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You searched for subject:(Cardiomyocyte). Showing records 1 – 30 of 174 total matches.

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Tampere University

1. Pölönen, Risto-Pekka. Effects of uniaxial strain in hypertrophic cardiomyopathy -patient specific human induced pluripotent stem cell -derived cardiomyocytes .

Degree: 2014, Tampere University

 Background and aims: Hypertrophic cardiomyopathy (HCM) is genetically inherited autosomal-dominant disease with significant genotypic and phenotypic heterogeneity. HCM is one of the most common inherited… (more)

Subjects/Keywords: HCM; hiPSC; cardiomyocyte

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APA (6th Edition):

Pölönen, R. (2014). Effects of uniaxial strain in hypertrophic cardiomyopathy -patient specific human induced pluripotent stem cell -derived cardiomyocytes . (Masters Thesis). Tampere University. Retrieved from https://trepo.tuni.fi/handle/10024/95880

Chicago Manual of Style (16th Edition):

Pölönen, Risto-Pekka. “Effects of uniaxial strain in hypertrophic cardiomyopathy -patient specific human induced pluripotent stem cell -derived cardiomyocytes .” 2014. Masters Thesis, Tampere University. Accessed April 05, 2020. https://trepo.tuni.fi/handle/10024/95880.

MLA Handbook (7th Edition):

Pölönen, Risto-Pekka. “Effects of uniaxial strain in hypertrophic cardiomyopathy -patient specific human induced pluripotent stem cell -derived cardiomyocytes .” 2014. Web. 05 Apr 2020.

Vancouver:

Pölönen R. Effects of uniaxial strain in hypertrophic cardiomyopathy -patient specific human induced pluripotent stem cell -derived cardiomyocytes . [Internet] [Masters thesis]. Tampere University; 2014. [cited 2020 Apr 05]. Available from: https://trepo.tuni.fi/handle/10024/95880.

Council of Science Editors:

Pölönen R. Effects of uniaxial strain in hypertrophic cardiomyopathy -patient specific human induced pluripotent stem cell -derived cardiomyocytes . [Masters Thesis]. Tampere University; 2014. Available from: https://trepo.tuni.fi/handle/10024/95880


University of Southern California

2. Gu, Jie. A robust adult zebrafish cardiomyocyte isolation protocol and its application in calcium transients imaging.

Degree: MS, Biomedical Engineering, 2012, University of Southern California

 The zebrafish is an emerging vertebral animal as a genetically tractable model for human-related disease studies. They serve as models for angiogenesis, drug screening (Serbedzija,… (more)

Subjects/Keywords: zebrafish; calcium transients; cardiomyocyte; isolation

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APA (6th Edition):

Gu, J. (2012). A robust adult zebrafish cardiomyocyte isolation protocol and its application in calcium transients imaging. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/29227/rec/350

Chicago Manual of Style (16th Edition):

Gu, Jie. “A robust adult zebrafish cardiomyocyte isolation protocol and its application in calcium transients imaging.” 2012. Masters Thesis, University of Southern California. Accessed April 05, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/29227/rec/350.

MLA Handbook (7th Edition):

Gu, Jie. “A robust adult zebrafish cardiomyocyte isolation protocol and its application in calcium transients imaging.” 2012. Web. 05 Apr 2020.

Vancouver:

Gu J. A robust adult zebrafish cardiomyocyte isolation protocol and its application in calcium transients imaging. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Apr 05]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/29227/rec/350.

Council of Science Editors:

Gu J. A robust adult zebrafish cardiomyocyte isolation protocol and its application in calcium transients imaging. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/29227/rec/350


Université de Sherbrooke

3. Bastien, Jean-Guillaume. Impact de la protéine ADAP1 sur la survie des cardiomyocytes .

Degree: 2015, Université de Sherbrooke

 Résumé: Une des maladies cardiaques les plus sévères est l’insuffisance cardiaque (IC). Un facteur important à la base de l’IC est la mort des cardiomyocytes… (more)

Subjects/Keywords: ADAP1; AKT; Arf6; Insuffisance cardiaque; Cardiomyocyte; Métabolisme; Heart failure; Cardiomyocyte; Metabolism

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APA (6th Edition):

Bastien, J. (2015). Impact de la protéine ADAP1 sur la survie des cardiomyocytes . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/7968

Chicago Manual of Style (16th Edition):

Bastien, Jean-Guillaume. “Impact de la protéine ADAP1 sur la survie des cardiomyocytes .” 2015. Masters Thesis, Université de Sherbrooke. Accessed April 05, 2020. http://hdl.handle.net/11143/7968.

MLA Handbook (7th Edition):

Bastien, Jean-Guillaume. “Impact de la protéine ADAP1 sur la survie des cardiomyocytes .” 2015. Web. 05 Apr 2020.

Vancouver:

Bastien J. Impact de la protéine ADAP1 sur la survie des cardiomyocytes . [Internet] [Masters thesis]. Université de Sherbrooke; 2015. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/11143/7968.

Council of Science Editors:

Bastien J. Impact de la protéine ADAP1 sur la survie des cardiomyocytes . [Masters Thesis]. Université de Sherbrooke; 2015. Available from: http://hdl.handle.net/11143/7968


Dalhousie University

4. Adamczyk, Andrew. CHARACTERIZATION OF THE PHOSPHODIESTERASE SUBTYPES THAT REGULATE MOUSE ATRIAL MYOCYTE ELECTROPHYSIOLOGY.

Degree: MS, Department of Physiology & Biophysics, 2012, Dalhousie University

 Phosphodiesterases (PDEs) are the enzymes responsible for the hydrolysis of cyclic nucleotides including cAMP and cGMP. We recently discovered that natriuretic peptides elicit effects in… (more)

Subjects/Keywords: Electrophysiology; Cardiomyocyte; Action Potential; Calcium Current; Phosphodiesterase

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APA (6th Edition):

Adamczyk, A. (2012). CHARACTERIZATION OF THE PHOSPHODIESTERASE SUBTYPES THAT REGULATE MOUSE ATRIAL MYOCYTE ELECTROPHYSIOLOGY. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/15708

Chicago Manual of Style (16th Edition):

Adamczyk, Andrew. “CHARACTERIZATION OF THE PHOSPHODIESTERASE SUBTYPES THAT REGULATE MOUSE ATRIAL MYOCYTE ELECTROPHYSIOLOGY.” 2012. Masters Thesis, Dalhousie University. Accessed April 05, 2020. http://hdl.handle.net/10222/15708.

MLA Handbook (7th Edition):

Adamczyk, Andrew. “CHARACTERIZATION OF THE PHOSPHODIESTERASE SUBTYPES THAT REGULATE MOUSE ATRIAL MYOCYTE ELECTROPHYSIOLOGY.” 2012. Web. 05 Apr 2020.

Vancouver:

Adamczyk A. CHARACTERIZATION OF THE PHOSPHODIESTERASE SUBTYPES THAT REGULATE MOUSE ATRIAL MYOCYTE ELECTROPHYSIOLOGY. [Internet] [Masters thesis]. Dalhousie University; 2012. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/10222/15708.

Council of Science Editors:

Adamczyk A. CHARACTERIZATION OF THE PHOSPHODIESTERASE SUBTYPES THAT REGULATE MOUSE ATRIAL MYOCYTE ELECTROPHYSIOLOGY. [Masters Thesis]. Dalhousie University; 2012. Available from: http://hdl.handle.net/10222/15708


Boston University

5. Schwab, John H. Prolonged alterations of cardiomyocyte gene expression following low dose high charge and energy particle radiation – implications for future deep space travel.

Degree: MS, Medical Sciences, 2015, Boston University

 INTRODUCTION: Space exploration is ultra-hazardous and unpredictably dangerous. One area of significant concern is the exposure of astronauts to deep space radiation and the development… (more)

Subjects/Keywords: Medicine; Cardiology; Cardiomyocyte; Physiology; Radiation; Space

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APA (6th Edition):

Schwab, J. H. (2015). Prolonged alterations of cardiomyocyte gene expression following low dose high charge and energy particle radiation – implications for future deep space travel. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/16289

Chicago Manual of Style (16th Edition):

Schwab, John H. “Prolonged alterations of cardiomyocyte gene expression following low dose high charge and energy particle radiation – implications for future deep space travel.” 2015. Masters Thesis, Boston University. Accessed April 05, 2020. http://hdl.handle.net/2144/16289.

MLA Handbook (7th Edition):

Schwab, John H. “Prolonged alterations of cardiomyocyte gene expression following low dose high charge and energy particle radiation – implications for future deep space travel.” 2015. Web. 05 Apr 2020.

Vancouver:

Schwab JH. Prolonged alterations of cardiomyocyte gene expression following low dose high charge and energy particle radiation – implications for future deep space travel. [Internet] [Masters thesis]. Boston University; 2015. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/2144/16289.

Council of Science Editors:

Schwab JH. Prolonged alterations of cardiomyocyte gene expression following low dose high charge and energy particle radiation – implications for future deep space travel. [Masters Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16289


University of California – Irvine

6. Davis, Chase. Design and Development of a Tool to Investigate the Cellular Response to Dynamic Mechanical Stimulation.

Degree: Biomedical Engineering, 2014, University of California – Irvine

 A novel, inexpensive, and uniformly characterized uniaxial stretch device was developed to study the cellular response to mechanical stretch. This device showed a pure uniaxial… (more)

Subjects/Keywords: Biomedical engineering; Biomechanics; Cardiomyocyte; macrophage; Strain; Stretch

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APA (6th Edition):

Davis, C. (2014). Design and Development of a Tool to Investigate the Cellular Response to Dynamic Mechanical Stimulation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/662115pr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davis, Chase. “Design and Development of a Tool to Investigate the Cellular Response to Dynamic Mechanical Stimulation.” 2014. Thesis, University of California – Irvine. Accessed April 05, 2020. http://www.escholarship.org/uc/item/662115pr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davis, Chase. “Design and Development of a Tool to Investigate the Cellular Response to Dynamic Mechanical Stimulation.” 2014. Web. 05 Apr 2020.

Vancouver:

Davis C. Design and Development of a Tool to Investigate the Cellular Response to Dynamic Mechanical Stimulation. [Internet] [Thesis]. University of California – Irvine; 2014. [cited 2020 Apr 05]. Available from: http://www.escholarship.org/uc/item/662115pr.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davis C. Design and Development of a Tool to Investigate the Cellular Response to Dynamic Mechanical Stimulation. [Thesis]. University of California – Irvine; 2014. Available from: http://www.escholarship.org/uc/item/662115pr

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Osaka University

7. 于, 涛; Yu, Tao. In vivo differentiation of induced pluripotent stem cell-derived Cardiomyocytes : 人工多能性幹細胞由来心筋細胞の生体内における分化; ジンコウ タノウセイ サイボウ ユライ シンキン サイボウ ノ セイタイ 二 オケル ブンカ.

Degree: 博士(保健学), 2013, Osaka University

Tao Yu, Shigeru Miyagawa, Kenji Miki, Atsuhiro Saito, Satsuki Fukushima, Takahiro Higuchi, Masashi Kawamura, Takuji Kawamura, Emiko Ito, Naomasa Kawaguchi, Yoshiki Sawa, Nariaki Matsuura, In Vivo Differentiation of Induced Pluripotent Stem Cell-Derived Cardiomyocytes. Circulation Journal, 77(5), 1297-1306, 2013

博士(保健学)

2013-03-25

大阪大学

14401甲第16340号

26126

Subjects/Keywords: iPS; Cardiomyocyte; Adhesion molecules; Cytoskeleton; Ultrastructure

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APA (6th Edition):

于, 涛; Yu, T. (2013). In vivo differentiation of induced pluripotent stem cell-derived Cardiomyocytes : 人工多能性幹細胞由来心筋細胞の生体内における分化; ジンコウ タノウセイ サイボウ ユライ シンキン サイボウ ノ セイタイ 二 オケル ブンカ. (Thesis). Osaka University. Retrieved from http://hdl.handle.net/11094/50611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

于, 涛; Yu, Tao. “In vivo differentiation of induced pluripotent stem cell-derived Cardiomyocytes : 人工多能性幹細胞由来心筋細胞の生体内における分化; ジンコウ タノウセイ サイボウ ユライ シンキン サイボウ ノ セイタイ 二 オケル ブンカ.” 2013. Thesis, Osaka University. Accessed April 05, 2020. http://hdl.handle.net/11094/50611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

于, 涛; Yu, Tao. “In vivo differentiation of induced pluripotent stem cell-derived Cardiomyocytes : 人工多能性幹細胞由来心筋細胞の生体内における分化; ジンコウ タノウセイ サイボウ ユライ シンキン サイボウ ノ セイタイ 二 オケル ブンカ.” 2013. Web. 05 Apr 2020.

Vancouver:

于, 涛; Yu T. In vivo differentiation of induced pluripotent stem cell-derived Cardiomyocytes : 人工多能性幹細胞由来心筋細胞の生体内における分化; ジンコウ タノウセイ サイボウ ユライ シンキン サイボウ ノ セイタイ 二 オケル ブンカ. [Internet] [Thesis]. Osaka University; 2013. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/11094/50611.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

于, 涛; Yu T. In vivo differentiation of induced pluripotent stem cell-derived Cardiomyocytes : 人工多能性幹細胞由来心筋細胞の生体内における分化; ジンコウ タノウセイ サイボウ ユライ シンキン サイボウ ノ セイタイ 二 オケル ブンカ. [Thesis]. Osaka University; 2013. Available from: http://hdl.handle.net/11094/50611

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Sherbrooke

8. Emond-Boisjoly, Marc-Alexandre. Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes .

Degree: 2016, Université de Sherbrooke

 Résumé: L’autophagie est un processus essentiel au maintien de l’homéostasie cellulaire. Elle permet de dégrader et recycler aussi bien des organelles entières que des composants… (more)

Subjects/Keywords: Autophagie; ERK; DUSP; Cardiomyocyte; Autophagy; p38; Starvation

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APA (6th Edition):

Emond-Boisjoly, M. (2016). Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes . (Masters Thesis). Université de Sherbrooke. Retrieved from http://hdl.handle.net/11143/8908

Chicago Manual of Style (16th Edition):

Emond-Boisjoly, Marc-Alexandre. “Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes .” 2016. Masters Thesis, Université de Sherbrooke. Accessed April 05, 2020. http://hdl.handle.net/11143/8908.

MLA Handbook (7th Edition):

Emond-Boisjoly, Marc-Alexandre. “Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes .” 2016. Web. 05 Apr 2020.

Vancouver:

Emond-Boisjoly M. Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes . [Internet] [Masters thesis]. Université de Sherbrooke; 2016. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/11143/8908.

Council of Science Editors:

Emond-Boisjoly M. Rôle de la protéine DUSP5 dans l’autophagie des cardiomyocytes . [Masters Thesis]. Université de Sherbrooke; 2016. Available from: http://hdl.handle.net/11143/8908


Université de Sherbrooke

9. Plamondon, Philippe. La MAP kinase p38γ influence la structure des cardiomyocytes .

Degree: 2014, Université de Sherbrooke

 Le cœur est un organe central au fonctionnement du système cardiovasculaire. Il est physiologiquement compartimenté et est constitué de cellules spécialisées qui régulent les impulsions… (more)

Subjects/Keywords: MAP kinase; p38; Cardiomyocyte; Localisation; Domaine PDZ

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APA (6th Edition):

Plamondon, P. (2014). La MAP kinase p38γ influence la structure des cardiomyocytes . (Masters Thesis). Université de Sherbrooke. Retrieved from http://savoirs.usherbrooke.ca/handle/11143/5307

Chicago Manual of Style (16th Edition):

Plamondon, Philippe. “La MAP kinase p38γ influence la structure des cardiomyocytes .” 2014. Masters Thesis, Université de Sherbrooke. Accessed April 05, 2020. http://savoirs.usherbrooke.ca/handle/11143/5307.

MLA Handbook (7th Edition):

Plamondon, Philippe. “La MAP kinase p38γ influence la structure des cardiomyocytes .” 2014. Web. 05 Apr 2020.

Vancouver:

Plamondon P. La MAP kinase p38γ influence la structure des cardiomyocytes . [Internet] [Masters thesis]. Université de Sherbrooke; 2014. [cited 2020 Apr 05]. Available from: http://savoirs.usherbrooke.ca/handle/11143/5307.

Council of Science Editors:

Plamondon P. La MAP kinase p38γ influence la structure des cardiomyocytes . [Masters Thesis]. Université de Sherbrooke; 2014. Available from: http://savoirs.usherbrooke.ca/handle/11143/5307


Vanderbilt University

10. Feaster, Tromondae Kenta. Implementation of human-induced pluripotent stem cell-derived cardiomyocyte to model excitation-contraction coupling in health and disease.

Degree: PhD, Pharmacology, 2015, Vanderbilt University

 Human induced pluripotent stem cell derived cardiomyocytes (hiPSC-CMs) have the potential to be an important tool for cardiovascular disease modeling, pre-clinical cardiotoxicity evaluation, and drug… (more)

Subjects/Keywords: excitation-contraction coupling; stem cell-derived cardiomyocyte

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APA (6th Edition):

Feaster, T. K. (2015). Implementation of human-induced pluripotent stem cell-derived cardiomyocyte to model excitation-contraction coupling in health and disease. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-11182015-125322/ ;

Chicago Manual of Style (16th Edition):

Feaster, Tromondae Kenta. “Implementation of human-induced pluripotent stem cell-derived cardiomyocyte to model excitation-contraction coupling in health and disease.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 05, 2020. http://etd.library.vanderbilt.edu/available/etd-11182015-125322/ ;.

MLA Handbook (7th Edition):

Feaster, Tromondae Kenta. “Implementation of human-induced pluripotent stem cell-derived cardiomyocyte to model excitation-contraction coupling in health and disease.” 2015. Web. 05 Apr 2020.

Vancouver:

Feaster TK. Implementation of human-induced pluripotent stem cell-derived cardiomyocyte to model excitation-contraction coupling in health and disease. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2020 Apr 05]. Available from: http://etd.library.vanderbilt.edu/available/etd-11182015-125322/ ;.

Council of Science Editors:

Feaster TK. Implementation of human-induced pluripotent stem cell-derived cardiomyocyte to model excitation-contraction coupling in health and disease. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu/available/etd-11182015-125322/ ;


Clemson University

11. Yu, Tiffany. Microfabricated Substrate to Achieve In Vivo-Like Cardiomyocyte Morphology and Electrical Propagation in Neonatal Cell Culture.

Degree: MS, Bioengineering, 2019, Clemson University

  Because of the complexity of the in vivo environment, much physiological and pathological understanding of the human body is obtained through cell culture. To… (more)

Subjects/Keywords: calcium cycling; cardiomyocyte; micropatterning; morphology; polydimethylsiloxane; wrinkles

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APA (6th Edition):

Yu, T. (2019). Microfabricated Substrate to Achieve In Vivo-Like Cardiomyocyte Morphology and Electrical Propagation in Neonatal Cell Culture. (Masters Thesis). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_theses/3219

Chicago Manual of Style (16th Edition):

Yu, Tiffany. “Microfabricated Substrate to Achieve In Vivo-Like Cardiomyocyte Morphology and Electrical Propagation in Neonatal Cell Culture.” 2019. Masters Thesis, Clemson University. Accessed April 05, 2020. https://tigerprints.clemson.edu/all_theses/3219.

MLA Handbook (7th Edition):

Yu, Tiffany. “Microfabricated Substrate to Achieve In Vivo-Like Cardiomyocyte Morphology and Electrical Propagation in Neonatal Cell Culture.” 2019. Web. 05 Apr 2020.

Vancouver:

Yu T. Microfabricated Substrate to Achieve In Vivo-Like Cardiomyocyte Morphology and Electrical Propagation in Neonatal Cell Culture. [Internet] [Masters thesis]. Clemson University; 2019. [cited 2020 Apr 05]. Available from: https://tigerprints.clemson.edu/all_theses/3219.

Council of Science Editors:

Yu T. Microfabricated Substrate to Achieve In Vivo-Like Cardiomyocyte Morphology and Electrical Propagation in Neonatal Cell Culture. [Masters Thesis]. Clemson University; 2019. Available from: https://tigerprints.clemson.edu/all_theses/3219


University of Louisville

12. Kilfoil, Peter Joseph. Modulation of cardiac Kv currents by Kvbeta2 and pyridine nucleotides.

Degree: PhD, 2016, University of Louisville

  Myocardial voltage-gated potassium (Kv) channels regulate the resting membrane potential and the repolarization phase of the action potential. Members of the Kv1 and Kv4… (more)

Subjects/Keywords: electrophysiology; action potential; potassium; cardiomyocyte; repolarization; Cardiology

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APA (6th Edition):

Kilfoil, P. J. (2016). Modulation of cardiac Kv currents by Kvbeta2 and pyridine nucleotides. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2400 ; https://ir.library.louisville.edu/etd/2400

Chicago Manual of Style (16th Edition):

Kilfoil, Peter Joseph. “Modulation of cardiac Kv currents by Kvbeta2 and pyridine nucleotides.” 2016. Doctoral Dissertation, University of Louisville. Accessed April 05, 2020. 10.18297/etd/2400 ; https://ir.library.louisville.edu/etd/2400.

MLA Handbook (7th Edition):

Kilfoil, Peter Joseph. “Modulation of cardiac Kv currents by Kvbeta2 and pyridine nucleotides.” 2016. Web. 05 Apr 2020.

Vancouver:

Kilfoil PJ. Modulation of cardiac Kv currents by Kvbeta2 and pyridine nucleotides. [Internet] [Doctoral dissertation]. University of Louisville; 2016. [cited 2020 Apr 05]. Available from: 10.18297/etd/2400 ; https://ir.library.louisville.edu/etd/2400.

Council of Science Editors:

Kilfoil PJ. Modulation of cardiac Kv currents by Kvbeta2 and pyridine nucleotides. [Doctoral Dissertation]. University of Louisville; 2016. Available from: 10.18297/etd/2400 ; https://ir.library.louisville.edu/etd/2400

13. Cho, Hee Ho. Investigation of Mechanisms Responsible for Myocyte Cell Death in Metabolic Syndrome.

Degree: MSc -MS, Biology, 2018, York University

 The aim of study is to investigate the mechanism involved in myocyte cell death in metabolic syndrome. Specific metabolic syndrome conditions focused on in this… (more)

Subjects/Keywords: Metabolic syndrome; Autophagy; Apoptosis; Skeletal muscle; Cardiomyocyte

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APA (6th Edition):

Cho, H. H. (2018). Investigation of Mechanisms Responsible for Myocyte Cell Death in Metabolic Syndrome. (Masters Thesis). York University. Retrieved from http://hdl.handle.net/10315/34523

Chicago Manual of Style (16th Edition):

Cho, Hee Ho. “Investigation of Mechanisms Responsible for Myocyte Cell Death in Metabolic Syndrome.” 2018. Masters Thesis, York University. Accessed April 05, 2020. http://hdl.handle.net/10315/34523.

MLA Handbook (7th Edition):

Cho, Hee Ho. “Investigation of Mechanisms Responsible for Myocyte Cell Death in Metabolic Syndrome.” 2018. Web. 05 Apr 2020.

Vancouver:

Cho HH. Investigation of Mechanisms Responsible for Myocyte Cell Death in Metabolic Syndrome. [Internet] [Masters thesis]. York University; 2018. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/10315/34523.

Council of Science Editors:

Cho HH. Investigation of Mechanisms Responsible for Myocyte Cell Death in Metabolic Syndrome. [Masters Thesis]. York University; 2018. Available from: http://hdl.handle.net/10315/34523


University of Toronto

14. Buchsbaum, Diana. Characterizing Function of Transmembrane Protein 65 in Mouse Neonatal Ventricular Cardiomycytes.

Degree: 2016, University of Toronto

The cardiomyocyte (CM) intercalated disc (ICD).is fundamental for signal propagation, cell adhesion, and transfer of molecules. Here we investigate ventricular protein Transmembrane protein 65 (Tmem65),… (more)

Subjects/Keywords: Cardiomyocyte; Cx43; Intercalated Disc; Ventricle; 0379

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APA (6th Edition):

Buchsbaum, D. (2016). Characterizing Function of Transmembrane Protein 65 in Mouse Neonatal Ventricular Cardiomycytes. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/79699

Chicago Manual of Style (16th Edition):

Buchsbaum, Diana. “Characterizing Function of Transmembrane Protein 65 in Mouse Neonatal Ventricular Cardiomycytes.” 2016. Masters Thesis, University of Toronto. Accessed April 05, 2020. http://hdl.handle.net/1807/79699.

MLA Handbook (7th Edition):

Buchsbaum, Diana. “Characterizing Function of Transmembrane Protein 65 in Mouse Neonatal Ventricular Cardiomycytes.” 2016. Web. 05 Apr 2020.

Vancouver:

Buchsbaum D. Characterizing Function of Transmembrane Protein 65 in Mouse Neonatal Ventricular Cardiomycytes. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/1807/79699.

Council of Science Editors:

Buchsbaum D. Characterizing Function of Transmembrane Protein 65 in Mouse Neonatal Ventricular Cardiomycytes. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/79699


University of Minnesota

15. Wheelwright, Matthew. Human iPSC-Derived Cardiac Myocytes: Toward an In Vitro Model of Cardiac Physiology.

Degree: PhD, Integrative Biology and Physiology, 2017, University of Minnesota

 Cardiovascular Disease is a growing public health issue in the modern world, with a high incidence rate that continues to increase, and poor mortality rates.… (more)

Subjects/Keywords: Cardiomyocyte; Development; Force; Genome Editing; Stem Cell

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APA (6th Edition):

Wheelwright, M. (2017). Human iPSC-Derived Cardiac Myocytes: Toward an In Vitro Model of Cardiac Physiology. (Doctoral Dissertation). University of Minnesota. Retrieved from http://hdl.handle.net/11299/188928

Chicago Manual of Style (16th Edition):

Wheelwright, Matthew. “Human iPSC-Derived Cardiac Myocytes: Toward an In Vitro Model of Cardiac Physiology.” 2017. Doctoral Dissertation, University of Minnesota. Accessed April 05, 2020. http://hdl.handle.net/11299/188928.

MLA Handbook (7th Edition):

Wheelwright, Matthew. “Human iPSC-Derived Cardiac Myocytes: Toward an In Vitro Model of Cardiac Physiology.” 2017. Web. 05 Apr 2020.

Vancouver:

Wheelwright M. Human iPSC-Derived Cardiac Myocytes: Toward an In Vitro Model of Cardiac Physiology. [Internet] [Doctoral dissertation]. University of Minnesota; 2017. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/11299/188928.

Council of Science Editors:

Wheelwright M. Human iPSC-Derived Cardiac Myocytes: Toward an In Vitro Model of Cardiac Physiology. [Doctoral Dissertation]. University of Minnesota; 2017. Available from: http://hdl.handle.net/11299/188928

16. Cauquil, Marie. Rôle d'éphrine-B1 dans la morphologie et l'état post-mitotique du cardiomyocyte adulte : potentiel en régénération cardiaque : Role of Ephrin-B1 in adult CM morphology and post-mitotic state : potential in heart regeneration.

Degree: Docteur es, Physiopathologie, 2018, Université Toulouse III – Paul Sabatier

L'insuffisance cardiaque (IC) est l'évolution terminale de nombreuses pathologies cardiaques, notamment de l'infarctus du myocarde (IDM). Elle se caractérise par la mort des cellules contractiles… (more)

Subjects/Keywords: Cardiomyocyte; Prolifération; Régénération; Insuffisance cardiaque; Polarité; Cardiomyocyte; Proliferation; Regeneration; Heart failure; Polarity

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APA (6th Edition):

Cauquil, M. (2018). Rôle d'éphrine-B1 dans la morphologie et l'état post-mitotique du cardiomyocyte adulte : potentiel en régénération cardiaque : Role of Ephrin-B1 in adult CM morphology and post-mitotic state : potential in heart regeneration. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2018TOU30280

Chicago Manual of Style (16th Edition):

Cauquil, Marie. “Rôle d'éphrine-B1 dans la morphologie et l'état post-mitotique du cardiomyocyte adulte : potentiel en régénération cardiaque : Role of Ephrin-B1 in adult CM morphology and post-mitotic state : potential in heart regeneration.” 2018. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed April 05, 2020. http://www.theses.fr/2018TOU30280.

MLA Handbook (7th Edition):

Cauquil, Marie. “Rôle d'éphrine-B1 dans la morphologie et l'état post-mitotique du cardiomyocyte adulte : potentiel en régénération cardiaque : Role of Ephrin-B1 in adult CM morphology and post-mitotic state : potential in heart regeneration.” 2018. Web. 05 Apr 2020.

Vancouver:

Cauquil M. Rôle d'éphrine-B1 dans la morphologie et l'état post-mitotique du cardiomyocyte adulte : potentiel en régénération cardiaque : Role of Ephrin-B1 in adult CM morphology and post-mitotic state : potential in heart regeneration. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2018. [cited 2020 Apr 05]. Available from: http://www.theses.fr/2018TOU30280.

Council of Science Editors:

Cauquil M. Rôle d'éphrine-B1 dans la morphologie et l'état post-mitotique du cardiomyocyte adulte : potentiel en régénération cardiaque : Role of Ephrin-B1 in adult CM morphology and post-mitotic state : potential in heart regeneration. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2018. Available from: http://www.theses.fr/2018TOU30280


University of Toronto

17. Lee, Jee Hoon. Modeling the Development of Human Atrial and Ventricular Cardiomyocyte Lineages from Human Pluripotent Stem Cells.

Degree: PhD, 2018, University of Toronto

 The ability to direct the differentiation of human pluripotent stem cells (hPSCs) to the different cardiovascular lineages is a prerequisite to modeling specific forms of… (more)

Subjects/Keywords: atrial cardiomyocyte; heart; Human pluripotent stem cell; regenerative medicine; ventricular cardiomyocyte; 0306

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APA (6th Edition):

Lee, J. H. (2018). Modeling the Development of Human Atrial and Ventricular Cardiomyocyte Lineages from Human Pluripotent Stem Cells. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/91983

Chicago Manual of Style (16th Edition):

Lee, Jee Hoon. “Modeling the Development of Human Atrial and Ventricular Cardiomyocyte Lineages from Human Pluripotent Stem Cells.” 2018. Doctoral Dissertation, University of Toronto. Accessed April 05, 2020. http://hdl.handle.net/1807/91983.

MLA Handbook (7th Edition):

Lee, Jee Hoon. “Modeling the Development of Human Atrial and Ventricular Cardiomyocyte Lineages from Human Pluripotent Stem Cells.” 2018. Web. 05 Apr 2020.

Vancouver:

Lee JH. Modeling the Development of Human Atrial and Ventricular Cardiomyocyte Lineages from Human Pluripotent Stem Cells. [Internet] [Doctoral dissertation]. University of Toronto; 2018. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/1807/91983.

Council of Science Editors:

Lee JH. Modeling the Development of Human Atrial and Ventricular Cardiomyocyte Lineages from Human Pluripotent Stem Cells. [Doctoral Dissertation]. University of Toronto; 2018. Available from: http://hdl.handle.net/1807/91983


Temple University

18. Wang, Fang. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.

Degree: PhD, 2012, Temple University

Physiology

Cardiovascular disease remains the number one cause or mortally in the western world. Heart failure is the most rapidly growing cardiovascular disease (Hobbs, 2004;… (more)

Subjects/Keywords: Physiology; Calcium Current; Cardiomyocyte Proliferation; Electrophysiology; Flow Cytometry; Neonatal Mice Ventricular Cardiomyocyte; T-type calcium channel

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APA (6th Edition):

Wang, F. (2012). DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214814

Chicago Manual of Style (16th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Doctoral Dissertation, Temple University. Accessed April 05, 2020. http://digital.library.temple.edu/u?/p245801coll10,214814.

MLA Handbook (7th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Web. 05 Apr 2020.

Vancouver:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Apr 05]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814.

Council of Science Editors:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814

19. Pasqualin, Côme. Dynamique calcique dans les cardiomyocytes de veines pulmonaires de rat : une hétérogénéité source d'arythmie ? : Calcium dynamics in pulmonary vein cardiac myocytes of the rat : an heterogeneity related source of arrhythmias ?.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2016, Université François-Rabelais de Tours

Les activités électriques ectopiques à l’origine des épisodes de fibrillation atriale pourraient être dues à des échanges calciques anormaux dans les cardiomyocytes (CM) de veine… (more)

Subjects/Keywords: Fibrillation atriale; Cardiomyocyte; Dynamique calcique; Contraction; Microscopie confocale; Analyse d’image; Atrial fibrillation; Cardiomyocyte; Calcium dynamics; Contraction; Confocal microscopy; Image analysis

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APA (6th Edition):

Pasqualin, C. (2016). Dynamique calcique dans les cardiomyocytes de veines pulmonaires de rat : une hétérogénéité source d'arythmie ? : Calcium dynamics in pulmonary vein cardiac myocytes of the rat : an heterogeneity related source of arrhythmias ?. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2016TOUR3808

Chicago Manual of Style (16th Edition):

Pasqualin, Côme. “Dynamique calcique dans les cardiomyocytes de veines pulmonaires de rat : une hétérogénéité source d'arythmie ? : Calcium dynamics in pulmonary vein cardiac myocytes of the rat : an heterogeneity related source of arrhythmias ?.” 2016. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed April 05, 2020. http://www.theses.fr/2016TOUR3808.

MLA Handbook (7th Edition):

Pasqualin, Côme. “Dynamique calcique dans les cardiomyocytes de veines pulmonaires de rat : une hétérogénéité source d'arythmie ? : Calcium dynamics in pulmonary vein cardiac myocytes of the rat : an heterogeneity related source of arrhythmias ?.” 2016. Web. 05 Apr 2020.

Vancouver:

Pasqualin C. Dynamique calcique dans les cardiomyocytes de veines pulmonaires de rat : une hétérogénéité source d'arythmie ? : Calcium dynamics in pulmonary vein cardiac myocytes of the rat : an heterogeneity related source of arrhythmias ?. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2016. [cited 2020 Apr 05]. Available from: http://www.theses.fr/2016TOUR3808.

Council of Science Editors:

Pasqualin C. Dynamique calcique dans les cardiomyocytes de veines pulmonaires de rat : une hétérogénéité source d'arythmie ? : Calcium dynamics in pulmonary vein cardiac myocytes of the rat : an heterogeneity related source of arrhythmias ?. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2016. Available from: http://www.theses.fr/2016TOUR3808


Brno University of Technology

20. Boudová, Markéta. Segmentace buněk ve snímcích z vysokorychlostní digitální kamery .

Degree: 2019, Brno University of Technology

 V první části se práce zabývá mikroskopickými metodami snímání srdečních buněk. V druhé části jsou popsány segmentační techniky zpracování obrazu. Praktická část je zaměřena na… (more)

Subjects/Keywords: Mikroskopie; kardiomyocyt; zpracování obrazu; segmentace obrazu; kontrakce kardiomyocytu; analýza kontraktility; Microscopy; cardiomyocyte; image processing; image segmentation; cardiomyocyte contraction; contractility analysis

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APA (6th Edition):

Boudová, M. (2019). Segmentace buněk ve snímcích z vysokorychlostní digitální kamery . (Thesis). Brno University of Technology. Retrieved from http://hdl.handle.net/11012/173638

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Boudová, Markéta. “Segmentace buněk ve snímcích z vysokorychlostní digitální kamery .” 2019. Thesis, Brno University of Technology. Accessed April 05, 2020. http://hdl.handle.net/11012/173638.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Boudová, Markéta. “Segmentace buněk ve snímcích z vysokorychlostní digitální kamery .” 2019. Web. 05 Apr 2020.

Vancouver:

Boudová M. Segmentace buněk ve snímcích z vysokorychlostní digitální kamery . [Internet] [Thesis]. Brno University of Technology; 2019. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/11012/173638.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Boudová M. Segmentace buněk ve snímcích z vysokorychlostní digitální kamery . [Thesis]. Brno University of Technology; 2019. Available from: http://hdl.handle.net/11012/173638

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Chadet, Stéphanie. Rôle du récepteur purinergique P2Y11 dans la modulation du phénotype des cellules dendritiques et la survie des cardiomyocytes en situation d'hypoxie/réoxygénation : The role of P2Y11 receptor in the modulation of dendritic cell phenotype and cardiomyocyte survival during hypoxia/reoxygenation.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2015, Université François-Rabelais de Tours

Les cellules dendritiques (DCs) possèdent des rôles clés dans la modulation de la réponse inflammatoire. Leur implication dans la réponse inflammatoire post-ischémie/reperfusion semble claire. Cependant,… (more)

Subjects/Keywords: Cellule dendritique; Cardiomyocyte; Récepteur P2Y11; Immunomodulation; Signaux de danger; Dendritic cell; Cardiomyocyte; P2Y11 receptor; Immunomodulation; Danger signal

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APA (6th Edition):

Chadet, S. (2015). Rôle du récepteur purinergique P2Y11 dans la modulation du phénotype des cellules dendritiques et la survie des cardiomyocytes en situation d'hypoxie/réoxygénation : The role of P2Y11 receptor in the modulation of dendritic cell phenotype and cardiomyocyte survival during hypoxia/reoxygenation. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2015TOUR3306

Chicago Manual of Style (16th Edition):

Chadet, Stéphanie. “Rôle du récepteur purinergique P2Y11 dans la modulation du phénotype des cellules dendritiques et la survie des cardiomyocytes en situation d'hypoxie/réoxygénation : The role of P2Y11 receptor in the modulation of dendritic cell phenotype and cardiomyocyte survival during hypoxia/reoxygenation.” 2015. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed April 05, 2020. http://www.theses.fr/2015TOUR3306.

MLA Handbook (7th Edition):

Chadet, Stéphanie. “Rôle du récepteur purinergique P2Y11 dans la modulation du phénotype des cellules dendritiques et la survie des cardiomyocytes en situation d'hypoxie/réoxygénation : The role of P2Y11 receptor in the modulation of dendritic cell phenotype and cardiomyocyte survival during hypoxia/reoxygenation.” 2015. Web. 05 Apr 2020.

Vancouver:

Chadet S. Rôle du récepteur purinergique P2Y11 dans la modulation du phénotype des cellules dendritiques et la survie des cardiomyocytes en situation d'hypoxie/réoxygénation : The role of P2Y11 receptor in the modulation of dendritic cell phenotype and cardiomyocyte survival during hypoxia/reoxygenation. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2015. [cited 2020 Apr 05]. Available from: http://www.theses.fr/2015TOUR3306.

Council of Science Editors:

Chadet S. Rôle du récepteur purinergique P2Y11 dans la modulation du phénotype des cellules dendritiques et la survie des cardiomyocytes en situation d'hypoxie/réoxygénation : The role of P2Y11 receptor in the modulation of dendritic cell phenotype and cardiomyocyte survival during hypoxia/reoxygenation. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2015. Available from: http://www.theses.fr/2015TOUR3306

22. Benoist, Lauriane. Rôle du récepteur purinergique P2Y11 dans la modulation des lésions d'Ischémie/Reperfusion myocardique : Role of P2Y11 purinergic receptor on the modulation of myocardial ischemia/reperfusion injuries.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2017, Université François-Rabelais de Tours

L’ischémie/reperfusion (I/R) induit des lésions impliquées dans la physiopathologie de la transplantation cardiaque où elles contribuent à augmenter le rejet de greffe. Le stress induit… (more)

Subjects/Keywords: Ischémie/Reperfusion; Cellule dendritique; Cardiomyocyte; Récepteur P2Y11; Immunomodulation; Cardioprotection; Transplantation; Ischemia/Reperfusion; Dendritic cell; Cardiomyocyte; P2Y11 receptor; Immunomodulation; Cardioprotection; Transplantation

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APA (6th Edition):

Benoist, L. (2017). Rôle du récepteur purinergique P2Y11 dans la modulation des lésions d'Ischémie/Reperfusion myocardique : Role of P2Y11 purinergic receptor on the modulation of myocardial ischemia/reperfusion injuries. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2017TOUR3310

Chicago Manual of Style (16th Edition):

Benoist, Lauriane. “Rôle du récepteur purinergique P2Y11 dans la modulation des lésions d'Ischémie/Reperfusion myocardique : Role of P2Y11 purinergic receptor on the modulation of myocardial ischemia/reperfusion injuries.” 2017. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed April 05, 2020. http://www.theses.fr/2017TOUR3310.

MLA Handbook (7th Edition):

Benoist, Lauriane. “Rôle du récepteur purinergique P2Y11 dans la modulation des lésions d'Ischémie/Reperfusion myocardique : Role of P2Y11 purinergic receptor on the modulation of myocardial ischemia/reperfusion injuries.” 2017. Web. 05 Apr 2020.

Vancouver:

Benoist L. Rôle du récepteur purinergique P2Y11 dans la modulation des lésions d'Ischémie/Reperfusion myocardique : Role of P2Y11 purinergic receptor on the modulation of myocardial ischemia/reperfusion injuries. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2017. [cited 2020 Apr 05]. Available from: http://www.theses.fr/2017TOUR3310.

Council of Science Editors:

Benoist L. Rôle du récepteur purinergique P2Y11 dans la modulation des lésions d'Ischémie/Reperfusion myocardique : Role of P2Y11 purinergic receptor on the modulation of myocardial ischemia/reperfusion injuries. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2017. Available from: http://www.theses.fr/2017TOUR3310


University of Pennsylvania

23. Majkut, Stephanie Feldman. Mechanical Development and Functional Mechanosensitivity During Early Cardiogenesis.

Degree: 2013, University of Pennsylvania

 This thesis addresses the questions of when and how mechanical stiffness arises during embryonic heart development and how mechanics affects early cardiomyocyte and myocardium contractile… (more)

Subjects/Keywords: cardiomyocyte; cytoskeletal organization; ECM; embryo; Lamin; mechanosensitivity; Biophysics; Developmental Biology

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APA (6th Edition):

Majkut, S. F. (2013). Mechanical Development and Functional Mechanosensitivity During Early Cardiogenesis. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/895

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Majkut, Stephanie Feldman. “Mechanical Development and Functional Mechanosensitivity During Early Cardiogenesis.” 2013. Thesis, University of Pennsylvania. Accessed April 05, 2020. https://repository.upenn.edu/edissertations/895.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Majkut, Stephanie Feldman. “Mechanical Development and Functional Mechanosensitivity During Early Cardiogenesis.” 2013. Web. 05 Apr 2020.

Vancouver:

Majkut SF. Mechanical Development and Functional Mechanosensitivity During Early Cardiogenesis. [Internet] [Thesis]. University of Pennsylvania; 2013. [cited 2020 Apr 05]. Available from: https://repository.upenn.edu/edissertations/895.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Majkut SF. Mechanical Development and Functional Mechanosensitivity During Early Cardiogenesis. [Thesis]. University of Pennsylvania; 2013. Available from: https://repository.upenn.edu/edissertations/895

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

24. Zhang, Lianghui. Mechanisms for Phospholipase Cε-Dependent Regulation of Cardiac Hypertrophy.

Degree: PhD, 2013, University of Rochester

 To explore the mechanisms of phospholipase Cε (PLCε) signaling in cardiomyocyte hypertrophy, PLCε protein was depleted by siRNA in neonatal rat ventricular myocytes (NRVMs). Endothelin-1… (more)

Subjects/Keywords: Phospholipase Cε; Hypertrophy; Cardiomyocyte; PkD; Nuclear Calcium; Inducible; Knock-out; Mice

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APA (6th Edition):

Zhang, L. (2013). Mechanisms for Phospholipase Cε-Dependent Regulation of Cardiac Hypertrophy. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/26774

Chicago Manual of Style (16th Edition):

Zhang, Lianghui. “Mechanisms for Phospholipase Cε-Dependent Regulation of Cardiac Hypertrophy.” 2013. Doctoral Dissertation, University of Rochester. Accessed April 05, 2020. http://hdl.handle.net/1802/26774.

MLA Handbook (7th Edition):

Zhang, Lianghui. “Mechanisms for Phospholipase Cε-Dependent Regulation of Cardiac Hypertrophy.” 2013. Web. 05 Apr 2020.

Vancouver:

Zhang L. Mechanisms for Phospholipase Cε-Dependent Regulation of Cardiac Hypertrophy. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/1802/26774.

Council of Science Editors:

Zhang L. Mechanisms for Phospholipase Cε-Dependent Regulation of Cardiac Hypertrophy. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/26774


Boston University

25. Estrella, Nelsa Leonor. Gene programs regulated by MEF2 transcription factors in rodent striated muscle cells.

Degree: PhD, Biology, 2015, Boston University

 Transcriptional programs regulating myogenesis are multi-layered, requiring carefully orchestrated temporal activation of a wide range of myogenic transcription factors for proper muscle formation. The MEF2… (more)

Subjects/Keywords: Cellular biology; Cardiomyocyte; Cell cycle; Differentiation; Transcription factor; Transcriptomics; Cell survival

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APA (6th Edition):

Estrella, N. L. (2015). Gene programs regulated by MEF2 transcription factors in rodent striated muscle cells. (Doctoral Dissertation). Boston University. Retrieved from http://hdl.handle.net/2144/16346

Chicago Manual of Style (16th Edition):

Estrella, Nelsa Leonor. “Gene programs regulated by MEF2 transcription factors in rodent striated muscle cells.” 2015. Doctoral Dissertation, Boston University. Accessed April 05, 2020. http://hdl.handle.net/2144/16346.

MLA Handbook (7th Edition):

Estrella, Nelsa Leonor. “Gene programs regulated by MEF2 transcription factors in rodent striated muscle cells.” 2015. Web. 05 Apr 2020.

Vancouver:

Estrella NL. Gene programs regulated by MEF2 transcription factors in rodent striated muscle cells. [Internet] [Doctoral dissertation]. Boston University; 2015. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/2144/16346.

Council of Science Editors:

Estrella NL. Gene programs regulated by MEF2 transcription factors in rodent striated muscle cells. [Doctoral Dissertation]. Boston University; 2015. Available from: http://hdl.handle.net/2144/16346


Universiteit Utrecht

26. Vliet, Patrick van. Progenitor cells from the heart - Cellulae Progenitores Cordis.

Degree: 2009, Universiteit Utrecht

 Myocardial infarction is a leading cause of high mortality rates in Western countries. After ischemia, lost cardiomyocytes are replaced by fibrotic tissue, while remaining cardiomyocytes… (more)

Subjects/Keywords: Geneeskunde; Cardiomyocyte; progenitor cell; proliferation; differentiation; multipotency; electrophysiology; microRNA; Survivin

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APA (6th Edition):

Vliet, P. v. (2009). Progenitor cells from the heart - Cellulae Progenitores Cordis. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/33258

Chicago Manual of Style (16th Edition):

Vliet, Patrick van. “Progenitor cells from the heart - Cellulae Progenitores Cordis.” 2009. Doctoral Dissertation, Universiteit Utrecht. Accessed April 05, 2020. http://dspace.library.uu.nl:8080/handle/1874/33258.

MLA Handbook (7th Edition):

Vliet, Patrick van. “Progenitor cells from the heart - Cellulae Progenitores Cordis.” 2009. Web. 05 Apr 2020.

Vancouver:

Vliet Pv. Progenitor cells from the heart - Cellulae Progenitores Cordis. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2009. [cited 2020 Apr 05]. Available from: http://dspace.library.uu.nl:8080/handle/1874/33258.

Council of Science Editors:

Vliet Pv. Progenitor cells from the heart - Cellulae Progenitores Cordis. [Doctoral Dissertation]. Universiteit Utrecht; 2009. Available from: http://dspace.library.uu.nl:8080/handle/1874/33258


University of California – Irvine

27. Fong, Ashley Heather. The role of the cardiac microenvironment during cardiomyocyte development and maturation.

Degree: Biological Sciences, 2016, University of California – Irvine

 The use of induced pluripotent stem cell (iPSC)-derived cardiomyocytes (CM) has great potential for developing novel therapies to treat cardiovascular disease (CVD). Unfortunately, CM derived… (more)

Subjects/Keywords: Biology; 3D culturing; cardiomyocyte; endothelial cells; extracellular matrix; maturation; stem cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fong, A. H. (2016). The role of the cardiac microenvironment during cardiomyocyte development and maturation. (Thesis). University of California – Irvine. Retrieved from http://www.escholarship.org/uc/item/9fp225c1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fong, Ashley Heather. “The role of the cardiac microenvironment during cardiomyocyte development and maturation.” 2016. Thesis, University of California – Irvine. Accessed April 05, 2020. http://www.escholarship.org/uc/item/9fp225c1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fong, Ashley Heather. “The role of the cardiac microenvironment during cardiomyocyte development and maturation.” 2016. Web. 05 Apr 2020.

Vancouver:

Fong AH. The role of the cardiac microenvironment during cardiomyocyte development and maturation. [Internet] [Thesis]. University of California – Irvine; 2016. [cited 2020 Apr 05]. Available from: http://www.escholarship.org/uc/item/9fp225c1.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fong AH. The role of the cardiac microenvironment during cardiomyocyte development and maturation. [Thesis]. University of California – Irvine; 2016. Available from: http://www.escholarship.org/uc/item/9fp225c1

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Pellman, Jason. A Desmosomal Protein Interaction Screen Uncovers a Novel Mechanism Underlying Cardiac Rhythm Disorders.

Degree: Biomedical Sciences, 2017, University of California – San Diego

 Arrhythmogenic right ventricular cardiomyopathy (ARVC) is a genetic based cardiac disease, which is characterized by ventricular dysfunction, fibrofatty accumulation, and ventricular arrhythmias culminating in sudden… (more)

Subjects/Keywords: Cellular biology; Physiology; Autophagy; Cardiac Arrhythmia; Cardiomyocyte; Desmosome; Gap Junction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pellman, J. (2017). A Desmosomal Protein Interaction Screen Uncovers a Novel Mechanism Underlying Cardiac Rhythm Disorders. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/6jm2q2jn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pellman, Jason. “A Desmosomal Protein Interaction Screen Uncovers a Novel Mechanism Underlying Cardiac Rhythm Disorders.” 2017. Thesis, University of California – San Diego. Accessed April 05, 2020. http://www.escholarship.org/uc/item/6jm2q2jn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pellman, Jason. “A Desmosomal Protein Interaction Screen Uncovers a Novel Mechanism Underlying Cardiac Rhythm Disorders.” 2017. Web. 05 Apr 2020.

Vancouver:

Pellman J. A Desmosomal Protein Interaction Screen Uncovers a Novel Mechanism Underlying Cardiac Rhythm Disorders. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2020 Apr 05]. Available from: http://www.escholarship.org/uc/item/6jm2q2jn.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pellman J. A Desmosomal Protein Interaction Screen Uncovers a Novel Mechanism Underlying Cardiac Rhythm Disorders. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/6jm2q2jn

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Clemson University

29. Liu, Honghai. Assembly and Disassembly of Myofibrils in Dissociated Cardiomyocytes.

Degree: PhD, Bioengineering, 2012, Clemson University

 Significance 1) We have developed a hybrid TPEF-SHG imaging system with an onstage incubator for long-term living-cell imaging. Using the imaging system, the assembly of… (more)

Subjects/Keywords: Cardiomyocyte; Dedifferentiation; Myofibrillogenesis; Second Harmonic Generation; Biomedical Engineering and Bioengineering

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liu, H. (2012). Assembly and Disassembly of Myofibrils in Dissociated Cardiomyocytes. (Doctoral Dissertation). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_dissertations/939

Chicago Manual of Style (16th Edition):

Liu, Honghai. “Assembly and Disassembly of Myofibrils in Dissociated Cardiomyocytes.” 2012. Doctoral Dissertation, Clemson University. Accessed April 05, 2020. https://tigerprints.clemson.edu/all_dissertations/939.

MLA Handbook (7th Edition):

Liu, Honghai. “Assembly and Disassembly of Myofibrils in Dissociated Cardiomyocytes.” 2012. Web. 05 Apr 2020.

Vancouver:

Liu H. Assembly and Disassembly of Myofibrils in Dissociated Cardiomyocytes. [Internet] [Doctoral dissertation]. Clemson University; 2012. [cited 2020 Apr 05]. Available from: https://tigerprints.clemson.edu/all_dissertations/939.

Council of Science Editors:

Liu H. Assembly and Disassembly of Myofibrils in Dissociated Cardiomyocytes. [Doctoral Dissertation]. Clemson University; 2012. Available from: https://tigerprints.clemson.edu/all_dissertations/939


University of Illinois – Chicago

30. Mkrtschjan, Michael Andrew. Mechanical Inputs to Cardiac Fibroblasts and Myocytes Affect Structure, Function, and Signaling Response.

Degree: 2018, University of Illinois – Chicago

 Tissues within the body are subject to a variety of physical and chemical cues, many of which play roles in maintaining physiological homeostasis or triggering… (more)

Subjects/Keywords: Mechanobiology; Stiffness; Actin; CapZ; PIP2; PKC epsilon; Fibroblast; Cardiomyocyte; Migration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mkrtschjan, M. A. (2018). Mechanical Inputs to Cardiac Fibroblasts and Myocytes Affect Structure, Function, and Signaling Response. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/23166

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mkrtschjan, Michael Andrew. “Mechanical Inputs to Cardiac Fibroblasts and Myocytes Affect Structure, Function, and Signaling Response.” 2018. Thesis, University of Illinois – Chicago. Accessed April 05, 2020. http://hdl.handle.net/10027/23166.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mkrtschjan, Michael Andrew. “Mechanical Inputs to Cardiac Fibroblasts and Myocytes Affect Structure, Function, and Signaling Response.” 2018. Web. 05 Apr 2020.

Vancouver:

Mkrtschjan MA. Mechanical Inputs to Cardiac Fibroblasts and Myocytes Affect Structure, Function, and Signaling Response. [Internet] [Thesis]. University of Illinois – Chicago; 2018. [cited 2020 Apr 05]. Available from: http://hdl.handle.net/10027/23166.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mkrtschjan MA. Mechanical Inputs to Cardiac Fibroblasts and Myocytes Affect Structure, Function, and Signaling Response. [Thesis]. University of Illinois – Chicago; 2018. Available from: http://hdl.handle.net/10027/23166

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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