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You searched for subject:(Bortezomib). Showing records 1 – 30 of 48 total matches.

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Princeton University

1. Arlow, Tim. Ubiquitination of the DNA Mismatch Repair Protein Msh2 .

Degree: PhD, 2012, Princeton University

 MSH2 is required for DNA mismatch repair recognition in eukaryotes. Deleterious mutations in human MSH2 account for approximately half of the alleles associated with a… (more)

Subjects/Keywords: Bortezomib; DNA Mismatch Repair; Ubiquitination

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APA (6th Edition):

Arlow, T. (2012). Ubiquitination of the DNA Mismatch Repair Protein Msh2 . (Doctoral Dissertation). Princeton University. Retrieved from http://arks.princeton.edu/ark:/88435/dsp012n49t172r

Chicago Manual of Style (16th Edition):

Arlow, Tim. “Ubiquitination of the DNA Mismatch Repair Protein Msh2 .” 2012. Doctoral Dissertation, Princeton University. Accessed December 10, 2018. http://arks.princeton.edu/ark:/88435/dsp012n49t172r.

MLA Handbook (7th Edition):

Arlow, Tim. “Ubiquitination of the DNA Mismatch Repair Protein Msh2 .” 2012. Web. 10 Dec 2018.

Vancouver:

Arlow T. Ubiquitination of the DNA Mismatch Repair Protein Msh2 . [Internet] [Doctoral dissertation]. Princeton University; 2012. [cited 2018 Dec 10]. Available from: http://arks.princeton.edu/ark:/88435/dsp012n49t172r.

Council of Science Editors:

Arlow T. Ubiquitination of the DNA Mismatch Repair Protein Msh2 . [Doctoral Dissertation]. Princeton University; 2012. Available from: http://arks.princeton.edu/ark:/88435/dsp012n49t172r


Harvard University

2. Sprague, Jared Michael. TRPV1 Sensitization in Primary Sensory Neurons.

Degree: PhD, Biological Sciences in Dental Medicine, 2014, Harvard University

 Pain is a major personal and community burden throughout the world with currently limited treatment options for persistent pain due to unacceptable side effects, dependence… (more)

Subjects/Keywords: Neurosciences; Bortezomib; Optovin; Sensitization; TRPA1; TRPV1

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APA (6th Edition):

Sprague, J. M. (2014). TRPV1 Sensitization in Primary Sensory Neurons. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115

Chicago Manual of Style (16th Edition):

Sprague, Jared Michael. “TRPV1 Sensitization in Primary Sensory Neurons.” 2014. Doctoral Dissertation, Harvard University. Accessed December 10, 2018. http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115.

MLA Handbook (7th Edition):

Sprague, Jared Michael. “TRPV1 Sensitization in Primary Sensory Neurons.” 2014. Web. 10 Dec 2018.

Vancouver:

Sprague JM. TRPV1 Sensitization in Primary Sensory Neurons. [Internet] [Doctoral dissertation]. Harvard University; 2014. [cited 2018 Dec 10]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115.

Council of Science Editors:

Sprague JM. TRPV1 Sensitization in Primary Sensory Neurons. [Doctoral Dissertation]. Harvard University; 2014. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:12274115


Universitat Autònoma de Barcelona

3. Bruna Escuer, Jordi. Neuropatia induïda per Bortezomib. Caracterització i factors de risc en un model experimental.

Degree: Departament de Biologia Cel·lular i de Fisiologia, 2012, Universitat Autònoma de Barcelona

 Introduction Peripheral neuropathy (PN) is a dose-limiting adverse event of most chemotherapeutic drugs. The chemotherapy neurotoxic side effect may be permanent with a negative impact… (more)

Subjects/Keywords: Bortezomib; Neuropatia; Citostatics; Ciències Experimentals; 616.8

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APA (6th Edition):

Bruna Escuer, J. (2012). Neuropatia induïda per Bortezomib. Caracterització i factors de risc en un model experimental. (Thesis). Universitat Autònoma de Barcelona. Retrieved from http://hdl.handle.net/10803/97366

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bruna Escuer, Jordi. “Neuropatia induïda per Bortezomib. Caracterització i factors de risc en un model experimental.” 2012. Thesis, Universitat Autònoma de Barcelona. Accessed December 10, 2018. http://hdl.handle.net/10803/97366.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bruna Escuer, Jordi. “Neuropatia induïda per Bortezomib. Caracterització i factors de risc en un model experimental.” 2012. Web. 10 Dec 2018.

Vancouver:

Bruna Escuer J. Neuropatia induïda per Bortezomib. Caracterització i factors de risc en un model experimental. [Internet] [Thesis]. Universitat Autònoma de Barcelona; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10803/97366.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bruna Escuer J. Neuropatia induïda per Bortezomib. Caracterització i factors de risc en un model experimental. [Thesis]. Universitat Autònoma de Barcelona; 2012. Available from: http://hdl.handle.net/10803/97366

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. Mizuno, Hiroki; Nakayama, Takayuki; Miyata, Yasuhiko; Saito, Shigeki; Nishiwaki, Satoshi; Nakao, Norihiko; Takeshita, Kyosuke. Mast cells promote the growth of Hodgkin's lymphoma cell tumor by modifying the tumor microenvironment that can be perturbed by bortezomib.

Degree: 博士(医学), 2018, Nagoya University / 名古屋大学

Hodgkin's lymphoma is frequently associated with mast cell infiltration that correlates directly with disease severity, but the mechanisms underlying this relationship remain unclear. Here, we… (more)

Subjects/Keywords: Hodgkin's lymphoma; mast cells; angiogenesis; fibrosis; bortezomib

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APA (6th Edition):

Mizuno, Hiroki; Nakayama, Takayuki; Miyata, Yasuhiko; Saito, Shigeki; Nishiwaki, Satoshi; Nakao, Norihiko; Takeshita, K. (2018). Mast cells promote the growth of Hodgkin's lymphoma cell tumor by modifying the tumor microenvironment that can be perturbed by bortezomib. (Thesis). Nagoya University / 名古屋大学. Retrieved from http://hdl.handle.net/2237/18834

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mizuno, Hiroki; Nakayama, Takayuki; Miyata, Yasuhiko; Saito, Shigeki; Nishiwaki, Satoshi; Nakao, Norihiko; Takeshita, Kyosuke. “Mast cells promote the growth of Hodgkin's lymphoma cell tumor by modifying the tumor microenvironment that can be perturbed by bortezomib.” 2018. Thesis, Nagoya University / 名古屋大学. Accessed December 10, 2018. http://hdl.handle.net/2237/18834.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mizuno, Hiroki; Nakayama, Takayuki; Miyata, Yasuhiko; Saito, Shigeki; Nishiwaki, Satoshi; Nakao, Norihiko; Takeshita, Kyosuke. “Mast cells promote the growth of Hodgkin's lymphoma cell tumor by modifying the tumor microenvironment that can be perturbed by bortezomib.” 2018. Web. 10 Dec 2018.

Vancouver:

Mizuno, Hiroki; Nakayama, Takayuki; Miyata, Yasuhiko; Saito, Shigeki; Nishiwaki, Satoshi; Nakao, Norihiko; Takeshita K. Mast cells promote the growth of Hodgkin's lymphoma cell tumor by modifying the tumor microenvironment that can be perturbed by bortezomib. [Internet] [Thesis]. Nagoya University / 名古屋大学; 2018. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/2237/18834.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mizuno, Hiroki; Nakayama, Takayuki; Miyata, Yasuhiko; Saito, Shigeki; Nishiwaki, Satoshi; Nakao, Norihiko; Takeshita K. Mast cells promote the growth of Hodgkin's lymphoma cell tumor by modifying the tumor microenvironment that can be perturbed by bortezomib. [Thesis]. Nagoya University / 名古屋大学; 2018. Available from: http://hdl.handle.net/2237/18834

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Boston University

5. Lee, Saem. Efficacy and safety of bortezomib with dexamethasone regimen in elderly newly diagnosed multiple myeloma patients with co-morbidities.

Degree: MS, Medical Sciences, 2014, Boston University

Bortezomib-based induction therapies have shown to increase complete response rates and are used as an upfront therapy for newly diagnosed multiple myeloma patients. The standard… (more)

Subjects/Keywords: Oncology; Bortezomib; Multiple myeloma; Newly diagnosed

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APA (6th Edition):

Lee, S. (2014). Efficacy and safety of bortezomib with dexamethasone regimen in elderly newly diagnosed multiple myeloma patients with co-morbidities. (Masters Thesis). Boston University. Retrieved from http://hdl.handle.net/2144/14373

Chicago Manual of Style (16th Edition):

Lee, Saem. “Efficacy and safety of bortezomib with dexamethasone regimen in elderly newly diagnosed multiple myeloma patients with co-morbidities.” 2014. Masters Thesis, Boston University. Accessed December 10, 2018. http://hdl.handle.net/2144/14373.

MLA Handbook (7th Edition):

Lee, Saem. “Efficacy and safety of bortezomib with dexamethasone regimen in elderly newly diagnosed multiple myeloma patients with co-morbidities.” 2014. Web. 10 Dec 2018.

Vancouver:

Lee S. Efficacy and safety of bortezomib with dexamethasone regimen in elderly newly diagnosed multiple myeloma patients with co-morbidities. [Internet] [Masters thesis]. Boston University; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/2144/14373.

Council of Science Editors:

Lee S. Efficacy and safety of bortezomib with dexamethasone regimen in elderly newly diagnosed multiple myeloma patients with co-morbidities. [Masters Thesis]. Boston University; 2014. Available from: http://hdl.handle.net/2144/14373


University of South Florida

6. Yarde, Danielle N. The Fanconi Anemia (FA/BRCA DNA damage repair pathway is reglated by NF-[kappa]B and mediates drug resistance in multiple myeloma.

Degree: 2010, University of South Florida

 The Fanconi Anemia (FA)/BRCA DNA damage repair pathway plays a critical role in the cellular response to stress induced by DNA alkylating agents and greatly… (more)

Subjects/Keywords: Hematologic malignancies; Re1B; p50; Melphalan; Bortezomib; American Studies; Arts and Humanities

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APA (6th Edition):

Yarde, D. N. (2010). The Fanconi Anemia (FA/BRCA DNA damage repair pathway is reglated by NF-[kappa]B and mediates drug resistance in multiple myeloma. (Thesis). University of South Florida. Retrieved from http://scholarcommons.usf.edu/etd/1818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yarde, Danielle N. “The Fanconi Anemia (FA/BRCA DNA damage repair pathway is reglated by NF-[kappa]B and mediates drug resistance in multiple myeloma.” 2010. Thesis, University of South Florida. Accessed December 10, 2018. http://scholarcommons.usf.edu/etd/1818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yarde, Danielle N. “The Fanconi Anemia (FA/BRCA DNA damage repair pathway is reglated by NF-[kappa]B and mediates drug resistance in multiple myeloma.” 2010. Web. 10 Dec 2018.

Vancouver:

Yarde DN. The Fanconi Anemia (FA/BRCA DNA damage repair pathway is reglated by NF-[kappa]B and mediates drug resistance in multiple myeloma. [Internet] [Thesis]. University of South Florida; 2010. [cited 2018 Dec 10]. Available from: http://scholarcommons.usf.edu/etd/1818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yarde DN. The Fanconi Anemia (FA/BRCA DNA damage repair pathway is reglated by NF-[kappa]B and mediates drug resistance in multiple myeloma. [Thesis]. University of South Florida; 2010. Available from: http://scholarcommons.usf.edu/etd/1818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Araújo, Karla Patrícia Cardoso. Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular.

Degree: PhD, Anatomia dos Animais Domésticos e Silvestres, 2009, University of São Paulo

A distrofia muscular progressiva no cão Golden Retriever (GRMD) é uma miopatia genética homóloga à Distrofia Muscular de Duchenne (DMD) que acomete humanos. A utilização… (more)

Subjects/Keywords: Bortezomib; Bortezomibe; Distrofina; Dystrophin; GRMD; GRMD model; Proteasome; Proteassoma

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APA (6th Edition):

Araújo, K. P. C. (2009). Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular. (Doctoral Dissertation). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;

Chicago Manual of Style (16th Edition):

Araújo, Karla Patrícia Cardoso. “Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular.” 2009. Doctoral Dissertation, University of São Paulo. Accessed December 10, 2018. http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;.

MLA Handbook (7th Edition):

Araújo, Karla Patrícia Cardoso. “Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular.” 2009. Web. 10 Dec 2018.

Vancouver:

Araújo KPC. Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular. [Internet] [Doctoral dissertation]. University of São Paulo; 2009. [cited 2018 Dec 10]. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;.

Council of Science Editors:

Araújo KPC. Uso de Bortezomibe (PS-341) em cães da raça Golden Retriever afetados pela Distrofia Muscular Progressiva (GRMD) - Avaliação da viabilidade da terapia e reestruturação da Distrofina Muscular. [Doctoral Dissertation]. University of São Paulo; 2009. Available from: http://www.teses.usp.br/teses/disponiveis/10/10132/tde-04022011-081208/ ;


University of Sydney

8. Al-Eisawi, Zaynab. Combinations between platinum drugs and bortezomib and changes in nature of administration in ovarian tumour models .

Degree: 2013, University of Sydney

 Currently, chemotherapy based on cisplatin and carboplatin is the primary treatment for ovarian cancer. Although cisplatin is carried into the cell by CTR1, the drug… (more)

Subjects/Keywords: Keywords: cisplatin; bortezomib; CTR1; resistance; combination; ovarian cancer

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APA (6th Edition):

Al-Eisawi, Z. (2013). Combinations between platinum drugs and bortezomib and changes in nature of administration in ovarian tumour models . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/9465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Al-Eisawi, Zaynab. “Combinations between platinum drugs and bortezomib and changes in nature of administration in ovarian tumour models .” 2013. Thesis, University of Sydney. Accessed December 10, 2018. http://hdl.handle.net/2123/9465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Al-Eisawi, Zaynab. “Combinations between platinum drugs and bortezomib and changes in nature of administration in ovarian tumour models .” 2013. Web. 10 Dec 2018.

Vancouver:

Al-Eisawi Z. Combinations between platinum drugs and bortezomib and changes in nature of administration in ovarian tumour models . [Internet] [Thesis]. University of Sydney; 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/2123/9465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Al-Eisawi Z. Combinations between platinum drugs and bortezomib and changes in nature of administration in ovarian tumour models . [Thesis]. University of Sydney; 2013. Available from: http://hdl.handle.net/2123/9465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. 이, 동민. Nutlin-3 sensitizes cancer cells to bortezomib via induction of paraptosis-associated cell death.

Degree: 2016, Ajou University

The ubiquitin-proteasome system is a complex and tightly controlled system in charge of degrading 80-90% of proteins, and is central to regulating cellular function and… (more)

Subjects/Keywords: bortezomib; nutlin-3; cancer; paraptosis; endoplasmic reticulum; mitochondria

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APA (6th Edition):

이, . (2016). Nutlin-3 sensitizes cancer cells to bortezomib via induction of paraptosis-associated cell death. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/13032 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000022078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

이, 동민. “Nutlin-3 sensitizes cancer cells to bortezomib via induction of paraptosis-associated cell death.” 2016. Thesis, Ajou University. Accessed December 10, 2018. http://repository.ajou.ac.kr/handle/201003/13032 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000022078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

이, 동민. “Nutlin-3 sensitizes cancer cells to bortezomib via induction of paraptosis-associated cell death.” 2016. Web. 10 Dec 2018.

Vancouver:

이 . Nutlin-3 sensitizes cancer cells to bortezomib via induction of paraptosis-associated cell death. [Internet] [Thesis]. Ajou University; 2016. [cited 2018 Dec 10]. Available from: http://repository.ajou.ac.kr/handle/201003/13032 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000022078.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

이 . Nutlin-3 sensitizes cancer cells to bortezomib via induction of paraptosis-associated cell death. [Thesis]. Ajou University; 2016. Available from: http://repository.ajou.ac.kr/handle/201003/13032 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000022078

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Debrecen

10. Mbamali, Monsobundu Michael. Pharmacotherapy of multiple myeloma .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 Multiple Myeloma is an incurable but treatable disease which predominantly affects the elderly. It constitutes about 10 percent of hematologic malignancies and 1 percent of… (more)

Subjects/Keywords: Multiple Myeloma; Lenalidomide; Bortezomib

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APA (6th Edition):

Mbamali, M. M. (n.d.). Pharmacotherapy of multiple myeloma . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/233612

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mbamali, Monsobundu Michael. “Pharmacotherapy of multiple myeloma .” Thesis, University of Debrecen. Accessed December 10, 2018. http://hdl.handle.net/2437/233612.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mbamali, Monsobundu Michael. “Pharmacotherapy of multiple myeloma .” Web. 10 Dec 2018.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Mbamali MM. Pharmacotherapy of multiple myeloma . [Internet] [Thesis]. University of Debrecen; [cited 2018 Dec 10]. Available from: http://hdl.handle.net/2437/233612.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Mbamali MM. Pharmacotherapy of multiple myeloma . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/233612

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.


University of Southern California

11. Sainz, Marcos A. Design, synthesis, and biological evaluation of novel therapeutics for cancer.

Degree: PhD, Chemistry, 2015, University of Southern California

 This dissertation details my efforts towards the design, synthesis and biological evaluation of novel, small molecule anti-cancer agents. This was accomplished with a joint collaboration… (more)

Subjects/Keywords: organic chemistry; synthesis; medicinal chemistry; dUTPase; PNA; bortezomib; MEF2

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APA (6th Edition):

Sainz, M. A. (2015). Design, synthesis, and biological evaluation of novel therapeutics for cancer. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/518515/rec/1873

Chicago Manual of Style (16th Edition):

Sainz, Marcos A. “Design, synthesis, and biological evaluation of novel therapeutics for cancer.” 2015. Doctoral Dissertation, University of Southern California. Accessed December 10, 2018. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/518515/rec/1873.

MLA Handbook (7th Edition):

Sainz, Marcos A. “Design, synthesis, and biological evaluation of novel therapeutics for cancer.” 2015. Web. 10 Dec 2018.

Vancouver:

Sainz MA. Design, synthesis, and biological evaluation of novel therapeutics for cancer. [Internet] [Doctoral dissertation]. University of Southern California; 2015. [cited 2018 Dec 10]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/518515/rec/1873.

Council of Science Editors:

Sainz MA. Design, synthesis, and biological evaluation of novel therapeutics for cancer. [Doctoral Dissertation]. University of Southern California; 2015. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/518515/rec/1873


University of Michigan

12. Wolter, Keith Garret. Mechanisms of Melanoma Cell Survival and Maintenance: Targeting Apoptosis and Authophagy to Improve Disease Treatment.

Degree: PhD, Cellular & Molecular Biology, 2011, University of Michigan

 Metastatic melanoma has proven exceptionally difficult to treat, in part due to consistent defects in cell death pathways. Overexpression of anti-apoptosis proteins from the Bcl-2… (more)

Subjects/Keywords: Melanoma; Autophagy; Apoptosis; Gossypol; Bortezomib; Molecular, Cellular and Developmental Biology; Science

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APA (6th Edition):

Wolter, K. G. (2011). Mechanisms of Melanoma Cell Survival and Maintenance: Targeting Apoptosis and Authophagy to Improve Disease Treatment. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/84630

Chicago Manual of Style (16th Edition):

Wolter, Keith Garret. “Mechanisms of Melanoma Cell Survival and Maintenance: Targeting Apoptosis and Authophagy to Improve Disease Treatment.” 2011. Doctoral Dissertation, University of Michigan. Accessed December 10, 2018. http://hdl.handle.net/2027.42/84630.

MLA Handbook (7th Edition):

Wolter, Keith Garret. “Mechanisms of Melanoma Cell Survival and Maintenance: Targeting Apoptosis and Authophagy to Improve Disease Treatment.” 2011. Web. 10 Dec 2018.

Vancouver:

Wolter KG. Mechanisms of Melanoma Cell Survival and Maintenance: Targeting Apoptosis and Authophagy to Improve Disease Treatment. [Internet] [Doctoral dissertation]. University of Michigan; 2011. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/2027.42/84630.

Council of Science Editors:

Wolter KG. Mechanisms of Melanoma Cell Survival and Maintenance: Targeting Apoptosis and Authophagy to Improve Disease Treatment. [Doctoral Dissertation]. University of Michigan; 2011. Available from: http://hdl.handle.net/2027.42/84630


University of Ottawa

13. Jary, Calvin. Pre-Clinical Assessment of the Proteasomal Inhibitor Bortezomib as a Generalized Therapeutic Approach for Recessively Inherited Disorders .

Degree: 2017, University of Ottawa

 The number of known monogenic rare diseases (~7000) exceeds the number of effective treatments (~500) by more than an order of magnitude underlining the pressing… (more)

Subjects/Keywords: rare diseases; bortezomib; recessive disorders; velcade; drug repurposing; proteasome inhibitor

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APA (6th Edition):

Jary, C. (2017). Pre-Clinical Assessment of the Proteasomal Inhibitor Bortezomib as a Generalized Therapeutic Approach for Recessively Inherited Disorders . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/36066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jary, Calvin. “Pre-Clinical Assessment of the Proteasomal Inhibitor Bortezomib as a Generalized Therapeutic Approach for Recessively Inherited Disorders .” 2017. Thesis, University of Ottawa. Accessed December 10, 2018. http://hdl.handle.net/10393/36066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jary, Calvin. “Pre-Clinical Assessment of the Proteasomal Inhibitor Bortezomib as a Generalized Therapeutic Approach for Recessively Inherited Disorders .” 2017. Web. 10 Dec 2018.

Vancouver:

Jary C. Pre-Clinical Assessment of the Proteasomal Inhibitor Bortezomib as a Generalized Therapeutic Approach for Recessively Inherited Disorders . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10393/36066.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jary C. Pre-Clinical Assessment of the Proteasomal Inhibitor Bortezomib as a Generalized Therapeutic Approach for Recessively Inherited Disorders . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/36066

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

14. Manton, Christa A. Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma.

Degree: PhD, 2015, Texas Medical Center

  New therapeutic options are needed for glioblastoma, a deadly disease with a median survival of only 14 months with current treatment. The proteasome inhibitor… (more)

Subjects/Keywords: bortezomib; marizomib; proteasome; proteasome inhibitor; glioblastoma; caspase; apoptosis; vorinostat; histone deacetylase inhibitor; Cancer Biology

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APA (6th Edition):

Manton, C. A. (2015). Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/545

Chicago Manual of Style (16th Edition):

Manton, Christa A. “Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma.” 2015. Doctoral Dissertation, Texas Medical Center. Accessed December 10, 2018. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/545.

MLA Handbook (7th Edition):

Manton, Christa A. “Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma.” 2015. Web. 10 Dec 2018.

Vancouver:

Manton CA. Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma. [Internet] [Doctoral dissertation]. Texas Medical Center; 2015. [cited 2018 Dec 10]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/545.

Council of Science Editors:

Manton CA. Induction of caspase-dependent death by proteasome targeted therapy in glioblastoma. [Doctoral Dissertation]. Texas Medical Center; 2015. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/545


Kent State University

15. Shyam, Sunitha. S-phase Synchronization Promotes Chemoradiotherapy-induced Apoptosis in Prostate Cancer Cell Lines.

Degree: MS, College of Biomedical Sciences, 2007, Kent State University

 Prostate cancer is the second leading cause of death in men in the USA for the year 2007 and cells are generally resistant to treatments.… (more)

Subjects/Keywords: Biology, Molecular; Apo2L/TRAIL; Bortezomib; C4-2; PARP-1; aphidicolin; Cell; C4-2 cells

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APA (6th Edition):

Shyam, S. (2007). S-phase Synchronization Promotes Chemoradiotherapy-induced Apoptosis in Prostate Cancer Cell Lines. (Masters Thesis). Kent State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=kent1185835523

Chicago Manual of Style (16th Edition):

Shyam, Sunitha. “S-phase Synchronization Promotes Chemoradiotherapy-induced Apoptosis in Prostate Cancer Cell Lines.” 2007. Masters Thesis, Kent State University. Accessed December 10, 2018. http://rave.ohiolink.edu/etdc/view?acc_num=kent1185835523.

MLA Handbook (7th Edition):

Shyam, Sunitha. “S-phase Synchronization Promotes Chemoradiotherapy-induced Apoptosis in Prostate Cancer Cell Lines.” 2007. Web. 10 Dec 2018.

Vancouver:

Shyam S. S-phase Synchronization Promotes Chemoradiotherapy-induced Apoptosis in Prostate Cancer Cell Lines. [Internet] [Masters thesis]. Kent State University; 2007. [cited 2018 Dec 10]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1185835523.

Council of Science Editors:

Shyam S. S-phase Synchronization Promotes Chemoradiotherapy-induced Apoptosis in Prostate Cancer Cell Lines. [Masters Thesis]. Kent State University; 2007. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=kent1185835523


Universidade de Lisboa

16. Esteves, Susana Luísa Augusto. Utilização de metodologia Bayesiana e métodos de comparação indirecta em meta-análise: uma aplicação ao tratamento de mieloma múltiplo.

Degree: 2009, Universidade de Lisboa

Tese de mestrado, Bioestatística, Universidade de Lisboa, Faculdade de Ciências, 2009

Nos últimos anos, os fármacos talidomida, bortezomib e lenalidomida vieram alterar o paradigma do… (more)

Subjects/Keywords: Meta-análise; Comparação indirecta; Mieloma múltiplo; Talidomida; Bortezomib; Lenalidomida; Teses de mestrado - 2009

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APA (6th Edition):

Esteves, S. L. A. (2009). Utilização de metodologia Bayesiana e métodos de comparação indirecta em meta-análise: uma aplicação ao tratamento de mieloma múltiplo. (Thesis). Universidade de Lisboa. Retrieved from http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/4920

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Esteves, Susana Luísa Augusto. “Utilização de metodologia Bayesiana e métodos de comparação indirecta em meta-análise: uma aplicação ao tratamento de mieloma múltiplo.” 2009. Thesis, Universidade de Lisboa. Accessed December 10, 2018. http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/4920.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Esteves, Susana Luísa Augusto. “Utilização de metodologia Bayesiana e métodos de comparação indirecta em meta-análise: uma aplicação ao tratamento de mieloma múltiplo.” 2009. Web. 10 Dec 2018.

Vancouver:

Esteves SLA. Utilização de metodologia Bayesiana e métodos de comparação indirecta em meta-análise: uma aplicação ao tratamento de mieloma múltiplo. [Internet] [Thesis]. Universidade de Lisboa; 2009. [cited 2018 Dec 10]. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/4920.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Esteves SLA. Utilização de metodologia Bayesiana e métodos de comparação indirecta em meta-análise: uma aplicação ao tratamento de mieloma múltiplo. [Thesis]. Universidade de Lisboa; 2009. Available from: http://www.rcaap.pt/detail.jsp?id=oai:repositorio.ul.pt:10451/4920

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Zarouli, Chrysothea. Μελέτη των νευρολογικών εκδηλώσεων σε ασθενείς με πλασματοκυτταρικές δυσκρασίες ως αποτέλεσμα εκδήλωσης της βασικής νόσου ή νευροτοξικής θεραπείας.

Degree: 2013, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Paraprotein related peripheral neuropathy (PN) is frequently associated with treatment administration. Vincristine, thalidomide and more recently bortezomib are common neurotoxic agents inducing PN. The purpose… (more)

Subjects/Keywords: Νευροτοξική περιφερική νευροπάθεια; Πολλαπλούν μυέλωμα; Bortezomib; Drug induced peripheral neuropathy; Multiple myeloma

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APA (6th Edition):

Zarouli, C. (2013). Μελέτη των νευρολογικών εκδηλώσεων σε ασθενείς με πλασματοκυτταρικές δυσκρασίες ως αποτέλεσμα εκδήλωσης της βασικής νόσου ή νευροτοξικής θεραπείας. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/40455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zarouli, Chrysothea. “Μελέτη των νευρολογικών εκδηλώσεων σε ασθενείς με πλασματοκυτταρικές δυσκρασίες ως αποτέλεσμα εκδήλωσης της βασικής νόσου ή νευροτοξικής θεραπείας.” 2013. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed December 10, 2018. http://hdl.handle.net/10442/hedi/40455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zarouli, Chrysothea. “Μελέτη των νευρολογικών εκδηλώσεων σε ασθενείς με πλασματοκυτταρικές δυσκρασίες ως αποτέλεσμα εκδήλωσης της βασικής νόσου ή νευροτοξικής θεραπείας.” 2013. Web. 10 Dec 2018.

Vancouver:

Zarouli C. Μελέτη των νευρολογικών εκδηλώσεων σε ασθενείς με πλασματοκυτταρικές δυσκρασίες ως αποτέλεσμα εκδήλωσης της βασικής νόσου ή νευροτοξικής θεραπείας. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10442/hedi/40455.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zarouli C. Μελέτη των νευρολογικών εκδηλώσεων σε ασθενείς με πλασματοκυτταρικές δυσκρασίες ως αποτέλεσμα εκδήλωσης της βασικής νόσου ή νευροτοξικής θεραπείας. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2013. Available from: http://hdl.handle.net/10442/hedi/40455

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Liapis, Konstantinos. Η μελέτη των μεταβολών που επάγονται απο τους νεότερους φαρμακευτικούς παράγοντες στα πλασματοκύτταρα και στο μικροπεριβάλλον των ασθενών με πολλαπλούν μυέλωμα και η συσχέτιση τους με κλινικά δεδομένα.

Degree: 2016, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Purpose: Maximizing the response rate to first-line therapy in multiple myeloma (MM) is important because it leads to improved outcome. Gene-expression studies have identified prognostic… (more)

Subjects/Keywords: Βορτεζομίμπη; Πολλαπλούν μυέλωμα; Γονιδιακή έκφραση; Φαρμακογενετική ανάλυση; Bortezomib; Multiple myeloma; Gene expression; Pharmacogenomics; Survivin

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APA (6th Edition):

Liapis, K. (2016). Η μελέτη των μεταβολών που επάγονται απο τους νεότερους φαρμακευτικούς παράγοντες στα πλασματοκύτταρα και στο μικροπεριβάλλον των ασθενών με πολλαπλούν μυέλωμα και η συσχέτιση τους με κλινικά δεδομένα. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/37061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liapis, Konstantinos. “Η μελέτη των μεταβολών που επάγονται απο τους νεότερους φαρμακευτικούς παράγοντες στα πλασματοκύτταρα και στο μικροπεριβάλλον των ασθενών με πολλαπλούν μυέλωμα και η συσχέτιση τους με κλινικά δεδομένα.” 2016. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed December 10, 2018. http://hdl.handle.net/10442/hedi/37061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liapis, Konstantinos. “Η μελέτη των μεταβολών που επάγονται απο τους νεότερους φαρμακευτικούς παράγοντες στα πλασματοκύτταρα και στο μικροπεριβάλλον των ασθενών με πολλαπλούν μυέλωμα και η συσχέτιση τους με κλινικά δεδομένα.” 2016. Web. 10 Dec 2018.

Vancouver:

Liapis K. Η μελέτη των μεταβολών που επάγονται απο τους νεότερους φαρμακευτικούς παράγοντες στα πλασματοκύτταρα και στο μικροπεριβάλλον των ασθενών με πολλαπλούν μυέλωμα και η συσχέτιση τους με κλινικά δεδομένα. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10442/hedi/37061.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liapis K. Η μελέτη των μεταβολών που επάγονται απο τους νεότερους φαρμακευτικούς παράγοντες στα πλασματοκύτταρα και στο μικροπεριβάλλον των ασθενών με πολλαπλούν μυέλωμα και η συσχέτιση τους με κλινικά δεδομένα. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2016. Available from: http://hdl.handle.net/10442/hedi/37061

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of South Florida

19. Cross, Brittany Lynne. Inhibition of p53 DNA binding function by the MDM2 acidic domain.

Degree: 2011, University of South Florida

 MDM2 regulates p53 predominantly by promoting p53 ubiquitination. However, ubiquitination-independent mechanisms of MDM2 have also been implicated. Here we show that MDM2 inhibits p53 DNA… (more)

Subjects/Keywords: ARF; Bortezomib; conformation; MDMX; Nutlin; SUV39H1; American Studies; Arts and Humanities; Cell Biology; Molecular Biology; Oncology

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APA (6th Edition):

Cross, B. L. (2011). Inhibition of p53 DNA binding function by the MDM2 acidic domain. (Thesis). University of South Florida. Retrieved from http://scholarcommons.usf.edu/etd/3050

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cross, Brittany Lynne. “Inhibition of p53 DNA binding function by the MDM2 acidic domain.” 2011. Thesis, University of South Florida. Accessed December 10, 2018. http://scholarcommons.usf.edu/etd/3050.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cross, Brittany Lynne. “Inhibition of p53 DNA binding function by the MDM2 acidic domain.” 2011. Web. 10 Dec 2018.

Vancouver:

Cross BL. Inhibition of p53 DNA binding function by the MDM2 acidic domain. [Internet] [Thesis]. University of South Florida; 2011. [cited 2018 Dec 10]. Available from: http://scholarcommons.usf.edu/etd/3050.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cross BL. Inhibition of p53 DNA binding function by the MDM2 acidic domain. [Thesis]. University of South Florida; 2011. Available from: http://scholarcommons.usf.edu/etd/3050

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. Franke, N.E. Molecular mechanisms of bortezomib resistance in acute leukemia .

Degree: 2017, Vrije Universiteit Amsterdam

Subjects/Keywords: acute leukemia; bortezomib; carfilzomib; drug resistance; proteasome inhibitors.

Page 1 Page 2 Page 3

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APA (6th Edition):

Franke, N. E. (2017). Molecular mechanisms of bortezomib resistance in acute leukemia . (Doctoral Dissertation). Vrije Universiteit Amsterdam. Retrieved from http://hdl.handle.net/1871/55349

Chicago Manual of Style (16th Edition):

Franke, N E. “Molecular mechanisms of bortezomib resistance in acute leukemia .” 2017. Doctoral Dissertation, Vrije Universiteit Amsterdam. Accessed December 10, 2018. http://hdl.handle.net/1871/55349.

MLA Handbook (7th Edition):

Franke, N E. “Molecular mechanisms of bortezomib resistance in acute leukemia .” 2017. Web. 10 Dec 2018.

Vancouver:

Franke NE. Molecular mechanisms of bortezomib resistance in acute leukemia . [Internet] [Doctoral dissertation]. Vrije Universiteit Amsterdam; 2017. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/1871/55349.

Council of Science Editors:

Franke NE. Molecular mechanisms of bortezomib resistance in acute leukemia . [Doctoral Dissertation]. Vrije Universiteit Amsterdam; 2017. Available from: http://hdl.handle.net/1871/55349


Texas Medical Center

21. Kosturakis, Alyssa K. MULTIPLE MYELOMA AND ITS TREATMENT ALTER PERIPHERAL NERVOUS SYSTEM STRUCTURE AND FUNCTION.

Degree: MS, 2014, Texas Medical Center

  MULTIPLE MYELOMA AND ITS TREATMENT ALTER PERIPHERAL NERVOUS SYSTEM STRUCTURE AND FUNCTION Alyssa Katarina Kosturakis, BA Supervisory Professor: Patrick M. Dougherty, PhD Peripheral neuropathy… (more)

Subjects/Keywords: multiple myeloma; bortezomib; chemotherapy-induced peripheral neuropathy; quantitative sensory testing; Meissner's corpuscle; minocycline; Medical Neurobiology; Medicine and Health Sciences

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APA (6th Edition):

Kosturakis, A. K. (2014). MULTIPLE MYELOMA AND ITS TREATMENT ALTER PERIPHERAL NERVOUS SYSTEM STRUCTURE AND FUNCTION. (Masters Thesis). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/475

Chicago Manual of Style (16th Edition):

Kosturakis, Alyssa K. “MULTIPLE MYELOMA AND ITS TREATMENT ALTER PERIPHERAL NERVOUS SYSTEM STRUCTURE AND FUNCTION.” 2014. Masters Thesis, Texas Medical Center. Accessed December 10, 2018. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/475.

MLA Handbook (7th Edition):

Kosturakis, Alyssa K. “MULTIPLE MYELOMA AND ITS TREATMENT ALTER PERIPHERAL NERVOUS SYSTEM STRUCTURE AND FUNCTION.” 2014. Web. 10 Dec 2018.

Vancouver:

Kosturakis AK. MULTIPLE MYELOMA AND ITS TREATMENT ALTER PERIPHERAL NERVOUS SYSTEM STRUCTURE AND FUNCTION. [Internet] [Masters thesis]. Texas Medical Center; 2014. [cited 2018 Dec 10]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/475.

Council of Science Editors:

Kosturakis AK. MULTIPLE MYELOMA AND ITS TREATMENT ALTER PERIPHERAL NERVOUS SYSTEM STRUCTURE AND FUNCTION. [Masters Thesis]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/475


Texas Medical Center

22. Robinson, Caleb R. Mechanisms of Astrocyte Contribution to Bortezomib-Induced Peripheral Neuropathy.

Degree: PhD, 2014, Texas Medical Center

Bortezomib is a proteasome inhibitor used in the treatment of multiple myeloma and other non-solid malignancies, alone or in combination with other chemotherapy drugs.… (more)

Subjects/Keywords: bortezomib; astrocyte; CIPN; chemotherapy-induced peripheral neuropathy; Medicine and Health Sciences; Nervous System Diseases; Neuroscience and Neurobiology; Other Neuroscience and Neurobiology

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APA (6th Edition):

Robinson, C. R. (2014). Mechanisms of Astrocyte Contribution to Bortezomib-Induced Peripheral Neuropathy. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/485

Chicago Manual of Style (16th Edition):

Robinson, Caleb R. “Mechanisms of Astrocyte Contribution to Bortezomib-Induced Peripheral Neuropathy.” 2014. Doctoral Dissertation, Texas Medical Center. Accessed December 10, 2018. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/485.

MLA Handbook (7th Edition):

Robinson, Caleb R. “Mechanisms of Astrocyte Contribution to Bortezomib-Induced Peripheral Neuropathy.” 2014. Web. 10 Dec 2018.

Vancouver:

Robinson CR. Mechanisms of Astrocyte Contribution to Bortezomib-Induced Peripheral Neuropathy. [Internet] [Doctoral dissertation]. Texas Medical Center; 2014. [cited 2018 Dec 10]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/485.

Council of Science Editors:

Robinson CR. Mechanisms of Astrocyte Contribution to Bortezomib-Induced Peripheral Neuropathy. [Doctoral Dissertation]. Texas Medical Center; 2014. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/485

23. Αναργύρου, Κωνσταντίνος. Μελέτη της προγνωστικής αξίας της αγγειογένεσης του μυελού των οστών σε ασθενείς με πολλαπλούν μυέλωμα.

Degree: 2008, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ)

Multiple myeloma (MM) is a B-cell lymphoproliferative disease with a median survival of approximately 3 years. Intensive research of the last decade has enlightened the… (more)

Subjects/Keywords: Πολλαπλούν μυέλωμα; Πρόγνωση; Αγγειογένεση; Μπορτεζομίμπη; Λόγος αγειοποιητίνη-1 / αγγειοποιητίνη-2; Multiple myeloma; Prognosis; Angiogenesis; Bortezomib; Angiopoietin-1 / angiopoietin-2 ratio

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APA (6th Edition):

Αναργύρου, . . (2008). Μελέτη της προγνωστικής αξίας της αγγειογένεσης του μυελού των οστών σε ασθενείς με πολλαπλούν μυέλωμα. (Thesis). National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Retrieved from http://hdl.handle.net/10442/hedi/23894

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Αναργύρου, Κωνσταντίνος. “Μελέτη της προγνωστικής αξίας της αγγειογένεσης του μυελού των οστών σε ασθενείς με πολλαπλούν μυέλωμα.” 2008. Thesis, National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ). Accessed December 10, 2018. http://hdl.handle.net/10442/hedi/23894.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Αναργύρου, Κωνσταντίνος. “Μελέτη της προγνωστικής αξίας της αγγειογένεσης του μυελού των οστών σε ασθενείς με πολλαπλούν μυέλωμα.” 2008. Web. 10 Dec 2018.

Vancouver:

Αναργύρου . Μελέτη της προγνωστικής αξίας της αγγειογένεσης του μυελού των οστών σε ασθενείς με πολλαπλούν μυέλωμα. [Internet] [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2008. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10442/hedi/23894.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Αναργύρου . Μελέτη της προγνωστικής αξίας της αγγειογένεσης του μυελού των οστών σε ασθενείς με πολλαπλούν μυέλωμα. [Thesis]. National and Kapodistrian University of Athens; Εθνικό και Καποδιστριακό Πανεπιστήμιο Αθηνών (ΕΚΠΑ); 2008. Available from: http://hdl.handle.net/10442/hedi/23894

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Μπεφάνη, Χριστίνα. Διερεύνηση του ρόλου του HIF-2α στην κυτταρική ομοιοστασία.

Degree: 2013, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

Τhe hypoxia inducible transcription factors, HIF-1α and HIF-2α, play central roles in the cellular response to hypoxia. In the present thesis, the molecular mechanisms of… (more)

Subjects/Keywords: Υποξία; Ηπατοκαρκίνωμα; Ερυθροποιητίνη; Κοβάλτιο; Κινάση της καζεΐνης; Βορτεζομίδη; Hypoxia; HIF-2α; Hepatocellular carcinoma; Erythropoetin; Cobalt; Casein kinase; ERK1/2; Bortezomib

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Μπεφάνη, . . (2013). Διερεύνηση του ρόλου του HIF-2α στην κυτταρική ομοιοστασία. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/33294

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Μπεφάνη, Χριστίνα. “Διερεύνηση του ρόλου του HIF-2α στην κυτταρική ομοιοστασία.” 2013. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed December 10, 2018. http://hdl.handle.net/10442/hedi/33294.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Μπεφάνη, Χριστίνα. “Διερεύνηση του ρόλου του HIF-2α στην κυτταρική ομοιοστασία.” 2013. Web. 10 Dec 2018.

Vancouver:

Μπεφάνη . Διερεύνηση του ρόλου του HIF-2α στην κυτταρική ομοιοστασία. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2013. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10442/hedi/33294.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Μπεφάνη . Διερεύνηση του ρόλου του HIF-2α στην κυτταρική ομοιοστασία. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2013. Available from: http://hdl.handle.net/10442/hedi/33294

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Τσαπακίδης, Κωνσταντίνος. Διερεύνηση των σχέσεων των ρυθμιστών κυτταρικού κύκλου και απόπτωσης καθώς και του συστήματος πρωτεασώματος / NFkB στον καρκίνο του προστάτη.

Degree: 2012, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

Objectives: Neuropeptides (NPs) are important signal initiators in advanced prostate cancer (PC) acting partially through activation of nuclear factor kappa B (NFκB). Central to NFκB… (more)

Subjects/Keywords: Βομβεσίνη; Μπορτεζομίμπη; Ενδοθηλίνη-1 (ΕΤ-1); Καρκίνος προστάτη; Πρωτεάσωμα; Bombesin; Bortezomib; Endothelin-1; Prostate cancer; Proteasome

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APA (6th Edition):

Τσαπακίδης, . . (2012). Διερεύνηση των σχέσεων των ρυθμιστών κυτταρικού κύκλου και απόπτωσης καθώς και του συστήματος πρωτεασώματος / NFkB στον καρκίνο του προστάτη. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/27593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Τσαπακίδης, Κωνσταντίνος. “Διερεύνηση των σχέσεων των ρυθμιστών κυτταρικού κύκλου και απόπτωσης καθώς και του συστήματος πρωτεασώματος / NFkB στον καρκίνο του προστάτη.” 2012. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed December 10, 2018. http://hdl.handle.net/10442/hedi/27593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Τσαπακίδης, Κωνσταντίνος. “Διερεύνηση των σχέσεων των ρυθμιστών κυτταρικού κύκλου και απόπτωσης καθώς και του συστήματος πρωτεασώματος / NFkB στον καρκίνο του προστάτη.” 2012. Web. 10 Dec 2018.

Vancouver:

Τσαπακίδης . Διερεύνηση των σχέσεων των ρυθμιστών κυτταρικού κύκλου και απόπτωσης καθώς και του συστήματος πρωτεασώματος / NFkB στον καρκίνο του προστάτη. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10442/hedi/27593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Τσαπακίδης . Διερεύνηση των σχέσεων των ρυθμιστών κυτταρικού κύκλου και απόπτωσης καθώς και του συστήματος πρωτεασώματος / NFkB στον καρκίνο του προστάτη. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2012. Available from: http://hdl.handle.net/10442/hedi/27593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Βουτσαδάκης, Ιωάννης. Παθογένεση του κολο-ορθικού καρκίνου: ο ρόλος των αναστολέων της κυκλοοξυγενάσης, του συστήματος ουβικουϊτίνης-πρωτεασώματος και πειραματικές εφαρμογές.

Degree: 2008, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας

Background: Cyclooxygenase-2 (Cox-2) is an important enzyme in colorectal carcinogenesis and Cox inhibitors such as aspirin (acetylsalicylic acid, ASA) have shown both Cox-2 dependent and… (more)

Subjects/Keywords: Καρκίνος, κολο-ορθικός; Κυκλοοξυγενάση; Πρωτεάσωμα; Ασπιρίνη; Μπορτεζομίμπη; Κυτταρικές σειρές; Colorectal cancer; Cyclooxygenase-2; Proteasome; Aspirin; Bortezomib; Cell lines; HT-29; HCT116

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Βουτσαδάκης, . . (2008). Παθογένεση του κολο-ορθικού καρκίνου: ο ρόλος των αναστολέων της κυκλοοξυγενάσης, του συστήματος ουβικουϊτίνης-πρωτεασώματος και πειραματικές εφαρμογές. (Thesis). University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Retrieved from http://hdl.handle.net/10442/hedi/17179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Βουτσαδάκης, Ιωάννης. “Παθογένεση του κολο-ορθικού καρκίνου: ο ρόλος των αναστολέων της κυκλοοξυγενάσης, του συστήματος ουβικουϊτίνης-πρωτεασώματος και πειραματικές εφαρμογές.” 2008. Thesis, University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας. Accessed December 10, 2018. http://hdl.handle.net/10442/hedi/17179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Βουτσαδάκης, Ιωάννης. “Παθογένεση του κολο-ορθικού καρκίνου: ο ρόλος των αναστολέων της κυκλοοξυγενάσης, του συστήματος ουβικουϊτίνης-πρωτεασώματος και πειραματικές εφαρμογές.” 2008. Web. 10 Dec 2018.

Vancouver:

Βουτσαδάκης . Παθογένεση του κολο-ορθικού καρκίνου: ο ρόλος των αναστολέων της κυκλοοξυγενάσης, του συστήματος ουβικουϊτίνης-πρωτεασώματος και πειραματικές εφαρμογές. [Internet] [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2008. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10442/hedi/17179.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Βουτσαδάκης . Παθογένεση του κολο-ορθικού καρκίνου: ο ρόλος των αναστολέων της κυκλοοξυγενάσης, του συστήματος ουβικουϊτίνης-πρωτεασώματος και πειραματικές εφαρμογές. [Thesis]. University of Thessaly (UTH); Πανεπιστήμιο Θεσσαλίας; 2008. Available from: http://hdl.handle.net/10442/hedi/17179

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Carmony, Kimberly C. Elucidating Proteasome Catalytic Subunit Composition and Its Role in Proteasome Inhibitor Resistance.

Degree: 2016, University of Kentucky

 Proteasome inhibitors bortezomib and carfilzomib are FDA-approved anticancer agents that have contributed to significant improvements in treatment outcomes. However, the eventual onset of acquired resistance… (more)

Subjects/Keywords: Proteasome Inhibitor; Bortezomib; Carfilzomib; Proteasome Subtype; Bifunctional Proteasome Probe; Biochemistry; Cancer Biology; Molecular Biology; Other Pharmacy and Pharmaceutical Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carmony, K. C. (2016). Elucidating Proteasome Catalytic Subunit Composition and Its Role in Proteasome Inhibitor Resistance. (Doctoral Dissertation). University of Kentucky. Retrieved from http://uknowledge.uky.edu/pharmacy_etds/56

Chicago Manual of Style (16th Edition):

Carmony, Kimberly C. “Elucidating Proteasome Catalytic Subunit Composition and Its Role in Proteasome Inhibitor Resistance.” 2016. Doctoral Dissertation, University of Kentucky. Accessed December 10, 2018. http://uknowledge.uky.edu/pharmacy_etds/56.

MLA Handbook (7th Edition):

Carmony, Kimberly C. “Elucidating Proteasome Catalytic Subunit Composition and Its Role in Proteasome Inhibitor Resistance.” 2016. Web. 10 Dec 2018.

Vancouver:

Carmony KC. Elucidating Proteasome Catalytic Subunit Composition and Its Role in Proteasome Inhibitor Resistance. [Internet] [Doctoral dissertation]. University of Kentucky; 2016. [cited 2018 Dec 10]. Available from: http://uknowledge.uky.edu/pharmacy_etds/56.

Council of Science Editors:

Carmony KC. Elucidating Proteasome Catalytic Subunit Composition and Its Role in Proteasome Inhibitor Resistance. [Doctoral Dissertation]. University of Kentucky; 2016. Available from: http://uknowledge.uky.edu/pharmacy_etds/56


University of Debrecen

28. Kristjánsdóttir, Sigrún Katrín. Multiple Myeloma: Pathogenesis, Diagnosis and Specific Treatment .

Degree: DE – Általános Orvostudományi Kar, University of Debrecen

 Multiple myeloma, also known as plasma cell myeloma is the most important subgroup amongst the plasma cell dyscrasisas. It is an incurable disease which although… (more)

Subjects/Keywords: Multiple myeloma; Bortezomib; Carfilzomib; Mephalan; Thalidomide; Lenalidomide; Pomalidomide

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APA (6th Edition):

Kristjánsdóttir, S. K. (n.d.). Multiple Myeloma: Pathogenesis, Diagnosis and Specific Treatment . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/233643

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kristjánsdóttir, Sigrún Katrín. “Multiple Myeloma: Pathogenesis, Diagnosis and Specific Treatment .” Thesis, University of Debrecen. Accessed December 10, 2018. http://hdl.handle.net/2437/233643.

Note: this citation may be lacking information needed for this citation format:
No year of publication.
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kristjánsdóttir, Sigrún Katrín. “Multiple Myeloma: Pathogenesis, Diagnosis and Specific Treatment .” Web. 10 Dec 2018.

Note: this citation may be lacking information needed for this citation format:
No year of publication.

Vancouver:

Kristjánsdóttir SK. Multiple Myeloma: Pathogenesis, Diagnosis and Specific Treatment . [Internet] [Thesis]. University of Debrecen; [cited 2018 Dec 10]. Available from: http://hdl.handle.net/2437/233643.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

Council of Science Editors:

Kristjánsdóttir SK. Multiple Myeloma: Pathogenesis, Diagnosis and Specific Treatment . [Thesis]. University of Debrecen; Available from: http://hdl.handle.net/2437/233643

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation
No year of publication.

29. CHIRIVA INTERNATI; MAURIZIO. PRECLINICAL TESTING OF GALECTIN-3C FOR MULTIPLE MYELOMA.

Degree: 2012, Università degli Studi di Milano

 The American Cancer Society expects that there will be more than 20,000 new cases of multiple myeloma (MM) in the US in 2011 and despite… (more)

Subjects/Keywords: multiple myeloma; cancer; B cells; galectin-3C; bortezomib; animal model; nod/scid; Settore BIO/16 - Anatomia Umana

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

MAURIZIO, C. I. (2012). PRECLINICAL TESTING OF GALECTIN-3C FOR MULTIPLE MYELOMA. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/168390

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

MAURIZIO, CHIRIVA INTERNATI;. “PRECLINICAL TESTING OF GALECTIN-3C FOR MULTIPLE MYELOMA.” 2012. Thesis, Università degli Studi di Milano. Accessed December 10, 2018. http://hdl.handle.net/2434/168390.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

MAURIZIO, CHIRIVA INTERNATI;. “PRECLINICAL TESTING OF GALECTIN-3C FOR MULTIPLE MYELOMA.” 2012. Web. 10 Dec 2018.

Vancouver:

MAURIZIO CI. PRECLINICAL TESTING OF GALECTIN-3C FOR MULTIPLE MYELOMA. [Internet] [Thesis]. Università degli Studi di Milano; 2012. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/2434/168390.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

MAURIZIO CI. PRECLINICAL TESTING OF GALECTIN-3C FOR MULTIPLE MYELOMA. [Thesis]. Università degli Studi di Milano; 2012. Available from: http://hdl.handle.net/2434/168390

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidad de Cantabria

30. Palanca Cuñado, Ana. Respuesta de las neuronas sensitivas al estrés genotóxico inducido por radiaciones ionizantes y por la inhibición del proteasoma.

Degree: Departamento de Anatomía y Biología Celular, 2014, Universidad de Cantabria

 Neurons are highly vulnerable to genotoxic stress. This vulnerability is particularly relevant in sensory ganglion neurons (SGN) since ganglia lacks of blood-brain barrier that prevents… (more)

Subjects/Keywords: Neuronas sensitivas; Estrés genotóxico; Neuropatía periférica; Radiación ionizante; Bortezomib; Sensory neurons; Genotoxic stress; Peripheral neuropathy; Ionizing radiation; 08; 576; 615; 616.8

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APA (6th Edition):

Palanca Cuñado, A. (2014). Respuesta de las neuronas sensitivas al estrés genotóxico inducido por radiaciones ionizantes y por la inhibición del proteasoma. (Thesis). Universidad de Cantabria. Retrieved from http://hdl.handle.net/10803/309129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Palanca Cuñado, Ana. “Respuesta de las neuronas sensitivas al estrés genotóxico inducido por radiaciones ionizantes y por la inhibición del proteasoma.” 2014. Thesis, Universidad de Cantabria. Accessed December 10, 2018. http://hdl.handle.net/10803/309129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Palanca Cuñado, Ana. “Respuesta de las neuronas sensitivas al estrés genotóxico inducido por radiaciones ionizantes y por la inhibición del proteasoma.” 2014. Web. 10 Dec 2018.

Vancouver:

Palanca Cuñado A. Respuesta de las neuronas sensitivas al estrés genotóxico inducido por radiaciones ionizantes y por la inhibición del proteasoma. [Internet] [Thesis]. Universidad de Cantabria; 2014. [cited 2018 Dec 10]. Available from: http://hdl.handle.net/10803/309129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Palanca Cuñado A. Respuesta de las neuronas sensitivas al estrés genotóxico inducido por radiaciones ionizantes y por la inhibición del proteasoma. [Thesis]. Universidad de Cantabria; 2014. Available from: http://hdl.handle.net/10803/309129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

.