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Dept: Biomedical Sciences

You searched for subject:(Cellular Biology). Showing records 1 – 30 of 130 total matches.

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University of California – San Diego

1. Chou, Annie Chiazong. Characterization of an internal ribosomal entry site in the coding sequence of tyrosyl-DNA phosphodiesterase 2.

Degree: Biomedical Sciences, 2018, University of California – San Diego

 Tyrosyl DNA-phosphodiesterase 2 (TDP2) is a multifunctional protein that has been implicated in a myriad of cellular pathways. While most well-known for its phosphodiesterase activity… (more)

Subjects/Keywords: Cellular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chou, A. C. (2018). Characterization of an internal ribosomal entry site in the coding sequence of tyrosyl-DNA phosphodiesterase 2. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/5n965003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chou, Annie Chiazong. “Characterization of an internal ribosomal entry site in the coding sequence of tyrosyl-DNA phosphodiesterase 2.” 2018. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/5n965003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chou, Annie Chiazong. “Characterization of an internal ribosomal entry site in the coding sequence of tyrosyl-DNA phosphodiesterase 2.” 2018. Web. 20 Aug 2019.

Vancouver:

Chou AC. Characterization of an internal ribosomal entry site in the coding sequence of tyrosyl-DNA phosphodiesterase 2. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/5n965003.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chou AC. Characterization of an internal ribosomal entry site in the coding sequence of tyrosyl-DNA phosphodiesterase 2. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/5n965003

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

2. Farber-Katz, Suzette Elena. DNA Damage Triggers Golgi Dispersal via GOLPH3 and DNA-PK.

Degree: Biomedical Sciences, 2012, University of California – San Diego

 Golgi morphology and function has fascinated scientists for decades; however, it was unclear how the two were related. Our research delving into the function of… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Farber-Katz, S. E. (2012). DNA Damage Triggers Golgi Dispersal via GOLPH3 and DNA-PK. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/0p0476db

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farber-Katz, Suzette Elena. “DNA Damage Triggers Golgi Dispersal via GOLPH3 and DNA-PK.” 2012. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/0p0476db.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farber-Katz, Suzette Elena. “DNA Damage Triggers Golgi Dispersal via GOLPH3 and DNA-PK.” 2012. Web. 20 Aug 2019.

Vancouver:

Farber-Katz SE. DNA Damage Triggers Golgi Dispersal via GOLPH3 and DNA-PK. [Internet] [Thesis]. University of California – San Diego; 2012. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/0p0476db.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farber-Katz SE. DNA Damage Triggers Golgi Dispersal via GOLPH3 and DNA-PK. [Thesis]. University of California – San Diego; 2012. Available from: http://www.escholarship.org/uc/item/0p0476db

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

3. Lyons, Jesse Stolberg. The Melanocortin 1 Receptor and cAMP Signaling Regulate Mitotic Entry in Melanoma Through Inhibition of cdc25B.

Degree: Biomedical Sciences, 2011, University of California – San Francisco

 Melanoma is an aggressive and treatment resistant tumor of the melanocyte lineage. In humans, melanocytes are responsible for skin pigmentation and the tanning response. Pigmentation… (more)

Subjects/Keywords: Cellular Biology

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APA (6th Edition):

Lyons, J. S. (2011). The Melanocortin 1 Receptor and cAMP Signaling Regulate Mitotic Entry in Melanoma Through Inhibition of cdc25B. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/4v83t5gs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lyons, Jesse Stolberg. “The Melanocortin 1 Receptor and cAMP Signaling Regulate Mitotic Entry in Melanoma Through Inhibition of cdc25B.” 2011. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/4v83t5gs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lyons, Jesse Stolberg. “The Melanocortin 1 Receptor and cAMP Signaling Regulate Mitotic Entry in Melanoma Through Inhibition of cdc25B.” 2011. Web. 20 Aug 2019.

Vancouver:

Lyons JS. The Melanocortin 1 Receptor and cAMP Signaling Regulate Mitotic Entry in Melanoma Through Inhibition of cdc25B. [Internet] [Thesis]. University of California – San Francisco; 2011. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/4v83t5gs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lyons JS. The Melanocortin 1 Receptor and cAMP Signaling Regulate Mitotic Entry in Melanoma Through Inhibition of cdc25B. [Thesis]. University of California – San Francisco; 2011. Available from: http://www.escholarship.org/uc/item/4v83t5gs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

4. Lock, Rebecca. Novel functions for autophagy during Ras transformation.

Degree: Biomedical Sciences, 2012, University of California – San Francisco

 Autophagy is a highly regulated catabolic process that degrades cytosolic organelles and macromolecules. Deregulation of the autophagic process has been linked to the development of… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Lock, R. (2012). Novel functions for autophagy during Ras transformation. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/6wk914ww

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lock, Rebecca. “Novel functions for autophagy during Ras transformation.” 2012. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/6wk914ww.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lock, Rebecca. “Novel functions for autophagy during Ras transformation.” 2012. Web. 20 Aug 2019.

Vancouver:

Lock R. Novel functions for autophagy during Ras transformation. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/6wk914ww.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lock R. Novel functions for autophagy during Ras transformation. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/6wk914ww

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

5. Saeteurn, Koy Yoon. Dissecting the Roles of TGF-b Receptors in TGF-b-Induced PI3K-Akt Activation.

Degree: Biomedical Sciences, 2016, University of California – San Francisco

 TGF-β plays many roles in growth and development, and aberrant TGF-β signaling contributes to the pathogenesis and progression of various diseases, including fibrosis and the… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Saeteurn, K. Y. (2016). Dissecting the Roles of TGF-b Receptors in TGF-b-Induced PI3K-Akt Activation. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/5zt3w31c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saeteurn, Koy Yoon. “Dissecting the Roles of TGF-b Receptors in TGF-b-Induced PI3K-Akt Activation.” 2016. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/5zt3w31c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saeteurn, Koy Yoon. “Dissecting the Roles of TGF-b Receptors in TGF-b-Induced PI3K-Akt Activation.” 2016. Web. 20 Aug 2019.

Vancouver:

Saeteurn KY. Dissecting the Roles of TGF-b Receptors in TGF-b-Induced PI3K-Akt Activation. [Internet] [Thesis]. University of California – San Francisco; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/5zt3w31c.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saeteurn KY. Dissecting the Roles of TGF-b Receptors in TGF-b-Induced PI3K-Akt Activation. [Thesis]. University of California – San Francisco; 2016. Available from: http://www.escholarship.org/uc/item/5zt3w31c

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

6. Lee, Albert Chuh-kai. Otubain 1 in T cell homeostasis and programmed cell death.

Degree: Biomedical Sciences, 2013, University of California – San Francisco

 Ubiquitination is a process used by cells to tightly control signaling pathways by covalently attaching a ubiquitin molecule to a target substrate. This process is… (more)

Subjects/Keywords: Immunology; Cellular biology

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APA (6th Edition):

Lee, A. C. (2013). Otubain 1 in T cell homeostasis and programmed cell death. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/259066bj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Albert Chuh-kai. “Otubain 1 in T cell homeostasis and programmed cell death.” 2013. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/259066bj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Albert Chuh-kai. “Otubain 1 in T cell homeostasis and programmed cell death.” 2013. Web. 20 Aug 2019.

Vancouver:

Lee AC. Otubain 1 in T cell homeostasis and programmed cell death. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/259066bj.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee AC. Otubain 1 in T cell homeostasis and programmed cell death. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/259066bj

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

7. Kramer, Michael Harris. Transformation of High-Throughput Data into Hierarchical Cellular Models Enables Biological Prediction and Discovery.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 A holy grail of bioinformatics is the creation of whole-cell models with the ability to enhance human understanding and facilitate discovery. To this end, a… (more)

Subjects/Keywords: Bioinformatics; Cellular biology; Molecular biology

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APA (6th Edition):

Kramer, M. H. (2016). Transformation of High-Throughput Data into Hierarchical Cellular Models Enables Biological Prediction and Discovery. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/6nm9r56v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kramer, Michael Harris. “Transformation of High-Throughput Data into Hierarchical Cellular Models Enables Biological Prediction and Discovery.” 2016. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/6nm9r56v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kramer, Michael Harris. “Transformation of High-Throughput Data into Hierarchical Cellular Models Enables Biological Prediction and Discovery.” 2016. Web. 20 Aug 2019.

Vancouver:

Kramer MH. Transformation of High-Throughput Data into Hierarchical Cellular Models Enables Biological Prediction and Discovery. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/6nm9r56v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kramer MH. Transformation of High-Throughput Data into Hierarchical Cellular Models Enables Biological Prediction and Discovery. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/6nm9r56v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

8. Liggins, Marc Christopher. Elucidating the Role of Phosphoinositides in Melanogenesis: PIKfyve Regulates Melanogenic Processes through the Synthesis of PI(3,5)P2 and PI(5)P.

Degree: Biomedical Sciences, 2016, University of California – Irvine

 Epidermal melanocytes synthesize melanin within specialized lysosome-related organelles known as melanosomes. These melanosomes are transferred to neighboring keratinocytes where they surround nuclei to protect against… (more)

Subjects/Keywords: Biology; Cellular biology; Biochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liggins, M. C. (2016). Elucidating the Role of Phosphoinositides in Melanogenesis: PIKfyve Regulates Melanogenic Processes through the Synthesis of PI(3,5)P2 and PI(5)P. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/9q19w9h3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liggins, Marc Christopher. “Elucidating the Role of Phosphoinositides in Melanogenesis: PIKfyve Regulates Melanogenic Processes through the Synthesis of PI(3,5)P2 and PI(5)P.” 2016. Thesis, University of California – Irvine. Accessed August 20, 2019. http://www.escholarship.org/uc/item/9q19w9h3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liggins, Marc Christopher. “Elucidating the Role of Phosphoinositides in Melanogenesis: PIKfyve Regulates Melanogenic Processes through the Synthesis of PI(3,5)P2 and PI(5)P.” 2016. Web. 20 Aug 2019.

Vancouver:

Liggins MC. Elucidating the Role of Phosphoinositides in Melanogenesis: PIKfyve Regulates Melanogenic Processes through the Synthesis of PI(3,5)P2 and PI(5)P. [Internet] [Thesis]. University of California – Irvine; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/9q19w9h3.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liggins MC. Elucidating the Role of Phosphoinositides in Melanogenesis: PIKfyve Regulates Melanogenic Processes through the Synthesis of PI(3,5)P2 and PI(5)P. [Thesis]. University of California – Irvine; 2016. Available from: http://www.escholarship.org/uc/item/9q19w9h3

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

9. Ward, Jacqueline Marie. Spinocerebellar Ataxia Type 7 is Characterized by Defects in Mitochondrial and Metabolic Function.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 Spinocerebellar ataxia type 7 (SCA7) is an inherited neurodegenerative disease caused by a CAG/polyglutamine (polyQ) repeat expansion in the ataxin-7 gene. Adult patient clinical features… (more)

Subjects/Keywords: Molecular biology; Neurosciences; Cellular biology

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APA (6th Edition):

Ward, J. M. (2016). Spinocerebellar Ataxia Type 7 is Characterized by Defects in Mitochondrial and Metabolic Function. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/02b7m809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ward, Jacqueline Marie. “Spinocerebellar Ataxia Type 7 is Characterized by Defects in Mitochondrial and Metabolic Function.” 2016. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/02b7m809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ward, Jacqueline Marie. “Spinocerebellar Ataxia Type 7 is Characterized by Defects in Mitochondrial and Metabolic Function.” 2016. Web. 20 Aug 2019.

Vancouver:

Ward JM. Spinocerebellar Ataxia Type 7 is Characterized by Defects in Mitochondrial and Metabolic Function. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/02b7m809.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ward JM. Spinocerebellar Ataxia Type 7 is Characterized by Defects in Mitochondrial and Metabolic Function. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/02b7m809

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

10. Guan, Zhiyun. Epigenetic Regulation of White-Opaque Switching in Candida albicans.

Degree: Biomedical Sciences, 2014, University of California – Irvine

 Candida albicans, a significant human fungal pathogen, switches between two phenotypic states, white and opaque. The white-opaque switch is a unique and tractable system for… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Genetics

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APA (6th Edition):

Guan, Z. (2014). Epigenetic Regulation of White-Opaque Switching in Candida albicans. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/8qv7w8zv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guan, Zhiyun. “Epigenetic Regulation of White-Opaque Switching in Candida albicans.” 2014. Thesis, University of California – Irvine. Accessed August 20, 2019. http://www.escholarship.org/uc/item/8qv7w8zv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guan, Zhiyun. “Epigenetic Regulation of White-Opaque Switching in Candida albicans.” 2014. Web. 20 Aug 2019.

Vancouver:

Guan Z. Epigenetic Regulation of White-Opaque Switching in Candida albicans. [Internet] [Thesis]. University of California – Irvine; 2014. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/8qv7w8zv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guan Z. Epigenetic Regulation of White-Opaque Switching in Candida albicans. [Thesis]. University of California – Irvine; 2014. Available from: http://www.escholarship.org/uc/item/8qv7w8zv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

11. Moon, Hyang Mi. Molecular Genetics of Lissencephaly and Functions of LIS1 in Cell Division.

Degree: Biomedical Sciences, 2012, University of California – San Francisco

 Lissencephaly (smooth brain) is a brain malformation disorder resulted from defective neuronal migration during early development. Deletion or mutation of many genes involved in the… (more)

Subjects/Keywords: Developmental biology; Cellular biology; Genetics

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APA (6th Edition):

Moon, H. M. (2012). Molecular Genetics of Lissencephaly and Functions of LIS1 in Cell Division. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/2x1698sv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Moon, Hyang Mi. “Molecular Genetics of Lissencephaly and Functions of LIS1 in Cell Division.” 2012. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/2x1698sv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Moon, Hyang Mi. “Molecular Genetics of Lissencephaly and Functions of LIS1 in Cell Division.” 2012. Web. 20 Aug 2019.

Vancouver:

Moon HM. Molecular Genetics of Lissencephaly and Functions of LIS1 in Cell Division. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/2x1698sv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Moon HM. Molecular Genetics of Lissencephaly and Functions of LIS1 in Cell Division. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/2x1698sv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

12. Pitt, Cameron L.W. Targeting Ras in Cancer: Mutation-Specific Dependencies in Oncogenic Signaling.

Degree: Biomedical Sciences, 2015, University of California – San Francisco

 Ras mutations drive approximately one third of human cancers by aberrant regulation of the mitogen-activated protein kinase (MAPK) signaling cascade and other effector pathways. While… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Biochemistry

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APA (6th Edition):

Pitt, C. L. W. (2015). Targeting Ras in Cancer: Mutation-Specific Dependencies in Oncogenic Signaling. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/5wd3q3kr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pitt, Cameron L W. “Targeting Ras in Cancer: Mutation-Specific Dependencies in Oncogenic Signaling.” 2015. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/5wd3q3kr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pitt, Cameron L W. “Targeting Ras in Cancer: Mutation-Specific Dependencies in Oncogenic Signaling.” 2015. Web. 20 Aug 2019.

Vancouver:

Pitt CLW. Targeting Ras in Cancer: Mutation-Specific Dependencies in Oncogenic Signaling. [Internet] [Thesis]. University of California – San Francisco; 2015. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/5wd3q3kr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pitt CLW. Targeting Ras in Cancer: Mutation-Specific Dependencies in Oncogenic Signaling. [Thesis]. University of California – San Francisco; 2015. Available from: http://www.escholarship.org/uc/item/5wd3q3kr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

13. Qi, Jenny. Targeting the Unfolded Protein Response in Pancreatic Neuroendocrine Tumors.

Degree: Biomedical Sciences, 2017, University of California – San Francisco

 A critical regulator of the unfolded protein response (UPR), the IRE1α kinase/endoribonuclease promotes adaptation or apoptosis depending on the level of upstream endoplasmic reticulum (ER)… (more)

Subjects/Keywords: Cellular biology; Oncology; Molecular biology

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APA (6th Edition):

Qi, J. (2017). Targeting the Unfolded Protein Response in Pancreatic Neuroendocrine Tumors. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/7v38v30g

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Qi, Jenny. “Targeting the Unfolded Protein Response in Pancreatic Neuroendocrine Tumors.” 2017. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/7v38v30g.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Qi, Jenny. “Targeting the Unfolded Protein Response in Pancreatic Neuroendocrine Tumors.” 2017. Web. 20 Aug 2019.

Vancouver:

Qi J. Targeting the Unfolded Protein Response in Pancreatic Neuroendocrine Tumors. [Internet] [Thesis]. University of California – San Francisco; 2017. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/7v38v30g.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Qi J. Targeting the Unfolded Protein Response in Pancreatic Neuroendocrine Tumors. [Thesis]. University of California – San Francisco; 2017. Available from: http://www.escholarship.org/uc/item/7v38v30g

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

14. Camarda, Roman. Metabolic requirements necessitate microenvironmental crosstalk in breast cancer.

Degree: Biomedical Sciences, 2018, University of California – San Francisco

 Despite marked advancements in targeted therapeutics, breast cancer remainsthe most diagnosed cancer and second leading cause of cancer-related death inwomen in the United States. The… (more)

Subjects/Keywords: Oncology; Cellular biology; Biochemistry

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APA (6th Edition):

Camarda, R. (2018). Metabolic requirements necessitate microenvironmental crosstalk in breast cancer. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/6g24523m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Camarda, Roman. “Metabolic requirements necessitate microenvironmental crosstalk in breast cancer.” 2018. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/6g24523m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Camarda, Roman. “Metabolic requirements necessitate microenvironmental crosstalk in breast cancer.” 2018. Web. 20 Aug 2019.

Vancouver:

Camarda R. Metabolic requirements necessitate microenvironmental crosstalk in breast cancer. [Internet] [Thesis]. University of California – San Francisco; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/6g24523m.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Camarda R. Metabolic requirements necessitate microenvironmental crosstalk in breast cancer. [Thesis]. University of California – San Francisco; 2018. Available from: http://www.escholarship.org/uc/item/6g24523m

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

15. Richardson, Amelia Katherine. Defining the Cellular Consequences of Loss of the Cohesin Complex Subunit STAG2 in Cancer Cells.

Degree: Biomedical Sciences, 2018, University of California – San Diego

 Inactivating mutations in the cohesin subunit STAG2 are frequent in many cancers, but the consequences of these mutations have not been clearly defined. STAG2 loss… (more)

Subjects/Keywords: Cellular biology; Biology; Cancer; Cohesin; STAG2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Richardson, A. K. (2018). Defining the Cellular Consequences of Loss of the Cohesin Complex Subunit STAG2 in Cancer Cells. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/8pj470md

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richardson, Amelia Katherine. “Defining the Cellular Consequences of Loss of the Cohesin Complex Subunit STAG2 in Cancer Cells.” 2018. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/8pj470md.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richardson, Amelia Katherine. “Defining the Cellular Consequences of Loss of the Cohesin Complex Subunit STAG2 in Cancer Cells.” 2018. Web. 20 Aug 2019.

Vancouver:

Richardson AK. Defining the Cellular Consequences of Loss of the Cohesin Complex Subunit STAG2 in Cancer Cells. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/8pj470md.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richardson AK. Defining the Cellular Consequences of Loss of the Cohesin Complex Subunit STAG2 in Cancer Cells. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/8pj470md

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

16. Kostova, Kamena Kamenova. The role of the Ribosome Quality Control Complex (RQC) in Maintaining Protein Homeostasis in Yeast and Higher Eukaryotes.

Degree: Biomedical Sciences, 2018, University of California – San Francisco

 Protein biosynthesis is the most energy-consuming process during cellular proliferation and any event that interferes with protein production jeopardizes cell viability. It has been recently… (more)

Subjects/Keywords: Cellular biology; Molecular biology; ribosome; RQC; translation

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APA (6th Edition):

Kostova, K. K. (2018). The role of the Ribosome Quality Control Complex (RQC) in Maintaining Protein Homeostasis in Yeast and Higher Eukaryotes. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/6b38941p

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kostova, Kamena Kamenova. “The role of the Ribosome Quality Control Complex (RQC) in Maintaining Protein Homeostasis in Yeast and Higher Eukaryotes.” 2018. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/6b38941p.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kostova, Kamena Kamenova. “The role of the Ribosome Quality Control Complex (RQC) in Maintaining Protein Homeostasis in Yeast and Higher Eukaryotes.” 2018. Web. 20 Aug 2019.

Vancouver:

Kostova KK. The role of the Ribosome Quality Control Complex (RQC) in Maintaining Protein Homeostasis in Yeast and Higher Eukaryotes. [Internet] [Thesis]. University of California – San Francisco; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/6b38941p.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kostova KK. The role of the Ribosome Quality Control Complex (RQC) in Maintaining Protein Homeostasis in Yeast and Higher Eukaryotes. [Thesis]. University of California – San Francisco; 2018. Available from: http://www.escholarship.org/uc/item/6b38941p

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

17. Salt, Megan. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.

Degree: Biomedical Sciences, 2014, University of California – San Francisco

 Tumors showing evidence of epithelial to mesenchymal transition (EMT) have been associated with metastasis, drug resistance, and poor prognosis. Heterogeneity along the EMT spectrum is… (more)

Subjects/Keywords: Cellular biology; Biology; EMT; ERBB3; PI3K

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APA (6th Edition):

Salt, M. (2014). Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/52s838hv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Salt, Megan. “Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.” 2014. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/52s838hv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Salt, Megan. “Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival.” 2014. Web. 20 Aug 2019.

Vancouver:

Salt M. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. [Internet] [Thesis]. University of California – San Francisco; 2014. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/52s838hv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Salt M. Epithelial to mesenchymal transition rewires the mechanisms of proliferation and survival. [Thesis]. University of California – San Francisco; 2014. Available from: http://www.escholarship.org/uc/item/52s838hv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

18. Starobinets, Hanna Sonia. The Role of Autophagy in Tumor Immunology and Vasculature.

Degree: Biomedical Sciences, 2016, University of California – San Francisco

 Cancer therapy of the last hundred years has mainly aimed to quell cancer cells’ intrinsic abnormalities. Although chemotherapies and targeted therapies have been successful at… (more)

Subjects/Keywords: Cellular biology; Immunology; Medicine; autophagy; cancer biology; immunology; tumor biology; vasculature

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APA (6th Edition):

Starobinets, H. S. (2016). The Role of Autophagy in Tumor Immunology and Vasculature. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/9nq8h0ts

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Starobinets, Hanna Sonia. “The Role of Autophagy in Tumor Immunology and Vasculature.” 2016. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/9nq8h0ts.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Starobinets, Hanna Sonia. “The Role of Autophagy in Tumor Immunology and Vasculature.” 2016. Web. 20 Aug 2019.

Vancouver:

Starobinets HS. The Role of Autophagy in Tumor Immunology and Vasculature. [Internet] [Thesis]. University of California – San Francisco; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/9nq8h0ts.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Starobinets HS. The Role of Autophagy in Tumor Immunology and Vasculature. [Thesis]. University of California – San Francisco; 2016. Available from: http://www.escholarship.org/uc/item/9nq8h0ts

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

19. Samocha, Alexandr. Mammary Epithelial Cell Fate & EGFR-Rasgrp1-Ras Effector Signals.

Degree: Biomedical Sciences, 2018, University of California – San Francisco

 Mammary stem cells (MaSC) are a heterogeneous population that give rise to progenitor cells subsequently developing into differentiated cells. The exact mammary epithelial cell (MEC)… (more)

Subjects/Keywords: Developmental biology; Cellular biology; Molecular biology; development; epithelium; mammary; Ras; Rasgrp1

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APA (6th Edition):

Samocha, A. (2018). Mammary Epithelial Cell Fate & EGFR-Rasgrp1-Ras Effector Signals. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/4rn233xf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Samocha, Alexandr. “Mammary Epithelial Cell Fate & EGFR-Rasgrp1-Ras Effector Signals.” 2018. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/4rn233xf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Samocha, Alexandr. “Mammary Epithelial Cell Fate & EGFR-Rasgrp1-Ras Effector Signals.” 2018. Web. 20 Aug 2019.

Vancouver:

Samocha A. Mammary Epithelial Cell Fate & EGFR-Rasgrp1-Ras Effector Signals. [Internet] [Thesis]. University of California – San Francisco; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/4rn233xf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Samocha A. Mammary Epithelial Cell Fate & EGFR-Rasgrp1-Ras Effector Signals. [Thesis]. University of California – San Francisco; 2018. Available from: http://www.escholarship.org/uc/item/4rn233xf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Chen, Kuan-Hui. Regulation of BRCA1/p21 Axis by Prolactin.

Degree: Biomedical Sciences, 2011, University of California – Riverside

 Tumor formation/progression is determined by a wide array of factors including downregulation of tumor suppressors and upregulation of oncogenes. In breast cancers, mutation of the… (more)

Subjects/Keywords: Cellular biology

…p21, it is reported that the function of p21 depends on its cellular distribution. p21 in… …apoptosis (Asada et al., 1999). The cellular distribution of p21 is determined by a… …correlation between Stat5a signaling and p21 cellular distribution (Santos et al., 2010)… …However, the cellular distribution of p21 was altered such that p21 was sequestered in the… 

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APA (6th Edition):

Chen, K. (2011). Regulation of BRCA1/p21 Axis by Prolactin. (Thesis). University of California – Riverside. Retrieved from http://www.escholarship.org/uc/item/4972q945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Kuan-Hui. “Regulation of BRCA1/p21 Axis by Prolactin.” 2011. Thesis, University of California – Riverside. Accessed August 20, 2019. http://www.escholarship.org/uc/item/4972q945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Kuan-Hui. “Regulation of BRCA1/p21 Axis by Prolactin.” 2011. Web. 20 Aug 2019.

Vancouver:

Chen K. Regulation of BRCA1/p21 Axis by Prolactin. [Internet] [Thesis]. University of California – Riverside; 2011. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/4972q945.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen K. Regulation of BRCA1/p21 Axis by Prolactin. [Thesis]. University of California – Riverside; 2011. Available from: http://www.escholarship.org/uc/item/4972q945

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

21. Greer, Alexandra. Human dendritic cells accumulate in lung disease and contribute to serum IgE clearance via FcERI.

Degree: Biomedical Sciences, 2013, University of California – San Francisco

 Dendritic cells are crucial for the development of the adaptive immune response and have also been implicated in a number of inflammatory diseases. Much of… (more)

Subjects/Keywords: Immunology; Cellular biology; dendritic cell; FcERI; IgE

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APA (6th Edition):

Greer, A. (2013). Human dendritic cells accumulate in lung disease and contribute to serum IgE clearance via FcERI. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/7r7500rb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Greer, Alexandra. “Human dendritic cells accumulate in lung disease and contribute to serum IgE clearance via FcERI.” 2013. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/7r7500rb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Greer, Alexandra. “Human dendritic cells accumulate in lung disease and contribute to serum IgE clearance via FcERI.” 2013. Web. 20 Aug 2019.

Vancouver:

Greer A. Human dendritic cells accumulate in lung disease and contribute to serum IgE clearance via FcERI. [Internet] [Thesis]. University of California – San Francisco; 2013. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/7r7500rb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Greer A. Human dendritic cells accumulate in lung disease and contribute to serum IgE clearance via FcERI. [Thesis]. University of California – San Francisco; 2013. Available from: http://www.escholarship.org/uc/item/7r7500rb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

22. Goldsmith, Juliet Eve. Translation regulation by autophagy and nutrient stress.

Degree: Biomedical Sciences, 2018, University of California – San Francisco

 Protein translation is necessary for cell function, but it is an incredibly energy demanding process, and is therefore tightly regulated by the metabolic state of… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Autophagy; BRCA2; Cellular metabolism; Protein translation; RNA binding proteins

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APA (6th Edition):

Goldsmith, J. E. (2018). Translation regulation by autophagy and nutrient stress. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/59h0f8gw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Goldsmith, Juliet Eve. “Translation regulation by autophagy and nutrient stress.” 2018. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/59h0f8gw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Goldsmith, Juliet Eve. “Translation regulation by autophagy and nutrient stress.” 2018. Web. 20 Aug 2019.

Vancouver:

Goldsmith JE. Translation regulation by autophagy and nutrient stress. [Internet] [Thesis]. University of California – San Francisco; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/59h0f8gw.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Goldsmith JE. Translation regulation by autophagy and nutrient stress. [Thesis]. University of California – San Francisco; 2018. Available from: http://www.escholarship.org/uc/item/59h0f8gw

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

23. Smith, Thomas Horace. Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 G protein-coupled receptors (GPCRs) are transmembrane proteins that allow cells to respond to extracellular stimuli. GPCR activation occurs when a ligand binds to the extracellular… (more)

Subjects/Keywords: Pharmacology; Molecular biology; Cellular biology; dimerization; endocytosis; GPCR; thrombin

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APA (6th Edition):

Smith, T. H. (2016). Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/93g0t82v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Smith, Thomas Horace. “Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling.” 2016. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/93g0t82v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Smith, Thomas Horace. “Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling.” 2016. Web. 20 Aug 2019.

Vancouver:

Smith TH. Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/93g0t82v.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Smith TH. Characterization of protease-activated receptor-4 trafficking and heterodimerization in modulating receptor signaling. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/93g0t82v

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

24. Chapman, Dail. Regulation of motor proteins by signaling kinases.

Degree: Biomedical Sciences, 2018, University of California – Irvine

 Movement is an essential part of life. On the cellular level, movement of intracellular components is essential for a cell's vitality. Most eukaryotic cells are… (more)

Subjects/Keywords: Biophysics; Cellular biology; Molecular biology; cdk5; dynein; force; NudEL; phosphorylation

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APA (6th Edition):

Chapman, D. (2018). Regulation of motor proteins by signaling kinases. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/4sj2c8h9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chapman, Dail. “Regulation of motor proteins by signaling kinases.” 2018. Thesis, University of California – Irvine. Accessed August 20, 2019. http://www.escholarship.org/uc/item/4sj2c8h9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chapman, Dail. “Regulation of motor proteins by signaling kinases.” 2018. Web. 20 Aug 2019.

Vancouver:

Chapman D. Regulation of motor proteins by signaling kinases. [Internet] [Thesis]. University of California – Irvine; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/4sj2c8h9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chapman D. Regulation of motor proteins by signaling kinases. [Thesis]. University of California – Irvine; 2018. Available from: http://www.escholarship.org/uc/item/4sj2c8h9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

25. Judson, Robert Laird. MicroRNAs Promote Induced Pluripotency Through the Regulation of Cooperative Gene Networks.

Degree: Biomedical Sciences, 2012, University of California – San Francisco

 MicroRNAs (miRNAs) are a class of endogenous non-coding RNAs that post-transcriptionally co‐regulate networks of genes. The evolutionary history of miRNAs suggests they may play major… (more)

Subjects/Keywords: Cellular biology; Molecular biology; iPS; microRNA; Reprogramming; Stem Cells

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APA (6th Edition):

Judson, R. L. (2012). MicroRNAs Promote Induced Pluripotency Through the Regulation of Cooperative Gene Networks. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/3595s812

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Judson, Robert Laird. “MicroRNAs Promote Induced Pluripotency Through the Regulation of Cooperative Gene Networks.” 2012. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/3595s812.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Judson, Robert Laird. “MicroRNAs Promote Induced Pluripotency Through the Regulation of Cooperative Gene Networks.” 2012. Web. 20 Aug 2019.

Vancouver:

Judson RL. MicroRNAs Promote Induced Pluripotency Through the Regulation of Cooperative Gene Networks. [Internet] [Thesis]. University of California – San Francisco; 2012. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/3595s812.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Judson RL. MicroRNAs Promote Induced Pluripotency Through the Regulation of Cooperative Gene Networks. [Thesis]. University of California – San Francisco; 2012. Available from: http://www.escholarship.org/uc/item/3595s812

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Francisco

26. Zape, Joan. Characterization of Early Hematopoietic Stem and Progenitor Cell Formation, Maturation, and Function.

Degree: Biomedical Sciences, 2017, University of California – San Francisco

 Hemogenic endothelial (HE) cells are a unique subset of vascular endothelial cells endowed with the capacity to generate hematopoietic stem and progenitor cells (HSPCs) through… (more)

Subjects/Keywords: Developmental biology; Cellular biology; Blood Development; Hematopoietic Stem Cells; Hemogenic Endothelium

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APA (6th Edition):

Zape, J. (2017). Characterization of Early Hematopoietic Stem and Progenitor Cell Formation, Maturation, and Function. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/2w97h18d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zape, Joan. “Characterization of Early Hematopoietic Stem and Progenitor Cell Formation, Maturation, and Function.” 2017. Thesis, University of California – San Francisco. Accessed August 20, 2019. http://www.escholarship.org/uc/item/2w97h18d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zape, Joan. “Characterization of Early Hematopoietic Stem and Progenitor Cell Formation, Maturation, and Function.” 2017. Web. 20 Aug 2019.

Vancouver:

Zape J. Characterization of Early Hematopoietic Stem and Progenitor Cell Formation, Maturation, and Function. [Internet] [Thesis]. University of California – San Francisco; 2017. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/2w97h18d.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zape J. Characterization of Early Hematopoietic Stem and Progenitor Cell Formation, Maturation, and Function. [Thesis]. University of California – San Francisco; 2017. Available from: http://www.escholarship.org/uc/item/2w97h18d

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

27. Arul Nambi Rajan, Kanaga Sundari Rajam. Sirt1 in Trophoblast Differentiation and Placental Development.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 The placenta is a fetal-derived transient organ critical for pregnancy and fetal development. During gestation, this organ is responsible for implantation into the maternal uterus… (more)

Subjects/Keywords: Developmental biology; Molecular biology; Cellular biology; Differentiation; Placenta; Sirt1; Trophoblast; Trophoblast stem cells

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APA (6th Edition):

Arul Nambi Rajan, K. S. R. (2016). Sirt1 in Trophoblast Differentiation and Placental Development. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/98s8g5kf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Arul Nambi Rajan, Kanaga Sundari Rajam. “Sirt1 in Trophoblast Differentiation and Placental Development.” 2016. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/98s8g5kf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Arul Nambi Rajan, Kanaga Sundari Rajam. “Sirt1 in Trophoblast Differentiation and Placental Development.” 2016. Web. 20 Aug 2019.

Vancouver:

Arul Nambi Rajan KSR. Sirt1 in Trophoblast Differentiation and Placental Development. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/98s8g5kf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Arul Nambi Rajan KSR. Sirt1 in Trophoblast Differentiation and Placental Development. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/98s8g5kf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

28. Schneider, Kevin Scott. Functional analysis of Nrf1 and Nrf2 transcription factors in adipose tissue.

Degree: Biomedical Sciences, 2016, University of California – Irvine

 The Nrf1 and Nrf2 (also known as NFE2L1 and NFE2L2) transcription factors are critical regulators of genes involved in defense against oxidative stress. Previous studies… (more)

Subjects/Keywords: Biology; Cellular biology; Molecular biology; adipose tissue; antioxidant; oxidative stress; reactive oxygen species; uncoupling protein

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APA (6th Edition):

Schneider, K. S. (2016). Functional analysis of Nrf1 and Nrf2 transcription factors in adipose tissue. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/9xx4b4rs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schneider, Kevin Scott. “Functional analysis of Nrf1 and Nrf2 transcription factors in adipose tissue.” 2016. Thesis, University of California – Irvine. Accessed August 20, 2019. http://www.escholarship.org/uc/item/9xx4b4rs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schneider, Kevin Scott. “Functional analysis of Nrf1 and Nrf2 transcription factors in adipose tissue.” 2016. Web. 20 Aug 2019.

Vancouver:

Schneider KS. Functional analysis of Nrf1 and Nrf2 transcription factors in adipose tissue. [Internet] [Thesis]. University of California – Irvine; 2016. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/9xx4b4rs.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schneider KS. Functional analysis of Nrf1 and Nrf2 transcription factors in adipose tissue. [Thesis]. University of California – Irvine; 2016. Available from: http://www.escholarship.org/uc/item/9xx4b4rs

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Irvine

29. Ruiz-Vega, Rolando. Melanoma growth is regulated by the RhoJ GTPase.

Degree: Biomedical Sciences, 2017, University of California – Irvine

 The main function of the mammalian skin is to protect the organism against various deleterious environmental factors such as UVR. The skin contains specialized pigment… (more)

Subjects/Keywords: Biology; Cellular biology; Molecular biology; ATR; Immunotherapy; Melanoma; Pak; RhoJ; Skin Cancer

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APA (6th Edition):

Ruiz-Vega, R. (2017). Melanoma growth is regulated by the RhoJ GTPase. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/59k0g0jv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ruiz-Vega, Rolando. “Melanoma growth is regulated by the RhoJ GTPase.” 2017. Thesis, University of California – Irvine. Accessed August 20, 2019. http://www.escholarship.org/uc/item/59k0g0jv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ruiz-Vega, Rolando. “Melanoma growth is regulated by the RhoJ GTPase.” 2017. Web. 20 Aug 2019.

Vancouver:

Ruiz-Vega R. Melanoma growth is regulated by the RhoJ GTPase. [Internet] [Thesis]. University of California – Irvine; 2017. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/59k0g0jv.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ruiz-Vega R. Melanoma growth is regulated by the RhoJ GTPase. [Thesis]. University of California – Irvine; 2017. Available from: http://www.escholarship.org/uc/item/59k0g0jv

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

30. Garcia, Daniel Abraham. The Regulation of Epithelial-Mesenchymal Transition by the Ubiquitin-Proteasome System.

Degree: Biomedical Sciences, 2018, University of California – San Diego

 Epithelial-mesenchymal transition (EMT) is a conserved cellular plasticity program that is reactivated in carcinoma cells and drives metastasis. EMT is well studied, but its regulatory… (more)

Subjects/Keywords: Biology; Cellular biology; Molecular biology; DUB; EMT; epithelial-mesenchymal transition; metastasis; proteasome; TGF

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Garcia, D. A. (2018). The Regulation of Epithelial-Mesenchymal Transition by the Ubiquitin-Proteasome System. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/7jh563pn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Garcia, Daniel Abraham. “The Regulation of Epithelial-Mesenchymal Transition by the Ubiquitin-Proteasome System.” 2018. Thesis, University of California – San Diego. Accessed August 20, 2019. http://www.escholarship.org/uc/item/7jh563pn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Garcia, Daniel Abraham. “The Regulation of Epithelial-Mesenchymal Transition by the Ubiquitin-Proteasome System.” 2018. Web. 20 Aug 2019.

Vancouver:

Garcia DA. The Regulation of Epithelial-Mesenchymal Transition by the Ubiquitin-Proteasome System. [Internet] [Thesis]. University of California – San Diego; 2018. [cited 2019 Aug 20]. Available from: http://www.escholarship.org/uc/item/7jh563pn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Garcia DA. The Regulation of Epithelial-Mesenchymal Transition by the Ubiquitin-Proteasome System. [Thesis]. University of California – San Diego; 2018. Available from: http://www.escholarship.org/uc/item/7jh563pn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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