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You searched for subject:(Cardiac Hypertrophy). Showing records 1 – 30 of 169 total matches.

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Universiteit Utrecht

1. Pruissen, F. The role of miRNAs in cardiac hypertrophy.

Degree: 2013, Universiteit Utrecht

Cardiac hypertrophy and increased cardiomyocytes size are important triggers in the pathophysiology in heart failure. In this review, the role of miRNAs in cardiac hypertrophy(more)

Subjects/Keywords: microRNA; cardiac hypertrophy

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APA (6th Edition):

Pruissen, F. (2013). The role of miRNAs in cardiac hypertrophy. (Masters Thesis). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/272438

Chicago Manual of Style (16th Edition):

Pruissen, F. “The role of miRNAs in cardiac hypertrophy.” 2013. Masters Thesis, Universiteit Utrecht. Accessed September 21, 2019. http://dspace.library.uu.nl:8080/handle/1874/272438.

MLA Handbook (7th Edition):

Pruissen, F. “The role of miRNAs in cardiac hypertrophy.” 2013. Web. 21 Sep 2019.

Vancouver:

Pruissen F. The role of miRNAs in cardiac hypertrophy. [Internet] [Masters thesis]. Universiteit Utrecht; 2013. [cited 2019 Sep 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/272438.

Council of Science Editors:

Pruissen F. The role of miRNAs in cardiac hypertrophy. [Masters Thesis]. Universiteit Utrecht; 2013. Available from: http://dspace.library.uu.nl:8080/handle/1874/272438


University of Ottawa

2. Putinski, Charis. Caspase-dependent Signaling as an Inductive Cue for Cardiac Hypertrophy .

Degree: 2018, University of Ottawa

 The heart has the remarkable ability to adjust in response to varying stress stimuli and myocardium enlargement, referred to as cardiac hypertrophy, is a common… (more)

Subjects/Keywords: Cardiac Hypertrophy; Caspase

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APA (6th Edition):

Putinski, C. (2018). Caspase-dependent Signaling as an Inductive Cue for Cardiac Hypertrophy . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/37728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Putinski, Charis. “Caspase-dependent Signaling as an Inductive Cue for Cardiac Hypertrophy .” 2018. Thesis, University of Ottawa. Accessed September 21, 2019. http://hdl.handle.net/10393/37728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Putinski, Charis. “Caspase-dependent Signaling as an Inductive Cue for Cardiac Hypertrophy .” 2018. Web. 21 Sep 2019.

Vancouver:

Putinski C. Caspase-dependent Signaling as an Inductive Cue for Cardiac Hypertrophy . [Internet] [Thesis]. University of Ottawa; 2018. [cited 2019 Sep 21]. Available from: http://hdl.handle.net/10393/37728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Putinski C. Caspase-dependent Signaling as an Inductive Cue for Cardiac Hypertrophy . [Thesis]. University of Ottawa; 2018. Available from: http://hdl.handle.net/10393/37728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

3. Stiles, Rebecca. A Novel Non-Apoptotic Role for Caspase Activity during Cardiac Hypertrophy .

Degree: 2011, University of Ottawa

Cardiac hypertrophy is an adaptive response in which the heart grows to normalize output during times of increased demand. This increase in size originates from… (more)

Subjects/Keywords: Caspase; Cardiac hypertrophy

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APA (6th Edition):

Stiles, R. (2011). A Novel Non-Apoptotic Role for Caspase Activity during Cardiac Hypertrophy . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/20023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stiles, Rebecca. “A Novel Non-Apoptotic Role for Caspase Activity during Cardiac Hypertrophy .” 2011. Thesis, University of Ottawa. Accessed September 21, 2019. http://hdl.handle.net/10393/20023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stiles, Rebecca. “A Novel Non-Apoptotic Role for Caspase Activity during Cardiac Hypertrophy .” 2011. Web. 21 Sep 2019.

Vancouver:

Stiles R. A Novel Non-Apoptotic Role for Caspase Activity during Cardiac Hypertrophy . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2019 Sep 21]. Available from: http://hdl.handle.net/10393/20023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stiles R. A Novel Non-Apoptotic Role for Caspase Activity during Cardiac Hypertrophy . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/20023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

4. Siauw, Willy Soesanto. Effect of high fat low carbohydrate diet on blood pressure and the development of cardiac hypertrophy in spontaneously hypertensive rats.

Degree: MS;, Nutrition;, 2009, University of Utah

 The current dietary recommendation for a healthy heart and prevention of heart disease focuses mostly on low fat and high carbohydrate foods. However, previous studies… (more)

Subjects/Keywords: Blood pressure; Heart; Cardiac hypertrophy

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APA (6th Edition):

Siauw, W. S. (2009). Effect of high fat low carbohydrate diet on blood pressure and the development of cardiac hypertrophy in spontaneously hypertensive rats. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/2096/rec/374

Chicago Manual of Style (16th Edition):

Siauw, Willy Soesanto. “Effect of high fat low carbohydrate diet on blood pressure and the development of cardiac hypertrophy in spontaneously hypertensive rats.” 2009. Masters Thesis, University of Utah. Accessed September 21, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/2096/rec/374.

MLA Handbook (7th Edition):

Siauw, Willy Soesanto. “Effect of high fat low carbohydrate diet on blood pressure and the development of cardiac hypertrophy in spontaneously hypertensive rats.” 2009. Web. 21 Sep 2019.

Vancouver:

Siauw WS. Effect of high fat low carbohydrate diet on blood pressure and the development of cardiac hypertrophy in spontaneously hypertensive rats. [Internet] [Masters thesis]. University of Utah; 2009. [cited 2019 Sep 21]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/2096/rec/374.

Council of Science Editors:

Siauw WS. Effect of high fat low carbohydrate diet on blood pressure and the development of cardiac hypertrophy in spontaneously hypertensive rats. [Masters Thesis]. University of Utah; 2009. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/2096/rec/374


University of Alberta

5. Alsaad,Abdulaziz M. Role of CYP-mediated Arachidonic Acid Metabolites in Development of Cardiac Hypertrophy and Chronic Doxorubicin-induced Cardiotoxicity.

Degree: MS, Faculty of Pharmacy and Pharmaceutical Sciences, 2012, University of Alberta

 Drug-induced cardiotoxicity is classified as a predisposing factor leading to cardiac hypertrophy and heart failure. Of importance, doxorubicin (DOX, adriamycin) is considered as an effective… (more)

Subjects/Keywords: Arachidonic acid; cardiac hypertrophy; cardiotoxicity

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APA (6th Edition):

M, A. (2012). Role of CYP-mediated Arachidonic Acid Metabolites in Development of Cardiac Hypertrophy and Chronic Doxorubicin-induced Cardiotoxicity. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/n870zs18z

Chicago Manual of Style (16th Edition):

M, Alsaad,Abdulaziz. “Role of CYP-mediated Arachidonic Acid Metabolites in Development of Cardiac Hypertrophy and Chronic Doxorubicin-induced Cardiotoxicity.” 2012. Masters Thesis, University of Alberta. Accessed September 21, 2019. https://era.library.ualberta.ca/files/n870zs18z.

MLA Handbook (7th Edition):

M, Alsaad,Abdulaziz. “Role of CYP-mediated Arachidonic Acid Metabolites in Development of Cardiac Hypertrophy and Chronic Doxorubicin-induced Cardiotoxicity.” 2012. Web. 21 Sep 2019.

Vancouver:

M A. Role of CYP-mediated Arachidonic Acid Metabolites in Development of Cardiac Hypertrophy and Chronic Doxorubicin-induced Cardiotoxicity. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2019 Sep 21]. Available from: https://era.library.ualberta.ca/files/n870zs18z.

Council of Science Editors:

M A. Role of CYP-mediated Arachidonic Acid Metabolites in Development of Cardiac Hypertrophy and Chronic Doxorubicin-induced Cardiotoxicity. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/n870zs18z


University of Alberta

6. Jesus Cadete, Virgilio Jorge. Energy metabolism in the hypertrophied newborn rabbit heart.

Degree: MS, Medical Sciences - Pediatrics, 2010, University of Alberta

 The newborn heart possesses a higher tolerance to ischemia in comparison to adult hearts. Post-ischemic interventions that increase energy production are beneficial for recovery. These… (more)

Subjects/Keywords: heart metabolism; cardiac hypertrophy; newborn

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APA (6th Edition):

Jesus Cadete, V. J. (2010). Energy metabolism in the hypertrophied newborn rabbit heart. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/7m01bk919

Chicago Manual of Style (16th Edition):

Jesus Cadete, Virgilio Jorge. “Energy metabolism in the hypertrophied newborn rabbit heart.” 2010. Masters Thesis, University of Alberta. Accessed September 21, 2019. https://era.library.ualberta.ca/files/7m01bk919.

MLA Handbook (7th Edition):

Jesus Cadete, Virgilio Jorge. “Energy metabolism in the hypertrophied newborn rabbit heart.” 2010. Web. 21 Sep 2019.

Vancouver:

Jesus Cadete VJ. Energy metabolism in the hypertrophied newborn rabbit heart. [Internet] [Masters thesis]. University of Alberta; 2010. [cited 2019 Sep 21]. Available from: https://era.library.ualberta.ca/files/7m01bk919.

Council of Science Editors:

Jesus Cadete VJ. Energy metabolism in the hypertrophied newborn rabbit heart. [Masters Thesis]. University of Alberta; 2010. Available from: https://era.library.ualberta.ca/files/7m01bk919

7. Jenie Riris Istighfari. Increased ubiquitin-conjugation in cardiac myocytes and the involvement of crosstalk of G protein signaling : 心筋細胞におけるユビキチン化の増加とGタンパク質シグナルのクロストーク; シンキン サイボウ ニ オケル ユビキチンカ ノ ゾウカ ト Gタンパクシツ シグナル ノ クロストーク.

Degree: 博士(バイオサイエンス), Nara Institute of Science and Technology / 奈良先端科学技術大学院大学

Subjects/Keywords: Cardiac hypertrophy

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APA (6th Edition):

Istighfari, J. R. (n.d.). Increased ubiquitin-conjugation in cardiac myocytes and the involvement of crosstalk of G protein signaling : 心筋細胞におけるユビキチン化の増加とGタンパク質シグナルのクロストーク; シンキン サイボウ ニ オケル ユビキチンカ ノ ゾウカ ト Gタンパクシツ シグナル ノ クロストーク. (Thesis). Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Retrieved from http://hdl.handle.net/10061/9178

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Istighfari, Jenie Riris. “Increased ubiquitin-conjugation in cardiac myocytes and the involvement of crosstalk of G protein signaling : 心筋細胞におけるユビキチン化の増加とGタンパク質シグナルのクロストーク; シンキン サイボウ ニ オケル ユビキチンカ ノ ゾウカ ト Gタンパクシツ シグナル ノ クロストーク.” Thesis, Nara Institute of Science and Technology / 奈良先端科学技術大学院大学. Accessed September 21, 2019. http://hdl.handle.net/10061/9178.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Istighfari, Jenie Riris. “Increased ubiquitin-conjugation in cardiac myocytes and the involvement of crosstalk of G protein signaling : 心筋細胞におけるユビキチン化の増加とGタンパク質シグナルのクロストーク; シンキン サイボウ ニ オケル ユビキチンカ ノ ゾウカ ト Gタンパクシツ シグナル ノ クロストーク.” Web. 21 Sep 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Istighfari JR. Increased ubiquitin-conjugation in cardiac myocytes and the involvement of crosstalk of G protein signaling : 心筋細胞におけるユビキチン化の増加とGタンパク質シグナルのクロストーク; シンキン サイボウ ニ オケル ユビキチンカ ノ ゾウカ ト Gタンパクシツ シグナル ノ クロストーク. [Internet] [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; [cited 2019 Sep 21]. Available from: http://hdl.handle.net/10061/9178.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Istighfari JR. Increased ubiquitin-conjugation in cardiac myocytes and the involvement of crosstalk of G protein signaling : 心筋細胞におけるユビキチン化の増加とGタンパク質シグナルのクロストーク; シンキン サイボウ ニ オケル ユビキチンカ ノ ゾウカ ト Gタンパクシツ シグナル ノ クロストーク. [Thesis]. Nara Institute of Science and Technology / 奈良先端科学技術大学院大学; Available from: http://hdl.handle.net/10061/9178

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

8. Holden, Patricia. Characterization Of Pregnancy Induced Cardiac Hypertrophy In Rats.

Degree: MS, Biological Sciences, 2011, Encompass Digital Archive, Eastern Kentucky University

  During pregnancy, an increase in blood volume occurs to compensate for fetal development, resulting in cardiac hypertrophy. For the majority of women, this physiological… (more)

Subjects/Keywords: Cardiac; Cardiac Hypertrophy; Gene Expression; Hypertrophy; miRNA; Pregnancy; Molecular Biology

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APA (6th Edition):

Holden, P. (2011). Characterization Of Pregnancy Induced Cardiac Hypertrophy In Rats. (Masters Thesis). Encompass Digital Archive, Eastern Kentucky University. Retrieved from https://encompass.eku.edu/etd/35

Chicago Manual of Style (16th Edition):

Holden, Patricia. “Characterization Of Pregnancy Induced Cardiac Hypertrophy In Rats.” 2011. Masters Thesis, Encompass Digital Archive, Eastern Kentucky University. Accessed September 21, 2019. https://encompass.eku.edu/etd/35.

MLA Handbook (7th Edition):

Holden, Patricia. “Characterization Of Pregnancy Induced Cardiac Hypertrophy In Rats.” 2011. Web. 21 Sep 2019.

Vancouver:

Holden P. Characterization Of Pregnancy Induced Cardiac Hypertrophy In Rats. [Internet] [Masters thesis]. Encompass Digital Archive, Eastern Kentucky University; 2011. [cited 2019 Sep 21]. Available from: https://encompass.eku.edu/etd/35.

Council of Science Editors:

Holden P. Characterization Of Pregnancy Induced Cardiac Hypertrophy In Rats. [Masters Thesis]. Encompass Digital Archive, Eastern Kentucky University; 2011. Available from: https://encompass.eku.edu/etd/35


University of Utah

9. Huang, Yujia. Effect of antioxidant treatment on cardiac hypertrophy and antioxidant enzyme expression in cardiac specific glut4 deleted mice.

Degree: MS, Nutrition, 2011, University of Utah

 We examined mice with cardiac specific deletion of the gene encoding glucose transporter 4 (GLUT4, G4H-/-). These mice develop moderate cardiac hypertrophy with normal basal… (more)

Subjects/Keywords: Antioxidant; Cardiac hypertrophy; Glut4 deleted mice

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APA (6th Edition):

Huang, Y. (2011). Effect of antioxidant treatment on cardiac hypertrophy and antioxidant enzyme expression in cardiac specific glut4 deleted mice. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/261/rec/794

Chicago Manual of Style (16th Edition):

Huang, Yujia. “Effect of antioxidant treatment on cardiac hypertrophy and antioxidant enzyme expression in cardiac specific glut4 deleted mice.” 2011. Masters Thesis, University of Utah. Accessed September 21, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/261/rec/794.

MLA Handbook (7th Edition):

Huang, Yujia. “Effect of antioxidant treatment on cardiac hypertrophy and antioxidant enzyme expression in cardiac specific glut4 deleted mice.” 2011. Web. 21 Sep 2019.

Vancouver:

Huang Y. Effect of antioxidant treatment on cardiac hypertrophy and antioxidant enzyme expression in cardiac specific glut4 deleted mice. [Internet] [Masters thesis]. University of Utah; 2011. [cited 2019 Sep 21]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/261/rec/794.

Council of Science Editors:

Huang Y. Effect of antioxidant treatment on cardiac hypertrophy and antioxidant enzyme expression in cardiac specific glut4 deleted mice. [Masters Thesis]. University of Utah; 2011. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/261/rec/794


University of Alberta

10. Wang, Xiang. Metalloproteinases in the Development of Hypertension and Cardiac Remodeling.

Degree: PhD, Department of Biochemistry, 2013, University of Alberta

 Despite many decades of research and drug development, the diseases of the cardiovascular system remain a major health threat in the modern world. Hypertensive cardiac(more)

Subjects/Keywords: Metalloproteinases; cardaic fibrosis; cardiac hypertrophy; Hypertension

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APA (6th Edition):

Wang, X. (2013). Metalloproteinases in the Development of Hypertension and Cardiac Remodeling. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/gt54kp54k

Chicago Manual of Style (16th Edition):

Wang, Xiang. “Metalloproteinases in the Development of Hypertension and Cardiac Remodeling.” 2013. Doctoral Dissertation, University of Alberta. Accessed September 21, 2019. https://era.library.ualberta.ca/files/gt54kp54k.

MLA Handbook (7th Edition):

Wang, Xiang. “Metalloproteinases in the Development of Hypertension and Cardiac Remodeling.” 2013. Web. 21 Sep 2019.

Vancouver:

Wang X. Metalloproteinases in the Development of Hypertension and Cardiac Remodeling. [Internet] [Doctoral dissertation]. University of Alberta; 2013. [cited 2019 Sep 21]. Available from: https://era.library.ualberta.ca/files/gt54kp54k.

Council of Science Editors:

Wang X. Metalloproteinases in the Development of Hypertension and Cardiac Remodeling. [Doctoral Dissertation]. University of Alberta; 2013. Available from: https://era.library.ualberta.ca/files/gt54kp54k


Temple University

11. Troupes, Constantine. The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction.

Degree: PhD, 2016, Temple University

Biomedical Sciences

Increases in cardiac afterload caused by disease conditions results in remodeling of heart structure by hypertrophy and alterations in the molecular regulation of… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Troupes, C. (2016). The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,403786

Chicago Manual of Style (16th Edition):

Troupes, Constantine. “The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction.” 2016. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,403786.

MLA Handbook (7th Edition):

Troupes, Constantine. “The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction.” 2016. Web. 21 Sep 2019.

Vancouver:

Troupes C. The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,403786.

Council of Science Editors:

Troupes C. The Role of STIM1 in Hypertrophy-Related Contractile Dysfunction. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,403786


University of Manchester

12. Stafford, Nicholas Pierre. The independent roles of PMCA1 and PMCA4 in the development and progression of left ventricular hypertrophy and failure.

Degree: PhD, 2014, University of Manchester

 Heart failure is responsible for one in twenty deaths in the UK, and as the average age of the general population increases that number is… (more)

Subjects/Keywords: 616.1; PMCA; cardiac hypertrophy; heart failure

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APA (6th Edition):

Stafford, N. P. (2014). The independent roles of PMCA1 and PMCA4 in the development and progression of left ventricular hypertrophy and failure. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/the-independent-roles-of-pmca1-and-pmca4-in-the-development-and-progression-of-left-ventricular-hypertrophy-and-failure(bd746ae0-8189-433a-8a0e-ecb4506afe62).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607415

Chicago Manual of Style (16th Edition):

Stafford, Nicholas Pierre. “The independent roles of PMCA1 and PMCA4 in the development and progression of left ventricular hypertrophy and failure.” 2014. Doctoral Dissertation, University of Manchester. Accessed September 21, 2019. https://www.research.manchester.ac.uk/portal/en/theses/the-independent-roles-of-pmca1-and-pmca4-in-the-development-and-progression-of-left-ventricular-hypertrophy-and-failure(bd746ae0-8189-433a-8a0e-ecb4506afe62).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607415.

MLA Handbook (7th Edition):

Stafford, Nicholas Pierre. “The independent roles of PMCA1 and PMCA4 in the development and progression of left ventricular hypertrophy and failure.” 2014. Web. 21 Sep 2019.

Vancouver:

Stafford NP. The independent roles of PMCA1 and PMCA4 in the development and progression of left ventricular hypertrophy and failure. [Internet] [Doctoral dissertation]. University of Manchester; 2014. [cited 2019 Sep 21]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-independent-roles-of-pmca1-and-pmca4-in-the-development-and-progression-of-left-ventricular-hypertrophy-and-failure(bd746ae0-8189-433a-8a0e-ecb4506afe62).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607415.

Council of Science Editors:

Stafford NP. The independent roles of PMCA1 and PMCA4 in the development and progression of left ventricular hypertrophy and failure. [Doctoral Dissertation]. University of Manchester; 2014. Available from: https://www.research.manchester.ac.uk/portal/en/theses/the-independent-roles-of-pmca1-and-pmca4-in-the-development-and-progression-of-left-ventricular-hypertrophy-and-failure(bd746ae0-8189-433a-8a0e-ecb4506afe62).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.607415


Boston University

13. Sepulveda, Sean Matthew. The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes.

Degree: 2015, Boston University

 Glycogen synthase kinase-3 (GSK3) is a ubiquitously expressed protein kinase with key roles in controlling proliferation, differentiation and survival of a wide variety of mammalian… (more)

Subjects/Keywords: Biology; Cardiac hypertrophy; Glycogen synthase kinase 3

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APA (6th Edition):

Sepulveda, S. M. (2015). The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes. (Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/15686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sepulveda, Sean Matthew. “The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes.” 2015. Thesis, Boston University. Accessed September 21, 2019. http://hdl.handle.net/2144/15686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sepulveda, Sean Matthew. “The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes.” 2015. Web. 21 Sep 2019.

Vancouver:

Sepulveda SM. The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes. [Internet] [Thesis]. Boston University; 2015. [cited 2019 Sep 21]. Available from: http://hdl.handle.net/2144/15686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sepulveda SM. The role of glycogen synthase kinase-3 and camp response element-binding protein in the induction and regulation of cardiac hypertrophy in neonatal rat ventricular myocytes. [Thesis]. Boston University; 2015. Available from: http://hdl.handle.net/2144/15686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queens University

14. Armstrong, David. DEVELOPMENTAL ORIGINS OF CARDIOVASCULAR DISEASE: ATRIAL NATRIURETIC PEPTIDE GENE DISRUPTED MICE AS A MODEL OF GESTATIONAL HYPERTENSION .

Degree: Anatomy and Cell Biology, 2012, Queens University

 Introduction: Developmental origins of disease refers to the theory that adverse maternal environments influence fetal development and the risk of cardiovascular disease (CVD) in adulthood.… (more)

Subjects/Keywords: Cardiac hypertrophy; Natriuretic peptides; Hypertension; Developmental origins

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APA (6th Edition):

Armstrong, D. (2012). DEVELOPMENTAL ORIGINS OF CARDIOVASCULAR DISEASE: ATRIAL NATRIURETIC PEPTIDE GENE DISRUPTED MICE AS A MODEL OF GESTATIONAL HYPERTENSION . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/7559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Armstrong, David. “DEVELOPMENTAL ORIGINS OF CARDIOVASCULAR DISEASE: ATRIAL NATRIURETIC PEPTIDE GENE DISRUPTED MICE AS A MODEL OF GESTATIONAL HYPERTENSION .” 2012. Thesis, Queens University. Accessed September 21, 2019. http://hdl.handle.net/1974/7559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Armstrong, David. “DEVELOPMENTAL ORIGINS OF CARDIOVASCULAR DISEASE: ATRIAL NATRIURETIC PEPTIDE GENE DISRUPTED MICE AS A MODEL OF GESTATIONAL HYPERTENSION .” 2012. Web. 21 Sep 2019.

Vancouver:

Armstrong D. DEVELOPMENTAL ORIGINS OF CARDIOVASCULAR DISEASE: ATRIAL NATRIURETIC PEPTIDE GENE DISRUPTED MICE AS A MODEL OF GESTATIONAL HYPERTENSION . [Internet] [Thesis]. Queens University; 2012. [cited 2019 Sep 21]. Available from: http://hdl.handle.net/1974/7559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Armstrong D. DEVELOPMENTAL ORIGINS OF CARDIOVASCULAR DISEASE: ATRIAL NATRIURETIC PEPTIDE GENE DISRUPTED MICE AS A MODEL OF GESTATIONAL HYPERTENSION . [Thesis]. Queens University; 2012. Available from: http://hdl.handle.net/1974/7559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Clemson University

15. Yuan, Cai. Polarization-Resolved Second Harmonic Generation Microscopy Study of Cardiac Hypertrophy.

Degree: PhD, Bioengineering, 2019, Clemson University

  As the most important structural and functional protein of cardiomyocyte, myosin is sensitive to mechanical and hormonal stimulus during cardiac hypertrophy. Accumulating evidence suggests… (more)

Subjects/Keywords: Cardiac Hypertrophy; Myosin; Polarization; Second Harmonic Generation

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APA (6th Edition):

Yuan, C. (2019). Polarization-Resolved Second Harmonic Generation Microscopy Study of Cardiac Hypertrophy. (Doctoral Dissertation). Clemson University. Retrieved from https://tigerprints.clemson.edu/all_dissertations/2418

Chicago Manual of Style (16th Edition):

Yuan, Cai. “Polarization-Resolved Second Harmonic Generation Microscopy Study of Cardiac Hypertrophy.” 2019. Doctoral Dissertation, Clemson University. Accessed September 21, 2019. https://tigerprints.clemson.edu/all_dissertations/2418.

MLA Handbook (7th Edition):

Yuan, Cai. “Polarization-Resolved Second Harmonic Generation Microscopy Study of Cardiac Hypertrophy.” 2019. Web. 21 Sep 2019.

Vancouver:

Yuan C. Polarization-Resolved Second Harmonic Generation Microscopy Study of Cardiac Hypertrophy. [Internet] [Doctoral dissertation]. Clemson University; 2019. [cited 2019 Sep 21]. Available from: https://tigerprints.clemson.edu/all_dissertations/2418.

Council of Science Editors:

Yuan C. Polarization-Resolved Second Harmonic Generation Microscopy Study of Cardiac Hypertrophy. [Doctoral Dissertation]. Clemson University; 2019. Available from: https://tigerprints.clemson.edu/all_dissertations/2418


University of Alberta

16. Odenbach, Jeffrey. Matrix metalloproteinase-2 mediates angiotensin II-induced hypertension.

Degree: MS, Department of Biochemistry, 2011, University of Alberta

 Angiotensin II signals cardiovascular disease through metalloproteinases including MMP-2, MMP-7 and ADAM-17/TACE. We hypothesized that these metalloproteinases regulate each other at the transcriptional level. Further,… (more)

Subjects/Keywords: RNA interference; cardiac fibrosis; hypertension; angiotensin II; cardiac hypertrophy; matrix metalloproteinase; hypertensive cardiac remodelling

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APA (6th Edition):

Odenbach, J. (2011). Matrix metalloproteinase-2 mediates angiotensin II-induced hypertension. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/kw52j8687

Chicago Manual of Style (16th Edition):

Odenbach, Jeffrey. “Matrix metalloproteinase-2 mediates angiotensin II-induced hypertension.” 2011. Masters Thesis, University of Alberta. Accessed September 21, 2019. https://era.library.ualberta.ca/files/kw52j8687.

MLA Handbook (7th Edition):

Odenbach, Jeffrey. “Matrix metalloproteinase-2 mediates angiotensin II-induced hypertension.” 2011. Web. 21 Sep 2019.

Vancouver:

Odenbach J. Matrix metalloproteinase-2 mediates angiotensin II-induced hypertension. [Internet] [Masters thesis]. University of Alberta; 2011. [cited 2019 Sep 21]. Available from: https://era.library.ualberta.ca/files/kw52j8687.

Council of Science Editors:

Odenbach J. Matrix metalloproteinase-2 mediates angiotensin II-induced hypertension. [Masters Thesis]. University of Alberta; 2011. Available from: https://era.library.ualberta.ca/files/kw52j8687


University of Oulu

17. Kelloniemi, A. (Annina). Novel factors regulating cardiac remodeling in experimental models of cardiac hypertrophy and failure.

Degree: 2018, University of Oulu

Abstract Cardiac loading induces left ventricular hypertrophy and cardiac remodeling which when prolonged, leads to heart failure, a complex syndrome affecting approximately 1-2% of the… (more)

Subjects/Keywords: cardiac hypertrophy; cardiac remodeling; gene expression; myocardial infarction; geenien ilmentyminen; hypertrofia; infarkti; sydämen uudelleenmuovautuminen

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APA (6th Edition):

Kelloniemi, A. (. (2018). Novel factors regulating cardiac remodeling in experimental models of cardiac hypertrophy and failure. (Doctoral Dissertation). University of Oulu. Retrieved from http://urn.fi/urn:isbn:9789526220291

Chicago Manual of Style (16th Edition):

Kelloniemi, A (Annina). “Novel factors regulating cardiac remodeling in experimental models of cardiac hypertrophy and failure.” 2018. Doctoral Dissertation, University of Oulu. Accessed September 21, 2019. http://urn.fi/urn:isbn:9789526220291.

MLA Handbook (7th Edition):

Kelloniemi, A (Annina). “Novel factors regulating cardiac remodeling in experimental models of cardiac hypertrophy and failure.” 2018. Web. 21 Sep 2019.

Vancouver:

Kelloniemi A(. Novel factors regulating cardiac remodeling in experimental models of cardiac hypertrophy and failure. [Internet] [Doctoral dissertation]. University of Oulu; 2018. [cited 2019 Sep 21]. Available from: http://urn.fi/urn:isbn:9789526220291.

Council of Science Editors:

Kelloniemi A(. Novel factors regulating cardiac remodeling in experimental models of cardiac hypertrophy and failure. [Doctoral Dissertation]. University of Oulu; 2018. Available from: http://urn.fi/urn:isbn:9789526220291


University of Kentucky

18. Byse, Miranda Jean. THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS.

Degree: 2010, University of Kentucky

 In the heart, the primary role of the L-type calcium channel (LTCC) CaV1.2 is to conduct calcium into cardiomyocytes and initiate contraction. However, part of… (more)

Subjects/Keywords: L-type Calcium Channel; Calcium Channel Block; Transcription; Cardiac Development; Cardiac Hypertrophy; Medical Physiology

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APA (6th Edition):

Byse, M. J. (2010). THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/gradschool_diss/20

Chicago Manual of Style (16th Edition):

Byse, Miranda Jean. “THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS.” 2010. Doctoral Dissertation, University of Kentucky. Accessed September 21, 2019. https://uknowledge.uky.edu/gradschool_diss/20.

MLA Handbook (7th Edition):

Byse, Miranda Jean. “THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS.” 2010. Web. 21 Sep 2019.

Vancouver:

Byse MJ. THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS. [Internet] [Doctoral dissertation]. University of Kentucky; 2010. [cited 2019 Sep 21]. Available from: https://uknowledge.uky.edu/gradschool_diss/20.

Council of Science Editors:

Byse MJ. THE ROLE OF THE L-TYPE CALCIUM CHANNEL AND ITS CARBOXYL-TERMINUS. [Doctoral Dissertation]. University of Kentucky; 2010. Available from: https://uknowledge.uky.edu/gradschool_diss/20


University of Utah

19. Bosse, John David. Effects of high-fat vs. low-fat diets on mammalian target of rapamycin signaling and metabolic measures in spontaneously hypertensive rats.

Degree: MS;, Nutrition;, 2010, University of Utah

 Preliminary studies in the Division of Nutrition;’s Nutrition; Sciences lab have shown that spontaneously hypertensive rats (SHRs) fed high-fat diets (HF; 60% kcals) have a… (more)

Subjects/Keywords: Cardiac hypertrophy; Hypertension; Insulin signaling; MTOR; Protein synthesis; High-fat diet

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APA (6th Edition):

Bosse, J. D. (2010). Effects of high-fat vs. low-fat diets on mammalian target of rapamycin signaling and metabolic measures in spontaneously hypertensive rats. (Masters Thesis). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/632/rec/396

Chicago Manual of Style (16th Edition):

Bosse, John David. “Effects of high-fat vs. low-fat diets on mammalian target of rapamycin signaling and metabolic measures in spontaneously hypertensive rats.” 2010. Masters Thesis, University of Utah. Accessed September 21, 2019. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/632/rec/396.

MLA Handbook (7th Edition):

Bosse, John David. “Effects of high-fat vs. low-fat diets on mammalian target of rapamycin signaling and metabolic measures in spontaneously hypertensive rats.” 2010. Web. 21 Sep 2019.

Vancouver:

Bosse JD. Effects of high-fat vs. low-fat diets on mammalian target of rapamycin signaling and metabolic measures in spontaneously hypertensive rats. [Internet] [Masters thesis]. University of Utah; 2010. [cited 2019 Sep 21]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/632/rec/396.

Council of Science Editors:

Bosse JD. Effects of high-fat vs. low-fat diets on mammalian target of rapamycin signaling and metabolic measures in spontaneously hypertensive rats. [Masters Thesis]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/632/rec/396

20. Camilla Pedreira da Silva. Papel do 17 β-estradiol no modelo de hipertrofia cardíaca induzida pelo isoproterenol em ratas.

Degree: 2009, Universidade Federal Rural do Rio de Janeiro

Um estudo pouco frequente no modelo de hipertrofia cardíaca induzida pelo isoproterenol, é aquele referente ao papel dos estrógenos no desenvolvimento da hipertrofia miocárdica. Portanto,… (more)

Subjects/Keywords: isoproterenol; hipertrofia; estradiol; MEDICINA VETERINARIA; cardiac hypertrophy, isoproterenol, 17 β; -estradiol

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APA (6th Edition):

Silva, C. P. d. (2009). Papel do 17 β-estradiol no modelo de hipertrofia cardíaca induzida pelo isoproterenol em ratas. (Thesis). Universidade Federal Rural do Rio de Janeiro. Retrieved from http://bdtd.ufrrj.br//tde_busca/arquivo.php?codArquivo=1331

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Camilla Pedreira da. “Papel do 17 β-estradiol no modelo de hipertrofia cardíaca induzida pelo isoproterenol em ratas.” 2009. Thesis, Universidade Federal Rural do Rio de Janeiro. Accessed September 21, 2019. http://bdtd.ufrrj.br//tde_busca/arquivo.php?codArquivo=1331.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Camilla Pedreira da. “Papel do 17 β-estradiol no modelo de hipertrofia cardíaca induzida pelo isoproterenol em ratas.” 2009. Web. 21 Sep 2019.

Vancouver:

Silva CPd. Papel do 17 β-estradiol no modelo de hipertrofia cardíaca induzida pelo isoproterenol em ratas. [Internet] [Thesis]. Universidade Federal Rural do Rio de Janeiro; 2009. [cited 2019 Sep 21]. Available from: http://bdtd.ufrrj.br//tde_busca/arquivo.php?codArquivo=1331.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva CPd. Papel do 17 β-estradiol no modelo de hipertrofia cardíaca induzida pelo isoproterenol em ratas. [Thesis]. Universidade Federal Rural do Rio de Janeiro; 2009. Available from: http://bdtd.ufrrj.br//tde_busca/arquivo.php?codArquivo=1331

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Amadeu, Marco Aurélio. Expressão de microRNAs no coração de ratos espontaneamente hipertensos (SHR) submetidos a treinamento físico aeróbio.

Degree: Mestrado, Biodinâmica do Movimento Humano, 2011, University of São Paulo

A hipertrofia cardíaca é um dos principais mecanismos de adaptação do coração frente a uma sobrecarga de trabalho e pode advir de estímulos patológicos como… (more)

Subjects/Keywords: Cardiac Hypertrophy.; Hipertrofia cardíaca; MicroRNAs; MicroRNAs; Physical training; Treinamento físico

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APA (6th Edition):

Amadeu, M. A. (2011). Expressão de microRNAs no coração de ratos espontaneamente hipertensos (SHR) submetidos a treinamento físico aeróbio. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/39/39132/tde-30012012-151733/ ;

Chicago Manual of Style (16th Edition):

Amadeu, Marco Aurélio. “Expressão de microRNAs no coração de ratos espontaneamente hipertensos (SHR) submetidos a treinamento físico aeróbio.” 2011. Masters Thesis, University of São Paulo. Accessed September 21, 2019. http://www.teses.usp.br/teses/disponiveis/39/39132/tde-30012012-151733/ ;.

MLA Handbook (7th Edition):

Amadeu, Marco Aurélio. “Expressão de microRNAs no coração de ratos espontaneamente hipertensos (SHR) submetidos a treinamento físico aeróbio.” 2011. Web. 21 Sep 2019.

Vancouver:

Amadeu MA. Expressão de microRNAs no coração de ratos espontaneamente hipertensos (SHR) submetidos a treinamento físico aeróbio. [Internet] [Masters thesis]. University of São Paulo; 2011. [cited 2019 Sep 21]. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-30012012-151733/ ;.

Council of Science Editors:

Amadeu MA. Expressão de microRNAs no coração de ratos espontaneamente hipertensos (SHR) submetidos a treinamento físico aeróbio. [Masters Thesis]. University of São Paulo; 2011. Available from: http://www.teses.usp.br/teses/disponiveis/39/39132/tde-30012012-151733/ ;


University of Alberta

22. Althurwi, Hassan N. THE ROLE OF CYTOCHROME P450 AND THEIR ASSOCIATED ARACHIDONIC ACID METABOLITES IN THE INITIATION AND PROGRESSION OF CARDIAC HYPERTROPHY.

Degree: PhD, Faculty of Pharmacy and Pharmaceutical Sciences, 2015, University of Alberta

 Heart failure (HF) is the leading cause of mortality and disability in adults worldwide. Cardiac hypertrophy is an independent risk factor and one of the… (more)

Subjects/Keywords: Cytochrome P450; cardiac hypertrophy; Soluble epoxide hydrolase; Epoxyeicosatrienoic acids

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APA (6th Edition):

Althurwi, H. N. (2015). THE ROLE OF CYTOCHROME P450 AND THEIR ASSOCIATED ARACHIDONIC ACID METABOLITES IN THE INITIATION AND PROGRESSION OF CARDIAC HYPERTROPHY. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/c3b591860n

Chicago Manual of Style (16th Edition):

Althurwi, Hassan N. “THE ROLE OF CYTOCHROME P450 AND THEIR ASSOCIATED ARACHIDONIC ACID METABOLITES IN THE INITIATION AND PROGRESSION OF CARDIAC HYPERTROPHY.” 2015. Doctoral Dissertation, University of Alberta. Accessed September 21, 2019. https://era.library.ualberta.ca/files/c3b591860n.

MLA Handbook (7th Edition):

Althurwi, Hassan N. “THE ROLE OF CYTOCHROME P450 AND THEIR ASSOCIATED ARACHIDONIC ACID METABOLITES IN THE INITIATION AND PROGRESSION OF CARDIAC HYPERTROPHY.” 2015. Web. 21 Sep 2019.

Vancouver:

Althurwi HN. THE ROLE OF CYTOCHROME P450 AND THEIR ASSOCIATED ARACHIDONIC ACID METABOLITES IN THE INITIATION AND PROGRESSION OF CARDIAC HYPERTROPHY. [Internet] [Doctoral dissertation]. University of Alberta; 2015. [cited 2019 Sep 21]. Available from: https://era.library.ualberta.ca/files/c3b591860n.

Council of Science Editors:

Althurwi HN. THE ROLE OF CYTOCHROME P450 AND THEIR ASSOCIATED ARACHIDONIC ACID METABOLITES IN THE INITIATION AND PROGRESSION OF CARDIAC HYPERTROPHY. [Doctoral Dissertation]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/c3b591860n


Universidade do Rio Grande do Sul

23. Soares, Douglas dos Santos. Hipertrofia cardíaca em camundongos submetidos à natação em diferentes volumes e intensidades de treinamento : avaliação do sistema renina angiotensina.

Degree: 2017, Universidade do Rio Grande do Sul

O exercício físico modula o sistema renina angiotensina (SRA), que tem um importante papel na fisiologia cardíaca, especialmente na promoção da hipertrofia cardíaca. O SRA… (more)

Subjects/Keywords: renin angiotensin system; Exercício; Sistema renina-angiotensina; Exercise; Cardiomegalia; Cardiac hypertrophy

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APA (6th Edition):

Soares, D. d. S. (2017). Hipertrofia cardíaca em camundongos submetidos à natação em diferentes volumes e intensidades de treinamento : avaliação do sistema renina angiotensina. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/169701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Soares, Douglas dos Santos. “Hipertrofia cardíaca em camundongos submetidos à natação em diferentes volumes e intensidades de treinamento : avaliação do sistema renina angiotensina.” 2017. Thesis, Universidade do Rio Grande do Sul. Accessed September 21, 2019. http://hdl.handle.net/10183/169701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Soares, Douglas dos Santos. “Hipertrofia cardíaca em camundongos submetidos à natação em diferentes volumes e intensidades de treinamento : avaliação do sistema renina angiotensina.” 2017. Web. 21 Sep 2019.

Vancouver:

Soares DdS. Hipertrofia cardíaca em camundongos submetidos à natação em diferentes volumes e intensidades de treinamento : avaliação do sistema renina angiotensina. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2017. [cited 2019 Sep 21]. Available from: http://hdl.handle.net/10183/169701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Soares DdS. Hipertrofia cardíaca em camundongos submetidos à natação em diferentes volumes e intensidades de treinamento : avaliação do sistema renina angiotensina. [Thesis]. Universidade do Rio Grande do Sul; 2017. Available from: http://hdl.handle.net/10183/169701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

24. Chiu, Diana Yuan Yng. Cardiac risk assessment using 2D and 3D transthoracic echocardiography in patients undergoing haemodialysis.

Degree: PhD, 2016, University of Manchester

 Haemodialysis (HD) patients have a high mortality risk and most have echocardiographic evidence of abnormal cardiac structure or function. Markers, such as left ventricular hypertrophy(more)

Subjects/Keywords: 616.1; Dialysis; Mortality; Cardiac; Echocardiography; Left ventricular hypertrophy; Strain

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APA (6th Edition):

Chiu, D. Y. Y. (2016). Cardiac risk assessment using 2D and 3D transthoracic echocardiography in patients undergoing haemodialysis. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/cardiac-risk-assessment-using-2d-and-3d-transthoracic-echocardiography-in-patients-undergoing-haemodialysis(162625bc-5375-4e8f-be10-6b1b0fd768bc).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.686795

Chicago Manual of Style (16th Edition):

Chiu, Diana Yuan Yng. “Cardiac risk assessment using 2D and 3D transthoracic echocardiography in patients undergoing haemodialysis.” 2016. Doctoral Dissertation, University of Manchester. Accessed September 21, 2019. https://www.research.manchester.ac.uk/portal/en/theses/cardiac-risk-assessment-using-2d-and-3d-transthoracic-echocardiography-in-patients-undergoing-haemodialysis(162625bc-5375-4e8f-be10-6b1b0fd768bc).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.686795.

MLA Handbook (7th Edition):

Chiu, Diana Yuan Yng. “Cardiac risk assessment using 2D and 3D transthoracic echocardiography in patients undergoing haemodialysis.” 2016. Web. 21 Sep 2019.

Vancouver:

Chiu DYY. Cardiac risk assessment using 2D and 3D transthoracic echocardiography in patients undergoing haemodialysis. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Sep 21]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/cardiac-risk-assessment-using-2d-and-3d-transthoracic-echocardiography-in-patients-undergoing-haemodialysis(162625bc-5375-4e8f-be10-6b1b0fd768bc).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.686795.

Council of Science Editors:

Chiu DYY. Cardiac risk assessment using 2D and 3D transthoracic echocardiography in patients undergoing haemodialysis. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/cardiac-risk-assessment-using-2d-and-3d-transthoracic-echocardiography-in-patients-undergoing-haemodialysis(162625bc-5375-4e8f-be10-6b1b0fd768bc).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.686795


University of Cincinnati

25. Woltz, John W. Cardiovascular Risk and Left Ventricular Hypertrophy in Firefighters.

Degree: MS, Medicine: Occupational Medicine, 2013, University of Cincinnati

 Purpose: The National Institute for Occupational Safety and Health (NIOSH) Firefighter Fatality Investigation and Prevention Program (FFFIPP) has investigated hundreds of firefighter deaths since it… (more)

Subjects/Keywords: Surgery; Firefighter; Cardiovascular Risk; Left Ventricular Hypertrophy; Sudden Cardiac Death

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APA (6th Edition):

Woltz, J. W. (2013). Cardiovascular Risk and Left Ventricular Hypertrophy in Firefighters. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1367940479

Chicago Manual of Style (16th Edition):

Woltz, John W. “Cardiovascular Risk and Left Ventricular Hypertrophy in Firefighters.” 2013. Masters Thesis, University of Cincinnati. Accessed September 21, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1367940479.

MLA Handbook (7th Edition):

Woltz, John W. “Cardiovascular Risk and Left Ventricular Hypertrophy in Firefighters.” 2013. Web. 21 Sep 2019.

Vancouver:

Woltz JW. Cardiovascular Risk and Left Ventricular Hypertrophy in Firefighters. [Internet] [Masters thesis]. University of Cincinnati; 2013. [cited 2019 Sep 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1367940479.

Council of Science Editors:

Woltz JW. Cardiovascular Risk and Left Ventricular Hypertrophy in Firefighters. [Masters Thesis]. University of Cincinnati; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1367940479


The Ohio State University

26. Varian, Kenneth Dean. Cardiac Myofilament Calcium Sensitivity in Health and Disease.

Degree: PhD, Integrated Biomedical Sciences, 2008, The Ohio State University

 Heart failure is the leading cause of death in much of the western world. Progressive deterioration of cardiac contractility and/or relaxation, resulting in cardiac output… (more)

Subjects/Keywords: Health; Cardiac; Heart Failure; Hypertrophy; Force Frequency Relationship; Myofilament Calcium Sensitivity

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APA (6th Edition):

Varian, K. D. (2008). Cardiac Myofilament Calcium Sensitivity in Health and Disease. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1211898886

Chicago Manual of Style (16th Edition):

Varian, Kenneth Dean. “Cardiac Myofilament Calcium Sensitivity in Health and Disease.” 2008. Doctoral Dissertation, The Ohio State University. Accessed September 21, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1211898886.

MLA Handbook (7th Edition):

Varian, Kenneth Dean. “Cardiac Myofilament Calcium Sensitivity in Health and Disease.” 2008. Web. 21 Sep 2019.

Vancouver:

Varian KD. Cardiac Myofilament Calcium Sensitivity in Health and Disease. [Internet] [Doctoral dissertation]. The Ohio State University; 2008. [cited 2019 Sep 21]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1211898886.

Council of Science Editors:

Varian KD. Cardiac Myofilament Calcium Sensitivity in Health and Disease. [Doctoral Dissertation]. The Ohio State University; 2008. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1211898886


Universiteit Utrecht

27. Bourajjaj, M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.

Degree: 2008, Universiteit Utrecht

 Despite significant progress in the prevention and treatment of cardiovascular diseases, heart failure is still a leading cause of morbidity and mortality in industrial countries.… (more)

Subjects/Keywords: Geneeskunde; cardiac hypertrophy; heart failure; calcineurin; NFAT; myogenesis; MyoD

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APA (6th Edition):

Bourajjaj, M. (2008). Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/29300

Chicago Manual of Style (16th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Doctoral Dissertation, Universiteit Utrecht. Accessed September 21, 2019. http://dspace.library.uu.nl:8080/handle/1874/29300.

MLA Handbook (7th Edition):

Bourajjaj, M. “Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation.” 2008. Web. 21 Sep 2019.

Vancouver:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2008. [cited 2019 Sep 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/29300.

Council of Science Editors:

Bourajjaj M. Nuclear Factor of Activated T cells (NFAT): key regulator of cardiac hypertrophy and skeletal muscle adaptation. [Doctoral Dissertation]. Universiteit Utrecht; 2008. Available from: http://dspace.library.uu.nl:8080/handle/1874/29300


Universiteit Utrecht

28. Oort, R.J. van. The transcription factor myocyte enhancer factor-2 (MEF2) in cardiac hypertrophy and heart failure.

Degree: 2007, Universiteit Utrecht

 The heart responds to stress signals by hypertrophic growth, which is the first step towards heart failure (HF). The genetic pattern underlying HF remains largely… (more)

Subjects/Keywords: Geneeskunde; heart failure; cardiac hypertrophy; MEF2; transcription; signal transduction; gene analysis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oort, R. J. v. (2007). The transcription factor myocyte enhancer factor-2 (MEF2) in cardiac hypertrophy and heart failure. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/20974

Chicago Manual of Style (16th Edition):

Oort, R J van. “The transcription factor myocyte enhancer factor-2 (MEF2) in cardiac hypertrophy and heart failure.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed September 21, 2019. http://dspace.library.uu.nl:8080/handle/1874/20974.

MLA Handbook (7th Edition):

Oort, R J van. “The transcription factor myocyte enhancer factor-2 (MEF2) in cardiac hypertrophy and heart failure.” 2007. Web. 21 Sep 2019.

Vancouver:

Oort RJv. The transcription factor myocyte enhancer factor-2 (MEF2) in cardiac hypertrophy and heart failure. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2019 Sep 21]. Available from: http://dspace.library.uu.nl:8080/handle/1874/20974.

Council of Science Editors:

Oort RJv. The transcription factor myocyte enhancer factor-2 (MEF2) in cardiac hypertrophy and heart failure. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/20974


University of Western Australia

29. Spence, Angela. Comparative impacts of endurance and resistance exercise on the cardiovascular system in humans : a prospective reandomised 6-month intervention.

Degree: PhD, 2012, University of Western Australia

[Truncated abstract] In recent years, sophisticated imaging modalities have evolved which considerably enhance our capacity to describe human anatomy and physiology. These approaches, including magnetic… (more)

Subjects/Keywords: Cardiac hypertrophy; Magnetic resonance imaging; Athlete's heart; Exercise training

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APA (6th Edition):

Spence, A. (2012). Comparative impacts of endurance and resistance exercise on the cardiovascular system in humans : a prospective reandomised 6-month intervention. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=34196&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Spence, Angela. “Comparative impacts of endurance and resistance exercise on the cardiovascular system in humans : a prospective reandomised 6-month intervention.” 2012. Doctoral Dissertation, University of Western Australia. Accessed September 21, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=34196&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Spence, Angela. “Comparative impacts of endurance and resistance exercise on the cardiovascular system in humans : a prospective reandomised 6-month intervention.” 2012. Web. 21 Sep 2019.

Vancouver:

Spence A. Comparative impacts of endurance and resistance exercise on the cardiovascular system in humans : a prospective reandomised 6-month intervention. [Internet] [Doctoral dissertation]. University of Western Australia; 2012. [cited 2019 Sep 21]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=34196&local_base=GEN01-INS01.

Council of Science Editors:

Spence A. Comparative impacts of endurance and resistance exercise on the cardiovascular system in humans : a prospective reandomised 6-month intervention. [Doctoral Dissertation]. University of Western Australia; 2012. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=34196&local_base=GEN01-INS01


University of Louisville

30. Givvimani, Srikanth. The role of matrix metalloproteinases induced angiogenesis during transition from compensatory cardiac hypertrophy to cardiac failure : therapeutic effects of hydrogen sulfide.

Degree: PhD, 2010, University of Louisville

  Background: Although matrix metalloproteinase (MMPs) and tissue inhibitor of metalloproteinase (TIMPs) play a vital role in tumor angiogenesis and TIMP-3 causes apoptosis, their role… (more)

Subjects/Keywords: Angiogenesis; Hydrogen sulfide; Heart failure; Hypertension; Cardiac hypertrophy

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APA (6th Edition):

Givvimani, S. (2010). The role of matrix metalloproteinases induced angiogenesis during transition from compensatory cardiac hypertrophy to cardiac failure : therapeutic effects of hydrogen sulfide. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/503 ; https://ir.library.louisville.edu/etd/503

Chicago Manual of Style (16th Edition):

Givvimani, Srikanth. “The role of matrix metalloproteinases induced angiogenesis during transition from compensatory cardiac hypertrophy to cardiac failure : therapeutic effects of hydrogen sulfide.” 2010. Doctoral Dissertation, University of Louisville. Accessed September 21, 2019. 10.18297/etd/503 ; https://ir.library.louisville.edu/etd/503.

MLA Handbook (7th Edition):

Givvimani, Srikanth. “The role of matrix metalloproteinases induced angiogenesis during transition from compensatory cardiac hypertrophy to cardiac failure : therapeutic effects of hydrogen sulfide.” 2010. Web. 21 Sep 2019.

Vancouver:

Givvimani S. The role of matrix metalloproteinases induced angiogenesis during transition from compensatory cardiac hypertrophy to cardiac failure : therapeutic effects of hydrogen sulfide. [Internet] [Doctoral dissertation]. University of Louisville; 2010. [cited 2019 Sep 21]. Available from: 10.18297/etd/503 ; https://ir.library.louisville.edu/etd/503.

Council of Science Editors:

Givvimani S. The role of matrix metalloproteinases induced angiogenesis during transition from compensatory cardiac hypertrophy to cardiac failure : therapeutic effects of hydrogen sulfide. [Doctoral Dissertation]. University of Louisville; 2010. Available from: 10.18297/etd/503 ; https://ir.library.louisville.edu/etd/503

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