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You searched for subject:(Cell signaling). Showing records 1 – 30 of 645 total matches.

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1. Leydon, Alexander Rivinus. Three MYB transcription factors coordinate expression of an array of secreted proteins that promote pollen tube identity and reproductive cell signaling.

Degree: PhD, Molecular Biology, Cell Biology, and Biochemistry, 2015, Brown University

 Successful plant reproduction is the lynchpin of agricultural food production. With high susceptibility to climatic variations and other stress conditions, plant reproduction demands further investigation… (more)

Subjects/Keywords: cell signaling

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APA (6th Edition):

Leydon, A. R. (2015). Three MYB transcription factors coordinate expression of an array of secreted proteins that promote pollen tube identity and reproductive cell signaling. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419495/

Chicago Manual of Style (16th Edition):

Leydon, Alexander Rivinus. “Three MYB transcription factors coordinate expression of an array of secreted proteins that promote pollen tube identity and reproductive cell signaling.” 2015. Doctoral Dissertation, Brown University. Accessed September 16, 2019. https://repository.library.brown.edu/studio/item/bdr:419495/.

MLA Handbook (7th Edition):

Leydon, Alexander Rivinus. “Three MYB transcription factors coordinate expression of an array of secreted proteins that promote pollen tube identity and reproductive cell signaling.” 2015. Web. 16 Sep 2019.

Vancouver:

Leydon AR. Three MYB transcription factors coordinate expression of an array of secreted proteins that promote pollen tube identity and reproductive cell signaling. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 Sep 16]. Available from: https://repository.library.brown.edu/studio/item/bdr:419495/.

Council of Science Editors:

Leydon AR. Three MYB transcription factors coordinate expression of an array of secreted proteins that promote pollen tube identity and reproductive cell signaling. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419495/


University of New South Wales

2. Barbour, Jayne. Mitochondrial stress communication in mammalian cells.

Degree: Medical Sciences, 2017, University of New South Wales

 Mitochondria are crucial organelles in energy transduction, with an emerging role in cell signaling beginning to be appreciated. Due to the endosymbiotic nature of mitochondria… (more)

Subjects/Keywords: Mitochondria; Cell signaling

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APA (6th Edition):

Barbour, J. (2017). Mitochondrial stress communication in mammalian cells. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/60039 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51370/SOURCE2?view=true

Chicago Manual of Style (16th Edition):

Barbour, Jayne. “Mitochondrial stress communication in mammalian cells.” 2017. Doctoral Dissertation, University of New South Wales. Accessed September 16, 2019. http://handle.unsw.edu.au/1959.4/60039 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51370/SOURCE2?view=true.

MLA Handbook (7th Edition):

Barbour, Jayne. “Mitochondrial stress communication in mammalian cells.” 2017. Web. 16 Sep 2019.

Vancouver:

Barbour J. Mitochondrial stress communication in mammalian cells. [Internet] [Doctoral dissertation]. University of New South Wales; 2017. [cited 2019 Sep 16]. Available from: http://handle.unsw.edu.au/1959.4/60039 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51370/SOURCE2?view=true.

Council of Science Editors:

Barbour J. Mitochondrial stress communication in mammalian cells. [Doctoral Dissertation]. University of New South Wales; 2017. Available from: http://handle.unsw.edu.au/1959.4/60039 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:51370/SOURCE2?view=true


University of Sydney

3. Domanova, Westa. Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics .

Degree: 2017, University of Sydney

 Homeostasis is essential for normal function of the mammalian body. On a cellular scale biological processes are tightly controlled by dynamic intracellular signalling mechanisms. Cells… (more)

Subjects/Keywords: phosphoproteomics; kinase; cell signaling

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APA (6th Edition):

Domanova, W. (2017). Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/16704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Domanova, Westa. “Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics .” 2017. Thesis, University of Sydney. Accessed September 16, 2019. http://hdl.handle.net/2123/16704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Domanova, Westa. “Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics .” 2017. Web. 16 Sep 2019.

Vancouver:

Domanova W. Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics . [Internet] [Thesis]. University of Sydney; 2017. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2123/16704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Domanova W. Unraveling the regulation of phosphorylation in insulin singaling from temporal large-scale phosphoproteomics . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/16704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

4. Zekas, Erin. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.

Degree: Biomedical Sciences Graduate Program, 2014, University of New Mexico

 17β-estradiol (estrogen) has been demonstrated to regulate survival in breast cancer cells, which is partially mediated by its nuclear receptors ERα and ERβ and the… (more)

Subjects/Keywords: Breast Cancer Cell Signaling

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APA (6th Edition):

Zekas, E. (2014). GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/153

Chicago Manual of Style (16th Edition):

Zekas, Erin. “GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.” 2014. Doctoral Dissertation, University of New Mexico. Accessed September 16, 2019. https://digitalrepository.unm.edu/biom_etds/153.

MLA Handbook (7th Edition):

Zekas, Erin. “GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING.” 2014. Web. 16 Sep 2019.

Vancouver:

Zekas E. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. [Internet] [Doctoral dissertation]. University of New Mexico; 2014. [cited 2019 Sep 16]. Available from: https://digitalrepository.unm.edu/biom_etds/153.

Council of Science Editors:

Zekas E. GPER-MEDIATED REGULATION OF NUCLEAR AKT/FOXO3A SIGNALING. [Doctoral Dissertation]. University of New Mexico; 2014. Available from: https://digitalrepository.unm.edu/biom_etds/153


University of Rochester

5. Betzenhauser, Matthew John. Regulation of Type-2 and Type-3 Inositol (1,4,5)-Trisphosphate Receptors by cAMP-Dependent Protein Kinase Phosphorylation and ATP Binding.

Degree: PhD, 2009, University of Rochester

 Hormones, neurotransmitters, growth factors and pharmacological agents bind to cellular receptors that initiate the production of the second messenger inositol (1,4,5)-trisphosphate (InsP3). InsP3 induces the… (more)

Subjects/Keywords: Calcium; Cell signaling; Exocrine gland

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APA (6th Edition):

Betzenhauser, M. J. (2009). Regulation of Type-2 and Type-3 Inositol (1,4,5)-Trisphosphate Receptors by cAMP-Dependent Protein Kinase Phosphorylation and ATP Binding. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/8454

Chicago Manual of Style (16th Edition):

Betzenhauser, Matthew John. “Regulation of Type-2 and Type-3 Inositol (1,4,5)-Trisphosphate Receptors by cAMP-Dependent Protein Kinase Phosphorylation and ATP Binding.” 2009. Doctoral Dissertation, University of Rochester. Accessed September 16, 2019. http://hdl.handle.net/1802/8454.

MLA Handbook (7th Edition):

Betzenhauser, Matthew John. “Regulation of Type-2 and Type-3 Inositol (1,4,5)-Trisphosphate Receptors by cAMP-Dependent Protein Kinase Phosphorylation and ATP Binding.” 2009. Web. 16 Sep 2019.

Vancouver:

Betzenhauser MJ. Regulation of Type-2 and Type-3 Inositol (1,4,5)-Trisphosphate Receptors by cAMP-Dependent Protein Kinase Phosphorylation and ATP Binding. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1802/8454.

Council of Science Editors:

Betzenhauser MJ. Regulation of Type-2 and Type-3 Inositol (1,4,5)-Trisphosphate Receptors by cAMP-Dependent Protein Kinase Phosphorylation and ATP Binding. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/8454

6. Helou, Ynes. Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics.

Degree: PhD, Molecular Pharmacology, Physiology, and Biotechnology, 2015, Brown University

 Activation of T cells via the T cell receptor (TCR) is a crucial event in the adaptive immune response, and is mediated by a delicate… (more)

Subjects/Keywords: T cell receptor signaling

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APA (6th Edition):

Helou, Y. (2015). Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:419500/

Chicago Manual of Style (16th Edition):

Helou, Ynes. “Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics.” 2015. Doctoral Dissertation, Brown University. Accessed September 16, 2019. https://repository.library.brown.edu/studio/item/bdr:419500/.

MLA Handbook (7th Edition):

Helou, Ynes. “Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics.” 2015. Web. 16 Sep 2019.

Vancouver:

Helou Y. Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics. [Internet] [Doctoral dissertation]. Brown University; 2015. [cited 2019 Sep 16]. Available from: https://repository.library.brown.edu/studio/item/bdr:419500/.

Council of Science Editors:

Helou Y. Dissection of T cell receptor-mediated signaling pathways using quantitative phosphoproteomics. [Doctoral Dissertation]. Brown University; 2015. Available from: https://repository.library.brown.edu/studio/item/bdr:419500/


Tampere University

7. Pulkkinen, Kati. HIV-1 Nef protein and the Nef-associating kinase PAK2 in cell signaling .

Degree: Lääketieteellisen teknologian instituutti - Institute of Medical Technology, 2010, Tampere University

 HIV- eli immuunikatovirustartuntaa kantaa maailmassa arviolta yli 30 miljoonaa ihmistä. Suomessa ilmoituksia HIV-positiivisista henkilöistä oli vuoden 2010 alkuun mennessä kirjattu runsas kolme tuhatta, ja määrä… (more)

Subjects/Keywords: HIV; soluviestintä; Nef; cell signaling

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APA (6th Edition):

Pulkkinen, K. (2010). HIV-1 Nef protein and the Nef-associating kinase PAK2 in cell signaling . (Doctoral Dissertation). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/66577

Chicago Manual of Style (16th Edition):

Pulkkinen, Kati. “HIV-1 Nef protein and the Nef-associating kinase PAK2 in cell signaling .” 2010. Doctoral Dissertation, Tampere University. Accessed September 16, 2019. https://trepo.tuni.fi/handle/10024/66577.

MLA Handbook (7th Edition):

Pulkkinen, Kati. “HIV-1 Nef protein and the Nef-associating kinase PAK2 in cell signaling .” 2010. Web. 16 Sep 2019.

Vancouver:

Pulkkinen K. HIV-1 Nef protein and the Nef-associating kinase PAK2 in cell signaling . [Internet] [Doctoral dissertation]. Tampere University; 2010. [cited 2019 Sep 16]. Available from: https://trepo.tuni.fi/handle/10024/66577.

Council of Science Editors:

Pulkkinen K. HIV-1 Nef protein and the Nef-associating kinase PAK2 in cell signaling . [Doctoral Dissertation]. Tampere University; 2010. Available from: https://trepo.tuni.fi/handle/10024/66577


Queens University

8. Wilson, Lindsay Shea. INTEGRATING PHOSPHOLIPID AND CYCLIC NUCLEOTIDE SIGNALING: ROLES OF PHOSPHODIESTERASES AS ENZYMES AND TETHERS .

Degree: Pathology and Molecular Medicine, 2011, Queens University

 Cells of the cardiovascular system translate incoming extracellular signals from hormones and drugs through binding of cell surface receptors, and activation of intracellular signaling cascades… (more)

Subjects/Keywords: cAMP/cGMP; cell signaling; cell biology; phosphodiesterases

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APA (6th Edition):

Wilson, L. S. (2011). INTEGRATING PHOSPHOLIPID AND CYCLIC NUCLEOTIDE SIGNALING: ROLES OF PHOSPHODIESTERASES AS ENZYMES AND TETHERS . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/6581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wilson, Lindsay Shea. “INTEGRATING PHOSPHOLIPID AND CYCLIC NUCLEOTIDE SIGNALING: ROLES OF PHOSPHODIESTERASES AS ENZYMES AND TETHERS .” 2011. Thesis, Queens University. Accessed September 16, 2019. http://hdl.handle.net/1974/6581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wilson, Lindsay Shea. “INTEGRATING PHOSPHOLIPID AND CYCLIC NUCLEOTIDE SIGNALING: ROLES OF PHOSPHODIESTERASES AS ENZYMES AND TETHERS .” 2011. Web. 16 Sep 2019.

Vancouver:

Wilson LS. INTEGRATING PHOSPHOLIPID AND CYCLIC NUCLEOTIDE SIGNALING: ROLES OF PHOSPHODIESTERASES AS ENZYMES AND TETHERS . [Internet] [Thesis]. Queens University; 2011. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1974/6581.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wilson LS. INTEGRATING PHOSPHOLIPID AND CYCLIC NUCLEOTIDE SIGNALING: ROLES OF PHOSPHODIESTERASES AS ENZYMES AND TETHERS . [Thesis]. Queens University; 2011. Available from: http://hdl.handle.net/1974/6581

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

9. Stancill, Jennifer Susan. Dissecting Pancreatic β-cell Stress Using Whole Transcriptome Sequencing.

Degree: PhD, Cell and Developmental Biology, 2017, Vanderbilt University

 Type 2 diabetes is characterized by failure of pancreatic β-cells to secrete adequate insulin to meet the needs of the body. This β-cell failure is… (more)

Subjects/Keywords: cell signaling; pancreas; calcium signaling; RNA-sequencing; beta cell; Diabetes

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APA (6th Edition):

Stancill, J. S. (2017). Dissecting Pancreatic β-cell Stress Using Whole Transcriptome Sequencing. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-03162017-155618/ ;

Chicago Manual of Style (16th Edition):

Stancill, Jennifer Susan. “Dissecting Pancreatic β-cell Stress Using Whole Transcriptome Sequencing.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed September 16, 2019. http://etd.library.vanderbilt.edu/available/etd-03162017-155618/ ;.

MLA Handbook (7th Edition):

Stancill, Jennifer Susan. “Dissecting Pancreatic β-cell Stress Using Whole Transcriptome Sequencing.” 2017. Web. 16 Sep 2019.

Vancouver:

Stancill JS. Dissecting Pancreatic β-cell Stress Using Whole Transcriptome Sequencing. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2019 Sep 16]. Available from: http://etd.library.vanderbilt.edu/available/etd-03162017-155618/ ;.

Council of Science Editors:

Stancill JS. Dissecting Pancreatic β-cell Stress Using Whole Transcriptome Sequencing. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://etd.library.vanderbilt.edu/available/etd-03162017-155618/ ;


Boston University

10. Chow, Jennifer Marie. Quantitative analysis of RET signaling dynamics and crosstalk.

Degree: PhD, Chemistry, 2018, Boston University

 Most existing studies of receptor signaling are qualitative, which can lead scientists to misinterpret or overlook key information about the extent and timing of key… (more)

Subjects/Keywords: Biochemistry; Cell signaling; Growth factor receptor; mRNA dynamics; Protein dynamics; Signaling dynamics; Signaling kinetics

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APA (6th Edition):

Chow, J. M. (2018). Quantitative analysis of RET signaling dynamics and crosstalk. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/27853

Chicago Manual of Style (16th Edition):

Chow, Jennifer Marie. “Quantitative analysis of RET signaling dynamics and crosstalk.” 2018. Doctoral Dissertation, Boston University. Accessed September 16, 2019. http://hdl.handle.net/2144/27853.

MLA Handbook (7th Edition):

Chow, Jennifer Marie. “Quantitative analysis of RET signaling dynamics and crosstalk.” 2018. Web. 16 Sep 2019.

Vancouver:

Chow JM. Quantitative analysis of RET signaling dynamics and crosstalk. [Internet] [Doctoral dissertation]. Boston University; 2018. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2144/27853.

Council of Science Editors:

Chow JM. Quantitative analysis of RET signaling dynamics and crosstalk. [Doctoral Dissertation]. Boston University; 2018. Available from: http://hdl.handle.net/2144/27853


Deakin University

11. Trengove, Monique C. Suppressor of cytokine signaling 4 and 5 in zebrafish development.

Degree: 2015, Deakin University

 This research provides insight into the evolution and function of zebrafish cell signaling proteins, Suppressor of Cytokine Signaling 4a, 4b and 5a. These signaling proteins… (more)

Subjects/Keywords: zebrafish cell signaling proteins; SOCS4; SOCS5; suppressor of cytokine signaling evolution

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APA (6th Edition):

Trengove, M. C. (2015). Suppressor of cytokine signaling 4 and 5 in zebrafish development. (Thesis). Deakin University. Retrieved from http://hdl.handle.net/10536/DRO/DU:30079717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Trengove, Monique C. “Suppressor of cytokine signaling 4 and 5 in zebrafish development.” 2015. Thesis, Deakin University. Accessed September 16, 2019. http://hdl.handle.net/10536/DRO/DU:30079717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Trengove, Monique C. “Suppressor of cytokine signaling 4 and 5 in zebrafish development.” 2015. Web. 16 Sep 2019.

Vancouver:

Trengove MC. Suppressor of cytokine signaling 4 and 5 in zebrafish development. [Internet] [Thesis]. Deakin University; 2015. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/10536/DRO/DU:30079717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Trengove MC. Suppressor of cytokine signaling 4 and 5 in zebrafish development. [Thesis]. Deakin University; 2015. Available from: http://hdl.handle.net/10536/DRO/DU:30079717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New Mexico

12. Paz, Mario. Calcium Signaling Dynamics in RBL Cells Under Pulsed Ligand Exposure.

Degree: Nanoscience and Microsystems, 2015, University of New Mexico

 Understanding cellular signaling is important to learning about life at a fundamental level. As an extension of this, understanding the dysregulation of cell signaling is… (more)

Subjects/Keywords: cell signaling; microfluidics; calcium signaling; allergic response; fluorescence microscopy

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APA (6th Edition):

Paz, M. (2015). Calcium Signaling Dynamics in RBL Cells Under Pulsed Ligand Exposure. (Masters Thesis). University of New Mexico. Retrieved from http://hdl.handle.net/1928/27947

Chicago Manual of Style (16th Edition):

Paz, Mario. “Calcium Signaling Dynamics in RBL Cells Under Pulsed Ligand Exposure.” 2015. Masters Thesis, University of New Mexico. Accessed September 16, 2019. http://hdl.handle.net/1928/27947.

MLA Handbook (7th Edition):

Paz, Mario. “Calcium Signaling Dynamics in RBL Cells Under Pulsed Ligand Exposure.” 2015. Web. 16 Sep 2019.

Vancouver:

Paz M. Calcium Signaling Dynamics in RBL Cells Under Pulsed Ligand Exposure. [Internet] [Masters thesis]. University of New Mexico; 2015. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1928/27947.

Council of Science Editors:

Paz M. Calcium Signaling Dynamics in RBL Cells Under Pulsed Ligand Exposure. [Masters Thesis]. University of New Mexico; 2015. Available from: http://hdl.handle.net/1928/27947


University of Ontario Institute of Technology

13. Mannon, Sara. Effects of endosulfan on human MCF-7 breast cancer cells.

Degree: 2011, University of Ontario Institute of Technology

 Organochlorine pesticides (OCs) are environmental toxicants with important links to human health. They have been found to activate signalling pathways within cells and thereby affect… (more)

Subjects/Keywords: Endosulfan; Cell signaling; RANKL; Breast cancer; Proliferation

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APA (6th Edition):

Mannon, S. (2011). Effects of endosulfan on human MCF-7 breast cancer cells. (Thesis). University of Ontario Institute of Technology. Retrieved from http://hdl.handle.net/10155/193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mannon, Sara. “Effects of endosulfan on human MCF-7 breast cancer cells.” 2011. Thesis, University of Ontario Institute of Technology. Accessed September 16, 2019. http://hdl.handle.net/10155/193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mannon, Sara. “Effects of endosulfan on human MCF-7 breast cancer cells.” 2011. Web. 16 Sep 2019.

Vancouver:

Mannon S. Effects of endosulfan on human MCF-7 breast cancer cells. [Internet] [Thesis]. University of Ontario Institute of Technology; 2011. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/10155/193.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mannon S. Effects of endosulfan on human MCF-7 breast cancer cells. [Thesis]. University of Ontario Institute of Technology; 2011. Available from: http://hdl.handle.net/10155/193

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Georgia Tech

14. Peterson, Vincent. Progress towards the development of G-protein coupled receptor based logic gates.

Degree: MS, Chemistry and Biochemistry, 2016, Georgia Tech

 The yeast Saccharomyces cerevisiae along with the bacterium Escherichia coli have been popular model organisms for the creation or modification of chemical producing or chemical… (more)

Subjects/Keywords: Synthetic biology; GPCRs; Yeast; Cell signaling; Biosensors

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APA (6th Edition):

Peterson, V. (2016). Progress towards the development of G-protein coupled receptor based logic gates. (Masters Thesis). Georgia Tech. Retrieved from http://hdl.handle.net/1853/58141

Chicago Manual of Style (16th Edition):

Peterson, Vincent. “Progress towards the development of G-protein coupled receptor based logic gates.” 2016. Masters Thesis, Georgia Tech. Accessed September 16, 2019. http://hdl.handle.net/1853/58141.

MLA Handbook (7th Edition):

Peterson, Vincent. “Progress towards the development of G-protein coupled receptor based logic gates.” 2016. Web. 16 Sep 2019.

Vancouver:

Peterson V. Progress towards the development of G-protein coupled receptor based logic gates. [Internet] [Masters thesis]. Georgia Tech; 2016. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1853/58141.

Council of Science Editors:

Peterson V. Progress towards the development of G-protein coupled receptor based logic gates. [Masters Thesis]. Georgia Tech; 2016. Available from: http://hdl.handle.net/1853/58141


De Montfort University

15. Luo, Yi. Mechanisms of epidermal growth factor receptor signalling in primary rat hepatocytes.

Degree: PhD, 2009, De Montfort University

 In the U.K. deaths due to liver diseases, especially alcohol related diseases, have risen considerably over the last 20 years. In 2005 up to 13,000… (more)

Subjects/Keywords: 612.3; cell signaling; EGF receptor; hepatocyte; internalization

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APA (6th Edition):

Luo, Y. (2009). Mechanisms of epidermal growth factor receptor signalling in primary rat hepatocytes. (Doctoral Dissertation). De Montfort University. Retrieved from http://hdl.handle.net/2086/2413

Chicago Manual of Style (16th Edition):

Luo, Yi. “Mechanisms of epidermal growth factor receptor signalling in primary rat hepatocytes.” 2009. Doctoral Dissertation, De Montfort University. Accessed September 16, 2019. http://hdl.handle.net/2086/2413.

MLA Handbook (7th Edition):

Luo, Yi. “Mechanisms of epidermal growth factor receptor signalling in primary rat hepatocytes.” 2009. Web. 16 Sep 2019.

Vancouver:

Luo Y. Mechanisms of epidermal growth factor receptor signalling in primary rat hepatocytes. [Internet] [Doctoral dissertation]. De Montfort University; 2009. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2086/2413.

Council of Science Editors:

Luo Y. Mechanisms of epidermal growth factor receptor signalling in primary rat hepatocytes. [Doctoral Dissertation]. De Montfort University; 2009. Available from: http://hdl.handle.net/2086/2413

16. Nguyen, Nhan. Engineering a cell line for analysis of Type I interferon signaling.

Degree: 2015, California State University – San Marcos

 Controlling interferon production will reduce the frequency of relapses of multiple sclerosis and provide a better quality of life for patients. Finding the gatekeepers within… (more)

Subjects/Keywords: interferon signaling; gfp; engineering; cell line

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nguyen, N. (2015). Engineering a cell line for analysis of Type I interferon signaling. (Thesis). California State University – San Marcos. Retrieved from http://hdl.handle.net/10211.3/139190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nguyen, Nhan. “Engineering a cell line for analysis of Type I interferon signaling. ” 2015. Thesis, California State University – San Marcos. Accessed September 16, 2019. http://hdl.handle.net/10211.3/139190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nguyen, Nhan. “Engineering a cell line for analysis of Type I interferon signaling. ” 2015. Web. 16 Sep 2019.

Vancouver:

Nguyen N. Engineering a cell line for analysis of Type I interferon signaling. [Internet] [Thesis]. California State University – San Marcos; 2015. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/10211.3/139190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nguyen N. Engineering a cell line for analysis of Type I interferon signaling. [Thesis]. California State University – San Marcos; 2015. Available from: http://hdl.handle.net/10211.3/139190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Louisiana State University

17. Creekmore, Emily Qualls. Characterization of the effect of alcohol on recombinant proteins derived from mammalian adenylyl cyclase.

Degree: PhD, Medicine and Health Sciences, 2013, Louisiana State University

 Research suggests that the cyclic AMP (cAMP) signaling pathway is implicated in the development of alcoholism. Previous work in our laboratory has demonstrated that alcohol… (more)

Subjects/Keywords: cell signaling; cAMP; adenylyl cyclase; alcohol

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Creekmore, E. Q. (2013). Characterization of the effect of alcohol on recombinant proteins derived from mammalian adenylyl cyclase. (Doctoral Dissertation). Louisiana State University. Retrieved from etd-10302013-142308 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3707

Chicago Manual of Style (16th Edition):

Creekmore, Emily Qualls. “Characterization of the effect of alcohol on recombinant proteins derived from mammalian adenylyl cyclase.” 2013. Doctoral Dissertation, Louisiana State University. Accessed September 16, 2019. etd-10302013-142308 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3707.

MLA Handbook (7th Edition):

Creekmore, Emily Qualls. “Characterization of the effect of alcohol on recombinant proteins derived from mammalian adenylyl cyclase.” 2013. Web. 16 Sep 2019.

Vancouver:

Creekmore EQ. Characterization of the effect of alcohol on recombinant proteins derived from mammalian adenylyl cyclase. [Internet] [Doctoral dissertation]. Louisiana State University; 2013. [cited 2019 Sep 16]. Available from: etd-10302013-142308 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3707.

Council of Science Editors:

Creekmore EQ. Characterization of the effect of alcohol on recombinant proteins derived from mammalian adenylyl cyclase. [Doctoral Dissertation]. Louisiana State University; 2013. Available from: etd-10302013-142308 ; https://digitalcommons.lsu.edu/gradschool_dissertations/3707


University of California – San Diego

18. Handly, Linda Naomi. Epithelial wound response: How do cells know where they are and what to do after a wound?.

Degree: Chemistry, 2017, University of California – San Diego

 Wound healing begins as soon as the wound occurs and can continue for days, weeks, or even months. Although decades of research have established the… (more)

Subjects/Keywords: Biochemistry; cell signaling; quantitative biology; wound response

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APA (6th Edition):

Handly, L. N. (2017). Epithelial wound response: How do cells know where they are and what to do after a wound?. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/3bm9h3v8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Handly, Linda Naomi. “Epithelial wound response: How do cells know where they are and what to do after a wound?.” 2017. Thesis, University of California – San Diego. Accessed September 16, 2019. http://www.escholarship.org/uc/item/3bm9h3v8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Handly, Linda Naomi. “Epithelial wound response: How do cells know where they are and what to do after a wound?.” 2017. Web. 16 Sep 2019.

Vancouver:

Handly LN. Epithelial wound response: How do cells know where they are and what to do after a wound?. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2019 Sep 16]. Available from: http://www.escholarship.org/uc/item/3bm9h3v8.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Handly LN. Epithelial wound response: How do cells know where they are and what to do after a wound?. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/3bm9h3v8

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

19. Silva, Oscar. Discs Large Homolog 1: Molecular Scaffolds Guiding T Cell Activation and Effector Function.

Degree: Microbiology, Immunology, & Molecular Genetics, 2013, UCLA

 Discs large homolog 1 (Dlgh1) is a scaffold protein that couples T cell receptor (TCR) engagement to signal transduction and cytoskeletal reorganization. Specifically, Dlgh1 directs… (more)

Subjects/Keywords: Immunology; alternative splicing; Dlgh1; T cell signaling

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APA (6th Edition):

Silva, O. (2013). Discs Large Homolog 1: Molecular Scaffolds Guiding T Cell Activation and Effector Function. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/55k9p6qd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Oscar. “Discs Large Homolog 1: Molecular Scaffolds Guiding T Cell Activation and Effector Function.” 2013. Thesis, UCLA. Accessed September 16, 2019. http://www.escholarship.org/uc/item/55k9p6qd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Oscar. “Discs Large Homolog 1: Molecular Scaffolds Guiding T Cell Activation and Effector Function.” 2013. Web. 16 Sep 2019.

Vancouver:

Silva O. Discs Large Homolog 1: Molecular Scaffolds Guiding T Cell Activation and Effector Function. [Internet] [Thesis]. UCLA; 2013. [cited 2019 Sep 16]. Available from: http://www.escholarship.org/uc/item/55k9p6qd.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva O. Discs Large Homolog 1: Molecular Scaffolds Guiding T Cell Activation and Effector Function. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/55k9p6qd

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

20. Phillips, Matthew. MECHANISMS FOR THE STRUCTURAL ACTIVATION OF THE SMALL G PROTEIN CDC42 AND ITS CONSTITUTIVELY ACTIVATED MUTANT, CDC42 (Q61L) .

Degree: 2008, Cornell University

 The x-ray crystal structures for the GTP-bound forms of the small G protein Cdc42 and its constitutively activated isoform, Cdc42 (Q61L), were previously unkown. The… (more)

Subjects/Keywords: G protein structure; Cdc42; cell signaling

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APA (6th Edition):

Phillips, M. (2008). MECHANISMS FOR THE STRUCTURAL ACTIVATION OF THE SMALL G PROTEIN CDC42 AND ITS CONSTITUTIVELY ACTIVATED MUTANT, CDC42 (Q61L) . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/10913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Phillips, Matthew. “MECHANISMS FOR THE STRUCTURAL ACTIVATION OF THE SMALL G PROTEIN CDC42 AND ITS CONSTITUTIVELY ACTIVATED MUTANT, CDC42 (Q61L) .” 2008. Thesis, Cornell University. Accessed September 16, 2019. http://hdl.handle.net/1813/10913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Phillips, Matthew. “MECHANISMS FOR THE STRUCTURAL ACTIVATION OF THE SMALL G PROTEIN CDC42 AND ITS CONSTITUTIVELY ACTIVATED MUTANT, CDC42 (Q61L) .” 2008. Web. 16 Sep 2019.

Vancouver:

Phillips M. MECHANISMS FOR THE STRUCTURAL ACTIVATION OF THE SMALL G PROTEIN CDC42 AND ITS CONSTITUTIVELY ACTIVATED MUTANT, CDC42 (Q61L) . [Internet] [Thesis]. Cornell University; 2008. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1813/10913.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Phillips M. MECHANISMS FOR THE STRUCTURAL ACTIVATION OF THE SMALL G PROTEIN CDC42 AND ITS CONSTITUTIVELY ACTIVATED MUTANT, CDC42 (Q61L) . [Thesis]. Cornell University; 2008. Available from: http://hdl.handle.net/1813/10913

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

21. Chylek, Lily. Understanding Ige Receptor Signaling Through Computational Modeling And Quantitative Experiments .

Degree: 2016, Cornell University

 Fc[epsilon]RI is a multi-subunit receptor found on the surface of mast cells and basophils and binds immunoglobulin E (IgE) with high affinity. Stimulation of this… (more)

Subjects/Keywords: mast cells; computational modeling; cell signaling

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APA (6th Edition):

Chylek, L. (2016). Understanding Ige Receptor Signaling Through Computational Modeling And Quantitative Experiments . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/44334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chylek, Lily. “Understanding Ige Receptor Signaling Through Computational Modeling And Quantitative Experiments .” 2016. Thesis, Cornell University. Accessed September 16, 2019. http://hdl.handle.net/1813/44334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chylek, Lily. “Understanding Ige Receptor Signaling Through Computational Modeling And Quantitative Experiments .” 2016. Web. 16 Sep 2019.

Vancouver:

Chylek L. Understanding Ige Receptor Signaling Through Computational Modeling And Quantitative Experiments . [Internet] [Thesis]. Cornell University; 2016. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1813/44334.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chylek L. Understanding Ige Receptor Signaling Through Computational Modeling And Quantitative Experiments . [Thesis]. Cornell University; 2016. Available from: http://hdl.handle.net/1813/44334

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cornell University

22. Srinivasan, Tara. Notch Signaling: Mechanistic And Functional Studies In Intestinal Stem Cells And Colorectal Cancer Cells .

Degree: 2016, Cornell University

 : The study of stem cell regulation in intestinal and colonic tissues is an area of significant focus within the scientific community, providing mechanistic insight… (more)

Subjects/Keywords: colorectal cancer; stem cell; NOTCH signaling

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APA (6th Edition):

Srinivasan, T. (2016). Notch Signaling: Mechanistic And Functional Studies In Intestinal Stem Cells And Colorectal Cancer Cells . (Thesis). Cornell University. Retrieved from http://hdl.handle.net/1813/44394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Srinivasan, Tara. “Notch Signaling: Mechanistic And Functional Studies In Intestinal Stem Cells And Colorectal Cancer Cells .” 2016. Thesis, Cornell University. Accessed September 16, 2019. http://hdl.handle.net/1813/44394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Srinivasan, Tara. “Notch Signaling: Mechanistic And Functional Studies In Intestinal Stem Cells And Colorectal Cancer Cells .” 2016. Web. 16 Sep 2019.

Vancouver:

Srinivasan T. Notch Signaling: Mechanistic And Functional Studies In Intestinal Stem Cells And Colorectal Cancer Cells . [Internet] [Thesis]. Cornell University; 2016. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1813/44394.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Srinivasan T. Notch Signaling: Mechanistic And Functional Studies In Intestinal Stem Cells And Colorectal Cancer Cells . [Thesis]. Cornell University; 2016. Available from: http://hdl.handle.net/1813/44394

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Princeton University

23. Wu, Mingxuan. DECODING DIPHOSPHOINOSITOL POLYPHOSPHATE SIGNALING .

Degree: PhD, 2015, Princeton University

 Diphosphoinositol polyphosphates (PP-IPs) represent a unique class of high energy messengers controlling a wide variety of cellular processes. It is proposed that these messengers transduce… (more)

Subjects/Keywords: Cell Signaling; Diphosphoinositol Polyphosphate; Second Messengers

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APA (6th Edition):

Wu, M. (2015). DECODING DIPHOSPHOINOSITOL POLYPHOSPHATE SIGNALING . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp01kh04ds07v

Chicago Manual of Style (16th Edition):

Wu, Mingxuan. “DECODING DIPHOSPHOINOSITOL POLYPHOSPHATE SIGNALING .” 2015. Doctoral Dissertation, Princeton University. Accessed September 16, 2019. http://arks.princeton.edu/ark:/88435/dsp01kh04ds07v.

MLA Handbook (7th Edition):

Wu, Mingxuan. “DECODING DIPHOSPHOINOSITOL POLYPHOSPHATE SIGNALING .” 2015. Web. 16 Sep 2019.

Vancouver:

Wu M. DECODING DIPHOSPHOINOSITOL POLYPHOSPHATE SIGNALING . [Internet] [Doctoral dissertation]. Princeton University; 2015. [cited 2019 Sep 16]. Available from: http://arks.princeton.edu/ark:/88435/dsp01kh04ds07v.

Council of Science Editors:

Wu M. DECODING DIPHOSPHOINOSITOL POLYPHOSPHATE SIGNALING . [Doctoral Dissertation]. Princeton University; 2015. Available from: http://arks.princeton.edu/ark:/88435/dsp01kh04ds07v


University of Ottawa

24. Hashim, Irshaad. The Effect of Freud-1/CC2D1A Knockout on EGF Receptor Activation .

Degree: 2015, University of Ottawa

 CC2D1A (coiled-coil and C2 domain containing protein 1A), also known as Freud-1, has been identified as a transcriptional repressor of the serotonin receptor 5-HT1A, a… (more)

Subjects/Keywords: Neuroscience; Cell Signaling; Freud-1; Molecular Biology

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APA (6th Edition):

Hashim, I. (2015). The Effect of Freud-1/CC2D1A Knockout on EGF Receptor Activation . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/33400

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hashim, Irshaad. “The Effect of Freud-1/CC2D1A Knockout on EGF Receptor Activation .” 2015. Thesis, University of Ottawa. Accessed September 16, 2019. http://hdl.handle.net/10393/33400.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hashim, Irshaad. “The Effect of Freud-1/CC2D1A Knockout on EGF Receptor Activation .” 2015. Web. 16 Sep 2019.

Vancouver:

Hashim I. The Effect of Freud-1/CC2D1A Knockout on EGF Receptor Activation . [Internet] [Thesis]. University of Ottawa; 2015. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/10393/33400.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hashim I. The Effect of Freud-1/CC2D1A Knockout on EGF Receptor Activation . [Thesis]. University of Ottawa; 2015. Available from: http://hdl.handle.net/10393/33400

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

25. Binder Saldana, Pablo Antonio. Role of eIF3d on the mTOR signalling pathway.

Degree: 2016, University of Manchester

 The mammalian target of rapamycin (mTOR) is a serine/threonine kinase that belongs to the phosphoinositide 3-kinase (PI3K)-related kinase family and participates in sensing and integration… (more)

Subjects/Keywords: mTOR; eIF3; eIF3d; S6K; Cell Signaling

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APA (6th Edition):

Binder Saldana, P. A. (2016). Role of eIF3d on the mTOR signalling pathway. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:297340

Chicago Manual of Style (16th Edition):

Binder Saldana, Pablo Antonio. “Role of eIF3d on the mTOR signalling pathway.” 2016. Doctoral Dissertation, University of Manchester. Accessed September 16, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:297340.

MLA Handbook (7th Edition):

Binder Saldana, Pablo Antonio. “Role of eIF3d on the mTOR signalling pathway.” 2016. Web. 16 Sep 2019.

Vancouver:

Binder Saldana PA. Role of eIF3d on the mTOR signalling pathway. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Sep 16]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:297340.

Council of Science Editors:

Binder Saldana PA. Role of eIF3d on the mTOR signalling pathway. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:297340


University of Texas Medical Branch – Galveston

26. [No author]. A mechanism of activation of c-MET receptor tyrosine kinase .

Degree: 2006, University of Texas Medical Branch – Galveston

 c-MET receptor tyrosine kinase-mediated signaling governs numerous important cellular responses including cellular proliferation, differentiation, migration and apoptosis. Deregulation of these signals result in malignant behaviors,… (more)

Subjects/Keywords: cell signaling; cancer

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APA (6th Edition):

author], [. (2006). A mechanism of activation of c-MET receptor tyrosine kinase . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/181

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “A mechanism of activation of c-MET receptor tyrosine kinase .” 2006. Thesis, University of Texas Medical Branch – Galveston. Accessed September 16, 2019. http://hdl.handle.net/2152.3/181.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “A mechanism of activation of c-MET receptor tyrosine kinase .” 2006. Web. 16 Sep 2019.

Vancouver:

author] [. A mechanism of activation of c-MET receptor tyrosine kinase . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; 2006. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2152.3/181.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. A mechanism of activation of c-MET receptor tyrosine kinase . [Thesis]. University of Texas Medical Branch – Galveston; 2006. Available from: http://hdl.handle.net/2152.3/181

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Medical Branch – Galveston

27. [No author]. Regulation of hepatocyte growth factor receptor endocytic trafficking .

Degree: 2006, University of Texas Medical Branch – Galveston

 The Hepatocyte Growth Factor Receptor (HGFR/cMet) is a receptor tyrosine kinase that is essential for multiple cell responses, including cell proliferation, survival, motility and branching… (more)

Subjects/Keywords: endocytosis; cell signaling

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APA (6th Edition):

author], [. (2006). Regulation of hepatocyte growth factor receptor endocytic trafficking . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/97

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Regulation of hepatocyte growth factor receptor endocytic trafficking .” 2006. Thesis, University of Texas Medical Branch – Galveston. Accessed September 16, 2019. http://hdl.handle.net/2152.3/97.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Regulation of hepatocyte growth factor receptor endocytic trafficking .” 2006. Web. 16 Sep 2019.

Vancouver:

author] [. Regulation of hepatocyte growth factor receptor endocytic trafficking . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; 2006. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2152.3/97.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Regulation of hepatocyte growth factor receptor endocytic trafficking . [Thesis]. University of Texas Medical Branch – Galveston; 2006. Available from: http://hdl.handle.net/2152.3/97

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

28. Kraguljac, Alan P. Use of Phospho-flow Cytometry to Define Influence of High-Risk Genetic Abnormalities on Cytokine-responsiveness in Human B-cell Leukemia.

Degree: 2012, University of Toronto

B-cell acute lymphoblastic leukemia (B-ALL) represents a collection of diseases that are categorized into subtypes based on the presence of recurrent cytogenetic abnormalities. These abnormalities… (more)

Subjects/Keywords: Leukemia; Signaling; B cell; Cytokine; CRLF2; 0760

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APA (6th Edition):

Kraguljac, A. P. (2012). Use of Phospho-flow Cytometry to Define Influence of High-Risk Genetic Abnormalities on Cytokine-responsiveness in Human B-cell Leukemia. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33296

Chicago Manual of Style (16th Edition):

Kraguljac, Alan P. “Use of Phospho-flow Cytometry to Define Influence of High-Risk Genetic Abnormalities on Cytokine-responsiveness in Human B-cell Leukemia.” 2012. Masters Thesis, University of Toronto. Accessed September 16, 2019. http://hdl.handle.net/1807/33296.

MLA Handbook (7th Edition):

Kraguljac, Alan P. “Use of Phospho-flow Cytometry to Define Influence of High-Risk Genetic Abnormalities on Cytokine-responsiveness in Human B-cell Leukemia.” 2012. Web. 16 Sep 2019.

Vancouver:

Kraguljac AP. Use of Phospho-flow Cytometry to Define Influence of High-Risk Genetic Abnormalities on Cytokine-responsiveness in Human B-cell Leukemia. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/1807/33296.

Council of Science Editors:

Kraguljac AP. Use of Phospho-flow Cytometry to Define Influence of High-Risk Genetic Abnormalities on Cytokine-responsiveness in Human B-cell Leukemia. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33296


University of Illinois – Urbana-Champaign

29. Goldberg, Leah. The role of notch signaling in the regulation of proliferation and cell differentiation during embryonic and postnatal pituitary development.

Degree: PhD, 0325, 2014, University of Illinois – Urbana-Champaign

 The pituitary develops in two distinct waves in mice, one occurring during embryogenesis and a second taking place during early postnatal development. During both periods,… (more)

Subjects/Keywords: Pituitary development; Notch signaling; Proliferation; Cell specification

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APA (6th Edition):

Goldberg, L. (2014). The role of notch signaling in the regulation of proliferation and cell differentiation during embryonic and postnatal pituitary development. (Doctoral Dissertation). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/49851

Chicago Manual of Style (16th Edition):

Goldberg, Leah. “The role of notch signaling in the regulation of proliferation and cell differentiation during embryonic and postnatal pituitary development.” 2014. Doctoral Dissertation, University of Illinois – Urbana-Champaign. Accessed September 16, 2019. http://hdl.handle.net/2142/49851.

MLA Handbook (7th Edition):

Goldberg, Leah. “The role of notch signaling in the regulation of proliferation and cell differentiation during embryonic and postnatal pituitary development.” 2014. Web. 16 Sep 2019.

Vancouver:

Goldberg L. The role of notch signaling in the regulation of proliferation and cell differentiation during embryonic and postnatal pituitary development. [Internet] [Doctoral dissertation]. University of Illinois – Urbana-Champaign; 2014. [cited 2019 Sep 16]. Available from: http://hdl.handle.net/2142/49851.

Council of Science Editors:

Goldberg L. The role of notch signaling in the regulation of proliferation and cell differentiation during embryonic and postnatal pituitary development. [Doctoral Dissertation]. University of Illinois – Urbana-Champaign; 2014. Available from: http://hdl.handle.net/2142/49851

30. Schott, Micah. Gravin Regulates Crosstalk Between Calcium And PkA Dependent Signaling Pathways In Cultured Cells.

Degree: PhD, Biology, 2014, University of North Dakota

  A-Kinase Anchoring Proteins (AKAPs) comprise a family of roughly 70 scaffolds that anchor PKA and other enzymes to a variety of subcellular compartments. Although… (more)

Subjects/Keywords: Cell signaling; Compartmentalization; Kinase; Microscopy; Scaffold

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APA (6th Edition):

Schott, M. (2014). Gravin Regulates Crosstalk Between Calcium And PkA Dependent Signaling Pathways In Cultured Cells. (Doctoral Dissertation). University of North Dakota. Retrieved from https://commons.und.edu/theses/1709

Chicago Manual of Style (16th Edition):

Schott, Micah. “Gravin Regulates Crosstalk Between Calcium And PkA Dependent Signaling Pathways In Cultured Cells.” 2014. Doctoral Dissertation, University of North Dakota. Accessed September 16, 2019. https://commons.und.edu/theses/1709.

MLA Handbook (7th Edition):

Schott, Micah. “Gravin Regulates Crosstalk Between Calcium And PkA Dependent Signaling Pathways In Cultured Cells.” 2014. Web. 16 Sep 2019.

Vancouver:

Schott M. Gravin Regulates Crosstalk Between Calcium And PkA Dependent Signaling Pathways In Cultured Cells. [Internet] [Doctoral dissertation]. University of North Dakota; 2014. [cited 2019 Sep 16]. Available from: https://commons.und.edu/theses/1709.

Council of Science Editors:

Schott M. Gravin Regulates Crosstalk Between Calcium And PkA Dependent Signaling Pathways In Cultured Cells. [Doctoral Dissertation]. University of North Dakota; 2014. Available from: https://commons.und.edu/theses/1709

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