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You searched for subject:(Breast Neoplasms metabolism 60). Showing records 1 – 30 of 20903 total matches.

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1. Kwon, Yeon-Jin. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.

Degree: PhD, 2010, University of Alabama – Birmingham

Glioma-associated oncogene homolog 1 (Gli1) is a well-known oncogene and a transcription factor that mediates several signaling pathways important for tumor progression, such as hedgehog,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Movement<; br>; Estrogen Receptor alpha  – deficiency<; br>; Matrix Metalloproteinase 11  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Up-Regulation

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APA (6th Edition):

Kwon, Y. (2010). The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,913

Chicago Manual of Style (16th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,913.

MLA Handbook (7th Edition):

Kwon, Yeon-Jin. “The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis.” 2010. Web. 21 Jan 2020.

Vancouver:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913.

Council of Science Editors:

Kwon Y. The Role of Gli1 in ERα-negative breast cancer : promoting survival, migration, invasion, and metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,913

2. Gavin, Cristin. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.

Degree: PhD, 2012, University of Alabama – Birmingham

Dynamic changes to the actin cytoskeleton are required for synaptic plasticity and long-term memory formation. However, the molecular mechanisms that mediate filamentous actin (F-actin) dynamics… (more)

Subjects/Keywords: Bone Neoplasms – secondary<; br>; Breast Neoplasms – pathology<; br>; Cell Communication<; br>; Cyclic AMP-Dependent Protein Kinases – metabolism.<; br>; Gap Junctions – metabolism.<; br>; Osteoblasts – cytology.<; br>; Phosphatidylinositol 3-Kinases – metabolism.

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APA (6th Edition):

Gavin, C. (2012). Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1396

Chicago Manual of Style (16th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1396.

MLA Handbook (7th Edition):

Gavin, Cristin. “Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation.” 2012. Web. 21 Jan 2020.

Vancouver:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396.

Council of Science Editors:

Gavin C. Myosin Ii Regulates Actin Dynamics Critical For Structural Plasticity And Fear Memory Formation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1396

3. Evans, Lynda Michele. The effect of stearic acid on breast cancer development and progression.

Degree: PhD, 2009, University of Alabama – Birmingham

Stearate is an 18-carbon saturated fatty acid that is found in many foods in the western diet including beef and chocolate. Cell culture studies indicate… (more)

Subjects/Keywords: Antineoplastic Agents  – administration & dosage<; br>; Apoptosis<; br>; Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Dietary Fats<; br>; Neoplasm Metastasis<; br>; Stearates  – therapeutic use<; br>; Stearic Acids  – administration & dosage

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APA (6th Edition):

Evans, L. M. (2009). The effect of stearic acid on breast cancer development and progression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,906

Chicago Manual of Style (16th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,906.

MLA Handbook (7th Edition):

Evans, Lynda Michele. “The effect of stearic acid on breast cancer development and progression.” 2009. Web. 21 Jan 2020.

Vancouver:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906.

Council of Science Editors:

Evans LM. The effect of stearic acid on breast cancer development and progression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,906

4. Pandya, Ashka Y. Structural and functional analysis of KLF4.

Degree: PhD, 2007, University of Alabama – Birmingham

KLF4, a C2H2 type zinc finger transcription factor, plays an essential role in maturation of normal stratified squamous epithelium. In normal squamous epithelium its expression… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism<; br>; Breast Neoplasms  – pathology<; br>; Cell Nucleus  – metabolism<; br>; DNA-Binding Proteins  – biosynthesis<; br>; Kruppel-Like Transcription Factors<; br>; Prognosis Transcription Factors  – biosynthesis

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APA (6th Edition):

Pandya, A. Y. (2007). Structural and functional analysis of KLF4. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,531

Chicago Manual of Style (16th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,531.

MLA Handbook (7th Edition):

Pandya, Ashka Y. “Structural and functional analysis of KLF4.” 2007. Web. 21 Jan 2020.

Vancouver:

Pandya AY. Structural and functional analysis of KLF4. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531.

Council of Science Editors:

Pandya AY. Structural and functional analysis of KLF4. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,531

5. Liu, Zhaoli. KLF4 regulates notch1 expression and signaling during epithelial transformation.

Degree: PhD, 2006, University of Alabama – Birmingham

Notch1 and KLF4 function in the specification of epithelial cell fates, and each can act as a context-dependent oncogene or tumor suppressor. We report that… (more)

Subjects/Keywords: Breast Neoplasms <; br>; Epithelial Cells  – metabolism <; br>; Kruppel-Like Transcription Factors  – genetics <; br>; Receptor, Notch1  – metabolism

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APA (6th Edition):

Liu, Z. (2006). KLF4 regulates notch1 expression and signaling during epithelial transformation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,378

Chicago Manual of Style (16th Edition):

Liu, Zhaoli. “KLF4 regulates notch1 expression and signaling during epithelial transformation.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,378.

MLA Handbook (7th Edition):

Liu, Zhaoli. “KLF4 regulates notch1 expression and signaling during epithelial transformation.” 2006. Web. 21 Jan 2020.

Vancouver:

Liu Z. KLF4 regulates notch1 expression and signaling during epithelial transformation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,378.

Council of Science Editors:

Liu Z. KLF4 regulates notch1 expression and signaling during epithelial transformation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,378

6. Silveira, Alexandra C. Characterization of SUDS3 as a BRMS1 family member in breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

BRMS1 and SUDS3 belong to a protein family characterized by the Sds3-like domain. These proteins are core members of SIN3-HDAC chromatin remodeling complexes and thus,… (more)

Subjects/Keywords: Breast Neoplasms  – metabolism <; br>; Carrier Proteins  – metabolism <; br>; Chromatin Assembly and Disassembly <; br>; Histone Deacetylases  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Repressor Proteins/metabolism

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APA (6th Edition):

Silveira, A. C. (2008). Characterization of SUDS3 as a BRMS1 family member in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,339

Chicago Manual of Style (16th Edition):

Silveira, Alexandra C. “Characterization of SUDS3 as a BRMS1 family member in breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,339.

MLA Handbook (7th Edition):

Silveira, Alexandra C. “Characterization of SUDS3 as a BRMS1 family member in breast cancer.” 2008. Web. 21 Jan 2020.

Vancouver:

Silveira AC. Characterization of SUDS3 as a BRMS1 family member in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,339.

Council of Science Editors:

Silveira AC. Characterization of SUDS3 as a BRMS1 family member in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,339

7. Moore, Lakisha Dionne. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.

Degree: PhD, 2008, University of Alabama – Birmingham

Overexpresssion of transforming growth factor (TGF)-[beta] has been implicated in promoting immune suppression, tumor angiogenesis, tumor cell migration, and invasion in many cancers including carcinoma… (more)

Subjects/Keywords: Breast Neoplasms  – genetics <; br>; Neoplasm Metastasis <; br>; RNA Interference <; br>; Transforming Growth Factor beta1  – genetics <; br>; Transforming Growth Factor beta1  – metabolism

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APA (6th Edition):

Moore, L. D. (2008). Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,233

Chicago Manual of Style (16th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,233.

MLA Handbook (7th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Web. 21 Jan 2020.

Vancouver:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233.

Council of Science Editors:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233

8. Cook, Leah M. (Leah Marie). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.

Degree: PhD, 2011, University of Alabama – Birmingham

Morbidity and mortality of breast cancer patients are drastically increased when primary tumor cells metastasize to distant organ sites. Effective treatment of metastatic disease has… (more)

Subjects/Keywords: Breast Neoplasms  – pathology<; br>; Mammary Neoplasms, Animal<; br>; Mice, Transgenic<; br>; Neoplasm Metastasis  – pathology<; br>; Tumor Microenvironment<; br>; Tumor Suppressor Proteins  – genetics<; br>; Tumor Suppressor Proteins  – metabolism

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APA (6th Edition):

Cook, L. M. (. M. (2011). Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1045

Chicago Manual of Style (16th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1045.

MLA Handbook (7th Edition):

Cook, Leah M (Leah Marie). “Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice.” 2011. Web. 21 Jan 2020.

Vancouver:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045.

Council of Science Editors:

Cook LM(M. Understanding molecular mechanisms of breast cancer metastasis using genetically-engineered mice. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1045

9. Ling, Shiyun. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.

Degree: PhD, 2010, University of Alabama – Birmingham

Unrestricted cell proliferation and suppression of cell death are two essential events for tumor development. My dissertation research involves two proteins, 14-3-3 &tau and EDD… (more)

Subjects/Keywords: 14-3-3 Proteins – metabolism.<; br>; Breast Neoplasms<; br>; Down-Regulation<; br>; Membrane Proteins – genetics<; br>; Ovarian Neoplasms<; br>; Protein Processing, Post-Translational<; br>; Transcriptional Activation

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APA (6th Edition):

Ling, S. (2010). Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1419

Chicago Manual of Style (16th Edition):

Ling, Shiyun. “Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1419.

MLA Handbook (7th Edition):

Ling, Shiyun. “Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation.” 2010. Web. 21 Jan 2020.

Vancouver:

Ling S. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1419.

Council of Science Editors:

Ling S. Role Of 14-3-3&tau In Autophagy And Role Of Edd In P53 Regulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1419

10. Yuan, Kun. Effects of defucosylation on human breast cancer cells.

Degree: PhD, 2007, University of Alabama – Birmingham

Carbohydrates can be conjugated to protein or lipids and participate in multiple cell-cell and cell-matrix interactions. Altered glycosylation events may be associated with cancer development… (more)

Subjects/Keywords: alpha-L-Fucosidase  – metabolism<; br>; Breast Neoplasms  – physiopathology<; br>; Fucose  – metabolism<; br>; Fucose  – physiology<; br>; Glycoproteins  – physiology<; br>; Membrane Glycoproteins  – metabolism

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APA (6th Edition):

Yuan, K. (2007). Effects of defucosylation on human breast cancer cells. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,694

Chicago Manual of Style (16th Edition):

Yuan, Kun. “Effects of defucosylation on human breast cancer cells.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,694.

MLA Handbook (7th Edition):

Yuan, Kun. “Effects of defucosylation on human breast cancer cells.” 2007. Web. 21 Jan 2020.

Vancouver:

Yuan K. Effects of defucosylation on human breast cancer cells. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,694.

Council of Science Editors:

Yuan K. Effects of defucosylation on human breast cancer cells. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,694

11. Phipps, Matthew Christopher. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.

Degree: PhD, 2012, University of Alabama – Birmingham

Although bone has a dramatic capacity for regeneration, certain injuries and procedures present defects that are unable to heal properly, requiring surgical intervention to induce… (more)

Subjects/Keywords: Apoptosis – drug effects<; br>; beta Catenin – metabolism.<; br>; Breast Neoplasms – drug therapy<; br>; Cell Proliferation – drug effects.<; br>; Cyclic GMP-Dependent Protein Kinases – metabolism.<; br>; Cyclic Nucleotide Phosphodiesterases, Type 5 – genetics.<; br>; Phosphodiesterase 5 Inhibitors.<; br>; Sulindac – analogs & derivatives.<; br>; Wnt Proteins – metabolism.

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APA (6th Edition):

Phipps, M. C. (2012). Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1410

Chicago Manual of Style (16th Edition):

Phipps, Matthew Christopher. “Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1410.

MLA Handbook (7th Edition):

Phipps, Matthew Christopher. “Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications.” 2012. Web. 21 Jan 2020.

Vancouver:

Phipps MC. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1410.

Council of Science Editors:

Phipps MC. Development Of Electrospun Bone-Mimetic Matrices For Bone Regenerative Applications. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1410

12. Diers, Anne R. Regulation of redox signaling by lipid electrophiles in breast cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

A number of steps in breast cancer progression and metastasis are regulated by redox signaling pathways. Electrophilic lipids such as 15-deoxy-[delta]12,14-prostaglandin J2 (15d-PGJ2) are mediators… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – drug effects<; br>; Breast Neoplasms<; br>; Mitochondria  – drug effects<; br>; Mitochondria  – metabolism<; br>; Prostaglandin D2  – analogs & derivatives

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APA (6th Edition):

Diers, A. R. (2009). Regulation of redox signaling by lipid electrophiles in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,908

Chicago Manual of Style (16th Edition):

Diers, Anne R. “Regulation of redox signaling by lipid electrophiles in breast cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,908.

MLA Handbook (7th Edition):

Diers, Anne R. “Regulation of redox signaling by lipid electrophiles in breast cancer.” 2009. Web. 21 Jan 2020.

Vancouver:

Diers AR. Regulation of redox signaling by lipid electrophiles in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,908.

Council of Science Editors:

Diers AR. Regulation of redox signaling by lipid electrophiles in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,908

13. Ogunrinu, Toyin Adeyemi. Role of the cystine-glutamate exchanger in glioma cell biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Changes in the glioma microenvironment including oxygen (O2) levels, supply of amino acid such as L-glutamate and L-cystine and glutathione (GSH) concentrations play a critical… (more)

Subjects/Keywords: Anoxia  – metabolism<; br>; Brain Neoplasms  – metabolism<; br>; Glioblastoma  – metabolism<; br>; Glioma  – metabolism<; br>; Glutathione  – metabolism<; br>; Glutamic Acid  – metabolism

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APA (6th Edition):

Ogunrinu, T. A. (2010). Role of the cystine-glutamate exchanger in glioma cell biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,956

Chicago Manual of Style (16th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,956.

MLA Handbook (7th Edition):

Ogunrinu, Toyin Adeyemi. “Role of the cystine-glutamate exchanger in glioma cell biology.” 2010. Web. 21 Jan 2020.

Vancouver:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956.

Council of Science Editors:

Ogunrinu TA. Role of the cystine-glutamate exchanger in glioma cell biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,956

14. Steg, Adam. Analysis of the hedgehog pathway in pancreatic adenocarcinoma.

Degree: PhD, 2008, University of Alabama – Birmingham

Pancreatic adenocarcinoma (PAC) is the fourth leading cause of cancer mortality in the United States. Despite the use of highly aggressive treatment regimens (surgery, chemotherapy… (more)

Subjects/Keywords: Adenocarcinoma  – metabolism <; br>; Adenocarcinoma  – pathology <; br>; Hedgehog Proteins  – metabolism <; br>; Pancreatic Neoplasms  – metabolism <; br>; Pancreatic Neoplasms  – pathology

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APA (6th Edition):

Steg, A. (2008). Analysis of the hedgehog pathway in pancreatic adenocarcinoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,290

Chicago Manual of Style (16th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,290.

MLA Handbook (7th Edition):

Steg, Adam. “Analysis of the hedgehog pathway in pancreatic adenocarcinoma.” 2008. Web. 21 Jan 2020.

Vancouver:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290.

Council of Science Editors:

Steg A. Analysis of the hedgehog pathway in pancreatic adenocarcinoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,290

15. Jiang, Wen, Ph.D. KLF4 and retinoid receptor signaling in cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

The fight against cancer has generated wide interest in understanding the genetic mechanisms behind the disease. One group of oncogenes – transcription factors – offers… (more)

Subjects/Keywords: Carcinoma, Squamous Cell  – metabolism<; br>; Cell Nucleus  – metabolism<; br>; Kruppel-Like Transcription Factors  – metabolism<; br>; Mice, Transgenic<; br>; Skin Neoplasms  – metabolism

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APA (6th Edition):

Jiang, Wen, P. D. (2009). KLF4 and retinoid receptor signaling in cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,598

Chicago Manual of Style (16th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,598.

MLA Handbook (7th Edition):

Jiang, Wen, Ph D. “KLF4 and retinoid receptor signaling in cancer.” 2009. Web. 21 Jan 2020.

Vancouver:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598.

Council of Science Editors:

Jiang, Wen PD. KLF4 and retinoid receptor signaling in cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,598

16. Hensel, Jonathan Adam. The role of LL-37 in prostate cancer and its potential as a therapeutic target.

Degree: 2011, University of Alabama – Birmingham

LL-37 is the only cathelicidin-derived anti-microbial peptide in humans and has been shown to stimulate proliferation, angiogenesis, cellular migration and inhibit apoptosis, in addition to… (more)

Subjects/Keywords: Antimicrobial Cationic Peptides  – metabolism<; br>; Cathelicidins<; br>; Molecular Targeted Therapy  – methods<; br>; Neoplasms, Hormone-Dependent<; br>; Prostatic Neoplasms  – genetics<; br>; Prostatic Neoplasms  – metabolism<; br>; Prostatic Neoplasms  – pathology

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APA (6th Edition):

Hensel, J. A. (2011). The role of LL-37 in prostate cancer and its potential as a therapeutic target. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Thesis, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Web. 21 Jan 2020.

Vancouver:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Internet] [Thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Cuddapah, Vishnu Anand. Regulation Of Clc-3 In Human Malignant Glioma.

Degree: PhD, 2012, University of Alabama – Birmingham

Malignant gliomas are the most common and deadly form of primary brain cancer afflicting adults. Current treatment regimens, including surgical debulking, radiotherapy, and chemotherapy, have… (more)

Subjects/Keywords: Brain Neoplasms – metabolism<; br>; Calcium-Calmodulin-Dependent Protein Kinase Type 2 – metabolism.<; br>; Cell Movement – physiology<; br>; Chloride Channels – metabolism.<; br>; Gene Expression Regulation<; br>; Gene Expression Regulation, Enzymologic<; br>; Gene Expression Regulation, Neoplastic<; br>; Glioma – metabolism<; br>; Ion Channels – metabolism<; br>; Membrane Transport Proteins – metabolism.<; br>; Mitosis<; br>; Neoplasms – metabolism<; br>; Neoplasms – pathology

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APA (6th Edition):

Cuddapah, V. A. (2012). Regulation Of Clc-3 In Human Malignant Glioma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1394

Chicago Manual of Style (16th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1394.

MLA Handbook (7th Edition):

Cuddapah, Vishnu Anand. “Regulation Of Clc-3 In Human Malignant Glioma.” 2012. Web. 21 Jan 2020.

Vancouver:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394.

Council of Science Editors:

Cuddapah VA. Regulation Of Clc-3 In Human Malignant Glioma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1394

18. Shaikh, Faheem M. Role of variant sialylation in regulating tumor cell behavior.

Degree: PhD, 2008, University of Alabama – Birmingham

Many different tumors have been documented to have elevated levels of the enzyme ST6Gal I, a Golgi glycosyltransferase that adds [alpha]2-6 sialic acids to glycoproteins.… (more)

Subjects/Keywords: Cell Movement  – physiology <; br>; Colonic Neoplasms <; br>; Integrins  – chemistry <; br>; Integrins  – metabolism <; br>; Neoplasm Invasiveness <; br>; Sialic Acids  – metabolism

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APA (6th Edition):

Shaikh, F. M. (2008). Role of variant sialylation in regulating tumor cell behavior. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,266

Chicago Manual of Style (16th Edition):

Shaikh, Faheem M. “Role of variant sialylation in regulating tumor cell behavior.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,266.

MLA Handbook (7th Edition):

Shaikh, Faheem M. “Role of variant sialylation in regulating tumor cell behavior.” 2008. Web. 21 Jan 2020.

Vancouver:

Shaikh FM. Role of variant sialylation in regulating tumor cell behavior. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,266.

Council of Science Editors:

Shaikh FM. Role of variant sialylation in regulating tumor cell behavior. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,266

19. Robert, Stephanie Marie. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.

Degree: 2012, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) are the most prevalent and aggressive malignant brain tumors. Current treatment - a combination of radiation, chemotherapy and resection - has limited… (more)

Subjects/Keywords: Amino Acid Transport System X-AG – metabolism.<; br>; Brain Neoplasms – metabolism<; br>; Glioblastoma.<; br>; Glutathione<; br>; Seizures<; br>; Sulfasalazine.

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APA (6th Edition):

Robert, S. M. (2012). Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Robert, Stephanie Marie. “Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.” 2012. Thesis, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Robert, Stephanie Marie. “Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors.” 2012. Web. 21 Jan 2020.

Vancouver:

Robert SM. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. [Internet] [Thesis]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1791.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Robert SM. Cystine/glutamate Transporters As Prognostic & Therapeutic Markers Of Primary Brain Tumors. [Thesis]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1791

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Hertz, Marla (Marla Ilene). In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.

Degree: PhD, 2011, University of Alabama – Birmingham

Translation of the majority of eukaryotic mRNAs is initiated upon recognition of its 5′ cap structure by translation initiation factors in so-called cap-dependent translation. Capdependent… (more)

Subjects/Keywords: Dicistroviridae  – metabolism<; br>; Gene Expression Regulation<; br>; Hepacivirus  – metabolism<; br>; Prostatic Neoplasms<; br>; Protein Biosynthesis<; br>; Ribosomal Proteins  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – metabolism

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APA (6th Edition):

Hertz, M. (. I. (2011). In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,953

Chicago Manual of Style (16th Edition):

Hertz, Marla (Marla Ilene). “In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,953.

MLA Handbook (7th Edition):

Hertz, Marla (Marla Ilene). “In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation.” 2011. Web. 21 Jan 2020.

Vancouver:

Hertz M(I. In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,953.

Council of Science Editors:

Hertz M(I. In vivo analysis of the natural diversity of the IGR IRES family and characterizaton of the role of ribosomal protein S25 in IRES-mediated translation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,953

21. Swindall, Amanda F. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.

Degree: PhD, 2012, University of Alabama – Birmingham

The golgi glycosyltransferase, ST6Gal-I, adds a negatively-charged sialic acid in an alpha-2-6 linkage to N-linked glycans. ST6Gal-I is upregulated in many cancers, and is associated… (more)

Subjects/Keywords: Antigens, CD – metabolism.<; br>; Antigens, CD95 – metabolism.<; br>; Apoptosis<; br>; Tumor Cells, Cultured<; br>; Colonic Neoplasms<; br>; Neoplasm Proteins – metabolism.<; br>; Sialyltransferases – metabolism.

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APA (6th Edition):

Swindall, A. F. (2012). The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1392

Chicago Manual of Style (16th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1392.

MLA Handbook (7th Edition):

Swindall, Amanda F. “The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis.” 2012. Web. 21 Jan 2020.

Vancouver:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392.

Council of Science Editors:

Swindall AF. The Role Of St6gal-I Sialylation In Fas (cd95) Death Receptor Function And Tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1392

22. Zheng, Ying. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Janus Kinase (JAK)-Signal Transducer and Activator of Transcription (STAT) signaling is involved in regulation of cell survival, proliferation and differentiation. JAK tyrosine kinases can be… (more)

Subjects/Keywords: Casein Kinase II  – metabolism<; br>; Hematologic Neoplasms  – metabolism<; br>; JNK Mitogen-Activated Protein Kinases  – metabolism<; br>; Polycythemia Vera  – metabolism<; br>; STAT Transcription Factors  – metabolism<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Zheng, Y. (2010). A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1168

Chicago Manual of Style (16th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1168.

MLA Handbook (7th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Web. 21 Jan 2020.

Vancouver:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168.

Council of Science Editors:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168

23. Grunda, Jessica M. Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment.

Degree: PhD, 2009, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) is the most lethal form of primary brain neoplasm with average patient survival between 9 and 15 months even with the most… (more)

Subjects/Keywords: Brain Neoplasms<; br>; Drug Resistance, Neoplasm  – genetics<; br>; Endopeptidases  – metabolism<; br>; Gene Expression Profiling<; br>; Glioblastoma<; br>; Microtubule-Associated Proteins  – metabolism<; br>; Neoplasm Proteins  – metabolism<; br>; Thymidylate Synthase  – metabolism<; br>; Tumor Markers, Biological  – metabolism

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APA (6th Edition):

Grunda, J. M. (2009). Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1085

Chicago Manual of Style (16th Edition):

Grunda, Jessica M. “Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1085.

MLA Handbook (7th Edition):

Grunda, Jessica M. “Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment.” 2009. Web. 21 Jan 2020.

Vancouver:

Grunda JM. Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1085.

Council of Science Editors:

Grunda JM. Identification of new strategies for the treatment of glioblastoma multiforme utilizing a pharmacogenomic approach: genetic profiles associated with patient prognosis and outcome to capecitabine treatment. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1085

24. Brantley, Emily Claire. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.

Degree: PhD, 2007, University of Alabama – Birmingham

Glioblastoma multiforme (GBM) is the most common form of cancer in the central nervous system in adults. Because of the infiltrative and aggressive nature of… (more)

Subjects/Keywords: Brain Neoplasms  – metabolism <; br>; Brain Neoplasms  – physiopathology <; br>; Glioblastoma  – metabolism <; br>; Glioblastoma  – physiopathology <; br>; Protein Transport  – genetics <; br>; STAT3 Transcription Factor

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APA (6th Edition):

Brantley, E. C. (2007). Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,193

Chicago Manual of Style (16th Edition):

Brantley, Emily Claire. “Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,193.

MLA Handbook (7th Edition):

Brantley, Emily Claire. “Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression.” 2007. Web. 21 Jan 2020.

Vancouver:

Brantley EC. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,193.

Council of Science Editors:

Brantley EC. Loss of PIAS3 expression in glioblastoma multiforme tumors : implications for STAT-3 activation and gene expression. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,193

25. McCoy, Eric. Expression and function of aquaporins in malignant and non-malignant astrocytes.

Degree: PhD, 2008, University of Alabama – Birmingham

Aquaporins (AQP) constitute the primary pathway for water movement across cellular membrances. As a result, their expression and function are important for regulating cell volume.… (more)

Subjects/Keywords: Aquaporin 1 Aquaporin 4<; br>; Aquaporins  – metabolism<; br>; Astrocytes  – physiology<; br>; Brain Injuries  – physiopathology<; br>; Brain Neoplasms  – metabolism<; br>; Cell Movement<; br>; Neoplasm Invasiveness<; br>; Wounds, Stab  – physiopathology

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APA (6th Edition):

McCoy, E. (2008). Expression and function of aquaporins in malignant and non-malignant astrocytes. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,774

Chicago Manual of Style (16th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,774.

MLA Handbook (7th Edition):

McCoy, Eric. “Expression and function of aquaporins in malignant and non-malignant astrocytes.” 2008. Web. 21 Jan 2020.

Vancouver:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774.

Council of Science Editors:

McCoy E. Expression and function of aquaporins in malignant and non-malignant astrocytes. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,774

26. Fetterman, Jessica L. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.

Degree: PhD, 2011, University of Alabama – Birmingham

While progress has been made in understanding the development and progression of cardiovascular disease (CVD), the mechanisms of CVD risk and initiation are not completely… (more)

Subjects/Keywords: Antigens – metabolism.<; br>; Hematopoietic Stem Cell Transplantation – methods.<; br>; Hematopoietic Stem Cells – metabolism.<; br>; HIV-1<; br>; Immunologic Memory – genetics.<; br>; Lentivirus.<; br>; Neoplasms – immunology<; br>; T-Lymphocytes, Cytotoxic – immunology

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APA (6th Edition):

Fetterman, J. L. (2011). Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1395

Chicago Manual of Style (16th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1395.

MLA Handbook (7th Edition):

Fetterman, Jessica L. “Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility.” 2011. Web. 21 Jan 2020.

Vancouver:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395.

Council of Science Editors:

Fetterman JL. Mitochondrial Genetics And Function In Cardiovascular Disease Susceptibility. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1395

27. Szafran, April Adams. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

The development of molecular imaging technologies has allowed biomedical researchers to study the process of cancer metastasis in animal models of disease. Bioluminescence imaging has… (more)

Subjects/Keywords: Antibodies, Monoclonal  – pharmacology<; br>; Antineoplastic Combined Chemotherapy<; br>; Protocols  – pharmacology<; br>; Bone Neoplasms<; br>; Breast Neoplasms<; br>; Diphosphonates  – pharmacology<; br>; Imidazoles  – pharmacology<; br>; Mammary Neoplasms, Experimental  – pathology<; br>; Receptors, TNF-Related Apoptosis-Inducing Ligand  – agonists

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APA (6th Edition):

Szafran, A. A. (2008). The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,927

Chicago Manual of Style (16th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,927.

MLA Handbook (7th Edition):

Szafran, April Adams. “The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer.” 2008. Web. 21 Jan 2020.

Vancouver:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927.

Council of Science Editors:

Szafran AA. The use of molecular imaging to evaluate the efficacy of treatments for metastatic breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,927

28. Atkinson, George P. (George Prescott). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.

Degree: PhD, 2009, University of Alabama – Birmingham

Glioblastoma (GBM) is an incurable tumor of the central nervous system (CNS). Over the past 50 years, little progress has made in improving the quality… (more)

Subjects/Keywords: Glioblastoma  – pathology<; br>; Neoplasms<; br>; NF-kappa B  – metabolism<; br>; Peptidylprolyl Isomerase<; br>; STAT3 Transcription Factor

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APA (6th Edition):

Atkinson, G. P. (. P. (2009). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,812

Chicago Manual of Style (16th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,812.

MLA Handbook (7th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Web. 21 Jan 2020.

Vancouver:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812.

Council of Science Editors:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812

29. Cody, James Joseph. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.

Degree: PhD, 2008, University of Alabama – Birmingham

Most patients with advanced breast cancer develop osteolytic bone metastases, which have numerous complications. Because current therapies are not curative, new treatments are needed. Conditionally… (more)

Subjects/Keywords: Adenoviridae  – genetics <; br>; Bone Neoplasms  – secondary <; br>; Breast Neoplasms <; br>; Gene Expression Regulation, Neoplastic <; br>; Neoplasm Metastasis  – genetics <; br>; Neoplasm Metastasis  – therapy <; br>; Virus Replication  – genetics

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cody, J. J. (2008). A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,320

Chicago Manual of Style (16th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,320.

MLA Handbook (7th Edition):

Cody, James Joseph. “A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer.” 2008. Web. 21 Jan 2020.

Vancouver:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320.

Council of Science Editors:

Cody JJ. A dual-action, armed replicating adenovirus for the treatment of bone metastases of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,320

30. Anderson, Rashaan. The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies.

Degree: PhD, 2008, University of Alabama – Birmingham

In an effort to extrapolate the detail intricacies of how community-based programs impact public health a systematic review of the literature was performed. This research… (more)

Subjects/Keywords: African Americans<; br>; Breast Neoplasms<; br>; Community Health Services<; br>; Health Services Accessibility<; br>; Health Status Disparities<; br>; Uterine Cervical Neoplasms<; br>; Women

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Anderson, R. (2008). The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,429

Chicago Manual of Style (16th Edition):

Anderson, Rashaan. “The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed January 21, 2020. http://contentdm.mhsl.uab.edu/u?/etd,429.

MLA Handbook (7th Edition):

Anderson, Rashaan. “The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies.” 2008. Web. 21 Jan 2020.

Vancouver:

Anderson R. The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Jan 21]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,429.

Council of Science Editors:

Anderson R. The role of community-based programs in addressing health disparities as it relates to breast and cervical cancer in African American women : systematic review of studies. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,429

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