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You searched for subject:(Biology medical sciences ). Showing records 1 – 30 of 214970 total matches.

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University of Oxford

1. Hannes, Tobias. Spatial structure and variability of the cardiac t-tubular system and its contribution to electrophysiological heterogeneity.

Degree: PhD, 2012, University of Oxford

 Background: The cardiomyocyte cell membrane is composed of the surface membrane and a complex system of membrane invaginations, the t-tubules. In keeping with its role… (more)

Subjects/Keywords: Biology (medical sciences)

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APA (6th Edition):

Hannes, T. (2012). Spatial structure and variability of the cardiac t-tubular system and its contribution to electrophysiological heterogeneity. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:5294698e-0262-4e6d-bed5-d9c718da7577 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770317

Chicago Manual of Style (16th Edition):

Hannes, Tobias. “Spatial structure and variability of the cardiac t-tubular system and its contribution to electrophysiological heterogeneity.” 2012. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:5294698e-0262-4e6d-bed5-d9c718da7577 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770317.

MLA Handbook (7th Edition):

Hannes, Tobias. “Spatial structure and variability of the cardiac t-tubular system and its contribution to electrophysiological heterogeneity.” 2012. Web. 24 Aug 2019.

Vancouver:

Hannes T. Spatial structure and variability of the cardiac t-tubular system and its contribution to electrophysiological heterogeneity. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:5294698e-0262-4e6d-bed5-d9c718da7577 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770317.

Council of Science Editors:

Hannes T. Spatial structure and variability of the cardiac t-tubular system and its contribution to electrophysiological heterogeneity. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:5294698e-0262-4e6d-bed5-d9c718da7577 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.770317

2. Guen, Vincent. Découverte de la protéine kinase CDK10 / Cycline M et exploration de ses fonctions biologiques en lien avec le syndrome STAR : Discovery of the protein kinase CDK10 / Cyclin M and exploration of its biological functions linked to STAR syndrome.

Degree: Docteur es, Biologie, 2013, Rennes 1

Les kinases dépendantes des cyclines (CDKs) sont les régulateurs essentiels du cycle cellulaire. Au sein de la famille des cyclines, les fonctions de la cycline… (more)

Subjects/Keywords: Sciences médicales; Biologie moléculaire et cellulaire; Medical sciences; Molecular and cellular biology

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APA (6th Edition):

Guen, V. (2013). Découverte de la protéine kinase CDK10 / Cycline M et exploration de ses fonctions biologiques en lien avec le syndrome STAR : Discovery of the protein kinase CDK10 / Cyclin M and exploration of its biological functions linked to STAR syndrome. (Doctoral Dissertation). Rennes 1. Retrieved from http://www.theses.fr/2013REN1S084

Chicago Manual of Style (16th Edition):

Guen, Vincent. “Découverte de la protéine kinase CDK10 / Cycline M et exploration de ses fonctions biologiques en lien avec le syndrome STAR : Discovery of the protein kinase CDK10 / Cyclin M and exploration of its biological functions linked to STAR syndrome.” 2013. Doctoral Dissertation, Rennes 1. Accessed August 24, 2019. http://www.theses.fr/2013REN1S084.

MLA Handbook (7th Edition):

Guen, Vincent. “Découverte de la protéine kinase CDK10 / Cycline M et exploration de ses fonctions biologiques en lien avec le syndrome STAR : Discovery of the protein kinase CDK10 / Cyclin M and exploration of its biological functions linked to STAR syndrome.” 2013. Web. 24 Aug 2019.

Vancouver:

Guen V. Découverte de la protéine kinase CDK10 / Cycline M et exploration de ses fonctions biologiques en lien avec le syndrome STAR : Discovery of the protein kinase CDK10 / Cyclin M and exploration of its biological functions linked to STAR syndrome. [Internet] [Doctoral dissertation]. Rennes 1; 2013. [cited 2019 Aug 24]. Available from: http://www.theses.fr/2013REN1S084.

Council of Science Editors:

Guen V. Découverte de la protéine kinase CDK10 / Cycline M et exploration de ses fonctions biologiques en lien avec le syndrome STAR : Discovery of the protein kinase CDK10 / Cyclin M and exploration of its biological functions linked to STAR syndrome. [Doctoral Dissertation]. Rennes 1; 2013. Available from: http://www.theses.fr/2013REN1S084


University of Oxford

3. O'Flaherty, Linda H. Development of novel in vitro and in vivo models for determining primary events in HLRCC tumourigenesis.

Degree: PhD, 2012, University of Oxford

 Germline mutations of fumarate hydratase (FH), encoding an enzyme of the tricarboxylic acid (TCA) cycle, predispose affected individuals to hereditary leiomyomatosis and renal cell cancer… (more)

Subjects/Keywords: 616.994; Oncology; Biology; Medical Sciences

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APA (6th Edition):

O'Flaherty, L. H. (2012). Development of novel in vitro and in vivo models for determining primary events in HLRCC tumourigenesis. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:184beb13-615b-4bc3-ad2a-baa33ab040bc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.565984

Chicago Manual of Style (16th Edition):

O'Flaherty, Linda H. “Development of novel in vitro and in vivo models for determining primary events in HLRCC tumourigenesis.” 2012. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:184beb13-615b-4bc3-ad2a-baa33ab040bc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.565984.

MLA Handbook (7th Edition):

O'Flaherty, Linda H. “Development of novel in vitro and in vivo models for determining primary events in HLRCC tumourigenesis.” 2012. Web. 24 Aug 2019.

Vancouver:

O'Flaherty LH. Development of novel in vitro and in vivo models for determining primary events in HLRCC tumourigenesis. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:184beb13-615b-4bc3-ad2a-baa33ab040bc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.565984.

Council of Science Editors:

O'Flaherty LH. Development of novel in vitro and in vivo models for determining primary events in HLRCC tumourigenesis. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:184beb13-615b-4bc3-ad2a-baa33ab040bc ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.565984


University of Oxford

4. Badrinarayanan, Anjana. Role of MukBEF in Escherichia coli chromosome organization.

Degree: PhD, 2011, University of Oxford

 In E. coli cells, spatial chromosome organization reflects the genetic map, with the origin located at mid-cell and left and right replichores in either cell… (more)

Subjects/Keywords: 572.87; Biology (medical sciences); Microbiology

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APA (6th Edition):

Badrinarayanan, A. (2011). Role of MukBEF in Escherichia coli chromosome organization. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:8abe7a64-cf7d-433a-9165-0ca2ced915b6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558690

Chicago Manual of Style (16th Edition):

Badrinarayanan, Anjana. “Role of MukBEF in Escherichia coli chromosome organization.” 2011. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:8abe7a64-cf7d-433a-9165-0ca2ced915b6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558690.

MLA Handbook (7th Edition):

Badrinarayanan, Anjana. “Role of MukBEF in Escherichia coli chromosome organization.” 2011. Web. 24 Aug 2019.

Vancouver:

Badrinarayanan A. Role of MukBEF in Escherichia coli chromosome organization. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:8abe7a64-cf7d-433a-9165-0ca2ced915b6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558690.

Council of Science Editors:

Badrinarayanan A. Role of MukBEF in Escherichia coli chromosome organization. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:8abe7a64-cf7d-433a-9165-0ca2ced915b6 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.558690


Columbia University

5. Kuo, Chao-Ling. Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:.

Degree: 2011, Columbia University

 Atherosclerosis, the primary cause of heart attack, stroke, and peripheral vascular disease, is genetically complex and the genes that confer cardiovascular risk remain largely unknown.… (more)

Subjects/Keywords: Medical sciences; Genetics; Molecular biology

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APA (6th Edition):

Kuo, C. (2011). Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8CV4QXC

Chicago Manual of Style (16th Edition):

Kuo, Chao-Ling. “Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:.” 2011. Doctoral Dissertation, Columbia University. Accessed August 24, 2019. https://doi.org/10.7916/D8CV4QXC.

MLA Handbook (7th Edition):

Kuo, Chao-Ling. “Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:.” 2011. Web. 24 Aug 2019.

Vancouver:

Kuo C. Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:. [Internet] [Doctoral dissertation]. Columbia University; 2011. [cited 2019 Aug 24]. Available from: https://doi.org/10.7916/D8CV4QXC.

Council of Science Editors:

Kuo C. Characterization ofAthsq1, an Atherosclerosis Modifier Locus on Mouse Chromosome 4:. [Doctoral Dissertation]. Columbia University; 2011. Available from: https://doi.org/10.7916/D8CV4QXC


Florida State University

6. Zhang, Yujie. Characterization of Role of LARP6 Phosphorylation in Regulating Type I Collagen Biosynthesis in Liver Fibrosis.

Degree: PhD, Biomedical Sciences, 2016, Florida State University

 Liver fibrosis is the common end stage of all chronic liver diseases, such as chronic viral hepatitis, alcoholism, nonalcoholic fatty liver disease, autoimmune hepatitis, alpha… (more)

Subjects/Keywords: Medical sciences; Biology; Biochemistry

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APA (6th Edition):

Zhang, Y. (2016). Characterization of Role of LARP6 Phosphorylation in Regulating Type I Collagen Biosynthesis in Liver Fibrosis. (Doctoral Dissertation). Florida State University. Retrieved from http://purl.flvc.org/fsu/fd/FSU_2016SP_Zhang_fsu_0071E_13114 ;

Chicago Manual of Style (16th Edition):

Zhang, Yujie. “Characterization of Role of LARP6 Phosphorylation in Regulating Type I Collagen Biosynthesis in Liver Fibrosis.” 2016. Doctoral Dissertation, Florida State University. Accessed August 24, 2019. http://purl.flvc.org/fsu/fd/FSU_2016SP_Zhang_fsu_0071E_13114 ;.

MLA Handbook (7th Edition):

Zhang, Yujie. “Characterization of Role of LARP6 Phosphorylation in Regulating Type I Collagen Biosynthesis in Liver Fibrosis.” 2016. Web. 24 Aug 2019.

Vancouver:

Zhang Y. Characterization of Role of LARP6 Phosphorylation in Regulating Type I Collagen Biosynthesis in Liver Fibrosis. [Internet] [Doctoral dissertation]. Florida State University; 2016. [cited 2019 Aug 24]. Available from: http://purl.flvc.org/fsu/fd/FSU_2016SP_Zhang_fsu_0071E_13114 ;.

Council of Science Editors:

Zhang Y. Characterization of Role of LARP6 Phosphorylation in Regulating Type I Collagen Biosynthesis in Liver Fibrosis. [Doctoral Dissertation]. Florida State University; 2016. Available from: http://purl.flvc.org/fsu/fd/FSU_2016SP_Zhang_fsu_0071E_13114 ;


University of Oxford

7. Kang, Youn-Jung. Cell adhesion and signalling at implantation.

Degree: PhD, 2012, University of Oxford

 Implantation failure is one of the major causes of infertility and contributes a major barrier to assisted reproduction success. Establishing a detailed understanding of embryo… (more)

Subjects/Keywords: Biology (medical sciences); Biology; Endocrinology; Gynaecology; Medical Sciences; Obstetrics

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APA (6th Edition):

Kang, Y. (2012). Cell adhesion and signalling at implantation. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:ce5e8c65-c882-48d5-9d00-648c712d924c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711653

Chicago Manual of Style (16th Edition):

Kang, Youn-Jung. “Cell adhesion and signalling at implantation.” 2012. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:ce5e8c65-c882-48d5-9d00-648c712d924c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711653.

MLA Handbook (7th Edition):

Kang, Youn-Jung. “Cell adhesion and signalling at implantation.” 2012. Web. 24 Aug 2019.

Vancouver:

Kang Y. Cell adhesion and signalling at implantation. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:ce5e8c65-c882-48d5-9d00-648c712d924c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711653.

Council of Science Editors:

Kang Y. Cell adhesion and signalling at implantation. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:ce5e8c65-c882-48d5-9d00-648c712d924c ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.711653


University of Oxford

8. Deegan, Rachel Sarah. The role of histone modification in DNA double-strand break repair in Schizosaccharomyces pombe.

Degree: PhD, 2012, University of Oxford

 DNA double-strand breaks (DSBs) are highly genotoxic lesions, which when incorrectly repaired can lead to gross chromosomal rearrangements and tumourigenesis through oncogene activation or loss… (more)

Subjects/Keywords: 572.86459; Biology (medical sciences); Biology (medical sciences); DNA repair

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APA (6th Edition):

Deegan, R. S. (2012). The role of histone modification in DNA double-strand break repair in Schizosaccharomyces pombe. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:88998b84-da94-40dc-80d0-ef46c51d20d1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572469

Chicago Manual of Style (16th Edition):

Deegan, Rachel Sarah. “The role of histone modification in DNA double-strand break repair in Schizosaccharomyces pombe.” 2012. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:88998b84-da94-40dc-80d0-ef46c51d20d1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572469.

MLA Handbook (7th Edition):

Deegan, Rachel Sarah. “The role of histone modification in DNA double-strand break repair in Schizosaccharomyces pombe.” 2012. Web. 24 Aug 2019.

Vancouver:

Deegan RS. The role of histone modification in DNA double-strand break repair in Schizosaccharomyces pombe. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:88998b84-da94-40dc-80d0-ef46c51d20d1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572469.

Council of Science Editors:

Deegan RS. The role of histone modification in DNA double-strand break repair in Schizosaccharomyces pombe. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:88998b84-da94-40dc-80d0-ef46c51d20d1 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572469


University of Oxford

9. O'Brien, Emma Rosemary. The role of astrocytes in brain metastasis.

Degree: PhD, 2014, University of Oxford

 Brain metastasis is a significant clinical problem, and 20-40% of cancer patients will develop metastatic spread to the brain. There remains, however, an urgent need… (more)

Subjects/Keywords: 612.8; Medical Sciences; Biology (medical sciences); Oncology; Astrocytes; brain metastasis

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APA (6th Edition):

O'Brien, E. R. (2014). The role of astrocytes in brain metastasis. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:60efc7bd-4f00-4e84-a964-c4ef55009dfb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658455

Chicago Manual of Style (16th Edition):

O'Brien, Emma Rosemary. “The role of astrocytes in brain metastasis.” 2014. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:60efc7bd-4f00-4e84-a964-c4ef55009dfb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658455.

MLA Handbook (7th Edition):

O'Brien, Emma Rosemary. “The role of astrocytes in brain metastasis.” 2014. Web. 24 Aug 2019.

Vancouver:

O'Brien ER. The role of astrocytes in brain metastasis. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:60efc7bd-4f00-4e84-a964-c4ef55009dfb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658455.

Council of Science Editors:

O'Brien ER. The role of astrocytes in brain metastasis. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:60efc7bd-4f00-4e84-a964-c4ef55009dfb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.658455


University of Oxford

10. Zheng, Shunsheng. Arginine methylation in the E2F1 pathway.

Degree: PhD, 2013, University of Oxford

 The E2F1 pathway plays an important role in coordinating early cell cycle progression. Deregulation of the E2F1 pathway, which may be brought about by somatic… (more)

Subjects/Keywords: 616.994; Medical Sciences; Oncology; Biology (medical sciences); Pharmacology; Methylation; Cancer

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APA (6th Edition):

Zheng, S. (2013). Arginine methylation in the E2F1 pathway. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:0a0e3d43-dedc-490c-92d0-44442c9be1f2 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604391

Chicago Manual of Style (16th Edition):

Zheng, Shunsheng. “Arginine methylation in the E2F1 pathway.” 2013. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:0a0e3d43-dedc-490c-92d0-44442c9be1f2 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604391.

MLA Handbook (7th Edition):

Zheng, Shunsheng. “Arginine methylation in the E2F1 pathway.” 2013. Web. 24 Aug 2019.

Vancouver:

Zheng S. Arginine methylation in the E2F1 pathway. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:0a0e3d43-dedc-490c-92d0-44442c9be1f2 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604391.

Council of Science Editors:

Zheng S. Arginine methylation in the E2F1 pathway. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:0a0e3d43-dedc-490c-92d0-44442c9be1f2 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604391


McMaster University

11. Collins, AF Celeste. The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death.

Degree: MSc, 2013, McMaster University

Pleckstrin homology like domain family A, member 1 (PHLDA1) is a member of the PHLDA family of homologous proteins recognized for their role in… (more)

Subjects/Keywords: PHLDA1; TDAG51; Medical Biochemistry; Medical Cell Biology; Medical Sciences; Reproductive and Urinary Physiology; Medical Biochemistry

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APA (6th Edition):

Collins, A. C. (2013). The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/15325

Chicago Manual of Style (16th Edition):

Collins, AF Celeste. “The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death.” 2013. Masters Thesis, McMaster University. Accessed August 24, 2019. http://hdl.handle.net/11375/15325.

MLA Handbook (7th Edition):

Collins, AF Celeste. “The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death.” 2013. Web. 24 Aug 2019.

Vancouver:

Collins AC. The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death. [Internet] [Masters thesis]. McMaster University; 2013. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/11375/15325.

Council of Science Editors:

Collins AC. The Functional Domains of PHLDA1: Modulation of Intracellular Localization Impacts Apoptotic Cell Death. [Masters Thesis]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/15325


University of Oxford

12. Potgieter, Dawid. Using fast-scan cyclic voltammetry to investigate monoamine release in BAC transgenic rodent models of Parkinson's disease.

Degree: PhD, 2014, University of Oxford

 Parkinson's disease (PD) is the most common neurodegenerative movement disorder. Nigrostriatal dopamine (DA) neurons are particularly susceptible to degeneration in PD, and the concomitant loss… (more)

Subjects/Keywords: 616.8; Neuropathology; Neurogenetics; Biology (medical sciences)

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APA (6th Edition):

Potgieter, D. (2014). Using fast-scan cyclic voltammetry to investigate monoamine release in BAC transgenic rodent models of Parkinson's disease. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:8f83c8b8-7800-4607-98b3-f4a7f6841716 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.665164

Chicago Manual of Style (16th Edition):

Potgieter, Dawid. “Using fast-scan cyclic voltammetry to investigate monoamine release in BAC transgenic rodent models of Parkinson's disease.” 2014. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:8f83c8b8-7800-4607-98b3-f4a7f6841716 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.665164.

MLA Handbook (7th Edition):

Potgieter, Dawid. “Using fast-scan cyclic voltammetry to investigate monoamine release in BAC transgenic rodent models of Parkinson's disease.” 2014. Web. 24 Aug 2019.

Vancouver:

Potgieter D. Using fast-scan cyclic voltammetry to investigate monoamine release in BAC transgenic rodent models of Parkinson's disease. [Internet] [Doctoral dissertation]. University of Oxford; 2014. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:8f83c8b8-7800-4607-98b3-f4a7f6841716 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.665164.

Council of Science Editors:

Potgieter D. Using fast-scan cyclic voltammetry to investigate monoamine release in BAC transgenic rodent models of Parkinson's disease. [Doctoral Dissertation]. University of Oxford; 2014. Available from: http://ora.ox.ac.uk/objects/uuid:8f83c8b8-7800-4607-98b3-f4a7f6841716 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.665164


University of Oxford

13. Lodhia, Kunal. The role of IGF-1R signaling in the DNA damage response of prostate cancer.

Degree: PhD, 2013, University of Oxford

 The type 1 insulin like growth factor receptor (IGF-1R) is a cell surface receptor that mediates proliferation and cell survival. The aim of this project… (more)

Subjects/Keywords: 616.97; Oncology; Urology; Biology; Medical Sciences

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APA (6th Edition):

Lodhia, K. (2013). The role of IGF-1R signaling in the DNA damage response of prostate cancer. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:78bb4539-9873-40d6-84e1-38187e58f585 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669862

Chicago Manual of Style (16th Edition):

Lodhia, Kunal. “The role of IGF-1R signaling in the DNA damage response of prostate cancer.” 2013. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:78bb4539-9873-40d6-84e1-38187e58f585 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669862.

MLA Handbook (7th Edition):

Lodhia, Kunal. “The role of IGF-1R signaling in the DNA damage response of prostate cancer.” 2013. Web. 24 Aug 2019.

Vancouver:

Lodhia K. The role of IGF-1R signaling in the DNA damage response of prostate cancer. [Internet] [Doctoral dissertation]. University of Oxford; 2013. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:78bb4539-9873-40d6-84e1-38187e58f585 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669862.

Council of Science Editors:

Lodhia K. The role of IGF-1R signaling in the DNA damage response of prostate cancer. [Doctoral Dissertation]. University of Oxford; 2013. Available from: http://ora.ox.ac.uk/objects/uuid:78bb4539-9873-40d6-84e1-38187e58f585 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.669862


University of Oxford

14. Roberts, Thomas C. Duchenne muscular dystrophy : RNA-based therapeutics and microRNA biology.

Degree: PhD, 2012, University of Oxford

 Duchenne muscular dystrophy (DMD) is a progressive muscle wasting disorder caused by absence of functional dystrophin protein. This thesis describes investigations into the role of… (more)

Subjects/Keywords: 616.748; Biology (medical sciences); microRNA; epigenetics

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APA (6th Edition):

Roberts, T. C. (2012). Duchenne muscular dystrophy : RNA-based therapeutics and microRNA biology. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:f53ea1f3-92db-4f90-ba95-01f2a56eae8f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572842

Chicago Manual of Style (16th Edition):

Roberts, Thomas C. “Duchenne muscular dystrophy : RNA-based therapeutics and microRNA biology.” 2012. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:f53ea1f3-92db-4f90-ba95-01f2a56eae8f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572842.

MLA Handbook (7th Edition):

Roberts, Thomas C. “Duchenne muscular dystrophy : RNA-based therapeutics and microRNA biology.” 2012. Web. 24 Aug 2019.

Vancouver:

Roberts TC. Duchenne muscular dystrophy : RNA-based therapeutics and microRNA biology. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:f53ea1f3-92db-4f90-ba95-01f2a56eae8f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572842.

Council of Science Editors:

Roberts TC. Duchenne muscular dystrophy : RNA-based therapeutics and microRNA biology. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:f53ea1f3-92db-4f90-ba95-01f2a56eae8f ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.572842


University of Oxford

15. Notari, Mario. Investigation of the biological role of iASPP in vivo.

Degree: PhD, 2011, University of Oxford

 The p53 family of transcription factors, which comprises the products of the TP53, TP63 and TP73 genes, is at the hub of different signalling pathways… (more)

Subjects/Keywords: 572.8; Biology; Cardiovascular disease; Medical Sciences

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APA (6th Edition):

Notari, M. (2011). Investigation of the biological role of iASPP in vivo. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:7fba0cb4-ca7a-4cd4-8d0e-d6a5d231a95a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589601

Chicago Manual of Style (16th Edition):

Notari, Mario. “Investigation of the biological role of iASPP in vivo.” 2011. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:7fba0cb4-ca7a-4cd4-8d0e-d6a5d231a95a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589601.

MLA Handbook (7th Edition):

Notari, Mario. “Investigation of the biological role of iASPP in vivo.” 2011. Web. 24 Aug 2019.

Vancouver:

Notari M. Investigation of the biological role of iASPP in vivo. [Internet] [Doctoral dissertation]. University of Oxford; 2011. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:7fba0cb4-ca7a-4cd4-8d0e-d6a5d231a95a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589601.

Council of Science Editors:

Notari M. Investigation of the biological role of iASPP in vivo. [Doctoral Dissertation]. University of Oxford; 2011. Available from: http://ora.ox.ac.uk/objects/uuid:7fba0cb4-ca7a-4cd4-8d0e-d6a5d231a95a ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.589601


Columbia University

16. Kumar, Brahma Vencel. Identification and characterization of tissue-resident memory T cells in humans.

Degree: 2018, Columbia University

 Memory T cells are critical for maintaining lifelong immunity by protecting against reinfection with previously encountered pathogens. In recent years, a subset of memory T… (more)

Subjects/Keywords: Immunology; T cells; Medical sciences; Molecular biology

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APA (6th Edition):

Kumar, B. V. (2018). Identification and characterization of tissue-resident memory T cells in humans. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8Q2563Z

Chicago Manual of Style (16th Edition):

Kumar, Brahma Vencel. “Identification and characterization of tissue-resident memory T cells in humans.” 2018. Doctoral Dissertation, Columbia University. Accessed August 24, 2019. https://doi.org/10.7916/D8Q2563Z.

MLA Handbook (7th Edition):

Kumar, Brahma Vencel. “Identification and characterization of tissue-resident memory T cells in humans.” 2018. Web. 24 Aug 2019.

Vancouver:

Kumar BV. Identification and characterization of tissue-resident memory T cells in humans. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2019 Aug 24]. Available from: https://doi.org/10.7916/D8Q2563Z.

Council of Science Editors:

Kumar BV. Identification and characterization of tissue-resident memory T cells in humans. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8Q2563Z


University of Oxford

17. Tomlins, Christine Helen. Induction of anti-apoptotic factors by cutaneous Human Papillomaviruses.

Degree: PhD, 2010, University of Oxford

 Human Papillomaviruses (HPVs) are small DNA viruses which specifically infect keratinocytes at different body sites. An association between cutaneous Squamous Cell Carcinoma (SCC) formation, UV… (more)

Subjects/Keywords: 616.994; Biology; Medical Sciences; Biology (medical sciences); Oncology; Viruses; Human papillomavirus; ultraviolet; apoptosis

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APA (6th Edition):

Tomlins, C. H. (2010). Induction of anti-apoptotic factors by cutaneous Human Papillomaviruses. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:121b0494-9cca-4fee-a800-b54fc36fe43e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.531788

Chicago Manual of Style (16th Edition):

Tomlins, Christine Helen. “Induction of anti-apoptotic factors by cutaneous Human Papillomaviruses.” 2010. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:121b0494-9cca-4fee-a800-b54fc36fe43e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.531788.

MLA Handbook (7th Edition):

Tomlins, Christine Helen. “Induction of anti-apoptotic factors by cutaneous Human Papillomaviruses.” 2010. Web. 24 Aug 2019.

Vancouver:

Tomlins CH. Induction of anti-apoptotic factors by cutaneous Human Papillomaviruses. [Internet] [Doctoral dissertation]. University of Oxford; 2010. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:121b0494-9cca-4fee-a800-b54fc36fe43e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.531788.

Council of Science Editors:

Tomlins CH. Induction of anti-apoptotic factors by cutaneous Human Papillomaviruses. [Doctoral Dissertation]. University of Oxford; 2010. Available from: http://ora.ox.ac.uk/objects/uuid:121b0494-9cca-4fee-a800-b54fc36fe43e ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.531788

18. Aggarwal, Sudhir. Role of Extracellular Signal-Regulated Kinase (ERK) in Regulation of Intestinal Epithelial Tight Junctions.

Degree: PhD, Biomedical Sciences, 2010, University of Tennessee Health Science Center

  Evidence indicates that MAP kinase (ERK1/2) is involved in regulation of epithelial tight junctions. There are different opinions expressed by investigators as to whether… (more)

Subjects/Keywords: ERK; Tight Junction; MAPK; Medical Biochemistry; Medical Cell Biology; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Aggarwal, S. (2010). Role of Extracellular Signal-Regulated Kinase (ERK) in Regulation of Intestinal Epithelial Tight Junctions. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/9

Chicago Manual of Style (16th Edition):

Aggarwal, Sudhir. “Role of Extracellular Signal-Regulated Kinase (ERK) in Regulation of Intestinal Epithelial Tight Junctions.” 2010. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed August 24, 2019. https://dc.uthsc.edu/dissertations/9.

MLA Handbook (7th Edition):

Aggarwal, Sudhir. “Role of Extracellular Signal-Regulated Kinase (ERK) in Regulation of Intestinal Epithelial Tight Junctions.” 2010. Web. 24 Aug 2019.

Vancouver:

Aggarwal S. Role of Extracellular Signal-Regulated Kinase (ERK) in Regulation of Intestinal Epithelial Tight Junctions. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2010. [cited 2019 Aug 24]. Available from: https://dc.uthsc.edu/dissertations/9.

Council of Science Editors:

Aggarwal S. Role of Extracellular Signal-Regulated Kinase (ERK) in Regulation of Intestinal Epithelial Tight Junctions. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2010. Available from: https://dc.uthsc.edu/dissertations/9


Johannes Gutenberg Universität Mainz

19. Hussein Allami, Risala. Influence of the chemokine CXCL12 on the progression and the signaling in colorectal cancer.

Degree: 2013, Johannes Gutenberg Universität Mainz

 Das Chemokin CXCL12 (auch bekannt als SDF-1) ist ein kleines Protein (8-14) KDa, das in sechs Isoformen exprimiert wird (SDF-1α, SDF-1β, SDF-1γ, SDF- 1δ, SDF-1ε… (more)

Subjects/Keywords: Biologie; Life sciences

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APA (6th Edition):

Hussein Allami, R. (2013). Influence of the chemokine CXCL12 on the progression and the signaling in colorectal cancer. (Doctoral Dissertation). Johannes Gutenberg Universität Mainz. Retrieved from http://ubm.opus.hbz-nrw.de/volltexte/2013/3489/

Chicago Manual of Style (16th Edition):

Hussein Allami, Risala. “Influence of the chemokine CXCL12 on the progression and the signaling in colorectal cancer.” 2013. Doctoral Dissertation, Johannes Gutenberg Universität Mainz. Accessed August 24, 2019. http://ubm.opus.hbz-nrw.de/volltexte/2013/3489/.

MLA Handbook (7th Edition):

Hussein Allami, Risala. “Influence of the chemokine CXCL12 on the progression and the signaling in colorectal cancer.” 2013. Web. 24 Aug 2019.

Vancouver:

Hussein Allami R. Influence of the chemokine CXCL12 on the progression and the signaling in colorectal cancer. [Internet] [Doctoral dissertation]. Johannes Gutenberg Universität Mainz; 2013. [cited 2019 Aug 24]. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2013/3489/.

Council of Science Editors:

Hussein Allami R. Influence of the chemokine CXCL12 on the progression and the signaling in colorectal cancer. [Doctoral Dissertation]. Johannes Gutenberg Universität Mainz; 2013. Available from: http://ubm.opus.hbz-nrw.de/volltexte/2013/3489/


University of Oxford

20. Javid, Mahsa. Gene modifiers and novel therapies for multiple endocrine neoplasia type 1.

Degree: PhD, 2012, University of Oxford

 Multiple Endocrine Neoplasia Type 1 (MEN1) is an autosomal dominant disorder characterized by the combined occurrence of pituitary, pancreatic islet and parathyroid tumours. MEN1-associated tumours… (more)

Subjects/Keywords: 616.4; Medical Sciences; Biology (medical sciences); Endocrinology; Genetics (life sciences); endocrine; genetics

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APA (6th Edition):

Javid, M. (2012). Gene modifiers and novel therapies for multiple endocrine neoplasia type 1. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373

Chicago Manual of Style (16th Edition):

Javid, Mahsa. “Gene modifiers and novel therapies for multiple endocrine neoplasia type 1.” 2012. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373.

MLA Handbook (7th Edition):

Javid, Mahsa. “Gene modifiers and novel therapies for multiple endocrine neoplasia type 1.” 2012. Web. 24 Aug 2019.

Vancouver:

Javid M. Gene modifiers and novel therapies for multiple endocrine neoplasia type 1. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373.

Council of Science Editors:

Javid M. Gene modifiers and novel therapies for multiple endocrine neoplasia type 1. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:baa5b96d-e186-4811-a867-945cee3c85eb ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.604373

21. Bolen, Alyssa Lynn Jefferson. The Biochemical Pathway Leading to Lpa Generation Upon Blood Coagulation.

Degree: PhD, Biomedical Sciences, 2011, University of Tennessee Health Science Center

  Platelet activation initiates an upsurge in 18:2 and 20:4 lysophosphatidic acid (LPA) production. The biochemical pathway responsible for LPA production during blood clotting is… (more)

Subjects/Keywords: Autotaxin; Lysophosphatidic acid; Phospholipase A1; Medical Cell Biology; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Bolen, A. L. J. (2011). The Biochemical Pathway Leading to Lpa Generation Upon Blood Coagulation. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/27

Chicago Manual of Style (16th Edition):

Bolen, Alyssa Lynn Jefferson. “The Biochemical Pathway Leading to Lpa Generation Upon Blood Coagulation.” 2011. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed August 24, 2019. https://dc.uthsc.edu/dissertations/27.

MLA Handbook (7th Edition):

Bolen, Alyssa Lynn Jefferson. “The Biochemical Pathway Leading to Lpa Generation Upon Blood Coagulation.” 2011. Web. 24 Aug 2019.

Vancouver:

Bolen ALJ. The Biochemical Pathway Leading to Lpa Generation Upon Blood Coagulation. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2011. [cited 2019 Aug 24]. Available from: https://dc.uthsc.edu/dissertations/27.

Council of Science Editors:

Bolen ALJ. The Biochemical Pathway Leading to Lpa Generation Upon Blood Coagulation. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2011. Available from: https://dc.uthsc.edu/dissertations/27

22. Elmeliegy, Mohamed. CNS Penetration of Tyrosine Kinase Inhibitors in Mouse Models.

Degree: PhD, Biomedical Sciences, 2012, University of Tennessee Health Science Center

  For the past three decades, advances in the treatment of central nervous system (CNS) tumors such as malignant glioma have only been modest. One… (more)

Subjects/Keywords: CNS penetration; Crenolanib; Erlotinib; Microdialysis; Tyrosine Kinase Inhibitors; Medical Cell Biology; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Elmeliegy, M. (2012). CNS Penetration of Tyrosine Kinase Inhibitors in Mouse Models. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/74

Chicago Manual of Style (16th Edition):

Elmeliegy, Mohamed. “CNS Penetration of Tyrosine Kinase Inhibitors in Mouse Models.” 2012. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed August 24, 2019. https://dc.uthsc.edu/dissertations/74.

MLA Handbook (7th Edition):

Elmeliegy, Mohamed. “CNS Penetration of Tyrosine Kinase Inhibitors in Mouse Models.” 2012. Web. 24 Aug 2019.

Vancouver:

Elmeliegy M. CNS Penetration of Tyrosine Kinase Inhibitors in Mouse Models. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2012. [cited 2019 Aug 24]. Available from: https://dc.uthsc.edu/dissertations/74.

Council of Science Editors:

Elmeliegy M. CNS Penetration of Tyrosine Kinase Inhibitors in Mouse Models. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2012. Available from: https://dc.uthsc.edu/dissertations/74

23. Beard, Jordan A. Investigating the Regulation and Function of the NR4A Nuclear Receptors in Cancer.

Degree: PhD, Biomedical Sciences, 2016, University of Tennessee Health Science Center

  The nuclear receptor (NR) superfamily represents a structurally-conserved group of ligand-regulated transcription factors. These proteins have critical roles in various physiological and pathological processes,… (more)

Subjects/Keywords: nuclear receptor; NR4A2; miRNA; miR-34; p53; rhabdomyosarcoma; Medical Cell Biology; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences

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APA (6th Edition):

Beard, J. A. (2016). Investigating the Regulation and Function of the NR4A Nuclear Receptors in Cancer. (Doctoral Dissertation). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/378

Chicago Manual of Style (16th Edition):

Beard, Jordan A. “Investigating the Regulation and Function of the NR4A Nuclear Receptors in Cancer.” 2016. Doctoral Dissertation, University of Tennessee Health Science Center. Accessed August 24, 2019. https://dc.uthsc.edu/dissertations/378.

MLA Handbook (7th Edition):

Beard, Jordan A. “Investigating the Regulation and Function of the NR4A Nuclear Receptors in Cancer.” 2016. Web. 24 Aug 2019.

Vancouver:

Beard JA. Investigating the Regulation and Function of the NR4A Nuclear Receptors in Cancer. [Internet] [Doctoral dissertation]. University of Tennessee Health Science Center; 2016. [cited 2019 Aug 24]. Available from: https://dc.uthsc.edu/dissertations/378.

Council of Science Editors:

Beard JA. Investigating the Regulation and Function of the NR4A Nuclear Receptors in Cancer. [Doctoral Dissertation]. University of Tennessee Health Science Center; 2016. Available from: https://dc.uthsc.edu/dissertations/378


University of Oxford

24. Vachtsevanos, Athanasios. Probing the molecular basis of melanopsin induced light sensitivity.

Degree: PhD, 2012, University of Oxford

 It has been demonstrated that retinal photoreception among mammals extends beyond rods and cones to include a small number of intrinsically photosensitive retinal ganglion cells… (more)

Subjects/Keywords: 617.7; Medical Sciences; Clinical laboratory sciences; Clinical genetics; Immunodiagnostics; Biology (medical sciences); Genetics (medical sciences); Ophthamology; ophthalmology; melanospin; siRNA; pupillometry

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APA (6th Edition):

Vachtsevanos, A. (2012). Probing the molecular basis of melanopsin induced light sensitivity. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770

Chicago Manual of Style (16th Edition):

Vachtsevanos, Athanasios. “Probing the molecular basis of melanopsin induced light sensitivity.” 2012. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770.

MLA Handbook (7th Edition):

Vachtsevanos, Athanasios. “Probing the molecular basis of melanopsin induced light sensitivity.” 2012. Web. 24 Aug 2019.

Vancouver:

Vachtsevanos A. Probing the molecular basis of melanopsin induced light sensitivity. [Internet] [Doctoral dissertation]. University of Oxford; 2012. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770.

Council of Science Editors:

Vachtsevanos A. Probing the molecular basis of melanopsin induced light sensitivity. [Doctoral Dissertation]. University of Oxford; 2012. Available from: http://ora.ox.ac.uk/objects/uuid:631deeeb-90c0-4e90-b24e-f03e1b318d8b ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.627770


McMaster University

25. Wang, David Yu Chang. Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli.

Degree: MSc, 2011, McMaster University

In atherosclerotic plaques, macrophages ingest modified LDL and turn to foam cells. Others have shown that SR-BI expression levels inversely correlated with cellular cholesterol… (more)

Subjects/Keywords: Atherosclerosis receptor cholesterol inflammation macrophage lipoprotein; Biochemistry; Biology; Cell Biology; Immunity; Medical Biochemistry; Medical Cell Biology; Medical Molecular Biology; Medical Sciences; Molecular Biology; Biochemistry

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APA (6th Edition):

Wang, D. Y. C. (2011). Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11219

Chicago Manual of Style (16th Edition):

Wang, David Yu Chang. “Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli.” 2011. Masters Thesis, McMaster University. Accessed August 24, 2019. http://hdl.handle.net/11375/11219.

MLA Handbook (7th Edition):

Wang, David Yu Chang. “Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli.” 2011. Web. 24 Aug 2019.

Vancouver:

Wang DYC. Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2019 Aug 24]. Available from: http://hdl.handle.net/11375/11219.

Council of Science Editors:

Wang DYC. Regulation of macrophage SR-BI by lipoproteins and inflammatory stimuli. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/11219


University of Arkansas

26. Alismail, Hanan Abdulaziz. Enhanced Pancreatic beta-cells Proliferation and Functionality.

Degree: MS, 2013, University of Arkansas

  Biologically functional beta-cells proliferate at an extremely low rate with limited turnover capacity. This cellular property hinders cell-based therapy for clinical applications. Many attempts… (more)

Subjects/Keywords: Biological sciences; Cells; niche; pancreas; proliferation; Medical Cell Biology; Medical Molecular Biology

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APA (6th Edition):

Alismail, H. A. (2013). Enhanced Pancreatic beta-cells Proliferation and Functionality. (Masters Thesis). University of Arkansas. Retrieved from https://scholarworks.uark.edu/etd/955

Chicago Manual of Style (16th Edition):

Alismail, Hanan Abdulaziz. “Enhanced Pancreatic beta-cells Proliferation and Functionality.” 2013. Masters Thesis, University of Arkansas. Accessed August 24, 2019. https://scholarworks.uark.edu/etd/955.

MLA Handbook (7th Edition):

Alismail, Hanan Abdulaziz. “Enhanced Pancreatic beta-cells Proliferation and Functionality.” 2013. Web. 24 Aug 2019.

Vancouver:

Alismail HA. Enhanced Pancreatic beta-cells Proliferation and Functionality. [Internet] [Masters thesis]. University of Arkansas; 2013. [cited 2019 Aug 24]. Available from: https://scholarworks.uark.edu/etd/955.

Council of Science Editors:

Alismail HA. Enhanced Pancreatic beta-cells Proliferation and Functionality. [Masters Thesis]. University of Arkansas; 2013. Available from: https://scholarworks.uark.edu/etd/955


University of South Florida

27. Nelson, Nadine D. Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer.

Degree: 2015, University of South Florida

 Pancreatic cancer is one of the deadliest cancers with a five-year survival rate of 6%. Pancreatic cancer is resistant to conventional chemotherapy and is usually… (more)

Subjects/Keywords: Transcription Factor; CK2; PP1; Ubiquitination; Apigenin; Cell Biology; Medical Molecular Biology; Medical Sciences

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APA (6th Edition):

Nelson, N. D. (2015). Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/5810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nelson, Nadine D. “Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer.” 2015. Thesis, University of South Florida. Accessed August 24, 2019. https://scholarcommons.usf.edu/etd/5810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nelson, Nadine D. “Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer.” 2015. Web. 24 Aug 2019.

Vancouver:

Nelson ND. Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer. [Internet] [Thesis]. University of South Florida; 2015. [cited 2019 Aug 24]. Available from: https://scholarcommons.usf.edu/etd/5810.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nelson ND. Ikaros Deficiency Leads To An Imbalance in Effector and Regulatory T Cell Homeostasis in Murine Pancreatic Cancer. [Thesis]. University of South Florida; 2015. Available from: https://scholarcommons.usf.edu/etd/5810

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Lee, Ein. Transcriptional Regulation of NLRC4 Inflammasome by IRF8.

Degree: MS, Biomedical Sciences, 2019, University of Tennessee Health Science Center

  The NLRC4 inflammasome is a crucial part of the innate immune response against bacterial infections. We found that NLRC4 inflammasome activation in bone marrow-derived… (more)

Subjects/Keywords: Inflammasome; Interferon regulatory factor; IRF8; NAIP; NLRC4; Salmonella; Medical Immunology; Medical Microbiology; Medical Molecular Biology; Medical Sciences; Medicine and Health Sciences

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, E. (2019). Transcriptional Regulation of NLRC4 Inflammasome by IRF8. (Thesis). University of Tennessee Health Science Center. Retrieved from https://dc.uthsc.edu/dissertations/465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Ein. “Transcriptional Regulation of NLRC4 Inflammasome by IRF8.” 2019. Thesis, University of Tennessee Health Science Center. Accessed August 24, 2019. https://dc.uthsc.edu/dissertations/465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Ein. “Transcriptional Regulation of NLRC4 Inflammasome by IRF8.” 2019. Web. 24 Aug 2019.

Vancouver:

Lee E. Transcriptional Regulation of NLRC4 Inflammasome by IRF8. [Internet] [Thesis]. University of Tennessee Health Science Center; 2019. [cited 2019 Aug 24]. Available from: https://dc.uthsc.edu/dissertations/465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee E. Transcriptional Regulation of NLRC4 Inflammasome by IRF8. [Thesis]. University of Tennessee Health Science Center; 2019. Available from: https://dc.uthsc.edu/dissertations/465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oxford

29. Chen, Ye. Induced regulatory T cells in transplantation tolerance.

Degree: PhD, 2010, University of Oxford

 Induced regulatory T cells (iTreg) play an important role in the induction of tolerance to self and non-self antigens. Harnessing their suppressive potential has therapeutic… (more)

Subjects/Keywords: 616.079; Life Sciences; Biology; Cell Biology (see also Plant sciences); Medical Sciences; Immunology; Transplantation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, Y. (2010). Induced regulatory T cells in transplantation tolerance. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:cffc275b-d32c-495e-a1da-55421a57e7e7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534150

Chicago Manual of Style (16th Edition):

Chen, Ye. “Induced regulatory T cells in transplantation tolerance.” 2010. Doctoral Dissertation, University of Oxford. Accessed August 24, 2019. http://ora.ox.ac.uk/objects/uuid:cffc275b-d32c-495e-a1da-55421a57e7e7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534150.

MLA Handbook (7th Edition):

Chen, Ye. “Induced regulatory T cells in transplantation tolerance.” 2010. Web. 24 Aug 2019.

Vancouver:

Chen Y. Induced regulatory T cells in transplantation tolerance. [Internet] [Doctoral dissertation]. University of Oxford; 2010. [cited 2019 Aug 24]. Available from: http://ora.ox.ac.uk/objects/uuid:cffc275b-d32c-495e-a1da-55421a57e7e7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534150.

Council of Science Editors:

Chen Y. Induced regulatory T cells in transplantation tolerance. [Doctoral Dissertation]. University of Oxford; 2010. Available from: http://ora.ox.ac.uk/objects/uuid:cffc275b-d32c-495e-a1da-55421a57e7e7 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534150


Rhode Island College

30. Lamkin, Jamie. Investigating the Role of p53 in the Germ Cell Apoptotic Pathway.

Degree: MA, 2011, Rhode Island College

Investigation of the role of p53 in germ cell apoptosis pathways.

Subjects/Keywords: Biology, Science; Biology; Medical Sciences; p53; apoptotic; germ

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lamkin, J. (2011). Investigating the Role of p53 in the Germ Cell Apoptotic Pathway. (Masters Thesis). Rhode Island College. Retrieved from https://digitalcommons.ric.edu/etd/47

Chicago Manual of Style (16th Edition):

Lamkin, Jamie. “Investigating the Role of p53 in the Germ Cell Apoptotic Pathway.” 2011. Masters Thesis, Rhode Island College. Accessed August 24, 2019. https://digitalcommons.ric.edu/etd/47.

MLA Handbook (7th Edition):

Lamkin, Jamie. “Investigating the Role of p53 in the Germ Cell Apoptotic Pathway.” 2011. Web. 24 Aug 2019.

Vancouver:

Lamkin J. Investigating the Role of p53 in the Germ Cell Apoptotic Pathway. [Internet] [Masters thesis]. Rhode Island College; 2011. [cited 2019 Aug 24]. Available from: https://digitalcommons.ric.edu/etd/47.

Council of Science Editors:

Lamkin J. Investigating the Role of p53 in the Germ Cell Apoptotic Pathway. [Masters Thesis]. Rhode Island College; 2011. Available from: https://digitalcommons.ric.edu/etd/47

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