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You searched for subject:(Cellular biology). Showing records 1 – 30 of 2726 total matches.

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Wright State University

1. Battini, Vishnu Priya Chowdary. Accurate splicing of HDAC6 requires Son.

Degree: MS, Biological Sciences, 2013, Wright State University

 Pre-mRNA splicing requires proper splice site selection mediated by many factors including snRNPs and serine-arginine rich (SR) splicing factors. Son is the largest known SR… (more)

Subjects/Keywords: Cellular Biology; cellular biology

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APA (6th Edition):

Battini, V. P. C. (2013). Accurate splicing of HDAC6 requires Son. (Masters Thesis). Wright State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=wright1390306890

Chicago Manual of Style (16th Edition):

Battini, Vishnu Priya Chowdary. “Accurate splicing of HDAC6 requires Son.” 2013. Masters Thesis, Wright State University. Accessed November 19, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=wright1390306890.

MLA Handbook (7th Edition):

Battini, Vishnu Priya Chowdary. “Accurate splicing of HDAC6 requires Son.” 2013. Web. 19 Nov 2017.

Vancouver:

Battini VPC. Accurate splicing of HDAC6 requires Son. [Internet] [Masters thesis]. Wright State University; 2013. [cited 2017 Nov 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1390306890.

Council of Science Editors:

Battini VPC. Accurate splicing of HDAC6 requires Son. [Masters Thesis]. Wright State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=wright1390306890


Temple University

2. Frara, Nagat. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.

Degree: PhD, 2015, Temple University

Cell Biology

Osteoactivin (OA) is a novel osteogenic and repair factor. It has the ability to regulate cell proliferation, adhesion, differentiation, and synthesis and regulation… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Frara, N. (2015). THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,336629

Chicago Manual of Style (16th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Doctoral Dissertation, Temple University. Accessed November 19, 2017. http://digital.library.temple.edu/u?/p245801coll10,336629.

MLA Handbook (7th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Web. 19 Nov 2017.

Vancouver:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2017 Nov 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629.

Council of Science Editors:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629


University of California – San Diego

3. Williams, Wesley. Elucidating Hepatitis C Virus Core Interactions with the Host Environment, Specifically Phosphorylated IkappaB-alpha.

Degree: Biology (Joint Doctoral SDSU), 2017, University of California – San Diego

 Hepatitis C Virus (HCV) is a blood-borne virus found worldwide, though most prevalent in third world countries. Currently, there is no vaccine and chronic infection… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Williams, W. (2017). Elucidating Hepatitis C Virus Core Interactions with the Host Environment, Specifically Phosphorylated IkappaB-alpha. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/8cd809c3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Williams, Wesley. “Elucidating Hepatitis C Virus Core Interactions with the Host Environment, Specifically Phosphorylated IkappaB-alpha.” 2017. Thesis, University of California – San Diego. Accessed November 19, 2017. http://www.escholarship.org/uc/item/8cd809c3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Williams, Wesley. “Elucidating Hepatitis C Virus Core Interactions with the Host Environment, Specifically Phosphorylated IkappaB-alpha.” 2017. Web. 19 Nov 2017.

Vancouver:

Williams W. Elucidating Hepatitis C Virus Core Interactions with the Host Environment, Specifically Phosphorylated IkappaB-alpha. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2017 Nov 19]. Available from: http://www.escholarship.org/uc/item/8cd809c3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Williams W. Elucidating Hepatitis C Virus Core Interactions with the Host Environment, Specifically Phosphorylated IkappaB-alpha. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/8cd809c3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

4. Sullivan, William Juster. Hyaluronidase Promotes Glucose Metabolism: Identification of the Extracellular Matrix as a Node of Cell-Extrinsic Metabolic Regulation.

Degree: Molecular and Medical Pharmacology, 2017, UCLA

 The metabolic state of a cell can be determined by cell-extrinsic factors, including nutrient availability and growth factor signaling. Here, we define extracellular matrix (ECM)… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Sullivan, W. J. (2017). Hyaluronidase Promotes Glucose Metabolism: Identification of the Extracellular Matrix as a Node of Cell-Extrinsic Metabolic Regulation. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/4fj8f40n

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sullivan, William Juster. “Hyaluronidase Promotes Glucose Metabolism: Identification of the Extracellular Matrix as a Node of Cell-Extrinsic Metabolic Regulation.” 2017. Thesis, UCLA. Accessed November 19, 2017. http://www.escholarship.org/uc/item/4fj8f40n.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sullivan, William Juster. “Hyaluronidase Promotes Glucose Metabolism: Identification of the Extracellular Matrix as a Node of Cell-Extrinsic Metabolic Regulation.” 2017. Web. 19 Nov 2017.

Vancouver:

Sullivan WJ. Hyaluronidase Promotes Glucose Metabolism: Identification of the Extracellular Matrix as a Node of Cell-Extrinsic Metabolic Regulation. [Internet] [Thesis]. UCLA; 2017. [cited 2017 Nov 19]. Available from: http://www.escholarship.org/uc/item/4fj8f40n.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sullivan WJ. Hyaluronidase Promotes Glucose Metabolism: Identification of the Extracellular Matrix as a Node of Cell-Extrinsic Metabolic Regulation. [Thesis]. UCLA; 2017. Available from: http://www.escholarship.org/uc/item/4fj8f40n

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Washington State University

5. [No author]. The Role of Stress Response Factors in the Development and Stress Resistance of the Zebrafish, Danio rerio .

Degree: 2011, Washington State University

 Cells respond to physiological and chemical insults by the upregulation and activation of the stress response. This response, also termed the heat shock response, is… (more)

Subjects/Keywords: Cellular Biology

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APA (6th Edition):

author], [. (2011). The Role of Stress Response Factors in the Development and Stress Resistance of the Zebrafish, Danio rerio . (Thesis). Washington State University. Retrieved from http://hdl.handle.net/2376/2860

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “The Role of Stress Response Factors in the Development and Stress Resistance of the Zebrafish, Danio rerio .” 2011. Thesis, Washington State University. Accessed November 19, 2017. http://hdl.handle.net/2376/2860.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “The Role of Stress Response Factors in the Development and Stress Resistance of the Zebrafish, Danio rerio .” 2011. Web. 19 Nov 2017.

Vancouver:

author] [. The Role of Stress Response Factors in the Development and Stress Resistance of the Zebrafish, Danio rerio . [Internet] [Thesis]. Washington State University; 2011. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/2376/2860.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. The Role of Stress Response Factors in the Development and Stress Resistance of the Zebrafish, Danio rerio . [Thesis]. Washington State University; 2011. Available from: http://hdl.handle.net/2376/2860

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


The Ohio State University

6. Sever, Nurettin Ilter. Investigation of the Role of Different Regions of the Cbl Protein in its Function.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2013, The Ohio State University

 The duration and amplitude of any signal transduction pathway should be tightly regulated for the maintenance of homeostasis in any organism. Cbl protein is one… (more)

Subjects/Keywords: Cellular Biology

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APA (6th Edition):

Sever, N. I. (2013). Investigation of the Role of Different Regions of the Cbl Protein in its Function. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1386059140

Chicago Manual of Style (16th Edition):

Sever, Nurettin Ilter. “Investigation of the Role of Different Regions of the Cbl Protein in its Function.” 2013. Doctoral Dissertation, The Ohio State University. Accessed November 19, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1386059140.

MLA Handbook (7th Edition):

Sever, Nurettin Ilter. “Investigation of the Role of Different Regions of the Cbl Protein in its Function.” 2013. Web. 19 Nov 2017.

Vancouver:

Sever NI. Investigation of the Role of Different Regions of the Cbl Protein in its Function. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2017 Nov 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1386059140.

Council of Science Editors:

Sever NI. Investigation of the Role of Different Regions of the Cbl Protein in its Function. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1386059140


University of Cincinnati

7. WILLIAMS, JON. EFFECTS OF LOSS OF NF1 GENE ON PERIPHERAL NERVOUS SYSTEM PROGENITORS AND TUMORIGENESIS.

Degree: PhD, Medicine : Neuroscience/Medical Science Scholars Interdisiplinary, 2008, University of Cincinnati

 This dissertation contains three independent studies involving the developmental cell biology of neural crest-derived glial lineage cells in the context of neurofibromatosis, type 1 (NF1).… (more)

Subjects/Keywords: Cellular Biology

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APA (6th Edition):

WILLIAMS, J. (2008). EFFECTS OF LOSS OF NF1 GENE ON PERIPHERAL NERVOUS SYSTEM PROGENITORS AND TUMORIGENESIS. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1212181112

Chicago Manual of Style (16th Edition):

WILLIAMS, JON. “EFFECTS OF LOSS OF NF1 GENE ON PERIPHERAL NERVOUS SYSTEM PROGENITORS AND TUMORIGENESIS.” 2008. Doctoral Dissertation, University of Cincinnati. Accessed November 19, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1212181112.

MLA Handbook (7th Edition):

WILLIAMS, JON. “EFFECTS OF LOSS OF NF1 GENE ON PERIPHERAL NERVOUS SYSTEM PROGENITORS AND TUMORIGENESIS.” 2008. Web. 19 Nov 2017.

Vancouver:

WILLIAMS J. EFFECTS OF LOSS OF NF1 GENE ON PERIPHERAL NERVOUS SYSTEM PROGENITORS AND TUMORIGENESIS. [Internet] [Doctoral dissertation]. University of Cincinnati; 2008. [cited 2017 Nov 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1212181112.

Council of Science Editors:

WILLIAMS J. EFFECTS OF LOSS OF NF1 GENE ON PERIPHERAL NERVOUS SYSTEM PROGENITORS AND TUMORIGENESIS. [Doctoral Dissertation]. University of Cincinnati; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1212181112


The Ohio State University

8. Yang, Jennifer. Targeted Therapeutics against Biliary Cancer Elicit Concurrent Tumor Extrinsic Effects on Immune Suppression and Cancer Cachexia.

Degree: PhD, Molecular, Cellular and Developmental Biology, 2016, The Ohio State University

 Biliary tract cancer (BTC) represents a rare but deadly disease due to difficulties in diagnosis and identifying effective treatment regimens. Even with continual improvement in… (more)

Subjects/Keywords: Cellular Biology

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APA (6th Edition):

Yang, J. (2016). Targeted Therapeutics against Biliary Cancer Elicit Concurrent Tumor Extrinsic Effects on Immune Suppression and Cancer Cachexia. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1461777433

Chicago Manual of Style (16th Edition):

Yang, Jennifer. “Targeted Therapeutics against Biliary Cancer Elicit Concurrent Tumor Extrinsic Effects on Immune Suppression and Cancer Cachexia.” 2016. Doctoral Dissertation, The Ohio State University. Accessed November 19, 2017. http://rave.ohiolink.edu/etdc/view?acc_num=osu1461777433.

MLA Handbook (7th Edition):

Yang, Jennifer. “Targeted Therapeutics against Biliary Cancer Elicit Concurrent Tumor Extrinsic Effects on Immune Suppression and Cancer Cachexia.” 2016. Web. 19 Nov 2017.

Vancouver:

Yang J. Targeted Therapeutics against Biliary Cancer Elicit Concurrent Tumor Extrinsic Effects on Immune Suppression and Cancer Cachexia. [Internet] [Doctoral dissertation]. The Ohio State University; 2016. [cited 2017 Nov 19]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1461777433.

Council of Science Editors:

Yang J. Targeted Therapeutics against Biliary Cancer Elicit Concurrent Tumor Extrinsic Effects on Immune Suppression and Cancer Cachexia. [Doctoral Dissertation]. The Ohio State University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1461777433


Duke University

9. Chen, Hsin. Cytoskeletal Dynamics and the Temporal Control of Yeast Morphogenesis.

Degree: 2012, Duke University

  The cells of the budding yeast Saccharomyces cerevisiae undergo a robust morphological cycle, involving reorganization of the actin cytoskeleton, septin ring formation, and polarized… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Chen, H. (2012). Cytoskeletal Dynamics and the Temporal Control of Yeast Morphogenesis. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5494

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Hsin. “Cytoskeletal Dynamics and the Temporal Control of Yeast Morphogenesis.” 2012. Thesis, Duke University. Accessed November 19, 2017. http://hdl.handle.net/10161/5494.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Hsin. “Cytoskeletal Dynamics and the Temporal Control of Yeast Morphogenesis.” 2012. Web. 19 Nov 2017.

Vancouver:

Chen H. Cytoskeletal Dynamics and the Temporal Control of Yeast Morphogenesis. [Internet] [Thesis]. Duke University; 2012. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/10161/5494.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen H. Cytoskeletal Dynamics and the Temporal Control of Yeast Morphogenesis. [Thesis]. Duke University; 2012. Available from: http://hdl.handle.net/10161/5494

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

10. Gemberling, Matthew P. Mechanisms that drive cardiomyocyte proliferation during zebrafish heart regeneration.

Degree: 2014, Duke University

  Heart disease is the leading cause of death in the developed world. Adult mammals cannot replace lost cardiac tissue after injury, leading to reduced… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Gemberling, M. P. (2014). Mechanisms that drive cardiomyocyte proliferation during zebrafish heart regeneration. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gemberling, Matthew P. “Mechanisms that drive cardiomyocyte proliferation during zebrafish heart regeneration.” 2014. Thesis, Duke University. Accessed November 19, 2017. http://hdl.handle.net/10161/9412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gemberling, Matthew P. “Mechanisms that drive cardiomyocyte proliferation during zebrafish heart regeneration.” 2014. Web. 19 Nov 2017.

Vancouver:

Gemberling MP. Mechanisms that drive cardiomyocyte proliferation during zebrafish heart regeneration. [Internet] [Thesis]. Duke University; 2014. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/10161/9412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gemberling MP. Mechanisms that drive cardiomyocyte proliferation during zebrafish heart regeneration. [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/9412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

11. McClure, Allison Wolff. Pheromone Gradient Tracking Mechanisms During Yeast Mating.

Degree: 2014, Duke University

  Many cell types are remarkably adept at tracking chemical gradients, but they use different mechanisms in order to properly migrate or grow up-gradient. Bacteria… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

McClure, A. W. (2014). Pheromone Gradient Tracking Mechanisms During Yeast Mating. (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9419

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McClure, Allison Wolff. “Pheromone Gradient Tracking Mechanisms During Yeast Mating.” 2014. Thesis, Duke University. Accessed November 19, 2017. http://hdl.handle.net/10161/9419.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McClure, Allison Wolff. “Pheromone Gradient Tracking Mechanisms During Yeast Mating.” 2014. Web. 19 Nov 2017.

Vancouver:

McClure AW. Pheromone Gradient Tracking Mechanisms During Yeast Mating. [Internet] [Thesis]. Duke University; 2014. [cited 2017 Nov 19]. Available from: http://hdl.handle.net/10161/9419.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McClure AW. Pheromone Gradient Tracking Mechanisms During Yeast Mating. [Thesis]. Duke University; 2014. Available from: http://hdl.handle.net/10161/9419

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Columbia University

12. Guilfoyle, Eileen M. Stressed Astrocytes: Insights on the Pathology of Alexander Disease.

Degree: PhD, Cellular, Molecular and Biomedical StudiesPathology, 2013, Columbia University

 Alexander disease (AxD) is a rare and fatal neurological disorder caused by mutations in the gene that encodes glial fibrillary acidic protein (GFAP), an intermediate… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Guilfoyle, E. M. (2013). Stressed Astrocytes: Insights on the Pathology of Alexander Disease. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:174689 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Guilfoyle, Eileen M. “Stressed Astrocytes: Insights on the Pathology of Alexander Disease.” 2013. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:174689 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Guilfoyle, Eileen M. “Stressed Astrocytes: Insights on the Pathology of Alexander Disease.” 2013. Web. 19 Nov 2017.

Vancouver:

Guilfoyle EM. Stressed Astrocytes: Insights on the Pathology of Alexander Disease. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:174689 %%% handle:10022/AC.

Council of Science Editors:

Guilfoyle EM. Stressed Astrocytes: Insights on the Pathology of Alexander Disease. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://academiccommons.columbia.edu/catalog/ac:174689 %%% handle:10022/AC


Columbia University

13. Vevea, Jason D. Cellular Response to Membrane Phospholipid Imbalance, in Yeast and in Human Disease.

Degree: PhD, Pathobiology and Molecular Medicine, 2015, Columbia University

 Organelles sequester biological phenomena within the cell, and allow an additional layer of complexity to life. The presence and maintenance of these organelles is crucial… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Vevea, J. D. (2015). Cellular Response to Membrane Phospholipid Imbalance, in Yeast and in Human Disease. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:195460 %%% doi:10.7916/D8SQ8Z75

Chicago Manual of Style (16th Edition):

Vevea, Jason D. “Cellular Response to Membrane Phospholipid Imbalance, in Yeast and in Human Disease.” 2015. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:195460 %%% doi:10.7916/D8SQ8Z75.

MLA Handbook (7th Edition):

Vevea, Jason D. “Cellular Response to Membrane Phospholipid Imbalance, in Yeast and in Human Disease.” 2015. Web. 19 Nov 2017.

Vancouver:

Vevea JD. Cellular Response to Membrane Phospholipid Imbalance, in Yeast and in Human Disease. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:195460 %%% doi:10.7916/D8SQ8Z75.

Council of Science Editors:

Vevea JD. Cellular Response to Membrane Phospholipid Imbalance, in Yeast and in Human Disease. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:195460 %%% doi:10.7916/D8SQ8Z75


Columbia University

14. Uh, Minji. Notch Signaling Determines Lymphatic Cell Fate and Regulates Sprouting Lymphangiogenesis.

Degree: PhD, Pharmacology and Molecular SignalingPathology and Cell BiologyDigestive and Liver Diseases, 2013, Columbia University

 The lymphatic vascular system is necessary for physiological regulation of tissue fluid homeostasis and absorption of dietary fat. Lymphatics also function in the inflammatory response… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Uh, M. (2013). Notch Signaling Determines Lymphatic Cell Fate and Regulates Sprouting Lymphangiogenesis. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:167031 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Uh, Minji. “Notch Signaling Determines Lymphatic Cell Fate and Regulates Sprouting Lymphangiogenesis.” 2013. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:167031 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Uh, Minji. “Notch Signaling Determines Lymphatic Cell Fate and Regulates Sprouting Lymphangiogenesis.” 2013. Web. 19 Nov 2017.

Vancouver:

Uh M. Notch Signaling Determines Lymphatic Cell Fate and Regulates Sprouting Lymphangiogenesis. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:167031 %%% handle:10022/AC.

Council of Science Editors:

Uh M. Notch Signaling Determines Lymphatic Cell Fate and Regulates Sprouting Lymphangiogenesis. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://academiccommons.columbia.edu/catalog/ac:167031 %%% handle:10022/AC


Columbia University

15. Mathur, Deepti. PTEN regulates glutamine flux to pyrimidine synthesis and sensitivity to dihydroorotate dehydrogenase inhibition.

Degree: PhD, Cellular, Molecular and Biomedical StudiesInstitute for Cancer Genetics, 2017, Columbia University

 The importance of metabolism in tumor initiation and progression is becoming increasingly clear. Metabolic changes induced by oncogenic drivers of cancer contribute to tumor growth… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Mathur, D. (2017). PTEN regulates glutamine flux to pyrimidine synthesis and sensitivity to dihydroorotate dehydrogenase inhibition. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:206873 %%% doi:10.7916/D86114ZT

Chicago Manual of Style (16th Edition):

Mathur, Deepti. “PTEN regulates glutamine flux to pyrimidine synthesis and sensitivity to dihydroorotate dehydrogenase inhibition.” 2017. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:206873 %%% doi:10.7916/D86114ZT.

MLA Handbook (7th Edition):

Mathur, Deepti. “PTEN regulates glutamine flux to pyrimidine synthesis and sensitivity to dihydroorotate dehydrogenase inhibition.” 2017. Web. 19 Nov 2017.

Vancouver:

Mathur D. PTEN regulates glutamine flux to pyrimidine synthesis and sensitivity to dihydroorotate dehydrogenase inhibition. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:206873 %%% doi:10.7916/D86114ZT.

Council of Science Editors:

Mathur D. PTEN regulates glutamine flux to pyrimidine synthesis and sensitivity to dihydroorotate dehydrogenase inhibition. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://academiccommons.columbia.edu/catalog/ac:206873 %%% doi:10.7916/D86114ZT


Columbia University

16. Viswanathan, Vasanthi. Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling.

Degree: PhD, Biological Sciences, 2015, Columbia University

 Ferroptosis is a novel non-apoptotic, oxidative form of regulated cell death that can be triggered by diverse small-molecule ferroptosis inducers (FINs) and genetic perturbations. Current… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Viswanathan, V. (2015). Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:207014 %%% doi:10.7916/D8H9940V

Chicago Manual of Style (16th Edition):

Viswanathan, Vasanthi. “Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling.” 2015. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:207014 %%% doi:10.7916/D8H9940V.

MLA Handbook (7th Edition):

Viswanathan, Vasanthi. “Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling.” 2015. Web. 19 Nov 2017.

Vancouver:

Viswanathan V. Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:207014 %%% doi:10.7916/D8H9940V.

Council of Science Editors:

Viswanathan V. Cellular features predicting susceptibility to ferroptosis: insights from cancer cell-line profiling. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:207014 %%% doi:10.7916/D8H9940V


Columbia University

17. Zhang, Shuobo. Transcription factor activating protein 4 is synthetic lethal and a master regulator of MYCN amplified neuroblastoma.

Degree: PhD, Pathobiology and Molecular MedicinePediatrics, 2015, Columbia University

 Despite the identification of MYCN amplification as an adverse prognostic marker in neuroblastoma, no drugs that target MYCN have yet been developed. Here, by combining… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Zhang, S. (2015). Transcription factor activating protein 4 is synthetic lethal and a master regulator of MYCN amplified neuroblastoma. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:207931 %%% doi:10.7916/D8VX0FPF

Chicago Manual of Style (16th Edition):

Zhang, Shuobo. “Transcription factor activating protein 4 is synthetic lethal and a master regulator of MYCN amplified neuroblastoma.” 2015. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:207931 %%% doi:10.7916/D8VX0FPF.

MLA Handbook (7th Edition):

Zhang, Shuobo. “Transcription factor activating protein 4 is synthetic lethal and a master regulator of MYCN amplified neuroblastoma.” 2015. Web. 19 Nov 2017.

Vancouver:

Zhang S. Transcription factor activating protein 4 is synthetic lethal and a master regulator of MYCN amplified neuroblastoma. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:207931 %%% doi:10.7916/D8VX0FPF.

Council of Science Editors:

Zhang S. Transcription factor activating protein 4 is synthetic lethal and a master regulator of MYCN amplified neuroblastoma. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:207931 %%% doi:10.7916/D8VX0FPF


Columbia University

18. Tattersall, Ian William. Notch signaling regulates myeloid cell function and contribution to angiogenesis.

Degree: PhD, Cellular, Molecular and Biomedical StudiesObstetrics and Gynecology, 2015, Columbia University

 We investigated the role of Notch signaling in the vascular microenvironment, with particular attention paid to the vascular consequences of Notch signaling disruption in myeloid… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Tattersall, I. W. (2015). Notch signaling regulates myeloid cell function and contribution to angiogenesis. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:188049 %%% doi:10.7916/D8PV6JHG

Chicago Manual of Style (16th Edition):

Tattersall, Ian William. “Notch signaling regulates myeloid cell function and contribution to angiogenesis.” 2015. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:188049 %%% doi:10.7916/D8PV6JHG.

MLA Handbook (7th Edition):

Tattersall, Ian William. “Notch signaling regulates myeloid cell function and contribution to angiogenesis.” 2015. Web. 19 Nov 2017.

Vancouver:

Tattersall IW. Notch signaling regulates myeloid cell function and contribution to angiogenesis. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:188049 %%% doi:10.7916/D8PV6JHG.

Council of Science Editors:

Tattersall IW. Notch signaling regulates myeloid cell function and contribution to angiogenesis. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:188049 %%% doi:10.7916/D8PV6JHG


Columbia University

19. Torres, Ciara. Macroautophagy Modulates Synaptic Function in the Striatum.

Degree: PhD, Cellular, Molecular and Biomedical StudiesNeurology, 2014, Columbia University

 The kinase mechanistic target of rapamycin (mTOR) is a regulator of cell growth and survival, protein synthesis-dependent synaptic plasticity, and macroautophagic degradation of cellular components.… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Torres, C. (2014). Macroautophagy Modulates Synaptic Function in the Striatum. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:207601 %%% doi:10.7916/D85T3HJC

Chicago Manual of Style (16th Edition):

Torres, Ciara. “Macroautophagy Modulates Synaptic Function in the Striatum.” 2014. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:207601 %%% doi:10.7916/D85T3HJC.

MLA Handbook (7th Edition):

Torres, Ciara. “Macroautophagy Modulates Synaptic Function in the Striatum.” 2014. Web. 19 Nov 2017.

Vancouver:

Torres C. Macroautophagy Modulates Synaptic Function in the Striatum. [Internet] [Doctoral dissertation]. Columbia University; 2014. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:207601 %%% doi:10.7916/D85T3HJC.

Council of Science Editors:

Torres C. Macroautophagy Modulates Synaptic Function in the Striatum. [Doctoral Dissertation]. Columbia University; 2014. Available from: https://academiccommons.columbia.edu/catalog/ac:207601 %%% doi:10.7916/D85T3HJC


Columbia University

20. Wang, Shang-Jui. Dissecting the non-canonical functions of p53 through novel target identification and p53 acetylation.

Degree: PhD, Cellular, Molecular and Biomedical StudiesPathology and Cell Biology, 2014, Columbia University

 It is well established that the p53 tumor suppressor plays a crucial role in controlling cell proliferation and apoptosis upon various types of stress. There… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Wang, S. (2014). Dissecting the non-canonical functions of p53 through novel target identification and p53 acetylation. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:176032 %%% doi:10.7916/D85B00M3

Chicago Manual of Style (16th Edition):

Wang, Shang-Jui. “Dissecting the non-canonical functions of p53 through novel target identification and p53 acetylation.” 2014. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:176032 %%% doi:10.7916/D85B00M3.

MLA Handbook (7th Edition):

Wang, Shang-Jui. “Dissecting the non-canonical functions of p53 through novel target identification and p53 acetylation.” 2014. Web. 19 Nov 2017.

Vancouver:

Wang S. Dissecting the non-canonical functions of p53 through novel target identification and p53 acetylation. [Internet] [Doctoral dissertation]. Columbia University; 2014. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:176032 %%% doi:10.7916/D85B00M3.

Council of Science Editors:

Wang S. Dissecting the non-canonical functions of p53 through novel target identification and p53 acetylation. [Doctoral Dissertation]. Columbia University; 2014. Available from: https://academiccommons.columbia.edu/catalog/ac:176032 %%% doi:10.7916/D85B00M3


Columbia University

21. McFaline Figueroa, Jose Ricardo. Mitochondrial Inheritance and Function in the Lifespan Control of Budding Yeast.

Degree: PhD, MedicinePathology and Cell Biology, 2012, Columbia University

 Mitochondria are essential organelles that cannot be synthesized de novo and must be inherited by daughter cells. During cell division, mitochondria align along the mother-… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

McFaline Figueroa, J. R. (2012). Mitochondrial Inheritance and Function in the Lifespan Control of Budding Yeast. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:153280 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

McFaline Figueroa, Jose Ricardo. “Mitochondrial Inheritance and Function in the Lifespan Control of Budding Yeast.” 2012. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:153280 %%% handle:10022/AC.

MLA Handbook (7th Edition):

McFaline Figueroa, Jose Ricardo. “Mitochondrial Inheritance and Function in the Lifespan Control of Budding Yeast.” 2012. Web. 19 Nov 2017.

Vancouver:

McFaline Figueroa JR. Mitochondrial Inheritance and Function in the Lifespan Control of Budding Yeast. [Internet] [Doctoral dissertation]. Columbia University; 2012. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:153280 %%% handle:10022/AC.

Council of Science Editors:

McFaline Figueroa JR. Mitochondrial Inheritance and Function in the Lifespan Control of Budding Yeast. [Doctoral Dissertation]. Columbia University; 2012. Available from: https://academiccommons.columbia.edu/catalog/ac:153280 %%% handle:10022/AC


Columbia University

22. Lutz, Regina Anna. Regulation of Polarity by Microtubules.

Degree: PhD, Cellular, Molecular and Biomedical Studies, 2015, Columbia University

 Cell polarity is essential for cellular functions, growth, development, and formation of multicellular organisms. Cell polarization is often regulated during the cell division cycle. For… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Lutz, R. A. (2015). Regulation of Polarity by Microtubules. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:206019 %%% doi:10.7916/D85Q4TT1

Chicago Manual of Style (16th Edition):

Lutz, Regina Anna. “Regulation of Polarity by Microtubules.” 2015. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:206019 %%% doi:10.7916/D85Q4TT1.

MLA Handbook (7th Edition):

Lutz, Regina Anna. “Regulation of Polarity by Microtubules.” 2015. Web. 19 Nov 2017.

Vancouver:

Lutz RA. Regulation of Polarity by Microtubules. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:206019 %%% doi:10.7916/D85Q4TT1.

Council of Science Editors:

Lutz RA. Regulation of Polarity by Microtubules. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:206019 %%% doi:10.7916/D85Q4TT1


Columbia University

23. Palmer, Colin James. The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation.

Degree: PhD, Cellular, Molecular, Structural, and Genetic StudiesMicrobiology, 2013, Columbia University

 The Hedgehog (Hh) signaling pathway regulates normal development and cell proliferation across the metazoa. Upon its aberrant activation, mammalian Hh signaling can also cause tumor… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Palmer, C. J. (2013). The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:165186 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Palmer, Colin James. “The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation.” 2013. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:165186 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Palmer, Colin James. “The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation.” 2013. Web. 19 Nov 2017.

Vancouver:

Palmer CJ. The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:165186 %%% handle:10022/AC.

Council of Science Editors:

Palmer CJ. The transcription factor Zfx is required for tumorigenesis caused by Hedgehog pathway activation. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://academiccommons.columbia.edu/catalog/ac:165186 %%% handle:10022/AC


Columbia University

24. Hu, Daniel Jun-Kit. Roles for Cytoplasmic Dynein and the Unconventional Kinesin, KIF1a, during Cortical Development.

Degree: PhD, Biological SciencesPathology and Cell Biology, 2015, Columbia University

 Radial glial progenitor (RGP) cells are neural stem cells that give rise to the majority of neurons, glia, and adult stem cells during cortical development.… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Hu, D. J. (2015). Roles for Cytoplasmic Dynein and the Unconventional Kinesin, KIF1a, during Cortical Development. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:187905 %%% doi:10.7916/D8Z89BG9

Chicago Manual of Style (16th Edition):

Hu, Daniel Jun-Kit. “Roles for Cytoplasmic Dynein and the Unconventional Kinesin, KIF1a, during Cortical Development.” 2015. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:187905 %%% doi:10.7916/D8Z89BG9.

MLA Handbook (7th Edition):

Hu, Daniel Jun-Kit. “Roles for Cytoplasmic Dynein and the Unconventional Kinesin, KIF1a, during Cortical Development.” 2015. Web. 19 Nov 2017.

Vancouver:

Hu DJ. Roles for Cytoplasmic Dynein and the Unconventional Kinesin, KIF1a, during Cortical Development. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:187905 %%% doi:10.7916/D8Z89BG9.

Council of Science Editors:

Hu DJ. Roles for Cytoplasmic Dynein and the Unconventional Kinesin, KIF1a, during Cortical Development. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:187905 %%% doi:10.7916/D8Z89BG9


Columbia University

25. Tambini, Marc D. Apolipoprotein E and Mitochondria-associated Endoplasmic Reticulum Membrane Dysfunction.

Degree: PhD, Cellular, Molecular and Biomedical StudiesNeurology, 2015, Columbia University

 Despite the tremendous advances of the last century, the cause of Alzheimer disease (AD) remains unclear. Genetic analysis of families with Alzheimer disease has revealed… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Tambini, M. D. (2015). Apolipoprotein E and Mitochondria-associated Endoplasmic Reticulum Membrane Dysfunction. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:192644 %%% doi:10.7916/D84M9484

Chicago Manual of Style (16th Edition):

Tambini, Marc D. “Apolipoprotein E and Mitochondria-associated Endoplasmic Reticulum Membrane Dysfunction.” 2015. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:192644 %%% doi:10.7916/D84M9484.

MLA Handbook (7th Edition):

Tambini, Marc D. “Apolipoprotein E and Mitochondria-associated Endoplasmic Reticulum Membrane Dysfunction.” 2015. Web. 19 Nov 2017.

Vancouver:

Tambini MD. Apolipoprotein E and Mitochondria-associated Endoplasmic Reticulum Membrane Dysfunction. [Internet] [Doctoral dissertation]. Columbia University; 2015. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:192644 %%% doi:10.7916/D84M9484.

Council of Science Editors:

Tambini MD. Apolipoprotein E and Mitochondria-associated Endoplasmic Reticulum Membrane Dysfunction. [Doctoral Dissertation]. Columbia University; 2015. Available from: https://academiccommons.columbia.edu/catalog/ac:192644 %%% doi:10.7916/D84M9484


Columbia University

26. Hahn, Philip L. A. Mitochondrial Diseases, Treatments, and FDA Orphan Legislation.

Degree: MA, Biological SciencesBiotechnology, 2012, Columbia University

 Mitochondria originated during a key endosymbiotic event, when an enveloped bacterium or invasive parasite adjusted to its intracellular surroundings and formed the first eukaryotic cell.… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Hahn, P. L. A. (2012). Mitochondrial Diseases, Treatments, and FDA Orphan Legislation. (Masters Thesis). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:153604 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Hahn, Philip L A. “Mitochondrial Diseases, Treatments, and FDA Orphan Legislation.” 2012. Masters Thesis, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:153604 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Hahn, Philip L A. “Mitochondrial Diseases, Treatments, and FDA Orphan Legislation.” 2012. Web. 19 Nov 2017.

Vancouver:

Hahn PLA. Mitochondrial Diseases, Treatments, and FDA Orphan Legislation. [Internet] [Masters thesis]. Columbia University; 2012. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:153604 %%% handle:10022/AC.

Council of Science Editors:

Hahn PLA. Mitochondrial Diseases, Treatments, and FDA Orphan Legislation. [Masters Thesis]. Columbia University; 2012. Available from: https://academiccommons.columbia.edu/catalog/ac:153604 %%% handle:10022/AC


Columbia University

27. Pan, Kally Zhang. Cell Size Control in Fission Yeast.

Degree: PhD, Genetics and DevelopmentMicrobiology, 2013, Columbia University

 Among all living organisms, there is almost much variety in cell size as there is for cell function and cell type. However, within each cell… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Pan, K. Z. (2013). Cell Size Control in Fission Yeast. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:163318 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Pan, Kally Zhang. “Cell Size Control in Fission Yeast.” 2013. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:163318 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Pan, Kally Zhang. “Cell Size Control in Fission Yeast.” 2013. Web. 19 Nov 2017.

Vancouver:

Pan KZ. Cell Size Control in Fission Yeast. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:163318 %%% handle:10022/AC.

Council of Science Editors:

Pan KZ. Cell Size Control in Fission Yeast. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://academiccommons.columbia.edu/catalog/ac:163318 %%% handle:10022/AC


Columbia University

28. Crouch, Elizabeth. Adult Neural Stem Cells and Their Perivascular Niche.

Degree: PhD, Neurobiology and BehaviorNeuroscience, 2013, Columbia University

 Stem cells reside in specialized niches that support their selfrenewal and differentiation. A balance between intrinsic and extrinsic signals mediates stem cell quiescence, activation and… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Crouch, E. (2013). Adult Neural Stem Cells and Their Perivascular Niche. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:188858 %%% doi:10.7916/D8T1531D

Chicago Manual of Style (16th Edition):

Crouch, Elizabeth. “Adult Neural Stem Cells and Their Perivascular Niche.” 2013. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:188858 %%% doi:10.7916/D8T1531D.

MLA Handbook (7th Edition):

Crouch, Elizabeth. “Adult Neural Stem Cells and Their Perivascular Niche.” 2013. Web. 19 Nov 2017.

Vancouver:

Crouch E. Adult Neural Stem Cells and Their Perivascular Niche. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:188858 %%% doi:10.7916/D8T1531D.

Council of Science Editors:

Crouch E. Adult Neural Stem Cells and Their Perivascular Niche. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://academiccommons.columbia.edu/catalog/ac:188858 %%% doi:10.7916/D8T1531D


Columbia University

29. Devereaux, Kelly Anne. The role of phosphatidylinositol 3-kinases in autophagy regulation.

Degree: PhD, Pathobiology and Molecular MedicinePathology and Cell Biology, 2014, Columbia University

 Autophagy requires the biogenesis of autophagosomes (APs), which are large multilamellar vesicles that sequester cytoplasmic substrates and undergo a maturation process that ultimately leads to… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Devereaux, K. A. (2014). The role of phosphatidylinositol 3-kinases in autophagy regulation. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:200589 %%% doi:10.7916/D8QZ2842

Chicago Manual of Style (16th Edition):

Devereaux, Kelly Anne. “The role of phosphatidylinositol 3-kinases in autophagy regulation.” 2014. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:200589 %%% doi:10.7916/D8QZ2842.

MLA Handbook (7th Edition):

Devereaux, Kelly Anne. “The role of phosphatidylinositol 3-kinases in autophagy regulation.” 2014. Web. 19 Nov 2017.

Vancouver:

Devereaux KA. The role of phosphatidylinositol 3-kinases in autophagy regulation. [Internet] [Doctoral dissertation]. Columbia University; 2014. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:200589 %%% doi:10.7916/D8QZ2842.

Council of Science Editors:

Devereaux KA. The role of phosphatidylinositol 3-kinases in autophagy regulation. [Doctoral Dissertation]. Columbia University; 2014. Available from: https://academiccommons.columbia.edu/catalog/ac:200589 %%% doi:10.7916/D8QZ2842


Columbia University

30. Guo, Yige. Molecular Mechanisms of Mitotic Spindle Assembly and Accurate Chromosome Segregation.

Degree: PhD, Pathobiology and Molecular MedicinePathology, 2013, Columbia University

 During the cell cycle, duplicated DNA in S phase is segregated, in the form of chromatids, into two daughter cells in mitosis. The accuracy of… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Guo, Y. (2013). Molecular Mechanisms of Mitotic Spindle Assembly and Accurate Chromosome Segregation. (Doctoral Dissertation). Columbia University. Retrieved from https://academiccommons.columbia.edu/catalog/ac:165153 %%% handle:10022/AC

Chicago Manual of Style (16th Edition):

Guo, Yige. “Molecular Mechanisms of Mitotic Spindle Assembly and Accurate Chromosome Segregation.” 2013. Doctoral Dissertation, Columbia University. Accessed November 19, 2017. https://academiccommons.columbia.edu/catalog/ac:165153 %%% handle:10022/AC.

MLA Handbook (7th Edition):

Guo, Yige. “Molecular Mechanisms of Mitotic Spindle Assembly and Accurate Chromosome Segregation.” 2013. Web. 19 Nov 2017.

Vancouver:

Guo Y. Molecular Mechanisms of Mitotic Spindle Assembly and Accurate Chromosome Segregation. [Internet] [Doctoral dissertation]. Columbia University; 2013. [cited 2017 Nov 19]. Available from: https://academiccommons.columbia.edu/catalog/ac:165153 %%% handle:10022/AC.

Council of Science Editors:

Guo Y. Molecular Mechanisms of Mitotic Spindle Assembly and Accurate Chromosome Segregation. [Doctoral Dissertation]. Columbia University; 2013. Available from: https://academiccommons.columbia.edu/catalog/ac:165153 %%% handle:10022/AC

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