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You searched for subject:(Cancer progression). Showing records 1 – 30 of 93 total matches.

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1. Bougnoux, Anne-Claire. Identification et caractérisation de biomarqueurs associés à la progression tumorale du mélanome malin cutané : implication de TRAP1 et PDIA4 : Identification and characterization of biomarkers associated with tumor progression of cutaneous malignant melanoma : involvement of TRAP1 and PDIA4.

Degree: Docteur es, Biologie Santé, 2014, Université Montpellier I

Bien que le mélanome malin cutané (MMC) ne représente que 10% des cancers cutanés, il en est la forme la plus agressive et est responsable… (more)

Subjects/Keywords: Cancer; Biomarqueurs; Mélanome; Progression tumorale; Cancer; Biomarkers; Melanoma; Tumor progression

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bougnoux, A. (2014). Identification et caractérisation de biomarqueurs associés à la progression tumorale du mélanome malin cutané : implication de TRAP1 et PDIA4 : Identification and characterization of biomarkers associated with tumor progression of cutaneous malignant melanoma : involvement of TRAP1 and PDIA4. (Doctoral Dissertation). Université Montpellier I. Retrieved from http://www.theses.fr/2014MON1T014

Chicago Manual of Style (16th Edition):

Bougnoux, Anne-Claire. “Identification et caractérisation de biomarqueurs associés à la progression tumorale du mélanome malin cutané : implication de TRAP1 et PDIA4 : Identification and characterization of biomarkers associated with tumor progression of cutaneous malignant melanoma : involvement of TRAP1 and PDIA4.” 2014. Doctoral Dissertation, Université Montpellier I. Accessed March 06, 2021. http://www.theses.fr/2014MON1T014.

MLA Handbook (7th Edition):

Bougnoux, Anne-Claire. “Identification et caractérisation de biomarqueurs associés à la progression tumorale du mélanome malin cutané : implication de TRAP1 et PDIA4 : Identification and characterization of biomarkers associated with tumor progression of cutaneous malignant melanoma : involvement of TRAP1 and PDIA4.” 2014. Web. 06 Mar 2021.

Vancouver:

Bougnoux A. Identification et caractérisation de biomarqueurs associés à la progression tumorale du mélanome malin cutané : implication de TRAP1 et PDIA4 : Identification and characterization of biomarkers associated with tumor progression of cutaneous malignant melanoma : involvement of TRAP1 and PDIA4. [Internet] [Doctoral dissertation]. Université Montpellier I; 2014. [cited 2021 Mar 06]. Available from: http://www.theses.fr/2014MON1T014.

Council of Science Editors:

Bougnoux A. Identification et caractérisation de biomarqueurs associés à la progression tumorale du mélanome malin cutané : implication de TRAP1 et PDIA4 : Identification and characterization of biomarkers associated with tumor progression of cutaneous malignant melanoma : involvement of TRAP1 and PDIA4. [Doctoral Dissertation]. Université Montpellier I; 2014. Available from: http://www.theses.fr/2014MON1T014

2. Jouida, Amina. Contrôle de la progression tumorale broncho-pulmonaire par FHIT : Implication du récepteur HER2 : Control of lung tumor progression by FHIT : Involvement of HER2 receptor.

Degree: Docteur es, Biochimie, biologie cellulaire et moléculaire, physiologie et nutrition, 2017, Reims

Dans les cancers du poumon, une des altérations les plus souvent observées est la perte ou l’atténuation de l’expression du gène FHIT (Fragile Histidine Triad).… (more)

Subjects/Keywords: Cancer broncho-Pulmonaire; Fhit; Her2; Progression tumorale; Tem; Lung cancer; Fhit; Her2; Cancer progression; Emt

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APA (6th Edition):

Jouida, A. (2017). Contrôle de la progression tumorale broncho-pulmonaire par FHIT : Implication du récepteur HER2 : Control of lung tumor progression by FHIT : Involvement of HER2 receptor. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2017REIMS008

Chicago Manual of Style (16th Edition):

Jouida, Amina. “Contrôle de la progression tumorale broncho-pulmonaire par FHIT : Implication du récepteur HER2 : Control of lung tumor progression by FHIT : Involvement of HER2 receptor.” 2017. Doctoral Dissertation, Reims. Accessed March 06, 2021. http://www.theses.fr/2017REIMS008.

MLA Handbook (7th Edition):

Jouida, Amina. “Contrôle de la progression tumorale broncho-pulmonaire par FHIT : Implication du récepteur HER2 : Control of lung tumor progression by FHIT : Involvement of HER2 receptor.” 2017. Web. 06 Mar 2021.

Vancouver:

Jouida A. Contrôle de la progression tumorale broncho-pulmonaire par FHIT : Implication du récepteur HER2 : Control of lung tumor progression by FHIT : Involvement of HER2 receptor. [Internet] [Doctoral dissertation]. Reims; 2017. [cited 2021 Mar 06]. Available from: http://www.theses.fr/2017REIMS008.

Council of Science Editors:

Jouida A. Contrôle de la progression tumorale broncho-pulmonaire par FHIT : Implication du récepteur HER2 : Control of lung tumor progression by FHIT : Involvement of HER2 receptor. [Doctoral Dissertation]. Reims; 2017. Available from: http://www.theses.fr/2017REIMS008

3. Sugie, Satoru; Mukai, Shoichiro; Yamasaki, Koji; Kamibeppu, Toyoharu; Tukino, Hiromasa. Significant association of caveolin-1 and caveolin-2 with prostate cancer progression.

Degree: 博士(医学), 2015, University of Miyazaki / 宮崎大学

学位論文の一部を構成しているため、http://hdl.handle.net/10458/5829に本文を掲載。

Background/Aim: Up-regulation of caveolin (CAV)-1 is associated with aggressive prostate cancer. Recently, it has been inferred that CAV2, a co-factor sub-type of CAV1, cross-talks… (more)

Subjects/Keywords: caveolin-1; caveolin-2; prostate cancer progression

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APA (6th Edition):

Sugie, Satoru; Mukai, Shoichiro; Yamasaki, Koji; Kamibeppu, Toyoharu; Tukino, H. (2015). Significant association of caveolin-1 and caveolin-2 with prostate cancer progression. (Thesis). University of Miyazaki / 宮崎大学. Retrieved from http://hdl.handle.net/10458/5828

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sugie, Satoru; Mukai, Shoichiro; Yamasaki, Koji; Kamibeppu, Toyoharu; Tukino, Hiromasa. “Significant association of caveolin-1 and caveolin-2 with prostate cancer progression.” 2015. Thesis, University of Miyazaki / 宮崎大学. Accessed March 06, 2021. http://hdl.handle.net/10458/5828.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sugie, Satoru; Mukai, Shoichiro; Yamasaki, Koji; Kamibeppu, Toyoharu; Tukino, Hiromasa. “Significant association of caveolin-1 and caveolin-2 with prostate cancer progression.” 2015. Web. 06 Mar 2021.

Vancouver:

Sugie, Satoru; Mukai, Shoichiro; Yamasaki, Koji; Kamibeppu, Toyoharu; Tukino H. Significant association of caveolin-1 and caveolin-2 with prostate cancer progression. [Internet] [Thesis]. University of Miyazaki / 宮崎大学; 2015. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10458/5828.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sugie, Satoru; Mukai, Shoichiro; Yamasaki, Koji; Kamibeppu, Toyoharu; Tukino H. Significant association of caveolin-1 and caveolin-2 with prostate cancer progression. [Thesis]. University of Miyazaki / 宮崎大学; 2015. Available from: http://hdl.handle.net/10458/5828

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Vermont

4. Ojemann, Alexandra. Understanding the Role of Runx2 in a Breast Cancer Progression Cell Model.

Degree: MS, Biochemistry, 2017, University of Vermont

  Runx2 is a transcription factor required for bone formation and osteoblastic differentiation during normal development and is implicated in metastatic disease during breast cancer(more)

Subjects/Keywords: Breast; Cancer; MCF10AT1; MCF10CA1a; Progression; Runx2; Biochemistry

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APA (6th Edition):

Ojemann, A. (2017). Understanding the Role of Runx2 in a Breast Cancer Progression Cell Model. (Thesis). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/741

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ojemann, Alexandra. “Understanding the Role of Runx2 in a Breast Cancer Progression Cell Model.” 2017. Thesis, University of Vermont. Accessed March 06, 2021. https://scholarworks.uvm.edu/graddis/741.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ojemann, Alexandra. “Understanding the Role of Runx2 in a Breast Cancer Progression Cell Model.” 2017. Web. 06 Mar 2021.

Vancouver:

Ojemann A. Understanding the Role of Runx2 in a Breast Cancer Progression Cell Model. [Internet] [Thesis]. University of Vermont; 2017. [cited 2021 Mar 06]. Available from: https://scholarworks.uvm.edu/graddis/741.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ojemann A. Understanding the Role of Runx2 in a Breast Cancer Progression Cell Model. [Thesis]. University of Vermont; 2017. Available from: https://scholarworks.uvm.edu/graddis/741

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. Patel, Ketan Vinodbhai. The development of an automated, fluorescent, quantitative three-dimensional in vitro organotypic invasion assay.

Degree: PhD, 2017, Queen Mary, University of London

 Tumour metastasis is a complex multi-step biological process, which involves dispersion of cancer cells to various organ sites within the body. The tumour-stroma interface plays… (more)

Subjects/Keywords: Tumour Biology; Tumour metastasis; cancer progression

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APA (6th Edition):

Patel, K. V. (2017). The development of an automated, fluorescent, quantitative three-dimensional in vitro organotypic invasion assay. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/31860 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786332

Chicago Manual of Style (16th Edition):

Patel, Ketan Vinodbhai. “The development of an automated, fluorescent, quantitative three-dimensional in vitro organotypic invasion assay.” 2017. Doctoral Dissertation, Queen Mary, University of London. Accessed March 06, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/31860 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786332.

MLA Handbook (7th Edition):

Patel, Ketan Vinodbhai. “The development of an automated, fluorescent, quantitative three-dimensional in vitro organotypic invasion assay.” 2017. Web. 06 Mar 2021.

Vancouver:

Patel KV. The development of an automated, fluorescent, quantitative three-dimensional in vitro organotypic invasion assay. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2017. [cited 2021 Mar 06]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/31860 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786332.

Council of Science Editors:

Patel KV. The development of an automated, fluorescent, quantitative three-dimensional in vitro organotypic invasion assay. [Doctoral Dissertation]. Queen Mary, University of London; 2017. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/31860 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.786332


University of Southern California

6. Oghamian, Shirley. The role of DNA methylation in early detection and progression of pancreatic cancer.

Degree: PhD, Biochemistry & Molecular Biology, 2011, University of Southern California

Cancer is the second leading cause of death in the United States and each year nearly half a million Americans die of the disease. The… (more)

Subjects/Keywords: DNA methylation; pancreatic cancer; progression; early detection

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APA (6th Edition):

Oghamian, S. (2011). The role of DNA methylation in early detection and progression of pancreatic cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/242078/rec/7214

Chicago Manual of Style (16th Edition):

Oghamian, Shirley. “The role of DNA methylation in early detection and progression of pancreatic cancer.” 2011. Doctoral Dissertation, University of Southern California. Accessed March 06, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/242078/rec/7214.

MLA Handbook (7th Edition):

Oghamian, Shirley. “The role of DNA methylation in early detection and progression of pancreatic cancer.” 2011. Web. 06 Mar 2021.

Vancouver:

Oghamian S. The role of DNA methylation in early detection and progression of pancreatic cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2011. [cited 2021 Mar 06]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/242078/rec/7214.

Council of Science Editors:

Oghamian S. The role of DNA methylation in early detection and progression of pancreatic cancer. [Doctoral Dissertation]. University of Southern California; 2011. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/242078/rec/7214

7. I. Forno. EXPRESSION ANALYSIS OF MICRORNA IN PROSTATE CANCER AND IDENTIFICATION OF NOVEL DIAGNOSTIC BIOMARKER.

Degree: 2014, Università degli Studi di Milano

 Introduzione: Il tumore alla prostata (PCa) è una delle neoplasie più comuni tra gli uomini nel mondo occidentale e, globalmente, rappresenta la sesta causa di… (more)

Subjects/Keywords: prostate cancer; microRNA; PIN; cancer progression; Settore MED/08 - Anatomia Patologica

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APA (6th Edition):

Forno, I. (2014). EXPRESSION ANALYSIS OF MICRORNA IN PROSTATE CANCER AND IDENTIFICATION OF NOVEL DIAGNOSTIC BIOMARKER. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/231097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Forno, I.. “EXPRESSION ANALYSIS OF MICRORNA IN PROSTATE CANCER AND IDENTIFICATION OF NOVEL DIAGNOSTIC BIOMARKER.” 2014. Thesis, Università degli Studi di Milano. Accessed March 06, 2021. http://hdl.handle.net/2434/231097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Forno, I.. “EXPRESSION ANALYSIS OF MICRORNA IN PROSTATE CANCER AND IDENTIFICATION OF NOVEL DIAGNOSTIC BIOMARKER.” 2014. Web. 06 Mar 2021.

Vancouver:

Forno I. EXPRESSION ANALYSIS OF MICRORNA IN PROSTATE CANCER AND IDENTIFICATION OF NOVEL DIAGNOSTIC BIOMARKER. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/2434/231097.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Forno I. EXPRESSION ANALYSIS OF MICRORNA IN PROSTATE CANCER AND IDENTIFICATION OF NOVEL DIAGNOSTIC BIOMARKER. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/231097

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Deakin University

8. Liem-Weits, Marije. Tumour microenvironment interactions in breast cancer and the role of caveolin-1.

Degree: School of Life and Environmental Sciences, 2017, Deakin University

This study contributes to knowledge about the role of caveolin-1 in the microenvironment of breast cancer tumours. An important findings is that differentiated monocytes contribute to the regulation of caveolin-1 levels in co-cultures of fibroblasts and breast cancer cells. Advisors/Committee Members: Ackland M. Leigh, Michalczyk, Agnes.

Subjects/Keywords: caveolin-1 (CAV1); breast cancer; tumour progression; cancer cell physiology

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APA (6th Edition):

Liem-Weits, M. (2017). Tumour microenvironment interactions in breast cancer and the role of caveolin-1. (Thesis). Deakin University. Retrieved from http://hdl.handle.net/10536/DRO/DU:30105453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liem-Weits, Marije. “Tumour microenvironment interactions in breast cancer and the role of caveolin-1.” 2017. Thesis, Deakin University. Accessed March 06, 2021. http://hdl.handle.net/10536/DRO/DU:30105453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liem-Weits, Marije. “Tumour microenvironment interactions in breast cancer and the role of caveolin-1.” 2017. Web. 06 Mar 2021.

Vancouver:

Liem-Weits M. Tumour microenvironment interactions in breast cancer and the role of caveolin-1. [Internet] [Thesis]. Deakin University; 2017. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10536/DRO/DU:30105453.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liem-Weits M. Tumour microenvironment interactions in breast cancer and the role of caveolin-1. [Thesis]. Deakin University; 2017. Available from: http://hdl.handle.net/10536/DRO/DU:30105453

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

9. Musrap, Natasha. Proteomic Identification of Mediators Implicated in the Metastatic Progression of Ovarian Cancer.

Degree: PhD, 2015, University of Toronto

 Ovarian cancer (OvCa) is the leading cause of death among gynecological malignancies, and is characterized by peritoneal metastasis and increased resistance to chemotherapy. Acquired drug… (more)

Subjects/Keywords: Cancer Progression; Mass Spectrometry; Metastasis; Ovarian Cancer; Proteomics; 0992

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APA (6th Edition):

Musrap, N. (2015). Proteomic Identification of Mediators Implicated in the Metastatic Progression of Ovarian Cancer. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/71578

Chicago Manual of Style (16th Edition):

Musrap, Natasha. “Proteomic Identification of Mediators Implicated in the Metastatic Progression of Ovarian Cancer.” 2015. Doctoral Dissertation, University of Toronto. Accessed March 06, 2021. http://hdl.handle.net/1807/71578.

MLA Handbook (7th Edition):

Musrap, Natasha. “Proteomic Identification of Mediators Implicated in the Metastatic Progression of Ovarian Cancer.” 2015. Web. 06 Mar 2021.

Vancouver:

Musrap N. Proteomic Identification of Mediators Implicated in the Metastatic Progression of Ovarian Cancer. [Internet] [Doctoral dissertation]. University of Toronto; 2015. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1807/71578.

Council of Science Editors:

Musrap N. Proteomic Identification of Mediators Implicated in the Metastatic Progression of Ovarian Cancer. [Doctoral Dissertation]. University of Toronto; 2015. Available from: http://hdl.handle.net/1807/71578

10. VanderVeen, Brandon N. The Regulation of Skeletal Muscle Fatigue During the Progression of Cancer Cachexia.

Degree: PhD, Exercise Science, 2019, University of South Carolina

  Cachexia is the unintentional loss of body weight secondary to chronic disease and is prevalent is roughly 50% of cancer patients. The loss of… (more)

Subjects/Keywords: Exercise Science; Public Health; cancer cachexia; fatigue; skeletal; muscle; cancer; progression

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APA (6th Edition):

VanderVeen, B. N. (2019). The Regulation of Skeletal Muscle Fatigue During the Progression of Cancer Cachexia. (Doctoral Dissertation). University of South Carolina. Retrieved from https://scholarcommons.sc.edu/etd/5249

Chicago Manual of Style (16th Edition):

VanderVeen, Brandon N. “The Regulation of Skeletal Muscle Fatigue During the Progression of Cancer Cachexia.” 2019. Doctoral Dissertation, University of South Carolina. Accessed March 06, 2021. https://scholarcommons.sc.edu/etd/5249.

MLA Handbook (7th Edition):

VanderVeen, Brandon N. “The Regulation of Skeletal Muscle Fatigue During the Progression of Cancer Cachexia.” 2019. Web. 06 Mar 2021.

Vancouver:

VanderVeen BN. The Regulation of Skeletal Muscle Fatigue During the Progression of Cancer Cachexia. [Internet] [Doctoral dissertation]. University of South Carolina; 2019. [cited 2021 Mar 06]. Available from: https://scholarcommons.sc.edu/etd/5249.

Council of Science Editors:

VanderVeen BN. The Regulation of Skeletal Muscle Fatigue During the Progression of Cancer Cachexia. [Doctoral Dissertation]. University of South Carolina; 2019. Available from: https://scholarcommons.sc.edu/etd/5249


University of Louisville

11. Leggett, Carmine Simone. Role of human arylamine N-acetyltransferase in carcinogen metabolism and human breast cancer progression.

Degree: PhD, 2012, University of Louisville

 Human arylamine N-acetyltransferase (NAT) is a phase II cytosolic enzyme that occurs as two isozymes, NAT1 and NAT2. This family of polymorphic enzymes catalyzes the… (more)

Subjects/Keywords: Arylamine N-acetyltransferase; cancer; environmental carcinogens; cancer progression; carcinogen metabolism

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APA (6th Edition):

Leggett, C. S. (2012). Role of human arylamine N-acetyltransferase in carcinogen metabolism and human breast cancer progression. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/812 ; https://ir.library.louisville.edu/etd/812

Chicago Manual of Style (16th Edition):

Leggett, Carmine Simone. “Role of human arylamine N-acetyltransferase in carcinogen metabolism and human breast cancer progression.” 2012. Doctoral Dissertation, University of Louisville. Accessed March 06, 2021. 10.18297/etd/812 ; https://ir.library.louisville.edu/etd/812.

MLA Handbook (7th Edition):

Leggett, Carmine Simone. “Role of human arylamine N-acetyltransferase in carcinogen metabolism and human breast cancer progression.” 2012. Web. 06 Mar 2021.

Vancouver:

Leggett CS. Role of human arylamine N-acetyltransferase in carcinogen metabolism and human breast cancer progression. [Internet] [Doctoral dissertation]. University of Louisville; 2012. [cited 2021 Mar 06]. Available from: 10.18297/etd/812 ; https://ir.library.louisville.edu/etd/812.

Council of Science Editors:

Leggett CS. Role of human arylamine N-acetyltransferase in carcinogen metabolism and human breast cancer progression. [Doctoral Dissertation]. University of Louisville; 2012. Available from: 10.18297/etd/812 ; https://ir.library.louisville.edu/etd/812


Université de Lorraine

12. Chamard-Jovenin, Clémence. Impact d’une surexpression d’ERα36 et/ou d’une exposition aux alkylphénols sur la physiopathologie de la glande mammaire : Consequences of of ERα36 overexpression and/or alkylphenols exposure on mammary gland physiopathology.

Degree: Docteur es, Sciences de la vie et de la santé, 2016, Université de Lorraine

Durant ma thèse, j’ai étudié l’implication d’un variant du récepteur aux œstrogènes α, ERα36, dans l’initiation et la progression du cancer du sein. Au laboratoire,… (more)

Subjects/Keywords: Cancer du sein; Erα36; Initiation; Progression; Perturbateurs endocriniens; Effets transgénérationnels; Breast cancer; Erα36; Initiation; Progression; Endocrine disruptors; Transgenerational effects

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APA (6th Edition):

Chamard-Jovenin, C. (2016). Impact d’une surexpression d’ERα36 et/ou d’une exposition aux alkylphénols sur la physiopathologie de la glande mammaire : Consequences of of ERα36 overexpression and/or alkylphenols exposure on mammary gland physiopathology. (Doctoral Dissertation). Université de Lorraine. Retrieved from http://www.theses.fr/2016LORR0232

Chicago Manual of Style (16th Edition):

Chamard-Jovenin, Clémence. “Impact d’une surexpression d’ERα36 et/ou d’une exposition aux alkylphénols sur la physiopathologie de la glande mammaire : Consequences of of ERα36 overexpression and/or alkylphenols exposure on mammary gland physiopathology.” 2016. Doctoral Dissertation, Université de Lorraine. Accessed March 06, 2021. http://www.theses.fr/2016LORR0232.

MLA Handbook (7th Edition):

Chamard-Jovenin, Clémence. “Impact d’une surexpression d’ERα36 et/ou d’une exposition aux alkylphénols sur la physiopathologie de la glande mammaire : Consequences of of ERα36 overexpression and/or alkylphenols exposure on mammary gland physiopathology.” 2016. Web. 06 Mar 2021.

Vancouver:

Chamard-Jovenin C. Impact d’une surexpression d’ERα36 et/ou d’une exposition aux alkylphénols sur la physiopathologie de la glande mammaire : Consequences of of ERα36 overexpression and/or alkylphenols exposure on mammary gland physiopathology. [Internet] [Doctoral dissertation]. Université de Lorraine; 2016. [cited 2021 Mar 06]. Available from: http://www.theses.fr/2016LORR0232.

Council of Science Editors:

Chamard-Jovenin C. Impact d’une surexpression d’ERα36 et/ou d’une exposition aux alkylphénols sur la physiopathologie de la glande mammaire : Consequences of of ERα36 overexpression and/or alkylphenols exposure on mammary gland physiopathology. [Doctoral Dissertation]. Université de Lorraine; 2016. Available from: http://www.theses.fr/2016LORR0232

13. Toulet, Aurélie. Dissémination du cancer de la prostate : un chemin pavé de gras : Dissemination of prostate cancer : a way paved of fat.

Degree: Docteur es, Cancérologie, 2018, Université Toulouse III – Paul Sabatier

Le cancer de la prostate (CaP) est la première cause de cancer chez l'homme dans les pays industrialisés. Il s'agit d'une maladie hétérogène pouvant être… (more)

Subjects/Keywords: Cancer de la prostate; Microenvironnement tumoral; Adipocytes; Progression tumorale; Matrice extracellulaire; Prostate cancer; Tumor microenvironment; Adipocytes; Tumor progression; Extracellular matrix

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APA (6th Edition):

Toulet, A. (2018). Dissémination du cancer de la prostate : un chemin pavé de gras : Dissemination of prostate cancer : a way paved of fat. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2018TOU30237

Chicago Manual of Style (16th Edition):

Toulet, Aurélie. “Dissémination du cancer de la prostate : un chemin pavé de gras : Dissemination of prostate cancer : a way paved of fat.” 2018. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed March 06, 2021. http://www.theses.fr/2018TOU30237.

MLA Handbook (7th Edition):

Toulet, Aurélie. “Dissémination du cancer de la prostate : un chemin pavé de gras : Dissemination of prostate cancer : a way paved of fat.” 2018. Web. 06 Mar 2021.

Vancouver:

Toulet A. Dissémination du cancer de la prostate : un chemin pavé de gras : Dissemination of prostate cancer : a way paved of fat. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2018. [cited 2021 Mar 06]. Available from: http://www.theses.fr/2018TOU30237.

Council of Science Editors:

Toulet A. Dissémination du cancer de la prostate : un chemin pavé de gras : Dissemination of prostate cancer : a way paved of fat. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2018. Available from: http://www.theses.fr/2018TOU30237


University of Windsor

14. Abedalrhman, Alkhateeb. Machine Learning Approaches for Cancer Analysis.

Degree: PhD, Computer Science, 2018, University of Windsor

  In addition, we propose many machine learning models that serve as contributions to solve a biological problem. First, we present Zseq, a linear time… (more)

Subjects/Keywords: Breast Cancer; Cancer Progression; Machine Learning; Prostate Cancer; RNA-Seq; Survivability; Computer Sciences

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APA (6th Edition):

Abedalrhman, A. (2018). Machine Learning Approaches for Cancer Analysis. (Doctoral Dissertation). University of Windsor. Retrieved from https://scholar.uwindsor.ca/etd/7597

Chicago Manual of Style (16th Edition):

Abedalrhman, Alkhateeb. “Machine Learning Approaches for Cancer Analysis.” 2018. Doctoral Dissertation, University of Windsor. Accessed March 06, 2021. https://scholar.uwindsor.ca/etd/7597.

MLA Handbook (7th Edition):

Abedalrhman, Alkhateeb. “Machine Learning Approaches for Cancer Analysis.” 2018. Web. 06 Mar 2021.

Vancouver:

Abedalrhman A. Machine Learning Approaches for Cancer Analysis. [Internet] [Doctoral dissertation]. University of Windsor; 2018. [cited 2021 Mar 06]. Available from: https://scholar.uwindsor.ca/etd/7597.

Council of Science Editors:

Abedalrhman A. Machine Learning Approaches for Cancer Analysis. [Doctoral Dissertation]. University of Windsor; 2018. Available from: https://scholar.uwindsor.ca/etd/7597


University of Michigan

15. Moore, Heather Marie. The Role of EZH2 in Breast Cancer Progression and Metastasis.

Degree: PhD, Cellular and Molecular Biology, 2013, University of Michigan

 Understanding how breast cancer cells disseminate and metastasize is essential to develop better treatments and to improve survival. Enhancer of Zeste Homolog 2 (EZH2) is… (more)

Subjects/Keywords: Breast Cancer Progression and Metastasis; EZH2; Polycomb; Pathology; Health Sciences

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APA (6th Edition):

Moore, H. M. (2013). The Role of EZH2 in Breast Cancer Progression and Metastasis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/100098

Chicago Manual of Style (16th Edition):

Moore, Heather Marie. “The Role of EZH2 in Breast Cancer Progression and Metastasis.” 2013. Doctoral Dissertation, University of Michigan. Accessed March 06, 2021. http://hdl.handle.net/2027.42/100098.

MLA Handbook (7th Edition):

Moore, Heather Marie. “The Role of EZH2 in Breast Cancer Progression and Metastasis.” 2013. Web. 06 Mar 2021.

Vancouver:

Moore HM. The Role of EZH2 in Breast Cancer Progression and Metastasis. [Internet] [Doctoral dissertation]. University of Michigan; 2013. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/2027.42/100098.

Council of Science Editors:

Moore HM. The Role of EZH2 in Breast Cancer Progression and Metastasis. [Doctoral Dissertation]. University of Michigan; 2013. Available from: http://hdl.handle.net/2027.42/100098


Cornell University

16. Mohanan Nair Padmini, Sunish. Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation.

Degree: PhD, Veterinary Medicine, 2014, Cornell University

 Numerous recent studies have shown that epigenetic modifications play a significant role in cancer pathogenesis. The PADs are a family of epigenetic enzymes that catalyze… (more)

Subjects/Keywords: Peptidylarginine deiminase 2 (PAD2); Cancer progression; Extracellular chromatin traps (ETosis)

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APA (6th Edition):

Mohanan Nair Padmini, S. (2014). Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation. (Doctoral Dissertation). Cornell University. Retrieved from http://hdl.handle.net/1813/36176

Chicago Manual of Style (16th Edition):

Mohanan Nair Padmini, Sunish. “Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation.” 2014. Doctoral Dissertation, Cornell University. Accessed March 06, 2021. http://hdl.handle.net/1813/36176.

MLA Handbook (7th Edition):

Mohanan Nair Padmini, Sunish. “Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation.” 2014. Web. 06 Mar 2021.

Vancouver:

Mohanan Nair Padmini S. Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation. [Internet] [Doctoral dissertation]. Cornell University; 2014. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1813/36176.

Council of Science Editors:

Mohanan Nair Padmini S. Role Of Peptidylarginine Deiminase 2 (Pad2) In Epithelial Carcinogenesis And Tumor-Associated Inflammation. [Doctoral Dissertation]. Cornell University; 2014. Available from: http://hdl.handle.net/1813/36176


Harvard University

17. McFarland, Christopher Dennis. The role of deleterious passengers in cancer.

Degree: PhD, Biophysics, 2014, Harvard University

 The development of cancer from a population of precancerous cells is a rapid evolutionary process. During progression, cells evolve several new traits for survive and… (more)

Subjects/Keywords: Biophysics; Evolution & development; Genetics; Cancer Progression; Evolutionary Modeling; Genomics

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APA (6th Edition):

McFarland, C. D. (2014). The role of deleterious passengers in cancer. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070047

Chicago Manual of Style (16th Edition):

McFarland, Christopher Dennis. “The role of deleterious passengers in cancer.” 2014. Doctoral Dissertation, Harvard University. Accessed March 06, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070047.

MLA Handbook (7th Edition):

McFarland, Christopher Dennis. “The role of deleterious passengers in cancer.” 2014. Web. 06 Mar 2021.

Vancouver:

McFarland CD. The role of deleterious passengers in cancer. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2021 Mar 06]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070047.

Council of Science Editors:

McFarland CD. The role of deleterious passengers in cancer. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:13070047


University of Toronto

18. Liu, Li Yang. Association of Tissue Promoter Methylation Levels of APC, RASSF1A, CYP26A1, and TBX15 with Prostate Cancer Progression.

Degree: 2012, University of Toronto

Aberrant promoter methylation is known to silence tumor-suppressor genes in prostate cancer. Using a quantitative real-time PCR assay(MethyLight), I determined promoter methylation levels of APC,… (more)

Subjects/Keywords: DNA Methylation; Prostate Cancer; Epigenetic Mechanism; Progression; Biochemical Recurrence; 0369; 0307

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APA (6th Edition):

Liu, L. Y. (2012). Association of Tissue Promoter Methylation Levels of APC, RASSF1A, CYP26A1, and TBX15 with Prostate Cancer Progression. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/33724

Chicago Manual of Style (16th Edition):

Liu, Li Yang. “Association of Tissue Promoter Methylation Levels of APC, RASSF1A, CYP26A1, and TBX15 with Prostate Cancer Progression.” 2012. Masters Thesis, University of Toronto. Accessed March 06, 2021. http://hdl.handle.net/1807/33724.

MLA Handbook (7th Edition):

Liu, Li Yang. “Association of Tissue Promoter Methylation Levels of APC, RASSF1A, CYP26A1, and TBX15 with Prostate Cancer Progression.” 2012. Web. 06 Mar 2021.

Vancouver:

Liu LY. Association of Tissue Promoter Methylation Levels of APC, RASSF1A, CYP26A1, and TBX15 with Prostate Cancer Progression. [Internet] [Masters thesis]. University of Toronto; 2012. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1807/33724.

Council of Science Editors:

Liu LY. Association of Tissue Promoter Methylation Levels of APC, RASSF1A, CYP26A1, and TBX15 with Prostate Cancer Progression. [Masters Thesis]. University of Toronto; 2012. Available from: http://hdl.handle.net/1807/33724


University of California – San Francisco

19. Brown, Christopher Michael. Technologies for Following Active Serine Proteases in Cancer Progression and Metastasis.

Degree: Biochemistry and Molecular Biology, 2010, University of California – San Francisco

 Serine proteases acting at the cell surface are a unique class of enzymes that carry out important cell signaling functions. By acting upstream of proteolytic… (more)

Subjects/Keywords: Biochemistry; activity-based probes; cancer progression; serine protease

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APA (6th Edition):

Brown, C. M. (2010). Technologies for Following Active Serine Proteases in Cancer Progression and Metastasis. (Thesis). University of California – San Francisco. Retrieved from http://www.escholarship.org/uc/item/2sh048jb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brown, Christopher Michael. “Technologies for Following Active Serine Proteases in Cancer Progression and Metastasis.” 2010. Thesis, University of California – San Francisco. Accessed March 06, 2021. http://www.escholarship.org/uc/item/2sh048jb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brown, Christopher Michael. “Technologies for Following Active Serine Proteases in Cancer Progression and Metastasis.” 2010. Web. 06 Mar 2021.

Vancouver:

Brown CM. Technologies for Following Active Serine Proteases in Cancer Progression and Metastasis. [Internet] [Thesis]. University of California – San Francisco; 2010. [cited 2021 Mar 06]. Available from: http://www.escholarship.org/uc/item/2sh048jb.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brown CM. Technologies for Following Active Serine Proteases in Cancer Progression and Metastasis. [Thesis]. University of California – San Francisco; 2010. Available from: http://www.escholarship.org/uc/item/2sh048jb

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Queen Mary, University of London

20. Hervieu Vilches, Alexia. Understanding and targeting PI3K downstream of oncogenic Met mutant.

Degree: PhD, 2015, Queen Mary, University of London

 The Receptor Tyrosine Kinase (RTK) Met, overexpressed or mutated in cancer, plays a major role in cancer progression and represents an attractive target for cancer(more)

Subjects/Keywords: 616.99; Tumour Biology; Receptor Tyrosine Kinase; cancer progression; Met oncogenicity

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APA (6th Edition):

Hervieu Vilches, A. (2015). Understanding and targeting PI3K downstream of oncogenic Met mutant. (Doctoral Dissertation). Queen Mary, University of London. Retrieved from http://qmro.qmul.ac.uk/xmlui/handle/123456789/33935 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667429

Chicago Manual of Style (16th Edition):

Hervieu Vilches, Alexia. “Understanding and targeting PI3K downstream of oncogenic Met mutant.” 2015. Doctoral Dissertation, Queen Mary, University of London. Accessed March 06, 2021. http://qmro.qmul.ac.uk/xmlui/handle/123456789/33935 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667429.

MLA Handbook (7th Edition):

Hervieu Vilches, Alexia. “Understanding and targeting PI3K downstream of oncogenic Met mutant.” 2015. Web. 06 Mar 2021.

Vancouver:

Hervieu Vilches A. Understanding and targeting PI3K downstream of oncogenic Met mutant. [Internet] [Doctoral dissertation]. Queen Mary, University of London; 2015. [cited 2021 Mar 06]. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/33935 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667429.

Council of Science Editors:

Hervieu Vilches A. Understanding and targeting PI3K downstream of oncogenic Met mutant. [Doctoral Dissertation]. Queen Mary, University of London; 2015. Available from: http://qmro.qmul.ac.uk/xmlui/handle/123456789/33935 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.667429


University of Southern California

21. Tsen, Shao Hung Fred. The mechanism by which extracellular Hsp90α promotes cell migration: implications in wound healing and cancer.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2013, University of Southern California

 Extracellular heat shock protein-90 (eHsp90) proteins, which include the membrane-bound, released and secreted forms were first cited in scientific literature late in the 70s. It… (more)

Subjects/Keywords: wound healing; cell motility; cancer progression; secreted Hsp90; and LRP-1

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APA (6th Edition):

Tsen, S. H. F. (2013). The mechanism by which extracellular Hsp90α promotes cell migration: implications in wound healing and cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/236027/rec/6979

Chicago Manual of Style (16th Edition):

Tsen, Shao Hung Fred. “The mechanism by which extracellular Hsp90α promotes cell migration: implications in wound healing and cancer.” 2013. Doctoral Dissertation, University of Southern California. Accessed March 06, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/236027/rec/6979.

MLA Handbook (7th Edition):

Tsen, Shao Hung Fred. “The mechanism by which extracellular Hsp90α promotes cell migration: implications in wound healing and cancer.” 2013. Web. 06 Mar 2021.

Vancouver:

Tsen SHF. The mechanism by which extracellular Hsp90α promotes cell migration: implications in wound healing and cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2013. [cited 2021 Mar 06]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/236027/rec/6979.

Council of Science Editors:

Tsen SHF. The mechanism by which extracellular Hsp90α promotes cell migration: implications in wound healing and cancer. [Doctoral Dissertation]. University of Southern California; 2013. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/236027/rec/6979


Queensland University of Technology

22. Shokoohmand, Ali. Identifying the molecular mediators of VN and the IGF:VN complex-stimulated breast cancer cell survival.

Degree: 2015, Queensland University of Technology

 This project has identified a molecular signature involved in functions critical to breast cancer progression and metastasis mediated by vitronectin, an abundant protein in human… (more)

Subjects/Keywords: vitronectin; breast cancer; cancer progression; extracellular matrix; insulin-like growth factor; Attachment; vitronectin-binding integrins

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APA (6th Edition):

Shokoohmand, A. (2015). Identifying the molecular mediators of VN and the IGF:VN complex-stimulated breast cancer cell survival. (Thesis). Queensland University of Technology. Retrieved from https://eprints.qut.edu.au/85436/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shokoohmand, Ali. “Identifying the molecular mediators of VN and the IGF:VN complex-stimulated breast cancer cell survival.” 2015. Thesis, Queensland University of Technology. Accessed March 06, 2021. https://eprints.qut.edu.au/85436/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shokoohmand, Ali. “Identifying the molecular mediators of VN and the IGF:VN complex-stimulated breast cancer cell survival.” 2015. Web. 06 Mar 2021.

Vancouver:

Shokoohmand A. Identifying the molecular mediators of VN and the IGF:VN complex-stimulated breast cancer cell survival. [Internet] [Thesis]. Queensland University of Technology; 2015. [cited 2021 Mar 06]. Available from: https://eprints.qut.edu.au/85436/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shokoohmand A. Identifying the molecular mediators of VN and the IGF:VN complex-stimulated breast cancer cell survival. [Thesis]. Queensland University of Technology; 2015. Available from: https://eprints.qut.edu.au/85436/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Dardenne, Étienne. Rôle des ARN hélicases Ddx5 et Ddx17 dans la progression tumorale : Role of RNA helicases DDX5 and DDX17 in tumor progression.

Degree: Docteur es, Biologie moléculaire, 2014, Université Claude Bernard – Lyon I

La progression tumorale, qui conduit à la formation de métastases, est le résultat de profondes modifications des différents niveaux de régulation de l'expression des gènes… (more)

Subjects/Keywords: DDX5; DDX17; Épissage alternatif; Cancer du sein; Transition épithélio-mésenchymateuse; Progression tumorale; Transcription; MicroARN; DDX5; DDX17; Splicing; Breast cancer; Epithelial-to-mesenchymal transition; Tumor progression; Transcription; MicroRNA; 572.8

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APA (6th Edition):

Dardenne, E. (2014). Rôle des ARN hélicases Ddx5 et Ddx17 dans la progression tumorale : Role of RNA helicases DDX5 and DDX17 in tumor progression. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2014LYO10048

Chicago Manual of Style (16th Edition):

Dardenne, Étienne. “Rôle des ARN hélicases Ddx5 et Ddx17 dans la progression tumorale : Role of RNA helicases DDX5 and DDX17 in tumor progression.” 2014. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed March 06, 2021. http://www.theses.fr/2014LYO10048.

MLA Handbook (7th Edition):

Dardenne, Étienne. “Rôle des ARN hélicases Ddx5 et Ddx17 dans la progression tumorale : Role of RNA helicases DDX5 and DDX17 in tumor progression.” 2014. Web. 06 Mar 2021.

Vancouver:

Dardenne E. Rôle des ARN hélicases Ddx5 et Ddx17 dans la progression tumorale : Role of RNA helicases DDX5 and DDX17 in tumor progression. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2014. [cited 2021 Mar 06]. Available from: http://www.theses.fr/2014LYO10048.

Council of Science Editors:

Dardenne E. Rôle des ARN hélicases Ddx5 et Ddx17 dans la progression tumorale : Role of RNA helicases DDX5 and DDX17 in tumor progression. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2014. Available from: http://www.theses.fr/2014LYO10048

24. Pez, Floriane. Implication de la lysyl oxydase dans la réponse hypoxique et dans la progression tumorale des cellules de carcinome colorectal humain : Role of lysyl oxidase in hypoxic response and tumor progression of Human colorectal carcinoma.

Degree: Docteur es, Biologie cellulaire, 2010, Université Claude Bernard – Lyon I

Au sein d’une tumeur des régions hypoxiques se forment ce qui conduit à l’activation du facteur de transcription HIF1. HIF1, composé des deux protéines HIF1a… (more)

Subjects/Keywords: Lysyl oxydase; Hypoxie; Cancer; HIF1; PI3K/AKT; Progression tumorale; Métastases; Carcinome colorectal; Lysyl oxidase; Hypoxia; Cancer; HIF1; PI3K/AKT; Tumor progression; Metastasis; Colorectal carcinoma; 614.5999

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APA (6th Edition):

Pez, F. (2010). Implication de la lysyl oxydase dans la réponse hypoxique et dans la progression tumorale des cellules de carcinome colorectal humain : Role of lysyl oxidase in hypoxic response and tumor progression of Human colorectal carcinoma. (Doctoral Dissertation). Université Claude Bernard – Lyon I. Retrieved from http://www.theses.fr/2010LYO10141

Chicago Manual of Style (16th Edition):

Pez, Floriane. “Implication de la lysyl oxydase dans la réponse hypoxique et dans la progression tumorale des cellules de carcinome colorectal humain : Role of lysyl oxidase in hypoxic response and tumor progression of Human colorectal carcinoma.” 2010. Doctoral Dissertation, Université Claude Bernard – Lyon I. Accessed March 06, 2021. http://www.theses.fr/2010LYO10141.

MLA Handbook (7th Edition):

Pez, Floriane. “Implication de la lysyl oxydase dans la réponse hypoxique et dans la progression tumorale des cellules de carcinome colorectal humain : Role of lysyl oxidase in hypoxic response and tumor progression of Human colorectal carcinoma.” 2010. Web. 06 Mar 2021.

Vancouver:

Pez F. Implication de la lysyl oxydase dans la réponse hypoxique et dans la progression tumorale des cellules de carcinome colorectal humain : Role of lysyl oxidase in hypoxic response and tumor progression of Human colorectal carcinoma. [Internet] [Doctoral dissertation]. Université Claude Bernard – Lyon I; 2010. [cited 2021 Mar 06]. Available from: http://www.theses.fr/2010LYO10141.

Council of Science Editors:

Pez F. Implication de la lysyl oxydase dans la réponse hypoxique et dans la progression tumorale des cellules de carcinome colorectal humain : Role of lysyl oxidase in hypoxic response and tumor progression of Human colorectal carcinoma. [Doctoral Dissertation]. Université Claude Bernard – Lyon I; 2010. Available from: http://www.theses.fr/2010LYO10141

25. Da silva, Jordan. Relation entre HER2 et FHIT dans la progression tumorale des cancers broncho-pulmonaires : Relationship between HER2 and FHIT during lung tumor progression.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2020, Reims

FHIT (fragile histidine triad) est un gène fréquemment inactivé dans le cancer du poumon. Nos travaux précédents ont montré que FHIT contrôle l’invasion des cellules… (more)

Subjects/Keywords: Cancer du poumon; Fhit; Her2; Progression tumorale; Transition épithélio-Mésenchymateuse; Thérapies ciblées; Lung cancer; Fhit; Her2; Tumor progression; Epithelial-Mesemchymal transition; Targeted therapies; 616

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APA (6th Edition):

Da silva, J. (2020). Relation entre HER2 et FHIT dans la progression tumorale des cancers broncho-pulmonaires : Relationship between HER2 and FHIT during lung tumor progression. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2020REIMS021

Chicago Manual of Style (16th Edition):

Da silva, Jordan. “Relation entre HER2 et FHIT dans la progression tumorale des cancers broncho-pulmonaires : Relationship between HER2 and FHIT during lung tumor progression.” 2020. Doctoral Dissertation, Reims. Accessed March 06, 2021. http://www.theses.fr/2020REIMS021.

MLA Handbook (7th Edition):

Da silva, Jordan. “Relation entre HER2 et FHIT dans la progression tumorale des cancers broncho-pulmonaires : Relationship between HER2 and FHIT during lung tumor progression.” 2020. Web. 06 Mar 2021.

Vancouver:

Da silva J. Relation entre HER2 et FHIT dans la progression tumorale des cancers broncho-pulmonaires : Relationship between HER2 and FHIT during lung tumor progression. [Internet] [Doctoral dissertation]. Reims; 2020. [cited 2021 Mar 06]. Available from: http://www.theses.fr/2020REIMS021.

Council of Science Editors:

Da silva J. Relation entre HER2 et FHIT dans la progression tumorale des cancers broncho-pulmonaires : Relationship between HER2 and FHIT during lung tumor progression. [Doctoral Dissertation]. Reims; 2020. Available from: http://www.theses.fr/2020REIMS021


University of Illinois – Chicago

26. Pandey, Akshay. FoxM1 in Liver Cancer.

Degree: 2017, University of Illinois – Chicago

 The work presented in this thesis elucidates the role of transcription factor FoxM1 in progression of hepatocellular carcinoma. In this work, I provide the genetic… (more)

Subjects/Keywords: Liver Cancer; Hepatocellular carcinoma; HCC; FoxM1; Ras; High grade progression; FoxA1; FoxA2

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APA (6th Edition):

Pandey, A. (2017). FoxM1 in Liver Cancer. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/22070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pandey, Akshay. “FoxM1 in Liver Cancer.” 2017. Thesis, University of Illinois – Chicago. Accessed March 06, 2021. http://hdl.handle.net/10027/22070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pandey, Akshay. “FoxM1 in Liver Cancer.” 2017. Web. 06 Mar 2021.

Vancouver:

Pandey A. FoxM1 in Liver Cancer. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10027/22070.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pandey A. FoxM1 in Liver Cancer. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/22070

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Western Ontario

27. Souter, Lesley H. A Model System for Rapid Identification and Functional Testing of Genes Involved in Early Breast Cancer Progression.

Degree: 2011, University of Western Ontario

 Early breast cancer progression involves advancement through specific morphologic stages including atypical ductal hyperplasia (ADH), ductal carcinoma in situ (DCIS) and invasive mammary carcinoma (IMC),… (more)

Subjects/Keywords: breast cancer progression; atypical ductal hperplasia; ductal carcinoma in situ; invasive mammary carcinoma; Matrigel; Neoplasms

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Souter, L. H. (2011). A Model System for Rapid Identification and Functional Testing of Genes Involved in Early Breast Cancer Progression. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Souter, Lesley H. “A Model System for Rapid Identification and Functional Testing of Genes Involved in Early Breast Cancer Progression.” 2011. Thesis, University of Western Ontario. Accessed March 06, 2021. https://ir.lib.uwo.ca/etd/122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Souter, Lesley H. “A Model System for Rapid Identification and Functional Testing of Genes Involved in Early Breast Cancer Progression.” 2011. Web. 06 Mar 2021.

Vancouver:

Souter LH. A Model System for Rapid Identification and Functional Testing of Genes Involved in Early Breast Cancer Progression. [Internet] [Thesis]. University of Western Ontario; 2011. [cited 2021 Mar 06]. Available from: https://ir.lib.uwo.ca/etd/122.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Souter LH. A Model System for Rapid Identification and Functional Testing of Genes Involved in Early Breast Cancer Progression. [Thesis]. University of Western Ontario; 2011. Available from: https://ir.lib.uwo.ca/etd/122

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

28. Kazazian, Karineh. Polo-Like Kinase 4 (PLK4) Function in Cancer Progression.

Degree: PhD, 2017, University of Toronto

 Invasion and metastasis are responsible for the majority of deaths from cancer, but the molecular mechanisms that facilitate execution of the metastatic cascade remain incompletely… (more)

Subjects/Keywords: Arp2/3; BioID; Cancer Progression; Invasion and Metastasis; Plk4; Plk4 inhibitor; 0307

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kazazian, K. (2017). Polo-Like Kinase 4 (PLK4) Function in Cancer Progression. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/77481

Chicago Manual of Style (16th Edition):

Kazazian, Karineh. “Polo-Like Kinase 4 (PLK4) Function in Cancer Progression.” 2017. Doctoral Dissertation, University of Toronto. Accessed March 06, 2021. http://hdl.handle.net/1807/77481.

MLA Handbook (7th Edition):

Kazazian, Karineh. “Polo-Like Kinase 4 (PLK4) Function in Cancer Progression.” 2017. Web. 06 Mar 2021.

Vancouver:

Kazazian K. Polo-Like Kinase 4 (PLK4) Function in Cancer Progression. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/1807/77481.

Council of Science Editors:

Kazazian K. Polo-Like Kinase 4 (PLK4) Function in Cancer Progression. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/77481


University of Windsor

29. Singi Reddy, Siva Charan Reddy. USING MACHINE LEARNING TECHNIQUES FOR FINDING MEANINGFUL TRANSCRIPTS IN PROSTATE CANCER PROGRESSION.

Degree: MS, Computer Science, 2015, University of Windsor

  Prostate Cancer is one of the most common types of cancer among Canadian men. Next generation sequencing that uses RNA-Seq can be valuable in… (more)

Subjects/Keywords: Alternative Splicing; Classification; Feature Selection; Machine Learning; Next Generation Sequencing; Prostate Cancer Progression

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Singi Reddy, S. C. R. (2015). USING MACHINE LEARNING TECHNIQUES FOR FINDING MEANINGFUL TRANSCRIPTS IN PROSTATE CANCER PROGRESSION. (Masters Thesis). University of Windsor. Retrieved from https://scholar.uwindsor.ca/etd/5433

Chicago Manual of Style (16th Edition):

Singi Reddy, Siva Charan Reddy. “USING MACHINE LEARNING TECHNIQUES FOR FINDING MEANINGFUL TRANSCRIPTS IN PROSTATE CANCER PROGRESSION.” 2015. Masters Thesis, University of Windsor. Accessed March 06, 2021. https://scholar.uwindsor.ca/etd/5433.

MLA Handbook (7th Edition):

Singi Reddy, Siva Charan Reddy. “USING MACHINE LEARNING TECHNIQUES FOR FINDING MEANINGFUL TRANSCRIPTS IN PROSTATE CANCER PROGRESSION.” 2015. Web. 06 Mar 2021.

Vancouver:

Singi Reddy SCR. USING MACHINE LEARNING TECHNIQUES FOR FINDING MEANINGFUL TRANSCRIPTS IN PROSTATE CANCER PROGRESSION. [Internet] [Masters thesis]. University of Windsor; 2015. [cited 2021 Mar 06]. Available from: https://scholar.uwindsor.ca/etd/5433.

Council of Science Editors:

Singi Reddy SCR. USING MACHINE LEARNING TECHNIQUES FOR FINDING MEANINGFUL TRANSCRIPTS IN PROSTATE CANCER PROGRESSION. [Masters Thesis]. University of Windsor; 2015. Available from: https://scholar.uwindsor.ca/etd/5433

30. Kaparelou, Maria. Μελέτη της απουσίας της ισομορφής Ec του IGF-1 (MGF).

Degree: 2015, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

IGF-1 is one of the key molecules in cancer biology; however, little is known about the role of the preferential expression of the premature IGF-1… (more)

Subjects/Keywords: Πεπτίδιο Ec; Καρκίνος προστάτη; Εξέληξη όγκου; Igf1ec; Ec peptide; Prostate cancer; Tumor progression

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kaparelou, M. (2015). Μελέτη της απουσίας της ισομορφής Ec του IGF-1 (MGF). (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/39741

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kaparelou, Maria. “Μελέτη της απουσίας της ισομορφής Ec του IGF-1 (MGF).” 2015. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed March 06, 2021. http://hdl.handle.net/10442/hedi/39741.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kaparelou, Maria. “Μελέτη της απουσίας της ισομορφής Ec του IGF-1 (MGF).” 2015. Web. 06 Mar 2021.

Vancouver:

Kaparelou M. Μελέτη της απουσίας της ισομορφής Ec του IGF-1 (MGF). [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. [cited 2021 Mar 06]. Available from: http://hdl.handle.net/10442/hedi/39741.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kaparelou M. Μελέτη της απουσίας της ισομορφής Ec του IGF-1 (MGF). [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2015. Available from: http://hdl.handle.net/10442/hedi/39741

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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