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You searched for subject:(Cell Cycle physiology 60). Showing records 1 – 30 of 60221 total matches.

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1. Habela, Christa Whelan. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.

Degree: PhD, 2008, University of Alabama – Birmingham

The hypothesis that cell volume and the progression of the cell cycle are interdependent has surfaced off and on in the cell cycle literature for… (more)

Subjects/Keywords: Cell Cycle  – physiology<; br>; Cell Movement  – physiology<; br>; Cell Proliferation<; br>; Cell Size<; br>; Chloride Channels  – physiology<; br>; Chlorides  – metabolism<; br>; Cytokinesis  – physiology<; br>; Glioma  – physiopathology<; br>; Mitosis  – physiology<; br>; Neuroglia  – physiology

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APA (6th Edition):

Habela, C. W. (2008). Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,444

Chicago Manual of Style (16th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,444.

MLA Handbook (7th Edition):

Habela, Christa Whelan. “Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes.” 2008. Web. 07 Mar 2021.

Vancouver:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444.

Council of Science Editors:

Habela CW. Progression through the cell cycle is regulated by dynamic chloride dependent changes in cell volumes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,444

2. Helton, Eric Scott. A role for p63 in the regulation of cell cycle progression and cell death.

Degree: PhD, 2007, University of Alabama – Birmingham

p63 is a member of the p53 family of transcription factors that is a critical regulator of epithelial development. Studies have shown that p63 does… (more)

Subjects/Keywords: Cell Cycle  – physiology <; br>; Cell Death  – physiology <; br>; Transcription Factors <; br>; Tumor Suppressor Protein p53  – physiology

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APA (6th Edition):

Helton, E. S. (2007). A role for p63 in the regulation of cell cycle progression and cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,279

Chicago Manual of Style (16th Edition):

Helton, Eric Scott. “A role for p63 in the regulation of cell cycle progression and cell death.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,279.

MLA Handbook (7th Edition):

Helton, Eric Scott. “A role for p63 in the regulation of cell cycle progression and cell death.” 2007. Web. 07 Mar 2021.

Vancouver:

Helton ES. A role for p63 in the regulation of cell cycle progression and cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,279.

Council of Science Editors:

Helton ES. A role for p63 in the regulation of cell cycle progression and cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,279

3. Parks, Brian W. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.

Degree: PhD, 2008, University of Alabama – Birmingham

The G protein-coupled receptor, G2A, is expressed by multiple cell-types involved in atherosclerosis and is activated by structurally related lysophospholipids generated during low-density lipoprotein (LDL)… (more)

Subjects/Keywords: Apolipoproteins E  – metabolism<; br>; Arteriosclerosis<; br>; Bone Marrow Cells  – metabolism<; br>; Cell Cycle Proteins<; br>; Hypercholesterolemia  – metabolism<; br>; Lysophosphatidylcholines  – metabolism<; br>; Macrophages  – physiology<; br>; Receptors, G-Protein-Coupled<; br>; Receptors, LDL

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APA (6th Edition):

Parks, B. W. (2008). Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,768

Chicago Manual of Style (16th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,768.

MLA Handbook (7th Edition):

Parks, Brian W. “Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A.” 2008. Web. 07 Mar 2021.

Vancouver:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768.

Council of Science Editors:

Parks BW. Modulation of lipoprotein metabolism and atherosclerosis by the G protein-coupled receptor, G2A. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,768

4. Genovese, Nicholas J. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.

Degree: PhD, 2010, University of Alabama – Birmingham

Though human papillomavirus infection of the human epidermis is epidemiologically widespread and typically benign, manipulation of the cell cycle within host tissues during infections can… (more)

Subjects/Keywords: Cell Cycle<; br>; Cell Transformation, Viral<; br>; Human papillomavirus 16  – metabolism<; br>; Keratinocytes<; br>; Oncogene Proteins, Viral  – metabolism<; br>; Papillomaviridae  – physiology<; br>; Papillomavirus E7 Proteins  – metabolism<; br>; Receptors, Estrogen  – metabolism<; br>; Retinoblastoma-Like Protein p130  – metabolism<; br>; S Phase

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APA (6th Edition):

Genovese, N. J. (2010). The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1158

Chicago Manual of Style (16th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1158.

MLA Handbook (7th Edition):

Genovese, Nicholas J. “The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium.” 2010. Web. 07 Mar 2021.

Vancouver:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158.

Council of Science Editors:

Genovese NJ. The mechanism of human papillomavirus E7 protein mediated S-phase reentry in the squamous epithelium. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1158

5. Wohler, Jillian E. The role of the [beta]2-integrin family on T cell subsets.

Degree: PhD, 2009, University of Alabama – Birmingham

Members of the [beta]2-integrin family of adhesion molecules, CD11a, CD11b, and CD11c, have all been shown to play a role in the pathogenesis of experimental… (more)

Subjects/Keywords: Antigens, CD11  – analysis<; br>; Cell Cycle Proteins  – physiology<; br>; Cell Proliferation<; br>; Encephalomyelitis, Autoimmune, Experimental  – genetics<; br>; Encephalomyelitis, Autoimmune, Experimental  – immunology<; br>; T-Lymphocytes  – cytology

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APA (6th Edition):

Wohler, J. E. (2009). The role of the [beta]2-integrin family on T cell subsets. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,409

Chicago Manual of Style (16th Edition):

Wohler, Jillian E. “The role of the [beta]2-integrin family on T cell subsets.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,409.

MLA Handbook (7th Edition):

Wohler, Jillian E. “The role of the [beta]2-integrin family on T cell subsets.” 2009. Web. 07 Mar 2021.

Vancouver:

Wohler JE. The role of the [beta]2-integrin family on T cell subsets. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,409.

Council of Science Editors:

Wohler JE. The role of the [beta]2-integrin family on T cell subsets. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,409

6. Belenky, Michael L. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.

Degree: PhD, 2011, University of Alabama – Birmingham

Our laboratory reported that a combination of ionizing radiation (IR) or temozolomide (Tmz), a DNA methylating agent clinically approved against glioblastoma multiforme (GBM), a type… (more)

Subjects/Keywords: Antibodies, Monoclonal  – therapeutic use<; br>; Apoptosis  – physiology<; br>; Brain Neoplasms  – drug therapy<; br>; Brain Neoplasms  – radiotherapy<; br>; Cell Cycle<; br>; Glioma  – drug therapy<; br>; Glioma  – radiotherapy<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Belenky, M. L. (2011). Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1032

Chicago Manual of Style (16th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1032.

MLA Handbook (7th Edition):

Belenky, Michael L. “Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism.” 2011. Web. 07 Mar 2021.

Vancouver:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032.

Council of Science Editors:

Belenky ML. Sensitization of glioma to death receptor 5-mediated apoptosis through genotoxic stress and cell cycle disruption: a study of mechanism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1032

7. Matthews, Tori A. Pathological modifications of tau induce toxicity and facilitate cell death.

Degree: PhD, 2009, University of Alabama – Birmingham

lzheimer’s disease (AD) is a progressive neurodegenerative disorder characterized by two major pathophysiological hallmarks, beta-amyloid (A[Beta]) plaques and tau tangles. In AD and other tau… (more)

Subjects/Keywords: Caspases  – metabolism<; br>; Cell Death  – physiology<; br>; Microtubules  – metabolism<; br>; tau Proteins  – physiology

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APA (6th Edition):

Matthews, T. A. (2009). Pathological modifications of tau induce toxicity and facilitate cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,607

Chicago Manual of Style (16th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,607.

MLA Handbook (7th Edition):

Matthews, Tori A. “Pathological modifications of tau induce toxicity and facilitate cell death.” 2009. Web. 07 Mar 2021.

Vancouver:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607.

Council of Science Editors:

Matthews TA. Pathological modifications of tau induce toxicity and facilitate cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,607

8. Keifenheim, Daniel L. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2015, U of Massachusetts : Med

  The coordination between cell growth and division is a highly regulated process that is intimately linked to the cell cycle. Efforts to identify an… (more)

Subjects/Keywords: Cell Size; Cell Cycle; Cell Cycle Checkpoints; Cell Division; Schizosaccharomyces; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Keifenheim, D. L. (2015). Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/784

Chicago Manual of Style (16th Edition):

Keifenheim, Daniel L. “Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2015. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/784.

MLA Handbook (7th Edition):

Keifenheim, Daniel L. “Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation.” 2015. Web. 07 Mar 2021.

Vancouver:

Keifenheim DL. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2015. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/784.

Council of Science Editors:

Keifenheim DL. Cell Size Control in the Fission Yeast Schizosaccharomyces pombe: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2015. Available from: http://escholarship.umassmed.edu/gsbs_diss/784

9. Marsh, Amie. Localization of proteins involved in trafficking in frog and mouse retina.

Degree: MS, 2012, University of Alabama – Birmingham

The connecting cilium serves as the major route for protein transport from the inner to outer segment of photoreceptor cells. The hypothesis that all Congenital… (more)

Subjects/Keywords: Cilia<; br>; Night Blindness<; br>; Protein Transport  – physiology<; br>; Rod Cell Outer Segment  – ultrastructure<; br>; Rhodopsin  – metabolism<; br>; Xenopus laevis

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APA (6th Edition):

Marsh, A. (2012). Localization of proteins involved in trafficking in frog and mouse retina. (Masters Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1186

Chicago Manual of Style (16th Edition):

Marsh, Amie. “Localization of proteins involved in trafficking in frog and mouse retina.” 2012. Masters Thesis, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1186.

MLA Handbook (7th Edition):

Marsh, Amie. “Localization of proteins involved in trafficking in frog and mouse retina.” 2012. Web. 07 Mar 2021.

Vancouver:

Marsh A. Localization of proteins involved in trafficking in frog and mouse retina. [Internet] [Masters thesis]. University of Alabama – Birmingham; 2012. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1186.

Council of Science Editors:

Marsh A. Localization of proteins involved in trafficking in frog and mouse retina. [Masters Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1186

10. Xayarath, Bobbi. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.

Degree: PhD, 2007, University of Alabama – Birmingham

 The polysaccharide capsules of Streptococcus pneumoniae represent the most important virulence determinant produced by this organism. Ninety-one different serotypes have been identified, but only a… (more)

Subjects/Keywords: Bacterial Capsules  – metabolism <; br>; Cell Wall  – metabolism <; br>; Genes, Essential <; br>; Mutation <; br>; Polysaccharides, Bacterial  – metabolism <; br>; Streptococcus pneumoniae  – physiology

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APA (6th Edition):

Xayarath, B. (2007). Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,190

Chicago Manual of Style (16th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,190.

MLA Handbook (7th Edition):

Xayarath, Bobbi. “Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence.” 2007. Web. 07 Mar 2021.

Vancouver:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190.

Council of Science Editors:

Xayarath B. Effects of specific alterations in capsule structure on Streptococcus pneumoniae capsule assembly and virulence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,190

11. Shaikh, Faheem M. Role of variant sialylation in regulating tumor cell behavior.

Degree: PhD, 2008, University of Alabama – Birmingham

Many different tumors have been documented to have elevated levels of the enzyme ST6Gal I, a Golgi glycosyltransferase that adds [alpha]2-6 sialic acids to glycoproteins.… (more)

Subjects/Keywords: Cell Movement  – physiology <; br>; Colonic Neoplasms <; br>; Integrins  – chemistry <; br>; Integrins  – metabolism <; br>; Neoplasm Invasiveness <; br>; Sialic Acids  – metabolism

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APA (6th Edition):

Shaikh, F. M. (2008). Role of variant sialylation in regulating tumor cell behavior. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,266

Chicago Manual of Style (16th Edition):

Shaikh, Faheem M. “Role of variant sialylation in regulating tumor cell behavior.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,266.

MLA Handbook (7th Edition):

Shaikh, Faheem M. “Role of variant sialylation in regulating tumor cell behavior.” 2008. Web. 07 Mar 2021.

Vancouver:

Shaikh FM. Role of variant sialylation in regulating tumor cell behavior. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,266.

Council of Science Editors:

Shaikh FM. Role of variant sialylation in regulating tumor cell behavior. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,266

12. Walls, Ken C. Molecular characterization of neural apoptosis.

Degree: PhD, 2009, University of Alabama – Birmingham

Neural cell death plays a critical role in normal nervous system development and dysregulated neural stem cell death contributes to brain malformation, tumorgenesis, and possibly,… (more)

Subjects/Keywords: Apoptosis<; br>; Autophagy  – genetics<; br>; Cell Death<; br>; Lysosomes <; br>; physiology Neurons  – pathology<; br>; Tumor Suppressor Protein p53

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APA (6th Edition):

Walls, K. C. (2009). Molecular characterization of neural apoptosis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,504

Chicago Manual of Style (16th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,504.

MLA Handbook (7th Edition):

Walls, Ken C. “Molecular characterization of neural apoptosis.” 2009. Web. 07 Mar 2021.

Vancouver:

Walls KC. Molecular characterization of neural apoptosis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504.

Council of Science Editors:

Walls KC. Molecular characterization of neural apoptosis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,504

13. Lai, Yun-Ju. Role of TRIP6 in LPA-induced cell migration.

Degree: PhD, 2007, University of Alabama – Birmingham

The LIM domain-containing Thyroid Receptor-Interacting Protein 6 (TRIP6) is a zyxin family member that has been implicated in actin dynamics and cell motility. In this… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing<; br>; Carrier Proteins  – physiology<; br>; Cell Movement<; br>; Feedback, Biochemical<; br>; Lysophospholipids<; br>; Protein-Tyrosine Kinases  – physiology<; br>; Receptors, G-Protein-Coupled  – metabolism<; br>; Transcription Factors

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APA (6th Edition):

Lai, Y. (2007). Role of TRIP6 in LPA-induced cell migration. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,453

Chicago Manual of Style (16th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,453.

MLA Handbook (7th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Web. 07 Mar 2021.

Vancouver:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453.

Council of Science Editors:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453

14. Nie, Shuyi. Regulation of vertebrate gastrulation by ErbB signaling.

Degree: PhD, 2007, University of Alabama – Birmingham

ErbB receptor tyrosine kinases have long been implicated in cancer formation and progression by regulating cell division, migration, and survival. ErbBs are also essential in… (more)

Subjects/Keywords: Cell Movement  – physiology <; br>; Gastrula  – physiology <; br>; Morphogenesis  – physiology <; br>; Receptor, Epidermal Growth Factor  – metabolism <; br>; Signal Transduction  – physiology <; br>; Xenopus  – embryology

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APA (6th Edition):

Nie, S. (2007). Regulation of vertebrate gastrulation by ErbB signaling. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,93

Chicago Manual of Style (16th Edition):

Nie, Shuyi. “Regulation of vertebrate gastrulation by ErbB signaling.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,93.

MLA Handbook (7th Edition):

Nie, Shuyi. “Regulation of vertebrate gastrulation by ErbB signaling.” 2007. Web. 07 Mar 2021.

Vancouver:

Nie S. Regulation of vertebrate gastrulation by ErbB signaling. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,93.

Council of Science Editors:

Nie S. Regulation of vertebrate gastrulation by ErbB signaling. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,93

15. Meares, Gordon P. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.

Degree: PhD, 2007, University of Alabama – Birmingham

Proper regulation of survival signaling is critical for all organisms. One important signaling cascade involved in the coordinated effort to control signals influencing cell fate… (more)

Subjects/Keywords: Apoptosis  – physiology <; br>; Cell Nucleus  – metabolism <; br>; Glycogen Synthase Kinase 3  – metabolism <; br>; HSP90 Heat-Shock Proteins  – physiology <; br>; Insulin  – physiology <; br>; Insulin-Like Growth Factor I  – physiology <; br>; Nuclear Localization Signals <; br>; Signal Transduction  – physiology

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APA (6th Edition):

Meares, G. P. (2007). Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,134

Chicago Manual of Style (16th Edition):

Meares, Gordon P. “Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,134.

MLA Handbook (7th Edition):

Meares, Gordon P. “Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival.” 2007. Web. 07 Mar 2021.

Vancouver:

Meares GP. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,134.

Council of Science Editors:

Meares GP. Directing Akt and GSK3[beta] : molecular insights into cell signaling and survival. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,134

16. Pivtoraiko, Violetta N. Regulation of neuronal death by the autophagy lysosomal pathway: implications for Parkinson disease.

Degree: PhD, 2011, University of Alabama – Birmingham

Parkinson Disease (PD) is the second most common age-related neurodegenerative disorder and is characterized pathologically by the loss of dopaminergic (DA) neurons in the stubstantia… (more)

Subjects/Keywords: Autophagy<; br>; Cell Death  – physiology<; br>; Cytoprotection  – drug effects<; br>; Lysosomes<; br>; Macrolides  – pharmacology<; br>; Neurons  – cytology<; br>; Oxidative Stress<; br>; Rotenone<; br>; Signal Transduction  – drug effects

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APA (6th Edition):

Pivtoraiko, V. N. (2011). Regulation of neuronal death by the autophagy lysosomal pathway: implications for Parkinson disease. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,957

Chicago Manual of Style (16th Edition):

Pivtoraiko, Violetta N. “Regulation of neuronal death by the autophagy lysosomal pathway: implications for Parkinson disease.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,957.

MLA Handbook (7th Edition):

Pivtoraiko, Violetta N. “Regulation of neuronal death by the autophagy lysosomal pathway: implications for Parkinson disease.” 2011. Web. 07 Mar 2021.

Vancouver:

Pivtoraiko VN. Regulation of neuronal death by the autophagy lysosomal pathway: implications for Parkinson disease. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,957.

Council of Science Editors:

Pivtoraiko VN. Regulation of neuronal death by the autophagy lysosomal pathway: implications for Parkinson disease. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,957

17. Ernest, Nola Jean Sieber. The role of chloride in the volume regulation of human glioma cells.

Degree: PhD, 2007, University of Alabama – Birmingham

According to the Central Brain Tumor Registry of the United States, the most common primary brain tumors are gliomas, tumors composed of cells of glial… (more)

Subjects/Keywords: Brain Neoplasms  – physiopathology <; br>; Cell Size <; br>; Chloride Channels  – physiology <; br>; Glioma  – physiopathology <; br>

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APA (6th Edition):

Ernest, N. J. S. (2007). The role of chloride in the volume regulation of human glioma cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,86

Chicago Manual of Style (16th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,86.

MLA Handbook (7th Edition):

Ernest, Nola Jean Sieber. “The role of chloride in the volume regulation of human glioma cells.” 2007. Web. 07 Mar 2021.

Vancouver:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86.

Council of Science Editors:

Ernest NJS. The role of chloride in the volume regulation of human glioma cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,86

18. Ko, Rose Marie. The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function.

Degree: PhD, 2007, University of Alabama – Birmingham

The t(8;21)(q22;q22) AML1-ETO translocation is one of the most frequent translocations in acute myeloid leukemia (AML), occurring in approximately 12% of cases. Our laboratory has… (more)

Subjects/Keywords: Cell Differentiation  – physiology <; br>; Core Binding Factor Alpha 2 Subunit  – physiology <; br>; Cyclin-Dependent Kinase Inhibitor p15  – physiology <; br>; Hematopoietic Stem Cells <; br>; Leukemia, Myeloid, Acute  – physiopathology <; br>; Myeloid Cells  – pathology

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APA (6th Edition):

Ko, R. M. (2007). The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,375

Chicago Manual of Style (16th Edition):

Ko, Rose Marie. “The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,375.

MLA Handbook (7th Edition):

Ko, Rose Marie. “The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function.” 2007. Web. 07 Mar 2021.

Vancouver:

Ko RM. The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,375.

Council of Science Editors:

Ko RM. The effect of the AML1-ETO translocation on cell cycle tumor suppressor gene function. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,375

19. Qian, Yingjuan, Ph.D. The role of DEC1 in P53-dependent cellular senescence.

Degree: PhD, 2008, University of Alabama – Birmingham

The p53 tumor suppressor is the most commonly mutated gene in human cancers. As a transcription factor, p53 exerts its tumor suppressor function through the… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors  – genetics<; br>; Basic Helix-Loop-Helix Transcription Factors  – physiology<; br>; Cell Aging  – physiology<; br>; DNA Damage<; br>; Tumor Suppressor Protein p53  – physiology<; br>; Tumor Suppressor Proteins  – genetics

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APA (6th Edition):

Qian, Yingjuan, P. D. (2008). The role of DEC1 in P53-dependent cellular senescence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,462

Chicago Manual of Style (16th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,462.

MLA Handbook (7th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Web. 07 Mar 2021.

Vancouver:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462.

Council of Science Editors:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462

20. McCoy, Eric. Expression and function of aquaporins in malignant and non-malignant astrocytes.

Degree: PhD, 2008, University of Alabama – Birmingham

Aquaporins (AQP) constitute the primary pathway for water movement across cellular membrances. As a result, their expression and function are important for regulating cell volume.… (more)

Subjects/Keywords: Aquaporin 1 Aquaporin 4<; br>; Aquaporins  – metabolism<; br>; Astrocytes  – physiology<; br>; Brain Injuries  – physiopathology<; br>; Brain Neoplasms  – metabolism<; br>; Cell Movement<; br>; Neoplasm Invasiveness<; br>; Wounds, Stab  – physiopathology

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APA (6th Edition):

McCoy, E. (2008). Expression and function of aquaporins in malignant and non-malignant astrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,774

Chicago Manual of Style (16th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,774.

MLA Handbook (7th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Web. 07 Mar 2021.

Vancouver:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774.

Council of Science Editors:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774

21. Goldstein, Rebecca F. Protein interaction and cell surface trafficking differences between wild-type and [Delta]F508 cystic fibrosis transmembrane conductance regulator.

Degree: PhD, 2007, University of Alabama – Birmingham

Cystic fibrosis (CF) is caused by mutation of one protein, the cystic fibrosis transmembrane conductance regulator (CFTR), which normally functions as a chloride channel at… (more)

Subjects/Keywords: Adenosine Triphosphatases  – metabolism <; br>; Cell Cycle Proteins  – metabolism <; br>; Cystic Fibrosis Transmembrane Conductance Regulator  – metabolism <; br>; Protein Interaction Mapping

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APA (6th Edition):

Goldstein, R. F. (2007). Protein interaction and cell surface trafficking differences between wild-type and [Delta]F508 cystic fibrosis transmembrane conductance regulator. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,123

Chicago Manual of Style (16th Edition):

Goldstein, Rebecca F. “Protein interaction and cell surface trafficking differences between wild-type and [Delta]F508 cystic fibrosis transmembrane conductance regulator.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,123.

MLA Handbook (7th Edition):

Goldstein, Rebecca F. “Protein interaction and cell surface trafficking differences between wild-type and [Delta]F508 cystic fibrosis transmembrane conductance regulator.” 2007. Web. 07 Mar 2021.

Vancouver:

Goldstein RF. Protein interaction and cell surface trafficking differences between wild-type and [Delta]F508 cystic fibrosis transmembrane conductance regulator. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,123.

Council of Science Editors:

Goldstein RF. Protein interaction and cell surface trafficking differences between wild-type and [Delta]F508 cystic fibrosis transmembrane conductance regulator. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,123

22. Bomben, Valerie Christine. Role of transient receptor potential canonical channels in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Gliomas, primary brain tumors derived from glial cells, constitute the majority of malignant tumors within the central nervous system. The most malignant of these tumors,… (more)

Subjects/Keywords: Brain Neoplasms  – pathology<; br>; Cell Cycle<; br>; Glioma  – pathology<; br>; Transient Receptor Potential Channels  – antagonists & inhibitors

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APA (6th Edition):

Bomben, V. C. (2010). Role of transient receptor potential canonical channels in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,642

Chicago Manual of Style (16th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,642.

MLA Handbook (7th Edition):

Bomben, Valerie Christine. “Role of transient receptor potential canonical channels in glioma cell biology.” 2010. Web. 07 Mar 2021.

Vancouver:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642.

Council of Science Editors:

Bomben VC. Role of transient receptor potential canonical channels in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,642

23. Woodard-Grice, Alencia V. (Alencia Vanay). Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.

Degree: PhD, 2008, University of Alabama – Birmingham

During monocyte activation and differentiation along the macrophage lineage, the activity of [alpha]4[beta]1 integrins is upregulated, which promotes extravasation from the vasculature and migration through… (more)

Subjects/Keywords: Amyloid Precursor Protein Secretases  – metabolism<; br>; Antigens, CD  – biosynthesis<; br>; Aspartic Endopeptidases  – metabolism<; br>; Cell Differentiation  – physiology<; br>; Integrin alpha4beta1  – metabolism<; br>; Macrophages  – enzymology<; br>; Monocytes  – enzymology<; br>; Protein Modification, Translational  – physiology<; br>; Sialyltransferases  – biosynthesis

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APA (6th Edition):

Woodard-Grice, A. V. (. V. (2008). Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,473

Chicago Manual of Style (16th Edition):

Woodard-Grice, Alencia V (Alencia Vanay). “Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,473.

MLA Handbook (7th Edition):

Woodard-Grice, Alencia V (Alencia Vanay). “Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1.” 2008. Web. 07 Mar 2021.

Vancouver:

Woodard-Grice AV(V. Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,473.

Council of Science Editors:

Woodard-Grice AV(V. Hyposialylation regulates [alpha]4[beta]1 integrin binding to VCAM-1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,473

24. Gupta, Sneha. Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation.

Degree: Interdisciplinary Graduate Program, Biochemistry and Molecular Pharmacology, 2013, U of Massachusetts : Med

  The fission yeast Schizosaccharomyces pombe has become a powerful model system for studying cytokinesis, a process of cytoplasmic division by which one cell divides… (more)

Subjects/Keywords: Cytokinesis; Cell Cycle; Cell Cycle Proteins; Cytoskeleton; Cytoskeletal Proteins; Schizosaccharomyces; Schizosaccharomyces pombe Proteins; Cellular and Molecular Physiology

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APA (6th Edition):

Gupta, S. (2013). Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/693

Chicago Manual of Style (16th Edition):

Gupta, Sneha. “Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/693.

MLA Handbook (7th Edition):

Gupta, Sneha. “Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation.” 2013. Web. 07 Mar 2021.

Vancouver:

Gupta S. Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/693.

Council of Science Editors:

Gupta S. Understanding Regulation of the Cytoskeleton during Cell Cycle Transitions through Examination of Crosstalk between Homologous Fission Yeast Pathways, Septation Initiation Network and Morphogenesis ORB6 Network: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/693

25. Kuo, Tse-Chun. Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation.

Degree: Interdisciplinary Graduate Program, Program in Molecular Medicine, 2013, U of Massachusetts : Med

  The midbody (MB) is a proteinaceous complex formed between the two daughter cells during cell division and is required for the final cell separation… (more)

Subjects/Keywords: Autophagy; Cell Division; Cell Differentiation; Cell Cycle Proteins; Nuclear Proteins; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

Kuo, T. (2013). Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/670

Chicago Manual of Style (16th Edition):

Kuo, Tse-Chun. “Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation.” 2013. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/670.

MLA Handbook (7th Edition):

Kuo, Tse-Chun. “Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation.” 2013. Web. 07 Mar 2021.

Vancouver:

Kuo T. Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2013. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/670.

Council of Science Editors:

Kuo T. Role of Autophagy in Post-Mitotic Midbody Fate and Function: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2013. Available from: http://escholarship.umassmed.edu/gsbs_diss/670

26. VanOudenhove, Jennifer J. Mechanisms Regulating Early Mesendodermal Differentiation of Human Embryonic Stem Cells: A Dissertation.

Degree: Cell Biology, Radiology, 2016, U of Massachusetts : Med

  Key regulatory events take place at very early stages of human embryonic stem cell (hESC) differentiation to accommodate their ability to differentiate into different… (more)

Subjects/Keywords: Cell Differentiation; Human Embryonic Stem Cells; Cell Cycle; Cell Lineage; Cell Biology; Cellular and Molecular Physiology

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APA (6th Edition):

VanOudenhove, J. J. (2016). Mechanisms Regulating Early Mesendodermal Differentiation of Human Embryonic Stem Cells: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from http://escholarship.umassmed.edu/gsbs_diss/849

Chicago Manual of Style (16th Edition):

VanOudenhove, Jennifer J. “Mechanisms Regulating Early Mesendodermal Differentiation of Human Embryonic Stem Cells: A Dissertation.” 2016. Doctoral Dissertation, U of Massachusetts : Med. Accessed March 07, 2021. http://escholarship.umassmed.edu/gsbs_diss/849.

MLA Handbook (7th Edition):

VanOudenhove, Jennifer J. “Mechanisms Regulating Early Mesendodermal Differentiation of Human Embryonic Stem Cells: A Dissertation.” 2016. Web. 07 Mar 2021.

Vancouver:

VanOudenhove JJ. Mechanisms Regulating Early Mesendodermal Differentiation of Human Embryonic Stem Cells: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2016. [cited 2021 Mar 07]. Available from: http://escholarship.umassmed.edu/gsbs_diss/849.

Council of Science Editors:

VanOudenhove JJ. Mechanisms Regulating Early Mesendodermal Differentiation of Human Embryonic Stem Cells: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2016. Available from: http://escholarship.umassmed.edu/gsbs_diss/849

27. Brooks, William Samuel. Localization and function of G2E3.

Degree: PhD, 2007, University of Alabama – Birmingham

A microarray study was performed which identified G2E3 as a novel, putative ubiquitin ligase that was both G2/M-specific in expression and down regulated at the… (more)

Subjects/Keywords: Cell Nucleus  – metabolism <; br>; DNA Damage  – physiology <; br>; Hela Cells <; br>; Nuclear Localization Signals  – analysis <; br>; Ubiquitin-Protein Ligases  – metabolism <; br>; Ubiquitin-Protein Ligases  – physiology

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APA (6th Edition):

Brooks, W. S. (2007). Localization and function of G2E3. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,175

Chicago Manual of Style (16th Edition):

Brooks, William Samuel. “Localization and function of G2E3.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,175.

MLA Handbook (7th Edition):

Brooks, William Samuel. “Localization and function of G2E3.” 2007. Web. 07 Mar 2021.

Vancouver:

Brooks WS. Localization and function of G2E3. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,175.

Council of Science Editors:

Brooks WS. Localization and function of G2E3. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,175

28. Wang, Hsu-Kun. A novel & efficient method to produce high titers of infectious HPV in organotypic cultures of primary human keratinocytes.

Degree: PhD, 2009, University of Alabama – Birmingham

Infections by the high-risk human papillomavirus type 18 (HPV-18) or type 16 (HPV-16) can lead to cancers of the anogenital tract. Because the propagation of… (more)

Subjects/Keywords: Cell Culture Techniques  – methods<; br>; DNA Replication<; br>; Human papillomavirus 18  – physiology<; br>; Keratinocytes  – virology<; br>; Papillomavirus Infections  – virology<; br>; Virus Replication  – physiology

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APA (6th Edition):

Wang, H. (2009). A novel & efficient method to produce high titers of infectious HPV in organotypic cultures of primary human keratinocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1012

Chicago Manual of Style (16th Edition):

Wang, Hsu-Kun. “A novel & efficient method to produce high titers of infectious HPV in organotypic cultures of primary human keratinocytes.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1012.

MLA Handbook (7th Edition):

Wang, Hsu-Kun. “A novel & efficient method to produce high titers of infectious HPV in organotypic cultures of primary human keratinocytes.” 2009. Web. 07 Mar 2021.

Vancouver:

Wang H. A novel & efficient method to produce high titers of infectious HPV in organotypic cultures of primary human keratinocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1012.

Council of Science Editors:

Wang H. A novel & efficient method to produce high titers of infectious HPV in organotypic cultures of primary human keratinocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1012

29. Schreeder, Daniel M. Biological characterization of Fc receptor-like 6 (FCRL6).

Degree: PhD, 2009, University of Alabama – Birmingham

Members of the Fc receptor-like (FCRL) family are cell-surface proteins with ancient conservation, distinct lymphocyte expression patterns, and tyrosine-based signaling capabilities that imply a fundamental… (more)

Subjects/Keywords: CD8-Positive T-Lymphocytes  – immunology<; br>; HLA-DR Antigens  – metabolism<; br>; Killer Cells, Natural  – immunology<; br>; Leukemia, Lymphocytic, Chronic, B-Cell  – immunology<; br>; Receptors, Cell Surface  – physiology<; br>; Receptors, Fc  – metabolism

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APA (6th Edition):

Schreeder, D. M. (2009). Biological characterization of Fc receptor-like 6 (FCRL6). (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1112

Chicago Manual of Style (16th Edition):

Schreeder, Daniel M. “Biological characterization of Fc receptor-like 6 (FCRL6).” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 07, 2021. http://contentdm.mhsl.uab.edu/u?/etd,1112.

MLA Handbook (7th Edition):

Schreeder, Daniel M. “Biological characterization of Fc receptor-like 6 (FCRL6).” 2009. Web. 07 Mar 2021.

Vancouver:

Schreeder DM. Biological characterization of Fc receptor-like 6 (FCRL6). [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2021 Mar 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1112.

Council of Science Editors:

Schreeder DM. Biological characterization of Fc receptor-like 6 (FCRL6). [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1112


York University

30. Shiell, Mitchell Anthony. Electrical Pulse Stimulation of MCF7 Breast Cancer Coordinates Autophagy Reprogramming and Proliferative Failure Leading to Cellular Senescence.

Degree: MSc -MS, Kinesiology & Health Science, 2020, York University

 The utilization of Electrical Pulse Stimulation (EPS) has been predominantly used to study the physiological, cellular, and molecular responses of excitable cells such as nerve… (more)

Subjects/Keywords: Physiology; Electrical Pulse Stimulation; Electrical Stimulation; EPS; MCF7; MCF-7; Breast Cancer; Cancer; Autophagy; Proteostasis; Proliferative Failure; Cellular Senescence; Cell cycle; Cell cycle control; Cell cycle arrest; Cell cycle exit; Cell cycle regulation; G2/M

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shiell, M. A. (2020). Electrical Pulse Stimulation of MCF7 Breast Cancer Coordinates Autophagy Reprogramming and Proliferative Failure Leading to Cellular Senescence. (Masters Thesis). York University. Retrieved from https://yorkspace.library.yorku.ca/xmlui/handle/10315/37430

Chicago Manual of Style (16th Edition):

Shiell, Mitchell Anthony. “Electrical Pulse Stimulation of MCF7 Breast Cancer Coordinates Autophagy Reprogramming and Proliferative Failure Leading to Cellular Senescence.” 2020. Masters Thesis, York University. Accessed March 07, 2021. https://yorkspace.library.yorku.ca/xmlui/handle/10315/37430.

MLA Handbook (7th Edition):

Shiell, Mitchell Anthony. “Electrical Pulse Stimulation of MCF7 Breast Cancer Coordinates Autophagy Reprogramming and Proliferative Failure Leading to Cellular Senescence.” 2020. Web. 07 Mar 2021.

Vancouver:

Shiell MA. Electrical Pulse Stimulation of MCF7 Breast Cancer Coordinates Autophagy Reprogramming and Proliferative Failure Leading to Cellular Senescence. [Internet] [Masters thesis]. York University; 2020. [cited 2021 Mar 07]. Available from: https://yorkspace.library.yorku.ca/xmlui/handle/10315/37430.

Council of Science Editors:

Shiell MA. Electrical Pulse Stimulation of MCF7 Breast Cancer Coordinates Autophagy Reprogramming and Proliferative Failure Leading to Cellular Senescence. [Masters Thesis]. York University; 2020. Available from: https://yorkspace.library.yorku.ca/xmlui/handle/10315/37430

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