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You searched for subject:(Cancer Biology). Showing records 1 – 30 of 2238 total matches.

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Université Laval

1. Milanese, Jean-Sébastien. Développement d'un algorithme permettant la prédiction des métastases à partir de mutations germinales et celles du clone fondateur chez des patients atteints du cancer.

Degree: 2018, Université Laval

Avec les constantes avancées du séquençage de nouvelle génération (NGS), la quantité de données disponibles devient massive. En parallèle, les méthodes de détection au cancer(more)

Subjects/Keywords: Mutation (Biologie); Algorithmes; Cancer  – Dépistage; Cancer  – Pronostic

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APA (6th Edition):

Milanese, J. (2018). Développement d'un algorithme permettant la prédiction des métastases à partir de mutations germinales et celles du clone fondateur chez des patients atteints du cancer. (Thesis). Université Laval. Retrieved from http://hdl.handle.net/20.500.11794/28327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Milanese, Jean-Sébastien. “Développement d'un algorithme permettant la prédiction des métastases à partir de mutations germinales et celles du clone fondateur chez des patients atteints du cancer.” 2018. Thesis, Université Laval. Accessed February 19, 2020. http://hdl.handle.net/20.500.11794/28327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Milanese, Jean-Sébastien. “Développement d'un algorithme permettant la prédiction des métastases à partir de mutations germinales et celles du clone fondateur chez des patients atteints du cancer.” 2018. Web. 19 Feb 2020.

Vancouver:

Milanese J. Développement d'un algorithme permettant la prédiction des métastases à partir de mutations germinales et celles du clone fondateur chez des patients atteints du cancer. [Internet] [Thesis]. Université Laval; 2018. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/20.500.11794/28327.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Milanese J. Développement d'un algorithme permettant la prédiction des métastases à partir de mutations germinales et celles du clone fondateur chez des patients atteints du cancer. [Thesis]. Université Laval; 2018. Available from: http://hdl.handle.net/20.500.11794/28327

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Karjalainen, Katja. Candidate therapeutic targets against acute myeloid leukemia identified via screening combinatorial peptide and chemical libraries.

Degree: Department of Biosciences, Biokemia ja biotekniikka; The University of Texas - M. D. Anderson Cancer Center, Houston, Texas, USA, 2015, University of Helsinki

 Acute myeloid leukemia (AML), although rare, is highly malignant neoplasms that account for a majority of leukemia-associated deaths. AML results from an over-growth of immature… (more)

Subjects/Keywords: Cancer Biology; Cancer Biology

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APA (6th Edition):

Karjalainen, K. (2015). Candidate therapeutic targets against acute myeloid leukemia identified via screening combinatorial peptide and chemical libraries. (Doctoral Dissertation). University of Helsinki. Retrieved from http://hdl.handle.net/10138/154447

Chicago Manual of Style (16th Edition):

Karjalainen, Katja. “Candidate therapeutic targets against acute myeloid leukemia identified via screening combinatorial peptide and chemical libraries.” 2015. Doctoral Dissertation, University of Helsinki. Accessed February 19, 2020. http://hdl.handle.net/10138/154447.

MLA Handbook (7th Edition):

Karjalainen, Katja. “Candidate therapeutic targets against acute myeloid leukemia identified via screening combinatorial peptide and chemical libraries.” 2015. Web. 19 Feb 2020.

Vancouver:

Karjalainen K. Candidate therapeutic targets against acute myeloid leukemia identified via screening combinatorial peptide and chemical libraries. [Internet] [Doctoral dissertation]. University of Helsinki; 2015. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/10138/154447.

Council of Science Editors:

Karjalainen K. Candidate therapeutic targets against acute myeloid leukemia identified via screening combinatorial peptide and chemical libraries. [Doctoral Dissertation]. University of Helsinki; 2015. Available from: http://hdl.handle.net/10138/154447


Texas Medical Center

3. Zhang, Sicong. FUNCTION AND MECHANISM OF ALKBH5 IN N6-METHYL-ADENOSINE RNA MODIFICATION IN GLIOBLASTOMA.

Degree: PhD, 2016, Texas Medical Center

  N6-methyl-adenosine (m6A) is the most prevalent internal chemical modification of mRNAs in eukaryotes. In mammals, m6A installed by m6A methyltransferases METTL3 and METTL14 is… (more)

Subjects/Keywords: Cancer Biology

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APA (6th Edition):

Zhang, S. (2016). FUNCTION AND MECHANISM OF ALKBH5 IN N6-METHYL-ADENOSINE RNA MODIFICATION IN GLIOBLASTOMA. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/698

Chicago Manual of Style (16th Edition):

Zhang, Sicong. “FUNCTION AND MECHANISM OF ALKBH5 IN N6-METHYL-ADENOSINE RNA MODIFICATION IN GLIOBLASTOMA.” 2016. Doctoral Dissertation, Texas Medical Center. Accessed February 19, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/698.

MLA Handbook (7th Edition):

Zhang, Sicong. “FUNCTION AND MECHANISM OF ALKBH5 IN N6-METHYL-ADENOSINE RNA MODIFICATION IN GLIOBLASTOMA.” 2016. Web. 19 Feb 2020.

Vancouver:

Zhang S. FUNCTION AND MECHANISM OF ALKBH5 IN N6-METHYL-ADENOSINE RNA MODIFICATION IN GLIOBLASTOMA. [Internet] [Doctoral dissertation]. Texas Medical Center; 2016. [cited 2020 Feb 19]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/698.

Council of Science Editors:

Zhang S. FUNCTION AND MECHANISM OF ALKBH5 IN N6-METHYL-ADENOSINE RNA MODIFICATION IN GLIOBLASTOMA. [Doctoral Dissertation]. Texas Medical Center; 2016. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/698


University of Arizona

4. Lee, Kristy. Targeting Copper: A Therapeutic Strategy In Lymphoma .

Degree: 2013, University of Arizona

 Non-Hodgkin lymphomas often arise at sites of chronic inflammation, exposing them to oxidative stress, or increased levels of reactive oxygen species (ROS). Increases in ROS… (more)

Subjects/Keywords: Cancer Biology

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APA (6th Edition):

Lee, K. (2013). Targeting Copper: A Therapeutic Strategy In Lymphoma . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/294030

Chicago Manual of Style (16th Edition):

Lee, Kristy. “Targeting Copper: A Therapeutic Strategy In Lymphoma .” 2013. Doctoral Dissertation, University of Arizona. Accessed February 19, 2020. http://hdl.handle.net/10150/294030.

MLA Handbook (7th Edition):

Lee, Kristy. “Targeting Copper: A Therapeutic Strategy In Lymphoma .” 2013. Web. 19 Feb 2020.

Vancouver:

Lee K. Targeting Copper: A Therapeutic Strategy In Lymphoma . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/10150/294030.

Council of Science Editors:

Lee K. Targeting Copper: A Therapeutic Strategy In Lymphoma . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/294030


University of Arizona

5. Alizadeh, Darya. Doxorubicin and T Helper Lymphocytes: Unexpected Allies Against Cancer .

Degree: 2013, University of Arizona

 Despite considerable progress in conventional cancer therapies, major challenges persist in the treatment of patients with advanced stage malignancies. Cancer immunotherapy (harnessing the immune system… (more)

Subjects/Keywords: Cancer Biology

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APA (6th Edition):

Alizadeh, D. (2013). Doxorubicin and T Helper Lymphocytes: Unexpected Allies Against Cancer . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/307049

Chicago Manual of Style (16th Edition):

Alizadeh, Darya. “Doxorubicin and T Helper Lymphocytes: Unexpected Allies Against Cancer .” 2013. Doctoral Dissertation, University of Arizona. Accessed February 19, 2020. http://hdl.handle.net/10150/307049.

MLA Handbook (7th Edition):

Alizadeh, Darya. “Doxorubicin and T Helper Lymphocytes: Unexpected Allies Against Cancer .” 2013. Web. 19 Feb 2020.

Vancouver:

Alizadeh D. Doxorubicin and T Helper Lymphocytes: Unexpected Allies Against Cancer . [Internet] [Doctoral dissertation]. University of Arizona; 2013. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/10150/307049.

Council of Science Editors:

Alizadeh D. Doxorubicin and T Helper Lymphocytes: Unexpected Allies Against Cancer . [Doctoral Dissertation]. University of Arizona; 2013. Available from: http://hdl.handle.net/10150/307049


University of Arizona

6. Hanke, Neale T. Molecular Regulation of the Tumor Killing Activity of Dendritic Cells .

Degree: 2012, University of Arizona

 Primarily defined by their antigen presenting function, dendritic cells (DC) are equipped with the unique ability to initiate and regulate immune responses. A less conventional… (more)

Subjects/Keywords: Cancer Biology

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APA (6th Edition):

Hanke, N. T. (2012). Molecular Regulation of the Tumor Killing Activity of Dendritic Cells . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/255197

Chicago Manual of Style (16th Edition):

Hanke, Neale T. “Molecular Regulation of the Tumor Killing Activity of Dendritic Cells .” 2012. Doctoral Dissertation, University of Arizona. Accessed February 19, 2020. http://hdl.handle.net/10150/255197.

MLA Handbook (7th Edition):

Hanke, Neale T. “Molecular Regulation of the Tumor Killing Activity of Dendritic Cells .” 2012. Web. 19 Feb 2020.

Vancouver:

Hanke NT. Molecular Regulation of the Tumor Killing Activity of Dendritic Cells . [Internet] [Doctoral dissertation]. University of Arizona; 2012. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/10150/255197.

Council of Science Editors:

Hanke NT. Molecular Regulation of the Tumor Killing Activity of Dendritic Cells . [Doctoral Dissertation]. University of Arizona; 2012. Available from: http://hdl.handle.net/10150/255197


University of Arizona

7. Tseng, Roger Sean. High-risk HPV: From Infection to Cervical Cancer Progression .

Degree: 2015, University of Arizona

 Human papillomaviruses (HPV) are small non-enveloped viruses that infect basal cells. Most HPV infections are mild and develop warts at the site of infection. However,… (more)

Subjects/Keywords: Cancer Biology

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APA (6th Edition):

Tseng, R. S. (2015). High-risk HPV: From Infection to Cervical Cancer Progression . (Masters Thesis). University of Arizona. Retrieved from http://hdl.handle.net/10150/604865

Chicago Manual of Style (16th Edition):

Tseng, Roger Sean. “High-risk HPV: From Infection to Cervical Cancer Progression .” 2015. Masters Thesis, University of Arizona. Accessed February 19, 2020. http://hdl.handle.net/10150/604865.

MLA Handbook (7th Edition):

Tseng, Roger Sean. “High-risk HPV: From Infection to Cervical Cancer Progression .” 2015. Web. 19 Feb 2020.

Vancouver:

Tseng RS. High-risk HPV: From Infection to Cervical Cancer Progression . [Internet] [Masters thesis]. University of Arizona; 2015. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/10150/604865.

Council of Science Editors:

Tseng RS. High-risk HPV: From Infection to Cervical Cancer Progression . [Masters Thesis]. University of Arizona; 2015. Available from: http://hdl.handle.net/10150/604865

8. Jackson, Robert. Knockout of the trans-acting riboswitches in Listeria monocytogenes to disrupt downstream virulence factors.

Degree: MS, Biological Sciences, 2019, Encompass Digital Archive, Eastern Kentucky University

  Listeria Monocytogenes (L. monocytogenes) is a ubiquitous bacterium that can be found in both soil and water. This opportunistic intercellular pathogen is often food-borne… (more)

Subjects/Keywords: Cancer Biology

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APA (6th Edition):

Jackson, R. (2019). Knockout of the trans-acting riboswitches in Listeria monocytogenes to disrupt downstream virulence factors. (Masters Thesis). Encompass Digital Archive, Eastern Kentucky University. Retrieved from https://encompass.eku.edu/etd/610

Chicago Manual of Style (16th Edition):

Jackson, Robert. “Knockout of the trans-acting riboswitches in Listeria monocytogenes to disrupt downstream virulence factors.” 2019. Masters Thesis, Encompass Digital Archive, Eastern Kentucky University. Accessed February 19, 2020. https://encompass.eku.edu/etd/610.

MLA Handbook (7th Edition):

Jackson, Robert. “Knockout of the trans-acting riboswitches in Listeria monocytogenes to disrupt downstream virulence factors.” 2019. Web. 19 Feb 2020.

Vancouver:

Jackson R. Knockout of the trans-acting riboswitches in Listeria monocytogenes to disrupt downstream virulence factors. [Internet] [Masters thesis]. Encompass Digital Archive, Eastern Kentucky University; 2019. [cited 2020 Feb 19]. Available from: https://encompass.eku.edu/etd/610.

Council of Science Editors:

Jackson R. Knockout of the trans-acting riboswitches in Listeria monocytogenes to disrupt downstream virulence factors. [Masters Thesis]. Encompass Digital Archive, Eastern Kentucky University; 2019. Available from: https://encompass.eku.edu/etd/610

9. Cabanettes, Aurore. Caractérisation et modification de lectines fongiques pour la reconnaissance spécifique de motifs glucidiques principalement associés aux cancers : Characterization and modification of fungal lectins for specific binding to carbohydrates patterns mainly involved in cancers.

Degree: Docteur es, Chimie biologie, 2019, Grenoble Alpes

Les lectines sont des protéines ubiquitaires qui se lient spécifiquement et de manière réversible aux sucres sans les modifier. Elles peuvent déchiffrer le glycocode :… (more)

Subjects/Keywords: Biomarqueurs; Champignon; Cancer; Biologie structurale; Lectins; Biomarkers; Mushroom; Cancer; Structural biology; 570

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APA (6th Edition):

Cabanettes, A. (2019). Caractérisation et modification de lectines fongiques pour la reconnaissance spécifique de motifs glucidiques principalement associés aux cancers : Characterization and modification of fungal lectins for specific binding to carbohydrates patterns mainly involved in cancers. (Doctoral Dissertation). Grenoble Alpes. Retrieved from http://www.theses.fr/2019GREAV021

Chicago Manual of Style (16th Edition):

Cabanettes, Aurore. “Caractérisation et modification de lectines fongiques pour la reconnaissance spécifique de motifs glucidiques principalement associés aux cancers : Characterization and modification of fungal lectins for specific binding to carbohydrates patterns mainly involved in cancers.” 2019. Doctoral Dissertation, Grenoble Alpes. Accessed February 19, 2020. http://www.theses.fr/2019GREAV021.

MLA Handbook (7th Edition):

Cabanettes, Aurore. “Caractérisation et modification de lectines fongiques pour la reconnaissance spécifique de motifs glucidiques principalement associés aux cancers : Characterization and modification of fungal lectins for specific binding to carbohydrates patterns mainly involved in cancers.” 2019. Web. 19 Feb 2020.

Vancouver:

Cabanettes A. Caractérisation et modification de lectines fongiques pour la reconnaissance spécifique de motifs glucidiques principalement associés aux cancers : Characterization and modification of fungal lectins for specific binding to carbohydrates patterns mainly involved in cancers. [Internet] [Doctoral dissertation]. Grenoble Alpes; 2019. [cited 2020 Feb 19]. Available from: http://www.theses.fr/2019GREAV021.

Council of Science Editors:

Cabanettes A. Caractérisation et modification de lectines fongiques pour la reconnaissance spécifique de motifs glucidiques principalement associés aux cancers : Characterization and modification of fungal lectins for specific binding to carbohydrates patterns mainly involved in cancers. [Doctoral Dissertation]. Grenoble Alpes; 2019. Available from: http://www.theses.fr/2019GREAV021


California State University – Sacramento

10. Keeling, Michael Thomas. HMA is potently cytotoxic against apoptosis-resistant CSC irrespective of tumor type.

Degree: MS, (Biological Sciences (Molecular and Cellular Biology, 2019, California State University – Sacramento

 Major advances in our understanding of the molecular mechanisms underlying the malignant progression of human cancers have been borne over the last decade. Such insight… (more)

Subjects/Keywords: Cancer; Cancer biology; Lysosome

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APA (6th Edition):

Keeling, M. T. (2019). HMA is potently cytotoxic against apoptosis-resistant CSC irrespective of tumor type. (Masters Thesis). California State University – Sacramento. Retrieved from http://hdl.handle.net/10211.3/207970

Chicago Manual of Style (16th Edition):

Keeling, Michael Thomas. “HMA is potently cytotoxic against apoptosis-resistant CSC irrespective of tumor type.” 2019. Masters Thesis, California State University – Sacramento. Accessed February 19, 2020. http://hdl.handle.net/10211.3/207970.

MLA Handbook (7th Edition):

Keeling, Michael Thomas. “HMA is potently cytotoxic against apoptosis-resistant CSC irrespective of tumor type.” 2019. Web. 19 Feb 2020.

Vancouver:

Keeling MT. HMA is potently cytotoxic against apoptosis-resistant CSC irrespective of tumor type. [Internet] [Masters thesis]. California State University – Sacramento; 2019. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/10211.3/207970.

Council of Science Editors:

Keeling MT. HMA is potently cytotoxic against apoptosis-resistant CSC irrespective of tumor type. [Masters Thesis]. California State University – Sacramento; 2019. Available from: http://hdl.handle.net/10211.3/207970


Drexel University

11. Feng, Yayi. Targeting Aurora A kinase (AURKA) and p21-activated kinase 1 (PAK1) in hormone receptor-positive breast cancer.

Degree: 2016, Drexel University

Estrogen receptor [alpha] (ER[alpha]) is a hormone-dependent nuclear and cytoplasmic receptor that regulates many physiological processes, such as reproduction, bone integrity, cardiovascular health, cognition, and… (more)

Subjects/Keywords: Cancer biology; Biology; Cancer; Biochemical Phenomena; Neoplasms

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APA (6th Edition):

Feng, Y. (2016). Targeting Aurora A kinase (AURKA) and p21-activated kinase 1 (PAK1) in hormone receptor-positive breast cancer. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7173

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Feng, Yayi. “Targeting Aurora A kinase (AURKA) and p21-activated kinase 1 (PAK1) in hormone receptor-positive breast cancer.” 2016. Thesis, Drexel University. Accessed February 19, 2020. http://hdl.handle.net/1860/idea:7173.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Feng, Yayi. “Targeting Aurora A kinase (AURKA) and p21-activated kinase 1 (PAK1) in hormone receptor-positive breast cancer.” 2016. Web. 19 Feb 2020.

Vancouver:

Feng Y. Targeting Aurora A kinase (AURKA) and p21-activated kinase 1 (PAK1) in hormone receptor-positive breast cancer. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/1860/idea:7173.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Feng Y. Targeting Aurora A kinase (AURKA) and p21-activated kinase 1 (PAK1) in hormone receptor-positive breast cancer. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7173

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

12. Deihimi, Safoora. Mutation profiling in colorectal cancer patients with microsatellite instability: High frequency of BRCA2 and EGFR somatic mutations.

Degree: 2016, Drexel University

Microsatellite instability (MSI) is a hallmark of deficient mismatch repair (MMR) and contributes to ~15% of colorectal cancer (CRCs). MSI can lead to mutations in… (more)

Subjects/Keywords: Cancer biology; Biochemical Phenomena; Neoplasms; Biology; Cancer

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APA (6th Edition):

Deihimi, S. (2016). Mutation profiling in colorectal cancer patients with microsatellite instability: High frequency of BRCA2 and EGFR somatic mutations. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Deihimi, Safoora. “Mutation profiling in colorectal cancer patients with microsatellite instability: High frequency of BRCA2 and EGFR somatic mutations.” 2016. Thesis, Drexel University. Accessed February 19, 2020. http://hdl.handle.net/1860/idea:7170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Deihimi, Safoora. “Mutation profiling in colorectal cancer patients with microsatellite instability: High frequency of BRCA2 and EGFR somatic mutations.” 2016. Web. 19 Feb 2020.

Vancouver:

Deihimi S. Mutation profiling in colorectal cancer patients with microsatellite instability: High frequency of BRCA2 and EGFR somatic mutations. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/1860/idea:7170.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Deihimi S. Mutation profiling in colorectal cancer patients with microsatellite instability: High frequency of BRCA2 and EGFR somatic mutations. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7170

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Drexel University

13. Rollins, Dustin. Netrin-G1 Enhances in Desmoplastic Fibroblasts Enhances Interactions with Pancreatic Cancer Cells.

Degree: 2016, Drexel University

Pancreatic Ductal Adenocarcinoma (PDAC) is amongst the most lethal forms of cancer encountered in patients. One of the defining characteristics of this neoplasia is a… (more)

Subjects/Keywords: Cancer biology; Cancer; Biochemical Phenomena; Neoplasms; Biology

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APA (6th Edition):

Rollins, D. (2016). Netrin-G1 Enhances in Desmoplastic Fibroblasts Enhances Interactions with Pancreatic Cancer Cells. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/idea:7164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rollins, Dustin. “Netrin-G1 Enhances in Desmoplastic Fibroblasts Enhances Interactions with Pancreatic Cancer Cells.” 2016. Thesis, Drexel University. Accessed February 19, 2020. http://hdl.handle.net/1860/idea:7164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rollins, Dustin. “Netrin-G1 Enhances in Desmoplastic Fibroblasts Enhances Interactions with Pancreatic Cancer Cells.” 2016. Web. 19 Feb 2020.

Vancouver:

Rollins D. Netrin-G1 Enhances in Desmoplastic Fibroblasts Enhances Interactions with Pancreatic Cancer Cells. [Internet] [Thesis]. Drexel University; 2016. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/1860/idea:7164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rollins D. Netrin-G1 Enhances in Desmoplastic Fibroblasts Enhances Interactions with Pancreatic Cancer Cells. [Thesis]. Drexel University; 2016. Available from: http://hdl.handle.net/1860/idea:7164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Spencer, Myla Renee. Identification of Mutation Profiles in Ovarian Cancer and Clinically Actionable Biomarkers.

Degree: PhD, Biology, 2015, Catholic U of America

Sixty-five percent of the 22,000 women diagnosed with ovarian cancer in the United States this year will succumb to the disease because most women are… (more)

Subjects/Keywords: Biology; Molecular biology; Cancer; Genomics

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APA (6th Edition):

Spencer, M. R. (2015). Identification of Mutation Profiles in Ovarian Cancer and Clinically Actionable Biomarkers. (Doctoral Dissertation). Catholic U of America. Retrieved from http://hdl.handle.net/1961/cuislandora:28279

Chicago Manual of Style (16th Edition):

Spencer, Myla Renee. “Identification of Mutation Profiles in Ovarian Cancer and Clinically Actionable Biomarkers.” 2015. Doctoral Dissertation, Catholic U of America. Accessed February 19, 2020. http://hdl.handle.net/1961/cuislandora:28279.

MLA Handbook (7th Edition):

Spencer, Myla Renee. “Identification of Mutation Profiles in Ovarian Cancer and Clinically Actionable Biomarkers.” 2015. Web. 19 Feb 2020.

Vancouver:

Spencer MR. Identification of Mutation Profiles in Ovarian Cancer and Clinically Actionable Biomarkers. [Internet] [Doctoral dissertation]. Catholic U of America; 2015. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/1961/cuislandora:28279.

Council of Science Editors:

Spencer MR. Identification of Mutation Profiles in Ovarian Cancer and Clinically Actionable Biomarkers. [Doctoral Dissertation]. Catholic U of America; 2015. Available from: http://hdl.handle.net/1961/cuislandora:28279

15. Baur, Brittany. Identifying Regulators from Multiple Types of Biological Data in Cancer.

Degree: 2017, Marquette University

Cancer genomes accumulate alterations that promote cancer cell proliferation and survival. Structural, genetic and epigenetic alterations that have a selective advantage for tumorigenesis affect key… (more)

Subjects/Keywords: Cancer Biology; Genomics

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APA (6th Edition):

Baur, B. (2017). Identifying Regulators from Multiple Types of Biological Data in Cancer. (Thesis). Marquette University. Retrieved from https://epublications.marquette.edu/dissertations_mu/706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Baur, Brittany. “Identifying Regulators from Multiple Types of Biological Data in Cancer.” 2017. Thesis, Marquette University. Accessed February 19, 2020. https://epublications.marquette.edu/dissertations_mu/706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Baur, Brittany. “Identifying Regulators from Multiple Types of Biological Data in Cancer.” 2017. Web. 19 Feb 2020.

Vancouver:

Baur B. Identifying Regulators from Multiple Types of Biological Data in Cancer. [Internet] [Thesis]. Marquette University; 2017. [cited 2020 Feb 19]. Available from: https://epublications.marquette.edu/dissertations_mu/706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Baur B. Identifying Regulators from Multiple Types of Biological Data in Cancer. [Thesis]. Marquette University; 2017. Available from: https://epublications.marquette.edu/dissertations_mu/706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cambridge

16. Batty, Elizabeth. Fusion genes in breast cancer.

Degree: PhD, 2012, University of Cambridge

 Fusion genes caused by chromosomal rearrangements are a common and important feature in haematological malignancies, but have until recently been seen as unimportant in epithelial… (more)

Subjects/Keywords: Cancer; Molecular biology

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APA (6th Edition):

Batty, E. (2012). Fusion genes in breast cancer. (Doctoral Dissertation). University of Cambridge. Retrieved from http://www.dspace.cam.ac.uk/handle/1810/241721https://www.repository.cam.ac.uk/bitstream/1810/241721/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/3/license_url ; https://www.repository.cam.ac.uk/bitstream/1810/241721/4/license_text ; https://www.repository.cam.ac.uk/bitstream/1810/241721/5/license_rdf ; https://www.repository.cam.ac.uk/bitstream/1810/241721/8/thesis_corrected.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/9/thesis_corrected.pdf.jpg

Chicago Manual of Style (16th Edition):

Batty, Elizabeth. “Fusion genes in breast cancer.” 2012. Doctoral Dissertation, University of Cambridge. Accessed February 19, 2020. http://www.dspace.cam.ac.uk/handle/1810/241721https://www.repository.cam.ac.uk/bitstream/1810/241721/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/3/license_url ; https://www.repository.cam.ac.uk/bitstream/1810/241721/4/license_text ; https://www.repository.cam.ac.uk/bitstream/1810/241721/5/license_rdf ; https://www.repository.cam.ac.uk/bitstream/1810/241721/8/thesis_corrected.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/9/thesis_corrected.pdf.jpg.

MLA Handbook (7th Edition):

Batty, Elizabeth. “Fusion genes in breast cancer.” 2012. Web. 19 Feb 2020.

Vancouver:

Batty E. Fusion genes in breast cancer. [Internet] [Doctoral dissertation]. University of Cambridge; 2012. [cited 2020 Feb 19]. Available from: http://www.dspace.cam.ac.uk/handle/1810/241721https://www.repository.cam.ac.uk/bitstream/1810/241721/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/3/license_url ; https://www.repository.cam.ac.uk/bitstream/1810/241721/4/license_text ; https://www.repository.cam.ac.uk/bitstream/1810/241721/5/license_rdf ; https://www.repository.cam.ac.uk/bitstream/1810/241721/8/thesis_corrected.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/9/thesis_corrected.pdf.jpg.

Council of Science Editors:

Batty E. Fusion genes in breast cancer. [Doctoral Dissertation]. University of Cambridge; 2012. Available from: http://www.dspace.cam.ac.uk/handle/1810/241721https://www.repository.cam.ac.uk/bitstream/1810/241721/2/license.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/3/license_url ; https://www.repository.cam.ac.uk/bitstream/1810/241721/4/license_text ; https://www.repository.cam.ac.uk/bitstream/1810/241721/5/license_rdf ; https://www.repository.cam.ac.uk/bitstream/1810/241721/8/thesis_corrected.pdf.txt ; https://www.repository.cam.ac.uk/bitstream/1810/241721/9/thesis_corrected.pdf.jpg


Arizona State University

17. ARIYASINGHE, NETHMI KANCHANA. Biophysical Characterization of Cancer Cell Phenotypes.

Degree: Physics, 2019, Arizona State University

Cancer is a serious health concern. Current treatments are limited due to certain subpopulations of cancer cells being resistant to chemotherapy and radiation. These subpopulations… (more)

Subjects/Keywords: Biophysics; Cancer biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

ARIYASINGHE, N. K. (2019). Biophysical Characterization of Cancer Cell Phenotypes. (Doctoral Dissertation). Arizona State University. Retrieved from http://repository.asu.edu/items/53955

Chicago Manual of Style (16th Edition):

ARIYASINGHE, NETHMI KANCHANA. “Biophysical Characterization of Cancer Cell Phenotypes.” 2019. Doctoral Dissertation, Arizona State University. Accessed February 19, 2020. http://repository.asu.edu/items/53955.

MLA Handbook (7th Edition):

ARIYASINGHE, NETHMI KANCHANA. “Biophysical Characterization of Cancer Cell Phenotypes.” 2019. Web. 19 Feb 2020.

Vancouver:

ARIYASINGHE NK. Biophysical Characterization of Cancer Cell Phenotypes. [Internet] [Doctoral dissertation]. Arizona State University; 2019. [cited 2020 Feb 19]. Available from: http://repository.asu.edu/items/53955.

Council of Science Editors:

ARIYASINGHE NK. Biophysical Characterization of Cancer Cell Phenotypes. [Doctoral Dissertation]. Arizona State University; 2019. Available from: http://repository.asu.edu/items/53955


University of Central Florida

18. Carr, Ana. Chaperonin Containing TCP1 (CCT) as a Target for Cancer Therapy.

Degree: 2017, University of Central Florida

 Treatments for aggressive cancers like triple negative breast cancer (TNBC) and small-cell lung cancer (SCLC) have not improved and remain associated with debilitating side effects.… (more)

Subjects/Keywords: Biotechnology; Cancer Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carr, A. (2017). Chaperonin Containing TCP1 (CCT) as a Target for Cancer Therapy. (Doctoral Dissertation). University of Central Florida. Retrieved from https://stars.library.ucf.edu/etd/6032

Chicago Manual of Style (16th Edition):

Carr, Ana. “Chaperonin Containing TCP1 (CCT) as a Target for Cancer Therapy.” 2017. Doctoral Dissertation, University of Central Florida. Accessed February 19, 2020. https://stars.library.ucf.edu/etd/6032.

MLA Handbook (7th Edition):

Carr, Ana. “Chaperonin Containing TCP1 (CCT) as a Target for Cancer Therapy.” 2017. Web. 19 Feb 2020.

Vancouver:

Carr A. Chaperonin Containing TCP1 (CCT) as a Target for Cancer Therapy. [Internet] [Doctoral dissertation]. University of Central Florida; 2017. [cited 2020 Feb 19]. Available from: https://stars.library.ucf.edu/etd/6032.

Council of Science Editors:

Carr A. Chaperonin Containing TCP1 (CCT) as a Target for Cancer Therapy. [Doctoral Dissertation]. University of Central Florida; 2017. Available from: https://stars.library.ucf.edu/etd/6032


University of Central Florida

19. Higgins, Lyn. The Identification and Segmentation of Astrocytoma Prior to Critical Mass, by means of a Volumetric/Subregion Regression Analysis of Normal and Neoplastic Brain Tissue.

Degree: 2018, University of Central Florida

 As the underlying cause of Glioblastoma Multiforme (GBM) is presently unclear, this research implements a new approach to identifying and segmenting plausible instances of GBM… (more)

Subjects/Keywords: Cancer Biology; Oncology

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APA (6th Edition):

Higgins, L. (2018). The Identification and Segmentation of Astrocytoma Prior to Critical Mass, by means of a Volumetric/Subregion Regression Analysis of Normal and Neoplastic Brain Tissue. (Masters Thesis). University of Central Florida. Retrieved from https://stars.library.ucf.edu/etd/6254

Chicago Manual of Style (16th Edition):

Higgins, Lyn. “The Identification and Segmentation of Astrocytoma Prior to Critical Mass, by means of a Volumetric/Subregion Regression Analysis of Normal and Neoplastic Brain Tissue.” 2018. Masters Thesis, University of Central Florida. Accessed February 19, 2020. https://stars.library.ucf.edu/etd/6254.

MLA Handbook (7th Edition):

Higgins, Lyn. “The Identification and Segmentation of Astrocytoma Prior to Critical Mass, by means of a Volumetric/Subregion Regression Analysis of Normal and Neoplastic Brain Tissue.” 2018. Web. 19 Feb 2020.

Vancouver:

Higgins L. The Identification and Segmentation of Astrocytoma Prior to Critical Mass, by means of a Volumetric/Subregion Regression Analysis of Normal and Neoplastic Brain Tissue. [Internet] [Masters thesis]. University of Central Florida; 2018. [cited 2020 Feb 19]. Available from: https://stars.library.ucf.edu/etd/6254.

Council of Science Editors:

Higgins L. The Identification and Segmentation of Astrocytoma Prior to Critical Mass, by means of a Volumetric/Subregion Regression Analysis of Normal and Neoplastic Brain Tissue. [Masters Thesis]. University of Central Florida; 2018. Available from: https://stars.library.ucf.edu/etd/6254


Université Laval

20. Guedaoura, Sonya. Pratiques de dépistage du cancer des femmes non porteuses de mutations familiales des gènes BRCA1/2.

Degree: 2017, Université Laval

 Les femmes non porteuses de mutations familiales des gènes BRCA1 ou BRCA2 présentent un risque de cancers du sein et de l'ovaire généralement comparable à… (more)

Subjects/Keywords: Sein  – Cancer  – Dépistage; Ovaires  – Cancer  – Dépistage; Mutation (Biologie)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Guedaoura, S. (2017). Pratiques de dépistage du cancer des femmes non porteuses de mutations familiales des gènes BRCA1/2. (Thesis). Université Laval. Retrieved from http://hdl.handle.net/20.500.11794/27686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Guedaoura, Sonya. “Pratiques de dépistage du cancer des femmes non porteuses de mutations familiales des gènes BRCA1/2.” 2017. Thesis, Université Laval. Accessed February 19, 2020. http://hdl.handle.net/20.500.11794/27686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Guedaoura, Sonya. “Pratiques de dépistage du cancer des femmes non porteuses de mutations familiales des gènes BRCA1/2.” 2017. Web. 19 Feb 2020.

Vancouver:

Guedaoura S. Pratiques de dépistage du cancer des femmes non porteuses de mutations familiales des gènes BRCA1/2. [Internet] [Thesis]. Université Laval; 2017. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/20.500.11794/27686.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Guedaoura S. Pratiques de dépistage du cancer des femmes non porteuses de mutations familiales des gènes BRCA1/2. [Thesis]. Université Laval; 2017. Available from: http://hdl.handle.net/20.500.11794/27686

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

21. Muradyan, Artur. Die Rolle von SPON2 im Rahmen der epithelial-mesenchymalen Transition.

Degree: 2010, Freie Universität Berlin

 Der Prozess in dem Epithelzellen in Zellen mit mesenchymalen Eigenschaften übergehen wird allgemein als Epitheliale-Mesenchymale Transition (EMT) bezeichnet. EMT ist unerlässlich für normales Wachstum, einschließlich… (more)

Subjects/Keywords: Epithelial-mesenchymal transition; cancer; metastasis; functional genomics; systems; biology; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Muradyan, A. (2010). Die Rolle von SPON2 im Rahmen der epithelial-mesenchymalen Transition. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-14059

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muradyan, Artur. “Die Rolle von SPON2 im Rahmen der epithelial-mesenchymalen Transition.” 2010. Thesis, Freie Universität Berlin. Accessed February 19, 2020. http://dx.doi.org/10.17169/refubium-14059.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muradyan, Artur. “Die Rolle von SPON2 im Rahmen der epithelial-mesenchymalen Transition.” 2010. Web. 19 Feb 2020.

Vancouver:

Muradyan A. Die Rolle von SPON2 im Rahmen der epithelial-mesenchymalen Transition. [Internet] [Thesis]. Freie Universität Berlin; 2010. [cited 2020 Feb 19]. Available from: http://dx.doi.org/10.17169/refubium-14059.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muradyan A. Die Rolle von SPON2 im Rahmen der epithelial-mesenchymalen Transition. [Thesis]. Freie Universität Berlin; 2010. Available from: http://dx.doi.org/10.17169/refubium-14059

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Loyola University Chicago

22. Davidson, Valerie. Emetine as an Anti-Cancer Therapeutic in Bladder Cancer.

Degree: MS, Molecular Biology, 2015, Loyola University Chicago

  Bladder cancer is a serious health concern among the older population, as it is responsible for thousands of deaths annually in the United States.… (more)

Subjects/Keywords: Bladder Cancer; Cancer Biology; Cancer Therapeutics; Molecular Biology; Molecular Biology

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APA (6th Edition):

Davidson, V. (2015). Emetine as an Anti-Cancer Therapeutic in Bladder Cancer. (Thesis). Loyola University Chicago. Retrieved from http://ecommons.luc.edu/luc_theses/3129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davidson, Valerie. “Emetine as an Anti-Cancer Therapeutic in Bladder Cancer.” 2015. Thesis, Loyola University Chicago. Accessed February 19, 2020. http://ecommons.luc.edu/luc_theses/3129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davidson, Valerie. “Emetine as an Anti-Cancer Therapeutic in Bladder Cancer.” 2015. Web. 19 Feb 2020.

Vancouver:

Davidson V. Emetine as an Anti-Cancer Therapeutic in Bladder Cancer. [Internet] [Thesis]. Loyola University Chicago; 2015. [cited 2020 Feb 19]. Available from: http://ecommons.luc.edu/luc_theses/3129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davidson V. Emetine as an Anti-Cancer Therapeutic in Bladder Cancer. [Thesis]. Loyola University Chicago; 2015. Available from: http://ecommons.luc.edu/luc_theses/3129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Freie Universität Berlin

23. Schade, Sophia. Molekulare und funktionale Charakterisierung des Galectin-3-bindenden Proteins LGALS3BP in Krebs und der Zentrosomenbiologie.

Degree: 2017, Freie Universität Berlin

 Das Zentrosom stellt das wesentliche Mikrotubuli-organisierende Zentrum in eukaryotischen Zellen dar. Neben seiner Funktion bei der Mikrotubulinukleation ist es beteiligt an der Integration von zellulären… (more)

Subjects/Keywords: LGALS3BP; centrosome; cancer; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

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APA (6th Edition):

Schade, S. (2017). Molekulare und funktionale Charakterisierung des Galectin-3-bindenden Proteins LGALS3BP in Krebs und der Zentrosomenbiologie. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-6313

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schade, Sophia. “Molekulare und funktionale Charakterisierung des Galectin-3-bindenden Proteins LGALS3BP in Krebs und der Zentrosomenbiologie.” 2017. Thesis, Freie Universität Berlin. Accessed February 19, 2020. http://dx.doi.org/10.17169/refubium-6313.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schade, Sophia. “Molekulare und funktionale Charakterisierung des Galectin-3-bindenden Proteins LGALS3BP in Krebs und der Zentrosomenbiologie.” 2017. Web. 19 Feb 2020.

Vancouver:

Schade S. Molekulare und funktionale Charakterisierung des Galectin-3-bindenden Proteins LGALS3BP in Krebs und der Zentrosomenbiologie. [Internet] [Thesis]. Freie Universität Berlin; 2017. [cited 2020 Feb 19]. Available from: http://dx.doi.org/10.17169/refubium-6313.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schade S. Molekulare und funktionale Charakterisierung des Galectin-3-bindenden Proteins LGALS3BP in Krebs und der Zentrosomenbiologie. [Thesis]. Freie Universität Berlin; 2017. Available from: http://dx.doi.org/10.17169/refubium-6313

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

24. Spears, Erick. Tumor Suppressor APC and Musashi1: Double-Negative Feedback, Wnt Signaling and Colon Cancer.

Degree: PhD, Molecular Biosciences, 2011, University of Kansas

 Colorectal cancer is the second leading cause of cancer related deaths in the United States. Approximately 80% of all colon cancers are associated with a… (more)

Subjects/Keywords: Biology; Cellular biology; Molecular biology; Cancer

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APA (6th Edition):

Spears, E. (2011). Tumor Suppressor APC and Musashi1: Double-Negative Feedback, Wnt Signaling and Colon Cancer. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/8198

Chicago Manual of Style (16th Edition):

Spears, Erick. “Tumor Suppressor APC and Musashi1: Double-Negative Feedback, Wnt Signaling and Colon Cancer.” 2011. Doctoral Dissertation, University of Kansas. Accessed February 19, 2020. http://hdl.handle.net/1808/8198.

MLA Handbook (7th Edition):

Spears, Erick. “Tumor Suppressor APC and Musashi1: Double-Negative Feedback, Wnt Signaling and Colon Cancer.” 2011. Web. 19 Feb 2020.

Vancouver:

Spears E. Tumor Suppressor APC and Musashi1: Double-Negative Feedback, Wnt Signaling and Colon Cancer. [Internet] [Doctoral dissertation]. University of Kansas; 2011. [cited 2020 Feb 19]. Available from: http://hdl.handle.net/1808/8198.

Council of Science Editors:

Spears E. Tumor Suppressor APC and Musashi1: Double-Negative Feedback, Wnt Signaling and Colon Cancer. [Doctoral Dissertation]. University of Kansas; 2011. Available from: http://hdl.handle.net/1808/8198


University of Western Ontario

25. Evered, Caitlin L. Inhibition Of MT1-MMP Proteolytic Function And ERK1/2 Signalling Influences Breast Cancer Cell Migration And Invasion Through Changes In MMP-2 And MMP-9 Expression.

Degree: 2016, University of Western Ontario

 Membrane-type 1 matrix metalloproteinase (MT1-MMP) is a multifunctional protease that invokes changes extracellularly via cleavages of ECM substrates, and intracellularly through induction of cell signalling… (more)

Subjects/Keywords: Cancer Biology; Cell Biology; Developmental Biology

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APA (6th Edition):

Evered, C. L. (2016). Inhibition Of MT1-MMP Proteolytic Function And ERK1/2 Signalling Influences Breast Cancer Cell Migration And Invasion Through Changes In MMP-2 And MMP-9 Expression. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/3900

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Evered, Caitlin L. “Inhibition Of MT1-MMP Proteolytic Function And ERK1/2 Signalling Influences Breast Cancer Cell Migration And Invasion Through Changes In MMP-2 And MMP-9 Expression.” 2016. Thesis, University of Western Ontario. Accessed February 19, 2020. https://ir.lib.uwo.ca/etd/3900.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Evered, Caitlin L. “Inhibition Of MT1-MMP Proteolytic Function And ERK1/2 Signalling Influences Breast Cancer Cell Migration And Invasion Through Changes In MMP-2 And MMP-9 Expression.” 2016. Web. 19 Feb 2020.

Vancouver:

Evered CL. Inhibition Of MT1-MMP Proteolytic Function And ERK1/2 Signalling Influences Breast Cancer Cell Migration And Invasion Through Changes In MMP-2 And MMP-9 Expression. [Internet] [Thesis]. University of Western Ontario; 2016. [cited 2020 Feb 19]. Available from: https://ir.lib.uwo.ca/etd/3900.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Evered CL. Inhibition Of MT1-MMP Proteolytic Function And ERK1/2 Signalling Influences Breast Cancer Cell Migration And Invasion Through Changes In MMP-2 And MMP-9 Expression. [Thesis]. University of Western Ontario; 2016. Available from: https://ir.lib.uwo.ca/etd/3900

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Raux, Brigitt. Développement d'inhibiteurs d'interaction protéine-protéine ciblant les protéines à bromodomaines : implications en épigénétique et dans le développement de cancers : Development of protein-protein interaction inhibitors targeting bromodomain-containing proteins : implications in epigenetics and cancer development.

Degree: Docteur es, Biologie. Biochimie structurale, 2017, Aix Marseille Université

Les protéines à bromodomaines (BCPs) sont notamment impliquées dans la régulation de la transcription de gènes et la signalisation cellulaire. Leur dérégulation conduit au développement… (more)

Subjects/Keywords: Interactions protéine-Protéine; Inhibiteurs; Bromodomaines; Épigénétique; Cancer; Biologie structurale; : protein-Protein interaction; Inhibitors; Bromodomains; Epigenetics; Cancer; Structural biology

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APA (6th Edition):

Raux, B. (2017). Développement d'inhibiteurs d'interaction protéine-protéine ciblant les protéines à bromodomaines : implications en épigénétique et dans le développement de cancers : Development of protein-protein interaction inhibitors targeting bromodomain-containing proteins : implications in epigenetics and cancer development. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2017AIXM0521

Chicago Manual of Style (16th Edition):

Raux, Brigitt. “Développement d'inhibiteurs d'interaction protéine-protéine ciblant les protéines à bromodomaines : implications en épigénétique et dans le développement de cancers : Development of protein-protein interaction inhibitors targeting bromodomain-containing proteins : implications in epigenetics and cancer development.” 2017. Doctoral Dissertation, Aix Marseille Université. Accessed February 19, 2020. http://www.theses.fr/2017AIXM0521.

MLA Handbook (7th Edition):

Raux, Brigitt. “Développement d'inhibiteurs d'interaction protéine-protéine ciblant les protéines à bromodomaines : implications en épigénétique et dans le développement de cancers : Development of protein-protein interaction inhibitors targeting bromodomain-containing proteins : implications in epigenetics and cancer development.” 2017. Web. 19 Feb 2020.

Vancouver:

Raux B. Développement d'inhibiteurs d'interaction protéine-protéine ciblant les protéines à bromodomaines : implications en épigénétique et dans le développement de cancers : Development of protein-protein interaction inhibitors targeting bromodomain-containing proteins : implications in epigenetics and cancer development. [Internet] [Doctoral dissertation]. Aix Marseille Université 2017. [cited 2020 Feb 19]. Available from: http://www.theses.fr/2017AIXM0521.

Council of Science Editors:

Raux B. Développement d'inhibiteurs d'interaction protéine-protéine ciblant les protéines à bromodomaines : implications en épigénétique et dans le développement de cancers : Development of protein-protein interaction inhibitors targeting bromodomain-containing proteins : implications in epigenetics and cancer development. [Doctoral Dissertation]. Aix Marseille Université 2017. Available from: http://www.theses.fr/2017AIXM0521


Université Paris-Sud – Paris XI

27. Barjon, Clément. Caractérisation biochimique et fonctionnelle de nouveaux anticorps monoclonaux anti-galectine-9 en vue d'applications diagnostiques et thérapeutiques : Biochemical and functional charcterization of new monoclonal antibodies targeted against galectin-9 for diagnostic and therapeutic applications.

Degree: Docteur es, Cancérologie, Immunologie, Biologie cellulaire, Biochimie, 2013, Université Paris-Sud – Paris XI

La galectine-9 est une lectine animale principalement exprimée dans un contexte inflammatoire et possédant des propriétés à la fois pro-inflammatoires et immunosuppressives. Elle induit la… (more)

Subjects/Keywords: Galectine-9; Cancer; Immunologie; Biologie cellulaire; Anticorps monoclonaux; Galectin-9; Cancer; Immunology; Cellular biology; Monoclonal antibodies

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APA (6th Edition):

Barjon, C. (2013). Caractérisation biochimique et fonctionnelle de nouveaux anticorps monoclonaux anti-galectine-9 en vue d'applications diagnostiques et thérapeutiques : Biochemical and functional charcterization of new monoclonal antibodies targeted against galectin-9 for diagnostic and therapeutic applications. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T001

Chicago Manual of Style (16th Edition):

Barjon, Clément. “Caractérisation biochimique et fonctionnelle de nouveaux anticorps monoclonaux anti-galectine-9 en vue d'applications diagnostiques et thérapeutiques : Biochemical and functional charcterization of new monoclonal antibodies targeted against galectin-9 for diagnostic and therapeutic applications.” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed February 19, 2020. http://www.theses.fr/2013PA11T001.

MLA Handbook (7th Edition):

Barjon, Clément. “Caractérisation biochimique et fonctionnelle de nouveaux anticorps monoclonaux anti-galectine-9 en vue d'applications diagnostiques et thérapeutiques : Biochemical and functional charcterization of new monoclonal antibodies targeted against galectin-9 for diagnostic and therapeutic applications.” 2013. Web. 19 Feb 2020.

Vancouver:

Barjon C. Caractérisation biochimique et fonctionnelle de nouveaux anticorps monoclonaux anti-galectine-9 en vue d'applications diagnostiques et thérapeutiques : Biochemical and functional charcterization of new monoclonal antibodies targeted against galectin-9 for diagnostic and therapeutic applications. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2020 Feb 19]. Available from: http://www.theses.fr/2013PA11T001.

Council of Science Editors:

Barjon C. Caractérisation biochimique et fonctionnelle de nouveaux anticorps monoclonaux anti-galectine-9 en vue d'applications diagnostiques et thérapeutiques : Biochemical and functional charcterization of new monoclonal antibodies targeted against galectin-9 for diagnostic and therapeutic applications. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T001


Université Paris-Sud – Paris XI

28. Bacquin, Agathe. Régulation de l’hélicase FBH1 et conséquences sur le maintien de la stabilité génétique chez l’homme : Regulation of FBH1 helicase and consequences on human genome stability.

Degree: Docteur es, Biologie cellulaire, 2012, Université Paris-Sud – Paris XI

Bien que la recombinaison homologue (RH) soit requise, notamment, pour la réparation fidèle des cassures double brins et la prise en charge des fourches de… (more)

Subjects/Keywords: ADN-Réparation; Cancer; Biologie céllulaire; Thèses et écrits académiques; DNA Repair; Cancer; Cell biology; Thesis and academic report

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bacquin, A. (2012). Régulation de l’hélicase FBH1 et conséquences sur le maintien de la stabilité génétique chez l’homme : Regulation of FBH1 helicase and consequences on human genome stability. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2012PA11T060

Chicago Manual of Style (16th Edition):

Bacquin, Agathe. “Régulation de l’hélicase FBH1 et conséquences sur le maintien de la stabilité génétique chez l’homme : Regulation of FBH1 helicase and consequences on human genome stability.” 2012. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed February 19, 2020. http://www.theses.fr/2012PA11T060.

MLA Handbook (7th Edition):

Bacquin, Agathe. “Régulation de l’hélicase FBH1 et conséquences sur le maintien de la stabilité génétique chez l’homme : Regulation of FBH1 helicase and consequences on human genome stability.” 2012. Web. 19 Feb 2020.

Vancouver:

Bacquin A. Régulation de l’hélicase FBH1 et conséquences sur le maintien de la stabilité génétique chez l’homme : Regulation of FBH1 helicase and consequences on human genome stability. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2012. [cited 2020 Feb 19]. Available from: http://www.theses.fr/2012PA11T060.

Council of Science Editors:

Bacquin A. Régulation de l’hélicase FBH1 et conséquences sur le maintien de la stabilité génétique chez l’homme : Regulation of FBH1 helicase and consequences on human genome stability. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2012. Available from: http://www.theses.fr/2012PA11T060


Freie Universität Berlin

29. Hallung, Nicole. A molecular and cell biological analysis of potential breast cancer candidates via 3D cell culture.

Degree: 2016, Freie Universität Berlin

 Breast cancer is one of the most common cancers in women worldwide and accounted for 25 % of all new female cancers in 2012. The… (more)

Subjects/Keywords: breast cancer; 3D cell culture; apoptosis; TNFRSF6B; DAPL1; molecular biology; cell biology; 500 Naturwissenschaften und Mathematik::570 Biowissenschaften; Biologie::570 Biowissenschaften; Biologie

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hallung, N. (2016). A molecular and cell biological analysis of potential breast cancer candidates via 3D cell culture. (Thesis). Freie Universität Berlin. Retrieved from http://dx.doi.org/10.17169/refubium-12962

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hallung, Nicole. “A molecular and cell biological analysis of potential breast cancer candidates via 3D cell culture.” 2016. Thesis, Freie Universität Berlin. Accessed February 19, 2020. http://dx.doi.org/10.17169/refubium-12962.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hallung, Nicole. “A molecular and cell biological analysis of potential breast cancer candidates via 3D cell culture.” 2016. Web. 19 Feb 2020.

Vancouver:

Hallung N. A molecular and cell biological analysis of potential breast cancer candidates via 3D cell culture. [Internet] [Thesis]. Freie Universität Berlin; 2016. [cited 2020 Feb 19]. Available from: http://dx.doi.org/10.17169/refubium-12962.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hallung N. A molecular and cell biological analysis of potential breast cancer candidates via 3D cell culture. [Thesis]. Freie Universität Berlin; 2016. Available from: http://dx.doi.org/10.17169/refubium-12962

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

30. Yang, Jun. DEVELOPMENTAL DEREGULATION AND TUMORIGENESIS INHIBITION IN 14-3-3ZETA KNOCKOUT MOUSE.

Degree: PhD, 2011, Texas Medical Center

Cancer is second leading cause of death in the United States. Improving cancer care through patient care, research, education and prevention not only saves… (more)

Subjects/Keywords: 14-3-3zeta; development; cancer; Cancer Biology; Developmental Biology; Molecular Biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, J. (2011). DEVELOPMENTAL DEREGULATION AND TUMORIGENESIS INHIBITION IN 14-3-3ZETA KNOCKOUT MOUSE. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/175

Chicago Manual of Style (16th Edition):

Yang, Jun. “DEVELOPMENTAL DEREGULATION AND TUMORIGENESIS INHIBITION IN 14-3-3ZETA KNOCKOUT MOUSE.” 2011. Doctoral Dissertation, Texas Medical Center. Accessed February 19, 2020. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/175.

MLA Handbook (7th Edition):

Yang, Jun. “DEVELOPMENTAL DEREGULATION AND TUMORIGENESIS INHIBITION IN 14-3-3ZETA KNOCKOUT MOUSE.” 2011. Web. 19 Feb 2020.

Vancouver:

Yang J. DEVELOPMENTAL DEREGULATION AND TUMORIGENESIS INHIBITION IN 14-3-3ZETA KNOCKOUT MOUSE. [Internet] [Doctoral dissertation]. Texas Medical Center; 2011. [cited 2020 Feb 19]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/175.

Council of Science Editors:

Yang J. DEVELOPMENTAL DEREGULATION AND TUMORIGENESIS INHIBITION IN 14-3-3ZETA KNOCKOUT MOUSE. [Doctoral Dissertation]. Texas Medical Center; 2011. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/175

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