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You searched for subject:(DNA double strand breaks DSB ). Showing records 1 – 30 of 18506 total matches.

[1] [2] [3] [4] [5] … [617]

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University of Illinois – Chicago

1. Li, Jing. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).

Degree: 2017, University of Illinois – Chicago

 The repair of DNA double-strand breaks (DSB) is central to the maintenance of genomic integrity. Major DSB repair pathways in mammalian cells include homologous recombination… (more)

Subjects/Keywords: tyrosyl-DNA phosphodiesterase 1 (TDP1); non-homologous end joining (NHEJ); DNA double-strand breaks (DSB)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, J. (2017). Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Jing. “Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).” 2017. Thesis, University of Illinois – Chicago. Accessed April 11, 2021. http://hdl.handle.net/10027/22078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Jing. “Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ).” 2017. Web. 11 Apr 2021.

Vancouver:

Li J. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10027/22078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li J. Investigating Role of Tyrosyl-DNA Phosphodiesterase 1 (TDP1) In Non-Homologous End Joining (NHEJ). [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/22078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

2. Lee, Eva I-Hua. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.

Degree: 2016, University of Toronto

Fanconi anemia (FA) patients are hypersensitive to ionizing radiation and other agents that generate DNA double-strand breaks (DSBs). The major error-free DSB repair pathway in… (more)

Subjects/Keywords: DNA repair; Double strand breaks; DSB resection; FANCD2; FANCJ; Fanconi Anemia; 0369

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APA (6th Edition):

Lee, E. I. (2016). Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/72732

Chicago Manual of Style (16th Edition):

Lee, Eva I-Hua. “Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.” 2016. Masters Thesis, University of Toronto. Accessed April 11, 2021. http://hdl.handle.net/1807/72732.

MLA Handbook (7th Edition):

Lee, Eva I-Hua. “Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells.” 2016. Web. 11 Apr 2021.

Vancouver:

Lee EI. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1807/72732.

Council of Science Editors:

Lee EI. Distinctive Roles for FANCD2 and FANCJ in the Resection of Radiation-induced DNA Double-strand Breaks in Human Cells. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/72732

3. Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.

Degree: 博士(医学), 2017, Nara Medical University / 奈良県立医科大学

DNA-dependent protein kinase (DNA-PK) is a serine/threonine kinase that plays an essential role in the repair of DNA double-strand breaks (DSBs) in the non-homologous end-joining… (more)

Subjects/Keywords: DNA double-strand breaks; Non-homologous end-joining; Acetylation; DNA-PKcs

Page 1

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APA (6th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, M. (2017). Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/3342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. “Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.” 2017. Thesis, Nara Medical University / 奈良県立医科大学. Accessed April 11, 2021. http://hdl.handle.net/10564/3342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mori, Eiichiro; Davis, Anthony J.; Hasegawa, Masatoshi. “Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である.” 2017. Web. 11 Apr 2021.

Vancouver:

Mori, Eiichiro; Davis, Anthony J.; Hasegawa M. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10564/3342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mori, Eiichiro; Davis, Anthony J.; Hasegawa M. Lysines 3241 and 3260 of DNA-PKcs are important for genomic stability and radioresistance. : DNA-PKcsのリジン3241と3260はゲノムの安定性と放射線抵抗性に重要である. [Thesis]. Nara Medical University / 奈良県立医科大学; 2017. Available from: http://hdl.handle.net/10564/3342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Giunta, Simona. DNA damage responses in the context of the cell division cycle.

Degree: PhD, 2010, University of Cambridge

 During my PhD, I have investigated aspects of the DNA damage response (DDR) in the context of three different cellular scenarios: DNA damage signalling in… (more)

Subjects/Keywords: DNA damage response; Cell cycle; Mitosis; DNA double strand breaks

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APA (6th Edition):

Giunta, S. (2010). DNA damage responses in the context of the cell division cycle. (Doctoral Dissertation). University of Cambridge. Retrieved from http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg

Chicago Manual of Style (16th Edition):

Giunta, Simona. “DNA damage responses in the context of the cell division cycle.” 2010. Doctoral Dissertation, University of Cambridge. Accessed April 11, 2021. http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg.

MLA Handbook (7th Edition):

Giunta, Simona. “DNA damage responses in the context of the cell division cycle.” 2010. Web. 11 Apr 2021.

Vancouver:

Giunta S. DNA damage responses in the context of the cell division cycle. [Internet] [Doctoral dissertation]. University of Cambridge; 2010. [cited 2021 Apr 11]. Available from: http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg.

Council of Science Editors:

Giunta S. DNA damage responses in the context of the cell division cycle. [Doctoral Dissertation]. University of Cambridge; 2010. Available from: http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg


University of Edinburgh

5. Vasianovich, Yuliya. Investigating the roles of the Srs2 and Pif1 helicases in DNA double-strand break repair in Saccharomyces cerevisiae.

Degree: PhD, 2015, University of Edinburgh

DNA double strand breaks (DSBs), which may occur during DNA replication or due to the action of genotoxic agents, are extremely dangerous DNA lesions as… (more)

Subjects/Keywords: 572.8; DNA; DNA double-stranded breaks; DSB; DSB repair; homologous recombination; de novo telomere addition; break-induced replication

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APA (6th Edition):

Vasianovich, Y. (2015). Investigating the roles of the Srs2 and Pif1 helicases in DNA double-strand break repair in Saccharomyces cerevisiae. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17984

Chicago Manual of Style (16th Edition):

Vasianovich, Yuliya. “Investigating the roles of the Srs2 and Pif1 helicases in DNA double-strand break repair in Saccharomyces cerevisiae.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed April 11, 2021. http://hdl.handle.net/1842/17984.

MLA Handbook (7th Edition):

Vasianovich, Yuliya. “Investigating the roles of the Srs2 and Pif1 helicases in DNA double-strand break repair in Saccharomyces cerevisiae.” 2015. Web. 11 Apr 2021.

Vancouver:

Vasianovich Y. Investigating the roles of the Srs2 and Pif1 helicases in DNA double-strand break repair in Saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1842/17984.

Council of Science Editors:

Vasianovich Y. Investigating the roles of the Srs2 and Pif1 helicases in DNA double-strand break repair in Saccharomyces cerevisiae. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/17984

6. Takabayashi, Hiroaki. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.

Degree: 博士(医学), 2013, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3807号. 学位記番号: 新大院博(医)甲第559号. 学位授与年月日: 平成25年9月20日

Human Pathology. 2013, 44(6), 1038–1046

p53-binding protein 1

Subjects/Keywords: Colorectal carcinoma; Double-strand breaks; DNA damage response; γH2AX

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APA (6th Edition):

Takabayashi, H. (2013). Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/24550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takabayashi, Hiroaki. “Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.” 2013. Thesis, Niigata University / 新潟大学. Accessed April 11, 2021. http://hdl.handle.net/10191/24550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takabayashi, Hiroaki. “Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.” 2013. Web. 11 Apr 2021.

Vancouver:

Takabayashi H. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. [Internet] [Thesis]. Niigata University / 新潟大学; 2013. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10191/24550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takabayashi H. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. [Thesis]. Niigata University / 新潟大学; 2013. Available from: http://hdl.handle.net/10191/24550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

7. Ku, Chuan-Chih. TCP6, a regulator in Arabidopsis gametophyte development and DNA damage response.

Degree: PhD, 2014, University of Edinburgh

 Plants have developed intricate mechanisms to control growth in response to a variety of environmental cues, to compensate its immobility and to survive in both… (more)

Subjects/Keywords: 572; TCP; gametophyte; pollen; embryo sac; cell cycle; DNA double-stranded breaks; DSB

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APA (6th Edition):

Ku, C. (2014). TCP6, a regulator in Arabidopsis gametophyte development and DNA damage response. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17892

Chicago Manual of Style (16th Edition):

Ku, Chuan-Chih. “TCP6, a regulator in Arabidopsis gametophyte development and DNA damage response.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed April 11, 2021. http://hdl.handle.net/1842/17892.

MLA Handbook (7th Edition):

Ku, Chuan-Chih. “TCP6, a regulator in Arabidopsis gametophyte development and DNA damage response.” 2014. Web. 11 Apr 2021.

Vancouver:

Ku C. TCP6, a regulator in Arabidopsis gametophyte development and DNA damage response. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1842/17892.

Council of Science Editors:

Ku C. TCP6, a regulator in Arabidopsis gametophyte development and DNA damage response. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/17892


University of California – Berkeley

8. Stamper, Ericca Lyn. Meiotic Double-Strand Break Formation in C. elegans.

Degree: Molecular & Cell Biology, 2014, University of California – Berkeley

 For most eukaryotes, recombination between homologous chromosomes during meiosis is an essential aspect of sexual reproduction. Meiotic recombination is initiated by programmed double-strand breaks (DSBs)… (more)

Subjects/Keywords: Cellular biology; Genetics; Molecular biology; C. elegans; crossover assurance; double-strand breaks; DSB-1; Meiosis; meiotic recombination

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APA (6th Edition):

Stamper, E. L. (2014). Meiotic Double-Strand Break Formation in C. elegans. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/3hn058dz

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stamper, Ericca Lyn. “Meiotic Double-Strand Break Formation in C. elegans.” 2014. Thesis, University of California – Berkeley. Accessed April 11, 2021. http://www.escholarship.org/uc/item/3hn058dz.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stamper, Ericca Lyn. “Meiotic Double-Strand Break Formation in C. elegans.” 2014. Web. 11 Apr 2021.

Vancouver:

Stamper EL. Meiotic Double-Strand Break Formation in C. elegans. [Internet] [Thesis]. University of California – Berkeley; 2014. [cited 2021 Apr 11]. Available from: http://www.escholarship.org/uc/item/3hn058dz.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stamper EL. Meiotic Double-Strand Break Formation in C. elegans. [Thesis]. University of California – Berkeley; 2014. Available from: http://www.escholarship.org/uc/item/3hn058dz

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. Passos, Joana. Implications of histone H2AX abundance in breast cancer.

Degree: 2020, NUI Galway

Histones are responsible for the packaging of DNA into a eukaryotic cell nucleus, and the H2A variant H2AX plays an important role in the DNA(more)

Subjects/Keywords: breast cancer; H2AX; copy number variation; DNA damage response; DNA double strand breaks; Medicine; Anatomy

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APA (6th Edition):

Passos, J. (2020). Implications of histone H2AX abundance in breast cancer. (Thesis). NUI Galway. Retrieved from http://hdl.handle.net/10379/15784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Passos, Joana. “Implications of histone H2AX abundance in breast cancer.” 2020. Thesis, NUI Galway. Accessed April 11, 2021. http://hdl.handle.net/10379/15784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Passos, Joana. “Implications of histone H2AX abundance in breast cancer.” 2020. Web. 11 Apr 2021.

Vancouver:

Passos J. Implications of histone H2AX abundance in breast cancer. [Internet] [Thesis]. NUI Galway; 2020. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10379/15784.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Passos J. Implications of histone H2AX abundance in breast cancer. [Thesis]. NUI Galway; 2020. Available from: http://hdl.handle.net/10379/15784

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

10. Young, Jordan. A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair.

Degree: PhD, 2016, University of Toronto

 All living organisms are continuously challenged by agents in their normal cellular environment that can inflict damage to their genetic material. DNA damage can have… (more)

Subjects/Keywords: Cell microarrays; DNA double-strand breaks; DNA repair; High-content screening; Homologous recombination; Microcephaly; 0379

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APA (6th Edition):

Young, J. (2016). A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/77384

Chicago Manual of Style (16th Edition):

Young, Jordan. “A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair.” 2016. Doctoral Dissertation, University of Toronto. Accessed April 11, 2021. http://hdl.handle.net/1807/77384.

MLA Handbook (7th Edition):

Young, Jordan. “A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair.” 2016. Web. 11 Apr 2021.

Vancouver:

Young J. A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1807/77384.

Council of Science Editors:

Young J. A Genome-scale RNA Interference Screen Identifies Novel Regulators of DNA Double-strand Break Repair. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/77384


University of Texas – Austin

11. -2937-0948. Mechanism of repair of Mu DNA insertions.

Degree: PhD, Cell and Molecular Biology, 2015, University of Texas – Austin

 Transposable elements are ubiquitous, occupying as much as 85% of the genome of some species, and nearly 50% of the human genome, and causing DNA(more)

Subjects/Keywords: Mu DNA transposition; Post-integration; DNA replication and repair; Double-strand breaks

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APA (6th Edition):

-2937-0948. (2015). Mechanism of repair of Mu DNA insertions. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/46541

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-2937-0948. “Mechanism of repair of Mu DNA insertions.” 2015. Doctoral Dissertation, University of Texas – Austin. Accessed April 11, 2021. http://hdl.handle.net/2152/46541.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-2937-0948. “Mechanism of repair of Mu DNA insertions.” 2015. Web. 11 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-2937-0948. Mechanism of repair of Mu DNA insertions. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2015. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2152/46541.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-2937-0948. Mechanism of repair of Mu DNA insertions. [Doctoral Dissertation]. University of Texas – Austin; 2015. Available from: http://hdl.handle.net/2152/46541

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

12. Moretton, Amandine. Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks.

Degree: Docteur es, Physiologie et Génétique Moléculaire, 2017, Université Clermont Auvergne‎ (2017-2020)

Les mitochondries sont des organites qui possèdent leur propre ADN (ADNmt), codant pour des gènes de la chaine respiratoire. La réparation des dommages dus aux… (more)

Subjects/Keywords: ADN mitochondrial; Cassures double-brin; Dégradation de l’ADN; Réparation de l’ADN; Mitochondrial DNA; Double-strand breaks; DNA degradation; DNA repair

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APA (6th Edition):

Moretton, A. (2017). Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks. (Doctoral Dissertation). Université Clermont Auvergne‎ (2017-2020). Retrieved from http://www.theses.fr/2017CLFAC047

Chicago Manual of Style (16th Edition):

Moretton, Amandine. “Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks.” 2017. Doctoral Dissertation, Université Clermont Auvergne‎ (2017-2020). Accessed April 11, 2021. http://www.theses.fr/2017CLFAC047.

MLA Handbook (7th Edition):

Moretton, Amandine. “Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks.” 2017. Web. 11 Apr 2021.

Vancouver:

Moretton A. Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks. [Internet] [Doctoral dissertation]. Université Clermont Auvergne‎ (2017-2020); 2017. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2017CLFAC047.

Council of Science Editors:

Moretton A. Mécanismes de maintenance de l'intégrité de l'ADN mitochondrial humain suite à des cassures double-brin : Maintenance of human mitochondrial DNA after double-strand breaks. [Doctoral Dissertation]. Université Clermont Auvergne‎ (2017-2020); 2017. Available from: http://www.theses.fr/2017CLFAC047


NSYSU

13. Yang, Chun-feng. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

 Breast cancer is a leading cause of death in women worldwide, and chemotherapy is one of the primary strategies for breast cancer treatment. However, the… (more)

Subjects/Keywords: DNA double-strand breaks; camptothecin; Phthalate; Bis(2-ethylhexyl) phthalate; DEHP; zebrafish; chemoresistance; breast cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, C. (2016). The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Thesis, NSYSU. Accessed April 11, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Web. 11 Apr 2021.

Vancouver:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Apr 11]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

14. Martin, Nathan. Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP.

Degree: Biomedical Physics, 2014, UCLA

 ABSTRACT OF THE DISSERTATIONRepair of DNA double strand breaks and radiosensitivity:modification of DNA repair and radiosensitivity bymicroRNA-335 and mtPAPby Nathan Thomas MartinDoctor of Philosophy in… (more)

Subjects/Keywords: Cellular biology; Biophysics; cancer; DNA repair; double strand breaks; radiobiology; Radiosensitivity; XCIND

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APA (6th Edition):

Martin, N. (2014). Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/26w5v41k

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martin, Nathan. “Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP.” 2014. Thesis, UCLA. Accessed April 11, 2021. http://www.escholarship.org/uc/item/26w5v41k.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martin, Nathan. “Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP.” 2014. Web. 11 Apr 2021.

Vancouver:

Martin N. Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP. [Internet] [Thesis]. UCLA; 2014. [cited 2021 Apr 11]. Available from: http://www.escholarship.org/uc/item/26w5v41k.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martin N. Repair of DNA double strand breaks and radiosensitivity: modulation of DNA repair and radiosensitivity by microRNA-335 and mtPAP. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/26w5v41k

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

15. Andreani, Matteo. Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden.

Degree: 2020, Freie Universität Berlin

 Das Rap1-interacting factor 1 (RIF1) ist ein multifunktionales Protein, welches unter anderem den Prozess der DNA-Replikation zeitlich koordiniert sowie am Neustart blockierter Replikationsgabeln, der Auflösung… (more)

Subjects/Keywords: RIF1; DNA repair; Class Switch Recombination; BRCA1-deficiency; Double-strand breaks; ddc:570

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APA (6th Edition):

Andreani, M. (2020). Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-26996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andreani, Matteo. “Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden.” 2020. Thesis, Freie Universität Berlin. Accessed April 11, 2021. http://dx.doi.org/10.17169/refubium-26996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andreani, Matteo. “Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden.” 2020. Web. 11 Apr 2021.

Vancouver:

Andreani M. Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2021 Apr 11]. Available from: http://dx.doi.org/10.17169/refubium-26996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andreani M. Molekulare Anforderungen an die Rolle von RIF1 beim Schutz von DNA-Doppelstrang-Bruchenden. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-26996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

16. Baldascini, Marta. Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks .

Degree: 2019, National University of Ireland – Galway

 The integrity of our genome is very important therefore, our cells have developed efficient DNA repair mechanisms to control this integrity and avoid mutations. Although… (more)

Subjects/Keywords: DNA damage; zinc finger proteins; ZC3H8; ZC3H11A; ZC3H14; double strand breaks; Biochemistry; Science; Natural Sciences

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APA (6th Edition):

Baldascini, M. (2019). Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/15652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baldascini, Marta. “Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks .” 2019. Thesis, National University of Ireland – Galway. Accessed April 11, 2021. http://hdl.handle.net/10379/15652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baldascini, Marta. “Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks .” 2019. Web. 11 Apr 2021.

Vancouver:

Baldascini M. Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks . [Internet] [Thesis]. National University of Ireland – Galway; 2019. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10379/15652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baldascini M. Characterisation of three zinc finger proteins ZC3H8, ZC3H11A and ZC3H14, identified as new partners of ATM, the central regulator of biological response to DNA double strand breaks . [Thesis]. National University of Ireland – Galway; 2019. Available from: http://hdl.handle.net/10379/15652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


York University

17. D'Angelo, Jessica Sarah. Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway.

Degree: MSc -MS, Biology, 2019, York University

 In response to DNA double-strand breaks (DSBs), H2A.X undergoes three post-translational modifications (PTMs) reported to be important for DSB signaling: phosphorylation at serine 139, monoubiquitylation… (more)

Subjects/Keywords: Biology; H2A.X; H2A.X ubiquitylation; H2A.X monoubiquitylation; DNA damage; double-strand breaks; H2A.X phosphorylation

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APA (6th Edition):

D'Angelo, J. S. (2019). Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway. (Masters Thesis). York University. Retrieved from http://hdl.handle.net/10315/35812

Chicago Manual of Style (16th Edition):

D'Angelo, Jessica Sarah. “Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway.” 2019. Masters Thesis, York University. Accessed April 11, 2021. http://hdl.handle.net/10315/35812.

MLA Handbook (7th Edition):

D'Angelo, Jessica Sarah. “Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway.” 2019. Web. 11 Apr 2021.

Vancouver:

D'Angelo JS. Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway. [Internet] [Masters thesis]. York University; 2019. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10315/35812.

Council of Science Editors:

D'Angelo JS. Elucidating the Mechanisms Surrounding H2A.X Monoubiquitylation Marks in the DNA Damage Response Pathway. [Masters Thesis]. York University; 2019. Available from: http://hdl.handle.net/10315/35812


Louisiana State University

18. Trieu, Tommy Nhan. Saccharomyces cerevisiae Tel1 & Tel2 role in DNA double-strand break repairs.

Degree: MSChE, Chemical Engineering, 2015, Louisiana State University

 Genomic DNA is constantly in danger of being damaged by endogenous cellular processes and exogenous agents. Eukaryotes have mechanisms, collectively termed the DNA damage response,… (more)

Subjects/Keywords: RESECTION; DSB; qPCR; Yeast; quantitative real-time PCR; SACCHAROMYCES CEREVISIAE; TEL1; TEL2; DNA REPAIR; DNA damage; DOUBLE-STRAND BREAK

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APA (6th Edition):

Trieu, T. N. (2015). Saccharomyces cerevisiae Tel1 & Tel2 role in DNA double-strand break repairs. (Masters Thesis). Louisiana State University. Retrieved from etd-08242015-155429 ; https://digitalcommons.lsu.edu/gradschool_theses/3434

Chicago Manual of Style (16th Edition):

Trieu, Tommy Nhan. “Saccharomyces cerevisiae Tel1 & Tel2 role in DNA double-strand break repairs.” 2015. Masters Thesis, Louisiana State University. Accessed April 11, 2021. etd-08242015-155429 ; https://digitalcommons.lsu.edu/gradschool_theses/3434.

MLA Handbook (7th Edition):

Trieu, Tommy Nhan. “Saccharomyces cerevisiae Tel1 & Tel2 role in DNA double-strand break repairs.” 2015. Web. 11 Apr 2021.

Vancouver:

Trieu TN. Saccharomyces cerevisiae Tel1 & Tel2 role in DNA double-strand break repairs. [Internet] [Masters thesis]. Louisiana State University; 2015. [cited 2021 Apr 11]. Available from: etd-08242015-155429 ; https://digitalcommons.lsu.edu/gradschool_theses/3434.

Council of Science Editors:

Trieu TN. Saccharomyces cerevisiae Tel1 & Tel2 role in DNA double-strand break repairs. [Masters Thesis]. Louisiana State University; 2015. Available from: etd-08242015-155429 ; https://digitalcommons.lsu.edu/gradschool_theses/3434


University of Texas – Austin

19. Zhou, Yi, Ph. D. Regulation of DNA damage response by ATM and DNA-PKcs.

Degree: PhD, Cell and Molecular Biology, 2015, University of Texas – Austin

 The 5’ strand resection of DNA double strand breaks (DSBs) initiates homologous recombination (HR) and is critical for genomic stability. To date there is no… (more)

Subjects/Keywords: DNA repair; DNA damage response; Double strand breaks; ATM; DNA-PKcs; MRN; Exo1; DNA end resection

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APA (6th Edition):

Zhou, Yi, P. D. (2015). Regulation of DNA damage response by ATM and DNA-PKcs. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/63857

Chicago Manual of Style (16th Edition):

Zhou, Yi, Ph D. “Regulation of DNA damage response by ATM and DNA-PKcs.” 2015. Doctoral Dissertation, University of Texas – Austin. Accessed April 11, 2021. http://hdl.handle.net/2152/63857.

MLA Handbook (7th Edition):

Zhou, Yi, Ph D. “Regulation of DNA damage response by ATM and DNA-PKcs.” 2015. Web. 11 Apr 2021.

Vancouver:

Zhou, Yi PD. Regulation of DNA damage response by ATM and DNA-PKcs. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2015. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2152/63857.

Council of Science Editors:

Zhou, Yi PD. Regulation of DNA damage response by ATM and DNA-PKcs. [Doctoral Dissertation]. University of Texas – Austin; 2015. Available from: http://hdl.handle.net/2152/63857

20. Cohen, Sarah. Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks.

Degree: Docteur es, Biologie cellulaire, 2019, Université Toulouse III – Paul Sabatier

 Les gènes transcriptionellement actifs peuvent être la source de l'instabilité du génome via de nombreux mécanismes. Ces gènes sont caractérisés par la formation de structures… (more)

Subjects/Keywords: ADN; Cassures double-brins; Hybrides ARN: ADN; Résolution; Senataxine; DNA; Double strand breaks; RNA : DNA hybrids; Resolution; Senataxin

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APA (6th Edition):

Cohen, S. (2019). Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2019TOU30125

Chicago Manual of Style (16th Edition):

Cohen, Sarah. “Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks.” 2019. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed April 11, 2021. http://www.theses.fr/2019TOU30125.

MLA Handbook (7th Edition):

Cohen, Sarah. “Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks.” 2019. Web. 11 Apr 2021.

Vancouver:

Cohen S. Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2019. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2019TOU30125.

Council of Science Editors:

Cohen S. Le rôle de senataxine dans la résolution des hybrides ARN : ADN aux cassures double brins de l'ADN : Role of senataxin in RNA : DNA hybrids resolution at DNA double strand breaks. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2019. Available from: http://www.theses.fr/2019TOU30125

21. Gelot, Camille. Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends.

Degree: Docteur es, Immunologie, 2014, Université Pierre et Marie Curie – Paris VI

Au cours de la réplication, la réparation des cassures double brin (CDB) par recombinaison homologue (RH), basée sur la synthèse d’ADN à partir de la… (more)

Subjects/Keywords: Cassures double-brin; Réparation; Nhej; Cohésines; Translocations; Mobilité des extrémités; DNA double-strand breaks; C-NHEJ; 616.079

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APA (6th Edition):

Gelot, C. (2014). Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2014PA066300

Chicago Manual of Style (16th Edition):

Gelot, Camille. “Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends.” 2014. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed April 11, 2021. http://www.theses.fr/2014PA066300.

MLA Handbook (7th Edition):

Gelot, Camille. “Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends.” 2014. Web. 11 Apr 2021.

Vancouver:

Gelot C. Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2014PA066300.

Council of Science Editors:

Gelot C. Rôle du complexe de cohésion sur la ligature d'extrémités d'ADN non homologues et la stabilité du génome : The cohesin complex protects against genome rearrangements by preventing the end-joining of distal DNA double-strand-ends. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2014. Available from: http://www.theses.fr/2014PA066300


University of Edinburgh

22. Amarh, Vincent. Visualization of replication-dependent DNA double-strand break repair in Escherichia coli.

Degree: PhD, 2017, University of Edinburgh

 Chromosomal replication is a source of spontaneous DNA double-strand breaks (DSBs). In E. coli, DSBs are repaired by homologous recombination using an undamaged sister template.… (more)

Subjects/Keywords: 572.8; DNA double-strand breaks; e. coli; Escherichia coli; replicated DNA; rod-shaped E. coli; fluorescence microscopy; RecA protein; lacZ locus

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APA (6th Edition):

Amarh, V. (2017). Visualization of replication-dependent DNA double-strand break repair in Escherichia coli. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/29596

Chicago Manual of Style (16th Edition):

Amarh, Vincent. “Visualization of replication-dependent DNA double-strand break repair in Escherichia coli.” 2017. Doctoral Dissertation, University of Edinburgh. Accessed April 11, 2021. http://hdl.handle.net/1842/29596.

MLA Handbook (7th Edition):

Amarh, Vincent. “Visualization of replication-dependent DNA double-strand break repair in Escherichia coli.” 2017. Web. 11 Apr 2021.

Vancouver:

Amarh V. Visualization of replication-dependent DNA double-strand break repair in Escherichia coli. [Internet] [Doctoral dissertation]. University of Edinburgh; 2017. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/1842/29596.

Council of Science Editors:

Amarh V. Visualization of replication-dependent DNA double-strand break repair in Escherichia coli. [Doctoral Dissertation]. University of Edinburgh; 2017. Available from: http://hdl.handle.net/1842/29596


Harvard University

23. Verkhedkar, Ketki Dinesh. Quantitative Analysis of DNA Repair and p53 in Individual Human Cells.

Degree: PhD, Systems Biology, 2012, Harvard University

 The goal of my research was to obtain a quantitative understanding of the mechanisms of DNA double-strand break (DSB) repair, and the activation of the… (more)

Subjects/Keywords: Systematic biology; Biology; Cellular biology; DNA damage response; DNA repair; Double-strand breaks; Live cell imaging; p53; Single cell analysis

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APA (6th Edition):

Verkhedkar, K. D. (2012). Quantitative Analysis of DNA Repair and p53 in Individual Human Cells. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476

Chicago Manual of Style (16th Edition):

Verkhedkar, Ketki Dinesh. “Quantitative Analysis of DNA Repair and p53 in Individual Human Cells.” 2012. Doctoral Dissertation, Harvard University. Accessed April 11, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476.

MLA Handbook (7th Edition):

Verkhedkar, Ketki Dinesh. “Quantitative Analysis of DNA Repair and p53 in Individual Human Cells.” 2012. Web. 11 Apr 2021.

Vancouver:

Verkhedkar KD. Quantitative Analysis of DNA Repair and p53 in Individual Human Cells. [Internet] [Doctoral dissertation]. Harvard University; 2012. [cited 2021 Apr 11]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476.

Council of Science Editors:

Verkhedkar KD. Quantitative Analysis of DNA Repair and p53 in Individual Human Cells. [Doctoral Dissertation]. Harvard University; 2012. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:10433476

24. C. Rawal. ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE.

Degree: 2015, Università degli Studi di Milano

 The integrity of genomic DNA is continuously jeopardized through of environmental stresses such as UV light, ionizing radiations and various chemicals in addition to cellular… (more)

Subjects/Keywords: DNA damage checkpoint; DNA double strand breaks; Polo kinases/Cdc5; Slx4-Rtt107 complex; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Rawal, C. (2015). ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/335192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rawal, C.. “ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE.” 2015. Thesis, Università degli Studi di Milano. Accessed April 11, 2021. http://hdl.handle.net/2434/335192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rawal, C.. “ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE.” 2015. Web. 11 Apr 2021.

Vancouver:

Rawal C. ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/2434/335192.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rawal C. ROLE OF POLO KINASE CDC5 AND SLX4-RTT107 COMPLEX IN CHECKPOINT SIGNALING DURING DNA DAMAGE IN S. CEREVISIAE. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/335192

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

25. Walden, Elizabeth A. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.

Degree: 2017, University of Western Ontario

 Ku is a key component of the Non-Homologous End Joining DNA repair pathway. Recently, a function for Ku in DNA damage response (DDR) signalling was… (more)

Subjects/Keywords: DNA double strand breaks; Non-homologous end joining; DNA damage response; Cell cycle arrest; Ku; Aurora B; Biochemistry

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APA (6th Edition):

Walden, E. A. (2017). Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walden, Elizabeth A. “Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.” 2017. Thesis, University of Western Ontario. Accessed April 11, 2021. https://ir.lib.uwo.ca/etd/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walden, Elizabeth A. “Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response.” 2017. Web. 11 Apr 2021.

Vancouver:

Walden EA. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2021 Apr 11]. Available from: https://ir.lib.uwo.ca/etd/4462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walden EA. Assessing the role of Ku70 vWA domain phosphorylation in the inhibition of Aurora B and activation of the DNA damage response. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/4462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

26. Odebunmi, Oluwaseun. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .

Degree: 2020, University of Ottawa

 There is substantial evidence on the carcinogenic properties of high doses of Ionizing Radiation (IR), however, whether such risks exist following exposure to low doses… (more)

Subjects/Keywords: Ionizing Radiation; Myoblasts; Myogenicity; DNA damage; DNA repair; Double-strand Breaks; LDR priming; Homologous Recombination; Non-Homologous End Joining; Genomic Instability

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APA (6th Edition):

Odebunmi, O. (2020). Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/40413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Odebunmi, Oluwaseun. “Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .” 2020. Thesis, University of Ottawa. Accessed April 11, 2021. http://hdl.handle.net/10393/40413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Odebunmi, Oluwaseun. “Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells .” 2020. Web. 11 Apr 2021.

Vancouver:

Odebunmi O. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . [Internet] [Thesis]. University of Ottawa; 2020. [cited 2021 Apr 11]. Available from: http://hdl.handle.net/10393/40413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Odebunmi O. Effects of Low Dose Ionizing Radiation on DNA Damage and Repair Response in Proliferating Muscle Stem Cells . [Thesis]. University of Ottawa; 2020. Available from: http://hdl.handle.net/10393/40413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Meylan, Sylvain. Développement d'un outil de simulation multi-échelle adapté au calcul des dommages radio-induits précoces dans des cellules exposées à des irradiations d'ions légers (proton et alpha) : Development of a multi-scale simulation tool for early radio-induced damage assessment in cells exposed to light ions irradiations (proton and alpha).

Degree: Docteur es, Astrophysique, plasmas, nucléaire, 2016, Bordeaux

Ce travail de thèse, réalisé dans le cadre des projets de recherche ROSIRIS (IRSN) et Geant4-DNA, porte sur la construction d’une simulation multi-échelle dédiée au… (more)

Subjects/Keywords: ADN; Irradiation; Génome humain; Geant4; Geant4-DNA; DnaFabric; SSB; DSB; Cassure double brin; Cassure simple brin; Multi-échelle; 3D; Simulation; DNA; Irradiation; Human genome; Geant4; Geant4-DNA; DnaFabric; SSB; DSB; Double strand beak; Single strand break; Multi-scale; 3D; Simulation

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APA (6th Edition):

Meylan, S. (2016). Développement d'un outil de simulation multi-échelle adapté au calcul des dommages radio-induits précoces dans des cellules exposées à des irradiations d'ions légers (proton et alpha) : Development of a multi-scale simulation tool for early radio-induced damage assessment in cells exposed to light ions irradiations (proton and alpha). (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2016BORD0184

Chicago Manual of Style (16th Edition):

Meylan, Sylvain. “Développement d'un outil de simulation multi-échelle adapté au calcul des dommages radio-induits précoces dans des cellules exposées à des irradiations d'ions légers (proton et alpha) : Development of a multi-scale simulation tool for early radio-induced damage assessment in cells exposed to light ions irradiations (proton and alpha).” 2016. Doctoral Dissertation, Bordeaux. Accessed April 11, 2021. http://www.theses.fr/2016BORD0184.

MLA Handbook (7th Edition):

Meylan, Sylvain. “Développement d'un outil de simulation multi-échelle adapté au calcul des dommages radio-induits précoces dans des cellules exposées à des irradiations d'ions légers (proton et alpha) : Development of a multi-scale simulation tool for early radio-induced damage assessment in cells exposed to light ions irradiations (proton and alpha).” 2016. Web. 11 Apr 2021.

Vancouver:

Meylan S. Développement d'un outil de simulation multi-échelle adapté au calcul des dommages radio-induits précoces dans des cellules exposées à des irradiations d'ions légers (proton et alpha) : Development of a multi-scale simulation tool for early radio-induced damage assessment in cells exposed to light ions irradiations (proton and alpha). [Internet] [Doctoral dissertation]. Bordeaux; 2016. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2016BORD0184.

Council of Science Editors:

Meylan S. Développement d'un outil de simulation multi-échelle adapté au calcul des dommages radio-induits précoces dans des cellules exposées à des irradiations d'ions légers (proton et alpha) : Development of a multi-scale simulation tool for early radio-induced damage assessment in cells exposed to light ions irradiations (proton and alpha). [Doctoral Dissertation]. Bordeaux; 2016. Available from: http://www.theses.fr/2016BORD0184

28. Serra, Heïdi. Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana.

Degree: Docteur es, Physiologie et Génétique Moléculaires, 2014, Université Blaise-Pascale, Clermont-Ferrand II

La réparation des cassures double brin (CDB) de l'ADN par recombinaison est essentielle au maintien de l'intégrité du génome de tous les être vivants. Ce… (more)

Subjects/Keywords: Réparation de l'ADN; Cassure double brin; Recombinaison homologue; Single Strand Annealing; Synthesis Dependent Strand Annealing; XRCC2; RAD51B; RAD51C; XPF-ERCC1; Arabidopsis thaliana; DNA repair; Double-Strand DNA Breaks; Homologous DNA recombination; Single Strand Annealing; Synthesis Dependent Strand Annealing; XRCC2; RAD51B; RAD51C; XPF-ERCC1

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APA (6th Edition):

Serra, H. (2014). Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana. (Doctoral Dissertation). Université Blaise-Pascale, Clermont-Ferrand II. Retrieved from http://www.theses.fr/2014CLF22483

Chicago Manual of Style (16th Edition):

Serra, Heïdi. “Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana.” 2014. Doctoral Dissertation, Université Blaise-Pascale, Clermont-Ferrand II. Accessed April 11, 2021. http://www.theses.fr/2014CLF22483.

MLA Handbook (7th Edition):

Serra, Heïdi. “Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana.” 2014. Web. 11 Apr 2021.

Vancouver:

Serra H. Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana. [Internet] [Doctoral dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2014. [cited 2021 Apr 11]. Available from: http://www.theses.fr/2014CLF22483.

Council of Science Editors:

Serra H. Etude des acteurs et des interactions entre les voies de recombinaison chez Arabidopsis thaliana : Study of the actors and of the interactions between the recombination pathways of Arabidopsis thaliana. [Doctoral Dissertation]. Université Blaise-Pascale, Clermont-Ferrand II; 2014. Available from: http://www.theses.fr/2014CLF22483


Wright State University

29. Gadgil, Rujuta Yashodhan. Instability at Trinucleotide Repeat DNAs.

Degree: MS, Biochemistry and Molecular Biology, 2016, Wright State University

 Trinucleotide repeats (TNRs) are sequences prone to formation of non-B DNAstructures and mutations; undergo expansions in vivo to cause various inheritedneurodegenerative diseases. Hairpin structures formed… (more)

Subjects/Keywords: Biochemistry; Molecular Biology; Tri-nucleotide; repeats; non-B DNA; structures; inherited; neurodegenerative; replication; fork; stalling; single; double; strand; DNA; breaks; color; marker; gene; assay; detect; DNA; breaks

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APA (6th Edition):

Gadgil, R. Y. (2016). Instability at Trinucleotide Repeat DNAs. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204

Chicago Manual of Style (16th Edition):

Gadgil, Rujuta Yashodhan. “Instability at Trinucleotide Repeat DNAs.” 2016. Masters Thesis, Wright State University. Accessed April 11, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

MLA Handbook (7th Edition):

Gadgil, Rujuta Yashodhan. “Instability at Trinucleotide Repeat DNAs.” 2016. Web. 11 Apr 2021.

Vancouver:

Gadgil RY. Instability at Trinucleotide Repeat DNAs. [Internet] [Masters thesis]. Wright State University; 2016. [cited 2021 Apr 11]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204.

Council of Science Editors:

Gadgil RY. Instability at Trinucleotide Repeat DNAs. [Masters Thesis]. Wright State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1472231204


Indian Institute of Science

30. Mishra, Anup. Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance.

Degree: PhD, Faculty of Science, 2018, Indian Institute of Science

 To counteract the potentially calamitous effects of genomic instability in the form of double-strand breaks (DSBs), cells have evolved with two major mechanisms. First, DNA(more)

Subjects/Keywords: DNA Single-strand Repair Pathway; Mitochondrial DNA; Nucleoid Organization; mtDNA Replisome; RAD51C/XRCC3; DNA Double-strand Breaks (DSBs); DNA Double-strand Break Repair (DSRB) Pathway; DNA Repair Pathway; PARP1 Inhibitors; Mitochondrial Genome; FANCJ DNA Helicase; RAD51; Biochemistry

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APA (6th Edition):

Mishra, A. (2018). Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/4155

Chicago Manual of Style (16th Edition):

Mishra, Anup. “Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance.” 2018. Doctoral Dissertation, Indian Institute of Science. Accessed April 11, 2021. http://etd.iisc.ac.in/handle/2005/4155.

MLA Handbook (7th Edition):

Mishra, Anup. “Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance.” 2018. Web. 11 Apr 2021.

Vancouver:

Mishra A. Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2018. [cited 2021 Apr 11]. Available from: http://etd.iisc.ac.in/handle/2005/4155.

Council of Science Editors:

Mishra A. Targeting RAD51C Pathological Mutants by Synthetic Lethality and Extended Functions of RAD51C/XRCC3 in Mitochondrial Genome Maintenance. [Doctoral Dissertation]. Indian Institute of Science; 2018. Available from: http://etd.iisc.ac.in/handle/2005/4155

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