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Jönköping University
1.
Elofsson, Jonathan.
Utvärdering av ett kortare dehydreringsprogram för stansbiopsier från human hud och portio : Har storleken betydelse?.
Degree: Dep. of Natural Science and Biomedicine, 2015, Jönköping University
URL: http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-27420 
► På histopatologiska laboratoriet vid Länssjukhuset Ryhov i Jönköping är dehydreringsprocessen i dagsläget förlagd över natt. För upprätthållande av god kvalitet på diagnos med förbättrat…
(more)
▼ På histopatologiska laboratoriet vid Länssjukhuset Ryhov i Jönköping är dehydreringsprocessen i dagsläget förlagd över natt. För upprätthållande av god kvalitet på diagnos med förbättrat provflöde och utnyttjande av instrumentens fulla kapacitet önskas att ett kortare dehydreringsprogram implementeras och förläggs dagtid. Syftet med denna studie var således att utvärdera ett kortare dehydreringsprogram för stansbiopsier från human hud och portio. Detta har gjorts utifrån cellmorfologiska bedömningsgrunder och bedömning av färgkvalité av rutinfärgning med Hematoxylin och Eosin samt specialfärgningarna polykrommetylenblått enligt Unna, Periodic acid-Schiff och Giemsa. Ett material om 87 biopsier från human hud samt portio dehydrerades fördelat över gällande rutinprogram samt utvärderat kortprogram. Preparaten bedömdes sedan utifrån snittbarhet, morfologisk kvalité samt färgbarhet och diagnostiserbarhet. Resultatet av undersökningen visade att mindre stansbiopsier från kortprogrammet varit enkla att snitta samt att dessa erhållit genomgående goda resultat från övriga bedömningar. Däremot var de större biopsierna mer svårsnittade samt uppvisade fler dehydreringsartefakter när dessa dehydrerats i kortprogram. Sammantaget konstaterades att det korta dehydreringsprogrammet skulle kunna implementeras för mindre stansbiopsier från hud och portio på histopatologiska laboratoriet vid Länssjukhuset Ryhov i Jönköping.
At the histopathological laboratory at Ryhov County Hospital in Jönköping the dehydration process is currently scheduled during nights. To maintain good diagnostic quality with improved sample flow and use of the instruments full capacity an implementation of a shorter dehydration program scheduled during daytime was needed. The purpose was thus to evaluate a shorter dehydration program for punch biopsies of human skin and portio. The evaluation was based on ratings of cellular morphology and quality of routine staining with Hematoxylin and Eosin as well as special staining with polychrome methylene blue according to Unna, Periodic acid-Schiff and Giemsa. A material containing 87 biopsies was dehydrated distributed between current routine program and the evaluated shorter program. The specimens were rated on the basis of ability to section, morphological and staining quality along with diagnostic ability. The results of the survey showed that smaller punch biopsies run in the shorter dehydration program were easy to section and received good results in general. In contrast, larger biopsies were more difficult to section and showed more dehydration artefacts when dehydrated in the shorter program. Altogether it was concluded that the short dehydration program could be implemented for smaller punch biopsies from skin and portio at the histopathological laboratory at Ryhov County Hospital in Jönköping.
Subjects/Keywords: Dehydration; punch biopsy; histotechnical process; staining; cellular morphology; Dehydrering; stansbiopsi; histoteknisk process; färgning; cellmorfologi
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APA (6th Edition):
Elofsson, J. (2015). Utvärdering av ett kortare dehydreringsprogram för stansbiopsier från human hud och portio : Har storleken betydelse?. (Thesis). Jönköping University. Retrieved from http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-27420
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Chicago Manual of Style (16th Edition):
Elofsson, Jonathan. “Utvärdering av ett kortare dehydreringsprogram för stansbiopsier från human hud och portio : Har storleken betydelse?.” 2015. Thesis, Jönköping University. Accessed January 16, 2021.
http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-27420.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
MLA Handbook (7th Edition):
Elofsson, Jonathan. “Utvärdering av ett kortare dehydreringsprogram för stansbiopsier från human hud och portio : Har storleken betydelse?.” 2015. Web. 16 Jan 2021.
Vancouver:
Elofsson J. Utvärdering av ett kortare dehydreringsprogram för stansbiopsier från human hud och portio : Har storleken betydelse?. [Internet] [Thesis]. Jönköping University; 2015. [cited 2021 Jan 16].
Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-27420.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Council of Science Editors:
Elofsson J. Utvärdering av ett kortare dehydreringsprogram för stansbiopsier från human hud och portio : Har storleken betydelse?. [Thesis]. Jönköping University; 2015. Available from: http://urn.kb.se/resolve?urn=urn:nbn:se:hj:diva-27420
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
University of Melbourne
2.
LEAW, BRYAN.
Sodium channels and epilepsy: neuronal dysfunction in genetic mouse models.
Degree: 2014, University of Melbourne
URL: http://hdl.handle.net/11343/40951
► Mutations in sodium channels have long been linked to inherited epilepsies. Recent clinical findings identified patients with Dravet syndrome that were homozygous for a mutation…
(more)
▼ Mutations in sodium channels have long been linked to inherited epilepsies. Recent clinical findings identified patients with Dravet syndrome that were homozygous for a mutation in SCN1B which encodes the β1 auxiliary subunit of sodium channels. Dravet syndrome is a severe childhood epileptic encephalopathy, and patients commonly present with frequent seizures, developmental regression, ataxia with associated gait abnormalities, and shorter lifespans. We have engineered a mouse model based on the human C121W epilepsy mutation (β1-C121W). Mice homozygous for this C121W mutation displayed similar deficits in health and motor skills to Dravet syndrome. Our experiments showed that β1-C121W homozygous neurons fired more action potentials per current injection, had significantly higher membrane resistance, and were more prone to demonstrate a bursting subtype. These hallmarks of neuronal excitability may contribute to the increased sensitivity to thermal seizures in the homozygous mice. Neuron morphology analysis also revealed that neurons within the subiculum of these animals were significantly smaller in size, consistent with the observed increased input resistance. Application of a new anti-epileptic drug, retigabine, successfully reversed the input resistance in homozygous animals down to wildtype levels, and dampened neuronal excitability. Retigabine injected intraperitoneally into homozygous mice was extremely efficient at reducing thermal seizure susceptibility. These findings highlight the potential utility of applying disease-mechanism based strategies to aid anti-epileptic therapy.
In order to examine network excitability in another genetic model of epilepsy, the function of the Nav1.2 sodium channel alpha subunit during development was studied. The NaV1.2 gene has two developmentally regulated splice variants; the ‘neonatal’ and ‘adult’ isoforms. A mutation discovered in patients with benign familial neonatal-infantile epilepsy (BFNIE) increases the excitability of the ‘neonatal’ isoform such that it resembles the adult isoform. Moreover, previous work from the current laboratory using human NaV1.2 expressed in HEK293 cells showed that the ‘neonatal’ form is less excitable than the ‘adult’ form. Based on these data and because the proportion of the neonatal Nav1.2 mRNAs gradually decreases with age during development we hypothesize that the ‘neonatal’ NaV1.2 isoform reduces neuronal excitability in infant brain and therefore plays a protective physiological role. To test this the current laboratory engineered a mouse line which continuously expresses the adult form of Nav1.2 from birth (NaV1.2adult) and investigated seizure susceptibility and neuronal phenotypes. Homozygous NaV1.2adult mice were of normal size and had no obvious seizures under observation during routine video analysis. However, NaV1.2adult mice had increased susceptibility to PTZ-induced seizures, suggesting that the neonatal isoform of NaV1.2 may confer an a novel form of seizure protection. Pyramidal neurons recorded from cortical layers…
Subjects/Keywords: neuroscience; epilepsy; sodium channel; genetic; mouse model; animal model; patching; cellular biology; neurophysiology; network; excitability; development; splice variant; behavior; immunohistochemistry; staining; confocal; microscopy; tracing; morphology
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APA (6th Edition):
LEAW, B. (2014). Sodium channels and epilepsy: neuronal dysfunction in genetic mouse models. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/40951
Chicago Manual of Style (16th Edition):
LEAW, BRYAN. “Sodium channels and epilepsy: neuronal dysfunction in genetic mouse models.” 2014. Doctoral Dissertation, University of Melbourne. Accessed January 16, 2021.
http://hdl.handle.net/11343/40951.
MLA Handbook (7th Edition):
LEAW, BRYAN. “Sodium channels and epilepsy: neuronal dysfunction in genetic mouse models.” 2014. Web. 16 Jan 2021.
Vancouver:
LEAW B. Sodium channels and epilepsy: neuronal dysfunction in genetic mouse models. [Internet] [Doctoral dissertation]. University of Melbourne; 2014. [cited 2021 Jan 16].
Available from: http://hdl.handle.net/11343/40951.
Council of Science Editors:
LEAW B. Sodium channels and epilepsy: neuronal dysfunction in genetic mouse models. [Doctoral Dissertation]. University of Melbourne; 2014. Available from: http://hdl.handle.net/11343/40951
3.
Choi, Sungmoon.
Fluorescent noble metal nanodots for biological applications.
Degree: PhD, Chemistry and Biochemistry, 2010, Georgia Tech
URL: http://hdl.handle.net/1853/37195
► Commercial organic dyes are widely used for cellular staining due to their small size, high brightness, and chemical functionality. However, their blinking and photobleaching are…
(more)
▼ Commercial organic dyes are widely used for
cellular staining due to their small size, high brightness, and chemical functionality. However, their blinking and photobleaching are not ideal for studying dynamics inside live cells. An improvement over organics and much larger quantum dots, silver nanodots (Ag NDs) exhibit low cytotoxicity and excellent brightness and photostability, while retaining small size. We have utilized ssDNA hairpin structures to encapsulate Ag NDs with excellent spectral purity, high concentration, and good chemical and photophysical stability in a variety of biological media. Multi-color
staining of fixed and live cells has been achieved, suggesting the promise of Ag NDs as good fluorophores for intracellular imaging. The great brightness and photostability of Ag nanodots indicate that they might be outstanding imaging agents for in vivo studies when encapsulated in delivery vehicles. In addition, Ag NDs can be optically modulated, resulting in increased sensitivity within high backgrounds. These good characteristics are combined with delivery vehicles such as PLGA and nanogels. After encapsulation, Ag nanodots still retain their good photophysical properties and modulation. It might be useful for in vivo applications in the near future
Advisors/Committee Members: Robert M. Dickson (Committee Chair), Christoph Fahrni (Committee Member), L. Andrew Lyon (Committee Member), Mostafa A. El-Sayed (Committee Member), Niren Murthy (Committee Member).
Subjects/Keywords: DNA; Nanodots; Drug delivery; Cellular staining; Fluorophores; Silver; Drug delivery systems; Biomedical materials Imaging compatibility; Diagnostic imaging
…Commercial organic dyes are widely used for cellular staining due to their small
size, high… …alternative to
commercial organic dyes and quantum dots for cellular staining with excellent… …chemical and photophysical stability. Their potential application as cellular
staining agent was… …Modern
imaging
and
biological
studies
require
better
fluorophores.
Cellular staining… …cellular staining, with high chromophore loading concentration.99-103 However, as
mentioned above…
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APA (6th Edition):
Choi, S. (2010). Fluorescent noble metal nanodots for biological applications. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/37195
Chicago Manual of Style (16th Edition):
Choi, Sungmoon. “Fluorescent noble metal nanodots for biological applications.” 2010. Doctoral Dissertation, Georgia Tech. Accessed January 16, 2021.
http://hdl.handle.net/1853/37195.
MLA Handbook (7th Edition):
Choi, Sungmoon. “Fluorescent noble metal nanodots for biological applications.” 2010. Web. 16 Jan 2021.
Vancouver:
Choi S. Fluorescent noble metal nanodots for biological applications. [Internet] [Doctoral dissertation]. Georgia Tech; 2010. [cited 2021 Jan 16].
Available from: http://hdl.handle.net/1853/37195.
Council of Science Editors:
Choi S. Fluorescent noble metal nanodots for biological applications. [Doctoral Dissertation]. Georgia Tech; 2010. Available from: http://hdl.handle.net/1853/37195
4.
Νικολάου, Μαργαρίτα.
Μελέτη της κυτταρικής ανοσίας στην ηπατίτιδα C.
Degree: 2007, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)
URL: http://hdl.handle.net/10442/hedi/18506
► Hepatitis C virus (HCV) is a major cause of chronic hepatitis worldwide. The immune mechanisms that promote viral persistence in HCV infection are not fully…
(more)
▼ Hepatitis C virus (HCV) is a major cause of chronic hepatitis worldwide. The immune mechanisms that promote viral persistence in HCV infection are not fully clarified. However, cellular immune responses appear to be crucial in viral control and outcome of chronic HCV infection. We studied the peripheral cellular immune responses in 34 patients with chronic HCV infection (25 with elevated and 9 with persistently normal ALT levels) and compared them with those observed in 11 individuals with past HCV infection and 24 healthy volunteers who served as controls. T cell responses to recombinant HCV proteins and synthetic peptides corresponding to known amino acid sequences of HCV core, NS3 and NS4 were analysed after specific and maximal non specific stimulation. IFN-# production was assessed using Elispot and intracellular cytokine staining and CD4+ T cell activity was measured using proliferating assays. Individuals with past HCV infection exhibited the most frequent proliferating T-cell responses against mainly non-structural HCV proteins while IFN-# production by CD8 T cells was relatively weak in comparison to patients with chronic HCV infection. Patients with chronic HCV infection and elevated ALT levels displayed an overall increased production of IFN-# after specific and non-specific stimulation. Patients with chronic HCV infection and normal ALT levels had minimal proliferating responses against whole HCV proteins but not HCV derived peptides while they exhibited an increased number of Core21-40aa-specific T cells. Conclusively, we detected enhanced proliferating activity of peripheral T cells in individuals with past HCV infection. Patients with chronic HCV infection and elevated ALT levels displayed vigorous CD8+ T cell effector responses, which appear to contribute to the establishment of HCV persistence in chronic HCV infection.
Subjects/Keywords: Ηπατίτιδα C, Ιός (HCV); Ανοσία; Τ κύτταρα; Κυτταρική απάντηση; Ιντερφερόνη-γ; Λεμφοκυτταρικές καλλιέργειες; Κυτταρομετρία ροής; HCV; Cellular immune responses; CD4; CD8; IFN-γ; Lymphoproliferative assays; Intracellular staining; T cells
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APA ·
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Manager
APA (6th Edition):
Νικολάου, . . (2007). Μελέτη της κυτταρικής ανοσίας στην ηπατίτιδα C. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/18506
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Chicago Manual of Style (16th Edition):
Νικολάου, Μαργαρίτα. “Μελέτη της κυτταρικής ανοσίας στην ηπατίτιδα C.” 2007. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed January 16, 2021.
http://hdl.handle.net/10442/hedi/18506.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
MLA Handbook (7th Edition):
Νικολάου, Μαργαρίτα. “Μελέτη της κυτταρικής ανοσίας στην ηπατίτιδα C.” 2007. Web. 16 Jan 2021.
Vancouver:
Νικολάου . Μελέτη της κυτταρικής ανοσίας στην ηπατίτιδα C. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2007. [cited 2021 Jan 16].
Available from: http://hdl.handle.net/10442/hedi/18506.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Council of Science Editors:
Νικολάου . Μελέτη της κυτταρικής ανοσίας στην ηπατίτιδα C. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2007. Available from: http://hdl.handle.net/10442/hedi/18506
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
5.
Bhat, Samerna.
Impact of Nicotine and PPARd-agonist on Human Mesenchymal
Stem Cells.
Degree: MSBS, College of Medicine, 2013, University of Toledo Health Science Campus
URL: http://rave.ohiolink.edu/etdc/view?acc_num=mco1366337315
► Smoking is a well established factor in many diseases and has multiple systemic effects due to more than 4000 different molecular species present in cigarette…
(more)
▼ Smoking is a well established factor in many diseases
and has multiple systemic effects due to more than 4000 different
molecular species present in cigarette smoke. Osteoporosis, low
bone mineral density, increased nonunion and delayed union, and
increased risk of bone fracture have been associated with cigarette
smoking. Nicotine, the main component of cigarette smoke is
responsible for addiction. Previous studies have demonstrated
deleterious impact of nicotine on skeletal remodeling and bone
metabolism. Although many studies in the past have used osteoblasts
and osteoblast like cells to study the impact of nicotine on bone,
the use of human mesenchymal stem cells to determine the effects of
nicotine has been rare. Therefore, learning whether or not these
cells that go on to differentiate into osteoblasts and chondrocytes
are also affected by nicotine will be particularly valuable in
predicting the prognosis of a smoker undergoing orthopedic
surgery/procedure. In this context, we examined the impact of
nicotine in physiological range (0.1µM to 10 µM) on: a) hMSC
proliferation b) Calcium deposition by osteoblasts (Alizarin red
staining) c) Alkaline phosphatase activity (ALP assay on day) d)
expression of canonical genes during differentiation of hMSCs
(western blot analysis). Our results demonstrated a dose dependent
decrease in hMSC proliferation, calcium deposition, ALP activity
and expression of BMP-2 and HO-1. Interestingly, induction of heme
oxygenase-1 (HO-1) by peroxisome proliferator-activated receptor
delta (PPARd) agonist, GW0742, prevented the negative effect of
nicotine. These results led to the conclusion that nicotine has a
damaging effect on hMSCs proliferation and osteogenic
differentiation and the induction of HO-1 by GW0742 results in the
reversal of these effects. This offers an opportunity for HO-1
inducers to be used as therapeutic agents to improve bone fusion
and fracture healing in smokers and non-smokers.
Advisors/Committee Members: Ebraheim, Nabil (Committee Chair).
Subjects/Keywords: Biomechanics; Biomedical Research; Cellular Biology; mesenchymal stem cell; smoking; nicotine; PPARd-agonist; HO-1; BMP-2; ALP; osteoblast; Alizarin red staining
…a free radical causing a chain reaction. Cellular damage or death can be caused
by both… …cigarettes.29 At
cellular level, nicotine has deleterious effects on osteoblasts, macrophages and… …characterized.
PPARs regulate a variety of cellular functions through isotype specific tissue… …proliferator-activated receptors are
nuclear receptors at the crossroads of key cellular functions… …1 can be induced by numerous pharmacological agents as well as by
conditions like cellular…
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APA (6th Edition):
Bhat, S. (2013). Impact of Nicotine and PPARd-agonist on Human Mesenchymal
Stem Cells. (Masters Thesis). University of Toledo Health Science Campus. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=mco1366337315
Chicago Manual of Style (16th Edition):
Bhat, Samerna. “Impact of Nicotine and PPARd-agonist on Human Mesenchymal
Stem Cells.” 2013. Masters Thesis, University of Toledo Health Science Campus. Accessed January 16, 2021.
http://rave.ohiolink.edu/etdc/view?acc_num=mco1366337315.
MLA Handbook (7th Edition):
Bhat, Samerna. “Impact of Nicotine and PPARd-agonist on Human Mesenchymal
Stem Cells.” 2013. Web. 16 Jan 2021.
Vancouver:
Bhat S. Impact of Nicotine and PPARd-agonist on Human Mesenchymal
Stem Cells. [Internet] [Masters thesis]. University of Toledo Health Science Campus; 2013. [cited 2021 Jan 16].
Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1366337315.
Council of Science Editors:
Bhat S. Impact of Nicotine and PPARd-agonist on Human Mesenchymal
Stem Cells. [Masters Thesis]. University of Toledo Health Science Campus; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=mco1366337315
Queensland University of Technology
6.
Bui, Loan Thuy.
Localisation of kallikreins in the prostate and association with prostate cancer progression.
Degree: 2006, Queensland University of Technology
URL: https://eprints.qut.edu.au/16276/
► At present, prostate cancer is a significant public health issue throughout the world and is the second leading cause of cancer deaths in older men.…
(more)
▼ At present, prostate cancer is a significant public health issue throughout the world and is the second leading cause of cancer deaths in older men. The prostate specific antigen or PSA (which is encoded by the kallikrein 3/KLK3 gene) test is the current most valuable tool for the diagnosis and management of prostate cancer. However, it is insufficiently sensitive and specific for early diagnosis, for staging of prostate cancer or for discriminating between benign prostatic hyperplasia (BPH) and prostate cancer. Recent research has revealed another potential tumour marker, glandular kallikrein 2 (KLK2 gene/hK2 protein), which may be used alone or in conjunction with PSA to overcome some of the limitations of the PSA test. Twelve new kallikrein gene family members have been recently identified and, like hK2 and PSA, many of these genes have been suggested to be involved in carcinogenesis. In this study, the cellular localisation and level of expression of several of these newer kallikreins (KLK4, KLK5, KLK7, KLK8 and KLK11) was examined in prostate tissue, to provide an understanding of the association of their expression with prostatic diseases and their potential as additional biomarkers. Like PSA and hK2, the present observation indicated that each of these proteins, hK4, hK5, hK7, hK8 and hK11, was detected within the cytoplasm of the secretory cells of the prostate glands. For the first time, all of these newly-identified proteins were shown to be expressed in prostatic intraepithelial neoplasia (PIN) lesions, in comparison to normal glands and cancer lesions. In addition to cytoplasmic secretory cell expression, the localisation of hK4 to the basal cells and nuclei in prostatic lesions was intriguing. The intensity of hK4 staining in prostate tissue was strongest in comparison to the other newly-identified kallikrein proteins (hK5, hK7, hK8 and hK11). Therefore, KLK4/hK4 expression was characterised further to define this cellular localisation and examined in non-prostatic tissue and also in a larger number of prostate tissues in an attempt to determine its potential value as a biomarker for prostate disease. Three hK4 antipeptide polyclonal antibodies, derived against N-terminal, mid-region and C-terminal hK4 amino acid sequences, were used. The hK4 N-terminal antipeptide antibody was used to demonstrate the cellular localisation of hK4 in kidney, salivary glands, liver, testis, colon carcinoma, heart, endometrium and ovarian cancer, for the first time. The presence of hK4 in these non-prostate tissues was consistent with the previous reports using RT-PCR. The dual cytoplasmic and nuclear localisation of hK4 observed in the prostate above was also seen in these tissues. Although hK4 was found widely expressed in many human tissue types, indicating that it is not prostate specific in its expression, the highest expression level of hK4 was seen in the prostate. Therefore, detailed expression patterns and levels of KLK4 mRNA and hK4 protein in the normal prostate and prostatic diseases and…
Subjects/Keywords: Prostate cancer; Prostate cancer progression; Gleason grade; Benign prostatic hyperplasia (BPH); Prostatic intraepithelial neoplasia (PIN); Bone metastases from prostate cancer; Transgenic adenocarcinoma mouse prostate (TRAMP); Kallikreins; KLK4/hK4 (prostase; KLK-L1 protein; EMSP-1); Prostate specific antigen (PSA; KLK3); Human glandular kallikrein (KLK2/hK2); KLK5/hK5 (KLK-L2; HSCTE); KLK7/hK7 (PRSS6; HSCCE); KLK8/hK8 (PRSS19; Neuropsin; Ovasin); KLK11/hK11 (PRSS20; TLSP; Hippostasin); Immunohistochemistry; In situ hybridisation; Secretory cells; Basal cells; Nuclear staining; Cellular localisation
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APA ·
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CSE |
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Manager
APA (6th Edition):
Bui, L. T. (2006). Localisation of kallikreins in the prostate and association with prostate cancer progression. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/16276/
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Chicago Manual of Style (16th Edition):
Bui, Loan Thuy. “Localisation of kallikreins in the prostate and association with prostate cancer progression.” 2006. Thesis, Queensland University of Technology. Accessed January 16, 2021.
https://eprints.qut.edu.au/16276/.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
MLA Handbook (7th Edition):
Bui, Loan Thuy. “Localisation of kallikreins in the prostate and association with prostate cancer progression.” 2006. Web. 16 Jan 2021.
Vancouver:
Bui LT. Localisation of kallikreins in the prostate and association with prostate cancer progression. [Internet] [Thesis]. Queensland University of Technology; 2006. [cited 2021 Jan 16].
Available from: https://eprints.qut.edu.au/16276/.
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
Council of Science Editors:
Bui LT. Localisation of kallikreins in the prostate and association with prostate cancer progression. [Thesis]. Queensland University of Technology; 2006. Available from: https://eprints.qut.edu.au/16276/
Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
. 
Open Access Theses and Dissertations
