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You searched for subject:(Cell Proliferation 60). Showing records 1 – 5 of 5 total matches.

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1. Habela, Christa Whelan. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.

Degree: PhD, 2008, University of Alabama – Birmingham

The hypothesis that cell volume and the progression of the cell cycle are interdependent has surfaced off and on in the cell cycle literature for… (more)

Subjects/Keywords: Cell Cycle  – physiology<; br>; Cell Movement  – physiology<; br>; Cell Proliferation<; br>; Cell Size<; br>; Chloride Channels  – physiology<; br>; Chlorides  – metabolism<; br>; Cytokinesis  – physiology<; br>; Glioma  – physiopathology<; br>; Mitosis  – physiology<; br>; Neuroglia  – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Habela, C. W. (2008). Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,444

Chicago Manual of Style (16th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 05, 2021. http://contentdm.mhsl.uab.edu/u?/etd,444.

MLA Handbook (7th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Web. 05 Mar 2021.

Vancouver:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 05]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444.

Council of Science Editors:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444

2. Lee, Joo Hyoung. Structural and functional studies on group A Streptococcal bacteriophage hyaluronidase.

Degree: PhD, 2011, University of Alabama – Birmingham

Aberrant hyaluronan production is frequently associated with tumors where the elevated levels of tumoral hyaluronan are often associated with higher expression levels of cellular hyaluronidases… (more)

Subjects/Keywords: Cell Movement  – drug effects<; br>; Cell Proliferation  – drug effects<; br>; Extracellular Signal-Regulated MAP Kinases  – metabolism<; br>; Hyaluronic Acid  – pharmacology<; br>; Hyaluronoglucosaminidase  – metabolism<; br>; rhoA GTP-Binding Protein  – metabolism

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APA (6th Edition):

Lee, J. H. (2011). Structural and functional studies on group A Streptococcal bacteriophage hyaluronidase. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1163

Chicago Manual of Style (16th Edition):

Lee, Joo Hyoung. “Structural and functional studies on group A Streptococcal bacteriophage hyaluronidase.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 05, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1163.

MLA Handbook (7th Edition):

Lee, Joo Hyoung. “Structural and functional studies on group A Streptococcal bacteriophage hyaluronidase.” 2011. Web. 05 Mar 2021.

Vancouver:

Lee JH. Structural and functional studies on group A Streptococcal bacteriophage hyaluronidase. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 05]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1163.

Council of Science Editors:

Lee JH. Structural and functional studies on group A Streptococcal bacteriophage hyaluronidase. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1163

3. Wohler, Jillian E. The role of the [beta]2-integrin family on T cell subsets.

Degree: PhD, 2009, University of Alabama – Birmingham

Members of the [beta]2-integrin family of adhesion molecules, CD11a, CD11b, and CD11c, have all been shown to play a role in the pathogenesis of experimental… (more)

Subjects/Keywords: Antigens, CD11  – analysis<; br>; Cell Cycle Proteins  – physiology<; br>; Cell Proliferation<; br>; Encephalomyelitis, Autoimmune, Experimental  – genetics<; br>; Encephalomyelitis, Autoimmune, Experimental  – immunology<; br>; T-Lymphocytes  – cytology

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APA (6th Edition):

Wohler, J. E. (2009). The role of the [beta]2-integrin family on T cell subsets. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,409

Chicago Manual of Style (16th Edition):

Wohler, Jillian E. “The role of the [beta]2-integrin family on T cell subsets.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 05, 2021. http://contentdm.mhsl.uab.edu/u?/etd,409.

MLA Handbook (7th Edition):

Wohler, Jillian E. “The role of the [beta]2-integrin family on T cell subsets.” 2009. Web. 05 Mar 2021.

Vancouver:

Wohler JE. The role of the [beta]2-integrin family on T cell subsets. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 05]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,409.

Council of Science Editors:

Wohler JE. The role of the [beta]2-integrin family on T cell subsets. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,409

4. Phipps, Matthew Christopher. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.

Degree: PhD, 2012, University of Alabama – Birmingham

Although bone has a dramatic capacity for regeneration, certain injuries and procedures present defects that are unable to heal properly, requiring surgical intervention to induce… (more)

Subjects/Keywords: Apoptosis – drug effects<; br>; beta Catenin – metabolism.<; br>; Breast Neoplasms – drug therapy<; br>; Cell Proliferation – drug effects.<; br>; Cyclic GMP-Dependent Protein Kinases – metabolism.<; br>; Cyclic Nucleotide Phosphodiesterases, Type 5 – genetics.<; br>; Phosphodiesterase 5 Inhibitors.<; br>; Sulindac – analogs & derivatives.<; br>; Wnt Proteins – metabolism.

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APA (6th Edition):

Phipps, M. C. (2012). Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1410

Chicago Manual of Style (16th Edition):

Phipps, Matthew Christopher. “Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 05, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1410.

MLA Handbook (7th Edition):

Phipps, Matthew Christopher. “Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.” 2012. Web. 05 Mar 2021.

Vancouver:

Phipps MC. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 05]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1410.

Council of Science Editors:

Phipps MC. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1410

5. Perez, Jessica. Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor.

Degree: PhD, 2010, University of Alabama – Birmingham

Growth factors are important inducers of vascular cell growth whose regulation is altered during the pathogenesis of atherosclerosis. An increase in growth factor and cytokine… (more)

Subjects/Keywords: Cell Proliferation  – drug effects<; br>; Glucose  – metabolism<; br>; Muscle, Smooth, Vascular  – cytology<; br>; Myocytes, Smooth Muscle  – cytology<; br>; Myocytes, Smooth Muscle  – drug effects<; br>; Phosphatidylinositol 3-Kinases  – antagonists & inhibitors<; br>; Platelet-Derived Growth Factor  – pharmacology<; br>

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Perez, J. (2010). Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,928

Chicago Manual of Style (16th Edition):

Perez, Jessica. “Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 05, 2021. http://contentdm.mhsl.uab.edu/u?/etd,928.

MLA Handbook (7th Edition):

Perez, Jessica. “Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor.” 2010. Web. 05 Mar 2021.

Vancouver:

Perez J. Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 05]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,928.

Council of Science Editors:

Perez J. Regulation of vascular smooth muscle cell signaling and metabolism by platelet-derived growth factor. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,928

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