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You searched for subject:(DNA PK). Showing records 1 – 25 of 25 total matches.

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University of Melbourne

1. AZAD, ARUN. Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells.

Degree: 2012, University of Melbourne

 Ionizing radiation is a widely used anti-cancer modality. Unfortunately however, relapse rates are high following radiation treatment indicating an urgent need for novel radiosensitizing strategies.… (more)

Subjects/Keywords: DNA-PK; DNA-PKCs; senescence; radiation; BEZ235; DNA damage

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APA (6th Edition):

AZAD, A. (2012). Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/37954

Chicago Manual of Style (16th Edition):

AZAD, ARUN. “Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells.” 2012. Doctoral Dissertation, University of Melbourne. Accessed April 17, 2021. http://hdl.handle.net/11343/37954.

MLA Handbook (7th Edition):

AZAD, ARUN. “Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells.” 2012. Web. 17 Apr 2021.

Vancouver:

AZAD A. Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells. [Internet] [Doctoral dissertation]. University of Melbourne; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11343/37954.

Council of Science Editors:

AZAD A. Targeting DNA-dependent protein kinase promotes accelerated senescence of irradiated human cancer cells. [Doctoral Dissertation]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/37954


Vanderbilt University

2. Nam, Edward Adam. Phospho-regulation of the DNA Damage Response Kinase ATR.

Degree: PhD, Cancer Biology, 2011, Vanderbilt University

 Understanding how cells maintain genome integrity is necessary to gain insight into the pathology of cancer and to identify therapeutic targets and biomarkers. The DNA(more)

Subjects/Keywords: replication stress; DNA-PK; ATR; ATM; DNA damage

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APA (6th Edition):

Nam, E. A. (2011). Phospho-regulation of the DNA Damage Response Kinase ATR. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14069

Chicago Manual of Style (16th Edition):

Nam, Edward Adam. “Phospho-regulation of the DNA Damage Response Kinase ATR.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 17, 2021. http://hdl.handle.net/1803/14069.

MLA Handbook (7th Edition):

Nam, Edward Adam. “Phospho-regulation of the DNA Damage Response Kinase ATR.” 2011. Web. 17 Apr 2021.

Vancouver:

Nam EA. Phospho-regulation of the DNA Damage Response Kinase ATR. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1803/14069.

Council of Science Editors:

Nam EA. Phospho-regulation of the DNA Damage Response Kinase ATR. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/14069


University of Edinburgh

3. Parker, James M. Structural studies of two proteins involved in the maintenance of genomic stability, FEN 1 and DNA-PKcs.

Degree: PhD, 2016, University of Edinburgh

 Genomic stability refers to an organism’s ability to maintain and pass forward its genetic information. There are a raft of proteins and pathways whose sole… (more)

Subjects/Keywords: 572.8; cellular repair pathways; DNA-PK; FEN 1; genome maintenance; protein

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APA (6th Edition):

Parker, J. M. (2016). Structural studies of two proteins involved in the maintenance of genomic stability, FEN 1 and DNA-PKcs. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/21709

Chicago Manual of Style (16th Edition):

Parker, James M. “Structural studies of two proteins involved in the maintenance of genomic stability, FEN 1 and DNA-PKcs.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed April 17, 2021. http://hdl.handle.net/1842/21709.

MLA Handbook (7th Edition):

Parker, James M. “Structural studies of two proteins involved in the maintenance of genomic stability, FEN 1 and DNA-PKcs.” 2016. Web. 17 Apr 2021.

Vancouver:

Parker JM. Structural studies of two proteins involved in the maintenance of genomic stability, FEN 1 and DNA-PKcs. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1842/21709.

Council of Science Editors:

Parker JM. Structural studies of two proteins involved in the maintenance of genomic stability, FEN 1 and DNA-PKcs. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/21709


Université Paris-Sud – Paris XI

4. Kotula, Ewa. The role of DNA-dependent protein kinase in tumor metastasis : Le rôle de la protéine kinase dépendante de l’ADN (DNA-PK) dans le processus métastatique.

Degree: Docteur es, Cancérologie / Radiobiologie, 2014, Université Paris-Sud – Paris XI

La protéine kinase dépendante de l’ADN (DNA-PK) est une sérine-thréonine kinase qui est un élément essentiel dans la voie de réparation de l’ADN endommagé par… (more)

Subjects/Keywords: DNA-PK; Réparation d’ADN; Dbait; Vimentine; Métastase; La migration; L’invasion; La sécrétion; DNA-PK; DNA repair; Dbait; Vimentin; Metastasis; Migration; Invasion; Secretion

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APA (6th Edition):

Kotula, E. (2014). The role of DNA-dependent protein kinase in tumor metastasis : Le rôle de la protéine kinase dépendante de l’ADN (DNA-PK) dans le processus métastatique. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA11T021

Chicago Manual of Style (16th Edition):

Kotula, Ewa. “The role of DNA-dependent protein kinase in tumor metastasis : Le rôle de la protéine kinase dépendante de l’ADN (DNA-PK) dans le processus métastatique.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 17, 2021. http://www.theses.fr/2014PA11T021.

MLA Handbook (7th Edition):

Kotula, Ewa. “The role of DNA-dependent protein kinase in tumor metastasis : Le rôle de la protéine kinase dépendante de l’ADN (DNA-PK) dans le processus métastatique.” 2014. Web. 17 Apr 2021.

Vancouver:

Kotula E. The role of DNA-dependent protein kinase in tumor metastasis : Le rôle de la protéine kinase dépendante de l’ADN (DNA-PK) dans le processus métastatique. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2014PA11T021.

Council of Science Editors:

Kotula E. The role of DNA-dependent protein kinase in tumor metastasis : Le rôle de la protéine kinase dépendante de l’ADN (DNA-PK) dans le processus métastatique. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA11T021

5. Cristini, Agnese. DNA double-strand break formation and signalling in response to transcription-blocking topoisomerase I complexes : Formation et signalisation des cassures double-brin de l'ADN lors d'un blocage de la transcription.

Degree: Docteur es, Biotechnologies, cancérologie, 2015, Université Toulouse III – Paul Sabatier

La topoisomérase I (Top1) élimine les surenroulements de l'ADN générés lors de la transcription en produisant transitoirement des complexes de clivage Top1-ADN (Top1cc). Ces Top1cc… (more)

Subjects/Keywords: Topoisomérase; Cassures double-brin; DNA-PK; Ubiquitine; Camptothécine; Transcription; Tdp1; Dommage à l'ADN; Topoisomerase; Double-strand breaks; DNA-PK; Ubiquitin; Camptothecin; Transcription; Tdp1; DNA damage

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APA (6th Edition):

Cristini, A. (2015). DNA double-strand break formation and signalling in response to transcription-blocking topoisomerase I complexes : Formation et signalisation des cassures double-brin de l'ADN lors d'un blocage de la transcription. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2015TOU30276

Chicago Manual of Style (16th Edition):

Cristini, Agnese. “DNA double-strand break formation and signalling in response to transcription-blocking topoisomerase I complexes : Formation et signalisation des cassures double-brin de l'ADN lors d'un blocage de la transcription.” 2015. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed April 17, 2021. http://www.theses.fr/2015TOU30276.

MLA Handbook (7th Edition):

Cristini, Agnese. “DNA double-strand break formation and signalling in response to transcription-blocking topoisomerase I complexes : Formation et signalisation des cassures double-brin de l'ADN lors d'un blocage de la transcription.” 2015. Web. 17 Apr 2021.

Vancouver:

Cristini A. DNA double-strand break formation and signalling in response to transcription-blocking topoisomerase I complexes : Formation et signalisation des cassures double-brin de l'ADN lors d'un blocage de la transcription. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2015. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2015TOU30276.

Council of Science Editors:

Cristini A. DNA double-strand break formation and signalling in response to transcription-blocking topoisomerase I complexes : Formation et signalisation des cassures double-brin de l'ADN lors d'un blocage de la transcription. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2015. Available from: http://www.theses.fr/2015TOU30276


University of Washington

6. Burleigh, Katelyn. Human DNA-PK activates a STING-independent DNA sensing pathway.

Degree: PhD, 2019, University of Washington

 Recognition of foreign nucleic acids is critical for antiviral defense. Detection of DNA is mediated by the cGAS-STING pathway, which activates a potent type I… (more)

Subjects/Keywords: cGAS-STING; DNA-PK; DNA sensing; HSPA8; STING-independent DNA sensing pathway; type I interferons; Immunology; Molecular and cellular biology

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APA (6th Edition):

Burleigh, K. (2019). Human DNA-PK activates a STING-independent DNA sensing pathway. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/44396

Chicago Manual of Style (16th Edition):

Burleigh, Katelyn. “Human DNA-PK activates a STING-independent DNA sensing pathway.” 2019. Doctoral Dissertation, University of Washington. Accessed April 17, 2021. http://hdl.handle.net/1773/44396.

MLA Handbook (7th Edition):

Burleigh, Katelyn. “Human DNA-PK activates a STING-independent DNA sensing pathway.” 2019. Web. 17 Apr 2021.

Vancouver:

Burleigh K. Human DNA-PK activates a STING-independent DNA sensing pathway. [Internet] [Doctoral dissertation]. University of Washington; 2019. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1773/44396.

Council of Science Editors:

Burleigh K. Human DNA-PK activates a STING-independent DNA sensing pathway. [Doctoral Dissertation]. University of Washington; 2019. Available from: http://hdl.handle.net/1773/44396


Duke University

7. Li, Shenduo. Nuclear Basic Fibroblast Growth Factor Regulation of Triple-­Negative Breast Cancer Dormancy/Recurrence .

Degree: 2014, Duke University

  Chemotherapy remains the only available treatment for triple-negative (TN) breast cancer. Although some TN breast cancers respond initially to neoadjuvant chemotherapy, the majority of… (more)

Subjects/Keywords: Pathology; breast cancer; cancer biology; DNA-PK; FGF-2; recurrence; triple-negative breast cancer

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APA (6th Edition):

Li, S. (2014). Nuclear Basic Fibroblast Growth Factor Regulation of Triple-­Negative Breast Cancer Dormancy/Recurrence . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Shenduo. “Nuclear Basic Fibroblast Growth Factor Regulation of Triple-­Negative Breast Cancer Dormancy/Recurrence .” 2014. Thesis, Duke University. Accessed April 17, 2021. http://hdl.handle.net/10161/9411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Shenduo. “Nuclear Basic Fibroblast Growth Factor Regulation of Triple-­Negative Breast Cancer Dormancy/Recurrence .” 2014. Web. 17 Apr 2021.

Vancouver:

Li S. Nuclear Basic Fibroblast Growth Factor Regulation of Triple-­Negative Breast Cancer Dormancy/Recurrence . [Internet] [Thesis]. Duke University; 2014. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10161/9411.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li S. Nuclear Basic Fibroblast Growth Factor Regulation of Triple-­Negative Breast Cancer Dormancy/Recurrence . [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/9411

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

8. Chatterjee, Rachita. DNA-PK sustains autophagy and pancreatic cancer cell growth: DNA-PK supporte l’autophagie et la croissance des cellules pancréatiques tumorales.

Degree: 2021, Université de Sherbrooke

 Selon les récentes statistiques, le cancer pancréatique est la quatrième cause de décès par cancer au Canada avec une survie à 5 ans de 8%.… (more)

Subjects/Keywords: Pancreatic cancer; DNA-PK; Autophagy; NU7441; Cell growth; Treatment; Cancer pancréatique; Autophagie; Croissance cellulaire; Traitement

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APA (6th Edition):

Chatterjee, R. (2021). DNA-PK sustains autophagy and pancreatic cancer cell growth: DNA-PK supporte l’autophagie et la croissance des cellules pancréatiques tumorales. (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/17874

Chicago Manual of Style (16th Edition):

Chatterjee, Rachita. “DNA-PK sustains autophagy and pancreatic cancer cell growth: DNA-PK supporte l’autophagie et la croissance des cellules pancréatiques tumorales.” 2021. Masters Thesis, Université de Sherbrooke. Accessed April 17, 2021. http://hdl.handle.net/11143/17874.

MLA Handbook (7th Edition):

Chatterjee, Rachita. “DNA-PK sustains autophagy and pancreatic cancer cell growth: DNA-PK supporte l’autophagie et la croissance des cellules pancréatiques tumorales.” 2021. Web. 17 Apr 2021.

Vancouver:

Chatterjee R. DNA-PK sustains autophagy and pancreatic cancer cell growth: DNA-PK supporte l’autophagie et la croissance des cellules pancréatiques tumorales. [Internet] [Masters thesis]. Université de Sherbrooke; 2021. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/11143/17874.

Council of Science Editors:

Chatterjee R. DNA-PK sustains autophagy and pancreatic cancer cell growth: DNA-PK supporte l’autophagie et la croissance des cellules pancréatiques tumorales. [Masters Thesis]. Université de Sherbrooke; 2021. Available from: http://hdl.handle.net/11143/17874

9. Heberle, Eléa. Etude du rôle et de la régulation de la Poly(ADP-ribose) Glycohydrolase(PARG) dans la réponse cellulaire aux dommages à l'ADN : Role and regulation of the Poly(ADP-ribose)Glycohydrolase (PARG) in the cell response to DNA damages.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2017, Université de Strasbourg

La Poly(ADP-ribosyl)ation est une modification post-traductionnelle de protéines, impliquée dans un grand nombre de processus biologiques, dont la réparation de l’ADN. Alors que la fonction… (more)

Subjects/Keywords: Poly(ADP-ribose); PAR; PARG; DNA-PK; Régulation; Isoformes; Réponse aux dommages à l’ADN; Réparation; Réplication; Phosphorylation; Modification post-traductionnelle; Poly(ADP-ribose); PAR; PARG; DNA-PK; Régulation; Isoformes; DNA damage response; Repair; Replication; Phosphorylation; Post-translational modification; 572.4; 572.8

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APA (6th Edition):

Heberle, E. (2017). Etude du rôle et de la régulation de la Poly(ADP-ribose) Glycohydrolase(PARG) dans la réponse cellulaire aux dommages à l'ADN : Role and regulation of the Poly(ADP-ribose)Glycohydrolase (PARG) in the cell response to DNA damages. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2017STRAJ106

Chicago Manual of Style (16th Edition):

Heberle, Eléa. “Etude du rôle et de la régulation de la Poly(ADP-ribose) Glycohydrolase(PARG) dans la réponse cellulaire aux dommages à l'ADN : Role and regulation of the Poly(ADP-ribose)Glycohydrolase (PARG) in the cell response to DNA damages.” 2017. Doctoral Dissertation, Université de Strasbourg. Accessed April 17, 2021. http://www.theses.fr/2017STRAJ106.

MLA Handbook (7th Edition):

Heberle, Eléa. “Etude du rôle et de la régulation de la Poly(ADP-ribose) Glycohydrolase(PARG) dans la réponse cellulaire aux dommages à l'ADN : Role and regulation of the Poly(ADP-ribose)Glycohydrolase (PARG) in the cell response to DNA damages.” 2017. Web. 17 Apr 2021.

Vancouver:

Heberle E. Etude du rôle et de la régulation de la Poly(ADP-ribose) Glycohydrolase(PARG) dans la réponse cellulaire aux dommages à l'ADN : Role and regulation of the Poly(ADP-ribose)Glycohydrolase (PARG) in the cell response to DNA damages. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2017. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2017STRAJ106.

Council of Science Editors:

Heberle E. Etude du rôle et de la régulation de la Poly(ADP-ribose) Glycohydrolase(PARG) dans la réponse cellulaire aux dommages à l'ADN : Role and regulation of the Poly(ADP-ribose)Glycohydrolase (PARG) in the cell response to DNA damages. [Doctoral Dissertation]. Université de Strasbourg; 2017. Available from: http://www.theses.fr/2017STRAJ106


University of Southern California

10. Li, Angela Ying-Jian. Molecular analysis of high mobility group A2 (HMGA2) oncogenic function.

Degree: PhD, Pharmaceutical Sciences, 2008, University of Southern California

 HMGA2 is located on chromosome 12q13-15, which is frequently amplified or subjected to chromosomal rearrangements in human cancers. However, the multitudinous tumorigenic roles of HMGA2… (more)

Subjects/Keywords: HMGA2; NHEJ; DNA-PK; DSBs; hTERT; telomere

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APA (6th Edition):

Li, A. Y. (2008). Molecular analysis of high mobility group A2 (HMGA2) oncogenic function. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/86635/rec/4176

Chicago Manual of Style (16th Edition):

Li, Angela Ying-Jian. “Molecular analysis of high mobility group A2 (HMGA2) oncogenic function.” 2008. Doctoral Dissertation, University of Southern California. Accessed April 17, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/86635/rec/4176.

MLA Handbook (7th Edition):

Li, Angela Ying-Jian. “Molecular analysis of high mobility group A2 (HMGA2) oncogenic function.” 2008. Web. 17 Apr 2021.

Vancouver:

Li AY. Molecular analysis of high mobility group A2 (HMGA2) oncogenic function. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2021 Apr 17]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/86635/rec/4176.

Council of Science Editors:

Li AY. Molecular analysis of high mobility group A2 (HMGA2) oncogenic function. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/86635/rec/4176


Université Paris-Sud – Paris XI

11. Croset, Amélie. Identification et caractérisation des mécanismes d'action des molécules appats, les SiDNA, dans l'inhibition des voies de réparation des cassures simple-brin : Identification and characterization of bait molecules mechanisms of action, the SIDNA, in the inhibition of single strand break repair pathway.

Degree: Docteur es, Cancérologie, radiobiologie, 2013, Université Paris-Sud – Paris XI

La plupart des traitements anticancéreux, comme la chimiothérapie ou la radiothérapie, sont cytotoxiques et causent des dommages à l'ADN dans le but d’induire la mort… (more)

Subjects/Keywords: SiDNA; Inhibiteurs des voies de réparation de l’ADN; Poly Adenosine Diphosphate Ribose Polymerase (PARP); Protéine Kinase ADN Dépendant (DNA-PK); Signalisation des dommages à l'ADN; Protéines BRCA; Instabilité génétique; Traitement des Cancers; SiDNA; DNA Repair Inhibitors; Poly Adenosine Diphosphate Ribose Polymerase (PARP); DNA-Activated Protein Kinase (DNA-PK); DNA Damage Signaling; BReast CAncer; Genetic Instability; Cancer Treatement

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APA (6th Edition):

Croset, A. (2013). Identification et caractérisation des mécanismes d'action des molécules appats, les SiDNA, dans l'inhibition des voies de réparation des cassures simple-brin : Identification and characterization of bait molecules mechanisms of action, the SIDNA, in the inhibition of single strand break repair pathway. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T018

Chicago Manual of Style (16th Edition):

Croset, Amélie. “Identification et caractérisation des mécanismes d'action des molécules appats, les SiDNA, dans l'inhibition des voies de réparation des cassures simple-brin : Identification and characterization of bait molecules mechanisms of action, the SIDNA, in the inhibition of single strand break repair pathway.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 17, 2021. http://www.theses.fr/2013PA11T018.

MLA Handbook (7th Edition):

Croset, Amélie. “Identification et caractérisation des mécanismes d'action des molécules appats, les SiDNA, dans l'inhibition des voies de réparation des cassures simple-brin : Identification and characterization of bait molecules mechanisms of action, the SIDNA, in the inhibition of single strand break repair pathway.” 2013. Web. 17 Apr 2021.

Vancouver:

Croset A. Identification et caractérisation des mécanismes d'action des molécules appats, les SiDNA, dans l'inhibition des voies de réparation des cassures simple-brin : Identification and characterization of bait molecules mechanisms of action, the SIDNA, in the inhibition of single strand break repair pathway. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2013PA11T018.

Council of Science Editors:

Croset A. Identification et caractérisation des mécanismes d'action des molécules appats, les SiDNA, dans l'inhibition des voies de réparation des cassures simple-brin : Identification and characterization of bait molecules mechanisms of action, the SIDNA, in the inhibition of single strand break repair pathway. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T018


University of Cincinnati

12. Kavanaugh, Gina M. The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair.

Degree: PhD, Medicine: Cell and Molecular Biology, 2011, University of Cincinnati

 Altered chromatin integrity and improper DNA damage repair are commonly associated with the formation and progression of human cancers. Here, we investigate the contribution of… (more)

Subjects/Keywords: Molecular Biology; DEK; DNA-PK; NHEJ; DNA damage repair; histone modification

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APA (6th Edition):

Kavanaugh, G. M. (2011). The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047

Chicago Manual of Style (16th Edition):

Kavanaugh, Gina M. “The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair.” 2011. Doctoral Dissertation, University of Cincinnati. Accessed April 17, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047.

MLA Handbook (7th Edition):

Kavanaugh, Gina M. “The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair.” 2011. Web. 17 Apr 2021.

Vancouver:

Kavanaugh GM. The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair. [Internet] [Doctoral dissertation]. University of Cincinnati; 2011. [cited 2021 Apr 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047.

Council of Science Editors:

Kavanaugh GM. The Role of the Human DEK Oncogene in the Regulation of DNA Damage Response and Repair. [Doctoral Dissertation]. University of Cincinnati; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305893047


Queensland University of Technology

13. Ashton, Nicholas W. Characterisation of human single-stranded DNA-binding protein 1 (hSSB1) regulation by post-translational modifications.

Degree: 2016, Queensland University of Technology

 Human single-stranded DNA-binding protein 1 (hSSB1) is required for the timely repair of double-strand DNA breaks, as well as the stabilisation and restart of stalled… (more)

Subjects/Keywords: Human single-stranded DNA-binding protein 1; Nucleic acid-binding protein 2; OB-fold containing protein 2B; Sensor of single-stranded DNA complex subunit B; DNA-dependent protein kinase (DNA-PK); PPP-family protein phosphatases; Integrator complex subunit 3 (INTS3); hSSB1; NABP2; OBFC2B

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APA (6th Edition):

Ashton, N. W. (2016). Characterisation of human single-stranded DNA-binding protein 1 (hSSB1) regulation by post-translational modifications. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/98660/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ashton, Nicholas W. “Characterisation of human single-stranded DNA-binding protein 1 (hSSB1) regulation by post-translational modifications.” 2016. Thesis, Queensland University of Technology. Accessed April 17, 2021. https://eprints.qut.edu.au/98660/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ashton, Nicholas W. “Characterisation of human single-stranded DNA-binding protein 1 (hSSB1) regulation by post-translational modifications.” 2016. Web. 17 Apr 2021.

Vancouver:

Ashton NW. Characterisation of human single-stranded DNA-binding protein 1 (hSSB1) regulation by post-translational modifications. [Internet] [Thesis]. Queensland University of Technology; 2016. [cited 2021 Apr 17]. Available from: https://eprints.qut.edu.au/98660/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ashton NW. Characterisation of human single-stranded DNA-binding protein 1 (hSSB1) regulation by post-translational modifications. [Thesis]. Queensland University of Technology; 2016. Available from: https://eprints.qut.edu.au/98660/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Chalasani, Sri Lakshmi. RESOLUTION OF PROXIMAL OXIDATIVE BASE DAMAGE AND 3′-PHOSPHATE TERMINI FOR NONHOMOLOGOUS END JOINING OF FREE RADICAL-MEDIATED DNA DOUBLE-STRAND BREAKS.

Degree: PhD, Pharmacology & Toxicology, 2018, Virginia Commonwealth University

  Clustered damage to DNA is a signature mark of radiation-induced damage, which involves damage to the nucleobases and/or DNA backbone. Double-strand breaks created by… (more)

Subjects/Keywords: NHEJ; BER; hNTH1; Thymine glycol; PNKP; DNA-PK; XLF

…9.1: Role of DNA-PK in NHEJ and DNA DSB processing …... 136 Fig. 9.2: Role of XLF-XRCC4… …response DNA Deoxyribonucleic acid DNA - PK DNA-dependent protein kinase DNA - PKcs DNA… …cell lines. NU7441, a DNA-PK inhibitor significantly blocked the activity in both wild-type… …core NHEJ proteins that form DNA-dependent protein kinase (DNA-PK). XRCC5 and XRCC6… …DNA-PK), and XRCC7 encodes the 470 kDa DNA-catalytic subunit of protein kinase DNA-PKcs… 

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APA (6th Edition):

Chalasani, S. L. (2018). RESOLUTION OF PROXIMAL OXIDATIVE BASE DAMAGE AND 3′-PHOSPHATE TERMINI FOR NONHOMOLOGOUS END JOINING OF FREE RADICAL-MEDIATED DNA DOUBLE-STRAND BREAKS. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/KQ3H-CN65 ; https://scholarscompass.vcu.edu/etd/5237

Chicago Manual of Style (16th Edition):

Chalasani, Sri Lakshmi. “RESOLUTION OF PROXIMAL OXIDATIVE BASE DAMAGE AND 3′-PHOSPHATE TERMINI FOR NONHOMOLOGOUS END JOINING OF FREE RADICAL-MEDIATED DNA DOUBLE-STRAND BREAKS.” 2018. Doctoral Dissertation, Virginia Commonwealth University. Accessed April 17, 2021. https://doi.org/10.25772/KQ3H-CN65 ; https://scholarscompass.vcu.edu/etd/5237.

MLA Handbook (7th Edition):

Chalasani, Sri Lakshmi. “RESOLUTION OF PROXIMAL OXIDATIVE BASE DAMAGE AND 3′-PHOSPHATE TERMINI FOR NONHOMOLOGOUS END JOINING OF FREE RADICAL-MEDIATED DNA DOUBLE-STRAND BREAKS.” 2018. Web. 17 Apr 2021.

Vancouver:

Chalasani SL. RESOLUTION OF PROXIMAL OXIDATIVE BASE DAMAGE AND 3′-PHOSPHATE TERMINI FOR NONHOMOLOGOUS END JOINING OF FREE RADICAL-MEDIATED DNA DOUBLE-STRAND BREAKS. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2018. [cited 2021 Apr 17]. Available from: https://doi.org/10.25772/KQ3H-CN65 ; https://scholarscompass.vcu.edu/etd/5237.

Council of Science Editors:

Chalasani SL. RESOLUTION OF PROXIMAL OXIDATIVE BASE DAMAGE AND 3′-PHOSPHATE TERMINI FOR NONHOMOLOGOUS END JOINING OF FREE RADICAL-MEDIATED DNA DOUBLE-STRAND BREAKS. [Doctoral Dissertation]. Virginia Commonwealth University; 2018. Available from: https://doi.org/10.25772/KQ3H-CN65 ; https://scholarscompass.vcu.edu/etd/5237


Universidade de Lisboa

15. Gonçalves, Maria Inês Cardoso. O papel do DNA-PKcs na transcrição mediada pelo promotor LTR do HIV-1.

Degree: 2017, Universidade de Lisboa

Tese de mestrado em Biologia Molecular e Genética, apresentada à Universidade de Lisboa, através da Faculdade de Ciências, 2017

O DNA-PK (Cinase dependente de DNA)… (more)

Subjects/Keywords: Vírus da Imunodeficiência Humana tipo 1 (HIV-1); Cinase dependente de DNA (DNA-PK); Transativador do LTR do HIV-1 (Tat); Elementos cis; Proteína de especificidade 1 (Pp1); Teses de mestrado - 2017; Domínio/Área Científica::Ciências Naturais::Ciências Biológicas

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APA (6th Edition):

Gonçalves, M. I. C. (2017). O papel do DNA-PKcs na transcrição mediada pelo promotor LTR do HIV-1. (Thesis). Universidade de Lisboa. Retrieved from https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/31528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gonçalves, Maria Inês Cardoso. “O papel do DNA-PKcs na transcrição mediada pelo promotor LTR do HIV-1.” 2017. Thesis, Universidade de Lisboa. Accessed April 17, 2021. https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/31528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gonçalves, Maria Inês Cardoso. “O papel do DNA-PKcs na transcrição mediada pelo promotor LTR do HIV-1.” 2017. Web. 17 Apr 2021.

Vancouver:

Gonçalves MIC. O papel do DNA-PKcs na transcrição mediada pelo promotor LTR do HIV-1. [Internet] [Thesis]. Universidade de Lisboa; 2017. [cited 2021 Apr 17]. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/31528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gonçalves MIC. O papel do DNA-PKcs na transcrição mediada pelo promotor LTR do HIV-1. [Thesis]. Universidade de Lisboa; 2017. Available from: https://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/31528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

16. Jeblonski, Frank. Regulation of cell cycle dependent transcription by E2F and stress pathways.

Degree: 2006, Freie Universität Berlin

 E2F activity controls the expression of a variety of genes that encode proteins essential for DNA replication and cell cycle progression. The E2F family consists… (more)

Subjects/Keywords: Cell cycle DNA repair E2F DNA-PK; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

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APA (6th Edition):

Jeblonski, F. (2006). Regulation of cell cycle dependent transcription by E2F and stress pathways. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-9598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jeblonski, Frank. “Regulation of cell cycle dependent transcription by E2F and stress pathways.” 2006. Thesis, Freie Universität Berlin. Accessed April 17, 2021. http://dx.doi.org/10.17169/refubium-9598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jeblonski, Frank. “Regulation of cell cycle dependent transcription by E2F and stress pathways.” 2006. Web. 17 Apr 2021.

Vancouver:

Jeblonski F. Regulation of cell cycle dependent transcription by E2F and stress pathways. [Internet] [Thesis]. Freie Universität Berlin; 2006. [cited 2021 Apr 17]. Available from: http://dx.doi.org/10.17169/refubium-9598.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jeblonski F. Regulation of cell cycle dependent transcription by E2F and stress pathways. [Thesis]. Freie Universität Berlin; 2006. Available from: http://dx.doi.org/10.17169/refubium-9598

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Sutcu, Haser. Implication of DNA damage and repair in viability and differentiation of muscle stem cells : Implication des dommages à l’ADN et leur réparation sur la viabilité et la différentiation des cellules souches musculaires.

Degree: Docteur es, Biologie cellulaire, 2018, Sorbonne université

Les cassures double-brin (DSB) sont des dommages dangereux de l’ADN et représentent un facteur de risque pour la stabilité du génome. Le maintien de l'intégrité… (more)

Subjects/Keywords: Réparation des cassures double-brin de l’ADN; Cellules souches musculaires; Myogenèse; Kinase DNA-PK; Kinase AKT; Kinase ATM; DNA double strand break repair; Muscle stem cells; Myogenesis; DNAPK kinase; AKT kinase; ATM kinase; 571.6

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APA (6th Edition):

Sutcu, H. (2018). Implication of DNA damage and repair in viability and differentiation of muscle stem cells : Implication des dommages à l’ADN et leur réparation sur la viabilité et la différentiation des cellules souches musculaires. (Doctoral Dissertation). Sorbonne université. Retrieved from http://www.theses.fr/2018SORUS125

Chicago Manual of Style (16th Edition):

Sutcu, Haser. “Implication of DNA damage and repair in viability and differentiation of muscle stem cells : Implication des dommages à l’ADN et leur réparation sur la viabilité et la différentiation des cellules souches musculaires.” 2018. Doctoral Dissertation, Sorbonne université. Accessed April 17, 2021. http://www.theses.fr/2018SORUS125.

MLA Handbook (7th Edition):

Sutcu, Haser. “Implication of DNA damage and repair in viability and differentiation of muscle stem cells : Implication des dommages à l’ADN et leur réparation sur la viabilité et la différentiation des cellules souches musculaires.” 2018. Web. 17 Apr 2021.

Vancouver:

Sutcu H. Implication of DNA damage and repair in viability and differentiation of muscle stem cells : Implication des dommages à l’ADN et leur réparation sur la viabilité et la différentiation des cellules souches musculaires. [Internet] [Doctoral dissertation]. Sorbonne université; 2018. [cited 2021 Apr 17]. Available from: http://www.theses.fr/2018SORUS125.

Council of Science Editors:

Sutcu H. Implication of DNA damage and repair in viability and differentiation of muscle stem cells : Implication des dommages à l’ADN et leur réparation sur la viabilité et la différentiation des cellules souches musculaires. [Doctoral Dissertation]. Sorbonne université; 2018. Available from: http://www.theses.fr/2018SORUS125


Wright State University

18. Lehman, Jason Alexander. Novel Redox and DNA-Dependent Conformational Changes in Human Ku, a DNA-Double Strand Break Repair Protein.

Degree: PhD, Biomedical Sciences PhD, 2008, Wright State University

  Ionizing radiation (IR) and radiomimetic drugs used in cancer chemotherapy cause DNA double-strand breaks which are repaired by the nonhomologous end joining (NHEJ) pathway.… (more)

Subjects/Keywords: Biochemistry; DNA repair; Ku; DNA-PK; non-homologous end joining; mass spectrometry; conformational changes; redox

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APA (6th Edition):

Lehman, J. A. (2008). Novel Redox and DNA-Dependent Conformational Changes in Human Ku, a DNA-Double Strand Break Repair Protein. (Doctoral Dissertation). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1211323245

Chicago Manual of Style (16th Edition):

Lehman, Jason Alexander. “Novel Redox and DNA-Dependent Conformational Changes in Human Ku, a DNA-Double Strand Break Repair Protein.” 2008. Doctoral Dissertation, Wright State University. Accessed April 17, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=wright1211323245.

MLA Handbook (7th Edition):

Lehman, Jason Alexander. “Novel Redox and DNA-Dependent Conformational Changes in Human Ku, a DNA-Double Strand Break Repair Protein.” 2008. Web. 17 Apr 2021.

Vancouver:

Lehman JA. Novel Redox and DNA-Dependent Conformational Changes in Human Ku, a DNA-Double Strand Break Repair Protein. [Internet] [Doctoral dissertation]. Wright State University; 2008. [cited 2021 Apr 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1211323245.

Council of Science Editors:

Lehman JA. Novel Redox and DNA-Dependent Conformational Changes in Human Ku, a DNA-Double Strand Break Repair Protein. [Doctoral Dissertation]. Wright State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1211323245


East Carolina University

19. Rushing, Amanda Williams. Human T-Cell Leukemia Virus Type I Basic Leucine Zipper Factor (HBZ) Modulates Cellular DNA Damage Repair and Antioxidant Responses to Promote Host Cell Survival and Leukemogenesis.

Degree: PhD, PHD-Microbiology & Immunology, 2018, East Carolina University

 Approximately twenty million people worldwide are infected with Human T-cell Leukemia Virus type 1 (HTLV-1). HTLV-1 establishes a life-long, chronic infection which can result in… (more)

Subjects/Keywords: HBZ; leukemogenesis; NHEJ; Ku70; Ku80; DNA-PK; HMOX-1; small Maf; antioxidant; oxidative stress; Human T-lymphotropic virus 1; Cell Survival; Leucine Zippers; Leukemia, T-Cell; DNA Damage; Gene Products, tax

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APA (6th Edition):

Rushing, A. W. (2018). Human T-Cell Leukemia Virus Type I Basic Leucine Zipper Factor (HBZ) Modulates Cellular DNA Damage Repair and Antioxidant Responses to Promote Host Cell Survival and Leukemogenesis. (Doctoral Dissertation). East Carolina University. Retrieved from http://hdl.handle.net/10342/6782

Chicago Manual of Style (16th Edition):

Rushing, Amanda Williams. “Human T-Cell Leukemia Virus Type I Basic Leucine Zipper Factor (HBZ) Modulates Cellular DNA Damage Repair and Antioxidant Responses to Promote Host Cell Survival and Leukemogenesis.” 2018. Doctoral Dissertation, East Carolina University. Accessed April 17, 2021. http://hdl.handle.net/10342/6782.

MLA Handbook (7th Edition):

Rushing, Amanda Williams. “Human T-Cell Leukemia Virus Type I Basic Leucine Zipper Factor (HBZ) Modulates Cellular DNA Damage Repair and Antioxidant Responses to Promote Host Cell Survival and Leukemogenesis.” 2018. Web. 17 Apr 2021.

Vancouver:

Rushing AW. Human T-Cell Leukemia Virus Type I Basic Leucine Zipper Factor (HBZ) Modulates Cellular DNA Damage Repair and Antioxidant Responses to Promote Host Cell Survival and Leukemogenesis. [Internet] [Doctoral dissertation]. East Carolina University; 2018. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/10342/6782.

Council of Science Editors:

Rushing AW. Human T-Cell Leukemia Virus Type I Basic Leucine Zipper Factor (HBZ) Modulates Cellular DNA Damage Repair and Antioxidant Responses to Promote Host Cell Survival and Leukemogenesis. [Doctoral Dissertation]. East Carolina University; 2018. Available from: http://hdl.handle.net/10342/6782


University of Gothenburg / Göteborgs Universitet

20. [No author]. Repair of DNA double-stranded breaks in human cells.

Degree: 2005, University of Gothenburg / Göteborgs Universitet

DNA is continuously subjected to degradation. Therefore, our cells need to constantly repair its DNA to prevent mutations and in the long run cancer. In… (more)

Subjects/Keywords: DNA-PK; DNA double-stranded break; DSB; DNA single-stranded break; SSB; DNA damage; ionizing radiation; IR; DNA repair; calicheamicin gamma1; bleomycin; etoposide; topoisomerase II; radiotherapy; radiosensitizer; ATM

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APA (6th Edition):

author], [. (2005). Repair of DNA double-stranded breaks in human cells. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Repair of DNA double-stranded breaks in human cells.” 2005. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 17, 2021. http://hdl.handle.net/2077/16596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Repair of DNA double-stranded breaks in human cells.” 2005. Web. 17 Apr 2021.

Vancouver:

author] [. Repair of DNA double-stranded breaks in human cells. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2077/16596.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Repair of DNA double-stranded breaks in human cells. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2005. Available from: http://hdl.handle.net/2077/16596

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

21. Ismail, Ismail Hassan 1975-. Recognition and signaling of DNA double-strand breaks in human cells.

Degree: 2006, University of Gothenburg / Göteborgs Universitet

DNA double-strand breaks (DSBs) are the most toxic type of DNA damage. Fortunately, our cells have highly conserved pathways that detect and repair DSBs. Defects… (more)

Subjects/Keywords: DNA-PK; DNA double-strand break; DSB; DNA single-strand break; SSB; DNA damage; ionizing radiation; IR; DNA repair; calicheamicin kappa1; ATM; hydrogen peroxide; H2AX; radiotherapy; radiosensitizer; flow cytometry

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APA (6th Edition):

Ismail, I. H. 1. (2006). Recognition and signaling of DNA double-strand breaks in human cells. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/16855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ismail, Ismail Hassan 1975-. “Recognition and signaling of DNA double-strand breaks in human cells.” 2006. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 17, 2021. http://hdl.handle.net/2077/16855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ismail, Ismail Hassan 1975-. “Recognition and signaling of DNA double-strand breaks in human cells.” 2006. Web. 17 Apr 2021.

Vancouver:

Ismail IH1. Recognition and signaling of DNA double-strand breaks in human cells. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2077/16855.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ismail IH1. Recognition and signaling of DNA double-strand breaks in human cells. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2006. Available from: http://hdl.handle.net/2077/16855

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

22. Björk-Eriksson, Thomas 1960-. Potential biological markers of tumour response to radiation therapy in head and neck cancers.

Degree: 1999, University of Gothenburg / Göteborgs Universitet

 The rationale behind studying biological markers of tumour response to radiation therapy in head and neck cancers is to increase the individualisation of radiation therapy.… (more)

Subjects/Keywords: Predictive assay; inherent radiosensitivity; SF2; proliferation; Tpot; TP53; DNA-PK; Ku; head and neck cancer

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APA (6th Edition):

Björk-Eriksson, T. 1. (1999). Potential biological markers of tumour response to radiation therapy in head and neck cancers. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/15273

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Björk-Eriksson, Thomas 1960-. “Potential biological markers of tumour response to radiation therapy in head and neck cancers.” 1999. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 17, 2021. http://hdl.handle.net/2077/15273.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Björk-Eriksson, Thomas 1960-. “Potential biological markers of tumour response to radiation therapy in head and neck cancers.” 1999. Web. 17 Apr 2021.

Vancouver:

Björk-Eriksson T1. Potential biological markers of tumour response to radiation therapy in head and neck cancers. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 1999. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/2077/15273.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Björk-Eriksson T1. Potential biological markers of tumour response to radiation therapy in head and neck cancers. [Thesis]. University of Gothenburg / Göteborgs Universitet; 1999. Available from: http://hdl.handle.net/2077/15273

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Minnesota

23. Fattah, Farjana Jahan. The impact of loss of function mutations of NHEJ genes on gene targeting and DNA DSB repair in human somatic cells.

Degree: PhD, Biochemistry, Molecular Biology, and Biophysics, 2009, University of Minnesota

 Non-homologous end-joining (NHEJ) is the predominant repair pathway for DNA double-strand breaks (DSBs) in human cells. The core NHEJ pathway is composed of seven factors:… (more)

Subjects/Keywords: DNA-PK; Gene Targeting; Ku86/Ku70; Non-Homologous End Joining; Biochemistry, Molecular Biology, and Biophysics

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APA (6th Edition):

Fattah, F. J. (2009). The impact of loss of function mutations of NHEJ genes on gene targeting and DNA DSB repair in human somatic cells. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/50889

Chicago Manual of Style (16th Edition):

Fattah, Farjana Jahan. “The impact of loss of function mutations of NHEJ genes on gene targeting and DNA DSB repair in human somatic cells.” 2009. Doctoral Dissertation, University of Minnesota. Accessed April 17, 2021. http://purl.umn.edu/50889.

MLA Handbook (7th Edition):

Fattah, Farjana Jahan. “The impact of loss of function mutations of NHEJ genes on gene targeting and DNA DSB repair in human somatic cells.” 2009. Web. 17 Apr 2021.

Vancouver:

Fattah FJ. The impact of loss of function mutations of NHEJ genes on gene targeting and DNA DSB repair in human somatic cells. [Internet] [Doctoral dissertation]. University of Minnesota; 2009. [cited 2021 Apr 17]. Available from: http://purl.umn.edu/50889.

Council of Science Editors:

Fattah FJ. The impact of loss of function mutations of NHEJ genes on gene targeting and DNA DSB repair in human somatic cells. [Doctoral Dissertation]. University of Minnesota; 2009. Available from: http://purl.umn.edu/50889


Université de Sherbrooke

24. Roy, Evelyne. Modulation de la réponse inflammatoire intestinale par la kinase DNA-PK.

Degree: 2007, Université de Sherbrooke

 Des résultats obtenus antérieurement au laboratoire ont démontré l'induction des facteurs de transcription C/EBPs par l'IL-1? ainsi que leur implication dans la régulation de la… (more)

Subjects/Keywords: Facteur de transcription C/EBP; Facteur de transcription NF-kB; DNA-PK; Réponse inflammatoire intestinale; Haptoglobine

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APA (6th Edition):

Roy, E. (2007). Modulation de la réponse inflammatoire intestinale par la kinase DNA-PK. (Masters Thesis). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/3931

Chicago Manual of Style (16th Edition):

Roy, Evelyne. “Modulation de la réponse inflammatoire intestinale par la kinase DNA-PK.” 2007. Masters Thesis, Université de Sherbrooke. Accessed April 17, 2021. http://savoirs.usherbrooke.ca/handle/11143/3931.

MLA Handbook (7th Edition):

Roy, Evelyne. “Modulation de la réponse inflammatoire intestinale par la kinase DNA-PK.” 2007. Web. 17 Apr 2021.

Vancouver:

Roy E. Modulation de la réponse inflammatoire intestinale par la kinase DNA-PK. [Internet] [Masters thesis]. Université de Sherbrooke; 2007. [cited 2021 Apr 17]. Available from: http://savoirs.usherbrooke.ca/handle/11143/3931.

Council of Science Editors:

Roy E. Modulation de la réponse inflammatoire intestinale par la kinase DNA-PK. [Masters Thesis]. Université de Sherbrooke; 2007. Available from: http://savoirs.usherbrooke.ca/handle/11143/3931

25. Woods, Derek S. The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors.

Degree: 2014, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

In mammalian cells DNA double strand breaks (DSBs) are highly variable with respect to sequence and structure all of which… (more)

Subjects/Keywords: DNA Repair,; NHEJ; DNA-PK; Ku70/80; Ku80 Carboxy Terminus; DNA  – Research  – Methodology; DNA-protein interactions; Protein-protein interactions; DNA  – Synthesis; Enzymes  – Analysis; Protein kinases  – Research  – Evaluation; DNA repair; DNA damage; DNA-binding proteins; Cellular control mechanisms; Nucleotide sequence; Mutagenesis; Biochemical genetics

…NHEJ requires the formation and activation of the DNA-dependent protein kinase (DNA-PK… …x29;. Once activated, DNA-PK functions to regulate NHEJ pathway progression as well as… …addition to its direct role in NHEJ, recent work has demonstrated that DNA-PK also regulates HR… …DNA termini. DNA-PKcs is recruited to form the DNA-PK heterotrimer. DNA-PK undergoes… …autophosphorylation as well as phosphorylation of downstream molecules. DNA termini are processed and DNA-PK… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Woods, D. S. (2014). The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/4665

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Woods, Derek S. “The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors.” 2014. Thesis, IUPUI. Accessed April 17, 2021. http://hdl.handle.net/1805/4665.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Woods, Derek S. “The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors.” 2014. Web. 17 Apr 2021.

Vancouver:

Woods DS. The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors. [Internet] [Thesis]. IUPUI; 2014. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1805/4665.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Woods DS. The influence of the Ku80 carboxy-terminus on activation of the DNA-dependent protein kinase and DNA repair is dependent on the structure of DNA cofactors. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/4665

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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