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You searched for subject:(Curriculum in Toxicology). Showing records 1 – 30 of 69 total matches.

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University of North Carolina

1. Bhavaraju, Laya. Examination of diesel and biodiesel exhaust exposure induced disruption of arachidonic acid metabolism.

Degree: 2015, University of North Carolina

 Diesel combustion emissions contributes a large amount of particulate matter found in air pollution collected in high-traffic urban areas. Current epidemiology studies indicate a strong… (more)

Subjects/Keywords: Toxicology; School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Bhavaraju, L. (2015). Examination of diesel and biodiesel exhaust exposure induced disruption of arachidonic acid metabolism. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9dd75e93-d3fb-402c-85d6-148c4ffacce1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bhavaraju, Laya. “Examination of diesel and biodiesel exhaust exposure induced disruption of arachidonic acid metabolism.” 2015. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:9dd75e93-d3fb-402c-85d6-148c4ffacce1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bhavaraju, Laya. “Examination of diesel and biodiesel exhaust exposure induced disruption of arachidonic acid metabolism.” 2015. Web. 21 Jan 2021.

Vancouver:

Bhavaraju L. Examination of diesel and biodiesel exhaust exposure induced disruption of arachidonic acid metabolism. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:9dd75e93-d3fb-402c-85d6-148c4ffacce1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bhavaraju L. Examination of diesel and biodiesel exhaust exposure induced disruption of arachidonic acid metabolism. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:9dd75e93-d3fb-402c-85d6-148c4ffacce1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

2. Wei, Darmood. Elucidating SNF5 Regulated Gene Expression in Malignant Rhabdoid Tumor Development.

Degree: 2014, University of North Carolina

 Malignant Rhabdoid Tumors (MRTs), a pediatric renal cancer, lack SNF5, a subunit of the SWI/SNF chromatin remodeling complex which regulates nucleosome positioning and gene expression.… (more)

Subjects/Keywords: Toxicology; School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Wei, D. (2014). Elucidating SNF5 Regulated Gene Expression in Malignant Rhabdoid Tumor Development. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:bcca99e3-3b24-4f32-a7b1-38eb1515c585

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wei, Darmood. “Elucidating SNF5 Regulated Gene Expression in Malignant Rhabdoid Tumor Development.” 2014. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:bcca99e3-3b24-4f32-a7b1-38eb1515c585.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wei, Darmood. “Elucidating SNF5 Regulated Gene Expression in Malignant Rhabdoid Tumor Development.” 2014. Web. 21 Jan 2021.

Vancouver:

Wei D. Elucidating SNF5 Regulated Gene Expression in Malignant Rhabdoid Tumor Development. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:bcca99e3-3b24-4f32-a7b1-38eb1515c585.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wei D. Elucidating SNF5 Regulated Gene Expression in Malignant Rhabdoid Tumor Development. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:bcca99e3-3b24-4f32-a7b1-38eb1515c585

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

3. Filgo, Adam. Early Life Chemical Exposures and Latent Mammary Effects in Male and Female Rats.

Degree: 2015, University of North Carolina

 Volatile organic compounds (VOCs) are common industrial solvents used in a number of cleaning agents and solvents. In the United States, spikes in birth defects,… (more)

Subjects/Keywords: Toxicology; School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Filgo, A. (2015). Early Life Chemical Exposures and Latent Mammary Effects in Male and Female Rats. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:3c15ffd0-0e71-4dc9-b76f-e1827831c212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Filgo, Adam. “Early Life Chemical Exposures and Latent Mammary Effects in Male and Female Rats.” 2015. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:3c15ffd0-0e71-4dc9-b76f-e1827831c212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Filgo, Adam. “Early Life Chemical Exposures and Latent Mammary Effects in Male and Female Rats.” 2015. Web. 21 Jan 2021.

Vancouver:

Filgo A. Early Life Chemical Exposures and Latent Mammary Effects in Male and Female Rats. [Internet] [Thesis]. University of North Carolina; 2015. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:3c15ffd0-0e71-4dc9-b76f-e1827831c212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Filgo A. Early Life Chemical Exposures and Latent Mammary Effects in Male and Female Rats. [Thesis]. University of North Carolina; 2015. Available from: https://cdr.lib.unc.edu/record/uuid:3c15ffd0-0e71-4dc9-b76f-e1827831c212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

4. High, Monica Denise. A Murine Model of Genetic Determinants of RSV Disease.

Degree: 2010, University of North Carolina

 Respiratory Syncytial Virus (RSV) is the leading cause of lower respiratory tract infection (LRTI) in infants. Approximately 70% of infants are infected with RSV within… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

High, M. D. (2010). A Murine Model of Genetic Determinants of RSV Disease. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:1b32dbfb-3d96-48bc-8270-f2cf5e01b6f5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

High, Monica Denise. “A Murine Model of Genetic Determinants of RSV Disease.” 2010. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:1b32dbfb-3d96-48bc-8270-f2cf5e01b6f5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

High, Monica Denise. “A Murine Model of Genetic Determinants of RSV Disease.” 2010. Web. 21 Jan 2021.

Vancouver:

High MD. A Murine Model of Genetic Determinants of RSV Disease. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:1b32dbfb-3d96-48bc-8270-f2cf5e01b6f5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

High MD. A Murine Model of Genetic Determinants of RSV Disease. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:1b32dbfb-3d96-48bc-8270-f2cf5e01b6f5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

5. Weed, Melanie B. The impact of epidermal growth factor receptor inhibition on energy homeostasis.

Degree: 2010, University of North Carolina

 As a result of the worldwide rise in obesity and obesity-related complications such as diabetes, stroke, and cardiovascular disease, understanding the mechanisms associated with this… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Weed, M. B. (2010). The impact of epidermal growth factor receptor inhibition on energy homeostasis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:df7baf0e-8c55-426f-875d-8a0e76c69872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Weed, Melanie B. “The impact of epidermal growth factor receptor inhibition on energy homeostasis.” 2010. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:df7baf0e-8c55-426f-875d-8a0e76c69872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Weed, Melanie B. “The impact of epidermal growth factor receptor inhibition on energy homeostasis.” 2010. Web. 21 Jan 2021.

Vancouver:

Weed MB. The impact of epidermal growth factor receptor inhibition on energy homeostasis. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:df7baf0e-8c55-426f-875d-8a0e76c69872.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Weed MB. The impact of epidermal growth factor receptor inhibition on energy homeostasis. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:df7baf0e-8c55-426f-875d-8a0e76c69872

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

6. Askew, Emily B. Mechanisms that regulate androgen receptor transcriptional activity.

Degree: 2010, University of North Carolina

 Testosterone (T) and dihydrotestosterone (DHT) are natural ligands for the androgen receptor (AR), an intracellular transcription factor and nuclear receptor. DHT is a more potent… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Askew, E. B. (2010). Mechanisms that regulate androgen receptor transcriptional activity. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:d0d09358-5988-497d-adc7-6e64263528d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Askew, Emily B. “Mechanisms that regulate androgen receptor transcriptional activity.” 2010. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:d0d09358-5988-497d-adc7-6e64263528d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Askew, Emily B. “Mechanisms that regulate androgen receptor transcriptional activity.” 2010. Web. 21 Jan 2021.

Vancouver:

Askew EB. Mechanisms that regulate androgen receptor transcriptional activity. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:d0d09358-5988-497d-adc7-6e64263528d3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Askew EB. Mechanisms that regulate androgen receptor transcriptional activity. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:d0d09358-5988-497d-adc7-6e64263528d3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

7. Godin, Elizabeth Anne. Magnetic resonance imaging, in situ hybridization, and immunohistochemistry-based analyses of early prenatal ethanol exposure-induced central nervous system abnormalities.

Degree: 2010, University of North Carolina

 Fetal alcohol spectrum disorders (FASD), the collection of defects resulting from prenatal alcohol (ethanol) exposure, has been the subject of basic and clinical investigation for… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Godin, E. A. (2010). Magnetic resonance imaging, in situ hybridization, and immunohistochemistry-based analyses of early prenatal ethanol exposure-induced central nervous system abnormalities. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ad950cd6-95f8-4e9a-aa45-339d97e9dca8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Godin, Elizabeth Anne. “Magnetic resonance imaging, in situ hybridization, and immunohistochemistry-based analyses of early prenatal ethanol exposure-induced central nervous system abnormalities.” 2010. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:ad950cd6-95f8-4e9a-aa45-339d97e9dca8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Godin, Elizabeth Anne. “Magnetic resonance imaging, in situ hybridization, and immunohistochemistry-based analyses of early prenatal ethanol exposure-induced central nervous system abnormalities.” 2010. Web. 21 Jan 2021.

Vancouver:

Godin EA. Magnetic resonance imaging, in situ hybridization, and immunohistochemistry-based analyses of early prenatal ethanol exposure-induced central nervous system abnormalities. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:ad950cd6-95f8-4e9a-aa45-339d97e9dca8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Godin EA. Magnetic resonance imaging, in situ hybridization, and immunohistochemistry-based analyses of early prenatal ethanol exposure-induced central nervous system abnormalities. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:ad950cd6-95f8-4e9a-aa45-339d97e9dca8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

8. Smith, Lindsay Kay. Glucocorticoid regulation of microRNA expression modulates lymphocyte apoptosis.

Degree: 2010, University of North Carolina

 Apoptosis of immature thymocytes is a carefully coordinated process that is controlled by the actions of pro-apoptotic and anti-apoptotic effectors. Endogenous glucocorticoids modulate immune development… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Smith, L. K. (2010). Glucocorticoid regulation of microRNA expression modulates lymphocyte apoptosis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:8e1b427f-ab95-4941-9946-49be83b103e8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Lindsay Kay. “Glucocorticoid regulation of microRNA expression modulates lymphocyte apoptosis.” 2010. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:8e1b427f-ab95-4941-9946-49be83b103e8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Lindsay Kay. “Glucocorticoid regulation of microRNA expression modulates lymphocyte apoptosis.” 2010. Web. 21 Jan 2021.

Vancouver:

Smith LK. Glucocorticoid regulation of microRNA expression modulates lymphocyte apoptosis. [Internet] [Thesis]. University of North Carolina; 2010. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:8e1b427f-ab95-4941-9946-49be83b103e8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith LK. Glucocorticoid regulation of microRNA expression modulates lymphocyte apoptosis. [Thesis]. University of North Carolina; 2010. Available from: https://cdr.lib.unc.edu/record/uuid:8e1b427f-ab95-4941-9946-49be83b103e8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

9. Shannahan, Jonathan H. Susceptibility to libby amphibole-induced pulmonary disease in the cardiovascular-compromised subpopulation.

Degree: 2011, University of North Carolina

 Human exposure to asbestos is known to cause the development of mesothelioma, asbestosis and lung cancer. Asbestos toxicity is thought to be mediated through reactive… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Shannahan, J. H. (2011). Susceptibility to libby amphibole-induced pulmonary disease in the cardiovascular-compromised subpopulation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:be3894a2-8a7d-4cd5-9408-0f32aaf3bfd0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shannahan, Jonathan H. “Susceptibility to libby amphibole-induced pulmonary disease in the cardiovascular-compromised subpopulation.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:be3894a2-8a7d-4cd5-9408-0f32aaf3bfd0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shannahan, Jonathan H. “Susceptibility to libby amphibole-induced pulmonary disease in the cardiovascular-compromised subpopulation.” 2011. Web. 21 Jan 2021.

Vancouver:

Shannahan JH. Susceptibility to libby amphibole-induced pulmonary disease in the cardiovascular-compromised subpopulation. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:be3894a2-8a7d-4cd5-9408-0f32aaf3bfd0.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shannahan JH. Susceptibility to libby amphibole-induced pulmonary disease in the cardiovascular-compromised subpopulation. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:be3894a2-8a7d-4cd5-9408-0f32aaf3bfd0

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

10. Griggs, Jennifer Lynn. The effects of diesel exhaust particles or titanium dioxide nanoparticles on pulmonary inflammation.

Degree: 2011, University of North Carolina

 Pulmonary exposure to particulate matter has been associated with development of pulmonary inflammation which can lead to pulmonary illnesses and diseases. Various cultures have turned… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Griggs, J. L. (2011). The effects of diesel exhaust particles or titanium dioxide nanoparticles on pulmonary inflammation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:82b9ca29-bbe5-4c8b-b155-c83e33bd4d14

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Griggs, Jennifer Lynn. “The effects of diesel exhaust particles or titanium dioxide nanoparticles on pulmonary inflammation.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:82b9ca29-bbe5-4c8b-b155-c83e33bd4d14.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Griggs, Jennifer Lynn. “The effects of diesel exhaust particles or titanium dioxide nanoparticles on pulmonary inflammation.” 2011. Web. 21 Jan 2021.

Vancouver:

Griggs JL. The effects of diesel exhaust particles or titanium dioxide nanoparticles on pulmonary inflammation. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:82b9ca29-bbe5-4c8b-b155-c83e33bd4d14.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Griggs JL. The effects of diesel exhaust particles or titanium dioxide nanoparticles on pulmonary inflammation. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:82b9ca29-bbe5-4c8b-b155-c83e33bd4d14

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

11. Marion, Tracy L. Role of bile acids and compensatory hepatic transport proteins in troglitazone-mediated hepatotoxicity.

Degree: 2011, University of North Carolina

 Drug-induced hepatotoxicity is the most common reason pharmaceuticals are removed from clinical use. Understanding mechanisms of hepatotoxicity, and in vitro models to predict hepatotoxicity in… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Marion, T. L. (2011). Role of bile acids and compensatory hepatic transport proteins in troglitazone-mediated hepatotoxicity. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c46741cd-042a-423f-837f-d17b36b00e2f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marion, Tracy L. “Role of bile acids and compensatory hepatic transport proteins in troglitazone-mediated hepatotoxicity.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:c46741cd-042a-423f-837f-d17b36b00e2f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marion, Tracy L. “Role of bile acids and compensatory hepatic transport proteins in troglitazone-mediated hepatotoxicity.” 2011. Web. 21 Jan 2021.

Vancouver:

Marion TL. Role of bile acids and compensatory hepatic transport proteins in troglitazone-mediated hepatotoxicity. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:c46741cd-042a-423f-837f-d17b36b00e2f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marion TL. Role of bile acids and compensatory hepatic transport proteins in troglitazone-mediated hepatotoxicity. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:c46741cd-042a-423f-837f-d17b36b00e2f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

12. Horvath, Katherine M. Cigarette Smoke Alters Influenza Induced Immune Responses of the Respiratory Epithelium.

Degree: 2011, University of North Carolina

 Epidemiological evidence demonstrates that smokers are at increased risk for and suffer greater morbidity and mortality from influenza infection but the mechanism underlying this susceptibility… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Horvath, K. M. (2011). Cigarette Smoke Alters Influenza Induced Immune Responses of the Respiratory Epithelium. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:981de7e0-3f52-4962-8ed1-cbfae9a3e18d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Horvath, Katherine M. “Cigarette Smoke Alters Influenza Induced Immune Responses of the Respiratory Epithelium.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:981de7e0-3f52-4962-8ed1-cbfae9a3e18d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Horvath, Katherine M. “Cigarette Smoke Alters Influenza Induced Immune Responses of the Respiratory Epithelium.” 2011. Web. 21 Jan 2021.

Vancouver:

Horvath KM. Cigarette Smoke Alters Influenza Induced Immune Responses of the Respiratory Epithelium. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:981de7e0-3f52-4962-8ed1-cbfae9a3e18d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Horvath KM. Cigarette Smoke Alters Influenza Induced Immune Responses of the Respiratory Epithelium. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:981de7e0-3f52-4962-8ed1-cbfae9a3e18d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

13. Davies, Brian Robert. Identification of novel binding targets of the SWI/SNF complex member SNF5 in malignant rhabdoid tumors.

Degree: 2011, University of North Carolina

 Malignant Rhabdoid Tumors (MRTs) show a loss of SNF5, a core subunit of the SWI/SNF chromatin remodeling complex. These cancers are genetically stable, but epigenetically… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Davies, B. R. (2011). Identification of novel binding targets of the SWI/SNF complex member SNF5 in malignant rhabdoid tumors. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:70665bc8-31e6-49d1-83f5-b237e68ba809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davies, Brian Robert. “Identification of novel binding targets of the SWI/SNF complex member SNF5 in malignant rhabdoid tumors.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:70665bc8-31e6-49d1-83f5-b237e68ba809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davies, Brian Robert. “Identification of novel binding targets of the SWI/SNF complex member SNF5 in malignant rhabdoid tumors.” 2011. Web. 21 Jan 2021.

Vancouver:

Davies BR. Identification of novel binding targets of the SWI/SNF complex member SNF5 in malignant rhabdoid tumors. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:70665bc8-31e6-49d1-83f5-b237e68ba809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davies BR. Identification of novel binding targets of the SWI/SNF complex member SNF5 in malignant rhabdoid tumors. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:70665bc8-31e6-49d1-83f5-b237e68ba809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

14. Irons, Terra D. Defining Behavioral Phenotypes Associated with Dopaminergic Nervous System Toxicity in Zebrafish Larvae.

Degree: 2011, University of North Carolina

 Parkinson's disease and attention deficit hyperactivity disorder are motor conditions characterized by dysfunction of dopaminergic signaling in the brain. The origins of these dopamine-related movement… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Irons, T. D. (2011). Defining Behavioral Phenotypes Associated with Dopaminergic Nervous System Toxicity in Zebrafish Larvae. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:bcb10a8a-1d89-4bd6-b65e-080811a56696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Irons, Terra D. “Defining Behavioral Phenotypes Associated with Dopaminergic Nervous System Toxicity in Zebrafish Larvae.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:bcb10a8a-1d89-4bd6-b65e-080811a56696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Irons, Terra D. “Defining Behavioral Phenotypes Associated with Dopaminergic Nervous System Toxicity in Zebrafish Larvae.” 2011. Web. 21 Jan 2021.

Vancouver:

Irons TD. Defining Behavioral Phenotypes Associated with Dopaminergic Nervous System Toxicity in Zebrafish Larvae. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:bcb10a8a-1d89-4bd6-b65e-080811a56696.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Irons TD. Defining Behavioral Phenotypes Associated with Dopaminergic Nervous System Toxicity in Zebrafish Larvae. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:bcb10a8a-1d89-4bd6-b65e-080811a56696

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

15. Troutman, John M. Molecular dosimetry of n7-guanine adducts in male and female mice exposed to 1,3-butadiene by inhalation.

Degree: 2011, University of North Carolina

 1,3-Butadiene (BD) is a well-characterized carcinogen that is both an occupational and environmental hazard and an important industrial chemical widely used in the production of… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Troutman, J. M. (2011). Molecular dosimetry of n7-guanine adducts in male and female mice exposed to 1,3-butadiene by inhalation. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:8eeb2252-e3b5-4caa-b5b8-9ee05c2af885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Troutman, John M. “Molecular dosimetry of n7-guanine adducts in male and female mice exposed to 1,3-butadiene by inhalation.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:8eeb2252-e3b5-4caa-b5b8-9ee05c2af885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Troutman, John M. “Molecular dosimetry of n7-guanine adducts in male and female mice exposed to 1,3-butadiene by inhalation.” 2011. Web. 21 Jan 2021.

Vancouver:

Troutman JM. Molecular dosimetry of n7-guanine adducts in male and female mice exposed to 1,3-butadiene by inhalation. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:8eeb2252-e3b5-4caa-b5b8-9ee05c2af885.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Troutman JM. Molecular dosimetry of n7-guanine adducts in male and female mice exposed to 1,3-butadiene by inhalation. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:8eeb2252-e3b5-4caa-b5b8-9ee05c2af885

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

16. Sproul, Christopher D. Biological Responses to Ultraviolet Radiation and the Effect of Intra-S Checkpoint Inhibition on Ultraviolet Radiation-Induced Mutagenesis.

Degree: 2013, University of North Carolina

 Sunlight emits a spectrum of electromagnetic radiation that includes ultraviolet, visible, and infrared wavelengths. It is widely accepted that the ultraviolet radiation (UVR) component is… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Sproul, C. D. (2013). Biological Responses to Ultraviolet Radiation and the Effect of Intra-S Checkpoint Inhibition on Ultraviolet Radiation-Induced Mutagenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:04393838-15df-43e6-a74f-9e026ef01dd8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sproul, Christopher D. “Biological Responses to Ultraviolet Radiation and the Effect of Intra-S Checkpoint Inhibition on Ultraviolet Radiation-Induced Mutagenesis.” 2013. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:04393838-15df-43e6-a74f-9e026ef01dd8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sproul, Christopher D. “Biological Responses to Ultraviolet Radiation and the Effect of Intra-S Checkpoint Inhibition on Ultraviolet Radiation-Induced Mutagenesis.” 2013. Web. 21 Jan 2021.

Vancouver:

Sproul CD. Biological Responses to Ultraviolet Radiation and the Effect of Intra-S Checkpoint Inhibition on Ultraviolet Radiation-Induced Mutagenesis. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:04393838-15df-43e6-a74f-9e026ef01dd8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sproul CD. Biological Responses to Ultraviolet Radiation and the Effect of Intra-S Checkpoint Inhibition on Ultraviolet Radiation-Induced Mutagenesis. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:04393838-15df-43e6-a74f-9e026ef01dd8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

17. Bauer, Rebecca N. The role of the inflammasome in the respiratory innate immune response to viruses and pollutants: insights for asthma pathogenesis.

Degree: 2014, University of North Carolina

 The respiratory mucosal innate immune system is composed of both structural cells, such as airway epithelial cells, and immune cells, such as macrophages, that together… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Bauer, R. N. (2014). The role of the inflammasome in the respiratory innate immune response to viruses and pollutants: insights for asthma pathogenesis. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:a5baa79b-e6d4-4950-ad16-f1207b8018c6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bauer, Rebecca N. “The role of the inflammasome in the respiratory innate immune response to viruses and pollutants: insights for asthma pathogenesis.” 2014. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:a5baa79b-e6d4-4950-ad16-f1207b8018c6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bauer, Rebecca N. “The role of the inflammasome in the respiratory innate immune response to viruses and pollutants: insights for asthma pathogenesis.” 2014. Web. 21 Jan 2021.

Vancouver:

Bauer RN. The role of the inflammasome in the respiratory innate immune response to viruses and pollutants: insights for asthma pathogenesis. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:a5baa79b-e6d4-4950-ad16-f1207b8018c6.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bauer RN. The role of the inflammasome in the respiratory innate immune response to viruses and pollutants: insights for asthma pathogenesis. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:a5baa79b-e6d4-4950-ad16-f1207b8018c6

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

18. Macon, Madisa. Mechanistic Pathways Underlying Low-Dose Perfluorooctanoic Acid (PFOA) Effects in Murine Mammary Tissues.

Degree: 2014, University of North Carolina

 Perfluorooctanoic acid (PFOA) is a synthetic surfactant that is as a prominent environmental toxicant. Previous studies have characterized the morphological effects of prenatal PFOA exposure… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Macon, M. (2014). Mechanistic Pathways Underlying Low-Dose Perfluorooctanoic Acid (PFOA) Effects in Murine Mammary Tissues. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:99cdf32b-1ce2-4ce5-ba02-7506de540d3f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Macon, Madisa. “Mechanistic Pathways Underlying Low-Dose Perfluorooctanoic Acid (PFOA) Effects in Murine Mammary Tissues.” 2014. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:99cdf32b-1ce2-4ce5-ba02-7506de540d3f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Macon, Madisa. “Mechanistic Pathways Underlying Low-Dose Perfluorooctanoic Acid (PFOA) Effects in Murine Mammary Tissues.” 2014. Web. 21 Jan 2021.

Vancouver:

Macon M. Mechanistic Pathways Underlying Low-Dose Perfluorooctanoic Acid (PFOA) Effects in Murine Mammary Tissues. [Internet] [Thesis]. University of North Carolina; 2014. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:99cdf32b-1ce2-4ce5-ba02-7506de540d3f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Macon M. Mechanistic Pathways Underlying Low-Dose Perfluorooctanoic Acid (PFOA) Effects in Murine Mammary Tissues. [Thesis]. University of North Carolina; 2014. Available from: https://cdr.lib.unc.edu/record/uuid:99cdf32b-1ce2-4ce5-ba02-7506de540d3f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

19. Szabo, David. TOXICOKINETICS AND EFFECTS OF 1,2,5,6,9,10-HEXABROMOCYCLODODECANE (HBCD) STEREOISOMERS IN MICE.

Degree: 2011, University of North Carolina

 Despite its small contribution to global production and usage of hexabromocyclododecane, α-HBCD is the major flame -retardant diastereomer found in environmental samples and human tissue.… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Szabo, D. (2011). TOXICOKINETICS AND EFFECTS OF 1,2,5,6,9,10-HEXABROMOCYCLODODECANE (HBCD) STEREOISOMERS IN MICE. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:4abc0883-76e2-46f9-835a-cd5ad50c126e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Szabo, David. “TOXICOKINETICS AND EFFECTS OF 1,2,5,6,9,10-HEXABROMOCYCLODODECANE (HBCD) STEREOISOMERS IN MICE.” 2011. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:4abc0883-76e2-46f9-835a-cd5ad50c126e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Szabo, David. “TOXICOKINETICS AND EFFECTS OF 1,2,5,6,9,10-HEXABROMOCYCLODODECANE (HBCD) STEREOISOMERS IN MICE.” 2011. Web. 21 Jan 2021.

Vancouver:

Szabo D. TOXICOKINETICS AND EFFECTS OF 1,2,5,6,9,10-HEXABROMOCYCLODODECANE (HBCD) STEREOISOMERS IN MICE. [Internet] [Thesis]. University of North Carolina; 2011. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:4abc0883-76e2-46f9-835a-cd5ad50c126e.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Szabo D. TOXICOKINETICS AND EFFECTS OF 1,2,5,6,9,10-HEXABROMOCYCLODODECANE (HBCD) STEREOISOMERS IN MICE. [Thesis]. University of North Carolina; 2011. Available from: https://cdr.lib.unc.edu/record/uuid:4abc0883-76e2-46f9-835a-cd5ad50c126e

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

20. Wages, Phillip. Peroxide dependent effects in human airway epithelial cells exposed to oxidant air pollutants.

Degree: 2016, University of North Carolina

 Air pollution is a global public health problem. The induction of oxidative stress, or the cellular response to avoid cytotoxicity due to an increase in… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Wages, P. (2016). Peroxide dependent effects in human airway epithelial cells exposed to oxidant air pollutants. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b542bdfd-91db-4656-810f-dc5fb1f899a3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wages, Phillip. “Peroxide dependent effects in human airway epithelial cells exposed to oxidant air pollutants.” 2016. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:b542bdfd-91db-4656-810f-dc5fb1f899a3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wages, Phillip. “Peroxide dependent effects in human airway epithelial cells exposed to oxidant air pollutants.” 2016. Web. 21 Jan 2021.

Vancouver:

Wages P. Peroxide dependent effects in human airway epithelial cells exposed to oxidant air pollutants. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:b542bdfd-91db-4656-810f-dc5fb1f899a3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wages P. Peroxide dependent effects in human airway epithelial cells exposed to oxidant air pollutants. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:b542bdfd-91db-4656-810f-dc5fb1f899a3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

21. Holman, Natalie. Investigating the role of hepatocyte-derived exosomes in drug-induced liver injury.

Degree: 2016, University of North Carolina

 Drug-induced liver injury (DILI) is the primary cause of acute liver failure in the United States and is responsible for a substantial number of drug… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Holman, N. (2016). Investigating the role of hepatocyte-derived exosomes in drug-induced liver injury. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:b23d5a8a-894a-4150-9ece-fa32359b7160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Holman, Natalie. “Investigating the role of hepatocyte-derived exosomes in drug-induced liver injury.” 2016. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:b23d5a8a-894a-4150-9ece-fa32359b7160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Holman, Natalie. “Investigating the role of hepatocyte-derived exosomes in drug-induced liver injury.” 2016. Web. 21 Jan 2021.

Vancouver:

Holman N. Investigating the role of hepatocyte-derived exosomes in drug-induced liver injury. [Internet] [Thesis]. University of North Carolina; 2016. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:b23d5a8a-894a-4150-9ece-fa32359b7160.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Holman N. Investigating the role of hepatocyte-derived exosomes in drug-induced liver injury. [Thesis]. University of North Carolina; 2016. Available from: https://cdr.lib.unc.edu/record/uuid:b23d5a8a-894a-4150-9ece-fa32359b7160

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

22. Suen, Alisa. ROLE OF THE SIX1 ONCOPROTEIN IN ENDOMETRIAL CANCER CAUSED BY NEONATAL XENOESTROGEN EXPOSURE.

Degree: 2017, University of North Carolina

 Exposure to xenoestrogens during key windows of development can lead to adverse female reproductive health outcomes, including cancer. In a classical model of latent hormonal… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Suen, A. (2017). ROLE OF THE SIX1 ONCOPROTEIN IN ENDOMETRIAL CANCER CAUSED BY NEONATAL XENOESTROGEN EXPOSURE. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:e3618968-6b80-4652-b735-a98ad7704d58

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suen, Alisa. “ROLE OF THE SIX1 ONCOPROTEIN IN ENDOMETRIAL CANCER CAUSED BY NEONATAL XENOESTROGEN EXPOSURE.” 2017. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:e3618968-6b80-4652-b735-a98ad7704d58.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suen, Alisa. “ROLE OF THE SIX1 ONCOPROTEIN IN ENDOMETRIAL CANCER CAUSED BY NEONATAL XENOESTROGEN EXPOSURE.” 2017. Web. 21 Jan 2021.

Vancouver:

Suen A. ROLE OF THE SIX1 ONCOPROTEIN IN ENDOMETRIAL CANCER CAUSED BY NEONATAL XENOESTROGEN EXPOSURE. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:e3618968-6b80-4652-b735-a98ad7704d58.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suen A. ROLE OF THE SIX1 ONCOPROTEIN IN ENDOMETRIAL CANCER CAUSED BY NEONATAL XENOESTROGEN EXPOSURE. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:e3618968-6b80-4652-b735-a98ad7704d58

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

23. Vaughan, Rachel. Biomarkers of Lung Epithelial Damage After Inhalation Injury.

Degree: 2017, University of North Carolina

 Burn and inhalation injury affects almost half a million people in the United States each year and it is one of the most complicated forms… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Vaughan, R. (2017). Biomarkers of Lung Epithelial Damage After Inhalation Injury. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:57d36a55-b30f-4874-8394-3115d5b83ef8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vaughan, Rachel. “Biomarkers of Lung Epithelial Damage After Inhalation Injury.” 2017. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:57d36a55-b30f-4874-8394-3115d5b83ef8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vaughan, Rachel. “Biomarkers of Lung Epithelial Damage After Inhalation Injury.” 2017. Web. 21 Jan 2021.

Vancouver:

Vaughan R. Biomarkers of Lung Epithelial Damage After Inhalation Injury. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:57d36a55-b30f-4874-8394-3115d5b83ef8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vaughan R. Biomarkers of Lung Epithelial Damage After Inhalation Injury. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:57d36a55-b30f-4874-8394-3115d5b83ef8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

24. Bowers, Emma. Harnessing Inter-Individual Variability to Identify Molecular Mechanisms Shaping Airway Epithelial Cell Transcriptional Responses to Ozone Exposure.

Degree: 2017, University of North Carolina

 Millions of people are exposed to levels of the ambient air pollutant ozone that are known to produce pulmonary inflammation; however, inflammatory responses exhibit extensive… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bowers, E. (2017). Harnessing Inter-Individual Variability to Identify Molecular Mechanisms Shaping Airway Epithelial Cell Transcriptional Responses to Ozone Exposure. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:32407efc-b9cd-4bb8-a5bc-6c11fd30afc5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bowers, Emma. “Harnessing Inter-Individual Variability to Identify Molecular Mechanisms Shaping Airway Epithelial Cell Transcriptional Responses to Ozone Exposure.” 2017. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:32407efc-b9cd-4bb8-a5bc-6c11fd30afc5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bowers, Emma. “Harnessing Inter-Individual Variability to Identify Molecular Mechanisms Shaping Airway Epithelial Cell Transcriptional Responses to Ozone Exposure.” 2017. Web. 21 Jan 2021.

Vancouver:

Bowers E. Harnessing Inter-Individual Variability to Identify Molecular Mechanisms Shaping Airway Epithelial Cell Transcriptional Responses to Ozone Exposure. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:32407efc-b9cd-4bb8-a5bc-6c11fd30afc5.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bowers E. Harnessing Inter-Individual Variability to Identify Molecular Mechanisms Shaping Airway Epithelial Cell Transcriptional Responses to Ozone Exposure. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:32407efc-b9cd-4bb8-a5bc-6c11fd30afc5

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

25. Henriquez Coria, Andres. OZONE-INDUCED LUNG INJURY AND INFLAMMATION ARE MODULATED BY CIRCULATING STRESS HORMONES.

Degree: 2017, University of North Carolina

 Air pollution has been associated with a wide-range of health effects. Pulmonary effects of acute ozone inhalation have been widely characterized in humans and animals.… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Henriquez Coria, A. (2017). OZONE-INDUCED LUNG INJURY AND INFLAMMATION ARE MODULATED BY CIRCULATING STRESS HORMONES. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:8d81df74-fe9f-46ed-b00d-3cd2efe6fdd3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Henriquez Coria, Andres. “OZONE-INDUCED LUNG INJURY AND INFLAMMATION ARE MODULATED BY CIRCULATING STRESS HORMONES.” 2017. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:8d81df74-fe9f-46ed-b00d-3cd2efe6fdd3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Henriquez Coria, Andres. “OZONE-INDUCED LUNG INJURY AND INFLAMMATION ARE MODULATED BY CIRCULATING STRESS HORMONES.” 2017. Web. 21 Jan 2021.

Vancouver:

Henriquez Coria A. OZONE-INDUCED LUNG INJURY AND INFLAMMATION ARE MODULATED BY CIRCULATING STRESS HORMONES. [Internet] [Thesis]. University of North Carolina; 2017. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:8d81df74-fe9f-46ed-b00d-3cd2efe6fdd3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Henriquez Coria A. OZONE-INDUCED LUNG INJURY AND INFLAMMATION ARE MODULATED BY CIRCULATING STRESS HORMONES. [Thesis]. University of North Carolina; 2017. Available from: https://cdr.lib.unc.edu/record/uuid:8d81df74-fe9f-46ed-b00d-3cd2efe6fdd3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

26. Jones, Shannon Zenia. The Identification of Novel Mechanisms to Regulate B cell Responses During Adaptive Immunity.

Degree: 2012, University of North Carolina

 Initiation of the germinal center reaction during T-dependent adaptive immune responses gives rise to long-lived plasma cells (PCs) that produce high affinity, class switched antibodies.… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jones, S. Z. (2012). The Identification of Novel Mechanisms to Regulate B cell Responses During Adaptive Immunity. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:574f1a68-0b2c-4abf-b2ba-04f861ad2c68

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jones, Shannon Zenia. “The Identification of Novel Mechanisms to Regulate B cell Responses During Adaptive Immunity.” 2012. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:574f1a68-0b2c-4abf-b2ba-04f861ad2c68.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jones, Shannon Zenia. “The Identification of Novel Mechanisms to Regulate B cell Responses During Adaptive Immunity.” 2012. Web. 21 Jan 2021.

Vancouver:

Jones SZ. The Identification of Novel Mechanisms to Regulate B cell Responses During Adaptive Immunity. [Internet] [Thesis]. University of North Carolina; 2012. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:574f1a68-0b2c-4abf-b2ba-04f861ad2c68.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jones SZ. The Identification of Novel Mechanisms to Regulate B cell Responses During Adaptive Immunity. [Thesis]. University of North Carolina; 2012. Available from: https://cdr.lib.unc.edu/record/uuid:574f1a68-0b2c-4abf-b2ba-04f861ad2c68

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

27. Gibbs-Flournoy, Eugene. Examination of the Adverse Effects of Exposure to Gaseous and Particulate Oxidant Air Pollutants in Human Airway Epithelial Cells.

Degree: 2013, University of North Carolina

 Human exposure to ambient air pollution is a pervasive global public health problem. Ambient levels of air pollutants, such as particulate matter and ozone, are… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gibbs-Flournoy, E. (2013). Examination of the Adverse Effects of Exposure to Gaseous and Particulate Oxidant Air Pollutants in Human Airway Epithelial Cells. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:c35e6feb-4fcb-47a3-afdb-70ed6164466f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gibbs-Flournoy, Eugene. “Examination of the Adverse Effects of Exposure to Gaseous and Particulate Oxidant Air Pollutants in Human Airway Epithelial Cells.” 2013. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:c35e6feb-4fcb-47a3-afdb-70ed6164466f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gibbs-Flournoy, Eugene. “Examination of the Adverse Effects of Exposure to Gaseous and Particulate Oxidant Air Pollutants in Human Airway Epithelial Cells.” 2013. Web. 21 Jan 2021.

Vancouver:

Gibbs-Flournoy E. Examination of the Adverse Effects of Exposure to Gaseous and Particulate Oxidant Air Pollutants in Human Airway Epithelial Cells. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:c35e6feb-4fcb-47a3-afdb-70ed6164466f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gibbs-Flournoy E. Examination of the Adverse Effects of Exposure to Gaseous and Particulate Oxidant Air Pollutants in Human Airway Epithelial Cells. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:c35e6feb-4fcb-47a3-afdb-70ed6164466f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

28. Snow, Samantha Jean. Ultrafine Particles Alter Endothelial Phenotype Through Oxidant Signaling.

Degree: 2013, University of North Carolina

 Mechanisms that underlie the strong association between air pollution exposure and cardiovascular (CV) morbidity and mortality remain unknown. Particulate matter (PM) is a major component… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Snow, S. J. (2013). Ultrafine Particles Alter Endothelial Phenotype Through Oxidant Signaling. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:9b309ef2-a0e8-4e11-bd0e-3fce7631a807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Snow, Samantha Jean. “Ultrafine Particles Alter Endothelial Phenotype Through Oxidant Signaling.” 2013. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:9b309ef2-a0e8-4e11-bd0e-3fce7631a807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Snow, Samantha Jean. “Ultrafine Particles Alter Endothelial Phenotype Through Oxidant Signaling.” 2013. Web. 21 Jan 2021.

Vancouver:

Snow SJ. Ultrafine Particles Alter Endothelial Phenotype Through Oxidant Signaling. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:9b309ef2-a0e8-4e11-bd0e-3fce7631a807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Snow SJ. Ultrafine Particles Alter Endothelial Phenotype Through Oxidant Signaling. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:9b309ef2-a0e8-4e11-bd0e-3fce7631a807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

29. Perez, Christina Margaret. The Role of Hypoxia in Air Pollutant-Induced Cardiovascular Dysfunction.

Degree: 2013, University of North Carolina

 Diesel exhaust (DE) is a major contributor to traffic-related urban air pollution and has been associated with cardiovascular dysfunction in humans, especially susceptible individuals. DE… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Perez, C. M. (2013). The Role of Hypoxia in Air Pollutant-Induced Cardiovascular Dysfunction. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:2f503bb3-78a0-454f-aa1e-aa00d6e1ed7f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Perez, Christina Margaret. “The Role of Hypoxia in Air Pollutant-Induced Cardiovascular Dysfunction.” 2013. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:2f503bb3-78a0-454f-aa1e-aa00d6e1ed7f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Perez, Christina Margaret. “The Role of Hypoxia in Air Pollutant-Induced Cardiovascular Dysfunction.” 2013. Web. 21 Jan 2021.

Vancouver:

Perez CM. The Role of Hypoxia in Air Pollutant-Induced Cardiovascular Dysfunction. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:2f503bb3-78a0-454f-aa1e-aa00d6e1ed7f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Perez CM. The Role of Hypoxia in Air Pollutant-Induced Cardiovascular Dysfunction. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:2f503bb3-78a0-454f-aa1e-aa00d6e1ed7f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of North Carolina

30. Currier, Jenna. Novel Biomarkers for the Risk Assessment of Exposure to Arsenic in Drinking Water.

Degree: 2013, University of North Carolina

 The toxic methylated trivalent metabolites of inorganic arsenic (iAs), methylarsonite (MAsIII) and dimethylarsinite (DMAsIII), play an important role in the etiology of As-induced diseases, including… (more)

Subjects/Keywords: School of Medicine; Curriculum in Toxicology

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APA (6th Edition):

Currier, J. (2013). Novel Biomarkers for the Risk Assessment of Exposure to Arsenic in Drinking Water. (Thesis). University of North Carolina. Retrieved from https://cdr.lib.unc.edu/record/uuid:ca54006e-7b76-4da5-8579-5e2bbe21b42a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Currier, Jenna. “Novel Biomarkers for the Risk Assessment of Exposure to Arsenic in Drinking Water.” 2013. Thesis, University of North Carolina. Accessed January 21, 2021. https://cdr.lib.unc.edu/record/uuid:ca54006e-7b76-4da5-8579-5e2bbe21b42a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Currier, Jenna. “Novel Biomarkers for the Risk Assessment of Exposure to Arsenic in Drinking Water.” 2013. Web. 21 Jan 2021.

Vancouver:

Currier J. Novel Biomarkers for the Risk Assessment of Exposure to Arsenic in Drinking Water. [Internet] [Thesis]. University of North Carolina; 2013. [cited 2021 Jan 21]. Available from: https://cdr.lib.unc.edu/record/uuid:ca54006e-7b76-4da5-8579-5e2bbe21b42a.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Currier J. Novel Biomarkers for the Risk Assessment of Exposure to Arsenic in Drinking Water. [Thesis]. University of North Carolina; 2013. Available from: https://cdr.lib.unc.edu/record/uuid:ca54006e-7b76-4da5-8579-5e2bbe21b42a

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3]

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