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You searched for subject:(DNA damage checkpoint). Showing records 1 – 30 of 78 total matches.

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Queens University

1. Frazer, Corey Thomas. Regulation of the Cdc25 mitotic inducer following replication arrest and DNA damage .

Degree: Biology, 2011, Queens University

 Dephosphorylation of the Cdc2 kinase by the Cdc25 tyrosine phosphatase is the universally conserved trigger for mitotic entry. Cdc25 is also the point of convergence… (more)

Subjects/Keywords: Cdc25 ; Fission Yeast ; DNA Damage Checkpoint ; DNA Replication Checkpoint

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APA (6th Edition):

Frazer, C. T. (2011). Regulation of the Cdc25 mitotic inducer following replication arrest and DNA damage . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/6563

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Frazer, Corey Thomas. “Regulation of the Cdc25 mitotic inducer following replication arrest and DNA damage .” 2011. Thesis, Queens University. Accessed March 03, 2021. http://hdl.handle.net/1974/6563.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Frazer, Corey Thomas. “Regulation of the Cdc25 mitotic inducer following replication arrest and DNA damage .” 2011. Web. 03 Mar 2021.

Vancouver:

Frazer CT. Regulation of the Cdc25 mitotic inducer following replication arrest and DNA damage . [Internet] [Thesis]. Queens University; 2011. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1974/6563.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Frazer CT. Regulation of the Cdc25 mitotic inducer following replication arrest and DNA damage . [Thesis]. Queens University; 2011. Available from: http://hdl.handle.net/1974/6563

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

2. Hengeveld, R.C.C. Loading of cohesin and cohesion generation in response to DNA damage.

Degree: 2011, Universiteit Utrecht

 The sister chromatids are held together by the conserved cohesin complex from DNA replication in S phase until the metaphase to anaphase transition during mitosis.… (more)

Subjects/Keywords: DNA damage; cohesion; DNA repair; DNA damage response; Checkpoint recovery

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APA (6th Edition):

Hengeveld, R. C. C. (2011). Loading of cohesin and cohesion generation in response to DNA damage. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/218765

Chicago Manual of Style (16th Edition):

Hengeveld, R C C. “Loading of cohesin and cohesion generation in response to DNA damage.” 2011. Masters Thesis, Universiteit Utrecht. Accessed March 03, 2021. http://dspace.library.uu.nl:8080/handle/1874/218765.

MLA Handbook (7th Edition):

Hengeveld, R C C. “Loading of cohesin and cohesion generation in response to DNA damage.” 2011. Web. 03 Mar 2021.

Vancouver:

Hengeveld RCC. Loading of cohesin and cohesion generation in response to DNA damage. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2021 Mar 03]. Available from: http://dspace.library.uu.nl:8080/handle/1874/218765.

Council of Science Editors:

Hengeveld RCC. Loading of cohesin and cohesion generation in response to DNA damage. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/218765


University of Cambridge

3. Zhang, Yue. Understanding the activation mechanism of the Chk1 kinase.

Degree: PhD, 2019, University of Cambridge

 Faithful replication of DNA and correct segregation of duplicated chromosomes into two daughter cells are essential to ensure genome integrity and cell survival. Genome integrity… (more)

Subjects/Keywords: Cell-cycle checkpoint; DNA damage response; Chk1

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APA (6th Edition):

Zhang, Y. (2019). Understanding the activation mechanism of the Chk1 kinase. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776

Chicago Manual of Style (16th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Doctoral Dissertation, University of Cambridge. Accessed March 03, 2021. https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776.

MLA Handbook (7th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Web. 03 Mar 2021.

Vancouver:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Mar 03]. Available from: https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776.

Council of Science Editors:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://doi.org/10.17863/CAM.40444 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.782776


University of Cambridge

4. Zhang, Yue. Understanding the activation mechanism of the Chk1 kinase.

Degree: PhD, 2019, University of Cambridge

 Faithful replication of DNA and correct segregation of duplicated chromosomes into two daughter cells are essential to ensure genome integrity and cell survival. Genome integrity… (more)

Subjects/Keywords: Cell-cycle checkpoint; DNA damage response; Chk1

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APA (6th Edition):

Zhang, Y. (2019). Understanding the activation mechanism of the Chk1 kinase. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/293290

Chicago Manual of Style (16th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Doctoral Dissertation, University of Cambridge. Accessed March 03, 2021. https://www.repository.cam.ac.uk/handle/1810/293290.

MLA Handbook (7th Edition):

Zhang, Yue. “Understanding the activation mechanism of the Chk1 kinase.” 2019. Web. 03 Mar 2021.

Vancouver:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Internet] [Doctoral dissertation]. University of Cambridge; 2019. [cited 2021 Mar 03]. Available from: https://www.repository.cam.ac.uk/handle/1810/293290.

Council of Science Editors:

Zhang Y. Understanding the activation mechanism of the Chk1 kinase. [Doctoral Dissertation]. University of Cambridge; 2019. Available from: https://www.repository.cam.ac.uk/handle/1810/293290


University of Edinburgh

5. Van Der Sar, Sjaak. Proteomics of spindle checkpoint complexes and characterisation of novel interactors.

Degree: PhD, 2014, University of Edinburgh

 The eukaryotic cell cycle is governed by molecular checkpoints that ensure genomic integrity and the faithful transmission of chromosomes to daughter cells. They inhibit the… (more)

Subjects/Keywords: 572.8; spindle checkpoint; DNA damage control; proteomics

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APA (6th Edition):

Van Der Sar, S. (2014). Proteomics of spindle checkpoint complexes and characterisation of novel interactors. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/11677

Chicago Manual of Style (16th Edition):

Van Der Sar, Sjaak. “Proteomics of spindle checkpoint complexes and characterisation of novel interactors.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed March 03, 2021. http://hdl.handle.net/1842/11677.

MLA Handbook (7th Edition):

Van Der Sar, Sjaak. “Proteomics of spindle checkpoint complexes and characterisation of novel interactors.” 2014. Web. 03 Mar 2021.

Vancouver:

Van Der Sar S. Proteomics of spindle checkpoint complexes and characterisation of novel interactors. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1842/11677.

Council of Science Editors:

Van Der Sar S. Proteomics of spindle checkpoint complexes and characterisation of novel interactors. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/11677

6. Kermi, Chames. Interaction fontionnelle entre le système de tolérance des lésions et le checkpoint des dommages à l'ADN : conséquences sur la stabilité du génome et l'oncogenèse : Functional interaction between the DNA damage tolerance pathway and the DNA damage checkpoint : implications for genome stability and oncogenesis.

Degree: Docteur es, Biologie Santé, 2016, Montpellier

 Notre génome subit constamment les effets néfastes des agents endommageant de l'ADN. Afin de se protéger de ces effets délétères, les cellules disposent d’un système… (more)

Subjects/Keywords: Système de tolérance; Checkpoint; Oncogenèse; DNA damage tolerance pathway; DNA damage checkpoint; Oncogenesis; 577

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APA (6th Edition):

Kermi, C. (2016). Interaction fontionnelle entre le système de tolérance des lésions et le checkpoint des dommages à l'ADN : conséquences sur la stabilité du génome et l'oncogenèse : Functional interaction between the DNA damage tolerance pathway and the DNA damage checkpoint : implications for genome stability and oncogenesis. (Doctoral Dissertation). Montpellier. Retrieved from http://www.theses.fr/2016MONT3520

Chicago Manual of Style (16th Edition):

Kermi, Chames. “Interaction fontionnelle entre le système de tolérance des lésions et le checkpoint des dommages à l'ADN : conséquences sur la stabilité du génome et l'oncogenèse : Functional interaction between the DNA damage tolerance pathway and the DNA damage checkpoint : implications for genome stability and oncogenesis.” 2016. Doctoral Dissertation, Montpellier. Accessed March 03, 2021. http://www.theses.fr/2016MONT3520.

MLA Handbook (7th Edition):

Kermi, Chames. “Interaction fontionnelle entre le système de tolérance des lésions et le checkpoint des dommages à l'ADN : conséquences sur la stabilité du génome et l'oncogenèse : Functional interaction between the DNA damage tolerance pathway and the DNA damage checkpoint : implications for genome stability and oncogenesis.” 2016. Web. 03 Mar 2021.

Vancouver:

Kermi C. Interaction fontionnelle entre le système de tolérance des lésions et le checkpoint des dommages à l'ADN : conséquences sur la stabilité du génome et l'oncogenèse : Functional interaction between the DNA damage tolerance pathway and the DNA damage checkpoint : implications for genome stability and oncogenesis. [Internet] [Doctoral dissertation]. Montpellier; 2016. [cited 2021 Mar 03]. Available from: http://www.theses.fr/2016MONT3520.

Council of Science Editors:

Kermi C. Interaction fontionnelle entre le système de tolérance des lésions et le checkpoint des dommages à l'ADN : conséquences sur la stabilité du génome et l'oncogenèse : Functional interaction between the DNA damage tolerance pathway and the DNA damage checkpoint : implications for genome stability and oncogenesis. [Doctoral Dissertation]. Montpellier; 2016. Available from: http://www.theses.fr/2016MONT3520

7. 조, 수진. A functional role of chromatin remodeling factor, Rsf-1 in the DNA damage signaling pathway.

Degree: 2014, Ajou University

As a member of imitation switch (ISWI) family in ATP-dependent chromatin remodeling factors, RSF complex consists of SNF2h ATPase and Rsf-1. Although it has been… (more)

Subjects/Keywords: DNA damage; chromatin remodeling factor; Rsf-1; PARP1; DNA damage checkpoint

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APA (6th Edition):

조, . (2014). A functional role of chromatin remodeling factor, Rsf-1 in the DNA damage signaling pathway. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/10857 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

조, 수진. “A functional role of chromatin remodeling factor, Rsf-1 in the DNA damage signaling pathway.” 2014. Thesis, Ajou University. Accessed March 03, 2021. http://repository.ajou.ac.kr/handle/201003/10857 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

조, 수진. “A functional role of chromatin remodeling factor, Rsf-1 in the DNA damage signaling pathway.” 2014. Web. 03 Mar 2021.

Vancouver:

조 . A functional role of chromatin remodeling factor, Rsf-1 in the DNA damage signaling pathway. [Internet] [Thesis]. Ajou University; 2014. [cited 2021 Mar 03]. Available from: http://repository.ajou.ac.kr/handle/201003/10857 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

조 . A functional role of chromatin remodeling factor, Rsf-1 in the DNA damage signaling pathway. [Thesis]. Ajou University; 2014. Available from: http://repository.ajou.ac.kr/handle/201003/10857 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000016596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

8. Lopez Mosqueda, Jaime. Damage-dependent Inhibition of Late Origin Activation in Saccharomyces cerevisiae.

Degree: Biomedical Sciences, 2011, University of California – San Francisco

 Origins of replication are activated throughout the S phase of the cell cycle such that some origins fire early and others fire late to ensure… (more)

Subjects/Keywords: Cellular Biology; Genetics; DNA damage; DNA replication; Intra-S-phase Checkpoint

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APA (6th Edition):

Lopez Mosqueda, J. (2011). Damage-dependent Inhibition of Late Origin Activation in Saccharomyces cerevisiae. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/89s8v9vn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lopez Mosqueda, Jaime. “Damage-dependent Inhibition of Late Origin Activation in Saccharomyces cerevisiae.” 2011. Thesis, University of California – San Francisco. Accessed March 03, 2021. http://www.escholarship.org/uc/item/89s8v9vn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lopez Mosqueda, Jaime. “Damage-dependent Inhibition of Late Origin Activation in Saccharomyces cerevisiae.” 2011. Web. 03 Mar 2021.

Vancouver:

Lopez Mosqueda J. Damage-dependent Inhibition of Late Origin Activation in Saccharomyces cerevisiae. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2021 Mar 03]. Available from: http://www.escholarship.org/uc/item/89s8v9vn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lopez Mosqueda J. Damage-dependent Inhibition of Late Origin Activation in Saccharomyces cerevisiae. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/89s8v9vn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

9. Bruinsma, W. Controlling kinase activity during the cell cycle: from the DNA damage response to mitosis.

Degree: 2014, Universiteit Utrecht

 The cell cycle is the process through which cells execute cell division. This is essential for many basal processes such as organismal development, tissue maintenance… (more)

Subjects/Keywords: Plk1; Tlk2; mitosis; DNA damage; checkpoint recovery; cell cycle

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APA (6th Edition):

Bruinsma, W. (2014). Controlling kinase activity during the cell cycle: from the DNA damage response to mitosis. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/296439

Chicago Manual of Style (16th Edition):

Bruinsma, W. “Controlling kinase activity during the cell cycle: from the DNA damage response to mitosis.” 2014. Doctoral Dissertation, Universiteit Utrecht. Accessed March 03, 2021. http://dspace.library.uu.nl:8080/handle/1874/296439.

MLA Handbook (7th Edition):

Bruinsma, W. “Controlling kinase activity during the cell cycle: from the DNA damage response to mitosis.” 2014. Web. 03 Mar 2021.

Vancouver:

Bruinsma W. Controlling kinase activity during the cell cycle: from the DNA damage response to mitosis. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2014. [cited 2021 Mar 03]. Available from: http://dspace.library.uu.nl:8080/handle/1874/296439.

Council of Science Editors:

Bruinsma W. Controlling kinase activity during the cell cycle: from the DNA damage response to mitosis. [Doctoral Dissertation]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/296439

10. Krenning, L. Cell cycle checkpoints: reversible when possible, irreversible when needed.

Degree: 2015, Universiteit Utrecht

 Cell cycle checkpoints are reversible in nature, and can prevent progression into the next cell cycle phase if needed. In the case of DNA damage,… (more)

Subjects/Keywords: Cell cycle checkpoints; DNA damage; Checkpoint Recovery; Cell fate; Mitosis

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APA (6th Edition):

Krenning, L. (2015). Cell cycle checkpoints: reversible when possible, irreversible when needed. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/322216

Chicago Manual of Style (16th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed March 03, 2021. http://dspace.library.uu.nl:8080/handle/1874/322216.

MLA Handbook (7th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Web. 03 Mar 2021.

Vancouver:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2021 Mar 03]. Available from: http://dspace.library.uu.nl:8080/handle/1874/322216.

Council of Science Editors:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/322216

11. Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.

Degree: 博士(医学), 2014, Nara Medical University / 奈良県立医科大学

Objective Appropriate cell cycle checkpoints are essential for the maintenance of normal cells and chemosensitivity of cancer cells. Clear cell adenocarcinoma (CCA) of the ovary… (more)

Subjects/Keywords: Transcription factors; DNA damage response; Cell cycle; Checkpoint control; Chemoresistance

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APA (6th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, N. (2014). Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. “Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.” 2014. Thesis, Nara Medical University / 奈良県立医科大学. Accessed March 03, 2021. http://hdl.handle.net/10564/2749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi, Noboru. “Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する.” 2014. Web. 03 Mar 2021.

Vancouver:

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi N. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10564/2749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shigetomi, Hiroshi; Sudo, Tamotsu; Shimada, Keiji; Uekuri, Chiharu; Tsuji, Yoriko; Kanayama, Seiji; Naruse, Katsuhiko; Yamada, Yoshihiko; Konishi N. Inhibition of cell death and induction of G2 arrest accumulation in human ovarian clear cells by HNF-1β transcription factor: chemosensitivity is regulated by checkpoint kinase CHK1. : 卵巣明細胞腺癌における転写因子HNF-1βはDNA損傷チェックポイント機構の一つであるCHK1タンパクを制御し、抗癌剤耐性を獲得する. [Thesis]. Nara Medical University / 奈良県立医科大学; 2014. Available from: http://hdl.handle.net/10564/2749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

12. Lim, Pei Xin. Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity.

Degree: PhD, Genetics, 2015, Cornell University

 The mammalian RAD9A-HUS1-RAD1 (9-1-1) complex is a heterotrimeric clamp that promotes checkpoint signaling and DNA repair by functioning as a scaffold at DNA damage sites.… (more)

Subjects/Keywords: RAD9-HUS1-RAD1; DNA damage response; checkpoint-repair coordination

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APA (6th Edition):

Lim, P. X. (2015). Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/40589

Chicago Manual of Style (16th Edition):

Lim, Pei Xin. “Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity.” 2015. Doctoral Dissertation, Cornell University. Accessed March 03, 2021. http://hdl.handle.net/1813/40589.

MLA Handbook (7th Edition):

Lim, Pei Xin. “Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity.” 2015. Web. 03 Mar 2021.

Vancouver:

Lim PX. Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity. [Internet] [Doctoral dissertation]. Cornell University; 2015. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1813/40589.

Council of Science Editors:

Lim PX. Mechanistic Functions Of The 9-1-1 Complex Subunit Hus1 In The Maintenance Of Genomic Integrity. [Doctoral Dissertation]. Cornell University; 2015. Available from: http://hdl.handle.net/1813/40589

13. LIN, XIAOZENG. BMI1 REDUCES ATM AND ATR ACTIVATION DURING DNA DAMAGE RESPONSE THROUGH BINDING TO NBS1 AND TOPBP1.

Degree: PhD, 2017, McMaster University

DNA damage response (DDR) maintains genome integrity through checkpoint activation and lesion repair. While ATM and ATR are essential in DDR, mechanisms regulating their activation… (more)

Subjects/Keywords: BMI1; DNA damage response; ATM and ATR; checkpoint activation; Microvesicles

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APA (6th Edition):

LIN, X. (2017). BMI1 REDUCES ATM AND ATR ACTIVATION DURING DNA DAMAGE RESPONSE THROUGH BINDING TO NBS1 AND TOPBP1. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/22055

Chicago Manual of Style (16th Edition):

LIN, XIAOZENG. “BMI1 REDUCES ATM AND ATR ACTIVATION DURING DNA DAMAGE RESPONSE THROUGH BINDING TO NBS1 AND TOPBP1.” 2017. Doctoral Dissertation, McMaster University. Accessed March 03, 2021. http://hdl.handle.net/11375/22055.

MLA Handbook (7th Edition):

LIN, XIAOZENG. “BMI1 REDUCES ATM AND ATR ACTIVATION DURING DNA DAMAGE RESPONSE THROUGH BINDING TO NBS1 AND TOPBP1.” 2017. Web. 03 Mar 2021.

Vancouver:

LIN X. BMI1 REDUCES ATM AND ATR ACTIVATION DURING DNA DAMAGE RESPONSE THROUGH BINDING TO NBS1 AND TOPBP1. [Internet] [Doctoral dissertation]. McMaster University; 2017. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/11375/22055.

Council of Science Editors:

LIN X. BMI1 REDUCES ATM AND ATR ACTIVATION DURING DNA DAMAGE RESPONSE THROUGH BINDING TO NBS1 AND TOPBP1. [Doctoral Dissertation]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/22055


University of California – San Francisco

14. Edenberg, Ellen R. Fine-tuned regulation of the mitotic genes in response to DNA damage.

Degree: Genetics, 2014, University of California – San Francisco

 One of the biggest challenges a cell faces is stably maintaining its genome in the face of constant exposure to DNA damage from both exogenous… (more)

Subjects/Keywords: Genetics; Molecular biology; Biochemistry; DNA damage checkpoint; Hst3; Ndd1; Rad53; SCF

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APA (6th Edition):

Edenberg, E. R. (2014). Fine-tuned regulation of the mitotic genes in response to DNA damage. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/74h8126s

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Edenberg, Ellen R. “Fine-tuned regulation of the mitotic genes in response to DNA damage.” 2014. Thesis, University of California – San Francisco. Accessed March 03, 2021. http://www.escholarship.org/uc/item/74h8126s.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Edenberg, Ellen R. “Fine-tuned regulation of the mitotic genes in response to DNA damage.” 2014. Web. 03 Mar 2021.

Vancouver:

Edenberg ER. Fine-tuned regulation of the mitotic genes in response to DNA damage. [Internet] [Thesis]. University of California – San Francisco; 2014. [cited 2021 Mar 03]. Available from: http://www.escholarship.org/uc/item/74h8126s.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Edenberg ER. Fine-tuned regulation of the mitotic genes in response to DNA damage. [Thesis]. University of California – San Francisco; 2014. Available from: http://www.escholarship.org/uc/item/74h8126s

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

15. Inanc, Burcu. The control of centrosome duplication after genotoxic stress .

Degree: 2011, National University of Ireland – Galway

 The centrosome is the major microtubule organising center in animal cells. Following DNA damage, centrosome amplification can occur, which in turn can result in the… (more)

Subjects/Keywords: Centrosome; DNA damage response; Cell cycle; Checkpoint; Ionizing radiation; Cep135.

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APA (6th Edition):

Inanc, B. (2011). The control of centrosome duplication after genotoxic stress . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/2862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Inanc, Burcu. “The control of centrosome duplication after genotoxic stress .” 2011. Thesis, National University of Ireland – Galway. Accessed March 03, 2021. http://hdl.handle.net/10379/2862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Inanc, Burcu. “The control of centrosome duplication after genotoxic stress .” 2011. Web. 03 Mar 2021.

Vancouver:

Inanc B. The control of centrosome duplication after genotoxic stress . [Internet] [Thesis]. National University of Ireland – Galway; 2011. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10379/2862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Inanc B. The control of centrosome duplication after genotoxic stress . [Thesis]. National University of Ireland – Galway; 2011. Available from: http://hdl.handle.net/10379/2862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

16. Mastro, Tara. Response to alkylation damage linked to meiotic progression.

Degree: PhD, Molecular Biology, 2015, University of Southern California

 Mechanisms that maintain genome stability are essential for human health. Loss of genome stability is associated with cancer and birth defects. This dissertation uses a… (more)

Subjects/Keywords: meiosis; S. pombe; DNA damage; checkpoint; chromosomes; genetics

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APA (6th Edition):

Mastro, T. (2015). Response to alkylation damage linked to meiotic progression. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/535465/rec/5571

Chicago Manual of Style (16th Edition):

Mastro, Tara. “Response to alkylation damage linked to meiotic progression.” 2015. Doctoral Dissertation, University of Southern California. Accessed March 03, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/535465/rec/5571.

MLA Handbook (7th Edition):

Mastro, Tara. “Response to alkylation damage linked to meiotic progression.” 2015. Web. 03 Mar 2021.

Vancouver:

Mastro T. Response to alkylation damage linked to meiotic progression. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2021 Mar 03]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/535465/rec/5571.

Council of Science Editors:

Mastro T. Response to alkylation damage linked to meiotic progression. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/535465/rec/5571

17. -0653-1123. LINE-1 Retrotransposon Activation in Mouse Fetal Oocytes.

Degree: PhD, Biology, 2020, Johns Hopkins University

 After her pivotal discovery of transposable elements or “jumping genes”, Barbara McClintock left future generations of scientists with the challenge of understanding how such elements… (more)

Subjects/Keywords: LINE-1 retrotransposon; Oocyte; DNA damage checkpoint; Meiosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

-0653-1123. (2020). LINE-1 Retrotransposon Activation in Mouse Fetal Oocytes. (Doctoral Dissertation). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/63437

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-0653-1123. “LINE-1 Retrotransposon Activation in Mouse Fetal Oocytes.” 2020. Doctoral Dissertation, Johns Hopkins University. Accessed March 03, 2021. http://jhir.library.jhu.edu/handle/1774.2/63437.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-0653-1123. “LINE-1 Retrotransposon Activation in Mouse Fetal Oocytes.” 2020. Web. 03 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-0653-1123. LINE-1 Retrotransposon Activation in Mouse Fetal Oocytes. [Internet] [Doctoral dissertation]. Johns Hopkins University; 2020. [cited 2021 Mar 03]. Available from: http://jhir.library.jhu.edu/handle/1774.2/63437.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-0653-1123. LINE-1 Retrotransposon Activation in Mouse Fetal Oocytes. [Doctoral Dissertation]. Johns Hopkins University; 2020. Available from: http://jhir.library.jhu.edu/handle/1774.2/63437

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Vanderbilt University

18. Couch, Frank Benjamin IV. Regulation of Stalled Replication Forks by ATR.

Degree: PhD, Biochemistry, 2014, Vanderbilt University

 Errors during DNA replication lead to mutations which contribute to cancer development. To deal with these challenges, cells contain an innate machinery known as the… (more)

Subjects/Keywords: cell cycle checkpoint; SMARCAL1; replication stress; DNA replication; DNA damage response; ATR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Couch, F. B. I. (2014). Regulation of Stalled Replication Forks by ATR. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11323

Chicago Manual of Style (16th Edition):

Couch, Frank Benjamin IV. “Regulation of Stalled Replication Forks by ATR.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed March 03, 2021. http://hdl.handle.net/1803/11323.

MLA Handbook (7th Edition):

Couch, Frank Benjamin IV. “Regulation of Stalled Replication Forks by ATR.” 2014. Web. 03 Mar 2021.

Vancouver:

Couch FBI. Regulation of Stalled Replication Forks by ATR. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1803/11323.

Council of Science Editors:

Couch FBI. Regulation of Stalled Replication Forks by ATR. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://hdl.handle.net/1803/11323

19. Jalili, Espanta E. Spy1 Regulation of the DNA Damage Response; Checkpoint Activation and Cellular Senescence.

Degree: PhD, Biological Sciences, 2013, National Library of Canada

  Cyclin-dependent kinases (Cdks) control progression through the cell cycle. Proper regulation of the events associated with each phase is critical for the cellular response… (more)

Subjects/Keywords: Pure sciences; Biological sciences; DNA damage; Cellular senescence; Cyclin-dependent kinases; DNA damage response; Checkpoint activation

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APA (6th Edition):

Jalili, E. E. (2013). Spy1 Regulation of the DNA Damage Response; Checkpoint Activation and Cellular Senescence. (Doctoral Dissertation). National Library of Canada. Retrieved from http://scholar.uwindsor.ca/etd/4906

Chicago Manual of Style (16th Edition):

Jalili, Espanta E. “Spy1 Regulation of the DNA Damage Response; Checkpoint Activation and Cellular Senescence.” 2013. Doctoral Dissertation, National Library of Canada. Accessed March 03, 2021. http://scholar.uwindsor.ca/etd/4906.

MLA Handbook (7th Edition):

Jalili, Espanta E. “Spy1 Regulation of the DNA Damage Response; Checkpoint Activation and Cellular Senescence.” 2013. Web. 03 Mar 2021.

Vancouver:

Jalili EE. Spy1 Regulation of the DNA Damage Response; Checkpoint Activation and Cellular Senescence. [Internet] [Doctoral dissertation]. National Library of Canada; 2013. [cited 2021 Mar 03]. Available from: http://scholar.uwindsor.ca/etd/4906.

Council of Science Editors:

Jalili EE. Spy1 Regulation of the DNA Damage Response; Checkpoint Activation and Cellular Senescence. [Doctoral Dissertation]. National Library of Canada; 2013. Available from: http://scholar.uwindsor.ca/etd/4906

20. A. Dondi. ADAPTATION TO THE DNA DAMAGE CHECKPOINT REQUIRES THE REWIRING OF THE CELL CYCLE MACHINERY.

Degree: 2019, Università degli Studi di Milano

 The DNA damage checkpoint is a surveillance mechanism evolved to preserve genome integrity in response to DNA damaging agents. The DNA damage checkpoint senses DNA(more)

Subjects/Keywords: Mitosis; Genomic instability; DNA damage; G2/M DNA damage checkpoint; Adaptation; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Dondi, A. (2019). ADAPTATION TO THE DNA DAMAGE CHECKPOINT REQUIRES THE REWIRING OF THE CELL CYCLE MACHINERY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/609526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dondi, A.. “ADAPTATION TO THE DNA DAMAGE CHECKPOINT REQUIRES THE REWIRING OF THE CELL CYCLE MACHINERY.” 2019. Thesis, Università degli Studi di Milano. Accessed March 03, 2021. http://hdl.handle.net/2434/609526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dondi, A.. “ADAPTATION TO THE DNA DAMAGE CHECKPOINT REQUIRES THE REWIRING OF THE CELL CYCLE MACHINERY.” 2019. Web. 03 Mar 2021.

Vancouver:

Dondi A. ADAPTATION TO THE DNA DAMAGE CHECKPOINT REQUIRES THE REWIRING OF THE CELL CYCLE MACHINERY. [Internet] [Thesis]. Università degli Studi di Milano; 2019. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2434/609526.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dondi A. ADAPTATION TO THE DNA DAMAGE CHECKPOINT REQUIRES THE REWIRING OF THE CELL CYCLE MACHINERY. [Thesis]. Università degli Studi di Milano; 2019. Available from: http://hdl.handle.net/2434/609526

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

21. Bindermann, Robert. relevance for oncogenesis, tumor biology and treatment resistance.

Degree: 2013, Freie Universität Berlin

 Background: The development of malignant tumours goes hand in hand with the accumulation of mutations and chromosomal aberrations. Defects in the cellular DNA damage response… (more)

Subjects/Keywords: chk2; chek2; cell cycle; dna damage repair; dna damage checkpoint; cell cycle checkpoint; 600 Technik, Medizin, angewandte Wissenschaften::610 Medizin und Gesundheit::610 Medizin und Gesundheit

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APA (6th Edition):

Bindermann, R. (2013). relevance for oncogenesis, tumor biology and treatment resistance. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-8399

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bindermann, Robert. “relevance for oncogenesis, tumor biology and treatment resistance.” 2013. Thesis, Freie Universität Berlin. Accessed March 03, 2021. http://dx.doi.org/10.17169/refubium-8399.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bindermann, Robert. “relevance for oncogenesis, tumor biology and treatment resistance.” 2013. Web. 03 Mar 2021.

Vancouver:

Bindermann R. relevance for oncogenesis, tumor biology and treatment resistance. [Internet] [Thesis]. Freie Universität Berlin; 2013. [cited 2021 Mar 03]. Available from: http://dx.doi.org/10.17169/refubium-8399.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bindermann R. relevance for oncogenesis, tumor biology and treatment resistance. [Thesis]. Freie Universität Berlin; 2013. Available from: http://dx.doi.org/10.17169/refubium-8399

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

22. Kim, Jihoon. F-box proteins, cell cycle and cancer.

Degree: 2015, Universiteit Utrecht

 E3 ubiquitin ligases are enzymes that confer specificity to the ubiquitin-proteasome system by directly binding the proteins that are targeted for degradation. E3s are encoded… (more)

Subjects/Keywords: Geneeskunde; Ubiquitin; Proteasome; Proteolysis; F-box protein; Cell cycle; cancer; checkpoint recovery; DNA damage

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APA (6th Edition):

Kim, J. (2015). F-box proteins, cell cycle and cancer. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/302855

Chicago Manual of Style (16th Edition):

Kim, Jihoon. “F-box proteins, cell cycle and cancer.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed March 03, 2021. http://dspace.library.uu.nl:8080/handle/1874/302855.

MLA Handbook (7th Edition):

Kim, Jihoon. “F-box proteins, cell cycle and cancer.” 2015. Web. 03 Mar 2021.

Vancouver:

Kim J. F-box proteins, cell cycle and cancer. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2021 Mar 03]. Available from: http://dspace.library.uu.nl:8080/handle/1874/302855.

Council of Science Editors:

Kim J. F-box proteins, cell cycle and cancer. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/302855

23. Rinaldi, Vera Da Silva Garcia. Genetically dissecting the meiotic checkpoint active during prophase I in female mice.

Degree: PhD, Comparative Biomedical Sciences, 2017, Cornell University

 Females have a non-renewable number of gametes at birth. These oocytes are extremely sensitive to environmental factors that generate DNA damage. Oocyte death due to… (more)

Subjects/Keywords: teaching; Developmental biology; meiosis; Biology; Genetics; reproduction; DNA-damage checkpoint; fertility; gametogenesis

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APA (6th Edition):

Rinaldi, V. D. S. G. (2017). Genetically dissecting the meiotic checkpoint active during prophase I in female mice. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/56716

Chicago Manual of Style (16th Edition):

Rinaldi, Vera Da Silva Garcia. “Genetically dissecting the meiotic checkpoint active during prophase I in female mice.” 2017. Doctoral Dissertation, Cornell University. Accessed March 03, 2021. http://hdl.handle.net/1813/56716.

MLA Handbook (7th Edition):

Rinaldi, Vera Da Silva Garcia. “Genetically dissecting the meiotic checkpoint active during prophase I in female mice.” 2017. Web. 03 Mar 2021.

Vancouver:

Rinaldi VDSG. Genetically dissecting the meiotic checkpoint active during prophase I in female mice. [Internet] [Doctoral dissertation]. Cornell University; 2017. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1813/56716.

Council of Science Editors:

Rinaldi VDSG. Genetically dissecting the meiotic checkpoint active during prophase I in female mice. [Doctoral Dissertation]. Cornell University; 2017. Available from: http://hdl.handle.net/1813/56716

24. M. Ferrari. CHARACTERIZATION OF FACTORS INVOLVED IN DNA DAMAGE CHECKPOINT RECOVERY AND ADAPTATION IN YEAST.

Degree: 2013, Università degli Studi di Milano

 Genome maintenance and stability are essential goals for all the organisms in order to transfer the correct genetic information to the progeny and to keep… (more)

Subjects/Keywords: DNA Damage Checkpoint; Cdc5; Adaptation; Recovery; Tid1; Sae2; Rad9; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Ferrari, M. (2013). CHARACTERIZATION OF FACTORS INVOLVED IN DNA DAMAGE CHECKPOINT RECOVERY AND ADAPTATION IN YEAST. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ferrari, M.. “CHARACTERIZATION OF FACTORS INVOLVED IN DNA DAMAGE CHECKPOINT RECOVERY AND ADAPTATION IN YEAST.” 2013. Thesis, Università degli Studi di Milano. Accessed March 03, 2021. http://hdl.handle.net/2434/229588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ferrari, M.. “CHARACTERIZATION OF FACTORS INVOLVED IN DNA DAMAGE CHECKPOINT RECOVERY AND ADAPTATION IN YEAST.” 2013. Web. 03 Mar 2021.

Vancouver:

Ferrari M. CHARACTERIZATION OF FACTORS INVOLVED IN DNA DAMAGE CHECKPOINT RECOVERY AND ADAPTATION IN YEAST. [Internet] [Thesis]. Università degli Studi di Milano; 2013. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2434/229588.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ferrari M. CHARACTERIZATION OF FACTORS INVOLVED IN DNA DAMAGE CHECKPOINT RECOVERY AND ADAPTATION IN YEAST. [Thesis]. Università degli Studi di Milano; 2013. Available from: http://hdl.handle.net/2434/229588

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

25. Robeson, Alexander. TRAF Regulation of Caspase-2-Dependent Apoptosis in Response to DNA Damage .

Degree: 2016, Duke University

  The DNA of a cell operates as its blueprint, providing coded information for the production of the RNA and proteins that allow the cell… (more)

Subjects/Keywords: Molecular biology; Cellular biology; bimolecular fluorescence complementation; caspase-2; DNA damage; phosphatase; spindle checkpoint

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APA (6th Edition):

Robeson, A. (2016). TRAF Regulation of Caspase-2-Dependent Apoptosis in Response to DNA Damage . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/13401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robeson, Alexander. “TRAF Regulation of Caspase-2-Dependent Apoptosis in Response to DNA Damage .” 2016. Thesis, Duke University. Accessed March 03, 2021. http://hdl.handle.net/10161/13401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robeson, Alexander. “TRAF Regulation of Caspase-2-Dependent Apoptosis in Response to DNA Damage .” 2016. Web. 03 Mar 2021.

Vancouver:

Robeson A. TRAF Regulation of Caspase-2-Dependent Apoptosis in Response to DNA Damage . [Internet] [Thesis]. Duke University; 2016. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10161/13401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robeson A. TRAF Regulation of Caspase-2-Dependent Apoptosis in Response to DNA Damage . [Thesis]. Duke University; 2016. Available from: http://hdl.handle.net/10161/13401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

26. Foss, Kristen. Regulation of the DNA Damage Response and Spindle Checkpoint Signaling Pathways .

Degree: 2015, Duke University

  The ultimate goal of any living cell is to pass on a complete, unaltered copy of its DNA to its daughter cell. The DNA(more)

Subjects/Keywords: Molecular biology; Biochemistry; Cellular biology; Cyclin F; DNA damage; mitosis; phosphatases; Spindle checkpoint

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APA (6th Edition):

Foss, K. (2015). Regulation of the DNA Damage Response and Spindle Checkpoint Signaling Pathways . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/11342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Foss, Kristen. “Regulation of the DNA Damage Response and Spindle Checkpoint Signaling Pathways .” 2015. Thesis, Duke University. Accessed March 03, 2021. http://hdl.handle.net/10161/11342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Foss, Kristen. “Regulation of the DNA Damage Response and Spindle Checkpoint Signaling Pathways .” 2015. Web. 03 Mar 2021.

Vancouver:

Foss K. Regulation of the DNA Damage Response and Spindle Checkpoint Signaling Pathways . [Internet] [Thesis]. Duke University; 2015. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/10161/11342.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Foss K. Regulation of the DNA Damage Response and Spindle Checkpoint Signaling Pathways . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/11342

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

27. Campbell, Callum James. Time to quit? : non-genetic heterogeneity in cell fate propensity after DNA damage.

Degree: PhD, 2018, University of Cambridge

 Cellular checkpoints are typically considered to both facilitate the ordered execution of the cell cycle and to act as a barrier to oncogene driven cell… (more)

Subjects/Keywords: 571.6; Cell cycle; DNA damage; Lineage tracing; microscopy; FUCCI; checkpoint; G1; G2; S phase; mitosis

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APA (6th Edition):

Campbell, C. J. (2018). Time to quit? : non-genetic heterogeneity in cell fate propensity after DNA damage. (Doctoral Dissertation). University of Cambridge. Retrieved from https://doi.org/10.17863/CAM.22851 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744813

Chicago Manual of Style (16th Edition):

Campbell, Callum James. “Time to quit? : non-genetic heterogeneity in cell fate propensity after DNA damage.” 2018. Doctoral Dissertation, University of Cambridge. Accessed March 03, 2021. https://doi.org/10.17863/CAM.22851 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744813.

MLA Handbook (7th Edition):

Campbell, Callum James. “Time to quit? : non-genetic heterogeneity in cell fate propensity after DNA damage.” 2018. Web. 03 Mar 2021.

Vancouver:

Campbell CJ. Time to quit? : non-genetic heterogeneity in cell fate propensity after DNA damage. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2021 Mar 03]. Available from: https://doi.org/10.17863/CAM.22851 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744813.

Council of Science Editors:

Campbell CJ. Time to quit? : non-genetic heterogeneity in cell fate propensity after DNA damage. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://doi.org/10.17863/CAM.22851 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.744813


University of Michigan

28. Rogawski, Mary. Regulation of ATM and CDK2 Kinases by the MRN Complex in DNA Damage Signaling and Cell Cycle Checkpoint Control.

Degree: PhD, Molecular & Cellular Pathology, 2018, University of Michigan

 Double-stranded breaks (DSBs) are toxic DNA lesions that if left unrepaired, lead to mutations, chromosomal aberrations, and oncogenesis. The DNA Damage Response (DDR) is a… (more)

Subjects/Keywords: DNA double-strand break; DNA damage response; DNA repair; Cell cycle checkpoint; Cancer; Pathology; Science (General); Health Sciences; Science

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APA (6th Edition):

Rogawski, M. (2018). Regulation of ATM and CDK2 Kinases by the MRN Complex in DNA Damage Signaling and Cell Cycle Checkpoint Control. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/143961

Chicago Manual of Style (16th Edition):

Rogawski, Mary. “Regulation of ATM and CDK2 Kinases by the MRN Complex in DNA Damage Signaling and Cell Cycle Checkpoint Control.” 2018. Doctoral Dissertation, University of Michigan. Accessed March 03, 2021. http://hdl.handle.net/2027.42/143961.

MLA Handbook (7th Edition):

Rogawski, Mary. “Regulation of ATM and CDK2 Kinases by the MRN Complex in DNA Damage Signaling and Cell Cycle Checkpoint Control.” 2018. Web. 03 Mar 2021.

Vancouver:

Rogawski M. Regulation of ATM and CDK2 Kinases by the MRN Complex in DNA Damage Signaling and Cell Cycle Checkpoint Control. [Internet] [Doctoral dissertation]. University of Michigan; 2018. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/2027.42/143961.

Council of Science Editors:

Rogawski M. Regulation of ATM and CDK2 Kinases by the MRN Complex in DNA Damage Signaling and Cell Cycle Checkpoint Control. [Doctoral Dissertation]. University of Michigan; 2018. Available from: http://hdl.handle.net/2027.42/143961


Wright State University

29. Abrefa, Darlington Osei. Genetic Study of Checkpoint Defects of the Mus81-1 Mutant in the Fission Yeast Schizosaccharomyces Pombe.

Degree: MS, Microbiology and Immunology, 2019, Wright State University

 In response to various perturbations of DNA replication, the DNA replication checkpoint is activated in eukaryotes to stimulate a cascade of cellular responses that are… (more)

Subjects/Keywords: Molecular Biology; Genetics; Microbiology; DNA replication checkpoint; DNA damage checkpoint; Mus81; DNA repair; genetic mutation; cell cycle; phosphorylation; Mrc1; Cds1; Chk1; Fission yeast mutant; MMS; UV; HU; BLM

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APA (6th Edition):

Abrefa, D. O. (2019). Genetic Study of Checkpoint Defects of the Mus81-1 Mutant in the Fission Yeast Schizosaccharomyces Pombe. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1578464554813491

Chicago Manual of Style (16th Edition):

Abrefa, Darlington Osei. “Genetic Study of Checkpoint Defects of the Mus81-1 Mutant in the Fission Yeast Schizosaccharomyces Pombe.” 2019. Masters Thesis, Wright State University. Accessed March 03, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1578464554813491.

MLA Handbook (7th Edition):

Abrefa, Darlington Osei. “Genetic Study of Checkpoint Defects of the Mus81-1 Mutant in the Fission Yeast Schizosaccharomyces Pombe.” 2019. Web. 03 Mar 2021.

Vancouver:

Abrefa DO. Genetic Study of Checkpoint Defects of the Mus81-1 Mutant in the Fission Yeast Schizosaccharomyces Pombe. [Internet] [Masters thesis]. Wright State University; 2019. [cited 2021 Mar 03]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1578464554813491.

Council of Science Editors:

Abrefa DO. Genetic Study of Checkpoint Defects of the Mus81-1 Mutant in the Fission Yeast Schizosaccharomyces Pombe. [Masters Thesis]. Wright State University; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1578464554813491


Cornell University

30. Patel, Darshil R. MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS.

Degree: PhD, Biochemistry, Molecular and Cell Biology, 2018, Cornell University

DNA damage checkpoint pathways are part of the broader cellular DNA damage response (DDR) that promotes genome maintenance when cells experience DNA damage. The RAD9A-HUS1-RAD1… (more)

Subjects/Keywords: Biochemistry; Cellular biology; Molecular biology; Checkpoint; DNA Damage Response; DNA Repair; Genomic Stability; Replication Fork Stability; Tumorigenesis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Patel, D. R. (2018). MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/59785

Chicago Manual of Style (16th Edition):

Patel, Darshil R. “MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS.” 2018. Doctoral Dissertation, Cornell University. Accessed March 03, 2021. http://hdl.handle.net/1813/59785.

MLA Handbook (7th Edition):

Patel, Darshil R. “MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS.” 2018. Web. 03 Mar 2021.

Vancouver:

Patel DR. MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS. [Internet] [Doctoral dissertation]. Cornell University; 2018. [cited 2021 Mar 03]. Available from: http://hdl.handle.net/1813/59785.

Council of Science Editors:

Patel DR. MOLECULAR FUNCTIONS OF THE RAD9A-RAD1-HUS1 DNA DAMAGE CHECKPOINT CLAMP IN GENOME MAINTENANCE AND TUMORIGENESIS. [Doctoral Dissertation]. Cornell University; 2018. Available from: http://hdl.handle.net/1813/59785

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