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You searched for subject:(Cell fate). Showing records 1 – 30 of 101 total matches.

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University of Manchester

1. Sloss, Olivia. The functional role of Mcl-1 in the dynamics of mitotic cell fate.

Degree: 2015, University of Manchester

Drugs that alter microtubule dynamics are often used in chemotherapy regimes in combination with other agents in order to treat various cancers. Despite the success… (more)

Subjects/Keywords: mitosis; Mcl-1; cell fate

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sloss, O. (2015). The functional role of Mcl-1 in the dynamics of mitotic cell fate. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:291383

Chicago Manual of Style (16th Edition):

Sloss, Olivia. “The functional role of Mcl-1 in the dynamics of mitotic cell fate.” 2015. Doctoral Dissertation, University of Manchester. Accessed March 18, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:291383.

MLA Handbook (7th Edition):

Sloss, Olivia. “The functional role of Mcl-1 in the dynamics of mitotic cell fate.” 2015. Web. 18 Mar 2019.

Vancouver:

Sloss O. The functional role of Mcl-1 in the dynamics of mitotic cell fate. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2019 Mar 18]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:291383.

Council of Science Editors:

Sloss O. The functional role of Mcl-1 in the dynamics of mitotic cell fate. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:291383


University of Edinburgh

2. Bai, Yu. A novel technique for manipulating cell fate.

Degree: PhD, 2014, University of Edinburgh

 The demonstration that simply by introducing four selected proteins it is possible to change mammalian somatic cells from one phenotype to another is providing important… (more)

Subjects/Keywords: 571.6; cell fate; reprogramming

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APA (6th Edition):

Bai, Y. (2014). A novel technique for manipulating cell fate. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/17860

Chicago Manual of Style (16th Edition):

Bai, Yu. “A novel technique for manipulating cell fate.” 2014. Doctoral Dissertation, University of Edinburgh. Accessed March 18, 2019. http://hdl.handle.net/1842/17860.

MLA Handbook (7th Edition):

Bai, Yu. “A novel technique for manipulating cell fate.” 2014. Web. 18 Mar 2019.

Vancouver:

Bai Y. A novel technique for manipulating cell fate. [Internet] [Doctoral dissertation]. University of Edinburgh; 2014. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1842/17860.

Council of Science Editors:

Bai Y. A novel technique for manipulating cell fate. [Doctoral Dissertation]. University of Edinburgh; 2014. Available from: http://hdl.handle.net/1842/17860


University of Toronto

3. Alexson, Tania O. Redefining Pluripotent Stem Cells: A Cell Cycle Perspective.

Degree: PhD, 2018, University of Toronto

 As it is classically defined, the term stem cell seems at odds with the process of early development where, at least initially, there are no… (more)

Subjects/Keywords: Cell Cycle; Cell Fate; Embryonic Stem Cell; Pluripotency; Stem Cell; 0758

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APA (6th Edition):

Alexson, T. O. (2018). Redefining Pluripotent Stem Cells: A Cell Cycle Perspective. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89792

Chicago Manual of Style (16th Edition):

Alexson, Tania O. “Redefining Pluripotent Stem Cells: A Cell Cycle Perspective.” 2018. Doctoral Dissertation, University of Toronto. Accessed March 18, 2019. http://hdl.handle.net/1807/89792.

MLA Handbook (7th Edition):

Alexson, Tania O. “Redefining Pluripotent Stem Cells: A Cell Cycle Perspective.” 2018. Web. 18 Mar 2019.

Vancouver:

Alexson TO. Redefining Pluripotent Stem Cells: A Cell Cycle Perspective. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1807/89792.

Council of Science Editors:

Alexson TO. Redefining Pluripotent Stem Cells: A Cell Cycle Perspective. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/89792


University of Manchester

4. Sloss, Olivia. The functional role of Mcl-1 in the dynamics of mitotic cell fate.

Degree: PhD, 2015, University of Manchester

 Drugs that alter microtubule dynamics are often used in chemotherapy regimes in combination with other agents in order to treat various cancers. Despite the success… (more)

Subjects/Keywords: 571.8; mitosis; Mcl-1; cell fate

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sloss, O. (2015). The functional role of Mcl-1 in the dynamics of mitotic cell fate. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-functional-role-of-mcl1-in-the-dynamics-of-mitotic-cell-fate(d567e84b-3a51-4ec9-8f5c-779704d26bae).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701101

Chicago Manual of Style (16th Edition):

Sloss, Olivia. “The functional role of Mcl-1 in the dynamics of mitotic cell fate.” 2015. Doctoral Dissertation, University of Manchester. Accessed March 18, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-functional-role-of-mcl1-in-the-dynamics-of-mitotic-cell-fate(d567e84b-3a51-4ec9-8f5c-779704d26bae).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701101.

MLA Handbook (7th Edition):

Sloss, Olivia. “The functional role of Mcl-1 in the dynamics of mitotic cell fate.” 2015. Web. 18 Mar 2019.

Vancouver:

Sloss O. The functional role of Mcl-1 in the dynamics of mitotic cell fate. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2019 Mar 18]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-functional-role-of-mcl1-in-the-dynamics-of-mitotic-cell-fate(d567e84b-3a51-4ec9-8f5c-779704d26bae).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701101.

Council of Science Editors:

Sloss O. The functional role of Mcl-1 in the dynamics of mitotic cell fate. [Doctoral Dissertation]. University of Manchester; 2015. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-functional-role-of-mcl1-in-the-dynamics-of-mitotic-cell-fate(d567e84b-3a51-4ec9-8f5c-779704d26bae).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.701101


University of Windsor

5. Davis, Gordon Omar. Regulation of Tuberin in Cell Fate Acquisition within Developing Neural Tissues.

Degree: MS, Biological Sciences, 2012, University of Windsor

  Inhibition or misregulation of the tumour suppressor protein Tuberin is known to cause the benign tumour disorder Tuberous Sclerosis, involving developmental defects in many… (more)

Subjects/Keywords: Cell Fate; Neural Development; Tuberin; Tuberous Sclerosis

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APA (6th Edition):

Davis, G. O. (2012). Regulation of Tuberin in Cell Fate Acquisition within Developing Neural Tissues. (Masters Thesis). University of Windsor. Retrieved from https://scholar.uwindsor.ca/etd/5424

Chicago Manual of Style (16th Edition):

Davis, Gordon Omar. “Regulation of Tuberin in Cell Fate Acquisition within Developing Neural Tissues.” 2012. Masters Thesis, University of Windsor. Accessed March 18, 2019. https://scholar.uwindsor.ca/etd/5424.

MLA Handbook (7th Edition):

Davis, Gordon Omar. “Regulation of Tuberin in Cell Fate Acquisition within Developing Neural Tissues.” 2012. Web. 18 Mar 2019.

Vancouver:

Davis GO. Regulation of Tuberin in Cell Fate Acquisition within Developing Neural Tissues. [Internet] [Masters thesis]. University of Windsor; 2012. [cited 2019 Mar 18]. Available from: https://scholar.uwindsor.ca/etd/5424.

Council of Science Editors:

Davis GO. Regulation of Tuberin in Cell Fate Acquisition within Developing Neural Tissues. [Masters Thesis]. University of Windsor; 2012. Available from: https://scholar.uwindsor.ca/etd/5424

6. Krenning, L. Cell cycle checkpoints: reversible when possible, irreversible when needed.

Degree: 2015, Universiteit Utrecht

Cell cycle checkpoints are reversible in nature, and can prevent progression into the next cell cycle phase if needed. In the case of DNA damage,… (more)

Subjects/Keywords: Cell cycle checkpoints; DNA damage; Checkpoint Recovery; Cell fate; Mitosis

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APA (6th Edition):

Krenning, L. (2015). Cell cycle checkpoints: reversible when possible, irreversible when needed. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/322216

Chicago Manual of Style (16th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed March 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/322216.

MLA Handbook (7th Edition):

Krenning, L. “Cell cycle checkpoints: reversible when possible, irreversible when needed.” 2015. Web. 18 Mar 2019.

Vancouver:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2019 Mar 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/322216.

Council of Science Editors:

Krenning L. Cell cycle checkpoints: reversible when possible, irreversible when needed. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/322216


University of Illinois – Urbana-Champaign

7. Choi, Ji Sun. Substrate elasticity regulates the biophysical properties of hematopoietic stem and progenitor cells.

Degree: MS, 0300, 2012, University of Illinois – Urbana-Champaign

 Physiological environments of the HSC niches exhibit a range of stiffness, ranging from soft marrow (< 1 Pa) to adipose tissue (1~3 kPa) to non-mineralized… (more)

Subjects/Keywords: Hematopoietic stem cell; stem cell fate; stem cell niche; cell spreading; substrate elasticity; hydrogels

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APA (6th Edition):

Choi, J. S. (2012). Substrate elasticity regulates the biophysical properties of hematopoietic stem and progenitor cells. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/29417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Choi, Ji Sun. “Substrate elasticity regulates the biophysical properties of hematopoietic stem and progenitor cells.” 2012. Thesis, University of Illinois – Urbana-Champaign. Accessed March 18, 2019. http://hdl.handle.net/2142/29417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Choi, Ji Sun. “Substrate elasticity regulates the biophysical properties of hematopoietic stem and progenitor cells.” 2012. Web. 18 Mar 2019.

Vancouver:

Choi JS. Substrate elasticity regulates the biophysical properties of hematopoietic stem and progenitor cells. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2012. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/2142/29417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Choi JS. Substrate elasticity regulates the biophysical properties of hematopoietic stem and progenitor cells. [Thesis]. University of Illinois – Urbana-Champaign; 2012. Available from: http://hdl.handle.net/2142/29417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

8. Stapel, L.C. The first cell fate specification event in mouse development.

Degree: 2011, Universiteit Utrecht

 The separation of inner cell mass and trophectoderm is the first cell fate specification event in mammals. The inner cell mass will form the embryo… (more)

Subjects/Keywords: mouse development; cell fate specification; cell polarity; regulative development; inner cell mass; trophectoderm; Cdx-2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stapel, L. C. (2011). The first cell fate specification event in mouse development. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/222737

Chicago Manual of Style (16th Edition):

Stapel, L C. “The first cell fate specification event in mouse development.” 2011. Masters Thesis, Universiteit Utrecht. Accessed March 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/222737.

MLA Handbook (7th Edition):

Stapel, L C. “The first cell fate specification event in mouse development.” 2011. Web. 18 Mar 2019.

Vancouver:

Stapel LC. The first cell fate specification event in mouse development. [Internet] [Masters thesis]. Universiteit Utrecht; 2011. [cited 2019 Mar 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/222737.

Council of Science Editors:

Stapel LC. The first cell fate specification event in mouse development. [Masters Thesis]. Universiteit Utrecht; 2011. Available from: http://dspace.library.uu.nl:8080/handle/1874/222737


University of Ottawa

9. Zhang, Yan. In vitro Functional Properties and In vivo Local Effects of Transplanted Human Progenitor Cells in Ischemic Tissues .

Degree: 2011, University of Ottawa

 Growing evidence from animal and clinical studies suggests that cardiac cell therapy can restore perfusion and improve function in the ischemic/infarcted myocardium. However, cell therapy… (more)

Subjects/Keywords: Stem cell therapy; Ischemic heart disease; Cell fate; Cell expansion; Positron emission tomography

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APA (6th Edition):

Zhang, Y. (2011). In vitro Functional Properties and In vivo Local Effects of Transplanted Human Progenitor Cells in Ischemic Tissues . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Yan. “In vitro Functional Properties and In vivo Local Effects of Transplanted Human Progenitor Cells in Ischemic Tissues .” 2011. Thesis, University of Ottawa. Accessed March 18, 2019. http://hdl.handle.net/10393/20216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Yan. “In vitro Functional Properties and In vivo Local Effects of Transplanted Human Progenitor Cells in Ischemic Tissues .” 2011. Web. 18 Mar 2019.

Vancouver:

Zhang Y. In vitro Functional Properties and In vivo Local Effects of Transplanted Human Progenitor Cells in Ischemic Tissues . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/10393/20216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang Y. In vitro Functional Properties and In vivo Local Effects of Transplanted Human Progenitor Cells in Ischemic Tissues . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/20216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

10. Stypulkowski, Ewa. Palmitoylation And Polarity: Regulation Of Asymmetric Partitioning Of Notch And Wnt Signaling By Reversible Lipid Modification In Dividing Cells.

Degree: 2018, University of Pennsylvania

 Protein palmitoylation is a reversible lipid modification that regulates protein-membrane interaction, activity, trafficking, and stability in a spatio-temporal manner similar to phosphorylation and ubiquitination. Asymmetric… (more)

Subjects/Keywords: Asymmetric cell division; Cell fate; Notch; Palmitoylation; Tumor heterogeneity; Wnt; Cell Biology

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APA (6th Edition):

Stypulkowski, E. (2018). Palmitoylation And Polarity: Regulation Of Asymmetric Partitioning Of Notch And Wnt Signaling By Reversible Lipid Modification In Dividing Cells. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/2821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stypulkowski, Ewa. “Palmitoylation And Polarity: Regulation Of Asymmetric Partitioning Of Notch And Wnt Signaling By Reversible Lipid Modification In Dividing Cells.” 2018. Thesis, University of Pennsylvania. Accessed March 18, 2019. https://repository.upenn.edu/edissertations/2821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stypulkowski, Ewa. “Palmitoylation And Polarity: Regulation Of Asymmetric Partitioning Of Notch And Wnt Signaling By Reversible Lipid Modification In Dividing Cells.” 2018. Web. 18 Mar 2019.

Vancouver:

Stypulkowski E. Palmitoylation And Polarity: Regulation Of Asymmetric Partitioning Of Notch And Wnt Signaling By Reversible Lipid Modification In Dividing Cells. [Internet] [Thesis]. University of Pennsylvania; 2018. [cited 2019 Mar 18]. Available from: https://repository.upenn.edu/edissertations/2821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stypulkowski E. Palmitoylation And Polarity: Regulation Of Asymmetric Partitioning Of Notch And Wnt Signaling By Reversible Lipid Modification In Dividing Cells. [Thesis]. University of Pennsylvania; 2018. Available from: https://repository.upenn.edu/edissertations/2821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

11. Soete, G.A.J. Cell fate determination in the Caenorhabditis elegans epidermal lineages.

Degree: 2007, Universiteit Utrecht

 The starting point for this work was to use the hypodermal seam of C. elegans as a model system to study cell fate determination. Even… (more)

Subjects/Keywords: Biologie; C. elegans; cell fate; patterning; chromatin remodeling; trithorax; cancer

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APA (6th Edition):

Soete, G. A. J. (2007). Cell fate determination in the Caenorhabditis elegans epidermal lineages. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/21400

Chicago Manual of Style (16th Edition):

Soete, G A J. “Cell fate determination in the Caenorhabditis elegans epidermal lineages.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed March 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/21400.

MLA Handbook (7th Edition):

Soete, G A J. “Cell fate determination in the Caenorhabditis elegans epidermal lineages.” 2007. Web. 18 Mar 2019.

Vancouver:

Soete GAJ. Cell fate determination in the Caenorhabditis elegans epidermal lineages. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2019 Mar 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/21400.

Council of Science Editors:

Soete GAJ. Cell fate determination in the Caenorhabditis elegans epidermal lineages. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/21400


Universiteit Utrecht

12. Long, Y. Transcription factor movement and tissue patterning in Arabidopsis root meristem.

Degree: 2015, Universiteit Utrecht

Cell-cell communication is key to coordinated cellular functions in multicellular organisms. In addition to the signaling molecules found in animals, plants also frequently recruit mobile… (more)

Subjects/Keywords: tissue boundary; cell fate; transcriptional network; proten interaction; protein movement

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APA (6th Edition):

Long, Y. (2015). Transcription factor movement and tissue patterning in Arabidopsis root meristem. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/320600

Chicago Manual of Style (16th Edition):

Long, Y. “Transcription factor movement and tissue patterning in Arabidopsis root meristem.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed March 18, 2019. http://dspace.library.uu.nl:8080/handle/1874/320600.

MLA Handbook (7th Edition):

Long, Y. “Transcription factor movement and tissue patterning in Arabidopsis root meristem.” 2015. Web. 18 Mar 2019.

Vancouver:

Long Y. Transcription factor movement and tissue patterning in Arabidopsis root meristem. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2019 Mar 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/320600.

Council of Science Editors:

Long Y. Transcription factor movement and tissue patterning in Arabidopsis root meristem. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/320600


University of Hawaii – Manoa

13. Yamaguchi, Emi. Cell fate specification and eye development in the polychaete capitella teleta.

Degree: 2016, University of Hawaii – Manoa

M.S. University of Hawaii at Manoa 2012.

A fundamental question of developmental biology is how an animal begins as an uncleaved zygote and ends up… (more)

Subjects/Keywords: capitella teleta; polychaete; cell fate specification; eye development

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APA (6th Edition):

Yamaguchi, E. (2016). Cell fate specification and eye development in the polychaete capitella teleta. (Thesis). University of Hawaii – Manoa. Retrieved from http://hdl.handle.net/10125/100789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yamaguchi, Emi. “Cell fate specification and eye development in the polychaete capitella teleta.” 2016. Thesis, University of Hawaii – Manoa. Accessed March 18, 2019. http://hdl.handle.net/10125/100789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yamaguchi, Emi. “Cell fate specification and eye development in the polychaete capitella teleta.” 2016. Web. 18 Mar 2019.

Vancouver:

Yamaguchi E. Cell fate specification and eye development in the polychaete capitella teleta. [Internet] [Thesis]. University of Hawaii – Manoa; 2016. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/10125/100789.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yamaguchi E. Cell fate specification and eye development in the polychaete capitella teleta. [Thesis]. University of Hawaii – Manoa; 2016. Available from: http://hdl.handle.net/10125/100789

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

14. Peissig, Kristen Beverly. Parathyroid cell fate instability:: an investigation of parathyroid cell cycle and order of fate switching.

Degree: MS, Genetics, 2017, University of Georgia

 The parathyroid glands are essential for regulating calcium homeostasis in the body. During mouse development, the parathyroid develops in the 3rd pharyngeal pouch along with… (more)

Subjects/Keywords: Parathyroid; 3rd pharyngeal pouch; cervical thymi; cell fate instability

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APA (6th Edition):

Peissig, K. B. (2017). Parathyroid cell fate instability:: an investigation of parathyroid cell cycle and order of fate switching. (Masters Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/38238

Chicago Manual of Style (16th Edition):

Peissig, Kristen Beverly. “Parathyroid cell fate instability:: an investigation of parathyroid cell cycle and order of fate switching.” 2017. Masters Thesis, University of Georgia. Accessed March 18, 2019. http://hdl.handle.net/10724/38238.

MLA Handbook (7th Edition):

Peissig, Kristen Beverly. “Parathyroid cell fate instability:: an investigation of parathyroid cell cycle and order of fate switching.” 2017. Web. 18 Mar 2019.

Vancouver:

Peissig KB. Parathyroid cell fate instability:: an investigation of parathyroid cell cycle and order of fate switching. [Internet] [Masters thesis]. University of Georgia; 2017. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/10724/38238.

Council of Science Editors:

Peissig KB. Parathyroid cell fate instability:: an investigation of parathyroid cell cycle and order of fate switching. [Masters Thesis]. University of Georgia; 2017. Available from: http://hdl.handle.net/10724/38238


University of Toronto

15. Kucharczuk, Aaron. Ectopic Notch1 Activation Alters Mammary Cell Fate During Puberty and Promotes the Development of Lactating Adenomas during Pregnancy.

Degree: 2009, University of Toronto

The role that each of the Notch receptors play in controlling alveolar development and cell fate determination in the mouse mammary gland has remained unclear.… (more)

Subjects/Keywords: Notch; Mammary Gland; Lactating Adenoma; ELF5+/ER- Cell Fate; 0369

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APA (6th Edition):

Kucharczuk, A. (2009). Ectopic Notch1 Activation Alters Mammary Cell Fate During Puberty and Promotes the Development of Lactating Adenomas during Pregnancy. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/18800

Chicago Manual of Style (16th Edition):

Kucharczuk, Aaron. “Ectopic Notch1 Activation Alters Mammary Cell Fate During Puberty and Promotes the Development of Lactating Adenomas during Pregnancy.” 2009. Masters Thesis, University of Toronto. Accessed March 18, 2019. http://hdl.handle.net/1807/18800.

MLA Handbook (7th Edition):

Kucharczuk, Aaron. “Ectopic Notch1 Activation Alters Mammary Cell Fate During Puberty and Promotes the Development of Lactating Adenomas during Pregnancy.” 2009. Web. 18 Mar 2019.

Vancouver:

Kucharczuk A. Ectopic Notch1 Activation Alters Mammary Cell Fate During Puberty and Promotes the Development of Lactating Adenomas during Pregnancy. [Internet] [Masters thesis]. University of Toronto; 2009. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1807/18800.

Council of Science Editors:

Kucharczuk A. Ectopic Notch1 Activation Alters Mammary Cell Fate During Puberty and Promotes the Development of Lactating Adenomas during Pregnancy. [Masters Thesis]. University of Toronto; 2009. Available from: http://hdl.handle.net/1807/18800


Washington University in St. Louis

16. Salazar, Valerie. Interactions between BMP and Canonical Wnt Signaling Regulate Critical Stages of the Osteoblast Lifecycle.

Degree: PhD, Biology and Biomedical Sciences: Molecular Cell Biology, 2011, Washington University in St. Louis

 Skeletal development and post-natal bone homeostasis are dependent on the coordinated activity of bone-forming cells called osteoblasts and bone-resorbing cells called osteoclasts. Over 10 million… (more)

Subjects/Keywords: Biology; apoptosis; BMP; cell fate; osteoblast; proliferation; Wnt

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APA (6th Edition):

Salazar, V. (2011). Interactions between BMP and Canonical Wnt Signaling Regulate Critical Stages of the Osteoblast Lifecycle. (Doctoral Dissertation). Washington University in St. Louis. Retrieved from https://openscholarship.wustl.edu/etd/637

Chicago Manual of Style (16th Edition):

Salazar, Valerie. “Interactions between BMP and Canonical Wnt Signaling Regulate Critical Stages of the Osteoblast Lifecycle.” 2011. Doctoral Dissertation, Washington University in St. Louis. Accessed March 18, 2019. https://openscholarship.wustl.edu/etd/637.

MLA Handbook (7th Edition):

Salazar, Valerie. “Interactions between BMP and Canonical Wnt Signaling Regulate Critical Stages of the Osteoblast Lifecycle.” 2011. Web. 18 Mar 2019.

Vancouver:

Salazar V. Interactions between BMP and Canonical Wnt Signaling Regulate Critical Stages of the Osteoblast Lifecycle. [Internet] [Doctoral dissertation]. Washington University in St. Louis; 2011. [cited 2019 Mar 18]. Available from: https://openscholarship.wustl.edu/etd/637.

Council of Science Editors:

Salazar V. Interactions between BMP and Canonical Wnt Signaling Regulate Critical Stages of the Osteoblast Lifecycle. [Doctoral Dissertation]. Washington University in St. Louis; 2011. Available from: https://openscholarship.wustl.edu/etd/637


Duke University

17. Jameson, Samantha Ann. Understanding Cell Fate Decisions in the Embryonic Gonad .

Degree: 2011, Duke University

  The divergence of distinct cell populations from multipotent progenitors is poorly understood, particularly in vivo. The gonad is an ideal place to study this… (more)

Subjects/Keywords: Cellular biology; cell fate; Fgf9; gonad; lineage priming; sex determination; Wnt4

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APA (6th Edition):

Jameson, S. A. (2011). Understanding Cell Fate Decisions in the Embryonic Gonad . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/5028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jameson, Samantha Ann. “Understanding Cell Fate Decisions in the Embryonic Gonad .” 2011. Thesis, Duke University. Accessed March 18, 2019. http://hdl.handle.net/10161/5028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jameson, Samantha Ann. “Understanding Cell Fate Decisions in the Embryonic Gonad .” 2011. Web. 18 Mar 2019.

Vancouver:

Jameson SA. Understanding Cell Fate Decisions in the Embryonic Gonad . [Internet] [Thesis]. Duke University; 2011. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/10161/5028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jameson SA. Understanding Cell Fate Decisions in the Embryonic Gonad . [Thesis]. Duke University; 2011. Available from: http://hdl.handle.net/10161/5028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oregon

18. Finley, Jennifer. Cell Fate Maintenance and Presynaptic Development in the Drosophila Eye.

Degree: 2013, University of Oregon

 Neurons in the central nervous system are typically not replaced and must therefore maintain their choice of fate and their synaptic connections throughout the life… (more)

Subjects/Keywords: Cell Fate; Drosophila; Polycomb Group; R7; Syd-1; Synaptic Targetting

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APA (6th Edition):

Finley, J. (2013). Cell Fate Maintenance and Presynaptic Development in the Drosophila Eye. (Thesis). University of Oregon. Retrieved from http://hdl.handle.net/1794/13254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Finley, Jennifer. “Cell Fate Maintenance and Presynaptic Development in the Drosophila Eye.” 2013. Thesis, University of Oregon. Accessed March 18, 2019. http://hdl.handle.net/1794/13254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Finley, Jennifer. “Cell Fate Maintenance and Presynaptic Development in the Drosophila Eye.” 2013. Web. 18 Mar 2019.

Vancouver:

Finley J. Cell Fate Maintenance and Presynaptic Development in the Drosophila Eye. [Internet] [Thesis]. University of Oregon; 2013. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1794/13254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Finley J. Cell Fate Maintenance and Presynaptic Development in the Drosophila Eye. [Thesis]. University of Oregon; 2013. Available from: http://hdl.handle.net/1794/13254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Illinois – Urbana-Champaign

19. Mahadik, Bhushan Prakash P. Multigradient hydrogels to decode extrinsic regulation of hematopoietic stem cell fate.

Degree: MS, 0300, 2011, University of Illinois – Urbana-Champaign

 Hematopoiesis is a physiological process responsible for the generation of all the blood and immune cells of the body. The cells responsible for this process… (more)

Subjects/Keywords: Biomaterial; gradient hydrogel; hematopoietic stem cells (HSCs); stem cell fate regulation

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APA (6th Edition):

Mahadik, B. P. P. (2011). Multigradient hydrogels to decode extrinsic regulation of hematopoietic stem cell fate. (Thesis). University of Illinois – Urbana-Champaign. Retrieved from http://hdl.handle.net/2142/18358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mahadik, Bhushan Prakash P. “Multigradient hydrogels to decode extrinsic regulation of hematopoietic stem cell fate.” 2011. Thesis, University of Illinois – Urbana-Champaign. Accessed March 18, 2019. http://hdl.handle.net/2142/18358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mahadik, Bhushan Prakash P. “Multigradient hydrogels to decode extrinsic regulation of hematopoietic stem cell fate.” 2011. Web. 18 Mar 2019.

Vancouver:

Mahadik BPP. Multigradient hydrogels to decode extrinsic regulation of hematopoietic stem cell fate. [Internet] [Thesis]. University of Illinois – Urbana-Champaign; 2011. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/2142/18358.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mahadik BPP. Multigradient hydrogels to decode extrinsic regulation of hematopoietic stem cell fate. [Thesis]. University of Illinois – Urbana-Champaign; 2011. Available from: http://hdl.handle.net/2142/18358

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Notre Dame

20. Christina N. Cheng. Analysis of the Genetic and Molecular Mechanisms That Regulate Renal Progenitor Cell Fate Specification</h1>.

Degree: PhD, Biological Sciences, 2017, University of Notre Dame

  Kidney disease is the 9th leading cause of death in the United States alone and can arise at any stage in life (American Kidney… (more)

Subjects/Keywords: irx2a; zebrafish; sim1a; development; kidney; cell fate specification

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APA (6th Edition):

Cheng, C. N. (2017). Analysis of the Genetic and Molecular Mechanisms That Regulate Renal Progenitor Cell Fate Specification</h1>. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/dn39x061p5k

Chicago Manual of Style (16th Edition):

Cheng, Christina N.. “Analysis of the Genetic and Molecular Mechanisms That Regulate Renal Progenitor Cell Fate Specification</h1>.” 2017. Doctoral Dissertation, University of Notre Dame. Accessed March 18, 2019. https://curate.nd.edu/show/dn39x061p5k.

MLA Handbook (7th Edition):

Cheng, Christina N.. “Analysis of the Genetic and Molecular Mechanisms That Regulate Renal Progenitor Cell Fate Specification</h1>.” 2017. Web. 18 Mar 2019.

Vancouver:

Cheng CN. Analysis of the Genetic and Molecular Mechanisms That Regulate Renal Progenitor Cell Fate Specification</h1>. [Internet] [Doctoral dissertation]. University of Notre Dame; 2017. [cited 2019 Mar 18]. Available from: https://curate.nd.edu/show/dn39x061p5k.

Council of Science Editors:

Cheng CN. Analysis of the Genetic and Molecular Mechanisms That Regulate Renal Progenitor Cell Fate Specification</h1>. [Doctoral Dissertation]. University of Notre Dame; 2017. Available from: https://curate.nd.edu/show/dn39x061p5k


University of Southern California

21. Hazen, Virginia M. The diverse roles of smads in dorsal spinal cord development.

Degree: PhD, Neuroscience, 2011, University of Southern California

 Bone Morphogenetic Proteins (BMPs) have disparate functions establishing neural circuitry in the dorsal spinal cord. BMPs first induce specific neuronal cell fates, including the most… (more)

Subjects/Keywords: axon guidance; cell fate; Smads; spinal cord; Bone Morphogenetic Proteins ❧

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APA (6th Edition):

Hazen, V. M. (2011). The diverse roles of smads in dorsal spinal cord development. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569

Chicago Manual of Style (16th Edition):

Hazen, Virginia M. “The diverse roles of smads in dorsal spinal cord development.” 2011. Doctoral Dissertation, University of Southern California. Accessed March 18, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569.

MLA Handbook (7th Edition):

Hazen, Virginia M. “The diverse roles of smads in dorsal spinal cord development.” 2011. Web. 18 Mar 2019.

Vancouver:

Hazen VM. The diverse roles of smads in dorsal spinal cord development. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2019 Mar 18]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569.

Council of Science Editors:

Hazen VM. The diverse roles of smads in dorsal spinal cord development. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/656050/rec/6569


University of Edinburgh

22. Laing, Adam. TLE proteins in mouse embryonic stem cell self renewal and early lineage specification.

Degree: PhD, 2011, University of Edinburgh

 TLE proteins are a closely related family of vertebrate corepressors. They have no intrinsic DNA binding ability, but are recruited as transcriptional repressors by other… (more)

Subjects/Keywords: 572.6; TLE proteins; early lineage specification; cell fate decisions; point mutations; ES cell differentiation

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APA (6th Edition):

Laing, A. (2011). TLE proteins in mouse embryonic stem cell self renewal and early lineage specification. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/5729

Chicago Manual of Style (16th Edition):

Laing, Adam. “TLE proteins in mouse embryonic stem cell self renewal and early lineage specification.” 2011. Doctoral Dissertation, University of Edinburgh. Accessed March 18, 2019. http://hdl.handle.net/1842/5729.

MLA Handbook (7th Edition):

Laing, Adam. “TLE proteins in mouse embryonic stem cell self renewal and early lineage specification.” 2011. Web. 18 Mar 2019.

Vancouver:

Laing A. TLE proteins in mouse embryonic stem cell self renewal and early lineage specification. [Internet] [Doctoral dissertation]. University of Edinburgh; 2011. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1842/5729.

Council of Science Editors:

Laing A. TLE proteins in mouse embryonic stem cell self renewal and early lineage specification. [Doctoral Dissertation]. University of Edinburgh; 2011. Available from: http://hdl.handle.net/1842/5729


University of Virginia

23. Dong, Han. The Function of CD27 Costimulation in the Activation and Fate Decisions of CD8+ T Cells.

Degree: PhD, 2015, University of Virginia

 CD8+ cytotoxic T lymphocytes are critical components of adaptive immunity against a variety of intracellular pathogens, and can play a key role in the control… (more)

Subjects/Keywords: CD27; inflammation; IL-12; type 1 interferon; CD8 T cell activation; CD8 T cell fate decisions; CD8 T cell memory

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APA (6th Edition):

Dong, H. (2015). The Function of CD27 Costimulation in the Activation and Fate Decisions of CD8+ T Cells. (Doctoral Dissertation). University of Virginia. Retrieved from http://libra.virginia.edu/catalog/libra-oa:8897

Chicago Manual of Style (16th Edition):

Dong, Han. “The Function of CD27 Costimulation in the Activation and Fate Decisions of CD8+ T Cells.” 2015. Doctoral Dissertation, University of Virginia. Accessed March 18, 2019. http://libra.virginia.edu/catalog/libra-oa:8897.

MLA Handbook (7th Edition):

Dong, Han. “The Function of CD27 Costimulation in the Activation and Fate Decisions of CD8+ T Cells.” 2015. Web. 18 Mar 2019.

Vancouver:

Dong H. The Function of CD27 Costimulation in the Activation and Fate Decisions of CD8+ T Cells. [Internet] [Doctoral dissertation]. University of Virginia; 2015. [cited 2019 Mar 18]. Available from: http://libra.virginia.edu/catalog/libra-oa:8897.

Council of Science Editors:

Dong H. The Function of CD27 Costimulation in the Activation and Fate Decisions of CD8+ T Cells. [Doctoral Dissertation]. University of Virginia; 2015. Available from: http://libra.virginia.edu/catalog/libra-oa:8897


University of Vienna

24. Göppert, Anne. Mechanism of asymmetric PIE-1 segregation in C. elegans one-cell embryo.

Degree: 2010, University of Vienna

Die Zelldifferenzierungsdeterminante PIE-1 bestimmt die Entwicklung und Identität der Keimbahn-Stammzellen in C. elegans, in dem die Transkription der Stammzellen inhibiert wird. PIE-1 akkumuliert in den… (more)

Subjects/Keywords: 42.13 Molekularbiologie; 42.23 Entwicklungsbiologie; Asymmetrische Zellteilung / Zellpolarität / C. elegans; asymmetric cell division / cell polarity / C. elegans / cell fate determinant gradient

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APA (6th Edition):

Göppert, A. (2010). Mechanism of asymmetric PIE-1 segregation in C. elegans one-cell embryo. (Thesis). University of Vienna. Retrieved from http://othes.univie.ac.at/10073/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Göppert, Anne. “Mechanism of asymmetric PIE-1 segregation in C. elegans one-cell embryo.” 2010. Thesis, University of Vienna. Accessed March 18, 2019. http://othes.univie.ac.at/10073/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Göppert, Anne. “Mechanism of asymmetric PIE-1 segregation in C. elegans one-cell embryo.” 2010. Web. 18 Mar 2019.

Vancouver:

Göppert A. Mechanism of asymmetric PIE-1 segregation in C. elegans one-cell embryo. [Internet] [Thesis]. University of Vienna; 2010. [cited 2019 Mar 18]. Available from: http://othes.univie.ac.at/10073/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Göppert A. Mechanism of asymmetric PIE-1 segregation in C. elegans one-cell embryo. [Thesis]. University of Vienna; 2010. Available from: http://othes.univie.ac.at/10073/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

25. Edwards, Nicole A. The Role of p66Shc in Mouse Blastocyst Development.

Degree: 2018, University of Western Ontario

 The earliest cell fate specification events during mammalian development occur in the blastocyst-stage preimplantation embryo, during which a pluripotent cell population is established. These cells… (more)

Subjects/Keywords: Blastocyst; preimplantation embryo; embryonic stem cells; cell fate; pluripotency; p66Shc; Developmental Biology

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APA (6th Edition):

Edwards, N. A. (2018). The Role of p66Shc in Mouse Blastocyst Development. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Edwards, Nicole A. “The Role of p66Shc in Mouse Blastocyst Development.” 2018. Thesis, University of Western Ontario. Accessed March 18, 2019. https://ir.lib.uwo.ca/etd/5403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Edwards, Nicole A. “The Role of p66Shc in Mouse Blastocyst Development.” 2018. Web. 18 Mar 2019.

Vancouver:

Edwards NA. The Role of p66Shc in Mouse Blastocyst Development. [Internet] [Thesis]. University of Western Ontario; 2018. [cited 2019 Mar 18]. Available from: https://ir.lib.uwo.ca/etd/5403.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Edwards NA. The Role of p66Shc in Mouse Blastocyst Development. [Thesis]. University of Western Ontario; 2018. Available from: https://ir.lib.uwo.ca/etd/5403

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. OGUTCEN, EZGI. Repression of the blood endothelial marker CD146 by the homeobox gene PROX1.

Degree: Biochemistry and Medical Genetics, 2010, University of Manitoba

 CD146 is a cell adhesion molecule that has been shown to regulate cell adhesion, migration and proliferation of different cell types. It is highly expressed… (more)

Subjects/Keywords: lymphatic differentiation; endothelial cell fate

…Another
aspect
of
Prox1
during
lymphatic
development
is
its
role
as
a
 cellfate
switch
(… …addition
 to
 its
 roles
 during
 cardiovascular
 development
 and
 cellfate
 determination… …Ampicillin
 Antennapedia
 Ammonium
Persulfate
 Blood
Endothelial
CellCell
Adhesion
molecule… …Dithiothreitol
 Embryonic
day
8
 Endothelial
Cell
Basal
Medium‐2
 Endothelial
Cell
 Extracellular… …Matrix
 Ethylenediaminetetraacetic
acid
 Enhanced
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Protein
 Endothelial
Cell… 

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APA (6th Edition):

OGUTCEN, E. (2010). Repression of the blood endothelial marker CD146 by the homeobox gene PROX1. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4038

Chicago Manual of Style (16th Edition):

OGUTCEN, EZGI. “Repression of the blood endothelial marker CD146 by the homeobox gene PROX1.” 2010. Masters Thesis, University of Manitoba. Accessed March 18, 2019. http://hdl.handle.net/1993/4038.

MLA Handbook (7th Edition):

OGUTCEN, EZGI. “Repression of the blood endothelial marker CD146 by the homeobox gene PROX1.” 2010. Web. 18 Mar 2019.

Vancouver:

OGUTCEN E. Repression of the blood endothelial marker CD146 by the homeobox gene PROX1. [Internet] [Masters thesis]. University of Manitoba; 2010. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1993/4038.

Council of Science Editors:

OGUTCEN E. Repression of the blood endothelial marker CD146 by the homeobox gene PROX1. [Masters Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/4038


University of Washington

27. Huang, Albert Chih-Pei. Two perspectives of cell fate determination: Systems dynamics and molecular mechanisms.

Degree: PhD, 2012, University of Washington

Cell fate determination, the process of transforming a unicellular embryonic cell into lineage-restricted specific tissues of a multicellular organism, is fundamental to the process of… (more)

Subjects/Keywords: attractor; cell fate; molecular mechanism; systems dynamics; T-bet; Molecular and cellular biology

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APA (6th Edition):

Huang, A. C. (2012). Two perspectives of cell fate determination: Systems dynamics and molecular mechanisms. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/19741

Chicago Manual of Style (16th Edition):

Huang, Albert Chih-Pei. “Two perspectives of cell fate determination: Systems dynamics and molecular mechanisms.” 2012. Doctoral Dissertation, University of Washington. Accessed March 18, 2019. http://hdl.handle.net/1773/19741.

MLA Handbook (7th Edition):

Huang, Albert Chih-Pei. “Two perspectives of cell fate determination: Systems dynamics and molecular mechanisms.” 2012. Web. 18 Mar 2019.

Vancouver:

Huang AC. Two perspectives of cell fate determination: Systems dynamics and molecular mechanisms. [Internet] [Doctoral dissertation]. University of Washington; 2012. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1773/19741.

Council of Science Editors:

Huang AC. Two perspectives of cell fate determination: Systems dynamics and molecular mechanisms. [Doctoral Dissertation]. University of Washington; 2012. Available from: http://hdl.handle.net/1773/19741


University of Cambridge

28. Johnson, Jo-Anne. Investigating factors governing cell fate decisions in respiratory epithelium.

Degree: PhD, 2018, University of Cambridge

 The maintenance of the airway/respiratory epithelium during adult homeostasis and repair and its construction during embryonic development require tightly regulated cell fate decisions. This regulation… (more)

Subjects/Keywords: respiratory epithelium; cilia; ciliogenesis; multiciliogenesis; human lung development; cell fate; Fank1; Jazf1; human lung organoids

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APA (6th Edition):

Johnson, J. (2018). Investigating factors governing cell fate decisions in respiratory epithelium. (Doctoral Dissertation). University of Cambridge. Retrieved from https://www.repository.cam.ac.uk/handle/1810/278966 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753423

Chicago Manual of Style (16th Edition):

Johnson, Jo-Anne. “Investigating factors governing cell fate decisions in respiratory epithelium.” 2018. Doctoral Dissertation, University of Cambridge. Accessed March 18, 2019. https://www.repository.cam.ac.uk/handle/1810/278966 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753423.

MLA Handbook (7th Edition):

Johnson, Jo-Anne. “Investigating factors governing cell fate decisions in respiratory epithelium.” 2018. Web. 18 Mar 2019.

Vancouver:

Johnson J. Investigating factors governing cell fate decisions in respiratory epithelium. [Internet] [Doctoral dissertation]. University of Cambridge; 2018. [cited 2019 Mar 18]. Available from: https://www.repository.cam.ac.uk/handle/1810/278966 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753423.

Council of Science Editors:

Johnson J. Investigating factors governing cell fate decisions in respiratory epithelium. [Doctoral Dissertation]. University of Cambridge; 2018. Available from: https://www.repository.cam.ac.uk/handle/1810/278966 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.753423


University of Toronto

29. Han, Hong. High-throughput Analysis of Alternative Splicing Regulatory Networks that Control Cell Fate.

Degree: PhD, 2016, University of Toronto

Advances in high-throughput profiling technologies have revolutionized our view of transcriptome complexity and regulation. Alternative splicing (AS) is increasingly recognized as a widespread mechanism for… (more)

Subjects/Keywords: alternative splicing; cell fate control; gene regulation; high-throughput screening; RNA biology; 0307

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APA (6th Edition):

Han, H. (2016). High-throughput Analysis of Alternative Splicing Regulatory Networks that Control Cell Fate. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/80386

Chicago Manual of Style (16th Edition):

Han, Hong. “High-throughput Analysis of Alternative Splicing Regulatory Networks that Control Cell Fate.” 2016. Doctoral Dissertation, University of Toronto. Accessed March 18, 2019. http://hdl.handle.net/1807/80386.

MLA Handbook (7th Edition):

Han, Hong. “High-throughput Analysis of Alternative Splicing Regulatory Networks that Control Cell Fate.” 2016. Web. 18 Mar 2019.

Vancouver:

Han H. High-throughput Analysis of Alternative Splicing Regulatory Networks that Control Cell Fate. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1807/80386.

Council of Science Editors:

Han H. High-throughput Analysis of Alternative Splicing Regulatory Networks that Control Cell Fate. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/80386


Rice University

30. Jolly, Mohit Kumar. The hybrid epithelial/mesenchymal phenotype and its implications in cancer metastasis.

Degree: PhD, Engineering, 2016, Rice University

 More than 90% of cancer-related deaths occur because cancer cells metastasize, i.e. invade the surrounding tissue, travel throughout the body, and form tumors at distant… (more)

Subjects/Keywords: Cancer metastasis; Epithelial-mesenchymal transition; Multistability; Systems Biology; Cell-fate decisons; Hybrid epithelial/mesenchymal phenotype

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APA (6th Edition):

Jolly, M. K. (2016). The hybrid epithelial/mesenchymal phenotype and its implications in cancer metastasis. (Doctoral Dissertation). Rice University. Retrieved from http://hdl.handle.net/1911/95959

Chicago Manual of Style (16th Edition):

Jolly, Mohit Kumar. “The hybrid epithelial/mesenchymal phenotype and its implications in cancer metastasis.” 2016. Doctoral Dissertation, Rice University. Accessed March 18, 2019. http://hdl.handle.net/1911/95959.

MLA Handbook (7th Edition):

Jolly, Mohit Kumar. “The hybrid epithelial/mesenchymal phenotype and its implications in cancer metastasis.” 2016. Web. 18 Mar 2019.

Vancouver:

Jolly MK. The hybrid epithelial/mesenchymal phenotype and its implications in cancer metastasis. [Internet] [Doctoral dissertation]. Rice University; 2016. [cited 2019 Mar 18]. Available from: http://hdl.handle.net/1911/95959.

Council of Science Editors:

Jolly MK. The hybrid epithelial/mesenchymal phenotype and its implications in cancer metastasis. [Doctoral Dissertation]. Rice University; 2016. Available from: http://hdl.handle.net/1911/95959

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