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You searched for subject:(Cftr). Showing records 1 – 30 of 125 total matches.

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1. Becdelièvre, Alix de. Contribution à l’amélioration des connaissances sur la relation génotype-phénotype dans la mucoviscidose et caractérisation phénotypique de l’inflammation pulmonaire : Contribution to the appreciation of the genotype‐phenotype correlation in cystic fibrosis and phenotypic characterization of lung inflammation.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2011, Université Paris-Est

La mucoviscidose est la maladie autosomique récessive grave la plus fréquente dans la population d'origine caucasienne. Elle est due a des anomalies du gène CFTR,… (more)

Subjects/Keywords: Cftr; Promoteur; Cftr; Promoter

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Becdelièvre, A. d. (2011). Contribution à l’amélioration des connaissances sur la relation génotype-phénotype dans la mucoviscidose et caractérisation phénotypique de l’inflammation pulmonaire : Contribution to the appreciation of the genotype‐phenotype correlation in cystic fibrosis and phenotypic characterization of lung inflammation. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2011PEST0084

Chicago Manual of Style (16th Edition):

Becdelièvre, Alix de. “Contribution à l’amélioration des connaissances sur la relation génotype-phénotype dans la mucoviscidose et caractérisation phénotypique de l’inflammation pulmonaire : Contribution to the appreciation of the genotype‐phenotype correlation in cystic fibrosis and phenotypic characterization of lung inflammation.” 2011. Doctoral Dissertation, Université Paris-Est. Accessed July 16, 2019. http://www.theses.fr/2011PEST0084.

MLA Handbook (7th Edition):

Becdelièvre, Alix de. “Contribution à l’amélioration des connaissances sur la relation génotype-phénotype dans la mucoviscidose et caractérisation phénotypique de l’inflammation pulmonaire : Contribution to the appreciation of the genotype‐phenotype correlation in cystic fibrosis and phenotypic characterization of lung inflammation.” 2011. Web. 16 Jul 2019.

Vancouver:

Becdelièvre Ad. Contribution à l’amélioration des connaissances sur la relation génotype-phénotype dans la mucoviscidose et caractérisation phénotypique de l’inflammation pulmonaire : Contribution to the appreciation of the genotype‐phenotype correlation in cystic fibrosis and phenotypic characterization of lung inflammation. [Internet] [Doctoral dissertation]. Université Paris-Est; 2011. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2011PEST0084.

Council of Science Editors:

Becdelièvre Ad. Contribution à l’amélioration des connaissances sur la relation génotype-phénotype dans la mucoviscidose et caractérisation phénotypique de l’inflammation pulmonaire : Contribution to the appreciation of the genotype‐phenotype correlation in cystic fibrosis and phenotypic characterization of lung inflammation. [Doctoral Dissertation]. Université Paris-Est; 2011. Available from: http://www.theses.fr/2011PEST0084

2. Le henaff, Carole. La protéine CFTR : Implication et cible thérapeutique dans la maladie osseuse chez les patients atteints de mucoviscidose. : CFTR : Involvement and therapeutic target in osteoporosis.

Degree: Docteur es, Sciences - STS, 2012, Reims

La mutation F508del dans la protéine CFTR (Cystic Fibrosis Transmembrane Conductance Regulator) est considérée comme un facteur de risque indépendant de maladie osseuse liée à… (more)

Subjects/Keywords: Cftr; Ostéoporose; Cf; Cftr; Osteoporosis

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APA (6th Edition):

Le henaff, C. (2012). La protéine CFTR : Implication et cible thérapeutique dans la maladie osseuse chez les patients atteints de mucoviscidose. : CFTR : Involvement and therapeutic target in osteoporosis. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2012REIMS022

Chicago Manual of Style (16th Edition):

Le henaff, Carole. “La protéine CFTR : Implication et cible thérapeutique dans la maladie osseuse chez les patients atteints de mucoviscidose. : CFTR : Involvement and therapeutic target in osteoporosis.” 2012. Doctoral Dissertation, Reims. Accessed July 16, 2019. http://www.theses.fr/2012REIMS022.

MLA Handbook (7th Edition):

Le henaff, Carole. “La protéine CFTR : Implication et cible thérapeutique dans la maladie osseuse chez les patients atteints de mucoviscidose. : CFTR : Involvement and therapeutic target in osteoporosis.” 2012. Web. 16 Jul 2019.

Vancouver:

Le henaff C. La protéine CFTR : Implication et cible thérapeutique dans la maladie osseuse chez les patients atteints de mucoviscidose. : CFTR : Involvement and therapeutic target in osteoporosis. [Internet] [Doctoral dissertation]. Reims; 2012. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2012REIMS022.

Council of Science Editors:

Le henaff C. La protéine CFTR : Implication et cible thérapeutique dans la maladie osseuse chez les patients atteints de mucoviscidose. : CFTR : Involvement and therapeutic target in osteoporosis. [Doctoral Dissertation]. Reims; 2012. Available from: http://www.theses.fr/2012REIMS022


University of Toronto

3. Hung, Maurita. The Development and Characterization of VX-770 Molecular Probes.

Degree: 2016, University of Toronto

Cystic fibrosis is the most common genetic disease affecting the Canadian population. It is caused by mutations in the cystic fibrosis transmembrane conductance regulator gene,… (more)

Subjects/Keywords: CFTR; cystic fibrosis; potentiator; 0719

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APA (6th Edition):

Hung, M. (2016). The Development and Characterization of VX-770 Molecular Probes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/89009

Chicago Manual of Style (16th Edition):

Hung, Maurita. “The Development and Characterization of VX-770 Molecular Probes.” 2016. Masters Thesis, University of Toronto. Accessed July 16, 2019. http://hdl.handle.net/1807/89009.

MLA Handbook (7th Edition):

Hung, Maurita. “The Development and Characterization of VX-770 Molecular Probes.” 2016. Web. 16 Jul 2019.

Vancouver:

Hung M. The Development and Characterization of VX-770 Molecular Probes. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1807/89009.

Council of Science Editors:

Hung M. The Development and Characterization of VX-770 Molecular Probes. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/89009

4. Paulet, Damien. Variation d'hydrophobicité et structure secondaire des protéines transmembranaires : Variation of hydrophobicity and secondary structure of integral membrane proteins.

Degree: Docteur es, Biologie Santé, 2010, Université Montpellier I

Contexte. Les protéines transmembranaires ont une importance considérable tant au niveau de la survie d'une cellule qu'au niveau de ces interactions avec les autres cellules.… (more)

Subjects/Keywords: Cftr; Protéine transmembranaire; Canal; Cftr; Transmembrane protein; Channel

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APA (6th Edition):

Paulet, D. (2010). Variation d'hydrophobicité et structure secondaire des protéines transmembranaires : Variation of hydrophobicity and secondary structure of integral membrane proteins. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2010MON13518

Chicago Manual of Style (16th Edition):

Paulet, Damien. “Variation d'hydrophobicité et structure secondaire des protéines transmembranaires : Variation of hydrophobicity and secondary structure of integral membrane proteins.” 2010. Doctoral Dissertation, Université Montpellier I. Accessed July 16, 2019. http://www.theses.fr/2010MON13518.

MLA Handbook (7th Edition):

Paulet, Damien. “Variation d'hydrophobicité et structure secondaire des protéines transmembranaires : Variation of hydrophobicity and secondary structure of integral membrane proteins.” 2010. Web. 16 Jul 2019.

Vancouver:

Paulet D. Variation d'hydrophobicité et structure secondaire des protéines transmembranaires : Variation of hydrophobicity and secondary structure of integral membrane proteins. [Internet] [Doctoral dissertation]. Université Montpellier I; 2010. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2010MON13518.

Council of Science Editors:

Paulet D. Variation d'hydrophobicité et structure secondaire des protéines transmembranaires : Variation of hydrophobicity and secondary structure of integral membrane proteins. [Doctoral Dissertation]. Université Montpellier I; 2010. Available from: http://www.theses.fr/2010MON13518

5. Aissat, Abdelrazak. Variabilité phénotypique et épissage : combinaison d'analyses in vitro et in silico du gène CFTR : Phenotypic variability and splicing : combined in vitro and in silico analyses of the CFTR gene.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2012, Université Paris-Est

 Depuis plusieurs décennies, l’étude des conséquences des mutations pathogéniques a permisnon seulement de définir l’origine de nombreuses maladies génétiques humaines, héréditaires ou non,mais également de… (more)

Subjects/Keywords: Mucoviscidose; Cftr; Épissage; Variabilité phénotypique; Cystic fibrosis; Cftr; Splicing; Phenotype variability

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aissat, A. (2012). Variabilité phénotypique et épissage : combinaison d'analyses in vitro et in silico du gène CFTR : Phenotypic variability and splicing : combined in vitro and in silico analyses of the CFTR gene. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2012PEST0060

Chicago Manual of Style (16th Edition):

Aissat, Abdelrazak. “Variabilité phénotypique et épissage : combinaison d'analyses in vitro et in silico du gène CFTR : Phenotypic variability and splicing : combined in vitro and in silico analyses of the CFTR gene.” 2012. Doctoral Dissertation, Université Paris-Est. Accessed July 16, 2019. http://www.theses.fr/2012PEST0060.

MLA Handbook (7th Edition):

Aissat, Abdelrazak. “Variabilité phénotypique et épissage : combinaison d'analyses in vitro et in silico du gène CFTR : Phenotypic variability and splicing : combined in vitro and in silico analyses of the CFTR gene.” 2012. Web. 16 Jul 2019.

Vancouver:

Aissat A. Variabilité phénotypique et épissage : combinaison d'analyses in vitro et in silico du gène CFTR : Phenotypic variability and splicing : combined in vitro and in silico analyses of the CFTR gene. [Internet] [Doctoral dissertation]. Université Paris-Est; 2012. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2012PEST0060.

Council of Science Editors:

Aissat A. Variabilité phénotypique et épissage : combinaison d'analyses in vitro et in silico du gène CFTR : Phenotypic variability and splicing : combined in vitro and in silico analyses of the CFTR gene. [Doctoral Dissertation]. Université Paris-Est; 2012. Available from: http://www.theses.fr/2012PEST0060

6. Drévillon, Loïc. Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination.

Degree: Docteur es, Physiopathologie, 2009, Université Paris-Est

 La mucoviscidose (CF, Cystic Fibrosis) est la maladie génétique la plus fréquente dans les populations d’origine caucasienne. Les malades présentent une symptomatologie variée, dominée par… (more)

Subjects/Keywords: CFTR; Ubiquitinylation; COMMD1; NF-?B; CFTR; Ubiquitination; COMMD1; NF-?B

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APA (6th Edition):

Drévillon, L. (2009). Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination. (Doctoral Dissertation). Université Paris-Est. Retrieved from http://www.theses.fr/2009PEST0005

Chicago Manual of Style (16th Edition):

Drévillon, Loïc. “Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination.” 2009. Doctoral Dissertation, Université Paris-Est. Accessed July 16, 2019. http://www.theses.fr/2009PEST0005.

MLA Handbook (7th Edition):

Drévillon, Loïc. “Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination.” 2009. Web. 16 Jul 2019.

Vancouver:

Drévillon L. Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination. [Internet] [Doctoral dissertation]. Université Paris-Est; 2009. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2009PEST0005.

Council of Science Editors:

Drévillon L. Contribution à l’étude du rôle de COMMD1 dans la physiopathologie de la mucoviscidose : COMMD1 promotes CFTR trafficking through inhibition of ubiquitination. [Doctoral Dissertation]. Université Paris-Est; 2009. Available from: http://www.theses.fr/2009PEST0005


Universitat Autònoma de Barcelona

7. Masvidal Sanz, Laia. Estudi dels mecanismes moleculars subjacents en mutacions CFTR que afecten l’eficiència de l’splicing. Desenvolupament de tècniques complementàries per a la caracterització a nivell de DNA, RNA i proteïna en cèl·lules epitelials.

Degree: Departament de Genètica i de Microbiologia, 2011, Universitat Autònoma de Barcelona

 Cystic fibrosis (CF) is the most common recessive genetic disease in Caucasian population. Over 1,600 cystic fibrosis transmembrane conductance regulator (CFTR) gene sequence variations have… (more)

Subjects/Keywords: CFTR; Splicing; mRNA; Ciències Experimentals; 575

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APA (6th Edition):

Masvidal Sanz, L. (2011). Estudi dels mecanismes moleculars subjacents en mutacions CFTR que afecten l’eficiència de l’splicing. Desenvolupament de tècniques complementàries per a la caracterització a nivell de DNA, RNA i proteïna en cèl·lules epitelials. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/82073

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Masvidal Sanz, Laia. “Estudi dels mecanismes moleculars subjacents en mutacions CFTR que afecten l’eficiència de l’splicing. Desenvolupament de tècniques complementàries per a la caracterització a nivell de DNA, RNA i proteïna en cèl·lules epitelials.” 2011. Thesis, Universitat Autònoma de Barcelona. Accessed July 16, 2019. http://hdl.handle.net/10803/82073.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Masvidal Sanz, Laia. “Estudi dels mecanismes moleculars subjacents en mutacions CFTR que afecten l’eficiència de l’splicing. Desenvolupament de tècniques complementàries per a la caracterització a nivell de DNA, RNA i proteïna en cèl·lules epitelials.” 2011. Web. 16 Jul 2019.

Vancouver:

Masvidal Sanz L. Estudi dels mecanismes moleculars subjacents en mutacions CFTR que afecten l’eficiència de l’splicing. Desenvolupament de tècniques complementàries per a la caracterització a nivell de DNA, RNA i proteïna en cèl·lules epitelials. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2011. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10803/82073.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Masvidal Sanz L. Estudi dels mecanismes moleculars subjacents en mutacions CFTR que afecten l’eficiència de l’splicing. Desenvolupament de tècniques complementàries per a la caracterització a nivell de DNA, RNA i proteïna en cèl·lules epitelials. [Thesis]. Universitat Autònoma de Barcelona; 2011. Available from: http://hdl.handle.net/10803/82073

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

8. Anthony Chen, Kar Ki. Role of FGF Receptors in Rescue of ΔF508-CFTR.

Degree: 2015, University of Toronto

ΔF508-CFTR is the most common mutation causing cystic fibrosis (CF), where it exhibits folding defects and is unable to reach the plasma membrane. To identify… (more)

Subjects/Keywords: cystic fibrosis; FGFR; kinase; ΔF508-CFTR; 0487

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APA (6th Edition):

Anthony Chen, K. K. (2015). Role of FGF Receptors in Rescue of ΔF508-CFTR. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/70251

Chicago Manual of Style (16th Edition):

Anthony Chen, Kar Ki. “Role of FGF Receptors in Rescue of ΔF508-CFTR.” 2015. Masters Thesis, University of Toronto. Accessed July 16, 2019. http://hdl.handle.net/1807/70251.

MLA Handbook (7th Edition):

Anthony Chen, Kar Ki. “Role of FGF Receptors in Rescue of ΔF508-CFTR.” 2015. Web. 16 Jul 2019.

Vancouver:

Anthony Chen KK. Role of FGF Receptors in Rescue of ΔF508-CFTR. [Internet] [Masters thesis]. University of Toronto; 2015. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/1807/70251.

Council of Science Editors:

Anthony Chen KK. Role of FGF Receptors in Rescue of ΔF508-CFTR. [Masters Thesis]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/70251

9. MARLENE GUIMARÃES SANTOS. O GENE CFTR E A MUTAÇÃO R1162X EM DIFERENTES GRUPOS POPULACIONAIS DE PORTO VELHO - RONDÔNIA.

Degree: 2005, Fundação Universidade Federal de Rondônia

Fibrose Cística (FC) é uma doença recessiva potencialmente letal, sendo a doença genética mais freqüente em populações caucasianas. Sua incidência é variável e está relacionada… (more)

Subjects/Keywords: GENETICA HUMANA E MEDICA; Fibrose Cística; Cystic Fibrosis; Mutation R1162X; CFTR; CFTR; Mutação R1162X

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APA (6th Edition):

SANTOS, M. G. (2005). O GENE CFTR E A MUTAÇÃO R1162X EM DIFERENTES GRUPOS POPULACIONAIS DE PORTO VELHO - RONDÔNIA. (Thesis). Fundação Universidade Federal de Rondônia. Retrieved from http://www.bdtd.unir.br/tde_busca/arquivo.php?codArquivo=12

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

SANTOS, MARLENE GUIMARÃES. “O GENE CFTR E A MUTAÇÃO R1162X EM DIFERENTES GRUPOS POPULACIONAIS DE PORTO VELHO - RONDÔNIA.” 2005. Thesis, Fundação Universidade Federal de Rondônia. Accessed July 16, 2019. http://www.bdtd.unir.br/tde_busca/arquivo.php?codArquivo=12.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

SANTOS, MARLENE GUIMARÃES. “O GENE CFTR E A MUTAÇÃO R1162X EM DIFERENTES GRUPOS POPULACIONAIS DE PORTO VELHO - RONDÔNIA.” 2005. Web. 16 Jul 2019.

Vancouver:

SANTOS MG. O GENE CFTR E A MUTAÇÃO R1162X EM DIFERENTES GRUPOS POPULACIONAIS DE PORTO VELHO - RONDÔNIA. [Internet] [Thesis]. Fundação Universidade Federal de Rondônia; 2005. [cited 2019 Jul 16]. Available from: http://www.bdtd.unir.br/tde_busca/arquivo.php?codArquivo=12.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SANTOS MG. O GENE CFTR E A MUTAÇÃO R1162X EM DIFERENTES GRUPOS POPULACIONAIS DE PORTO VELHO - RONDÔNIA. [Thesis]. Fundação Universidade Federal de Rondônia; 2005. Available from: http://www.bdtd.unir.br/tde_busca/arquivo.php?codArquivo=12

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Huguet, Florentin. Impact de la modulation de TRPM7 et ATF6 sur le cystic fibrosis transmembrane conductance regulator : Impact of TRPM7 and ATF6 modulation on cystic fibrosis transmembrane conductance regulator.

Degree: Docteur es, Biologie-santé, 2017, Brest

La mucoviscidose est une maladie causée par des mutations du gène cftr entraînant des défauts importants de la protéine CFTR. La mutation la plus fréquente… (more)

Subjects/Keywords: Mucoviscidose; CFTR; F508del; UPR; TRPM7; Mg2+; Cystic fibrosis; CFTR; F508del; TRPM7; Mg2+; 616.372

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APA (6th Edition):

Huguet, F. (2017). Impact de la modulation de TRPM7 et ATF6 sur le cystic fibrosis transmembrane conductance regulator : Impact of TRPM7 and ATF6 modulation on cystic fibrosis transmembrane conductance regulator. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2017BRES0058

Chicago Manual of Style (16th Edition):

Huguet, Florentin. “Impact de la modulation de TRPM7 et ATF6 sur le cystic fibrosis transmembrane conductance regulator : Impact of TRPM7 and ATF6 modulation on cystic fibrosis transmembrane conductance regulator.” 2017. Doctoral Dissertation, Brest. Accessed July 16, 2019. http://www.theses.fr/2017BRES0058.

MLA Handbook (7th Edition):

Huguet, Florentin. “Impact de la modulation de TRPM7 et ATF6 sur le cystic fibrosis transmembrane conductance regulator : Impact of TRPM7 and ATF6 modulation on cystic fibrosis transmembrane conductance regulator.” 2017. Web. 16 Jul 2019.

Vancouver:

Huguet F. Impact de la modulation de TRPM7 et ATF6 sur le cystic fibrosis transmembrane conductance regulator : Impact of TRPM7 and ATF6 modulation on cystic fibrosis transmembrane conductance regulator. [Internet] [Doctoral dissertation]. Brest; 2017. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2017BRES0058.

Council of Science Editors:

Huguet F. Impact de la modulation de TRPM7 et ATF6 sur le cystic fibrosis transmembrane conductance regulator : Impact of TRPM7 and ATF6 modulation on cystic fibrosis transmembrane conductance regulator. [Doctoral Dissertation]. Brest; 2017. Available from: http://www.theses.fr/2017BRES0058


University of Toledo Health Science Campus

11. Saxena, Anita. Role of Hsp105 in CFTR Biogenesis.

Degree: PhD, College of Medicine, 2010, University of Toledo Health Science Campus

 Cystic fibrosis (CF) is a life-threatening genetic disease arising from mutations in the gene encoding cystic fibrosis transmembrane conductance regulator (CFTR). The mutant ¿F508 CFTR(more)

Subjects/Keywords: Biology; Molecular Biology; Pharmacology; Heat Shock Proteins; Hsp105; Cystic Fibrosis; CFTR; CFTR Biogenesis; Molecular Chaperones

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APA (6th Edition):

Saxena, A. (2010). Role of Hsp105 in CFTR Biogenesis. (Doctoral Dissertation). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1279120195

Chicago Manual of Style (16th Edition):

Saxena, Anita. “Role of Hsp105 in CFTR Biogenesis.” 2010. Doctoral Dissertation, University of Toledo Health Science Campus. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=mco1279120195.

MLA Handbook (7th Edition):

Saxena, Anita. “Role of Hsp105 in CFTR Biogenesis.” 2010. Web. 16 Jul 2019.

Vancouver:

Saxena A. Role of Hsp105 in CFTR Biogenesis. [Internet] [Doctoral dissertation]. University of Toledo Health Science Campus; 2010. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1279120195.

Council of Science Editors:

Saxena A. Role of Hsp105 in CFTR Biogenesis. [Doctoral Dissertation]. University of Toledo Health Science Campus; 2010. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1279120195


Universidade de Lisboa

12. Ferreira, Vera Filipa Cerqueira. A molecular imaging approach to cystic fibrosis.

Degree: 2013, Universidade de Lisboa

Tese de mestrado em Bioquímica, apresentada à Universidade de Lisboa, através da Faculdade de Ciências, 2013

A Fibrose Quística (FQ) é a doença autossómica recessiva… (more)

Subjects/Keywords: Fibrose quística; Imagiologia molecular; Tecnécio; Anticorpos anti-CFTR; Inibidores da CFTR; Teses de mestrado - 2013

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APA (6th Edition):

Ferreira, V. F. C. (2013). A molecular imaging approach to cystic fibrosis. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ferreira, Vera Filipa Cerqueira. “A molecular imaging approach to cystic fibrosis.” 2013. Thesis, Universidade de Lisboa. Accessed July 16, 2019. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ferreira, Vera Filipa Cerqueira. “A molecular imaging approach to cystic fibrosis.” 2013. Web. 16 Jul 2019.

Vancouver:

Ferreira VFC. A molecular imaging approach to cystic fibrosis. [Internet] [Thesis]. Universidade de Lisboa; 2013. [cited 2019 Jul 16]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9438.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ferreira VFC. A molecular imaging approach to cystic fibrosis. [Thesis]. Universidade de Lisboa; 2013. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/9438

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Gonzalez, Gaëlle. Transfert du CFTR par vecteurs de gènes dérivés des adénovirus ou par trogocytose de microparticules membranaires : mécanismes moléculaires et applications à la mucoviscidose : Transfer of CFTR by gene tranfer vectors derived from adenoviruses or by trogocytosis of membrane-derived microparticle : molecular mechanisms and applications in cystic fibrosis.

Degree: Docteur es, Virologie, 2011, Université Claude Bernard – Lyon I

La mucoviscidose est une maladie génétique due à des mutations du gène CFTR, conduisant à une altération de la fonction de canal à ions chlorure… (more)

Subjects/Keywords: Adénovirus; Microparticule membranaire; GFP-CFTR; Trogocytose; Exosome; Adenovirus; Microparticle membrane; GFP-CFTR; Trogocytosis; Exosome; 616.9101

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APA (6th Edition):

Gonzalez, G. (2011). Transfert du CFTR par vecteurs de gènes dérivés des adénovirus ou par trogocytose de microparticules membranaires : mécanismes moléculaires et applications à la mucoviscidose : Transfer of CFTR by gene tranfer vectors derived from adenoviruses or by trogocytosis of membrane-derived microparticle : molecular mechanisms and applications in cystic fibrosis. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2011LYO10261

Chicago Manual of Style (16th Edition):

Gonzalez, Gaëlle. “Transfert du CFTR par vecteurs de gènes dérivés des adénovirus ou par trogocytose de microparticules membranaires : mécanismes moléculaires et applications à la mucoviscidose : Transfer of CFTR by gene tranfer vectors derived from adenoviruses or by trogocytosis of membrane-derived microparticle : molecular mechanisms and applications in cystic fibrosis.” 2011. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed July 16, 2019. http://www.theses.fr/2011LYO10261.

MLA Handbook (7th Edition):

Gonzalez, Gaëlle. “Transfert du CFTR par vecteurs de gènes dérivés des adénovirus ou par trogocytose de microparticules membranaires : mécanismes moléculaires et applications à la mucoviscidose : Transfer of CFTR by gene tranfer vectors derived from adenoviruses or by trogocytosis of membrane-derived microparticle : molecular mechanisms and applications in cystic fibrosis.” 2011. Web. 16 Jul 2019.

Vancouver:

Gonzalez G. Transfert du CFTR par vecteurs de gènes dérivés des adénovirus ou par trogocytose de microparticules membranaires : mécanismes moléculaires et applications à la mucoviscidose : Transfer of CFTR by gene tranfer vectors derived from adenoviruses or by trogocytosis of membrane-derived microparticle : molecular mechanisms and applications in cystic fibrosis. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2011. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2011LYO10261.

Council of Science Editors:

Gonzalez G. Transfert du CFTR par vecteurs de gènes dérivés des adénovirus ou par trogocytose de microparticules membranaires : mécanismes moléculaires et applications à la mucoviscidose : Transfer of CFTR by gene tranfer vectors derived from adenoviruses or by trogocytosis of membrane-derived microparticle : molecular mechanisms and applications in cystic fibrosis. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2011. Available from: http://www.theses.fr/2011LYO10261

14. Benz, Nathalie. Stimulation de cellules épithéliales bronchiques humaines par la GnRH : effet sur le transport ionique médié par le CFTR : No title.

Degree: Docteur es, Biologie-santé, 2013, Brest

Introduction : La mucoviscidose est une maladie génétique autosomale récessive causée par des mutations dans le gène CFTR (cystic fibrosis transmembrane conductance regulator). Ce dernier… (more)

Subjects/Keywords: CFTR; Mutation F508del; Transport ionique; AnxA5; GnRH; Récepteur de la GnR; CFTR; F508del mutation; Ion transport; AnxA5; GnRH; GnRH recepto

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APA (6th Edition):

Benz, N. (2013). Stimulation de cellules épithéliales bronchiques humaines par la GnRH : effet sur le transport ionique médié par le CFTR : No title. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2013BRES0021

Chicago Manual of Style (16th Edition):

Benz, Nathalie. “Stimulation de cellules épithéliales bronchiques humaines par la GnRH : effet sur le transport ionique médié par le CFTR : No title.” 2013. Doctoral Dissertation, Brest. Accessed July 16, 2019. http://www.theses.fr/2013BRES0021.

MLA Handbook (7th Edition):

Benz, Nathalie. “Stimulation de cellules épithéliales bronchiques humaines par la GnRH : effet sur le transport ionique médié par le CFTR : No title.” 2013. Web. 16 Jul 2019.

Vancouver:

Benz N. Stimulation de cellules épithéliales bronchiques humaines par la GnRH : effet sur le transport ionique médié par le CFTR : No title. [Internet] [Doctoral dissertation]. Brest; 2013. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2013BRES0021.

Council of Science Editors:

Benz N. Stimulation de cellules épithéliales bronchiques humaines par la GnRH : effet sur le transport ionique médié par le CFTR : No title. [Doctoral Dissertation]. Brest; 2013. Available from: http://www.theses.fr/2013BRES0021


Université de Grenoble

15. Maurin, Bruno. Pharmacologie du canal CFTR : développement de molécules activatrices du canal : Pharmacology of CFTR chanel : development of new activators.

Degree: Docteur es, Sciences du médicament, 2012, Université de Grenoble

PHARMACOLOGIE DU CANAL CFTR : SYNTHESE DE NOUVEAUX COMPOSES ACTIVATEURS DE L'EFFLUX DES IONS CHLORURE.Après la découverte dans notre laboratoire d'une nouvelle réaction entre le… (more)

Subjects/Keywords: Mucoviscidose; Canal CFTR; Α-oxoaldehyde; Α-aminoazahétérocycle; Cystic fibrosis; CFTR chanel; Α-oxoaldehyde; Α-aminoazaheterocycle; 570

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APA (6th Edition):

Maurin, B. (2012). Pharmacologie du canal CFTR : développement de molécules activatrices du canal : Pharmacology of CFTR chanel : development of new activators. (Doctoral Dissertation). Université de Grenoble. Retrieved from http://www.theses.fr/2012GRENV085

Chicago Manual of Style (16th Edition):

Maurin, Bruno. “Pharmacologie du canal CFTR : développement de molécules activatrices du canal : Pharmacology of CFTR chanel : development of new activators.” 2012. Doctoral Dissertation, Université de Grenoble. Accessed July 16, 2019. http://www.theses.fr/2012GRENV085.

MLA Handbook (7th Edition):

Maurin, Bruno. “Pharmacologie du canal CFTR : développement de molécules activatrices du canal : Pharmacology of CFTR chanel : development of new activators.” 2012. Web. 16 Jul 2019.

Vancouver:

Maurin B. Pharmacologie du canal CFTR : développement de molécules activatrices du canal : Pharmacology of CFTR chanel : development of new activators. [Internet] [Doctoral dissertation]. Université de Grenoble; 2012. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2012GRENV085.

Council of Science Editors:

Maurin B. Pharmacologie du canal CFTR : développement de molécules activatrices du canal : Pharmacology of CFTR chanel : development of new activators. [Doctoral Dissertation]. Université de Grenoble; 2012. Available from: http://www.theses.fr/2012GRENV085

16. Viart, Victoria. Etude de la régulation transcriptionnelle et post-transcriptionnelle du gène CFTR : identification de facteurs de transcription et de microARNs : Transcriptional and post-transcriptional regulation of the CFTR gene expression : identification of transcription factors and microRNAs.

Degree: Docteur es, Biologie Santé, 2011, Université Montpellier I

Le gène CFTR, impliqué lorsqu'il est muté dans la mucoviscidose, est finement régulé au niveau tissulaire (principalement exprimé dans les organes cibles de la mucoviscidose)… (more)

Subjects/Keywords: Génétique; Régulation transcriptionnelle; Mucoviscidose; Cftr; Facteurs de transcription; MicroARN; Genetics; Transcriptional regulation; Cystic fibrosis; Cftr; Transcription factors; MicroRNA

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APA (6th Edition):

Viart, V. (2011). Etude de la régulation transcriptionnelle et post-transcriptionnelle du gène CFTR : identification de facteurs de transcription et de microARNs : Transcriptional and post-transcriptional regulation of the CFTR gene expression : identification of transcription factors and microRNAs. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2011MON1T031

Chicago Manual of Style (16th Edition):

Viart, Victoria. “Etude de la régulation transcriptionnelle et post-transcriptionnelle du gène CFTR : identification de facteurs de transcription et de microARNs : Transcriptional and post-transcriptional regulation of the CFTR gene expression : identification of transcription factors and microRNAs.” 2011. Doctoral Dissertation, Université Montpellier I. Accessed July 16, 2019. http://www.theses.fr/2011MON1T031.

MLA Handbook (7th Edition):

Viart, Victoria. “Etude de la régulation transcriptionnelle et post-transcriptionnelle du gène CFTR : identification de facteurs de transcription et de microARNs : Transcriptional and post-transcriptional regulation of the CFTR gene expression : identification of transcription factors and microRNAs.” 2011. Web. 16 Jul 2019.

Vancouver:

Viart V. Etude de la régulation transcriptionnelle et post-transcriptionnelle du gène CFTR : identification de facteurs de transcription et de microARNs : Transcriptional and post-transcriptional regulation of the CFTR gene expression : identification of transcription factors and microRNAs. [Internet] [Doctoral dissertation]. Université Montpellier I; 2011. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2011MON1T031.

Council of Science Editors:

Viart V. Etude de la régulation transcriptionnelle et post-transcriptionnelle du gène CFTR : identification de facteurs de transcription et de microARNs : Transcriptional and post-transcriptional regulation of the CFTR gene expression : identification of transcription factors and microRNAs. [Doctoral Dissertation]. Université Montpellier I; 2011. Available from: http://www.theses.fr/2011MON1T031

17. Moisan, Stéphanie. Étude des mécanismes de régulation à distance du gène CFTR : Analysis of long-range regulatory mechanisms of the CFTR gene.

Degree: Docteur es, Biologie santé, 2014, Brest

Le gène CFTR (Cystic Fibrosis Transmembrane conductance Regulator) a été identifié en 1989. Vingt-cinq ans après, les mécanismes contrôlant sa fine expression, sont encore mal… (more)

Subjects/Keywords: CFTR; Régulation transcriptionnelle; Chromatine; Techniques 3C / 5C; CFTR; Transcriptional regulation; Chromatin; 3C / 5C methods; 611.018 16

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APA (6th Edition):

Moisan, S. (2014). Étude des mécanismes de régulation à distance du gène CFTR : Analysis of long-range regulatory mechanisms of the CFTR gene. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2014BRES0018

Chicago Manual of Style (16th Edition):

Moisan, Stéphanie. “Étude des mécanismes de régulation à distance du gène CFTR : Analysis of long-range regulatory mechanisms of the CFTR gene.” 2014. Doctoral Dissertation, Brest. Accessed July 16, 2019. http://www.theses.fr/2014BRES0018.

MLA Handbook (7th Edition):

Moisan, Stéphanie. “Étude des mécanismes de régulation à distance du gène CFTR : Analysis of long-range regulatory mechanisms of the CFTR gene.” 2014. Web. 16 Jul 2019.

Vancouver:

Moisan S. Étude des mécanismes de régulation à distance du gène CFTR : Analysis of long-range regulatory mechanisms of the CFTR gene. [Internet] [Doctoral dissertation]. Brest; 2014. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2014BRES0018.

Council of Science Editors:

Moisan S. Étude des mécanismes de régulation à distance du gène CFTR : Analysis of long-range regulatory mechanisms of the CFTR gene. [Doctoral Dissertation]. Brest; 2014. Available from: http://www.theses.fr/2014BRES0018

18. Philippe, Réginald. Etude des dérégulations de la signalisation calcique et du rôle de la caluménine dans la mucoviscidose : Calcium signaling deregulation and role of calumenin in CF cells.

Degree: Docteur es, Biologie Santé, 2016, Brest

La mucoviscidose (Cystic Fibrosis, CF) est caractérisée par des mutations dans le gène CFTR codant pour un canal chlorure situé à la membrane apicale des… (more)

Subjects/Keywords: Mucoviscidose; CFTR; Mutation F508del; Signalisation calcique; Caluménine; Cystic Fibrosis; CFTR; F508del mutation; Calcium signaling; Calumenin; 616.372; 572.516

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APA (6th Edition):

Philippe, R. (2016). Etude des dérégulations de la signalisation calcique et du rôle de la caluménine dans la mucoviscidose : Calcium signaling deregulation and role of calumenin in CF cells. (Doctoral Dissertation). Brest. Retrieved from http://www.theses.fr/2016BRES0010

Chicago Manual of Style (16th Edition):

Philippe, Réginald. “Etude des dérégulations de la signalisation calcique et du rôle de la caluménine dans la mucoviscidose : Calcium signaling deregulation and role of calumenin in CF cells.” 2016. Doctoral Dissertation, Brest. Accessed July 16, 2019. http://www.theses.fr/2016BRES0010.

MLA Handbook (7th Edition):

Philippe, Réginald. “Etude des dérégulations de la signalisation calcique et du rôle de la caluménine dans la mucoviscidose : Calcium signaling deregulation and role of calumenin in CF cells.” 2016. Web. 16 Jul 2019.

Vancouver:

Philippe R. Etude des dérégulations de la signalisation calcique et du rôle de la caluménine dans la mucoviscidose : Calcium signaling deregulation and role of calumenin in CF cells. [Internet] [Doctoral dissertation]. Brest; 2016. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2016BRES0010.

Council of Science Editors:

Philippe R. Etude des dérégulations de la signalisation calcique et du rôle de la caluménine dans la mucoviscidose : Calcium signaling deregulation and role of calumenin in CF cells. [Doctoral Dissertation]. Brest; 2016. Available from: http://www.theses.fr/2016BRES0010

19. Delion, Martial. COROS - Correction du déficit osseux dans la mucoviscidose. : Effects of CFTR correctors in CF bone disease.

Degree: Docteur es, Pharmacie - STS, 2016, Reims

La maladie osseuse est une complication sévère pour les patients atteints de mucoviscidose (Cystic Fibrosis, CF). Les fractures vertébrales et costales impactent les capacités pulmonaires… (more)

Subjects/Keywords: Mucoviscidose; F508del; Ostéoblastes; Rankl/opg; Pge2; Correcteur de CFTR; Cystic Fibrosis; F508del; Osteoblasts; Rankl/opg; Pge2; CFTR corrector; 615

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APA (6th Edition):

Delion, M. (2016). COROS - Correction du déficit osseux dans la mucoviscidose. : Effects of CFTR correctors in CF bone disease. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2016REIMP203

Chicago Manual of Style (16th Edition):

Delion, Martial. “COROS - Correction du déficit osseux dans la mucoviscidose. : Effects of CFTR correctors in CF bone disease.” 2016. Doctoral Dissertation, Reims. Accessed July 16, 2019. http://www.theses.fr/2016REIMP203.

MLA Handbook (7th Edition):

Delion, Martial. “COROS - Correction du déficit osseux dans la mucoviscidose. : Effects of CFTR correctors in CF bone disease.” 2016. Web. 16 Jul 2019.

Vancouver:

Delion M. COROS - Correction du déficit osseux dans la mucoviscidose. : Effects of CFTR correctors in CF bone disease. [Internet] [Doctoral dissertation]. Reims; 2016. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2016REIMP203.

Council of Science Editors:

Delion M. COROS - Correction du déficit osseux dans la mucoviscidose. : Effects of CFTR correctors in CF bone disease. [Doctoral Dissertation]. Reims; 2016. Available from: http://www.theses.fr/2016REIMP203

20. Zhang, Shaoyi. Role of Chloride in Modulating Autophagy : Cystic Fibrosis as a Disease Model : Rôle du chlorure dans la modulation de l'autophagie : la mucoviscidose en tant que modèle de maladie.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Paris Saclay

Les niveaux de chlorure sont rigoureusement régulés par des canaux de chlorure tels que le régulateur transmembranaire de mucoviscidose (CFTR), la famille de canaux CLC… (more)

Subjects/Keywords: Autophagie; Canal de chlorure; Mucoviscidose; Cftr; Clcn7; Mitochondrie; Autophagy; Chloride Channel; Cystic Fibrosis; Cftr; Clcn7; Mitochondria

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APA (6th Edition):

Zhang, S. (2018). Role of Chloride in Modulating Autophagy : Cystic Fibrosis as a Disease Model : Rôle du chlorure dans la modulation de l'autophagie : la mucoviscidose en tant que modèle de maladie. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2018SACLS178

Chicago Manual of Style (16th Edition):

Zhang, Shaoyi. “Role of Chloride in Modulating Autophagy : Cystic Fibrosis as a Disease Model : Rôle du chlorure dans la modulation de l'autophagie : la mucoviscidose en tant que modèle de maladie.” 2018. Doctoral Dissertation, Paris Saclay. Accessed July 16, 2019. http://www.theses.fr/2018SACLS178.

MLA Handbook (7th Edition):

Zhang, Shaoyi. “Role of Chloride in Modulating Autophagy : Cystic Fibrosis as a Disease Model : Rôle du chlorure dans la modulation de l'autophagie : la mucoviscidose en tant que modèle de maladie.” 2018. Web. 16 Jul 2019.

Vancouver:

Zhang S. Role of Chloride in Modulating Autophagy : Cystic Fibrosis as a Disease Model : Rôle du chlorure dans la modulation de l'autophagie : la mucoviscidose en tant que modèle de maladie. [Internet] [Doctoral dissertation]. Paris Saclay; 2018. [cited 2019 Jul 16]. Available from: http://www.theses.fr/2018SACLS178.

Council of Science Editors:

Zhang S. Role of Chloride in Modulating Autophagy : Cystic Fibrosis as a Disease Model : Rôle du chlorure dans la modulation de l'autophagie : la mucoviscidose en tant que modèle de maladie. [Doctoral Dissertation]. Paris Saclay; 2018. Available from: http://www.theses.fr/2018SACLS178


Dalhousie University

21. Conrad, Dustin. Increased VIP Receptor Expression Mediates CFTR Membrane Localization in Response to VIP Treatment in VIP Knockout Mice.

Degree: MS, Department of Physiology & Biophysics, 2011, Dalhousie University

 Cystic Fibrosis (CF) is caused by mutations in CFTR, a protein for chloride efflux in epithelial cells. VIP is a peptide that activates CFTR and… (more)

Subjects/Keywords: VIP; CFTR; VIPKO; Cystic Fibrosis; VPAC1; VPAC2; PAC1; Lung; Duodenum

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APA (6th Edition):

Conrad, D. (2011). Increased VIP Receptor Expression Mediates CFTR Membrane Localization in Response to VIP Treatment in VIP Knockout Mice. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14231

Chicago Manual of Style (16th Edition):

Conrad, Dustin. “Increased VIP Receptor Expression Mediates CFTR Membrane Localization in Response to VIP Treatment in VIP Knockout Mice.” 2011. Masters Thesis, Dalhousie University. Accessed July 16, 2019. http://hdl.handle.net/10222/14231.

MLA Handbook (7th Edition):

Conrad, Dustin. “Increased VIP Receptor Expression Mediates CFTR Membrane Localization in Response to VIP Treatment in VIP Knockout Mice.” 2011. Web. 16 Jul 2019.

Vancouver:

Conrad D. Increased VIP Receptor Expression Mediates CFTR Membrane Localization in Response to VIP Treatment in VIP Knockout Mice. [Internet] [Masters thesis]. Dalhousie University; 2011. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10222/14231.

Council of Science Editors:

Conrad D. Increased VIP Receptor Expression Mediates CFTR Membrane Localization in Response to VIP Treatment in VIP Knockout Mice. [Masters Thesis]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14231


Universiteit Utrecht

22. Dekkers, J.F. Intestinal organoids as model for cystic fibrosis.

Degree: 2015, Universiteit Utrecht

 Recent advances in adult stem cell culture technology have enabled long-term in vitro expansion of intestinal organoids or ‘mini-guts’. In this thesis, we used the… (more)

Subjects/Keywords: Cystic fibrosis; intestinal organoids; CFTR; correctors; potentiators; personalized/precision medicine

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APA (6th Edition):

Dekkers, J. F. (2015). Intestinal organoids as model for cystic fibrosis. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/321710

Chicago Manual of Style (16th Edition):

Dekkers, J F. “Intestinal organoids as model for cystic fibrosis.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed July 16, 2019. http://dspace.library.uu.nl:8080/handle/1874/321710.

MLA Handbook (7th Edition):

Dekkers, J F. “Intestinal organoids as model for cystic fibrosis.” 2015. Web. 16 Jul 2019.

Vancouver:

Dekkers JF. Intestinal organoids as model for cystic fibrosis. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2019 Jul 16]. Available from: http://dspace.library.uu.nl:8080/handle/1874/321710.

Council of Science Editors:

Dekkers JF. Intestinal organoids as model for cystic fibrosis. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/321710


University of Southern California

23. Salinas, Danieli Barino. Genotype-phenotype associations in children with a positive cystic fibrosis newborn screening.

Degree: MS, Clinical and Biomedical Investigations, 2015, University of Southern California

 Cystic Fibrosis (CF) is an autosomal recessive disorder caused by a defective trans-epithelial channel called CF Transmembrane Conductance Regulator (CFTR). Of the nearly 2000 CFTR(more)

Subjects/Keywords: cystic fibrosis newborn screening; CFTR mutations; genotype-phenotype associations

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APA (6th Edition):

Salinas, D. B. (2015). Genotype-phenotype associations in children with a positive cystic fibrosis newborn screening. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/597264/rec/3023

Chicago Manual of Style (16th Edition):

Salinas, Danieli Barino. “Genotype-phenotype associations in children with a positive cystic fibrosis newborn screening.” 2015. Masters Thesis, University of Southern California. Accessed July 16, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/597264/rec/3023.

MLA Handbook (7th Edition):

Salinas, Danieli Barino. “Genotype-phenotype associations in children with a positive cystic fibrosis newborn screening.” 2015. Web. 16 Jul 2019.

Vancouver:

Salinas DB. Genotype-phenotype associations in children with a positive cystic fibrosis newborn screening. [Internet] [Masters thesis]. University of Southern California; 2015. [cited 2019 Jul 16]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/597264/rec/3023.

Council of Science Editors:

Salinas DB. Genotype-phenotype associations in children with a positive cystic fibrosis newborn screening. [Masters Thesis]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/597264/rec/3023


Universidade de Lisboa

24. Martins, Joana Raquel Delgado, 1984-. Calcium-activated chloride in cystic fibrosis.

Degree: 2011, Universidade de Lisboa

Tese de doutoramento, Bioquímica (Genética Molecular), Universidade de Lisboa, Faculdade de Ciências, 2011

Proper ion transport is crucial for maintenance, hydration and also protection against… (more)

Subjects/Keywords: Fibrose quística; Proteína CFTR; Transporte iónico; Bestrophin; Teses de doutoramento - 2011

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APA (6th Edition):

Martins, Joana Raquel Delgado, 1. (2011). Calcium-activated chloride in cystic fibrosis. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Martins, Joana Raquel Delgado, 1984-. “Calcium-activated chloride in cystic fibrosis.” 2011. Thesis, Universidade de Lisboa. Accessed July 16, 2019. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Martins, Joana Raquel Delgado, 1984-. “Calcium-activated chloride in cystic fibrosis.” 2011. Web. 16 Jul 2019.

Vancouver:

Martins, Joana Raquel Delgado 1. Calcium-activated chloride in cystic fibrosis. [Internet] [Thesis]. Universidade de Lisboa; 2011. [cited 2019 Jul 16]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Martins, Joana Raquel Delgado 1. Calcium-activated chloride in cystic fibrosis. [Thesis]. Universidade de Lisboa; 2011. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/3533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

25. Pócsi, Marianna. A humán epididymis protein 4 (HE4) labordiagnosztikai vizsgálatának jelentősége cisztás fibrózisban .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 A cisztás fibrózis (CF) az egyik leggyakoribb autoszomális recesszív öröklődésmenetű genetikai anyagcsere betegség. A betegség hátterében a CF transzmembrán konduktancia regulátor (CFTR) gén funkcióvesztéssel járó… (more)

Subjects/Keywords: cisztás fibrózis; CFTR; HE4

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pócsi, M. (n.d.). A humán epididymis protein 4 (HE4) labordiagnosztikai vizsgálatának jelentősége cisztás fibrózisban . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/214593

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pócsi, Marianna. “A humán epididymis protein 4 (HE4) labordiagnosztikai vizsgálatának jelentősége cisztás fibrózisban .” Thesis, University of Debrecen. Accessed July 16, 2019. http://hdl.handle.net/2437/214593.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pócsi, Marianna. “A humán epididymis protein 4 (HE4) labordiagnosztikai vizsgálatának jelentősége cisztás fibrózisban .” Web. 16 Jul 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Pócsi M. A humán epididymis protein 4 (HE4) labordiagnosztikai vizsgálatának jelentősége cisztás fibrózisban . [Internet] [Thesis]. University of Debrecen; [cited 2019 Jul 16]. Available from: http://hdl.handle.net/2437/214593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Pócsi M. A humán epididymis protein 4 (HE4) labordiagnosztikai vizsgálatának jelentősége cisztás fibrózisban . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/214593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Duke University

26. Navis, Adam. Roles of Cftr-dependent Fluid Secretion During Organ Morphogenesis and Function .

Degree: 2014, Duke University

  Fluid secretion is essential to organ development and function, yet relatively little is known about the roles of fluid secretion in vivo. Early in… (more)

Subjects/Keywords: Cellular biology; Developmental biology; cftr; fluid secretion; Kupffer's vesicle; pancreas

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Navis, A. (2014). Roles of Cftr-dependent Fluid Secretion During Organ Morphogenesis and Function . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/8705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Navis, Adam. “Roles of Cftr-dependent Fluid Secretion During Organ Morphogenesis and Function .” 2014. Thesis, Duke University. Accessed July 16, 2019. http://hdl.handle.net/10161/8705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Navis, Adam. “Roles of Cftr-dependent Fluid Secretion During Organ Morphogenesis and Function .” 2014. Web. 16 Jul 2019.

Vancouver:

Navis A. Roles of Cftr-dependent Fluid Secretion During Organ Morphogenesis and Function . [Internet] [Thesis]. Duke University; 2014. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/10161/8705.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Navis A. Roles of Cftr-dependent Fluid Secretion During Organ Morphogenesis and Function . [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/8705

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

27. Rimington, Tracy. Expression, Purification and Characterisation of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) in Saccharomyces cerevisiae.

Degree: 2014, University of Manchester

 Mutations in the eukaryotic integral membrane protein Cystic Fibrosis Transmembrane conductance Regulator (CFTR) cause the hereditary disease cystic fibrosis (CF). CFTR functions as an ion… (more)

Subjects/Keywords: CFTR; ABCC7; cystic fibrosis; membrane protein; GFP; ion channel; ABC-transporter

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rimington, T. (2014). Expression, Purification and Characterisation of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) in Saccharomyces cerevisiae. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231349

Chicago Manual of Style (16th Edition):

Rimington, Tracy. “Expression, Purification and Characterisation of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) in Saccharomyces cerevisiae.” 2014. Doctoral Dissertation, University of Manchester. Accessed July 16, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231349.

MLA Handbook (7th Edition):

Rimington, Tracy. “Expression, Purification and Characterisation of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) in Saccharomyces cerevisiae.” 2014. Web. 16 Jul 2019.

Vancouver:

Rimington T. Expression, Purification and Characterisation of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) in Saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Jul 16]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231349.

Council of Science Editors:

Rimington T. Expression, Purification and Characterisation of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) in Saccharomyces cerevisiae. [Doctoral Dissertation]. University of Manchester; 2014. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:231349


Case Western Reserve University

28. Vitko, Megan Sue. Intestinal Dysfunction in Cystic Fibrosis.

Degree: PhD, Genetics, 2016, Case Western Reserve University

 Intestinal Dysfunction in Cystic FibrosisAbstract By MEGAN VITKOCystic Fibrosis (CF) is an autosomal recessive genetic disease that is caused by mutations in the cystic fibrosis… (more)

Subjects/Keywords: Genetics; CFTR; Intestinal Obstruction; Cystic Fibrosis; mouse models

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vitko, M. S. (2016). Intestinal Dysfunction in Cystic Fibrosis. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266

Chicago Manual of Style (16th Edition):

Vitko, Megan Sue. “Intestinal Dysfunction in Cystic Fibrosis.” 2016. Doctoral Dissertation, Case Western Reserve University. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266.

MLA Handbook (7th Edition):

Vitko, Megan Sue. “Intestinal Dysfunction in Cystic Fibrosis.” 2016. Web. 16 Jul 2019.

Vancouver:

Vitko MS. Intestinal Dysfunction in Cystic Fibrosis. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2016. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266.

Council of Science Editors:

Vitko MS. Intestinal Dysfunction in Cystic Fibrosis. [Doctoral Dissertation]. Case Western Reserve University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1459248266


University of British Columbia

29. Chan, Becky Ka Man. Expression of beta subunit of epithelium sodium channel and cystic fibrosis transmembrane regulator in small airways obstruction in chronic obstructive pulmonary disease .

Degree: 2008, University of British Columbia

 Background: Excess plugging of small airways is associated with premature death in chronic obstructive pulmonary disease (COPD). Over-expression of beta-epithelial sodium channel (β-ENaC) in airway… (more)

Subjects/Keywords: ENaC; CFTR; Mucin expression; COPD

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chan, B. K. M. (2008). Expression of beta subunit of epithelium sodium channel and cystic fibrosis transmembrane regulator in small airways obstruction in chronic obstructive pulmonary disease . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/4072

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chan, Becky Ka Man. “Expression of beta subunit of epithelium sodium channel and cystic fibrosis transmembrane regulator in small airways obstruction in chronic obstructive pulmonary disease .” 2008. Thesis, University of British Columbia. Accessed July 16, 2019. http://hdl.handle.net/2429/4072.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chan, Becky Ka Man. “Expression of beta subunit of epithelium sodium channel and cystic fibrosis transmembrane regulator in small airways obstruction in chronic obstructive pulmonary disease .” 2008. Web. 16 Jul 2019.

Vancouver:

Chan BKM. Expression of beta subunit of epithelium sodium channel and cystic fibrosis transmembrane regulator in small airways obstruction in chronic obstructive pulmonary disease . [Internet] [Thesis]. University of British Columbia; 2008. [cited 2019 Jul 16]. Available from: http://hdl.handle.net/2429/4072.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chan BKM. Expression of beta subunit of epithelium sodium channel and cystic fibrosis transmembrane regulator in small airways obstruction in chronic obstructive pulmonary disease . [Thesis]. University of British Columbia; 2008. Available from: http://hdl.handle.net/2429/4072

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

30. Chettiar, Somsundaram Natrajan. Design and Synthesis of Small Molecules as Potential Therapeutic Agents for the Treatment of Cancer and Cystic Fibrosis.

Degree: PhD, Pharmacy, 2013, The Ohio State University

 Cancer is a complex group of distinct genetic disease having common hallmarks. Cancer is characterized by abnormal and uncontrolled cell growth, and may acquire the… (more)

Subjects/Keywords: Pharmaceuticals; Pharmacy Sciences; Survivin, Mps1 kinase, JMJD2 enzyme, CFTR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chettiar, S. N. (2013). Design and Synthesis of Small Molecules as Potential Therapeutic Agents for the Treatment of Cancer and Cystic Fibrosis. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1385559040

Chicago Manual of Style (16th Edition):

Chettiar, Somsundaram Natrajan. “Design and Synthesis of Small Molecules as Potential Therapeutic Agents for the Treatment of Cancer and Cystic Fibrosis.” 2013. Doctoral Dissertation, The Ohio State University. Accessed July 16, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1385559040.

MLA Handbook (7th Edition):

Chettiar, Somsundaram Natrajan. “Design and Synthesis of Small Molecules as Potential Therapeutic Agents for the Treatment of Cancer and Cystic Fibrosis.” 2013. Web. 16 Jul 2019.

Vancouver:

Chettiar SN. Design and Synthesis of Small Molecules as Potential Therapeutic Agents for the Treatment of Cancer and Cystic Fibrosis. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2019 Jul 16]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1385559040.

Council of Science Editors:

Chettiar SN. Design and Synthesis of Small Molecules as Potential Therapeutic Agents for the Treatment of Cancer and Cystic Fibrosis. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1385559040

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