Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Chemokines). Showing records 1 – 30 of 366 total matches.

[1] [2] [3] [4] [5] … [13]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Aberdeen

1. Yu, Tian. Role of atypical chemokine receptor-2 in ocular inflammation.

Degree: PhD, 2015, University of Aberdeen

 The atypical chemokine receptor-2 (ACKR2) is a chemokine decoy receptor that recognises pro-inflammatory CC chemokines. Many studies showed up-regulated inflammation and delayed resolution of inflammatory… (more)

Subjects/Keywords: 617.7; Eye; Chemokines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, T. (2015). Role of atypical chemokine receptor-2 in ocular inflammation. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229021 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996

Chicago Manual of Style (16th Edition):

Yu, Tian. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Doctoral Dissertation, University of Aberdeen. Accessed February 22, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229021 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996.

MLA Handbook (7th Edition):

Yu, Tian. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Web. 22 Feb 2020.

Vancouver:

Yu T. Role of atypical chemokine receptor-2 in ocular inflammation. [Internet] [Doctoral dissertation]. University of Aberdeen; 2015. [cited 2020 Feb 22]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229021 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996.

Council of Science Editors:

Yu T. Role of atypical chemokine receptor-2 in ocular inflammation. [Doctoral Dissertation]. University of Aberdeen; 2015. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=229021 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.680996


University of Hong Kong

2. 宋蘭。; Sung, Lan, Fion. Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1).

Degree: M. Phil., 1999, University of Hong Kong

published_or_final_version

Pharmacology

Master

Master of Philosophy

Subjects/Keywords: Chemokines.; Homocysteine.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

宋蘭。; Sung, Lan, F. (1999). Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). (Masters Thesis). University of Hong Kong. Retrieved from Sung, L. F. [宋蘭。]. (1999). Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122165 ; http://dx.doi.org/10.5353/th_b3122165 ; http://hdl.handle.net/10722/56679

Chicago Manual of Style (16th Edition):

宋蘭。; Sung, Lan, Fion. “Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1).” 1999. Masters Thesis, University of Hong Kong. Accessed February 22, 2020. Sung, L. F. [宋蘭。]. (1999). Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122165 ; http://dx.doi.org/10.5353/th_b3122165 ; http://hdl.handle.net/10722/56679.

MLA Handbook (7th Edition):

宋蘭。; Sung, Lan, Fion. “Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1).” 1999. Web. 22 Feb 2020.

Vancouver:

宋蘭。; Sung, Lan F. Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). [Internet] [Masters thesis]. University of Hong Kong; 1999. [cited 2020 Feb 22]. Available from: Sung, L. F. [宋蘭。]. (1999). Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122165 ; http://dx.doi.org/10.5353/th_b3122165 ; http://hdl.handle.net/10722/56679.

Council of Science Editors:

宋蘭。; Sung, Lan F. Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). [Masters Thesis]. University of Hong Kong; 1999. Available from: Sung, L. F. [宋蘭。]. (1999). Role of homocysteine in the expression of monocyte Chemoattractant protein-1 (MCP-1). (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122165 ; http://dx.doi.org/10.5353/th_b3122165 ; http://hdl.handle.net/10722/56679


University of Louisville

3. Mosley, LaSharon Denise. Mechanisms mediated by CXCL12 signaling through CXCR4 and CXCR7 in breast cancer.

Degree: PhD, 2009, University of Louisville

 Interactions between chemokines and their receptors are involved in organ- specific homing and propagation of metastatic breast cancer (BrCa) cells. BrCa cells express higher levels… (more)

Subjects/Keywords: Breast cancer; Chemokines; CXCR4; CXCR7

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mosley, L. D. (2009). Mechanisms mediated by CXCL12 signaling through CXCR4 and CXCR7 in breast cancer. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/1012 ; https://ir.library.louisville.edu/etd/1012

Chicago Manual of Style (16th Edition):

Mosley, LaSharon Denise. “Mechanisms mediated by CXCL12 signaling through CXCR4 and CXCR7 in breast cancer.” 2009. Doctoral Dissertation, University of Louisville. Accessed February 22, 2020. 10.18297/etd/1012 ; https://ir.library.louisville.edu/etd/1012.

MLA Handbook (7th Edition):

Mosley, LaSharon Denise. “Mechanisms mediated by CXCL12 signaling through CXCR4 and CXCR7 in breast cancer.” 2009. Web. 22 Feb 2020.

Vancouver:

Mosley LD. Mechanisms mediated by CXCL12 signaling through CXCR4 and CXCR7 in breast cancer. [Internet] [Doctoral dissertation]. University of Louisville; 2009. [cited 2020 Feb 22]. Available from: 10.18297/etd/1012 ; https://ir.library.louisville.edu/etd/1012.

Council of Science Editors:

Mosley LD. Mechanisms mediated by CXCL12 signaling through CXCR4 and CXCR7 in breast cancer. [Doctoral Dissertation]. University of Louisville; 2009. Available from: 10.18297/etd/1012 ; https://ir.library.louisville.edu/etd/1012


University of Aberdeen

4. Dagkalis, Athanasios. CCR2 and CX3CR1 in monocyte trafficking in experimental autoimmune uveoretinitis.

Degree: 2008, University of Aberdeen

 We used Experimental Autoimmune Uveoretinitis (EAU) as a model system to investigate the involvement of CCR2 and CX3CR1 in regulating the trafficking and function of… (more)

Subjects/Keywords: 616.079; Chemokines : Cell receptors : Inflammation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dagkalis, A. (2008). CCR2 and CX3CR1 in monocyte trafficking in experimental autoimmune uveoretinitis. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=24809 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495031

Chicago Manual of Style (16th Edition):

Dagkalis, Athanasios. “CCR2 and CX3CR1 in monocyte trafficking in experimental autoimmune uveoretinitis.” 2008. Doctoral Dissertation, University of Aberdeen. Accessed February 22, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=24809 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495031.

MLA Handbook (7th Edition):

Dagkalis, Athanasios. “CCR2 and CX3CR1 in monocyte trafficking in experimental autoimmune uveoretinitis.” 2008. Web. 22 Feb 2020.

Vancouver:

Dagkalis A. CCR2 and CX3CR1 in monocyte trafficking in experimental autoimmune uveoretinitis. [Internet] [Doctoral dissertation]. University of Aberdeen; 2008. [cited 2020 Feb 22]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=24809 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495031.

Council of Science Editors:

Dagkalis A. CCR2 and CX3CR1 in monocyte trafficking in experimental autoimmune uveoretinitis. [Doctoral Dissertation]. University of Aberdeen; 2008. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?pid=24809 ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495031


North Carolina State University

5. Bost, Phillip Chapman. Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis.

Degree: MS, Toxicology, 2009, North Carolina State University

 The purpose of this research was to investigate the potential of a therapeutic vanadium compound in the modification and regulation of common repair mechanisms associated… (more)

Subjects/Keywords: STAT; chemokines; fibrosis; vanadium

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bost, P. C. (2009). Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis. (Thesis). North Carolina State University. Retrieved from http://www.lib.ncsu.edu/resolver/1840.16/851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bost, Phillip Chapman. “Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis.” 2009. Thesis, North Carolina State University. Accessed February 22, 2020. http://www.lib.ncsu.edu/resolver/1840.16/851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bost, Phillip Chapman. “Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis.” 2009. Web. 22 Feb 2020.

Vancouver:

Bost PC. Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis. [Internet] [Thesis]. North Carolina State University; 2009. [cited 2020 Feb 22]. Available from: http://www.lib.ncsu.edu/resolver/1840.16/851.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bost PC. Bis(maltolato)oxovanadium(IV) Activation of STAT-1 Signaling in Fibroblasts as a Potential Therapy for Pulmonary Fibrosis. [Thesis]. North Carolina State University; 2009. Available from: http://www.lib.ncsu.edu/resolver/1840.16/851

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

6. Thach, Chia Tha. Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure.

Degree: PhD, 2013, University of Rochester

 With the increase in the production of engineered nanomaterials, researchers are discovering that there is a direct impact of these nanomaterials on the cell membrane.… (more)

Subjects/Keywords: Nanoparticles; Cellular signaling; Chemokines; Cholesterol

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Thach, C. T. (2013). Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/26782

Chicago Manual of Style (16th Edition):

Thach, Chia Tha. “Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure.” 2013. Doctoral Dissertation, University of Rochester. Accessed February 22, 2020. http://hdl.handle.net/1802/26782.

MLA Handbook (7th Edition):

Thach, Chia Tha. “Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure.” 2013. Web. 22 Feb 2020.

Vancouver:

Thach CT. Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/1802/26782.

Council of Science Editors:

Thach CT. Modulation of Cellular Signaling Upon Cholesterol Depletion and Nanoparticle Exposure. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/26782


University of Southern California

7. Tam, Yvonne. Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics.

Degree: MS, Cranio-Facial Biology, 2012, University of Southern California

 Background: Some investigators have detected edema, swelling and histological changes in the soft tissue under pontics, while others have demonstrated that exceptional plaque control can… (more)

Subjects/Keywords: cytokines; chemokines; implants; pontics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tam, Y. (2012). Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1298

Chicago Manual of Style (16th Edition):

Tam, Yvonne. “Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics.” 2012. Masters Thesis, University of Southern California. Accessed February 22, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1298.

MLA Handbook (7th Edition):

Tam, Yvonne. “Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics.” 2012. Web. 22 Feb 2020.

Vancouver:

Tam Y. Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Feb 22]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1298.

Council of Science Editors:

Tam Y. Characterization of cytokine/chemokine and microbiology profiles of peri-implant sulci and implant-supported ridge lap pontics. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93541/rec/1298


University of Vermont

8. Arms, Lauren. Inflammation-Induced Plasticity of Micturition Reflex Pathways.

Degree: PhD, Neuroscience, 2011, University of Vermont

 Although a seemingly basic and simple behavior, micturition necessitates precise integration and coordination of multiple divisions of the nervous system: visceral sensory, somatic motor, sympathetic,… (more)

Subjects/Keywords: Cyclophosphamide; Cystometry; pAKT; Chemokines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Arms, L. (2011). Inflammation-Induced Plasticity of Micturition Reflex Pathways. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/9

Chicago Manual of Style (16th Edition):

Arms, Lauren. “Inflammation-Induced Plasticity of Micturition Reflex Pathways.” 2011. Doctoral Dissertation, University of Vermont. Accessed February 22, 2020. https://scholarworks.uvm.edu/graddis/9.

MLA Handbook (7th Edition):

Arms, Lauren. “Inflammation-Induced Plasticity of Micturition Reflex Pathways.” 2011. Web. 22 Feb 2020.

Vancouver:

Arms L. Inflammation-Induced Plasticity of Micturition Reflex Pathways. [Internet] [Doctoral dissertation]. University of Vermont; 2011. [cited 2020 Feb 22]. Available from: https://scholarworks.uvm.edu/graddis/9.

Council of Science Editors:

Arms L. Inflammation-Induced Plasticity of Micturition Reflex Pathways. [Doctoral Dissertation]. University of Vermont; 2011. Available from: https://scholarworks.uvm.edu/graddis/9


University of Vermont

9. Guo, Michael. The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse.

Degree: Neurological Sciences, 2017, University of Vermont

  Interstitial cystitis is a serious chronic condition that causes bladder pain and increased voiding frequency in millions of adults in the US, most of… (more)

Subjects/Keywords: chemokines; cystitis; urinary bladder; inflammation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guo, M. (2017). The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/hcoltheses/193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guo, Michael. “The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse.” 2017. Thesis, University of Vermont. Accessed February 22, 2020. https://scholarworks.uvm.edu/hcoltheses/193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guo, Michael. “The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse.” 2017. Web. 22 Feb 2020.

Vancouver:

Guo M. The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse. [Internet] [Thesis]. University of Vermont; 2017. [cited 2020 Feb 22]. Available from: https://scholarworks.uvm.edu/hcoltheses/193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guo M. The Expression and Regulation of Chemokines (CXCL9, CXCL10, CXCL11) in Urinary Bladder Inflammation of the Mouse. [Thesis]. University of Vermont; 2017. Available from: https://scholarworks.uvm.edu/hcoltheses/193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Aberdeen

10. Yu, Tian.; University of Aberdeen.Dept. of Immunity, Infection and Inflammation.; University of Aberdeen.Development Trust. Role of atypical chemokine receptor-2 in ocular inflammation.

Degree: Dept. of Immunity, Infection and Inflammation., 2015, University of Aberdeen

Subjects/Keywords: Eye; Chemokines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, Tian.; University of Aberdeen.Dept. of Immunity, I. a. I. ;. U. o. A. D. T. (2015). Role of atypical chemokine receptor-2 in ocular inflammation. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool2.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Yu, Tian.; University of Aberdeen.Dept. of Immunity, Infection and Inflammation ; University of Aberdeen Development Trust. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Doctoral Dissertation, University of Aberdeen. Accessed February 22, 2020. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool2.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Yu, Tian.; University of Aberdeen.Dept. of Immunity, Infection and Inflammation ; University of Aberdeen Development Trust. “Role of atypical chemokine receptor-2 in ocular inflammation.” 2015. Web. 22 Feb 2020.

Vancouver:

Yu, Tian.; University of Aberdeen.Dept. of Immunity IaI;UoADT. Role of atypical chemokine receptor-2 in ocular inflammation. [Internet] [Doctoral dissertation]. University of Aberdeen; 2015. [cited 2020 Feb 22]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool2.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer.

Council of Science Editors:

Yu, Tian.; University of Aberdeen.Dept. of Immunity IaI;UoADT. Role of atypical chemokine receptor-2 in ocular inflammation. [Doctoral Dissertation]. University of Aberdeen; 2015. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=229021 ; http://digitool2.abdn.ac.uk:1801/webclient/DeliveryManager?pid=229021&custom_att_2=simple_viewer


University of North Carolina – Greensboro

11. Paila, Hari Srinivas Kalyan. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.

Degree: 2016, University of North Carolina – Greensboro

 Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization by mass spectrometry, and phosphoproteomic methods… (more)

Subjects/Keywords: Chemokines – Receptors; Cellular signal transduction

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paila, H. S. K. (2016). Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. (Doctoral Dissertation). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370

Chicago Manual of Style (16th Edition):

Paila, Hari Srinivas Kalyan. “Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.” 2016. Doctoral Dissertation, University of North Carolina – Greensboro. Accessed February 22, 2020. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370.

MLA Handbook (7th Edition):

Paila, Hari Srinivas Kalyan. “Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway.” 2016. Web. 22 Feb 2020.

Vancouver:

Paila HSK. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. [Internet] [Doctoral dissertation]. University of North Carolina – Greensboro; 2016. [cited 2020 Feb 22]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370.

Council of Science Editors:

Paila HSK. Studies of the human CCR3 chemokine receptor: development of a cell line stably expressing CCR3, receptor purification and characterization, and phosphopeptide enrichment methods to study the CCR3 GPCR signaling pathway. [Doctoral Dissertation]. University of North Carolina – Greensboro; 2016. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=21370


University of California – San Francisco

12. Kelly, Lisa. EBI2 positions naïve and activated B cells.

Degree: Biomedical Sciences, 2011, University of California – San Francisco

 The immune system is organized to allow lymphocytes to survey for antigen and rapidly respond to an infection. B lymphocytes reside in B cell follicles… (more)

Subjects/Keywords: Immunology; B cells; chemokines; GPCR

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kelly, L. (2011). EBI2 positions naïve and activated B cells. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/0cf2v6js

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kelly, Lisa. “EBI2 positions naïve and activated B cells.” 2011. Thesis, University of California – San Francisco. Accessed February 22, 2020. http://www.escholarship.org/uc/item/0cf2v6js.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kelly, Lisa. “EBI2 positions naïve and activated B cells.” 2011. Web. 22 Feb 2020.

Vancouver:

Kelly L. EBI2 positions naïve and activated B cells. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2020 Feb 22]. Available from: http://www.escholarship.org/uc/item/0cf2v6js.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kelly L. EBI2 positions naïve and activated B cells. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/0cf2v6js

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Chicago

13. Patel, Ankur S. The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth.

Degree: 2016, University of Illinois – Chicago

 This study investigated the role of chemokines in Cyclosporine (CsA) induced gingival overgrowth (GO). 14 patients with renal transplants undergoing CsA treatment and 3 healthy… (more)

Subjects/Keywords: gingival overgrowth; cyclosporine; chemokines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Patel, A. S. (2016). The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Patel, Ankur S. “The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth.” 2016. Thesis, University of Illinois – Chicago. Accessed February 22, 2020. http://hdl.handle.net/10027/21278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Patel, Ankur S. “The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth.” 2016. Web. 22 Feb 2020.

Vancouver:

Patel AS. The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth. [Internet] [Thesis]. University of Illinois – Chicago; 2016. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/10027/21278.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Patel AS. The Role of Chemokines in Cyclosporine Induced Gingival Overgrowth. [Thesis]. University of Illinois – Chicago; 2016. Available from: http://hdl.handle.net/10027/21278

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tennessee – Knoxville

14. Dogra, Pranay. CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil.

Degree: 2015, University of Tennessee – Knoxville

 Human Cytomegalovirus (HCMV) is the leading cause of both non-hereditary mental retardation and hearing loss, and CMV infection/reactivation causes serious complications in transplant and immune… (more)

Subjects/Keywords: Cytomegalovirus; Chemokines; Co-infection; Viral Chemokines; Antiviral Peptides; Other Microbiology; Virology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dogra, P. (2015). CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil. (Doctoral Dissertation). University of Tennessee – Knoxville. Retrieved from https://trace.tennessee.edu/utk_graddiss/3497

Chicago Manual of Style (16th Edition):

Dogra, Pranay. “CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil.” 2015. Doctoral Dissertation, University of Tennessee – Knoxville. Accessed February 22, 2020. https://trace.tennessee.edu/utk_graddiss/3497.

MLA Handbook (7th Edition):

Dogra, Pranay. “CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil.” 2015. Web. 22 Feb 2020.

Vancouver:

Dogra P. CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil. [Internet] [Doctoral dissertation]. University of Tennessee – Knoxville; 2015. [cited 2020 Feb 22]. Available from: https://trace.tennessee.edu/utk_graddiss/3497.

Council of Science Editors:

Dogra P. CMV Chemokines and Co-infection: A Dissemination Plot that Peptides Can Foil. [Doctoral Dissertation]. University of Tennessee – Knoxville; 2015. Available from: https://trace.tennessee.edu/utk_graddiss/3497

15. Sipert, Carla Renata. Produção de MIP-1alfa e SDF-1 por fibroblastos de polpa dental humana em cultura frente ao desafio com Enterococcus faecalis inativado por calor.

Degree: Mestrado, Endodontia, 2007, University of São Paulo

A polpa dental é formada de tecido conjuntivo frouxo sendo constituída por diversas células, dentre as quais os fibroblastos são as mais numerosas. Ao serem… (more)

Subjects/Keywords: Chemokines; Fibroblastos; Fibroblasts; Inflamação; Inflammation; Quimiocinas

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sipert, C. R. (2007). Produção de MIP-1alfa e SDF-1 por fibroblastos de polpa dental humana em cultura frente ao desafio com Enterococcus faecalis inativado por calor. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/25/25138/tde-15102008-164844/ ;

Chicago Manual of Style (16th Edition):

Sipert, Carla Renata. “Produção de MIP-1alfa e SDF-1 por fibroblastos de polpa dental humana em cultura frente ao desafio com Enterococcus faecalis inativado por calor.” 2007. Masters Thesis, University of São Paulo. Accessed February 22, 2020. http://www.teses.usp.br/teses/disponiveis/25/25138/tde-15102008-164844/ ;.

MLA Handbook (7th Edition):

Sipert, Carla Renata. “Produção de MIP-1alfa e SDF-1 por fibroblastos de polpa dental humana em cultura frente ao desafio com Enterococcus faecalis inativado por calor.” 2007. Web. 22 Feb 2020.

Vancouver:

Sipert CR. Produção de MIP-1alfa e SDF-1 por fibroblastos de polpa dental humana em cultura frente ao desafio com Enterococcus faecalis inativado por calor. [Internet] [Masters thesis]. University of São Paulo; 2007. [cited 2020 Feb 22]. Available from: http://www.teses.usp.br/teses/disponiveis/25/25138/tde-15102008-164844/ ;.

Council of Science Editors:

Sipert CR. Produção de MIP-1alfa e SDF-1 por fibroblastos de polpa dental humana em cultura frente ao desafio com Enterococcus faecalis inativado por calor. [Masters Thesis]. University of São Paulo; 2007. Available from: http://www.teses.usp.br/teses/disponiveis/25/25138/tde-15102008-164844/ ;


Temple University

16. Ellison, Stephen Patrick. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.

Degree: PhD, 2015, Temple University

Physiology

BACKGROUND: Despite aggressive dietary modification, lipid lowering medications, and other medical therapy, vascular proliferative diseases continue to account for 50% of all mortality in… (more)

Subjects/Keywords: Physiology;

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ellison, S. P. (2015). THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253270

Chicago Manual of Style (16th Edition):

Ellison, Stephen Patrick. “THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.” 2015. Doctoral Dissertation, Temple University. Accessed February 22, 2020. http://digital.library.temple.edu/u?/p245801coll10,253270.

MLA Handbook (7th Edition):

Ellison, Stephen Patrick. “THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.” 2015. Web. 22 Feb 2020.

Vancouver:

Ellison SP. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Feb 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253270.

Council of Science Editors:

Ellison SP. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,253270

17. 石川, 桂二郎. Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用.

Degree: 博士(医学), 2013, Kyushu University / 九州大学

 Recent clinical observations have indicated that vascular endothelial growth factor (VEGF) is a key factor that stimulates the development of preretinal pathological neovascularization (NV). However,… (more)

Subjects/Keywords: angiogenesis; chemokines; inflammation; ischemic retinopathy; macrophage/microglia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

石川, . (2013). Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用. (Thesis). Kyushu University / 九州大学. Retrieved from http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

石川, 桂二郎. “Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用.” 2013. Thesis, Kyushu University / 九州大学. Accessed February 22, 2020. http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

石川, 桂二郎. “Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用.” 2013. Web. 22 Feb 2020.

Vancouver:

石川 . Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用. [Internet] [Thesis]. Kyushu University / 九州大学; 2013. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

石川 . Bone marrow-derived monocyte lineage cells recruited by MIP-1β promote physiological revascularization in mouse model of oxygen-induced retinopathy : 酸素負荷網膜症モデルマウス網膜における、MIP-1β誘導性骨髄由来単球系細胞の生理的血管再生促進作用. [Thesis]. Kyushu University / 九州大学; 2013. Available from: http://hdl.handle.net/2324/26669 ; http://dx.doi.org/10.15017/26669

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

18. Kang, Yuanxi. Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides.

Degree: PhD, 2012, University of Hong Kong

R5-tropic HIV-1 is predominantly transmitted during unprotected sexual contacts, rendering CCR5 antagonist as an attractive agent not only for antiretroviral therapy but also for prevention.… (more)

Subjects/Keywords: Chemokines - Receptors.; HIV infections - Treatment.; Antiviral agents.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kang, Y. (2012). Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides. (Doctoral Dissertation). University of Hong Kong. Retrieved from Kang, Y. [康元曦]. (2012). Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4784939 ; http://dx.doi.org/10.5353/th_b4784939 ; http://hdl.handle.net/10722/182280

Chicago Manual of Style (16th Edition):

Kang, Yuanxi. “Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides.” 2012. Doctoral Dissertation, University of Hong Kong. Accessed February 22, 2020. Kang, Y. [康元曦]. (2012). Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4784939 ; http://dx.doi.org/10.5353/th_b4784939 ; http://hdl.handle.net/10722/182280.

MLA Handbook (7th Edition):

Kang, Yuanxi. “Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides.” 2012. Web. 22 Feb 2020.

Vancouver:

Kang Y. Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides. [Internet] [Doctoral dissertation]. University of Hong Kong; 2012. [cited 2020 Feb 22]. Available from: Kang, Y. [康元曦]. (2012). Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4784939 ; http://dx.doi.org/10.5353/th_b4784939 ; http://hdl.handle.net/10722/182280.

Council of Science Editors:

Kang Y. Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides. [Doctoral Dissertation]. University of Hong Kong; 2012. Available from: Kang, Y. [康元曦]. (2012). Mechanism study of novel CCR5 antagonists and their potential as anti-HIV-1 microbicides. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4784939 ; http://dx.doi.org/10.5353/th_b4784939 ; http://hdl.handle.net/10722/182280


University of Edinburgh

19. Rhodes, Jonathan K. J. Investigation of chemokine expression and modulation following traumatic brain injury.

Degree: 2010, University of Edinburgh

 Over the last 20 years, advances in our understanding of the pathophysiology of severe traumatic brain injury (TBI) and in particular the contribution of secondary… (more)

Subjects/Keywords: 616.8; brain trauma; trauma; chemokines; modulation

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rhodes, J. K. J. (2010). Investigation of chemokine expression and modulation following traumatic brain injury. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/4513

Chicago Manual of Style (16th Edition):

Rhodes, Jonathan K J. “Investigation of chemokine expression and modulation following traumatic brain injury.” 2010. Doctoral Dissertation, University of Edinburgh. Accessed February 22, 2020. http://hdl.handle.net/1842/4513.

MLA Handbook (7th Edition):

Rhodes, Jonathan K J. “Investigation of chemokine expression and modulation following traumatic brain injury.” 2010. Web. 22 Feb 2020.

Vancouver:

Rhodes JKJ. Investigation of chemokine expression and modulation following traumatic brain injury. [Internet] [Doctoral dissertation]. University of Edinburgh; 2010. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/1842/4513.

Council of Science Editors:

Rhodes JKJ. Investigation of chemokine expression and modulation following traumatic brain injury. [Doctoral Dissertation]. University of Edinburgh; 2010. Available from: http://hdl.handle.net/1842/4513


Brigham Young University

20. Lew, Cynthia S. Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms.

Degree: MS, 2012, Brigham Young University

  In order to better understand the mechanism of antimicrobial chemokine activity, including binding to and killing of bacteria, random transposon mutagenesis was performed in… (more)

Subjects/Keywords: antimicrobial chemokines; Y. pseudotuberculosis; rfaD; Microbiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lew, C. S. (2012). Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms. (Masters Thesis). Brigham Young University. Retrieved from https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd

Chicago Manual of Style (16th Edition):

Lew, Cynthia S. “Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms.” 2012. Masters Thesis, Brigham Young University. Accessed February 22, 2020. https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd.

MLA Handbook (7th Edition):

Lew, Cynthia S. “Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms.” 2012. Web. 22 Feb 2020.

Vancouver:

Lew CS. Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms. [Internet] [Masters thesis]. Brigham Young University; 2012. [cited 2020 Feb 22]. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd.

Council of Science Editors:

Lew CS. Loss of the Lipopolysaccharide Core Biosynthesis rfaD Gene Increases Antimicrobial Chemokine Binding and Bacterial Susceptibility to CCL28 and Polymyxin: A Model for Understanding the Interface of Antimicrobial Chemokines and Bacterial Host Defense Avoidance Mechanisms. [Masters Thesis]. Brigham Young University; 2012. Available from: https://scholarsarchive.byu.edu/cgi/viewcontent.cgi?article=4755&context=etd

21. 足立, 育子. Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes.

Degree: 博士(医学), 2016, Oita University / 大分大学

 Previous studies have demonstrated that the activation of peroxisome proliferator-activated receptors (PPARs) not only has positive effects on permeability barrier homoeostasis but also has anti-inflammatory… (more)

Subjects/Keywords: barrier abrogation; chemokines; IL-4; PPARa

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

足立, . (2016). Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes. (Thesis). Oita University / 大分大学. Retrieved from http://hdl.handle.net/10559/15621

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

足立, 育子. “Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes.” 2016. Thesis, Oita University / 大分大学. Accessed February 22, 2020. http://hdl.handle.net/10559/15621.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

足立, 育子. “Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes.” 2016. Web. 22 Feb 2020.

Vancouver:

足立 . Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes. [Internet] [Thesis]. Oita University / 大分大学; 2016. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/10559/15621.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

足立 . Expressions of peroxisome proliferator-activated receptors (PPARs) are directly influenced by permeability barrier abrogation and inflammatory cytokines and depressed PPARα modulates expressions of chemokines and epidermal differentiation-related molecules in keratinocytes. [Thesis]. Oita University / 大分大学; 2016. Available from: http://hdl.handle.net/10559/15621

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Regenass, Pierre. Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands.

Degree: Docteur es, Chimie biologique et thérapeutique, 2015, Université de Strasbourg

Les chimiokines forment une vaste famille de cytokines chimioattractantes surtout connues pour leur implication dans des phénomènes pro-inflammatoires. Ainsi, l’obtention de composés modulant l’action de… (more)

Subjects/Keywords: Chimiokines; Neutraligands; Chemokines; Neutraligands; 547.7; 615.19

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Regenass, P. (2015). Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2015STRAF057

Chicago Manual of Style (16th Edition):

Regenass, Pierre. “Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands.” 2015. Doctoral Dissertation, Université de Strasbourg. Accessed February 22, 2020. http://www.theses.fr/2015STRAF057.

MLA Handbook (7th Edition):

Regenass, Pierre. “Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands.” 2015. Web. 22 Feb 2020.

Vancouver:

Regenass P. Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2015. [cited 2020 Feb 22]. Available from: http://www.theses.fr/2015STRAF057.

Council of Science Editors:

Regenass P. Conception de plateformes hétérocycliques originales et application à la découverte de nouveaux neutraligands des chimiokines CXCL12 et CCL17 : Design and synthesis of new heterocyclic scaffolds and application to the discovery of chemokine CXCL12 and CCLl7 neutraligands. [Doctoral Dissertation]. Université de Strasbourg; 2015. Available from: http://www.theses.fr/2015STRAF057


Case Western Reserve University

23. Kerstetter Fogle, Amber E. ROLE OF CHEMOKINES IN REGULATING OLIGODENDROCYTE DEVELOPMENT, ASTROGLIOSIS, AND DEMYELINATING DISEASES.

Degree: PhD, Neurosciences, 2010, Case Western Reserve University

  Role of Chemokines in Regulating Oligodendrocyte Development, Astrogliosis and Demyelinating Disorders AbstractbyAMBER E. KERSTETTER-FOGLE Oligodendrocyte development and maturation is crucial for efficient axonal transduction… (more)

Subjects/Keywords: Biomedical Research; demyelination; chemokines; Multiple sclerosis; astrogliosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kerstetter Fogle, A. E. (2010). ROLE OF CHEMOKINES IN REGULATING OLIGODENDROCYTE DEVELOPMENT, ASTROGLIOSIS, AND DEMYELINATING DISEASES. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1258139447

Chicago Manual of Style (16th Edition):

Kerstetter Fogle, Amber E. “ROLE OF CHEMOKINES IN REGULATING OLIGODENDROCYTE DEVELOPMENT, ASTROGLIOSIS, AND DEMYELINATING DISEASES.” 2010. Doctoral Dissertation, Case Western Reserve University. Accessed February 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1258139447.

MLA Handbook (7th Edition):

Kerstetter Fogle, Amber E. “ROLE OF CHEMOKINES IN REGULATING OLIGODENDROCYTE DEVELOPMENT, ASTROGLIOSIS, AND DEMYELINATING DISEASES.” 2010. Web. 22 Feb 2020.

Vancouver:

Kerstetter Fogle AE. ROLE OF CHEMOKINES IN REGULATING OLIGODENDROCYTE DEVELOPMENT, ASTROGLIOSIS, AND DEMYELINATING DISEASES. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2010. [cited 2020 Feb 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1258139447.

Council of Science Editors:

Kerstetter Fogle AE. ROLE OF CHEMOKINES IN REGULATING OLIGODENDROCYTE DEVELOPMENT, ASTROGLIOSIS, AND DEMYELINATING DISEASES. [Doctoral Dissertation]. Case Western Reserve University; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1258139447


Case Western Reserve University

24. Lin, Michelle. Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines.

Degree: PhD, Pathology, 2008, Case Western Reserve University

 Corneal opacification caused by extracellular damage and remodeling is a symptom of bacterial keratitis in which neutrophils are a critical part of its pathogenesis. While… (more)

Subjects/Keywords: neutrophil; matrix metalloproteinases; corneal inflammation; chemokines; migration

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, M. (2008). Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168

Chicago Manual of Style (16th Edition):

Lin, Michelle. “Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines.” 2008. Doctoral Dissertation, Case Western Reserve University. Accessed February 22, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168.

MLA Handbook (7th Edition):

Lin, Michelle. “Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines.” 2008. Web. 22 Feb 2020.

Vancouver:

Lin M. Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2008. [cited 2020 Feb 22]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168.

Council of Science Editors:

Lin M. Facilitation of Neutrophil Migration Through the Corneal Stroma During Keratitis - Mmp8 and Chemokines. [Doctoral Dissertation]. Case Western Reserve University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1191012168


University of California – Merced

25. Tian, Wei. Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI.

Degree: Quantitative and Systems Biology, 2009, University of California – Merced

 Eotaxin belongs to the chemokine family of proteins, which are involved in inflammation and in the development of the immune system, regulating activation and chemotaxis… (more)

Subjects/Keywords: Biology; chemokines; eotaxin; CC-chemokine inhibitor

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tian, W. (2009). Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI. (Thesis). University of California – Merced. Retrieved from http://www.escholarship.org/uc/item/0fs1p96r

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tian, Wei. “Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI.” 2009. Thesis, University of California – Merced. Accessed February 22, 2020. http://www.escholarship.org/uc/item/0fs1p96r.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tian, Wei. “Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI.” 2009. Web. 22 Feb 2020.

Vancouver:

Tian W. Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI. [Internet] [Thesis]. University of California – Merced; 2009. [cited 2020 Feb 22]. Available from: http://www.escholarship.org/uc/item/0fs1p96r.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tian W. Investigation of the interaction between the CC-chemokine eotaxin and the viral CC-chemokine inhibitor vCCI. [Thesis]. University of California – Merced; 2009. Available from: http://www.escholarship.org/uc/item/0fs1p96r

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Azzaoui, Imane. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.

Degree: Docteur es, Immunologie (Médecine), 2011, Université Lille II – Droit et Santé

 Les chimiokines sont un élément essentiel du trafic cellulaire aussi bien homéostatique que dans des situations pathologiques. Outre cette fonction chimiotactique spécifique à ce type… (more)

Subjects/Keywords: Chimiokine CCL18; Allergie; Chemokines; Allergy; Immunology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Azzaoui, I. (2011). CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. (Doctoral Dissertation). Université Lille II – Droit et Santé. Retrieved from http://www.theses.fr/2011LIL2S019

Chicago Manual of Style (16th Edition):

Azzaoui, Imane. “CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.” 2011. Doctoral Dissertation, Université Lille II – Droit et Santé. Accessed February 22, 2020. http://www.theses.fr/2011LIL2S019.

MLA Handbook (7th Edition):

Azzaoui, Imane. “CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy.” 2011. Web. 22 Feb 2020.

Vancouver:

Azzaoui I. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. [Internet] [Doctoral dissertation]. Université Lille II – Droit et Santé 2011. [cited 2020 Feb 22]. Available from: http://www.theses.fr/2011LIL2S019.

Council of Science Editors:

Azzaoui I. CCL18 et réponse régulatrice, de la situation physiologique à l'atopie : CCL18 and regulatory responses from steady state to atopy. [Doctoral Dissertation]. Université Lille II – Droit et Santé 2011. Available from: http://www.theses.fr/2011LIL2S019


Drexel University

27. Festa, Lindsay Kathryne. Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV.

Degree: 2018, Drexel University

Despite the introduction of antiretroviral therapy (ART), approximately 50% of HIV+ patients still experience some degree of neurological dysfunction. The loss of dendritic spines, the… (more)

Subjects/Keywords: Pharmacology; Physiology; Adaptation (Biology); Chemokines; Dendritic cells

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Festa, L. K. (2018). Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV. (Thesis). Drexel University. Retrieved from https://idea.library.drexel.edu/islandora/object/idea%3A7966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Festa, Lindsay Kathryne. “Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV.” 2018. Thesis, Drexel University. Accessed February 22, 2020. https://idea.library.drexel.edu/islandora/object/idea%3A7966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Festa, Lindsay Kathryne. “Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV.” 2018. Web. 22 Feb 2020.

Vancouver:

Festa LK. Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV. [Internet] [Thesis]. Drexel University; 2018. [cited 2020 Feb 22]. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A7966.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Festa LK. Defining the Molecular Pathways Involved in CXCL12-mediated Rescue of Dendritic Spines and Cognitive Deficits in an Animal Model of neuroHIV. [Thesis]. Drexel University; 2018. Available from: https://idea.library.drexel.edu/islandora/object/idea%3A7966

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Emmanouil, Georgios. Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου.

Degree: 2018, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Colorectal cancer is the second leading etiology of cancer death in Western countries as almost half of the patients die of metastatic disease after curative… (more)

Subjects/Keywords: Αγγειογένεση όγκου; Χημειοκίνες; Angiogenens index; Chemokines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Emmanouil, G. (2018). Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/43650

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Emmanouil, Georgios. “Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου.” 2018. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed February 22, 2020. http://hdl.handle.net/10442/hedi/43650.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Emmanouil, Georgios. “Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου.” 2018. Web. 22 Feb 2020.

Vancouver:

Emmanouil G. Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2018. [cited 2020 Feb 22]. Available from: http://hdl.handle.net/10442/hedi/43650.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Emmanouil G. Ο ρόλος των αγγειογενών και αγγειοστατικών C-X-C χημειοκινών στην ανάπτυξη του καρκίνου του παχέος εντέρου. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2018. Available from: http://hdl.handle.net/10442/hedi/43650

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

29. Poupel, Lucie. Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis.

Degree: Docteur es, Immunologie, 2013, Université Paris-Sud – Paris XI

: L’athérosclérose est une maladie inflammatoire chronique des grosses artères à localisation intimale. Elle est probablement la résultante d’une réaction inflammatoire mal contrôlée ayant pour… (more)

Subjects/Keywords: Chimiokine; Monocytes; Atherosclerose; Chemokines; Monocytes; Atherosclerosis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Poupel, L. (2013). Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T032

Chicago Manual of Style (16th Edition):

Poupel, Lucie. “Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed February 22, 2020. http://www.theses.fr/2013PA11T032.

MLA Handbook (7th Edition):

Poupel, Lucie. “Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis.” 2013. Web. 22 Feb 2020.

Vancouver:

Poupel L. Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2020 Feb 22]. Available from: http://www.theses.fr/2013PA11T032.

Council of Science Editors:

Poupel L. Rôle des chimiokines dans la mobilisation monocytaire au cours de l’athérosclérose : Role of chemokines in monocyte mobilization during atherosclerosis. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T032


Université Paris-Sud – Paris XI

30. Weiss, Julia Miriam. Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13.

Degree: Docteur es, Immunologie et biothérapies, 2011, Université Paris-Sud – Paris XI

 La myasthénie (Myasthenia Gravis) est une maladie neuromusculaire impliquant des auto-anticorps dirigés majoritairement contre le récepteur à l’acétylcholine (RACh) et entrainant une fatigabilité musculaire. Ces… (more)

Subjects/Keywords: Auto-immunité; Autoimmunity; Myasthenia Gravis; Thymus; Chemokines

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Weiss, J. M. (2011). Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA114831

Chicago Manual of Style (16th Edition):

Weiss, Julia Miriam. “Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed February 22, 2020. http://www.theses.fr/2011PA114831.

MLA Handbook (7th Edition):

Weiss, Julia Miriam. “Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13.” 2011. Web. 22 Feb 2020.

Vancouver:

Weiss JM. Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2020 Feb 22]. Available from: http://www.theses.fr/2011PA114831.

Council of Science Editors:

Weiss JM. Characterization of thymic hyperplasia associated with autoimmune Myasthenia Gravis : role of the chemokines CXCL12 and CXCL13 : Caractérisation de l’hyperplasie thymique associée à la myasthénie : rôle des chimiokines CXCL12 et CXCL13. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA114831

[1] [2] [3] [4] [5] … [13]

.