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You searched for subject:(DNA damage response). Showing records 1 – 30 of 253 total matches.

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Queens University

1. Kelly, Ryan. The interaction between Rad9 and Tousled-like kinase 1 in the cell cycle and the DNA damage response .

Degree: Biochemistry, 2013, Queens University

 Genomic integrity is preserved by checkpoints, which are signal transduction pathways that serve to delay cell cycle progression in the presence of DNA damage or… (more)

Subjects/Keywords: cancer biology ; DNA damage response

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APA (6th Edition):

Kelly, R. (2013). The interaction between Rad9 and Tousled-like kinase 1 in the cell cycle and the DNA damage response . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kelly, Ryan. “The interaction between Rad9 and Tousled-like kinase 1 in the cell cycle and the DNA damage response .” 2013. Thesis, Queens University. Accessed April 20, 2021. http://hdl.handle.net/1974/8536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kelly, Ryan. “The interaction between Rad9 and Tousled-like kinase 1 in the cell cycle and the DNA damage response .” 2013. Web. 20 Apr 2021.

Vancouver:

Kelly R. The interaction between Rad9 and Tousled-like kinase 1 in the cell cycle and the DNA damage response . [Internet] [Thesis]. Queens University; 2013. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1974/8536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kelly R. The interaction between Rad9 and Tousled-like kinase 1 in the cell cycle and the DNA damage response . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

2. Luo, Yong. MOLECULAR MECHANISMS OF THE DNA DAMAGE RESPONSE INDUCED DURING PARVOVIRUS INFECTION.

Degree: PhD, Microbiology, Molecular Genetics & Immunology, 2012, University of Kansas

DNA damage response (DDR) is a critical safeguarding system to protect genomic stability and integrality through a cascade of phosphorylation events of three PI-3-kinase-like kinases:… (more)

Subjects/Keywords: Microbiology; Dna damage response; Dna replication; Parvovirus

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APA (6th Edition):

Luo, Y. (2012). MOLECULAR MECHANISMS OF THE DNA DAMAGE RESPONSE INDUCED DURING PARVOVIRUS INFECTION. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/14837

Chicago Manual of Style (16th Edition):

Luo, Yong. “MOLECULAR MECHANISMS OF THE DNA DAMAGE RESPONSE INDUCED DURING PARVOVIRUS INFECTION.” 2012. Doctoral Dissertation, University of Kansas. Accessed April 20, 2021. http://hdl.handle.net/1808/14837.

MLA Handbook (7th Edition):

Luo, Yong. “MOLECULAR MECHANISMS OF THE DNA DAMAGE RESPONSE INDUCED DURING PARVOVIRUS INFECTION.” 2012. Web. 20 Apr 2021.

Vancouver:

Luo Y. MOLECULAR MECHANISMS OF THE DNA DAMAGE RESPONSE INDUCED DURING PARVOVIRUS INFECTION. [Internet] [Doctoral dissertation]. University of Kansas; 2012. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1808/14837.

Council of Science Editors:

Luo Y. MOLECULAR MECHANISMS OF THE DNA DAMAGE RESPONSE INDUCED DURING PARVOVIRUS INFECTION. [Doctoral Dissertation]. University of Kansas; 2012. Available from: http://hdl.handle.net/1808/14837


Universiteit Utrecht

3. Hengeveld, R.C.C. Loading of cohesin and cohesion generation in response to DNA damage.

Degree: 2011, Universiteit Utrecht

 The sister chromatids are held together by the conserved cohesin complex from DNA replication in S phase until the metaphase to anaphase transition during mitosis.… (more)

Subjects/Keywords: DNA damage; cohesion; DNA repair; DNA damage response; Checkpoint recovery

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APA (6th Edition):

Hengeveld, R. C. C. (2011). Loading of cohesin and cohesion generation in response to DNA damage. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/218765

Chicago Manual of Style (16th Edition):

Hengeveld, R C C. “Loading of cohesin and cohesion generation in response to DNA damage.” 2011. Masters Thesis, Universiteit Utrecht. Accessed April 20, 2021. http://dspace.library.uu.nl:8080/handle/1874/218765.

MLA Handbook (7th Edition):

Hengeveld, R C C. “Loading of cohesin and cohesion generation in response to DNA damage.” 2011. Web. 20 Apr 2021.

Vancouver:

Hengeveld RCC. Loading of cohesin and cohesion generation in response to DNA damage. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2021 Apr 20]. Available from: http://dspace.library.uu.nl:8080/handle/1874/218765.

Council of Science Editors:

Hengeveld RCC. Loading of cohesin and cohesion generation in response to DNA damage. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/218765


Brandeis University

4. Brand, Kirsten. Characterization of the Repair Protein Complex: Putative Helicase YoaA and its Associated DNA Proteins.

Degree: 2019, Brandeis University

DNA replication is a conserved process that must be highly regulated. DNA replication involves a complex known as the replisome that interacts with proteins to… (more)

Subjects/Keywords: DNA damage; YoaA; HolC; DNA damage response complex

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APA (6th Edition):

Brand, K. (2019). Characterization of the Repair Protein Complex: Putative Helicase YoaA and its Associated DNA Proteins. (Thesis). Brandeis University. Retrieved from http://hdl.handle.net/10192/36554

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brand, Kirsten. “Characterization of the Repair Protein Complex: Putative Helicase YoaA and its Associated DNA Proteins.” 2019. Thesis, Brandeis University. Accessed April 20, 2021. http://hdl.handle.net/10192/36554.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brand, Kirsten. “Characterization of the Repair Protein Complex: Putative Helicase YoaA and its Associated DNA Proteins.” 2019. Web. 20 Apr 2021.

Vancouver:

Brand K. Characterization of the Repair Protein Complex: Putative Helicase YoaA and its Associated DNA Proteins. [Internet] [Thesis]. Brandeis University; 2019. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10192/36554.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brand K. Characterization of the Repair Protein Complex: Putative Helicase YoaA and its Associated DNA Proteins. [Thesis]. Brandeis University; 2019. Available from: http://hdl.handle.net/10192/36554

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Massoni, Shawn Christopher. Reca Dynamics & the SOS Response in Escherichia Coli: Cellular Limitation of Inducing Filaments.

Degree: PhD, Microbiology, 2013, U of Massachusetts : PhD

  During the course of normal DNA replication, replication forks are constantly encountering "housekeeping" types of routine damage to the DNA template that may cause… (more)

Subjects/Keywords: DNA damage; RadA; RecA; SOS response; Microbiology

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APA (6th Edition):

Massoni, S. C. (2013). Reca Dynamics & the SOS Response in Escherichia Coli: Cellular Limitation of Inducing Filaments. (Doctoral Dissertation). U of Massachusetts : PhD. Retrieved from https://scholarworks.umass.edu/open_access_dissertations/695

Chicago Manual of Style (16th Edition):

Massoni, Shawn Christopher. “Reca Dynamics & the SOS Response in Escherichia Coli: Cellular Limitation of Inducing Filaments.” 2013. Doctoral Dissertation, U of Massachusetts : PhD. Accessed April 20, 2021. https://scholarworks.umass.edu/open_access_dissertations/695.

MLA Handbook (7th Edition):

Massoni, Shawn Christopher. “Reca Dynamics & the SOS Response in Escherichia Coli: Cellular Limitation of Inducing Filaments.” 2013. Web. 20 Apr 2021.

Vancouver:

Massoni SC. Reca Dynamics & the SOS Response in Escherichia Coli: Cellular Limitation of Inducing Filaments. [Internet] [Doctoral dissertation]. U of Massachusetts : PhD; 2013. [cited 2021 Apr 20]. Available from: https://scholarworks.umass.edu/open_access_dissertations/695.

Council of Science Editors:

Massoni SC. Reca Dynamics & the SOS Response in Escherichia Coli: Cellular Limitation of Inducing Filaments. [Doctoral Dissertation]. U of Massachusetts : PhD; 2013. Available from: https://scholarworks.umass.edu/open_access_dissertations/695


Kyoto University / 京都大学

6. Ishii, Kei. The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells : Trp53-Trp53inp1-Tnfrsf10b経路がマウス精子幹細胞の放射線に対する応答を制御する.

Degree: 博士(医学), 2015, Kyoto University / 京都大学

Kei Ishii, Masamichi Ishiai, Hiroko Morimoto, Mito Kanatsu-Shinohara, Ohtsura Niwa, Minoru Takata, Takashi Shinohara, The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells, Stem Cell Reports, Volume 3, Issue 4, 14 October 2014, Pages 676-689, ISSN 2213-6711

新制・課程博士

甲第18685号

医博第3957号

Subjects/Keywords: Apoptosis; DNA damage response; Genotoxicity; Trp53; Radiation

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APA (6th Edition):

Ishii, K. (2015). The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells : Trp53-Trp53inp1-Tnfrsf10b経路がマウス精子幹細胞の放射線に対する応答を制御する. (Thesis). Kyoto University / 京都大学. Retrieved from http://hdl.handle.net/2433/195971 ; http://dx.doi.org/10.14989/doctor.k18685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ishii, Kei. “The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells : Trp53-Trp53inp1-Tnfrsf10b経路がマウス精子幹細胞の放射線に対する応答を制御する.” 2015. Thesis, Kyoto University / 京都大学. Accessed April 20, 2021. http://hdl.handle.net/2433/195971 ; http://dx.doi.org/10.14989/doctor.k18685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ishii, Kei. “The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells : Trp53-Trp53inp1-Tnfrsf10b経路がマウス精子幹細胞の放射線に対する応答を制御する.” 2015. Web. 20 Apr 2021.

Vancouver:

Ishii K. The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells : Trp53-Trp53inp1-Tnfrsf10b経路がマウス精子幹細胞の放射線に対する応答を制御する. [Internet] [Thesis]. Kyoto University / 京都大学; 2015. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/2433/195971 ; http://dx.doi.org/10.14989/doctor.k18685.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ishii K. The Trp53-Trp53inp1-Tnfrsf10b Pathway Regulates the Radiation Response of Mouse Spermatogonial Stem Cells : Trp53-Trp53inp1-Tnfrsf10b経路がマウス精子幹細胞の放射線に対する応答を制御する. [Thesis]. Kyoto University / 京都大学; 2015. Available from: http://hdl.handle.net/2433/195971 ; http://dx.doi.org/10.14989/doctor.k18685

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Leventakos, Konstantinos. Μελέτη των παραγόντων απόκρισης σε βλάβες του DNA στην ενδοεπιθηλιακή νεοπλασία του τραχήλου της μήτρας.

Degree: 2017, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 There is an ongoing quest for biomarkers improving the diagnosis of HPV-related cervical low- (LGSIL) and high-grade squamous intraepithelial lesions (HGSIL) and carcinomas, as well… (more)

Subjects/Keywords: Ενδοεπιθηλιακή νεοπλασία του τραχήλου; DNA damage response

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APA (6th Edition):

Leventakos, K. (2017). Μελέτη των παραγόντων απόκρισης σε βλάβες του DNA στην ενδοεπιθηλιακή νεοπλασία του τραχήλου της μήτρας. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/42347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Leventakos, Konstantinos. “Μελέτη των παραγόντων απόκρισης σε βλάβες του DNA στην ενδοεπιθηλιακή νεοπλασία του τραχήλου της μήτρας.” 2017. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 20, 2021. http://hdl.handle.net/10442/hedi/42347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Leventakos, Konstantinos. “Μελέτη των παραγόντων απόκρισης σε βλάβες του DNA στην ενδοεπιθηλιακή νεοπλασία του τραχήλου της μήτρας.” 2017. Web. 20 Apr 2021.

Vancouver:

Leventakos K. Μελέτη των παραγόντων απόκρισης σε βλάβες του DNA στην ενδοεπιθηλιακή νεοπλασία του τραχήλου της μήτρας. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10442/hedi/42347.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Leventakos K. Μελέτη των παραγόντων απόκρισης σε βλάβες του DNA στην ενδοεπιθηλιακή νεοπλασία του τραχήλου της μήτρας. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2017. Available from: http://hdl.handle.net/10442/hedi/42347

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

8. Robbins, Carol Bansbach. Identifying and defining the genome maintenance functions of SMARCAL1.

Degree: PhD, Biochemistry, 2012, Vanderbilt University

 In this dissertation I identify and define SWI/SNF, matrix-associated, actin-dependent regulator of chromatin, A-like 1 (SMARCAL1) as a genome maintenance protein. First, I introduce a… (more)

Subjects/Keywords: phosphorylation; DNA damage response; replication stress

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APA (6th Edition):

Robbins, C. B. (2012). Identifying and defining the genome maintenance functions of SMARCAL1. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12388

Chicago Manual of Style (16th Edition):

Robbins, Carol Bansbach. “Identifying and defining the genome maintenance functions of SMARCAL1.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed April 20, 2021. http://hdl.handle.net/1803/12388.

MLA Handbook (7th Edition):

Robbins, Carol Bansbach. “Identifying and defining the genome maintenance functions of SMARCAL1.” 2012. Web. 20 Apr 2021.

Vancouver:

Robbins CB. Identifying and defining the genome maintenance functions of SMARCAL1. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1803/12388.

Council of Science Editors:

Robbins CB. Identifying and defining the genome maintenance functions of SMARCAL1. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/12388


Texas Medical Center

9. Lee, Hong-Jen. TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE.

Degree: PhD, 2014, Texas Medical Center

  Ataxia telangiectasia-mutated (ATM) mediates DNA damage response by controlling irradiation (IR)-induced foci formation, cell cycle checkpoint, and apoptosis. However, how upstream signaling regulates ATM… (more)

Subjects/Keywords: EGFR; ATM; DNA Damage Response; Cancer Biology

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APA (6th Edition):

Lee, H. (2014). TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487

Chicago Manual of Style (16th Edition):

Lee, Hong-Jen. “TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE.” 2014. Doctoral Dissertation, Texas Medical Center. Accessed April 20, 2021. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487.

MLA Handbook (7th Edition):

Lee, Hong-Jen. “TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE.” 2014. Web. 20 Apr 2021.

Vancouver:

Lee H. TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2014. [cited 2021 Apr 20]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487.

Council of Science Editors:

Lee H. TYROSINE 370 PHOSPHORYLATION OF ATM POSITIVELY REGULATES DNA DAMAGE RESPONSE. [Doctoral Dissertation]. Texas Medical Center; 2014. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/487


University of Cambridge

10. Zhang, Yue. Understanding the activation mechanism of the Chk1 kinase.

Degree: PhD, 2019, University of Cambridge

 Faithful replication of DNA and correct segregation of duplicated chromosomes into two daughter cells are essential to ensure genome integrity and cell survival. Genome integrity… (more)

Subjects/Keywords: Cell-cycle checkpoint; DNA damage response; Chk1

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APA (6th Edition):

Zhang, Y. (2019). Understanding the activation mechanism of the Chk1 kinase. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776

Chicago Manual of Style (16th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 20, 2021. https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776.

MLA Handbook (7th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Web. 20 Apr 2021.

Vancouver:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Apr 20]. Available from: https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776.

Council of Science Editors:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776


University of Cambridge

11. Zhang, Yue. Understanding the activation mechanism of the Chk1 kinase.

Degree: PhD, 2019, University of Cambridge

 Faithful replication of DNA and correct segregation of duplicated chromosomes into two daughter cells are essential to ensure genome integrity and cell survival. Genome integrity… (more)

Subjects/Keywords: Cell-cycle checkpoint; DNA damage response; Chk1

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APA (6th Edition):

Zhang, Y. (2019). Understanding the activation mechanism of the Chk1 kinase. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/293290

Chicago Manual of Style (16th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Doctoral Dissertation, University of Cambridge. Accessed April 20, 2021. https://www.repository.cam.ac.uk/handle/1810/293290.

MLA Handbook (7th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Web. 20 Apr 2021.

Vancouver:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Apr 20]. Available from: https://www.repository.cam.ac.uk/handle/1810/293290.

Council of Science Editors:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/293290


University of Edinburgh

12. Din, Shahida. Constitutive activation of the ATM DNA damage response pathway in cancer represents a deregulated pathway.

Degree: PhD, 2014, University of Edinburgh

 Constitutive activation of the ATM dependent DNA damage response and repair pathways have been reported in pre-malignant and malignant human tissues and may undermine the… (more)

Subjects/Keywords: 616.99; DNA damage response; cancer; ATM inhibitors

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APA (6th Edition):

Din, S. (2014). Constitutive activation of the ATM DNA damage response pathway in cancer represents a deregulated pathway. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/18016

Chicago Manual of Style (16th Edition):

Din, Shahida. “Constitutive activation of the ATM DNA damage response pathway in cancer represents a deregulated pathway.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed April 20, 2021. http://hdl.handle.net/1842/18016.

MLA Handbook (7th Edition):

Din, Shahida. “Constitutive activation of the ATM DNA damage response pathway in cancer represents a deregulated pathway.” 2014. Web. 20 Apr 2021.

Vancouver:

Din S. Constitutive activation of the ATM DNA damage response pathway in cancer represents a deregulated pathway. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1842/18016.

Council of Science Editors:

Din S. Constitutive activation of the ATM DNA damage response pathway in cancer represents a deregulated pathway. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/18016


National University of Ireland – Galway

13. Walsh, Mary. Characterisation of novel Histone H3 RAD9 mutants defective in the DNA damage response in Saccharomyces cerevisiae .

Degree: 2013, National University of Ireland – Galway

 The DNA damage response (DDR) is a signal transduction cascade, which regulates cell cycle progression, gene transcription and DNA repair. Histones also play a vital… (more)

Subjects/Keywords: DNA damage response; Histones; RAD9; Biochemistry

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APA (6th Edition):

Walsh, M. (2013). Characterisation of novel Histone H3 RAD9 mutants defective in the DNA damage response in Saccharomyces cerevisiae . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/4254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walsh, Mary. “Characterisation of novel Histone H3 RAD9 mutants defective in the DNA damage response in Saccharomyces cerevisiae .” 2013. Thesis, National University of Ireland – Galway. Accessed April 20, 2021. http://hdl.handle.net/10379/4254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walsh, Mary. “Characterisation of novel Histone H3 RAD9 mutants defective in the DNA damage response in Saccharomyces cerevisiae .” 2013. Web. 20 Apr 2021.

Vancouver:

Walsh M. Characterisation of novel Histone H3 RAD9 mutants defective in the DNA damage response in Saccharomyces cerevisiae . [Internet] [Thesis]. National University of Ireland – Galway; 2013. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10379/4254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walsh M. Characterisation of novel Histone H3 RAD9 mutants defective in the DNA damage response in Saccharomyces cerevisiae . [Thesis]. National University of Ireland – Galway; 2013. Available from: http://hdl.handle.net/10379/4254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

14. Ho, Ting-Hin. Identification of Epstein-Barr virus proteins that inhibit the DNA damage response.

Degree: PhD, 2018, University of Toronto

 Epstein-Barr virus (EBV) is a common herpesvirus that is the causative agent of mononucleosis and can also induce a variety of B cell lymphomas and… (more)

Subjects/Keywords: DNA damage response; Epstein-Barr virus; 0307

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APA (6th Edition):

Ho, T. (2018). Identification of Epstein-Barr virus proteins that inhibit the DNA damage response. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/92161

Chicago Manual of Style (16th Edition):

Ho, Ting-Hin. “Identification of Epstein-Barr virus proteins that inhibit the DNA damage response.” 2018. Doctoral Dissertation, University of Toronto. Accessed April 20, 2021. http://hdl.handle.net/1807/92161.

MLA Handbook (7th Edition):

Ho, Ting-Hin. “Identification of Epstein-Barr virus proteins that inhibit the DNA damage response.” 2018. Web. 20 Apr 2021.

Vancouver:

Ho T. Identification of Epstein-Barr virus proteins that inhibit the DNA damage response. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1807/92161.

Council of Science Editors:

Ho T. Identification of Epstein-Barr virus proteins that inhibit the DNA damage response. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/92161


University of Toronto

15. Ramachandran, Shaliny. Characterizing the Role of the DNA Damage Response in Class switch recombination.

Degree: 2014, University of Toronto

B cells undergo secondary antibody diversification to elicit an effective immune response. Secondary antibody diversification generates antibodies with high affinity for the cognate antigen, and… (more)

Subjects/Keywords: Class switch recombination; DNA damage response; 0982

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APA (6th Edition):

Ramachandran, S. (2014). Characterizing the Role of the DNA Damage Response in Class switch recombination. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/69062

Chicago Manual of Style (16th Edition):

Ramachandran, Shaliny. “Characterizing the Role of the DNA Damage Response in Class switch recombination.” 2014. Doctoral Dissertation, University of Toronto. Accessed April 20, 2021. http://hdl.handle.net/1807/69062.

MLA Handbook (7th Edition):

Ramachandran, Shaliny. “Characterizing the Role of the DNA Damage Response in Class switch recombination.” 2014. Web. 20 Apr 2021.

Vancouver:

Ramachandran S. Characterizing the Role of the DNA Damage Response in Class switch recombination. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1807/69062.

Council of Science Editors:

Ramachandran S. Characterizing the Role of the DNA Damage Response in Class switch recombination. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/69062


Colorado State University

16. Nelson, Christopher Boulanger. Telomeric double strand breaks undergo resection - but not repair - in G1 human cells.

Degree: PhD, Cell and Molecular Biology, 2017, Colorado State University

 Telomeres are specialized G-rich repetitive regions at the ends of eukaryotic chromosomes (TTAGGGn in mammalian cells). Telomeres function to prevent double strand break (DSB) repair… (more)

Subjects/Keywords: DNA damage response; double strand break; telomeres; genomic stability; DNA repair; DNA damage

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APA (6th Edition):

Nelson, C. B. (2017). Telomeric double strand breaks undergo resection - but not repair - in G1 human cells. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/184022

Chicago Manual of Style (16th Edition):

Nelson, Christopher Boulanger. “Telomeric double strand breaks undergo resection - but not repair - in G1 human cells.” 2017. Doctoral Dissertation, Colorado State University. Accessed April 20, 2021. http://hdl.handle.net/10217/184022.

MLA Handbook (7th Edition):

Nelson, Christopher Boulanger. “Telomeric double strand breaks undergo resection - but not repair - in G1 human cells.” 2017. Web. 20 Apr 2021.

Vancouver:

Nelson CB. Telomeric double strand breaks undergo resection - but not repair - in G1 human cells. [Internet] [Doctoral dissertation]. Colorado State University; 2017. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10217/184022.

Council of Science Editors:

Nelson CB. Telomeric double strand breaks undergo resection - but not repair - in G1 human cells. [Doctoral Dissertation]. Colorado State University; 2017. Available from: http://hdl.handle.net/10217/184022


University of Oxford

17. Olcina del Molino, Mónica. Hypoxia-induced chromatin changes and ATM signalling.

Degree: PhD, 2014, University of Oxford

 The DNA damage response (DDR) is a complex signalling cascade triggered in response to stress, in an attempt to maintain genomic integrity. Components of this… (more)

Subjects/Keywords: 572.8; DNA damage signalling; Oncology; Biology (medical sciences); hypoxia; chromatin; DNA replication; DNA damage response

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APA (6th Edition):

Olcina del Molino, M. (2014). Hypoxia-induced chromatin changes and ATM signalling. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:dbef3c19-b7f6-42a4-a321-97d00c572ae3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604527

Chicago Manual of Style (16th Edition):

Olcina del Molino, Mónica. “Hypoxia-induced chromatin changes and ATM signalling.” 2014. Doctoral Dissertation, University of Oxford. Accessed April 20, 2021. http://ora.ox.ac.uk/objects/uuid:dbef3c19-b7f6-42a4-a321-97d00c572ae3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604527.

MLA Handbook (7th Edition):

Olcina del Molino, Mónica. “Hypoxia-induced chromatin changes and ATM signalling.” 2014. Web. 20 Apr 2021.

Vancouver:

Olcina del Molino M. Hypoxia-induced chromatin changes and ATM signalling. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2021 Apr 20]. Available from: http://ora.ox.ac.uk/objects/uuid:dbef3c19-b7f6-42a4-a321-97d00c572ae3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604527.

Council of Science Editors:

Olcina del Molino M. Hypoxia-induced chromatin changes and ATM signalling. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:dbef3c19-b7f6-42a4-a321-97d00c572ae3 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604527


University of Texas – Austin

18. Gong, Fade. Role of bromodomain containing proteins in the DNA damage response.

Degree: PhD, Cell and Molecular Biology, 2016, University of Texas – Austin

 Chromatin-based DNA damage response (DDR) mechanisms are fundamental for preventing genome and epigenome instability, which are hallmarks of cancer. How chromatin promotes genome-epigenome integrity in… (more)

Subjects/Keywords: DNA damage response; DNA repair; Chromatin; Acetylation; Bromodomain

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APA (6th Edition):

Gong, F. (2016). Role of bromodomain containing proteins in the DNA damage response. (Doctoral Dissertation). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/68576

Chicago Manual of Style (16th Edition):

Gong, Fade. “Role of bromodomain containing proteins in the DNA damage response.” 2016. Doctoral Dissertation, University of Texas – Austin. Accessed April 20, 2021. http://hdl.handle.net/2152/68576.

MLA Handbook (7th Edition):

Gong, Fade. “Role of bromodomain containing proteins in the DNA damage response.” 2016. Web. 20 Apr 2021.

Vancouver:

Gong F. Role of bromodomain containing proteins in the DNA damage response. [Internet] [Doctoral dissertation]. University of Texas – Austin; 2016. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/2152/68576.

Council of Science Editors:

Gong F. Role of bromodomain containing proteins in the DNA damage response. [Doctoral Dissertation]. University of Texas – Austin; 2016. Available from: http://hdl.handle.net/2152/68576

19. Παναγόπουλος, Ανδρέας. Διερεύνηση της κινητικής συσσώρευσης της πρωτεΐνης Cdt2 μετά από εντοπισμένη βλάβη στο DNA.

Degree: 2014, University of Patras

 Η αδειοδότηση της αντιγραφής αποτελεί μία ιδιαίτερα σημαντική διαδικασία, η οποία λαμβάνει χώρα στις θέσεις έναρξης της αντιγραφής με το σχηματισμό του προ-αντιγραφικού συμπλόκου, που… (more)

Subjects/Keywords: Βλάβη στο DNA; 571.84; DNA damage response; Cdt2

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APA (6th Edition):

Παναγόπουλος, . (2014). Διερεύνηση της κινητικής συσσώρευσης της πρωτεΐνης Cdt2 μετά από εντοπισμένη βλάβη στο DNA. (Masters Thesis). University of Patras. Retrieved from http://hdl.handle.net/10889/8497

Chicago Manual of Style (16th Edition):

Παναγόπουλος, Ανδρέας. “Διερεύνηση της κινητικής συσσώρευσης της πρωτεΐνης Cdt2 μετά από εντοπισμένη βλάβη στο DNA.” 2014. Masters Thesis, University of Patras. Accessed April 20, 2021. http://hdl.handle.net/10889/8497.

MLA Handbook (7th Edition):

Παναγόπουλος, Ανδρέας. “Διερεύνηση της κινητικής συσσώρευσης της πρωτεΐνης Cdt2 μετά από εντοπισμένη βλάβη στο DNA.” 2014. Web. 20 Apr 2021.

Vancouver:

Παναγόπουλος . Διερεύνηση της κινητικής συσσώρευσης της πρωτεΐνης Cdt2 μετά από εντοπισμένη βλάβη στο DNA. [Internet] [Masters thesis]. University of Patras; 2014. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10889/8497.

Council of Science Editors:

Παναγόπουλος . Διερεύνηση της κινητικής συσσώρευσης της πρωτεΐνης Cdt2 μετά από εντοπισμένη βλάβη στο DNA. [Masters Thesis]. University of Patras; 2014. Available from: http://hdl.handle.net/10889/8497


Vanderbilt University

20. Puccetti, Matthew Vincent. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.

Degree: PhD, Pathology, 2018, Vanderbilt University

 Genome maintenance is essential for the viability of eukaryotic cells and for the prevention of tumorigenesis. DNA replication stress is a significant contributor to genomic… (more)

Subjects/Keywords: DNA translocases; DNA damage response; genomic instability; replication fork remodeling; tumorigenesis

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APA (6th Edition):

Puccetti, M. V. (2018). The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15446

Chicago Manual of Style (16th Edition):

Puccetti, Matthew Vincent. “The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed April 20, 2021. http://hdl.handle.net/1803/15446.

MLA Handbook (7th Edition):

Puccetti, Matthew Vincent. “The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress.” 2018. Web. 20 Apr 2021.

Vancouver:

Puccetti MV. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1803/15446.

Council of Science Editors:

Puccetti MV. The role of DNA replication fork remodeling proteins in lymphomagenesis and hematopoietic cell replication stress. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/15446

21. Giunta, Simona. DNA damage responses in the context of the cell division cycle.

Degree: PhD, 2010, University of Cambridge

 During my PhD, I have investigated aspects of the DNA damage response (DDR) in the context of three different cellular scenarios: DNA damage signalling in… (more)

Subjects/Keywords: DNA damage response; Cell cycle; Mitosis; DNA double strand breaks

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APA (6th Edition):

Giunta, S. (2010). DNA damage responses in the context of the cell division cycle. (Doctoral Dissertation). University of Cambridge. Retrieved from http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg

Chicago Manual of Style (16th Edition):

Giunta, Simona. “DNA damage responses in the context of the cell division cycle.” 2010. Doctoral Dissertation, University of Cambridge. Accessed April 20, 2021. http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg.

MLA Handbook (7th Edition):

Giunta, Simona. “DNA damage responses in the context of the cell division cycle.” 2010. Web. 20 Apr 2021.

Vancouver:

Giunta S. DNA damage responses in the context of the cell division cycle. [Internet] [Doctoral dissertation]. University of Cambridge; 2010. [cited 2021 Apr 20]. Available from: http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg.

Council of Science Editors:

Giunta S. DNA damage responses in the context of the cell division cycle. [Doctoral Dissertation]. University of Cambridge; 2010. Available from: http://www.dspace.cam.ac.uk/handle/1810/228687https://www.repository.cam.ac.uk/bitstream/1810/228687/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/5/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/228687/6/PhD%20Thesis%20Simona%20Giunta_EDeposit%26PanelsRED.pdf.jpg


University of Oxford

22. Carlos, A. R. DNA damage responses to loss of telomere integrity.

Degree: PhD, 2013, University of Oxford

 Linear genomes end in characteristic structures consisting of repetitive DNA and proteins: the telomeres. These play two critical roles: on one hand they avoid the… (more)

Subjects/Keywords: 572.8; Biology (medical sciences); DNA damage signalling; Telomere integrity; DNA damage response; BRCA1; CtIP; EXO1

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APA (6th Edition):

Carlos, A. R. (2013). DNA damage responses to loss of telomere integrity. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:27bcf3b6-edb9-47e2-af7c-c7ba9b431572 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588451

Chicago Manual of Style (16th Edition):

Carlos, A R. “DNA damage responses to loss of telomere integrity.” 2013. Doctoral Dissertation, University of Oxford. Accessed April 20, 2021. http://ora.ox.ac.uk/objects/uuid:27bcf3b6-edb9-47e2-af7c-c7ba9b431572 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588451.

MLA Handbook (7th Edition):

Carlos, A R. “DNA damage responses to loss of telomere integrity.” 2013. Web. 20 Apr 2021.

Vancouver:

Carlos AR. DNA damage responses to loss of telomere integrity. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2021 Apr 20]. Available from: http://ora.ox.ac.uk/objects/uuid:27bcf3b6-edb9-47e2-af7c-c7ba9b431572 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588451.

Council of Science Editors:

Carlos AR. DNA damage responses to loss of telomere integrity. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:27bcf3b6-edb9-47e2-af7c-c7ba9b431572 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.588451

23. Champeris-Tsaniras, Spyridon. Διερεύνηση των παραγόντων αδειοδότησης της αντιγραφής CDT1 και Geminin στα καρκινικά βλαστικά κύτταρα και την καρκινογένεση.

Degree: 2017, University of Patras; Πανεπιστήμιο Πατρών

Geminin, a DNA replication licensing inhibitor, together with its interacting partner Cdt1, ensure faithful DNA replication in vertebrates. Several studies have shown that Cdt1 overexpression… (more)

Subjects/Keywords: Χημική καρκινογένεση; αντιγραφή DNA; Geminin; DNA replication; DNA damage response; Carcinogenesis model

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APA (6th Edition):

Champeris-Tsaniras, S. (2017). Διερεύνηση των παραγόντων αδειοδότησης της αντιγραφής CDT1 και Geminin στα καρκινικά βλαστικά κύτταρα και την καρκινογένεση. (Thesis). University of Patras; Πανεπιστήμιο Πατρών. Retrieved from http://hdl.handle.net/10442/hedi/42667

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Champeris-Tsaniras, Spyridon. “Διερεύνηση των παραγόντων αδειοδότησης της αντιγραφής CDT1 και Geminin στα καρκινικά βλαστικά κύτταρα και την καρκινογένεση.” 2017. Thesis, University of Patras; Πανεπιστήμιο Πατρών. Accessed April 20, 2021. http://hdl.handle.net/10442/hedi/42667.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Champeris-Tsaniras, Spyridon. “Διερεύνηση των παραγόντων αδειοδότησης της αντιγραφής CDT1 και Geminin στα καρκινικά βλαστικά κύτταρα και την καρκινογένεση.” 2017. Web. 20 Apr 2021.

Vancouver:

Champeris-Tsaniras S. Διερεύνηση των παραγόντων αδειοδότησης της αντιγραφής CDT1 και Geminin στα καρκινικά βλαστικά κύτταρα και την καρκινογένεση. [Internet] [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2017. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10442/hedi/42667.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Champeris-Tsaniras S. Διερεύνηση των παραγόντων αδειοδότησης της αντιγραφής CDT1 και Geminin στα καρκινικά βλαστικά κύτταρα και την καρκινογένεση. [Thesis]. University of Patras; Πανεπιστήμιο Πατρών; 2017. Available from: http://hdl.handle.net/10442/hedi/42667

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Takabayashi, Hiroaki. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.

Degree: 博士(医学), 2013, Niigata University / 新潟大学

学位の種類: 博士(医学). 報告番号: 甲第3807号. 学位記番号: 新大院博(医)甲第559号. 学位授与年月日: 平成25年9月20日

Human Pathology. 2013, 44(6), 1038–1046

p53-binding protein 1

Subjects/Keywords: Colorectal carcinoma; Double-strand breaks; DNA damage response; γH2AX

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APA (6th Edition):

Takabayashi, H. (2013). Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. (Thesis). Niigata University / 新潟大学. Retrieved from http://hdl.handle.net/10191/24550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Takabayashi, Hiroaki. “Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.” 2013. Thesis, Niigata University / 新潟大学. Accessed April 20, 2021. http://hdl.handle.net/10191/24550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Takabayashi, Hiroaki. “Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化.” 2013. Web. 20 Apr 2021.

Vancouver:

Takabayashi H. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. [Internet] [Thesis]. Niigata University / 新潟大学; 2013. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10191/24550.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Takabayashi H. Alteration of the DNA damage response in colorectal tumor progression : 大腸腫瘍の発育過程におけるDNA損傷応答の変化. [Thesis]. Niigata University / 新潟大学; 2013. Available from: http://hdl.handle.net/10191/24550

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.

Degree: 博士(医学), 2014, Nara Medical University / 奈良県立医科大学

Objective Appropriate cell cycle checkpoints are essential for the maintenance of normal cells and chemosensitivity of cancer cells. Clear cell adenocarcinoma (CCA) of the ovary… (more)

Subjects/Keywords: Transcription factors; DNA damage response; Cell cycle; Checkpoint control; Chemoresistance

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APA (6th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, N. (2014). Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. “Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.” 2014. Thesis, Nara Medical University / 奈良県立医科大学. Accessed April 20, 2021. http://hdl.handle.net/10564/2749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. “Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.” 2014. Web. 20 Apr 2021.

Vancouver:

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi N. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/10564/2749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi N. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. Available from: http://hdl.handle.net/10564/2749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Alberta

26. Minard, Laura. Chromatin regulation by histone chaperone Asf1.

Degree: PhD, Department of Biochemistry, 2010, University of Alberta

 Asf1 is a conserved H3/H4 histone chaperone with multiple functions in chromatin modulation. Using budding yeast as a model, we identify new pathways of Asf1… (more)

Subjects/Keywords: Asf1; SWI/SNF; histone chaperone; hydroxyurea; chromatin; DNA damage response

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Minard, L. (2010). Chromatin regulation by histone chaperone Asf1. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/44558d82z

Chicago Manual of Style (16th Edition):

Minard, Laura. “Chromatin regulation by histone chaperone Asf1.” 2010. Doctoral Dissertation, University of Alberta. Accessed April 20, 2021. https://era.library.ualberta.ca/files/44558d82z.

MLA Handbook (7th Edition):

Minard, Laura. “Chromatin regulation by histone chaperone Asf1.” 2010. Web. 20 Apr 2021.

Vancouver:

Minard L. Chromatin regulation by histone chaperone Asf1. [Internet] [Doctoral dissertation]. University of Alberta; 2010. [cited 2021 Apr 20]. Available from: https://era.library.ualberta.ca/files/44558d82z.

Council of Science Editors:

Minard L. Chromatin regulation by histone chaperone Asf1. [Doctoral Dissertation]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/44558d82z


University of Michigan

27. Festerling, Todd Allen. The Mre11-CDK2 Interaction in the DNA Damage Response.

Degree: PhD, Toxicology, 2014, University of Michigan

 MRN (Mre11, Rad50, and NBS1) complex consists of highly conserved proteins integral to DNA double-strand break (DSB) signaling and repair. The unique MRN complex architecture… (more)

Subjects/Keywords: DNA Damage Response; Cell Cycle CDK2; Public Health; Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Festerling, T. A. (2014). The Mre11-CDK2 Interaction in the DNA Damage Response. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/108856

Chicago Manual of Style (16th Edition):

Festerling, Todd Allen. “The Mre11-CDK2 Interaction in the DNA Damage Response.” 2014. Doctoral Dissertation, University of Michigan. Accessed April 20, 2021. http://hdl.handle.net/2027.42/108856.

MLA Handbook (7th Edition):

Festerling, Todd Allen. “The Mre11-CDK2 Interaction in the DNA Damage Response.” 2014. Web. 20 Apr 2021.

Vancouver:

Festerling TA. The Mre11-CDK2 Interaction in the DNA Damage Response. [Internet] [Doctoral dissertation]. University of Michigan; 2014. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/2027.42/108856.

Council of Science Editors:

Festerling TA. The Mre11-CDK2 Interaction in the DNA Damage Response. [Doctoral Dissertation]. University of Michigan; 2014. Available from: http://hdl.handle.net/2027.42/108856


Cornell University

28. Lim, Pei Xin. Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity.

Degree: PhD, Genetics, 2015, Cornell University

 The mammalian RAD9A-HUS1-RAD1 (9-1-1) complex is a heterotrimeric clamp that promotes checkpoint signaling and DNA repair by functioning as a scaffold at DNA damage sites.… (more)

Subjects/Keywords: RAD9-HUS1-RAD1; DNA damage response; checkpoint-repair coordination

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lim, P. X. (2015). Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/40589

Chicago Manual of Style (16th Edition):

Lim, Pei Xin. “Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity.” 2015. Doctoral Dissertation, Cornell University. Accessed April 20, 2021. http://hdl.handle.net/1813/40589.

MLA Handbook (7th Edition):

Lim, Pei Xin. “Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity.” 2015. Web. 20 Apr 2021.

Vancouver:

Lim PX. Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity. [Internet] [Doctoral dissertation]. Cornell University; 2015. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1813/40589.

Council of Science Editors:

Lim PX. Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity. [Doctoral Dissertation]. Cornell University; 2015. Available from: http://hdl.handle.net/1813/40589


Vanderbilt University

29. Luzwick, Jessica Whitney. Regulation of the ATR Pathway in the Replication Stress Response.

Degree: PhD, Biochemistry, 2016, Vanderbilt University

 Every cell divisions cycle, over 6.8 billion base pairs of DNA must be accurately replicated. To further complicate this process, the DNA is damaged at… (more)

Subjects/Keywords: replication; ATR inhibitor; DNA damage response; replication stress; ATR; ATRIP

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Luzwick, J. W. (2016). Regulation of the ATR Pathway in the Replication Stress Response. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13041

Chicago Manual of Style (16th Edition):

Luzwick, Jessica Whitney. “Regulation of the ATR Pathway in the Replication Stress Response.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed April 20, 2021. http://hdl.handle.net/1803/13041.

MLA Handbook (7th Edition):

Luzwick, Jessica Whitney. “Regulation of the ATR Pathway in the Replication Stress Response.” 2016. Web. 20 Apr 2021.

Vancouver:

Luzwick JW. Regulation of the ATR Pathway in the Replication Stress Response. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1803/13041.

Council of Science Editors:

Luzwick JW. Regulation of the ATR Pathway in the Replication Stress Response. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://hdl.handle.net/1803/13041


Vanderbilt University

30. Poole, Lisa Ann. SMARCAL1 Maintains Telomere Integrity During DNA Replication.

Degree: PhD, Biochemistry, 2017, Vanderbilt University

DNA replication is constantly challenged by several sources of replication stress that can affect the accuracy and timely completion of this essential biological process. The… (more)

Subjects/Keywords: DNA damage response; HLTF; telomeres; fork remodeling; SMARCAL1; ZRANB3

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Poole, L. A. (2017). SMARCAL1 Maintains Telomere Integrity During DNA Replication. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14466

Chicago Manual of Style (16th Edition):

Poole, Lisa Ann. “SMARCAL1 Maintains Telomere Integrity During DNA Replication.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed April 20, 2021. http://hdl.handle.net/1803/14466.

MLA Handbook (7th Edition):

Poole, Lisa Ann. “SMARCAL1 Maintains Telomere Integrity During DNA Replication.” 2017. Web. 20 Apr 2021.

Vancouver:

Poole LA. SMARCAL1 Maintains Telomere Integrity During DNA Replication. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Apr 20]. Available from: http://hdl.handle.net/1803/14466.

Council of Science Editors:

Poole LA. SMARCAL1 Maintains Telomere Integrity During DNA Replication. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/14466

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