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You searched for subject:(C jun n terminal kinase). Showing records 1 – 30 of 21185 total matches.

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1. Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, Kosuke. Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する.

Degree: 博士(医学), 2015, Nara Medical University / 奈良県立医科大学

Acute aortic dissection (AAD) is a life-threating disease; however, there is almost no effective pharmacotherapy for it. An increase in c-Jun N-terminal kinase (JNK) phosphorylation… (more)

Subjects/Keywords: Stretch; c-Jun N-terminal kinase; p38; Acute aortic dissection; Olmesartan

Page 1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, K. (2015). Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する. (Thesis). Nara Medical University / 奈良県立医科大学. Retrieved from http://hdl.handle.net/10564/2939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, Kosuke. “Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する.” 2015. Thesis, Nara Medical University / 奈良県立医科大学. Accessed April 25, 2019. http://hdl.handle.net/10564/2939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama, Kosuke. “Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する.” 2015. Web. 25 Apr 2019.

Vancouver:

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama K. Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する. [Internet] [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10564/2939.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ito, Satoyasu; Ozawa, Kentaro; Zhao, Jing; Kyotani, Yoji; Nagayama K. Olmesartan inhibits cultured rat aortic smooth muscle cell death induced by cyclic mechanical stretch through the inhibition of the c-Jun N-terminal kinase and p38 signaling pathways. : オルメサルタンは、JNK及びp38シグナル経路を抑制することで、周期的機械的伸展による培養大動脈平滑筋細胞死を抑制する. [Thesis]. Nara Medical University / 奈良県立医科大学; 2015. Available from: http://hdl.handle.net/10564/2939

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kansas

2. Saito, Chieko. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.

Degree: PhD, Pharmacology, Toxicology & Therapeutics, 2010, University of Kansas

 Acetaminophen (APAP) is a widely used analgesic, which is safe at therapeutic levels. APAP is mainly conjugated with glucuronic acid and sulfate to form water-soluble,… (more)

Subjects/Keywords: Health sciences; Toxicology; Acetaminophen; C-jun n-terminal kinase; Glutathione; Metallothionein; N-acetylcysteine

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APA (6th Edition):

Saito, C. (2010). PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. (Doctoral Dissertation). University of Kansas. Retrieved from http://hdl.handle.net/1808/6369

Chicago Manual of Style (16th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Doctoral Dissertation, University of Kansas. Accessed April 25, 2019. http://hdl.handle.net/1808/6369.

MLA Handbook (7th Edition):

Saito, Chieko. “PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY.” 2010. Web. 25 Apr 2019.

Vancouver:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Internet] [Doctoral dissertation]. University of Kansas; 2010. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1808/6369.

Council of Science Editors:

Saito C. PROTECTIVE STRATEGIES AGAINST ACETAMINOPHEN INDUCED HEPATOTOXICITY. [Doctoral Dissertation]. University of Kansas; 2010. Available from: http://hdl.handle.net/1808/6369


University of Illinois – Chicago

3. Lowry, Jessica L. A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium.

Degree: 2013, University of Illinois – Chicago

 High levels of nitric oxide (NO) generated in the vasculature under inflammatory conditions are usually attributed to inducible nitric oxide synthase (iNOS), but the role… (more)

Subjects/Keywords: Nitric Oxide; c-Jun-N-Terminal Kinase; endothelial nitric oxide synthase; inflammation; migration

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APA (6th Edition):

Lowry, J. L. (2013). A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium. (Thesis). University of Illinois – Chicago. Retrieved from http://hdl.handle.net/10027/10054

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lowry, Jessica L. “A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium.” 2013. Thesis, University of Illinois – Chicago. Accessed April 25, 2019. http://hdl.handle.net/10027/10054.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lowry, Jessica L. “A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium.” 2013. Web. 25 Apr 2019.

Vancouver:

Lowry JL. A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium. [Internet] [Thesis]. University of Illinois – Chicago; 2013. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10027/10054.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lowry JL. A Novel c-Jun-N-Terminal Kinase Pathway Stimulates High Output eNOS-derived NO in Inflamed Endothelium. [Thesis]. University of Illinois – Chicago; 2013. Available from: http://hdl.handle.net/10027/10054

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Washington

4. Kuhar, Jamie Rose. Mechanisms of Opioid Receptor Desensitization.

Degree: PhD, 2015, University of Washington

 Agonists targeting MOR are effective analgesics, but their clinical use is hindered by side effects, including tolerance and addiction. KOR agonists also produce analgesia, but… (more)

Subjects/Keywords: Arrestin; c-Jun N-terminal kinase; Fentanyl; Morphine; Opioid; p38; Pharmacology; pharmacology

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APA (6th Edition):

Kuhar, J. R. (2015). Mechanisms of Opioid Receptor Desensitization. (Doctoral Dissertation). University of Washington. Retrieved from http://hdl.handle.net/1773/34122

Chicago Manual of Style (16th Edition):

Kuhar, Jamie Rose. “Mechanisms of Opioid Receptor Desensitization.” 2015. Doctoral Dissertation, University of Washington. Accessed April 25, 2019. http://hdl.handle.net/1773/34122.

MLA Handbook (7th Edition):

Kuhar, Jamie Rose. “Mechanisms of Opioid Receptor Desensitization.” 2015. Web. 25 Apr 2019.

Vancouver:

Kuhar JR. Mechanisms of Opioid Receptor Desensitization. [Internet] [Doctoral dissertation]. University of Washington; 2015. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1773/34122.

Council of Science Editors:

Kuhar JR. Mechanisms of Opioid Receptor Desensitization. [Doctoral Dissertation]. University of Washington; 2015. Available from: http://hdl.handle.net/1773/34122


University of Southern Mississippi

5. Chen, Qichuan. The Drosophila T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival.

Degree: MS, Biological Sciences, 2014, University of Southern Mississippi

  From a genetic and allelic modifier screen, we report that the Drosophila melanogaster T-box transcription factor midline (mid), a homolog to the human TBX20… (more)

Subjects/Keywords: mid; midline; Tbx20; dFOXO; C-Jun-N-terminal kinase; insulin receptor; Developmental Biology

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APA (6th Edition):

Chen, Q. (2014). The Drosophila T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival. (Masters Thesis). University of Southern Mississippi. Retrieved from https://aquila.usm.edu/masters_theses/76

Chicago Manual of Style (16th Edition):

Chen, Qichuan. “The Drosophila T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival.” 2014. Masters Thesis, University of Southern Mississippi. Accessed April 25, 2019. https://aquila.usm.edu/masters_theses/76.

MLA Handbook (7th Edition):

Chen, Qichuan. “The Drosophila T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival.” 2014. Web. 25 Apr 2019.

Vancouver:

Chen Q. The Drosophila T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival. [Internet] [Masters thesis]. University of Southern Mississippi; 2014. [cited 2019 Apr 25]. Available from: https://aquila.usm.edu/masters_theses/76.

Council of Science Editors:

Chen Q. The Drosophila T-box Transcription Factor Midline Functions within the Insulin/AKT and c-Jun-N-terminal Kinase Signaling Pathways to Regulate Interomatidial Bristle Formation and Cell Survival. [Masters Thesis]. University of Southern Mississippi; 2014. Available from: https://aquila.usm.edu/masters_theses/76


University of Toronto

6. Tang, Christine. Mechanisms of High Glucose-induced Decrease in β-cell Function.

Degree: 2010, University of Toronto

Chronic hyperglycemia, a hallmark of type 2 diabetes, can decrease β-cell function and mass (β-cell glucotoxicity); however, the mechanisms are incompletely understood. The objective was… (more)

Subjects/Keywords: Glucotoxicity; Oxidative Stress; Type 2 Diabetes; Endoplasmic Reticulum Stress; c-jun N-terminal kinase; 0719

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APA (6th Edition):

Tang, C. (2010). Mechanisms of High Glucose-induced Decrease in β-cell Function. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/26331

Chicago Manual of Style (16th Edition):

Tang, Christine. “Mechanisms of High Glucose-induced Decrease in β-cell Function.” 2010. Doctoral Dissertation, University of Toronto. Accessed April 25, 2019. http://hdl.handle.net/1807/26331.

MLA Handbook (7th Edition):

Tang, Christine. “Mechanisms of High Glucose-induced Decrease in β-cell Function.” 2010. Web. 25 Apr 2019.

Vancouver:

Tang C. Mechanisms of High Glucose-induced Decrease in β-cell Function. [Internet] [Doctoral dissertation]. University of Toronto; 2010. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/1807/26331.

Council of Science Editors:

Tang C. Mechanisms of High Glucose-induced Decrease in β-cell Function. [Doctoral Dissertation]. University of Toronto; 2010. Available from: http://hdl.handle.net/1807/26331


University of Notre Dame

7. Anthony E Clemons. Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>.

Degree: PhD, Biological Sciences, 2014, University of Notre Dame

  Gene expression during embryogenesis impacts adult mosquito fitness, the ability of an organism to survive and transmit its genotype to offspring as compared to… (more)

Subjects/Keywords: Aeded aegypti; Puckered; C-Jun N-terminal Kinase; insulin signaling pathway; stress signaling pathway

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APA (6th Edition):

Clemons, A. E. (2014). Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>. (Doctoral Dissertation). University of Notre Dame. Retrieved from https://curate.nd.edu/show/w089280462m

Chicago Manual of Style (16th Edition):

Clemons, Anthony E. “Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>.” 2014. Doctoral Dissertation, University of Notre Dame. Accessed April 25, 2019. https://curate.nd.edu/show/w089280462m.

MLA Handbook (7th Edition):

Clemons, Anthony E. “Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>.” 2014. Web. 25 Apr 2019.

Vancouver:

Clemons AE. Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>. [Internet] [Doctoral dissertation]. University of Notre Dame; 2014. [cited 2019 Apr 25]. Available from: https://curate.nd.edu/show/w089280462m.

Council of Science Editors:

Clemons AE. Conserved Developmental Cues of Aedes aegypti and their Impact on the Mosquito Vector</h1>. [Doctoral Dissertation]. University of Notre Dame; 2014. Available from: https://curate.nd.edu/show/w089280462m


Vanderbilt University

8. Spurlock III, Charles Floyd. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.

Degree: PhD, Microbiology and Immunology, 2014, Vanderbilt University

 Rheumatoid arthritis is the most common serious autoimmune disease affecting almost one percent of the human population worldwide. Methotrexate is the most commonly used disease-modifying… (more)

Subjects/Keywords: methotrexate; autoimmune disease; inflammation; rheumatoid arthritis; cell cycle checkpoints; c-Jun-N-terminal kinase; p53; tetrahydrobiopterin; long non-coding RNA

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APA (6th Edition):

Spurlock III, C. F. (2014). Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;

Chicago Manual of Style (16th Edition):

Spurlock III, Charles Floyd. “Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.” 2014. Doctoral Dissertation, Vanderbilt University. Accessed April 25, 2019. http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;.

MLA Handbook (7th Edition):

Spurlock III, Charles Floyd. “Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints.” 2014. Web. 25 Apr 2019.

Vancouver:

Spurlock III CF. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. [Internet] [Doctoral dissertation]. Vanderbilt University; 2014. [cited 2019 Apr 25]. Available from: http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;.

Council of Science Editors:

Spurlock III CF. Methotrexate and Rheumatoid Arthritis: At the Crossroads Between Inflammation and Defects in Cell Cycle Checkpoints. [Doctoral Dissertation]. Vanderbilt University; 2014. Available from: http://etd.library.vanderbilt.edu//available/etd-03212014-091439/ ;


Indian Institute of Science

9. Prasad, Karothu Durga. Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications.

Degree: 2014, Indian Institute of Science

 Pyrazoloanthrone and its analogues form the central core of the thesis and the work is focused on the evaluation of chemical and biological applications of… (more)

Subjects/Keywords: Pyrazoloanthrones; Pyrazoloanthrone; Anthrapyrazolones; C-Jun N-Terminal Kinase (JNK); Protein Kinase Inhibition; Cyanide/Fluoride Ion Detection; Picric Acid; Fluoremetric Chemosensor; Septic Shock; JNK Signals; 1, 9-pyrazoloanthrone; Organic Chemistry

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APA (6th Edition):

Prasad, K. D. (2014). Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications. (Thesis). Indian Institute of Science. Retrieved from http://etd.iisc.ernet.in/handle/2005/2980 ; http://etd.ncsi.iisc.ernet.in/abstracts/3842/G26706-Abs.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Prasad, Karothu Durga. “Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications.” 2014. Thesis, Indian Institute of Science. Accessed April 25, 2019. http://etd.iisc.ernet.in/handle/2005/2980 ; http://etd.ncsi.iisc.ernet.in/abstracts/3842/G26706-Abs.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Prasad, Karothu Durga. “Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications.” 2014. Web. 25 Apr 2019.

Vancouver:

Prasad KD. Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications. [Internet] [Thesis]. Indian Institute of Science; 2014. [cited 2019 Apr 25]. Available from: http://etd.iisc.ernet.in/handle/2005/2980 ; http://etd.ncsi.iisc.ernet.in/abstracts/3842/G26706-Abs.pdf.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Prasad KD. Exploration of 1,9-Pyrazoloanthrones as a Copious Reserve for Multifarious Chemical and Biological Applications. [Thesis]. Indian Institute of Science; 2014. Available from: http://etd.iisc.ernet.in/handle/2005/2980 ; http://etd.ncsi.iisc.ernet.in/abstracts/3842/G26706-Abs.pdf

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Gothenburg / Göteborgs Universitet

10. Olausson, Josefin. Studies on microcirculation in insulin resistance.

Degree: 2015, University of Gothenburg / Göteborgs Universitet

 The overall aim of this thesis was to investigate the microcirculation in insulin resistance, with focus on the expression of endothelin-1, through a translational approach.… (more)

Subjects/Keywords: endothelin-1; type 2 diabetes; coronary heart disease; phosphodiesterase-5 inhibition; tadalafil; insulin resistance; endothelial dysfunction; c-Jun N-terminal kinase; nitric oxide; microdialysis

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APA (6th Edition):

Olausson, J. (2015). Studies on microcirculation in insulin resistance. (Thesis). University of Gothenburg / Göteborgs Universitet. Retrieved from http://hdl.handle.net/2077/39546

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Olausson, Josefin. “Studies on microcirculation in insulin resistance.” 2015. Thesis, University of Gothenburg / Göteborgs Universitet. Accessed April 25, 2019. http://hdl.handle.net/2077/39546.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Olausson, Josefin. “Studies on microcirculation in insulin resistance.” 2015. Web. 25 Apr 2019.

Vancouver:

Olausson J. Studies on microcirculation in insulin resistance. [Internet] [Thesis]. University of Gothenburg / Göteborgs Universitet; 2015. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/2077/39546.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Olausson J. Studies on microcirculation in insulin resistance. [Thesis]. University of Gothenburg / Göteborgs Universitet; 2015. Available from: http://hdl.handle.net/2077/39546

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

11. Melino, Michelle. The role of c-jun N-terminal kinase (JNK) in human T cell function.

Degree: 2009, University of Adelaide

 T cells are involved in cellular pathways which enable the immune system to protect us against infection and cancer. However, the same mechanisms also allow… (more)

Subjects/Keywords: T cells; cytokines; c-jun N-terminal kinase; mitogen-activated protein kinases

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APA (6th Edition):

Melino, M. (2009). The role of c-jun N-terminal kinase (JNK) in human T cell function. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/56209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Melino, Michelle. “The role of c-jun N-terminal kinase (JNK) in human T cell function.” 2009. Thesis, University of Adelaide. Accessed April 25, 2019. http://hdl.handle.net/2440/56209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Melino, Michelle. “The role of c-jun N-terminal kinase (JNK) in human T cell function.” 2009. Web. 25 Apr 2019.

Vancouver:

Melino M. The role of c-jun N-terminal kinase (JNK) in human T cell function. [Internet] [Thesis]. University of Adelaide; 2009. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/2440/56209.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Melino M. The role of c-jun N-terminal kinase (JNK) in human T cell function. [Thesis]. University of Adelaide; 2009. Available from: http://hdl.handle.net/2440/56209

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. 鈴木, 彰子. c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する.

Degree: 医学博士, 2017, Iwate Medical University / 岩手医科大学

2016

Subjects/Keywords: Ballooned hepatocyte; Caspase 9; c-Jun N-terminal kinase; Rubicon; SP600125; Ballooned hepatocyte; Caspase 9; c-Jun N-terminal kinase; Rubicon; SP600125

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APA (6th Edition):

鈴木, . (2017). c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する. (Thesis). Iwate Medical University / 岩手医科大学. Retrieved from http://id.nii.ac.jp/1181/00008785/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

鈴木, 彰子. “c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する.” 2017. Thesis, Iwate Medical University / 岩手医科大学. Accessed April 25, 2019. http://id.nii.ac.jp/1181/00008785/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

鈴木, 彰子. “c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する.” 2017. Web. 25 Apr 2019.

Vancouver:

鈴木 . c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する. [Internet] [Thesis]. Iwate Medical University / 岩手医科大学; 2017. [cited 2019 Apr 25]. Available from: http://id.nii.ac.jp/1181/00008785/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

鈴木 . c-Jun N-terminal kinase-mediated Rubicon expression enhances hepatocyte lipoapoptosis and promotes hepatocyte ballooning. : c-Jun N末端キナーゼ依存性のRubicon発現は肝細胞のlipoapoptosisと肝細胞のballooningを促進する. [Thesis]. Iwate Medical University / 岩手医科大学; 2017. Available from: http://id.nii.ac.jp/1181/00008785/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of St. Andrews

13. Borger, Eva. New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia .

Degree: 2012, University of St. Andrews

 Dementia causes an increasing social and economic burden worldwide, demanding action regarding its diagnosis, treatment and everyday management. Recent years have seen many advances in… (more)

Subjects/Keywords: Alzheimer's disease; Frontotemporal dementia; Endophilin; Peroxiredoxin; EFHD2; C-Jun N-tmerinal kinase

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APA (6th Edition):

Borger, E. (2012). New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia . (Thesis). University of St. Andrews. Retrieved from http://hdl.handle.net/10023/3092

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Borger, Eva. “New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia .” 2012. Thesis, University of St. Andrews. Accessed April 25, 2019. http://hdl.handle.net/10023/3092.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Borger, Eva. “New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia .” 2012. Web. 25 Apr 2019.

Vancouver:

Borger E. New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia . [Internet] [Thesis]. University of St. Andrews; 2012. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10023/3092.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Borger E. New intracellular mechanisms involved in Alzheimer's disease and frontotemporal dementia . [Thesis]. University of St. Andrews; 2012. Available from: http://hdl.handle.net/10023/3092

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Gkirtzimanaki, Aikaterini. Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function.

Degree: 2012, University of Crete (UOC); Πανεπιστήμιο Κρήτης

 Cancer can be perceived as a disease of communication between and within cells. The aberrations are pleiotropic, but mitogen‐activated protein kinase (MAPK) pathways feature prominently.… (more)

Subjects/Keywords: Καρκινογένεση πνεύμονα; Σηματοδότηση; Πρωτο-ογκογονίδια; Ογκοκατασταλτικό γονίδιο; Κινάση ενεργοποιούμενη από μιττογόνα; Απώλεια ετεροζυγωτίας; Επιγενετική ρύθμιση; Επιθηλιακά κύτταρα; MAP3K8 (Mittogen activated protein 3 kinase 8); TPL2 (Tumor progression locus 2); COT (Cancer Osaka Thyroid); p53; JNK (C-Jun n-Terminal kinase); ras; NPM (Nucleophosmin); MIR370 (Micro-RNA 370)

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APA (6th Edition):

Gkirtzimanaki, A. (2012). Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/29220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gkirtzimanaki, Aikaterini. “Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function.” 2012. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed April 25, 2019. http://hdl.handle.net/10442/hedi/29220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gkirtzimanaki, Aikaterini. “Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function.” 2012. Web. 25 Apr 2019.

Vancouver:

Gkirtzimanaki A. Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10442/hedi/29220.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gkirtzimanaki A. Study of the role and regulation of TpL2 / COT (MAP3K8) in lung carcinogenesis: a proto-oncogene with a tumor suppressive function. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2012. Available from: http://hdl.handle.net/10442/hedi/29220

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

15. Wang, Fang. Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression.

Degree: Clinical School - St George Hospital, 2006, University of New South Wales

 To explore the potential mechanisms of tendon degeneration, we investigated the role of c-Jun N-terminal Kinase (JNK) activation and the regulation of matrix metalloproteinase 1… (more)

Subjects/Keywords: supraspinatus tendon; c-Jun N-terminal kinase (JNK); matrix metalloproteinase 1 (MMP1); rotator cuff tear; oxidative stress; hydrogen peroxide

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APA (6th Edition):

Wang, F. (2006). Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression. (Masters Thesis). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Wang, Fang. “Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression.” 2006. Masters Thesis, University of New South Wales. Accessed April 25, 2019. http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true.

MLA Handbook (7th Edition):

Wang, Fang. “Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression.” 2006. Web. 25 Apr 2019.

Vancouver:

Wang F. Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression. [Internet] [Masters thesis]. University of New South Wales; 2006. [cited 2019 Apr 25]. Available from: http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true.

Council of Science Editors:

Wang F. Oxidative stress induced C-Jun N-terminal Kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression. [Masters Thesis]. University of New South Wales; 2006. Available from: http://handle.unsw.edu.au/1959.4/28815 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:1348/SOURCE02?view=true


University of Arizona

16. Orton, Christopher R. Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling .

Degree: 2006, University of Arizona

 Defining the mechanics and consequences of protein adduction is crucial to understanding the toxicity of reactive electrophiles. Application of tandem mass spectrometry and data analysis… (more)

Subjects/Keywords: Proteomics; Protein Adduction Kinetics; Quantitative Mass Spectrometry; Glutathione S-transferase; c-Jun N-terminal Kinase Signaling

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APA (6th Edition):

Orton, C. R. (2006). Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling . (Doctoral Dissertation). University of Arizona. Retrieved from http://hdl.handle.net/10150/194247

Chicago Manual of Style (16th Edition):

Orton, Christopher R. “Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling .” 2006. Doctoral Dissertation, University of Arizona. Accessed April 25, 2019. http://hdl.handle.net/10150/194247.

MLA Handbook (7th Edition):

Orton, Christopher R. “Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling .” 2006. Web. 25 Apr 2019.

Vancouver:

Orton CR. Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling . [Internet] [Doctoral dissertation]. University of Arizona; 2006. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10150/194247.

Council of Science Editors:

Orton CR. Analysis of Protein Adduction Kinetics and the Effects of Protein Adduction on C-Jun N-Terminal Kinase Signaling . [Doctoral Dissertation]. University of Arizona; 2006. Available from: http://hdl.handle.net/10150/194247

17. Καρκαλή, Αικατερίνη. Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.

Degree: 2012, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

 MAPK (Mitogen Activated Protein Kinases) signaling pathways have been extensively studied and are known to control multiple biological processes, such as proliferation, apoptosis, differentiation and… (more)

Subjects/Keywords: Φωσφατάσες διπλής εξειδίκευσης της Drosophila melanogaster; Ενεργοποιούµενες από µιτογόνα ερεθίσµατα κινάσες; Οξειδωτικό στρες; ΦΩΣΦΟΡΥΛΙΩΣΗ ΠΡΩΤΕΙΝΩΝ; Εµβρυϊκό Νευρικό Σύστηµα της Drosophila melanogaster; Φωσφατάση Puckered; Κινάση του N-τελικού άκρου του µεταγραφικού παράγοντα c-Jun; Κινάση p38; Dual Specificity Phosphatase’s of Drosophila melanogaster; Mitogen – Activated Protein Kinases (MAPKs); Oxidative stress; PROTEIN PHOSPHORYLATION; Embryonic Nervous System of Drosophila melanogaster; Puckered phosphatase; c-Jun N-terminal kinase (JNK); p38 kinase

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APA (6th Edition):

Καρκαλή, . . (2012). Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/36150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Καρκαλή, Αικατερίνη. “Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.” 2012. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 25, 2019. http://hdl.handle.net/10442/hedi/36150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Καρκαλή, Αικατερίνη. “Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster.” 2012. Web. 25 Apr 2019.

Vancouver:

Καρκαλή . Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10442/hedi/36150.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Καρκαλή . Λειτουργική ανάλυση των φωσφατασών διπλής εξειδίκευσης (DUSPs) στη drosophila melanogaster. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2012. Available from: http://hdl.handle.net/10442/hedi/36150

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Texas – Austin

18. Gopalan, Archana. Targeting breast cancer with natural forms of vitamin E and simvastatin.

Degree: Nutritional Sciences, 2012, University of Texas – Austin

 Breast cancer is the second leading cause of death due to cancer in women. A number of effective therapeutic strategies have been implemented in clinics… (more)

Subjects/Keywords: Breast cancer; Vitamin E; Simvastatin; Apoptosis; Stem cells; TIC; Mevalonate pathway; Ceramide synthesis pathways; Death receptor 5 (DR5); c-Jun N-terminal kinase/C/EBP homologous protein (JNK/CHOP); FLICE inhibitory protein (c-FLIP); B-cell lymphoma 2 (Bcl-2); Survivin; Signal transducer and activator of transcription 3 (Stat3); Bcl-xL; Cyclin D1; c-Myc

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APA (6th Edition):

Gopalan, A. (2012). Targeting breast cancer with natural forms of vitamin E and simvastatin. (Thesis). University of Texas – Austin. Retrieved from http://hdl.handle.net/2152/ETD-UT-2012-05-5520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gopalan, Archana. “Targeting breast cancer with natural forms of vitamin E and simvastatin.” 2012. Thesis, University of Texas – Austin. Accessed April 25, 2019. http://hdl.handle.net/2152/ETD-UT-2012-05-5520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gopalan, Archana. “Targeting breast cancer with natural forms of vitamin E and simvastatin.” 2012. Web. 25 Apr 2019.

Vancouver:

Gopalan A. Targeting breast cancer with natural forms of vitamin E and simvastatin. [Internet] [Thesis]. University of Texas – Austin; 2012. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5520.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gopalan A. Targeting breast cancer with natural forms of vitamin E and simvastatin. [Thesis]. University of Texas – Austin; 2012. Available from: http://hdl.handle.net/2152/ETD-UT-2012-05-5520

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. Henrie, Hélène. Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate.

Degree: Docteur es, Physiologie, physiopathologie, 2017, Paris Saclay

Les microtubules sont des éléments dynamiques du cytosquelette qui contrôlent à la fois l’organisation du cytoplasme, la polarité, la migration et la division cellulaire. Notre… (more)

Subjects/Keywords: Microtubules; JNK (c-Jun NH2-Terminal Kinase); CLIP-170 (Cytoplasmic Linker Protein of 170 kDa); Beta-Tubuline; Sauvetage microtubulaire; Cellules épithéliales; Microtubules; JNK (c-Jun NH2-Terminal Kinase); CLIP-170 (Cytoplasmic Linker Protein of 170 kDa); Beta-Tubulin; Microtubule rescue; Epithelial cells

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APA (6th Edition):

Henrie, H. (2017). Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate. (Doctoral Dissertation). Paris Saclay. Retrieved from http://www.theses.fr/2017SACLS461

Chicago Manual of Style (16th Edition):

Henrie, Hélène. “Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate.” 2017. Doctoral Dissertation, Paris Saclay. Accessed April 25, 2019. http://www.theses.fr/2017SACLS461.

MLA Handbook (7th Edition):

Henrie, Hélène. “Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate.” 2017. Web. 25 Apr 2019.

Vancouver:

Henrie H. Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate. [Internet] [Doctoral dissertation]. Paris Saclay; 2017. [cited 2019 Apr 25]. Available from: http://www.theses.fr/2017SACLS461.

Council of Science Editors:

Henrie H. Régulation de la dynamique des microtubules par la kinase de stress JNK dans les cellules épithéliales : caractérisation de CLIP-170 comme un nouveau substrat. : Microtubule dynamics regulation by the stress kinase JNK in epithelial cells : characterization of CLIP-170 as a new substrate. [Doctoral Dissertation]. Paris Saclay; 2017. Available from: http://www.theses.fr/2017SACLS461


University of Western Australia

20. Pang, Wei Wei. The role of mitochondria in regulating MAPK signalling pathways during oxidative stress.

Degree: PhD, 2006, University of Western Australia

[Truncated abstract] Reactive oxygen species (ROS) have been implicated to play a major role in many pathological conditions including heart attack and stroke. Their ability… (more)

Subjects/Keywords: Mitochondria; Protein kinases; Oxidative stress; Mitochondrial pathology; C-Jun N-terminal kinase (JNK); MAPK signalling pathways; Extracellular-signal regulated protein Kinase (ERK)

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APA (6th Edition):

Pang, W. W. (2006). The role of mitochondria in regulating MAPK signalling pathways during oxidative stress. (Doctoral Dissertation). University of Western Australia. Retrieved from http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=8462&local_base=GEN01-INS01

Chicago Manual of Style (16th Edition):

Pang, Wei Wei. “The role of mitochondria in regulating MAPK signalling pathways during oxidative stress.” 2006. Doctoral Dissertation, University of Western Australia. Accessed April 25, 2019. http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=8462&local_base=GEN01-INS01.

MLA Handbook (7th Edition):

Pang, Wei Wei. “The role of mitochondria in regulating MAPK signalling pathways during oxidative stress.” 2006. Web. 25 Apr 2019.

Vancouver:

Pang WW. The role of mitochondria in regulating MAPK signalling pathways during oxidative stress. [Internet] [Doctoral dissertation]. University of Western Australia; 2006. [cited 2019 Apr 25]. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=8462&local_base=GEN01-INS01.

Council of Science Editors:

Pang WW. The role of mitochondria in regulating MAPK signalling pathways during oxidative stress. [Doctoral Dissertation]. University of Western Australia; 2006. Available from: http://repository.uwa.edu.au:80/R/?func=dbin-jump-full&object_id=8462&local_base=GEN01-INS01

21. NGUYEN THI KIM OANH. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.

Degree: 2018, Ajou University

Mitochondria are dynamic organelles essential for the life and death of the cells. The dynamic nature of mitochondria allows them to organize a network of… (more)

Subjects/Keywords: DNA damage signaling; Mitochondria fission; Nuclear; JNK, Jun N-terminal Kinase; SIRT1, Sirtuin 1; ATM, ataxia-telangiectasia mutated; DNA 손상신호; 미토콘드리아 분열;

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APA (6th Edition):

OANH, N. T. K. (2018). Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

OANH, NGUYEN THI KIM. “Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.” 2018. Thesis, Ajou University. Accessed April 25, 2019. http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

OANH, NGUYEN THI KIM. “Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses.” 2018. Web. 25 Apr 2019.

Vancouver:

OANH NTK. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. [Internet] [Thesis]. Ajou University; 2018. [cited 2019 Apr 25]. Available from: http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

OANH NTK. Roles of mitochondria morphodynamic changes in the DNA damage signaling and cellular stresses. [Thesis]. Ajou University; 2018. Available from: http://repository.ajou.ac.kr/handle/201003/16574 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000027412

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. Λαγκαδινού, Ελένη. Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος.

Degree: 2008, University of Patras

Η θεραπεία της Οξείας Μυελογενούς Λευχαιμίας (ΟΜΛ) είναι συχνά ανεπιτυχής λόγω ανάπτυξης κυτταρικής αντίστασης στα αντιλευχαιμικά φάρμακα. Εκτός από την έκφραση Ρ-γλυκοπρωτείνης στα λευχαιμικά κύτταρα,… (more)

Subjects/Keywords: Οξεία μυελογενής λευχαιμία (ΟΜΛ); Χημειοαντίσταση; Μηχανισμοί μεταγωγής σήματος; 616.994 190 61; Acute myeloid leukemia (AML); Drug resistance; Jun N-terminal Kinase (JNK)

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APA (6th Edition):

Λαγκαδινού, . (2008). Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος. (Doctoral Dissertation). University of Patras. Retrieved from http://nemertes.lis.upatras.gr/jspui/handle/10889/2208

Chicago Manual of Style (16th Edition):

Λαγκαδινού, Ελένη. “Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος.” 2008. Doctoral Dissertation, University of Patras. Accessed April 25, 2019. http://nemertes.lis.upatras.gr/jspui/handle/10889/2208.

MLA Handbook (7th Edition):

Λαγκαδινού, Ελένη. “Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος.” 2008. Web. 25 Apr 2019.

Vancouver:

Λαγκαδινού . Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος. [Internet] [Doctoral dissertation]. University of Patras; 2008. [cited 2019 Apr 25]. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/2208.

Council of Science Editors:

Λαγκαδινού . Διερεύνηση μηχανισμών χημειοαντίστασης στην οξεία μυελογενή λευχαιμία με έμφαση στο ρόλο ενδοκυττάριων μονοπατιών μεταγωγής σήματος. [Doctoral Dissertation]. University of Patras; 2008. Available from: http://nemertes.lis.upatras.gr/jspui/handle/10889/2208

23. Zamani, Marzieh. The Role of the JNK/AP-1 Pathway in the Induction of iNOS and CATs in Vascular Cells .

Degree: 2013, University of Hertfordshire

 Nitric oxide (NO) is an important biological molecule within the body, which over production of this molecule in response to different stimulations can cause various… (more)

Subjects/Keywords: Nitric Oxide (NO); Nitric Oxide Synthase (NOS); Inducible Nitric Oxide Synthase (iNOS); c-Jun N-terminal Kinase (JNK); Activator Protein-1 (AP-1); Mitogen activated Protein Kinase (MAPK); Cationic Amino Acid Transporters (CAT); Vascular Smooth Muscle Cells (VSMCs); J774 macrophages; Cardiovascular disease; Inflammation; soluble Guanylate Cyclase (sGC); L-arginine; Bacterial Lipopolysaccharide (LPS); Interferon-gamma (IFN-???); TAM-67; a-Fos; SP600125; MAP kinase kinases (MKK4)

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APA (6th Edition):

Zamani, M. (2013). The Role of the JNK/AP-1 Pathway in the Induction of iNOS and CATs in Vascular Cells . (Thesis). University of Hertfordshire. Retrieved from http://hdl.handle.net/2299/10626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zamani, Marzieh. “The Role of the JNK/AP-1 Pathway in the Induction of iNOS and CATs in Vascular Cells .” 2013. Thesis, University of Hertfordshire. Accessed April 25, 2019. http://hdl.handle.net/2299/10626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zamani, Marzieh. “The Role of the JNK/AP-1 Pathway in the Induction of iNOS and CATs in Vascular Cells .” 2013. Web. 25 Apr 2019.

Vancouver:

Zamani M. The Role of the JNK/AP-1 Pathway in the Induction of iNOS and CATs in Vascular Cells . [Internet] [Thesis]. University of Hertfordshire; 2013. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/2299/10626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zamani M. The Role of the JNK/AP-1 Pathway in the Induction of iNOS and CATs in Vascular Cells . [Thesis]. University of Hertfordshire; 2013. Available from: http://hdl.handle.net/2299/10626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Οικονομίδου, Όλγα. Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο.

Degree: 2005, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Subjects/Keywords: Υποδοχέας γλυκοκορτικοειδών GR; Πρωτεΐνη ενεργοποίησης-1; Πυρηνικός παράγοντας κΒ; c-jun N-terminal κινάση; Πρωτεΐνη αναστολέας του πυρηνικού παράγοντα; Συστηματικός ερυθηματώδης λύκος ( ΣΕΛ); Μονοπάτι μεταγωγής σήματος; Λεμφοκύτταρα; Glucocorticoid receptor (GR); Activating protein-1; Nuclear factor kB; c-jun N-terminal kinase; IkBa; Systemic lupus erythematosus ( SLE); Signal transduction; Lymphocytes

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Οικονομίδου, . . (2005). Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/20025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Οικονομίδου, Όλγα. “Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο.” 2005. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 25, 2019. http://hdl.handle.net/10442/hedi/20025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Οικονομίδου, Όλγα. “Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο.” 2005. Web. 25 Apr 2019.

Vancouver:

Οικονομίδου . Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2005. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10442/hedi/20025.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Οικονομίδου . Διερεύνηση της μοριακής αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες στο συστηματικό ερυθηματώδη λύκο. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2005. Available from: http://hdl.handle.net/10442/hedi/20025

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Southern California

25. Tao, Litao. Investigation of the molecular mechanisms of ototoxicity.

Degree: PhD, Genetic, Molecular and Cellular Biology, 2014, University of Southern California

 Sensory hair cells are essential for transforming the mechanical vibrations of sound into electric signals that our nervous system can interpret. However, sensory hair cells… (more)

Subjects/Keywords: aminoglycoside antibiotics; ototoxicity; cyclin-dependent kinase 2; c-Jun; RNA sequencing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tao, L. (2014). Investigation of the molecular mechanisms of ototoxicity. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3645

Chicago Manual of Style (16th Edition):

Tao, Litao. “Investigation of the molecular mechanisms of ototoxicity.” 2014. Doctoral Dissertation, University of Southern California. Accessed April 25, 2019. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3645.

MLA Handbook (7th Edition):

Tao, Litao. “Investigation of the molecular mechanisms of ototoxicity.” 2014. Web. 25 Apr 2019.

Vancouver:

Tao L. Investigation of the molecular mechanisms of ototoxicity. [Internet] [Doctoral dissertation]. University of Southern California; 2014. [cited 2019 Apr 25]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3645.

Council of Science Editors:

Tao L. Investigation of the molecular mechanisms of ototoxicity. [Doctoral Dissertation]. University of Southern California; 2014. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/426847/rec/3645

26. Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, Hisashi. p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells.

Degree: 博士(歯学), 2015, Fukuoka Dental College / 福岡歯科大学

To investigate the involvement of stress-activated protein kinases, JNK and p38 MAPK, in the assembly of tight junctions in keratinocytes, we treated HaCaT cells with… (more)

Subjects/Keywords: MAPK; JNK; p38; タイト結合; ZO-1; HaCaT細胞; mitogen-activated protein kinase (MAPK); c-Jun NH2-terminal protein kinase (JNK); p38 MAPK; tight junction; ZO-1; HaCaT cell

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APA (6th Edition):

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, H. (2015). p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. (Thesis). Fukuoka Dental College / 福岡歯科大学. Retrieved from http://id.nii.ac.jp/1167/00000024/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, Hisashi. “p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells.” 2015. Thesis, Fukuoka Dental College / 福岡歯科大学. Accessed April 25, 2019. http://id.nii.ac.jp/1167/00000024/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan, Hisashi. “p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells.” 2015. Web. 25 Apr 2019.

Vancouver:

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan H. p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. [Internet] [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. [cited 2019 Apr 25]. Available from: http://id.nii.ac.jp/1167/00000024/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Minakami, Masahiko; Kitagawa, Norio; Iida, Hiroshi; Anan H. p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. : p38 Mitogen-activated protein kinase and c-Jun NH2-terminal protein kinase regulate the accumulation of a tight junction protein, ZO-1, in cell-cell contacts in HaCaT cells. [Thesis]. Fukuoka Dental College / 福岡歯科大学; 2015. Available from: http://id.nii.ac.jp/1167/00000024/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Paris-Sud – Paris XI

27. He, Tiantian. Studying the Role of Peroxiredoxin 1 in ROS Modulation and Drug Resistance : Etude du rôle de la Peroxiredoxine 1 dans la modulation redox et la résistance aux drogues anticancéreuses.

Degree: Docteur es, Biologie moléculaire et cellulaire, 2014, Université Paris-Sud – Paris XI

 Les peroxyrédoxines sont des enzymes essentielles de la cellule. Outre leur rôle d’antioxydant, elles sont aussi des régulateurs de la signalisation cellulaire et des suppresseurs… (more)

Subjects/Keywords: Espèces réactives de l’oxygène; Péroxyredoxine 1; Β-lapachone; Ménadione; NADPH quinone oxydoréductase; NRH: quinone oxydoréductase 2; Thioredoxine 1; Thioredoxine 2; La mort des cellules cancéreuses; HyPer; Necropotose; Reactive oxygen species; Peroxiredoxin 1; Β-lapachone; Menadione; NAD(P)H:quinone oxidoreductase 1; NRH:quinone oxidoreductase 2; Thioredoxin 1; Thioredoxin 2; Mitogen-activated protein kinase; C-Jun N-terminal kinases; Extracellular signal-regulated kinases; Anti-cancer; HyPer; Redox western blot; Cell death; Necroptosis

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APA (6th Edition):

He, T. (2014). Studying the Role of Peroxiredoxin 1 in ROS Modulation and Drug Resistance : Etude du rôle de la Peroxiredoxine 1 dans la modulation redox et la résistance aux drogues anticancéreuses. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA112139

Chicago Manual of Style (16th Edition):

He, Tiantian. “Studying the Role of Peroxiredoxin 1 in ROS Modulation and Drug Resistance : Etude du rôle de la Peroxiredoxine 1 dans la modulation redox et la résistance aux drogues anticancéreuses.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed April 25, 2019. http://www.theses.fr/2014PA112139.

MLA Handbook (7th Edition):

He, Tiantian. “Studying the Role of Peroxiredoxin 1 in ROS Modulation and Drug Resistance : Etude du rôle de la Peroxiredoxine 1 dans la modulation redox et la résistance aux drogues anticancéreuses.” 2014. Web. 25 Apr 2019.

Vancouver:

He T. Studying the Role of Peroxiredoxin 1 in ROS Modulation and Drug Resistance : Etude du rôle de la Peroxiredoxine 1 dans la modulation redox et la résistance aux drogues anticancéreuses. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2019 Apr 25]. Available from: http://www.theses.fr/2014PA112139.

Council of Science Editors:

He T. Studying the Role of Peroxiredoxin 1 in ROS Modulation and Drug Resistance : Etude du rôle de la Peroxiredoxine 1 dans la modulation redox et la résistance aux drogues anticancéreuses. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA112139

28. Gandhi, Adarsh. Role of Inflammation in Metabolism and Hepatotoxicity of Prototypical Drugs.

Degree: Pharmacological and Pharmaceutical Sciences, Department of, 2012, University of Houston

 Inflammation leads to altered drug metabolism and augments drug-induced hepatotoxicity. However, the mechanisms are not fully understood. Inflammation is known to alter the gene expression… (more)

Subjects/Keywords: Inflammation; Toll-like receptor; c-Jun-N-terminal kinase; chlorpromazine; acetaminophen; midazolam; cytochrome P4503A; tumor necrosis factor-alpha; lipopolysaccharide; lipoteichoic acid.

…146 6. Role of c-jun N-terminal kinase (JNK) in regulating tumor necrosis factor… …MAPK such as the c-Jun-N-terminal kinase (JNK), as well as dendritic cell… …transcription of MAPK genes such as c-Jun, activator protein (AP-1), etc. 14 In the MyD88… …99 Table 4.3 PK parameters of (A) MDZ, (B) 1’-OHMDZ and (C) 1… …observed after C. rodentium infection in mice (Chaluvadi et al., 2009) .There was a… 

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APA (6th Edition):

Gandhi, A. (2012). Role of Inflammation in Metabolism and Hepatotoxicity of Prototypical Drugs. (Thesis). University of Houston. Retrieved from http://hdl.handle.net/10657/ETD-UH-2012-08-457

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gandhi, Adarsh. “Role of Inflammation in Metabolism and Hepatotoxicity of Prototypical Drugs.” 2012. Thesis, University of Houston. Accessed April 25, 2019. http://hdl.handle.net/10657/ETD-UH-2012-08-457.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gandhi, Adarsh. “Role of Inflammation in Metabolism and Hepatotoxicity of Prototypical Drugs.” 2012. Web. 25 Apr 2019.

Vancouver:

Gandhi A. Role of Inflammation in Metabolism and Hepatotoxicity of Prototypical Drugs. [Internet] [Thesis]. University of Houston; 2012. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10657/ETD-UH-2012-08-457.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gandhi A. Role of Inflammation in Metabolism and Hepatotoxicity of Prototypical Drugs. [Thesis]. University of Houston; 2012. Available from: http://hdl.handle.net/10657/ETD-UH-2012-08-457

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Σπηλιωτάκη, Μαρία. Διερεύνηση της αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες και κινάσες στην κατάθλιψη.

Degree: 2005, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Subjects/Keywords: Υποδοχέας γλυκοκορτικοειδών; Πρωτεΐνη ενεργοποίησης-1; c-jun αμινοτελική κινάση; Πυρηνικός παράγοντας κΒ; Διπολική συναισθηματική διαταραχή; Κατάθλιψη; Νορμοθυμία; Ευθυμία; Glucocorticoid receptor (GR); Activator protein -1; c-jun N-terminal kinase; Nuclear factor kB; Bipolar affective disorder; Depression; Normothymia; Eythymia

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APA (6th Edition):

Σπηλιωτάκη, . . (2005). Διερεύνηση της αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες και κινάσες στην κατάθλιψη. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/25513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Σπηλιωτάκη, Μαρία. “Διερεύνηση της αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες και κινάσες στην κατάθλιψη.” 2005. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed April 25, 2019. http://hdl.handle.net/10442/hedi/25513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Σπηλιωτάκη, Μαρία. “Διερεύνηση της αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες και κινάσες στην κατάθλιψη.” 2005. Web. 25 Apr 2019.

Vancouver:

Σπηλιωτάκη . Διερεύνηση της αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες και κινάσες στην κατάθλιψη. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2005. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10442/hedi/25513.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Σπηλιωτάκη . Διερεύνηση της αλληλεπίδρασης του υποδοχέα των γλυκοκορτικοειδών με μεταγραφικούς παράγοντες και κινάσες στην κατάθλιψη. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2005. Available from: http://hdl.handle.net/10442/hedi/25513

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Δροσάτος, Κωνσταντίνος. Γονιδιακή ρύθμιση και λειτουργίες της απολιποπρωτεΐνης Ε.

Degree: 2007, University of Crete (UOC); Πανεπιστήμιο Κρήτης

Subjects/Keywords: Απολιποπρωτεΐνη Ε; Γονιδιακή ρύθμιση; Μεταγραφικός παράγοντας c-Jun; Αμινο-τελική κινάση; Υπερλιποπρωτεϊναιμία τύπου ΙΙΙ; Βιοσύνθεση λιποπρωτεϊνών υψηλής πυκνότητας; Αδενοϊοί; Γονιδιακή μεταφορά; Apolipoprotein E; Gene regulation; Transcription factor c-Jun; N-terminal kinase; Hyperlipoproteinemia type III; Biogenesis of high density lipoproteins; Adenoviruses; Gene transfer

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APA (6th Edition):

Δροσάτος, . . (2007). Γονιδιακή ρύθμιση και λειτουργίες της απολιποπρωτεΐνης Ε. (Thesis). University of Crete (UOC); Πανεπιστήμιο Κρήτης. Retrieved from http://hdl.handle.net/10442/hedi/15792

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Δροσάτος, Κωνσταντίνος. “Γονιδιακή ρύθμιση και λειτουργίες της απολιποπρωτεΐνης Ε.” 2007. Thesis, University of Crete (UOC); Πανεπιστήμιο Κρήτης. Accessed April 25, 2019. http://hdl.handle.net/10442/hedi/15792.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Δροσάτος, Κωνσταντίνος. “Γονιδιακή ρύθμιση και λειτουργίες της απολιποπρωτεΐνης Ε.” 2007. Web. 25 Apr 2019.

Vancouver:

Δροσάτος . Γονιδιακή ρύθμιση και λειτουργίες της απολιποπρωτεΐνης Ε. [Internet] [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2007. [cited 2019 Apr 25]. Available from: http://hdl.handle.net/10442/hedi/15792.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Δροσάτος . Γονιδιακή ρύθμιση και λειτουργίες της απολιποπρωτεΐνης Ε. [Thesis]. University of Crete (UOC); Πανεπιστήμιο Κρήτης; 2007. Available from: http://hdl.handle.net/10442/hedi/15792

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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