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You searched for subject:(CaMKII). Showing records 1 – 30 of 96 total matches.

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Vanderbilt University

1. Hoffman, Laurel R. Investigation of CaMKII activation: a model of self-regulation.

Degree: PhD, Molecular Physiology and Biophysics, 2011, Vanderbilt University

 Presently we do not completely understand how memories are able to store, retain, and recall information and experiences. The investigation is just beginning and proteins… (more)

Subjects/Keywords: CaMKII; EPR

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APA (6th Edition):

Hoffman, L. R. (2011). Investigation of CaMKII activation: a model of self-regulation. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-11302011-144712/ ;

Chicago Manual of Style (16th Edition):

Hoffman, Laurel R. “Investigation of CaMKII activation: a model of self-regulation.” 2011. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu//available/etd-11302011-144712/ ;.

MLA Handbook (7th Edition):

Hoffman, Laurel R. “Investigation of CaMKII activation: a model of self-regulation.” 2011. Web. 23 Sep 2019.

Vancouver:

Hoffman LR. Investigation of CaMKII activation: a model of self-regulation. [Internet] [Doctoral dissertation]. Vanderbilt University; 2011. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-11302011-144712/ ;.

Council of Science Editors:

Hoffman LR. Investigation of CaMKII activation: a model of self-regulation. [Doctoral Dissertation]. Vanderbilt University; 2011. Available from: http://etd.library.vanderbilt.edu//available/etd-11302011-144712/ ;


The Ohio State University

2. Glynn, Patric Joseph. Integrative Studies on the Role of CaMKII in Cardiac Disease and Arrhythmias.

Degree: PhD, Biomedical Engineering, 2015, The Ohio State University

 Heart disease accounts for one of every three deaths in the United States annually. The majority of these deaths are caused by disturbances in normal… (more)

Subjects/Keywords: Biomedical Engineering; Cardiac; CaMKII; Modeling

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APA (6th Edition):

Glynn, P. J. (2015). Integrative Studies on the Role of CaMKII in Cardiac Disease and Arrhythmias. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1429132697

Chicago Manual of Style (16th Edition):

Glynn, Patric Joseph. “Integrative Studies on the Role of CaMKII in Cardiac Disease and Arrhythmias.” 2015. Doctoral Dissertation, The Ohio State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1429132697.

MLA Handbook (7th Edition):

Glynn, Patric Joseph. “Integrative Studies on the Role of CaMKII in Cardiac Disease and Arrhythmias.” 2015. Web. 23 Sep 2019.

Vancouver:

Glynn PJ. Integrative Studies on the Role of CaMKII in Cardiac Disease and Arrhythmias. [Internet] [Doctoral dissertation]. The Ohio State University; 2015. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1429132697.

Council of Science Editors:

Glynn PJ. Integrative Studies on the Role of CaMKII in Cardiac Disease and Arrhythmias. [Doctoral Dissertation]. The Ohio State University; 2015. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1429132697


University of Illinois – Chicago

3. Hu, Xiaoyu. Molecular Mechanisms and Experimental Therapies of Pain in Multiple Sclerosis.

Degree: 2017, University of Illinois – Chicago

 Multiple sclerosis is an autoimmune disorder in central nervous system with manifestations of neuroinflammation and demyelination. Pain associated with multiple sclerosis is a common and… (more)

Subjects/Keywords: pain; multiple sclerosis; CaMKII; curcumin

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APA (6th Edition):

Hu, X. (2017). Molecular Mechanisms and Experimental Therapies of Pain in Multiple Sclerosis. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/21777

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hu, Xiaoyu. “Molecular Mechanisms and Experimental Therapies of Pain in Multiple Sclerosis.” 2017. Thesis, University of Illinois – Chicago. Accessed September 23, 2019. http://hdl.handle.net/10027/21777.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hu, Xiaoyu. “Molecular Mechanisms and Experimental Therapies of Pain in Multiple Sclerosis.” 2017. Web. 23 Sep 2019.

Vancouver:

Hu X. Molecular Mechanisms and Experimental Therapies of Pain in Multiple Sclerosis. [Internet] [Thesis]. University of Illinois – Chicago; 2017. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10027/21777.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hu X. Molecular Mechanisms and Experimental Therapies of Pain in Multiple Sclerosis. [Thesis]. University of Illinois – Chicago; 2017. Available from: http://hdl.handle.net/10027/21777

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

4. Mora, Rudy J. Genetically-encoded probes for visualizing protein trafficking dynamics in living neurons.

Degree: PhD, Neuroscience, 2012, University of Southern California

 Here I describe the development of genetically-encoded probes for visualizing the trafficking of neuronal proteins in living cells. The probes are derived from a diverse… (more)

Subjects/Keywords: probe; calcium; traffic; neuron; CaMKII

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APA (6th Edition):

Mora, R. J. (2012). Genetically-encoded probes for visualizing protein trafficking dynamics in living neurons. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8143/rec/3010

Chicago Manual of Style (16th Edition):

Mora, Rudy J. “Genetically-encoded probes for visualizing protein trafficking dynamics in living neurons.” 2012. Doctoral Dissertation, University of Southern California. Accessed September 23, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8143/rec/3010.

MLA Handbook (7th Edition):

Mora, Rudy J. “Genetically-encoded probes for visualizing protein trafficking dynamics in living neurons.” 2012. Web. 23 Sep 2019.

Vancouver:

Mora RJ. Genetically-encoded probes for visualizing protein trafficking dynamics in living neurons. [Internet] [Doctoral dissertation]. University of Southern California; 2012. [cited 2019 Sep 23]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8143/rec/3010.

Council of Science Editors:

Mora RJ. Genetically-encoded probes for visualizing protein trafficking dynamics in living neurons. [Doctoral Dissertation]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/8143/rec/3010


Virginia Commonwealth University

5. Waggener, Christopher. Calcium/Calmodulin-Dependent Protein Kinase II Beta (CaMKIIβ): A Regulator of Oligodendrocyte Maturation and Myelination.

Degree: PhD, Integrative Life Sciences, 2013, Virginia Commonwealth University

  Oligodendrocytes are cells located in the central nervous system (CNS) that are responsible for the production of the lipid rich membrane, myelin. Myelin and… (more)

Subjects/Keywords: Oligodendrocyte; CaMKII; Myelin; Life Sciences

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APA (6th Edition):

Waggener, C. (2013). Calcium/Calmodulin-Dependent Protein Kinase II Beta (CaMKIIβ): A Regulator of Oligodendrocyte Maturation and Myelination. (Doctoral Dissertation). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/527

Chicago Manual of Style (16th Edition):

Waggener, Christopher. “Calcium/Calmodulin-Dependent Protein Kinase II Beta (CaMKIIβ): A Regulator of Oligodendrocyte Maturation and Myelination.” 2013. Doctoral Dissertation, Virginia Commonwealth University. Accessed September 23, 2019. https://scholarscompass.vcu.edu/etd/527.

MLA Handbook (7th Edition):

Waggener, Christopher. “Calcium/Calmodulin-Dependent Protein Kinase II Beta (CaMKIIβ): A Regulator of Oligodendrocyte Maturation and Myelination.” 2013. Web. 23 Sep 2019.

Vancouver:

Waggener C. Calcium/Calmodulin-Dependent Protein Kinase II Beta (CaMKIIβ): A Regulator of Oligodendrocyte Maturation and Myelination. [Internet] [Doctoral dissertation]. Virginia Commonwealth University; 2013. [cited 2019 Sep 23]. Available from: https://scholarscompass.vcu.edu/etd/527.

Council of Science Editors:

Waggener C. Calcium/Calmodulin-Dependent Protein Kinase II Beta (CaMKIIβ): A Regulator of Oligodendrocyte Maturation and Myelination. [Doctoral Dissertation]. Virginia Commonwealth University; 2013. Available from: https://scholarscompass.vcu.edu/etd/527


Penn State University

6. Lee, Hyun-Gwan. ROLES OF THE OCTOPAMINE RECEPTOR OAMB IN ASSOCIATIVE LEARNING, MEMORY, AND REPRODUCTION IN DROSOPHILA MELANOGASTER.

Degree: PhD, Genetics, 2008, Penn State University

 Octopamine, a biogenic monoamine, is a major neuromodulator in invertebrates. It plays a crucial role in various adaptive behaviors and in female reproduction. To mediate… (more)

Subjects/Keywords: octpamine receptor; OAMB; Conditioned courtship; Ovulation; CaMKII

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APA (6th Edition):

Lee, H. (2008). ROLES OF THE OCTOPAMINE RECEPTOR OAMB IN ASSOCIATIVE LEARNING, MEMORY, AND REPRODUCTION IN DROSOPHILA MELANOGASTER. (Doctoral Dissertation). Penn State University. Retrieved from https://etda.libraries.psu.edu/catalog/8440

Chicago Manual of Style (16th Edition):

Lee, Hyun-Gwan. “ROLES OF THE OCTOPAMINE RECEPTOR OAMB IN ASSOCIATIVE LEARNING, MEMORY, AND REPRODUCTION IN DROSOPHILA MELANOGASTER.” 2008. Doctoral Dissertation, Penn State University. Accessed September 23, 2019. https://etda.libraries.psu.edu/catalog/8440.

MLA Handbook (7th Edition):

Lee, Hyun-Gwan. “ROLES OF THE OCTOPAMINE RECEPTOR OAMB IN ASSOCIATIVE LEARNING, MEMORY, AND REPRODUCTION IN DROSOPHILA MELANOGASTER.” 2008. Web. 23 Sep 2019.

Vancouver:

Lee H. ROLES OF THE OCTOPAMINE RECEPTOR OAMB IN ASSOCIATIVE LEARNING, MEMORY, AND REPRODUCTION IN DROSOPHILA MELANOGASTER. [Internet] [Doctoral dissertation]. Penn State University; 2008. [cited 2019 Sep 23]. Available from: https://etda.libraries.psu.edu/catalog/8440.

Council of Science Editors:

Lee H. ROLES OF THE OCTOPAMINE RECEPTOR OAMB IN ASSOCIATIVE LEARNING, MEMORY, AND REPRODUCTION IN DROSOPHILA MELANOGASTER. [Doctoral Dissertation]. Penn State University; 2008. Available from: https://etda.libraries.psu.edu/catalog/8440


University of California – Berkeley

7. Chao, Luke H. Structural Studies of Calcium/Calmodulin Depenedent Protein Kinase II Activation.

Degree: Molecular & Cell Biology, 2010, University of California – Berkeley

 Cell signaling utilizes the frequency of calcium stimuli to produce diverse physiological outcomes. From fertilization of oocytes and cardiac facilitation, to muscle contraction and action… (more)

Subjects/Keywords: Biophysics, General; Chemistry, Biochemistry; CaMKII; Structural Biology

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APA (6th Edition):

Chao, L. H. (2010). Structural Studies of Calcium/Calmodulin Depenedent Protein Kinase II Activation. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/9bt8t2n7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chao, Luke H. “Structural Studies of Calcium/Calmodulin Depenedent Protein Kinase II Activation.” 2010. Thesis, University of California – Berkeley. Accessed September 23, 2019. http://www.escholarship.org/uc/item/9bt8t2n7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chao, Luke H. “Structural Studies of Calcium/Calmodulin Depenedent Protein Kinase II Activation.” 2010. Web. 23 Sep 2019.

Vancouver:

Chao LH. Structural Studies of Calcium/Calmodulin Depenedent Protein Kinase II Activation. [Internet] [Thesis]. University of California – Berkeley; 2010. [cited 2019 Sep 23]. Available from: http://www.escholarship.org/uc/item/9bt8t2n7.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chao LH. Structural Studies of Calcium/Calmodulin Depenedent Protein Kinase II Activation. [Thesis]. University of California – Berkeley; 2010. Available from: http://www.escholarship.org/uc/item/9bt8t2n7

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – Berkeley

8. McSpadden, Ethan DeNardo. Structural and evolutionary studies on CaMKII oligomerization.

Degree: Molecular & Cell Biology, 2018, University of California – Berkeley

 Calcium-calmodulin-dependent protein kinase II (CaMKII) plays a critical role in animal learning and memory (Giese et al. 1998). CaMKII operates as a multi-subunit holoenzyme and… (more)

Subjects/Keywords: Biochemistry; CaMKII; Chlamydomonas; Human; Protein; Structure

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APA (6th Edition):

McSpadden, E. D. (2018). Structural and evolutionary studies on CaMKII oligomerization. (Thesis). University of California – Berkeley. Retrieved from http://www.escholarship.org/uc/item/00k0r2pt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McSpadden, Ethan DeNardo. “Structural and evolutionary studies on CaMKII oligomerization.” 2018. Thesis, University of California – Berkeley. Accessed September 23, 2019. http://www.escholarship.org/uc/item/00k0r2pt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McSpadden, Ethan DeNardo. “Structural and evolutionary studies on CaMKII oligomerization.” 2018. Web. 23 Sep 2019.

Vancouver:

McSpadden ED. Structural and evolutionary studies on CaMKII oligomerization. [Internet] [Thesis]. University of California – Berkeley; 2018. [cited 2019 Sep 23]. Available from: http://www.escholarship.org/uc/item/00k0r2pt.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McSpadden ED. Structural and evolutionary studies on CaMKII oligomerization. [Thesis]. University of California – Berkeley; 2018. Available from: http://www.escholarship.org/uc/item/00k0r2pt

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

9. Klug, Jason Richard. Role of CaMKII and the GluN2B subunit of the NMDAR in dorsal striatal glutamatergic synaptic transmission, MSN morphology and striatal-based behaviors.

Degree: PhD, Neuroscience, 2012, Vanderbilt University

 The role of striatal Ca2+/calmodulin-dependent protein kinase II (CaMKII) and the GluN2B subunit of the NMDA receptor in excitatory glutamatergic transmission, morphology and behavior are… (more)

Subjects/Keywords: CaMKII; plasticity; electrophysiology; excitability; morphology; behavior; striatum

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APA (6th Edition):

Klug, J. R. (2012). Role of CaMKII and the GluN2B subunit of the NMDAR in dorsal striatal glutamatergic synaptic transmission, MSN morphology and striatal-based behaviors. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-09242012-210803/ ;

Chicago Manual of Style (16th Edition):

Klug, Jason Richard. “Role of CaMKII and the GluN2B subunit of the NMDAR in dorsal striatal glutamatergic synaptic transmission, MSN morphology and striatal-based behaviors.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu//available/etd-09242012-210803/ ;.

MLA Handbook (7th Edition):

Klug, Jason Richard. “Role of CaMKII and the GluN2B subunit of the NMDAR in dorsal striatal glutamatergic synaptic transmission, MSN morphology and striatal-based behaviors.” 2012. Web. 23 Sep 2019.

Vancouver:

Klug JR. Role of CaMKII and the GluN2B subunit of the NMDAR in dorsal striatal glutamatergic synaptic transmission, MSN morphology and striatal-based behaviors. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-09242012-210803/ ;.

Council of Science Editors:

Klug JR. Role of CaMKII and the GluN2B subunit of the NMDAR in dorsal striatal glutamatergic synaptic transmission, MSN morphology and striatal-based behaviors. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://etd.library.vanderbilt.edu//available/etd-09242012-210803/ ;


Brandeis University

10. Serebrenik, Joyce. The Role of CaMKII and CASK-β in the Regulation of TH and Dopamine Synthesis.

Degree: 2019, Brandeis University

 Phosphorylation at Threonine 306 and Threonine 307 results in a constitutively inactive CaMKII. This phosphorylation has been shown to be stimulated by CASK-β in low… (more)

Subjects/Keywords: CaMKII; CASK; Tyrosine Hydroxylase; Dopamine; Drosophila; Regulation

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APA (6th Edition):

Serebrenik, J. (2019). The Role of CaMKII and CASK-β in the Regulation of TH and Dopamine Synthesis. (Thesis). Brandeis University. Retrieved from http://hdl.handle.net/10192/36785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Serebrenik, Joyce. “The Role of CaMKII and CASK-β in the Regulation of TH and Dopamine Synthesis.” 2019. Thesis, Brandeis University. Accessed September 23, 2019. http://hdl.handle.net/10192/36785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Serebrenik, Joyce. “The Role of CaMKII and CASK-β in the Regulation of TH and Dopamine Synthesis.” 2019. Web. 23 Sep 2019.

Vancouver:

Serebrenik J. The Role of CaMKII and CASK-β in the Regulation of TH and Dopamine Synthesis. [Internet] [Thesis]. Brandeis University; 2019. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10192/36785.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Serebrenik J. The Role of CaMKII and CASK-β in the Regulation of TH and Dopamine Synthesis. [Thesis]. Brandeis University; 2019. Available from: http://hdl.handle.net/10192/36785

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Guelph

11. Wideman, Cassidy. Evidence that muscarinic receptor activation destabilizes object memories via a mechanism involving CaMKII and synaptic protein degradation .

Degree: 2019, University of Guelph

 Recently acquired information is stabilized for storage in long-term memory through the process of consolidation. However, when reactivated, consolidated memories can destabilize, rendering them labile… (more)

Subjects/Keywords: Memory; Reconsolidation; Destabilization; Acetylcholine; CaMKII; Synaptic Proteins

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APA (6th Edition):

Wideman, C. (2019). Evidence that muscarinic receptor activation destabilizes object memories via a mechanism involving CaMKII and synaptic protein degradation . (Thesis). University of Guelph. Retrieved from https://atrium.lib.uoguelph.ca/xmlui/handle/10214/16942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wideman, Cassidy. “Evidence that muscarinic receptor activation destabilizes object memories via a mechanism involving CaMKII and synaptic protein degradation .” 2019. Thesis, University of Guelph. Accessed September 23, 2019. https://atrium.lib.uoguelph.ca/xmlui/handle/10214/16942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wideman, Cassidy. “Evidence that muscarinic receptor activation destabilizes object memories via a mechanism involving CaMKII and synaptic protein degradation .” 2019. Web. 23 Sep 2019.

Vancouver:

Wideman C. Evidence that muscarinic receptor activation destabilizes object memories via a mechanism involving CaMKII and synaptic protein degradation . [Internet] [Thesis]. University of Guelph; 2019. [cited 2019 Sep 23]. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/16942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wideman C. Evidence that muscarinic receptor activation destabilizes object memories via a mechanism involving CaMKII and synaptic protein degradation . [Thesis]. University of Guelph; 2019. Available from: https://atrium.lib.uoguelph.ca/xmlui/handle/10214/16942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Mones, Saleh. Création et caractérisation des modèles animaux pré-clinique de CMTX : Creation and characterization of pre-clinic CMTX animal models.

Degree: Docteur es, Pathologie humaine. Génétique humaine, 2014, Aix Marseille Université

La maladie de Charcot-Marie-Tooth liée à l'X (CMTX), deuxième cause, en fréquence, de neuropathies héréditaire, est due à des mutations dans le gène Gjb1 codant… (more)

Subjects/Keywords: Cmtx; Connexine32; CaMKII; Souris transgéniques; Instabilité mitotiques; Cmtx; Connexin32; CaMKII; Transgenic mice; Mitotic instability

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APA (6th Edition):

Mones, S. (2014). Création et caractérisation des modèles animaux pré-clinique de CMTX : Creation and characterization of pre-clinic CMTX animal models. (Doctoral Dissertation). Aix Marseille Université. Retrieved from http://www.theses.fr/2014AIXM5007

Chicago Manual of Style (16th Edition):

Mones, Saleh. “Création et caractérisation des modèles animaux pré-clinique de CMTX : Creation and characterization of pre-clinic CMTX animal models.” 2014. Doctoral Dissertation, Aix Marseille Université. Accessed September 23, 2019. http://www.theses.fr/2014AIXM5007.

MLA Handbook (7th Edition):

Mones, Saleh. “Création et caractérisation des modèles animaux pré-clinique de CMTX : Creation and characterization of pre-clinic CMTX animal models.” 2014. Web. 23 Sep 2019.

Vancouver:

Mones S. Création et caractérisation des modèles animaux pré-clinique de CMTX : Creation and characterization of pre-clinic CMTX animal models. [Internet] [Doctoral dissertation]. Aix Marseille Université 2014. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2014AIXM5007.

Council of Science Editors:

Mones S. Création et caractérisation des modèles animaux pré-clinique de CMTX : Creation and characterization of pre-clinic CMTX animal models. [Doctoral Dissertation]. Aix Marseille Université 2014. Available from: http://www.theses.fr/2014AIXM5007


Kaunas University of Technology

13. Slivko, Giedrė. Biologinių neuronų mokymosi savybių tyrimas.

Degree: Master, Informatics, 2008, Kaunas University of Technology

Ankstesni tyrimai, susiję su laikinėmis sinapsinio efektyvumo pasikeitimo taisyklėmis per nuo veikimo potencialų poravimo priklausantį plastiškumą, praktiškai nekreipė jokio dėmesio į neuronų erdvines savybes. Mes… (more)

Subjects/Keywords: Sinapsinis plastiškumas; Ca2+; CaM; CaMKII; CaN; Synaptic plasticity; Ca2+; CaM; CaMKII; CaN

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APA (6th Edition):

Slivko, Giedrė. (2008). Biologinių neuronų mokymosi savybių tyrimas. (Masters Thesis). Kaunas University of Technology. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2008~D_20080902_100150-63821 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Slivko, Giedrė. “Biologinių neuronų mokymosi savybių tyrimas.” 2008. Masters Thesis, Kaunas University of Technology. Accessed September 23, 2019. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2008~D_20080902_100150-63821 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Slivko, Giedrė. “Biologinių neuronų mokymosi savybių tyrimas.” 2008. Web. 23 Sep 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Slivko, Giedrė. Biologinių neuronų mokymosi savybių tyrimas. [Internet] [Masters thesis]. Kaunas University of Technology; 2008. [cited 2019 Sep 23]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2008~D_20080902_100150-63821 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Slivko, Giedrė. Biologinių neuronų mokymosi savybių tyrimas. [Masters Thesis]. Kaunas University of Technology; 2008. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2008~D_20080902_100150-63821 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Université Paris-Sud – Paris XI

14. Breckler, Magali. Mise en évidence des effets anti-hypertrophiques de carabine dans le cardiomyocyte et caractérisation de ses voies de signalisation : Study of Signalling Pathway Regulated by Carabin in the Cardiomyocyte and Highlights its Anti-Hypertrophic Role.

Degree: Docteur es, Biologie Cellulaire et Moléculaire, Science de la Santé, 2012, Université Paris-Sud – Paris XI

L’hypertrophie cardiaque est un mécanisme adaptatif qui se développe en réponse à une surcharge de travail hémodynamique. Lors d’un stress chronique, elle peut mener à… (more)

Subjects/Keywords: Carabine; Récepteurs α et β-adrénergiques; CaN; Ras; CaMKII; Hypertrophie cardiaque; Carabin; Cardiac hypertrophy and failure; Calcineurine; Ras; CaMKII; Epac

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APA (6th Edition):

Breckler, M. (2012). Mise en évidence des effets anti-hypertrophiques de carabine dans le cardiomyocyte et caractérisation de ses voies de signalisation : Study of Signalling Pathway Regulated by Carabin in the Cardiomyocyte and Highlights its Anti-Hypertrophic Role. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2012PA11T014

Chicago Manual of Style (16th Edition):

Breckler, Magali. “Mise en évidence des effets anti-hypertrophiques de carabine dans le cardiomyocyte et caractérisation de ses voies de signalisation : Study of Signalling Pathway Regulated by Carabin in the Cardiomyocyte and Highlights its Anti-Hypertrophic Role.” 2012. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed September 23, 2019. http://www.theses.fr/2012PA11T014.

MLA Handbook (7th Edition):

Breckler, Magali. “Mise en évidence des effets anti-hypertrophiques de carabine dans le cardiomyocyte et caractérisation de ses voies de signalisation : Study of Signalling Pathway Regulated by Carabin in the Cardiomyocyte and Highlights its Anti-Hypertrophic Role.” 2012. Web. 23 Sep 2019.

Vancouver:

Breckler M. Mise en évidence des effets anti-hypertrophiques de carabine dans le cardiomyocyte et caractérisation de ses voies de signalisation : Study of Signalling Pathway Regulated by Carabin in the Cardiomyocyte and Highlights its Anti-Hypertrophic Role. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2012. [cited 2019 Sep 23]. Available from: http://www.theses.fr/2012PA11T014.

Council of Science Editors:

Breckler M. Mise en évidence des effets anti-hypertrophiques de carabine dans le cardiomyocyte et caractérisation de ses voies de signalisation : Study of Signalling Pathway Regulated by Carabin in the Cardiomyocyte and Highlights its Anti-Hypertrophic Role. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2012. Available from: http://www.theses.fr/2012PA11T014


University of Edinburgh

15. Jama, Abdirahman Mohamud. Functional regulation of kisspeptin receptor by calmodulin and Ca2+/calmodulin-dependent protein kinase II.

Degree: PhD, 2015, University of Edinburgh

 The kisspeptin receptor (KISS1R), functioning as a metastasis suppressor and gatekeeper of GnRH neurons, is a potent activator of intracellular Ca2+. The surge in cytoplasmic… (more)

Subjects/Keywords: 572; kisspeptin receptor; GPCR; calcium; Ca2+; CaMKII; calmodulin; CaM

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APA (6th Edition):

Jama, A. M. (2015). Functional regulation of kisspeptin receptor by calmodulin and Ca2+/calmodulin-dependent protein kinase II. (Doctoral Dissertation). University of Edinburgh. Retrieved from http://hdl.handle.net/1842/15914

Chicago Manual of Style (16th Edition):

Jama, Abdirahman Mohamud. “Functional regulation of kisspeptin receptor by calmodulin and Ca2+/calmodulin-dependent protein kinase II.” 2015. Doctoral Dissertation, University of Edinburgh. Accessed September 23, 2019. http://hdl.handle.net/1842/15914.

MLA Handbook (7th Edition):

Jama, Abdirahman Mohamud. “Functional regulation of kisspeptin receptor by calmodulin and Ca2+/calmodulin-dependent protein kinase II.” 2015. Web. 23 Sep 2019.

Vancouver:

Jama AM. Functional regulation of kisspeptin receptor by calmodulin and Ca2+/calmodulin-dependent protein kinase II. [Internet] [Doctoral dissertation]. University of Edinburgh; 2015. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1842/15914.

Council of Science Editors:

Jama AM. Functional regulation of kisspeptin receptor by calmodulin and Ca2+/calmodulin-dependent protein kinase II. [Doctoral Dissertation]. University of Edinburgh; 2015. Available from: http://hdl.handle.net/1842/15914


The Ohio State University

16. Tang, Lifei. Effects of Neuronal Nitric Oxide Synthase Signaling on Myocyte Contraction during Beta-Adrenergic Stimulation.

Degree: PhD, Biophysics, 2013, The Ohio State University

 Stimulation of the beta-adrenergic (beta-AR) pathway leads to positive inotropy, and is the major regulator of heart function. The purpose of this study is to… (more)

Subjects/Keywords: Cellular Biology; Physiology; Biophysics; NOS1; CaMKII; RyR; beta Adrenergic stimulation; Akt

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APA (6th Edition):

Tang, L. (2013). Effects of Neuronal Nitric Oxide Synthase Signaling on Myocyte Contraction during Beta-Adrenergic Stimulation. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1385336408

Chicago Manual of Style (16th Edition):

Tang, Lifei. “Effects of Neuronal Nitric Oxide Synthase Signaling on Myocyte Contraction during Beta-Adrenergic Stimulation.” 2013. Doctoral Dissertation, The Ohio State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1385336408.

MLA Handbook (7th Edition):

Tang, Lifei. “Effects of Neuronal Nitric Oxide Synthase Signaling on Myocyte Contraction during Beta-Adrenergic Stimulation.” 2013. Web. 23 Sep 2019.

Vancouver:

Tang L. Effects of Neuronal Nitric Oxide Synthase Signaling on Myocyte Contraction during Beta-Adrenergic Stimulation. [Internet] [Doctoral dissertation]. The Ohio State University; 2013. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1385336408.

Council of Science Editors:

Tang L. Effects of Neuronal Nitric Oxide Synthase Signaling on Myocyte Contraction during Beta-Adrenergic Stimulation. [Doctoral Dissertation]. The Ohio State University; 2013. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1385336408


University of Alberta

17. Yang, Kaiyuan. Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats.

Degree: PhD, Department of Agricultural, Food, and Nutritional Science, 2015, University of Alberta

 Proanthocyanidins (PAC) belong to a highly consumed class of flavonoids and their consumption has been linked to beneficial effects on glycemic control in type 2… (more)

Subjects/Keywords: Proanthocyanidins; Bioavailability; CaMKII; Hepatic glucose production; Glucose homeostasis; Pancreatic β-cells

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APA (6th Edition):

Yang, K. (2015). Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats. (Doctoral Dissertation). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/n870zt826

Chicago Manual of Style (16th Edition):

Yang, Kaiyuan. “Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats.” 2015. Doctoral Dissertation, University of Alberta. Accessed September 23, 2019. https://era.library.ualberta.ca/files/n870zt826.

MLA Handbook (7th Edition):

Yang, Kaiyuan. “Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats.” 2015. Web. 23 Sep 2019.

Vancouver:

Yang K. Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats. [Internet] [Doctoral dissertation]. University of Alberta; 2015. [cited 2019 Sep 23]. Available from: https://era.library.ualberta.ca/files/n870zt826.

Council of Science Editors:

Yang K. Proanthocyanidins and Glucose Homeostasis: An Analysis of Bioavailability and Mechanisms in Rats. [Doctoral Dissertation]. University of Alberta; 2015. Available from: https://era.library.ualberta.ca/files/n870zt826


University of California – San Diego

18. Gray, Charles Burdis Burns. The role of CaMKII-delta subtypes in myocardial ischemia/reperfusion injury.

Degree: Biomedical Sciences, 2016, University of California – San Diego

 Ca2+ dysregulation underlies many forms of cardiac stress and contributes significantly to arrhythmia and dysfunction during heart disease. Ca2+/CaM-dependent protein kinase II (CaMKII) is a… (more)

Subjects/Keywords: Pharmacology; Cellular biology; CaMKII; Ischemia; NF-kB; Reperfusion; TNF-a

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APA (6th Edition):

Gray, C. B. B. (2016). The role of CaMKII-delta subtypes in myocardial ischemia/reperfusion injury. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/8gk4k9ps

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gray, Charles Burdis Burns. “The role of CaMKII-delta subtypes in myocardial ischemia/reperfusion injury.” 2016. Thesis, University of California – San Diego. Accessed September 23, 2019. http://www.escholarship.org/uc/item/8gk4k9ps.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gray, Charles Burdis Burns. “The role of CaMKII-delta subtypes in myocardial ischemia/reperfusion injury.” 2016. Web. 23 Sep 2019.

Vancouver:

Gray CBB. The role of CaMKII-delta subtypes in myocardial ischemia/reperfusion injury. [Internet] [Thesis]. University of California – San Diego; 2016. [cited 2019 Sep 23]. Available from: http://www.escholarship.org/uc/item/8gk4k9ps.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gray CBB. The role of CaMKII-delta subtypes in myocardial ischemia/reperfusion injury. [Thesis]. University of California – San Diego; 2016. Available from: http://www.escholarship.org/uc/item/8gk4k9ps

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of California – San Diego

19. Willeford, Andrew. The Role of CaMKII delta in Pathophysiological Responses Induced by Beta-Adrenergic Receptor Stimulation and Angiotensin II.

Degree: Biomedical Sciences, 2017, University of California – San Diego

 The initial studies of my graduate research, which resulted in a co-authorship and for clarity reasons is presented in this dissertation in full, utilized calcium/calmodulin-dependent… (more)

Subjects/Keywords: Molecular biology; Pharmacology; angiotensin II; beta-adrenergic; CaMKII; cardiomyocyte; inflammasome; inflammation

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APA (6th Edition):

Willeford, A. (2017). The Role of CaMKII delta in Pathophysiological Responses Induced by Beta-Adrenergic Receptor Stimulation and Angiotensin II. (Thesis). University of California – San Diego. Retrieved from http://www.escholarship.org/uc/item/88x3c8w9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Willeford, Andrew. “The Role of CaMKII delta in Pathophysiological Responses Induced by Beta-Adrenergic Receptor Stimulation and Angiotensin II.” 2017. Thesis, University of California – San Diego. Accessed September 23, 2019. http://www.escholarship.org/uc/item/88x3c8w9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Willeford, Andrew. “The Role of CaMKII delta in Pathophysiological Responses Induced by Beta-Adrenergic Receptor Stimulation and Angiotensin II.” 2017. Web. 23 Sep 2019.

Vancouver:

Willeford A. The Role of CaMKII delta in Pathophysiological Responses Induced by Beta-Adrenergic Receptor Stimulation and Angiotensin II. [Internet] [Thesis]. University of California – San Diego; 2017. [cited 2019 Sep 23]. Available from: http://www.escholarship.org/uc/item/88x3c8w9.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Willeford A. The Role of CaMKII delta in Pathophysiological Responses Induced by Beta-Adrenergic Receptor Stimulation and Angiotensin II. [Thesis]. University of California – San Diego; 2017. Available from: http://www.escholarship.org/uc/item/88x3c8w9

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Duke University

20. Huang, Bofu. Metabolic Control of CaMKII-mediated Caspase-2 Suppression by B55β/PP2A .

Degree: 2015, Duke University

  Apoptosis is a programmed form of cell death, essential for maintaining tissue homeostasis and eliminating dysfunctional cells. The process of apoptosis is executed by… (more)

Subjects/Keywords: Biology; Biochemistry; CaMKII; caspase-2; metabolism; phosphatase; phosphorylation

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APA (6th Edition):

Huang, B. (2015). Metabolic Control of CaMKII-mediated Caspase-2 Suppression by B55β/PP2A . (Thesis). Duke University. Retrieved from http://hdl.handle.net/10161/9833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Bofu. “Metabolic Control of CaMKII-mediated Caspase-2 Suppression by B55β/PP2A .” 2015. Thesis, Duke University. Accessed September 23, 2019. http://hdl.handle.net/10161/9833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Bofu. “Metabolic Control of CaMKII-mediated Caspase-2 Suppression by B55β/PP2A .” 2015. Web. 23 Sep 2019.

Vancouver:

Huang B. Metabolic Control of CaMKII-mediated Caspase-2 Suppression by B55β/PP2A . [Internet] [Thesis]. Duke University; 2015. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10161/9833.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang B. Metabolic Control of CaMKII-mediated Caspase-2 Suppression by B55β/PP2A . [Thesis]. Duke University; 2015. Available from: http://hdl.handle.net/10161/9833

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Manchester

21. Shen, Weijian. Development of a Biophysically Detailed Mathematical Model of a Mouse Atrial Cell for the Study of Cellular Proarrhythmic Mechanisms.

Degree: 2015, University of Manchester

 Atrial fibrillation (AF), the most common sustained arrhythmia, is associated with abnormal intracellular Ca2+ handling. Understanding AF requires comprehensive understanding of ionic currents, Ca2+ handling,… (more)

Subjects/Keywords: Mouse Atrial Model; CaMKII; β-adrenergic signalling; IP3 signalling

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APA (6th Edition):

Shen, W. (2015). Development of a Biophysically Detailed Mathematical Model of a Mouse Atrial Cell for the Study of Cellular Proarrhythmic Mechanisms. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269811

Chicago Manual of Style (16th Edition):

Shen, Weijian. “Development of a Biophysically Detailed Mathematical Model of a Mouse Atrial Cell for the Study of Cellular Proarrhythmic Mechanisms.” 2015. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269811.

MLA Handbook (7th Edition):

Shen, Weijian. “Development of a Biophysically Detailed Mathematical Model of a Mouse Atrial Cell for the Study of Cellular Proarrhythmic Mechanisms.” 2015. Web. 23 Sep 2019.

Vancouver:

Shen W. Development of a Biophysically Detailed Mathematical Model of a Mouse Atrial Cell for the Study of Cellular Proarrhythmic Mechanisms. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2019 Sep 23]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269811.

Council of Science Editors:

Shen W. Development of a Biophysically Detailed Mathematical Model of a Mouse Atrial Cell for the Study of Cellular Proarrhythmic Mechanisms. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:269811


University of Texas Medical Branch – Galveston

22. [No author]. The role of Substance P and its signaling pathways in central sensitization .

Degree: 2007, University of Texas Medical Branch – Galveston

 Central sensitization of nociceptive dorsal horn neurons is an underlying mechanism of behavioral phenotype, such as secondary hyperalgesia. Substance P (SP) is regarded as an… (more)

Subjects/Keywords: NK-1 receptor; CaMKII; PKC

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APA (6th Edition):

author], [. (2007). The role of Substance P and its signaling pathways in central sensitization . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “The role of Substance P and its signaling pathways in central sensitization .” 2007. Thesis, University of Texas Medical Branch – Galveston. Accessed September 23, 2019. http://hdl.handle.net/2152.3/65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “The role of Substance P and its signaling pathways in central sensitization .” 2007. Web. 23 Sep 2019.

Vancouver:

author] [. The role of Substance P and its signaling pathways in central sensitization . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; 2007. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/2152.3/65.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. The role of Substance P and its signaling pathways in central sensitization . [Thesis]. University of Texas Medical Branch – Galveston; 2007. Available from: http://hdl.handle.net/2152.3/65

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vrije Universiteit Amsterdam

23. Eilers, W. Sensory pathways of muscle phenotypic plasticity: Calcium signalling through CaMKII .

Degree: 2013, Vrije Universiteit Amsterdam

Subjects/Keywords: Skeletal muscle; CaMKII; Adaptation

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APA (6th Edition):

Eilers, W. (2013). Sensory pathways of muscle phenotypic plasticity: Calcium signalling through CaMKII . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/40249

Chicago Manual of Style (16th Edition):

Eilers, W. “Sensory pathways of muscle phenotypic plasticity: Calcium signalling through CaMKII .” 2013. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed September 23, 2019. http://hdl.handle.net/1871/40249.

MLA Handbook (7th Edition):

Eilers, W. “Sensory pathways of muscle phenotypic plasticity: Calcium signalling through CaMKII .” 2013. Web. 23 Sep 2019.

Vancouver:

Eilers W. Sensory pathways of muscle phenotypic plasticity: Calcium signalling through CaMKII . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2013. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1871/40249.

Council of Science Editors:

Eilers W. Sensory pathways of muscle phenotypic plasticity: Calcium signalling through CaMKII . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2013. Available from: http://hdl.handle.net/1871/40249


University of Otago

24. Ebenebe, Obialunanma. Factors Affecting The Advancement Of Atherosclerotic Plaques .

Degree: University of Otago

 Cardiovascular disease (CVD) is the leading cause of death worldwide in adult men and women. Atherosclerotic cardiovascular disease (ASCVD) has been highlighted as the main… (more)

Subjects/Keywords: atherosclerosis; CaMKII; ApoE-/-; Estradiol

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APA (6th Edition):

Ebenebe, O. (n.d.). Factors Affecting The Advancement Of Atherosclerotic Plaques . (Doctoral Dissertation). University of Otago. Retrieved from http://hdl.handle.net/10523/8227

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Chicago Manual of Style (16th Edition):

Ebenebe, Obialunanma. “Factors Affecting The Advancement Of Atherosclerotic Plaques .” Doctoral Dissertation, University of Otago. Accessed September 23, 2019. http://hdl.handle.net/10523/8227.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

MLA Handbook (7th Edition):

Ebenebe, Obialunanma. “Factors Affecting The Advancement Of Atherosclerotic Plaques .” Web. 23 Sep 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Ebenebe O. Factors Affecting The Advancement Of Atherosclerotic Plaques . [Internet] [Doctoral dissertation]. University of Otago; [cited 2019 Sep 23]. Available from: http://hdl.handle.net/10523/8227.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Council of Science Editors:

Ebenebe O. Factors Affecting The Advancement Of Atherosclerotic Plaques . [Doctoral Dissertation]. University of Otago; Available from: http://hdl.handle.net/10523/8227

Note: this citation may be lacking information needed for this citation format:
No year of publication.


Vanderbilt University

25. Gustin, Richard Michael. The role of Ca2+/calmodulin-dependent kinase II in normal and abnormal early postnatal development.

Degree: PhD, Pharmacology, 2010, Vanderbilt University

 THE ROLE OF CA2+/CALMODULIN-DEPENDENT KINASE II IN NORMAL AND ABNORMAL EARLY POSTNATAL DEVELOPMENT RICHARD GUSTIN Dissertation under the direction of Professor Roger J. Colbran Ca2+/calmodulin-dependent… (more)

Subjects/Keywords: Angelman syndrome; Early-life stress; transgenic mouse; CaMKII

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APA (6th Edition):

Gustin, R. M. (2010). The role of Ca2+/calmodulin-dependent kinase II in normal and abnormal early postnatal development. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-06282010-154107/ ;

Chicago Manual of Style (16th Edition):

Gustin, Richard Michael. “The role of Ca2+/calmodulin-dependent kinase II in normal and abnormal early postnatal development.” 2010. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu//available/etd-06282010-154107/ ;.

MLA Handbook (7th Edition):

Gustin, Richard Michael. “The role of Ca2+/calmodulin-dependent kinase II in normal and abnormal early postnatal development.” 2010. Web. 23 Sep 2019.

Vancouver:

Gustin RM. The role of Ca2+/calmodulin-dependent kinase II in normal and abnormal early postnatal development. [Internet] [Doctoral dissertation]. Vanderbilt University; 2010. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu//available/etd-06282010-154107/ ;.

Council of Science Editors:

Gustin RM. The role of Ca2+/calmodulin-dependent kinase II in normal and abnormal early postnatal development. [Doctoral Dissertation]. Vanderbilt University; 2010. Available from: http://etd.library.vanderbilt.edu//available/etd-06282010-154107/ ;


Vanderbilt University

26. Rodriguez Mena, Francisco Guillermo. Spectroscopic Study of the Conformational Changes Involved in the Activation of CaMKII.

Degree: PhD, Chemistry, 2016, Vanderbilt University

 The CaMKII enzyme is involved in diverse biological processes including control of heart rate and synaptic plasticity. CaMKII is activated by binding of calmodulin (CaM)… (more)

Subjects/Keywords: protein dynamics; CaM; calmodulin; NMR; EPR; CaMKII; conformational change

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APA (6th Edition):

Rodriguez Mena, F. G. (2016). Spectroscopic Study of the Conformational Changes Involved in the Activation of CaMKII. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu/available/etd-07192016-152059/ ;

Chicago Manual of Style (16th Edition):

Rodriguez Mena, Francisco Guillermo. “Spectroscopic Study of the Conformational Changes Involved in the Activation of CaMKII.” 2016. Doctoral Dissertation, Vanderbilt University. Accessed September 23, 2019. http://etd.library.vanderbilt.edu/available/etd-07192016-152059/ ;.

MLA Handbook (7th Edition):

Rodriguez Mena, Francisco Guillermo. “Spectroscopic Study of the Conformational Changes Involved in the Activation of CaMKII.” 2016. Web. 23 Sep 2019.

Vancouver:

Rodriguez Mena FG. Spectroscopic Study of the Conformational Changes Involved in the Activation of CaMKII. [Internet] [Doctoral dissertation]. Vanderbilt University; 2016. [cited 2019 Sep 23]. Available from: http://etd.library.vanderbilt.edu/available/etd-07192016-152059/ ;.

Council of Science Editors:

Rodriguez Mena FG. Spectroscopic Study of the Conformational Changes Involved in the Activation of CaMKII. [Doctoral Dissertation]. Vanderbilt University; 2016. Available from: http://etd.library.vanderbilt.edu/available/etd-07192016-152059/ ;


University of Newcastle

27. Abdul Majeed, A'qilah Banu binte. Effects of multi-site phosphorylation on CaMKII function.

Degree: Medical Biochemistry, 2012, University of Newcastle

Masters Research - Master of Philosophy (Medical Biochemistry)

CaMKII, or calcium/calmodulin stimulated protein kinase II, is a multifunctional Serine/Threonine protein kinase. CaMKII regulates a number… (more)

Subjects/Keywords: CaMKII; posphorylation; protein interactions; targeting; T253; T286; T305/T306

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APA (6th Edition):

Abdul Majeed, A. B. b. (2012). Effects of multi-site phosphorylation on CaMKII function. (Masters Thesis). University of Newcastle. Retrieved from http://hdl.handle.net/1959.13/936838

Chicago Manual of Style (16th Edition):

Abdul Majeed, A'qilah Banu binte. “Effects of multi-site phosphorylation on CaMKII function.” 2012. Masters Thesis, University of Newcastle. Accessed September 23, 2019. http://hdl.handle.net/1959.13/936838.

MLA Handbook (7th Edition):

Abdul Majeed, A'qilah Banu binte. “Effects of multi-site phosphorylation on CaMKII function.” 2012. Web. 23 Sep 2019.

Vancouver:

Abdul Majeed ABb. Effects of multi-site phosphorylation on CaMKII function. [Internet] [Masters thesis]. University of Newcastle; 2012. [cited 2019 Sep 23]. Available from: http://hdl.handle.net/1959.13/936838.

Council of Science Editors:

Abdul Majeed ABb. Effects of multi-site phosphorylation on CaMKII function. [Masters Thesis]. University of Newcastle; 2012. Available from: http://hdl.handle.net/1959.13/936838


University of Manchester

28. Shen, Weijian. Development of a biophysically detailed mathematical model of a mouse atrial cell for the study of cellular proarrhythmic mechanisms.

Degree: PhD, 2016, University of Manchester

 Atrial fibrillation (AF), the most common sustained arrhythmia, is associated with abnormal intracellular Ca2+ handling. Understanding AF requires comprehensive understanding of ionic currents, Ca2+ handling,… (more)

Subjects/Keywords: 612.1; IP3 signalling; ß-adrenergic signalling; Mouse Atrial Model; CaMKII

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shen, W. (2016). Development of a biophysically detailed mathematical model of a mouse atrial cell for the study of cellular proarrhythmic mechanisms. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/development-of-a-biophysically-detailed-mathematical-model-of-a-mouse-atrial-cell-for-the-study-of-cellular-proarrhythmic-mechanisms(6d03f0a2-7c2e-4cb6-a8e7-39791cbe0136).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728090

Chicago Manual of Style (16th Edition):

Shen, Weijian. “Development of a biophysically detailed mathematical model of a mouse atrial cell for the study of cellular proarrhythmic mechanisms.” 2016. Doctoral Dissertation, University of Manchester. Accessed September 23, 2019. https://www.research.manchester.ac.uk/portal/en/theses/development-of-a-biophysically-detailed-mathematical-model-of-a-mouse-atrial-cell-for-the-study-of-cellular-proarrhythmic-mechanisms(6d03f0a2-7c2e-4cb6-a8e7-39791cbe0136).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728090.

MLA Handbook (7th Edition):

Shen, Weijian. “Development of a biophysically detailed mathematical model of a mouse atrial cell for the study of cellular proarrhythmic mechanisms.” 2016. Web. 23 Sep 2019.

Vancouver:

Shen W. Development of a biophysically detailed mathematical model of a mouse atrial cell for the study of cellular proarrhythmic mechanisms. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2019 Sep 23]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/development-of-a-biophysically-detailed-mathematical-model-of-a-mouse-atrial-cell-for-the-study-of-cellular-proarrhythmic-mechanisms(6d03f0a2-7c2e-4cb6-a8e7-39791cbe0136).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728090.

Council of Science Editors:

Shen W. Development of a biophysically detailed mathematical model of a mouse atrial cell for the study of cellular proarrhythmic mechanisms. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/development-of-a-biophysically-detailed-mathematical-model-of-a-mouse-atrial-cell-for-the-study-of-cellular-proarrhythmic-mechanisms(6d03f0a2-7c2e-4cb6-a8e7-39791cbe0136).html ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.728090


The Ohio State University

29. Howard, Taylor. Investigating the mechanism underlying CaMKII-induced arrhythmias in ischemia using optical mapping.

Degree: MS, Biomedical Engineering, 2018, The Ohio State University

 The mechanisms underlying CaMKII-induced arrhythmias in ischemia/reperfusion (I/R) are not fully understood. My thesis research tested the hypothesis that CaMKII increases late Na+ current (INa,L)… (more)

Subjects/Keywords: Biomedical Engineering; CaMKII; ischemia reperfusion; late sodium current; optical mapping; arrhythmia

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APA (6th Edition):

Howard, T. (2018). Investigating the mechanism underlying CaMKII-induced arrhythmias in ischemia using optical mapping. (Masters Thesis). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1523297730669223

Chicago Manual of Style (16th Edition):

Howard, Taylor. “Investigating the mechanism underlying CaMKII-induced arrhythmias in ischemia using optical mapping.” 2018. Masters Thesis, The Ohio State University. Accessed September 23, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=osu1523297730669223.

MLA Handbook (7th Edition):

Howard, Taylor. “Investigating the mechanism underlying CaMKII-induced arrhythmias in ischemia using optical mapping.” 2018. Web. 23 Sep 2019.

Vancouver:

Howard T. Investigating the mechanism underlying CaMKII-induced arrhythmias in ischemia using optical mapping. [Internet] [Masters thesis]. The Ohio State University; 2018. [cited 2019 Sep 23]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1523297730669223.

Council of Science Editors:

Howard T. Investigating the mechanism underlying CaMKII-induced arrhythmias in ischemia using optical mapping. [Masters Thesis]. The Ohio State University; 2018. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1523297730669223


University of Iowa

30. Dallapiazza, Robert Francis. The role of CaMKII binding NMDARs in synaptic plasticity and memory.

Degree: PhD, Pharmacology, 2010, University of Iowa

  Our memories are fundamental components of who we are as individuals. They influence almost every aspect of our lives such as our decisions, our… (more)

Subjects/Keywords: CaMKII; Learning; Memory; NMDA Receptors; Synaptic plasticity; Pharmacology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dallapiazza, R. F. (2010). The role of CaMKII binding NMDARs in synaptic plasticity and memory. (Doctoral Dissertation). University of Iowa. Retrieved from https://ir.uiowa.edu/etd/483

Chicago Manual of Style (16th Edition):

Dallapiazza, Robert Francis. “The role of CaMKII binding NMDARs in synaptic plasticity and memory.” 2010. Doctoral Dissertation, University of Iowa. Accessed September 23, 2019. https://ir.uiowa.edu/etd/483.

MLA Handbook (7th Edition):

Dallapiazza, Robert Francis. “The role of CaMKII binding NMDARs in synaptic plasticity and memory.” 2010. Web. 23 Sep 2019.

Vancouver:

Dallapiazza RF. The role of CaMKII binding NMDARs in synaptic plasticity and memory. [Internet] [Doctoral dissertation]. University of Iowa; 2010. [cited 2019 Sep 23]. Available from: https://ir.uiowa.edu/etd/483.

Council of Science Editors:

Dallapiazza RF. The role of CaMKII binding NMDARs in synaptic plasticity and memory. [Doctoral Dissertation]. University of Iowa; 2010. Available from: https://ir.uiowa.edu/etd/483

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