Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Cell death). Showing records 1 – 30 of 899 total matches.

[1] [2] [3] [4] [5] … [30]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


University of Rochester

1. Hwangbo, Dae-Sung. Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster.

Degree: PhD, 2012, University of Rochester

 Apoptosis and immune responses are crucial for tissue and organismal homeostasis in metazoans. Loss of proper regulation of these processes can cause serious defects in… (more)

Subjects/Keywords: Cell Death; WD40

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hwangbo, D. (2012). Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/25510

Chicago Manual of Style (16th Edition):

Hwangbo, Dae-Sung. “Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster.” 2012. Doctoral Dissertation, University of Rochester. Accessed October 19, 2020. http://hdl.handle.net/1802/25510.

MLA Handbook (7th Edition):

Hwangbo, Dae-Sung. “Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster.” 2012. Web. 19 Oct 2020.

Vancouver:

Hwangbo D. Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster. [Internet] [Doctoral dissertation]. University of Rochester; 2012. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1802/25510.

Council of Science Editors:

Hwangbo D. Regulation of Apoptosis, Innate Immunity and Longevity by dWDR40 in Drosophila melanogaster. [Doctoral Dissertation]. University of Rochester; 2012. Available from: http://hdl.handle.net/1802/25510


University of Rochester

2. Fornarola, Laura Beth. The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation.

Degree: PhD, 2014, University of Rochester

 In the developing nervous system, programmed cell death (PCD) is critical for shaping and refining appropriate neuronal connections present in adulthood. Although PCD is regulated… (more)

Subjects/Keywords: Programmed cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fornarola, L. B. (2014). The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/28436

Chicago Manual of Style (16th Edition):

Fornarola, Laura Beth. “The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation.” 2014. Doctoral Dissertation, University of Rochester. Accessed October 19, 2020. http://hdl.handle.net/1802/28436.

MLA Handbook (7th Edition):

Fornarola, Laura Beth. “The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation.” 2014. Web. 19 Oct 2020.

Vancouver:

Fornarola LB. The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation. [Internet] [Doctoral dissertation]. University of Rochester; 2014. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1802/28436.

Council of Science Editors:

Fornarola LB. The Role of PIN1 in neuronal programmed cell death following trophic factor deprivation. [Doctoral Dissertation]. University of Rochester; 2014. Available from: http://hdl.handle.net/1802/28436


University of Waterloo

3. Chong, Stacey. Cell death and proliferation characteristics of the retina after optic nerve section in chickens.

Degree: 2013, University of Waterloo

 Optic nerve section (ONS) is an experimental model for damage of the optic nerve associated with diseases such as glaucoma and optic neuritis. Damage to… (more)

Subjects/Keywords: proliferation; cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chong, S. (2013). Cell death and proliferation characteristics of the retina after optic nerve section in chickens. (Thesis). University of Waterloo. Retrieved from http://hdl.handle.net/10012/7959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chong, Stacey. “Cell death and proliferation characteristics of the retina after optic nerve section in chickens.” 2013. Thesis, University of Waterloo. Accessed October 19, 2020. http://hdl.handle.net/10012/7959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chong, Stacey. “Cell death and proliferation characteristics of the retina after optic nerve section in chickens.” 2013. Web. 19 Oct 2020.

Vancouver:

Chong S. Cell death and proliferation characteristics of the retina after optic nerve section in chickens. [Internet] [Thesis]. University of Waterloo; 2013. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/10012/7959.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chong S. Cell death and proliferation characteristics of the retina after optic nerve section in chickens. [Thesis]. University of Waterloo; 2013. Available from: http://hdl.handle.net/10012/7959

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

4. Park, Danielle. The noninvasive imaging of cell death using a Hsp90 ligand.

Degree: Prince of Wales Medical Research Institute, 2012, University of New South Wales

Cell death plays an integral role in physiology, including turnover of cells in the gastrointestinal tract, the menstrual cycle and the immune system. Imbalance of… (more)

Subjects/Keywords: Cell death; Imaging

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Park, D. (2012). The noninvasive imaging of cell death using a Hsp90 ligand. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true

Chicago Manual of Style (16th Edition):

Park, Danielle. “The noninvasive imaging of cell death using a Hsp90 ligand.” 2012. Doctoral Dissertation, University of New South Wales. Accessed October 19, 2020. http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true.

MLA Handbook (7th Edition):

Park, Danielle. “The noninvasive imaging of cell death using a Hsp90 ligand.” 2012. Web. 19 Oct 2020.

Vancouver:

Park D. The noninvasive imaging of cell death using a Hsp90 ligand. [Internet] [Doctoral dissertation]. University of New South Wales; 2012. [cited 2020 Oct 19]. Available from: http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true.

Council of Science Editors:

Park D. The noninvasive imaging of cell death using a Hsp90 ligand. [Doctoral Dissertation]. University of New South Wales; 2012. Available from: http://handle.unsw.edu.au/1959.4/51998 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:10668/SOURCE01?view=true


Hong Kong University of Science and Technology

5. Tang, Rui LIFS. Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death.

Degree: 2016, Hong Kong University of Science and Technology

 Anti-microtubule agents activate the spindle-assembly checkpoint by perturbing spindle formation and trapping cells in mitosis. Whether cells undergo mitotic cell death subsequently is an important… (more)

Subjects/Keywords: Cell cycle ; Mitosis ; Cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tang, R. L. (2016). Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tang, Rui LIFS. “Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death.” 2016. Thesis, Hong Kong University of Science and Technology. Accessed October 19, 2020. http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tang, Rui LIFS. “Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death.” 2016. Web. 19 Oct 2020.

Vancouver:

Tang RL. Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2016. [cited 2020 Oct 19]. Available from: http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tang RL. Anti-apoptotic functions of BCL-W, MCL-1, and A1 in mitotic cell death. [Thesis]. Hong Kong University of Science and Technology; 2016. Available from: http://repository.ust.hk/ir/Record/1783.1-100402 ; https://doi.org/10.14711/thesis-b1626285 ; http://repository.ust.hk/ir/bitstream/1783.1-100402/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

6. Wang, Lei. Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila.

Degree: PhD, Human Genetics, 2010, University of Utah

 The steroid hormone ecdysone triggers the stage-specific destruction of Drosophila larval tissues through transcriptional cascades that induce two distinct forms of programmed cell death: caspase-dependent… (more)

Subjects/Keywords: Drosophila; Cell Death; Steroids

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, L. (2010). Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526

Chicago Manual of Style (16th Edition):

Wang, Lei. “Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila.” 2010. Doctoral Dissertation, University of Utah. Accessed October 19, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526.

MLA Handbook (7th Edition):

Wang, Lei. “Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila.” 2010. Web. 19 Oct 2020.

Vancouver:

Wang L. Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Oct 19]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526.

Council of Science Editors:

Wang L. Genetic Screen and Functional Analysis of Steroid-Triggered Cell Death in Drosophila. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/137/rec/526

7. Purushothaman, Divya. Regulation of activated T cell death; -.

Degree: Chemical and Biotechnology, 2014, SASTRA University

Abstract included

Reference p116-134, List of publications p114-115, List of abbreviations p135-137, Summary included

Advisors/Committee Members: Sarin, Apurva.

Subjects/Keywords: Immune System; T cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Purushothaman, D. (2014). Regulation of activated T cell death; -. (Thesis). SASTRA University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/22846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Purushothaman, Divya. “Regulation of activated T cell death; -.” 2014. Thesis, SASTRA University. Accessed October 19, 2020. http://shodhganga.inflibnet.ac.in/handle/10603/22846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Purushothaman, Divya. “Regulation of activated T cell death; -.” 2014. Web. 19 Oct 2020.

Vancouver:

Purushothaman D. Regulation of activated T cell death; -. [Internet] [Thesis]. SASTRA University; 2014. [cited 2020 Oct 19]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/22846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Purushothaman D. Regulation of activated T cell death; -. [Thesis]. SASTRA University; 2014. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/22846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Saskatchewan

8. Bridge, Rylan. INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS.

Degree: 2016, University of Saskatchewan

 Cells of the Monocyte / Macrophage lineage are key players in innate and adaptive immunity. They eliminate pathogens through their phagocytic and antimicrobial properties, secretion… (more)

Subjects/Keywords: autophagy; cell death; kinome analysis

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bridge, R. (2016). INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS. (Thesis). University of Saskatchewan. Retrieved from http://hdl.handle.net/10388/7628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bridge, Rylan. “INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS.” 2016. Thesis, University of Saskatchewan. Accessed October 19, 2020. http://hdl.handle.net/10388/7628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bridge, Rylan. “INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS.” 2016. Web. 19 Oct 2020.

Vancouver:

Bridge R. INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS. [Internet] [Thesis]. University of Saskatchewan; 2016. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/10388/7628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bridge R. INVESTIGATING THE ROLE OF CELLULAR AUTOPHAGY IN HUMAN MONOCYTIC CELL DEATH BY KINOME ANALYSIS. [Thesis]. University of Saskatchewan; 2016. Available from: http://hdl.handle.net/10388/7628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Edinburgh

9. Paterson, Scott Ian. Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention.

Degree: PhD, 2016, University of Edinburgh

 During open orthopaedic surgical procedures, the articular cartilage covering the exposed joint surfaces can be exposed to air for prolonged periods. This exposure facilitates cartilage… (more)

Subjects/Keywords: 616.7; cartilage; drying; cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Paterson, S. I. (2016). Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/23386

Chicago Manual of Style (16th Edition):

Paterson, Scott Ian. “Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention.” 2016. Doctoral Dissertation, University of Edinburgh. Accessed October 19, 2020. http://hdl.handle.net/1842/23386.

MLA Handbook (7th Edition):

Paterson, Scott Ian. “Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention.” 2016. Web. 19 Oct 2020.

Vancouver:

Paterson SI. Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention. [Internet] [Doctoral dissertation]. University of Edinburgh; 2016. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1842/23386.

Council of Science Editors:

Paterson SI. Chondrocyte death during articular cartilage drying : an investigation of its effect, mechanism, and prevention. [Doctoral Dissertation]. University of Edinburgh; 2016. Available from: http://hdl.handle.net/1842/23386


University of Manitoba

10. Roveimiab, Zeinab. p53 mediates cell death in the heart.

Degree: Physiology and Pathophysiology, 2019, University of Manitoba

 During cellular stress, cells try to survive and overcome the stress by an essential process called autophagy. This process involves degrading and recycling organelles and… (more)

Subjects/Keywords: p53; Cell death; Heart; Autophagy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Roveimiab, Z. (2019). p53 mediates cell death in the heart. (Masters Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/34460

Chicago Manual of Style (16th Edition):

Roveimiab, Zeinab. “p53 mediates cell death in the heart.” 2019. Masters Thesis, University of Manitoba. Accessed October 19, 2020. http://hdl.handle.net/1993/34460.

MLA Handbook (7th Edition):

Roveimiab, Zeinab. “p53 mediates cell death in the heart.” 2019. Web. 19 Oct 2020.

Vancouver:

Roveimiab Z. p53 mediates cell death in the heart. [Internet] [Masters thesis]. University of Manitoba; 2019. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1993/34460.

Council of Science Editors:

Roveimiab Z. p53 mediates cell death in the heart. [Masters Thesis]. University of Manitoba; 2019. Available from: http://hdl.handle.net/1993/34460


University of Victoria

11. Nelson, Kimberlea Lynne. Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas.

Degree: Department of Biochemistry and Microbiology, 2018, University of Victoria

 Aerolysin is a channel-forming protein toxin secreted by virulent Aeromonas species. The toxin binds to receptors on cells, is proteolytically activated, and then assembles into… (more)

Subjects/Keywords: Aerolysin; Bacterial toxins; Cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nelson, K. L. (2018). Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas. (Thesis). University of Victoria. Retrieved from https://dspace.library.uvic.ca//handle/1828/9673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nelson, Kimberlea Lynne. “Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas.” 2018. Thesis, University of Victoria. Accessed October 19, 2020. https://dspace.library.uvic.ca//handle/1828/9673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nelson, Kimberlea Lynne. “Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas.” 2018. Web. 19 Oct 2020.

Vancouver:

Nelson KL. Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas. [Internet] [Thesis]. University of Victoria; 2018. [cited 2020 Oct 19]. Available from: https://dspace.library.uvic.ca//handle/1828/9673.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nelson KL. Elucidation of the mode of action of the pore-forming toxin aerolysin on T lymphomas. [Thesis]. University of Victoria; 2018. Available from: https://dspace.library.uvic.ca//handle/1828/9673

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


National University of Ireland – Galway

12. Pakos-Zebrucka, Karolina. Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells .

Degree: 2017, National University of Ireland – Galway

 Chronic or unresolved stress can lead to cell death, and induction of apoptosis is crucial when the cellular adaptive mechanisms are not able to resolve… (more)

Subjects/Keywords: Autophagy; Cell death; Cell stress; Apoptosis; Biochemistry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pakos-Zebrucka, K. (2017). Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells . (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/7114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pakos-Zebrucka, Karolina. “Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells .” 2017. Thesis, National University of Ireland – Galway. Accessed October 19, 2020. http://hdl.handle.net/10379/7114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pakos-Zebrucka, Karolina. “Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells .” 2017. Web. 19 Oct 2020.

Vancouver:

Pakos-Zebrucka K. Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells . [Internet] [Thesis]. National University of Ireland – Galway; 2017. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/10379/7114.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pakos-Zebrucka K. Cellular response to stress: role of autophagy and cell death in caspase-9 deficient cells . [Thesis]. National University of Ireland – Galway; 2017. Available from: http://hdl.handle.net/10379/7114

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Notre Dame

13. Randy Jeffrey. Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>.

Degree: Biological Sciences, 2012, University of Notre Dame

  The use of anti-androgen therapy for the treatment of invasive and metastatic prostate cancer is common practice. However, after an initial response, the tumor… (more)

Subjects/Keywords: cell death; prostate cancer; cell cycle; bicalutamide

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jeffrey, R. (2012). Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>. (Thesis). University of Notre Dame. Retrieved from https://curate.nd.edu/show/qf85n873c5q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jeffrey, Randy. “Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>.” 2012. Thesis, University of Notre Dame. Accessed October 19, 2020. https://curate.nd.edu/show/qf85n873c5q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jeffrey, Randy. “Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>.” 2012. Web. 19 Oct 2020.

Vancouver:

Jeffrey R. Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>. [Internet] [Thesis]. University of Notre Dame; 2012. [cited 2020 Oct 19]. Available from: https://curate.nd.edu/show/qf85n873c5q.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jeffrey R. Effects of the Anti-Androgen Bicalutamide on Prostate Cancer Cells</h1>. [Thesis]. University of Notre Dame; 2012. Available from: https://curate.nd.edu/show/qf85n873c5q

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Yang, Chenying. Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death.

Degree: 2017, University of Melbourne

 Salmonella enterica is a Gram-negative intracellular pathogen, which can cause typhoid fever and non-typhoidal salmonellosis. Every year ~22 million cases and ~200,000 deaths are reported… (more)

Subjects/Keywords: Salmonella; dendritic cell; cell death; IFN-γ

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, C. (2017). Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death. (Masters Thesis). University of Melbourne. Retrieved from http://hdl.handle.net/11343/194652

Chicago Manual of Style (16th Edition):

Yang, Chenying. “Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death.” 2017. Masters Thesis, University of Melbourne. Accessed October 19, 2020. http://hdl.handle.net/11343/194652.

MLA Handbook (7th Edition):

Yang, Chenying. “Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death.” 2017. Web. 19 Oct 2020.

Vancouver:

Yang C. Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death. [Internet] [Masters thesis]. University of Melbourne; 2017. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/11343/194652.

Council of Science Editors:

Yang C. Immunological checkpoints in the control of murine Salmonella enterica infection: IFN-γ pathways and early dendritic cell death. [Masters Thesis]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/194652


Texas Medical Center

15. White, Erin. Mechanisms of Adenovirus-Mediated Autophagy.

Degree: PhD, 2011, Texas Medical Center

  A patient diagnosed with a glioma, generally, has an average of 14 months year to live after implementation of conventional therapies such as surgery,… (more)

Subjects/Keywords: Adenovirus; Autophagy; Autophagic Cell Death; GCN2; Cell Death; Cancer Biology; Cell Biology; Virology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

White, E. (2011). Mechanisms of Adenovirus-Mediated Autophagy. (Doctoral Dissertation). Texas Medical Center. Retrieved from https://digitalcommons.library.tmc.edu/utgsbs_dissertations/180

Chicago Manual of Style (16th Edition):

White, Erin. “Mechanisms of Adenovirus-Mediated Autophagy.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed October 19, 2020. https://digitalcommons.library.tmc.edu/utgsbs_dissertations/180.

MLA Handbook (7th Edition):

White, Erin. “Mechanisms of Adenovirus-Mediated Autophagy.” 2011. Web. 19 Oct 2020.

Vancouver:

White E. Mechanisms of Adenovirus-Mediated Autophagy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2020 Oct 19]. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/180.

Council of Science Editors:

White E. Mechanisms of Adenovirus-Mediated Autophagy. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: https://digitalcommons.library.tmc.edu/utgsbs_dissertations/180


Vanderbilt University

16. Carboneau, Bethany Ann. Regulation of Beta-Cell Mass Expansion by Prostaglandin E2 Signaling.

Degree: PhD, Molecular Physiology and Biophysics, 2017, Vanderbilt University

 Type 2 diabetes is a major healthcare concern and is characterized by chronic hyperglycemia and low-grade inflammation. Hyperglycemia and systemic inflammation can induce the production… (more)

Subjects/Keywords: pancreatic beta cell; prostaglandins; proliferation; cell death; beta-cell mass

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carboneau, B. A. (2017). Regulation of Beta-Cell Mass Expansion by Prostaglandin E2 Signaling. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13906

Chicago Manual of Style (16th Edition):

Carboneau, Bethany Ann. “Regulation of Beta-Cell Mass Expansion by Prostaglandin E2 Signaling.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed October 19, 2020. http://hdl.handle.net/1803/13906.

MLA Handbook (7th Edition):

Carboneau, Bethany Ann. “Regulation of Beta-Cell Mass Expansion by Prostaglandin E2 Signaling.” 2017. Web. 19 Oct 2020.

Vancouver:

Carboneau BA. Regulation of Beta-Cell Mass Expansion by Prostaglandin E2 Signaling. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1803/13906.

Council of Science Editors:

Carboneau BA. Regulation of Beta-Cell Mass Expansion by Prostaglandin E2 Signaling. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/13906


University of Delaware

17. Pokrzywinski, Kaytee. Characterization of bacterial algicide IRI-160AA: insights on programmed death pathways and organelle impacts in harmful dinoflagellates.

Degree: PhD, University of Delaware, School of Marine Science and Policy, 2014, University of Delaware

 With increases in anthropogenic eutrophication, phytoplankton assemblages are shifting from healthy communities dominated by diatoms to communities dominated by flagellated phytoplankton. Among these are the… (more)

Subjects/Keywords: Algicides.; Dinoflagellates.; Cell death.; Cell nuclei.; Cell organelles.

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pokrzywinski, K. (2014). Characterization of bacterial algicide IRI-160AA: insights on programmed death pathways and organelle impacts in harmful dinoflagellates. (Doctoral Dissertation). University of Delaware. Retrieved from http://udspace.udel.edu/handle/19716/16775

Chicago Manual of Style (16th Edition):

Pokrzywinski, Kaytee. “Characterization of bacterial algicide IRI-160AA: insights on programmed death pathways and organelle impacts in harmful dinoflagellates.” 2014. Doctoral Dissertation, University of Delaware. Accessed October 19, 2020. http://udspace.udel.edu/handle/19716/16775.

MLA Handbook (7th Edition):

Pokrzywinski, Kaytee. “Characterization of bacterial algicide IRI-160AA: insights on programmed death pathways and organelle impacts in harmful dinoflagellates.” 2014. Web. 19 Oct 2020.

Vancouver:

Pokrzywinski K. Characterization of bacterial algicide IRI-160AA: insights on programmed death pathways and organelle impacts in harmful dinoflagellates. [Internet] [Doctoral dissertation]. University of Delaware; 2014. [cited 2020 Oct 19]. Available from: http://udspace.udel.edu/handle/19716/16775.

Council of Science Editors:

Pokrzywinski K. Characterization of bacterial algicide IRI-160AA: insights on programmed death pathways and organelle impacts in harmful dinoflagellates. [Doctoral Dissertation]. University of Delaware; 2014. Available from: http://udspace.udel.edu/handle/19716/16775


University of Rochester

18. Gundemir, Soner. Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress.

Degree: PhD, 2011, University of Rochester

 Transglutaminase 2 (TG2) is a multifunctional enzyme that has guanine nucleotide binding and GTP hydrolyzing activity in addition to its transamidating function. Studies show that… (more)

Subjects/Keywords: Hypoxior; Transglutaminase 2; Cell Death; Ischemia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gundemir, S. (2011). Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/14598

Chicago Manual of Style (16th Edition):

Gundemir, Soner. “Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress.” 2011. Doctoral Dissertation, University of Rochester. Accessed October 19, 2020. http://hdl.handle.net/1802/14598.

MLA Handbook (7th Edition):

Gundemir, Soner. “Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress.” 2011. Web. 19 Oct 2020.

Vancouver:

Gundemir S. Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress. [Internet] [Doctoral dissertation]. University of Rochester; 2011. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1802/14598.

Council of Science Editors:

Gundemir S. Elucidating the Role of Transglutaminase 2 in Cellular Response to Hypoxic Stress. [Doctoral Dissertation]. University of Rochester; 2011. Available from: http://hdl.handle.net/1802/14598


Columbia University

19. Venkatesh, Divya. Elucidating the abilities of MDM2, MDMX and p21 to regulate ferroptosis.

Degree: 2020, Columbia University

 In this thesis, I have explored the role of three genes related to p53, namely p21, MDM2 and MDMX, in regulating ferroptosis, a form of… (more)

Subjects/Keywords: Biology; Genetics; Lipids – Peroxidation; Cell death; Cancer

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Venkatesh, D. (2020). Elucidating the abilities of MDM2, MDMX and p21 to regulate ferroptosis. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-es1c-z783

Chicago Manual of Style (16th Edition):

Venkatesh, Divya. “Elucidating the abilities of MDM2, MDMX and p21 to regulate ferroptosis.” 2020. Doctoral Dissertation, Columbia University. Accessed October 19, 2020. https://doi.org/10.7916/d8-es1c-z783.

MLA Handbook (7th Edition):

Venkatesh, Divya. “Elucidating the abilities of MDM2, MDMX and p21 to regulate ferroptosis.” 2020. Web. 19 Oct 2020.

Vancouver:

Venkatesh D. Elucidating the abilities of MDM2, MDMX and p21 to regulate ferroptosis. [Internet] [Doctoral dissertation]. Columbia University; 2020. [cited 2020 Oct 19]. Available from: https://doi.org/10.7916/d8-es1c-z783.

Council of Science Editors:

Venkatesh D. Elucidating the abilities of MDM2, MDMX and p21 to regulate ferroptosis. [Doctoral Dissertation]. Columbia University; 2020. Available from: https://doi.org/10.7916/d8-es1c-z783


Vanderbilt University

20. Abreu, Maria Mercedes. C/EBPbeta3 (LIP) induces cell death in breast cancer cells.

Degree: PhD, Cancer Biology, 2012, Vanderbilt University

 C/EBPbeta is a member of a family of basic-leucine zipper transcription factors. It has been shown to be a key regulator of growth and differentiation… (more)

Subjects/Keywords: C/EBPbeta; breast cancer; cell death; autophagy

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abreu, M. M. (2012). C/EBPbeta3 (LIP) induces cell death in breast cancer cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12173

Chicago Manual of Style (16th Edition):

Abreu, Maria Mercedes. “C/EBPbeta3 (LIP) induces cell death in breast cancer cells.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed October 19, 2020. http://hdl.handle.net/1803/12173.

MLA Handbook (7th Edition):

Abreu, Maria Mercedes. “C/EBPbeta3 (LIP) induces cell death in breast cancer cells.” 2012. Web. 19 Oct 2020.

Vancouver:

Abreu MM. C/EBPbeta3 (LIP) induces cell death in breast cancer cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1803/12173.

Council of Science Editors:

Abreu MM. C/EBPbeta3 (LIP) induces cell death in breast cancer cells. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/12173


Nelson Mandela Metropolitan University

21. Venables, Luanne. In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone.

Degree: PhD, Faculty of Science, 2014, Nelson Mandela Metropolitan University

 Artemisia afra is one of the oldest, most well known and widely used traditional medicinal plants in South Africa. It is used to treat many… (more)

Subjects/Keywords: Medicinal plants  – South Africa; Cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Venables, L. (2014). In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone. (Doctoral Dissertation). Nelson Mandela Metropolitan University. Retrieved from http://hdl.handle.net/10948/d1021087

Chicago Manual of Style (16th Edition):

Venables, Luanne. “In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone.” 2014. Doctoral Dissertation, Nelson Mandela Metropolitan University. Accessed October 19, 2020. http://hdl.handle.net/10948/d1021087.

MLA Handbook (7th Edition):

Venables, Luanne. “In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone.” 2014. Web. 19 Oct 2020.

Vancouver:

Venables L. In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone. [Internet] [Doctoral dissertation]. Nelson Mandela Metropolitan University; 2014. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/10948/d1021087.

Council of Science Editors:

Venables L. In vitro induction of cell death pathways by artemisia afra extract and isolation of an active compound, isoalantolactone. [Doctoral Dissertation]. Nelson Mandela Metropolitan University; 2014. Available from: http://hdl.handle.net/10948/d1021087


Texas A&M University

22. Gray, Joel W. Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3.

Degree: PhD, Biochemistry, 2013, Texas A&M University

 Programmed cell death (PCD) is a fundamentally important process delicately coordinated throughout an organism’s life cycle. In plants, PCD is an integral part of development,… (more)

Subjects/Keywords: Plant biology; protein kinase; cell-death; metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gray, J. W. (2013). Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151164

Chicago Manual of Style (16th Edition):

Gray, Joel W. “Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3.” 2013. Doctoral Dissertation, Texas A&M University. Accessed October 19, 2020. http://hdl.handle.net/1969.1/151164.

MLA Handbook (7th Edition):

Gray, Joel W. “Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3.” 2013. Web. 19 Oct 2020.

Vancouver:

Gray JW. Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1969.1/151164.

Council of Science Editors:

Gray JW. Non-activation Loop Phosphorylation and Downstream Signaling of AGC1-3 the Arabidopsis thaliana Homologue of the Tomato Cell Death Suppressor Adi3. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151164


Texas A&M University

23. Turtle, Joel Dylan. Acute Pain after Spinal Cord Injury: Impaired Recovery through Inflammation, Cell Death, and Hemorrhage.

Degree: PhD, Neuroscience, 2018, Texas A&M University

 Over 90% of spinal cord injuries are caused by traumatic accidents and are often associated with secondary tissue damage that can provide a source of… (more)

Subjects/Keywords: spinal cord injury; multitrauma; pain; cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Turtle, J. D. (2018). Acute Pain after Spinal Cord Injury: Impaired Recovery through Inflammation, Cell Death, and Hemorrhage. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/173285

Chicago Manual of Style (16th Edition):

Turtle, Joel Dylan. “Acute Pain after Spinal Cord Injury: Impaired Recovery through Inflammation, Cell Death, and Hemorrhage.” 2018. Doctoral Dissertation, Texas A&M University. Accessed October 19, 2020. http://hdl.handle.net/1969.1/173285.

MLA Handbook (7th Edition):

Turtle, Joel Dylan. “Acute Pain after Spinal Cord Injury: Impaired Recovery through Inflammation, Cell Death, and Hemorrhage.” 2018. Web. 19 Oct 2020.

Vancouver:

Turtle JD. Acute Pain after Spinal Cord Injury: Impaired Recovery through Inflammation, Cell Death, and Hemorrhage. [Internet] [Doctoral dissertation]. Texas A&M University; 2018. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1969.1/173285.

Council of Science Editors:

Turtle JD. Acute Pain after Spinal Cord Injury: Impaired Recovery through Inflammation, Cell Death, and Hemorrhage. [Doctoral Dissertation]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/173285


Texas A&M University

24. Avila Pacheco, Julian Ricardo, 1983-. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.

Degree: PhD, Biochemistry, 2012, Texas A&M University

 Programmed cell death (PCD) is an active process by which organisms coordinate the controlled destruction of cells. In tomato, the protein kinase Adi3 (AvrPto-dependent Pto-interacting… (more)

Subjects/Keywords: Ubiquitination; Phosphorylation; Tomato; Programmed cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Avila Pacheco, Julian Ricardo, 1. (2012). Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148132

Chicago Manual of Style (16th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Doctoral Dissertation, Texas A&M University. Accessed October 19, 2020. http://hdl.handle.net/1969.1/148132.

MLA Handbook (7th Edition):

Avila Pacheco, Julian Ricardo, 1983-. “Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling.” 2012. Web. 19 Oct 2020.

Vancouver:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1969.1/148132.

Council of Science Editors:

Avila Pacheco, Julian Ricardo 1. Understanding Postranslational Modifications Involved in Adi3 Programmed Cell Death Signaling. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148132


McMaster University

25. Chi, Xiaoke. EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM.

Degree: PhD, 2016, McMaster University

Apoptosis is a type of programmed cell death which plays a fundamental role in maintaining homeostasis in multi-cellular organisms. The Bcl-2 family has been identified… (more)

Subjects/Keywords: apoptosis; cell death; cancer; Bcl-2 family

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chi, X. (2016). EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/20403

Chicago Manual of Style (16th Edition):

Chi, Xiaoke. “EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM.” 2016. Doctoral Dissertation, McMaster University. Accessed October 19, 2020. http://hdl.handle.net/11375/20403.

MLA Handbook (7th Edition):

Chi, Xiaoke. “EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM.” 2016. Web. 19 Oct 2020.

Vancouver:

Chi X. EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM. [Internet] [Doctoral dissertation]. McMaster University; 2016. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/11375/20403.

Council of Science Editors:

Chi X. EXPLORING THE PRO-APOPTOTIC FUNCTION OF BIM. [Doctoral Dissertation]. McMaster University; 2016. Available from: http://hdl.handle.net/11375/20403


Texas A&M University

26. Gillis, David Christopher. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.

Degree: MS, Biomedical Sciences, 2015, Texas A&M University

 Inflammation in the developing central nervous system (CNS) can contribute to numerous issues in the fully developed adult. Uncovering pathways responsible for the inflammation is… (more)

Subjects/Keywords: RIP3; neuroinflammation; LPS; programmed cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gillis, D. C. (2015). The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155480

Chicago Manual of Style (16th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Masters Thesis, Texas A&M University. Accessed October 19, 2020. http://hdl.handle.net/1969.1/155480.

MLA Handbook (7th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Web. 19 Oct 2020.

Vancouver:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Internet] [Masters thesis]. Texas A&M University; 2015. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1969.1/155480.

Council of Science Editors:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Masters Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155480


University of Texas Southwestern Medical Center

27. Wu, Cheng-Yang. Using C. Elegans as Model Organism to Study the Mode of Action of a Natural Toxin, Psymberin.

Degree: 2011, University of Texas Southwestern Medical Center

 Psymberin is an extremely potent cytotoxin isolated from the marine sponges Psammocinia and Ircinia ramose. Several cancer cell lines are sensitive to psymberin, including breast,… (more)

Subjects/Keywords: Pyrones; Caenorhabditis elegans Proteins; Cell Death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wu, C. (2011). Using C. Elegans as Model Organism to Study the Mode of Action of a Natural Toxin, Psymberin. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/956

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Cheng-Yang. “Using C. Elegans as Model Organism to Study the Mode of Action of a Natural Toxin, Psymberin.” 2011. Thesis, University of Texas Southwestern Medical Center. Accessed October 19, 2020. http://hdl.handle.net/2152.5/956.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Cheng-Yang. “Using C. Elegans as Model Organism to Study the Mode of Action of a Natural Toxin, Psymberin.” 2011. Web. 19 Oct 2020.

Vancouver:

Wu C. Using C. Elegans as Model Organism to Study the Mode of Action of a Natural Toxin, Psymberin. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2011. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/2152.5/956.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu C. Using C. Elegans as Model Organism to Study the Mode of Action of a Natural Toxin, Psymberin. [Thesis]. University of Texas Southwestern Medical Center; 2011. Available from: http://hdl.handle.net/2152.5/956

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Johannesburg

28. Mfouo-Tynga, Ivan Sosthene. Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili).

Degree: 2013, University of Johannesburg

M.Tech. (Biomedical Technology)

The uncontrolled growth of cells in the body is often associated with cancer. It constitutes a major health problem and is one… (more)

Subjects/Keywords: Phthalocyanines; Breast cancer - Photochemotherapy; Anthraquinones; Cell death

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mfouo-Tynga, I. S. (2013). Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili). (Thesis). University of Johannesburg. Retrieved from http://hdl.handle.net/10210/8735

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mfouo-Tynga, Ivan Sosthene. “Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili).” 2013. Thesis, University of Johannesburg. Accessed October 19, 2020. http://hdl.handle.net/10210/8735.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mfouo-Tynga, Ivan Sosthene. “Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili).” 2013. Web. 19 Oct 2020.

Vancouver:

Mfouo-Tynga IS. Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili). [Internet] [Thesis]. University of Johannesburg; 2013. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/10210/8735.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mfouo-Tynga IS. Effectiveness of zinc-phthalocyanine and hypericin in inducing cell death in human breast cancer cells (mcf-7) using low intensity laser irradiation (lili). [Thesis]. University of Johannesburg; 2013. Available from: http://hdl.handle.net/10210/8735

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

29. Oliveri, Stefanie. Characterizing the Mechanism of Cul7-mediated Inhibition of Cell Death.

Degree: 2011, University of Toronto

Cullin 7 (Cul7) is a member of the cullin protein family that is emerging as a complex anti-apoptotic player in tumourigenesis. We hypothesize that by… (more)

Subjects/Keywords: Cul7; Myc; Cell Death; 0307; 0992

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oliveri, S. (2011). Characterizing the Mechanism of Cul7-mediated Inhibition of Cell Death. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/31376

Chicago Manual of Style (16th Edition):

Oliveri, Stefanie. “Characterizing the Mechanism of Cul7-mediated Inhibition of Cell Death.” 2011. Masters Thesis, University of Toronto. Accessed October 19, 2020. http://hdl.handle.net/1807/31376.

MLA Handbook (7th Edition):

Oliveri, Stefanie. “Characterizing the Mechanism of Cul7-mediated Inhibition of Cell Death.” 2011. Web. 19 Oct 2020.

Vancouver:

Oliveri S. Characterizing the Mechanism of Cul7-mediated Inhibition of Cell Death. [Internet] [Masters thesis]. University of Toronto; 2011. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/1807/31376.

Council of Science Editors:

Oliveri S. Characterizing the Mechanism of Cul7-mediated Inhibition of Cell Death. [Masters Thesis]. University of Toronto; 2011. Available from: http://hdl.handle.net/1807/31376


University of Ottawa

30. Wachholz, Kristina Lora Catherine. Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface .

Degree: 2016, University of Ottawa

 Maternal tolerance during pregnancy increases the risk of infection with certain intracellular pathogens such as Salmonella enterica serovar Typhimurium (S.Tm). Systemic S.Tm infection during pregnancy… (more)

Subjects/Keywords: Pregnancy; Infection; Immunity; Cell Death; Inflammation; Salmonella

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wachholz, K. L. C. (2016). Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/34190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wachholz, Kristina Lora Catherine. “Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface .” 2016. Thesis, University of Ottawa. Accessed October 19, 2020. http://hdl.handle.net/10393/34190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wachholz, Kristina Lora Catherine. “Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface .” 2016. Web. 19 Oct 2020.

Vancouver:

Wachholz KLC. Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface . [Internet] [Thesis]. University of Ottawa; 2016. [cited 2020 Oct 19]. Available from: http://hdl.handle.net/10393/34190.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wachholz KLC. Placental Infection by Salmonella Typhimurium in a Murine Model: The Role of Innate Immune Mediators in Cell Death at the Fetal-Maternal Interface . [Thesis]. University of Ottawa; 2016. Available from: http://hdl.handle.net/10393/34190

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3] [4] [5] … [30]

.