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You searched for subject:(DNA damage repair). Showing records 1 – 30 of 305 total matches.

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University of Oxford

1. Baddock, Hannah. Understanding the role of the SNM1B and EXD2 in DNA damage repair.

Degree: PhD, 2017, University of Oxford

 Unrepaired, or misrepaired, DNA damage can be carcinogenic or mutagenic; thus functional DNA damage repair pathways are essential for the safeguarding of the genome. SNM1B… (more)

Subjects/Keywords: DNA damage repair

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APA (6th Edition):

Baddock, H. (2017). Understanding the role of the SNM1B and EXD2 in DNA damage repair. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895

Chicago Manual of Style (16th Edition):

Baddock, Hannah. “Understanding the role of the SNM1B and EXD2 in DNA damage repair.” 2017. Doctoral Dissertation, University of Oxford. Accessed January 22, 2020. http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895.

MLA Handbook (7th Edition):

Baddock, Hannah. “Understanding the role of the SNM1B and EXD2 in DNA damage repair.” 2017. Web. 22 Jan 2020.

Vancouver:

Baddock H. Understanding the role of the SNM1B and EXD2 in DNA damage repair. [Internet] [Doctoral dissertation]. University of Oxford; 2017. [cited 2020 Jan 22]. Available from: http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895.

Council of Science Editors:

Baddock H. Understanding the role of the SNM1B and EXD2 in DNA damage repair. [Doctoral Dissertation]. University of Oxford; 2017. Available from: http://ora.ox.ac.uk/objects/uuid:e120ab1f-d001-4b9b-9a03-c0852f4dfd21 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.748895


Rutgers University

2. Misenko, Sarah Marie, 1990-. 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism.

Degree: PhD, Biochemistry, 2017, Rutgers University

DNA end resection is believed to be a step that influences the choice between the two major DNA double-strand break (DSB) repair pathways: homologous recombination… (more)

Subjects/Keywords: DNA damage; DNA repair

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APA (6th Edition):

Misenko, Sarah Marie, 1. (2017). 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/55597/

Chicago Manual of Style (16th Edition):

Misenko, Sarah Marie, 1990-. “53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism.” 2017. Doctoral Dissertation, Rutgers University. Accessed January 22, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/55597/.

MLA Handbook (7th Edition):

Misenko, Sarah Marie, 1990-. “53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism.” 2017. Web. 22 Jan 2020.

Vancouver:

Misenko, Sarah Marie 1. 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2020 Jan 22]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55597/.

Council of Science Editors:

Misenko, Sarah Marie 1. 53BP1 regulates dna double-strand break repair in a mouse “BRCA-like” model by a non-resection mechanism. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55597/


University of Oxford

3. Kasparek, Torben Rudolf. Identification and characterisation of determinants of genome stability in response to a double-strand break.

Degree: PhD, 2013, University of Oxford

 Chromosomal rearrangements can lead to loss of heterozygosity (LOH) and oncogene activation, both of which represent possible causative events in cancer development. Such outcomes can… (more)

Subjects/Keywords: 616.994; Oncology; DNA damage repair

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APA (6th Edition):

Kasparek, T. R. (2013). Identification and characterisation of determinants of genome stability in response to a double-strand break. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454

Chicago Manual of Style (16th Edition):

Kasparek, Torben Rudolf. “Identification and characterisation of determinants of genome stability in response to a double-strand break.” 2013. Doctoral Dissertation, University of Oxford. Accessed January 22, 2020. http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454.

MLA Handbook (7th Edition):

Kasparek, Torben Rudolf. “Identification and characterisation of determinants of genome stability in response to a double-strand break.” 2013. Web. 22 Jan 2020.

Vancouver:

Kasparek TR. Identification and characterisation of determinants of genome stability in response to a double-strand break. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2020 Jan 22]. Available from: http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454.

Council of Science Editors:

Kasparek TR. Identification and characterisation of determinants of genome stability in response to a double-strand break. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:78e0a145-22c8-4abd-a746-e18c1939f5c9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604454

4. Jatsenko, Tatjana. Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads .

Degree: 2018, Tartu University

 Kahjustused DNA-s, mis tekivad kas rakkude normaalse elutegevuse käigus või erinevate keskonnategurite mõjul (näiteks UV-kiirgus, DNA-d kahjustavad kemikaalid), pärsivad genoomi replikatsiooni, takistades replikatiivse DNA polümeraasi… (more)

Subjects/Keywords: mutations; DNA damage; DNA polymerase; DNA repair

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APA (6th Edition):

Jatsenko, T. (2018). Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads . (Thesis). Tartu University. Retrieved from http://hdl.handle.net/10062/61278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jatsenko, Tatjana. “Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads .” 2018. Thesis, Tartu University. Accessed January 22, 2020. http://hdl.handle.net/10062/61278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jatsenko, Tatjana. “Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads .” 2018. Web. 22 Jan 2020.

Vancouver:

Jatsenko T. Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads . [Internet] [Thesis]. Tartu University; 2018. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10062/61278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jatsenko T. Role of translesion DNA polymerases in mutagenesis and DNA damage tolerance in Pseudomonads . [Thesis]. Tartu University; 2018. Available from: http://hdl.handle.net/10062/61278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

5. Li, Mingyang. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.

Degree: PhD, Laboratory and Basic Science Research, 2017, Louisiana State University

  In eukaryotes, DNA repair mechanisms detect and repair damaged DNA. DNA damage is primarily caused by a variety of exogenous and endogenous sources. Several… (more)

Subjects/Keywords: DNA repair; DNA damage; Base excision repair; Double-strand break repair

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APA (6th Edition):

Li, M. (2017). DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. (Doctoral Dissertation). Louisiana State University. Retrieved from https://digitalcommons.lsu.edu/gradschool_dissertations/4186

Chicago Manual of Style (16th Edition):

Li, Mingyang. “DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.” 2017. Doctoral Dissertation, Louisiana State University. Accessed January 22, 2020. https://digitalcommons.lsu.edu/gradschool_dissertations/4186.

MLA Handbook (7th Edition):

Li, Mingyang. “DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.” 2017. Web. 22 Jan 2020.

Vancouver:

Li M. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. [Internet] [Doctoral dissertation]. Louisiana State University; 2017. [cited 2020 Jan 22]. Available from: https://digitalcommons.lsu.edu/gradschool_dissertations/4186.

Council of Science Editors:

Li M. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. [Doctoral Dissertation]. Louisiana State University; 2017. Available from: https://digitalcommons.lsu.edu/gradschool_dissertations/4186


Hong Kong University of Science and Technology

6. Wong, Tin Yan CHEM. Formation of apurinic/apyrimidinic site in nucleic acids and its DNA-protein cross-links.

Degree: 2017, Hong Kong University of Science and Technology

 Apurinic/apyrimidinic sites, also called the abasic sites, are the most commonly found lesions in DNA and they are reported to be mutagenic and carcinogenic. Abasic… (more)

Subjects/Keywords: DNA repair ; DNA damage ; Identification ; Proteins ; Crosslinking

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APA (6th Edition):

Wong, T. Y. C. (2017). Formation of apurinic/apyrimidinic site in nucleic acids and its DNA-protein cross-links. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-91195 ; https://doi.org/10.14711/thesis-991012564468403412 ; http://repository.ust.hk/ir/bitstream/1783.1-91195/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wong, Tin Yan CHEM. “Formation of apurinic/apyrimidinic site in nucleic acids and its DNA-protein cross-links.” 2017. Thesis, Hong Kong University of Science and Technology. Accessed January 22, 2020. http://repository.ust.hk/ir/Record/1783.1-91195 ; https://doi.org/10.14711/thesis-991012564468403412 ; http://repository.ust.hk/ir/bitstream/1783.1-91195/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wong, Tin Yan CHEM. “Formation of apurinic/apyrimidinic site in nucleic acids and its DNA-protein cross-links.” 2017. Web. 22 Jan 2020.

Vancouver:

Wong TYC. Formation of apurinic/apyrimidinic site in nucleic acids and its DNA-protein cross-links. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2017. [cited 2020 Jan 22]. Available from: http://repository.ust.hk/ir/Record/1783.1-91195 ; https://doi.org/10.14711/thesis-991012564468403412 ; http://repository.ust.hk/ir/bitstream/1783.1-91195/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wong TYC. Formation of apurinic/apyrimidinic site in nucleic acids and its DNA-protein cross-links. [Thesis]. Hong Kong University of Science and Technology; 2017. Available from: http://repository.ust.hk/ir/Record/1783.1-91195 ; https://doi.org/10.14711/thesis-991012564468403412 ; http://repository.ust.hk/ir/bitstream/1783.1-91195/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

7. Hengeveld, R.C.C. Loading of cohesin and cohesion generation in response to DNA damage.

Degree: 2011, Universiteit Utrecht

 The sister chromatids are held together by the conserved cohesin complex from DNA replication in S phase until the metaphase to anaphase transition during mitosis.… (more)

Subjects/Keywords: DNA damage; cohesion; DNA repair; DNA damage response; Checkpoint recovery

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APA (6th Edition):

Hengeveld, R. C. C. (2011). Loading of cohesin and cohesion generation in response to DNA damage. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/218765

Chicago Manual of Style (16th Edition):

Hengeveld, R C C. “Loading of cohesin and cohesion generation in response to DNA damage.” 2011. Masters Thesis, Universiteit Utrecht. Accessed January 22, 2020. http://dspace.library.uu.nl:8080/handle/1874/218765.

MLA Handbook (7th Edition):

Hengeveld, R C C. “Loading of cohesin and cohesion generation in response to DNA damage.” 2011. Web. 22 Jan 2020.

Vancouver:

Hengeveld RCC. Loading of cohesin and cohesion generation in response to DNA damage. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2020 Jan 22]. Available from: http://dspace.library.uu.nl:8080/handle/1874/218765.

Council of Science Editors:

Hengeveld RCC. Loading of cohesin and cohesion generation in response to DNA damage. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/218765


Tartu University

8. Sidorenko, Julia. Combating DNA damage and maintenance of genome integrity in pseudomonads .

Degree: 2015, Tartu University

 Efektiivne DNA replikatsioon ja DNA kahjustuste eemaldamine on erakordse tähtsusega elusrakkude genoomi terviklikkuse säilitamisel. Kõrgelt koordineeritud DNA reparatsiooni mehhanismid võimaldavad rakkudel ellu jääda ka ulatuslike… (more)

Subjects/Keywords: DNA kahjustused; DNA reparatsioon; Pseudomonas; DNA damage; DNA repair

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APA (6th Edition):

Sidorenko, J. (2015). Combating DNA damage and maintenance of genome integrity in pseudomonads . (Thesis). Tartu University. Retrieved from http://hdl.handle.net/10062/48510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sidorenko, Julia. “Combating DNA damage and maintenance of genome integrity in pseudomonads .” 2015. Thesis, Tartu University. Accessed January 22, 2020. http://hdl.handle.net/10062/48510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sidorenko, Julia. “Combating DNA damage and maintenance of genome integrity in pseudomonads .” 2015. Web. 22 Jan 2020.

Vancouver:

Sidorenko J. Combating DNA damage and maintenance of genome integrity in pseudomonads . [Internet] [Thesis]. Tartu University; 2015. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10062/48510.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sidorenko J. Combating DNA damage and maintenance of genome integrity in pseudomonads . [Thesis]. Tartu University; 2015. Available from: http://hdl.handle.net/10062/48510

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toledo Health Science Campus

9. Edelbrock, Michael Aaron. Cell Cycle Regulation of DNA Mismatch Repair Protein Expression and Activity at the H-ras Oncogenic Hot Spot.

Degree: PhD, College of Graduate Studies, 2007, University of Toledo Health Science Campus

 Mismatch repair (MMR) corrects mispaired bases and insertion/deletion loops and has long been regarded as a post-replicative DNA repair process. MMR proteins also are involved… (more)

Subjects/Keywords: DNA Repair; Mismatch Repair; MMR; Hotspot; HRAS; DNA Damage

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APA (6th Edition):

Edelbrock, M. A. (2007). Cell Cycle Regulation of DNA Mismatch Repair Protein Expression and Activity at the H-ras Oncogenic Hot Spot. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1192129938

Chicago Manual of Style (16th Edition):

Edelbrock, Michael Aaron. “Cell Cycle Regulation of DNA Mismatch Repair Protein Expression and Activity at the H-ras Oncogenic Hot Spot.” 2007. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed January 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1192129938.

MLA Handbook (7th Edition):

Edelbrock, Michael Aaron. “Cell Cycle Regulation of DNA Mismatch Repair Protein Expression and Activity at the H-ras Oncogenic Hot Spot.” 2007. Web. 22 Jan 2020.

Vancouver:

Edelbrock MA. Cell Cycle Regulation of DNA Mismatch Repair Protein Expression and Activity at the H-ras Oncogenic Hot Spot. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2007. [cited 2020 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1192129938.

Council of Science Editors:

Edelbrock MA. Cell Cycle Regulation of DNA Mismatch Repair Protein Expression and Activity at the H-ras Oncogenic Hot Spot. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1192129938


Vanderbilt University

10. Andrews, Alex Michael. SAN1 - a novel nuclease involved in interstrand cross-link repair.

Degree: PhD, Cell and Developmental Biology, 2018, Vanderbilt University

 The DNA damage response consists of complex signaling pathways that act to recognize and remove various types of genomic insults. One particularly dangerous type of… (more)

Subjects/Keywords: ICL; interstrand cross-link repair; DNA damage; DNA repair

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APA (6th Edition):

Andrews, A. M. (2018). SAN1 - a novel nuclease involved in interstrand cross-link repair. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07022018-103550/ ;

Chicago Manual of Style (16th Edition):

Andrews, Alex Michael. “SAN1 - a novel nuclease involved in interstrand cross-link repair.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2020. http://etd.library.vanderbilt.edu/available/etd-07022018-103550/ ;.

MLA Handbook (7th Edition):

Andrews, Alex Michael. “SAN1 - a novel nuclease involved in interstrand cross-link repair.” 2018. Web. 22 Jan 2020.

Vancouver:

Andrews AM. SAN1 - a novel nuclease involved in interstrand cross-link repair. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2020 Jan 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-07022018-103550/ ;.

Council of Science Editors:

Andrews AM. SAN1 - a novel nuclease involved in interstrand cross-link repair. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://etd.library.vanderbilt.edu/available/etd-07022018-103550/ ;


McMaster University

11. Bowie, Laura. A Novel Type of Signalling from DNA Damage Under ATP Stress in Huntington’s Disease.

Degree: PhD, 2018, McMaster University

Huntington’s disease is an autosomal dominantly inherited neurodegenerative disorder characterized by degeneration of striatal and cortical neurons. The neurons in these regions are particularly energy-demanding… (more)

Subjects/Keywords: Huntington's disease; DNA damage; Bioenergetics; DNA damage repair

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APA (6th Edition):

Bowie, L. (2018). A Novel Type of Signalling from DNA Damage Under ATP Stress in Huntington’s Disease. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/24045

Chicago Manual of Style (16th Edition):

Bowie, Laura. “A Novel Type of Signalling from DNA Damage Under ATP Stress in Huntington’s Disease.” 2018. Doctoral Dissertation, McMaster University. Accessed January 22, 2020. http://hdl.handle.net/11375/24045.

MLA Handbook (7th Edition):

Bowie, Laura. “A Novel Type of Signalling from DNA Damage Under ATP Stress in Huntington’s Disease.” 2018. Web. 22 Jan 2020.

Vancouver:

Bowie L. A Novel Type of Signalling from DNA Damage Under ATP Stress in Huntington’s Disease. [Internet] [Doctoral dissertation]. McMaster University; 2018. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/11375/24045.

Council of Science Editors:

Bowie L. A Novel Type of Signalling from DNA Damage Under ATP Stress in Huntington’s Disease. [Doctoral Dissertation]. McMaster University; 2018. Available from: http://hdl.handle.net/11375/24045


Wesleyan University

12. Zhang, Shu. Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment.

Degree: Chemistry, 2014, Wesleyan University

  Certain types of cancer have defective repair mechanisms, which can cause the accumulation of damaged DNA. The persistent DNA damage can stall replication process… (more)

Subjects/Keywords: DNA damage; DNA damage repair; Synthetic Lethality; SSB; DSB

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APA (6th Edition):

Zhang, S. (2014). Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment. (Masters Thesis). Wesleyan University. Retrieved from https://wesscholar.wesleyan.edu/etd_mas_theses/78

Chicago Manual of Style (16th Edition):

Zhang, Shu. “Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment.” 2014. Masters Thesis, Wesleyan University. Accessed January 22, 2020. https://wesscholar.wesleyan.edu/etd_mas_theses/78.

MLA Handbook (7th Edition):

Zhang, Shu. “Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment.” 2014. Web. 22 Jan 2020.

Vancouver:

Zhang S. Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment. [Internet] [Masters thesis]. Wesleyan University; 2014. [cited 2020 Jan 22]. Available from: https://wesscholar.wesleyan.edu/etd_mas_theses/78.

Council of Science Editors:

Zhang S. Masking DNA Single-Strand Break (SSB): a New Approach to Cancer Treatment. [Masters Thesis]. Wesleyan University; 2014. Available from: https://wesscholar.wesleyan.edu/etd_mas_theses/78

13. Kosicki, Michal Konrad. Cas9-induced on-target genomic damage.

Degree: PhD, 2019, University of Cambridge

 CRISPR/Cas9 is the gene editing tool of choice in basic research and poised to become one in clinical context. However, current studies on the topic… (more)

Subjects/Keywords: CRISPR; DNA damage repair; Cas9; mutagenesis

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APA (6th Edition):

Kosicki, M. K. (2019). Cas9-induced on-target genomic damage. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289911

Chicago Manual of Style (16th Edition):

Kosicki, Michal Konrad. “Cas9-induced on-target genomic damage.” 2019. Doctoral Dissertation, University of Cambridge. Accessed January 22, 2020. https://www.repository.cam.ac.uk/handle/1810/289911.

MLA Handbook (7th Edition):

Kosicki, Michal Konrad. “Cas9-induced on-target genomic damage.” 2019. Web. 22 Jan 2020.

Vancouver:

Kosicki MK. Cas9-induced on-target genomic damage. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Jan 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/289911.

Council of Science Editors:

Kosicki MK. Cas9-induced on-target genomic damage. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289911


University of Cambridge

14. Kosicki, Michal Konrad. Cas9-induced on-target genomic damage.

Degree: PhD, 2019, University of Cambridge

 CRISPR/Cas9 is the gene editing tool of choice in basic research and poised to become one in clinical context. However, current studies on the topic… (more)

Subjects/Keywords: CRISPR; DNA damage repair; Cas9; mutagenesis

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APA (6th Edition):

Kosicki, M. K. (2019). Cas9-induced on-target genomic damage. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/289911 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767862

Chicago Manual of Style (16th Edition):

Kosicki, Michal Konrad. “Cas9-induced on-target genomic damage.” 2019. Doctoral Dissertation, University of Cambridge. Accessed January 22, 2020. https://www.repository.cam.ac.uk/handle/1810/289911 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767862.

MLA Handbook (7th Edition):

Kosicki, Michal Konrad. “Cas9-induced on-target genomic damage.” 2019. Web. 22 Jan 2020.

Vancouver:

Kosicki MK. Cas9-induced on-target genomic damage. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2020 Jan 22]. Available from: https://www.repository.cam.ac.uk/handle/1810/289911 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767862.

Council of Science Editors:

Kosicki MK. Cas9-induced on-target genomic damage. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/289911 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.767862


Deakin University

15. Vonarx, Edward J. The repair and tolerance of DNA damage in higher plants.

Degree: 2000, Deakin University

DNA repair mechanisms constitute an essential cellular response to DNA damage arising either from metabolic processes or from environmental sources such as ultraviolet radiation. Repair(more)

Subjects/Keywords: DNA repair; DNA damage; DNA

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APA (6th Edition):

Vonarx, E. J. (2000). The repair and tolerance of DNA damage in higher plants. (Thesis). Deakin University. Retrieved from http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051110.135641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vonarx, Edward J. “The repair and tolerance of DNA damage in higher plants.” 2000. Thesis, Deakin University. Accessed January 22, 2020. http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051110.135641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vonarx, Edward J. “The repair and tolerance of DNA damage in higher plants.” 2000. Web. 22 Jan 2020.

Vancouver:

Vonarx EJ. The repair and tolerance of DNA damage in higher plants. [Internet] [Thesis]. Deakin University; 2000. [cited 2020 Jan 22]. Available from: http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051110.135641.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vonarx EJ. The repair and tolerance of DNA damage in higher plants. [Thesis]. Deakin University; 2000. Available from: http://tux.lib.deakin.edu.au./adt-VDU/public/adt-VDU20051110.135641

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

16. Badu-Nkansah, Akosua Agyeman. Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks.

Degree: PhD, Biochemistry, 2016, Vanderbilt University

 Genomic replication is a highly challenging task. The DNA replication machinery must precisely duplicate billions of base pairs while tolerating a multitude of obstacles including… (more)

Subjects/Keywords: DNA Repair; Replication Stress; Replication Forks; DNA Replication; DNA Damage

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APA (6th Edition):

Badu-Nkansah, A. A. (2016). Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07192016-131516/ ;

Chicago Manual of Style (16th Edition):

Badu-Nkansah, Akosua Agyeman. “Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed January 22, 2020. http://etd.library.vanderbilt.edu/available/etd-07192016-131516/ ;.

MLA Handbook (7th Edition):

Badu-Nkansah, Akosua Agyeman. “Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks.” 2016. Web. 22 Jan 2020.

Vancouver:

Badu-Nkansah AA. Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2020 Jan 22]. Available from: http://etd.library.vanderbilt.edu/available/etd-07192016-131516/ ;.

Council of Science Editors:

Badu-Nkansah AA. Mechanisms of DNA Translocases in the Repair of Damaged Replication Forks. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu/available/etd-07192016-131516/ ;


University of Minnesota

17. Park, Daeyoon. DNA-Protein Cross-links: Formation in Cells and Tissues, Repair, and Inhibition of DNA Transcription.

Degree: MS, Biochemistry, Molecular Bio, and Biophysics, 2019, University of Minnesota

DNA is constantly damaged by exogenous and endogenous agents, generating a range of nucleobase lesions. It is important to understand the biological consequences and repair(more)

Subjects/Keywords: Crosslink; DNA adduct; DNA damage; DNA repair; mass spectrometry

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APA (6th Edition):

Park, D. (2019). DNA-Protein Cross-links: Formation in Cells and Tissues, Repair, and Inhibition of DNA Transcription. (Masters Thesis). University of Minnesota. Retrieved from http://hdl.handle.net/11299/203567

Chicago Manual of Style (16th Edition):

Park, Daeyoon. “DNA-Protein Cross-links: Formation in Cells and Tissues, Repair, and Inhibition of DNA Transcription.” 2019. Masters Thesis, University of Minnesota. Accessed January 22, 2020. http://hdl.handle.net/11299/203567.

MLA Handbook (7th Edition):

Park, Daeyoon. “DNA-Protein Cross-links: Formation in Cells and Tissues, Repair, and Inhibition of DNA Transcription.” 2019. Web. 22 Jan 2020.

Vancouver:

Park D. DNA-Protein Cross-links: Formation in Cells and Tissues, Repair, and Inhibition of DNA Transcription. [Internet] [Masters thesis]. University of Minnesota; 2019. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/11299/203567.

Council of Science Editors:

Park D. DNA-Protein Cross-links: Formation in Cells and Tissues, Repair, and Inhibition of DNA Transcription. [Masters Thesis]. University of Minnesota; 2019. Available from: http://hdl.handle.net/11299/203567


Florida International University

18. Beaver, Jill M. Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair.

Degree: PhD, Biochemistry, 2016, Florida International University

  Trinucleotide repeat (TNR) expansions are the cause of over 40 human neurodegenerative diseases, and are linked to DNA damage and base excision repair (BER).… (more)

Subjects/Keywords: DNA repair; trinucleotide repeats; DNA damage; trinucleotide repeat instability; DNA base excision repair; Biochemistry

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APA (6th Edition):

Beaver, J. M. (2016). Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair. (Doctoral Dissertation). Florida International University. Retrieved from https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178

Chicago Manual of Style (16th Edition):

Beaver, Jill M. “Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair.” 2016. Doctoral Dissertation, Florida International University. Accessed January 22, 2020. https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178.

MLA Handbook (7th Edition):

Beaver, Jill M. “Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair.” 2016. Web. 22 Jan 2020.

Vancouver:

Beaver JM. Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair. [Internet] [Doctoral dissertation]. Florida International University; 2016. [cited 2020 Jan 22]. Available from: https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178.

Council of Science Editors:

Beaver JM. Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair. [Doctoral Dissertation]. Florida International University; 2016. Available from: https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178


Colorado State University

19. Nelson, Christopher Boulanger. Telomeric double strand breaks undergo resection - but not repair - in G1 human cells.

Degree: PhD, Cell and Molecular Biology, 2017, Colorado State University

 Telomeres are specialized G-rich repetitive regions at the ends of eukaryotic chromosomes (TTAGGGn in mammalian cells). Telomeres function to prevent double strand break (DSB) repair(more)

Subjects/Keywords: DNA Damage Response; Double Strand Break; Telomeres; DNA Repair; DNA Damage; Genomic Stability

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APA (6th Edition):

Nelson, C. B. (2017). Telomeric double strand breaks undergo resection - but not repair - in G1 human cells. (Doctoral Dissertation). Colorado State University. Retrieved from http://hdl.handle.net/10217/184022

Chicago Manual of Style (16th Edition):

Nelson, Christopher Boulanger. “Telomeric double strand breaks undergo resection - but not repair - in G1 human cells.” 2017. Doctoral Dissertation, Colorado State University. Accessed January 22, 2020. http://hdl.handle.net/10217/184022.

MLA Handbook (7th Edition):

Nelson, Christopher Boulanger. “Telomeric double strand breaks undergo resection - but not repair - in G1 human cells.” 2017. Web. 22 Jan 2020.

Vancouver:

Nelson CB. Telomeric double strand breaks undergo resection - but not repair - in G1 human cells. [Internet] [Doctoral dissertation]. Colorado State University; 2017. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10217/184022.

Council of Science Editors:

Nelson CB. Telomeric double strand breaks undergo resection - but not repair - in G1 human cells. [Doctoral Dissertation]. Colorado State University; 2017. Available from: http://hdl.handle.net/10217/184022


University of Lethbridge

20. University of Lethbridge. Faculty of Arts and Science. Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria .

Degree: 2018, University of Lethbridge

 Formation of guanine oxidation products is among the most frequently occurring DNA damaging events, and has been suspected to be related to aging, Alzheimer’s disease… (more)

Subjects/Keywords: DNA repair; DNA repair – Computer simulation; DNA damage; Oxidation; oxidation of guanine; base excision repair; computer modelling; DNA glycosylases

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APA (6th Edition):

Science, U. o. L. F. o. A. a. (2018). Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria . (Thesis). University of Lethbridge. Retrieved from http://hdl.handle.net/10133/5093

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Science, University of Lethbridge. Faculty of Arts and. “Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria .” 2018. Thesis, University of Lethbridge. Accessed January 22, 2020. http://hdl.handle.net/10133/5093.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Science, University of Lethbridge. Faculty of Arts and. “Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria .” 2018. Web. 22 Jan 2020.

Vancouver:

Science UoLFoAa. Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria . [Internet] [Thesis]. University of Lethbridge; 2018. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10133/5093.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Science UoLFoAa. Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria . [Thesis]. University of Lethbridge; 2018. Available from: http://hdl.handle.net/10133/5093

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

21. Ahmad, Alya. Human targeted deletions and biological roles of genes involved in repair of alkylation damage.

Degree: MS, Medical Sciences, 2015, Boston University

DNA repair is not a single mechanism found within cells. There exists numerous different DNA repair mechanisms that function within every type of cell. The… (more)

Subjects/Keywords: Cellular biology; ALKBH; Alkylation damage; Direct reversal repair; DNA repair

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APA (6th Edition):

Ahmad, A. (2015). Human targeted deletions and biological roles of genes involved in repair of alkylation damage. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16247

Chicago Manual of Style (16th Edition):

Ahmad, Alya. “Human targeted deletions and biological roles of genes involved in repair of alkylation damage.” 2015. Masters Thesis, Boston University. Accessed January 22, 2020. http://hdl.handle.net/2144/16247.

MLA Handbook (7th Edition):

Ahmad, Alya. “Human targeted deletions and biological roles of genes involved in repair of alkylation damage.” 2015. Web. 22 Jan 2020.

Vancouver:

Ahmad A. Human targeted deletions and biological roles of genes involved in repair of alkylation damage. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/2144/16247.

Council of Science Editors:

Ahmad A. Human targeted deletions and biological roles of genes involved in repair of alkylation damage. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16247


Portland State University

22. Jeiranian, Harout Arthur. Use of Two-Dimensional Agarose-Gel Analysis to Characterize Processing of UV-Irradiated Plasmids and the Composition of the Replisome Following UV-induced Arrest.

Degree: PhD, Biology, 2012, Portland State University

  In this thesis, I address two fundamental questions related to our understanding of how DNA damage is processed and repaired during replication. Using Two-dimensional… (more)

Subjects/Keywords: DNA replication; DNA polymerases; DNA damage  – Research; DNA repair; Biology; Other Cell and Developmental Biology

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APA (6th Edition):

Jeiranian, H. A. (2012). Use of Two-Dimensional Agarose-Gel Analysis to Characterize Processing of UV-Irradiated Plasmids and the Composition of the Replisome Following UV-induced Arrest. (Doctoral Dissertation). Portland State University. Retrieved from http://pdxscholar.library.pdx.edu/open_access_etds/921

Chicago Manual of Style (16th Edition):

Jeiranian, Harout Arthur. “Use of Two-Dimensional Agarose-Gel Analysis to Characterize Processing of UV-Irradiated Plasmids and the Composition of the Replisome Following UV-induced Arrest.” 2012. Doctoral Dissertation, Portland State University. Accessed January 22, 2020. http://pdxscholar.library.pdx.edu/open_access_etds/921.

MLA Handbook (7th Edition):

Jeiranian, Harout Arthur. “Use of Two-Dimensional Agarose-Gel Analysis to Characterize Processing of UV-Irradiated Plasmids and the Composition of the Replisome Following UV-induced Arrest.” 2012. Web. 22 Jan 2020.

Vancouver:

Jeiranian HA. Use of Two-Dimensional Agarose-Gel Analysis to Characterize Processing of UV-Irradiated Plasmids and the Composition of the Replisome Following UV-induced Arrest. [Internet] [Doctoral dissertation]. Portland State University; 2012. [cited 2020 Jan 22]. Available from: http://pdxscholar.library.pdx.edu/open_access_etds/921.

Council of Science Editors:

Jeiranian HA. Use of Two-Dimensional Agarose-Gel Analysis to Characterize Processing of UV-Irradiated Plasmids and the Composition of the Replisome Following UV-induced Arrest. [Doctoral Dissertation]. Portland State University; 2012. Available from: http://pdxscholar.library.pdx.edu/open_access_etds/921


East Carolina University

23. Loftin, Charles William. Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells.

Degree: 2010, East Carolina University

DNA-dependent protein kinase (DNA-PK) is a holoenzyme of three subunits, Ku70, Ku80, and the DNA-Pk catalytic subunit (DNA-PKcs). DNA-PK serves a role in non-homologous end… (more)

Subjects/Keywords: Biology; DNA damage; Protein kinases; DNA repair; Tumor suppressor proteins

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APA (6th Edition):

Loftin, C. W. (2010). Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells. (Thesis). East Carolina University. Retrieved from http://hdl.handle.net/10342/2727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Loftin, Charles William. “Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells.” 2010. Thesis, East Carolina University. Accessed January 22, 2020. http://hdl.handle.net/10342/2727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Loftin, Charles William. “Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells.” 2010. Web. 22 Jan 2020.

Vancouver:

Loftin CW. Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells. [Internet] [Thesis]. East Carolina University; 2010. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10342/2727.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Loftin CW. Repair of Clustered DNA Lesions in Double Strand Break Repair Deficient Human Tumor Cells. [Thesis]. East Carolina University; 2010. Available from: http://hdl.handle.net/10342/2727

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Newcastle

24. Yuen, Wai Shan. DNA repair and the Fanconi Anemia pathway: insights into female meiosis and mitosis.

Degree: PhD, 2012, University of Newcastle

Research Doctorate - Doctor of Philosophy (PhD)

There are numerous intrinsic and extrinsic factors that cause DNA damage. Without proper DNA repair, such damage would… (more)

Subjects/Keywords: meiosis; mitosis; DNA repair; Fanconi Anemia; DNA damage

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APA (6th Edition):

Yuen, W. S. (2012). DNA repair and the Fanconi Anemia pathway: insights into female meiosis and mitosis. (Doctoral Dissertation). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/1036684

Chicago Manual of Style (16th Edition):

Yuen, Wai Shan. “DNA repair and the Fanconi Anemia pathway: insights into female meiosis and mitosis.” 2012. Doctoral Dissertation, University of Newcastle. Accessed January 22, 2020. http://hdl.handle.net/1959.13/1036684.

MLA Handbook (7th Edition):

Yuen, Wai Shan. “DNA repair and the Fanconi Anemia pathway: insights into female meiosis and mitosis.” 2012. Web. 22 Jan 2020.

Vancouver:

Yuen WS. DNA repair and the Fanconi Anemia pathway: insights into female meiosis and mitosis. [Internet] [Doctoral dissertation]. University of Newcastle; 2012. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1959.13/1036684.

Council of Science Editors:

Yuen WS. DNA repair and the Fanconi Anemia pathway: insights into female meiosis and mitosis. [Doctoral Dissertation]. University of Newcastle; 2012. Available from: http://hdl.handle.net/1959.13/1036684


University of Kansas

25. Lu, Shuai. CHARACTERIZATION OF THE COHESIN ACETYLTRANSFERASE ECO1 AND ITS ROLE IN NUCLEAR FUNCTIONS.

Degree: PhD, Biochemistry & Molecular Biology, 2013, University of Kansas

 Sister-chromatid cohesion plays a vital role in precise chromosome segregation and genome stability. This process is carried out by the cohesin complex and its associated… (more)

Subjects/Keywords: Biochemistry; Molecular biology; Cohesin; Dna damage repair; Dna replication; Eco1; Rdna

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APA (6th Edition):

Lu, S. (2013). CHARACTERIZATION OF THE COHESIN ACETYLTRANSFERASE ECO1 AND ITS ROLE IN NUCLEAR FUNCTIONS. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/14187

Chicago Manual of Style (16th Edition):

Lu, Shuai. “CHARACTERIZATION OF THE COHESIN ACETYLTRANSFERASE ECO1 AND ITS ROLE IN NUCLEAR FUNCTIONS.” 2013. Doctoral Dissertation, University of Kansas. Accessed January 22, 2020. http://hdl.handle.net/1808/14187.

MLA Handbook (7th Edition):

Lu, Shuai. “CHARACTERIZATION OF THE COHESIN ACETYLTRANSFERASE ECO1 AND ITS ROLE IN NUCLEAR FUNCTIONS.” 2013. Web. 22 Jan 2020.

Vancouver:

Lu S. CHARACTERIZATION OF THE COHESIN ACETYLTRANSFERASE ECO1 AND ITS ROLE IN NUCLEAR FUNCTIONS. [Internet] [Doctoral dissertation]. University of Kansas; 2013. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/1808/14187.

Council of Science Editors:

Lu S. CHARACTERIZATION OF THE COHESIN ACETYLTRANSFERASE ECO1 AND ITS ROLE IN NUCLEAR FUNCTIONS. [Doctoral Dissertation]. University of Kansas; 2013. Available from: http://hdl.handle.net/1808/14187


University of Cincinnati

26. Kavanaugh, Gina M. The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair.

Degree: PhD, Medicine: Cell and Molecular Biology, 2011, University of Cincinnati

 Altered chromatin integrity and improper DNA damage repair are commonly associated with the formation and progression of human cancers. Here, we investigate the contribution of… (more)

Subjects/Keywords: Molecular Biology; DEK; DNA-PK; NHEJ; DNA damage repair; histone modification

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APA (6th Edition):

Kavanaugh, G. M. (2011). The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047

Chicago Manual of Style (16th Edition):

Kavanaugh, Gina M. “The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair.” 2011. Doctoral Dissertation, University of Cincinnati. Accessed January 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047.

MLA Handbook (7th Edition):

Kavanaugh, Gina M. “The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair.” 2011. Web. 22 Jan 2020.

Vancouver:

Kavanaugh GM. The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair. [Internet] [Doctoral dissertation]. University of Cincinnati; 2011. [cited 2020 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047.

Council of Science Editors:

Kavanaugh GM. The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair. [Doctoral Dissertation]. University of Cincinnati; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047


University of Toledo Health Science Campus

27. Nadkarni, Aditi A. Functional analysis of the Rad51d (E233G) breast cancer associated polymorphism and a pharmacogenetic evaluation of RAD51D status.

Degree: PhD, College of Graduate Studies, 2008, University of Toledo Health Science Campus

 RAD51D performs functions in homologous recombination and telomere maintenance,two genome stabilization mechanisms. A gene variant in the human RAD51D gene thatreplaced glutamic acid (E) to… (more)

Subjects/Keywords: Genetics; Oncology; Pharmacology; DNA Repair; DNA Damage; cancer; genomics; chemotherapy; pharmacogenetics

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APA (6th Edition):

Nadkarni, A. A. (2008). Functional analysis of the Rad51d (E233G) breast cancer associated polymorphism and a pharmacogenetic evaluation of RAD51D status. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1222877984

Chicago Manual of Style (16th Edition):

Nadkarni, Aditi A. “Functional analysis of the Rad51d (E233G) breast cancer associated polymorphism and a pharmacogenetic evaluation of RAD51D status.” 2008. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed January 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=mco1222877984.

MLA Handbook (7th Edition):

Nadkarni, Aditi A. “Functional analysis of the Rad51d (E233G) breast cancer associated polymorphism and a pharmacogenetic evaluation of RAD51D status.” 2008. Web. 22 Jan 2020.

Vancouver:

Nadkarni AA. Functional analysis of the Rad51d (E233G) breast cancer associated polymorphism and a pharmacogenetic evaluation of RAD51D status. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2008. [cited 2020 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1222877984.

Council of Science Editors:

Nadkarni AA. Functional analysis of the Rad51d (E233G) breast cancer associated polymorphism and a pharmacogenetic evaluation of RAD51D status. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1222877984


Miami University

28. Connelly, Sandra J. Effects of Ultraviolet Radiation (UVR) Induced DNA Damage and Other Ecological Determinants on <i>cryptosporidium Parvum</i>, <i>Giardia Lamblia</i>, and <i>Daphnia</i> spp. in Freshwater Ecosystems.

Degree: PhD, Zoology, 2007, Miami University

  Freshwater ecosystems are especially susceptible to climatic change, including anthropogenic-induced changes, as they are directly influenced by the atmosphere and terrestrial ecosystems. A major… (more)

Subjects/Keywords: UV; DNA damage; DNA repair; Human pathogens; Disinfection; Daphnia; Cryptosporidium; Giardia

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APA (6th Edition):

Connelly, S. J. (2007). Effects of Ultraviolet Radiation (UVR) Induced DNA Damage and Other Ecological Determinants on <i>cryptosporidium Parvum</i>, <i>Giardia Lamblia</i>, and <i>Daphnia</i> spp. in Freshwater Ecosystems. (Doctoral Dissertation). Miami University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=miami1196353326

Chicago Manual of Style (16th Edition):

Connelly, Sandra J. “Effects of Ultraviolet Radiation (UVR) Induced DNA Damage and Other Ecological Determinants on <i>cryptosporidium Parvum</i>, <i>Giardia Lamblia</i>, and <i>Daphnia</i> spp. in Freshwater Ecosystems.” 2007. Doctoral Dissertation, Miami University. Accessed January 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=miami1196353326.

MLA Handbook (7th Edition):

Connelly, Sandra J. “Effects of Ultraviolet Radiation (UVR) Induced DNA Damage and Other Ecological Determinants on <i>cryptosporidium Parvum</i>, <i>Giardia Lamblia</i>, and <i>Daphnia</i> spp. in Freshwater Ecosystems.” 2007. Web. 22 Jan 2020.

Vancouver:

Connelly SJ. Effects of Ultraviolet Radiation (UVR) Induced DNA Damage and Other Ecological Determinants on <i>cryptosporidium Parvum</i>, <i>Giardia Lamblia</i>, and <i>Daphnia</i> spp. in Freshwater Ecosystems. [Internet] [Doctoral dissertation]. Miami University; 2007. [cited 2020 Jan 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1196353326.

Council of Science Editors:

Connelly SJ. Effects of Ultraviolet Radiation (UVR) Induced DNA Damage and Other Ecological Determinants on <i>cryptosporidium Parvum</i>, <i>Giardia Lamblia</i>, and <i>Daphnia</i> spp. in Freshwater Ecosystems. [Doctoral Dissertation]. Miami University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1196353326


University of California – Irvine

29. Alcaraz Silva, Barbara. Laser ablation of single telomeres in mitosis: effects and consequences.

Degree: Biological Sciences, 2014, University of California – Irvine

 Telomeres are essential for protecting chromosome ends not only from the replication-associated DNA loss, but also from unwanted DNA damage response (DDR) and repair by… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Cell cycle; DNA damage; DNA repair; Mitosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Alcaraz Silva, B. (2014). Laser ablation of single telomeres in mitosis: effects and consequences. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/8n31j119

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Alcaraz Silva, Barbara. “Laser ablation of single telomeres in mitosis: effects and consequences.” 2014. Thesis, University of California – Irvine. Accessed January 22, 2020. http://www.escholarship.org/uc/item/8n31j119.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Alcaraz Silva, Barbara. “Laser ablation of single telomeres in mitosis: effects and consequences.” 2014. Web. 22 Jan 2020.

Vancouver:

Alcaraz Silva B. Laser ablation of single telomeres in mitosis: effects and consequences. [Internet] [Thesis]. University of California – Irvine; 2014. [cited 2020 Jan 22]. Available from: http://www.escholarship.org/uc/item/8n31j119.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Alcaraz Silva B. Laser ablation of single telomeres in mitosis: effects and consequences. [Thesis]. University of California – Irvine; 2014. Available from: http://www.escholarship.org/uc/item/8n31j119

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

30. Andersen, Parker Lyng. DNA damage tolerance in mammalian cells.

Degree: 2009, University of Saskatchewan

DNA is susceptible to both exogenous and endogenous damaging agents. Damage is constantly reversed by a wide range of DNA repair pathways. Lesions which escape… (more)

Subjects/Keywords: Cell; Cancer; DNA damage tolerance; DNA repair; Mutation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Andersen, P. L. (2009). DNA damage tolerance in mammalian cells. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/etd-08262009-102011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Andersen, Parker Lyng. “DNA damage tolerance in mammalian cells.” 2009. Thesis, University of Saskatchewan. Accessed January 22, 2020. http://hdl.handle.net/10388/etd-08262009-102011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Andersen, Parker Lyng. “DNA damage tolerance in mammalian cells.” 2009. Web. 22 Jan 2020.

Vancouver:

Andersen PL. DNA damage tolerance in mammalian cells. [Internet] [Thesis]. University of Saskatchewan; 2009. [cited 2020 Jan 22]. Available from: http://hdl.handle.net/10388/etd-08262009-102011.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Andersen PL. DNA damage tolerance in mammalian cells. [Thesis]. University of Saskatchewan; 2009. Available from: http://hdl.handle.net/10388/etd-08262009-102011

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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