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You searched for subject:(Chloride Channels physiology 60). Showing records 1 – 30 of 24484 total matches.

[1] [2] [3] [4] [5] … [817]

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1. Ernest, Nola Jean Sieber. The role of chloride in the volume regulation of human glioma cells.

Degree: PhD, 2007, University of Alabama – Birmingham

According to the Central Brain Tumor Registry of the United States, the most common primary brain tumors are gliomas, tumors composed of cells of glial… (more)

Subjects/Keywords: Brain Neoplasms  – physiopathology <; br>; Cell Size <; br>; Chloride Channels  – physiology <; br>; Glioma  – physiopathology <; br>

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APA (6th Edition):

Ernest, N. J. S. (2007). The role of chloride in the volume regulation of human glioma cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,86

Chicago Manual of Style (16th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,86.

MLA Handbook (7th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Web. 07 Mar 2021.

Vancouver:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86.

Council of Science Editors:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86

2. Habela, Christa Whelan. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.

Degree: PhD, 2008, University of Alabama – Birmingham

The hypothesis that cell volume and the progression of the cell cycle are interdependent has surfaced off and on in the cell cycle literature for… (more)

Subjects/Keywords: Cell Cycle  – physiology<; br>; Cell Movement  – physiology<; br>; Cell Proliferation<; br>; Cell Size<; br>; Chloride Channels  – physiology<; br>; Chlorides  – metabolism<; br>; Cytokinesis  – physiology<; br>; Glioma  – physiopathology<; br>; Mitosis  – physiology<; br>; Neuroglia  – physiology

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APA (6th Edition):

Habela, C. W. (2008). Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,444

Chicago Manual of Style (16th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,444.

MLA Handbook (7th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Web. 07 Mar 2021.

Vancouver:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444.

Council of Science Editors:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444

3. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

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APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 07 Mar 2021.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

4. Kim, Min-Jung. Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line.

Degree: M.S. in Biomedical Sciences, Biomedical Sciences (graduate program), 2004, Creighton University

 Phagocytic cells serve critical roles in the body’s immune response by recognizing, phagocytosing and destroying foreign particles. A major class of phagocytic cells is composed… (more)

Subjects/Keywords: Chloride Channels – physiology; Monocytes – physiology

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APA (6th Edition):

Kim, M. (2004). Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line. (Masters Thesis). Creighton University. Retrieved from http://hdl.handle.net/10504/47222

Chicago Manual of Style (16th Edition):

Kim, Min-Jung. “Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line.” 2004. Masters Thesis, Creighton University. Accessed March 07, 2021. http://hdl.handle.net/10504/47222.

MLA Handbook (7th Edition):

Kim, Min-Jung. “Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line.” 2004. Web. 07 Mar 2021.

Vancouver:

Kim M. Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line. [Internet] [Masters thesis]. Creighton University; 2004. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10504/47222.

Council of Science Editors:

Kim M. Chloride Channels in Normal Human Blood Monocytes and a Macrophage-like Cell Line. [Masters Thesis]. Creighton University; 2004. Available from: http://hdl.handle.net/10504/47222


University of Adelaide

5. Bennetts, Brett. Structure function studies of muscle-type CIC chloride channels.

Degree: 2006, University of Adelaide

 ClC proteins are chloride channels and transporters that are found in a wide variety of prokaryotic and eukaryotic cell-types. The mammalian chloride channel ClC-1 is… (more)

Subjects/Keywords: physiology, ion-channel, neuroscience, biophysics; Chloride channels; Ion channels

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APA (6th Edition):

Bennetts, B. (2006). Structure function studies of muscle-type CIC chloride channels. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/48393

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bennetts, Brett. “Structure function studies of muscle-type CIC chloride channels.” 2006. Thesis, University of Adelaide. Accessed March 07, 2021. http://hdl.handle.net/2440/48393.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bennetts, Brett. “Structure function studies of muscle-type CIC chloride channels.” 2006. Web. 07 Mar 2021.

Vancouver:

Bennetts B. Structure function studies of muscle-type CIC chloride channels. [Internet] [Thesis]. University of Adelaide; 2006. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2440/48393.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bennetts B. Structure function studies of muscle-type CIC chloride channels. [Thesis]. University of Adelaide; 2006. Available from: http://hdl.handle.net/2440/48393

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Amaral, Michelle Dawn. TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse.

Degree: PhD, 2008, University of Alabama – Birmingham

Over the years, various roles for neurotrophins have been revealed, being initially described as survival signals for neurons making their initial synaptic contacts in the… (more)

Subjects/Keywords: Brain-Derived Neurotrophic Factor <; br>; Calcium  – metabolism <; br>; Dendritic Spines  – metabolism <; br>; Hippocampus <; br>; Ion Channels  – physiology <; br>; Pyramidal Cells  – metabolism <; br>; TRPC Cation Channels

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APA (6th Edition):

Amaral, M. D. (2008). TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,225

Chicago Manual of Style (16th Edition):

Amaral, Michelle Dawn. “TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,225.

MLA Handbook (7th Edition):

Amaral, Michelle Dawn. “TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse.” 2008. Web. 07 Mar 2021.

Vancouver:

Amaral MD. TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,225.

Council of Science Editors:

Amaral MD. TRP-ing down a TRK : a new role for transient receptor potential channels as novel mediators of brain-derived neurotrophic factor actions at both sides of the excitatory synapse. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,225

7. Albertson, Asher J. HCN channels and regulation of neocortical network activity.

Degree: PhD, 2012, University of Alabama – Birmingham

Hyperpolarization activated non-specifc cation (HCN) channels are unique channels that activate following membrane hyperpolarization. Expressed primarily along the apical dendrites of pyramidal neurons, they pass… (more)

Subjects/Keywords: Cerebral Cortex  – physiopathology<; br>; Cyclic Nucleotide-Gated Cation Channels  – physiology<; br>; Epilepsy  – drug therapy<; br>; Epilepsy  – physiopathology<; br>; Excitatory Postsynaptic Potentials<; br>; Malformations of Cortical Development  – physiopathology<; br>; Pyramidal Cells  – physiology

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APA (6th Edition):

Albertson, A. J. (2012). HCN channels and regulation of neocortical network activity. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1133

Chicago Manual of Style (16th Edition):

Albertson, Asher J. “HCN channels and regulation of neocortical network activity.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1133.

MLA Handbook (7th Edition):

Albertson, Asher J. “HCN channels and regulation of neocortical network activity.” 2012. Web. 07 Mar 2021.

Vancouver:

Albertson AJ. HCN channels and regulation of neocortical network activity. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1133.

Council of Science Editors:

Albertson AJ. HCN channels and regulation of neocortical network activity. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1133

8. Sarfare, Shanta. Overexpression of GARP2 increases phototransduction gain.

Degree: PhD, 2010, University of Alabama – Birmingham

The cGMP-gated cation channel (CNG) in the rod photoreceptors is an essential component of phototransduction, and may have a role in maintaining structural integrity of… (more)

Subjects/Keywords: Cyclic GMP  – physiology<; br>; Cyclic Nucleotide-Gated Cation Channels<; br>; Light Signal Transduction<; br>; Mice, Transgenic<; br>; Photoreceptor Cells<; br>; Retina<; br>; Retinal Rod Photoreceptor Cells  – physiology

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APA (6th Edition):

Sarfare, S. (2010). Overexpression of GARP2 increases phototransduction gain. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,922

Chicago Manual of Style (16th Edition):

Sarfare, Shanta. “Overexpression of GARP2 increases phototransduction gain.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,922.

MLA Handbook (7th Edition):

Sarfare, Shanta. “Overexpression of GARP2 increases phototransduction gain.” 2010. Web. 07 Mar 2021.

Vancouver:

Sarfare S. Overexpression of GARP2 increases phototransduction gain. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,922.

Council of Science Editors:

Sarfare S. Overexpression of GARP2 increases phototransduction gain. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,922

9. Gaurav, Rohit. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.

Degree: PhD, Biomedical Sciences (graduate program), 2014, Creighton University

 Allergic asthma is a complex and heterogeneous disease of the airways. Eosinophils migrate to the airways under the influence of chemokines to start an excessive… (more)

Subjects/Keywords: Chloride Channels – metabolism; Asthma – immunology; Airway Inflammation

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APA (6th Edition):

Gaurav, R. (2014). Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. (Doctoral Dissertation). Creighton University. Retrieved from http://hdl.handle.net/10504/62868

Chicago Manual of Style (16th Edition):

Gaurav, Rohit. “Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.” 2014. Doctoral Dissertation, Creighton University. Accessed March 07, 2021. http://hdl.handle.net/10504/62868.

MLA Handbook (7th Edition):

Gaurav, Rohit. “Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma.” 2014. Web. 07 Mar 2021.

Vancouver:

Gaurav R. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. [Internet] [Doctoral dissertation]. Creighton University; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10504/62868.

Council of Science Editors:

Gaurav R. Chloride Channel 3 (CLC3), an Immune Antiporter in the Migration and Activation of Human Eosinophils in Allergic Asthma. [Doctoral Dissertation]. Creighton University; 2014. Available from: http://hdl.handle.net/10504/62868


Columbia University

10. Mao, De Yu. Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling.

Degree: 2019, Columbia University

Chloride intracellular channel proteins have homology to ion channels and omega class of glutathione-S-transferases but channel activity is not well established, suggesting roles in other… (more)

Subjects/Keywords: Pharmacology; Chloride channels; Molecular biology; Cytology

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APA (6th Edition):

Mao, D. Y. (2019). Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-eyrq-5w43

Chicago Manual of Style (16th Edition):

Mao, De Yu. “Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling.” 2019. Doctoral Dissertation, Columbia University. Accessed March 07, 2021. https://doi.org/10.7916/d8-eyrq-5w43.

MLA Handbook (7th Edition):

Mao, De Yu. “Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling.” 2019. Web. 07 Mar 2021.

Vancouver:

Mao DY. Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2021 Mar 07]. Available from: https://doi.org/10.7916/d8-eyrq-5w43.

Council of Science Editors:

Mao DY. Chloride Intracellular Channel (CLIC) proteins function to modulate Rac1 and RhoA downstream of endothelial G-protein coupled receptors signaling. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-eyrq-5w43

11. Wadiche, Jacques Isaac. Properties of cloned excitatory amino acid transporters.

Degree: PhD, 1998, Oregon Health Sciences University

Subjects/Keywords: Excitatory Amino Acids; Cloning, Molecular; Carrier Proteins; Chloride Channels  – physiology

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APA (6th Edition):

Wadiche, J. I. (1998). Properties of cloned excitatory amino acid transporters. (Doctoral Dissertation). Oregon Health Sciences University. Retrieved from doi:10.6083/M43N21MJ ; http://digitalcommons.ohsu.edu/etd/2592

Chicago Manual of Style (16th Edition):

Wadiche, Jacques Isaac. “Properties of cloned excitatory amino acid transporters.” 1998. Doctoral Dissertation, Oregon Health Sciences University. Accessed March 07, 2021. doi:10.6083/M43N21MJ ; http://digitalcommons.ohsu.edu/etd/2592.

MLA Handbook (7th Edition):

Wadiche, Jacques Isaac. “Properties of cloned excitatory amino acid transporters.” 1998. Web. 07 Mar 2021.

Vancouver:

Wadiche JI. Properties of cloned excitatory amino acid transporters. [Internet] [Doctoral dissertation]. Oregon Health Sciences University; 1998. [cited 2021 Mar 07]. Available from: doi:10.6083/M43N21MJ ; http://digitalcommons.ohsu.edu/etd/2592.

Council of Science Editors:

Wadiche JI. Properties of cloned excitatory amino acid transporters. [Doctoral Dissertation]. Oregon Health Sciences University; 1998. Available from: doi:10.6083/M43N21MJ ; http://digitalcommons.ohsu.edu/etd/2592


University of Hong Kong

12. Lu, Lin. The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes.

Degree: 2014, University of Hong Kong

 The cystic fibrosis transmembrane conductance regulator (CFTR) was identified to be involved in acidosis-induced ATP release from skeletal myocytes in vitro and from contracting muscle… (more)

Subjects/Keywords: Muscle cells - Physiology; Cystic fibrosis - Pathophysiology; Connexins; Chloride channels; Adenosine triphosphate

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APA (6th Edition):

Lu, L. (2014). The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/208017

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lu, Lin. “The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes.” 2014. Thesis, University of Hong Kong. Accessed March 07, 2021. http://hdl.handle.net/10722/208017.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lu, Lin. “The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes.” 2014. Web. 07 Mar 2021.

Vancouver:

Lu L. The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes. [Internet] [Thesis]. University of Hong Kong; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10722/208017.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lu L. The involvement of connexin hemichannels and cystic fibrosis transmembrane conductance regulator in acidosis-induced ATP release from skeletal myocytes. [Thesis]. University of Hong Kong; 2014. Available from: http://hdl.handle.net/10722/208017

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

13. Cai, Weisong. Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle.

Degree: 2012, University of Hong Kong

 Contracting skeletal muscle releases ATP into the interstitial space where it is subsequently broken down to adenosine by the action of ecto-5’-nucleotidase. Both ATP and… (more)

Subjects/Keywords: Cystic fibrosis - Pathophysiology.; Muscle contraction - Physiology.; Adenosine triphosphate.; Chloride channels.

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APA (6th Edition):

Cai, W. (2012). Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/180987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cai, Weisong. “Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle.” 2012. Thesis, University of Hong Kong. Accessed March 07, 2021. http://hdl.handle.net/10722/180987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cai, Weisong. “Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle.” 2012. Web. 07 Mar 2021.

Vancouver:

Cai W. Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle. [Internet] [Thesis]. University of Hong Kong; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10722/180987.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cai W. Cystic fibrosis transmembrane conductance regulator is involved in therelease of ATP from contracting skeletal muscle. [Thesis]. University of Hong Kong; 2012. Available from: http://hdl.handle.net/10722/180987

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

14. 冼世隆. Chloride channel in glioma cell invasion.

Degree: 2008, University of Hong Kong

Subjects/Keywords: Gliomas.; Chloride channels.; Cancer invasiveness.

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APA (6th Edition):

冼世隆.. (2008). Chloride channel in glioma cell invasion. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/54467

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

冼世隆.. “Chloride channel in glioma cell invasion.” 2008. Thesis, University of Hong Kong. Accessed March 07, 2021. http://hdl.handle.net/10722/54467.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

冼世隆.. “Chloride channel in glioma cell invasion.” 2008. Web. 07 Mar 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

冼世隆.. Chloride channel in glioma cell invasion. [Internet] [Thesis]. University of Hong Kong; 2008. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10722/54467.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

冼世隆.. Chloride channel in glioma cell invasion. [Thesis]. University of Hong Kong; 2008. Available from: http://hdl.handle.net/10722/54467

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

15. Wang, Lynn. Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum.

Degree: Neuroscience, 2018, University of California – San Francisco

 The lateral septum (LS) plays an important role in regulating aggression. It is well recognized that LS lesions lead to a dramatic increase in aggressive… (more)

Subjects/Keywords: Neurosciences; ano2; anoctamin2; calcium-activated chloride channels; lateral septum; tmem16b

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APA (6th Edition):

Wang, L. (2018). Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/9tp5r4tm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Lynn. “Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum.” 2018. Thesis, University of California – San Francisco. Accessed March 07, 2021. http://www.escholarship.org/uc/item/9tp5r4tm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Lynn. “Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum.” 2018. Web. 07 Mar 2021.

Vancouver:

Wang L. Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum. [Internet] [Thesis]. University of California – San Francisco; 2018. [cited 2021 Mar 07]. Available from: http://www.escholarship.org/uc/item/9tp5r4tm.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang L. Functional Studies of the TMEM16B Calcium-activated Chloride Channel in the Lateral Septum. [Thesis]. University of California – San Francisco; 2018. Available from: http://www.escholarship.org/uc/item/9tp5r4tm

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

16. Harvey, Kordan. The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes.

Degree: 2012, University of Toronto

Sarcolemmal chloride channels and associated cell volume regulatory pathways have been shown to be important in local ischemic preconditioning (IPC) induced protection against myocardial ischemia/reperfusion… (more)

Subjects/Keywords: remote preconditioning; cardiomyocytes; chloride channels; volume regulation; IAA-94; 0379

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APA (6th Edition):

Harvey, K. (2012). The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33748

Chicago Manual of Style (16th Edition):

Harvey, Kordan. “The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes.” 2012. Masters Thesis, University of Toronto. Accessed March 07, 2021. http://hdl.handle.net/1807/33748.

MLA Handbook (7th Edition):

Harvey, Kordan. “The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes.” 2012. Web. 07 Mar 2021.

Vancouver:

Harvey K. The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/1807/33748.

Council of Science Editors:

Harvey K. The Role of Chloride Channels in Remote Ischemic Preconditioning of Ventricular Cardiomyocytes. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33748

17. M. Setti. SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC.

Degree: 2015, Università degli Studi di Milano

 ABSTRACT The treatment of glioblastoma (GBM) still represents a tremendous clinical challenge, with the average survival that is not exceeding 14 months. Given the lack… (more)

Subjects/Keywords: glioblastoma; chloride channels; extracellular vesicles; Settore BIO/11 - Biologia Molecolare

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APA (6th Edition):

Setti, M. (2015). SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/264914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Setti, M.. “SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC.” 2015. Thesis, Università degli Studi di Milano. Accessed March 07, 2021. http://hdl.handle.net/2434/264914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Setti, M.. “SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC.” 2015. Web. 07 Mar 2021.

Vancouver:

Setti M. SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2434/264914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Setti M. SHEDDING LIGHT ON GLIOBLASTOMA AND DERIVED EXTRACELLULAR VESICLES IN ONE CLIC. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/264914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

18. Wong, Xiu Ming. CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster.

Degree: Chemistry and Chemical Biology, 2013, University of California – San Francisco

 TMEM16A and TMEM16B are calcium-activated chloride channels (CaCCs) with important functions in mammalian physiology. Whether distant relatives of the vertebrate TMEM16 families also form CaCCs… (more)

Subjects/Keywords: Biology; Calcium-activated chloride channels; Drosophila; host defense

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APA (6th Edition):

Wong, X. M. (2013). CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/4jd0m5gb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wong, Xiu Ming. “CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster.” 2013. Thesis, University of California – San Francisco. Accessed March 07, 2021. http://www.escholarship.org/uc/item/4jd0m5gb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wong, Xiu Ming. “CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster.” 2013. Web. 07 Mar 2021.

Vancouver:

Wong XM. CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2021 Mar 07]. Available from: http://www.escholarship.org/uc/item/4jd0m5gb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wong XM. CG16718, A Calcium-Activated Chloride Channel of the TMEM16 Family in Drosophila melanogaster. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/4jd0m5gb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

19. Astill, David St. John. Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill.

Degree: 1996, University of Adelaide

Subjects/Keywords: 571.64/1 21; Chloride channels.

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APA (6th Edition):

Astill, D. S. J. (1996). Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/18707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Astill, David St John. “Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill.” 1996. Thesis, University of Adelaide. Accessed March 07, 2021. http://hdl.handle.net/2440/18707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Astill, David St John. “Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill.” 1996. Web. 07 Mar 2021.

Vancouver:

Astill DSJ. Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill. [Internet] [Thesis]. University of Adelaide; 1996. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/2440/18707.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Astill DSJ. Characteristics of baculovirus - expressed in CIC-1 / by David St.John Astill. [Thesis]. University of Adelaide; 1996. Available from: http://hdl.handle.net/2440/18707

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Indian Institute of Science

20. Harinath, S. Pharmacological Modulation Of Recombinant Human Two-Pore Domain K+ Channels : Whole-Cell patch-Clamp Analysis.

Degree: PhD, Faculty of Science, 2011, Indian Institute of Science

 Background potassium currents play an important role in the regulation of the resting membrane potential and excitability of mammalian neurons. Recently cloned two- pore domain… (more)

Subjects/Keywords: K Channels - Physiology; K Channels - Pharmacology; Cell Patch-Clamp Analysis; Ion Channels; Two-Pore Domain Potassium Channels; K+ Channels; TREK-1 Channels; TRAAK Channels; Caffeine; Theophylline; K2P Channels; Molecular Biology

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APA (6th Edition):

Harinath, S. (2011). Pharmacological Modulation Of Recombinant Human Two-Pore Domain K+ Channels : Whole-Cell patch-Clamp Analysis. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/1501

Chicago Manual of Style (16th Edition):

Harinath, S. “Pharmacological Modulation Of Recombinant Human Two-Pore Domain K+ Channels : Whole-Cell patch-Clamp Analysis.” 2011. Doctoral Dissertation, Indian Institute of Science. Accessed March 07, 2021. http://etd.iisc.ac.in/handle/2005/1501.

MLA Handbook (7th Edition):

Harinath, S. “Pharmacological Modulation Of Recombinant Human Two-Pore Domain K+ Channels : Whole-Cell patch-Clamp Analysis.” 2011. Web. 07 Mar 2021.

Vancouver:

Harinath S. Pharmacological Modulation Of Recombinant Human Two-Pore Domain K+ Channels : Whole-Cell patch-Clamp Analysis. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2011. [cited 2021 Mar 07]. Available from: http://etd.iisc.ac.in/handle/2005/1501.

Council of Science Editors:

Harinath S. Pharmacological Modulation Of Recombinant Human Two-Pore Domain K+ Channels : Whole-Cell patch-Clamp Analysis. [Doctoral Dissertation]. Indian Institute of Science; 2011. Available from: http://etd.iisc.ac.in/handle/2005/1501


Virginia Commonwealth University

21. Deng, Wu. ROLE OF ENDOTHELIN-1 IN THE REGULATION OF THE SWELLING-ACTIVATED Cl- CURRENT IN ATRIAL MYOCYTES.

Degree: PhD, Physiology, 2009, Virginia Commonwealth University

 Swelling-activated Cl- current (ICl,swell) is an outwardly rectifying Cl- current that influences cardiac electric activities and acts as a potential effector of mechanoelectrical feedback that… (more)

Subjects/Keywords: ion channels; endothelin; reactive oxygen species; NADPH oxidase; mitochondria; cardiac myocytes; osmotic swelling; chloride channels; atrial myocytes; HL-1 cells; Life Sciences; Physiology

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APA (6th Edition):

Deng, W. (2009). ROLE OF ENDOTHELIN-1 IN THE REGULATION OF THE SWELLING-ACTIVATED Cl- CURRENT IN ATRIAL MYOCYTES. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/Q669-NV98 ; https://scholarscompass.vcu.edu/etd/12

Chicago Manual of Style (16th Edition):

Deng, Wu. “ROLE OF ENDOTHELIN-1 IN THE REGULATION OF THE SWELLING-ACTIVATED Cl- CURRENT IN ATRIAL MYOCYTES.” 2009. Doctoral Dissertation, Virginia Commonwealth University. Accessed March 07, 2021. https://doi.org/10.25772/Q669-NV98 ; https://scholarscompass.vcu.edu/etd/12.

MLA Handbook (7th Edition):

Deng, Wu. “ROLE OF ENDOTHELIN-1 IN THE REGULATION OF THE SWELLING-ACTIVATED Cl- CURRENT IN ATRIAL MYOCYTES.” 2009. Web. 07 Mar 2021.

Vancouver:

Deng W. ROLE OF ENDOTHELIN-1 IN THE REGULATION OF THE SWELLING-ACTIVATED Cl- CURRENT IN ATRIAL MYOCYTES. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2009. [cited 2021 Mar 07]. Available from: https://doi.org/10.25772/Q669-NV98 ; https://scholarscompass.vcu.edu/etd/12.

Council of Science Editors:

Deng W. ROLE OF ENDOTHELIN-1 IN THE REGULATION OF THE SWELLING-ACTIVATED Cl- CURRENT IN ATRIAL MYOCYTES. [Doctoral Dissertation]. Virginia Commonwealth University; 2009. Available from: https://doi.org/10.25772/Q669-NV98 ; https://scholarscompass.vcu.edu/etd/12


Universitat de Barcelona

22. Pérez Rius, Carla. Model animal de malalties associades a canals de clorur en peix zebra.

Degree: Departament de Ciències Fisiològiques, 2017, Universitat de Barcelona

Chloride is the most abundant anion in body fluids. Chloride transport is involved in many different cellular processes, such as regulating the excitability of muscle… (more)

Subjects/Keywords: Fisiologia; Fisiología; Physiology; Aparell locomotor; Aparato locomotor; Musculoskeletal system; Canals de clorur; Canales de cloruros; Chloride channels; Peix zebra; Pez cebra; Zebra danio; Proteïnes; Proteínas; Proteins; Ciències de la Salut; 612

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APA (6th Edition):

Pérez Rius, C. (2017). Model animal de malalties associades a canals de clorur en peix zebra. (Thesis). Universitat de Barcelona. Retrieved from http://hdl.handle.net/10803/482037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pérez Rius, Carla. “Model animal de malalties associades a canals de clorur en peix zebra.” 2017. Thesis, Universitat de Barcelona. Accessed March 07, 2021. http://hdl.handle.net/10803/482037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pérez Rius, Carla. “Model animal de malalties associades a canals de clorur en peix zebra.” 2017. Web. 07 Mar 2021.

Vancouver:

Pérez Rius C. Model animal de malalties associades a canals de clorur en peix zebra. [Internet] [Thesis]. Universitat de Barcelona; 2017. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10803/482037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pérez Rius C. Model animal de malalties associades a canals de clorur en peix zebra. [Thesis]. Universitat de Barcelona; 2017. Available from: http://hdl.handle.net/10803/482037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Dobbins, George C. (George Clement). The role of the cytoskeleton in AChR clustering.

Degree: PhD, 2007, University of Alabama – Birmingham

The human brain contains billions of neurons which are designed to receive and integrate a range of electrical and biochemical signals. The ability to convey… (more)

Subjects/Keywords: Cytoskeleton  – physiology <; br>; Neuromuscular Junction  – physiology <; br>; Receptors, Cholinergic  – physiology

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APA (6th Edition):

Dobbins, G. C. (. C. (2007). The role of the cytoskeleton in AChR clustering. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,119

Chicago Manual of Style (16th Edition):

Dobbins, George C (George Clement). “The role of the cytoskeleton in AChR clustering.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,119.

MLA Handbook (7th Edition):

Dobbins, George C (George Clement). “The role of the cytoskeleton in AChR clustering.” 2007. Web. 07 Mar 2021.

Vancouver:

Dobbins GC(C. The role of the cytoskeleton in AChR clustering. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,119.

Council of Science Editors:

Dobbins GC(C. The role of the cytoskeleton in AChR clustering. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,119


University of Missouri – Columbia

24. Lett, Kawasi M. Loss of afferent input alters voltage gated ion channel and neuromodulator receptor expression in crustacean and mammalian neurons.

Degree: 2014, University of Missouri – Columbia

 [ACCESS RESTRICTED TO THE UNIVERSITY OF MISSOURI AT AUTHOR'S REQUEST.] Neural networks need to generate appropriate output regardless of external stimuli, to ensure survival of… (more)

Subjects/Keywords: Neurotransmitter receptors; Neurons  – Physiology; Homeostasis; Ion channels

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APA (6th Edition):

Lett, K. M. (2014). Loss of afferent input alters voltage gated ion channel and neuromodulator receptor expression in crustacean and mammalian neurons. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/45807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lett, Kawasi M. “Loss of afferent input alters voltage gated ion channel and neuromodulator receptor expression in crustacean and mammalian neurons.” 2014. Thesis, University of Missouri – Columbia. Accessed March 07, 2021. http://hdl.handle.net/10355/45807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lett, Kawasi M. “Loss of afferent input alters voltage gated ion channel and neuromodulator receptor expression in crustacean and mammalian neurons.” 2014. Web. 07 Mar 2021.

Vancouver:

Lett KM. Loss of afferent input alters voltage gated ion channel and neuromodulator receptor expression in crustacean and mammalian neurons. [Internet] [Thesis]. University of Missouri – Columbia; 2014. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10355/45807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lett KM. Loss of afferent input alters voltage gated ion channel and neuromodulator receptor expression in crustacean and mammalian neurons. [Thesis]. University of Missouri – Columbia; 2014. Available from: http://hdl.handle.net/10355/45807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Davison, Ryan C. The behavior of oculomotor neurons during conjugate and disconjugate eye movements.

Degree: PhD, 2007, University of Alabama – Birmingham

Conjugate eye movements are those that occur when both eyes move the same amount in the same direction. Saccades and smooth pursuit have classically been… (more)

Subjects/Keywords: Convergence, Ocular  – physiology<; br>; Eye Movements  – physiology<; br>; Motor Neurons  – physiology<; br>; Pursuit, Smooth  – physiology<; br>; Saccades  – physiology

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APA (6th Edition):

Davison, R. C. (2007). The behavior of oculomotor neurons during conjugate and disconjugate eye movements. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,435

Chicago Manual of Style (16th Edition):

Davison, Ryan C. “The behavior of oculomotor neurons during conjugate and disconjugate eye movements.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,435.

MLA Handbook (7th Edition):

Davison, Ryan C. “The behavior of oculomotor neurons during conjugate and disconjugate eye movements.” 2007. Web. 07 Mar 2021.

Vancouver:

Davison RC. The behavior of oculomotor neurons during conjugate and disconjugate eye movements. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,435.

Council of Science Editors:

Davison RC. The behavior of oculomotor neurons during conjugate and disconjugate eye movements. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,435

26. Cottingham, Christopher. Neuropharmacology Of The α2a Adrenergic Receptor In Disorders Of Mood And Cognition.

Degree: PhD, 2012, University of Alabama – Birmingham

Depressive disorders carry relatively high lifetime risks of greater than 10%, and the antidepressant drugs used in the pharmacotherapy of these mood/cognitive disorders are among… (more)

Subjects/Keywords: Actins – metabolism<; br>; Amygdala – physiology<; br>; Conditioning, Classical – physiology.<; br>; Fear – physiology<; br>; Long-Term Potentiation – physiology.<; br>; Memory – physiology<; br>; Nonmuscle Myosin Type IIB – metabolism.<; br>; Synapses – physiology

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APA (6th Edition):

Cottingham, C. (2012). Neuropharmacology Of The α2a Adrenergic Receptor In Disorders Of Mood And Cognition. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1393

Chicago Manual of Style (16th Edition):

Cottingham, Christopher. “Neuropharmacology Of The α2a Adrenergic Receptor In Disorders Of Mood And Cognition.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1393.

MLA Handbook (7th Edition):

Cottingham, Christopher. “Neuropharmacology Of The α2a Adrenergic Receptor In Disorders Of Mood And Cognition.” 2012. Web. 07 Mar 2021.

Vancouver:

Cottingham C. Neuropharmacology Of The α2a Adrenergic Receptor In Disorders Of Mood And Cognition. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1393.

Council of Science Editors:

Cottingham C. Neuropharmacology Of The α2a Adrenergic Receptor In Disorders Of Mood And Cognition. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1393

27. Olteanu, Dragos S. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.

Degree: PhD, 2007, University of Alabama – Birmingham

Polycystic kidney disease in both its recessive and dominant forms involves the remodeling of the kidney and extra-renal tissues where parts of the tissue break… (more)

Subjects/Keywords: Cilia  – metabolism<; br>; Epithelial Cells<; br>; Kidney<; br>; Polycystic Kidney, Autosomal Recessive  – metabolism<; br>; Sodium  – metabolism<; br>; Sodium Channels  – metabolism

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APA (6th Edition):

Olteanu, D. S. (2007). Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,610

Chicago Manual of Style (16th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,610.

MLA Handbook (7th Edition):

Olteanu, Dragos S. “Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease.” 2007. Web. 07 Mar 2021.

Vancouver:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610.

Council of Science Editors:

Olteanu DS. Dysregulated ENAC and NHE function in cilium-deficient renal collecting duct cell monolayers : a model of polycystic kidney disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,610


University of New South Wales

28. Warton, Kristina. Characterization of CLIC1 : a chloride ion channel which can exist in both a transmembrane and a soluble state.

Degree: Medicine. Clinical School - St Vincent's Hospital, 2002, University of New South Wales

Subjects/Keywords: Chloride channels; Ion channels; Cell membranes; Thesis Digitisation Program

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warton, K. (2002). Characterization of CLIC1 : a chloride ion channel which can exist in both a transmembrane and a soluble state. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/64448 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:62158/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Warton, Kristina. “Characterization of CLIC1 : a chloride ion channel which can exist in both a transmembrane and a soluble state.” 2002. Doctoral Dissertation, University of New South Wales. Accessed March 07, 2021. http://handle.unsw.edu.au/1959.4/64448 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:62158/SOURCE01?view=true.

MLA Handbook (7th Edition):

Warton, Kristina. “Characterization of CLIC1 : a chloride ion channel which can exist in both a transmembrane and a soluble state.” 2002. Web. 07 Mar 2021.

Vancouver:

Warton K. Characterization of CLIC1 : a chloride ion channel which can exist in both a transmembrane and a soluble state. [Internet] [Doctoral dissertation]. University of New South Wales; 2002. [cited 2021 Mar 07]. Available from: http://handle.unsw.edu.au/1959.4/64448 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:62158/SOURCE01?view=true.

Council of Science Editors:

Warton K. Characterization of CLIC1 : a chloride ion channel which can exist in both a transmembrane and a soluble state. [Doctoral Dissertation]. University of New South Wales; 2002. Available from: http://handle.unsw.edu.au/1959.4/64448 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:62158/SOURCE01?view=true

29. Rubio, Maria Dolores. Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms.

Degree: PhD, 2011, University of Alabama – Birmingham

Cytoskeletal actin filaments underlie dendritic spine plasticity, critical for several forms of learning and memory. Therefore, understanding the mechanisms that regulate actin dynamics is essential… (more)

Subjects/Keywords: Actins  – metabolism<; br>; Cardiac Myosins  – physiology<; br>; Long-Term Potentiation  – physiology<; br>; Memory  – physiology<; br>; Myosin Heavy Chains  – physiology<; br>; Neurons  – metabolism<; br>; Nonmuscle Myosin Type IIB  – metabolism<; br>; Synapses  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rubio, M. D. (2011). Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,878

Chicago Manual of Style (16th Edition):

Rubio, Maria Dolores. “Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,878.

MLA Handbook (7th Edition):

Rubio, Maria Dolores. “Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms.” 2011. Web. 07 Mar 2021.

Vancouver:

Rubio MD. Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,878.

Council of Science Editors:

Rubio MD. Myosin II in hippocampal synapses: regulation of synaptic plasticity, strength and actin dynamics by two distinct isoforms. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,878


University of Hong Kong

30. Li, Xiang. Using alpha-aminoxy acids as building blocks to construct anion receptors and synthetic chloride channels.

Degree: 2008, University of Hong Kong

Subjects/Keywords: Chloride channels.; Anions - Receptors.; Amino acids.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Li, X. (2008). Using alpha-aminoxy acids as building blocks to construct anion receptors and synthetic chloride channels. (Thesis). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/50389

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Li, Xiang. “Using alpha-aminoxy acids as building blocks to construct anion receptors and synthetic chloride channels.” 2008. Thesis, University of Hong Kong. Accessed March 07, 2021. http://hdl.handle.net/10722/50389.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Li, Xiang. “Using alpha-aminoxy acids as building blocks to construct anion receptors and synthetic chloride channels.” 2008. Web. 07 Mar 2021.

Vancouver:

Li X. Using alpha-aminoxy acids as building blocks to construct anion receptors and synthetic chloride channels. [Internet] [Thesis]. University of Hong Kong; 2008. [cited 2021 Mar 07]. Available from: http://hdl.handle.net/10722/50389.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li X. Using alpha-aminoxy acids as building blocks to construct anion receptors and synthetic chloride channels. [Thesis]. University of Hong Kong; 2008. Available from: http://hdl.handle.net/10722/50389

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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