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You searched for subject:(Cancer Molecular aspects). Showing records 1 – 30 of 126 total matches.

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University of Hong Kong

1. Tey, Sze Keong. Clinical significance and functional role of nuclear met in hepatocellular carcinoma.

Degree: PhD, 2015, University of Hong Kong

 Hepatocellular carcinoma (HCC) is the fifth most prevalent cancer and the third most common cause of cancer mortality worldwide. Understanding the underlying mechanisms that contributed… (more)

Subjects/Keywords: Liver - Cancer - Molecular aspects

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APA (6th Edition):

Tey, S. K. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140

Chicago Manual of Style (16th Edition):

Tey, Sze Keong. “Clinical significance and functional role of nuclear met in hepatocellular carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140.

MLA Handbook (7th Edition):

Tey, Sze Keong. “Clinical significance and functional role of nuclear met in hepatocellular carcinoma.” 2015. Web. 18 Aug 2019.

Vancouver:

Tey SK. Clinical significance and functional role of nuclear met in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Aug 18]. Available from: Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140.

Council of Science Editors:

Tey SK. Clinical significance and functional role of nuclear met in hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: Tey, S. K. [鄭思強]. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5699886 ; http://dx.doi.org/10.5353/th_b5699886 ; http://hdl.handle.net/10722/231140


University of Hong Kong

2. Chan, Yan-yan. The role of cyclin E1 in hepatocellular carcinoma.

Degree: M. Phil., 2014, University of Hong Kong

Hepatocellular carcinoma (HCC) accounts for 70-85% of liver cancer, which is the sixth most common cancer in the world. Prognosis of HCC is dismal with… (more)

Subjects/Keywords: Cyclins; Liver - Cancer - Molecular aspects

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APA (6th Edition):

Chan, Y. (2014). The role of cyclin E1 in hepatocellular carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Chan, Y. [陳茵茵]. (2014). The role of cyclin E1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5351033 ; http://dx.doi.org/10.5353/th_b5351033 ; http://hdl.handle.net/10722/208041

Chicago Manual of Style (16th Edition):

Chan, Yan-yan. “The role of cyclin E1 in hepatocellular carcinoma.” 2014. Masters Thesis, University of Hong Kong. Accessed August 18, 2019. Chan, Y. [陳茵茵]. (2014). The role of cyclin E1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5351033 ; http://dx.doi.org/10.5353/th_b5351033 ; http://hdl.handle.net/10722/208041.

MLA Handbook (7th Edition):

Chan, Yan-yan. “The role of cyclin E1 in hepatocellular carcinoma.” 2014. Web. 18 Aug 2019.

Vancouver:

Chan Y. The role of cyclin E1 in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2014. [cited 2019 Aug 18]. Available from: Chan, Y. [陳茵茵]. (2014). The role of cyclin E1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5351033 ; http://dx.doi.org/10.5353/th_b5351033 ; http://hdl.handle.net/10722/208041.

Council of Science Editors:

Chan Y. The role of cyclin E1 in hepatocellular carcinoma. [Masters Thesis]. University of Hong Kong; 2014. Available from: Chan, Y. [陳茵茵]. (2014). The role of cyclin E1 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5351033 ; http://dx.doi.org/10.5353/th_b5351033 ; http://hdl.handle.net/10722/208041


University of Hong Kong

3. Hung, Wing-yan. The role of TAX1BP2 in hepatocellular carcinoma.

Degree: M. Phil., 2012, University of Hong Kong

 TAX1 Binding Protein 2 (TAX1BP2) has been found to be a centrosome duplication regulating protein. Previous findings have demonstrated that over-expression of TAX1BP2 suppresses centrosome… (more)

Subjects/Keywords: Tumor suppressor proteins; Liver - Cancer - Molecular aspects

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APA (6th Edition):

Hung, W. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070

Chicago Manual of Style (16th Edition):

Hung, Wing-yan. “The role of TAX1BP2 in hepatocellular carcinoma.” 2012. Masters Thesis, University of Hong Kong. Accessed August 18, 2019. Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070.

MLA Handbook (7th Edition):

Hung, Wing-yan. “The role of TAX1BP2 in hepatocellular carcinoma.” 2012. Web. 18 Aug 2019.

Vancouver:

Hung W. The role of TAX1BP2 in hepatocellular carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2012. [cited 2019 Aug 18]. Available from: Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070.

Council of Science Editors:

Hung W. The role of TAX1BP2 in hepatocellular carcinoma. [Masters Thesis]. University of Hong Kong; 2012. Available from: Hung, W. [洪穎欣]. (2012). The role of TAX1BP2 in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4852155 ; http://dx.doi.org/10.5353/th_b4852155 ; http://hdl.handle.net/10722/193070


University of North Carolina – Greensboro

4. Vaidya, Himani. Role of DNA methylation in WNT5A Promoter B expression in Osteosarcoma (SaOS-2) cells.

Degree: 2013, University of North Carolina – Greensboro

 WNT5A, a member of the WNT family of secreted proteins, activates the non-canonical WNT pathway, regulates developmental events and is involved in tissue homeostasis. Misregulation… (more)

Subjects/Keywords: Wnt proteins; DNA – Methylation; Cancer – Molecular aspects

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APA (6th Edition):

Vaidya, H. (2013). Role of DNA methylation in WNT5A Promoter B expression in Osteosarcoma (SaOS-2) cells. (Masters Thesis). University of North Carolina – Greensboro. Retrieved from http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=15663

Chicago Manual of Style (16th Edition):

Vaidya, Himani. “Role of DNA methylation in WNT5A Promoter B expression in Osteosarcoma (SaOS-2) cells.” 2013. Masters Thesis, University of North Carolina – Greensboro. Accessed August 18, 2019. http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=15663.

MLA Handbook (7th Edition):

Vaidya, Himani. “Role of DNA methylation in WNT5A Promoter B expression in Osteosarcoma (SaOS-2) cells.” 2013. Web. 18 Aug 2019.

Vancouver:

Vaidya H. Role of DNA methylation in WNT5A Promoter B expression in Osteosarcoma (SaOS-2) cells. [Internet] [Masters thesis]. University of North Carolina – Greensboro; 2013. [cited 2019 Aug 18]. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=15663.

Council of Science Editors:

Vaidya H. Role of DNA methylation in WNT5A Promoter B expression in Osteosarcoma (SaOS-2) cells. [Masters Thesis]. University of North Carolina – Greensboro; 2013. Available from: http://libres.uncg.edu/ir/listing.aspx?styp=ti&id=15663


University of Hong Kong

5. Wong, Kit-man, Sunny. Isolation and characterization of cancer stem cells in non-small cell lung cancer.

Degree: M. Phil., 2011, University of Hong Kong

 Tumor heterogeneity has long been observed and recognized as a challenge to cancer therapy. The cancer stem cell (CSC) model is one of the hypotheses… (more)

Subjects/Keywords: Cancer cells.; Stem cells.; Lungs - Cancer - Molecular aspects.

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APA (6th Edition):

Wong, Kit-man, S. (2011). Isolation and characterization of cancer stem cells in non-small cell lung cancer. (Masters Thesis). University of Hong Kong. Retrieved from Wong, K. S. [王傑民]. (2011). Isolation and characterization of cancer stem cells in non-small cell lung cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4725066 ; http://dx.doi.org/10.5353/th_b4725066 ; http://hdl.handle.net/10722/180008

Chicago Manual of Style (16th Edition):

Wong, Kit-man, Sunny. “Isolation and characterization of cancer stem cells in non-small cell lung cancer.” 2011. Masters Thesis, University of Hong Kong. Accessed August 18, 2019. Wong, K. S. [王傑民]. (2011). Isolation and characterization of cancer stem cells in non-small cell lung cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4725066 ; http://dx.doi.org/10.5353/th_b4725066 ; http://hdl.handle.net/10722/180008.

MLA Handbook (7th Edition):

Wong, Kit-man, Sunny. “Isolation and characterization of cancer stem cells in non-small cell lung cancer.” 2011. Web. 18 Aug 2019.

Vancouver:

Wong, Kit-man S. Isolation and characterization of cancer stem cells in non-small cell lung cancer. [Internet] [Masters thesis]. University of Hong Kong; 2011. [cited 2019 Aug 18]. Available from: Wong, K. S. [王傑民]. (2011). Isolation and characterization of cancer stem cells in non-small cell lung cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4725066 ; http://dx.doi.org/10.5353/th_b4725066 ; http://hdl.handle.net/10722/180008.

Council of Science Editors:

Wong, Kit-man S. Isolation and characterization of cancer stem cells in non-small cell lung cancer. [Masters Thesis]. University of Hong Kong; 2011. Available from: Wong, K. S. [王傑民]. (2011). Isolation and characterization of cancer stem cells in non-small cell lung cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4725066 ; http://dx.doi.org/10.5353/th_b4725066 ; http://hdl.handle.net/10722/180008


Columbia University

6. Broyde, Joshua. Analysis of Oncogenic Signal Transduction with Application to KRAS Signaling Pathways.

Degree: 2018, Columbia University

 The discovery of novel members of tumorigenic pathways remains a critical step to fully dissect the molecular biology of cancer. Indeed, because a number of… (more)

Subjects/Keywords: Bioinformatics; Biometry; Molecular biology; Cancer – Molecular aspects; Cellular signal transduction; Oncology

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APA (6th Edition):

Broyde, J. (2018). Analysis of Oncogenic Signal Transduction with Application to KRAS Signaling Pathways. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8NZ8M76

Chicago Manual of Style (16th Edition):

Broyde, Joshua. “Analysis of Oncogenic Signal Transduction with Application to KRAS Signaling Pathways.” 2018. Doctoral Dissertation, Columbia University. Accessed August 18, 2019. https://doi.org/10.7916/D8NZ8M76.

MLA Handbook (7th Edition):

Broyde, Joshua. “Analysis of Oncogenic Signal Transduction with Application to KRAS Signaling Pathways.” 2018. Web. 18 Aug 2019.

Vancouver:

Broyde J. Analysis of Oncogenic Signal Transduction with Application to KRAS Signaling Pathways. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2019 Aug 18]. Available from: https://doi.org/10.7916/D8NZ8M76.

Council of Science Editors:

Broyde J. Analysis of Oncogenic Signal Transduction with Application to KRAS Signaling Pathways. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8NZ8M76


Florida Atlantic University

7. Liddle, Genevieve M. A Study on Reversing the Immunosuppressive Phenotype of Tumor Associated Macrophages.

Degree: MS, 2017, Florida Atlantic University

Summary: Extracellular stimuli may influence the M1/M2 phenotypic polarization of macrophages. We examined M1/M2 biomarkers, phagocytic activity, and tumoricidal activity in RAW 264.7 mouse macrophages.… (more)

Subjects/Keywords: Macrophages.; Breast – Cancer – Treatment.; Tumors – Immunological aspects.; Cancer – Immunological aspects.; Biological response modifiers.; Cancer – Molecular aspects.

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APA (6th Edition):

Liddle, G. M. (2017). A Study on Reversing the Immunosuppressive Phenotype of Tumor Associated Macrophages. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004867

Chicago Manual of Style (16th Edition):

Liddle, Genevieve M. “A Study on Reversing the Immunosuppressive Phenotype of Tumor Associated Macrophages.” 2017. Masters Thesis, Florida Atlantic University. Accessed August 18, 2019. http://purl.flvc.org/fau/fd/FA00004867.

MLA Handbook (7th Edition):

Liddle, Genevieve M. “A Study on Reversing the Immunosuppressive Phenotype of Tumor Associated Macrophages.” 2017. Web. 18 Aug 2019.

Vancouver:

Liddle GM. A Study on Reversing the Immunosuppressive Phenotype of Tumor Associated Macrophages. [Internet] [Masters thesis]. Florida Atlantic University; 2017. [cited 2019 Aug 18]. Available from: http://purl.flvc.org/fau/fd/FA00004867.

Council of Science Editors:

Liddle GM. A Study on Reversing the Immunosuppressive Phenotype of Tumor Associated Macrophages. [Masters Thesis]. Florida Atlantic University; 2017. Available from: http://purl.flvc.org/fau/fd/FA00004867


Florida Atlantic University

8. Onwuha-Ekpete, Lillian C. Inflammatory response in stress and the role of autophagy in breast cancer.

Degree: MS, 2012, Florida Atlantic University

Summary: We attempted to understand the molecular regulators that impact inflammation using a rat model of human sensation-seeking/risk-taking trait for drug and stress vulnerability, based… (more)

Subjects/Keywords: Breast – Cancer – Genetic aspects; Cancer – Molecular aspects; Carcinogenesis; Cellular signal transduction; Stress (Physiology)

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APA (6th Edition):

Onwuha-Ekpete, L. C. (2012). Inflammatory response in stress and the role of autophagy in breast cancer. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fcla/dt/3362042

Chicago Manual of Style (16th Edition):

Onwuha-Ekpete, Lillian C. “Inflammatory response in stress and the role of autophagy in breast cancer.” 2012. Masters Thesis, Florida Atlantic University. Accessed August 18, 2019. http://purl.flvc.org/fcla/dt/3362042.

MLA Handbook (7th Edition):

Onwuha-Ekpete, Lillian C. “Inflammatory response in stress and the role of autophagy in breast cancer.” 2012. Web. 18 Aug 2019.

Vancouver:

Onwuha-Ekpete LC. Inflammatory response in stress and the role of autophagy in breast cancer. [Internet] [Masters thesis]. Florida Atlantic University; 2012. [cited 2019 Aug 18]. Available from: http://purl.flvc.org/fcla/dt/3362042.

Council of Science Editors:

Onwuha-Ekpete LC. Inflammatory response in stress and the role of autophagy in breast cancer. [Masters Thesis]. Florida Atlantic University; 2012. Available from: http://purl.flvc.org/fcla/dt/3362042


Michigan State University

9. Gary, Joy. Phenotypic characterization of allelic variants of the mechanistic target of rapamycin (mTOR.

Degree: 2015, Michigan State University

Thesis Ph. D. Michigan State University. Pathobiology 2015

ABSTRACTPHENOTYPIC CHARACTERIZATION OF ALLELIC VARIANTS OF THE MECHANISTIC TARGET OF RAPAMYCIN (mTOR)ByJoy GarymTOR is a serine/threonine kinase… (more)

Subjects/Keywords: Protein kinases; Tumors – Genetic aspects; Cancer – Molecular aspects; Molecular biology; Genetics; Pathology

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APA (6th Edition):

Gary, J. (2015). Phenotypic characterization of allelic variants of the mechanistic target of rapamycin (mTOR. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:2887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gary, Joy. “Phenotypic characterization of allelic variants of the mechanistic target of rapamycin (mTOR.” 2015. Thesis, Michigan State University. Accessed August 18, 2019. http://etd.lib.msu.edu/islandora/object/etd:2887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gary, Joy. “Phenotypic characterization of allelic variants of the mechanistic target of rapamycin (mTOR.” 2015. Web. 18 Aug 2019.

Vancouver:

Gary J. Phenotypic characterization of allelic variants of the mechanistic target of rapamycin (mTOR. [Internet] [Thesis]. Michigan State University; 2015. [cited 2019 Aug 18]. Available from: http://etd.lib.msu.edu/islandora/object/etd:2887.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gary J. Phenotypic characterization of allelic variants of the mechanistic target of rapamycin (mTOR. [Thesis]. Michigan State University; 2015. Available from: http://etd.lib.msu.edu/islandora/object/etd:2887

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Durban University of Technology

10. Cele, Nosipho Magnificat. Computational studies on the identification and analyses of p53 cancer associated mutations.

Degree: 2017, Durban University of Technology

Submitted in the fulfillment of the requirement for the Degree of Master's in Chemistry, Durban University of Technology, 2017.

P53 is a tumour suppressor protein… (more)

Subjects/Keywords: p53 antioncogene; p53 protein; Tumor suppressor proteins; Cancer – Computer simulation; Mutation (Biology) – Computer simulation; Cancer – Genetic aspects; Cancer – Molecular aspects

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APA (6th Edition):

Cele, N. M. (2017). Computational studies on the identification and analyses of p53 cancer associated mutations. (Thesis). Durban University of Technology. Retrieved from http://hdl.handle.net/10321/2617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cele, Nosipho Magnificat. “Computational studies on the identification and analyses of p53 cancer associated mutations.” 2017. Thesis, Durban University of Technology. Accessed August 18, 2019. http://hdl.handle.net/10321/2617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cele, Nosipho Magnificat. “Computational studies on the identification and analyses of p53 cancer associated mutations.” 2017. Web. 18 Aug 2019.

Vancouver:

Cele NM. Computational studies on the identification and analyses of p53 cancer associated mutations. [Internet] [Thesis]. Durban University of Technology; 2017. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10321/2617.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cele NM. Computational studies on the identification and analyses of p53 cancer associated mutations. [Thesis]. Durban University of Technology; 2017. Available from: http://hdl.handle.net/10321/2617

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Lethbridge

11. University of Lethbridge. Faculty of Arts and Science. Analysis of colon cancer cells that survive checkpoint adaptation after treatment with a genotoxic age .

Degree: 2013, University of Lethbridge

 Most cancer treatments are genotoxic agents that target and damage DNA as part of their mechanism of action. Recently, it was discovered that cancer cells… (more)

Subjects/Keywords: Colon (Anatomy)  – Cancer; Colon (Anatomy)  – Cancer  – Cytopathology; Colon (Anatomy)  – Cancer  – Chemotherapy; Colon (Anatomy)  – Cancer  – Molecular aspects; Dissertations, Academic

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APA (6th Edition):

Science, U. o. L. F. o. A. a. (2013). Analysis of colon cancer cells that survive checkpoint adaptation after treatment with a genotoxic age . (Thesis). University of Lethbridge. Retrieved from http://hdl.handle.net/10133/3565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Science, University of Lethbridge. Faculty of Arts and. “Analysis of colon cancer cells that survive checkpoint adaptation after treatment with a genotoxic age .” 2013. Thesis, University of Lethbridge. Accessed August 18, 2019. http://hdl.handle.net/10133/3565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Science, University of Lethbridge. Faculty of Arts and. “Analysis of colon cancer cells that survive checkpoint adaptation after treatment with a genotoxic age .” 2013. Web. 18 Aug 2019.

Vancouver:

Science UoLFoAa. Analysis of colon cancer cells that survive checkpoint adaptation after treatment with a genotoxic age . [Internet] [Thesis]. University of Lethbridge; 2013. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10133/3565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Science UoLFoAa. Analysis of colon cancer cells that survive checkpoint adaptation after treatment with a genotoxic age . [Thesis]. University of Lethbridge; 2013. Available from: http://hdl.handle.net/10133/3565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of KwaZulu-Natal

12. [No author]. Gene expression analysis of squamous cell carcinoma of the oesophagus using a novel real time PCR probe system .

Degree: Medical microbiology, 2010, University of KwaZulu-Natal

 Squamous cell carcinoma of the oesophagus (OSCC) is a common malignancy that occurs with high frequency in certain parts of the world, including South Africa.… (more)

Subjects/Keywords: Cancer – Molecular aspects.; Squamous cell carcinoma.; Oesophagus – Cancer.; Gene expression.; Medical microbiology.

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APA (6th Edition):

author], [. (2010). Gene expression analysis of squamous cell carcinoma of the oesophagus using a novel real time PCR probe system . (Thesis). University of KwaZulu-Natal. Retrieved from http://hdl.handle.net/10413/706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Gene expression analysis of squamous cell carcinoma of the oesophagus using a novel real time PCR probe system .” 2010. Thesis, University of KwaZulu-Natal. Accessed August 18, 2019. http://hdl.handle.net/10413/706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Gene expression analysis of squamous cell carcinoma of the oesophagus using a novel real time PCR probe system .” 2010. Web. 18 Aug 2019.

Vancouver:

author] [. Gene expression analysis of squamous cell carcinoma of the oesophagus using a novel real time PCR probe system . [Internet] [Thesis]. University of KwaZulu-Natal; 2010. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10413/706.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Gene expression analysis of squamous cell carcinoma of the oesophagus using a novel real time PCR probe system . [Thesis]. University of KwaZulu-Natal; 2010. Available from: http://hdl.handle.net/10413/706

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

13. 黃澤蕾.; Wong, Chak-lui, Carmen. Regulations and functions of rho-kinases in hepatocellular carcinoma.

Degree: PhD, 2009, University of Hong Kong

The Best PhD Thesis in the Faculties of Dentistry, Engineering, Medicine and Science (University of Hong Kong), Li Ka Shing Prize,2008-2009

published_or_final_version

Pathology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Ng, IOL.

Subjects/Keywords: Cancer cells.; Metastasis.; Liver - Cancer - Molecular aspects.; Serine proteinases.; Protein kinases.; Tumor suppressor proteins.

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APA (6th Edition):

黃澤蕾.; Wong, Chak-lui, C. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132

Chicago Manual of Style (16th Edition):

黃澤蕾.; Wong, Chak-lui, Carmen. “Regulations and functions of rho-kinases in hepatocellular carcinoma.” 2009. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132.

MLA Handbook (7th Edition):

黃澤蕾.; Wong, Chak-lui, Carmen. “Regulations and functions of rho-kinases in hepatocellular carcinoma.” 2009. Web. 18 Aug 2019.

Vancouver:

黃澤蕾.; Wong, Chak-lui C. Regulations and functions of rho-kinases in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2009. [cited 2019 Aug 18]. Available from: Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132.

Council of Science Editors:

黃澤蕾.; Wong, Chak-lui C. Regulations and functions of rho-kinases in hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2009. Available from: Wong, C. C. [黃澤蕾]. (2009). Regulations and functions of rho-kinases in hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4218200 ; http://dx.doi.org/10.5353/th_b4218200 ; http://hdl.handle.net/10722/61132


Ryerson University

14. Premnath, Priyatha. Bio-functionalization of silicon and its applications in mammalian and cancer cell manipulation and proliferation.

Degree: 2015, Ryerson University

 Currently fabricated bio-matrices lack important characteristics such as nanometer scale, ‘bumpy’ morphology and an interlinked structure. Therefore, cells cultured on such matrices may not truly… (more)

Subjects/Keywords: Cancer cells  – Proliferation; Cell proliferation; Cancer  – Molecular aspects; Cell culture; Mammals  – Cytology; Pathology, Cellular

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APA (6th Edition):

Premnath, P. (2015). Bio-functionalization of silicon and its applications in mammalian and cancer cell manipulation and proliferation. (Thesis). Ryerson University. Retrieved from https://digital.library.ryerson.ca/islandora/object/RULA%3A3749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Premnath, Priyatha. “Bio-functionalization of silicon and its applications in mammalian and cancer cell manipulation and proliferation.” 2015. Thesis, Ryerson University. Accessed August 18, 2019. https://digital.library.ryerson.ca/islandora/object/RULA%3A3749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Premnath, Priyatha. “Bio-functionalization of silicon and its applications in mammalian and cancer cell manipulation and proliferation.” 2015. Web. 18 Aug 2019.

Vancouver:

Premnath P. Bio-functionalization of silicon and its applications in mammalian and cancer cell manipulation and proliferation. [Internet] [Thesis]. Ryerson University; 2015. [cited 2019 Aug 18]. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3749.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Premnath P. Bio-functionalization of silicon and its applications in mammalian and cancer cell manipulation and proliferation. [Thesis]. Ryerson University; 2015. Available from: https://digital.library.ryerson.ca/islandora/object/RULA%3A3749

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

15. 凌明達; Ling, Ming-tat, Patrick. A study of molecular and cell biology of prostate tumorigenesis in cell culture.

Degree: M. Phil., 2000, University of Hong Kong

published_or_final_version

Pathology

Master

Master of Philosophy

Subjects/Keywords: Prostate - Cancer - Molecular aspects.

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APA (6th Edition):

凌明達; Ling, Ming-tat, P. (2000). A study of molecular and cell biology of prostate tumorigenesis in cell culture. (Masters Thesis). University of Hong Kong. Retrieved from Ling, M. P. [凌明達]. (2000). A study of molecular and cell biology of prostate tumorigenesis in cell culture. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122310 ; http://dx.doi.org/10.5353/th_b3122310 ; http://hdl.handle.net/10722/33483

Chicago Manual of Style (16th Edition):

凌明達; Ling, Ming-tat, Patrick. “A study of molecular and cell biology of prostate tumorigenesis in cell culture.” 2000. Masters Thesis, University of Hong Kong. Accessed August 18, 2019. Ling, M. P. [凌明達]. (2000). A study of molecular and cell biology of prostate tumorigenesis in cell culture. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122310 ; http://dx.doi.org/10.5353/th_b3122310 ; http://hdl.handle.net/10722/33483.

MLA Handbook (7th Edition):

凌明達; Ling, Ming-tat, Patrick. “A study of molecular and cell biology of prostate tumorigenesis in cell culture.” 2000. Web. 18 Aug 2019.

Vancouver:

凌明達; Ling, Ming-tat P. A study of molecular and cell biology of prostate tumorigenesis in cell culture. [Internet] [Masters thesis]. University of Hong Kong; 2000. [cited 2019 Aug 18]. Available from: Ling, M. P. [凌明達]. (2000). A study of molecular and cell biology of prostate tumorigenesis in cell culture. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122310 ; http://dx.doi.org/10.5353/th_b3122310 ; http://hdl.handle.net/10722/33483.

Council of Science Editors:

凌明達; Ling, Ming-tat P. A study of molecular and cell biology of prostate tumorigenesis in cell culture. [Masters Thesis]. University of Hong Kong; 2000. Available from: Ling, M. P. [凌明達]. (2000). A study of molecular and cell biology of prostate tumorigenesis in cell culture. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b3122310 ; http://dx.doi.org/10.5353/th_b3122310 ; http://hdl.handle.net/10722/33483


Hong Kong University of Science and Technology

16. Sun, Yisuo. Diagnostics of human diseases by the one label extension method.

Degree: 2014, Hong Kong University of Science and Technology

 Genetic testing can provide important clinical benefits. Genetic testing has been done for diverse purposes, including the diagnostic testing; presymptomatic and predictive testing and drug-resistance… (more)

Subjects/Keywords: Human chromosome abnormalities; Diagnosis; Molecular diagnosis; Cancer; Genetic aspects; Testing

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APA (6th Edition):

Sun, Y. (2014). Diagnostics of human diseases by the one label extension method. (Thesis). Hong Kong University of Science and Technology. Retrieved from https://doi.org/10.14711/thesis-b1274546 ; http://repository.ust.hk/ir/bitstream/1783.1-76059/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sun, Yisuo. “Diagnostics of human diseases by the one label extension method.” 2014. Thesis, Hong Kong University of Science and Technology. Accessed August 18, 2019. https://doi.org/10.14711/thesis-b1274546 ; http://repository.ust.hk/ir/bitstream/1783.1-76059/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sun, Yisuo. “Diagnostics of human diseases by the one label extension method.” 2014. Web. 18 Aug 2019.

Vancouver:

Sun Y. Diagnostics of human diseases by the one label extension method. [Internet] [Thesis]. Hong Kong University of Science and Technology; 2014. [cited 2019 Aug 18]. Available from: https://doi.org/10.14711/thesis-b1274546 ; http://repository.ust.hk/ir/bitstream/1783.1-76059/1/th_redirect.html.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sun Y. Diagnostics of human diseases by the one label extension method. [Thesis]. Hong Kong University of Science and Technology; 2014. Available from: https://doi.org/10.14711/thesis-b1274546 ; http://repository.ust.hk/ir/bitstream/1783.1-76059/1/th_redirect.html

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Hong Kong

17. 林敬賢.; Lam, King-yin, Alfred. Pathology and molecular biology of malignant thyroid tumours.

Degree: Doctor of Medicine, 2004, University of Hong Kong

published_or_final_version

toc

abstract

Medicine

Master

Doctor of Medicine

Subjects/Keywords: Thyroid gland - Cancer - Molecular aspects.

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APA (6th Edition):

林敬賢.; Lam, King-yin, A. (2004). Pathology and molecular biology of malignant thyroid tumours. (Doctoral Dissertation). University of Hong Kong. Retrieved from Lam, K. A. [林敬賢]. (2004). Pathology and molecular biology of malignant thyroid tumours. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2986782 ; http://dx.doi.org/10.5353/th_b2986782 ; http://hdl.handle.net/10722/31886

Chicago Manual of Style (16th Edition):

林敬賢.; Lam, King-yin, Alfred. “Pathology and molecular biology of malignant thyroid tumours.” 2004. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Lam, K. A. [林敬賢]. (2004). Pathology and molecular biology of malignant thyroid tumours. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2986782 ; http://dx.doi.org/10.5353/th_b2986782 ; http://hdl.handle.net/10722/31886.

MLA Handbook (7th Edition):

林敬賢.; Lam, King-yin, Alfred. “Pathology and molecular biology of malignant thyroid tumours.” 2004. Web. 18 Aug 2019.

Vancouver:

林敬賢.; Lam, King-yin A. Pathology and molecular biology of malignant thyroid tumours. [Internet] [Doctoral dissertation]. University of Hong Kong; 2004. [cited 2019 Aug 18]. Available from: Lam, K. A. [林敬賢]. (2004). Pathology and molecular biology of malignant thyroid tumours. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2986782 ; http://dx.doi.org/10.5353/th_b2986782 ; http://hdl.handle.net/10722/31886.

Council of Science Editors:

林敬賢.; Lam, King-yin A. Pathology and molecular biology of malignant thyroid tumours. [Doctoral Dissertation]. University of Hong Kong; 2004. Available from: Lam, K. A. [林敬賢]. (2004). Pathology and molecular biology of malignant thyroid tumours. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b2986782 ; http://dx.doi.org/10.5353/th_b2986782 ; http://hdl.handle.net/10722/31886


University of Hong Kong

18. 紀思思.; Kee, Francis. Molecular pathology of hepatocellular carcinoma.

Degree: PhD, 2008, University of Hong Kong

published_or_final_version

Medicine

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Lai, CL.

Subjects/Keywords: Liver - Cancer - Molecular aspects.

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APA (6th Edition):

紀思思.; Kee, F. (2008). Molecular pathology of hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Kee, F. [紀思思]. (2008). Molecular pathology of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4020378 ; http://dx.doi.org/10.5353/th_b4020378 ; http://hdl.handle.net/10722/54654

Chicago Manual of Style (16th Edition):

紀思思.; Kee, Francis. “Molecular pathology of hepatocellular carcinoma.” 2008. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Kee, F. [紀思思]. (2008). Molecular pathology of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4020378 ; http://dx.doi.org/10.5353/th_b4020378 ; http://hdl.handle.net/10722/54654.

MLA Handbook (7th Edition):

紀思思.; Kee, Francis. “Molecular pathology of hepatocellular carcinoma.” 2008. Web. 18 Aug 2019.

Vancouver:

紀思思.; Kee F. Molecular pathology of hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2008. [cited 2019 Aug 18]. Available from: Kee, F. [紀思思]. (2008). Molecular pathology of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4020378 ; http://dx.doi.org/10.5353/th_b4020378 ; http://hdl.handle.net/10722/54654.

Council of Science Editors:

紀思思.; Kee F. Molecular pathology of hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2008. Available from: Kee, F. [紀思思]. (2008). Molecular pathology of hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4020378 ; http://dx.doi.org/10.5353/th_b4020378 ; http://hdl.handle.net/10722/54654


University of Hong Kong

19. Ko, Chi-fat. Molecular regulations of deleted in liver cancer (DLC) protein family.

Degree: PhD, 2009, University of Hong Kong

published_or_final_version

Pathology

Doctoral

Doctor of Philosophy

Advisors/Committee Members: Ng, IOL, Fan, ST, Yam, JWP.

Subjects/Keywords: Antioncogenes.; Liver - Cancer - Molecular aspects.

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APA (6th Edition):

Ko, C. (2009). Molecular regulations of deleted in liver cancer (DLC) protein family. (Doctoral Dissertation). University of Hong Kong. Retrieved from Ko, C. [高自發]. (2009). Molecular regulations of deleted in liver cancer (DLC) protein family. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4189688 ; http://dx.doi.org/10.5353/th_b4189688 ; http://hdl.handle.net/10722/57037

Chicago Manual of Style (16th Edition):

Ko, Chi-fat. “Molecular regulations of deleted in liver cancer (DLC) protein family.” 2009. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Ko, C. [高自發]. (2009). Molecular regulations of deleted in liver cancer (DLC) protein family. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4189688 ; http://dx.doi.org/10.5353/th_b4189688 ; http://hdl.handle.net/10722/57037.

MLA Handbook (7th Edition):

Ko, Chi-fat. “Molecular regulations of deleted in liver cancer (DLC) protein family.” 2009. Web. 18 Aug 2019.

Vancouver:

Ko C. Molecular regulations of deleted in liver cancer (DLC) protein family. [Internet] [Doctoral dissertation]. University of Hong Kong; 2009. [cited 2019 Aug 18]. Available from: Ko, C. [高自發]. (2009). Molecular regulations of deleted in liver cancer (DLC) protein family. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4189688 ; http://dx.doi.org/10.5353/th_b4189688 ; http://hdl.handle.net/10722/57037.

Council of Science Editors:

Ko C. Molecular regulations of deleted in liver cancer (DLC) protein family. [Doctoral Dissertation]. University of Hong Kong; 2009. Available from: Ko, C. [高自發]. (2009). Molecular regulations of deleted in liver cancer (DLC) protein family. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4189688 ; http://dx.doi.org/10.5353/th_b4189688 ; http://hdl.handle.net/10722/57037


University of Hong Kong

20. Chu, Ka-wan, Kevin. High-throughput molecular characterization of human non-small cell lung carcinoma.

Degree: Master of Research in Medicine, Medicine, 2007, University of Hong Kong

published_or_final_version

Clinical Oncology

Master

Master of Research in Medicine

Subjects/Keywords: Lungs - Cancer - Molecular aspects.

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APA (6th Edition):

Chu, Ka-wan, K. (2007). High-throughput molecular characterization of human non-small cell lung carcinoma. (Masters Thesis). University of Hong Kong. Retrieved from Chu, K. K. [朱嘉運]. (2007). High-throughput molecular characterization of human non-small cell lung carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501288 ; http://dx.doi.org/10.5353/th_b4501288 ; http://hdl.handle.net/10722/131694

Chicago Manual of Style (16th Edition):

Chu, Ka-wan, Kevin. “High-throughput molecular characterization of human non-small cell lung carcinoma.” 2007. Masters Thesis, University of Hong Kong. Accessed August 18, 2019. Chu, K. K. [朱嘉運]. (2007). High-throughput molecular characterization of human non-small cell lung carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501288 ; http://dx.doi.org/10.5353/th_b4501288 ; http://hdl.handle.net/10722/131694.

MLA Handbook (7th Edition):

Chu, Ka-wan, Kevin. “High-throughput molecular characterization of human non-small cell lung carcinoma.” 2007. Web. 18 Aug 2019.

Vancouver:

Chu, Ka-wan K. High-throughput molecular characterization of human non-small cell lung carcinoma. [Internet] [Masters thesis]. University of Hong Kong; 2007. [cited 2019 Aug 18]. Available from: Chu, K. K. [朱嘉運]. (2007). High-throughput molecular characterization of human non-small cell lung carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501288 ; http://dx.doi.org/10.5353/th_b4501288 ; http://hdl.handle.net/10722/131694.

Council of Science Editors:

Chu, Ka-wan K. High-throughput molecular characterization of human non-small cell lung carcinoma. [Masters Thesis]. University of Hong Kong; 2007. Available from: Chu, K. K. [朱嘉運]. (2007). High-throughput molecular characterization of human non-small cell lung carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4501288 ; http://dx.doi.org/10.5353/th_b4501288 ; http://hdl.handle.net/10722/131694


University of Hong Kong

21. Tey, Sze Keong. Clinical significance and functional role of nuclear met in hepatocellular carcinoma.

Degree: PhD, 2015, University of Hong Kong

abstract

Pathology

Doctoral

Doctor of Philosophy

Subjects/Keywords: Liver - Cancer - Molecular aspects

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APA (6th Edition):

Tey, S. K. (2015). Clinical significance and functional role of nuclear met in hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from http://hdl.handle.net/10722/222985

Chicago Manual of Style (16th Edition):

Tey, Sze Keong. “Clinical significance and functional role of nuclear met in hepatocellular carcinoma.” 2015. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. http://hdl.handle.net/10722/222985.

MLA Handbook (7th Edition):

Tey, Sze Keong. “Clinical significance and functional role of nuclear met in hepatocellular carcinoma.” 2015. Web. 18 Aug 2019.

Vancouver:

Tey SK. Clinical significance and functional role of nuclear met in hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2015. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/10722/222985.

Council of Science Editors:

Tey SK. Clinical significance and functional role of nuclear met in hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2015. Available from: http://hdl.handle.net/10722/222985


University of Hong Kong

22. Liu, Jia. Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival.

Degree: PhD, 2013, University of Hong Kong

 Esophageal cancer is the third most common digestive tract malignancy. Along with surgery, genotoxic drugs (e.g. cisplatin) and radiotherapy are the mainstays of treatment for… (more)

Subjects/Keywords: Protein kinases; Cellular signal transduction; Esophagus - Cancer - Molecular aspects

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APA (6th Edition):

Liu, J. (2013). Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival. (Doctoral Dissertation). University of Hong Kong. Retrieved from Liu, J. [劉佳]. (2013). Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5186496 ; http://dx.doi.org/10.5353/th_b5186496 ; http://hdl.handle.net/10722/205657

Chicago Manual of Style (16th Edition):

Liu, Jia. “Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Liu, J. [劉佳]. (2013). Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5186496 ; http://dx.doi.org/10.5353/th_b5186496 ; http://hdl.handle.net/10722/205657.

MLA Handbook (7th Edition):

Liu, Jia. “Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival.” 2013. Web. 18 Aug 2019.

Vancouver:

Liu J. Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Aug 18]. Available from: Liu, J. [劉佳]. (2013). Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5186496 ; http://dx.doi.org/10.5353/th_b5186496 ; http://hdl.handle.net/10722/205657.

Council of Science Editors:

Liu J. Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Liu, J. [劉佳]. (2013). Role of FBXO31 in regulating MAPK-mediated genotoxic stress response and cancer cell survival. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5186496 ; http://dx.doi.org/10.5353/th_b5186496 ; http://hdl.handle.net/10722/205657


University of Hong Kong

23. Ma, Wei. Study of the roles of RhoE in human hepatocellular carcinoma.

Degree: PhD, 2013, University of Hong Kong

 Hepatocellular carcinoma (HCC) is the seventh most prevalent cancer and the third leading cause of cancer-related mortality globally. Metastasis is a major cause of mortality.… (more)

Subjects/Keywords: Liver - Cancer - Molecular aspects; Rho GTPases; Protein kinases

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APA (6th Edition):

Ma, W. (2013). Study of the roles of RhoE in human hepatocellular carcinoma. (Doctoral Dissertation). University of Hong Kong. Retrieved from Ma, W. [馬威]. (2013). Study of the roles of RhoE in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108667 ; http://dx.doi.org/10.5353/th_b5108667 ; http://hdl.handle.net/10722/205869

Chicago Manual of Style (16th Edition):

Ma, Wei. “Study of the roles of RhoE in human hepatocellular carcinoma.” 2013. Doctoral Dissertation, University of Hong Kong. Accessed August 18, 2019. Ma, W. [馬威]. (2013). Study of the roles of RhoE in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108667 ; http://dx.doi.org/10.5353/th_b5108667 ; http://hdl.handle.net/10722/205869.

MLA Handbook (7th Edition):

Ma, Wei. “Study of the roles of RhoE in human hepatocellular carcinoma.” 2013. Web. 18 Aug 2019.

Vancouver:

Ma W. Study of the roles of RhoE in human hepatocellular carcinoma. [Internet] [Doctoral dissertation]. University of Hong Kong; 2013. [cited 2019 Aug 18]. Available from: Ma, W. [馬威]. (2013). Study of the roles of RhoE in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108667 ; http://dx.doi.org/10.5353/th_b5108667 ; http://hdl.handle.net/10722/205869.

Council of Science Editors:

Ma W. Study of the roles of RhoE in human hepatocellular carcinoma. [Doctoral Dissertation]. University of Hong Kong; 2013. Available from: Ma, W. [馬威]. (2013). Study of the roles of RhoE in human hepatocellular carcinoma. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b5108667 ; http://dx.doi.org/10.5353/th_b5108667 ; http://hdl.handle.net/10722/205869


University of Hong Kong

24. Wang, Neng. Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer.

Degree: M. Phil., 2012, University of Hong Kong

Angiogenesis is one of the essential hallmarks of cancer, typically breast cancer. Signaling from VEGFR-2 is necessary for the execution of VEGF-induced proliferation, migration, and… (more)

Subjects/Keywords: Plant polyphenols.; Neovascularization inhibitors.; Breast - Cancer - Molecular aspects.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, N. (2012). Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer. (Masters Thesis). University of Hong Kong. Retrieved from Wang, N. [王能]. (2012). Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786933 ; http://dx.doi.org/10.5353/th_b4786933 ; http://hdl.handle.net/10722/181909

Chicago Manual of Style (16th Edition):

Wang, Neng. “Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer.” 2012. Masters Thesis, University of Hong Kong. Accessed August 18, 2019. Wang, N. [王能]. (2012). Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786933 ; http://dx.doi.org/10.5353/th_b4786933 ; http://hdl.handle.net/10722/181909.

MLA Handbook (7th Edition):

Wang, Neng. “Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer.” 2012. Web. 18 Aug 2019.

Vancouver:

Wang N. Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer. [Internet] [Masters thesis]. University of Hong Kong; 2012. [cited 2019 Aug 18]. Available from: Wang, N. [王能]. (2012). Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786933 ; http://dx.doi.org/10.5353/th_b4786933 ; http://hdl.handle.net/10722/181909.

Council of Science Editors:

Wang N. Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer. [Masters Thesis]. University of Hong Kong; 2012. Available from: Wang, N. [王能]. (2012). Ellagic acid exerts anti-angiogenesis effects by blocking VEGFR-2 signaling pathway in breast cancer. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4786933 ; http://dx.doi.org/10.5353/th_b4786933 ; http://hdl.handle.net/10722/181909


Columbia University

25. Ziperstein, Michelle Joy. Regulation of Breast Cancer Cell Morphological and Invasive Characteristics by the Extracellular Environment.

Degree: 2016, Columbia University

 The aim of this thesis is to evaluate the role of the extracellular environment in regulating breast cancer cell morphological and invasive characteristics. In vitro… (more)

Subjects/Keywords: Breast – Cancer – Molecular aspects; Extracellular matrix – Research; Extracellular space; Chemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ziperstein, M. J. (2016). Regulation of Breast Cancer Cell Morphological and Invasive Characteristics by the Extracellular Environment. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8JD4X2Q

Chicago Manual of Style (16th Edition):

Ziperstein, Michelle Joy. “Regulation of Breast Cancer Cell Morphological and Invasive Characteristics by the Extracellular Environment.” 2016. Doctoral Dissertation, Columbia University. Accessed August 18, 2019. https://doi.org/10.7916/D8JD4X2Q.

MLA Handbook (7th Edition):

Ziperstein, Michelle Joy. “Regulation of Breast Cancer Cell Morphological and Invasive Characteristics by the Extracellular Environment.” 2016. Web. 18 Aug 2019.

Vancouver:

Ziperstein MJ. Regulation of Breast Cancer Cell Morphological and Invasive Characteristics by the Extracellular Environment. [Internet] [Doctoral dissertation]. Columbia University; 2016. [cited 2019 Aug 18]. Available from: https://doi.org/10.7916/D8JD4X2Q.

Council of Science Editors:

Ziperstein MJ. Regulation of Breast Cancer Cell Morphological and Invasive Characteristics by the Extracellular Environment. [Doctoral Dissertation]. Columbia University; 2016. Available from: https://doi.org/10.7916/D8JD4X2Q


Columbia University

26. Chung, Wei-Jen. Identification of microRNA Biogenesis Regulators and Activity Modulators.

Degree: 2014, Columbia University

 MicroRNAs play a key role in post-transcriptional gene regulation. They regulate target gene expression with mRNA degradation or translation repression. Each miRNA is estimated to… (more)

Subjects/Keywords: Cancer – Genetic aspects; Metastasis; Genetic regulation; RNA; Bioinformatics; Molecular biology

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APA (6th Edition):

Chung, W. (2014). Identification of microRNA Biogenesis Regulators and Activity Modulators. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D89Z92Z8

Chicago Manual of Style (16th Edition):

Chung, Wei-Jen. “Identification of microRNA Biogenesis Regulators and Activity Modulators.” 2014. Doctoral Dissertation, Columbia University. Accessed August 18, 2019. https://doi.org/10.7916/D89Z92Z8.

MLA Handbook (7th Edition):

Chung, Wei-Jen. “Identification of microRNA Biogenesis Regulators and Activity Modulators.” 2014. Web. 18 Aug 2019.

Vancouver:

Chung W. Identification of microRNA Biogenesis Regulators and Activity Modulators. [Internet] [Doctoral dissertation]. Columbia University; 2014. [cited 2019 Aug 18]. Available from: https://doi.org/10.7916/D89Z92Z8.

Council of Science Editors:

Chung W. Identification of microRNA Biogenesis Regulators and Activity Modulators. [Doctoral Dissertation]. Columbia University; 2014. Available from: https://doi.org/10.7916/D89Z92Z8


IUPUI

27. Qin, Li. Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer.

Degree: 2014, IUPUI

Indiana University-Purdue University (IUPUI)

Most pancreatic cancer patients receiving gemcitabine chemotherapy eventually develop resistance to gemcitabine. To improve survival and prognosis of pancreatic cancer patients,… (more)

Subjects/Keywords: 14-3-3sigma; PDGFD; YAP1; Uhrf1; DNMT1; Apoptosis; Chemotherapy; Drugs  – Side effects; Pancreas  – Diseases; Cancer  – Genetic aspects; Cancer  – Molecular aspects; Genomics

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APA (6th Edition):

Qin, L. (2014). Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/6295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Qin, Li. “Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer.” 2014. Thesis, IUPUI. Accessed August 18, 2019. http://hdl.handle.net/1805/6295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Qin, Li. “Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer.” 2014. Web. 18 Aug 2019.

Vancouver:

Qin L. Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer. [Internet] [Thesis]. IUPUI; 2014. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1805/6295.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Qin L. Molecular mechanisms of acquired gemcitabine resistance in pancreatic cancer. [Thesis]. IUPUI; 2014. Available from: http://hdl.handle.net/1805/6295

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Michigan State University

28. Abd El Fattah, Sherif. Fra-1 level in aggressive cancer cell lines under serum starved state and its impact on an autocrine loop.

Degree: 2015, Michigan State University

Thesis Ph. D. Michigan State University. Genetics 2015.

Fos related antigen 1 (Fra-1) is a component of the dimeric AP-1transcription factor that plays an important… (more)

Subjects/Keywords: Breast – Cancer – Genetic aspects; Cancer – Genetic aspects; Autocrine mechanisms; Cell cycle – Regulation; Antigens; Transcription factors; Genetics; Oncology; Molecular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Abd El Fattah, S. (2015). Fra-1 level in aggressive cancer cell lines under serum starved state and its impact on an autocrine loop. (Thesis). Michigan State University. Retrieved from http://etd.lib.msu.edu/islandora/object/etd:3776

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Abd El Fattah, Sherif. “Fra-1 level in aggressive cancer cell lines under serum starved state and its impact on an autocrine loop.” 2015. Thesis, Michigan State University. Accessed August 18, 2019. http://etd.lib.msu.edu/islandora/object/etd:3776.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Abd El Fattah, Sherif. “Fra-1 level in aggressive cancer cell lines under serum starved state and its impact on an autocrine loop.” 2015. Web. 18 Aug 2019.

Vancouver:

Abd El Fattah S. Fra-1 level in aggressive cancer cell lines under serum starved state and its impact on an autocrine loop. [Internet] [Thesis]. Michigan State University; 2015. [cited 2019 Aug 18]. Available from: http://etd.lib.msu.edu/islandora/object/etd:3776.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Abd El Fattah S. Fra-1 level in aggressive cancer cell lines under serum starved state and its impact on an autocrine loop. [Thesis]. Michigan State University; 2015. Available from: http://etd.lib.msu.edu/islandora/object/etd:3776

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida Atlantic University

29. Oseni, Saheed Oluwasina. Enhancement of the Chemopreventive and Chemotherapeutic Effects of Genistein and Beta-lapachone in Human Prostate Cancer Cells by Pyroelectrically Generated Very Low Dose Ionizing Radiation.

Degree: MS, 2015, Florida Atlantic University

Summary: An estimated 220,800 new prostate cancer cases and 27,540 deaths are expected to occur in US men by the end of 2015. Despite the… (more)

Subjects/Keywords: Apoptosis  – Molecular aspects; Genistein  – Therapeutic use; Phytochemicals  – Physiological effect; Phytochemicals  – Therapeutic use; Prostate  –  Cancer  – Adjuvant treatment; Prostate  –  Cancer  – Cryptopathology; Prostate  –  Cancer  – Molecular aspects

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APA (6th Edition):

Oseni, S. O. (2015). Enhancement of the Chemopreventive and Chemotherapeutic Effects of Genistein and Beta-lapachone in Human Prostate Cancer Cells by Pyroelectrically Generated Very Low Dose Ionizing Radiation. (Masters Thesis). Florida Atlantic University. Retrieved from http://purl.flvc.org/fau/fd/FA00004530 ; (URL) http://purl.flvc.org/fau/fd/FA00004530

Chicago Manual of Style (16th Edition):

Oseni, Saheed Oluwasina. “Enhancement of the Chemopreventive and Chemotherapeutic Effects of Genistein and Beta-lapachone in Human Prostate Cancer Cells by Pyroelectrically Generated Very Low Dose Ionizing Radiation.” 2015. Masters Thesis, Florida Atlantic University. Accessed August 18, 2019. http://purl.flvc.org/fau/fd/FA00004530 ; (URL) http://purl.flvc.org/fau/fd/FA00004530.

MLA Handbook (7th Edition):

Oseni, Saheed Oluwasina. “Enhancement of the Chemopreventive and Chemotherapeutic Effects of Genistein and Beta-lapachone in Human Prostate Cancer Cells by Pyroelectrically Generated Very Low Dose Ionizing Radiation.” 2015. Web. 18 Aug 2019.

Vancouver:

Oseni SO. Enhancement of the Chemopreventive and Chemotherapeutic Effects of Genistein and Beta-lapachone in Human Prostate Cancer Cells by Pyroelectrically Generated Very Low Dose Ionizing Radiation. [Internet] [Masters thesis]. Florida Atlantic University; 2015. [cited 2019 Aug 18]. Available from: http://purl.flvc.org/fau/fd/FA00004530 ; (URL) http://purl.flvc.org/fau/fd/FA00004530.

Council of Science Editors:

Oseni SO. Enhancement of the Chemopreventive and Chemotherapeutic Effects of Genistein and Beta-lapachone in Human Prostate Cancer Cells by Pyroelectrically Generated Very Low Dose Ionizing Radiation. [Masters Thesis]. Florida Atlantic University; 2015. Available from: http://purl.flvc.org/fau/fd/FA00004530 ; (URL) http://purl.flvc.org/fau/fd/FA00004530


IUPUI

30. Cao, Liyun. Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis.

Degree: 2012, IUPUI

Indiana University-Purdue University Indianapolis (IUPUI)

Ovarian cancer (OC) is a lethal disease due to metastasis and chemoresistance. Our laboratory previously reported that tissue transglutaminase (TG2)… (more)

Subjects/Keywords: tissue transglutaminase, TGF-beta, ovarian cancer; Ovaries  – Cancer  – Molecular aspects  – Research  – Methodology; Ovaries  – Cancer  – Genetic aspects  – Research  – Methodology; Metastasis; Tumor markers  – Research  – Methodology; Peritoneum  – Cancer; Transglutaminases

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APA (6th Edition):

Cao, L. (2012). Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis. (Thesis). IUPUI. Retrieved from http://hdl.handle.net/1805/2817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cao, Liyun. “Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis.” 2012. Thesis, IUPUI. Accessed August 18, 2019. http://hdl.handle.net/1805/2817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cao, Liyun. “Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis.” 2012. Web. 18 Aug 2019.

Vancouver:

Cao L. Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis. [Internet] [Thesis]. IUPUI; 2012. [cited 2019 Aug 18]. Available from: http://hdl.handle.net/1805/2817.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cao L. Mechanism of tissue transglutaminase upregulation and its role in ovarian cancer metastasis. [Thesis]. IUPUI; 2012. Available from: http://hdl.handle.net/1805/2817

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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