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You searched for subject:(CCR5). Showing records 1 – 30 of 82 total matches.

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California State University – Sacramento

1. Fury, Brian Patrick. Therapeutic potential for the inhibition of HIV from CD34+ hematopoietic stem cell derived induced pluripotent stem cells expressing three anti-HIV genes.

Degree: MA, Biological Sciences (Stem Cell, 2011, California State University – Sacramento

 Human immunodeficiency virus continues to persist in millions of people worldwide. While antiretroviral drug therapies have improved life for many, a cure remains elusive. Long-term… (more)

Subjects/Keywords: iPSC; CCR5

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fury, B. P. (2011). Therapeutic potential for the inhibition of HIV from CD34+ hematopoietic stem cell derived induced pluripotent stem cells expressing three anti-HIV genes. (Masters Thesis). California State University – Sacramento. Retrieved from http://hdl.handle.net/10211.9/1314

Chicago Manual of Style (16th Edition):

Fury, Brian Patrick. “Therapeutic potential for the inhibition of HIV from CD34+ hematopoietic stem cell derived induced pluripotent stem cells expressing three anti-HIV genes.” 2011. Masters Thesis, California State University – Sacramento. Accessed June 17, 2019. http://hdl.handle.net/10211.9/1314.

MLA Handbook (7th Edition):

Fury, Brian Patrick. “Therapeutic potential for the inhibition of HIV from CD34+ hematopoietic stem cell derived induced pluripotent stem cells expressing three anti-HIV genes.” 2011. Web. 17 Jun 2019.

Vancouver:

Fury BP. Therapeutic potential for the inhibition of HIV from CD34+ hematopoietic stem cell derived induced pluripotent stem cells expressing three anti-HIV genes. [Internet] [Masters thesis]. California State University – Sacramento; 2011. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/10211.9/1314.

Council of Science Editors:

Fury BP. Therapeutic potential for the inhibition of HIV from CD34+ hematopoietic stem cell derived induced pluripotent stem cells expressing three anti-HIV genes. [Masters Thesis]. California State University – Sacramento; 2011. Available from: http://hdl.handle.net/10211.9/1314

2. José de Pessoa Saldanha, Carlos. Avaliação da mutação ccr532 do receptor da quimiocina como marcador genético-histórico na população de Triunfo Pernambuco .

Degree: 2008, Universidade Federal de Pernambuco

 O gene ccr5 codifica o receptor b-quimiocina 5 (CCR5), uma proteína transmembrânica que age como principal co-receptor para os vírus HIV-1, Variola major e para… (more)

Subjects/Keywords: ccr5; ccr5$32; Mutação; População de Triunfo

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APA (6th Edition):

José de Pessoa Saldanha, C. (2008). Avaliação da mutação ccr532 do receptor da quimiocina como marcador genético-histórico na população de Triunfo Pernambuco . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/6316

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

José de Pessoa Saldanha, Carlos. “Avaliação da mutação ccr532 do receptor da quimiocina como marcador genético-histórico na população de Triunfo Pernambuco .” 2008. Thesis, Universidade Federal de Pernambuco. Accessed June 17, 2019. http://repositorio.ufpe.br/handle/123456789/6316.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

José de Pessoa Saldanha, Carlos. “Avaliação da mutação ccr532 do receptor da quimiocina como marcador genético-histórico na população de Triunfo Pernambuco .” 2008. Web. 17 Jun 2019.

Vancouver:

José de Pessoa Saldanha C. Avaliação da mutação ccr532 do receptor da quimiocina como marcador genético-histórico na população de Triunfo Pernambuco . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2008. [cited 2019 Jun 17]. Available from: http://repositorio.ufpe.br/handle/123456789/6316.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

José de Pessoa Saldanha C. Avaliação da mutação ccr532 do receptor da quimiocina como marcador genético-histórico na população de Triunfo Pernambuco . [Thesis]. Universidade Federal de Pernambuco; 2008. Available from: http://repositorio.ufpe.br/handle/123456789/6316

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. CAMPELO JUNIOR, Evônio de Barros. Coinfecção GBV-C e HIV em pacientes acompanhados no serviço de doenças infecciosas e parasitárias do HC-UFPE .

Degree: 2012, Universidade Federal de Pernambuco

 Desde 1967, momento do seu surgimento no mundo científico, o GB vírus C (GBV-C) permanece conhecido como vírus indolente, todavia não obstante,pesquisas têm reveladoadespeito da… (more)

Subjects/Keywords: HIV; GBV-C; CCR5; CXCR4

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APA (6th Edition):

CAMPELO JUNIOR, E. d. B. (2012). Coinfecção GBV-C e HIV em pacientes acompanhados no serviço de doenças infecciosas e parasitárias do HC-UFPE . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/18505

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CAMPELO JUNIOR, Evônio de Barros. “Coinfecção GBV-C e HIV em pacientes acompanhados no serviço de doenças infecciosas e parasitárias do HC-UFPE .” 2012. Thesis, Universidade Federal de Pernambuco. Accessed June 17, 2019. http://repositorio.ufpe.br/handle/123456789/18505.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CAMPELO JUNIOR, Evônio de Barros. “Coinfecção GBV-C e HIV em pacientes acompanhados no serviço de doenças infecciosas e parasitárias do HC-UFPE .” 2012. Web. 17 Jun 2019.

Vancouver:

CAMPELO JUNIOR EdB. Coinfecção GBV-C e HIV em pacientes acompanhados no serviço de doenças infecciosas e parasitárias do HC-UFPE . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2012. [cited 2019 Jun 17]. Available from: http://repositorio.ufpe.br/handle/123456789/18505.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CAMPELO JUNIOR EdB. Coinfecção GBV-C e HIV em pacientes acompanhados no serviço de doenças infecciosas e parasitárias do HC-UFPE . [Thesis]. Universidade Federal de Pernambuco; 2012. Available from: http://repositorio.ufpe.br/handle/123456789/18505

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pretoria

4. Barmania, Fatima. Analysis of CCR5 diversity in the South African population.

Degree: Immunology, 2012, University of Pretoria

 Infection with the human immunodeficiency virus (HIV) constitutes a global pandemic, and South Africa forms part of the region known to house over two-thirds of… (more)

Subjects/Keywords: UCTD; South African population; CCR5 diversity; CCR5 receptors

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APA (6th Edition):

Barmania, F. (2012). Analysis of CCR5 diversity in the South African population. (Masters Thesis). University of Pretoria. Retrieved from http://hdl.handle.net/2263/31127

Chicago Manual of Style (16th Edition):

Barmania, Fatima. “Analysis of CCR5 diversity in the South African population.” 2012. Masters Thesis, University of Pretoria. Accessed June 17, 2019. http://hdl.handle.net/2263/31127.

MLA Handbook (7th Edition):

Barmania, Fatima. “Analysis of CCR5 diversity in the South African population.” 2012. Web. 17 Jun 2019.

Vancouver:

Barmania F. Analysis of CCR5 diversity in the South African population. [Internet] [Masters thesis]. University of Pretoria; 2012. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/2263/31127.

Council of Science Editors:

Barmania F. Analysis of CCR5 diversity in the South African population. [Masters Thesis]. University of Pretoria; 2012. Available from: http://hdl.handle.net/2263/31127


University of Pretoria

5. Barmania, Fatima. Analysis of CCR5 diversity in the South African population .

Degree: 2012, University of Pretoria

 Infection with the human immunodeficiency virus (HIV) constitutes a global pandemic, and South Africa forms part of the region known to house over two-thirds of… (more)

Subjects/Keywords: UCTD; South African population; CCR5 diversity; CCR5 receptors

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APA (6th Edition):

Barmania, F. (2012). Analysis of CCR5 diversity in the South African population . (Masters Thesis). University of Pretoria. Retrieved from http://upetd.up.ac.za/thesis/available/etd-08072012-185416/

Chicago Manual of Style (16th Edition):

Barmania, Fatima. “Analysis of CCR5 diversity in the South African population .” 2012. Masters Thesis, University of Pretoria. Accessed June 17, 2019. http://upetd.up.ac.za/thesis/available/etd-08072012-185416/.

MLA Handbook (7th Edition):

Barmania, Fatima. “Analysis of CCR5 diversity in the South African population .” 2012. Web. 17 Jun 2019.

Vancouver:

Barmania F. Analysis of CCR5 diversity in the South African population . [Internet] [Masters thesis]. University of Pretoria; 2012. [cited 2019 Jun 17]. Available from: http://upetd.up.ac.za/thesis/available/etd-08072012-185416/.

Council of Science Editors:

Barmania F. Analysis of CCR5 diversity in the South African population . [Masters Thesis]. University of Pretoria; 2012. Available from: http://upetd.up.ac.za/thesis/available/etd-08072012-185416/

6. Koensgen, Florian. Modélisation du récepteur aux chimiokines C-C de type 5 : caractérisation des états conformationnels et conception rationnelle de modulateurs de la dimérisation : C-C chemokine receptor type 5 modelization : characterisation of conformational states and rational design of dimerization modulators.

Degree: Docteur es, Chimie informatique et théorique, 2018, Université de Strasbourg

De nombreuses études des RCPGs révèlent que leur activation n’implique pas que deux états conformationnels, l’un activé et l’autre inactivé, mais une diversité plus importante… (more)

Subjects/Keywords: Rcpg; CCR5; Criblage virtuel; Rcpg; CCR5; Virtual screening; Molecular dynamics; 541.2

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APA (6th Edition):

Koensgen, F. (2018). Modélisation du récepteur aux chimiokines C-C de type 5 : caractérisation des états conformationnels et conception rationnelle de modulateurs de la dimérisation : C-C chemokine receptor type 5 modelization : characterisation of conformational states and rational design of dimerization modulators. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2018STRAF026

Chicago Manual of Style (16th Edition):

Koensgen, Florian. “Modélisation du récepteur aux chimiokines C-C de type 5 : caractérisation des états conformationnels et conception rationnelle de modulateurs de la dimérisation : C-C chemokine receptor type 5 modelization : characterisation of conformational states and rational design of dimerization modulators.” 2018. Doctoral Dissertation, Université de Strasbourg. Accessed June 17, 2019. http://www.theses.fr/2018STRAF026.

MLA Handbook (7th Edition):

Koensgen, Florian. “Modélisation du récepteur aux chimiokines C-C de type 5 : caractérisation des états conformationnels et conception rationnelle de modulateurs de la dimérisation : C-C chemokine receptor type 5 modelization : characterisation of conformational states and rational design of dimerization modulators.” 2018. Web. 17 Jun 2019.

Vancouver:

Koensgen F. Modélisation du récepteur aux chimiokines C-C de type 5 : caractérisation des états conformationnels et conception rationnelle de modulateurs de la dimérisation : C-C chemokine receptor type 5 modelization : characterisation of conformational states and rational design of dimerization modulators. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2018. [cited 2019 Jun 17]. Available from: http://www.theses.fr/2018STRAF026.

Council of Science Editors:

Koensgen F. Modélisation du récepteur aux chimiokines C-C de type 5 : caractérisation des états conformationnels et conception rationnelle de modulateurs de la dimérisation : C-C chemokine receptor type 5 modelization : characterisation of conformational states and rational design of dimerization modulators. [Doctoral Dissertation]. Université de Strasbourg; 2018. Available from: http://www.theses.fr/2018STRAF026


Universidade de Brasília

7. Eduardo Lourenço da Silva. Marcadores moleculares associados à resistência/susceptibilidade ao HIV-1 no Distrito Federal (Brasil).

Degree: 2010, Universidade de Brasília

A Síndrome da Imuno-Deficiência Adquirida (AIDS), causada pelo vírus da imunodeficiência humana (HIV), caracteriza-se por ser uma infecção em que se observa uma alta taxa… (more)

Subjects/Keywords: HIV-1; CIENCIAS BIOLOGICAS; CCR5; Brasília; TNFs

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APA (6th Edition):

Silva, E. L. d. (2010). Marcadores moleculares associados à resistência/susceptibilidade ao HIV-1 no Distrito Federal (Brasil). (Thesis). Universidade de Brasília. Retrieved from http://bdtd.bce.unb.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=6401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Silva, Eduardo Lourenço da. “Marcadores moleculares associados à resistência/susceptibilidade ao HIV-1 no Distrito Federal (Brasil).” 2010. Thesis, Universidade de Brasília. Accessed June 17, 2019. http://bdtd.bce.unb.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=6401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Silva, Eduardo Lourenço da. “Marcadores moleculares associados à resistência/susceptibilidade ao HIV-1 no Distrito Federal (Brasil).” 2010. Web. 17 Jun 2019.

Vancouver:

Silva ELd. Marcadores moleculares associados à resistência/susceptibilidade ao HIV-1 no Distrito Federal (Brasil). [Internet] [Thesis]. Universidade de Brasília; 2010. [cited 2019 Jun 17]. Available from: http://bdtd.bce.unb.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=6401.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Silva ELd. Marcadores moleculares associados à resistência/susceptibilidade ao HIV-1 no Distrito Federal (Brasil). [Thesis]. Universidade de Brasília; 2010. Available from: http://bdtd.bce.unb.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=6401

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

8. Kroetz, Danielle N. The Role of CCR5 in Protection Against Histoplasma capsulatum Infection.

Degree: PhD, Medicine: Molecular Genetics, Biochemistry, and Microbiology, 2011, University of Cincinnati

 Histoplasma capsulatum is a dimorphic fungus that is found worldwide, but is endemic in the Midwest and Southeast regions of the United States. Infection with… (more)

Subjects/Keywords: Immunology; Histoplasma; CCR5; Treg; Th17; chemokine

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APA (6th Edition):

Kroetz, D. N. (2011). The Role of CCR5 in Protection Against Histoplasma capsulatum Infection. (Doctoral Dissertation). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305030008

Chicago Manual of Style (16th Edition):

Kroetz, Danielle N. “The Role of CCR5 in Protection Against Histoplasma capsulatum Infection.” 2011. Doctoral Dissertation, University of Cincinnati. Accessed June 17, 2019. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305030008.

MLA Handbook (7th Edition):

Kroetz, Danielle N. “The Role of CCR5 in Protection Against Histoplasma capsulatum Infection.” 2011. Web. 17 Jun 2019.

Vancouver:

Kroetz DN. The Role of CCR5 in Protection Against Histoplasma capsulatum Infection. [Internet] [Doctoral dissertation]. University of Cincinnati; 2011. [cited 2019 Jun 17]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305030008.

Council of Science Editors:

Kroetz DN. The Role of CCR5 in Protection Against Histoplasma capsulatum Infection. [Doctoral Dissertation]. University of Cincinnati; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1305030008


University of New South Wales

9. Ledger, Scott. Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection.

Degree: Clinical School - St Vincent's Hospital, 2016, University of New South Wales

 HIV currently infects 35 million people worldwide and antiretroviral treatments are expensive, lifelong, and fail to provide a cure. Gene therapy provides an alternate approach… (more)

Subjects/Keywords: shRNA; HIV; Gene therapy; CCR5; C46; lentivirus

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APA (6th Edition):

Ledger, S. (2016). Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Ledger, Scott. “Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection.” 2016. Doctoral Dissertation, University of New South Wales. Accessed June 17, 2019. http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true.

MLA Handbook (7th Edition):

Ledger, Scott. “Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection.” 2016. Web. 17 Jun 2019.

Vancouver:

Ledger S. Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection. [Internet] [Doctoral dissertation]. University of New South Wales; 2016. [cited 2019 Jun 17]. Available from: http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true.

Council of Science Editors:

Ledger S. Use of Short-Hairpin RNA to CCR5, and maC46 Entry Inhibitor Gene-Therapies against HIV infection. [Doctoral Dissertation]. University of New South Wales; 2016. Available from: http://handle.unsw.edu.au/1959.4/56230 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:40347/SOURCE02?view=true

10. Pakalnienė, Jolita. Genų, koduojančių penktą chemokino ir trečią Toll-like receptorius, polimorfizmų reikšmė erkinio encefalito viruso infekcijos metu.

Degree: Dissertation, Medicine, 2014, Lithuanian Academic Libraries Network (LABT)

Erkinis encefalitas (EE) – pati dažniausia ir sunkiausia virusinė nervų sistemos infekcija Lietuvoje, kuria per metus vidutiniškai suserga 400 žmonių. Užsikrėtus EE virusu (EEV), galima… (more)

Subjects/Keywords: Erkinis encefalitas; CCR5; TLR3; Genų polimorfizmai; Tick-borne encephalitis; CCR5; TLR3; Gene polymorphism

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APA (6th Edition):

Pakalnienė, J. (2014). Genų, koduojančių penktą chemokino ir trečią Toll-like receptorius, polimorfizmų reikšmė erkinio encefalito viruso infekcijos metu. (Doctoral Dissertation). Lithuanian Academic Libraries Network (LABT). Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2014~D_20140930_085230-33015 ;

Chicago Manual of Style (16th Edition):

Pakalnienė, Jolita. “Genų, koduojančių penktą chemokino ir trečią Toll-like receptorius, polimorfizmų reikšmė erkinio encefalito viruso infekcijos metu.” 2014. Doctoral Dissertation, Lithuanian Academic Libraries Network (LABT). Accessed June 17, 2019. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2014~D_20140930_085230-33015 ;.

MLA Handbook (7th Edition):

Pakalnienė, Jolita. “Genų, koduojančių penktą chemokino ir trečią Toll-like receptorius, polimorfizmų reikšmė erkinio encefalito viruso infekcijos metu.” 2014. Web. 17 Jun 2019.

Vancouver:

Pakalnienė J. Genų, koduojančių penktą chemokino ir trečią Toll-like receptorius, polimorfizmų reikšmė erkinio encefalito viruso infekcijos metu. [Internet] [Doctoral dissertation]. Lithuanian Academic Libraries Network (LABT); 2014. [cited 2019 Jun 17]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2014~D_20140930_085230-33015 ;.

Council of Science Editors:

Pakalnienė J. Genų, koduojančių penktą chemokino ir trečią Toll-like receptorius, polimorfizmų reikšmė erkinio encefalito viruso infekcijos metu. [Doctoral Dissertation]. Lithuanian Academic Libraries Network (LABT); 2014. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2014~D_20140930_085230-33015 ;


Université de Montréal

11. Asin Milan, Odalis. Mécanismes de résistance aux inhibiteurs de CCR5 .

Degree: 2015, Université de Montréal

 Le travail décrit dans ce manuscrit vise à caractériser les voies de résistance aux inhibiteurs de CCR5. Lors d’une première étape, nous avons développé un… (more)

Subjects/Keywords: VIH; résistance; inhibiteurs d’entrée; corécepteur CCR5; HIV; resistance; HIV inhibitors; coreceptor CCR5

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APA (6th Edition):

Asin Milan, O. (2015). Mécanismes de résistance aux inhibiteurs de CCR5 . (Thesis). Université de Montréal. Retrieved from http://hdl.handle.net/1866/12734

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Asin Milan, Odalis. “Mécanismes de résistance aux inhibiteurs de CCR5 .” 2015. Thesis, Université de Montréal. Accessed June 17, 2019. http://hdl.handle.net/1866/12734.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Asin Milan, Odalis. “Mécanismes de résistance aux inhibiteurs de CCR5 .” 2015. Web. 17 Jun 2019.

Vancouver:

Asin Milan O. Mécanismes de résistance aux inhibiteurs de CCR5 . [Internet] [Thesis]. Université de Montréal; 2015. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/1866/12734.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Asin Milan O. Mécanismes de résistance aux inhibiteurs de CCR5 . [Thesis]. Université de Montréal; 2015. Available from: http://hdl.handle.net/1866/12734

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Sul

12. Schauren, Juliana da Silveira. Estudo dos polimorfismos CCR2-64I, CCR5-59353, CCR5-59356, CCR5-59402 e CCR5-59653 em pacientes com lúpus eritematoso sistêmico do sul do Brasil.

Degree: 2013, Universidade do Rio Grande do Sul

 O Lúpus Eritematoso Sistêmico (LES) é uma doença autoimune inflamatória crônica que possui uma etiopatogênese complexa. Diversos fatores participam da patogênese da doença, dentre eles… (more)

Subjects/Keywords: CCR5; Polimorfismo; CCR5 promoter polymorphisms; Lupus eritematoso sistêmico; Genética humana; CCR2; Lupus; Systemic lupus erythematosus

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APA (6th Edition):

Schauren, J. d. S. (2013). Estudo dos polimorfismos CCR2-64I, CCR5-59353, CCR5-59356, CCR5-59402 e CCR5-59653 em pacientes com lúpus eritematoso sistêmico do sul do Brasil. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/78127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Schauren, Juliana da Silveira. “Estudo dos polimorfismos CCR2-64I, CCR5-59353, CCR5-59356, CCR5-59402 e CCR5-59653 em pacientes com lúpus eritematoso sistêmico do sul do Brasil.” 2013. Thesis, Universidade do Rio Grande do Sul. Accessed June 17, 2019. http://hdl.handle.net/10183/78127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Schauren, Juliana da Silveira. “Estudo dos polimorfismos CCR2-64I, CCR5-59353, CCR5-59356, CCR5-59402 e CCR5-59653 em pacientes com lúpus eritematoso sistêmico do sul do Brasil.” 2013. Web. 17 Jun 2019.

Vancouver:

Schauren JdS. Estudo dos polimorfismos CCR2-64I, CCR5-59353, CCR5-59356, CCR5-59402 e CCR5-59653 em pacientes com lúpus eritematoso sistêmico do sul do Brasil. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2013. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/10183/78127.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Schauren JdS. Estudo dos polimorfismos CCR2-64I, CCR5-59353, CCR5-59356, CCR5-59402 e CCR5-59653 em pacientes com lúpus eritematoso sistêmico do sul do Brasil. [Thesis]. Universidade do Rio Grande do Sul; 2013. Available from: http://hdl.handle.net/10183/78127

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. Maria do Socorro Queiroz Alves de Souza. Genetic susceptibility to osteomyelitis development associated to polymorphisms: IL1RN VNTR, IL1B -511C>T, IL8 -251A>T, IL6 -174G>C, TNFA -308G>A and to CCR5DELTA32 mutation.

Degree: PhD, 2015, Universidade Federal do Ceará

Osteomyelitis is an inflammatory process and a complication of bone traumas. Nowadays, this disease is a public health problem with a significant morbidity and mortality.… (more)

Subjects/Keywords: CIENCIAS DA SAUDE; Osteomielite; Staphylococcus aureus; Citocinas; Receptores CCR5; Osteomyelitis; Staphylococcus aureus; Cytokines; Receptors, CCR5

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APA (6th Edition):

Souza, M. d. S. Q. A. d. (2015). Genetic susceptibility to osteomyelitis development associated to polymorphisms: IL1RN VNTR, IL1B -511C>T, IL8 -251A>T, IL6 -174G>C, TNFA -308G>A and to CCR5DELTA32 mutation. (Doctoral Dissertation). Universidade Federal do Ceará. Retrieved from http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15100 ;

Chicago Manual of Style (16th Edition):

Souza, Maria do Socorro Queiroz Alves de. “Genetic susceptibility to osteomyelitis development associated to polymorphisms: IL1RN VNTR, IL1B -511C>T, IL8 -251A>T, IL6 -174G>C, TNFA -308G>A and to CCR5DELTA32 mutation.” 2015. Doctoral Dissertation, Universidade Federal do Ceará. Accessed June 17, 2019. http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15100 ;.

MLA Handbook (7th Edition):

Souza, Maria do Socorro Queiroz Alves de. “Genetic susceptibility to osteomyelitis development associated to polymorphisms: IL1RN VNTR, IL1B -511C>T, IL8 -251A>T, IL6 -174G>C, TNFA -308G>A and to CCR5DELTA32 mutation.” 2015. Web. 17 Jun 2019.

Vancouver:

Souza MdSQAd. Genetic susceptibility to osteomyelitis development associated to polymorphisms: IL1RN VNTR, IL1B -511C>T, IL8 -251A>T, IL6 -174G>C, TNFA -308G>A and to CCR5DELTA32 mutation. [Internet] [Doctoral dissertation]. Universidade Federal do Ceará 2015. [cited 2019 Jun 17]. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15100 ;.

Council of Science Editors:

Souza MdSQAd. Genetic susceptibility to osteomyelitis development associated to polymorphisms: IL1RN VNTR, IL1B -511C>T, IL8 -251A>T, IL6 -174G>C, TNFA -308G>A and to CCR5DELTA32 mutation. [Doctoral Dissertation]. Universidade Federal do Ceará 2015. Available from: http://www.teses.ufc.br/tde_busca/arquivo.php?codArquivo=15100 ;

14. Gata, Gabriel. Regulated export of G-protein coupled receptors : L’export régulé de récepteurs couplés aux protéines G.

Degree: Docteur es, Biologie cellulaire, 2014, Université Paris Descartes – Paris V

La plus grande famille de récepteurs membranaires est constituée par des récepteurs à sept domaines transmembranaires couplés aux protéines G (RCPG). Ces récepteurs sont impliqués… (more)

Subjects/Keywords: RCPG; Export; Rétention intracellulaire; Gatekkeper; GB1; CCR5; GPCR; Export; Intracellular retention; Gatekeeper; GB1; CCR5; 571.6

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APA (6th Edition):

Gata, G. (2014). Regulated export of G-protein coupled receptors : L’export régulé de récepteurs couplés aux protéines G. (Doctoral Dissertation). Université Paris Descartes – Paris V. Retrieved from http://www.theses.fr/2014PA05T066

Chicago Manual of Style (16th Edition):

Gata, Gabriel. “Regulated export of G-protein coupled receptors : L’export régulé de récepteurs couplés aux protéines G.” 2014. Doctoral Dissertation, Université Paris Descartes – Paris V. Accessed June 17, 2019. http://www.theses.fr/2014PA05T066.

MLA Handbook (7th Edition):

Gata, Gabriel. “Regulated export of G-protein coupled receptors : L’export régulé de récepteurs couplés aux protéines G.” 2014. Web. 17 Jun 2019.

Vancouver:

Gata G. Regulated export of G-protein coupled receptors : L’export régulé de récepteurs couplés aux protéines G. [Internet] [Doctoral dissertation]. Université Paris Descartes – Paris V; 2014. [cited 2019 Jun 17]. Available from: http://www.theses.fr/2014PA05T066.

Council of Science Editors:

Gata G. Regulated export of G-protein coupled receptors : L’export régulé de récepteurs couplés aux protéines G. [Doctoral Dissertation]. Université Paris Descartes – Paris V; 2014. Available from: http://www.theses.fr/2014PA05T066


Université Montpellier II

15. Duquenne, Charline. Modulation de l’activité des corécepteurs CCR5 et CXCR4 du VIH 1 comme stratégie thérapeutique : étude des deux isoformes de CXCR4 et interaction de CCR5 avec le récepteur S1P1 : Modulation of CCR5 and CXCR4 HIV-1 coreceptor activities as a therapeutic strategy : studying the two CXCR4 isoforms and the interaction of CCR5 with S1P1.

Degree: Docteur es, Biologie Santé, 2013, Université Montpellier II

CCR5 et CXCR4 sont les corécepteurs d'entrée du VIH utilisés par le virus in vivo en plus du récepteur principal CD4 pour infecter les cellules.… (more)

Subjects/Keywords: S1p1; Vih; Ccr5; Cxcr4; Rcpg; Fty720; S1p1; Hiv; Ccr5; Cxcr4; Gpcr; Fty720

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APA (6th Edition):

Duquenne, C. (2013). Modulation de l’activité des corécepteurs CCR5 et CXCR4 du VIH 1 comme stratégie thérapeutique : étude des deux isoformes de CXCR4 et interaction de CCR5 avec le récepteur S1P1 : Modulation of CCR5 and CXCR4 HIV-1 coreceptor activities as a therapeutic strategy : studying the two CXCR4 isoforms and the interaction of CCR5 with S1P1. (Doctoral Dissertation). Université Montpellier II. Retrieved from http://www.theses.fr/2013MON20188

Chicago Manual of Style (16th Edition):

Duquenne, Charline. “Modulation de l’activité des corécepteurs CCR5 et CXCR4 du VIH 1 comme stratégie thérapeutique : étude des deux isoformes de CXCR4 et interaction de CCR5 avec le récepteur S1P1 : Modulation of CCR5 and CXCR4 HIV-1 coreceptor activities as a therapeutic strategy : studying the two CXCR4 isoforms and the interaction of CCR5 with S1P1.” 2013. Doctoral Dissertation, Université Montpellier II. Accessed June 17, 2019. http://www.theses.fr/2013MON20188.

MLA Handbook (7th Edition):

Duquenne, Charline. “Modulation de l’activité des corécepteurs CCR5 et CXCR4 du VIH 1 comme stratégie thérapeutique : étude des deux isoformes de CXCR4 et interaction de CCR5 avec le récepteur S1P1 : Modulation of CCR5 and CXCR4 HIV-1 coreceptor activities as a therapeutic strategy : studying the two CXCR4 isoforms and the interaction of CCR5 with S1P1.” 2013. Web. 17 Jun 2019.

Vancouver:

Duquenne C. Modulation de l’activité des corécepteurs CCR5 et CXCR4 du VIH 1 comme stratégie thérapeutique : étude des deux isoformes de CXCR4 et interaction de CCR5 avec le récepteur S1P1 : Modulation of CCR5 and CXCR4 HIV-1 coreceptor activities as a therapeutic strategy : studying the two CXCR4 isoforms and the interaction of CCR5 with S1P1. [Internet] [Doctoral dissertation]. Université Montpellier II; 2013. [cited 2019 Jun 17]. Available from: http://www.theses.fr/2013MON20188.

Council of Science Editors:

Duquenne C. Modulation de l’activité des corécepteurs CCR5 et CXCR4 du VIH 1 comme stratégie thérapeutique : étude des deux isoformes de CXCR4 et interaction de CCR5 avec le récepteur S1P1 : Modulation of CCR5 and CXCR4 HIV-1 coreceptor activities as a therapeutic strategy : studying the two CXCR4 isoforms and the interaction of CCR5 with S1P1. [Doctoral Dissertation]. Université Montpellier II; 2013. Available from: http://www.theses.fr/2013MON20188

16. Manin, Graziele Zenaro. Identificação dos componentes do Sistema Imune que participam na resistência de camundongos em modelo de infecção letal por Legionella longbeachae.

Degree: Mestrado, Imunologia Básica e Aplicada, 2014, University of São Paulo

A doença dos legionários consiste em uma broncopneumonia severa e atípica, que acomete de 2 a 7% das pessoas infectadas com Legionella spp e que… (more)

Subjects/Keywords: Legionella longbeachae; Legionella longbeachae; CCR2; CCR2; CCR5; CCR5; Doença dos legionários; IL-17; IL-17; Legionnaires disease

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APA (6th Edition):

Manin, G. Z. (2014). Identificação dos componentes do Sistema Imune que participam na resistência de camundongos em modelo de infecção letal por Legionella longbeachae. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21052014-153321/ ;

Chicago Manual of Style (16th Edition):

Manin, Graziele Zenaro. “Identificação dos componentes do Sistema Imune que participam na resistência de camundongos em modelo de infecção letal por Legionella longbeachae.” 2014. Masters Thesis, University of São Paulo. Accessed June 17, 2019. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21052014-153321/ ;.

MLA Handbook (7th Edition):

Manin, Graziele Zenaro. “Identificação dos componentes do Sistema Imune que participam na resistência de camundongos em modelo de infecção letal por Legionella longbeachae.” 2014. Web. 17 Jun 2019.

Vancouver:

Manin GZ. Identificação dos componentes do Sistema Imune que participam na resistência de camundongos em modelo de infecção letal por Legionella longbeachae. [Internet] [Masters thesis]. University of São Paulo; 2014. [cited 2019 Jun 17]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21052014-153321/ ;.

Council of Science Editors:

Manin GZ. Identificação dos componentes do Sistema Imune que participam na resistência de camundongos em modelo de infecção letal por Legionella longbeachae. [Masters Thesis]. University of São Paulo; 2014. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-21052014-153321/ ;

17. Bonfá, Giuliano. Papel de CCR5 na infecção oral por Toxoplasma gondii.

Degree: Mestrado, Imunologia Básica e Aplicada, 2010, University of São Paulo

Toxoplasma gondii é um protozoário intracelular obrigatório que causa a toxoplasmose. Em modelo experimental, camundongos C57BL/6 infectados por via oral com 100 cistos de T.… (more)

Subjects/Keywords: Toxoplasma gondii; Toxolasma gondii; Chemokine receptor CCR5; Infecção oral; Lesão tecidual.; Oral infection; Receptor de quimiocina CCR5; Tissue injury

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APA (6th Edition):

Bonfá, G. (2010). Papel de CCR5 na infecção oral por Toxoplasma gondii. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/17/17147/tde-08072010-152818/ ;

Chicago Manual of Style (16th Edition):

Bonfá, Giuliano. “Papel de CCR5 na infecção oral por Toxoplasma gondii.” 2010. Masters Thesis, University of São Paulo. Accessed June 17, 2019. http://www.teses.usp.br/teses/disponiveis/17/17147/tde-08072010-152818/ ;.

MLA Handbook (7th Edition):

Bonfá, Giuliano. “Papel de CCR5 na infecção oral por Toxoplasma gondii.” 2010. Web. 17 Jun 2019.

Vancouver:

Bonfá G. Papel de CCR5 na infecção oral por Toxoplasma gondii. [Internet] [Masters thesis]. University of São Paulo; 2010. [cited 2019 Jun 17]. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-08072010-152818/ ;.

Council of Science Editors:

Bonfá G. Papel de CCR5 na infecção oral por Toxoplasma gondii. [Masters Thesis]. University of São Paulo; 2010. Available from: http://www.teses.usp.br/teses/disponiveis/17/17147/tde-08072010-152818/ ;

18. Ramalho, Rodrigo Fernandes. Análise do polimorfismo da região cis-reguladora do gene CCR5 em populações ameríndias.

Degree: Mestrado, Biologia (Genética), 2008, University of São Paulo

Populações nativas da América do Sul apresentam diversidade genética reduzida em relação às demais populações do mundo e alta diferenciação interpopulacional dentro do continente. As… (more)

Subjects/Keywords: Amerindians; Ameríndios; CCR5 5´cis-regulatory region; Demografia; Demography; Natural selection; Região cis-reguladora do gene CCR5; Seleção natural

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APA (6th Edition):

Ramalho, R. F. (2008). Análise do polimorfismo da região cis-reguladora do gene CCR5 em populações ameríndias. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/41/41131/tde-05032008-145950/ ;

Chicago Manual of Style (16th Edition):

Ramalho, Rodrigo Fernandes. “Análise do polimorfismo da região cis-reguladora do gene CCR5 em populações ameríndias.” 2008. Masters Thesis, University of São Paulo. Accessed June 17, 2019. http://www.teses.usp.br/teses/disponiveis/41/41131/tde-05032008-145950/ ;.

MLA Handbook (7th Edition):

Ramalho, Rodrigo Fernandes. “Análise do polimorfismo da região cis-reguladora do gene CCR5 em populações ameríndias.” 2008. Web. 17 Jun 2019.

Vancouver:

Ramalho RF. Análise do polimorfismo da região cis-reguladora do gene CCR5 em populações ameríndias. [Internet] [Masters thesis]. University of São Paulo; 2008. [cited 2019 Jun 17]. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-05032008-145950/ ;.

Council of Science Editors:

Ramalho RF. Análise do polimorfismo da região cis-reguladora do gene CCR5 em populações ameríndias. [Masters Thesis]. University of São Paulo; 2008. Available from: http://www.teses.usp.br/teses/disponiveis/41/41131/tde-05032008-145950/ ;


Virginia Commonwealth University

19. Ahmed, Tasrif. Discovery of a Novel CCR5 Antagonist as an Effective Therapeutic Agent for Prostate Cancer.

Degree: MS, Physiology, 2010, Virginia Commonwealth University

 Previously, the CCR5 receptor was found to be a good target for treating prostate cancer (PCa). Dr. Yan Zhang’s laboratory designed several CCR5 antagonists, which… (more)

Subjects/Keywords: prostate; prostate cancer; ccr5; ccl5; ccr5 antagonists; PCa; drug; drug discovery; tasrif; ahmed; chemokine; receptor; ligand; Life Sciences; Physiology

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APA (6th Edition):

Ahmed, T. (2010). Discovery of a Novel CCR5 Antagonist as an Effective Therapeutic Agent for Prostate Cancer. (Thesis). Virginia Commonwealth University. Retrieved from https://scholarscompass.vcu.edu/etd/2215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ahmed, Tasrif. “Discovery of a Novel CCR5 Antagonist as an Effective Therapeutic Agent for Prostate Cancer.” 2010. Thesis, Virginia Commonwealth University. Accessed June 17, 2019. https://scholarscompass.vcu.edu/etd/2215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ahmed, Tasrif. “Discovery of a Novel CCR5 Antagonist as an Effective Therapeutic Agent for Prostate Cancer.” 2010. Web. 17 Jun 2019.

Vancouver:

Ahmed T. Discovery of a Novel CCR5 Antagonist as an Effective Therapeutic Agent for Prostate Cancer. [Internet] [Thesis]. Virginia Commonwealth University; 2010. [cited 2019 Jun 17]. Available from: https://scholarscompass.vcu.edu/etd/2215.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ahmed T. Discovery of a Novel CCR5 Antagonist as an Effective Therapeutic Agent for Prostate Cancer. [Thesis]. Virginia Commonwealth University; 2010. Available from: https://scholarscompass.vcu.edu/etd/2215

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

20. Conforti, Rosa. Traitement anti tumoral par ciblage de TLR3 et découplage des effets opposés des chimiokines pour améliorer l’efficacité des agonistes de TLR3 : Anticancer treatment by TLR3 targeting and uncoupling of the chemokines opposing effects to optimize the efficacy of TLR3 agonists.

Degree: Docteur es, Cancérologie, 2011, Université Paris-Sud – Paris XI

Le rationnel pour l’utilisation des agonistes des Toll-Like Récepteurs (TLR) dans le traitement du cancer repose sur leurs effets bénéfiques au niveau des cellules du… (more)

Subjects/Keywords: Immunité tumorale; TLR3; Poly(A:U); CCR5; MetRANTES; CXCR3; Tumor immunity; TLR3; Poly(A:U); CCR5; MetRANTES; CXCR3

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APA (6th Edition):

Conforti, R. (2011). Traitement anti tumoral par ciblage de TLR3 et découplage des effets opposés des chimiokines pour améliorer l’efficacité des agonistes de TLR3 : Anticancer treatment by TLR3 targeting and uncoupling of the chemokines opposing effects to optimize the efficacy of TLR3 agonists. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2011PA11T067

Chicago Manual of Style (16th Edition):

Conforti, Rosa. “Traitement anti tumoral par ciblage de TLR3 et découplage des effets opposés des chimiokines pour améliorer l’efficacité des agonistes de TLR3 : Anticancer treatment by TLR3 targeting and uncoupling of the chemokines opposing effects to optimize the efficacy of TLR3 agonists.” 2011. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed June 17, 2019. http://www.theses.fr/2011PA11T067.

MLA Handbook (7th Edition):

Conforti, Rosa. “Traitement anti tumoral par ciblage de TLR3 et découplage des effets opposés des chimiokines pour améliorer l’efficacité des agonistes de TLR3 : Anticancer treatment by TLR3 targeting and uncoupling of the chemokines opposing effects to optimize the efficacy of TLR3 agonists.” 2011. Web. 17 Jun 2019.

Vancouver:

Conforti R. Traitement anti tumoral par ciblage de TLR3 et découplage des effets opposés des chimiokines pour améliorer l’efficacité des agonistes de TLR3 : Anticancer treatment by TLR3 targeting and uncoupling of the chemokines opposing effects to optimize the efficacy of TLR3 agonists. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2011. [cited 2019 Jun 17]. Available from: http://www.theses.fr/2011PA11T067.

Council of Science Editors:

Conforti R. Traitement anti tumoral par ciblage de TLR3 et découplage des effets opposés des chimiokines pour améliorer l’efficacité des agonistes de TLR3 : Anticancer treatment by TLR3 targeting and uncoupling of the chemokines opposing effects to optimize the efficacy of TLR3 agonists. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2011. Available from: http://www.theses.fr/2011PA11T067


Dalhousie University

21. Charette, Nicholle Jeanine. INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS.

Degree: MS, Department of Pharmacology, 2011, Dalhousie University

 Little is known about the outward trafficking of receptor dimers from the endoplasmic reticulum to the plasma membrane, or the role that trafficking plays in… (more)

Subjects/Keywords: G protein coupled receptor; CXCR4; CCR5; Rab GTPase; Trafficking; Dimerization

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APA (6th Edition):

Charette, N. J. (2011). INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS. (Masters Thesis). Dalhousie University. Retrieved from http://hdl.handle.net/10222/14023

Chicago Manual of Style (16th Edition):

Charette, Nicholle Jeanine. “INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS.” 2011. Masters Thesis, Dalhousie University. Accessed June 17, 2019. http://hdl.handle.net/10222/14023.

MLA Handbook (7th Edition):

Charette, Nicholle Jeanine. “INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS.” 2011. Web. 17 Jun 2019.

Vancouver:

Charette NJ. INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS. [Internet] [Masters thesis]. Dalhousie University; 2011. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/10222/14023.

Council of Science Editors:

Charette NJ. INVOLVEMENT OF DIFFERENT RAB GTPASES IN THE TRAFFICKING OF CXCR4 AND CCR5 HOMO- AND HETERODIMERS BETWEEN THE ENDOPLASMIC RETICULUM AND PLASMA MEMBRANE IN HEK293 AND JURKAT CELLS. [Masters Thesis]. Dalhousie University; 2011. Available from: http://hdl.handle.net/10222/14023

22. GOMES, Adriana Vieira. História Genético-Molecular da População Atual do Nordeste Brasileiro .

Degree: 2010, Universidade Federal de Pernambuco

 O Brasil tornou-se objeto de interesse de estudos genético-populacionais devido às diferenças, fenotípica e social, verificadas entre as populações das cinco regiões geográficas do país.… (more)

Subjects/Keywords: Polimorfismos Genéticos; STRs; ccr5-∆32; Nordeste Brasileiro; Genetic Polymorphism; Northeast Brazil

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APA (6th Edition):

GOMES, A. V. (2010). História Genético-Molecular da População Atual do Nordeste Brasileiro . (Thesis). Universidade Federal de Pernambuco. Retrieved from http://repositorio.ufpe.br/handle/123456789/18507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

GOMES, Adriana Vieira. “História Genético-Molecular da População Atual do Nordeste Brasileiro .” 2010. Thesis, Universidade Federal de Pernambuco. Accessed June 17, 2019. http://repositorio.ufpe.br/handle/123456789/18507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

GOMES, Adriana Vieira. “História Genético-Molecular da População Atual do Nordeste Brasileiro .” 2010. Web. 17 Jun 2019.

Vancouver:

GOMES AV. História Genético-Molecular da População Atual do Nordeste Brasileiro . [Internet] [Thesis]. Universidade Federal de Pernambuco; 2010. [cited 2019 Jun 17]. Available from: http://repositorio.ufpe.br/handle/123456789/18507.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

GOMES AV. História Genético-Molecular da População Atual do Nordeste Brasileiro . [Thesis]. Universidade Federal de Pernambuco; 2010. Available from: http://repositorio.ufpe.br/handle/123456789/18507

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. 古橋, 隆; フルハシ, タカシ. 移植前ドナー血輸血による免疫学的不応答状態の誘導、樹状細胞のフェノタイプ及び脾臓への特異的遊走 ・ 局在に関する研究 : イショクマエ ドナーケツ ユケツ ニヨル メンエキガクテキ フオウトウ ジョウタイ ノ ユウドウ ジュジョウ サイボウ ノ フェノタイプ オヨビ ヒゾウ エノ トクイテキ ユウソウ キョクザイ ニカンスル ケンキュウ; Induction of immunological unresponsiveness by donor blood transfusion prior to liver transplantation in rat-Role of dendritic cell migration from donor liver to recipient spleen.

Degree: Kumamoto University / 熊本大学

Subjects/Keywords: サイトカインプロフィール; ケモカインレセプター; CD62L; CCR5; CCR7

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APA (6th Edition):

古橋, 隆; フルハシ, . (n.d.). 移植前ドナー血輸血による免疫学的不応答状態の誘導、樹状細胞のフェノタイプ及び脾臓への特異的遊走 ・ 局在に関する研究 : イショクマエ ドナーケツ ユケツ ニヨル メンエキガクテキ フオウトウ ジョウタイ ノ ユウドウ ジュジョウ サイボウ ノ フェノタイプ オヨビ ヒゾウ エノ トクイテキ ユウソウ キョクザイ ニカンスル ケンキュウ; Induction of immunological unresponsiveness by donor blood transfusion prior to liver transplantation in rat-Role of dendritic cell migration from donor liver to recipient spleen. (Thesis). Kumamoto University / 熊本大学. Retrieved from http://hdl.handle.net/2298/3104

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

古橋, 隆; フルハシ, タカシ. “移植前ドナー血輸血による免疫学的不応答状態の誘導、樹状細胞のフェノタイプ及び脾臓への特異的遊走 ・ 局在に関する研究 : イショクマエ ドナーケツ ユケツ ニヨル メンエキガクテキ フオウトウ ジョウタイ ノ ユウドウ ジュジョウ サイボウ ノ フェノタイプ オヨビ ヒゾウ エノ トクイテキ ユウソウ キョクザイ ニカンスル ケンキュウ; Induction of immunological unresponsiveness by donor blood transfusion prior to liver transplantation in rat-Role of dendritic cell migration from donor liver to recipient spleen.” Thesis, Kumamoto University / 熊本大学. Accessed June 17, 2019. http://hdl.handle.net/2298/3104.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

古橋, 隆; フルハシ, タカシ. “移植前ドナー血輸血による免疫学的不応答状態の誘導、樹状細胞のフェノタイプ及び脾臓への特異的遊走 ・ 局在に関する研究 : イショクマエ ドナーケツ ユケツ ニヨル メンエキガクテキ フオウトウ ジョウタイ ノ ユウドウ ジュジョウ サイボウ ノ フェノタイプ オヨビ ヒゾウ エノ トクイテキ ユウソウ キョクザイ ニカンスル ケンキュウ; Induction of immunological unresponsiveness by donor blood transfusion prior to liver transplantation in rat-Role of dendritic cell migration from donor liver to recipient spleen.” Web. 17 Jun 2019.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

古橋, 隆; フルハシ . 移植前ドナー血輸血による免疫学的不応答状態の誘導、樹状細胞のフェノタイプ及び脾臓への特異的遊走 ・ 局在に関する研究 : イショクマエ ドナーケツ ユケツ ニヨル メンエキガクテキ フオウトウ ジョウタイ ノ ユウドウ ジュジョウ サイボウ ノ フェノタイプ オヨビ ヒゾウ エノ トクイテキ ユウソウ キョクザイ ニカンスル ケンキュウ; Induction of immunological unresponsiveness by donor blood transfusion prior to liver transplantation in rat-Role of dendritic cell migration from donor liver to recipient spleen. [Internet] [Thesis]. Kumamoto University / 熊本大学; [cited 2019 Jun 17]. Available from: http://hdl.handle.net/2298/3104.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

古橋, 隆; フルハシ . 移植前ドナー血輸血による免疫学的不応答状態の誘導、樹状細胞のフェノタイプ及び脾臓への特異的遊走 ・ 局在に関する研究 : イショクマエ ドナーケツ ユケツ ニヨル メンエキガクテキ フオウトウ ジョウタイ ノ ユウドウ ジュジョウ サイボウ ノ フェノタイプ オヨビ ヒゾウ エノ トクイテキ ユウソウ キョクザイ ニカンスル ケンキュウ; Induction of immunological unresponsiveness by donor blood transfusion prior to liver transplantation in rat-Role of dendritic cell migration from donor liver to recipient spleen. [Thesis]. Kumamoto University / 熊本大学; Available from: http://hdl.handle.net/2298/3104

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


Universidade do Rio Grande do Sul

24. Kohem, Charles Lubianca. Estudo do polimorfismo e expressão do CCR5 em pacientes com artrite reumatóide.

Degree: 2005, Universidade do Rio Grande do Sul

Resumo não disponível Advisors/Committee Members: Brenol, João Carlos Tavares.

Subjects/Keywords: Artrite reumatóide; Receptores CCR5; Polimorfismo

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kohem, C. L. (2005). Estudo do polimorfismo e expressão do CCR5 em pacientes com artrite reumatóide. (Thesis). Universidade do Rio Grande do Sul. Retrieved from http://hdl.handle.net/10183/5218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kohem, Charles Lubianca. “Estudo do polimorfismo e expressão do CCR5 em pacientes com artrite reumatóide.” 2005. Thesis, Universidade do Rio Grande do Sul. Accessed June 17, 2019. http://hdl.handle.net/10183/5218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kohem, Charles Lubianca. “Estudo do polimorfismo e expressão do CCR5 em pacientes com artrite reumatóide.” 2005. Web. 17 Jun 2019.

Vancouver:

Kohem CL. Estudo do polimorfismo e expressão do CCR5 em pacientes com artrite reumatóide. [Internet] [Thesis]. Universidade do Rio Grande do Sul; 2005. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/10183/5218.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kohem CL. Estudo do polimorfismo e expressão do CCR5 em pacientes com artrite reumatóide. [Thesis]. Universidade do Rio Grande do Sul; 2005. Available from: http://hdl.handle.net/10183/5218

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

25. Nahabedian, John Francis. Mapping the determinants in owl monkey CD4 and CCR5 that allow entry of early-stage HIV-1 Env variants.

Degree: 2016, University of Washington

 Many physiological and immunological aspects of non-human primates make them ideal candidates for animal models of HIV-1 transmission and vaccine studies. However, HIV-1 does not… (more)

Subjects/Keywords: animal; CCR5; CD4; Envelope; HIV; receptor; Virology; pathobiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Nahabedian, J. F. (2016). Mapping the determinants in owl monkey CD4 and CCR5 that allow entry of early-stage HIV-1 Env variants. (Thesis). University of Washington. Retrieved from http://hdl.handle.net/1773/36800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nahabedian, John Francis. “Mapping the determinants in owl monkey CD4 and CCR5 that allow entry of early-stage HIV-1 Env variants.” 2016. Thesis, University of Washington. Accessed June 17, 2019. http://hdl.handle.net/1773/36800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nahabedian, John Francis. “Mapping the determinants in owl monkey CD4 and CCR5 that allow entry of early-stage HIV-1 Env variants.” 2016. Web. 17 Jun 2019.

Vancouver:

Nahabedian JF. Mapping the determinants in owl monkey CD4 and CCR5 that allow entry of early-stage HIV-1 Env variants. [Internet] [Thesis]. University of Washington; 2016. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/1773/36800.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nahabedian JF. Mapping the determinants in owl monkey CD4 and CCR5 that allow entry of early-stage HIV-1 Env variants. [Thesis]. University of Washington; 2016. Available from: http://hdl.handle.net/1773/36800

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

26. Gao, Fei Darrin. CCL5-CCR5 Interactions in Breast Cancer Regulate Cell Metabolism.

Degree: PhD, 2017, University of Toronto

 Chemokines are chemotactic cytokines that recruit and activate leukocytes. It is now apparent that they also participate actively in the neoplastic process in cancer. This… (more)

Subjects/Keywords: Anabolism; Breast Cancer; CCL5; CCR5; Chemokine; Metabolism; 0982

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gao, F. D. (2017). CCL5-CCR5 Interactions in Breast Cancer Regulate Cell Metabolism. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/80877

Chicago Manual of Style (16th Edition):

Gao, Fei Darrin. “CCL5-CCR5 Interactions in Breast Cancer Regulate Cell Metabolism.” 2017. Doctoral Dissertation, University of Toronto. Accessed June 17, 2019. http://hdl.handle.net/1807/80877.

MLA Handbook (7th Edition):

Gao, Fei Darrin. “CCL5-CCR5 Interactions in Breast Cancer Regulate Cell Metabolism.” 2017. Web. 17 Jun 2019.

Vancouver:

Gao FD. CCL5-CCR5 Interactions in Breast Cancer Regulate Cell Metabolism. [Internet] [Doctoral dissertation]. University of Toronto; 2017. [cited 2019 Jun 17]. Available from: http://hdl.handle.net/1807/80877.

Council of Science Editors:

Gao FD. CCL5-CCR5 Interactions in Breast Cancer Regulate Cell Metabolism. [Doctoral Dissertation]. University of Toronto; 2017. Available from: http://hdl.handle.net/1807/80877


University of New Mexico

27. Hunter, Zoe. Induction of mucosal immunity using virus-like particle based vaccines.

Degree: Biomedical Sciences Graduate Program, 2011, University of New Mexico

 Many viral structural proteins are capable of spontaneously self-assembling into structures that resemble virus particles. These structures, called virus-like particles (VLPs), have multivalent, highly repetitive… (more)

Subjects/Keywords: virus-like particles; CCR5; mucosal immunity; vaccine; HPV16 L2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hunter, Z. (2011). Induction of mucosal immunity using virus-like particle based vaccines. (Doctoral Dissertation). University of New Mexico. Retrieved from https://digitalrepository.unm.edu/biom_etds/31

Chicago Manual of Style (16th Edition):

Hunter, Zoe. “Induction of mucosal immunity using virus-like particle based vaccines.” 2011. Doctoral Dissertation, University of New Mexico. Accessed June 17, 2019. https://digitalrepository.unm.edu/biom_etds/31.

MLA Handbook (7th Edition):

Hunter, Zoe. “Induction of mucosal immunity using virus-like particle based vaccines.” 2011. Web. 17 Jun 2019.

Vancouver:

Hunter Z. Induction of mucosal immunity using virus-like particle based vaccines. [Internet] [Doctoral dissertation]. University of New Mexico; 2011. [cited 2019 Jun 17]. Available from: https://digitalrepository.unm.edu/biom_etds/31.

Council of Science Editors:

Hunter Z. Induction of mucosal immunity using virus-like particle based vaccines. [Doctoral Dissertation]. University of New Mexico; 2011. Available from: https://digitalrepository.unm.edu/biom_etds/31


University of Pennsylvania

28. Riddick, Nadeene E. A Novel CCR5 Mutation in Sooty Mangabeys Reveals SIVsmm Infection of CCR5-null Natural Hosts: Examining the Potential Roles of Alternative Entry Pathways in HIV and SIV Infection.

Degree: 2012, University of Pennsylvania

 Natural hosts of SIV, such as sooty mangabeys (SM), maintain high levels of virus replication, but do not typically develop CD4+ T cell loss and… (more)

Subjects/Keywords: alternative; CCR5; coreceptors; HIV; mutants; SIV; Molecular Biology; Virology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Riddick, N. E. (2012). A Novel CCR5 Mutation in Sooty Mangabeys Reveals SIVsmm Infection of CCR5-null Natural Hosts: Examining the Potential Roles of Alternative Entry Pathways in HIV and SIV Infection. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Riddick, Nadeene E. “A Novel CCR5 Mutation in Sooty Mangabeys Reveals SIVsmm Infection of CCR5-null Natural Hosts: Examining the Potential Roles of Alternative Entry Pathways in HIV and SIV Infection.” 2012. Thesis, University of Pennsylvania. Accessed June 17, 2019. https://repository.upenn.edu/edissertations/566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Riddick, Nadeene E. “A Novel CCR5 Mutation in Sooty Mangabeys Reveals SIVsmm Infection of CCR5-null Natural Hosts: Examining the Potential Roles of Alternative Entry Pathways in HIV and SIV Infection.” 2012. Web. 17 Jun 2019.

Vancouver:

Riddick NE. A Novel CCR5 Mutation in Sooty Mangabeys Reveals SIVsmm Infection of CCR5-null Natural Hosts: Examining the Potential Roles of Alternative Entry Pathways in HIV and SIV Infection. [Internet] [Thesis]. University of Pennsylvania; 2012. [cited 2019 Jun 17]. Available from: https://repository.upenn.edu/edissertations/566.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Riddick NE. A Novel CCR5 Mutation in Sooty Mangabeys Reveals SIVsmm Infection of CCR5-null Natural Hosts: Examining the Potential Roles of Alternative Entry Pathways in HIV and SIV Infection. [Thesis]. University of Pennsylvania; 2012. Available from: https://repository.upenn.edu/edissertations/566

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Pennsylvania

29. Parker, Zahra Folasade. Insights into the Role of Chemokines and Chemokine Receptors During HIV-1 Pathogenesis.

Degree: 2015, University of Pennsylvania

 Sexual transmission of HIV-1 is often established by one genetic variant, the transmitted/founder (T/F) virus. T/F HIV-1 may have specific phenotypic properties that are selected… (more)

Subjects/Keywords: CCR5; Chemokine receptors; Chemokines; HIV-1; PF4; Microbiology; Virology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Parker, Z. F. (2015). Insights into the Role of Chemokines and Chemokine Receptors During HIV-1 Pathogenesis. (Thesis). University of Pennsylvania. Retrieved from https://repository.upenn.edu/edissertations/1931

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Parker, Zahra Folasade. “Insights into the Role of Chemokines and Chemokine Receptors During HIV-1 Pathogenesis.” 2015. Thesis, University of Pennsylvania. Accessed June 17, 2019. https://repository.upenn.edu/edissertations/1931.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Parker, Zahra Folasade. “Insights into the Role of Chemokines and Chemokine Receptors During HIV-1 Pathogenesis.” 2015. Web. 17 Jun 2019.

Vancouver:

Parker ZF. Insights into the Role of Chemokines and Chemokine Receptors During HIV-1 Pathogenesis. [Internet] [Thesis]. University of Pennsylvania; 2015. [cited 2019 Jun 17]. Available from: https://repository.upenn.edu/edissertations/1931.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Parker ZF. Insights into the Role of Chemokines and Chemokine Receptors During HIV-1 Pathogenesis. [Thesis]. University of Pennsylvania; 2015. Available from: https://repository.upenn.edu/edissertations/1931

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Ferreira Junior, Samuel de Barros. Modulação da severidade da doença periodontal experimental por células CCR5+.

Degree: Mestrado, Biologia Oral, 2009, University of São Paulo

As doenças periodontais (DP) afetam os tecidos de suporte dos dentes e são desencadeadas por micro-organismos gram-negativos anaeróbios presentes no biofilme periodontal. A evolução da… (more)

Subjects/Keywords: Aggregatibacter Actinomycetemcomitans; Aggregatibacter Actinomycetemcomitans; Bone resorption; CCR5 Receptors; Células Th1; Chemokines; Citocinas; Cytokines; Periodontite; Periodontitis; Quimiocinas; RANKL; RANKL; Reabsorção óssea; Receptores CCR5; Th1 cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ferreira Junior, S. d. B. (2009). Modulação da severidade da doença periodontal experimental por células CCR5+. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/25/25142/tde-02072009-112551/ ;

Chicago Manual of Style (16th Edition):

Ferreira Junior, Samuel de Barros. “Modulação da severidade da doença periodontal experimental por células CCR5+.” 2009. Masters Thesis, University of São Paulo. Accessed June 17, 2019. http://www.teses.usp.br/teses/disponiveis/25/25142/tde-02072009-112551/ ;.

MLA Handbook (7th Edition):

Ferreira Junior, Samuel de Barros. “Modulação da severidade da doença periodontal experimental por células CCR5+.” 2009. Web. 17 Jun 2019.

Vancouver:

Ferreira Junior SdB. Modulação da severidade da doença periodontal experimental por células CCR5+. [Internet] [Masters thesis]. University of São Paulo; 2009. [cited 2019 Jun 17]. Available from: http://www.teses.usp.br/teses/disponiveis/25/25142/tde-02072009-112551/ ;.

Council of Science Editors:

Ferreira Junior SdB. Modulação da severidade da doença periodontal experimental por células CCR5+. [Masters Thesis]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/25/25142/tde-02072009-112551/ ;

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