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You searched for subject:(DNA double strand breaks). Showing records 1 – 30 of 18524 total matches.

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1. Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.

Degree: 博士(医学), 2017, Nara Medical University / 奈良県立医科大学

DNA-dependent protein kinase (DNA-PK) is a serine/threonine kinase that plays an essential role in the repair of DNA double-strand breaks (DSBs) in the non-homologous end-joining… (more)

Subjects/Keywords: DNA double-strand breaks; Non-homologous end-joining; Acetylation; DNA-PKcs

Page 1

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APA (6th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, M. (2017). Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. “Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.” 2017. Thesis, Nara Medical University / 奈良県立医科大学. Accessed May 06, 2021. http://hdl.handle.net/10564/3342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. “Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.” 2017. Web. 06 May 2021.

Vancouver:

Mori, Eiichiro; Davis, Anthony J.; Hasegawa M. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. [cited 2021 May 06]. Available from: http://hdl.handle.net/10564/3342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mori, Eiichiro; Davis, Anthony J.; Hasegawa M. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. Available from: http://hdl.handle.net/10564/3342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Giunta, Simona. DNA damage responses in the context of the cell division cycle.

Degree: PhD, 2010, University of Cambridge

 During my PhD, I have investigated aspects of the DNA damage response (DDR) in the context of three different cellular scenarios: DNA damage signalling in… (more)

Subjects/Keywords: DNA damage response; Cell cycle; Mitosis; DNA double strand breaks

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APA (6th Edition):

Giunta, S. (2010). DNA damage responses in the context of the cell division cycle. (Doctoral Dissertation). University of Cambridge. Retrieved from http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg

Chicago Manual of Style (16th Edition):

Giunta, Simona. “DNA damage responses in the context of the cell division cycle.” 2010. Doctoral Dissertation, University of Cambridge. Accessed May 06, 2021. http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg.

MLA Handbook (7th Edition):

Giunta, Simona. “DNA damage responses in the context of the cell division cycle.” 2010. Web. 06 May 2021.

Vancouver:

Giunta S. DNA damage responses in the context of the cell division cycle. [Internet] [Doctoral dissertation]. University of Cambridge; 2010. [cited 2021 May 06]. Available from: http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg.

Council of Science Editors:

Giunta S. DNA damage responses in the context of the cell division cycle. [Doctoral Dissertation]. University of Cambridge; 2010. Available from: http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg

3. Takabayashi, Hiroaki. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.

Degree: 博士(医学), 2013, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3807号. 学位記番号: 新大院博(医)甲第559号. 学位授与年月日: 平成25年9月20日

Human Pathology. 2013, 44(6), 1038–1046

p53-binding protein 1

Subjects/Keywords: Colorectal carcinoma; Double-strand breaks; DNA damage response; γH2AX

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APA (6th Edition):

Takabayashi, H. (2013). Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/24550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takabayashi, Hiroaki. “Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.” 2013. Thesis, Niigata University / 新潟大学. Accessed May 06, 2021. http://hdl.handle.net/10191/24550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takabayashi, Hiroaki. “Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.” 2013. Web. 06 May 2021.

Vancouver:

Takabayashi H. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. [Internet] [Thesis]. Niigata University / 新潟大学; 2013. [cited 2021 May 06]. Available from: http://hdl.handle.net/10191/24550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takabayashi H. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. [Thesis]. Niigata University / 新潟大学; 2013. Available from: http://hdl.handle.net/10191/24550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Passos, Joana. Implications of histone H2AX abundance in breast cancer.

Degree: 2020, NUI Galway

Histones are responsible for the packaging of DNA into a eukaryotic cell nucleus, and the H2A variant H2AX plays an important role in the DNA(more)

Subjects/Keywords: breast cancer; H2AX; copy number variation; DNA damage response; DNA double strand breaks; Medicine; Anatomy

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APA (6th Edition):

Passos, J. (2020). Implications of histone H2AX abundance in breast cancer. (Thesis). NUI Galway. Retrieved from http://hdl.handle.net/10379/15784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Passos, Joana. “Implications of histone H2AX abundance in breast cancer.” 2020. Thesis, NUI Galway. Accessed May 06, 2021. http://hdl.handle.net/10379/15784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Passos, Joana. “Implications of histone H2AX abundance in breast cancer.” 2020. Web. 06 May 2021.

Vancouver:

Passos J. Implications of histone H2AX abundance in breast cancer. [Internet] [Thesis]. NUI Galway; 2020. [cited 2021 May 06]. Available from: http://hdl.handle.net/10379/15784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Passos J. Implications of histone H2AX abundance in breast cancer. [Thesis]. NUI Galway; 2020. Available from: http://hdl.handle.net/10379/15784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

5. Young, Jordan. A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair.

Degree: PhD, 2016, University of Toronto

 All living organisms are continuously challenged by agents in their normal cellular environment that can inflict damage to their genetic material. DNA damage can have… (more)

Subjects/Keywords: Cell microarrays; DNA double-strand breaks; DNA repair; High-content screening; Homologous recombination; Microcephaly; 0379

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APA (6th Edition):

Young, J. (2016). A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/77384

Chicago Manual of Style (16th Edition):

Young, Jordan. “A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair.” 2016. Doctoral Dissertation, University of Toronto. Accessed May 06, 2021. http://hdl.handle.net/1807/77384.

MLA Handbook (7th Edition):

Young, Jordan. “A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair.” 2016. Web. 06 May 2021.

Vancouver:

Young J. A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 May 06]. Available from: http://hdl.handle.net/1807/77384.

Council of Science Editors:

Young J. A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/77384


University of Texas – Austin

6. -2937-0948. Mechanism of repair of Mu DNA insertions.

Degree: PhD, Cell and Molecular Biology, 2015, University of Texas – Austin

 Transposable elements are ubiquitous, occupying as much as 85% of the genome of some species, and nearly 50% of the human genome, and causing DNA(more)

Subjects/Keywords: Mu DNA transposition; Post-integration; DNA replication and repair; Double-strand breaks

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APA (6th Edition):

-2937-0948. (2015). Mechanism of repair of Mu DNA insertions. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/46541

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-2937-0948. “Mechanism of repair of Mu DNA insertions.” 2015. Doctoral Dissertation, University of Texas – Austin. Accessed May 06, 2021. http://hdl.handle.net/2152/46541.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-2937-0948. “Mechanism of repair of Mu DNA insertions.” 2015. Web. 06 May 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-2937-0948. Mechanism of repair of Mu DNA insertions. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2015. [cited 2021 May 06]. Available from: http://hdl.handle.net/2152/46541.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-2937-0948. Mechanism of repair of Mu DNA insertions. [Doctoral Dissertation]. University of Texas – Austin; 2015. Available from: http://hdl.handle.net/2152/46541

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

7. Moretton, Amandine. Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks.

Degree: Docteur es, Physiologie et Génétique Moléculaire, 2017, Université Clermont Auvergne‎ (2017-2020)

Les mitochondries sont des organites qui possèdent leur propre ADN (ADNmt), codant pour des gènes de la chaine respiratoire. La réparation des dommages dus aux… (more)

Subjects/Keywords: ADN mitochondrial; Cassures double-brin; Dégradation de l’ADN; Réparation de l’ADN; Mitochondrial DNA; Double-strand breaks; DNA degradation; DNA repair

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APA (6th Edition):

Moretton, A. (2017). Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks. (Doctoral Dissertation). Université Clermont Auvergne‎ (2017-2020). Retrieved from http://www.theses.fr/2017CLFAC047

Chicago Manual of Style (16th Edition):

Moretton, Amandine. “Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks.” 2017. Doctoral Dissertation, Université Clermont Auvergne‎ (2017-2020). Accessed May 06, 2021. http://www.theses.fr/2017CLFAC047.

MLA Handbook (7th Edition):

Moretton, Amandine. “Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks.” 2017. Web. 06 May 2021.

Vancouver:

Moretton A. Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks. [Internet] [Doctoral dissertation]. Université Clermont Auvergne‎ (2017-2020); 2017. [cited 2021 May 06]. Available from: http://www.theses.fr/2017CLFAC047.

Council of Science Editors:

Moretton A. Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks. [Doctoral Dissertation]. Université Clermont Auvergne‎ (2017-2020); 2017. Available from: http://www.theses.fr/2017CLFAC047


NSYSU

8. Yang, Chun-feng. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

 Breast cancer is a leading cause of death in women worldwide, and chemotherapy is one of the primary strategies for breast cancer treatment. However, the… (more)

Subjects/Keywords: DNA double-strand breaks; camptothecin; Phthalate; Bis(2-ethylhexyl) phthalate; DEHP; zebrafish; chemoresistance; breast cancer

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APA (6th Edition):

Yang, C. (2016). The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Thesis, NSYSU. Accessed May 06, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Web. 06 May 2021.

Vancouver:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 May 06]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

9. Martin, Nathan. Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP.

Degree: Biomedical Physics, 2014, UCLA

 ABSTRACT OF THE DISSERTATIONRepair of DNA double strand breaks and radiosensitivity:modification of DNA repair and radiosensitivity bymicroRNA-335 and mtPAPby Nathan Thomas MartinDoctor of Philosophy in… (more)

Subjects/Keywords: Cellular biology; Biophysics; cancer; DNA repair; double strand breaks; radiobiology; Radiosensitivity; XCIND

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APA (6th Edition):

Martin, N. (2014). Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/26w5v41k

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Nathan. “Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP.” 2014. Thesis, UCLA. Accessed May 06, 2021. http://www.escholarship.org/uc/item/26w5v41k.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Nathan. “Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP.” 2014. Web. 06 May 2021.

Vancouver:

Martin N. Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP. [Internet] [Thesis]. UCLA; 2014. [cited 2021 May 06]. Available from: http://www.escholarship.org/uc/item/26w5v41k.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin N. Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/26w5v41k

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

10. Lee, Eva I-Hua. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.

Degree: 2016, University of Toronto

Fanconi anemia (FA) patients are hypersensitive to ionizing radiation and other agents that generate DNA double-strand breaks (DSBs). The major error-free DSB repair pathway in… (more)

Subjects/Keywords: DNA repair; Double strand breaks; DSB resection; FANCD2; FANCJ; Fanconi Anemia; 0369

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APA (6th Edition):

Lee, E. I. (2016). Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/72732

Chicago Manual of Style (16th Edition):

Lee, Eva I-Hua. “Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.” 2016. Masters Thesis, University of Toronto. Accessed May 06, 2021. http://hdl.handle.net/1807/72732.

MLA Handbook (7th Edition):

Lee, Eva I-Hua. “Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.” 2016. Web. 06 May 2021.

Vancouver:

Lee EI. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2021 May 06]. Available from: http://hdl.handle.net/1807/72732.

Council of Science Editors:

Lee EI. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72732


Freie Universität Berlin

11. Andreani, Matteo. Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden.

Degree: 2020, Freie Universität Berlin

 Das Rap1-interacting factor 1 (RIF1) ist ein multifunktionales Protein, welches unter anderem den Prozess der DNA-Replikation zeitlich koordiniert sowie am Neustart blockierter Replikationsgabeln, der Auflösung… (more)

Subjects/Keywords: RIF1; DNA repair; Class Switch Recombination; BRCA1-deficiency; Double-strand breaks; ddc:570

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APA (6th Edition):

Andreani, M. (2020). Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-26996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andreani, Matteo. “Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden.” 2020. Thesis, Freie Universität Berlin. Accessed May 06, 2021. http://dx.doi.org/10.17169/refubium-26996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andreani, Matteo. “Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden.” 2020. Web. 06 May 2021.

Vancouver:

Andreani M. Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2021 May 06]. Available from: http://dx.doi.org/10.17169/refubium-26996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andreani M. Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-26996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

12. Baldascini, Marta. Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks .

Degree: 2019, National University of Ireland – Galway

 The integrity of our genome is very important therefore, our cells have developed efficient DNA repair mechanisms to control this integrity and avoid mutations. Although… (more)

Subjects/Keywords: DNA damage; zinc finger proteins; ZC3H8; ZC3H11A; ZC3H14; double strand breaks; Biochemistry; Science; Natural Sciences

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APA (6th Edition):

Baldascini, M. (2019). Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/15652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baldascini, Marta. “Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks .” 2019. Thesis, National University of Ireland – Galway. Accessed May 06, 2021. http://hdl.handle.net/10379/15652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baldascini, Marta. “Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks .” 2019. Web. 06 May 2021.

Vancouver:

Baldascini M. Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks . [Internet] [Thesis]. National University of Ireland – Galway; 2019. [cited 2021 May 06]. Available from: http://hdl.handle.net/10379/15652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baldascini M. Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks . [Thesis]. National University of Ireland – Galway; 2019. Available from: http://hdl.handle.net/10379/15652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


York University

13. D'Angelo, Jessica Sarah. Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway.

Degree: MSc -MS, Biology, 2019, York University

 In response to DNA double-strand breaks (DSBs), H2A.X undergoes three post-translational modifications (PTMs) reported to be important for DSB signaling: phosphorylation at serine 139, monoubiquitylation… (more)

Subjects/Keywords: Biology; H2A.X; H2A.X ubiquitylation; H2A.X monoubiquitylation; DNA damage; double-strand breaks; H2A.X phosphorylation

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APA (6th Edition):

D'Angelo, J. S. (2019). Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway. (Masters Thesis). York University. Retrieved from http://hdl.handle.net/10315/35812

Chicago Manual of Style (16th Edition):

D'Angelo, Jessica Sarah. “Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway.” 2019. Masters Thesis, York University. Accessed May 06, 2021. http://hdl.handle.net/10315/35812.

MLA Handbook (7th Edition):

D'Angelo, Jessica Sarah. “Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway.” 2019. Web. 06 May 2021.

Vancouver:

D'Angelo JS. Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway. [Internet] [Masters thesis]. York University; 2019. [cited 2021 May 06]. Available from: http://hdl.handle.net/10315/35812.

Council of Science Editors:

D'Angelo JS. Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway. [Masters Thesis]. York University; 2019. Available from: http://hdl.handle.net/10315/35812


University of Texas – Austin

14. Zhou, Yi, Ph. D. Regulation of DNA damage response by ATM and DNA-PKcs.

Degree: PhD, Cell and Molecular Biology, 2015, University of Texas – Austin

 The 5’ strand resection of DNA double strand breaks (DSBs) initiates homologous recombination (HR) and is critical for genomic stability. To date there is no… (more)

Subjects/Keywords: DNA repair; DNA damage response; Double strand breaks; ATM; DNA-PKcs; MRN; Exo1; DNA end resection

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APA (6th Edition):

Zhou, Yi, P. D. (2015). Regulation of DNA damage response by ATM and DNA-PKcs. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/63857

Chicago Manual of Style (16th Edition):

Zhou, Yi, Ph D. “Regulation of DNA damage response by ATM and DNA-PKcs.” 2015. Doctoral Dissertation, University of Texas – Austin. Accessed May 06, 2021. http://hdl.handle.net/2152/63857.

MLA Handbook (7th Edition):

Zhou, Yi, Ph D. “Regulation of DNA damage response by ATM and DNA-PKcs.” 2015. Web. 06 May 2021.

Vancouver:

Zhou, Yi PD. Regulation of DNA damage response by ATM and DNA-PKcs. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2015. [cited 2021 May 06]. Available from: http://hdl.handle.net/2152/63857.

Council of Science Editors:

Zhou, Yi PD. Regulation of DNA damage response by ATM and DNA-PKcs. [Doctoral Dissertation]. University of Texas – Austin; 2015. Available from: http://hdl.handle.net/2152/63857

15. Cohen, Sarah. Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks.

Degree: Docteur es, Biologie cellulaire, 2019, Université Toulouse III – Paul Sabatier

 Les gènes transcriptionellement actifs peuvent être la source de l'instabilité du génome via de nombreux mécanismes. Ces gènes sont caractérisés par la formation de structures… (more)

Subjects/Keywords: ADN; Cassures double-brins; Hybrides ARN: ADN; Résolution; Senataxine; DNA; Double strand breaks; RNA : DNA hybrids; Resolution; Senataxin

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APA (6th Edition):

Cohen, S. (2019). Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2019TOU30125

Chicago Manual of Style (16th Edition):

Cohen, Sarah. “Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks.” 2019. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed May 06, 2021. http://www.theses.fr/2019TOU30125.

MLA Handbook (7th Edition):

Cohen, Sarah. “Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks.” 2019. Web. 06 May 2021.

Vancouver:

Cohen S. Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2019. [cited 2021 May 06]. Available from: http://www.theses.fr/2019TOU30125.

Council of Science Editors:

Cohen S. Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2019. Available from: http://www.theses.fr/2019TOU30125

16. Gelot, Camille. Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends.

Degree: Docteur es, Immunologie, 2014, Université Pierre et Marie Curie – Paris VI

Au cours de la réplication, la réparation des cassures double brin (CDB) par recombinaison homologue (RH), basée sur la synthèse d’ADN à partir de la… (more)

Subjects/Keywords: Cassures double-brin; Réparation; Nhej; Cohésines; Translocations; Mobilité des extrémités; DNA double-strand breaks; C-NHEJ; 616.079

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APA (6th Edition):

Gelot, C. (2014). Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2014PA066300

Chicago Manual of Style (16th Edition):

Gelot, Camille. “Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends.” 2014. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed May 06, 2021. http://www.theses.fr/2014PA066300.

MLA Handbook (7th Edition):

Gelot, Camille. “Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends.” 2014. Web. 06 May 2021.

Vancouver:

Gelot C. Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. [cited 2021 May 06]. Available from: http://www.theses.fr/2014PA066300.

Council of Science Editors:

Gelot C. Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. Available from: http://www.theses.fr/2014PA066300


University of Edinburgh

17. Amarh, Vincent. Visualization of replication-dependent DNA double-strand break repair in Escherichia coli.

Degree: PhD, 2017, University of Edinburgh

 Chromosomal replication is a source of spontaneous DNA double-strand breaks (DSBs). In E. coli, DSBs are repaired by homologous recombination using an undamaged sister template.… (more)

Subjects/Keywords: 572.8; DNA double-strand breaks; e. coli; Escherichia coli; replicated DNA; rod-shaped E. coli; fluorescence microscopy; RecA protein; lacZ locus

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APA (6th Edition):

Amarh, V. (2017). Visualization of replication-dependent DNA double-strand break repair in Escherichia coli. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/29596

Chicago Manual of Style (16th Edition):

Amarh, Vincent. “Visualization of replication-dependent DNA double-strand break repair in Escherichia coli.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed May 06, 2021. http://hdl.handle.net/1842/29596.

MLA Handbook (7th Edition):

Amarh, Vincent. “Visualization of replication-dependent DNA double-strand break repair in Escherichia coli.” 2017. Web. 06 May 2021.

Vancouver:

Amarh V. Visualization of replication-dependent DNA double-strand break repair in Escherichia coli. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2021 May 06]. Available from: http://hdl.handle.net/1842/29596.

Council of Science Editors:

Amarh V. Visualization of replication-dependent DNA double-strand break repair in Escherichia coli. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/29596


Harvard University

18. Verkhedkar, Ketki Dinesh. Quantitative Analysis of DNA Repair and p53 in Individual Human Cells.

Degree: PhD, Systems Biology, 2012, Harvard University

 The goal of my research was to obtain a quantitative understanding of the mechanisms of DNA double-strand break (DSB) repair, and the activation of the… (more)

Subjects/Keywords: Systematic biology; Biology; Cellular biology; DNA damage response; DNA repair; Double-strand breaks; Live cell imaging; p53; Single cell analysis

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APA (6th Edition):

Verkhedkar, K. D. (2012). Quantitative Analysis of DNA Repair and p53 in Individual Human Cells. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476

Chicago Manual of Style (16th Edition):

Verkhedkar, Ketki Dinesh. “Quantitative Analysis of DNA Repair and p53 in Individual Human Cells.” 2012. Doctoral Dissertation, Harvard University. Accessed May 06, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476.

MLA Handbook (7th Edition):

Verkhedkar, Ketki Dinesh. “Quantitative Analysis of DNA Repair and p53 in Individual Human Cells.” 2012. Web. 06 May 2021.

Vancouver:

Verkhedkar KD. Quantitative Analysis of DNA Repair and p53 in Individual Human Cells. [Internet] [Doctoral dissertation]. Harvard University; 2012. [cited 2021 May 06]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476.

Council of Science Editors:

Verkhedkar KD. Quantitative Analysis of DNA Repair and p53 in Individual Human Cells. [Doctoral Dissertation]. Harvard University; 2012. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476

19. C. Rawal. ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE.

Degree: 2015, Università degli Studi di Milano

 The integrity of genomic DNA is continuously jeopardized through of environmental stresses such as UV light, ionizing radiations and various chemicals in addition to cellular… (more)

Subjects/Keywords: DNA damage checkpoint; DNA double strand breaks; Polo kinases/Cdc5; Slx4-Rtt107 complex; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Rawal, C. (2015). ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/335192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rawal, C.. “ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE.” 2015. Thesis, Università degli Studi di Milano. Accessed May 06, 2021. http://hdl.handle.net/2434/335192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rawal, C.. “ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE.” 2015. Web. 06 May 2021.

Vancouver:

Rawal C. ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 May 06]. Available from: http://hdl.handle.net/2434/335192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rawal C. ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/335192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

20. Walden, Elizabeth A. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.

Degree: 2017, University of Western Ontario

 Ku is a key component of the Non-Homologous End Joining DNA repair pathway. Recently, a function for Ku in DNA damage response (DDR) signalling was… (more)

Subjects/Keywords: DNA double strand breaks; Non-homologous end joining; DNA damage response; Cell cycle arrest; Ku; Aurora B; Biochemistry

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APA (6th Edition):

Walden, E. A. (2017). Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walden, Elizabeth A. “Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.” 2017. Thesis, University of Western Ontario. Accessed May 06, 2021. https://ir.lib.uwo.ca/etd/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walden, Elizabeth A. “Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.” 2017. Web. 06 May 2021.

Vancouver:

Walden EA. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2021 May 06]. Available from: https://ir.lib.uwo.ca/etd/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walden EA. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

21. Odebunmi, Oluwaseun. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .

Degree: 2020, University of Ottawa

 There is substantial evidence on the carcinogenic properties of high doses of Ionizing Radiation (IR), however, whether such risks exist following exposure to low doses… (more)

Subjects/Keywords: Ionizing Radiation; Myoblasts; Myogenicity; DNA damage; DNA repair; Double-strand Breaks; LDR priming; Homologous Recombination; Non-Homologous End Joining; Genomic Instability

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APA (6th Edition):

Odebunmi, O. (2020). Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/40413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Odebunmi, Oluwaseun. “Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .” 2020. Thesis, University of Ottawa. Accessed May 06, 2021. http://hdl.handle.net/10393/40413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Odebunmi, Oluwaseun. “Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .” 2020. Web. 06 May 2021.

Vancouver:

Odebunmi O. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . [Internet] [Thesis]. University of Ottawa; 2020. [cited 2021 May 06]. Available from: http://hdl.handle.net/10393/40413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Odebunmi O. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . [Thesis]. University of Ottawa; 2020. Available from: http://hdl.handle.net/10393/40413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

22. Jing Li (10611). Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).

Degree: 2017, University of Illinois – Chicago

 The repair of DNA double-strand breaks (DSB) is central to the maintenance of genomic integrity. Major DSB repair pathways in mammalian cells include homologous recombination… (more)

Subjects/Keywords: Uncategorized; tyrosyl-DNA phosphodiesterase 1 (TDP1), non-homologous end joining (NHEJ), DNA double-strand breaks (DSB)

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APA (6th Edition):

(10611), J. L. (2017). Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

(10611), Jing Li. “Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).” 2017. Thesis, University of Illinois – Chicago. Accessed May 06, 2021. http://hdl.handle.net/10027/22078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

(10611), Jing Li. “Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).” 2017. Web. 06 May 2021.

Vancouver:

(10611) JL. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2021 May 06]. Available from: http://hdl.handle.net/10027/22078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

(10611) JL. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/22078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Serra, Heïdi. Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana.

Degree: Docteur es, Physiologie et Génétique Moléculaires, 2014, Université Blaise-Pascale, Clermont-Ferrand II

La réparation des cassures double brin (CDB) de l'ADN par recombinaison est essentielle au maintien de l'intégrité du génome de tous les être vivants. Ce… (more)

Subjects/Keywords: Réparation de l'ADN; Cassure double brin; Recombinaison homologue; Single Strand Annealing; Synthesis Dependent Strand Annealing; XRCC2; RAD51B; RAD51C; XPF-ERCC1; Arabidopsis thaliana; DNA repair; Double-Strand DNA Breaks; Homologous DNA recombination; Single Strand Annealing; Synthesis Dependent Strand Annealing; XRCC2; RAD51B; RAD51C; XPF-ERCC1

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APA (6th Edition):

Serra, H. (2014). Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana. (Doctoral Dissertation). Université Blaise-Pascale, Clermont-Ferrand II. Retrieved from http://www.theses.fr/2014CLF22483

Chicago Manual of Style (16th Edition):

Serra, Heïdi. “Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana.” 2014. Doctoral Dissertation, Université Blaise-Pascale, Clermont-Ferrand II. Accessed May 06, 2021. http://www.theses.fr/2014CLF22483.

MLA Handbook (7th Edition):

Serra, Heïdi. “Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana.” 2014. Web. 06 May 2021.

Vancouver:

Serra H. Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana. [Internet] [Doctoral dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2014. [cited 2021 May 06]. Available from: http://www.theses.fr/2014CLF22483.

Council of Science Editors:

Serra H. Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana. [Doctoral Dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2014. Available from: http://www.theses.fr/2014CLF22483


Wright State University

24. Gadgil, Rujuta Yashodhan. Instability at Trinucleotide Repeat DNAs.

Degree: MS, Biochemistry and Molecular Biology, 2016, Wright State University

 Trinucleotide repeats (TNRs) are sequences prone to formation of non-B DNAstructures and mutations; undergo expansions in vivo to cause various inheritedneurodegenerative diseases. Hairpin structures formed… (more)

Subjects/Keywords: Biochemistry; Molecular Biology; Tri-nucleotide; repeats; non-B DNA; structures; inherited; neurodegenerative; replication; fork; stalling; single; double; strand; DNA; breaks; color; marker; gene; assay; detect; DNA; breaks

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gadgil, R. Y. (2016). Instability at Trinucleotide Repeat DNAs. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204

Chicago Manual of Style (16th Edition):

Gadgil, Rujuta Yashodhan. “Instability at Trinucleotide Repeat DNAs.” 2016. Masters Thesis, Wright State University. Accessed May 06, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

MLA Handbook (7th Edition):

Gadgil, Rujuta Yashodhan. “Instability at Trinucleotide Repeat DNAs.” 2016. Web. 06 May 2021.

Vancouver:

Gadgil RY. Instability at Trinucleotide Repeat DNAs. [Internet] [Masters thesis]. Wright State University; 2016. [cited 2021 May 06]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

Council of Science Editors:

Gadgil RY. Instability at Trinucleotide Repeat DNAs. [Masters Thesis]. Wright State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204


Indian Institute of Science

25. Mishra, Anup. Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance.

Degree: PhD, Faculty of Science, 2018, Indian Institute of Science

 To counteract the potentially calamitous effects of genomic instability in the form of double-strand breaks (DSBs), cells have evolved with two major mechanisms. First, DNA(more)

Subjects/Keywords: DNA Single-strand Repair Pathway; Mitochondrial DNA; Nucleoid Organization; mtDNA Replisome; RAD51C/XRCC3; DNA Double-strand Breaks (DSBs); DNA Double-strand Break Repair (DSRB) Pathway; DNA Repair Pathway; PARP1 Inhibitors; Mitochondrial Genome; FANCJ DNA Helicase; RAD51; Biochemistry

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APA (6th Edition):

Mishra, A. (2018). Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/4155

Chicago Manual of Style (16th Edition):

Mishra, Anup. “Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance.” 2018. Doctoral Dissertation, Indian Institute of Science. Accessed May 06, 2021. http://etd.iisc.ac.in/handle/2005/4155.

MLA Handbook (7th Edition):

Mishra, Anup. “Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance.” 2018. Web. 06 May 2021.

Vancouver:

Mishra A. Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2018. [cited 2021 May 06]. Available from: http://etd.iisc.ac.in/handle/2005/4155.

Council of Science Editors:

Mishra A. Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance. [Doctoral Dissertation]. Indian Institute of Science; 2018. Available from: http://etd.iisc.ac.in/handle/2005/4155


Queens University

26. Cutler, Geoffrey Lloyd. Characterizing Valproic Acid-Induced DNA Double Strand Break Repair .

Degree: Pharmacology and Toxicology, 2012, Queens University

 The teratogenic effects of valproic acid (VPA) are well known, though its teratogenic mechanism remains unknown. VPA induces oxidative stress, which may lead to double(more)

Subjects/Keywords: pKZ1 ; Valproic Acid ; RAD51 ; XRCC4 ; DNA Double Strand Breaks ; Homologous Recombination ; Non-Homologous End Joining ; Neural Tube Defects

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APA (6th Edition):

Cutler, G. L. (2012). Characterizing Valproic Acid-Induced DNA Double Strand Break Repair . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cutler, Geoffrey Lloyd. “Characterizing Valproic Acid-Induced DNA Double Strand Break Repair .” 2012. Thesis, Queens University. Accessed May 06, 2021. http://hdl.handle.net/1974/7597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cutler, Geoffrey Lloyd. “Characterizing Valproic Acid-Induced DNA Double Strand Break Repair .” 2012. Web. 06 May 2021.

Vancouver:

Cutler GL. Characterizing Valproic Acid-Induced DNA Double Strand Break Repair . [Internet] [Thesis]. Queens University; 2012. [cited 2021 May 06]. Available from: http://hdl.handle.net/1974/7597.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cutler GL. Characterizing Valproic Acid-Induced DNA Double Strand Break Repair . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7597

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Brinkmeier, Julia. Propriétés biochimiques de la Topoisomérase VI d’Ectocarpus siliculosus et ses facteurs accessoires potentiels : Biochemical properties of the Topoisomerase VI from Ectocarpus siliculosus and its potential accessory factors.

Degree: Docteur es, Biologie Santé, 2020, Montpellier

Les topoisomérases d'ADN (appelées topoisomérases) sont des enzymes essentielles qui jouent un rôle crucial dans les processus biologiques tels que la réplication, la transcription, la… (more)

Subjects/Keywords: Recombinaison; Méiose; Reproduction; Stabilité du génome; Cassures de l’ADN; Recombination; Meiosis; Reproduction; Genome stability; DNA double strand breaks

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APA (6th Edition):

Brinkmeier, J. (2020). Propriétés biochimiques de la Topoisomérase VI d’Ectocarpus siliculosus et ses facteurs accessoires potentiels : Biochemical properties of the Topoisomerase VI from Ectocarpus siliculosus and its potential accessory factors. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2020MONTT002

Chicago Manual of Style (16th Edition):

Brinkmeier, Julia. “Propriétés biochimiques de la Topoisomérase VI d’Ectocarpus siliculosus et ses facteurs accessoires potentiels : Biochemical properties of the Topoisomerase VI from Ectocarpus siliculosus and its potential accessory factors.” 2020. Doctoral Dissertation, Montpellier. Accessed May 06, 2021. http://www.theses.fr/2020MONTT002.

MLA Handbook (7th Edition):

Brinkmeier, Julia. “Propriétés biochimiques de la Topoisomérase VI d’Ectocarpus siliculosus et ses facteurs accessoires potentiels : Biochemical properties of the Topoisomerase VI from Ectocarpus siliculosus and its potential accessory factors.” 2020. Web. 06 May 2021.

Vancouver:

Brinkmeier J. Propriétés biochimiques de la Topoisomérase VI d’Ectocarpus siliculosus et ses facteurs accessoires potentiels : Biochemical properties of the Topoisomerase VI from Ectocarpus siliculosus and its potential accessory factors. [Internet] [Doctoral dissertation]. Montpellier; 2020. [cited 2021 May 06]. Available from: http://www.theses.fr/2020MONTT002.

Council of Science Editors:

Brinkmeier J. Propriétés biochimiques de la Topoisomérase VI d’Ectocarpus siliculosus et ses facteurs accessoires potentiels : Biochemical properties of the Topoisomerase VI from Ectocarpus siliculosus and its potential accessory factors. [Doctoral Dissertation]. Montpellier; 2020. Available from: http://www.theses.fr/2020MONTT002

28. S. Aliprandi. UNRAVELLING THE ROLE OF NOVEL FACTORS INVOLVED IN DOUBLE-STRAND BREAK REPAIR.

Degree: 2018, Università degli Studi di Milano

 Each day every cell of a living organism is constantly exposed to numerous DNA damages deriving both from the environment but also from its own… (more)

Subjects/Keywords: DAXX; DNA repair; Double strand breaks; histone variant; SLX4; resection; homologous recombination; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Aliprandi, S. (2018). UNRAVELLING THE ROLE OF NOVEL FACTORS INVOLVED IN DOUBLE-STRAND BREAK REPAIR. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/570044

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aliprandi, S.. “UNRAVELLING THE ROLE OF NOVEL FACTORS INVOLVED IN DOUBLE-STRAND BREAK REPAIR.” 2018. Thesis, Università degli Studi di Milano. Accessed May 06, 2021. http://hdl.handle.net/2434/570044.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aliprandi, S.. “UNRAVELLING THE ROLE OF NOVEL FACTORS INVOLVED IN DOUBLE-STRAND BREAK REPAIR.” 2018. Web. 06 May 2021.

Vancouver:

Aliprandi S. UNRAVELLING THE ROLE OF NOVEL FACTORS INVOLVED IN DOUBLE-STRAND BREAK REPAIR. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 May 06]. Available from: http://hdl.handle.net/2434/570044.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aliprandi S. UNRAVELLING THE ROLE OF NOVEL FACTORS INVOLVED IN DOUBLE-STRAND BREAK REPAIR. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/570044

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

29. Hoffer, Sarah M. Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining.

Degree: 2015, University of Western Ontario

DNA double strand breaks (DSBs) are a toxic and dangerous form of DNA damage repaired primarily by non-homologous end joining (NHEJ) in mammals. The Ku70/80… (more)

Subjects/Keywords: Ku70/80; non-homologous end joining; DNA double strand breaks; microirradiation; protein-protein interactions; lysine signaling; ionizing radiation; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hoffer, S. M. (2015). Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/3015

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoffer, Sarah M. “Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining.” 2015. Thesis, University of Western Ontario. Accessed May 06, 2021. https://ir.lib.uwo.ca/etd/3015.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoffer, Sarah M. “Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining.” 2015. Web. 06 May 2021.

Vancouver:

Hoffer SM. Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining. [Internet] [Thesis]. University of Western Ontario; 2015. [cited 2021 May 06]. Available from: https://ir.lib.uwo.ca/etd/3015.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoffer SM. Characterizing the Function of Helix Five of the Ku70 von Willebrand A Domain in Non-Homologous End Joining. [Thesis]. University of Western Ontario; 2015. Available from: https://ir.lib.uwo.ca/etd/3015

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

30. Wang, Huasheng. Structural and Functional Characterization of Non-Homologous End Joining Factors.

Degree: 2017, University of Western Ontario

DNA double strand breaks represent the most toxic form of DNA damage. In mammals, non-homologous end-joining (NHEJ) is the primary DNA repair pathway for such… (more)

Subjects/Keywords: DNA double strand breaks; Non-homologous end joining; Protein expression; Protein purification; Protein structure; X-ray crystallography; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, H. (2017). Structural and Functional Characterization of Non-Homologous End Joining Factors. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Huasheng. “Structural and Functional Characterization of Non-Homologous End Joining Factors.” 2017. Thesis, University of Western Ontario. Accessed May 06, 2021. https://ir.lib.uwo.ca/etd/4887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Huasheng. “Structural and Functional Characterization of Non-Homologous End Joining Factors.” 2017. Web. 06 May 2021.

Vancouver:

Wang H. Structural and Functional Characterization of Non-Homologous End Joining Factors. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2021 May 06]. Available from: https://ir.lib.uwo.ca/etd/4887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang H. Structural and Functional Characterization of Non-Homologous End Joining Factors. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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