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You searched for subject:(Cuprizone). Showing records 1 – 21 of 21 total matches.

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Victoria University of Wellington

1. Templeton, Nicola. The effect of clozapine and MIS416 on demyelination, remyelination and immunomodulation in the murine cuprizone model.

Degree: 2017, Victoria University of Wellington

 Multiple Sclerosis (MS) is a disorder of the central nervous system that affects approximately 2.5 million people worldwide. Due to the heterogeneous nature of the… (more)

Subjects/Keywords: Multiple sclerosis; Cuprizone; Clozapine; Myelin

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APA (6th Edition):

Templeton, N. (2017). The effect of clozapine and MIS416 on demyelination, remyelination and immunomodulation in the murine cuprizone model. (Doctoral Dissertation). Victoria University of Wellington. Retrieved from http://hdl.handle.net/10063/6439

Chicago Manual of Style (16th Edition):

Templeton, Nicola. “The effect of clozapine and MIS416 on demyelination, remyelination and immunomodulation in the murine cuprizone model.” 2017. Doctoral Dissertation, Victoria University of Wellington. Accessed February 28, 2021. http://hdl.handle.net/10063/6439.

MLA Handbook (7th Edition):

Templeton, Nicola. “The effect of clozapine and MIS416 on demyelination, remyelination and immunomodulation in the murine cuprizone model.” 2017. Web. 28 Feb 2021.

Vancouver:

Templeton N. The effect of clozapine and MIS416 on demyelination, remyelination and immunomodulation in the murine cuprizone model. [Internet] [Doctoral dissertation]. Victoria University of Wellington; 2017. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10063/6439.

Council of Science Editors:

Templeton N. The effect of clozapine and MIS416 on demyelination, remyelination and immunomodulation in the murine cuprizone model. [Doctoral Dissertation]. Victoria University of Wellington; 2017. Available from: http://hdl.handle.net/10063/6439


University of Rochester

2. Szymaniak, Brittany M. Loss of ATM Induces Glial Cell Dysfunction in a Novel Murine Model of Ataxia-telangiectasia.

Degree: PhD, 2016, University of Rochester

 Ataxia-telangiectasia (A-T) is a rare, recessive, pediatric disorder that is primarily characterized by cerebellar degeneration of Purkinje neurons and results from mutations in the Ataxia-telangiectasia… (more)

Subjects/Keywords: ATM; Mouse model; Ataxia-telangiectasia; Cuprizone

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APA (6th Edition):

Szymaniak, B. M. (2016). Loss of ATM Induces Glial Cell Dysfunction in a Novel Murine Model of Ataxia-telangiectasia. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/31655

Chicago Manual of Style (16th Edition):

Szymaniak, Brittany M. “Loss of ATM Induces Glial Cell Dysfunction in a Novel Murine Model of Ataxia-telangiectasia.” 2016. Doctoral Dissertation, University of Rochester. Accessed February 28, 2021. http://hdl.handle.net/1802/31655.

MLA Handbook (7th Edition):

Szymaniak, Brittany M. “Loss of ATM Induces Glial Cell Dysfunction in a Novel Murine Model of Ataxia-telangiectasia.” 2016. Web. 28 Feb 2021.

Vancouver:

Szymaniak BM. Loss of ATM Induces Glial Cell Dysfunction in a Novel Murine Model of Ataxia-telangiectasia. [Internet] [Doctoral dissertation]. University of Rochester; 2016. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1802/31655.

Council of Science Editors:

Szymaniak BM. Loss of ATM Induces Glial Cell Dysfunction in a Novel Murine Model of Ataxia-telangiectasia. [Doctoral Dissertation]. University of Rochester; 2016. Available from: http://hdl.handle.net/1802/31655


University of Saskatchewan

3. Mooshekhian, Ali 1979-. Effects of Low Field Magnetic Stimulation on.

Degree: 2019, University of Saskatchewan

 Cognitive impairment (CI) is a leading cause of disability in patients with Multiple Sclerosis (MS). Although CI has catastrophic effects on patients’ quality of life,… (more)

Subjects/Keywords: Demyelination; Multiple Sclerosis; Cuprizone; Cognitive deficit

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APA (6th Edition):

Mooshekhian, A. 1. (2019). Effects of Low Field Magnetic Stimulation on. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/13084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mooshekhian, Ali 1979-. “Effects of Low Field Magnetic Stimulation on.” 2019. Thesis, University of Saskatchewan. Accessed February 28, 2021. http://hdl.handle.net/10388/13084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mooshekhian, Ali 1979-. “Effects of Low Field Magnetic Stimulation on.” 2019. Web. 28 Feb 2021.

Vancouver:

Mooshekhian A1. Effects of Low Field Magnetic Stimulation on. [Internet] [Thesis]. University of Saskatchewan; 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10388/13084.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mooshekhian A1. Effects of Low Field Magnetic Stimulation on. [Thesis]. University of Saskatchewan; 2019. Available from: http://hdl.handle.net/10388/13084

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manitoba

4. Palmer, Vanessa Leanne. Understanding white matter pathology through correlating longitudinal and quantitative MRI metrics weekly in the cuprizone mouse model of demyelination.

Degree: Biomedical Engineering, 2016, University of Manitoba

 Magnetic resonance imaging (MRI) methods thought to assess myelin and axon integrity are improving the understanding of white matter diseases like multiple sclerosis (MS). This… (more)

Subjects/Keywords: MRI; Electron Microscopy; Cuprizone mouse model; White matter

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APA (6th Edition):

Palmer, V. L. (2016). Understanding white matter pathology through correlating longitudinal and quantitative MRI metrics weekly in the cuprizone mouse model of demyelination. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/31207

Chicago Manual of Style (16th Edition):

Palmer, Vanessa Leanne. “Understanding white matter pathology through correlating longitudinal and quantitative MRI metrics weekly in the cuprizone mouse model of demyelination.” 2016. Masters Thesis, University of Manitoba. Accessed February 28, 2021. http://hdl.handle.net/1993/31207.

MLA Handbook (7th Edition):

Palmer, Vanessa Leanne. “Understanding white matter pathology through correlating longitudinal and quantitative MRI metrics weekly in the cuprizone mouse model of demyelination.” 2016. Web. 28 Feb 2021.

Vancouver:

Palmer VL. Understanding white matter pathology through correlating longitudinal and quantitative MRI metrics weekly in the cuprizone mouse model of demyelination. [Internet] [Masters thesis]. University of Manitoba; 2016. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1993/31207.

Council of Science Editors:

Palmer VL. Understanding white matter pathology through correlating longitudinal and quantitative MRI metrics weekly in the cuprizone mouse model of demyelination. [Masters Thesis]. University of Manitoba; 2016. Available from: http://hdl.handle.net/1993/31207


University of Melbourne

5. Xing, Yao Lulu. Investigation of the relative contribution of neural precursor cells and oligodendrocyte progenitor cells to myelin repair.

Degree: 2015, University of Melbourne

 Remyelination following demyelination of the central nervous system is mediated via the generation of new myelin-forming oligodendrocytes. It has been demonstrated that both parenchymal oligodendrocyte… (more)

Subjects/Keywords: neural precursor cells; oligodendrocyte progenitor cells; remyelination; multiple sclerosis; cuprizone

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APA (6th Edition):

Xing, Y. L. (2015). Investigation of the relative contribution of neural precursor cells and oligodendrocyte progenitor cells to myelin repair. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/59457

Chicago Manual of Style (16th Edition):

Xing, Yao Lulu. “Investigation of the relative contribution of neural precursor cells and oligodendrocyte progenitor cells to myelin repair.” 2015. Doctoral Dissertation, University of Melbourne. Accessed February 28, 2021. http://hdl.handle.net/11343/59457.

MLA Handbook (7th Edition):

Xing, Yao Lulu. “Investigation of the relative contribution of neural precursor cells and oligodendrocyte progenitor cells to myelin repair.” 2015. Web. 28 Feb 2021.

Vancouver:

Xing YL. Investigation of the relative contribution of neural precursor cells and oligodendrocyte progenitor cells to myelin repair. [Internet] [Doctoral dissertation]. University of Melbourne; 2015. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11343/59457.

Council of Science Editors:

Xing YL. Investigation of the relative contribution of neural precursor cells and oligodendrocyte progenitor cells to myelin repair. [Doctoral Dissertation]. University of Melbourne; 2015. Available from: http://hdl.handle.net/11343/59457


Kent State University

6. Shelestak, John Wesley. EVALUATION OF BLOOD-BRAIN BARRIER INTEGRITY UNDER CUPRIZONE ADMINISTRATION.

Degree: PhD, College of Arts and Sciences / School of Biomedical Sciences, 2019, Kent State University

 The blood-brain barrier is an extremely important protective mechanism for the central nervous system. The blood-brain barrier is often damaged in neurodegenerative diseases and understanding… (more)

Subjects/Keywords: Biology; Biomedical Research; Neurobiology; Blood-Brain Barrier; Cuprizone; Demyelination; Neurodegeneration; Neuroimmunology

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APA (6th Edition):

Shelestak, J. W. (2019). EVALUATION OF BLOOD-BRAIN BARRIER INTEGRITY UNDER CUPRIZONE ADMINISTRATION. (Doctoral Dissertation). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1574351981114558

Chicago Manual of Style (16th Edition):

Shelestak, John Wesley. “EVALUATION OF BLOOD-BRAIN BARRIER INTEGRITY UNDER CUPRIZONE ADMINISTRATION.” 2019. Doctoral Dissertation, Kent State University. Accessed February 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=kent1574351981114558.

MLA Handbook (7th Edition):

Shelestak, John Wesley. “EVALUATION OF BLOOD-BRAIN BARRIER INTEGRITY UNDER CUPRIZONE ADMINISTRATION.” 2019. Web. 28 Feb 2021.

Vancouver:

Shelestak JW. EVALUATION OF BLOOD-BRAIN BARRIER INTEGRITY UNDER CUPRIZONE ADMINISTRATION. [Internet] [Doctoral dissertation]. Kent State University; 2019. [cited 2021 Feb 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1574351981114558.

Council of Science Editors:

Shelestak JW. EVALUATION OF BLOOD-BRAIN BARRIER INTEGRITY UNDER CUPRIZONE ADMINISTRATION. [Doctoral Dissertation]. Kent State University; 2019. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1574351981114558

7. Prevost, Valentin. Validation du transfert d'aimantation inhomogène (ihMT) comme nouveau biomarqueur IRM de la myéline : Validation of inhomogeneous magnetization transfer (ihMT) as a new MRI myelin biomarker.

Degree: Docteur es, Neurosciences, 2018, Aix Marseille Université

L’imagerie par résonance magnétique (IRM) est une technique d’imagerie médicale largement utilisée pour son caractère non-invasif et pour sa capacité à explorer les tissus mous.… (more)

Subjects/Keywords: Imagerie; Irm; Myéline; IhMT; Transfert d'aimantation; Inhomogène; Modèle; Cuprizone; Validation; Biomarqueur; Imaging; Mri; Myelin; IhMT; Magnetization transfer; Inhomogeneous; Model; Cuprizone; Validation; Biomarker

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APA (6th Edition):

Prevost, V. (2018). Validation du transfert d'aimantation inhomogène (ihMT) comme nouveau biomarqueur IRM de la myéline : Validation of inhomogeneous magnetization transfer (ihMT) as a new MRI myelin biomarker. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2018AIXM0037

Chicago Manual of Style (16th Edition):

Prevost, Valentin. “Validation du transfert d'aimantation inhomogène (ihMT) comme nouveau biomarqueur IRM de la myéline : Validation of inhomogeneous magnetization transfer (ihMT) as a new MRI myelin biomarker.” 2018. Doctoral Dissertation, Aix Marseille Université. Accessed February 28, 2021. http://www.theses.fr/2018AIXM0037.

MLA Handbook (7th Edition):

Prevost, Valentin. “Validation du transfert d'aimantation inhomogène (ihMT) comme nouveau biomarqueur IRM de la myéline : Validation of inhomogeneous magnetization transfer (ihMT) as a new MRI myelin biomarker.” 2018. Web. 28 Feb 2021.

Vancouver:

Prevost V. Validation du transfert d'aimantation inhomogène (ihMT) comme nouveau biomarqueur IRM de la myéline : Validation of inhomogeneous magnetization transfer (ihMT) as a new MRI myelin biomarker. [Internet] [Doctoral dissertation]. Aix Marseille Université 2018. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2018AIXM0037.

Council of Science Editors:

Prevost V. Validation du transfert d'aimantation inhomogène (ihMT) comme nouveau biomarqueur IRM de la myéline : Validation of inhomogeneous magnetization transfer (ihMT) as a new MRI myelin biomarker. [Doctoral Dissertation]. Aix Marseille Université 2018. Available from: http://www.theses.fr/2018AIXM0037

8. Becquet, Laurine. Evaluation des effets thérapeutiques de neuropeptides contre la sclérose en plaques : les orexines, le vasoactive intestinal peptide, le pituitary adenylate cyclase-activating polypeptide et leurs analogues : Therapeutic effects of VIP, PACAP, orexins and their analogs in experimental models of multiple sclerosis.

Degree: Docteur es, Sciences de la vie et de la sante, 2018, Normandie

 La sclérose en plaques (SEP) est une maladie autoimmune inflammatoire et neurodégénérative du système nerveux central (SNC) chez le jeune adulte résultant d’une altération ciblée… (more)

Subjects/Keywords: Agoniste de VPAC2; Cuprizone; Encéphalomyélite autoimmune expérimentale; Orexine A; Sclérose en plaques; Neuropeptides; Cuprizone; Experimental autoimmune encephalomyelitis; Orexin A; Multiple sclerosis; Neuropeptides; VPAC2 agonist; 615.78

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APA (6th Edition):

Becquet, L. (2018). Evaluation des effets thérapeutiques de neuropeptides contre la sclérose en plaques : les orexines, le vasoactive intestinal peptide, le pituitary adenylate cyclase-activating polypeptide et leurs analogues : Therapeutic effects of VIP, PACAP, orexins and their analogs in experimental models of multiple sclerosis. (Doctoral Dissertation). Normandie. Retrieved from http://www.theses.fr/2018NORMR071

Chicago Manual of Style (16th Edition):

Becquet, Laurine. “Evaluation des effets thérapeutiques de neuropeptides contre la sclérose en plaques : les orexines, le vasoactive intestinal peptide, le pituitary adenylate cyclase-activating polypeptide et leurs analogues : Therapeutic effects of VIP, PACAP, orexins and their analogs in experimental models of multiple sclerosis.” 2018. Doctoral Dissertation, Normandie. Accessed February 28, 2021. http://www.theses.fr/2018NORMR071.

MLA Handbook (7th Edition):

Becquet, Laurine. “Evaluation des effets thérapeutiques de neuropeptides contre la sclérose en plaques : les orexines, le vasoactive intestinal peptide, le pituitary adenylate cyclase-activating polypeptide et leurs analogues : Therapeutic effects of VIP, PACAP, orexins and their analogs in experimental models of multiple sclerosis.” 2018. Web. 28 Feb 2021.

Vancouver:

Becquet L. Evaluation des effets thérapeutiques de neuropeptides contre la sclérose en plaques : les orexines, le vasoactive intestinal peptide, le pituitary adenylate cyclase-activating polypeptide et leurs analogues : Therapeutic effects of VIP, PACAP, orexins and their analogs in experimental models of multiple sclerosis. [Internet] [Doctoral dissertation]. Normandie; 2018. [cited 2021 Feb 28]. Available from: http://www.theses.fr/2018NORMR071.

Council of Science Editors:

Becquet L. Evaluation des effets thérapeutiques de neuropeptides contre la sclérose en plaques : les orexines, le vasoactive intestinal peptide, le pituitary adenylate cyclase-activating polypeptide et leurs analogues : Therapeutic effects of VIP, PACAP, orexins and their analogs in experimental models of multiple sclerosis. [Doctoral Dissertation]. Normandie; 2018. Available from: http://www.theses.fr/2018NORMR071


UCLA

9. Kim, Youn-Jung Roy. Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands.

Degree: Molec, Cell, & Integ Physiology, 2018, UCLA

 Currently, available drugs for multiple sclerosis (MS) are predominantly immune-modulatory. They are effective in reducing relapses, but not in slowing down or stopping progressive disease… (more)

Subjects/Keywords: Neurosciences; Physiology; Cuprizone; Estrogen Receptor Beta Ligand; Experimental Autoimmune Encephalomyelitis; Multiple Sclerosis; Myeloid cells; Oligodendrocytes

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APA (6th Edition):

Kim, Y. R. (2018). Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/09j2798f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kim, Youn-Jung Roy. “Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands.” 2018. Thesis, UCLA. Accessed February 28, 2021. http://www.escholarship.org/uc/item/09j2798f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kim, Youn-Jung Roy. “Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands.” 2018. Web. 28 Feb 2021.

Vancouver:

Kim YR. Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands. [Internet] [Thesis]. UCLA; 2018. [cited 2021 Feb 28]. Available from: http://www.escholarship.org/uc/item/09j2798f.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kim YR. Mechanisms of Neuroprotection and Remyelination in Demyelinating Disease Models of Multiple Sclerosis: A Lesson From Estrogen Receptor Specific Ligands. [Thesis]. UCLA; 2018. Available from: http://www.escholarship.org/uc/item/09j2798f

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


McMaster University

10. Moldovan, Nataliya. MEASUREMENT OF TRANSITION METALS IN THE RODENT BRAIN USING X-RAY FLUORESCENCE AND NEUTRON ACTIVATION ANALYSIS.

Degree: MSc, 2012, McMaster University

Transition metals, such as iron, manganese, and copper are essential in the development and function of biological systems. However, disrupted levels of transition metals… (more)

Subjects/Keywords: transition metal homeostasis; cuprizone; manganese enhanced MRI; manganese overexposure; demyelination; x-ray fluorescence; neutron activation analysis; Medical Biophysics; Medical Biophysics

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APA (6th Edition):

Moldovan, N. (2012). MEASUREMENT OF TRANSITION METALS IN THE RODENT BRAIN USING X-RAY FLUORESCENCE AND NEUTRON ACTIVATION ANALYSIS. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/12372

Chicago Manual of Style (16th Edition):

Moldovan, Nataliya. “MEASUREMENT OF TRANSITION METALS IN THE RODENT BRAIN USING X-RAY FLUORESCENCE AND NEUTRON ACTIVATION ANALYSIS.” 2012. Masters Thesis, McMaster University. Accessed February 28, 2021. http://hdl.handle.net/11375/12372.

MLA Handbook (7th Edition):

Moldovan, Nataliya. “MEASUREMENT OF TRANSITION METALS IN THE RODENT BRAIN USING X-RAY FLUORESCENCE AND NEUTRON ACTIVATION ANALYSIS.” 2012. Web. 28 Feb 2021.

Vancouver:

Moldovan N. MEASUREMENT OF TRANSITION METALS IN THE RODENT BRAIN USING X-RAY FLUORESCENCE AND NEUTRON ACTIVATION ANALYSIS. [Internet] [Masters thesis]. McMaster University; 2012. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/11375/12372.

Council of Science Editors:

Moldovan N. MEASUREMENT OF TRANSITION METALS IN THE RODENT BRAIN USING X-RAY FLUORESCENCE AND NEUTRON ACTIVATION ANALYSIS. [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/12372

11. Zhang, Lin. The effect of galanin alone and combined with estrogen in a cuprizone-induced demyelination mouse model of multiple sclerosis.

Degree: Human Anatomy and Cell Science, 2011, University of Manitoba

 Multiple Sclerosis (MS) is an autoimmune demyelination disease of the central nervous system (CNS). The traditional immunotherapies so far have not been able to stop… (more)

Subjects/Keywords: Multiple sclerosis; galanin; estrogen; cuprizone; demyelination; remyelination

…Abbreviations 12wCPZ twelve-week cuprizone (challenge) 12wCPZ+3wR twelve-week cuprizone… …x28;challenge) plus three-week recovery 6wCPZ six-week cuprizone (challenge)… …6wCPZ+3wR six-week cuprizone (challenge) plus three-week recovery aco anterior… …callosum cing cingulum bundle CNS central nervous system CP caudoputamen CPZ cuprizone… 

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APA (6th Edition):

Zhang, L. (2011). The effect of galanin alone and combined with estrogen in a cuprizone-induced demyelination mouse model of multiple sclerosis. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4749

Chicago Manual of Style (16th Edition):

Zhang, Lin. “The effect of galanin alone and combined with estrogen in a cuprizone-induced demyelination mouse model of multiple sclerosis.” 2011. Masters Thesis, University of Manitoba. Accessed February 28, 2021. http://hdl.handle.net/1993/4749.

MLA Handbook (7th Edition):

Zhang, Lin. “The effect of galanin alone and combined with estrogen in a cuprizone-induced demyelination mouse model of multiple sclerosis.” 2011. Web. 28 Feb 2021.

Vancouver:

Zhang L. The effect of galanin alone and combined with estrogen in a cuprizone-induced demyelination mouse model of multiple sclerosis. [Internet] [Masters thesis]. University of Manitoba; 2011. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1993/4749.

Council of Science Editors:

Zhang L. The effect of galanin alone and combined with estrogen in a cuprizone-induced demyelination mouse model of multiple sclerosis. [Masters Thesis]. University of Manitoba; 2011. Available from: http://hdl.handle.net/1993/4749


University of Akron

12. Ray, Lucille Alexandria. Live single cell fluorescence microscopy; from antibiotic resistance detection to mitochondrial dysfunction.

Degree: PhD, Chemistry, 2020, University of Akron

 Single-cell fluorescence microscopy is a powerful tool which can be used to investigate the nature of cellular responses to external stimuli. The application of single-cell… (more)

Subjects/Keywords: Biochemistry; Chemistry; Microbiology; microscopy; single-cell; antibiotic resistance; fluorescence; mitochondria; cuprizone; antibacterial susceptibility testing; antimicrobial polymer

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APA (6th Edition):

Ray, L. A. (2020). Live single cell fluorescence microscopy; from antibiotic resistance detection to mitochondrial dysfunction. (Doctoral Dissertation). University of Akron. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=akron1597342775751888

Chicago Manual of Style (16th Edition):

Ray, Lucille Alexandria. “Live single cell fluorescence microscopy; from antibiotic resistance detection to mitochondrial dysfunction.” 2020. Doctoral Dissertation, University of Akron. Accessed February 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=akron1597342775751888.

MLA Handbook (7th Edition):

Ray, Lucille Alexandria. “Live single cell fluorescence microscopy; from antibiotic resistance detection to mitochondrial dysfunction.” 2020. Web. 28 Feb 2021.

Vancouver:

Ray LA. Live single cell fluorescence microscopy; from antibiotic resistance detection to mitochondrial dysfunction. [Internet] [Doctoral dissertation]. University of Akron; 2020. [cited 2021 Feb 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=akron1597342775751888.

Council of Science Editors:

Ray LA. Live single cell fluorescence microscopy; from antibiotic resistance detection to mitochondrial dysfunction. [Doctoral Dissertation]. University of Akron; 2020. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=akron1597342775751888

13. Taraboletti, Alexandra Anna. Chemical and Metabolomic Analyses of Cuprizone-Induced Demyelination and Remyelination.

Degree: PhD, Chemistry, 2017, University of Akron

 The cuprizone intoxication model of demyelination and remyelination has long been used to test myelin regenerative therapies for neurodegenerative diseases such as multiple sclerosis. Mice… (more)

Subjects/Keywords: Biochemistry; Bioinformatics; Cellular Biology; Chemistry; metabolomics; cuprizone; multiple sclerosis; copper; transcriptomics; neurodegenerative; oligodendrocytes; myelin; rapamycin; mTOR; BCAA; TDO; demyelination; remyelination

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APA (6th Edition):

Taraboletti, A. A. (2017). Chemical and Metabolomic Analyses of Cuprizone-Induced Demyelination and Remyelination. (Doctoral Dissertation). University of Akron. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=akron1498535047689141

Chicago Manual of Style (16th Edition):

Taraboletti, Alexandra Anna. “Chemical and Metabolomic Analyses of Cuprizone-Induced Demyelination and Remyelination.” 2017. Doctoral Dissertation, University of Akron. Accessed February 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=akron1498535047689141.

MLA Handbook (7th Edition):

Taraboletti, Alexandra Anna. “Chemical and Metabolomic Analyses of Cuprizone-Induced Demyelination and Remyelination.” 2017. Web. 28 Feb 2021.

Vancouver:

Taraboletti AA. Chemical and Metabolomic Analyses of Cuprizone-Induced Demyelination and Remyelination. [Internet] [Doctoral dissertation]. University of Akron; 2017. [cited 2021 Feb 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=akron1498535047689141.

Council of Science Editors:

Taraboletti AA. Chemical and Metabolomic Analyses of Cuprizone-Induced Demyelination and Remyelination. [Doctoral Dissertation]. University of Akron; 2017. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=akron1498535047689141

14. Sen, Monokesh. An investigation of the effect of cuprizone-induced demyelination using histological, behavioural, proteomics and immunological approaches.

Degree: 2019, Western Sydney University

 Feeding cuprizone (CPZ) to animals has been long known to lead to regional degeneration of oligodendrocytes (OLG) and has been used to model the demyelinating… (more)

Subjects/Keywords: multiple sclerosis; demyelination; diseases; causes and theories of causation; proteomics; bioinformatics; mitochondria; cuprizone; Thesis (Ph.D.) – Western Sydney University, 2019

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sen, M. (2019). An investigation of the effect of cuprizone-induced demyelination using histological, behavioural, proteomics and immunological approaches. (Thesis). Western Sydney University. Retrieved from http://hdl.handle.net/1959.7/uws:58405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sen, Monokesh. “An investigation of the effect of cuprizone-induced demyelination using histological, behavioural, proteomics and immunological approaches.” 2019. Thesis, Western Sydney University. Accessed February 28, 2021. http://hdl.handle.net/1959.7/uws:58405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sen, Monokesh. “An investigation of the effect of cuprizone-induced demyelination using histological, behavioural, proteomics and immunological approaches.” 2019. Web. 28 Feb 2021.

Vancouver:

Sen M. An investigation of the effect of cuprizone-induced demyelination using histological, behavioural, proteomics and immunological approaches. [Internet] [Thesis]. Western Sydney University; 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1959.7/uws:58405.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sen M. An investigation of the effect of cuprizone-induced demyelination using histological, behavioural, proteomics and immunological approaches. [Thesis]. Western Sydney University; 2019. Available from: http://hdl.handle.net/1959.7/uws:58405

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Papazian, Irini. Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ.

Degree: 2019, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 TNF is a member of a large family of cytokines and has multiple roles in the CNS. Regulated TNF production is necessary for successful host… (more)

Subjects/Keywords: TNF υποδοχείς; TNF; Νευροεκφυλισμός; Νευροπροστασία; Σκλήρυνση κατά πλάκας; Γλουταμινικοί υποδοχείς; Απομυελίνωση απο κουπριζόνη; Πειραματική αυτοάνοση εγκεφαλομυελίτιδα; TNF receptors; TNF; Neurodegeneration; Neuroprotection; Multiple sclerosis; Glutamate receptors; Cuprizone model; Experimental autoimmune encephalomyelitis

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APA (6th Edition):

Papazian, I. (2019). Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/45595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Papazian, Irini. “Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ.” 2019. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed February 28, 2021. http://hdl.handle.net/10442/hedi/45595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Papazian, Irini. “Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ.” 2019. Web. 28 Feb 2021.

Vancouver:

Papazian I. Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2019. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/10442/hedi/45595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Papazian I. Μελέτη του ρόλου του διαλυτού και διαμεμβρανικού TNF κατά την παθολογία του ΚΝΣ. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2019. Available from: http://hdl.handle.net/10442/hedi/45595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Virginia Commonwealth University

16. Moffatt, David. INVESTIGATING THE RESPONSE OF OLIGODENDROCYTE PROGENITOR CELLS TO THE CUPRIZONE MODEL OF DEMYELINATION.

Degree: MS, Anatomy & Neurobiology, 2009, Virginia Commonwealth University

 Multiple sclerosis and other myelin diseases affect the quality of life many people. In the United States alone, multiple sclerosis afflicts as many as 400,000… (more)

Subjects/Keywords: oligodendrocyte progenitor cell; SVZ; cuprizone; demyelination; Anatomy; Medicine and Health Sciences; Nervous System

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APA (6th Edition):

Moffatt, D. (2009). INVESTIGATING THE RESPONSE OF OLIGODENDROCYTE PROGENITOR CELLS TO THE CUPRIZONE MODEL OF DEMYELINATION. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/FBVA-G038 ; https://scholarscompass.vcu.edu/etd/1850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moffatt, David. “INVESTIGATING THE RESPONSE OF OLIGODENDROCYTE PROGENITOR CELLS TO THE CUPRIZONE MODEL OF DEMYELINATION.” 2009. Thesis, Virginia Commonwealth University. Accessed February 28, 2021. https://doi.org/10.25772/FBVA-G038 ; https://scholarscompass.vcu.edu/etd/1850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moffatt, David. “INVESTIGATING THE RESPONSE OF OLIGODENDROCYTE PROGENITOR CELLS TO THE CUPRIZONE MODEL OF DEMYELINATION.” 2009. Web. 28 Feb 2021.

Vancouver:

Moffatt D. INVESTIGATING THE RESPONSE OF OLIGODENDROCYTE PROGENITOR CELLS TO THE CUPRIZONE MODEL OF DEMYELINATION. [Internet] [Thesis]. Virginia Commonwealth University; 2009. [cited 2021 Feb 28]. Available from: https://doi.org/10.25772/FBVA-G038 ; https://scholarscompass.vcu.edu/etd/1850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moffatt D. INVESTIGATING THE RESPONSE OF OLIGODENDROCYTE PROGENITOR CELLS TO THE CUPRIZONE MODEL OF DEMYELINATION. [Thesis]. Virginia Commonwealth University; 2009. Available from: https://doi.org/10.25772/FBVA-G038 ; https://scholarscompass.vcu.edu/etd/1850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Millet, Violeta. Dinámica celular en respuesta a la desmielinización inducida por intoxicación con cuprizona en diferentes condiciones experimentales.

Degree: Farmacia y Bioquímica, 2016, Universidad de Buenos Aires

Demyelination models have proven useful tools in the assessment of new therapies to target demyelinating disorders. Among these models, cuprizone (CPZ) intoxication is simple and… (more)

Subjects/Keywords: Cuprizona; Mielinización; Proteasoma; Galectinas 1y3; Microglia; Ratones CNP::EGFPasa; Oligodendrocitos; Desmielinización; Remielinización; Especies reactivas del oxígeno; Citoquinas pro-inflamatorias; Oligodendrocytes; Microglia; Myelination; Demyelination; Remyelination; Reactive oxygen species; Pro-inflammatory cytokines; Proteasome; Cuprizone; Galectins 1 and 3; EGFP::CNPase mice; Ciencia de la vida

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APA (6th Edition):

Millet, V. (2016). Dinámica celular en respuesta a la desmielinización inducida por intoxicación con cuprizona en diferentes condiciones experimentales. (Thesis). Universidad de Buenos Aires. Retrieved from http://repositoriouba.sisbi.uba.ar/gsdl/cgi-bin/library.cgi?a=d&c=posgrauba&cl=CL1&d=HWA_1752 ; http://repositoriouba.sisbi.uba.ar/gsdl/collect/posgrauba/index/assoc/HWA_1752.dir/1752.PDF

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Millet, Violeta. “Dinámica celular en respuesta a la desmielinización inducida por intoxicación con cuprizona en diferentes condiciones experimentales.” 2016. Thesis, Universidad de Buenos Aires. Accessed February 28, 2021. http://repositoriouba.sisbi.uba.ar/gsdl/cgi-bin/library.cgi?a=d&c=posgrauba&cl=CL1&d=HWA_1752 ; http://repositoriouba.sisbi.uba.ar/gsdl/collect/posgrauba/index/assoc/HWA_1752.dir/1752.PDF.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Millet, Violeta. “Dinámica celular en respuesta a la desmielinización inducida por intoxicación con cuprizona en diferentes condiciones experimentales.” 2016. Web. 28 Feb 2021.

Vancouver:

Millet V. Dinámica celular en respuesta a la desmielinización inducida por intoxicación con cuprizona en diferentes condiciones experimentales. [Internet] [Thesis]. Universidad de Buenos Aires; 2016. [cited 2021 Feb 28]. Available from: http://repositoriouba.sisbi.uba.ar/gsdl/cgi-bin/library.cgi?a=d&c=posgrauba&cl=CL1&d=HWA_1752 ; http://repositoriouba.sisbi.uba.ar/gsdl/collect/posgrauba/index/assoc/HWA_1752.dir/1752.PDF.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Millet V. Dinámica celular en respuesta a la desmielinización inducida por intoxicación con cuprizona en diferentes condiciones experimentales. [Thesis]. Universidad de Buenos Aires; 2016. Available from: http://repositoriouba.sisbi.uba.ar/gsdl/cgi-bin/library.cgi?a=d&c=posgrauba&cl=CL1&d=HWA_1752 ; http://repositoriouba.sisbi.uba.ar/gsdl/collect/posgrauba/index/assoc/HWA_1752.dir/1752.PDF

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Kuypers, Nicholas John. Functional implications of demyelination and the molecular control of remyelination in the adult mouse.

Degree: PhD, 2014, University of Louisville

  Oligodendrocyte (OL) loss contributes to the functional deficits underlying diseases with a demyelinating component (Gajjar et al., 1997; Miller et al., 2002). Remyelination can… (more)

Subjects/Keywords: MicroRNA; Cuprizone; Demyelination; Remyelination; Oligodendrocyte; Ethidium bromide; Anatomy; Neuroscience and Neurobiology

…adult mice with the vii copper chelator cuprizone demyelinates specific brain… …regions which remyelinate following cuprizone cessation (Torkildsen et al., 2008)… …Therefore, Chapter 4 focuses on the role of oligodendroglial-specific miRNAs in cuprizone-induced… …facilitates the quantification of cuprizone-induced de- and re-myelination. 115 10. FACS-based… …miRNAs are significantly decreased at 6 weeks on cuprizone compared to no cuprizone miRNA… 

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APA (6th Edition):

Kuypers, N. J. (2014). Functional implications of demyelination and the molecular control of remyelination in the adult mouse. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/786 ; https://ir.library.louisville.edu/etd/786

Chicago Manual of Style (16th Edition):

Kuypers, Nicholas John. “Functional implications of demyelination and the molecular control of remyelination in the adult mouse.” 2014. Doctoral Dissertation, University of Louisville. Accessed February 28, 2021. 10.18297/etd/786 ; https://ir.library.louisville.edu/etd/786.

MLA Handbook (7th Edition):

Kuypers, Nicholas John. “Functional implications of demyelination and the molecular control of remyelination in the adult mouse.” 2014. Web. 28 Feb 2021.

Vancouver:

Kuypers NJ. Functional implications of demyelination and the molecular control of remyelination in the adult mouse. [Internet] [Doctoral dissertation]. University of Louisville; 2014. [cited 2021 Feb 28]. Available from: 10.18297/etd/786 ; https://ir.library.louisville.edu/etd/786.

Council of Science Editors:

Kuypers NJ. Functional implications of demyelination and the molecular control of remyelination in the adult mouse. [Doctoral Dissertation]. University of Louisville; 2014. Available from: 10.18297/etd/786 ; https://ir.library.louisville.edu/etd/786


Kyoto University

19. Fukudome, Daisuke. Translocator protein (TSPO) and stress cascades in mouse models of psychosis with inflammatory disturbances .

Degree: 2020, Kyoto University

Subjects/Keywords: Cuprizone short-term exposure (CSE); Maternal immune activation (MIA); Inflammatory disturbances; Translocator protein 18 kDa (TSPO); Stress cascades; Psychosis

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APA (6th Edition):

Fukudome, D. (2020). Translocator protein (TSPO) and stress cascades in mouse models of psychosis with inflammatory disturbances . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/259710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fukudome, Daisuke. “Translocator protein (TSPO) and stress cascades in mouse models of psychosis with inflammatory disturbances .” 2020. Thesis, Kyoto University. Accessed February 28, 2021. http://hdl.handle.net/2433/259710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fukudome, Daisuke. “Translocator protein (TSPO) and stress cascades in mouse models of psychosis with inflammatory disturbances .” 2020. Web. 28 Feb 2021.

Vancouver:

Fukudome D. Translocator protein (TSPO) and stress cascades in mouse models of psychosis with inflammatory disturbances . [Internet] [Thesis]. Kyoto University; 2020. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/2433/259710.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fukudome D. Translocator protein (TSPO) and stress cascades in mouse models of psychosis with inflammatory disturbances . [Thesis]. Kyoto University; 2020. Available from: http://hdl.handle.net/2433/259710

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. 山本, 梓司. Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.

Degree: 博士(医学), 2018, Saitama Medical University / 埼玉医科大学

Multiple sclerosis is a neuroinflammatory demyelinating and neurodegenerative disease of the central nervous system characterized by recurrent and progressive demyelination/remyelination cycles, neuroinflammation, oligodendrocyte loss, demyelination,… (more)

Subjects/Keywords: Animals; Anti-Inflammatory Agents; Apoptosis; Cell Differentiation; Cell Line, Transformed; Cuprizone; Demyelinating Diseases; Disease Models, Animal; Female; Gene Expression Regulation; Humans; MAP Kinase Signaling System; Male; Mice; Mice, Inbred C57BL; Monoamine Oxidase Inhibitors; Myelin Sheath; NLR Family, Pyrin Domain-Containing 3 Protein; Phosphatidic Acids; Proto-Oncogene Proteins c-bcl-2; p38 Mitogen-Activated Protein Kinases

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APA (6th Edition):

山本, . (2018). Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. (Thesis). Saitama Medical University / 埼玉医科大学. Retrieved from http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Thesis, Saitama Medical University / 埼玉医科大学. Accessed February 28, 2021. http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山本, 梓司. “Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果.” 2018. Web. 28 Feb 2021.

Vancouver:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Internet] [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. [cited 2021 Feb 28]. Available from: http://id.nii.ac.jp/1386/00000615/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山本 . Protective and therapeutic role of 2-carba-cyclic phosphatidic acid in demyelinating disease. : 脱髄性疾患における環状ホスファチジン酸誘導体の脱髄抑制及び治療効果. [Thesis]. Saitama Medical University / 埼玉医科大学; 2018. Available from: http://id.nii.ac.jp/1386/00000615/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Thiessen, Jonathan. Development and application of quantitative MRI methods for assessing white matter integrity in the mouse brain.

Degree: Physics and Astronomy, 2012, University of Manitoba

 Healthy white matter in the brain and spinal cord is composed primarily of myelinated axons and glial cells. Myelinated axons transfer information between the peripheral… (more)

Subjects/Keywords: magnetic resonance imaging; diffusion tensor imaging; magnetization transfer imaging; relaxometry; demyelination; cuprizone; corpus callosum; myelin; electron microscopy; multiple sclerosis; white matter; image processing

…Discussion . . . . . . . . . . . . . . . . . . . . . 4.3 Cuprizone Mouse Model of Demyelination… …Cuprizone 105 . . . . . . 106 . . . . . . 106 . . . . . . 106 . . . . . . 107 . . . . . . 112… …157 5 Quantitative MRI and Ultrastructural Examination of the Cuprizone Model of… …163 5.2.1 Cuprizone Mouse Model . . . . . . . . . . . . . . . . . . . . . . . . . 163 5.2.2… …166 5.3.1 Cuprizone Diet . . . . . . . . . . . . . . . . . . . . . . . . . . . . . . 166… 

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APA (6th Edition):

Thiessen, J. (2012). Development and application of quantitative MRI methods for assessing white matter integrity in the mouse brain. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/9221

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thiessen, Jonathan. “Development and application of quantitative MRI methods for assessing white matter integrity in the mouse brain.” 2012. Thesis, University of Manitoba. Accessed February 28, 2021. http://hdl.handle.net/1993/9221.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thiessen, Jonathan. “Development and application of quantitative MRI methods for assessing white matter integrity in the mouse brain.” 2012. Web. 28 Feb 2021.

Vancouver:

Thiessen J. Development and application of quantitative MRI methods for assessing white matter integrity in the mouse brain. [Internet] [Thesis]. University of Manitoba; 2012. [cited 2021 Feb 28]. Available from: http://hdl.handle.net/1993/9221.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thiessen J. Development and application of quantitative MRI methods for assessing white matter integrity in the mouse brain. [Thesis]. University of Manitoba; 2012. Available from: http://hdl.handle.net/1993/9221

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.