctoral Dissertation, University of Rochester. Accessed February 24, 2020. http://hdl.handle.net/1802/14593.

MLA Handbook (7th Edition):

DeRan, Michael. “The Role of NPAT in S-Phase-Dependent Histone Gene Transcription and DNA Double-Strand Break Repair.” 2011. Web. 24 Feb 2020.

Vancouver:

DeRan M. The Role of NPAT in S-Phase-Dependent Histone Gene Transcription and DNA Double-Strand Break Repair. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1802/14593.

Council of Science Editors:

DeRan M. The Role of NPAT in S-Phase-Dependent Histone Gene Transcription and DNA Double-Strand Break Repair. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/14593


University of Southern California

19. Zawahir, Faathma. Targeting human base excision repair as a novel strategy in cancer therapeutics.

Degree: PhD, Pharmacy / Pharmaceutical Sciences, 2009, University of Southern California

 APE1 is an attractive target for the rational design of small-molecule inhibitors in the field of oncological therapeutic research. It is an essential enzyme in… (more)

Subjects/Keywords: APE1; DNA repair; cancer therapeutics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zawahir, F. (2009). Targeting human base excision repair as a novel strategy in cancer therapeutics. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/248516/rec/6318

Chicago Manual of Style (16th Edition):

Zawahir, Faathma. “Targeting human base excision repair as a novel strategy in cancer therapeutics.” 2009. Doctoral Dissertation, University of Southern California. Accessed February 24, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/248516/rec/6318.

MLA Handbook (7th Edition):

Zawahir, Faathma. “Targeting human base excision repair as a novel strategy in cancer therapeutics.” 2009. Web. 24 Feb 2020.

Vancouver:

Zawahir F. Targeting human base excision repair as a novel strategy in cancer therapeutics. [Internet] [Doctoral dissertation]. University of Southern California; 2009. [cited 2020 Feb 24]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/248516/rec/6318.

Council of Science Editors:

Zawahir F. Targeting human base excision repair as a novel strategy in cancer therapeutics. [Doctoral Dissertation]. University of Southern California; 2009. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/248516/rec/6318


Louisiana State University

20. Rahman, Sheikh Arafatur. Role of Histone H4 Mutations in DNA Repair Pathways.

Degree: MS, Medicine and Health Sciences, 2016, Louisiana State University

 Histone H3K79 methylation has been shown to play roles in different DNA repair pathways. Histone H4 residues serine 64 to threonine 80 surround histone H3K79… (more)

Subjects/Keywords: Histone H4 Mutations; DNA Repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rahman, S. A. (2016). Role of Histone H4 Mutations in DNA Repair Pathways. (Masters Thesis). Louisiana State University. Retrieved from etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565

Chicago Manual of Style (16th Edition):

Rahman, Sheikh Arafatur. “Role of Histone H4 Mutations in DNA Repair Pathways.” 2016. Masters Thesis, Louisiana State University. Accessed February 24, 2020. etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565.

MLA Handbook (7th Edition):

Rahman, Sheikh Arafatur. “Role of Histone H4 Mutations in DNA Repair Pathways.” 2016. Web. 24 Feb 2020.

Vancouver:

Rahman SA. Role of Histone H4 Mutations in DNA Repair Pathways. [Internet] [Masters thesis]. Louisiana State University; 2016. [cited 2020 Feb 24]. Available from: etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565.

Council of Science Editors:

Rahman SA. Role of Histone H4 Mutations in DNA Repair Pathways. [Masters Thesis]. Louisiana State University; 2016. Available from: etd-07112016-153156 ; https://digitalcommons.lsu.edu/gradschool_theses/1565


University of Southern California

21. Li, Sicong. Mechanisms of nucleases in non-homologous DNA end joining.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2013, University of Southern California

DNA double-stranded breaks (DSB) can occur through programmed mechanisms such as V(D)J recombination and class switch recombination or pathological mechanisms such as ionizing radiation. To… (more)

Subjects/Keywords: NHEJ; Artemis; nuclease; DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, S. (2013). Mechanisms of nucleases in non-homologous DNA end joining. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/342055/rec/4011

Chicago Manual of Style (16th Edition):

Li, Sicong. “Mechanisms of nucleases in non-homologous DNA end joining.” 2013. Doctoral Dissertation, University of Southern California. Accessed February 24, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/342055/rec/4011.

MLA Handbook (7th Edition):

Li, Sicong. “Mechanisms of nucleases in non-homologous DNA end joining.” 2013. Web. 24 Feb 2020.

Vancouver:

Li S. Mechanisms of nucleases in non-homologous DNA end joining. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2020 Feb 24]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/342055/rec/4011.

Council of Science Editors:

Li S. Mechanisms of nucleases in non-homologous DNA end joining. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/342055/rec/4011


McMaster University

22. Brown, Christopher, M. CHARACTERIZATION OF THE END BRIDGING COMPLEX OF NON-HOMOLOGOUS END JOINING REPAIR OF DNA DOUBLE STRAND BREAKS.

Degree: PhD, 2018, McMaster University

DNA double strand breaks represent the single most dangerous type of damage that can afflict the genome. Given the severity of such a lesion, higher… (more)

Subjects/Keywords: Structural biology; DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brown, Christopher, M. (2018). CHARACTERIZATION OF THE END BRIDGING COMPLEX OF NON-HOMOLOGOUS END JOINING REPAIR OF DNA DOUBLE STRAND BREAKS. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/22876

Chicago Manual of Style (16th Edition):

Brown, Christopher, M. “CHARACTERIZATION OF THE END BRIDGING COMPLEX OF NON-HOMOLOGOUS END JOINING REPAIR OF DNA DOUBLE STRAND BREAKS.” 2018. Doctoral Dissertation, McMaster University. Accessed February 24, 2020. http://hdl.handle.net/11375/22876.

MLA Handbook (7th Edition):

Brown, Christopher, M. “CHARACTERIZATION OF THE END BRIDGING COMPLEX OF NON-HOMOLOGOUS END JOINING REPAIR OF DNA DOUBLE STRAND BREAKS.” 2018. Web. 24 Feb 2020.

Vancouver:

Brown, Christopher M. CHARACTERIZATION OF THE END BRIDGING COMPLEX OF NON-HOMOLOGOUS END JOINING REPAIR OF DNA DOUBLE STRAND BREAKS. [Internet] [Doctoral dissertation]. McMaster University; 2018. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/11375/22876.

Council of Science Editors:

Brown, Christopher M. CHARACTERIZATION OF THE END BRIDGING COMPLEX OF NON-HOMOLOGOUS END JOINING REPAIR OF DNA DOUBLE STRAND BREAKS. [Doctoral Dissertation]. McMaster University; 2018. Available from: http://hdl.handle.net/11375/22876


University of Oxford

23. Kasparek, Torben Rudolf. Identification and characterisation of determinants of genome stability in response to a double-strand break.

Degree: PhD, 2013, University of Oxford

 Chromosomal rearrangements can lead to loss of heterozygosity (LOH) and oncogene activation, both of which represent possible causative events in cancer development. Such outcomes can… (more)

Subjects/Keywords: 616.994; Oncology; DNA damage repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kasparek, T. R. (2013). Identification and characterisation of determinants of genome stability in response to a double-strand break. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454

Chicago Manual of Style (16th Edition):

Kasparek, Torben Rudolf. “Identification and characterisation of determinants of genome stability in response to a double-strand break.” 2013. Doctoral Dissertation, University of Oxford. Accessed February 24, 2020. http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454.

MLA Handbook (7th Edition):

Kasparek, Torben Rudolf. “Identification and characterisation of determinants of genome stability in response to a double-strand break.” 2013. Web. 24 Feb 2020.

Vancouver:

Kasparek TR. Identification and characterisation of determinants of genome stability in response to a double-strand break. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Feb 24]. Available from: http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454.

Council of Science Editors:

Kasparek TR. Identification and characterisation of determinants of genome stability in response to a double-strand break. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454


Oregon State University

24. Wu, Shiau-Yin. An assay for screening cells for mismatch repair proficiency in vivo.

Degree: MS, Biochemistry and Biophysics, 2002, Oregon State University

Subjects/Keywords: DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, S. (2002). An assay for screening cells for mismatch repair proficiency in vivo. (Masters Thesis). Oregon State University. Retrieved from http://hdl.handle.net/1957/31049

Chicago Manual of Style (16th Edition):

Wu, Shiau-Yin. “An assay for screening cells for mismatch repair proficiency in vivo.” 2002. Masters Thesis, Oregon State University. Accessed February 24, 2020. http://hdl.handle.net/1957/31049.

MLA Handbook (7th Edition):

Wu, Shiau-Yin. “An assay for screening cells for mismatch repair proficiency in vivo.” 2002. Web. 24 Feb 2020.

Vancouver:

Wu S. An assay for screening cells for mismatch repair proficiency in vivo. [Internet] [Masters thesis]. Oregon State University; 2002. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1957/31049.

Council of Science Editors:

Wu S. An assay for screening cells for mismatch repair proficiency in vivo. [Masters Thesis]. Oregon State University; 2002. Available from: http://hdl.handle.net/1957/31049


Oregon State University

25. Shin-Darlak, Chi Y. Spontaneous and enviornmental [sic] mutagenesis in mismatch repair deficient cells.

Degree: PhD, Toxicology, 2002, Oregon State University

Subjects/Keywords: DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shin-Darlak, C. Y. (2002). Spontaneous and enviornmental [sic] mutagenesis in mismatch repair deficient cells. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/32158

Chicago Manual of Style (16th Edition):

Shin-Darlak, Chi Y. “Spontaneous and enviornmental [sic] mutagenesis in mismatch repair deficient cells.” 2002. Doctoral Dissertation, Oregon State University. Accessed February 24, 2020. http://hdl.handle.net/1957/32158.

MLA Handbook (7th Edition):

Shin-Darlak, Chi Y. “Spontaneous and enviornmental [sic] mutagenesis in mismatch repair deficient cells.” 2002. Web. 24 Feb 2020.

Vancouver:

Shin-Darlak CY. Spontaneous and enviornmental [sic] mutagenesis in mismatch repair deficient cells. [Internet] [Doctoral dissertation]. Oregon State University; 2002. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1957/32158.

Council of Science Editors:

Shin-Darlak CY. Spontaneous and enviornmental [sic] mutagenesis in mismatch repair deficient cells. [Doctoral Dissertation]. Oregon State University; 2002. Available from: http://hdl.handle.net/1957/32158


Oregon State University

26. Feng, Wen-yang. Recombinagenic and anti-mutagenic processing of UV-light photoproducts by the Escherichia coli methyl-directed mismatch-repair system.

Degree: PhD, Genetics, 1994, Oregon State University

Subjects/Keywords: DNA repair

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Feng, W. (1994). Recombinagenic and anti-mutagenic processing of UV-light photoproducts by the Escherichia coli methyl-directed mismatch-repair system. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/35075

Chicago Manual of Style (16th Edition):

Feng, Wen-yang. “Recombinagenic and anti-mutagenic processing of UV-light photoproducts by the Escherichia coli methyl-directed mismatch-repair system.” 1994. Doctoral Dissertation, Oregon State University. Accessed February 24, 2020. http://hdl.handle.net/1957/35075.

MLA Handbook (7th Edition):

Feng, Wen-yang. “Recombinagenic and anti-mutagenic processing of UV-light photoproducts by the Escherichia coli methyl-directed mismatch-repair system.” 1994. Web. 24 Feb 2020.

Vancouver:

Feng W. Recombinagenic and anti-mutagenic processing of UV-light photoproducts by the Escherichia coli methyl-directed mismatch-repair system. [Internet] [Doctoral dissertation]. Oregon State University; 1994. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1957/35075.

Council of Science Editors:

Feng W. Recombinagenic and anti-mutagenic processing of UV-light photoproducts by the Escherichia coli methyl-directed mismatch-repair system. [Doctoral Dissertation]. Oregon State University; 1994. Available from: http://hdl.handle.net/1957/35075


University of Cambridge

27. Morton, Christopher Robert. Structural and biophysical analysis of Human DNA repair protein CtIP.

Degree: PhD, 2019, University of Cambridge

 The integrity of our genome is constantly threatened by endogenous and exogenous sources of DNA damage. The successful repair of DNA lesions is necessary for… (more)

Subjects/Keywords: DNA Repair; Homologous Recombination; CtIP

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Morton, C. R. (2019). Structural and biophysical analysis of Human DNA repair protein CtIP. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/294355

Chicago Manual of Style (16th Edition):

Morton, Christopher Robert. “Structural and biophysical analysis of Human DNA repair protein CtIP.” 2019. Doctoral Dissertation, University of Cambridge. Accessed February 24, 2020. https://www.repository.cam.ac.uk/handle/1810/294355.

MLA Handbook (7th Edition):

Morton, Christopher Robert. “Structural and biophysical analysis of Human DNA repair protein CtIP.” 2019. Web. 24 Feb 2020.

Vancouver:

Morton CR. Structural and biophysical analysis of Human DNA repair protein CtIP. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Feb 24]. Available from: https://www.repository.cam.ac.uk/handle/1810/294355.

Council of Science Editors:

Morton CR. Structural and biophysical analysis of Human DNA repair protein CtIP. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/294355


Cornell University

28. Plys, Aaron. Analysis Of The Movement Of Saccharomyces Cerevisiae Mismatch Repair Proteins On Dna .

Degree: 2011, Cornell University

 Replication errors that escape DNA polymerase proof-reading activity are efficientl y recognized and repaired by conserved DNA mismatch repair factors. The overall result is a… (more)

Subjects/Keywords: DNA mismatch repair; Replication; Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Plys, A. (2011). Analysis Of The Movement Of Saccharomyces Cerevisiae Mismatch Repair Proteins On Dna . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/30604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Plys, Aaron. “Analysis Of The Movement Of Saccharomyces Cerevisiae Mismatch Repair Proteins On Dna .” 2011. Thesis, Cornell University. Accessed February 24, 2020. http://hdl.handle.net/1813/30604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Plys, Aaron. “Analysis Of The Movement Of Saccharomyces Cerevisiae Mismatch Repair Proteins On Dna .” 2011. Web. 24 Feb 2020.

Vancouver:

Plys A. Analysis Of The Movement Of Saccharomyces Cerevisiae Mismatch Repair Proteins On Dna . [Internet] [Thesis]. Cornell University; 2011. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/1813/30604.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Plys A. Analysis Of The Movement Of Saccharomyces Cerevisiae Mismatch Repair Proteins On Dna . [Thesis]. Cornell University; 2011. Available from: http://hdl.handle.net/1813/30604

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

29. Sugitani, Norie. Structural and Biophysical Characterization of the Nucleotide Excision Repair Factor XPA.

Degree: PhD, Chemistry, 2017, Vanderbilt University

 Maintaining the integrity of the genome is critical for the survival of all organisms. Genome maintenance must be efficient because we are constantly under exposure… (more)

Subjects/Keywords: NER; XPA; DNA repair; RPA

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sugitani, N. (2017). Structural and Biophysical Characterization of the Nucleotide Excision Repair Factor XPA. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03212017-135434/ ;

Chicago Manual of Style (16th Edition):

Sugitani, Norie. “Structural and Biophysical Characterization of the Nucleotide Excision Repair Factor XPA.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed February 24, 2020. http://etd.library.vanderbilt.edu//available/etd-03212017-135434/ ;.

MLA Handbook (7th Edition):

Sugitani, Norie. “Structural and Biophysical Characterization of the Nucleotide Excision Repair Factor XPA.” 2017. Web. 24 Feb 2020.

Vancouver:

Sugitani N. Structural and Biophysical Characterization of the Nucleotide Excision Repair Factor XPA. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Feb 24]. Available from: http://etd.library.vanderbilt.edu//available/etd-03212017-135434/ ;.

Council of Science Editors:

Sugitani N. Structural and Biophysical Characterization of the Nucleotide Excision Repair Factor XPA. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu//available/etd-03212017-135434/ ;


Washington State University

30. [No author]. Functional characterization of human hMRE11 and hMRE11-hMLH1 interplay in DNA mismatch repair .

Degree: 2006, Washington State University

Subjects/Keywords: DNA repair.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

author], [. (2006). Functional characterization of human hMRE11 and hMRE11-hMLH1 interplay in DNA mismatch repair . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/446

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Functional characterization of human hMRE11 and hMRE11-hMLH1 interplay in DNA mismatch repair .” 2006. Thesis, Washington State University. Accessed February 24, 2020. http://hdl.handle.net/2376/446.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Functional characterization of human hMRE11 and hMRE11-hMLH1 interplay in DNA mismatch repair .” 2006. Web. 24 Feb 2020.

Vancouver:

author] [. Functional characterization of human hMRE11 and hMRE11-hMLH1 interplay in DNA mismatch repair . [Internet] [Thesis]. Washington State University; 2006. [cited 2020 Feb 24]. Available from: http://hdl.handle.net/2376/446.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Functional characterization of human hMRE11 and hMRE11-hMLH1 interplay in DNA mismatch repair . [Thesis]. Washington State University; 2006. Available from: http://hdl.handle.net/2376/446

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [39]

.