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You searched for subject:(CTGF). Showing records 1 – 30 of 31 total matches.

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University of Manchester

1. Robinson, Richard. Biomarker and treatment target development in muscle invasive bladder cancer.

Degree: Thesis (M.D.), 2015, University of Manchester

 Introduction: The outcomes following radical treatment for bladder cancer (BC) remain poor, with 5 year overall survival (OS) rates of approximately 50% and over 5000… (more)

Subjects/Keywords: 616.99; Bladder cancer; CYR61; CTGF

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APA (6th Edition):

Robinson, R. (2015). Biomarker and treatment target development in muscle invasive bladder cancer. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/biomarker-and-treatment-target-development-in-muscle-invasive-bladder-cancer(9f7e3dba-9630-4d0d-b7aa-629718d02018).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.654813

Chicago Manual of Style (16th Edition):

Robinson, Richard. “Biomarker and treatment target development in muscle invasive bladder cancer.” 2015. Doctoral Dissertation, University of Manchester. Accessed August 18, 2018. https://www.research.manchester.ac.uk/portal/en/theses/biomarker-and-treatment-target-development-in-muscle-invasive-bladder-cancer(9f7e3dba-9630-4d0d-b7aa-629718d02018).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.654813.

MLA Handbook (7th Edition):

Robinson, Richard. “Biomarker and treatment target development in muscle invasive bladder cancer.” 2015. Web. 18 Aug 2018.

Vancouver:

Robinson R. Biomarker and treatment target development in muscle invasive bladder cancer. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2018 Aug 18]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/biomarker-and-treatment-target-development-in-muscle-invasive-bladder-cancer(9f7e3dba-9630-4d0d-b7aa-629718d02018).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.654813.

Council of Science Editors:

Robinson R. Biomarker and treatment target development in muscle invasive bladder cancer. [Doctoral Dissertation]. University of Manchester; 2015. Available from: https://www.research.manchester.ac.uk/portal/en/theses/biomarker-and-treatment-target-development-in-muscle-invasive-bladder-cancer(9f7e3dba-9630-4d0d-b7aa-629718d02018).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.654813


University of Sydney

2. Ren, Jing. Targeting non-alcoholic steatohepatitis induced by high fat feeding and type 2 diabetes in a preclinical model .

Degree: 2015, University of Sydney

 Non-alcoholic fatty liver disease (NAFLD) is the most common chronic liver disease in the developed world and it is strongly associated with metabolic derangements, such… (more)

Subjects/Keywords: NAFLD; type 2 diabetes; CTGF; sitagliptin; gliclazide

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APA (6th Edition):

Ren, J. (2015). Targeting non-alcoholic steatohepatitis induced by high fat feeding and type 2 diabetes in a preclinical model . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/15737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ren, Jing. “Targeting non-alcoholic steatohepatitis induced by high fat feeding and type 2 diabetes in a preclinical model .” 2015. Thesis, University of Sydney. Accessed August 18, 2018. http://hdl.handle.net/2123/15737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ren, Jing. “Targeting non-alcoholic steatohepatitis induced by high fat feeding and type 2 diabetes in a preclinical model .” 2015. Web. 18 Aug 2018.

Vancouver:

Ren J. Targeting non-alcoholic steatohepatitis induced by high fat feeding and type 2 diabetes in a preclinical model . [Internet] [Thesis]. University of Sydney; 2015. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/2123/15737.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ren J. Targeting non-alcoholic steatohepatitis induced by high fat feeding and type 2 diabetes in a preclinical model . [Thesis]. University of Sydney; 2015. Available from: http://hdl.handle.net/2123/15737

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Florida

3. SRIRAM,SRINIWAS. Knockdown of Connective Tissue Growth Factor(ctgf), Transforming Growth Factor Beta 1 (tgf-B1) and Transforming Growth Factor Beta Receptor 2 (tgf-Br2) by the Topical Application of Short Interfering Rna Molecules in Rabbit Corneal Fibroblasts.

Degree: Biomedical Engineering, 2011, University of Florida

 Purpose: Transforming Growth Factor ? (TGF-?) is a key mediator of the fibrotic response to wounding. It is up regulated during different types of wound… (more)

Subjects/Keywords: CTGF  – RBCF  – SIRNA  – TGF  – TGFBR2; Biomedical Engineering

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APA (6th Edition):

SRIRAM,SRINIWAS. (2011). Knockdown of Connective Tissue Growth Factor(ctgf), Transforming Growth Factor Beta 1 (tgf-B1) and Transforming Growth Factor Beta Receptor 2 (tgf-Br2) by the Topical Application of Short Interfering Rna Molecules in Rabbit Corneal Fibroblasts. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0043062

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

SRIRAM,SRINIWAS. “Knockdown of Connective Tissue Growth Factor(ctgf), Transforming Growth Factor Beta 1 (tgf-B1) and Transforming Growth Factor Beta Receptor 2 (tgf-Br2) by the Topical Application of Short Interfering Rna Molecules in Rabbit Corneal Fibroblasts.” 2011. Masters Thesis, University of Florida. Accessed August 18, 2018. http://ufdc.ufl.edu/UFE0043062.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

SRIRAM,SRINIWAS. “Knockdown of Connective Tissue Growth Factor(ctgf), Transforming Growth Factor Beta 1 (tgf-B1) and Transforming Growth Factor Beta Receptor 2 (tgf-Br2) by the Topical Application of Short Interfering Rna Molecules in Rabbit Corneal Fibroblasts.” 2011. Web. 18 Aug 2018.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

SRIRAM,SRINIWAS. Knockdown of Connective Tissue Growth Factor(ctgf), Transforming Growth Factor Beta 1 (tgf-B1) and Transforming Growth Factor Beta Receptor 2 (tgf-Br2) by the Topical Application of Short Interfering Rna Molecules in Rabbit Corneal Fibroblasts. [Internet] [Masters thesis]. University of Florida; 2011. [cited 2018 Aug 18]. Available from: http://ufdc.ufl.edu/UFE0043062.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

SRIRAM,SRINIWAS. Knockdown of Connective Tissue Growth Factor(ctgf), Transforming Growth Factor Beta 1 (tgf-B1) and Transforming Growth Factor Beta Receptor 2 (tgf-Br2) by the Topical Application of Short Interfering Rna Molecules in Rabbit Corneal Fibroblasts. [Masters Thesis]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0043062

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Harvard University

4. Han, Juliette. The Role of TSC in Oligodendrocyte Differentiation and Myelination.

Degree: PhD, Neurobiology, 2012, Harvard University

 Tuberous Sclerosis Complex (TSC) is an autosomal dominant syndrome characterized by epilepsy, intellectual disability, and autism. Recent studies have suggested that white matter abnormalities, including… (more)

Subjects/Keywords: autism; CTGF; myelin; oligodendrocytes; TSC; neurosciences

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APA (6th Edition):

Han, J. (2012). The Role of TSC in Oligodendrocyte Differentiation and Myelination. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128

Chicago Manual of Style (16th Edition):

Han, Juliette. “The Role of TSC in Oligodendrocyte Differentiation and Myelination.” 2012. Doctoral Dissertation, Harvard University. Accessed August 18, 2018. http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128.

MLA Handbook (7th Edition):

Han, Juliette. “The Role of TSC in Oligodendrocyte Differentiation and Myelination.” 2012. Web. 18 Aug 2018.

Vancouver:

Han J. The Role of TSC in Oligodendrocyte Differentiation and Myelination. [Internet] [Doctoral dissertation]. Harvard University; 2012. [cited 2018 Aug 18]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128.

Council of Science Editors:

Han J. The Role of TSC in Oligodendrocyte Differentiation and Myelination. [Doctoral Dissertation]. Harvard University; 2012. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:9556128


Johannes Gutenberg Universität Mainz

5. Pante, Saskia Veronika. Die Rolle von Chemokinrezeptor CXCR4 und Connective Tissue Growth Factor innerhalb der Tumor-Stroma-Interaktion in der akuten myeloischen Leukämie.

Degree: 2014, Johannes Gutenberg Universität Mainz

 Die akute myeloische Leukämie (AML) ist eine heterogene Erkrankung der hämatopoetischen Vorläuferzelle, die durch unkontrollierte Vermehrung und ein reduziertes Differenzierungsverhalten gekennzeichnet ist. Aufgrund von Therapieresistenzen… (more)

Subjects/Keywords: Leukämie; Therapieresistenz; Stroma; CXCR4; CTGF; leukemia; resistance to therapy; stroma; CXCR4; CTGF; Life sciences

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APA (6th Edition):

Pante, S. V. (2014). Die Rolle von Chemokinrezeptor CXCR4 und Connective Tissue Growth Factor innerhalb der Tumor-Stroma-Interaktion in der akuten myeloischen Leukämie. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2014/3758/

Chicago Manual of Style (16th Edition):

Pante, Saskia Veronika. “Die Rolle von Chemokinrezeptor CXCR4 und Connective Tissue Growth Factor innerhalb der Tumor-Stroma-Interaktion in der akuten myeloischen Leukämie.” 2014. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed August 18, 2018. http://ubm.opus.hbz-nrw.de/volltexte/2014/3758/.

MLA Handbook (7th Edition):

Pante, Saskia Veronika. “Die Rolle von Chemokinrezeptor CXCR4 und Connective Tissue Growth Factor innerhalb der Tumor-Stroma-Interaktion in der akuten myeloischen Leukämie.” 2014. Web. 18 Aug 2018.

Vancouver:

Pante SV. Die Rolle von Chemokinrezeptor CXCR4 und Connective Tissue Growth Factor innerhalb der Tumor-Stroma-Interaktion in der akuten myeloischen Leukämie. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2014. [cited 2018 Aug 18]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2014/3758/.

Council of Science Editors:

Pante SV. Die Rolle von Chemokinrezeptor CXCR4 und Connective Tissue Growth Factor innerhalb der Tumor-Stroma-Interaktion in der akuten myeloischen Leukämie. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2014. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2014/3758/

6. Έλληνα, Όλγα. Μελέτη των επιπέδων των γλυκοζαμινογλυκανών και αυξητικών παραγόντων (CTGF και VEGF) στον σακχαρώδη διαβήτη τύπου Ι: συσχέτισή τους με δείκτες φλεγμονής και την νεφρική λειτουργία.

Degree: 2011, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 Aim To evaluate the role of extracellular matrix-associated (Glycosaminoglycans - GAGs, tissue growth factor - CTGF), angiogenic (VEGF) and inflammatory factors (MCP-1, CD40, IFN-β) in… (more)

Subjects/Keywords: Διαβητική νευροπάθεια; Σακχαρώδης διαβήτης τύπου 1 (TIDM); Αυξητικός παράγοντας συνδετικού ιστού (CTGF); Γλυκοζαμινογλυκάνες (GAGs); Diabetic nephropathy; TIDM; CTGF; GAGs

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APA (6th Edition):

Έλληνα, . . (2011). Μελέτη των επιπέδων των γλυκοζαμινογλυκανών και αυξητικών παραγόντων (CTGF και VEGF) στον σακχαρώδη διαβήτη τύπου Ι: συσχέτισή τους με δείκτες φλεγμονής και την νεφρική λειτουργία. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/27029

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Έλληνα, Όλγα. “Μελέτη των επιπέδων των γλυκοζαμινογλυκανών και αυξητικών παραγόντων (CTGF και VEGF) στον σακχαρώδη διαβήτη τύπου Ι: συσχέτισή τους με δείκτες φλεγμονής και την νεφρική λειτουργία.” 2011. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed August 18, 2018. http://hdl.handle.net/10442/hedi/27029.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Έλληνα, Όλγα. “Μελέτη των επιπέδων των γλυκοζαμινογλυκανών και αυξητικών παραγόντων (CTGF και VEGF) στον σακχαρώδη διαβήτη τύπου Ι: συσχέτισή τους με δείκτες φλεγμονής και την νεφρική λειτουργία.” 2011. Web. 18 Aug 2018.

Vancouver:

Έλληνα . Μελέτη των επιπέδων των γλυκοζαμινογλυκανών και αυξητικών παραγόντων (CTGF και VEGF) στον σακχαρώδη διαβήτη τύπου Ι: συσχέτισή τους με δείκτες φλεγμονής και την νεφρική λειτουργία. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2011. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/10442/hedi/27029.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Έλληνα . Μελέτη των επιπέδων των γλυκοζαμινογλυκανών και αυξητικών παραγόντων (CTGF και VEGF) στον σακχαρώδη διαβήτη τύπου Ι: συσχέτισή τους με δείκτες φλεγμονής και την νεφρική λειτουργία. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2011. Available from: http://hdl.handle.net/10442/hedi/27029

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université de Montréal

7. Bernard, Monique. Rôle de MTORC2 dans la sénescence et la différenciation myofibroblastique induites par l'autophagie .

Degree: 2016, Université de Montréal

 Il a été suggéré que l’autophagie pouvait participer au processus fibrotique en favorisant la différenciation du fibroblaste en myofibroblaste. La sénescence cellulaire a aussi été… (more)

Subjects/Keywords: Autophagie; CTGF; Différenciation; Fibroblaste; Fibrose; MTORC2; Myofibroblaste; Rapamycine; ROS; Sénescence; Autophagy; CTGF; Differentiation; Fibroblast; Fibrosis; MTORC2; Myofibroblast; Rapamycin; ROS; Senescence

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APA (6th Edition):

Bernard, M. (2016). Rôle de MTORC2 dans la sénescence et la différenciation myofibroblastique induites par l'autophagie . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/13524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bernard, Monique. “Rôle de MTORC2 dans la sénescence et la différenciation myofibroblastique induites par l'autophagie .” 2016. Thesis, Université de Montréal. Accessed August 18, 2018. http://hdl.handle.net/1866/13524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bernard, Monique. “Rôle de MTORC2 dans la sénescence et la différenciation myofibroblastique induites par l'autophagie .” 2016. Web. 18 Aug 2018.

Vancouver:

Bernard M. Rôle de MTORC2 dans la sénescence et la différenciation myofibroblastique induites par l'autophagie . [Internet] [Thesis]. Université de Montréal; 2016. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/1866/13524.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bernard M. Rôle de MTORC2 dans la sénescence et la différenciation myofibroblastique induites par l'autophagie . [Thesis]. Université de Montréal; 2016. Available from: http://hdl.handle.net/1866/13524

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

8. Lambi, Alex G. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.

Degree: PhD, 2015, Temple University

Cell Biology

Connective tissue growth factor (CTGF/CCN2) is axiomatically necessary for proper skeletal development and function. We need not look further than the studies that… (more)

Subjects/Keywords: Cellular biology; Developmental biology;

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APA (6th Edition):

Lambi, A. G. (2015). The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,233693

Chicago Manual of Style (16th Edition):

Lambi, Alex G. “The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.” 2015. Doctoral Dissertation, Temple University. Accessed August 18, 2018. http://digital.library.temple.edu/u?/p245801coll10,233693.

MLA Handbook (7th Edition):

Lambi, Alex G. “The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.” 2015. Web. 18 Aug 2018.

Vancouver:

Lambi AG. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2018 Aug 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,233693.

Council of Science Editors:

Lambi AG. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,233693


Temple University

9. Hendesi, Honey. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.

Degree: PhD, 2014, Temple University

Cell Biology

Connective Tissue Growth Factor (CTGF) is a matricellular protein that has been shown to mediate cell adhesion, and as a consequence, it regulates… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Hendesi, H. (2014). CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,263674

Chicago Manual of Style (16th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Doctoral Dissertation, Temple University. Accessed August 18, 2018. http://digital.library.temple.edu/u?/p245801coll10,263674.

MLA Handbook (7th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Web. 18 Aug 2018.

Vancouver:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2018 Aug 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674.

Council of Science Editors:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674


Universiteit Utrecht

10. Lacle, M.M. Male versus Female Breast Cancer : differences hidden behind similarities.

Degree: 2014, Universiteit Utrecht

 Breast cancer in males is a rare disease. Due to its rarity, little research has been conducted on male breast cancer (MBC), especially when compared… (more)

Subjects/Keywords: Geneeskunde; male breast cancer; prognosis; BCL2; CTGF; MLPA; Chr16; Chr17; aCGH

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APA (6th Edition):

Lacle, M. M. (2014). Male versus Female Breast Cancer : differences hidden behind similarities. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/301933

Chicago Manual of Style (16th Edition):

Lacle, M M. “Male versus Female Breast Cancer : differences hidden behind similarities.” 2014. Doctoral Dissertation, Universiteit Utrecht. Accessed August 18, 2018. http://dspace.library.uu.nl:8080/handle/1874/301933.

MLA Handbook (7th Edition):

Lacle, M M. “Male versus Female Breast Cancer : differences hidden behind similarities.” 2014. Web. 18 Aug 2018.

Vancouver:

Lacle MM. Male versus Female Breast Cancer : differences hidden behind similarities. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2014. [cited 2018 Aug 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/301933.

Council of Science Editors:

Lacle MM. Male versus Female Breast Cancer : differences hidden behind similarities. [Doctoral Dissertation]. Universiteit Utrecht; 2014. Available from: http://dspace.library.uu.nl:8080/handle/1874/301933


UCLA

11. Ong, Jessica Renee. Overlapping and Discrete Functions of the Matricellular Proteins CCN1 and CCN2 during Endochondral Bone Formation.

Degree: Molec, Cell, & Dev Biology, 2013, UCLA

 The CCN (Cyr61/Ctgf/Nov) family of matricellular proteins integrates the extracellular environment with the intracellular environment, with involvement in many cellular processes such as adhesion, proliferation,… (more)

Subjects/Keywords: Biology; Histology; Developmental biology; CCN1; CCN2; ctgf; cyr61; endochondral; ossification

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APA (6th Edition):

Ong, J. R. (2013). Overlapping and Discrete Functions of the Matricellular Proteins CCN1 and CCN2 during Endochondral Bone Formation. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/1cf7827n

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ong, Jessica Renee. “Overlapping and Discrete Functions of the Matricellular Proteins CCN1 and CCN2 during Endochondral Bone Formation.” 2013. Thesis, UCLA. Accessed August 18, 2018. http://www.escholarship.org/uc/item/1cf7827n.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ong, Jessica Renee. “Overlapping and Discrete Functions of the Matricellular Proteins CCN1 and CCN2 during Endochondral Bone Formation.” 2013. Web. 18 Aug 2018.

Vancouver:

Ong JR. Overlapping and Discrete Functions of the Matricellular Proteins CCN1 and CCN2 during Endochondral Bone Formation. [Internet] [Thesis]. UCLA; 2013. [cited 2018 Aug 18]. Available from: http://www.escholarship.org/uc/item/1cf7827n.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ong JR. Overlapping and Discrete Functions of the Matricellular Proteins CCN1 and CCN2 during Endochondral Bone Formation. [Thesis]. UCLA; 2013. Available from: http://www.escholarship.org/uc/item/1cf7827n

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Robinson, Richard. Biomarker and Treatment Target Development in Muscle Invasive Bladder Cancer.

Degree: 2015, University of Manchester

 Introduction: The outcomes following radical treatment for bladder cancer (BC) remain poor, with 5 year overall survival (OS) rates of approximately 50% and over 5000… (more)

Subjects/Keywords: Bladder cancer; CYR61; CTGF

…Figure 3.51 - Representative CTGF expression in 1mm TMA cores .....................180 Figure… …185 Figure 3.56 - Expression of CTGF by stage and grade of tumour all cases 19692006… …186 8 Figure 3.57 - Expression of CTGF by stage and grade of tumour in cases post 1985… …191 Figure 3.60 - Mean IHC staining intensity of CTGF (top) and percentage of… …cases expressing CTGF at each level according to percentage thresholds (bottom)… 

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APA (6th Edition):

Robinson, R. (2015). Biomarker and Treatment Target Development in Muscle Invasive Bladder Cancer. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:264777

Chicago Manual of Style (16th Edition):

Robinson, Richard. “Biomarker and Treatment Target Development in Muscle Invasive Bladder Cancer.” 2015. Doctoral Dissertation, University of Manchester. Accessed August 18, 2018. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:264777.

MLA Handbook (7th Edition):

Robinson, Richard. “Biomarker and Treatment Target Development in Muscle Invasive Bladder Cancer.” 2015. Web. 18 Aug 2018.

Vancouver:

Robinson R. Biomarker and Treatment Target Development in Muscle Invasive Bladder Cancer. [Internet] [Doctoral dissertation]. University of Manchester; 2015. [cited 2018 Aug 18]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:264777.

Council of Science Editors:

Robinson R. Biomarker and Treatment Target Development in Muscle Invasive Bladder Cancer. [Doctoral Dissertation]. University of Manchester; 2015. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:264777


University of Otago

13. Alsaleh, Abdulmonem. Methods for Exploiting the Childhood Acute Lymphoblastic Leukaemia DNA Methylation Profile to Detect a Novel Population of Fetal Lymphocytes .

Degree: 2014, University of Otago

 The incidence of childhood acute lymphoblastic leukaemia (B-ALL) has increased since 1950s worldwide. The disease affects young children and it has physical and psychosocial impacts… (more)

Subjects/Keywords: ALL; acute lymphoblastic leukaemia; epigenetics; blood; lymphocytes; CTGF; TES; DNA methylation

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APA (6th Edition):

Alsaleh, A. (2014). Methods for Exploiting the Childhood Acute Lymphoblastic Leukaemia DNA Methylation Profile to Detect a Novel Population of Fetal Lymphocytes . (Masters Thesis). University of Otago. Retrieved from http://hdl.handle.net/10523/4667

Chicago Manual of Style (16th Edition):

Alsaleh, Abdulmonem. “Methods for Exploiting the Childhood Acute Lymphoblastic Leukaemia DNA Methylation Profile to Detect a Novel Population of Fetal Lymphocytes .” 2014. Masters Thesis, University of Otago. Accessed August 18, 2018. http://hdl.handle.net/10523/4667.

MLA Handbook (7th Edition):

Alsaleh, Abdulmonem. “Methods for Exploiting the Childhood Acute Lymphoblastic Leukaemia DNA Methylation Profile to Detect a Novel Population of Fetal Lymphocytes .” 2014. Web. 18 Aug 2018.

Vancouver:

Alsaleh A. Methods for Exploiting the Childhood Acute Lymphoblastic Leukaemia DNA Methylation Profile to Detect a Novel Population of Fetal Lymphocytes . [Internet] [Masters thesis]. University of Otago; 2014. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/10523/4667.

Council of Science Editors:

Alsaleh A. Methods for Exploiting the Childhood Acute Lymphoblastic Leukaemia DNA Methylation Profile to Detect a Novel Population of Fetal Lymphocytes . [Masters Thesis]. University of Otago; 2014. Available from: http://hdl.handle.net/10523/4667


University of Florida

14. Robinson, Paulette. Regulation of Corneal Scar Formation by Transforming Growth Factor Beta and Connective Tissue Growth Factor Roles of Proteolytic Processing and Development of a Gene Silencing Technique.

Degree: Medical Sciences
Genetics (IDP), 2012, University of Florida

 Previous research has demonstrated that corneal scarring following trauma, infection, or refractive surgery is the result of a complex cascade of multiple growth factors, cytokines,… (more)

Subjects/Keywords: cornea  – ctgf  – ribozyme  – scar  – sirna  – tgf-b; Genetics (IDP)

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APA (6th Edition):

Robinson, P. (2012). Regulation of Corneal Scar Formation by Transforming Growth Factor Beta and Connective Tissue Growth Factor Roles of Proteolytic Processing and Development of a Gene Silencing Technique. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0044056

Chicago Manual of Style (16th Edition):

Robinson, Paulette. “Regulation of Corneal Scar Formation by Transforming Growth Factor Beta and Connective Tissue Growth Factor Roles of Proteolytic Processing and Development of a Gene Silencing Technique.” 2012. Doctoral Dissertation, University of Florida. Accessed August 18, 2018. http://ufdc.ufl.edu/UFE0044056.

MLA Handbook (7th Edition):

Robinson, Paulette. “Regulation of Corneal Scar Formation by Transforming Growth Factor Beta and Connective Tissue Growth Factor Roles of Proteolytic Processing and Development of a Gene Silencing Technique.” 2012. Web. 18 Aug 2018.

Vancouver:

Robinson P. Regulation of Corneal Scar Formation by Transforming Growth Factor Beta and Connective Tissue Growth Factor Roles of Proteolytic Processing and Development of a Gene Silencing Technique. [Internet] [Doctoral dissertation]. University of Florida; 2012. [cited 2018 Aug 18]. Available from: http://ufdc.ufl.edu/UFE0044056.

Council of Science Editors:

Robinson P. Regulation of Corneal Scar Formation by Transforming Growth Factor Beta and Connective Tissue Growth Factor Roles of Proteolytic Processing and Development of a Gene Silencing Technique. [Doctoral Dissertation]. University of Florida; 2012. Available from: http://ufdc.ufl.edu/UFE0044056


Université de Montréal

15. Laplante, Patrick. La fibrose en deux parties : de la paillasse à la souris .

Degree: 2009, Université de Montréal

 L’apoptose des cellules endothéliales (CE) représente un évènement initial dans le développement de plusieurs pathologies fibrotiques telles que le rejet chronique d’allogreffe et la sclérose… (more)

Subjects/Keywords: Apoptose; Apoptosis; Différenciation; Differentiation; Cellule endothéliale; Endothelial cell; Fibroblaste; Fibroblast; Myofibroblaste; Myofibroblast; CTGF; CTGF; Perlécan; Perlecan; Pyk2; Pyk2; Src; Src; PI3K; PI3K

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APA (6th Edition):

Laplante, P. (2009). La fibrose en deux parties : de la paillasse à la souris . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/2859

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Laplante, Patrick. “La fibrose en deux parties : de la paillasse à la souris .” 2009. Thesis, Université de Montréal. Accessed August 18, 2018. http://hdl.handle.net/1866/2859.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Laplante, Patrick. “La fibrose en deux parties : de la paillasse à la souris .” 2009. Web. 18 Aug 2018.

Vancouver:

Laplante P. La fibrose en deux parties : de la paillasse à la souris . [Internet] [Thesis]. Université de Montréal; 2009. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/1866/2859.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Laplante P. La fibrose en deux parties : de la paillasse à la souris . [Thesis]. Université de Montréal; 2009. Available from: http://hdl.handle.net/1866/2859

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. 加藤, 真一郎. 不均一ながん細胞の相互作用による腫瘍悪性化機構の解明 : Functional cross-talks between heterogeneous cancer cells accelerate tumor malignant progression.

Degree: 博士(薬学), 2016, University of Toyama / 富山大学

富山大学・富医薬博甲第137号・加藤真一郎・2014/03/21・★論文非公開★

Subjects/Keywords: CTGF; MET; EMT; invasion; lung cancer

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APA (6th Edition):

加藤, . (2016). 不均一ながん細胞の相互作用による腫瘍悪性化機構の解明 : Functional cross-talks between heterogeneous cancer cells accelerate tumor malignant progression. (Thesis). University of Toyama / 富山大学. Retrieved from http://hdl.handle.net/10110/12971 ; http://dx.doi.org/10.15099/00005073

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

加藤, 真一郎. “不均一ながん細胞の相互作用による腫瘍悪性化機構の解明 : Functional cross-talks between heterogeneous cancer cells accelerate tumor malignant progression.” 2016. Thesis, University of Toyama / 富山大学. Accessed August 18, 2018. http://hdl.handle.net/10110/12971 ; http://dx.doi.org/10.15099/00005073.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

加藤, 真一郎. “不均一ながん細胞の相互作用による腫瘍悪性化機構の解明 : Functional cross-talks between heterogeneous cancer cells accelerate tumor malignant progression.” 2016. Web. 18 Aug 2018.

Vancouver:

加藤 . 不均一ながん細胞の相互作用による腫瘍悪性化機構の解明 : Functional cross-talks between heterogeneous cancer cells accelerate tumor malignant progression. [Internet] [Thesis]. University of Toyama / 富山大学; 2016. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/10110/12971 ; http://dx.doi.org/10.15099/00005073.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

加藤 . 不均一ながん細胞の相互作用による腫瘍悪性化機構の解明 : Functional cross-talks between heterogeneous cancer cells accelerate tumor malignant progression. [Thesis]. University of Toyama / 富山大学; 2016. Available from: http://hdl.handle.net/10110/12971 ; http://dx.doi.org/10.15099/00005073

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universiteit Utrecht

17. Roestenberg, Patricia Maria Henrica. The role of CTGF in diabetic nephropathy : Marker, pathogenic factor and target for therapeutic intervention.

Degree: 2007, Universiteit Utrecht

 Diabetic nephropathy (DN) is a common complication of diabetes mellitus (DM) and a major cause of end stage renal disease. DN is a progressive fibrotic… (more)

Subjects/Keywords: Geneeskunde; CTGF; diabetes; kidney; nephropathy; fibrosis; patients; mice; albuminuria; glomerular basement membrane; matrix metalloproteinases

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APA (6th Edition):

Roestenberg, P. M. H. (2007). The role of CTGF in diabetic nephropathy : Marker, pathogenic factor and target for therapeutic intervention. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/19350

Chicago Manual of Style (16th Edition):

Roestenberg, Patricia Maria Henrica. “The role of CTGF in diabetic nephropathy : Marker, pathogenic factor and target for therapeutic intervention.” 2007. Doctoral Dissertation, Universiteit Utrecht. Accessed August 18, 2018. http://dspace.library.uu.nl:8080/handle/1874/19350.

MLA Handbook (7th Edition):

Roestenberg, Patricia Maria Henrica. “The role of CTGF in diabetic nephropathy : Marker, pathogenic factor and target for therapeutic intervention.” 2007. Web. 18 Aug 2018.

Vancouver:

Roestenberg PMH. The role of CTGF in diabetic nephropathy : Marker, pathogenic factor and target for therapeutic intervention. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2007. [cited 2018 Aug 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/19350.

Council of Science Editors:

Roestenberg PMH. The role of CTGF in diabetic nephropathy : Marker, pathogenic factor and target for therapeutic intervention. [Doctoral Dissertation]. Universiteit Utrecht; 2007. Available from: http://dspace.library.uu.nl:8080/handle/1874/19350


Universiteit Utrecht

18. Fontes, Magda Sofia Cristóvão Martins Castro. Conductional remodeling and arrhythmias in the diseased heart.

Degree: 2015, Universiteit Utrecht

 Cardiovascular disease (CVD) is the main cause of death in Western society and it is a global public health problem, particularly taking into account the… (more)

Subjects/Keywords: arrhythmias; connexin43; fibrosis; hypertrophy; CTGF; calcineurin; calmodulin; CaMKII; cardiorenal disease; conduction velocity

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APA (6th Edition):

Fontes, M. S. C. M. C. (2015). Conductional remodeling and arrhythmias in the diseased heart. (Doctoral Dissertation). Universiteit Utrecht. Retrieved from http://dspace.library.uu.nl:8080/handle/1874/317828

Chicago Manual of Style (16th Edition):

Fontes, Magda Sofia Cristóvão Martins Castro. “Conductional remodeling and arrhythmias in the diseased heart.” 2015. Doctoral Dissertation, Universiteit Utrecht. Accessed August 18, 2018. http://dspace.library.uu.nl:8080/handle/1874/317828.

MLA Handbook (7th Edition):

Fontes, Magda Sofia Cristóvão Martins Castro. “Conductional remodeling and arrhythmias in the diseased heart.” 2015. Web. 18 Aug 2018.

Vancouver:

Fontes MSCMC. Conductional remodeling and arrhythmias in the diseased heart. [Internet] [Doctoral dissertation]. Universiteit Utrecht; 2015. [cited 2018 Aug 18]. Available from: http://dspace.library.uu.nl:8080/handle/1874/317828.

Council of Science Editors:

Fontes MSCMC. Conductional remodeling and arrhythmias in the diseased heart. [Doctoral Dissertation]. Universiteit Utrecht; 2015. Available from: http://dspace.library.uu.nl:8080/handle/1874/317828


University of Florida

19. Watson, Rachael. A Model of Arthrofibrosis Using Intra-Articular Gene Delivery of Transforming Growth Factor Beta 1.

Degree: Medical Sciences
Genetics (IDP), 2010, University of Florida

 Idiopathic adhesive capsulitis (IAC) of the shoulder is a disease of unknown etiology characterized by painful, chronic fibrotic expansion of the synovium and joint capsule,… (more)

Subjects/Keywords: adhesive, arthrofibrosis, capsulitis, chondrometaplasia, ctgf, differentiation, factor, fibrosis, growth, mmp, tgfbeta1, transforming; Genetics (IDP)

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APA (6th Edition):

Watson, R. (2010). A Model of Arthrofibrosis Using Intra-Articular Gene Delivery of Transforming Growth Factor Beta 1. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0041397

Chicago Manual of Style (16th Edition):

Watson, Rachael. “A Model of Arthrofibrosis Using Intra-Articular Gene Delivery of Transforming Growth Factor Beta 1.” 2010. Doctoral Dissertation, University of Florida. Accessed August 18, 2018. http://ufdc.ufl.edu/UFE0041397.

MLA Handbook (7th Edition):

Watson, Rachael. “A Model of Arthrofibrosis Using Intra-Articular Gene Delivery of Transforming Growth Factor Beta 1.” 2010. Web. 18 Aug 2018.

Vancouver:

Watson R. A Model of Arthrofibrosis Using Intra-Articular Gene Delivery of Transforming Growth Factor Beta 1. [Internet] [Doctoral dissertation]. University of Florida; 2010. [cited 2018 Aug 18]. Available from: http://ufdc.ufl.edu/UFE0041397.

Council of Science Editors:

Watson R. A Model of Arthrofibrosis Using Intra-Articular Gene Delivery of Transforming Growth Factor Beta 1. [Doctoral Dissertation]. University of Florida; 2010. Available from: http://ufdc.ufl.edu/UFE0041397


Kyoto University

20. Toda, Naohiro. Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis .

Degree: 2018, Kyoto University

Subjects/Keywords: CTGF; mesangial cells; macrophages; integrin; fibronectin

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APA (6th Edition):

Toda, N. (2018). Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis . (Thesis). Kyoto University. Retrieved from http://hdl.handle.net/2433/232131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Toda, Naohiro. “Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis .” 2018. Thesis, Kyoto University. Accessed August 18, 2018. http://hdl.handle.net/2433/232131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Toda, Naohiro. “Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis .” 2018. Web. 18 Aug 2018.

Vancouver:

Toda N. Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis . [Internet] [Thesis]. Kyoto University; 2018. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/2433/232131.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Toda N. Crucial Role of Mesangial Cell-derived Connective Tissue Growth Factor in a Mouse Model of Anti-Glomerular Basement Membrane Glomerulonephritis . [Thesis]. Kyoto University; 2018. Available from: http://hdl.handle.net/2433/232131

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

21. Thanigaimani, Shivshankar. Atrial reverse remodeling in hypertensive substrate.

Degree: 2015, University of Adelaide

 Hypertension is a major independent risk factor for atrial fibrillation (AF). Despite various clinical and experimental studies on atrial remodeling in hypertensive substrates, pre-clinical experimental… (more)

Subjects/Keywords: atrial fibrillation; hypertension; 1K1C ovine model; Fibrosis; inflammation; Connexin43; TGF-B1; CTGF; CFAE; DF; spatio-temporal complexity

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APA (6th Edition):

Thanigaimani, S. (2015). Atrial reverse remodeling in hypertensive substrate. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/98709

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Thanigaimani, Shivshankar. “Atrial reverse remodeling in hypertensive substrate.” 2015. Thesis, University of Adelaide. Accessed August 18, 2018. http://hdl.handle.net/2440/98709.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Thanigaimani, Shivshankar. “Atrial reverse remodeling in hypertensive substrate.” 2015. Web. 18 Aug 2018.

Vancouver:

Thanigaimani S. Atrial reverse remodeling in hypertensive substrate. [Internet] [Thesis]. University of Adelaide; 2015. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/2440/98709.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Thanigaimani S. Atrial reverse remodeling in hypertensive substrate. [Thesis]. University of Adelaide; 2015. Available from: http://hdl.handle.net/2440/98709

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. 梁川, 志保. 皮膚線維化過程におけるCTGF発現に対するSRF,MRTF関与の検討 : SRF acts as a crucial regulator of TGFβ1-induced CTGF expression in human dermal fibroblasts.

Degree: 医学博士, 2015, Iwate Medical University / 岩手医科大学

2015

Subjects/Keywords: TGFβ1; CTGF; SRF; human dermal fibroblast; fibrosis

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APA (6th Edition):

梁川, . (2015). 皮膚線維化過程におけるCTGF発現に対するSRF,MRTF関与の検討 : SRF acts as a crucial regulator of TGFβ1-induced CTGF expression in human dermal fibroblasts. (Thesis). Iwate Medical University / 岩手医科大学. Retrieved from http://id.nii.ac.jp/1181/00005438/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

梁川, 志保. “皮膚線維化過程におけるCTGF発現に対するSRF,MRTF関与の検討 : SRF acts as a crucial regulator of TGFβ1-induced CTGF expression in human dermal fibroblasts.” 2015. Thesis, Iwate Medical University / 岩手医科大学. Accessed August 18, 2018. http://id.nii.ac.jp/1181/00005438/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

梁川, 志保. “皮膚線維化過程におけるCTGF発現に対するSRF,MRTF関与の検討 : SRF acts as a crucial regulator of TGFβ1-induced CTGF expression in human dermal fibroblasts.” 2015. Web. 18 Aug 2018.

Vancouver:

梁川 . 皮膚線維化過程におけるCTGF発現に対するSRF,MRTF関与の検討 : SRF acts as a crucial regulator of TGFβ1-induced CTGF expression in human dermal fibroblasts. [Internet] [Thesis]. Iwate Medical University / 岩手医科大学; 2015. [cited 2018 Aug 18]. Available from: http://id.nii.ac.jp/1181/00005438/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

梁川 . 皮膚線維化過程におけるCTGF発現に対するSRF,MRTF関与の検討 : SRF acts as a crucial regulator of TGFβ1-induced CTGF expression in human dermal fibroblasts. [Thesis]. Iwate Medical University / 岩手医科大学; 2015. Available from: http://id.nii.ac.jp/1181/00005438/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Miami University

23. Zhou, Zhenqing. BIOLOGICAL SIGNIFICANCE OF HEPARIN-BINDING GROWTH FACTORS HB-EGF AND CTGF.

Degree: PhD, Zoology, 2009, Miami University

 Typically growth factors act as signaling molecules between cells, playing an important role in cellular proliferation and differentiation. A variety of growth factors are able… (more)

Subjects/Keywords: Biomedical Research; HB-EGF; CTGF; brown adipose tissue; white adipose tissue; testicular fibrosis; PRDM16; type I collagen; obesity; diabetes; transdifferentiation

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APA (6th Edition):

Zhou, Z. (2009). BIOLOGICAL SIGNIFICANCE OF HEPARIN-BINDING GROWTH FACTORS HB-EGF AND CTGF. (Doctoral Dissertation). Miami University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=miami1258498601

Chicago Manual of Style (16th Edition):

Zhou, Zhenqing. “BIOLOGICAL SIGNIFICANCE OF HEPARIN-BINDING GROWTH FACTORS HB-EGF AND CTGF.” 2009. Doctoral Dissertation, Miami University. Accessed August 18, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=miami1258498601.

MLA Handbook (7th Edition):

Zhou, Zhenqing. “BIOLOGICAL SIGNIFICANCE OF HEPARIN-BINDING GROWTH FACTORS HB-EGF AND CTGF.” 2009. Web. 18 Aug 2018.

Vancouver:

Zhou Z. BIOLOGICAL SIGNIFICANCE OF HEPARIN-BINDING GROWTH FACTORS HB-EGF AND CTGF. [Internet] [Doctoral dissertation]. Miami University; 2009. [cited 2018 Aug 18]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1258498601.

Council of Science Editors:

Zhou Z. BIOLOGICAL SIGNIFICANCE OF HEPARIN-BINDING GROWTH FACTORS HB-EGF AND CTGF. [Doctoral Dissertation]. Miami University; 2009. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=miami1258498601

24. He, Hongbin. Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum.

Degree: Docteur es, Pathologie humaine, 2010, Aix-Marseille 2

La bilharziose reste un problème de santé majeur. L'équipe du Pr Dessein a montré que les infections élevées étaient déterminées par un locus majeur en… (more)

Subjects/Keywords: Bilharziose; Génétique; Susceptibilité; Fibrose; Ctgf; Stat6; Schistosomiasis; Hepatic fibrosis; Connective tissue growth factor; Stat6 transcription factor

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APA (6th Edition):

He, H. (2010). Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2010AIX20720

Chicago Manual of Style (16th Edition):

He, Hongbin. “Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum.” 2010. Doctoral Dissertation, Aix-Marseille 2. Accessed August 18, 2018. http://www.theses.fr/2010AIX20720.

MLA Handbook (7th Edition):

He, Hongbin. “Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum.” 2010. Web. 18 Aug 2018.

Vancouver:

He H. Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2010. [cited 2018 Aug 18]. Available from: http://www.theses.fr/2010AIX20720.

Council of Science Editors:

He H. Studies on the genetic control of infection and hepatic disease in schistosoma haematobium and schistosoma japonicum infections in human : Etudes du contrôle génétique des niveaux d'infection et des atteintes hépatiques dans les infections par Schistosoma haematobium et Schistosoma japonicum. [Doctoral Dissertation]. Aix-Marseille 2; 2010. Available from: http://www.theses.fr/2010AIX20720


University of Florida

25. Gibson,Daniel James. A Cellular and Molecular Study of Corneal Scarring.

Degree: Medical Sciences
Biochemistry and Molecular Biology (IDP), 2011, University of Florida

Subjects/Keywords: anti; antisense; corneal; ctgf; emt; fibrosis; iontophoresis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

James, G. (2011). A Cellular and Molecular Study of Corneal Scarring. (Doctoral Dissertation). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0043264

Chicago Manual of Style (16th Edition):

James, Gibson,Daniel. “A Cellular and Molecular Study of Corneal Scarring.” 2011. Doctoral Dissertation, University of Florida. Accessed August 18, 2018. http://ufdc.ufl.edu/UFE0043264.

MLA Handbook (7th Edition):

James, Gibson,Daniel. “A Cellular and Molecular Study of Corneal Scarring.” 2011. Web. 18 Aug 2018.

Vancouver:

James G. A Cellular and Molecular Study of Corneal Scarring. [Internet] [Doctoral dissertation]. University of Florida; 2011. [cited 2018 Aug 18]. Available from: http://ufdc.ufl.edu/UFE0043264.

Council of Science Editors:

James G. A Cellular and Molecular Study of Corneal Scarring. [Doctoral Dissertation]. University of Florida; 2011. Available from: http://ufdc.ufl.edu/UFE0043264

26. Riley, Kimberly Ann Gooding. Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration.

Degree: PhD, Cell and Developmental Biology, 2015, Vanderbilt University

 As diabetes continues to affect millions of people within the United States, identification of novel factors that may enhance beta-cell proliferation and mass regeneration in… (more)

Subjects/Keywords: diabetes; regeneration; beta-cell; CTGF; pancreas; proliferation

CTGF induction shortens the replicative refractory period ....................... 90 Gene… …expression changes after two days CTGF treatment ..................... 94 Verification of gene… …102 V. ANALYZING THE ROLE OF THE IMMUNE SYSTEM IN CTGF-MEDIATED β-CELL REGENERATION… …107 Evaluation of immune cell populations after two days of CTGF treatment, the peak of β… …16 1-5. Structure of CTGF… 

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APA (6th Edition):

Riley, K. A. G. (2015). Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;

Chicago Manual of Style (16th Edition):

Riley, Kimberly Ann Gooding. “Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed August 18, 2018. http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;.

MLA Handbook (7th Edition):

Riley, Kimberly Ann Gooding. “Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration.” 2015. Web. 18 Aug 2018.

Vancouver:

Riley KAG. Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2018 Aug 18]. Available from: http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;.

Council of Science Editors:

Riley KAG. Modulation of Beta-Cell Intrinsic and Extrinsic Characteristics by CTGF to Promote Beta-Cell Mass Regeneration. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://etd.library.vanderbilt.edu//available/etd-03102015-132801/ ;

27. Kanakis, Georgios. Προγνωστικοί δείκτες της βιολογικής συμπεριφοράς των νευροενδοκρινών όγκων του πνεύμονα: η σημασία της παρουσίας πεπτιδικών μεμβρανικών υποδοχέων.

Degree: 2018, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Pulmonary carcinoids (PC) are among the most common neuroendocrine tumors. The current WHO classification (typical; TC and atypical carcinoids; AC) incorporates the concept of grading;… (more)

Subjects/Keywords: Υποδοχείς σωματοστατίνης; Υποδοχείς ντοπαμίνης; Καρκινοειδές πνεύμονα; Δείκτης κυτταρικού πολλαπλασιασμού Ki-67; Αυξητικός παράγοντας συνδετικού ιστού (CTGF); Somatostatin receptor subtypes; Dopamine receptors; Ki-67 cell proliferation index; Lung carcinoid; Connective tissue growth factor (CTGF)

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APA (6th Edition):

Kanakis, G. (2018). Προγνωστικοί δείκτες της βιολογικής συμπεριφοράς των νευροενδοκρινών όγκων του πνεύμονα: η σημασία της παρουσίας πεπτιδικών μεμβρανικών υποδοχέων. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/42968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kanakis, Georgios. “Προγνωστικοί δείκτες της βιολογικής συμπεριφοράς των νευροενδοκρινών όγκων του πνεύμονα: η σημασία της παρουσίας πεπτιδικών μεμβρανικών υποδοχέων.” 2018. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed August 18, 2018. http://hdl.handle.net/10442/hedi/42968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kanakis, Georgios. “Προγνωστικοί δείκτες της βιολογικής συμπεριφοράς των νευροενδοκρινών όγκων του πνεύμονα: η σημασία της παρουσίας πεπτιδικών μεμβρανικών υποδοχέων.” 2018. Web. 18 Aug 2018.

Vancouver:

Kanakis G. Προγνωστικοί δείκτες της βιολογικής συμπεριφοράς των νευροενδοκρινών όγκων του πνεύμονα: η σημασία της παρουσίας πεπτιδικών μεμβρανικών υποδοχέων. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/10442/hedi/42968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kanakis G. Προγνωστικοί δείκτες της βιολογικής συμπεριφοράς των νευροενδοκρινών όγκων του πνεύμονα: η σημασία της παρουσίας πεπτιδικών μεμβρανικών υποδοχέων. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2018. Available from: http://hdl.handle.net/10442/hedi/42968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Kannampuzha Francis, Jasmine. Regulation of Early Embryonic Development by Maternally Derived Embryokines.

Degree: Animal Molecular and Cellular Biology, 2016, University of Florida

 The reproductive tract secretes bioactive molecules known as embryokines that can regulate embryonic growth and development. Here, we tested actions of four proteins whose genes… (more)

Subjects/Keywords: blastocyst  – bovine  – csf2  – ctgf  – embryokines; Animal Molecular and Cellular Biology

…development ......................................................64 2-2 Actions of CTGF from Day… …connective tissue growth factor (CTGF), teratocarcinoma-derived growth factor (TDGF… …developed to the blastocyst whereas CTGF increased the number of inner cell mass cells in the… …through 13 mo of age without a difference in withers height. In conclusion, activin A and CTGF… 

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kannampuzha Francis, J. (2016). Regulation of Early Embryonic Development by Maternally Derived Embryokines. (Masters Thesis). University of Florida. Retrieved from http://ufdc.ufl.edu/UFE0049964

Chicago Manual of Style (16th Edition):

Kannampuzha Francis, Jasmine. “Regulation of Early Embryonic Development by Maternally Derived Embryokines.” 2016. Masters Thesis, University of Florida. Accessed August 18, 2018. http://ufdc.ufl.edu/UFE0049964.

MLA Handbook (7th Edition):

Kannampuzha Francis, Jasmine. “Regulation of Early Embryonic Development by Maternally Derived Embryokines.” 2016. Web. 18 Aug 2018.

Vancouver:

Kannampuzha Francis J. Regulation of Early Embryonic Development by Maternally Derived Embryokines. [Internet] [Masters thesis]. University of Florida; 2016. [cited 2018 Aug 18]. Available from: http://ufdc.ufl.edu/UFE0049964.

Council of Science Editors:

Kannampuzha Francis J. Regulation of Early Embryonic Development by Maternally Derived Embryokines. [Masters Thesis]. University of Florida; 2016. Available from: http://ufdc.ufl.edu/UFE0049964

29. 河田, かずみ. Possible role of LRP1, a CCN2 receptor, in chondrocytes : 軟骨細胞におけるLRP1,CCN2受容体の機能の可能性.

Degree: 博士(歯学), 2006, Okayama University / 岡山大学

 Low density lipoprotein receptor (LDLR)-related protein 1 (LRP1/CD91) is one of the receptors of CCN2 that conducts endochondral ossification and cartilage repair. LRP1 is a… (more)

Subjects/Keywords: Low density lipoprotein receptor-related protein 1 (LRP1); Cartilage; Chondrocyte differentiation; CCN2/connective tissue growth factor (CTGF); Endocytosis

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APA (6th Edition):

河田, . (2006). Possible role of LRP1, a CCN2 receptor, in chondrocytes : 軟骨細胞におけるLRP1,CCN2受容体の機能の可能性. (Thesis). Okayama University / 岡山大学. Retrieved from http://ousar.lib.okayama-u.ac.jp/12078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

河田, かずみ. “Possible role of LRP1, a CCN2 receptor, in chondrocytes : 軟骨細胞におけるLRP1,CCN2受容体の機能の可能性.” 2006. Thesis, Okayama University / 岡山大学. Accessed August 18, 2018. http://ousar.lib.okayama-u.ac.jp/12078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

河田, かずみ. “Possible role of LRP1, a CCN2 receptor, in chondrocytes : 軟骨細胞におけるLRP1,CCN2受容体の機能の可能性.” 2006. Web. 18 Aug 2018.

Vancouver:

河田 . Possible role of LRP1, a CCN2 receptor, in chondrocytes : 軟骨細胞におけるLRP1,CCN2受容体の機能の可能性. [Internet] [Thesis]. Okayama University / 岡山大学; 2006. [cited 2018 Aug 18]. Available from: http://ousar.lib.okayama-u.ac.jp/12078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

河田 . Possible role of LRP1, a CCN2 receptor, in chondrocytes : 軟骨細胞におけるLRP1,CCN2受容体の機能の可能性. [Thesis]. Okayama University / 岡山大学; 2006. Available from: http://ousar.lib.okayama-u.ac.jp/12078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

30. Σωτηριάδου, Φωτεινή. Κυτταροκίνες ηπατικής ίνωσης σε παιδιά με χρόνια ηπατοπάθεια.

Degree: 2011, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Subjects/Keywords: Κυτταροκίνες; Οικογένεια TGFβS; Πρωτεΐνες Smads; Αυξητικός παράγοντας συνδετικού ιστού (CTGF); Κυστική ίνωση; Στεατοηπατίτιδα; Ηπατική ίνωση σε παιδιά; Μονοπάτι TGF-βs; Cytocines; Family TGF-βs; Proteins Smads; Connective tissue growth factor (CTGF); Cystic fibrosis; Steatohepatitis; Hepatic fibrosis in children; Pathway TGF-βs

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APA (6th Edition):

Σωτηριάδου, . . (2011). Κυτταροκίνες ηπατικής ίνωσης σε παιδιά με χρόνια ηπατοπάθεια. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/27270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Σωτηριάδου, Φωτεινή. “Κυτταροκίνες ηπατικής ίνωσης σε παιδιά με χρόνια ηπατοπάθεια.” 2011. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed August 18, 2018. http://hdl.handle.net/10442/hedi/27270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Σωτηριάδου, Φωτεινή. “Κυτταροκίνες ηπατικής ίνωσης σε παιδιά με χρόνια ηπατοπάθεια.” 2011. Web. 18 Aug 2018.

Vancouver:

Σωτηριάδου . Κυτταροκίνες ηπατικής ίνωσης σε παιδιά με χρόνια ηπατοπάθεια. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2011. [cited 2018 Aug 18]. Available from: http://hdl.handle.net/10442/hedi/27270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Σωτηριάδου . Κυτταροκίνες ηπατικής ίνωσης σε παιδιά με χρόνια ηπατοπάθεια. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2011. Available from: http://hdl.handle.net/10442/hedi/27270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.