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You searched for subject:(Chondrocyte). Showing records 1 – 30 of 175 total matches.

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Mississippi State University

1. Mochal-King, Cathleen Ann. ANTIBIOTICS INDUCE PROSTAGLADIN E2 PRODUCTION AND CYTOTOXICITY IN EQUINE CHONDROCYTES THAT CAN BE INHIBITED BY AVOCADO SOYBEAN UNSAPONIFIABLES, GLUCOSAMINE, AND CHONDROITIN SULFATE.

Degree: MS, Veterinary Medicine, College of, 2011, Mississippi State University

  Amikacin (AK) and enrofloxacin (EF) concentrations consistent with intra-articular and regional limb perfusion were evaluated for their effects on equine chondrocytes. We evaluated the… (more)

Subjects/Keywords: amikacin; enrofloxacin; chondrocyte

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APA (6th Edition):

Mochal-King, C. A. (2011). ANTIBIOTICS INDUCE PROSTAGLADIN E2 PRODUCTION AND CYTOTOXICITY IN EQUINE CHONDROCYTES THAT CAN BE INHIBITED BY AVOCADO SOYBEAN UNSAPONIFIABLES, GLUCOSAMINE, AND CHONDROITIN SULFATE. (Masters Thesis). Mississippi State University. Retrieved from http://sun.library.msstate.edu/ETD-db/theses/available/etd-04042011-170643/ ;

Chicago Manual of Style (16th Edition):

Mochal-King, Cathleen Ann. “ANTIBIOTICS INDUCE PROSTAGLADIN E2 PRODUCTION AND CYTOTOXICITY IN EQUINE CHONDROCYTES THAT CAN BE INHIBITED BY AVOCADO SOYBEAN UNSAPONIFIABLES, GLUCOSAMINE, AND CHONDROITIN SULFATE.” 2011. Masters Thesis, Mississippi State University. Accessed April 13, 2021. http://sun.library.msstate.edu/ETD-db/theses/available/etd-04042011-170643/ ;.

MLA Handbook (7th Edition):

Mochal-King, Cathleen Ann. “ANTIBIOTICS INDUCE PROSTAGLADIN E2 PRODUCTION AND CYTOTOXICITY IN EQUINE CHONDROCYTES THAT CAN BE INHIBITED BY AVOCADO SOYBEAN UNSAPONIFIABLES, GLUCOSAMINE, AND CHONDROITIN SULFATE.” 2011. Web. 13 Apr 2021.

Vancouver:

Mochal-King CA. ANTIBIOTICS INDUCE PROSTAGLADIN E2 PRODUCTION AND CYTOTOXICITY IN EQUINE CHONDROCYTES THAT CAN BE INHIBITED BY AVOCADO SOYBEAN UNSAPONIFIABLES, GLUCOSAMINE, AND CHONDROITIN SULFATE. [Internet] [Masters thesis]. Mississippi State University; 2011. [cited 2021 Apr 13]. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-04042011-170643/ ;.

Council of Science Editors:

Mochal-King CA. ANTIBIOTICS INDUCE PROSTAGLADIN E2 PRODUCTION AND CYTOTOXICITY IN EQUINE CHONDROCYTES THAT CAN BE INHIBITED BY AVOCADO SOYBEAN UNSAPONIFIABLES, GLUCOSAMINE, AND CHONDROITIN SULFATE. [Masters Thesis]. Mississippi State University; 2011. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-04042011-170643/ ;


University of Manchester

2. Szkolar, Laura. The Development of Functional Peptide Scaffolds for Cell Culture.

Degree: 2016, University of Manchester

 Peptides and peptide derivatives have shown great scope as biomaterials and for biomedicaltherapy application. It has been demonstrated that classes of these peptides can form… (more)

Subjects/Keywords: peptide; chondrocyte; hydrogel

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APA (6th Edition):

Szkolar, L. (2016). The Development of Functional Peptide Scaffolds for Cell Culture. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:298798

Chicago Manual of Style (16th Edition):

Szkolar, Laura. “The Development of Functional Peptide Scaffolds for Cell Culture.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 13, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:298798.

MLA Handbook (7th Edition):

Szkolar, Laura. “The Development of Functional Peptide Scaffolds for Cell Culture.” 2016. Web. 13 Apr 2021.

Vancouver:

Szkolar L. The Development of Functional Peptide Scaffolds for Cell Culture. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 13]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:298798.

Council of Science Editors:

Szkolar L. The Development of Functional Peptide Scaffolds for Cell Culture. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:298798


University of Rochester

3. Chen, Tony (1981 - ). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering.

Degree: PhD, 2011, University of Rochester

 Injuries to articular cartilage are debilitating and typically require surgical intervention due to the tissue’s inability to self-repair. Current surgical options for focal cartilage defects… (more)

Subjects/Keywords: Collagen alignment; Chondrocyte; TNF-&alpha

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APA (6th Edition):

Chen, T. (. -. ). (2011). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/14143

Chicago Manual of Style (16th Edition):

Chen, Tony (1981 - ). “The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering.” 2011. Doctoral Dissertation, University of Rochester. Accessed April 13, 2021. http://hdl.handle.net/1802/14143.

MLA Handbook (7th Edition):

Chen, Tony (1981 - ). “The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering.” 2011. Web. 13 Apr 2021.

Vancouver:

Chen T(-). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1802/14143.

Council of Science Editors:

Chen T(-). The Role of interstitial fluid flow as a mediator of matrix anisotropy and as a protective mechanism against inflammation in cartilage tissue engineering. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/14143


University of Manchester

4. Szkolar, Laura. The development of functional peptide scaffolds for cell culture.

Degree: PhD, 2016, University of Manchester

 Peptides and peptide derivatives have shown great scope as biomaterials and for biomedicaltherapy application. It has been demonstrated that classes of these peptides can form… (more)

Subjects/Keywords: 610.28; peptide; chondrocyte; hydrogel

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APA (6th Edition):

Szkolar, L. (2016). The development of functional peptide scaffolds for cell culture. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-development-of-functional-peptide-scaffolds-for-cell-culture(6e7dffc3-030c-4c9e-873e-8ed1f48efa51).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728122

Chicago Manual of Style (16th Edition):

Szkolar, Laura. “The development of functional peptide scaffolds for cell culture.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 13, 2021. https://www.research.manchester.ac.uk/portal/en/theses/the-development-of-functional-peptide-scaffolds-for-cell-culture(6e7dffc3-030c-4c9e-873e-8ed1f48efa51).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728122.

MLA Handbook (7th Edition):

Szkolar, Laura. “The development of functional peptide scaffolds for cell culture.” 2016. Web. 13 Apr 2021.

Vancouver:

Szkolar L. The development of functional peptide scaffolds for cell culture. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 13]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-development-of-functional-peptide-scaffolds-for-cell-culture(6e7dffc3-030c-4c9e-873e-8ed1f48efa51).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728122.

Council of Science Editors:

Szkolar L. The development of functional peptide scaffolds for cell culture. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-development-of-functional-peptide-scaffolds-for-cell-culture(6e7dffc3-030c-4c9e-873e-8ed1f48efa51).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728122


University of Manchester

5. Jones, Rebecca. The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1.

Degree: 2019, University of Manchester

Osteoarthritis (OA) is one of the leading causes of morbidity worldwide. Single injurious mechanical impact to joints as seen in sports injury is known to… (more)

Subjects/Keywords: chondrocyte; cartilage; osteoarthritis; urocortin; piezo1

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APA (6th Edition):

Jones, R. (2019). The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322543

Chicago Manual of Style (16th Edition):

Jones, Rebecca. “The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1.” 2019. Doctoral Dissertation, University of Manchester. Accessed April 13, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322543.

MLA Handbook (7th Edition):

Jones, Rebecca. “The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1.” 2019. Web. 13 Apr 2021.

Vancouver:

Jones R. The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2021 Apr 13]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322543.

Council of Science Editors:

Jones R. The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1. [Doctoral Dissertation]. University of Manchester; 2019. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:322543


University of Manchester

6. Jones, Rebecca. The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1.

Degree: PhD, 2019, University of Manchester

 Osteoarthritis (OA) is one of the leading causes of morbidity worldwide. Single injurious mechanical impact to joints as seen in sports injury is known to… (more)

Subjects/Keywords: piezo1; chondrocyte; cartilage; osteoarthritis; urocortin

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APA (6th Edition):

Jones, R. (2019). The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-chondroprotective-effect-of-urocortin-involves-modulation-of-the-mechanosensitive-ion-channel-piezo1(a3216810-a542-4f27-bbdb-78bbdb86f56a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.820085

Chicago Manual of Style (16th Edition):

Jones, Rebecca. “The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1.” 2019. Doctoral Dissertation, University of Manchester. Accessed April 13, 2021. https://www.research.manchester.ac.uk/portal/en/theses/the-chondroprotective-effect-of-urocortin-involves-modulation-of-the-mechanosensitive-ion-channel-piezo1(a3216810-a542-4f27-bbdb-78bbdb86f56a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.820085.

MLA Handbook (7th Edition):

Jones, Rebecca. “The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1.” 2019. Web. 13 Apr 2021.

Vancouver:

Jones R. The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1. [Internet] [Doctoral dissertation]. University of Manchester; 2019. [cited 2021 Apr 13]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-chondroprotective-effect-of-urocortin-involves-modulation-of-the-mechanosensitive-ion-channel-piezo1(a3216810-a542-4f27-bbdb-78bbdb86f56a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.820085.

Council of Science Editors:

Jones R. The chondroprotective effect of urocortin involves modulation of the mechanosensitive ion channel Piezo1. [Doctoral Dissertation]. University of Manchester; 2019. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-chondroprotective-effect-of-urocortin-involves-modulation-of-the-mechanosensitive-ion-channel-piezo1(a3216810-a542-4f27-bbdb-78bbdb86f56a).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.820085

7. Guérit, David. Rôle des miR-29a et miR-574-3p au cours de la différenciation chondrocytaire de la cellule souche mésenchymateuse : Roles of miR-29a and miR-574-3p during the chondrogenic differentiation of mesenchymal stem cell.

Degree: Docteur es, Biologie Santé, 2012, Université Montpellier I

Avec l'augmentation de l'espérance de vie, les pathologies ostéo-articulaires comme l'arthrose ou la polyarthrite rhumatoïde, caractérisées par la dégradation du cartilage articulaire, deviennent de réels… (more)

Subjects/Keywords: Arthrose; MicroARN; Chondrocyte; Différenciation; Osteoarthritis; MicroRNA; Chondrocyte; Differentiation

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APA (6th Edition):

Guérit, D. (2012). Rôle des miR-29a et miR-574-3p au cours de la différenciation chondrocytaire de la cellule souche mésenchymateuse : Roles of miR-29a and miR-574-3p during the chondrogenic differentiation of mesenchymal stem cell. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2012MON1T013

Chicago Manual of Style (16th Edition):

Guérit, David. “Rôle des miR-29a et miR-574-3p au cours de la différenciation chondrocytaire de la cellule souche mésenchymateuse : Roles of miR-29a and miR-574-3p during the chondrogenic differentiation of mesenchymal stem cell.” 2012. Doctoral Dissertation, Université Montpellier I. Accessed April 13, 2021. http://www.theses.fr/2012MON1T013.

MLA Handbook (7th Edition):

Guérit, David. “Rôle des miR-29a et miR-574-3p au cours de la différenciation chondrocytaire de la cellule souche mésenchymateuse : Roles of miR-29a and miR-574-3p during the chondrogenic differentiation of mesenchymal stem cell.” 2012. Web. 13 Apr 2021.

Vancouver:

Guérit D. Rôle des miR-29a et miR-574-3p au cours de la différenciation chondrocytaire de la cellule souche mésenchymateuse : Roles of miR-29a and miR-574-3p during the chondrogenic differentiation of mesenchymal stem cell. [Internet] [Doctoral dissertation]. Université Montpellier I; 2012. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2012MON1T013.

Council of Science Editors:

Guérit D. Rôle des miR-29a et miR-574-3p au cours de la différenciation chondrocytaire de la cellule souche mésenchymateuse : Roles of miR-29a and miR-574-3p during the chondrogenic differentiation of mesenchymal stem cell. [Doctoral Dissertation]. Université Montpellier I; 2012. Available from: http://www.theses.fr/2012MON1T013


University of Rochester

8. Zhang, Yongchun. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.

Degree: PhD, 2015, University of Rochester

 This work addresses the central question of how B-CATENIN signaling regulates chondrocyte differentiation during endochondral bone formation and cartilage development through integration with BMP and… (more)

Subjects/Keywords: beta-CATENIN; CCN1; BMP2; Chondrocyte; Cartilage

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APA (6th Edition):

Zhang, Y. (2015). B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/30113

Chicago Manual of Style (16th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Doctoral Dissertation, University of Rochester. Accessed April 13, 2021. http://hdl.handle.net/1802/30113.

MLA Handbook (7th Edition):

Zhang, Yongchun. “B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate.” 2015. Web. 13 Apr 2021.

Vancouver:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Internet] [Doctoral dissertation]. University of Rochester; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1802/30113.

Council of Science Editors:

Zhang Y. B-CATENIN Signaling Integrates with BMP and CCN1 Signaling to Regulate Chondrocyte Differentiation and Cartilage Development ate. [Doctoral Dissertation]. University of Rochester; 2015. Available from: http://hdl.handle.net/1802/30113


Mississippi State University

9. Dycus, David Lee. Modulation of inflammation and oxidative stress in canine chondrocytes.

Degree: MS, Veterinary Medicine, College of, 2012, Mississippi State University

  Little research has focused on the involvement of oxidative stress as it relates to the pathophysiology of osteoarthritis (OA); while inflammation has been extensively… (more)

Subjects/Keywords: canine; chondrocyte; oxidative stress; inflammation; osteoarthritis

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APA (6th Edition):

Dycus, D. L. (2012). Modulation of inflammation and oxidative stress in canine chondrocytes. (Masters Thesis). Mississippi State University. Retrieved from http://sun.library.msstate.edu/ETD-db/theses/available/etd-09262012-163241/ ;

Chicago Manual of Style (16th Edition):

Dycus, David Lee. “Modulation of inflammation and oxidative stress in canine chondrocytes.” 2012. Masters Thesis, Mississippi State University. Accessed April 13, 2021. http://sun.library.msstate.edu/ETD-db/theses/available/etd-09262012-163241/ ;.

MLA Handbook (7th Edition):

Dycus, David Lee. “Modulation of inflammation and oxidative stress in canine chondrocytes.” 2012. Web. 13 Apr 2021.

Vancouver:

Dycus DL. Modulation of inflammation and oxidative stress in canine chondrocytes. [Internet] [Masters thesis]. Mississippi State University; 2012. [cited 2021 Apr 13]. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-09262012-163241/ ;.

Council of Science Editors:

Dycus DL. Modulation of inflammation and oxidative stress in canine chondrocytes. [Masters Thesis]. Mississippi State University; 2012. Available from: http://sun.library.msstate.edu/ETD-db/theses/available/etd-09262012-163241/ ;


University of Manchester

10. Soul, Jamie. A Systems Biology Approach To Knee Osteoarthritis.

Degree: 2017, University of Manchester

A hallmark of the joint disease osteoarthritis (OA) is the degradation of the articularcartilage in the affected joint, debilitating pain and decreased mobility. At present… (more)

Subjects/Keywords: Osteoarthritis; Cartilage; Chondrocyte; Bioinformatics; Transcriptomics; Network biology

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APA (6th Edition):

Soul, J. (2017). A Systems Biology Approach To Knee Osteoarthritis. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308443

Chicago Manual of Style (16th Edition):

Soul, Jamie. “A Systems Biology Approach To Knee Osteoarthritis.” 2017. Doctoral Dissertation, University of Manchester. Accessed April 13, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308443.

MLA Handbook (7th Edition):

Soul, Jamie. “A Systems Biology Approach To Knee Osteoarthritis.” 2017. Web. 13 Apr 2021.

Vancouver:

Soul J. A Systems Biology Approach To Knee Osteoarthritis. [Internet] [Doctoral dissertation]. University of Manchester; 2017. [cited 2021 Apr 13]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308443.

Council of Science Editors:

Soul J. A Systems Biology Approach To Knee Osteoarthritis. [Doctoral Dissertation]. University of Manchester; 2017. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:308443


Vanderbilt University

11. Wang, Weiguang. Function of Atf4 in Endochondral Bone Formation.

Degree: PhD, Pharmacology, 2011, Vanderbilt University

 Activating transcription factor 4 (Atf4) is a leucine zip transcription factor. Atf4 mutant (Atf4-/-) mice show severe low bone mass and short stature phenotype. Atf4… (more)

Subjects/Keywords: chondrocyte; Ihh; Endochondral ossification; Atf4; osteoblast

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APA (6th Edition):

Wang, W. (2011). Function of Atf4 in Endochondral Bone Formation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12726

Chicago Manual of Style (16th Edition):

Wang, Weiguang. “Function of Atf4 in Endochondral Bone Formation.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed April 13, 2021. http://hdl.handle.net/1803/12726.

MLA Handbook (7th Edition):

Wang, Weiguang. “Function of Atf4 in Endochondral Bone Formation.” 2011. Web. 13 Apr 2021.

Vancouver:

Wang W. Function of Atf4 in Endochondral Bone Formation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1803/12726.

Council of Science Editors:

Wang W. Function of Atf4 in Endochondral Bone Formation. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://hdl.handle.net/1803/12726

12. 山下, 美智子. Uhrf1は軟骨細胞分化を制御し正常な骨格形成に必須である.

Degree: 博士(医学), 2018, Ehime University / 愛媛大学

博士(医学)

甲医博第860号

Subjects/Keywords: Uhrf1; chondrocyte; differentiation

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APA (6th Edition):

山下, . (2018). Uhrf1は軟骨細胞分化を制御し正常な骨格形成に必須である. (Thesis). Ehime University / 愛媛大学. Retrieved from http://iyokan.lib.ehime-u.ac.jp/dspace/handle/iyokan/5401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

山下, 美智子. “Uhrf1は軟骨細胞分化を制御し正常な骨格形成に必須である.” 2018. Thesis, Ehime University / 愛媛大学. Accessed April 13, 2021. http://iyokan.lib.ehime-u.ac.jp/dspace/handle/iyokan/5401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

山下, 美智子. “Uhrf1は軟骨細胞分化を制御し正常な骨格形成に必須である.” 2018. Web. 13 Apr 2021.

Vancouver:

山下 . Uhrf1は軟骨細胞分化を制御し正常な骨格形成に必須である. [Internet] [Thesis]. Ehime University / 愛媛大学; 2018. [cited 2021 Apr 13]. Available from: http://iyokan.lib.ehime-u.ac.jp/dspace/handle/iyokan/5401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

山下 . Uhrf1は軟骨細胞分化を制御し正常な骨格形成に必須である. [Thesis]. Ehime University / 愛媛大学; 2018. Available from: http://iyokan.lib.ehime-u.ac.jp/dspace/handle/iyokan/5401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

13. Blank, Kevin. The effect of phosphate availability on chondrocyte metabolism.

Degree: MS, Medical Sciences, 2016, Boston University

 Dietary phosphate is essential for normal fracture healing and bone growth. Previous studies have established that mice given a phosphate deficient diet after a fracture… (more)

Subjects/Keywords: Biochemistry; Chondrocyte; Differentiation; Metabolism; Oxidative phosphorylation; Phosphate

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APA (6th Edition):

Blank, K. (2016). The effect of phosphate availability on chondrocyte metabolism. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16797

Chicago Manual of Style (16th Edition):

Blank, Kevin. “The effect of phosphate availability on chondrocyte metabolism.” 2016. Masters Thesis, Boston University. Accessed April 13, 2021. http://hdl.handle.net/2144/16797.

MLA Handbook (7th Edition):

Blank, Kevin. “The effect of phosphate availability on chondrocyte metabolism.” 2016. Web. 13 Apr 2021.

Vancouver:

Blank K. The effect of phosphate availability on chondrocyte metabolism. [Internet] [Masters thesis]. Boston University; 2016. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/2144/16797.

Council of Science Editors:

Blank K. The effect of phosphate availability on chondrocyte metabolism. [Masters Thesis]. Boston University; 2016. Available from: http://hdl.handle.net/2144/16797


University of Toronto

14. Parreno, Justin. The Role of Cytoskeletal Polymerization in the Regulation of the Chondrocyte Phenotype.

Degree: PhD, 2015, University of Toronto

In this study the role of the chondrocyte cytoskeleton in regulating cellular mRNA levels in chondrocytes was examined. Passaging of chondrocytes in traditional 2D monolayer… (more)

Subjects/Keywords: Actin; Bioengineering; Cartilage; Chondrocyte; Dedifferentiation; MRTF; 0379

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APA (6th Edition):

Parreno, J. (2015). The Role of Cytoskeletal Polymerization in the Regulation of the Chondrocyte Phenotype. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/80370

Chicago Manual of Style (16th Edition):

Parreno, Justin. “The Role of Cytoskeletal Polymerization in the Regulation of the Chondrocyte Phenotype.” 2015. Doctoral Dissertation, University of Toronto. Accessed April 13, 2021. http://hdl.handle.net/1807/80370.

MLA Handbook (7th Edition):

Parreno, Justin. “The Role of Cytoskeletal Polymerization in the Regulation of the Chondrocyte Phenotype.” 2015. Web. 13 Apr 2021.

Vancouver:

Parreno J. The Role of Cytoskeletal Polymerization in the Regulation of the Chondrocyte Phenotype. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1807/80370.

Council of Science Editors:

Parreno J. The Role of Cytoskeletal Polymerization in the Regulation of the Chondrocyte Phenotype. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/80370


University of Toronto

15. Aiken, Alison. The Fundamental Roles of the Tissue Inhibitor of Metalloproteinase Family in Mammalian Biology.

Degree: PhD, 2014, University of Toronto

The metalloproteinases are a family of extracellular proteases responsible for degrading components of the extracellular matrix as well as shedding growth factors, cytokines and their… (more)

Subjects/Keywords: chondrocyte; CXCL12; IHH; metalloproteinase; skeleton; TIMP; 0307

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APA (6th Edition):

Aiken, A. (2014). The Fundamental Roles of the Tissue Inhibitor of Metalloproteinase Family in Mammalian Biology. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/74489

Chicago Manual of Style (16th Edition):

Aiken, Alison. “The Fundamental Roles of the Tissue Inhibitor of Metalloproteinase Family in Mammalian Biology.” 2014. Doctoral Dissertation, University of Toronto. Accessed April 13, 2021. http://hdl.handle.net/1807/74489.

MLA Handbook (7th Edition):

Aiken, Alison. “The Fundamental Roles of the Tissue Inhibitor of Metalloproteinase Family in Mammalian Biology.” 2014. Web. 13 Apr 2021.

Vancouver:

Aiken A. The Fundamental Roles of the Tissue Inhibitor of Metalloproteinase Family in Mammalian Biology. [Internet] [Doctoral dissertation]. University of Toronto; 2014. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1807/74489.

Council of Science Editors:

Aiken A. The Fundamental Roles of the Tissue Inhibitor of Metalloproteinase Family in Mammalian Biology. [Doctoral Dissertation]. University of Toronto; 2014. Available from: http://hdl.handle.net/1807/74489


University of Melbourne

16. Falahati, Behzad. Development of an experimental loading and imaging protocol for studying cartilage cell morphology in a mouse model.

Degree: 2012, University of Melbourne

 A methodology was developed to apply controlled cyclic loads on mouse knees and to investigate resulting changes in chondrocyte morphology in vitro using confocal microscopy.… (more)

Subjects/Keywords: chondrocyte; mouse; ADAMTS5 gene; loading; confocal microscopy

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APA (6th Edition):

Falahati, B. (2012). Development of an experimental loading and imaging protocol for studying cartilage cell morphology in a mouse model. (Masters Thesis). University of Melbourne. Retrieved from http://hdl.handle.net/11343/38006

Chicago Manual of Style (16th Edition):

Falahati, Behzad. “Development of an experimental loading and imaging protocol for studying cartilage cell morphology in a mouse model.” 2012. Masters Thesis, University of Melbourne. Accessed April 13, 2021. http://hdl.handle.net/11343/38006.

MLA Handbook (7th Edition):

Falahati, Behzad. “Development of an experimental loading and imaging protocol for studying cartilage cell morphology in a mouse model.” 2012. Web. 13 Apr 2021.

Vancouver:

Falahati B. Development of an experimental loading and imaging protocol for studying cartilage cell morphology in a mouse model. [Internet] [Masters thesis]. University of Melbourne; 2012. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/11343/38006.

Council of Science Editors:

Falahati B. Development of an experimental loading and imaging protocol for studying cartilage cell morphology in a mouse model. [Masters Thesis]. University of Melbourne; 2012. Available from: http://hdl.handle.net/11343/38006


University of Edinburgh

17. Mohamad Yusof, Loqman. Longitudinal growth of mammalian bones : a possible role for membrane transporters in mediating chondrocyte hypertrophy.

Degree: PhD, 2012, University of Edinburgh

 Long bone lengthening occurs at the growth plate (GP) by well-regulated chondrocyte proliferation, hypertrophy and terminal matrix deposition. GP chondrocyte (GPC) hypertrophy has been implicated… (more)

Subjects/Keywords: 612; longitudinal growth; membrane transporter; chondrocyte hypertrophy

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APA (6th Edition):

Mohamad Yusof, L. (2012). Longitudinal growth of mammalian bones : a possible role for membrane transporters in mediating chondrocyte hypertrophy. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/6481

Chicago Manual of Style (16th Edition):

Mohamad Yusof, Loqman. “Longitudinal growth of mammalian bones : a possible role for membrane transporters in mediating chondrocyte hypertrophy.” 2012. Doctoral Dissertation, University of Edinburgh. Accessed April 13, 2021. http://hdl.handle.net/1842/6481.

MLA Handbook (7th Edition):

Mohamad Yusof, Loqman. “Longitudinal growth of mammalian bones : a possible role for membrane transporters in mediating chondrocyte hypertrophy.” 2012. Web. 13 Apr 2021.

Vancouver:

Mohamad Yusof L. Longitudinal growth of mammalian bones : a possible role for membrane transporters in mediating chondrocyte hypertrophy. [Internet] [Doctoral dissertation]. University of Edinburgh; 2012. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1842/6481.

Council of Science Editors:

Mohamad Yusof L. Longitudinal growth of mammalian bones : a possible role for membrane transporters in mediating chondrocyte hypertrophy. [Doctoral Dissertation]. University of Edinburgh; 2012. Available from: http://hdl.handle.net/1842/6481


Freie Universität Berlin

18. König, Josephine. Effekte von Polystyrol- und Polyetherimid-Zellkulturinserts mit verschiedenen Rauigkeitsstufen auf die Morphologie, die metabolische Aktivität und das Expressionsprofil artikulärer Chondrozyten.

Degree: 2020, Freie Universität Berlin

 Autologe Chondrozyten können in vitro kultiviert werden und zur Defektdeckung von Knorpelschäden implantiert werden. Dafür muss eine kleine Menge an autologen Knorpelzellen während einer Biopsie… (more)

Subjects/Keywords: cell culture inserts; chondrocyte; ddc:610

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APA (6th Edition):

König, J. (2020). Effekte von Polystyrol- und Polyetherimid-Zellkulturinserts mit verschiedenen Rauigkeitsstufen auf die Morphologie, die metabolische Aktivität und das Expressionsprofil artikulärer Chondrozyten. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-27583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

König, Josephine. “Effekte von Polystyrol- und Polyetherimid-Zellkulturinserts mit verschiedenen Rauigkeitsstufen auf die Morphologie, die metabolische Aktivität und das Expressionsprofil artikulärer Chondrozyten.” 2020. Thesis, Freie Universität Berlin. Accessed April 13, 2021. http://dx.doi.org/10.17169/refubium-27583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

König, Josephine. “Effekte von Polystyrol- und Polyetherimid-Zellkulturinserts mit verschiedenen Rauigkeitsstufen auf die Morphologie, die metabolische Aktivität und das Expressionsprofil artikulärer Chondrozyten.” 2020. Web. 13 Apr 2021.

Vancouver:

König J. Effekte von Polystyrol- und Polyetherimid-Zellkulturinserts mit verschiedenen Rauigkeitsstufen auf die Morphologie, die metabolische Aktivität und das Expressionsprofil artikulärer Chondrozyten. [Internet] [Thesis]. Freie Universität Berlin; 2020. [cited 2021 Apr 13]. Available from: http://dx.doi.org/10.17169/refubium-27583.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

König J. Effekte von Polystyrol- und Polyetherimid-Zellkulturinserts mit verschiedenen Rauigkeitsstufen auf die Morphologie, die metabolische Aktivität und das Expressionsprofil artikulärer Chondrozyten. [Thesis]. Freie Universität Berlin; 2020. Available from: http://dx.doi.org/10.17169/refubium-27583

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Oklahoma State University

19. Hooshmand-yazdi, Shirin. Genistein Reduces Production of Proinflammatory Molecules in Human Chondrocytes.

Degree: Department of Nutritional Sciences, 2006, Oklahoma State University

 Previously, we reported that cartilage is an estrogen receptor (ER) positive tissue and that mRNA levels of ER increase in postmenopausal women with osteoarthritis. Based… (more)

Subjects/Keywords: genistein; chondrocyte; inflammation

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APA (6th Edition):

Hooshmand-yazdi, S. (2006). Genistein Reduces Production of Proinflammatory Molecules in Human Chondrocytes. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/9233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hooshmand-yazdi, Shirin. “Genistein Reduces Production of Proinflammatory Molecules in Human Chondrocytes.” 2006. Thesis, Oklahoma State University. Accessed April 13, 2021. http://hdl.handle.net/11244/9233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hooshmand-yazdi, Shirin. “Genistein Reduces Production of Proinflammatory Molecules in Human Chondrocytes.” 2006. Web. 13 Apr 2021.

Vancouver:

Hooshmand-yazdi S. Genistein Reduces Production of Proinflammatory Molecules in Human Chondrocytes. [Internet] [Thesis]. Oklahoma State University; 2006. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/11244/9233.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hooshmand-yazdi S. Genistein Reduces Production of Proinflammatory Molecules in Human Chondrocytes. [Thesis]. Oklahoma State University; 2006. Available from: http://hdl.handle.net/11244/9233

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Durand, Anne-Laure. Rôle de l'épigénétique dans la régulation des collagènes dans les chondrocytes articulaires humains : nouveaux aspects pour la compréhension de l'homéostasie du cartilage : The role of epigenetics in the regulation of collagens in human articular chondrocytes : new insight for cartilage homeostasis.

Degree: Docteur es, Sciences de la vie, 2017, Lyon

Le cartilage articulaire est un tissu avasculaire ayant une faible capacité de régénération. Ce tissu est essentiellement constitué d’un type cellulaire, les chondrocytes, inclus dans… (more)

Subjects/Keywords: LSD1; Méthylation de l'ADN; Cartilage; Chondrocyte; LSD1; DNA methylation; Cartilage; Chondrocyte; 571.6

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APA (6th Edition):

Durand, A. (2017). Rôle de l'épigénétique dans la régulation des collagènes dans les chondrocytes articulaires humains : nouveaux aspects pour la compréhension de l'homéostasie du cartilage : The role of epigenetics in the regulation of collagens in human articular chondrocytes : new insight for cartilage homeostasis. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2017LYSE1270

Chicago Manual of Style (16th Edition):

Durand, Anne-Laure. “Rôle de l'épigénétique dans la régulation des collagènes dans les chondrocytes articulaires humains : nouveaux aspects pour la compréhension de l'homéostasie du cartilage : The role of epigenetics in the regulation of collagens in human articular chondrocytes : new insight for cartilage homeostasis.” 2017. Doctoral Dissertation, Lyon. Accessed April 13, 2021. http://www.theses.fr/2017LYSE1270.

MLA Handbook (7th Edition):

Durand, Anne-Laure. “Rôle de l'épigénétique dans la régulation des collagènes dans les chondrocytes articulaires humains : nouveaux aspects pour la compréhension de l'homéostasie du cartilage : The role of epigenetics in the regulation of collagens in human articular chondrocytes : new insight for cartilage homeostasis.” 2017. Web. 13 Apr 2021.

Vancouver:

Durand A. Rôle de l'épigénétique dans la régulation des collagènes dans les chondrocytes articulaires humains : nouveaux aspects pour la compréhension de l'homéostasie du cartilage : The role of epigenetics in the regulation of collagens in human articular chondrocytes : new insight for cartilage homeostasis. [Internet] [Doctoral dissertation]. Lyon; 2017. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2017LYSE1270.

Council of Science Editors:

Durand A. Rôle de l'épigénétique dans la régulation des collagènes dans les chondrocytes articulaires humains : nouveaux aspects pour la compréhension de l'homéostasie du cartilage : The role of epigenetics in the regulation of collagens in human articular chondrocytes : new insight for cartilage homeostasis. [Doctoral Dissertation]. Lyon; 2017. Available from: http://www.theses.fr/2017LYSE1270

21. El-Hayek, Elissar. Analyse à large échelle du profil d'expression des gènes dans des chondrocytes articulaires soumis à un stress mécanique de type étirement : la relaxine une nouvelle cible d'intérêt dans les pathologies ostéoarticulaires ? : Pas de titre traduit.

Degree: Docteur es, Biologie cellulaire et moléculaire, 2013, Université Paris Descartes – Paris V

Le cartilage articulaire est un tissu conjonctif spécialisé recouvrant les surfaces osseuses et assurant, avec d’autres tissus comme la membrane synoviale, le bon fonctionnement des… (more)

Subjects/Keywords: Chondrocyte; Étirement; Relaxine; Inflammation; Dédifférenciation; Arthrose; Chondrocyte; Stretching; Relaxin; Inflammation; Dedifferentiation; Osteoarthritis; 611.018 1

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APA (6th Edition):

El-Hayek, E. (2013). Analyse à large échelle du profil d'expression des gènes dans des chondrocytes articulaires soumis à un stress mécanique de type étirement : la relaxine une nouvelle cible d'intérêt dans les pathologies ostéoarticulaires ? : Pas de titre traduit. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2013PA05T077

Chicago Manual of Style (16th Edition):

El-Hayek, Elissar. “Analyse à large échelle du profil d'expression des gènes dans des chondrocytes articulaires soumis à un stress mécanique de type étirement : la relaxine une nouvelle cible d'intérêt dans les pathologies ostéoarticulaires ? : Pas de titre traduit.” 2013. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed April 13, 2021. http://www.theses.fr/2013PA05T077.

MLA Handbook (7th Edition):

El-Hayek, Elissar. “Analyse à large échelle du profil d'expression des gènes dans des chondrocytes articulaires soumis à un stress mécanique de type étirement : la relaxine une nouvelle cible d'intérêt dans les pathologies ostéoarticulaires ? : Pas de titre traduit.” 2013. Web. 13 Apr 2021.

Vancouver:

El-Hayek E. Analyse à large échelle du profil d'expression des gènes dans des chondrocytes articulaires soumis à un stress mécanique de type étirement : la relaxine une nouvelle cible d'intérêt dans les pathologies ostéoarticulaires ? : Pas de titre traduit. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2013. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2013PA05T077.

Council of Science Editors:

El-Hayek E. Analyse à large échelle du profil d'expression des gènes dans des chondrocytes articulaires soumis à un stress mécanique de type étirement : la relaxine une nouvelle cible d'intérêt dans les pathologies ostéoarticulaires ? : Pas de titre traduit. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2013. Available from: http://www.theses.fr/2013PA05T077


Université de Lorraine

22. Guibert, Mathilde. Altération du phénotype chondrocytaire : Rôle de l’homéostasie locale de facteurs modulant la balance Pi/Ppi : Alteration in chondrocyte phenotype : role of local homeostasis of Pi/PPi balance modulating factors.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université de Lorraine

L'arthrose (OA) est une maladie articulaire chronique qui résulte de changements complexes dans le phénotype des chondrocytes. La présence de microcristaux contenant du phosphate dans… (more)

Subjects/Keywords: Chondrocyte; Phénotype; Pyrophosphate inorganique; Phosphate inorganique; FGF23; Arthrose; Chondrocyte; Phenotype; Inorganic pyrophosphate; Inorganic phosphate; FGF23; Osteoarthritis; 571.75; 616.722 3; 612.392 4

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APA (6th Edition):

Guibert, M. (2016). Altération du phénotype chondrocytaire : Rôle de l’homéostasie locale de facteurs modulant la balance Pi/Ppi : Alteration in chondrocyte phenotype : role of local homeostasis of Pi/PPi balance modulating factors. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2016LORR0160

Chicago Manual of Style (16th Edition):

Guibert, Mathilde. “Altération du phénotype chondrocytaire : Rôle de l’homéostasie locale de facteurs modulant la balance Pi/Ppi : Alteration in chondrocyte phenotype : role of local homeostasis of Pi/PPi balance modulating factors.” 2016. Doctoral Dissertation, Université de Lorraine. Accessed April 13, 2021. http://www.theses.fr/2016LORR0160.

MLA Handbook (7th Edition):

Guibert, Mathilde. “Altération du phénotype chondrocytaire : Rôle de l’homéostasie locale de facteurs modulant la balance Pi/Ppi : Alteration in chondrocyte phenotype : role of local homeostasis of Pi/PPi balance modulating factors.” 2016. Web. 13 Apr 2021.

Vancouver:

Guibert M. Altération du phénotype chondrocytaire : Rôle de l’homéostasie locale de facteurs modulant la balance Pi/Ppi : Alteration in chondrocyte phenotype : role of local homeostasis of Pi/PPi balance modulating factors. [Internet] [Doctoral dissertation]. Université de Lorraine; 2016. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2016LORR0160.

Council of Science Editors:

Guibert M. Altération du phénotype chondrocytaire : Rôle de l’homéostasie locale de facteurs modulant la balance Pi/Ppi : Alteration in chondrocyte phenotype : role of local homeostasis of Pi/PPi balance modulating factors. [Doctoral Dissertation]. Université de Lorraine; 2016. Available from: http://www.theses.fr/2016LORR0160


Université de Lorraine

23. Xie, Zhe. Wnt signaling in human cartilage degeneration and chondrocytes de-differentiation : La signalisation de Wnt dans la dégradation du cartilage et la dédifférenciation chondrocytaire.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université de Lorraine

La dérégulation de la signalisation Wnt est impliquée dans les anomalies du développement et dans la pathogenèse de nombreuses maladies, y compris l'arthrose. Au cours… (more)

Subjects/Keywords: Wnt; Syndécane4; Chondrocyte; Différenciation; ADAMTS-4; Arthrose; Wnt; Syndecan4; Chondrocyte; Differentiation; ADAMTS-4; Osteoarthritis; 616.722 3; 611.018 1

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APA (6th Edition):

Xie, Z. (2016). Wnt signaling in human cartilage degeneration and chondrocytes de-differentiation : La signalisation de Wnt dans la dégradation du cartilage et la dédifférenciation chondrocytaire. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2016LORR0111

Chicago Manual of Style (16th Edition):

Xie, Zhe. “Wnt signaling in human cartilage degeneration and chondrocytes de-differentiation : La signalisation de Wnt dans la dégradation du cartilage et la dédifférenciation chondrocytaire.” 2016. Doctoral Dissertation, Université de Lorraine. Accessed April 13, 2021. http://www.theses.fr/2016LORR0111.

MLA Handbook (7th Edition):

Xie, Zhe. “Wnt signaling in human cartilage degeneration and chondrocytes de-differentiation : La signalisation de Wnt dans la dégradation du cartilage et la dédifférenciation chondrocytaire.” 2016. Web. 13 Apr 2021.

Vancouver:

Xie Z. Wnt signaling in human cartilage degeneration and chondrocytes de-differentiation : La signalisation de Wnt dans la dégradation du cartilage et la dédifférenciation chondrocytaire. [Internet] [Doctoral dissertation]. Université de Lorraine; 2016. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2016LORR0111.

Council of Science Editors:

Xie Z. Wnt signaling in human cartilage degeneration and chondrocytes de-differentiation : La signalisation de Wnt dans la dégradation du cartilage et la dédifférenciation chondrocytaire. [Doctoral Dissertation]. Université de Lorraine; 2016. Available from: http://www.theses.fr/2016LORR0111

24. Roszkowska, Monika. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.

Degree: Docteur es, Biochimie, 2018, Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne)

Chez les patients atteints d'athérosclérose, les calcifications vasculaires sont une caracteristique des plaques d'athérome. Elles résultent de la trans-différenciation des cellules musculaires lisses (CMLs) en… (more)

Subjects/Keywords: Cellule musculaire lisse; Phosphatase alcaline; Calcification vasculaire; Chondrocyte; Vascular smooth muscle cell; Tissue-nonspecific alkaline phosphatase; Vascular calcification; Chondrocyte; 572

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APA (6th Edition):

Roszkowska, M. (2018). Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. (Doctoral Dissertation). Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne). Retrieved from http://www.theses.fr/2018LYSE1059

Chicago Manual of Style (16th Edition):

Roszkowska, Monika. “Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.” 2018. Doctoral Dissertation, Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne). Accessed April 13, 2021. http://www.theses.fr/2018LYSE1059.

MLA Handbook (7th Edition):

Roszkowska, Monika. “Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis.” 2018. Web. 13 Apr 2021.

Vancouver:

Roszkowska M. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. [Internet] [Doctoral dissertation]. Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne); 2018. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2018LYSE1059.

Council of Science Editors:

Roszkowska M. Mécanismes moléculaires de la transdifférenciation des cellules musculaires lisses et calcification dans l'athérosclérose : Molecular mechanisms of vascular smooth muscle cell trans-differentiation and calcification in atherosclerosis. [Doctoral Dissertation]. Lyon; Instytut biologii doświadczalnej im. M. Nenckiego (Pologne); 2018. Available from: http://www.theses.fr/2018LYSE1059

25. Bouderlique, Thibault. Etude des propriétés ostéoinductrices et chondroinductrices de "l'Heparin affin regulatory peptide" sur les cellules stromales mésenchymateuses humaines, application en régénération osseuse : Study of the osteoinductive and chondroinductive properties of the heparin affin regulatory peptide on human mesenchymal stromal cells, application in bone regeneration.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2012, Université Paris-Est

 La régénération osseuse est un processus impliquant de nombreux types cellulaires comme les ostéoblastes, les chondrocytes ou les cellules stromales mésenchymateuses (CSM). Les CSM possèdent… (more)

Subjects/Keywords: Cellules souches mesenchymateuses; Harp; Ostéoblaste; Chondrocyte; Biomatériaux; Réparation osseuse; Mesenchymal stem cell; Harp; Osteoblast; Chondrocyte; Biomaterials; Bone repair

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APA (6th Edition):

Bouderlique, T. (2012). Etude des propriétés ostéoinductrices et chondroinductrices de "l'Heparin affin regulatory peptide" sur les cellules stromales mésenchymateuses humaines, application en régénération osseuse : Study of the osteoinductive and chondroinductive properties of the heparin affin regulatory peptide on human mesenchymal stromal cells, application in bone regeneration. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2012PEST0063

Chicago Manual of Style (16th Edition):

Bouderlique, Thibault. “Etude des propriétés ostéoinductrices et chondroinductrices de "l'Heparin affin regulatory peptide" sur les cellules stromales mésenchymateuses humaines, application en régénération osseuse : Study of the osteoinductive and chondroinductive properties of the heparin affin regulatory peptide on human mesenchymal stromal cells, application in bone regeneration.” 2012. Doctoral Dissertation, Université Paris-Est. Accessed April 13, 2021. http://www.theses.fr/2012PEST0063.

MLA Handbook (7th Edition):

Bouderlique, Thibault. “Etude des propriétés ostéoinductrices et chondroinductrices de "l'Heparin affin regulatory peptide" sur les cellules stromales mésenchymateuses humaines, application en régénération osseuse : Study of the osteoinductive and chondroinductive properties of the heparin affin regulatory peptide on human mesenchymal stromal cells, application in bone regeneration.” 2012. Web. 13 Apr 2021.

Vancouver:

Bouderlique T. Etude des propriétés ostéoinductrices et chondroinductrices de "l'Heparin affin regulatory peptide" sur les cellules stromales mésenchymateuses humaines, application en régénération osseuse : Study of the osteoinductive and chondroinductive properties of the heparin affin regulatory peptide on human mesenchymal stromal cells, application in bone regeneration. [Internet] [Doctoral dissertation]. Université Paris-Est; 2012. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2012PEST0063.

Council of Science Editors:

Bouderlique T. Etude des propriétés ostéoinductrices et chondroinductrices de "l'Heparin affin regulatory peptide" sur les cellules stromales mésenchymateuses humaines, application en régénération osseuse : Study of the osteoinductive and chondroinductive properties of the heparin affin regulatory peptide on human mesenchymal stromal cells, application in bone regeneration. [Doctoral Dissertation]. Université Paris-Est; 2012. Available from: http://www.theses.fr/2012PEST0063

26. Drevet, Sabine. Evaluation d'outils pour l'étude de l'arthrose à l'échelle protéique, cellulaire et animale : Evaluation of tools for the study of osteoarthritis at the protein, cellular and animal level.

Degree: Docteur es, Biologie cellulaire, 2020, Université Grenoble Alpes

L’arthrose est une pathologie chronique dégénérative invalidante, fréquente, multifactorielle, grave et coûteuse. Deux avancées conceptuelles récentes soulignent une vision pluritissulaire de la maladie et un… (more)

Subjects/Keywords: Arthrose; Chondrocyte; NADPH oxidase 4; Stress oxydant; Modèle; Animal; Osteoarthritis; Chondrocyte; NADPH oxidase 4; Oxidative stress; Model; Animal; 610

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APA (6th Edition):

Drevet, S. (2020). Evaluation d'outils pour l'étude de l'arthrose à l'échelle protéique, cellulaire et animale : Evaluation of tools for the study of osteoarthritis at the protein, cellular and animal level. (Doctoral Dissertation). Université Grenoble Alpes. Retrieved from http://www.theses.fr/2020GRALV019

Chicago Manual of Style (16th Edition):

Drevet, Sabine. “Evaluation d'outils pour l'étude de l'arthrose à l'échelle protéique, cellulaire et animale : Evaluation of tools for the study of osteoarthritis at the protein, cellular and animal level.” 2020. Doctoral Dissertation, Université Grenoble Alpes. Accessed April 13, 2021. http://www.theses.fr/2020GRALV019.

MLA Handbook (7th Edition):

Drevet, Sabine. “Evaluation d'outils pour l'étude de l'arthrose à l'échelle protéique, cellulaire et animale : Evaluation of tools for the study of osteoarthritis at the protein, cellular and animal level.” 2020. Web. 13 Apr 2021.

Vancouver:

Drevet S. Evaluation d'outils pour l'étude de l'arthrose à l'échelle protéique, cellulaire et animale : Evaluation of tools for the study of osteoarthritis at the protein, cellular and animal level. [Internet] [Doctoral dissertation]. Université Grenoble Alpes; 2020. [cited 2021 Apr 13]. Available from: http://www.theses.fr/2020GRALV019.

Council of Science Editors:

Drevet S. Evaluation d'outils pour l'étude de l'arthrose à l'échelle protéique, cellulaire et animale : Evaluation of tools for the study of osteoarthritis at the protein, cellular and animal level. [Doctoral Dissertation]. Université Grenoble Alpes; 2020. Available from: http://www.theses.fr/2020GRALV019


Universiteit Utrecht

27. Laar, A.M. van. The effect of intra-articular pressure and mechanical load on articular chondrocyte cellular signalling.

Degree: 2012, Universiteit Utrecht

 The various forms of joint loading have been found to differentially influence anatomical and molecular responses, which together are aimed to maintain the joint homeostasis.… (more)

Subjects/Keywords: articular; cartilage; chondrocyte; load; mechanical; stress; pressure; diarthrodial; joint; subchondral

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APA (6th Edition):

Laar, A. M. v. (2012). The effect of intra-articular pressure and mechanical load on articular chondrocyte cellular signalling. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/255225

Chicago Manual of Style (16th Edition):

Laar, A M van. “The effect of intra-articular pressure and mechanical load on articular chondrocyte cellular signalling.” 2012. Masters Thesis, Universiteit Utrecht. Accessed April 13, 2021. http://dspace.library.uu.nl:8080/handle/1874/255225.

MLA Handbook (7th Edition):

Laar, A M van. “The effect of intra-articular pressure and mechanical load on articular chondrocyte cellular signalling.” 2012. Web. 13 Apr 2021.

Vancouver:

Laar AMv. The effect of intra-articular pressure and mechanical load on articular chondrocyte cellular signalling. [Internet] [Masters thesis]. Universiteit Utrecht; 2012. [cited 2021 Apr 13]. Available from: http://dspace.library.uu.nl:8080/handle/1874/255225.

Council of Science Editors:

Laar AMv. The effect of intra-articular pressure and mechanical load on articular chondrocyte cellular signalling. [Masters Thesis]. Universiteit Utrecht; 2012. Available from: http://dspace.library.uu.nl:8080/handle/1874/255225


University of Rochester

28. Shen, Run; Chen, Di (1955 - ). Regulation of Runx2 protein stability in chondrocyte development.

Degree: PhD, 2009, University of Rochester

 During endochondral bone formation, cartilages provide the template for vascular invasion, which brings in osteoblasts to deposit bone matrix on the debris of apoptotic chondrocytes.… (more)

Subjects/Keywords: Ubiquitin; Runx2; Chondrocyte; PTHrP; Cartilage

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APA (6th Edition):

Shen, Run; Chen, D. (. -. ). (2009). Regulation of Runx2 protein stability in chondrocyte development. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6536

Chicago Manual of Style (16th Edition):

Shen, Run; Chen, Di (1955 - ). “Regulation of Runx2 protein stability in chondrocyte development.” 2009. Doctoral Dissertation, University of Rochester. Accessed April 13, 2021. http://hdl.handle.net/1802/6536.

MLA Handbook (7th Edition):

Shen, Run; Chen, Di (1955 - ). “Regulation of Runx2 protein stability in chondrocyte development.” 2009. Web. 13 Apr 2021.

Vancouver:

Shen, Run; Chen D(-). Regulation of Runx2 protein stability in chondrocyte development. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1802/6536.

Council of Science Editors:

Shen, Run; Chen D(-). Regulation of Runx2 protein stability in chondrocyte development. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6536


University of Rochester

29. Kotelsky, Alexander. Elucidating the factors that govern vulnerability of in situ articular chondrocytes to mechanical loading.

Degree: PhD, 2020, University of Rochester

 Articular cartilage (AC) is a load bearing tissue that covers and protects bones in synovial joints. Since resident cells (chondrocytes) in cartilage bear the responsibility… (more)

Subjects/Keywords: Cartilage; Cell volume; Cell vulnerability; Chondrocyte; Impact; Sub-impact

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kotelsky, A. (2020). Elucidating the factors that govern vulnerability of in situ articular chondrocytes to mechanical loading. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/35670

Chicago Manual of Style (16th Edition):

Kotelsky, Alexander. “Elucidating the factors that govern vulnerability of in situ articular chondrocytes to mechanical loading.” 2020. Doctoral Dissertation, University of Rochester. Accessed April 13, 2021. http://hdl.handle.net/1802/35670.

MLA Handbook (7th Edition):

Kotelsky, Alexander. “Elucidating the factors that govern vulnerability of in situ articular chondrocytes to mechanical loading.” 2020. Web. 13 Apr 2021.

Vancouver:

Kotelsky A. Elucidating the factors that govern vulnerability of in situ articular chondrocytes to mechanical loading. [Internet] [Doctoral dissertation]. University of Rochester; 2020. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1802/35670.

Council of Science Editors:

Kotelsky A. Elucidating the factors that govern vulnerability of in situ articular chondrocytes to mechanical loading. [Doctoral Dissertation]. University of Rochester; 2020. Available from: http://hdl.handle.net/1802/35670


University of Rochester

30. Catheline, Sarah Elizabeth. Inflammation in Osteoarthritis Pathogenesis: Contribution of IKKβ/NF-κB Signaling and Interplay with Chondrocyte Hypertrophy.

Degree: PhD, 2020, University of Rochester

 Osteoarthritis (OA) is a degenerative joint disease characterized by articular cartilage degradation that affects all tissues within the synovial joint. OA is associated with an… (more)

Subjects/Keywords: Aging; Cartilage; Chondrocyte; IKKβ; NF-κB signaling; Osteoarthritis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Catheline, S. E. (2020). Inflammation in Osteoarthritis Pathogenesis: Contribution of IKKβ/NF-κB Signaling and Interplay with Chondrocyte Hypertrophy. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/35815

Chicago Manual of Style (16th Edition):

Catheline, Sarah Elizabeth. “Inflammation in Osteoarthritis Pathogenesis: Contribution of IKKβ/NF-κB Signaling and Interplay with Chondrocyte Hypertrophy.” 2020. Doctoral Dissertation, University of Rochester. Accessed April 13, 2021. http://hdl.handle.net/1802/35815.

MLA Handbook (7th Edition):

Catheline, Sarah Elizabeth. “Inflammation in Osteoarthritis Pathogenesis: Contribution of IKKβ/NF-κB Signaling and Interplay with Chondrocyte Hypertrophy.” 2020. Web. 13 Apr 2021.

Vancouver:

Catheline SE. Inflammation in Osteoarthritis Pathogenesis: Contribution of IKKβ/NF-κB Signaling and Interplay with Chondrocyte Hypertrophy. [Internet] [Doctoral dissertation]. University of Rochester; 2020. [cited 2021 Apr 13]. Available from: http://hdl.handle.net/1802/35815.

Council of Science Editors:

Catheline SE. Inflammation in Osteoarthritis Pathogenesis: Contribution of IKKβ/NF-κB Signaling and Interplay with Chondrocyte Hypertrophy. [Doctoral Dissertation]. University of Rochester; 2020. Available from: http://hdl.handle.net/1802/35815

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