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You searched for subject:(CXCL10). Showing records 1 – 28 of 28 total matches.

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1. Torrezani, Anna. Estudo da expressão de genes relacionados à resposta inflamatória e autoimune em lesões de líquen plano oral do tipo reticular por meio de PCR-array.

Degree: PhD, Diagnóstico Bucal, 2014, University of São Paulo

O líquen plano oral (LPO) é uma doença inflamatória crônica que afeta em torno de 1 a 2% da população mundial adulta, entre 30 e… (more)

Subjects/Keywords: Chemokine CXCL10; Chemokine CXCL9; Expressão gênica; Gene expression; Lichen planus oral; Líquen plano oral; Quimiocina CXCL10; Quimiocina CXCL9

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APA (6th Edition):

Torrezani, A. (2014). Estudo da expressão de genes relacionados à resposta inflamatória e autoimune em lesões de líquen plano oral do tipo reticular por meio de PCR-array. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/23/23139/tde-18032015-111145/ ;

Chicago Manual of Style (16th Edition):

Torrezani, Anna. “Estudo da expressão de genes relacionados à resposta inflamatória e autoimune em lesões de líquen plano oral do tipo reticular por meio de PCR-array.” 2014. Doctoral Dissertation, University of São Paulo. Accessed January 20, 2020. http://www.teses.usp.br/teses/disponiveis/23/23139/tde-18032015-111145/ ;.

MLA Handbook (7th Edition):

Torrezani, Anna. “Estudo da expressão de genes relacionados à resposta inflamatória e autoimune em lesões de líquen plano oral do tipo reticular por meio de PCR-array.” 2014. Web. 20 Jan 2020.

Vancouver:

Torrezani A. Estudo da expressão de genes relacionados à resposta inflamatória e autoimune em lesões de líquen plano oral do tipo reticular por meio de PCR-array. [Internet] [Doctoral dissertation]. University of São Paulo; 2014. [cited 2020 Jan 20]. Available from: http://www.teses.usp.br/teses/disponiveis/23/23139/tde-18032015-111145/ ;.

Council of Science Editors:

Torrezani A. Estudo da expressão de genes relacionados à resposta inflamatória e autoimune em lesões de líquen plano oral do tipo reticular por meio de PCR-array. [Doctoral Dissertation]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/23/23139/tde-18032015-111145/ ;

2. Priscila Valera Guerra. Efeito protetor de CXCL10 em camundongos BALB/C infectados com Leishmania Braziliensis.

Degree: Master, 2013, Universidade Federal do Ceará

Leishmania braziliensis à o agente etiolÃgico mais comum da leishmaniose cutÃnea no Brasil. Camundongos BALB/c sÃo os mais susceptÃveis à infecÃÃo por L. braziliensis, embora… (more)

Subjects/Keywords: DOENCAS INFECCIOSAS E PARASITARIAS; Leishmania braziliensis; Camundongos; Quimiocina CXCL10; Citocinas; Leishmania braziliensis; Mice; Chemokine CXCL10; Cytokines

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APA (6th Edition):

Guerra, P. V. (2013). Efeito protetor de CXCL10 em camundongos BALB/C infectados com Leishmania Braziliensis. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=10063 ;

Chicago Manual of Style (16th Edition):

Guerra, Priscila Valera. “Efeito protetor de CXCL10 em camundongos BALB/C infectados com Leishmania Braziliensis.” 2013. Masters Thesis, Universidade Federal do Ceará. Accessed January 20, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=10063 ;.

MLA Handbook (7th Edition):

Guerra, Priscila Valera. “Efeito protetor de CXCL10 em camundongos BALB/C infectados com Leishmania Braziliensis.” 2013. Web. 20 Jan 2020.

Vancouver:

Guerra PV. Efeito protetor de CXCL10 em camundongos BALB/C infectados com Leishmania Braziliensis. [Internet] [Masters thesis]. Universidade Federal do Ceará 2013. [cited 2020 Jan 20]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=10063 ;.

Council of Science Editors:

Guerra PV. Efeito protetor de CXCL10 em camundongos BALB/C infectados com Leishmania Braziliensis. [Masters Thesis]. Universidade Federal do Ceará 2013. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=10063 ;

3. Riani, Meriem. Rôle de la chimiokine CXCL10 dans la réaction inflammatoire associée à l'autoimmunité : exemple de la pemphigoide bulleuse : Role of the chemokine CXCL10 in the inflammatory response associated with autoimmunity : example of bullous pemphigoid.

Degree: Docteur es, Médecine - STS, 2017, Reims

La pemphigoïde bulleuse (PB) est la plus fréquente et la plus grave des dermatoses bulleuses auto-immunes caractérisée par une cascade inflammatoire impliquant plusieurs cytokines et… (more)

Subjects/Keywords: Autoimmunité; Cxcl10; Inflammation; Mmp9; Polarisation de macrophages; Composé A; Autoimmunity; Cxcl10; Inflammation; Mmp9; Macrophage polarization; Compound A; 615

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APA (6th Edition):

Riani, M. (2017). Rôle de la chimiokine CXCL10 dans la réaction inflammatoire associée à l'autoimmunité : exemple de la pemphigoide bulleuse : Role of the chemokine CXCL10 in the inflammatory response associated with autoimmunity : example of bullous pemphigoid. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2017REIMM201

Chicago Manual of Style (16th Edition):

Riani, Meriem. “Rôle de la chimiokine CXCL10 dans la réaction inflammatoire associée à l'autoimmunité : exemple de la pemphigoide bulleuse : Role of the chemokine CXCL10 in the inflammatory response associated with autoimmunity : example of bullous pemphigoid.” 2017. Doctoral Dissertation, Reims. Accessed January 20, 2020. http://www.theses.fr/2017REIMM201.

MLA Handbook (7th Edition):

Riani, Meriem. “Rôle de la chimiokine CXCL10 dans la réaction inflammatoire associée à l'autoimmunité : exemple de la pemphigoide bulleuse : Role of the chemokine CXCL10 in the inflammatory response associated with autoimmunity : example of bullous pemphigoid.” 2017. Web. 20 Jan 2020.

Vancouver:

Riani M. Rôle de la chimiokine CXCL10 dans la réaction inflammatoire associée à l'autoimmunité : exemple de la pemphigoide bulleuse : Role of the chemokine CXCL10 in the inflammatory response associated with autoimmunity : example of bullous pemphigoid. [Internet] [Doctoral dissertation]. Reims; 2017. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2017REIMM201.

Council of Science Editors:

Riani M. Rôle de la chimiokine CXCL10 dans la réaction inflammatoire associée à l'autoimmunité : exemple de la pemphigoide bulleuse : Role of the chemokine CXCL10 in the inflammatory response associated with autoimmunity : example of bullous pemphigoid. [Doctoral Dissertation]. Reims; 2017. Available from: http://www.theses.fr/2017REIMM201


Universiteit Utrecht

4. van den Borne, P. Changing blood flow in peripheral artery disease.

Degree: 2014, Universiteit Utrecht

 Cardiovascular disease (CVD) is the leading cause of death globally and it is predicted this will remain to increase throughout 2030 to an estimated 23,3… (more)

Subjects/Keywords: Arteriogenesis; inflammation; CXCL10; CD200-CD200R; TLR4; exosomes; LTB4; atherosclerosis; AAA

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APA (6th Edition):

van den Borne, P. (2014). Changing blood flow in peripheral artery disease. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/294660

Chicago Manual of Style (16th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Doctoral Dissertation, Universiteit Utrecht. Accessed January 20, 2020. http://dspace.library.uu.nl:8080/handle/1874/294660.

MLA Handbook (7th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Web. 20 Jan 2020.

Vancouver:

van den Borne P. Changing blood flow in peripheral artery disease. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2014. [cited 2020 Jan 20]. Available from: http://dspace.library.uu.nl:8080/handle/1874/294660.

Council of Science Editors:

van den Borne P. Changing blood flow in peripheral artery disease. [Doctoral Dissertation]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/294660

5. Duruisseaux, Michaël. Signature moléculaire des adénocarcinomes pulmonaires de type lépidique prédominant et mucineux invasif et dérégulation : Molecular profile of lepidic predominant adenocarcinoma and invasive mucinous adenocarcinoma and deregulation.

Degree: Docteur es, Sciences du vivant, 2015, Université Pierre et Marie Curie – Paris VI

Les adénocarcinomes lépidiques prédominant (ALP) sont une entité originale sur le plan histologique, clinique et biologique parmi les adénocarcinomes pulmonaires. Il s’agit de tumeurs non-mucineuses… (more)

Subjects/Keywords: Cancer; Poumon; Lépidique; Mucine; Cytokine; Cxcl10; Cxcr3; Nrg1; Lepidic; Mucins; 571.6

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APA (6th Edition):

Duruisseaux, M. (2015). Signature moléculaire des adénocarcinomes pulmonaires de type lépidique prédominant et mucineux invasif et dérégulation : Molecular profile of lepidic predominant adenocarcinoma and invasive mucinous adenocarcinoma and deregulation. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2015PA066277

Chicago Manual of Style (16th Edition):

Duruisseaux, Michaël. “Signature moléculaire des adénocarcinomes pulmonaires de type lépidique prédominant et mucineux invasif et dérégulation : Molecular profile of lepidic predominant adenocarcinoma and invasive mucinous adenocarcinoma and deregulation.” 2015. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed January 20, 2020. http://www.theses.fr/2015PA066277.

MLA Handbook (7th Edition):

Duruisseaux, Michaël. “Signature moléculaire des adénocarcinomes pulmonaires de type lépidique prédominant et mucineux invasif et dérégulation : Molecular profile of lepidic predominant adenocarcinoma and invasive mucinous adenocarcinoma and deregulation.” 2015. Web. 20 Jan 2020.

Vancouver:

Duruisseaux M. Signature moléculaire des adénocarcinomes pulmonaires de type lépidique prédominant et mucineux invasif et dérégulation : Molecular profile of lepidic predominant adenocarcinoma and invasive mucinous adenocarcinoma and deregulation. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2015PA066277.

Council of Science Editors:

Duruisseaux M. Signature moléculaire des adénocarcinomes pulmonaires de type lépidique prédominant et mucineux invasif et dérégulation : Molecular profile of lepidic predominant adenocarcinoma and invasive mucinous adenocarcinoma and deregulation. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2015. Available from: http://www.theses.fr/2015PA066277


Oklahoma State University

6. Saffarian Tousi, Neda. Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells.

Degree: Department of Biochemistry and Molecular Biology, 2011, Oklahoma State University

 The findings from the current study concluded a cytokine-dependent regulation of astroglial CXCL10 and iNOS expression by alpha-synuclein and neuromelanin. Alpha-synuclein induced the expression of… (more)

Subjects/Keywords: alpha-synuclein; cxcl10; inos; neuromelanin; nf-kappab; parkinson's disease

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APA (6th Edition):

Saffarian Tousi, N. (2011). Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells. (Thesis). Oklahoma State University. Retrieved from http://hdl.handle.net/11244/6674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Saffarian Tousi, Neda. “Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells.” 2011. Thesis, Oklahoma State University. Accessed January 20, 2020. http://hdl.handle.net/11244/6674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Saffarian Tousi, Neda. “Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells.” 2011. Web. 20 Jan 2020.

Vancouver:

Saffarian Tousi N. Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells. [Internet] [Thesis]. Oklahoma State University; 2011. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/11244/6674.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Saffarian Tousi N. Neuromelanin and Alpha-synuclein Modulation of Inflammatory Signaling in Human Astroglial Cells. [Thesis]. Oklahoma State University; 2011. Available from: http://hdl.handle.net/11244/6674

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

7. Raitman, Irene. Characterization of the Role of CXCL10 and CXCR3 in Breast Cancer.

Degree: 2010, University of Toronto

Lymphocytic infiltration is a feature of basal breast cancer tumors. CXCL10 is a chemokine that was found expressed higher in basal tumor RNA compared to… (more)

Subjects/Keywords: Breast Cancer; Lymphocytic Infiltration; Chemokine; Basal; CXCL10; CXCR3; 0369

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APA (6th Edition):

Raitman, I. (2010). Characterization of the Role of CXCL10 and CXCR3 in Breast Cancer. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/24279

Chicago Manual of Style (16th Edition):

Raitman, Irene. “Characterization of the Role of CXCL10 and CXCR3 in Breast Cancer.” 2010. Masters Thesis, University of Toronto. Accessed January 20, 2020. http://hdl.handle.net/1807/24279.

MLA Handbook (7th Edition):

Raitman, Irene. “Characterization of the Role of CXCL10 and CXCR3 in Breast Cancer.” 2010. Web. 20 Jan 2020.

Vancouver:

Raitman I. Characterization of the Role of CXCL10 and CXCR3 in Breast Cancer. [Internet] [Masters thesis]. University of Toronto; 2010. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1807/24279.

Council of Science Editors:

Raitman I. Characterization of the Role of CXCL10 and CXCR3 in Breast Cancer. [Masters Thesis]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/24279

8. Abdalla, Ligia Fernandes. Infecção pelo vírus Zika em uma população da Amazônia Ocidental Brasileira: estudo da resposta imune, características clínicas e de diagnóstico.

Degree: 2018, Universidade Federal do Amazonas

O Zika virus (ZIKV) é um arbovírus emergente da família Flaviviridae e do gênero Flavivirus, que até 2007 estava restrito a alguns casos de doença… (more)

Subjects/Keywords: Zika virus; RT-qPCR; CXCL10; Saliva; Fibrilação; Atrial Clínica; CIÊNCIAS BIOLÓGICAS

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APA (6th Edition):

Abdalla, L. F. (2018). Infecção pelo vírus Zika em uma população da Amazônia Ocidental Brasileira: estudo da resposta imune, características clínicas e de diagnóstico. (Doctoral Dissertation). Universidade Federal do Amazonas. Retrieved from https://tede.ufam.edu.br/handle/tede/6727

Chicago Manual of Style (16th Edition):

Abdalla, Ligia Fernandes. “Infecção pelo vírus Zika em uma população da Amazônia Ocidental Brasileira: estudo da resposta imune, características clínicas e de diagnóstico.” 2018. Doctoral Dissertation, Universidade Federal do Amazonas. Accessed January 20, 2020. https://tede.ufam.edu.br/handle/tede/6727.

MLA Handbook (7th Edition):

Abdalla, Ligia Fernandes. “Infecção pelo vírus Zika em uma população da Amazônia Ocidental Brasileira: estudo da resposta imune, características clínicas e de diagnóstico.” 2018. Web. 20 Jan 2020.

Vancouver:

Abdalla LF. Infecção pelo vírus Zika em uma população da Amazônia Ocidental Brasileira: estudo da resposta imune, características clínicas e de diagnóstico. [Internet] [Doctoral dissertation]. Universidade Federal do Amazonas; 2018. [cited 2020 Jan 20]. Available from: https://tede.ufam.edu.br/handle/tede/6727.

Council of Science Editors:

Abdalla LF. Infecção pelo vírus Zika em uma população da Amazônia Ocidental Brasileira: estudo da resposta imune, características clínicas e de diagnóstico. [Doctoral Dissertation]. Universidade Federal do Amazonas; 2018. Available from: https://tede.ufam.edu.br/handle/tede/6727


University of Gothenburg / Göteborgs Universitet

9. Askarieh, Galia. Immunological and Genetic Markers Predicting Treatment Outcome in Hepatitits C Virus Infection.

Degree: 2012, University of Gothenburg / Göteborgs Universitet

 Hepatitis C is a blood-borne infection caused by the hepatitis C virus (HCV). A chronic infection, which develops in most infected subjects, may lead to… (more)

Subjects/Keywords: IP-10; CXCL10; CD26; hepatitis C virus; interferon; ribavirin; treatment; IL28B

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APA (6th Edition):

Askarieh, G. (2012). Immunological and Genetic Markers Predicting Treatment Outcome in Hepatitits C Virus Infection. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/27818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Askarieh, Galia. “Immunological and Genetic Markers Predicting Treatment Outcome in Hepatitits C Virus Infection.” 2012. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed January 20, 2020. http://hdl.handle.net/2077/27818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Askarieh, Galia. “Immunological and Genetic Markers Predicting Treatment Outcome in Hepatitits C Virus Infection.” 2012. Web. 20 Jan 2020.

Vancouver:

Askarieh G. Immunological and Genetic Markers Predicting Treatment Outcome in Hepatitits C Virus Infection. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2012. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/2077/27818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Askarieh G. Immunological and Genetic Markers Predicting Treatment Outcome in Hepatitits C Virus Infection. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2012. Available from: http://hdl.handle.net/2077/27818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Brunheld Maia Dutra. Effect immunomodulator of CXCL10 in mice BALB/C infected with Leishmania braziliensis refractory to antimony.

Degree: Master, 2015, Universidade Federal do Ceará

Leishmania braziliensis from antimony-resistant patient is able to induce high levels of IL-4 and Arginase in mice, contributing to the increased virulence of the strain… (more)

Subjects/Keywords: ANATOMIA PATOLOGICA E PATOLOGIA CLINICA; Leishmania braziliensis; ResistÃncia a Medicamentos; Camundongos EndogÃmicos BALB C; Quimiocina CXCL10; Leishmania braziliensis; Drug Resistance; Inbred BALB C Mice; Chemokine CXCL10.

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APA (6th Edition):

Dutra, B. M. (2015). Effect immunomodulator of CXCL10 in mice BALB/C infected with Leishmania braziliensis refractory to antimony. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15643 ;

Chicago Manual of Style (16th Edition):

Dutra, Brunheld Maia. “Effect immunomodulator of CXCL10 in mice BALB/C infected with Leishmania braziliensis refractory to antimony.” 2015. Masters Thesis, Universidade Federal do Ceará. Accessed January 20, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15643 ;.

MLA Handbook (7th Edition):

Dutra, Brunheld Maia. “Effect immunomodulator of CXCL10 in mice BALB/C infected with Leishmania braziliensis refractory to antimony.” 2015. Web. 20 Jan 2020.

Vancouver:

Dutra BM. Effect immunomodulator of CXCL10 in mice BALB/C infected with Leishmania braziliensis refractory to antimony. [Internet] [Masters thesis]. Universidade Federal do Ceará 2015. [cited 2020 Jan 20]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15643 ;.

Council of Science Editors:

Dutra BM. Effect immunomodulator of CXCL10 in mice BALB/C infected with Leishmania braziliensis refractory to antimony. [Masters Thesis]. Universidade Federal do Ceará 2015. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15643 ;


University of Kansas

11. Williams, Rachel. HIV-Encephalitis: Mechanisms for CXCL10 Induction in Astrocytes.

Degree: PhD, Molecular & Integrative Physiology, 2009, University of Kansas

 With the prevalence of HIV-associated neurocognitive disorders (HAND) increasing, understanding the mechanisms by which HIV induces neuro-inflammation and subsequent neuronal damage is of paramount importance.… (more)

Subjects/Keywords: Biology; Neurosciences; Molecular biology; Microbiology; Astrocytes; Cxcl10; Hiv; Hiv-associated dementia; Ifn-gamma; Tnf-alpha

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APA (6th Edition):

Williams, R. (2009). HIV-Encephalitis: Mechanisms for CXCL10 Induction in Astrocytes. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/5577

Chicago Manual of Style (16th Edition):

Williams, Rachel. “HIV-Encephalitis: Mechanisms for CXCL10 Induction in Astrocytes.” 2009. Doctoral Dissertation, University of Kansas. Accessed January 20, 2020. http://hdl.handle.net/1808/5577.

MLA Handbook (7th Edition):

Williams, Rachel. “HIV-Encephalitis: Mechanisms for CXCL10 Induction in Astrocytes.” 2009. Web. 20 Jan 2020.

Vancouver:

Williams R. HIV-Encephalitis: Mechanisms for CXCL10 Induction in Astrocytes. [Internet] [Doctoral dissertation]. University of Kansas; 2009. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1808/5577.

Council of Science Editors:

Williams R. HIV-Encephalitis: Mechanisms for CXCL10 Induction in Astrocytes. [Doctoral Dissertation]. University of Kansas; 2009. Available from: http://hdl.handle.net/1808/5577


University of Western Australia

12. Oliver, Benjamin Gregory. Biomarkers of immune restoration disease in HIV patients after they commence antiretroviral therapy.

Degree: PhD, 2011, University of Western Australia

[Truncated abstract] Initiation of antiretroviral therapy (ART) in HIV patients may result in the restoration of pathogen-specific immune responses which cause immunopathology. This clinical phenomenon… (more)

Subjects/Keywords: HIV; TB-IRIS; ART-TB; Biomarkers; Tuberulosis; Antiretroviral therapy; Immune restoration disease; CXCL10

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APA (6th Edition):

Oliver, B. G. (2011). Biomarkers of immune restoration disease in HIV patients after they commence antiretroviral therapy. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33231&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Oliver, Benjamin Gregory. “Biomarkers of immune restoration disease in HIV patients after they commence antiretroviral therapy.” 2011. Doctoral Dissertation, University of Western Australia. Accessed January 20, 2020. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33231&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Oliver, Benjamin Gregory. “Biomarkers of immune restoration disease in HIV patients after they commence antiretroviral therapy.” 2011. Web. 20 Jan 2020.

Vancouver:

Oliver BG. Biomarkers of immune restoration disease in HIV patients after they commence antiretroviral therapy. [Internet] [Doctoral dissertation]. University of Western Australia; 2011. [cited 2020 Jan 20]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33231&local_base=GEN01-INS01.

Council of Science Editors:

Oliver BG. Biomarkers of immune restoration disease in HIV patients after they commence antiretroviral therapy. [Doctoral Dissertation]. University of Western Australia; 2011. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=33231&local_base=GEN01-INS01

13. Lupieri, Adrien. Fonctions catalytique et non-catalytique de la PI3Ky dans la réponse artérielle aux stress : Catalytic and non-catalytic function of PI3Kgamma following arterial stress.

Degree: Docteur es, Physiopathologie, 2017, Université Toulouse III – Paul Sabatier

 La physiopathologie artérielle comprend différentes pathologies telles que l'hypertension, l'athérosclérose, l'hyperplasie intimale qui sont caractérisées par un remodelage artériel en réponse à différents stress environnementaux.… (more)

Subjects/Keywords: Phosphoinositide 3-kinase gamma; Remodelage; Cellules musculaires lisses; Cellules endothéliales; Lymphocytes; CXCL10; AMPc

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APA (6th Edition):

Lupieri, A. (2017). Fonctions catalytique et non-catalytique de la PI3Ky dans la réponse artérielle aux stress : Catalytic and non-catalytic function of PI3Kgamma following arterial stress. (Doctoral Dissertation). Université Toulouse III – Paul Sabatier. Retrieved from http://www.theses.fr/2017TOU30095

Chicago Manual of Style (16th Edition):

Lupieri, Adrien. “Fonctions catalytique et non-catalytique de la PI3Ky dans la réponse artérielle aux stress : Catalytic and non-catalytic function of PI3Kgamma following arterial stress.” 2017. Doctoral Dissertation, Université Toulouse III – Paul Sabatier. Accessed January 20, 2020. http://www.theses.fr/2017TOU30095.

MLA Handbook (7th Edition):

Lupieri, Adrien. “Fonctions catalytique et non-catalytique de la PI3Ky dans la réponse artérielle aux stress : Catalytic and non-catalytic function of PI3Kgamma following arterial stress.” 2017. Web. 20 Jan 2020.

Vancouver:

Lupieri A. Fonctions catalytique et non-catalytique de la PI3Ky dans la réponse artérielle aux stress : Catalytic and non-catalytic function of PI3Kgamma following arterial stress. [Internet] [Doctoral dissertation]. Université Toulouse III – Paul Sabatier; 2017. [cited 2020 Jan 20]. Available from: http://www.theses.fr/2017TOU30095.

Council of Science Editors:

Lupieri A. Fonctions catalytique et non-catalytique de la PI3Ky dans la réponse artérielle aux stress : Catalytic and non-catalytic function of PI3Kgamma following arterial stress. [Doctoral Dissertation]. Université Toulouse III – Paul Sabatier; 2017. Available from: http://www.theses.fr/2017TOU30095


Queens University

14. Au, Katrina. Role of STAT1 and CXCL10 in the Tumour Immune Microenvironment of High-Grade Serous Ovarian Cancer .

Degree: Biomedical and Molecular Sciences, 2016, Queens University

 High-grade serous ovarian cancer (HGSC) is the most prevalent epithelial ovarian cancer characterized by late detection, metastasis and resistance to chemotherapy. Previous studies on the… (more)

Subjects/Keywords: CXCL10; CD8+ T Cells; Cancer; STAT1; Tumour Immune Microenvironment; Biomarkers; High-Grade Serous Ovarian Cancer

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APA (6th Edition):

Au, K. (2016). Role of STAT1 and CXCL10 in the Tumour Immune Microenvironment of High-Grade Serous Ovarian Cancer . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/14676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Au, Katrina. “Role of STAT1 and CXCL10 in the Tumour Immune Microenvironment of High-Grade Serous Ovarian Cancer .” 2016. Thesis, Queens University. Accessed January 20, 2020. http://hdl.handle.net/1974/14676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Au, Katrina. “Role of STAT1 and CXCL10 in the Tumour Immune Microenvironment of High-Grade Serous Ovarian Cancer .” 2016. Web. 20 Jan 2020.

Vancouver:

Au K. Role of STAT1 and CXCL10 in the Tumour Immune Microenvironment of High-Grade Serous Ovarian Cancer . [Internet] [Thesis]. Queens University; 2016. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1974/14676.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Au K. Role of STAT1 and CXCL10 in the Tumour Immune Microenvironment of High-Grade Serous Ovarian Cancer . [Thesis]. Queens University; 2016. Available from: http://hdl.handle.net/1974/14676

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Georgia

15. Chai, Qingqing. Mechanism of BBB permeability enhancement in rabies virus infection.

Degree: PhD, Veterinary and Biomedical Sciences, 2014, University of Georgia

 Infection with laboratory-attenuated rabies virus (RABV) enhances Blood-brain Barrier (BBB) permeability, which has been demonstrated to be an important factor for host survival, since it… (more)

Subjects/Keywords: Rabies virus (RABV); Blood-brain Barrier (BBB); Tight junction (TJ) proteins; Chemokine; Cytokine; IFN-γ; CXCL10

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APA (6th Edition):

Chai, Q. (2014). Mechanism of BBB permeability enhancement in rabies virus infection. (Doctoral Dissertation). University of Georgia. Retrieved from http://purl.galileo.usg.edu/uga_etd/chai_qingqing_201408_phd

Chicago Manual of Style (16th Edition):

Chai, Qingqing. “Mechanism of BBB permeability enhancement in rabies virus infection.” 2014. Doctoral Dissertation, University of Georgia. Accessed January 20, 2020. http://purl.galileo.usg.edu/uga_etd/chai_qingqing_201408_phd.

MLA Handbook (7th Edition):

Chai, Qingqing. “Mechanism of BBB permeability enhancement in rabies virus infection.” 2014. Web. 20 Jan 2020.

Vancouver:

Chai Q. Mechanism of BBB permeability enhancement in rabies virus infection. [Internet] [Doctoral dissertation]. University of Georgia; 2014. [cited 2020 Jan 20]. Available from: http://purl.galileo.usg.edu/uga_etd/chai_qingqing_201408_phd.

Council of Science Editors:

Chai Q. Mechanism of BBB permeability enhancement in rabies virus infection. [Doctoral Dissertation]. University of Georgia; 2014. Available from: http://purl.galileo.usg.edu/uga_etd/chai_qingqing_201408_phd

16. TAN ANTHONY TANOTO. Immunodominance and immunoprotection of anti-viral specific CD8+ T cell response during HBV infection.

Degree: 2011, National University of Singapore

Subjects/Keywords: HBV; T cell; HLA; Hepatic flares; CXCL9; CXCL10

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APA (6th Edition):

TANOTO, T. A. (2011). Immunodominance and immunoprotection of anti-viral specific CD8+ T cell response during HBV infection. (Thesis). National University of Singapore. Retrieved from http://scholarbank.nus.edu.sg/handle/10635/30704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

TANOTO, TAN ANTHONY. “Immunodominance and immunoprotection of anti-viral specific CD8+ T cell response during HBV infection.” 2011. Thesis, National University of Singapore. Accessed January 20, 2020. http://scholarbank.nus.edu.sg/handle/10635/30704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

TANOTO, TAN ANTHONY. “Immunodominance and immunoprotection of anti-viral specific CD8+ T cell response during HBV infection.” 2011. Web. 20 Jan 2020.

Vancouver:

TANOTO TA. Immunodominance and immunoprotection of anti-viral specific CD8+ T cell response during HBV infection. [Internet] [Thesis]. National University of Singapore; 2011. [cited 2020 Jan 20]. Available from: http://scholarbank.nus.edu.sg/handle/10635/30704.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

TANOTO TA. Immunodominance and immunoprotection of anti-viral specific CD8+ T cell response during HBV infection. [Thesis]. National University of Singapore; 2011. Available from: http://scholarbank.nus.edu.sg/handle/10635/30704

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Louisville

17. Chheda, Zinal S. Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors.

Degree: PhD, 2015, University of Louisville

  Presence of increased numbers of CD8+ T cells in the tumors correspond to better overall survival in the patients. Variety of immuno-therapies have shown… (more)

Subjects/Keywords: anti-tumor immunity; melanoma; CTL infiltration to tumors; PD-1 blockade; LTB4; CXCL9 and CXCL10; Immunity

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APA (6th Edition):

Chheda, Z. S. (2015). Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294

Chicago Manual of Style (16th Edition):

Chheda, Zinal S. “Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors.” 2015. Doctoral Dissertation, University of Louisville. Accessed January 20, 2020. 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294.

MLA Handbook (7th Edition):

Chheda, Zinal S. “Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors.” 2015. Web. 20 Jan 2020.

Vancouver:

Chheda ZS. Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors. [Internet] [Doctoral dissertation]. University of Louisville; 2015. [cited 2020 Jan 20]. Available from: 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294.

Council of Science Editors:

Chheda ZS. Chemoattractant receptors BLT1 and CXCR3 regulate anti-tumor immunity by facilitating CD8+ T cell migration to tumors. [Doctoral Dissertation]. University of Louisville; 2015. Available from: 10.18297/etd/2294 ; https://ir.library.louisville.edu/etd/2294

18. Webertty Mayk EufrÃsio de Figueiredo. Efeito da Quimiocina CXCL10 na infecÃÃo por Leishmania infantum/chagasi em camundongos BALB/C.

Degree: Master, 2012, Universidade Federal do Ceará

A leishmaniose visceral causada por Leishmania infantum chagasi à caracterizada pela perda da habilidade do hospedeiro gerar uma resposta imunolÃgica eficaz. Neste estudo, foi investigado… (more)

Subjects/Keywords: DOENCAS INFECCIOSAS E PARASITARIAS; L. infantum chagasi; Quimiocina CXCL10; IFN-;

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APA (6th Edition):

Figueiredo, W. M. E. d. (2012). Efeito da Quimiocina CXCL10 na infecÃÃo por Leishmania infantum/chagasi em camundongos BALB/C. (Masters Thesis). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8565 ;

Chicago Manual of Style (16th Edition):

Figueiredo, Webertty Mayk EufrÃsio de. “Efeito da Quimiocina CXCL10 na infecÃÃo por Leishmania infantum/chagasi em camundongos BALB/C.” 2012. Masters Thesis, Universidade Federal do Ceará. Accessed January 20, 2020. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8565 ;.

MLA Handbook (7th Edition):

Figueiredo, Webertty Mayk EufrÃsio de. “Efeito da Quimiocina CXCL10 na infecÃÃo por Leishmania infantum/chagasi em camundongos BALB/C.” 2012. Web. 20 Jan 2020.

Vancouver:

Figueiredo WMEd. Efeito da Quimiocina CXCL10 na infecÃÃo por Leishmania infantum/chagasi em camundongos BALB/C. [Internet] [Masters thesis]. Universidade Federal do Ceará 2012. [cited 2020 Jan 20]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8565 ;.

Council of Science Editors:

Figueiredo WMEd. Efeito da Quimiocina CXCL10 na infecÃÃo por Leishmania infantum/chagasi em camundongos BALB/C. [Masters Thesis]. Universidade Federal do Ceará 2012. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=8565 ;


Universidade do Estado do Rio de Janeiro

19. Raquel da Silva Corrêa. Avaliação de marcadores biológicos com potencial para detecção da evolução para doença em tuberculose.

Degree: Master, 2012, Universidade do Estado do Rio de Janeiro

 A tuberculose (TB) é uma doença infecto-contagiosa obtida a partir da inalação de aerossóis contendo seu agente etiológico, o Mycobacterium tuberculosis. A TB acomete principalmente… (more)

Subjects/Keywords: Inflamação; Mycobacterium tuberculosis; Tuberculose ativa; Tuberculose latente; IFN- γ; CXCL10; Biomarcadores; Mycobacterium tuberculosis; Inflammation; Active tuberculosis; Latent tuberculosis; IFN-γ; CXCL10; Biomarkers; IMUNOLOGIA CELULAR; Mycobacterium tuberculosis Teses; Tuberculose Teses; Inflamação Teses

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APA (6th Edition):

Corrêa, R. d. S. (2012). Avaliação de marcadores biológicos com potencial para detecção da evolução para doença em tuberculose. (Masters Thesis). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8116 ;

Chicago Manual of Style (16th Edition):

Corrêa, Raquel da Silva. “Avaliação de marcadores biológicos com potencial para detecção da evolução para doença em tuberculose.” 2012. Masters Thesis, Universidade do Estado do Rio de Janeiro. Accessed January 20, 2020. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8116 ;.

MLA Handbook (7th Edition):

Corrêa, Raquel da Silva. “Avaliação de marcadores biológicos com potencial para detecção da evolução para doença em tuberculose.” 2012. Web. 20 Jan 2020.

Vancouver:

Corrêa RdS. Avaliação de marcadores biológicos com potencial para detecção da evolução para doença em tuberculose. [Internet] [Masters thesis]. Universidade do Estado do Rio de Janeiro; 2012. [cited 2020 Jan 20]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8116 ;.

Council of Science Editors:

Corrêa RdS. Avaliação de marcadores biológicos com potencial para detecção da evolução para doença em tuberculose. [Masters Thesis]. Universidade do Estado do Rio de Janeiro; 2012. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=8116 ;


Université de Montréal

20. Girard, Mélanie. Dégradation de CXCL11 et CXCL10 par les lymphocytes T activés .

Degree: 2017, Université de Montréal

 Suite à leur activation par des cellules présentatrices d’antigène, les lymphocytes T expriment le récepteur de chimiokines CXCR3 et peuvent alors infiltrer les tissus enflammés.… (more)

Subjects/Keywords: CXCR3; Lymphocytes T; Chimiokines; β-arrestine; chimiotaxie; Gradients de chimiokines; CXCL11; CXCL10; ACKR; CXCR3A; CXCR3B; β-arrestin; Chemotaxis; Chemokine gradient; Chemokine degradation; Self-generated gradients

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APA (6th Edition):

Girard, M. (2017). Dégradation de CXCL11 et CXCL10 par les lymphocytes T activés . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/19417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Girard, Mélanie. “Dégradation de CXCL11 et CXCL10 par les lymphocytes T activés .” 2017. Thesis, Université de Montréal. Accessed January 20, 2020. http://hdl.handle.net/1866/19417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Girard, Mélanie. “Dégradation de CXCL11 et CXCL10 par les lymphocytes T activés .” 2017. Web. 20 Jan 2020.

Vancouver:

Girard M. Dégradation de CXCL11 et CXCL10 par les lymphocytes T activés . [Internet] [Thesis]. Université de Montréal; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1866/19417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Girard M. Dégradation de CXCL11 et CXCL10 par les lymphocytes T activés . [Thesis]. Université de Montréal; 2017. Available from: http://hdl.handle.net/1866/19417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

21. Ramirez, Lorenzo Antonio. Enhancing T-Cell Responses to Vaccination of HIV-1 infected Subjects on Antiretroviral Therapy.

Degree: 2014, University of Pennsylvania

 With the advancement in anti-retroviral therapy (ART) regimens there has been a significant improvement in the quality of life and survival of those individuals infected… (more)

Subjects/Keywords: Antiretroviral Therapy; chemokine; CXCL10; HIV-1; IP-10; T-cell; Allergy and Immunology; Immunology and Infectious Disease; Medical Immunology; Medicine and Health Sciences; Virology

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APA (6th Edition):

Ramirez, L. A. (2014). Enhancing T-Cell Responses to Vaccination of HIV-1 infected Subjects on Antiretroviral Therapy. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ramirez, Lorenzo Antonio. “Enhancing T-Cell Responses to Vaccination of HIV-1 infected Subjects on Antiretroviral Therapy.” 2014. Thesis, University of Pennsylvania. Accessed January 20, 2020. https://repository.upenn.edu/edissertations/1413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ramirez, Lorenzo Antonio. “Enhancing T-Cell Responses to Vaccination of HIV-1 infected Subjects on Antiretroviral Therapy.” 2014. Web. 20 Jan 2020.

Vancouver:

Ramirez LA. Enhancing T-Cell Responses to Vaccination of HIV-1 infected Subjects on Antiretroviral Therapy. [Internet] [Thesis]. University of Pennsylvania; 2014. [cited 2020 Jan 20]. Available from: https://repository.upenn.edu/edissertations/1413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ramirez LA. Enhancing T-Cell Responses to Vaccination of HIV-1 infected Subjects on Antiretroviral Therapy. [Thesis]. University of Pennsylvania; 2014. Available from: https://repository.upenn.edu/edissertations/1413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. van den Borne, P. Changing blood flow in peripheral artery disease.

Degree: 2014, University Utrecht

 Cardiovascular disease (CVD) is the leading cause of death globally and it is predicted this will remain to increase throughout 2030 to an estimated 23,3… (more)

Subjects/Keywords: Arteriogenesis; inflammation; CXCL10; CD200-CD200R; TLR4; exosomes; LTB4; atherosclerosis; AAA

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APA (6th Edition):

van den Borne, P. (2014). Changing blood flow in peripheral artery disease. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660

Chicago Manual of Style (16th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Doctoral Dissertation, University Utrecht. Accessed January 20, 2020. http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660.

MLA Handbook (7th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Web. 20 Jan 2020.

Vancouver:

van den Borne P. Changing blood flow in peripheral artery disease. [Internet] [Doctoral dissertation]. University Utrecht; 2014. [cited 2020 Jan 20]. Available from: http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660.

Council of Science Editors:

van den Borne P. Changing blood flow in peripheral artery disease. [Doctoral Dissertation]. University Utrecht; 2014. Available from: http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660

23. van den Borne, P. Changing blood flow in peripheral artery disease.

Degree: 2014, University Utrecht

 Cardiovascular disease (CVD) is the leading cause of death globally and it is predicted this will remain to increase throughout 2030 to an estimated 23,3… (more)

Subjects/Keywords: Arteriogenesis; inflammation; CXCL10; CD200-CD200R; TLR4; exosomes; LTB4; atherosclerosis; AAA

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APA (6th Edition):

van den Borne, P. (2014). Changing blood flow in peripheral artery disease. (Doctoral Dissertation). University Utrecht. Retrieved from http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660

Chicago Manual of Style (16th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Doctoral Dissertation, University Utrecht. Accessed January 20, 2020. http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660.

MLA Handbook (7th Edition):

van den Borne, P. “Changing blood flow in peripheral artery disease.” 2014. Web. 20 Jan 2020.

Vancouver:

van den Borne P. Changing blood flow in peripheral artery disease. [Internet] [Doctoral dissertation]. University Utrecht; 2014. [cited 2020 Jan 20]. Available from: http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660.

Council of Science Editors:

van den Borne P. Changing blood flow in peripheral artery disease. [Doctoral Dissertation]. University Utrecht; 2014. Available from: http://dspace.library.uu.nl/handle/1874/294660 ; URN:NBN:NL:UI:10-1874-294660 ; urn:isbn:978-90-393-6157-3 ; URN:NBN:NL:UI:10-1874-294660 ; http://dspace.library.uu.nl/handle/1874/294660


Universidade do Estado do Rio de Janeiro

24. Mildred Ferreira Medeiros. Hanseníase neural, aspectos diagnósticos da forma neural pura e mecanismos imunopatogênicos da lesão do nervo na doença. Participação de quimiocinas CCL2 e CXCL10 e metaloproteinases 2 e 9.

Degree: PhD, 2014, Universidade do Estado do Rio de Janeiro

 O diagnóstico da hanseníase neural pura baseia-se em dados clínicos e laboratoriais do paciente, incluindo a histopatologia de espécimes de biópsia de nervo e detecção… (more)

Subjects/Keywords: ANATOMIA PATOLOGICA E PATOLOGIA CLINICA; Hanseníase; Mycobacterium leprae; Neuropatia; Imuno-histoquímica; Metaloproteinases de matriz extracelular (MMP); Quimiocinas; CXCL10; CCL2; Células inflamatórias; Lipoarabinomanana (LAM); Ácido fenólico glicolipídico-1. (PGL1). Hanseníase neural pura (HNP); Nervo periférico. Diagnóstico; Leprosy; Mycobacterium leprae; Neuropathy; Immunohistochemistry; Matrix metalloproteinase (MMP); Chemokines; CXCL10; CCL2; Inflammatory cells; Lipoarabinomannan (LAM); Phenolic glycolipid (PGL1); Pure neural leprosy; Peripheral nerve; Diagnosis; Hanseníase - Teses; Mycobacteriam leprae - Teses; Neuropatia - Teses; Metaloproteinases da Matriz

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APA (6th Edition):

Medeiros, M. F. (2014). Hanseníase neural, aspectos diagnósticos da forma neural pura e mecanismos imunopatogênicos da lesão do nervo na doença. Participação de quimiocinas CCL2 e CXCL10 e metaloproteinases 2 e 9. (Doctoral Dissertation). Universidade do Estado do Rio de Janeiro. Retrieved from http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7356 ;

Chicago Manual of Style (16th Edition):

Medeiros, Mildred Ferreira. “Hanseníase neural, aspectos diagnósticos da forma neural pura e mecanismos imunopatogênicos da lesão do nervo na doença. Participação de quimiocinas CCL2 e CXCL10 e metaloproteinases 2 e 9.” 2014. Doctoral Dissertation, Universidade do Estado do Rio de Janeiro. Accessed January 20, 2020. http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7356 ;.

MLA Handbook (7th Edition):

Medeiros, Mildred Ferreira. “Hanseníase neural, aspectos diagnósticos da forma neural pura e mecanismos imunopatogênicos da lesão do nervo na doença. Participação de quimiocinas CCL2 e CXCL10 e metaloproteinases 2 e 9.” 2014. Web. 20 Jan 2020.

Vancouver:

Medeiros MF. Hanseníase neural, aspectos diagnósticos da forma neural pura e mecanismos imunopatogênicos da lesão do nervo na doença. Participação de quimiocinas CCL2 e CXCL10 e metaloproteinases 2 e 9. [Internet] [Doctoral dissertation]. Universidade do Estado do Rio de Janeiro; 2014. [cited 2020 Jan 20]. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7356 ;.

Council of Science Editors:

Medeiros MF. Hanseníase neural, aspectos diagnósticos da forma neural pura e mecanismos imunopatogênicos da lesão do nervo na doença. Participação de quimiocinas CCL2 e CXCL10 e metaloproteinases 2 e 9. [Doctoral Dissertation]. Universidade do Estado do Rio de Janeiro; 2014. Available from: http://www.bdtd.uerj.br/tde_busca/arquivo.php?codArquivo=7356 ;


University of Florida

25. Dominguez, Paul. STAT1 Overexpression in Systemic Lupus Erythematosus Correlates with Enhanced Biomarker Expression, Therapy Resistance, and Occurrence of Anemia.

Degree: PhD, Medical Sciences - Molecular Cell Biology (IDP), 2013, University of Florida

Subjects/Keywords: Anemia; Arthritis; Biological markers; Chemokines; Diseases; Interferons; Lupus; MicroRNAs; RNA; Systemic lupus erythematosus; ccl2; cxcl10; mir146a; sle; sledai; stat1

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APA (6th Edition):

Dominguez, P. (2013). STAT1 Overexpression in Systemic Lupus Erythematosus Correlates with Enhanced Biomarker Expression, Therapy Resistance, and Occurrence of Anemia. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0044908

Chicago Manual of Style (16th Edition):

Dominguez, Paul. “STAT1 Overexpression in Systemic Lupus Erythematosus Correlates with Enhanced Biomarker Expression, Therapy Resistance, and Occurrence of Anemia.” 2013. Doctoral Dissertation, University of Florida. Accessed January 20, 2020. http://ufdc.ufl.edu/UFE0044908.

MLA Handbook (7th Edition):

Dominguez, Paul. “STAT1 Overexpression in Systemic Lupus Erythematosus Correlates with Enhanced Biomarker Expression, Therapy Resistance, and Occurrence of Anemia.” 2013. Web. 20 Jan 2020.

Vancouver:

Dominguez P. STAT1 Overexpression in Systemic Lupus Erythematosus Correlates with Enhanced Biomarker Expression, Therapy Resistance, and Occurrence of Anemia. [Internet] [Doctoral dissertation]. University of Florida; 2013. [cited 2020 Jan 20]. Available from: http://ufdc.ufl.edu/UFE0044908.

Council of Science Editors:

Dominguez P. STAT1 Overexpression in Systemic Lupus Erythematosus Correlates with Enhanced Biomarker Expression, Therapy Resistance, and Occurrence of Anemia. [Doctoral Dissertation]. University of Florida; 2013. Available from: http://ufdc.ufl.edu/UFE0044908

26. Ouwendijk, Werner. Dynamic Interplay between Varicelloviruses and their Primate Hosts: Dynamische Wisselwerking tussen Varicellovirussen en hun Primaten Gastheer.

Degree: 2013, Erasmus University Medical Center

 markdownabstract__Abstract__ Varicella-zoster virus (VZV) is a ubiquitous human alphaherpesvirus (αHHV). Most individuals become infected with VZV during childhood typically resulting in generalized vesicular skin rash,… (more)

Subjects/Keywords: Varicella-zoster virus; Simian varicella virus; Herpes simplex virus; Varicella; Herpes zoster; latency; T-cell immunity; Chinese rhesus macaques; oral shedding; CXCL10

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APA (6th Edition):

Ouwendijk, W. (2013). Dynamic Interplay between Varicelloviruses and their Primate Hosts: Dynamische Wisselwerking tussen Varicellovirussen en hun Primaten Gastheer. (Doctoral Dissertation). Erasmus University Medical Center. Retrieved from http://hdl.handle.net/1765/50152

Chicago Manual of Style (16th Edition):

Ouwendijk, Werner. “Dynamic Interplay between Varicelloviruses and their Primate Hosts: Dynamische Wisselwerking tussen Varicellovirussen en hun Primaten Gastheer.” 2013. Doctoral Dissertation, Erasmus University Medical Center. Accessed January 20, 2020. http://hdl.handle.net/1765/50152.

MLA Handbook (7th Edition):

Ouwendijk, Werner. “Dynamic Interplay between Varicelloviruses and their Primate Hosts: Dynamische Wisselwerking tussen Varicellovirussen en hun Primaten Gastheer.” 2013. Web. 20 Jan 2020.

Vancouver:

Ouwendijk W. Dynamic Interplay between Varicelloviruses and their Primate Hosts: Dynamische Wisselwerking tussen Varicellovirussen en hun Primaten Gastheer. [Internet] [Doctoral dissertation]. Erasmus University Medical Center; 2013. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1765/50152.

Council of Science Editors:

Ouwendijk W. Dynamic Interplay between Varicelloviruses and their Primate Hosts: Dynamische Wisselwerking tussen Varicellovirussen en hun Primaten Gastheer. [Doctoral Dissertation]. Erasmus University Medical Center; 2013. Available from: http://hdl.handle.net/1765/50152

27. Harris, Daniel Pellerin. PRMT5-CATALYZED ARGININE METHYLATION OF NF-kappaB p65 INTHE ENDOTHELIAL CELL INDUCTION OF PRO-INFLAMMATORYCHEMOKINES.

Degree: PhD, Physiology and Biophysics, 2016, Case Western Reserve University

 Inflammatory agonists differentially activate gene induction of chemokinesin endothelial cells (EC). The molecular mechanisms that produce distinctchemokine induction profiles following EC activation are incompletelyunderstood. The… (more)

Subjects/Keywords: Biochemistry; Molecular Biology; Physiology; Cellular Biology; PRMT5; arginine methylation; SDMA; NF-kappaB; p76; post-translational modification; endothelial cell; inflammation; CXCL10; IP-10; CXCL11; I-TAC; TNF-a; IFNg; transcription

…4.1 PRMT5 is necessary for the induction of CXCL10 and CX3CL1 in TNF-stimulated EC… …61 FIGURE 4.2 Knockdown of PRMT5 reduces CXCL10 transcription. .................. 63… …FIGURE 4.3 PRMT5 activity leads to the association of SDMA-containing proteins with the CXCL10… …p65 association with the CXCL10 promoter… …77 FIGURE 4.7 SDMA-p65 is critical for CXCL10 induction… 

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APA (6th Edition):

Harris, D. P. (2016). PRMT5-CATALYZED ARGININE METHYLATION OF NF-kappaB p65 INTHE ENDOTHELIAL CELL INDUCTION OF PRO-INFLAMMATORYCHEMOKINES. (Doctoral Dissertation). Case Western Reserve University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1449579234

Chicago Manual of Style (16th Edition):

Harris, Daniel Pellerin. “PRMT5-CATALYZED ARGININE METHYLATION OF NF-kappaB p65 INTHE ENDOTHELIAL CELL INDUCTION OF PRO-INFLAMMATORYCHEMOKINES.” 2016. Doctoral Dissertation, Case Western Reserve University. Accessed January 20, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1449579234.

MLA Handbook (7th Edition):

Harris, Daniel Pellerin. “PRMT5-CATALYZED ARGININE METHYLATION OF NF-kappaB p65 INTHE ENDOTHELIAL CELL INDUCTION OF PRO-INFLAMMATORYCHEMOKINES.” 2016. Web. 20 Jan 2020.

Vancouver:

Harris DP. PRMT5-CATALYZED ARGININE METHYLATION OF NF-kappaB p65 INTHE ENDOTHELIAL CELL INDUCTION OF PRO-INFLAMMATORYCHEMOKINES. [Internet] [Doctoral dissertation]. Case Western Reserve University; 2016. [cited 2020 Jan 20]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1449579234.

Council of Science Editors:

Harris DP. PRMT5-CATALYZED ARGININE METHYLATION OF NF-kappaB p65 INTHE ENDOTHELIAL CELL INDUCTION OF PRO-INFLAMMATORYCHEMOKINES. [Doctoral Dissertation]. Case Western Reserve University; 2016. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1449579234


Erasmus University Rotterdam

28. Polak, Roel. Molecular Drivers of Pediatric Acute Lymphoblastic Leukemia : Toward targeted therapy for children with ALL.

Degree: 2017, Erasmus University Rotterdam

textabstractThis thesis aims to identify leukemia-specific processes that can be used to target leukemic cells or the leukemic microenvironment.

Subjects/Keywords: Pediatric; Acute lymphoblastic leukemia; ALL; B-ALL; BCP-ALL; Targeted therapy; Selective targeting; Molecular drivers of leukemia; Pathobiology; Intrinsic characteristic(s); Extrinsic signal(s); PI3K; PKB / Akt; ETV6-RUNX1; TEL-AML1; Autophagy; Vps34; Autophagy inhibitors; Drug resistance; Chemotherapy resistance; L-Asparaginase resistance; Prednisolone resistance; LARG; ARHGEF 12; Migration; Leukemic niche; Bone marrow microenvironment; Leukemic microenvironment; Small molecule inhibition; CXCL12; CXCR4; Crosstalk; Tunneling nanotube(s); Mesenchymal stromal cell(s); IP10 / CXCL10; IL8 / CXCL8; MCP-1 / CCL2; CCR4; CXCR1; CXCR2; Ligands; Leukemic niche disruption

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Polak, R. (2017). Molecular Drivers of Pediatric Acute Lymphoblastic Leukemia : Toward targeted therapy for children with ALL. (Doctoral Dissertation). Erasmus University Rotterdam. Retrieved from http://hdl.handle.net/1765/102893

Chicago Manual of Style (16th Edition):

Polak, Roel. “Molecular Drivers of Pediatric Acute Lymphoblastic Leukemia : Toward targeted therapy for children with ALL.” 2017. Doctoral Dissertation, Erasmus University Rotterdam. Accessed January 20, 2020. http://hdl.handle.net/1765/102893.

MLA Handbook (7th Edition):

Polak, Roel. “Molecular Drivers of Pediatric Acute Lymphoblastic Leukemia : Toward targeted therapy for children with ALL.” 2017. Web. 20 Jan 2020.

Vancouver:

Polak R. Molecular Drivers of Pediatric Acute Lymphoblastic Leukemia : Toward targeted therapy for children with ALL. [Internet] [Doctoral dissertation]. Erasmus University Rotterdam; 2017. [cited 2020 Jan 20]. Available from: http://hdl.handle.net/1765/102893.

Council of Science Editors:

Polak R. Molecular Drivers of Pediatric Acute Lymphoblastic Leukemia : Toward targeted therapy for children with ALL. [Doctoral Dissertation]. Erasmus University Rotterdam; 2017. Available from: http://hdl.handle.net/1765/102893

.