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You searched for subject:(Cancer invasiveness ). Showing records 1 – 30 of 41 total matches.

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Ryerson University

1. Neshatian, Mehrnoosh. Cellular uptake and toxicity of gold nanoparticles in a tumor-like (hypoxic) environment.

Degree: 2015, Ryerson University

Cancer cells deprived adequate oxygen tension, are called hypoxic cells. Hypoxic shows resistance to both chemotherapy and/or radiotherapy. On the other hand gold Nanoparticles (GNPs)… (more)

Subjects/Keywords: Tumors; Anoxemia; Cancer invasiveness

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APA (6th Edition):

Neshatian, M. (2015). Cellular uptake and toxicity of gold nanoparticles in a tumor-like (hypoxic) environment. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A3771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Neshatian, Mehrnoosh. “Cellular uptake and toxicity of gold nanoparticles in a tumor-like (hypoxic) environment.” 2015. Thesis, Ryerson University. Accessed December 09, 2019. https://digital.library.ryerson.ca/islandora/object/RULA%3A3771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Neshatian, Mehrnoosh. “Cellular uptake and toxicity of gold nanoparticles in a tumor-like (hypoxic) environment.” 2015. Web. 09 Dec 2019.

Vancouver:

Neshatian M. Cellular uptake and toxicity of gold nanoparticles in a tumor-like (hypoxic) environment. [Internet] [Thesis]. Ryerson University; 2015. [cited 2019 Dec 09]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3771.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Neshatian M. Cellular uptake and toxicity of gold nanoparticles in a tumor-like (hypoxic) environment. [Thesis]. Ryerson University; 2015. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3771

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


IUPUI

2. Yan, Jingliang. Role of Rap1 in Angiogenesis and Tumor Invasion.

Degree: 2009, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Rap1a and Rap1b are two closely related members of the Ras family of small GTPases. Despite their high sequence similarity,… (more)

Subjects/Keywords: Hydrolases; Neovascularization; Cancer invasiveness

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APA (6th Edition):

Yan, J. (2009). Role of Rap1 in Angiogenesis and Tumor Invasion. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/1956

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yan, Jingliang. “Role of Rap1 in Angiogenesis and Tumor Invasion.” 2009. Thesis, IUPUI. Accessed December 09, 2019. http://hdl.handle.net/1805/1956.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yan, Jingliang. “Role of Rap1 in Angiogenesis and Tumor Invasion.” 2009. Web. 09 Dec 2019.

Vancouver:

Yan J. Role of Rap1 in Angiogenesis and Tumor Invasion. [Internet] [Thesis]. IUPUI; 2009. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/1805/1956.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yan J. Role of Rap1 in Angiogenesis and Tumor Invasion. [Thesis]. IUPUI; 2009. Available from: http://hdl.handle.net/1805/1956

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

3. Kasbekar, Priyanka Pratod. The Effects of Biomimetic Proteoglycans On Interstitial Flow-Induced Cancer Invasion.

Degree: 2013, Drexel University

Local microenvironments play a crucial role in cancer biology and the regulation of cellular behavior. A challenge that remains to be studied is the role… (more)

Subjects/Keywords: Biomedical engineering; Proteoglycans – Cancer invasiveness; Biomimetic polymers – Cancer invasiveness

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APA (6th Edition):

Kasbekar, P. P. (2013). The Effects of Biomimetic Proteoglycans On Interstitial Flow-Induced Cancer Invasion. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/4298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kasbekar, Priyanka Pratod. “The Effects of Biomimetic Proteoglycans On Interstitial Flow-Induced Cancer Invasion.” 2013. Thesis, Drexel University. Accessed December 09, 2019. http://hdl.handle.net/1860/4298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kasbekar, Priyanka Pratod. “The Effects of Biomimetic Proteoglycans On Interstitial Flow-Induced Cancer Invasion.” 2013. Web. 09 Dec 2019.

Vancouver:

Kasbekar PP. The Effects of Biomimetic Proteoglycans On Interstitial Flow-Induced Cancer Invasion. [Internet] [Thesis]. Drexel University; 2013. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/1860/4298.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kasbekar PP. The Effects of Biomimetic Proteoglycans On Interstitial Flow-Induced Cancer Invasion. [Thesis]. Drexel University; 2013. Available from: http://hdl.handle.net/1860/4298

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of KwaZulu-Natal

4. [No author]. Proteinases and extracellular matrix degradation in breast cancer.

Degree: Biochemistry, 2013, University of KwaZulu-Natal

 A variety of proteases have been shown to promote the progression of cancer by virtue of their ability to degrade extracellular proteinaceous barriers, such as… (more)

Subjects/Keywords: Breast – Cancer.; Cancer invasiveness.; Cathepsin D.; Proteolytic enzymes.; Extracellular matrix.; Biochemistry.

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APA (6th Edition):

author], [. (2013). Proteinases and extracellular matrix degradation in breast cancer. (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/9728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Proteinases and extracellular matrix degradation in breast cancer. ” 2013. Thesis, University of KwaZulu-Natal. Accessed December 09, 2019. http://hdl.handle.net/10413/9728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Proteinases and extracellular matrix degradation in breast cancer. ” 2013. Web. 09 Dec 2019.

Vancouver:

author] [. Proteinases and extracellular matrix degradation in breast cancer. [Internet] [Thesis]. University of KwaZulu-Natal; 2013. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10413/9728.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Proteinases and extracellular matrix degradation in breast cancer. [Thesis]. University of KwaZulu-Natal; 2013. Available from: http://hdl.handle.net/10413/9728

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

5. Giongo, Cíntia de Oliveira. Caracterização imunogenética de variantes dos genes CCR2, CCR5 e HLA-G como potenciais alvos para diagnóstico, prognóstico e tratamento do câncer de mama feminino esporádico e familial.

Degree: 2012, Universidade do Rio Grande do Sul

 O câncer de mama é a neoplasia mais comum entre as mulheres. Sua etiologia é complexa, onde tanto fatores ambientais como genéticos podem contribuir para… (more)

Subjects/Keywords: Familial breast cancer; Neoplasias da mama; Sporadic breast cancer; Invasiveness; Polymorphism

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APA (6th Edition):

Giongo, C. d. O. (2012). Caracterização imunogenética de variantes dos genes CCR2, CCR5 e HLA-G como potenciais alvos para diagnóstico, prognóstico e tratamento do câncer de mama feminino esporádico e familial. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/78137

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Giongo, Cíntia de Oliveira. “Caracterização imunogenética de variantes dos genes CCR2, CCR5 e HLA-G como potenciais alvos para diagnóstico, prognóstico e tratamento do câncer de mama feminino esporádico e familial.” 2012. Thesis, Universidade do Rio Grande do Sul. Accessed December 09, 2019. http://hdl.handle.net/10183/78137.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Giongo, Cíntia de Oliveira. “Caracterização imunogenética de variantes dos genes CCR2, CCR5 e HLA-G como potenciais alvos para diagnóstico, prognóstico e tratamento do câncer de mama feminino esporádico e familial.” 2012. Web. 09 Dec 2019.

Vancouver:

Giongo CdO. Caracterização imunogenética de variantes dos genes CCR2, CCR5 e HLA-G como potenciais alvos para diagnóstico, prognóstico e tratamento do câncer de mama feminino esporádico e familial. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2012. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10183/78137.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Giongo CdO. Caracterização imunogenética de variantes dos genes CCR2, CCR5 e HLA-G como potenciais alvos para diagnóstico, prognóstico e tratamento do câncer de mama feminino esporádico e familial. [Thesis]. Universidade do Rio Grande do Sul; 2012. Available from: http://hdl.handle.net/10183/78137

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

6. 吳磊.; Ng, Lui. Actopaxin: a novel regulator of cell migration and invasion in human hepatocellular carcinoma.

Degree: PhD, 2012, University of Hong Kong

 Invasion and metastasis are the major causes of treatment failure and high mortality rate in hepatocellular carcinoma (HCC) patients. Cell motility is crucial to tumor… (more)

Subjects/Keywords: Liver - Cancer.; Focal adhesion kinase.; Cell migration.; Cancer invasiveness.

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APA (6th Edition):

吳磊.; Ng, L. (2012). Actopaxin: a novel regulator of cell migration and invasion in human hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Ng, L. [吳磊]. (2012). Actopaxin : a novel regulator of cell migration and invasion in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775261 ; http://dx.doi.org/10.5353/th_b4775261 ; http://hdl.handle.net/10722/181471

Chicago Manual of Style (16th Edition):

吳磊.; Ng, Lui. “Actopaxin: a novel regulator of cell migration and invasion in human hepatocellular carcinoma.” 2012. Doctoral Dissertation, University of Hong Kong. Accessed December 09, 2019. Ng, L. [吳磊]. (2012). Actopaxin : a novel regulator of cell migration and invasion in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775261 ; http://dx.doi.org/10.5353/th_b4775261 ; http://hdl.handle.net/10722/181471.

MLA Handbook (7th Edition):

吳磊.; Ng, Lui. “Actopaxin: a novel regulator of cell migration and invasion in human hepatocellular carcinoma.” 2012. Web. 09 Dec 2019.

Vancouver:

吳磊.; Ng L. Actopaxin: a novel regulator of cell migration and invasion in human hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2012. [cited 2019 Dec 09]. Available from: Ng, L. [吳磊]. (2012). Actopaxin : a novel regulator of cell migration and invasion in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775261 ; http://dx.doi.org/10.5353/th_b4775261 ; http://hdl.handle.net/10722/181471.

Council of Science Editors:

吳磊.; Ng L. Actopaxin: a novel regulator of cell migration and invasion in human hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2012. Available from: Ng, L. [吳磊]. (2012). Actopaxin : a novel regulator of cell migration and invasion in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4775261 ; http://dx.doi.org/10.5353/th_b4775261 ; http://hdl.handle.net/10722/181471


Columbia University

7. Price, Jessica Caughman. Notch3 Signaling Promotes Adhesion and Tumor Progression in a Murine Epithelial Ovarian Cancer Model.

Degree: 2017, Columbia University

 Ovarian cancer is the 5th leading cause of cancer death in women in the United States and is the most fatal gynecological malignancy. High grade… (more)

Subjects/Keywords: Biology; Notch genes; Ovaries – Cancer; Cancer invasiveness; Oncology

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APA (6th Edition):

Price, J. C. (2017). Notch3 Signaling Promotes Adhesion and Tumor Progression in a Murine Epithelial Ovarian Cancer Model. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8708CWF

Chicago Manual of Style (16th Edition):

Price, Jessica Caughman. “Notch3 Signaling Promotes Adhesion and Tumor Progression in a Murine Epithelial Ovarian Cancer Model.” 2017. Doctoral Dissertation, Columbia University. Accessed December 09, 2019. https://doi.org/10.7916/D8708CWF.

MLA Handbook (7th Edition):

Price, Jessica Caughman. “Notch3 Signaling Promotes Adhesion and Tumor Progression in a Murine Epithelial Ovarian Cancer Model.” 2017. Web. 09 Dec 2019.

Vancouver:

Price JC. Notch3 Signaling Promotes Adhesion and Tumor Progression in a Murine Epithelial Ovarian Cancer Model. [Internet] [Doctoral dissertation]. Columbia University; 2017. [cited 2019 Dec 09]. Available from: https://doi.org/10.7916/D8708CWF.

Council of Science Editors:

Price JC. Notch3 Signaling Promotes Adhesion and Tumor Progression in a Murine Epithelial Ovarian Cancer Model. [Doctoral Dissertation]. Columbia University; 2017. Available from: https://doi.org/10.7916/D8708CWF


Rice University

8. Yang, Lifeng. Glutaminolysis: A Hallmark of Cancer Metabolism.

Degree: PhD, Engineering, 2016, Rice University

 The goals of these projects are to study the critical role of glutamine (Gln) in ovarian cancer growth, metastasis, drug resistance and sources of glutamine… (more)

Subjects/Keywords: Glutamine; Ovarian Cancer; Invasiveness; STAT3; Glutamine Synthetase; Cancer Associated Fibroblasts

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APA (6th Edition):

Yang, L. (2016). Glutaminolysis: A Hallmark of Cancer Metabolism. (Doctoral Dissertation). Rice University. Retrieved from http://hdl.handle.net/1911/96526

Chicago Manual of Style (16th Edition):

Yang, Lifeng. “Glutaminolysis: A Hallmark of Cancer Metabolism.” 2016. Doctoral Dissertation, Rice University. Accessed December 09, 2019. http://hdl.handle.net/1911/96526.

MLA Handbook (7th Edition):

Yang, Lifeng. “Glutaminolysis: A Hallmark of Cancer Metabolism.” 2016. Web. 09 Dec 2019.

Vancouver:

Yang L. Glutaminolysis: A Hallmark of Cancer Metabolism. [Internet] [Doctoral dissertation]. Rice University; 2016. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/1911/96526.

Council of Science Editors:

Yang L. Glutaminolysis: A Hallmark of Cancer Metabolism. [Doctoral Dissertation]. Rice University; 2016. Available from: http://hdl.handle.net/1911/96526


University of Hong Kong

9. Han, Liang. Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance.

Degree: PhD, 2015, University of Hong Kong

 Esophageal cancer is the eighth most frequent cancer and the sixth leading cause of cancer mortality in the world. Esophageal squamous cell carcinoma (ESCC) is… (more)

Subjects/Keywords: Cancer invasiveness; Small interfering RNA; Esophagus - Cancer - Chemotherapy; Drug resistance in cancer cells

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APA (6th Edition):

Han, L. (2015). Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. (Doctoral Dissertation). University of Hong Kong. Retrieved from Han, L. [韩亮]. (2015). Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5689261 ; http://dx.doi.org/10.5353/th_b5689261 ; http://hdl.handle.net/10722/235748

Chicago Manual of Style (16th Edition):

Han, Liang. “Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed December 09, 2019. Han, L. [韩亮]. (2015). Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5689261 ; http://dx.doi.org/10.5353/th_b5689261 ; http://hdl.handle.net/10722/235748.

MLA Handbook (7th Edition):

Han, Liang. “Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance.” 2015. Web. 09 Dec 2019.

Vancouver:

Han L. Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Dec 09]. Available from: Han, L. [韩亮]. (2015). Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5689261 ; http://dx.doi.org/10.5353/th_b5689261 ; http://hdl.handle.net/10722/235748.

Council of Science Editors:

Han L. Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Han, L. [韩亮]. (2015). Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5689261 ; http://dx.doi.org/10.5353/th_b5689261 ; http://hdl.handle.net/10722/235748


Dublin City University

10. Pierce, Aisling. Investigation into the molecular mechanisms underlying invasion in the lung carcinoma cell line DLKP and it's chemotherapeutic drug resistant variants.

Degree: National Institute for Cellular Biotechnology (NICB), 2006, Dublin City University

 This research described in thesis was undertaken in an attempt to increase our knowledge of mechanisms by which lung cancer cells acquire the capacity to… (more)

Subjects/Keywords: Cancer; Chemotherapeutic drugs; Resistance; Invasiveness

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APA (6th Edition):

Pierce, A. (2006). Investigation into the molecular mechanisms underlying invasion in the lung carcinoma cell line DLKP and it's chemotherapeutic drug resistant variants. (Thesis). Dublin City University. Retrieved from http://doras.dcu.ie/18157/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pierce, Aisling. “Investigation into the molecular mechanisms underlying invasion in the lung carcinoma cell line DLKP and it's chemotherapeutic drug resistant variants.” 2006. Thesis, Dublin City University. Accessed December 09, 2019. http://doras.dcu.ie/18157/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pierce, Aisling. “Investigation into the molecular mechanisms underlying invasion in the lung carcinoma cell line DLKP and it's chemotherapeutic drug resistant variants.” 2006. Web. 09 Dec 2019.

Vancouver:

Pierce A. Investigation into the molecular mechanisms underlying invasion in the lung carcinoma cell line DLKP and it's chemotherapeutic drug resistant variants. [Internet] [Thesis]. Dublin City University; 2006. [cited 2019 Dec 09]. Available from: http://doras.dcu.ie/18157/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pierce A. Investigation into the molecular mechanisms underlying invasion in the lung carcinoma cell line DLKP and it's chemotherapeutic drug resistant variants. [Thesis]. Dublin City University; 2006. Available from: http://doras.dcu.ie/18157/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

11. Sin, Sai-lung, Steven. Chloride channel in glioma cell invasion.

Degree: M. Phil., 2008, University of Hong Kong

published_or_final_version

Surgery

Master

Master of Philosophy

Advisors/Committee Members: Leung, GKK, Lui, VCH.

Subjects/Keywords: Gliomas.; Chloride channels.; Cancer invasiveness.

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APA (6th Edition):

Sin, Sai-lung, S. (2008). Chloride channel in glioma cell invasion. (Masters Thesis). University of Hong Kong. Retrieved from Sin, S. S. [冼世隆]. (2008). Chloride channel in glioma cell invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4150855 ; http://dx.doi.org/10.5353/th_b4150855 ; http://hdl.handle.net/10722/54467

Chicago Manual of Style (16th Edition):

Sin, Sai-lung, Steven. “Chloride channel in glioma cell invasion.” 2008. Masters Thesis, University of Hong Kong. Accessed December 09, 2019. Sin, S. S. [冼世隆]. (2008). Chloride channel in glioma cell invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4150855 ; http://dx.doi.org/10.5353/th_b4150855 ; http://hdl.handle.net/10722/54467.

MLA Handbook (7th Edition):

Sin, Sai-lung, Steven. “Chloride channel in glioma cell invasion.” 2008. Web. 09 Dec 2019.

Vancouver:

Sin, Sai-lung S. Chloride channel in glioma cell invasion. [Internet] [Masters thesis]. University of Hong Kong; 2008. [cited 2019 Dec 09]. Available from: Sin, S. S. [冼世隆]. (2008). Chloride channel in glioma cell invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4150855 ; http://dx.doi.org/10.5353/th_b4150855 ; http://hdl.handle.net/10722/54467.

Council of Science Editors:

Sin, Sai-lung S. Chloride channel in glioma cell invasion. [Masters Thesis]. University of Hong Kong; 2008. Available from: Sin, S. S. [冼世隆]. (2008). Chloride channel in glioma cell invasion. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4150855 ; http://dx.doi.org/10.5353/th_b4150855 ; http://hdl.handle.net/10722/54467


University of Hong Kong

12. Shih, Kendrick Co. Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation usingsmall-for-size grafts.

Degree: Master of Research in Medicine, Medicine, 2009, University of Hong Kong

published_or_final_version

Surgery

Master

Master of Research in Medicine

Subjects/Keywords: Liver transplantation.; Cancer invasiveness.; Genes.

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APA (6th Edition):

Shih, K. C. (2009). Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation usingsmall-for-size grafts. (Masters Thesis). University of Hong Kong. Retrieved from Shih, K. C. [施愷迪]. (2009). Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation using small-for-size grafts. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290529 ; http://dx.doi.org/10.5353/th_b4290529 ; http://hdl.handle.net/10722/56983

Chicago Manual of Style (16th Edition):

Shih, Kendrick Co. “Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation usingsmall-for-size grafts.” 2009. Masters Thesis, University of Hong Kong. Accessed December 09, 2019. Shih, K. C. [施愷迪]. (2009). Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation using small-for-size grafts. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290529 ; http://dx.doi.org/10.5353/th_b4290529 ; http://hdl.handle.net/10722/56983.

MLA Handbook (7th Edition):

Shih, Kendrick Co. “Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation usingsmall-for-size grafts.” 2009. Web. 09 Dec 2019.

Vancouver:

Shih KC. Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation usingsmall-for-size grafts. [Internet] [Masters thesis]. University of Hong Kong; 2009. [cited 2019 Dec 09]. Available from: Shih, K. C. [施愷迪]. (2009). Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation using small-for-size grafts. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290529 ; http://dx.doi.org/10.5353/th_b4290529 ; http://hdl.handle.net/10722/56983.

Council of Science Editors:

Shih KC. Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation usingsmall-for-size grafts. [Masters Thesis]. University of Hong Kong; 2009. Available from: Shih, K. C. [施愷迪]. (2009). Distinct gene signatures linked to acute phase injury and tumor invasiveness in tumor development after liver transplantation using small-for-size grafts. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4290529 ; http://dx.doi.org/10.5353/th_b4290529 ; http://hdl.handle.net/10722/56983


University of Arizona

13. GEHLSEN, KURT RONALD. IN VITRO AND IN VIVO STUDY OF MELANOMA TUMOR CELL INVASION AND METASTASIS.

Degree: 1986, University of Arizona

 The correlation of information obtained from in vitro investigations and in vivo experiments has frequently evaded researchers, especially in the area of tumor cell invasion… (more)

Subjects/Keywords: Cancer invasiveness.; Metastasis.; Melanoma.

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APA (6th Edition):

GEHLSEN, K. R. (1986). IN VITRO AND IN VIVO STUDY OF MELANOMA TUMOR CELL INVASION AND METASTASIS. (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/188148

Chicago Manual of Style (16th Edition):

GEHLSEN, KURT RONALD. “IN VITRO AND IN VIVO STUDY OF MELANOMA TUMOR CELL INVASION AND METASTASIS. ” 1986. Doctoral Dissertation, University of Arizona. Accessed December 09, 2019. http://hdl.handle.net/10150/188148.

MLA Handbook (7th Edition):

GEHLSEN, KURT RONALD. “IN VITRO AND IN VIVO STUDY OF MELANOMA TUMOR CELL INVASION AND METASTASIS. ” 1986. Web. 09 Dec 2019.

Vancouver:

GEHLSEN KR. IN VITRO AND IN VIVO STUDY OF MELANOMA TUMOR CELL INVASION AND METASTASIS. [Internet] [Doctoral dissertation]. University of Arizona; 1986. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10150/188148.

Council of Science Editors:

GEHLSEN KR. IN VITRO AND IN VIVO STUDY OF MELANOMA TUMOR CELL INVASION AND METASTASIS. [Doctoral Dissertation]. University of Arizona; 1986. Available from: http://hdl.handle.net/10150/188148


University of Arizona

14. Greeff, Christopher Whitney, 1961-. CYTOGENETIC ABNORMALITIES AND THE PROGRESSION TO INVASION IN A375P HUMAN MELANOMA CELLS IN VITRO .

Degree: 1987, University of Arizona

 A study was undertaken to determine whether cytogenetic abnormalities can be identified in an invasive melanoma cell population that has been selected in vitro out… (more)

Subjects/Keywords: Cytogenetics.; Melanoma.; Melanocytes.; Cancer invasiveness.

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APA (6th Edition):

Greeff, Christopher Whitney, 1. (1987). CYTOGENETIC ABNORMALITIES AND THE PROGRESSION TO INVASION IN A375P HUMAN MELANOMA CELLS IN VITRO . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/276462

Chicago Manual of Style (16th Edition):

Greeff, Christopher Whitney, 1961-. “CYTOGENETIC ABNORMALITIES AND THE PROGRESSION TO INVASION IN A375P HUMAN MELANOMA CELLS IN VITRO .” 1987. Masters Thesis, University of Arizona. Accessed December 09, 2019. http://hdl.handle.net/10150/276462.

MLA Handbook (7th Edition):

Greeff, Christopher Whitney, 1961-. “CYTOGENETIC ABNORMALITIES AND THE PROGRESSION TO INVASION IN A375P HUMAN MELANOMA CELLS IN VITRO .” 1987. Web. 09 Dec 2019.

Vancouver:

Greeff, Christopher Whitney 1. CYTOGENETIC ABNORMALITIES AND THE PROGRESSION TO INVASION IN A375P HUMAN MELANOMA CELLS IN VITRO . [Internet] [Masters thesis]. University of Arizona; 1987. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10150/276462.

Council of Science Editors:

Greeff, Christopher Whitney 1. CYTOGENETIC ABNORMALITIES AND THE PROGRESSION TO INVASION IN A375P HUMAN MELANOMA CELLS IN VITRO . [Masters Thesis]. University of Arizona; 1987. Available from: http://hdl.handle.net/10150/276462


University of KwaZulu-Natal

15. [No author]. Mathematical modeling of cancer treatments and the role of the immune system response to tumor invasion.

Degree: 2015, University of KwaZulu-Natal

 Despite the success of traditional cancer treatments, a definite cure to several cancers does not exist. Further, the traditional cancer treatments are highly toxic and… (more)

Subjects/Keywords: Cancer  – Mathematical models  – Treatment.; Cancer invasiveness.; Immune system  – Computer simulation.; Tumors  – Immunological aspects.; Applied mathematics.

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APA (6th Edition):

author], [. (2015). Mathematical modeling of cancer treatments and the role of the immune system response to tumor invasion. (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/13457

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Mathematical modeling of cancer treatments and the role of the immune system response to tumor invasion. ” 2015. Thesis, University of KwaZulu-Natal. Accessed December 09, 2019. http://hdl.handle.net/10413/13457.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Mathematical modeling of cancer treatments and the role of the immune system response to tumor invasion. ” 2015. Web. 09 Dec 2019.

Vancouver:

author] [. Mathematical modeling of cancer treatments and the role of the immune system response to tumor invasion. [Internet] [Thesis]. University of KwaZulu-Natal; 2015. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10413/13457.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Mathematical modeling of cancer treatments and the role of the immune system response to tumor invasion. [Thesis]. University of KwaZulu-Natal; 2015. Available from: http://hdl.handle.net/10413/13457

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

16. Zhao, Xin. Tumor burden induces a self-amplifying loop of myelopoiesis that Is mediated by NFκB-kit ligand signaling.

Degree: Microbiology and Molecular Genetics, 2012, Rutgers University

Subjects/Keywords: Cancer cells; Hematopoiesis; Cytokines; Cancer invasiveness

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APA (6th Edition):

Zhao, X. (2012). Tumor burden induces a self-amplifying loop of myelopoiesis that Is mediated by NFκB-kit ligand signaling. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065309

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhao, Xin. “Tumor burden induces a self-amplifying loop of myelopoiesis that Is mediated by NFκB-kit ligand signaling.” 2012. Thesis, Rutgers University. Accessed December 09, 2019. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065309.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhao, Xin. “Tumor burden induces a self-amplifying loop of myelopoiesis that Is mediated by NFκB-kit ligand signaling.” 2012. Web. 09 Dec 2019.

Vancouver:

Zhao X. Tumor burden induces a self-amplifying loop of myelopoiesis that Is mediated by NFκB-kit ligand signaling. [Internet] [Thesis]. Rutgers University; 2012. [cited 2019 Dec 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065309.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhao X. Tumor burden induces a self-amplifying loop of myelopoiesis that Is mediated by NFκB-kit ligand signaling. [Thesis]. Rutgers University; 2012. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065309

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Indian Institute of Science

17. Bashir, Mohsin. Role of Activin A Signaling in Breast Cancer.

Degree: 2014, Indian Institute of Science

 Activin-A is a member of transforming growth factor-β (TGF-β) superfamily of cytokines which includes TGF-βs, Activins, Nodal, bone morphogenetic proteins (BMPs), growth and differentiation factors… (more)

Subjects/Keywords: Breast Cancer; Activin-A Signaling; Breast Tumors - Activin- A Signaling; SMAD Signaling; SMAD3; Breast Cancer Invasiveness; Activin-A in Cancer; Oncology

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APA (6th Edition):

Bashir, M. (2014). Role of Activin A Signaling in Breast Cancer. (Thesis). Indian Institute of Science. Retrieved from http://hdl.handle.net/2005/2780

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bashir, Mohsin. “Role of Activin A Signaling in Breast Cancer.” 2014. Thesis, Indian Institute of Science. Accessed December 09, 2019. http://hdl.handle.net/2005/2780.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bashir, Mohsin. “Role of Activin A Signaling in Breast Cancer.” 2014. Web. 09 Dec 2019.

Vancouver:

Bashir M. Role of Activin A Signaling in Breast Cancer. [Internet] [Thesis]. Indian Institute of Science; 2014. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/2005/2780.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bashir M. Role of Activin A Signaling in Breast Cancer. [Thesis]. Indian Institute of Science; 2014. Available from: http://hdl.handle.net/2005/2780

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Indian Institute of Science

18. Bashir, Mohsin. Role of Activin A Signaling in Breast Cancer.

Degree: 2014, Indian Institute of Science

 Activin-A is a member of transforming growth factor-β (TGF-β) superfamily of cytokines which includes TGF-βs, Activins, Nodal, bone morphogenetic proteins (BMPs), growth and differentiation factors… (more)

Subjects/Keywords: Breast Cancer; Activin-A Signaling; Breast Tumors - Activin- A Signaling; SMAD Signaling; SMAD3; Breast Cancer Invasiveness; Activin-A in Cancer; Oncology

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APA (6th Edition):

Bashir, M. (2014). Role of Activin A Signaling in Breast Cancer. (Thesis). Indian Institute of Science. Retrieved from http://etd.iisc.ernet.in/handle/2005/2780 ; http://etd.ncsi.iisc.ernet.in/abstracts/3650/G26601-Abs.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bashir, Mohsin. “Role of Activin A Signaling in Breast Cancer.” 2014. Thesis, Indian Institute of Science. Accessed December 09, 2019. http://etd.iisc.ernet.in/handle/2005/2780 ; http://etd.ncsi.iisc.ernet.in/abstracts/3650/G26601-Abs.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bashir, Mohsin. “Role of Activin A Signaling in Breast Cancer.” 2014. Web. 09 Dec 2019.

Vancouver:

Bashir M. Role of Activin A Signaling in Breast Cancer. [Internet] [Thesis]. Indian Institute of Science; 2014. [cited 2019 Dec 09]. Available from: http://etd.iisc.ernet.in/handle/2005/2780 ; http://etd.ncsi.iisc.ernet.in/abstracts/3650/G26601-Abs.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bashir M. Role of Activin A Signaling in Breast Cancer. [Thesis]. Indian Institute of Science; 2014. Available from: http://etd.iisc.ernet.in/handle/2005/2780 ; http://etd.ncsi.iisc.ernet.in/abstracts/3650/G26601-Abs.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

19. 周穎嫻; Chow, Wing-han, Vivian. Genes associated with invasion and metastasis of head and neck cancer.

Degree: M. Phil., 2000, University of Hong Kong

published_or_final_version

Surgery

Master

Master of Philosophy

Subjects/Keywords: Head - Cancer.; Neck - Cancer.; Metastasis.; Cancer invasiveness.

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APA (6th Edition):

周穎嫻; Chow, Wing-han, V. (2000). Genes associated with invasion and metastasis of head and neck cancer. (Masters Thesis). University of Hong Kong. Retrieved from Chow, W. V. [周穎嫻]. (2000). Genes associated with invasion and metastasis of head and neck cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122246 ; http://dx.doi.org/10.5353/th_b3122246 ; http://hdl.handle.net/10722/33531

Chicago Manual of Style (16th Edition):

周穎嫻; Chow, Wing-han, Vivian. “Genes associated with invasion and metastasis of head and neck cancer.” 2000. Masters Thesis, University of Hong Kong. Accessed December 09, 2019. Chow, W. V. [周穎嫻]. (2000). Genes associated with invasion and metastasis of head and neck cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122246 ; http://dx.doi.org/10.5353/th_b3122246 ; http://hdl.handle.net/10722/33531.

MLA Handbook (7th Edition):

周穎嫻; Chow, Wing-han, Vivian. “Genes associated with invasion and metastasis of head and neck cancer.” 2000. Web. 09 Dec 2019.

Vancouver:

周穎嫻; Chow, Wing-han V. Genes associated with invasion and metastasis of head and neck cancer. [Internet] [Masters thesis]. University of Hong Kong; 2000. [cited 2019 Dec 09]. Available from: Chow, W. V. [周穎嫻]. (2000). Genes associated with invasion and metastasis of head and neck cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122246 ; http://dx.doi.org/10.5353/th_b3122246 ; http://hdl.handle.net/10722/33531.

Council of Science Editors:

周穎嫻; Chow, Wing-han V. Genes associated with invasion and metastasis of head and neck cancer. [Masters Thesis]. University of Hong Kong; 2000. Available from: Chow, W. V. [周穎嫻]. (2000). Genes associated with invasion and metastasis of head and neck cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122246 ; http://dx.doi.org/10.5353/th_b3122246 ; http://hdl.handle.net/10722/33531


Dublin City University

20. Murray, David W. Molecular mechanisms of human colorectal cancer metastasis: identification of novel biomarkers.

Degree: School of Biotechnology, 2004, Dublin City University

 Tumour metastasis represents the most lethal aspect o f cancer. It involves initial attachment of cells to the extracellular matrix (ECM), degradation of the ECM… (more)

Subjects/Keywords: Biotechnology; Biochemical markers; Cancer invasiveness; Colon (Anatomy) Cancer Pathophysiology

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APA (6th Edition):

Murray, D. W. (2004). Molecular mechanisms of human colorectal cancer metastasis: identification of novel biomarkers. (Thesis). Dublin City University. Retrieved from http://doras.dcu.ie/19168/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Murray, David W. “Molecular mechanisms of human colorectal cancer metastasis: identification of novel biomarkers.” 2004. Thesis, Dublin City University. Accessed December 09, 2019. http://doras.dcu.ie/19168/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Murray, David W. “Molecular mechanisms of human colorectal cancer metastasis: identification of novel biomarkers.” 2004. Web. 09 Dec 2019.

Vancouver:

Murray DW. Molecular mechanisms of human colorectal cancer metastasis: identification of novel biomarkers. [Internet] [Thesis]. Dublin City University; 2004. [cited 2019 Dec 09]. Available from: http://doras.dcu.ie/19168/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Murray DW. Molecular mechanisms of human colorectal cancer metastasis: identification of novel biomarkers. [Thesis]. Dublin City University; 2004. Available from: http://doras.dcu.ie/19168/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Montana State University

21. Hamner, Steve. Expression of ras and metastatic behavior in panel of cell lines derived from infection of NIH 3T3 cells with Kirsten murine sarcoma virus.

Degree: College of Agriculture, 1991, Montana State University

Subjects/Keywords: Ras oncogenes.; Cancer Genetic aspects.; Cancer invasiveness.; Metastasis.

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APA (6th Edition):

Hamner, S. (1991). Expression of ras and metastatic behavior in panel of cell lines derived from infection of NIH 3T3 cells with Kirsten murine sarcoma virus. (Thesis). Montana State University. Retrieved from https://scholarworks.montana.edu/xmlui/handle/1/6850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hamner, Steve. “Expression of ras and metastatic behavior in panel of cell lines derived from infection of NIH 3T3 cells with Kirsten murine sarcoma virus.” 1991. Thesis, Montana State University. Accessed December 09, 2019. https://scholarworks.montana.edu/xmlui/handle/1/6850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hamner, Steve. “Expression of ras and metastatic behavior in panel of cell lines derived from infection of NIH 3T3 cells with Kirsten murine sarcoma virus.” 1991. Web. 09 Dec 2019.

Vancouver:

Hamner S. Expression of ras and metastatic behavior in panel of cell lines derived from infection of NIH 3T3 cells with Kirsten murine sarcoma virus. [Internet] [Thesis]. Montana State University; 1991. [cited 2019 Dec 09]. Available from: https://scholarworks.montana.edu/xmlui/handle/1/6850.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hamner S. Expression of ras and metastatic behavior in panel of cell lines derived from infection of NIH 3T3 cells with Kirsten murine sarcoma virus. [Thesis]. Montana State University; 1991. Available from: https://scholarworks.montana.edu/xmlui/handle/1/6850

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Wannous, Ramez. L'inhibition du récepteur nucléaire PPARB diminue la croissance et l'invasivité des cellules cancéreuses mammaires humaines : Inhibition of the nuclear receptor PPARB reduces growth and invasiveness of human breast cancer cells.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2014, Université François-Rabelais de Tours

Les récepteurs nucléaires PPAR sont régulés par les acides gras polyinsaturés n-3 (AGPI n-3), dont l’acide docosahexaènoïque (DHA, 22:6n-3) qui inhibe la croissance des lignées… (more)

Subjects/Keywords: PPAR; DHA; Cancer du sein; Croissance; Invasivité; NaV 1.5; PPAR; DHA; Breast cancer; Growth; Invasiveness; NaV 1.5

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APA (6th Edition):

Wannous, R. (2014). L'inhibition du récepteur nucléaire PPARB diminue la croissance et l'invasivité des cellules cancéreuses mammaires humaines : Inhibition of the nuclear receptor PPARB reduces growth and invasiveness of human breast cancer cells. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2014TOUR3307

Chicago Manual of Style (16th Edition):

Wannous, Ramez. “L'inhibition du récepteur nucléaire PPARB diminue la croissance et l'invasivité des cellules cancéreuses mammaires humaines : Inhibition of the nuclear receptor PPARB reduces growth and invasiveness of human breast cancer cells.” 2014. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed December 09, 2019. http://www.theses.fr/2014TOUR3307.

MLA Handbook (7th Edition):

Wannous, Ramez. “L'inhibition du récepteur nucléaire PPARB diminue la croissance et l'invasivité des cellules cancéreuses mammaires humaines : Inhibition of the nuclear receptor PPARB reduces growth and invasiveness of human breast cancer cells.” 2014. Web. 09 Dec 2019.

Vancouver:

Wannous R. L'inhibition du récepteur nucléaire PPARB diminue la croissance et l'invasivité des cellules cancéreuses mammaires humaines : Inhibition of the nuclear receptor PPARB reduces growth and invasiveness of human breast cancer cells. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2014. [cited 2019 Dec 09]. Available from: http://www.theses.fr/2014TOUR3307.

Council of Science Editors:

Wannous R. L'inhibition du récepteur nucléaire PPARB diminue la croissance et l'invasivité des cellules cancéreuses mammaires humaines : Inhibition of the nuclear receptor PPARB reduces growth and invasiveness of human breast cancer cells. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2014. Available from: http://www.theses.fr/2014TOUR3307


University of KwaZulu-Natal

23. [No author]. A study of the proteinase, cathepsin L, in the context of tumour invasion.

Degree: Biochemistry, 1990, University of KwaZulu-Natal

 The proteinase, cathepsin L, has been strongly implicated in the processes of tumour invasion and metastasis. A new purification method, three-phase partitioning, characterised in terms… (more)

Subjects/Keywords: Cathepsin L – Purification.; Cysteine proteinases.; Cancer invasiveness.; Biochemistry.

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APA (6th Edition):

author], [. (1990). A study of the proteinase, cathepsin L, in the context of tumour invasion. (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/9536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “A study of the proteinase, cathepsin L, in the context of tumour invasion. ” 1990. Thesis, University of KwaZulu-Natal. Accessed December 09, 2019. http://hdl.handle.net/10413/9536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “A study of the proteinase, cathepsin L, in the context of tumour invasion. ” 1990. Web. 09 Dec 2019.

Vancouver:

author] [. A study of the proteinase, cathepsin L, in the context of tumour invasion. [Internet] [Thesis]. University of KwaZulu-Natal; 1990. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10413/9536.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. A study of the proteinase, cathepsin L, in the context of tumour invasion. [Thesis]. University of KwaZulu-Natal; 1990. Available from: http://hdl.handle.net/10413/9536

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Univerzitet u Beogradu

24. Milošević, Zorica, 1985-. Uloga mTOR i MAPK signalnih puteva u rezistenciji karcinoma štitaste žlezde na hemioterapiju.

Degree: Biološki fakultet, 2016, Univerzitet u Beogradu

Molekularna onkologija - Kancerogeneza / Molecular oncology - Cancerogenesis

Kаrcinomi štitаste žlezde su nаjčešći mаligniteti endokrinog sistemа. Klаsifikаcijа ovih mаlignitetа je izvršenа nа osnovu njihovih… (more)

Subjects/Keywords: Anaplastic thyroid cancer (ATC); RAS/MAPK/ERKpathway; PI3K/AKT/mTOR pathway; ATC chemoresistance; ATC invasiveness; dual mTOR inhibitor; AZD2014; paclitaxel (PTX)

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APA (6th Edition):

Milošević, Zorica, 1. (2016). Uloga mTOR i MAPK signalnih puteva u rezistenciji karcinoma štitaste žlezde na hemioterapiju. (Thesis). Univerzitet u Beogradu. Retrieved from https://fedorabg.bg.ac.rs/fedora/get/o:11053/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Milošević, Zorica, 1985-. “Uloga mTOR i MAPK signalnih puteva u rezistenciji karcinoma štitaste žlezde na hemioterapiju.” 2016. Thesis, Univerzitet u Beogradu. Accessed December 09, 2019. https://fedorabg.bg.ac.rs/fedora/get/o:11053/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Milošević, Zorica, 1985-. “Uloga mTOR i MAPK signalnih puteva u rezistenciji karcinoma štitaste žlezde na hemioterapiju.” 2016. Web. 09 Dec 2019.

Vancouver:

Milošević, Zorica 1. Uloga mTOR i MAPK signalnih puteva u rezistenciji karcinoma štitaste žlezde na hemioterapiju. [Internet] [Thesis]. Univerzitet u Beogradu; 2016. [cited 2019 Dec 09]. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:11053/bdef:Content/get.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Milošević, Zorica 1. Uloga mTOR i MAPK signalnih puteva u rezistenciji karcinoma štitaste žlezde na hemioterapiju. [Thesis]. Univerzitet u Beogradu; 2016. Available from: https://fedorabg.bg.ac.rs/fedora/get/o:11053/bdef:Content/get

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Hudson, Corey M. Informatic approaches to evolutionary systems biology.

Degree: 2013, University of Missouri – Columbia

 The sheer complexity of evolutionary systems biology requires us to develop more sophisticated tools for analysis, as well as more probing and biologically relevant representations… (more)

Subjects/Keywords: Systems biology; Evolution (Biology); Genetics; Cancer invasiveness

…prostate cancers). I found an overabundance of genes that share expression between cancer… 

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APA (6th Edition):

Hudson, C. M. (2013). Informatic approaches to evolutionary systems biology. (Thesis). University of Missouri – Columbia. Retrieved from http://hdl.handle.net/10355/40062

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hudson, Corey M. “Informatic approaches to evolutionary systems biology.” 2013. Thesis, University of Missouri – Columbia. Accessed December 09, 2019. http://hdl.handle.net/10355/40062.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hudson, Corey M. “Informatic approaches to evolutionary systems biology.” 2013. Web. 09 Dec 2019.

Vancouver:

Hudson CM. Informatic approaches to evolutionary systems biology. [Internet] [Thesis]. University of Missouri – Columbia; 2013. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10355/40062.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hudson CM. Informatic approaches to evolutionary systems biology. [Thesis]. University of Missouri – Columbia; 2013. Available from: http://hdl.handle.net/10355/40062

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Arizona

26. Bevacqua, Sandra Jean. An in vitro study of human melanoma tumor cell metastasis: Cytological and molecular events during extravasation.

Degree: 1989, University of Arizona

 In order to study the process by which human melanoma cells achieve invasion of basement membranes, a modification of the Membrane Invasion Culture System was… (more)

Subjects/Keywords: Cancer invasiveness.; Metastasis.; Membrane, Basement.; Melanoma.; Gene amplification.

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APA (6th Edition):

Bevacqua, S. J. (1989). An in vitro study of human melanoma tumor cell metastasis: Cytological and molecular events during extravasation. (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/184746

Chicago Manual of Style (16th Edition):

Bevacqua, Sandra Jean. “An in vitro study of human melanoma tumor cell metastasis: Cytological and molecular events during extravasation. ” 1989. Doctoral Dissertation, University of Arizona. Accessed December 09, 2019. http://hdl.handle.net/10150/184746.

MLA Handbook (7th Edition):

Bevacqua, Sandra Jean. “An in vitro study of human melanoma tumor cell metastasis: Cytological and molecular events during extravasation. ” 1989. Web. 09 Dec 2019.

Vancouver:

Bevacqua SJ. An in vitro study of human melanoma tumor cell metastasis: Cytological and molecular events during extravasation. [Internet] [Doctoral dissertation]. University of Arizona; 1989. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10150/184746.

Council of Science Editors:

Bevacqua SJ. An in vitro study of human melanoma tumor cell metastasis: Cytological and molecular events during extravasation. [Doctoral Dissertation]. University of Arizona; 1989. Available from: http://hdl.handle.net/10150/184746


University of Hong Kong

27. Han, Liang. Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance.

Degree: PhD, 2015, University of Hong Kong

abstract

Biomedical Sciences

Doctoral

Doctor of Philosophy

Subjects/Keywords: Esophagus - Cancer - Chemotherapy; Small interfering RNA; Cancer invasiveness; Drug resistance in cancer cells

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APA (6th Edition):

Han, L. (2015). Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/222337

Chicago Manual of Style (16th Edition):

Han, Liang. “Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed December 09, 2019. http://hdl.handle.net/10722/222337.

MLA Handbook (7th Edition):

Han, Liang. “Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance.” 2015. Web. 09 Dec 2019.

Vancouver:

Han L. Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10722/222337.

Council of Science Editors:

Han L. Role of miR-338-5p in targeting Id-1 and modulating esophageal cancer cell invasion and chemoresistance. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/222337


University of KwaZulu-Natal

28. Coetzer, Theresa Helen Taillefer. Type IV collagenase and cathepsins L and H : proteinases involved in tumour invasion.

Degree: PhD, 1992, University of KwaZulu-Natal

 The collagenolytic proteinases, type IV collagenase and cathepsins Land H, have been implicated in tumour invasion and metastasis, by virtue of their degradative action on… (more)

Subjects/Keywords: Cancer invasiveness.; Collagenases.; Cysteine proteinases.; Proteolytic enzymes.; Cathepsin L.; Cathepsin H.

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APA (6th Edition):

Coetzer, T. H. T. (1992). Type IV collagenase and cathepsins L and H : proteinases involved in tumour invasion. (Doctoral Dissertation). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/9532

Chicago Manual of Style (16th Edition):

Coetzer, Theresa Helen Taillefer. “Type IV collagenase and cathepsins L and H : proteinases involved in tumour invasion.” 1992. Doctoral Dissertation, University of KwaZulu-Natal. Accessed December 09, 2019. http://hdl.handle.net/10413/9532.

MLA Handbook (7th Edition):

Coetzer, Theresa Helen Taillefer. “Type IV collagenase and cathepsins L and H : proteinases involved in tumour invasion.” 1992. Web. 09 Dec 2019.

Vancouver:

Coetzer THT. Type IV collagenase and cathepsins L and H : proteinases involved in tumour invasion. [Internet] [Doctoral dissertation]. University of KwaZulu-Natal; 1992. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10413/9532.

Council of Science Editors:

Coetzer THT. Type IV collagenase and cathepsins L and H : proteinases involved in tumour invasion. [Doctoral Dissertation]. University of KwaZulu-Natal; 1992. Available from: http://hdl.handle.net/10413/9532

29. Liu, Lu. The effect of hypoxia and 3D culture conditions on heterogeneous ovarian cancer spheroids.

Degree: MS, Human Nutrition, Foods, and Exercise, 2017, Virginia Tech

 Epithelial ovarian cancer (EOC) is the leading cause of death from gynecological malignancy due to the insufficient accurate screening programs for the early detection of… (more)

Subjects/Keywords: ovarian cancer; metabolism; hypoxia; spheroids; stromal vascular fractions; invasiveness

…4 Figure 1-2 Progression of epithelial ovarian cancer… …12 Figure 1-5 The formation of cancer stem cells… …14 Figure 3-1 The glycolytic shift results of MOSE cancer cells in 3D culture under hypoxia… …30 Figure 3-2 The comparison of glycolytic shift results of MOSE cancer cells in 3D and 2D… …34 Figure 3-3 The impact of SVF cells and MS1 cells on glycolytic shift of MOSE cancer… 

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APA (6th Edition):

Liu, L. (2017). The effect of hypoxia and 3D culture conditions on heterogeneous ovarian cancer spheroids. (Masters Thesis). Virginia Tech. Retrieved from http://hdl.handle.net/10919/74235

Chicago Manual of Style (16th Edition):

Liu, Lu. “The effect of hypoxia and 3D culture conditions on heterogeneous ovarian cancer spheroids.” 2017. Masters Thesis, Virginia Tech. Accessed December 09, 2019. http://hdl.handle.net/10919/74235.

MLA Handbook (7th Edition):

Liu, Lu. “The effect of hypoxia and 3D culture conditions on heterogeneous ovarian cancer spheroids.” 2017. Web. 09 Dec 2019.

Vancouver:

Liu L. The effect of hypoxia and 3D culture conditions on heterogeneous ovarian cancer spheroids. [Internet] [Masters thesis]. Virginia Tech; 2017. [cited 2019 Dec 09]. Available from: http://hdl.handle.net/10919/74235.

Council of Science Editors:

Liu L. The effect of hypoxia and 3D culture conditions on heterogeneous ovarian cancer spheroids. [Masters Thesis]. Virginia Tech; 2017. Available from: http://hdl.handle.net/10919/74235

30. Brisson, Lucie. Modulation de l'échangeur Na+/H+ de type 1 (NHE1) par le canal sodique dépendant du voltage Nav1.5 : implication dans l'invasivité de cellules cancéreuses mammaires humaines : Modulation of type 1 Na+/H+ exchanger (NHE1) by Nav1.5 voltage-gated sodium channel : involvement in human breast cancer cells invasiveness.

Degree: Docteur es, Sciences de la Vie et de la Santé, 2012, Université François-Rabelais de Tours

Les cellules cancéreuses mammaires invasives expriment des canaux sodiques NaV1.5 dont l’activité semble être associée au développement métastatique. L’activité de ce canal dans les cellules… (more)

Subjects/Keywords: Canaux sodiques dépendants du voltage; Échangeurs sodium-proton; Invadopodes; Invasivité des cellules cancéreuses; Voltage-gated sodium channel; Sodium-proton exchangers; Invadopodia; Cancer cells invasiveness

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APA (6th Edition):

Brisson, L. (2012). Modulation de l'échangeur Na+/H+ de type 1 (NHE1) par le canal sodique dépendant du voltage Nav1.5 : implication dans l'invasivité de cellules cancéreuses mammaires humaines : Modulation of type 1 Na+/H+ exchanger (NHE1) by Nav1.5 voltage-gated sodium channel : involvement in human breast cancer cells invasiveness. (Doctoral Dissertation). Université François-Rabelais de Tours. Retrieved from http://www.theses.fr/2012TOUR4038

Chicago Manual of Style (16th Edition):

Brisson, Lucie. “Modulation de l'échangeur Na+/H+ de type 1 (NHE1) par le canal sodique dépendant du voltage Nav1.5 : implication dans l'invasivité de cellules cancéreuses mammaires humaines : Modulation of type 1 Na+/H+ exchanger (NHE1) by Nav1.5 voltage-gated sodium channel : involvement in human breast cancer cells invasiveness.” 2012. Doctoral Dissertation, Université François-Rabelais de Tours. Accessed December 09, 2019. http://www.theses.fr/2012TOUR4038.

MLA Handbook (7th Edition):

Brisson, Lucie. “Modulation de l'échangeur Na+/H+ de type 1 (NHE1) par le canal sodique dépendant du voltage Nav1.5 : implication dans l'invasivité de cellules cancéreuses mammaires humaines : Modulation of type 1 Na+/H+ exchanger (NHE1) by Nav1.5 voltage-gated sodium channel : involvement in human breast cancer cells invasiveness.” 2012. Web. 09 Dec 2019.

Vancouver:

Brisson L. Modulation de l'échangeur Na+/H+ de type 1 (NHE1) par le canal sodique dépendant du voltage Nav1.5 : implication dans l'invasivité de cellules cancéreuses mammaires humaines : Modulation of type 1 Na+/H+ exchanger (NHE1) by Nav1.5 voltage-gated sodium channel : involvement in human breast cancer cells invasiveness. [Internet] [Doctoral dissertation]. Université François-Rabelais de Tours; 2012. [cited 2019 Dec 09]. Available from: http://www.theses.fr/2012TOUR4038.

Council of Science Editors:

Brisson L. Modulation de l'échangeur Na+/H+ de type 1 (NHE1) par le canal sodique dépendant du voltage Nav1.5 : implication dans l'invasivité de cellules cancéreuses mammaires humaines : Modulation of type 1 Na+/H+ exchanger (NHE1) by Nav1.5 voltage-gated sodium channel : involvement in human breast cancer cells invasiveness. [Doctoral Dissertation]. Université François-Rabelais de Tours; 2012. Available from: http://www.theses.fr/2012TOUR4038

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