Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Chloride Channels metabolism 60). Showing records 1 – 30 of 16788 total matches.

[1] [2] [3] [4] [5] … [560]

Search Limiters

Last 2 Years | English Only

Degrees

Languages

Country

▼ Search Limiters

1. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 08 Mar 2021.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

2. Gaurav, Rohit. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.

Degree: PhD, Biomedical Sciences (graduate program), 2014, Creighton University

 Allergic asthma is a complex and heterogeneous disease of the airways. Eosinophils migrate to the airways under the influence of chemokines to start an excessive… (more)

Subjects/Keywords: Chloride Channels – metabolism; Asthma – immunology; Airway Inflammation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gaurav, R. (2014). Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. (Doctoral Dissertation). Creighton University. Retrieved from http://hdl.handle.net/10504/62868

Chicago Manual of Style (16th Edition):

Gaurav, Rohit. “Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.” 2014. Doctoral Dissertation, Creighton University. Accessed March 08, 2021. http://hdl.handle.net/10504/62868.

MLA Handbook (7th Edition):

Gaurav, Rohit. “Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.” 2014. Web. 08 Mar 2021.

Vancouver:

Gaurav R. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. [Internet] [Doctoral dissertation]. Creighton University; 2014. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10504/62868.

Council of Science Editors:

Gaurav R. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. [Doctoral Dissertation]. Creighton University; 2014. Available from: http://hdl.handle.net/10504/62868

3. Habela, Christa Whelan. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.

Degree: PhD, 2008, University of Alabama – Birmingham

The hypothesis that cell volume and the progression of the cell cycle are interdependent has surfaced off and on in the cell cycle literature for… (more)

Subjects/Keywords: Cell Cycle  – physiology<; br>; Cell Movement  – physiology<; br>; Cell Proliferation<; br>; Cell Size<; br>; Chloride Channels  – physiology<; br>; Chlorides  – metabolism<; br>; Cytokinesis  – physiology<; br>; Glioma  – physiopathology<; br>; Mitosis  – physiology<; br>; Neuroglia  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Habela, C. W. (2008). Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,444

Chicago Manual of Style (16th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,444.

MLA Handbook (7th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Web. 08 Mar 2021.

Vancouver:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444.

Council of Science Editors:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444

4. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 08 Mar 2021.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610

5. Ernest, Nola Jean Sieber. The role of chloride in the volume regulation of human glioma cells.

Degree: PhD, 2007, University of Alabama – Birmingham

According to the Central Brain Tumor Registry of the United States, the most common primary brain tumors are gliomas, tumors composed of cells of glial… (more)

Subjects/Keywords: Brain Neoplasms  – physiopathology <; br>; Cell Size <; br>; Chloride Channels  – physiology <; br>; Glioma  – physiopathology <; br>

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ernest, N. J. S. (2007). The role of chloride in the volume regulation of human glioma cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,86

Chicago Manual of Style (16th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,86.

MLA Handbook (7th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Web. 08 Mar 2021.

Vancouver:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86.

Council of Science Editors:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86

6. Amaral, Michelle Dawn. TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse.

Degree: PhD, 2008, University of Alabama – Birmingham

Over the years, various roles for neurotrophins have been revealed, being initially described as survival signals for neurons making their initial synaptic contacts in the… (more)

Subjects/Keywords: Brain-Derived Neurotrophic Factor <; br>; Calcium  – metabolism <; br>; Dendritic Spines  – metabolism <; br>; Hippocampus <; br>; Ion Channels  – physiology <; br>; Pyramidal Cells  – metabolism <; br>; TRPC Cation Channels

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Amaral, M. D. (2008). TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,225

Chicago Manual of Style (16th Edition):

Amaral, Michelle Dawn. “TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,225.

MLA Handbook (7th Edition):

Amaral, Michelle Dawn. “TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse.” 2008. Web. 08 Mar 2021.

Vancouver:

Amaral MD. TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,225.

Council of Science Editors:

Amaral MD. TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,225

7. Qadri, Yawar J. Small molecule inhibitors of acid sensing ion channel-1.

Degree: PhD, 2009, University of Alabama – Birmingham

Acid Sensing Ion Channel 1 is one of the many proteins in the Epithelial Sodium Channel/Degenerin family. The proteins in this family interact to form… (more)

Subjects/Keywords: Amiloride  – pharmacology<; br>; Nerve Tissue Proteins  – chemistry<; br>; Nerve Tissue Proteins  – metabolism<; br>; Sodium Channels  – chemistry<; br>; Spider Venoms  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qadri, Y. J. (2009). Small molecule inhibitors of acid sensing ion channel-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1174

Chicago Manual of Style (16th Edition):

Qadri, Yawar J. “Small molecule inhibitors of acid sensing ion channel-1.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1174.

MLA Handbook (7th Edition):

Qadri, Yawar J. “Small molecule inhibitors of acid sensing ion channel-1.” 2009. Web. 08 Mar 2021.

Vancouver:

Qadri YJ. Small molecule inhibitors of acid sensing ion channel-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1174.

Council of Science Editors:

Qadri YJ. Small molecule inhibitors of acid sensing ion channel-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1174

8. Haas, Brian Robert. The sodium-potassium-chloride cotransporter in glioma biology.

Degree: PhD, 2011, University of Alabama – Birmingham

The most common malignant primary brain tumor, gliomas usually derive from glial cells including oligodendrocytes and astrocytes. These tumors are characterized by high rates of… (more)

Subjects/Keywords: Brain Neoplasms<; br>; Bumetanide  – pharmacology<; br>; Glioma<; br>; Protein-Serine-Threonine Kinases  – metabolism<; br>; Sodium Potassium Chloride Symporter Inhibitors  – pharmacology<; br>; Sodium-Potassium-Chloride Symporters  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Haas, B. R. (2011). The sodium-potassium-chloride cotransporter in glioma biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1165

Chicago Manual of Style (16th Edition):

Haas, Brian Robert. “The sodium-potassium-chloride cotransporter in glioma biology.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1165.

MLA Handbook (7th Edition):

Haas, Brian Robert. “The sodium-potassium-chloride cotransporter in glioma biology.” 2011. Web. 08 Mar 2021.

Vancouver:

Haas BR. The sodium-potassium-chloride cotransporter in glioma biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1165.

Council of Science Editors:

Haas BR. The sodium-potassium-chloride cotransporter in glioma biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1165

9. Stout, Randy Franklin. Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans.

Degree: PhD, 2011, University of Alabama – Birmingham

A major challenge in neuroscience is understanding how the different neural cell types work together to process information and produce a behavioral output. Glial cells… (more)

Subjects/Keywords: Astrocytes  – metabolism<; br>; Caenorhabditis elegans<; br>; Caenorhabditis elegans Proteins  – metabolism<; br>; Calcium Channels, L-Type  – metabolism<; br>; Neuroglia  – metabolism<; br>; Oligodendroglia  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stout, R. F. (2011). Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1066

Chicago Manual of Style (16th Edition):

Stout, Randy Franklin. “Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1066.

MLA Handbook (7th Edition):

Stout, Randy Franklin. “Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans.” 2011. Web. 08 Mar 2021.

Vancouver:

Stout RF. Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1066.

Council of Science Editors:

Stout RF. Calcium dynamics of glial cells and genetic influences on behavior of the nematode Caenorhabditis elegans. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1066

10. McFerrin, Michael Bryan. Role of calcium-activated potassium channels in glioblastoma cell volume regulation.

Degree: PhD, 2011, University of Alabama – Birmingham

The most common and most malignant gliomas are the Grade IV Glioblastoma Multiforme (GBM), characterized by a highly proliferative tumor mass and extremely invasive phenotype… (more)

Subjects/Keywords: Apoptosis<; br>; Glioblastoma  – metabolism<; br>; Intermediate-Conductance Calcium-Activated Potassium Channels<; br>; Large-Conductance Calcium-Activated Potassium Channels<; br>; Potassium Channels, Calcium-Activated  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

McFerrin, M. B. (2011). Role of calcium-activated potassium channels in glioblastoma cell volume regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1054

Chicago Manual of Style (16th Edition):

McFerrin, Michael Bryan. “Role of calcium-activated potassium channels in glioblastoma cell volume regulation.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1054.

MLA Handbook (7th Edition):

McFerrin, Michael Bryan. “Role of calcium-activated potassium channels in glioblastoma cell volume regulation.” 2011. Web. 08 Mar 2021.

Vancouver:

McFerrin MB. Role of calcium-activated potassium channels in glioblastoma cell volume regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1054.

Council of Science Editors:

McFerrin MB. Role of calcium-activated potassium channels in glioblastoma cell volume regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1054

11. Kapoor, Niren. Role of epithelial sodium channel and acid sensing ion channel in glioblastoma multiforme.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioblastoma Multifrome is the most common and aggressive of the primary brain tumors. These tumors express multiple members of the Epithelial Sodium Channel (ENaC)/Degenerin (Deg)… (more)

Subjects/Keywords: Cell Movement<; br>; Epithelial Sodium Channel  – metabolism<; br>; Glioblastoma  – mortality<; br>; Membrane Potentials<; br>; Nerve Tissue Proteins  – metabolism<; br>; Sodium Channels  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kapoor, N. (2010). Role of epithelial sodium channel and acid sensing ion channel in glioblastoma multiforme. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,914

Chicago Manual of Style (16th Edition):

Kapoor, Niren. “Role of epithelial sodium channel and acid sensing ion channel in glioblastoma multiforme.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,914.

MLA Handbook (7th Edition):

Kapoor, Niren. “Role of epithelial sodium channel and acid sensing ion channel in glioblastoma multiforme.” 2010. Web. 08 Mar 2021.

Vancouver:

Kapoor N. Role of epithelial sodium channel and acid sensing ion channel in glioblastoma multiforme. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,914.

Council of Science Editors:

Kapoor N. Role of epithelial sodium channel and acid sensing ion channel in glioblastoma multiforme. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,914

12. Chapleau, Christopher Allen. The developmental functions of BDNF and MECP2 on dendritic and synaptic structure.

Degree: PhD, 2008, University of Alabama – Birmingham

Mutations in the transcriptional repressor MeCP2 cause Rett Syndrome (RTT), a mental retardation disease associated with reduced dendritic architecture of cortical pyramidal neurons, in addition… (more)

Subjects/Keywords: Blood Proteins  – pharmacology<; br>; Brain-Derived Neurotrophic Factor  – pharmacology<; br>; Culture Media  – pharmacology <; br>; Dendritic Spines  – drug effects <; br>; Hippocampus  – drug effects <; br>; Rett Syndrome  – metabolism <; br>; Transient Receptor Potential Channels  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chapleau, C. A. (2008). The developmental functions of BDNF and MECP2 on dendritic and synaptic structure. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,227

Chicago Manual of Style (16th Edition):

Chapleau, Christopher Allen. “The developmental functions of BDNF and MECP2 on dendritic and synaptic structure.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,227.

MLA Handbook (7th Edition):

Chapleau, Christopher Allen. “The developmental functions of BDNF and MECP2 on dendritic and synaptic structure.” 2008. Web. 08 Mar 2021.

Vancouver:

Chapleau CA. The developmental functions of BDNF and MECP2 on dendritic and synaptic structure. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,227.

Council of Science Editors:

Chapleau CA. The developmental functions of BDNF and MECP2 on dendritic and synaptic structure. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,227

13. Yuskaitis, Christopher Joseph. Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3.

Degree: PhD, 2009, University of Alabama – Birmingham

Neuroinflammation and Fragile X syndrome (FXS) are two particularly devastating neurologic conditions for which no adequate treatment exists and much is still unknown about the… (more)

Subjects/Keywords: Fragile X Mental Retardation Protein  – genetics<; br>; Gene Expression Regulation  – genetics<; br>; Glycogen Synthase Kinase 3  – antagonists & inhibitors<; br>; Inflammation Mediators  – metabolism<; br>; Lithium Chloride  – pharmacology<; br>; Microglia  – drug effects

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yuskaitis, C. J. (2009). Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,507

Chicago Manual of Style (16th Edition):

Yuskaitis, Christopher Joseph. “Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,507.

MLA Handbook (7th Edition):

Yuskaitis, Christopher Joseph. “Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3.” 2009. Web. 08 Mar 2021.

Vancouver:

Yuskaitis CJ. Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,507.

Council of Science Editors:

Yuskaitis CJ. Neuroinflammation and Fragile X syndrome: regulation by glycogen synthase kinase-3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,507


Columbia University

14. Mao, De Yu. Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling.

Degree: 2019, Columbia University

Chloride intracellular channel proteins have homology to ion channels and omega class of glutathione-S-transferases but channel activity is not well established, suggesting roles in other… (more)

Subjects/Keywords: Pharmacology; Chloride channels; Molecular biology; Cytology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mao, D. Y. (2019). Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-eyrq-5w43

Chicago Manual of Style (16th Edition):

Mao, De Yu. “Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling.” 2019. Doctoral Dissertation, Columbia University. Accessed March 08, 2021. https://doi.org/10.7916/d8-eyrq-5w43.

MLA Handbook (7th Edition):

Mao, De Yu. “Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling.” 2019. Web. 08 Mar 2021.

Vancouver:

Mao DY. Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2021 Mar 08]. Available from: https://doi.org/10.7916/d8-eyrq-5w43.

Council of Science Editors:

Mao DY. Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-eyrq-5w43

15. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 08 Mar 2021.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956


Oregon State University

16. Pang, Incheol Jonathan. Microcosm study of enhanced biotransformation of vinyl chloride to ethylene with TCE additions under anaerobic conditions from Point Mugu, California.

Degree: MS, Civil Engineering, 2000, Oregon State University

 This microcosm study demonstrated the enhanced anaerobic transformation of vinyl chloride (VC) to ethylene. A previous microcosm study from Point Mugu site showed the accumulation… (more)

Subjects/Keywords: Vinyl chloride  – Metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pang, I. J. (2000). Microcosm study of enhanced biotransformation of vinyl chloride to ethylene with TCE additions under anaerobic conditions from Point Mugu, California. (Masters Thesis). Oregon State University. Retrieved from http://hdl.handle.net/1957/33263

Chicago Manual of Style (16th Edition):

Pang, Incheol Jonathan. “Microcosm study of enhanced biotransformation of vinyl chloride to ethylene with TCE additions under anaerobic conditions from Point Mugu, California.” 2000. Masters Thesis, Oregon State University. Accessed March 08, 2021. http://hdl.handle.net/1957/33263.

MLA Handbook (7th Edition):

Pang, Incheol Jonathan. “Microcosm study of enhanced biotransformation of vinyl chloride to ethylene with TCE additions under anaerobic conditions from Point Mugu, California.” 2000. Web. 08 Mar 2021.

Vancouver:

Pang IJ. Microcosm study of enhanced biotransformation of vinyl chloride to ethylene with TCE additions under anaerobic conditions from Point Mugu, California. [Internet] [Masters thesis]. Oregon State University; 2000. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1957/33263.

Council of Science Editors:

Pang IJ. Microcosm study of enhanced biotransformation of vinyl chloride to ethylene with TCE additions under anaerobic conditions from Point Mugu, California. [Masters Thesis]. Oregon State University; 2000. Available from: http://hdl.handle.net/1957/33263

17. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 08 Mar 2021.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

18. Danilchanka, Olga V. Diffusion pathways through the outer membrane of mycobacteria.

Degree: PhD, 2009, University of Alabama – Birmingham

The extraordinary capacity of Mycobacterium tuberculosis (Mtb) to adapt to environmental changes during infection contributes to its success as a pathogen. While the unique outer… (more)

Subjects/Keywords: Anti-Bacterial Agents  – metabolism<; br>; Bacterial Proteins  – metabolism<; br>; Chloramphenicol  – metabolism<; br>; Fluoroquinolones  – metabolism<; br>; Membrane Transport Proteins  – metabolism<; br>; Mycobacterium smegmatis<; br>; Mycobacterium tuberculosis  – metabolism<; br>; Porins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Danilchanka, O. V. (2009). Diffusion pathways through the outer membrane of mycobacteria. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1150

Chicago Manual of Style (16th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1150.

MLA Handbook (7th Edition):

Danilchanka, Olga V. “Diffusion pathways through the outer membrane of mycobacteria.” 2009. Web. 08 Mar 2021.

Vancouver:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150.

Council of Science Editors:

Danilchanka OV. Diffusion pathways through the outer membrane of mycobacteria. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1150


University of Hong Kong

19. 冼世隆. Chloride channel in glioma cell invasion.

Degree: 2008, University of Hong Kong

Subjects/Keywords: Gliomas.; Chloride channels.; Cancer invasiveness.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

冼世隆.. (2008). Chloride channel in glioma cell invasion. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/54467

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

冼世隆.. “Chloride channel in glioma cell invasion.” 2008. Thesis, University of Hong Kong. Accessed March 08, 2021. http://hdl.handle.net/10722/54467.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

冼世隆.. “Chloride channel in glioma cell invasion.” 2008. Web. 08 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

冼世隆.. Chloride channel in glioma cell invasion. [Internet] [Thesis]. University of Hong Kong; 2008. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10722/54467.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

冼世隆.. Chloride channel in glioma cell invasion. [Thesis]. University of Hong Kong; 2008. Available from: http://hdl.handle.net/10722/54467

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

20. Kim, Min-Jung. Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line.

Degree: M.S. in Biomedical Sciences, Biomedical Sciences (graduate program), 2004, Creighton University

 Phagocytic cells serve critical roles in the body’s immune response by recognizing, phagocytosing and destroying foreign particles. A major class of phagocytic cells is composed… (more)

Subjects/Keywords: Chloride Channels – physiology; Monocytes – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kim, M. (2004). Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line. (Masters Thesis). Creighton University. Retrieved from http://hdl.handle.net/10504/47222

Chicago Manual of Style (16th Edition):

Kim, Min-Jung. “Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line.” 2004. Masters Thesis, Creighton University. Accessed March 08, 2021. http://hdl.handle.net/10504/47222.

MLA Handbook (7th Edition):

Kim, Min-Jung. “Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line.” 2004. Web. 08 Mar 2021.

Vancouver:

Kim M. Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line. [Internet] [Masters thesis]. Creighton University; 2004. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/10504/47222.

Council of Science Editors:

Kim M. Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line. [Masters Thesis]. Creighton University; 2004. Available from: http://hdl.handle.net/10504/47222


University of California – San Francisco

21. Wang, Lynn. Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum.

Degree: Neuroscience, 2018, University of California – San Francisco

 The lateral septum (LS) plays an important role in regulating aggression. It is well recognized that LS lesions lead to a dramatic increase in aggressive… (more)

Subjects/Keywords: Neurosciences; ano2; anoctamin2; calcium-activated chloride channels; lateral septum; tmem16b

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, L. (2018). Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/9tp5r4tm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Lynn. “Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum.” 2018. Thesis, University of California – San Francisco. Accessed March 08, 2021. http://www.escholarship.org/uc/item/9tp5r4tm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Lynn. “Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum.” 2018. Web. 08 Mar 2021.

Vancouver:

Wang L. Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum. [Internet] [Thesis]. University of California – San Francisco; 2018. [cited 2021 Mar 08]. Available from: http://www.escholarship.org/uc/item/9tp5r4tm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang L. Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum. [Thesis]. University of California – San Francisco; 2018. Available from: http://www.escholarship.org/uc/item/9tp5r4tm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

22. Harvey, Kordan. The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes.

Degree: 2012, University of Toronto

Sarcolemmal chloride channels and associated cell volume regulatory pathways have been shown to be important in local ischemic preconditioning (IPC) induced protection against myocardial ischemia/reperfusion… (more)

Subjects/Keywords: remote preconditioning; cardiomyocytes; chloride channels; volume regulation; IAA-94; 0379

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harvey, K. (2012). The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33748

Chicago Manual of Style (16th Edition):

Harvey, Kordan. “The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes.” 2012. Masters Thesis, University of Toronto. Accessed March 08, 2021. http://hdl.handle.net/1807/33748.

MLA Handbook (7th Edition):

Harvey, Kordan. “The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes.” 2012. Web. 08 Mar 2021.

Vancouver:

Harvey K. The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/1807/33748.

Council of Science Editors:

Harvey K. The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33748

23. M. Setti. SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC.

Degree: 2015, Università degli Studi di Milano

 ABSTRACT The treatment of glioblastoma (GBM) still represents a tremendous clinical challenge, with the average survival that is not exceeding 14 months. Given the lack… (more)

Subjects/Keywords: glioblastoma; chloride channels; extracellular vesicles; Settore BIO/11 - Biologia Molecolare

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Setti, M. (2015). SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/264914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Setti, M.. “SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC.” 2015. Thesis, Università degli Studi di Milano. Accessed March 08, 2021. http://hdl.handle.net/2434/264914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Setti, M.. “SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC.” 2015. Web. 08 Mar 2021.

Vancouver:

Setti M. SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2434/264914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Setti M. SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/264914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

24. Wong, Xiu Ming. CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster.

Degree: Chemistry and Chemical Biology, 2013, University of California – San Francisco

 TMEM16A and TMEM16B are calcium-activated chloride channels (CaCCs) with important functions in mammalian physiology. Whether distant relatives of the vertebrate TMEM16 families also form CaCCs… (more)

Subjects/Keywords: Biology; Calcium-activated chloride channels; Drosophila; host defense

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wong, X. M. (2013). CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/4jd0m5gb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wong, Xiu Ming. “CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster.” 2013. Thesis, University of California – San Francisco. Accessed March 08, 2021. http://www.escholarship.org/uc/item/4jd0m5gb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wong, Xiu Ming. “CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster.” 2013. Web. 08 Mar 2021.

Vancouver:

Wong XM. CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2021 Mar 08]. Available from: http://www.escholarship.org/uc/item/4jd0m5gb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wong XM. CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/4jd0m5gb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

25. Astill, David St. John. Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill.

Degree: 1996, University of Adelaide

Subjects/Keywords: 571.64/1 21; Chloride channels.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Astill, D. S. J. (1996). Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/18707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Astill, David St John. “Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill.” 1996. Thesis, University of Adelaide. Accessed March 08, 2021. http://hdl.handle.net/2440/18707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Astill, David St John. “Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill.” 1996. Web. 08 Mar 2021.

Vancouver:

Astill DSJ. Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill. [Internet] [Thesis]. University of Adelaide; 1996. [cited 2021 Mar 08]. Available from: http://hdl.handle.net/2440/18707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Astill DSJ. Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill. [Thesis]. University of Adelaide; 1996. Available from: http://hdl.handle.net/2440/18707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Salman, Emily Deanna. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.

Degree: PhD, 2011, University of Alabama – Birmingham

The human cytosolic sulfotransferases are a family of phase II drug-metabolizing enzymes that conjugate a sulfonate moiety from 3’-phosphoadenosine 5’-phosphosulfate (PAPS) to a hydroxyl moeity… (more)

Subjects/Keywords: Arylsulfotransferase  – metabolism<; br>; Brain  – enzymology<; br>; Cytosol  – enzymology<; br>; Immunohistochemistry<; br>; Sulfotransferases  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Salman, E. D. (2011). Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,960

Chicago Manual of Style (16th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,960.

MLA Handbook (7th Edition):

Salman, Emily Deanna. “Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain.” 2011. Web. 08 Mar 2021.

Vancouver:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960.

Council of Science Editors:

Salman ED. Human cytosolic sulfotransferase 2B1b: structure, function, and expression in human brain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,960

27. Yezdani, Gulam. Role of VDR in host immune response to Porphyromonas gingivalis infection.

Degree: MS, 2011, University of Alabama – Birmingham

Porphyromonas gingivalis is one of the etiologic factors of periodontal disease, a chronic inflammatory disorder characterized by the destruction of periodontal connective tissue and the… (more)

Subjects/Keywords: Mice<; br>; Porphyromonas gingivalis – metabolism<; br>; Receptors, Calcitriol – metabolism<; br>; Vitamin D – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yezdani, G. (2011). Role of VDR in host immune response to Porphyromonas gingivalis infection. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,991

Chicago Manual of Style (16th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Masters Thesis, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,991.

MLA Handbook (7th Edition):

Yezdani, Gulam. “Role of VDR in host immune response to Porphyromonas gingivalis infection.” 2011. Web. 08 Mar 2021.

Vancouver:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991.

Council of Science Editors:

Yezdani G. Role of VDR in host immune response to Porphyromonas gingivalis infection. [Masters Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,991

28. Funk, Adam J. Intracellular signaling abnormalities in schizophrenia.

Degree: PhD, 2011, University of Alabama – Birmingham

The pathophysiology of schizophrenia is complex and diverse, with many classes of receptors, neurotransmitters, and brain regions implicated in this illness. The many hypotheses proposed… (more)

Subjects/Keywords: Carrier Proteins  – metabolism<; br>; GTPase-Activating Proteins  – metabolism<; br>; Gyrus Cinguli  – metabolism<; br>; Intracellular Signaling Peptides and Proteins  – metabolism<; br>; Membrane Proteins  – metabolism<; br>; Prefrontal Cortex  – metabolism<; br>; Schizophrenia  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Funk, A. J. (2011). Intracellular signaling abnormalities in schizophrenia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1151

Chicago Manual of Style (16th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1151.

MLA Handbook (7th Edition):

Funk, Adam J. “Intracellular signaling abnormalities in schizophrenia.” 2011. Web. 08 Mar 2021.

Vancouver:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151.

Council of Science Editors:

Funk AJ. Intracellular signaling abnormalities in schizophrenia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1151

29. Ding, Huiping. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.

Degree: PhD, 2008, University of Alabama – Birmingham

Alzheimer’s disease (AD), the most common neurodegenerative disease, is pathologically characterized by senile plaques composed of amyloid [beta] peptide and neurofibrillary tangles composed of hyperphosphorylated… (more)

Subjects/Keywords: Alzheimer Disease  – metabolism<; br>; Brain  – metabolism<; br>; Caspases  – metabolism<; br>; Histone Deacetylases  – metabolism<; br>; Microtubules  – metabolism<; br>; Neurons  – metabolism<; br>; tau Proteins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ding, H. (2008). Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,439

Chicago Manual of Style (16th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,439.

MLA Handbook (7th Edition):

Ding, Huiping. “Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6.” 2008. Web. 08 Mar 2021.

Vancouver:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439.

Council of Science Editors:

Ding H. Regulation of tau functions by posttranslational modifications of tau and histone deacetylase 6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,439

30. Beagle, Brandon Richard. Canonical Wnt signaling by the proteolytic processing of LRP6.

Degree: PhD, 2010, University of Alabama – Birmingham

Low density Lipoprotein receptor Related 6 (LRP6) functions as an essential coreceptor for Wnt/β-catenin signaling as pathway activation, reflected by cytosolic β- catenin stabilization and… (more)

Subjects/Keywords: beta Catenin  – metabolism<; br>; Glycogen Synthase Kinase 3  – metabolism<; br>; LDL-Receptor Related Proteins  – metabolism<; br>; Lymphoid Enhancer-Binding Factor 1  – metabolism<; br>; Repressor Proteins  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Wnt Proteins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beagle, B. R. (2010). Canonical Wnt signaling by the proteolytic processing of LRP6. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,857

Chicago Manual of Style (16th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 08, 2021. http://contentdm.mhsl.uab.edu/u?/etd,857.

MLA Handbook (7th Edition):

Beagle, Brandon Richard. “Canonical Wnt signaling by the proteolytic processing of LRP6.” 2010. Web. 08 Mar 2021.

Vancouver:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 08]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857.

Council of Science Editors:

Beagle BR. Canonical Wnt signaling by the proteolytic processing of LRP6. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,857

[1] [2] [3] [4] [5] … [560]

.