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You searched for subject:(COUP TFI). Showing records 1 – 5 of 5 total matches.

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1. Parisot, Joséphine. COUP-TFI est nécessaire dans la différentiation et la migration des granules du gyrus denté : COUP-TFI is required in the differentiation and migration of granule cells in the developing dentate gyrus.

Degree: Docteur es, Interactions moléculaires et cellulaires, 2015, Nice

L’hippocampe est un composant majeur du cerveau des mammifères et joue d'importants rôles dans la mémoire, l’apprentissage et la navigation spatiale. Il comprend deux régions… (more)

Subjects/Keywords: Hippocampe; Gyrus denté; Développement; COUP-TFI; Hippocampus; Dentate gyrus; Development; COUP-TFI

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Parisot, J. (2015). COUP-TFI est nécessaire dans la différentiation et la migration des granules du gyrus denté : COUP-TFI is required in the differentiation and migration of granule cells in the developing dentate gyrus. (Doctoral Dissertation). Nice. Retrieved from http://www.theses.fr/2015NICE4089

Chicago Manual of Style (16th Edition):

Parisot, Joséphine. “COUP-TFI est nécessaire dans la différentiation et la migration des granules du gyrus denté : COUP-TFI is required in the differentiation and migration of granule cells in the developing dentate gyrus.” 2015. Doctoral Dissertation, Nice. Accessed September 25, 2020. http://www.theses.fr/2015NICE4089.

MLA Handbook (7th Edition):

Parisot, Joséphine. “COUP-TFI est nécessaire dans la différentiation et la migration des granules du gyrus denté : COUP-TFI is required in the differentiation and migration of granule cells in the developing dentate gyrus.” 2015. Web. 25 Sep 2020.

Vancouver:

Parisot J. COUP-TFI est nécessaire dans la différentiation et la migration des granules du gyrus denté : COUP-TFI is required in the differentiation and migration of granule cells in the developing dentate gyrus. [Internet] [Doctoral dissertation]. Nice; 2015. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2015NICE4089.

Council of Science Editors:

Parisot J. COUP-TFI est nécessaire dans la différentiation et la migration des granules du gyrus denté : COUP-TFI is required in the differentiation and migration of granule cells in the developing dentate gyrus. [Doctoral Dissertation]. Nice; 2015. Available from: http://www.theses.fr/2015NICE4089


Temple University

2. Pickens, Brandy S. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.

Degree: PhD, 2012, Temple University

Biochemistry

ABSTRACT Retinoic acid (RA) is a positive regulator of P19 EC cell differentiation. Pre-B cell leukemia transcription factors (PBXs) act in conjunction with homeobox… (more)

Subjects/Keywords: Biochemistry; COUP-TFI; Endodermal Differentiation; P19 Cells; Retinoic Acid

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APA (6th Edition):

Pickens, B. S. (2012). THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,196883

Chicago Manual of Style (16th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,196883.

MLA Handbook (7th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Web. 25 Sep 2020.

Vancouver:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883.

Council of Science Editors:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883

3. Magrinelli, Elia. Le récepteur nucléaire orphelin COUP-TFI contrôle l’identité sensorielle et l'activité neuronale dans les cellules post-mitotiques du néocortex chez la souris : The orphan nuclear receptor COUP-TFI controls sensory identity and neuronal activity in post-mitotic cells of the mouse neocortex.

Degree: Docteur es, Interactions moléculaires et cellulaires, 2016, Nice

Le néocortex est une région du cerveau qui traite toutes les entrées sensorielles et créé des réponses comportementales. Il est subdivisé en zones fonctionnelles, chacune… (more)

Subjects/Keywords: Cortex cérébral de la souris; Neocortex; Développement; Connectivité thalamo-cortical; Activité neuronale; COUP-TFI; Egr1; Mouse cerebral cortex; Neocortex; Development; Thalamo-cortical connectivity; Neuronal activity; COUP-TFI; Egr1

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APA (6th Edition):

Magrinelli, E. (2016). Le récepteur nucléaire orphelin COUP-TFI contrôle l’identité sensorielle et l'activité neuronale dans les cellules post-mitotiques du néocortex chez la souris : The orphan nuclear receptor COUP-TFI controls sensory identity and neuronal activity in post-mitotic cells of the mouse neocortex. (Doctoral Dissertation). Nice. Retrieved from http://www.theses.fr/2016NICE4037

Chicago Manual of Style (16th Edition):

Magrinelli, Elia. “Le récepteur nucléaire orphelin COUP-TFI contrôle l’identité sensorielle et l'activité neuronale dans les cellules post-mitotiques du néocortex chez la souris : The orphan nuclear receptor COUP-TFI controls sensory identity and neuronal activity in post-mitotic cells of the mouse neocortex.” 2016. Doctoral Dissertation, Nice. Accessed September 25, 2020. http://www.theses.fr/2016NICE4037.

MLA Handbook (7th Edition):

Magrinelli, Elia. “Le récepteur nucléaire orphelin COUP-TFI contrôle l’identité sensorielle et l'activité neuronale dans les cellules post-mitotiques du néocortex chez la souris : The orphan nuclear receptor COUP-TFI controls sensory identity and neuronal activity in post-mitotic cells of the mouse neocortex.” 2016. Web. 25 Sep 2020.

Vancouver:

Magrinelli E. Le récepteur nucléaire orphelin COUP-TFI contrôle l’identité sensorielle et l'activité neuronale dans les cellules post-mitotiques du néocortex chez la souris : The orphan nuclear receptor COUP-TFI controls sensory identity and neuronal activity in post-mitotic cells of the mouse neocortex. [Internet] [Doctoral dissertation]. Nice; 2016. [cited 2020 Sep 25]. Available from: http://www.theses.fr/2016NICE4037.

Council of Science Editors:

Magrinelli E. Le récepteur nucléaire orphelin COUP-TFI contrôle l’identité sensorielle et l'activité neuronale dans les cellules post-mitotiques du néocortex chez la souris : The orphan nuclear receptor COUP-TFI controls sensory identity and neuronal activity in post-mitotic cells of the mouse neocortex. [Doctoral Dissertation]. Nice; 2016. Available from: http://www.theses.fr/2016NICE4037

4. 村上, 真史. ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制.

Degree: 2018, Nagoya University / 名古屋大学

Ceramide is the central lipid in the sphingolipid metabolism. Ceramide kinase (CERK) and its product, ceramide 1-phosphate (C1P), have been implicated in various cellular functions.… (more)

Subjects/Keywords: All-trans retinoic acid; neuronal differentiation; SH-SY5Y cell; ceramide kinase; promoter analysis; COUP-TFI

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APA (6th Edition):

村上, . (2018). ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制. (Thesis). Nagoya University / 名古屋大学. Retrieved from http://hdl.handle.net/2237/14433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

村上, 真史. “ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制.” 2018. Thesis, Nagoya University / 名古屋大学. Accessed September 25, 2020. http://hdl.handle.net/2237/14433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

村上, 真史. “ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制.” 2018. Web. 25 Sep 2020.

Vancouver:

村上 . ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制. [Internet] [Thesis]. Nagoya University / 名古屋大学; 2018. [cited 2020 Sep 25]. Available from: http://hdl.handle.net/2237/14433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

村上 . ATRA inhibits ceramide kinase transcription through an ATRA-related transcription factor, COUP-TFI, in a human neuroblastoma cell line, SH-SY5Y : ヒト神経芽細胞腫細胞株SH-SY5Y細胞のATRA誘導性転写因子COUP-TFIによるセラミドキナーゼ遺伝子の発現抑制. [Thesis]. Nagoya University / 名古屋大学; 2018. Available from: http://hdl.handle.net/2237/14433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Indian Institute of Science

5. Harish, S. Transcriptional Regulation By Nuclear Receptor Homodimers Binding To The Direct Repeat Motif DR1 : Investigations In An in vitro Transcription System Derived From Rat Liver Nuclear Extracts.

Degree: PhD, Faculty of Science, 2005, Indian Institute of Science

 Nuclear receptors (NRs) are important transcription factors involved in the regulation of a variety of physiological processes such as embryonic development, cell differentiation and homeostasis… (more)

Subjects/Keywords: Biochemistry; Nuclear Receptors (NRs); Retinoid X Receptor (RXR); Electro Mobility Shift Assays (EMSA); Chromatin Assembly; Plasmids Constructs; Chicken Ovalbumin Upstream Promoter Transcription Factor I (COUP-TFI)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harish, S. (2005). Transcriptional Regulation By Nuclear Receptor Homodimers Binding To The Direct Repeat Motif DR1 : Investigations In An in vitro Transcription System Derived From Rat Liver Nuclear Extracts. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/164

Chicago Manual of Style (16th Edition):

Harish, S. “Transcriptional Regulation By Nuclear Receptor Homodimers Binding To The Direct Repeat Motif DR1 : Investigations In An in vitro Transcription System Derived From Rat Liver Nuclear Extracts.” 2005. Doctoral Dissertation, Indian Institute of Science. Accessed September 25, 2020. http://etd.iisc.ac.in/handle/2005/164.

MLA Handbook (7th Edition):

Harish, S. “Transcriptional Regulation By Nuclear Receptor Homodimers Binding To The Direct Repeat Motif DR1 : Investigations In An in vitro Transcription System Derived From Rat Liver Nuclear Extracts.” 2005. Web. 25 Sep 2020.

Vancouver:

Harish S. Transcriptional Regulation By Nuclear Receptor Homodimers Binding To The Direct Repeat Motif DR1 : Investigations In An in vitro Transcription System Derived From Rat Liver Nuclear Extracts. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2005. [cited 2020 Sep 25]. Available from: http://etd.iisc.ac.in/handle/2005/164.

Council of Science Editors:

Harish S. Transcriptional Regulation By Nuclear Receptor Homodimers Binding To The Direct Repeat Motif DR1 : Investigations In An in vitro Transcription System Derived From Rat Liver Nuclear Extracts. [Doctoral Dissertation]. Indian Institute of Science; 2005. Available from: http://etd.iisc.ac.in/handle/2005/164

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