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You searched for subject:(Carcinogenesis). Showing records 1 – 30 of 406 total matches.

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1. Khare, Shikha. Chemopreventive action of purified herbal agents in animal models of carcinogenesis.

Degree: 2005, Jamia Hamdard University

newline Advisors/Committee Members: Athar, Mohammad.

Subjects/Keywords: carcinogenesis

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APA (6th Edition):

Khare, S. (2005). Chemopreventive action of purified herbal agents in animal models of carcinogenesis. (Thesis). Jamia Hamdard University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/37388

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Khare, Shikha. “Chemopreventive action of purified herbal agents in animal models of carcinogenesis.” 2005. Thesis, Jamia Hamdard University. Accessed January 18, 2017. http://shodhganga.inflibnet.ac.in/handle/10603/37388.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Khare, Shikha. “Chemopreventive action of purified herbal agents in animal models of carcinogenesis.” 2005. Web. 18 Jan 2017.

Vancouver:

Khare S. Chemopreventive action of purified herbal agents in animal models of carcinogenesis. [Internet] [Thesis]. Jamia Hamdard University; 2005. [cited 2017 Jan 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/37388.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Khare S. Chemopreventive action of purified herbal agents in animal models of carcinogenesis. [Thesis]. Jamia Hamdard University; 2005. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/37388

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Oregon State University

2. Orner, Gayle A. Dehydroepiandrosterone carcinogenesis and tumor modulating effects in trout.

Degree: PhD, Toxicology, 1995, Oregon State University

Subjects/Keywords: Carcinogenesis

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APA (6th Edition):

Orner, G. A. (1995). Dehydroepiandrosterone carcinogenesis and tumor modulating effects in trout. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/34877

Chicago Manual of Style (16th Edition):

Orner, Gayle A. “Dehydroepiandrosterone carcinogenesis and tumor modulating effects in trout.” 1995. Doctoral Dissertation, Oregon State University. Accessed January 18, 2017. http://hdl.handle.net/1957/34877.

MLA Handbook (7th Edition):

Orner, Gayle A. “Dehydroepiandrosterone carcinogenesis and tumor modulating effects in trout.” 1995. Web. 18 Jan 2017.

Vancouver:

Orner GA. Dehydroepiandrosterone carcinogenesis and tumor modulating effects in trout. [Internet] [Doctoral dissertation]. Oregon State University; 1995. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/1957/34877.

Council of Science Editors:

Orner GA. Dehydroepiandrosterone carcinogenesis and tumor modulating effects in trout. [Doctoral Dissertation]. Oregon State University; 1995. Available from: http://hdl.handle.net/1957/34877


Hong Kong University of Science and Technology

3. Yang, Lei. Understanding the role of RGS20 in tumorigenesis and metastasis.

Degree: 2012, Hong Kong University of Science and Technology

 RGS (regulator of G protein signaling) proteins serve as negative regulators of G protein signaling. Since the discovery of the first RGS protein, more than… (more)

Subjects/Keywords: G proteins; Carcinogenesis

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APA (6th Edition):

Yang, L. (2012). Understanding the role of RGS20 in tumorigenesis and metastasis. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://dx.doi.org/10.14711/thesis-b1190240 ; http://repository.ust.hk/ir/bitstream/1783.1-63087/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Lei. “Understanding the role of RGS20 in tumorigenesis and metastasis.” 2012. Thesis, Hong Kong University of Science and Technology. Accessed January 18, 2017. http://dx.doi.org/10.14711/thesis-b1190240 ; http://repository.ust.hk/ir/bitstream/1783.1-63087/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Lei. “Understanding the role of RGS20 in tumorigenesis and metastasis.” 2012. Web. 18 Jan 2017.

Vancouver:

Yang L. Understanding the role of RGS20 in tumorigenesis and metastasis. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2012. [cited 2017 Jan 18]. Available from: http://dx.doi.org/10.14711/thesis-b1190240 ; http://repository.ust.hk/ir/bitstream/1783.1-63087/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang L. Understanding the role of RGS20 in tumorigenesis and metastasis. [Thesis]. Hong Kong University of Science and Technology; 2012. Available from: http://dx.doi.org/10.14711/thesis-b1190240 ; http://repository.ust.hk/ir/bitstream/1783.1-63087/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

4. Hu, Xiaobin. The role of Uhrf1 in development and tumorigenesis.

Degree: PhD, 2014, University of Hong Kong

published_or_final_version

Biochemistry

Doctoral

Doctor of Philosophy

Subjects/Keywords: Carcinogenesis; Ubiquitin

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APA (6th Edition):

Hu, X. (2014). The role of Uhrf1 in development and tumorigenesis. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/225205

Chicago Manual of Style (16th Edition):

Hu, Xiaobin. “The role of Uhrf1 in development and tumorigenesis.” 2014. Doctoral Dissertation, University of Hong Kong. Accessed January 18, 2017. http://hdl.handle.net/10722/225205.

MLA Handbook (7th Edition):

Hu, Xiaobin. “The role of Uhrf1 in development and tumorigenesis.” 2014. Web. 18 Jan 2017.

Vancouver:

Hu X. The role of Uhrf1 in development and tumorigenesis. [Internet] [Doctoral dissertation]. University of Hong Kong; 2014. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/10722/225205.

Council of Science Editors:

Hu X. The role of Uhrf1 in development and tumorigenesis. [Doctoral Dissertation]. University of Hong Kong; 2014. Available from: http://hdl.handle.net/10722/225205


NSYSU

5. Huang, Jian-yuan. Investigation of the Homeobox (HOX) gene expression in human gastric carcinogenesis.

Degree: Master, Institute of Biomedical Sciences, 2009, NSYSU

 In the past study, we used a human gastric stem cell clone, KMU-GI2, isolated from endoscopically biopsied gastric mucosa. As we maintained this KMU-GI2 cell… (more)

Subjects/Keywords: HOX; Homeobox; gastric carcinogenesis

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APA (6th Edition):

Huang, J. (2009). Investigation of the Homeobox (HOX) gene expression in human gastric carcinogenesis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623109-181128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Jian-yuan. “Investigation of the Homeobox (HOX) gene expression in human gastric carcinogenesis.” 2009. Thesis, NSYSU. Accessed January 18, 2017. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623109-181128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Jian-yuan. “Investigation of the Homeobox (HOX) gene expression in human gastric carcinogenesis.” 2009. Web. 18 Jan 2017.

Vancouver:

Huang J. Investigation of the Homeobox (HOX) gene expression in human gastric carcinogenesis. [Internet] [Thesis]. NSYSU; 2009. [cited 2017 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623109-181128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang J. Investigation of the Homeobox (HOX) gene expression in human gastric carcinogenesis. [Thesis]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623109-181128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Cape Peninsula University of Technology

6. Petrova, Antoinette. Modulation of ultrviolet light induced skin carcinogenesis by extracts of Rooibos and Honeybush using a mouse model:elucidating possible protective mechanisms.

Degree: 2009, Cape Peninsula University of Technology

 This thesis provides the first scientific evidence of the photoprotective properties of rooibos and honeybush herbal tea extracts and to some extent, two major honeybush… (more)

Subjects/Keywords: rooibos; honeybush; carcinogenesis; mouse model

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APA (6th Edition):

Petrova, A. (2009). Modulation of ultrviolet light induced skin carcinogenesis by extracts of Rooibos and Honeybush using a mouse model:elucidating possible protective mechanisms. (Thesis). Cape Peninsula University of Technology. Retrieved from http://digitalknowledge.cput.ac.za/xmlui/handle/11189/157

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petrova, Antoinette. “Modulation of ultrviolet light induced skin carcinogenesis by extracts of Rooibos and Honeybush using a mouse model:elucidating possible protective mechanisms.” 2009. Thesis, Cape Peninsula University of Technology. Accessed January 18, 2017. http://digitalknowledge.cput.ac.za/xmlui/handle/11189/157.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petrova, Antoinette. “Modulation of ultrviolet light induced skin carcinogenesis by extracts of Rooibos and Honeybush using a mouse model:elucidating possible protective mechanisms.” 2009. Web. 18 Jan 2017.

Vancouver:

Petrova A. Modulation of ultrviolet light induced skin carcinogenesis by extracts of Rooibos and Honeybush using a mouse model:elucidating possible protective mechanisms. [Internet] [Thesis]. Cape Peninsula University of Technology; 2009. [cited 2017 Jan 18]. Available from: http://digitalknowledge.cput.ac.za/xmlui/handle/11189/157.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petrova A. Modulation of ultrviolet light induced skin carcinogenesis by extracts of Rooibos and Honeybush using a mouse model:elucidating possible protective mechanisms. [Thesis]. Cape Peninsula University of Technology; 2009. Available from: http://digitalknowledge.cput.ac.za/xmlui/handle/11189/157

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Nayeem Bilal. Biochemical studies on the effects of chronic unpredictable stress on nitrosamine induced carcinogenesis; -.

Degree: Biochemistry, 2013, Aligarh Muslim University

None

Bibliography given

Advisors/Committee Members: Naheed Banu.

Subjects/Keywords: biochemistry; Carcinogenesis

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APA (6th Edition):

Bilal, N. (2013). Biochemical studies on the effects of chronic unpredictable stress on nitrosamine induced carcinogenesis; -. (Thesis). Aligarh Muslim University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/12844

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bilal, Nayeem. “Biochemical studies on the effects of chronic unpredictable stress on nitrosamine induced carcinogenesis; -.” 2013. Thesis, Aligarh Muslim University. Accessed January 18, 2017. http://shodhganga.inflibnet.ac.in/handle/10603/12844.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bilal, Nayeem. “Biochemical studies on the effects of chronic unpredictable stress on nitrosamine induced carcinogenesis; -.” 2013. Web. 18 Jan 2017.

Vancouver:

Bilal N. Biochemical studies on the effects of chronic unpredictable stress on nitrosamine induced carcinogenesis; -. [Internet] [Thesis]. Aligarh Muslim University; 2013. [cited 2017 Jan 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/12844.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bilal N. Biochemical studies on the effects of chronic unpredictable stress on nitrosamine induced carcinogenesis; -. [Thesis]. Aligarh Muslim University; 2013. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/12844

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Nida Suhail. Biochemical studies on the effect of stress on DMBA -TPA induced carcinogenesis; -.

Degree: Biochemistry, 2013, Aligarh Muslim University

Cancer is one of the leading causes of death in most well developed countries. It has been established that multiple stepwise alterations of the original… (more)

Subjects/Keywords: biochemistry; Carcinogenesis; Chemoprevention; Stress

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APA (6th Edition):

Suhail, N. (2013). Biochemical studies on the effect of stress on DMBA -TPA induced carcinogenesis; -. (Thesis). Aligarh Muslim University. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/12846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Suhail, Nida. “Biochemical studies on the effect of stress on DMBA -TPA induced carcinogenesis; -.” 2013. Thesis, Aligarh Muslim University. Accessed January 18, 2017. http://shodhganga.inflibnet.ac.in/handle/10603/12846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Suhail, Nida. “Biochemical studies on the effect of stress on DMBA -TPA induced carcinogenesis; -.” 2013. Web. 18 Jan 2017.

Vancouver:

Suhail N. Biochemical studies on the effect of stress on DMBA -TPA induced carcinogenesis; -. [Internet] [Thesis]. Aligarh Muslim University; 2013. [cited 2017 Jan 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/12846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Suhail N. Biochemical studies on the effect of stress on DMBA -TPA induced carcinogenesis; -. [Thesis]. Aligarh Muslim University; 2013. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/12846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Hong Kong University of Science and Technology

9. Wang, Yingchun. RGS19 inhibits oncogene-induced neoplastic transformation.

Degree: 2010, Hong Kong University of Science and Technology

 Many growth factors stimulate cell proliferation through mitogenic pathways such as the Ras/Raf/MEK/ERK cascade. Defective Ras signaling has long been associated with cancer progression and… (more)

Subjects/Keywords: G proteins; Cell transformation; Carcinogenesis

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APA (6th Edition):

Wang, Y. (2010). RGS19 inhibits oncogene-induced neoplastic transformation. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://dx.doi.org/10.14711/thesis-b1115676 ; http://repository.ust.hk/ir/bitstream/1783.1-7545/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Yingchun. “RGS19 inhibits oncogene-induced neoplastic transformation.” 2010. Thesis, Hong Kong University of Science and Technology. Accessed January 18, 2017. http://dx.doi.org/10.14711/thesis-b1115676 ; http://repository.ust.hk/ir/bitstream/1783.1-7545/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Yingchun. “RGS19 inhibits oncogene-induced neoplastic transformation.” 2010. Web. 18 Jan 2017.

Vancouver:

Wang Y. RGS19 inhibits oncogene-induced neoplastic transformation. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2010. [cited 2017 Jan 18]. Available from: http://dx.doi.org/10.14711/thesis-b1115676 ; http://repository.ust.hk/ir/bitstream/1783.1-7545/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang Y. RGS19 inhibits oncogene-induced neoplastic transformation. [Thesis]. Hong Kong University of Science and Technology; 2010. Available from: http://dx.doi.org/10.14711/thesis-b1115676 ; http://repository.ust.hk/ir/bitstream/1783.1-7545/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of British Columbia

10. Hanham, Ann Frances. The clastogenic activity of phenolic oxidation products .

Degree: 1983, University of British Columbia

 Several epidemiological studies .have demonstrated the importance of diet in the development of gastro-intestinal carcinomas in man. This study examines the role of plant phenolics,… (more)

Subjects/Keywords: Phenols; Carcinogenesis

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APA (6th Edition):

Hanham, A. F. (1983). The clastogenic activity of phenolic oxidation products . (Thesis). University of British Columbia. Retrieved from http://hdl.handle.net/2429/24298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hanham, Ann Frances. “The clastogenic activity of phenolic oxidation products .” 1983. Thesis, University of British Columbia. Accessed January 18, 2017. http://hdl.handle.net/2429/24298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hanham, Ann Frances. “The clastogenic activity of phenolic oxidation products .” 1983. Web. 18 Jan 2017.

Vancouver:

Hanham AF. The clastogenic activity of phenolic oxidation products . [Internet] [Thesis]. University of British Columbia; 1983. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/2429/24298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hanham AF. The clastogenic activity of phenolic oxidation products . [Thesis]. University of British Columbia; 1983. Available from: http://hdl.handle.net/2429/24298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas Southwestern Medical Center

11. Lee, Eunmyong. The Role of Autophagy in Early Development and Tumor Suppression Using a Zebrafish Model System.

Degree: 2013, University of Texas Southwestern Medical Center

 Autophagy is an evolutionarily conserved lysosomal degradation pathway which involves the sequestration of cytoplasmic components into a double membraned structure called the autophagosome. By using… (more)

Subjects/Keywords: Autophagy; Carcinogenesis; Morphogenesis; Zebrafish

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APA (6th Edition):

Lee, E. (2013). The Role of Autophagy in Early Development and Tumor Suppression Using a Zebrafish Model System. (Thesis). University of Texas Southwestern Medical Center. Retrieved from http://hdl.handle.net/2152.5/1738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Eunmyong. “The Role of Autophagy in Early Development and Tumor Suppression Using a Zebrafish Model System.” 2013. Thesis, University of Texas Southwestern Medical Center. Accessed January 18, 2017. http://hdl.handle.net/2152.5/1738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Eunmyong. “The Role of Autophagy in Early Development and Tumor Suppression Using a Zebrafish Model System.” 2013. Web. 18 Jan 2017.

Vancouver:

Lee E. The Role of Autophagy in Early Development and Tumor Suppression Using a Zebrafish Model System. [Internet] [Thesis]. University of Texas Southwestern Medical Center; 2013. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/2152.5/1738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee E. The Role of Autophagy in Early Development and Tumor Suppression Using a Zebrafish Model System. [Thesis]. University of Texas Southwestern Medical Center; 2013. Available from: http://hdl.handle.net/2152.5/1738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

12. Kalyuga , Maria. Exploration of the role of the ETS transcription factor ESE2 in breast cancer.

Degree: Clinical School - St Vincent's Hospital, 2011, University of New South Wales

 Thesis abstract: Breast cancer represents a leading cancer-related diagnosis of Australian women, with treatments ultimately still ineffective for many patients. Investigating the mechanisms behind normal… (more)

Subjects/Keywords: Breast cancer; Human breast carcinogenesis

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APA (6th Edition):

Kalyuga , M. (2011). Exploration of the role of the ETS transcription factor ESE2 in breast cancer. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/51251

Chicago Manual of Style (16th Edition):

Kalyuga , Maria. “Exploration of the role of the ETS transcription factor ESE2 in breast cancer.” 2011. Doctoral Dissertation, University of New South Wales. Accessed January 18, 2017. http://handle.unsw.edu.au/1959.4/51251.

MLA Handbook (7th Edition):

Kalyuga , Maria. “Exploration of the role of the ETS transcription factor ESE2 in breast cancer.” 2011. Web. 18 Jan 2017.

Vancouver:

Kalyuga M. Exploration of the role of the ETS transcription factor ESE2 in breast cancer. [Internet] [Doctoral dissertation]. University of New South Wales; 2011. [cited 2017 Jan 18]. Available from: http://handle.unsw.edu.au/1959.4/51251.

Council of Science Editors:

Kalyuga M. Exploration of the role of the ETS transcription factor ESE2 in breast cancer. [Doctoral Dissertation]. University of New South Wales; 2011. Available from: http://handle.unsw.edu.au/1959.4/51251


University of Southern California

13. Shahin, Sophia Allaf. Ascorbate and dehydroascorbate enhance the cytotoxicity and morphological transformation of C3H/10T½ C1 8 mouse embryo cells induced by soluble chromium (VI) compounds.

Degree: MS, Experimental and Molecular Pathology, 2014, University of Southern California

 Hexavalent chromium [Cr(VI)]‐containing compounds are human carcinogens. They cause cancers in the respiratory system when inhaled, and stomach, kidney/other internal cancers when ingested. Soluble and… (more)

Subjects/Keywords: chemical; chromium; physical carcinogenesis

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APA (6th Edition):

Shahin, S. A. (2014). Ascorbate and dehydroascorbate enhance the cytotoxicity and morphological transformation of C3H/10T½ C1 8 mouse embryo cells induced by soluble chromium (VI) compounds. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385505/rec/920

Chicago Manual of Style (16th Edition):

Shahin, Sophia Allaf. “Ascorbate and dehydroascorbate enhance the cytotoxicity and morphological transformation of C3H/10T½ C1 8 mouse embryo cells induced by soluble chromium (VI) compounds.” 2014. Masters Thesis, University of Southern California. Accessed January 18, 2017. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385505/rec/920.

MLA Handbook (7th Edition):

Shahin, Sophia Allaf. “Ascorbate and dehydroascorbate enhance the cytotoxicity and morphological transformation of C3H/10T½ C1 8 mouse embryo cells induced by soluble chromium (VI) compounds.” 2014. Web. 18 Jan 2017.

Vancouver:

Shahin SA. Ascorbate and dehydroascorbate enhance the cytotoxicity and morphological transformation of C3H/10T½ C1 8 mouse embryo cells induced by soluble chromium (VI) compounds. [Internet] [Masters thesis]. University of Southern California; 2014. [cited 2017 Jan 18]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385505/rec/920.

Council of Science Editors:

Shahin SA. Ascorbate and dehydroascorbate enhance the cytotoxicity and morphological transformation of C3H/10T½ C1 8 mouse embryo cells induced by soluble chromium (VI) compounds. [Masters Thesis]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/385505/rec/920

14. Jadhav, Rohit. ROLE OF OVARIAN CANCER-INITIATING CELLS IN HIGH-GRADE SEROUS OVARIAN CARCINOGENESIS.

Degree: 2012, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

A subpopulation of tumor cells known as ovarian cancer initiating cells (OCICs) have been shown to be the cells that… (more)

Subjects/Keywords: Ovarian Cancer; Epigenetics; DNA Methylation; Carcinogenesis; Ovaries  – Cancer; Carcinogenesis; Epigenetics

…events in ovarian carcinogenesis. x Chapter 1 INTRODUCTION Ovarian Cancer Ovarian cancer is… …ovarian carcinogenesis, have not been clearly identified (Dubeau, 2008). Second, current 2… …molecular changes in carcinogenesis (A. P. Bird & Wolffe, 1999), and as such can be applied for… …immortalization during carcinogenesis (Feinberg & Tycko, 2004). The components of the epigenome, DNA… …can be used to better understand the steps of carcinogenesis and their cells of origin… 

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APA (6th Edition):

Jadhav, R. (2012). ROLE OF OVARIAN CANCER-INITIATING CELLS IN HIGH-GRADE SEROUS OVARIAN CARCINOGENESIS. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2773

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jadhav, Rohit. “ROLE OF OVARIAN CANCER-INITIATING CELLS IN HIGH-GRADE SEROUS OVARIAN CARCINOGENESIS.” 2012. Thesis, IUPUI. Accessed January 18, 2017. http://hdl.handle.net/1805/2773.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jadhav, Rohit. “ROLE OF OVARIAN CANCER-INITIATING CELLS IN HIGH-GRADE SEROUS OVARIAN CARCINOGENESIS.” 2012. Web. 18 Jan 2017.

Vancouver:

Jadhav R. ROLE OF OVARIAN CANCER-INITIATING CELLS IN HIGH-GRADE SEROUS OVARIAN CARCINOGENESIS. [Internet] [Thesis]. IUPUI; 2012. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/1805/2773.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jadhav R. ROLE OF OVARIAN CANCER-INITIATING CELLS IN HIGH-GRADE SEROUS OVARIAN CARCINOGENESIS. [Thesis]. IUPUI; 2012. Available from: http://hdl.handle.net/1805/2773

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

15. JAFFE, DEBORAH RUTH. MOUSE SKIN TUMOR INITIATION BY IONIZING RADIATION AND THE DETECTION OF DOMINANT TRANSFORMING GENE(S).

Degree: 1987, University of Arizona

 The initiating potential of a range of 4 MeV X-rays was studied using the mouse skin two-stage model of carcinogenesis. A single dose of radiation… (more)

Subjects/Keywords: Radiation carcinogenesis.; Ionizing radiation.; Carcinogenesis.

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APA (6th Edition):

JAFFE, D. R. (1987). MOUSE SKIN TUMOR INITIATION BY IONIZING RADIATION AND THE DETECTION OF DOMINANT TRANSFORMING GENE(S). (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/184146

Chicago Manual of Style (16th Edition):

JAFFE, DEBORAH RUTH. “MOUSE SKIN TUMOR INITIATION BY IONIZING RADIATION AND THE DETECTION OF DOMINANT TRANSFORMING GENE(S). ” 1987. Doctoral Dissertation, University of Arizona. Accessed January 18, 2017. http://hdl.handle.net/10150/184146.

MLA Handbook (7th Edition):

JAFFE, DEBORAH RUTH. “MOUSE SKIN TUMOR INITIATION BY IONIZING RADIATION AND THE DETECTION OF DOMINANT TRANSFORMING GENE(S). ” 1987. Web. 18 Jan 2017.

Vancouver:

JAFFE DR. MOUSE SKIN TUMOR INITIATION BY IONIZING RADIATION AND THE DETECTION OF DOMINANT TRANSFORMING GENE(S). [Internet] [Doctoral dissertation]. University of Arizona; 1987. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/10150/184146.

Council of Science Editors:

JAFFE DR. MOUSE SKIN TUMOR INITIATION BY IONIZING RADIATION AND THE DETECTION OF DOMINANT TRANSFORMING GENE(S). [Doctoral Dissertation]. University of Arizona; 1987. Available from: http://hdl.handle.net/10150/184146


Penn State University

16. Markell, Lauren Mordasky. PHARMACOLOGICAL INHIBITION OF THE TGF-BETA TYPE I RECEPTOR REVEALS A DUAL ROLE OF TGF-BETA IN CARCINOGENESIS.

Degree: PhD, Integrative Biosciences, 2010, Penn State University

 Transforming growth factor-beta 1 (TGF-beta 1) is a member of a large family of regulatory molecules that play both positive and negative roles in epithelial… (more)

Subjects/Keywords: skin; skin carcinogenesis; TGF-beta; chemically-induced carcinogenesis

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APA (6th Edition):

Markell, L. M. (2010). PHARMACOLOGICAL INHIBITION OF THE TGF-BETA TYPE I RECEPTOR REVEALS A DUAL ROLE OF TGF-BETA IN CARCINOGENESIS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11358

Chicago Manual of Style (16th Edition):

Markell, Lauren Mordasky. “PHARMACOLOGICAL INHIBITION OF THE TGF-BETA TYPE I RECEPTOR REVEALS A DUAL ROLE OF TGF-BETA IN CARCINOGENESIS.” 2010. Doctoral Dissertation, Penn State University. Accessed January 18, 2017. https://etda.libraries.psu.edu/catalog/11358.

MLA Handbook (7th Edition):

Markell, Lauren Mordasky. “PHARMACOLOGICAL INHIBITION OF THE TGF-BETA TYPE I RECEPTOR REVEALS A DUAL ROLE OF TGF-BETA IN CARCINOGENESIS.” 2010. Web. 18 Jan 2017.

Vancouver:

Markell LM. PHARMACOLOGICAL INHIBITION OF THE TGF-BETA TYPE I RECEPTOR REVEALS A DUAL ROLE OF TGF-BETA IN CARCINOGENESIS. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2017 Jan 18]. Available from: https://etda.libraries.psu.edu/catalog/11358.

Council of Science Editors:

Markell LM. PHARMACOLOGICAL INHIBITION OF THE TGF-BETA TYPE I RECEPTOR REVEALS A DUAL ROLE OF TGF-BETA IN CARCINOGENESIS. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11358


Penn State University

17. Bility, Moses Turkle. MODULATION OF SKIN CANCER BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA.

Degree: PhD, Integrative Biosciences, 2008, Penn State University

 Pparb/d-null mice exhibit enhanced tumorigenesis in a two-stage carcinogenesis bioassay model when compared to wild-type mice, which is likely due in part to enhanced epidermal… (more)

Subjects/Keywords: PPARb/d; nuclear receptors; v-rasHa-induced skin carcinogenesis; chemically-induced skin carcinogenesis; skin carcinogenesis; PPARs

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APA (6th Edition):

Bility, M. T. (2008). MODULATION OF SKIN CANCER BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/9430

Chicago Manual of Style (16th Edition):

Bility, Moses Turkle. “MODULATION OF SKIN CANCER BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA.” 2008. Doctoral Dissertation, Penn State University. Accessed January 18, 2017. https://etda.libraries.psu.edu/catalog/9430.

MLA Handbook (7th Edition):

Bility, Moses Turkle. “MODULATION OF SKIN CANCER BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA.” 2008. Web. 18 Jan 2017.

Vancouver:

Bility MT. MODULATION OF SKIN CANCER BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2017 Jan 18]. Available from: https://etda.libraries.psu.edu/catalog/9430.

Council of Science Editors:

Bility MT. MODULATION OF SKIN CANCER BY PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/9430

18. Burke, Amy. Chronic nitric oxide exposure induces prostate cell carcinogenesis, involving genetic instability and a pro-tumorigenic secretory phenotype.

Degree: 2016, National University of Ireland – Galway

 Prostate cancer is the second most commonly diagnosed cancer, and ranked third in the causes of death from cancer in Irish men. Inflammation has been… (more)

Subjects/Keywords: Prostate cancer; Nitric oxide; Carcinogenesis; Transformation; Medicine

…cancer in Irish men. Inflammation has been implicated in prostate carcinogenesis, with the… …telomere shortening and genomic alterations have all been implicated in prostate carcinogenesis… …in carcinogenesis, there is no doubt that inflammation plays a key role in the initiation… …peroxynitrite induce inflammation-associated carcinogenesis include induction of DNA damage… …carcinogenesis (Ying and Hofseth, 2007). NOS, with NADPH oxidase (NOX) and cyclooxygenase (COX) mediate… 

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APA (6th Edition):

Burke, A. (2016). Chronic nitric oxide exposure induces prostate cell carcinogenesis, involving genetic instability and a pro-tumorigenic secretory phenotype. (Thesis). National University of Ireland – Galway. Retrieved from http://hdl.handle.net/10379/5532

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Burke, Amy. “Chronic nitric oxide exposure induces prostate cell carcinogenesis, involving genetic instability and a pro-tumorigenic secretory phenotype.” 2016. Thesis, National University of Ireland – Galway. Accessed January 18, 2017. http://hdl.handle.net/10379/5532.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Burke, Amy. “Chronic nitric oxide exposure induces prostate cell carcinogenesis, involving genetic instability and a pro-tumorigenic secretory phenotype.” 2016. Web. 18 Jan 2017.

Vancouver:

Burke A. Chronic nitric oxide exposure induces prostate cell carcinogenesis, involving genetic instability and a pro-tumorigenic secretory phenotype. [Internet] [Thesis]. National University of Ireland – Galway; 2016. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/10379/5532.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Burke A. Chronic nitric oxide exposure induces prostate cell carcinogenesis, involving genetic instability and a pro-tumorigenic secretory phenotype. [Thesis]. National University of Ireland – Galway; 2016. Available from: http://hdl.handle.net/10379/5532

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Soni, Bihari Lal. Global protein profiling during rat lingual carcinogenesis and validation of differentiator proteins in human tongue; -.

Degree: Life Science, 2015, INFLIBNET

None

Bibliography p.110 - 123 Appendix p.124 - 143 and Publication P.144

Advisors/Committee Members: Vaidya, Milind M.

Subjects/Keywords: Carcinogenesis; Global; Protein

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APA (6th Edition):

Soni, B. L. (2015). Global protein profiling during rat lingual carcinogenesis and validation of differentiator proteins in human tongue; -. (Thesis). INFLIBNET. Retrieved from http://shodhganga.inflibnet.ac.in/handle/10603/49148

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Soni, Bihari Lal. “Global protein profiling during rat lingual carcinogenesis and validation of differentiator proteins in human tongue; -.” 2015. Thesis, INFLIBNET. Accessed January 18, 2017. http://shodhganga.inflibnet.ac.in/handle/10603/49148.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Soni, Bihari Lal. “Global protein profiling during rat lingual carcinogenesis and validation of differentiator proteins in human tongue; -.” 2015. Web. 18 Jan 2017.

Vancouver:

Soni BL. Global protein profiling during rat lingual carcinogenesis and validation of differentiator proteins in human tongue; -. [Internet] [Thesis]. INFLIBNET; 2015. [cited 2017 Jan 18]. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/49148.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Soni BL. Global protein profiling during rat lingual carcinogenesis and validation of differentiator proteins in human tongue; -. [Thesis]. INFLIBNET; 2015. Available from: http://shodhganga.inflibnet.ac.in/handle/10603/49148

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

20. denDekker, Aaron D. Genetic mapping and mechanism of action of rat mammary carcinoma susceptibility quantitative trait locus Mcs1b.

Degree: PhD, 2013, University of Louisville

 Breast cancer is a complex disease that involves genetic, epigenetic, and environmental components. High and moderate penetrant genes have been identified that affect risk to… (more)

Subjects/Keywords: Breast cancer susceptibility; MIER3; Mammary carcinogenesis; Mcs1b

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APA (6th Edition):

denDekker, A. D. (2013). Genetic mapping and mechanism of action of rat mammary carcinoma susceptibility quantitative trait locus Mcs1b. (Doctoral Dissertation). University of Louisville. Retrieved from http://dx.doi.org/10.18297/etd/330 ; http://ir.library.louisville.edu/etd/330

Chicago Manual of Style (16th Edition):

denDekker, Aaron D. “Genetic mapping and mechanism of action of rat mammary carcinoma susceptibility quantitative trait locus Mcs1b.” 2013. Doctoral Dissertation, University of Louisville. Accessed January 18, 2017. http://dx.doi.org/10.18297/etd/330 ; http://ir.library.louisville.edu/etd/330.

MLA Handbook (7th Edition):

denDekker, Aaron D. “Genetic mapping and mechanism of action of rat mammary carcinoma susceptibility quantitative trait locus Mcs1b.” 2013. Web. 18 Jan 2017.

Vancouver:

denDekker AD. Genetic mapping and mechanism of action of rat mammary carcinoma susceptibility quantitative trait locus Mcs1b. [Internet] [Doctoral dissertation]. University of Louisville; 2013. [cited 2017 Jan 18]. Available from: http://dx.doi.org/10.18297/etd/330 ; http://ir.library.louisville.edu/etd/330.

Council of Science Editors:

denDekker AD. Genetic mapping and mechanism of action of rat mammary carcinoma susceptibility quantitative trait locus Mcs1b. [Doctoral Dissertation]. University of Louisville; 2013. Available from: http://dx.doi.org/10.18297/etd/330 ; http://ir.library.louisville.edu/etd/330


West Virginia University

21. Vongsutilers, Vorasit. The effect of C8-arylguanine adducts on B/Z-DNA equilibrium.

Degree: 2009, West Virginia University

 Aryl hydrazines and related compounds have been shown to be carcinogenic but the mechanism is still unclear. C8-Arylguanine adducts, formed from oxidative metabolites of aryl… (more)

Subjects/Keywords: Hydrazines.; Carcinogenesis.; Hydrazines.; Carcinogens.; DNA, Z-Form.

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APA (6th Edition):

Vongsutilers, V. (2009). The effect of C8-arylguanine adducts on B/Z-DNA equilibrium. (Thesis). West Virginia University. Retrieved from http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=22151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vongsutilers, Vorasit. “The effect of C8-arylguanine adducts on B/Z-DNA equilibrium.” 2009. Thesis, West Virginia University. Accessed January 18, 2017. http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=22151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vongsutilers, Vorasit. “The effect of C8-arylguanine adducts on B/Z-DNA equilibrium.” 2009. Web. 18 Jan 2017.

Vancouver:

Vongsutilers V. The effect of C8-arylguanine adducts on B/Z-DNA equilibrium. [Internet] [Thesis]. West Virginia University; 2009. [cited 2017 Jan 18]. Available from: http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=22151.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vongsutilers V. The effect of C8-arylguanine adducts on B/Z-DNA equilibrium. [Thesis]. West Virginia University; 2009. Available from: http://wvuscholar.wvu.edu:8881/R/?func=dbin-jump-full&object_id=22151

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Emory University

22. Guo, Junjie. Coffee and Tea Intake and Risk of Incident, Sporadic Colorectal Adenomas.

Degree: MPH, Epidemiology; Epidemiology, 2016, Emory University

Background: Coffee and tea are commonly consumed beverages that contain several bioactive compounds, and have been suggested to influence colorectal carcinogenesis. However, the findings from… (more)

Subjects/Keywords: Epidemiology; Colorectal adenoma; Colorectal carcinogenesis; Coffee; Tea

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APA (6th Edition):

Guo, J. (2016). Coffee and Tea Intake and Risk of Incident, Sporadic Colorectal Adenomas. (Masters Thesis). Emory University. Retrieved from http://pid.emory.edu/ark:/25593/rkd19

Chicago Manual of Style (16th Edition):

Guo, Junjie. “Coffee and Tea Intake and Risk of Incident, Sporadic Colorectal Adenomas.” 2016. Masters Thesis, Emory University. Accessed January 18, 2017. http://pid.emory.edu/ark:/25593/rkd19.

MLA Handbook (7th Edition):

Guo, Junjie. “Coffee and Tea Intake and Risk of Incident, Sporadic Colorectal Adenomas.” 2016. Web. 18 Jan 2017.

Vancouver:

Guo J. Coffee and Tea Intake and Risk of Incident, Sporadic Colorectal Adenomas. [Internet] [Masters thesis]. Emory University; 2016. [cited 2017 Jan 18]. Available from: http://pid.emory.edu/ark:/25593/rkd19.

Council of Science Editors:

Guo J. Coffee and Tea Intake and Risk of Incident, Sporadic Colorectal Adenomas. [Masters Thesis]. Emory University; 2016. Available from: http://pid.emory.edu/ark:/25593/rkd19


Hong Kong University of Science and Technology

23. Hu, Yaling. Fucoidin enhances dendritic cell-mediated T-cell cytotoxicity against NY-ESO-1 expressing human cancer cells.

Degree: 2008, Hong Kong University of Science and Technology

 Scavenger receptor A (SR-A) has been found able to negatively regulate antitumor immunity, and Fucoidin is a promising inhibitor of SR-A. Calreticulin (CRT) on dendritic… (more)

Subjects/Keywords: Cell-mediated cytotoxicity; T cells; Carcinogenesis

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APA (6th Edition):

Hu, Y. (2008). Fucoidin enhances dendritic cell-mediated T-cell cytotoxicity against NY-ESO-1 expressing human cancer cells. (Thesis). Hong Kong University of Science and Technology. Retrieved from http://dx.doi.org/10.14711/thesis-b1030064 ; http://repository.ust.hk/ir/bitstream/1783.1-3802/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Yaling. “Fucoidin enhances dendritic cell-mediated T-cell cytotoxicity against NY-ESO-1 expressing human cancer cells.” 2008. Thesis, Hong Kong University of Science and Technology. Accessed January 18, 2017. http://dx.doi.org/10.14711/thesis-b1030064 ; http://repository.ust.hk/ir/bitstream/1783.1-3802/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Yaling. “Fucoidin enhances dendritic cell-mediated T-cell cytotoxicity against NY-ESO-1 expressing human cancer cells.” 2008. Web. 18 Jan 2017.

Vancouver:

Hu Y. Fucoidin enhances dendritic cell-mediated T-cell cytotoxicity against NY-ESO-1 expressing human cancer cells. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2008. [cited 2017 Jan 18]. Available from: http://dx.doi.org/10.14711/thesis-b1030064 ; http://repository.ust.hk/ir/bitstream/1783.1-3802/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu Y. Fucoidin enhances dendritic cell-mediated T-cell cytotoxicity against NY-ESO-1 expressing human cancer cells. [Thesis]. Hong Kong University of Science and Technology; 2008. Available from: http://dx.doi.org/10.14711/thesis-b1030064 ; http://repository.ust.hk/ir/bitstream/1783.1-3802/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Oregon State University

24. Kelly, Jack D. Oxidative stress and carcinogenesis in trout.

Degree: PhD, Toxicology, 1992, Oregon State University

Subjects/Keywords: Carcinogenesis  – Animal models

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APA (6th Edition):

Kelly, J. D. (1992). Oxidative stress and carcinogenesis in trout. (Doctoral Dissertation). Oregon State University. Retrieved from http://hdl.handle.net/1957/37254

Chicago Manual of Style (16th Edition):

Kelly, Jack D. “Oxidative stress and carcinogenesis in trout.” 1992. Doctoral Dissertation, Oregon State University. Accessed January 18, 2017. http://hdl.handle.net/1957/37254.

MLA Handbook (7th Edition):

Kelly, Jack D. “Oxidative stress and carcinogenesis in trout.” 1992. Web. 18 Jan 2017.

Vancouver:

Kelly JD. Oxidative stress and carcinogenesis in trout. [Internet] [Doctoral dissertation]. Oregon State University; 1992. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/1957/37254.

Council of Science Editors:

Kelly JD. Oxidative stress and carcinogenesis in trout. [Doctoral Dissertation]. Oregon State University; 1992. Available from: http://hdl.handle.net/1957/37254


Penn State University

25. Borland, Michael Gregory. PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS.

Degree: PhD, Biochemistry, Microbiology, and Molecular Biology, 2010, Penn State University

 Since its identification in the early 1990fs, the physiological roles of the nuclear hormone receptor peroxisome proliferator-activated receptor-ƒÀ/ƒÂ (PPARƒÀ/ƒÂ) have become better understood. This ligand-activated… (more)

Subjects/Keywords: PPARBeta/Delta; AHR; PAH; Bioactivation; Skin carcinogenesis

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APA (6th Edition):

Borland, M. G. (2010). PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/11381

Chicago Manual of Style (16th Edition):

Borland, Michael Gregory. “PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS.” 2010. Doctoral Dissertation, Penn State University. Accessed January 18, 2017. https://etda.libraries.psu.edu/catalog/11381.

MLA Handbook (7th Edition):

Borland, Michael Gregory. “PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS.” 2010. Web. 18 Jan 2017.

Vancouver:

Borland MG. PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS. [Internet] [Doctoral dissertation]. Penn State University; 2010. [cited 2017 Jan 18]. Available from: https://etda.libraries.psu.edu/catalog/11381.

Council of Science Editors:

Borland MG. PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR BETA/DELTA MODULATES ARYL HYDROCARBON RECEPTOR-DEPENDENT SIGNALING AND SKIN CARCINOGENESIS. [Doctoral Dissertation]. Penn State University; 2010. Available from: https://etda.libraries.psu.edu/catalog/11381


University of Hong Kong

26. Chu, Ying-ying, Jamie. Molecular characterization of the novel oncogene human cell cycle-related kinase (CCRK) in glioblastoma multiforme.

Degree: PhD, 2011, University of Hong Kong

 Glioblastoma multiforme (GBMs) are the most common and severe form of malignant brain tumors. Despite recent advancement in the fields of surgical resection, radiotherapy and… (more)

Subjects/Keywords: Glioblastoma multiforme.; Carcinogenesis.; Cell cycle.; Oncogenes.

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APA (6th Edition):

Chu, Ying-ying, J. (2011). Molecular characterization of the novel oncogene human cell cycle-related kinase (CCRK) in glioblastoma multiforme. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/182308

Chicago Manual of Style (16th Edition):

Chu, Ying-ying, Jamie. “Molecular characterization of the novel oncogene human cell cycle-related kinase (CCRK) in glioblastoma multiforme.” 2011. Doctoral Dissertation, University of Hong Kong. Accessed January 18, 2017. http://hdl.handle.net/10722/182308.

MLA Handbook (7th Edition):

Chu, Ying-ying, Jamie. “Molecular characterization of the novel oncogene human cell cycle-related kinase (CCRK) in glioblastoma multiforme.” 2011. Web. 18 Jan 2017.

Vancouver:

Chu, Ying-ying J. Molecular characterization of the novel oncogene human cell cycle-related kinase (CCRK) in glioblastoma multiforme. [Internet] [Doctoral dissertation]. University of Hong Kong; 2011. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/10722/182308.

Council of Science Editors:

Chu, Ying-ying J. Molecular characterization of the novel oncogene human cell cycle-related kinase (CCRK) in glioblastoma multiforme. [Doctoral Dissertation]. University of Hong Kong; 2011. Available from: http://hdl.handle.net/10722/182308


University of Gothenburg / Göteborgs Universitet

27. Wang, Wanzhong. INFLAMMATION AND PROSTATIC CARCINOGENESIS – A MORPHOLOGICAL STUDY OF THE HUMAN PROSTATE.

Degree: 2008, University of Gothenburg / Göteborgs Universitet

 Chronic inflammation has been suggested to be linked to cancers. Inflammatory infiltrates are often found in and around foci of prostatic atrophy. These foci, called… (more)

Subjects/Keywords: prostate; carcinogenesis; chronic inflammation; atrophy; morphology; immunohistochemistry

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APA (6th Edition):

Wang, W. (2008). INFLAMMATION AND PROSTATIC CARCINOGENESIS – A MORPHOLOGICAL STUDY OF THE HUMAN PROSTATE. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/9634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Wanzhong. “INFLAMMATION AND PROSTATIC CARCINOGENESIS – A MORPHOLOGICAL STUDY OF THE HUMAN PROSTATE.” 2008. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 18, 2017. http://hdl.handle.net/2077/9634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Wanzhong. “INFLAMMATION AND PROSTATIC CARCINOGENESIS – A MORPHOLOGICAL STUDY OF THE HUMAN PROSTATE.” 2008. Web. 18 Jan 2017.

Vancouver:

Wang W. INFLAMMATION AND PROSTATIC CARCINOGENESIS – A MORPHOLOGICAL STUDY OF THE HUMAN PROSTATE. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2008. [cited 2017 Jan 18]. Available from: http://hdl.handle.net/2077/9634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang W. INFLAMMATION AND PROSTATIC CARCINOGENESIS – A MORPHOLOGICAL STUDY OF THE HUMAN PROSTATE. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2008. Available from: http://hdl.handle.net/2077/9634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Musser, Sarah Kipley. Structural Investigations of N3-(2-Hydroxy-3-buten-1-yl)-2'-deoxyuridine DNA Adducts.

Degree: PhD, Chemistry, 2009, Vanderbilt University

 Butadiene monoepoxide (BDO) is the major metabolite of butadiene (BD), a widely used petrochemical and known human and animal carcinogen. BD metabolites may be involved… (more)

Subjects/Keywords: butadiene; DNA adducts; carcinogenesis; NMR spectroscopy

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APA (6th Edition):

Musser, S. K. (2009). Structural Investigations of N3-(2-Hydroxy-3-buten-1-yl)-2'-deoxyuridine DNA Adducts. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-07222009-161202/ ;

Chicago Manual of Style (16th Edition):

Musser, Sarah Kipley. “Structural Investigations of N3-(2-Hydroxy-3-buten-1-yl)-2'-deoxyuridine DNA Adducts.” 2009. Doctoral Dissertation, Vanderbilt University. Accessed January 18, 2017. http://etd.library.vanderbilt.edu//available/etd-07222009-161202/ ;.

MLA Handbook (7th Edition):

Musser, Sarah Kipley. “Structural Investigations of N3-(2-Hydroxy-3-buten-1-yl)-2'-deoxyuridine DNA Adducts.” 2009. Web. 18 Jan 2017.

Vancouver:

Musser SK. Structural Investigations of N3-(2-Hydroxy-3-buten-1-yl)-2'-deoxyuridine DNA Adducts. [Internet] [Doctoral dissertation]. Vanderbilt University; 2009. [cited 2017 Jan 18]. Available from: http://etd.library.vanderbilt.edu//available/etd-07222009-161202/ ;.

Council of Science Editors:

Musser SK. Structural Investigations of N3-(2-Hydroxy-3-buten-1-yl)-2'-deoxyuridine DNA Adducts. [Doctoral Dissertation]. Vanderbilt University; 2009. Available from: http://etd.library.vanderbilt.edu//available/etd-07222009-161202/ ;


Rutgers University

29. Wilimczyk, Barbara J. Continuous expression of metabotropic glutamate receptor 1 is required for maintenance of tumorigenesis in GRM1 transformed mouse kidney epithelial cells.

Degree: MS, Microbiology and Molecular Genetics, 2009, Rutgers University

 Within the United States, skin cancer has become a predominant form of cancer that occurs across all age groups, racial backgrounds, and to both genders… (more)

Subjects/Keywords: Cancer cells – Proliferation; Carcinogenesis; Cancer – Treatment

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wilimczyk, B. J. (2009). Continuous expression of metabotropic glutamate receptor 1 is required for maintenance of tumorigenesis in GRM1 transformed mouse kidney epithelial cells. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051419

Chicago Manual of Style (16th Edition):

Wilimczyk, Barbara J. “Continuous expression of metabotropic glutamate receptor 1 is required for maintenance of tumorigenesis in GRM1 transformed mouse kidney epithelial cells.” 2009. Masters Thesis, Rutgers University. Accessed January 18, 2017. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051419.

MLA Handbook (7th Edition):

Wilimczyk, Barbara J. “Continuous expression of metabotropic glutamate receptor 1 is required for maintenance of tumorigenesis in GRM1 transformed mouse kidney epithelial cells.” 2009. Web. 18 Jan 2017.

Vancouver:

Wilimczyk BJ. Continuous expression of metabotropic glutamate receptor 1 is required for maintenance of tumorigenesis in GRM1 transformed mouse kidney epithelial cells. [Internet] [Masters thesis]. Rutgers University; 2009. [cited 2017 Jan 18]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051419.

Council of Science Editors:

Wilimczyk BJ. Continuous expression of metabotropic glutamate receptor 1 is required for maintenance of tumorigenesis in GRM1 transformed mouse kidney epithelial cells. [Masters Thesis]. Rutgers University; 2009. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051419


Rutgers University

30. Gomez, Yury Y., 1982-. Synergestic induction of NRF-2 gene by curcumin and sulforaphane and pharmacokinetics/metabolism of 13C/Dim in rats by UPLC/MS.

Degree: MS, Pharmaceutical Science, 2011, Rutgers University

 Curcumin (CUR) and Sulforaphane (SFN) have shown remarkable cancer chemopreventive effects in numerous studies and combinations of low doses of chemopreventive agents can reduce toxicity… (more)

Subjects/Keywords: Cancer – Chemoprevention; Cancer – Treatment; Carcinogenesis; Phytochemicals

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gomez, Yury Y., 1. (2011). Synergestic induction of NRF-2 gene by curcumin and sulforaphane and pharmacokinetics/metabolism of 13C/Dim in rats by UPLC/MS. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057585

Chicago Manual of Style (16th Edition):

Gomez, Yury Y., 1982-. “Synergestic induction of NRF-2 gene by curcumin and sulforaphane and pharmacokinetics/metabolism of 13C/Dim in rats by UPLC/MS.” 2011. Masters Thesis, Rutgers University. Accessed January 18, 2017. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057585.

MLA Handbook (7th Edition):

Gomez, Yury Y., 1982-. “Synergestic induction of NRF-2 gene by curcumin and sulforaphane and pharmacokinetics/metabolism of 13C/Dim in rats by UPLC/MS.” 2011. Web. 18 Jan 2017.

Vancouver:

Gomez, Yury Y. 1. Synergestic induction of NRF-2 gene by curcumin and sulforaphane and pharmacokinetics/metabolism of 13C/Dim in rats by UPLC/MS. [Internet] [Masters thesis]. Rutgers University; 2011. [cited 2017 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057585.

Council of Science Editors:

Gomez, Yury Y. 1. Synergestic induction of NRF-2 gene by curcumin and sulforaphane and pharmacokinetics/metabolism of 13C/Dim in rats by UPLC/MS. [Masters Thesis]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057585

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