Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for subject:(Biochemical Phenomena Metabolism AND Nutrition). Showing records 1 – 30 of 531 total matches.

[1] [2] [3] [4] [5] … [18]

Search Limiters

Last 2 Years | English Only

Degrees

Levels

Languages

Country

▼ Search Limiters


McMaster University

1. Hayes, Emily K. The effect of lifelong maternal obesity on pregnancy outcomes and placental development.

Degree: MSc, 2012, McMaster University

Maternal obesity is associated with an increased risk of pregnancy complications, including preeclampsia, miscarriage, birth of small for gestational age (SGA) babies, and stillbirth.… (more)

Subjects/Keywords: Obesity; Placenta; Vasculature; Oxidative Stress; Biochemical Phenomena, Metabolism, and Nutrition; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hayes, E. K. (2012). The effect of lifelong maternal obesity on pregnancy outcomes and placental development. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/11970

Chicago Manual of Style (16th Edition):

Hayes, Emily K. “The effect of lifelong maternal obesity on pregnancy outcomes and placental development.” 2012. Masters Thesis, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/11970.

MLA Handbook (7th Edition):

Hayes, Emily K. “The effect of lifelong maternal obesity on pregnancy outcomes and placental development.” 2012. Web. 22 Jul 2019.

Vancouver:

Hayes EK. The effect of lifelong maternal obesity on pregnancy outcomes and placental development. [Internet] [Masters thesis]. McMaster University; 2012. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/11970.

Council of Science Editors:

Hayes EK. The effect of lifelong maternal obesity on pregnancy outcomes and placental development. [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/11970


McMaster University

2. Wang, Nuo. THE ROLE OF SREBP-1 IN MEDIATING ANGIOTENSIN II- INDUCED KIDNEY FIBROSIS.

Degree: MSc, 2012, McMaster University

End stage renal disease (ESRD) has been a world-wide cause of renal disease-related fatality. Most patients with ESRD progress through chronic kidney disease (CKD).… (more)

Subjects/Keywords: renal firbosis SREBP angiotensin; Biochemical Phenomena, Metabolism, and Nutrition; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Wang, N. (2012). THE ROLE OF SREBP-1 IN MEDIATING ANGIOTENSIN II- INDUCED KIDNEY FIBROSIS. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/12621

Chicago Manual of Style (16th Edition):

Wang, Nuo. “THE ROLE OF SREBP-1 IN MEDIATING ANGIOTENSIN II- INDUCED KIDNEY FIBROSIS.” 2012. Masters Thesis, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/12621.

MLA Handbook (7th Edition):

Wang, Nuo. “THE ROLE OF SREBP-1 IN MEDIATING ANGIOTENSIN II- INDUCED KIDNEY FIBROSIS.” 2012. Web. 22 Jul 2019.

Vancouver:

Wang N. THE ROLE OF SREBP-1 IN MEDIATING ANGIOTENSIN II- INDUCED KIDNEY FIBROSIS. [Internet] [Masters thesis]. McMaster University; 2012. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/12621.

Council of Science Editors:

Wang N. THE ROLE OF SREBP-1 IN MEDIATING ANGIOTENSIN II- INDUCED KIDNEY FIBROSIS. [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/12621


McMaster University

3. Barra, Nicole G. Investigating the Role of Interleukin-15 in Modulating Adipose Tissue.

Degree: PhD, 2013, McMaster University

Obesity is a major global health concern and is associated with the development of numerous non-communicable diseases. A thorough understanding of the onset of… (more)

Subjects/Keywords: obesity; interleukin-15; adipose tissue; Biochemical Phenomena, Metabolism, and Nutrition; Biological Phenomena, Cell Phenomena, and Immunity; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barra, N. G. (2013). Investigating the Role of Interleukin-15 in Modulating Adipose Tissue. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/15319

Chicago Manual of Style (16th Edition):

Barra, Nicole G. “Investigating the Role of Interleukin-15 in Modulating Adipose Tissue.” 2013. Doctoral Dissertation, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/15319.

MLA Handbook (7th Edition):

Barra, Nicole G. “Investigating the Role of Interleukin-15 in Modulating Adipose Tissue.” 2013. Web. 22 Jul 2019.

Vancouver:

Barra NG. Investigating the Role of Interleukin-15 in Modulating Adipose Tissue. [Internet] [Doctoral dissertation]. McMaster University; 2013. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/15319.

Council of Science Editors:

Barra NG. Investigating the Role of Interleukin-15 in Modulating Adipose Tissue. [Doctoral Dissertation]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/15319


McMaster University

4. Mocellin, Joseph Nicholas. GLUCOSE SUPPLEMENTATION AND MEASURES OF OXIDATIVE STRESS IN PATIENTS WITH GLYCOGEN PHOSPHORYLASE DEFICIENCY (MCARDLE’S DISEASE).

Degree: MSc, 2012, McMaster University

Objective: We evaluated the potential effect of oral glucose supplementation on: (1) exercise performance and tolerance, (2) the concentrations of plasma uric acid and… (more)

Subjects/Keywords: McArdles disease; oxidative stress; exercise; glucose supplementation; Biochemical Phenomena, Metabolism, and Nutrition; Medical Nutrition; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mocellin, J. N. (2012). GLUCOSE SUPPLEMENTATION AND MEASURES OF OXIDATIVE STRESS IN PATIENTS WITH GLYCOGEN PHOSPHORYLASE DEFICIENCY (MCARDLE’S DISEASE). (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/12653

Chicago Manual of Style (16th Edition):

Mocellin, Joseph Nicholas. “GLUCOSE SUPPLEMENTATION AND MEASURES OF OXIDATIVE STRESS IN PATIENTS WITH GLYCOGEN PHOSPHORYLASE DEFICIENCY (MCARDLE’S DISEASE).” 2012. Masters Thesis, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/12653.

MLA Handbook (7th Edition):

Mocellin, Joseph Nicholas. “GLUCOSE SUPPLEMENTATION AND MEASURES OF OXIDATIVE STRESS IN PATIENTS WITH GLYCOGEN PHOSPHORYLASE DEFICIENCY (MCARDLE’S DISEASE).” 2012. Web. 22 Jul 2019.

Vancouver:

Mocellin JN. GLUCOSE SUPPLEMENTATION AND MEASURES OF OXIDATIVE STRESS IN PATIENTS WITH GLYCOGEN PHOSPHORYLASE DEFICIENCY (MCARDLE’S DISEASE). [Internet] [Masters thesis]. McMaster University; 2012. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/12653.

Council of Science Editors:

Mocellin JN. GLUCOSE SUPPLEMENTATION AND MEASURES OF OXIDATIVE STRESS IN PATIENTS WITH GLYCOGEN PHOSPHORYLASE DEFICIENCY (MCARDLE’S DISEASE). [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/12653


McMaster University

5. Chin, Jennifer. Method Development, Feasibility and Clinical Pilot Study of Air Displacement Plethysmography for Longitudinal Body Composition Measurements of Preterm Infants in Hospital.

Degree: MSMS, 2013, McMaster University

Background: Inadequate nutrition during the postnatal period may be associated with adverse outcomes in later life. Tailoring nutrition to promote optimal growth requires monitoring… (more)

Subjects/Keywords: Preterm Infant; Body Composition; Air Displacement Plethysmography; Longitudinal; Nutrition; Growth; Biochemical Phenomena, Metabolism, and Nutrition; Pediatrics; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chin, J. (2013). Method Development, Feasibility and Clinical Pilot Study of Air Displacement Plethysmography for Longitudinal Body Composition Measurements of Preterm Infants in Hospital. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/13452

Chicago Manual of Style (16th Edition):

Chin, Jennifer. “Method Development, Feasibility and Clinical Pilot Study of Air Displacement Plethysmography for Longitudinal Body Composition Measurements of Preterm Infants in Hospital.” 2013. Masters Thesis, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/13452.

MLA Handbook (7th Edition):

Chin, Jennifer. “Method Development, Feasibility and Clinical Pilot Study of Air Displacement Plethysmography for Longitudinal Body Composition Measurements of Preterm Infants in Hospital.” 2013. Web. 22 Jul 2019.

Vancouver:

Chin J. Method Development, Feasibility and Clinical Pilot Study of Air Displacement Plethysmography for Longitudinal Body Composition Measurements of Preterm Infants in Hospital. [Internet] [Masters thesis]. McMaster University; 2013. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/13452.

Council of Science Editors:

Chin J. Method Development, Feasibility and Clinical Pilot Study of Air Displacement Plethysmography for Longitudinal Body Composition Measurements of Preterm Infants in Hospital. [Masters Thesis]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/13452


McMaster University

6. Sikkema, Sarah R. High-Intensity Interval Training Improves Insulin Sensitivity Independent of Adipose Tissue Inflammation.

Degree: MSc, 2011, McMaster University

Obesity is associated with a state of chronic, low-grade inflammation that contributes to the development of insulin resistance. Exercise is known to improve insulin… (more)

Subjects/Keywords: obesity; adipose tissue; insulin resistance; exercise; inflammation; macrophage; Biochemical Phenomena, Metabolism, and Nutrition; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sikkema, S. R. (2011). High-Intensity Interval Training Improves Insulin Sensitivity Independent of Adipose Tissue Inflammation. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/10395

Chicago Manual of Style (16th Edition):

Sikkema, Sarah R. “High-Intensity Interval Training Improves Insulin Sensitivity Independent of Adipose Tissue Inflammation.” 2011. Masters Thesis, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/10395.

MLA Handbook (7th Edition):

Sikkema, Sarah R. “High-Intensity Interval Training Improves Insulin Sensitivity Independent of Adipose Tissue Inflammation.” 2011. Web. 22 Jul 2019.

Vancouver:

Sikkema SR. High-Intensity Interval Training Improves Insulin Sensitivity Independent of Adipose Tissue Inflammation. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/10395.

Council of Science Editors:

Sikkema SR. High-Intensity Interval Training Improves Insulin Sensitivity Independent of Adipose Tissue Inflammation. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/10395


McMaster University

7. Ramsingh, Laura. EXAMINING DETERMINANTS OF INSULIN RESISTANCE IN ADULTS BORN AT NORMAL AND EXTREMELY LOW BIRTH WEIGHT.

Degree: MSc, 2012, McMaster University

The association between low birth weight at term birth and an increased risk for insulin resistance (IR) in adulthood is well established. Less is… (more)

Subjects/Keywords: Preterm; fetal origins; insulin resistance; body composition; Biochemical Phenomena, Metabolism, and Nutrition; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ramsingh, L. (2012). EXAMINING DETERMINANTS OF INSULIN RESISTANCE IN ADULTS BORN AT NORMAL AND EXTREMELY LOW BIRTH WEIGHT. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/12355

Chicago Manual of Style (16th Edition):

Ramsingh, Laura. “EXAMINING DETERMINANTS OF INSULIN RESISTANCE IN ADULTS BORN AT NORMAL AND EXTREMELY LOW BIRTH WEIGHT.” 2012. Masters Thesis, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/12355.

MLA Handbook (7th Edition):

Ramsingh, Laura. “EXAMINING DETERMINANTS OF INSULIN RESISTANCE IN ADULTS BORN AT NORMAL AND EXTREMELY LOW BIRTH WEIGHT.” 2012. Web. 22 Jul 2019.

Vancouver:

Ramsingh L. EXAMINING DETERMINANTS OF INSULIN RESISTANCE IN ADULTS BORN AT NORMAL AND EXTREMELY LOW BIRTH WEIGHT. [Internet] [Masters thesis]. McMaster University; 2012. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/12355.

Council of Science Editors:

Ramsingh L. EXAMINING DETERMINANTS OF INSULIN RESISTANCE IN ADULTS BORN AT NORMAL AND EXTREMELY LOW BIRTH WEIGHT. [Masters Thesis]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/12355


McMaster University

8. Marcinko, Katarina. GENETIC DISRUPTION OF ACETYL COA CARBOXYLASE PHOSPHORYLATION BY AMP-ACTIVATED PROTEIN KINASE INCREASES LIVER LIPID ACCUMULATION AND INSULIN RESISTANCE.

Degree: MSc, 2011, McMaster University

In obesity, nonalcoholic fatty liver disease (NAFLD) has been associated with the development of hepatic insulin resistance. Acetyl coA carboxylase (ACC), which exists as… (more)

Subjects/Keywords: obesity; diabetes; fatty liver; NAFLD; insulin resistance; Biochemical Phenomena, Metabolism, and Nutrition; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Marcinko, K. (2011). GENETIC DISRUPTION OF ACETYL COA CARBOXYLASE PHOSPHORYLATION BY AMP-ACTIVATED PROTEIN KINASE INCREASES LIVER LIPID ACCUMULATION AND INSULIN RESISTANCE. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/10585

Chicago Manual of Style (16th Edition):

Marcinko, Katarina. “GENETIC DISRUPTION OF ACETYL COA CARBOXYLASE PHOSPHORYLATION BY AMP-ACTIVATED PROTEIN KINASE INCREASES LIVER LIPID ACCUMULATION AND INSULIN RESISTANCE.” 2011. Masters Thesis, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/10585.

MLA Handbook (7th Edition):

Marcinko, Katarina. “GENETIC DISRUPTION OF ACETYL COA CARBOXYLASE PHOSPHORYLATION BY AMP-ACTIVATED PROTEIN KINASE INCREASES LIVER LIPID ACCUMULATION AND INSULIN RESISTANCE.” 2011. Web. 22 Jul 2019.

Vancouver:

Marcinko K. GENETIC DISRUPTION OF ACETYL COA CARBOXYLASE PHOSPHORYLATION BY AMP-ACTIVATED PROTEIN KINASE INCREASES LIVER LIPID ACCUMULATION AND INSULIN RESISTANCE. [Internet] [Masters thesis]. McMaster University; 2011. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/10585.

Council of Science Editors:

Marcinko K. GENETIC DISRUPTION OF ACETYL COA CARBOXYLASE PHOSPHORYLATION BY AMP-ACTIVATED PROTEIN KINASE INCREASES LIVER LIPID ACCUMULATION AND INSULIN RESISTANCE. [Masters Thesis]. McMaster University; 2011. Available from: http://hdl.handle.net/11375/10585


McMaster University

9. Antolic, AnaMaria. MITOCHONDRIAL CONTRIBUTIONS TO BONE HEALTH, AN INVESTIGATION USING TWO DIFFERENT ANIMAL MODELS OF MITOCHONDRIAL CAPACITY.

Degree: PhD, 2013, McMaster University

Mitochondrial dysfunction has been linked to a number of age-associated disorders and recent evidence has shown that mitochondrial function is necessary for osteoblast differentiation… (more)

Subjects/Keywords: bone; mitochondria; exercise; aging; translational medicine; Biochemical Phenomena, Metabolism, and Nutrition; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Antolic, A. (2013). MITOCHONDRIAL CONTRIBUTIONS TO BONE HEALTH, AN INVESTIGATION USING TWO DIFFERENT ANIMAL MODELS OF MITOCHONDRIAL CAPACITY. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/13478

Chicago Manual of Style (16th Edition):

Antolic, AnaMaria. “MITOCHONDRIAL CONTRIBUTIONS TO BONE HEALTH, AN INVESTIGATION USING TWO DIFFERENT ANIMAL MODELS OF MITOCHONDRIAL CAPACITY.” 2013. Doctoral Dissertation, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/13478.

MLA Handbook (7th Edition):

Antolic, AnaMaria. “MITOCHONDRIAL CONTRIBUTIONS TO BONE HEALTH, AN INVESTIGATION USING TWO DIFFERENT ANIMAL MODELS OF MITOCHONDRIAL CAPACITY.” 2013. Web. 22 Jul 2019.

Vancouver:

Antolic A. MITOCHONDRIAL CONTRIBUTIONS TO BONE HEALTH, AN INVESTIGATION USING TWO DIFFERENT ANIMAL MODELS OF MITOCHONDRIAL CAPACITY. [Internet] [Doctoral dissertation]. McMaster University; 2013. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/13478.

Council of Science Editors:

Antolic A. MITOCHONDRIAL CONTRIBUTIONS TO BONE HEALTH, AN INVESTIGATION USING TWO DIFFERENT ANIMAL MODELS OF MITOCHONDRIAL CAPACITY. [Doctoral Dissertation]. McMaster University; 2013. Available from: http://hdl.handle.net/11375/13478


Texas Medical Center

10. Shiang, Christine. NOVEL THERAPEUTIC TARGETS IDENTIFIED BY HIGH-THROUGHPUT TECHNOLOGIES FOR TRIPLE-NEGATIVE BREAST CANCER.

Degree: PhD, 2013, Texas Medical Center

  Triple-negative breast cancers (TNBC) are characterized by the lack of or reduced expression of the estrogen and progesterone receptors, and normal expression of the… (more)

Subjects/Keywords: Biochemical Phenomena, Metabolism, and Nutrition; Medicine and Health Sciences; Oncology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shiang, C. (2013). NOVEL THERAPEUTIC TARGETS IDENTIFIED BY HIGH-THROUGHPUT TECHNOLOGIES FOR TRIPLE-NEGATIVE BREAST CANCER. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/346

Chicago Manual of Style (16th Edition):

Shiang, Christine. “NOVEL THERAPEUTIC TARGETS IDENTIFIED BY HIGH-THROUGHPUT TECHNOLOGIES FOR TRIPLE-NEGATIVE BREAST CANCER.” 2013. Doctoral Dissertation, Texas Medical Center. Accessed July 22, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/346.

MLA Handbook (7th Edition):

Shiang, Christine. “NOVEL THERAPEUTIC TARGETS IDENTIFIED BY HIGH-THROUGHPUT TECHNOLOGIES FOR TRIPLE-NEGATIVE BREAST CANCER.” 2013. Web. 22 Jul 2019.

Vancouver:

Shiang C. NOVEL THERAPEUTIC TARGETS IDENTIFIED BY HIGH-THROUGHPUT TECHNOLOGIES FOR TRIPLE-NEGATIVE BREAST CANCER. [Internet] [Doctoral dissertation]. Texas Medical Center; 2013. [cited 2019 Jul 22]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/346.

Council of Science Editors:

Shiang C. NOVEL THERAPEUTIC TARGETS IDENTIFIED BY HIGH-THROUGHPUT TECHNOLOGIES FOR TRIPLE-NEGATIVE BREAST CANCER. [Doctoral Dissertation]. Texas Medical Center; 2013. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/346


University of Kentucky

11. Carter, Lindsay G. Offspring and Maternal Health Benefits of Exercise during Pregnancy.

Degree: 2013, University of Kentucky

 Maternal lifestyle and nutrient intake during pregnancy can have long-lasting effects on the health of offspring as well as the mother. This dissertation focuses on… (more)

Subjects/Keywords: Pregnancy; Exercise; Offspring; Glucose; Insulin; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Carter, L. G. (2013). Offspring and Maternal Health Benefits of Exercise during Pregnancy. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/nutrisci_etds/6

Chicago Manual of Style (16th Edition):

Carter, Lindsay G. “Offspring and Maternal Health Benefits of Exercise during Pregnancy.” 2013. Doctoral Dissertation, University of Kentucky. Accessed July 22, 2019. https://uknowledge.uky.edu/nutrisci_etds/6.

MLA Handbook (7th Edition):

Carter, Lindsay G. “Offspring and Maternal Health Benefits of Exercise during Pregnancy.” 2013. Web. 22 Jul 2019.

Vancouver:

Carter LG. Offspring and Maternal Health Benefits of Exercise during Pregnancy. [Internet] [Doctoral dissertation]. University of Kentucky; 2013. [cited 2019 Jul 22]. Available from: https://uknowledge.uky.edu/nutrisci_etds/6.

Council of Science Editors:

Carter LG. Offspring and Maternal Health Benefits of Exercise during Pregnancy. [Doctoral Dissertation]. University of Kentucky; 2013. Available from: https://uknowledge.uky.edu/nutrisci_etds/6


University of Nevada – Las Vegas

12. Kachinski, Julie Jo. Use of an animal model to explore prenatal predictors of insulin and glucose metabolism in Southwestern Alaskan Yupiit.

Degree: MAin Anthropology, Anthropology, 2010, University of Nevada – Las Vegas

 Compared to other North American indigenous populations, Southwest Alaskan Yupiit exhibit very low rates of type 2 diabetes despite the occurrence of common risk factors.… (more)

Subjects/Keywords: Biochemical Phenomena, Metabolism, and Nutrition; Biological and Physical Anthropology; Medical Sciences; Nutrition; Obstetrics and Gynecology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kachinski, J. J. (2010). Use of an animal model to explore prenatal predictors of insulin and glucose metabolism in Southwestern Alaskan Yupiit. (Masters Thesis). University of Nevada – Las Vegas. Retrieved from https://digitalscholarship.unlv.edu/thesesdissertations/224

Chicago Manual of Style (16th Edition):

Kachinski, Julie Jo. “Use of an animal model to explore prenatal predictors of insulin and glucose metabolism in Southwestern Alaskan Yupiit.” 2010. Masters Thesis, University of Nevada – Las Vegas. Accessed July 22, 2019. https://digitalscholarship.unlv.edu/thesesdissertations/224.

MLA Handbook (7th Edition):

Kachinski, Julie Jo. “Use of an animal model to explore prenatal predictors of insulin and glucose metabolism in Southwestern Alaskan Yupiit.” 2010. Web. 22 Jul 2019.

Vancouver:

Kachinski JJ. Use of an animal model to explore prenatal predictors of insulin and glucose metabolism in Southwestern Alaskan Yupiit. [Internet] [Masters thesis]. University of Nevada – Las Vegas; 2010. [cited 2019 Jul 22]. Available from: https://digitalscholarship.unlv.edu/thesesdissertations/224.

Council of Science Editors:

Kachinski JJ. Use of an animal model to explore prenatal predictors of insulin and glucose metabolism in Southwestern Alaskan Yupiit. [Masters Thesis]. University of Nevada – Las Vegas; 2010. Available from: https://digitalscholarship.unlv.edu/thesesdissertations/224


McMaster University

13. Basseri, Sana. CONTRIBUTION OF THE UNFOLDED PROTEIN RESPONSE (UPR) TO ADIPOGENESIS AND WHOLE BODY ENERGY HOMEOSTASIS.

Degree: PhD, 2012, McMaster University

  The endoplasmic reticulum (ER) is a specialized organelle that facilitates correct protein folding and maturation. Disruptions in ER homeostasis lead to ER stress and… (more)

Subjects/Keywords: Endoplasmic Reticulum Stress; TDAG51; Adipocytes; Obesity; White Adipose Tissue; Biochemical Phenomena, Metabolism, and Nutrition; Endocrinology, Diabetes, and Metabolism; Medical Cell Biology; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Basseri, S. (2012). CONTRIBUTION OF THE UNFOLDED PROTEIN RESPONSE (UPR) TO ADIPOGENESIS AND WHOLE BODY ENERGY HOMEOSTASIS. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/11961

Chicago Manual of Style (16th Edition):

Basseri, Sana. “CONTRIBUTION OF THE UNFOLDED PROTEIN RESPONSE (UPR) TO ADIPOGENESIS AND WHOLE BODY ENERGY HOMEOSTASIS.” 2012. Doctoral Dissertation, McMaster University. Accessed July 22, 2019. http://hdl.handle.net/11375/11961.

MLA Handbook (7th Edition):

Basseri, Sana. “CONTRIBUTION OF THE UNFOLDED PROTEIN RESPONSE (UPR) TO ADIPOGENESIS AND WHOLE BODY ENERGY HOMEOSTASIS.” 2012. Web. 22 Jul 2019.

Vancouver:

Basseri S. CONTRIBUTION OF THE UNFOLDED PROTEIN RESPONSE (UPR) TO ADIPOGENESIS AND WHOLE BODY ENERGY HOMEOSTASIS. [Internet] [Doctoral dissertation]. McMaster University; 2012. [cited 2019 Jul 22]. Available from: http://hdl.handle.net/11375/11961.

Council of Science Editors:

Basseri S. CONTRIBUTION OF THE UNFOLDED PROTEIN RESPONSE (UPR) TO ADIPOGENESIS AND WHOLE BODY ENERGY HOMEOSTASIS. [Doctoral Dissertation]. McMaster University; 2012. Available from: http://hdl.handle.net/11375/11961


University of Kentucky

14. Eske, Katryn Elizabeth. COPLANAR PCB-INDUCED INFLAMMATION AND DIETARY INTERVENTIONS.

Degree: 2013, University of Kentucky

 Diseases, such as cardiovascular disease (CVD), are linked to chronic low levels of inflammation. This inflamed state is the product of risk factors including exposure… (more)

Subjects/Keywords: Polychlorinated biphenyls (PCBs); endothelial cells; inflammation; docosahexaenoic acid (DHA); caveolae; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Eske, K. E. (2013). COPLANAR PCB-INDUCED INFLAMMATION AND DIETARY INTERVENTIONS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/nutrisci_etds/8

Chicago Manual of Style (16th Edition):

Eske, Katryn Elizabeth. “COPLANAR PCB-INDUCED INFLAMMATION AND DIETARY INTERVENTIONS.” 2013. Doctoral Dissertation, University of Kentucky. Accessed July 22, 2019. https://uknowledge.uky.edu/nutrisci_etds/8.

MLA Handbook (7th Edition):

Eske, Katryn Elizabeth. “COPLANAR PCB-INDUCED INFLAMMATION AND DIETARY INTERVENTIONS.” 2013. Web. 22 Jul 2019.

Vancouver:

Eske KE. COPLANAR PCB-INDUCED INFLAMMATION AND DIETARY INTERVENTIONS. [Internet] [Doctoral dissertation]. University of Kentucky; 2013. [cited 2019 Jul 22]. Available from: https://uknowledge.uky.edu/nutrisci_etds/8.

Council of Science Editors:

Eske KE. COPLANAR PCB-INDUCED INFLAMMATION AND DIETARY INTERVENTIONS. [Doctoral Dissertation]. University of Kentucky; 2013. Available from: https://uknowledge.uky.edu/nutrisci_etds/8


University of Western Ontario

15. Johansen, Christopher T. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.

Degree: 2011, University of Western Ontario

 Cardiovascular disease (CVD) is the leading cause of morbidity and mortality in Canada. Among non-traditional risk factors, plasma triglyceride (TG) concentration is re-emerging as a… (more)

Subjects/Keywords: hypertriglyceridemia; hyperlipoproteinemia; genetic variation; cardiovascular disease; genome-wide association study; resequencing; Biochemical Phenomena, Metabolism, and Nutrition; Cardiovascular Diseases; Genetic Phenomena

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Johansen, C. T. (2011). Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Johansen, Christopher T. “Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.” 2011. Thesis, University of Western Ontario. Accessed July 22, 2019. https://ir.lib.uwo.ca/etd/219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Johansen, Christopher T. “Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia.” 2011. Web. 22 Jul 2019.

Vancouver:

Johansen CT. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. [Internet] [Thesis]. University of Western Ontario; 2011. [cited 2019 Jul 22]. Available from: https://ir.lib.uwo.ca/etd/219.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Johansen CT. Elucidating the genetic determinants of the archetypal complex disease hypertriglyceridemia. [Thesis]. University of Western Ontario; 2011. Available from: https://ir.lib.uwo.ca/etd/219

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. Shearstone, Jeffrey R. Global DNA Demethylation During Erythropoiesis: A Dissertation.

Degree: Cancer Biology, Molecular, Cell and Cancer Biology, 2011, U of Massachusetts : Med

  In the mammalian genome, 5‟-CpG-3‟ dinucleotides are frequently methylated, correlating with transcriptional silencing. Genome-wide waves of demethylation are thought to occur only twice during… (more)

Subjects/Keywords: Erythropoiesis; DNA Methylation; Biochemical Phenomena, Metabolism, and Nutrition; Cancer Biology; Cells; Circulatory and Respiratory Physiology; Genetic Phenomena; Genetics and Genomics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shearstone, J. R. (2011). Global DNA Demethylation During Erythropoiesis: A Dissertation. (Doctoral Dissertation). U of Massachusetts : Med. Retrieved from https://escholarship.umassmed.edu/gsbs_diss/549

Chicago Manual of Style (16th Edition):

Shearstone, Jeffrey R. “Global DNA Demethylation During Erythropoiesis: A Dissertation.” 2011. Doctoral Dissertation, U of Massachusetts : Med. Accessed July 22, 2019. https://escholarship.umassmed.edu/gsbs_diss/549.

MLA Handbook (7th Edition):

Shearstone, Jeffrey R. “Global DNA Demethylation During Erythropoiesis: A Dissertation.” 2011. Web. 22 Jul 2019.

Vancouver:

Shearstone JR. Global DNA Demethylation During Erythropoiesis: A Dissertation. [Internet] [Doctoral dissertation]. U of Massachusetts : Med; 2011. [cited 2019 Jul 22]. Available from: https://escholarship.umassmed.edu/gsbs_diss/549.

Council of Science Editors:

Shearstone JR. Global DNA Demethylation During Erythropoiesis: A Dissertation. [Doctoral Dissertation]. U of Massachusetts : Med; 2011. Available from: https://escholarship.umassmed.edu/gsbs_diss/549


Bond University

17. Koh, Andy Hsien Wei. Amphetamine-like compounds in pre-workout supplements.

Degree: 2017, Bond University

 Pre-workout supplements (PWS), like most nutritional supplements, are classified by the Therapeutic Goods Administration (TGA) in Australia as complementary medicines and are therefore subject to… (more)

Subjects/Keywords: Nutrition; Dietary supplements.; Health Sciences, Nutrition (0570); Alternative and Complementary Medicine; Amino Acids, Peptides, and Proteins; Biochemical Phenomena, Metabolism, and Nutrition; Medical Nutrition; Molecular, Genetic, and Biochemical Nutrition; Sports Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Koh, A. H. W. (2017). Amphetamine-like compounds in pre-workout supplements. (Thesis). Bond University. Retrieved from https://epublications.bond.edu.au/theses/159

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Koh, Andy Hsien Wei. “Amphetamine-like compounds in pre-workout supplements.” 2017. Thesis, Bond University. Accessed July 22, 2019. https://epublications.bond.edu.au/theses/159.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Koh, Andy Hsien Wei. “Amphetamine-like compounds in pre-workout supplements.” 2017. Web. 22 Jul 2019.

Vancouver:

Koh AHW. Amphetamine-like compounds in pre-workout supplements. [Internet] [Thesis]. Bond University; 2017. [cited 2019 Jul 22]. Available from: https://epublications.bond.edu.au/theses/159.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Koh AHW. Amphetamine-like compounds in pre-workout supplements. [Thesis]. Bond University; 2017. Available from: https://epublications.bond.edu.au/theses/159

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. Frederick, Armina-Lyn. Impact of Bodyweight on Tissue-Specific Folate Status, Genome Wide and Gene-Specific DNA Methylation in Normal Breast Tissues from Premenopausal Women.

Degree: 2018, University of Massachusetts

  Obesity has reached an epidemic level in the United States. A number of epidemiological studies have established obesity as a critical risk factor for… (more)

Subjects/Keywords: methylation; epigenetics; premenopausal breast cancer; folate; BMI; SFRP1; Biochemical Phenomena, Metabolism, and Nutrition; Human and Clinical Nutrition; Medical Molecular Biology; Medical Nutrition; Molecular, Genetic, and Biochemical Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Frederick, A. (2018). Impact of Bodyweight on Tissue-Specific Folate Status, Genome Wide and Gene-Specific DNA Methylation in Normal Breast Tissues from Premenopausal Women. (Thesis). University of Massachusetts. Retrieved from https://scholarworks.umass.edu/masters_theses_2/639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Frederick, Armina-Lyn. “Impact of Bodyweight on Tissue-Specific Folate Status, Genome Wide and Gene-Specific DNA Methylation in Normal Breast Tissues from Premenopausal Women.” 2018. Thesis, University of Massachusetts. Accessed July 22, 2019. https://scholarworks.umass.edu/masters_theses_2/639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Frederick, Armina-Lyn. “Impact of Bodyweight on Tissue-Specific Folate Status, Genome Wide and Gene-Specific DNA Methylation in Normal Breast Tissues from Premenopausal Women.” 2018. Web. 22 Jul 2019.

Vancouver:

Frederick A. Impact of Bodyweight on Tissue-Specific Folate Status, Genome Wide and Gene-Specific DNA Methylation in Normal Breast Tissues from Premenopausal Women. [Internet] [Thesis]. University of Massachusetts; 2018. [cited 2019 Jul 22]. Available from: https://scholarworks.umass.edu/masters_theses_2/639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Frederick A. Impact of Bodyweight on Tissue-Specific Folate Status, Genome Wide and Gene-Specific DNA Methylation in Normal Breast Tissues from Premenopausal Women. [Thesis]. University of Massachusetts; 2018. Available from: https://scholarworks.umass.edu/masters_theses_2/639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

19. Sen, Shiraj. Metabolic Regulation of mTOR Activation and Endoplasmic Reticulum Stress in the Heart.

Degree: PhD, 2012, Texas Medical Center

  When subjected to increased workload, the heart responds metabolically by increasing its reliance on glucose and structurally by increasing the size of myocytes. Whether… (more)

Subjects/Keywords: cardiac metabolism; mTOR; endoplasmic reticulum stress; hypertrophy; glucose; Biochemical Phenomena, Metabolism, and Nutrition; Cardiovascular Diseases; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sen, S. (2012). Metabolic Regulation of mTOR Activation and Endoplasmic Reticulum Stress in the Heart. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/230

Chicago Manual of Style (16th Edition):

Sen, Shiraj. “Metabolic Regulation of mTOR Activation and Endoplasmic Reticulum Stress in the Heart.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed July 22, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/230.

MLA Handbook (7th Edition):

Sen, Shiraj. “Metabolic Regulation of mTOR Activation and Endoplasmic Reticulum Stress in the Heart.” 2012. Web. 22 Jul 2019.

Vancouver:

Sen S. Metabolic Regulation of mTOR Activation and Endoplasmic Reticulum Stress in the Heart. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Jul 22]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/230.

Council of Science Editors:

Sen S. Metabolic Regulation of mTOR Activation and Endoplasmic Reticulum Stress in the Heart. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/230


University of Kentucky

20. Larson, Kara L. Regulation of Glucose Homeostasis by the PHLPP1 Phosphatase.

Degree: 2014, University of Kentucky

 Type 2 diabetes mellitus is a metabolic disease that affects one in ten people in the United States. It is caused by a combination of… (more)

Subjects/Keywords: Type 2 diabetes mellitus; PHLPP; Akt; glucose; animal model; Biochemical Phenomena, Metabolism, and Nutrition; Endocrinology, Diabetes, and Metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Larson, K. L. (2014). Regulation of Glucose Homeostasis by the PHLPP1 Phosphatase. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/biochem_etds/17

Chicago Manual of Style (16th Edition):

Larson, Kara L. “Regulation of Glucose Homeostasis by the PHLPP1 Phosphatase.” 2014. Doctoral Dissertation, University of Kentucky. Accessed July 22, 2019. https://uknowledge.uky.edu/biochem_etds/17.

MLA Handbook (7th Edition):

Larson, Kara L. “Regulation of Glucose Homeostasis by the PHLPP1 Phosphatase.” 2014. Web. 22 Jul 2019.

Vancouver:

Larson KL. Regulation of Glucose Homeostasis by the PHLPP1 Phosphatase. [Internet] [Doctoral dissertation]. University of Kentucky; 2014. [cited 2019 Jul 22]. Available from: https://uknowledge.uky.edu/biochem_etds/17.

Council of Science Editors:

Larson KL. Regulation of Glucose Homeostasis by the PHLPP1 Phosphatase. [Doctoral Dissertation]. University of Kentucky; 2014. Available from: https://uknowledge.uky.edu/biochem_etds/17


University of Arkansas

21. Williams, Terry L., Jr. A Comparison of Nebulized Vitamin B12 Versus Oral Vitamin B12 Supplementation.

Degree: PhD, 2013, University of Arkansas

  Vitamin B12 is important for various processes in the human body including DNA synthesis, red blood cell production, energy production, fatty acid synthesis, and… (more)

Subjects/Keywords: Health and environmental sciences; Nebulization; Oral supplements; Vitamin B12; Vitamin deficiency; Biochemical Phenomena, Metabolism, and Nutrition; Medical Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Williams, Terry L., J. (2013). A Comparison of Nebulized Vitamin B12 Versus Oral Vitamin B12 Supplementation. (Doctoral Dissertation). University of Arkansas. Retrieved from https://scholarworks.uark.edu/etd/954

Chicago Manual of Style (16th Edition):

Williams, Terry L., Jr. “A Comparison of Nebulized Vitamin B12 Versus Oral Vitamin B12 Supplementation.” 2013. Doctoral Dissertation, University of Arkansas. Accessed July 22, 2019. https://scholarworks.uark.edu/etd/954.

MLA Handbook (7th Edition):

Williams, Terry L., Jr. “A Comparison of Nebulized Vitamin B12 Versus Oral Vitamin B12 Supplementation.” 2013. Web. 22 Jul 2019.

Vancouver:

Williams, Terry L. J. A Comparison of Nebulized Vitamin B12 Versus Oral Vitamin B12 Supplementation. [Internet] [Doctoral dissertation]. University of Arkansas; 2013. [cited 2019 Jul 22]. Available from: https://scholarworks.uark.edu/etd/954.

Council of Science Editors:

Williams, Terry L. J. A Comparison of Nebulized Vitamin B12 Versus Oral Vitamin B12 Supplementation. [Doctoral Dissertation]. University of Arkansas; 2013. Available from: https://scholarworks.uark.edu/etd/954


University of Kentucky

22. Hoffman, Jared D. THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL.

Degree: 2018, University of Kentucky

 Alzheimer’s disease (AD) is the most common form of dementia and a growing disease burden that has seen pharmacological interventions primarily fail. Instead, it has… (more)

Subjects/Keywords: Alzheimer’s Disease; APOE4; Gut Microbiota; Gut-Brain Axis; Prebiotic Inulin; Amyloid β; Biochemical Phenomena, Metabolism, and Nutrition; Neuroscience and Neurobiology; Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hoffman, J. D. (2018). THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/pharmacol_etds/24

Chicago Manual of Style (16th Edition):

Hoffman, Jared D. “THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL.” 2018. Doctoral Dissertation, University of Kentucky. Accessed July 22, 2019. https://uknowledge.uky.edu/pharmacol_etds/24.

MLA Handbook (7th Edition):

Hoffman, Jared D. “THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL.” 2018. Web. 22 Jul 2019.

Vancouver:

Hoffman JD. THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL. [Internet] [Doctoral dissertation]. University of Kentucky; 2018. [cited 2019 Jul 22]. Available from: https://uknowledge.uky.edu/pharmacol_etds/24.

Council of Science Editors:

Hoffman JD. THE PREBIOTIC INULIN BENEFICIALLY MODULATES THE GUT-BRAIN AXIS BY ENHANCING METABOLISM IN AN APOE4 MOUSE MODEL. [Doctoral Dissertation]. University of Kentucky; 2018. Available from: https://uknowledge.uky.edu/pharmacol_etds/24


University of Western Ontario

23. Pena Diaz, Ana M. Protective Effect of Modified Human Fibroblast Growth Factor on Diabetic Nephropathy.

Degree: 2015, University of Western Ontario

 Oxidative stress is a key mechanism causing Diabetic Nephropathy (DN). Acidic fibroblast growth factor (aFGF) is known to confer protection from oxidative stress. However, it… (more)

Subjects/Keywords: modified acidic FGF; acidic FGF; diabetic nephropathy; oxidative stress; nitric oxide; endothelial nitric oxide synthase; Biochemical Phenomena, Metabolism, and Nutrition; Biological Phenomena, Cell Phenomena, and Immunity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pena Diaz, A. M. (2015). Protective Effect of Modified Human Fibroblast Growth Factor on Diabetic Nephropathy. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/2717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Pena Diaz, Ana M. “Protective Effect of Modified Human Fibroblast Growth Factor on Diabetic Nephropathy.” 2015. Thesis, University of Western Ontario. Accessed July 22, 2019. https://ir.lib.uwo.ca/etd/2717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Pena Diaz, Ana M. “Protective Effect of Modified Human Fibroblast Growth Factor on Diabetic Nephropathy.” 2015. Web. 22 Jul 2019.

Vancouver:

Pena Diaz AM. Protective Effect of Modified Human Fibroblast Growth Factor on Diabetic Nephropathy. [Internet] [Thesis]. University of Western Ontario; 2015. [cited 2019 Jul 22]. Available from: https://ir.lib.uwo.ca/etd/2717.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Pena Diaz AM. Protective Effect of Modified Human Fibroblast Growth Factor on Diabetic Nephropathy. [Thesis]. University of Western Ontario; 2015. Available from: https://ir.lib.uwo.ca/etd/2717

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Tennessee – Knoxville

24. Booker, Suzanne Lauren. Regional Differences in Adipose Tissue of the Sinclair Minipig.

Degree: MS, Animal Science, 2010, University of Tennessee – Knoxville

 Adipose tissue is an endocrine organ, and its homeostatic mechanisms in normal weight, overweight and obese subjects must be elucidated. We sought to determine the… (more)

Subjects/Keywords: Sinclair minipig; obesity; adipose tissue; Biochemical Phenomena, Metabolism, and Nutrition; Biological Phenomena, Cell Phenomena, and Immunity; Medicine and Health Sciences; Other Animal Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Booker, S. L. (2010). Regional Differences in Adipose Tissue of the Sinclair Minipig. (Thesis). University of Tennessee – Knoxville. Retrieved from https://trace.tennessee.edu/utk_gradthes/689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Booker, Suzanne Lauren. “Regional Differences in Adipose Tissue of the Sinclair Minipig.” 2010. Thesis, University of Tennessee – Knoxville. Accessed July 22, 2019. https://trace.tennessee.edu/utk_gradthes/689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Booker, Suzanne Lauren. “Regional Differences in Adipose Tissue of the Sinclair Minipig.” 2010. Web. 22 Jul 2019.

Vancouver:

Booker SL. Regional Differences in Adipose Tissue of the Sinclair Minipig. [Internet] [Thesis]. University of Tennessee – Knoxville; 2010. [cited 2019 Jul 22]. Available from: https://trace.tennessee.edu/utk_gradthes/689.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Booker SL. Regional Differences in Adipose Tissue of the Sinclair Minipig. [Thesis]. University of Tennessee – Knoxville; 2010. Available from: https://trace.tennessee.edu/utk_gradthes/689

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Yearell, Cheryl D. Synthesis of a series of 16, 16-dimethyl-prostacyclin and 6-keto prostaglandin analogs.

Degree: MS, Chemistry, 1978, Clark University Atlanta

  A synthesis is described for a number of 16,16-dimethyl analogs of the prostacyclin and related 6-keto prostaglandin types. Included are l6, l6-dimethyl-PGI2 sodium salt… (more)

Subjects/Keywords: Biochemical Phenomena, Metabolism, and Nutrition; Biological Phenomena, Cell Phenomena, and Immunity

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yearell, C. D. (1978). Synthesis of a series of 16, 16-dimethyl-prostacyclin and 6-keto prostaglandin analogs. (Thesis). Clark University Atlanta. Retrieved from http://digitalcommons.auctr.edu/dissertations/787

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yearell, Cheryl D. “Synthesis of a series of 16, 16-dimethyl-prostacyclin and 6-keto prostaglandin analogs.” 1978. Thesis, Clark University Atlanta. Accessed July 22, 2019. http://digitalcommons.auctr.edu/dissertations/787.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yearell, Cheryl D. “Synthesis of a series of 16, 16-dimethyl-prostacyclin and 6-keto prostaglandin analogs.” 1978. Web. 22 Jul 2019.

Vancouver:

Yearell CD. Synthesis of a series of 16, 16-dimethyl-prostacyclin and 6-keto prostaglandin analogs. [Internet] [Thesis]. Clark University Atlanta; 1978. [cited 2019 Jul 22]. Available from: http://digitalcommons.auctr.edu/dissertations/787.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yearell CD. Synthesis of a series of 16, 16-dimethyl-prostacyclin and 6-keto prostaglandin analogs. [Thesis]. Clark University Atlanta; 1978. Available from: http://digitalcommons.auctr.edu/dissertations/787

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas Medical Center

26. Sun, Hua. THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY.

Degree: PhD, 2012, Texas Medical Center

  Plasma low-density lipoprotein (LDL) levels are positively correlated with the incidence of coronary artery disease. In the circulation, the plasma LDL clearance is mainly… (more)

Subjects/Keywords: PCSK9; apolipoprotein; cholesterol; mouse model; autophgy; atherosclerosis; LDL; Biochemical Phenomena, Metabolism, and Nutrition; Medical Biochemistry; Medical Pathology; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Sun, H. (2012). THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY. (Doctoral Dissertation). Texas Medical Center. Retrieved from http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277

Chicago Manual of Style (16th Edition):

Sun, Hua. “THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY.” 2012. Doctoral Dissertation, Texas Medical Center. Accessed July 22, 2019. http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277.

MLA Handbook (7th Edition):

Sun, Hua. “THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY.” 2012. Web. 22 Jul 2019.

Vancouver:

Sun H. THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY. [Internet] [Doctoral dissertation]. Texas Medical Center; 2012. [cited 2019 Jul 22]. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277.

Council of Science Editors:

Sun H. THE NEW ROLE OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9: A CONNECTION OF PROPROTEIN CONVERTASE SUBTILISIN/KEXIN TYPE 9, APOLIPOPROTEIN B, and AUTOPHAGY. [Doctoral Dissertation]. Texas Medical Center; 2012. Available from: http://digitalcommons.library.tmc.edu/utgsbs_dissertations/277


Michigan Technological University

27. Mohan, Ramkumar. Role of miR-483 in pancreatic alpha and beta cells.

Degree: PhD, Department of Biological Sciences, 2016, Michigan Technological University

  Diabetes mellitus is a disease associated with impaired glucose homeostasis leading to chronic hyperglycemia (elevated blood glucose level). Insulin and Glucagon, hormones secreted from… (more)

Subjects/Keywords: miRNA; Diabetes; miR-483; SOCS3; KLF2; Biochemical Phenomena, Metabolism, and Nutrition; Endocrine System Diseases; Medical Molecular Biology; Molecular Biology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Mohan, R. (2016). Role of miR-483 in pancreatic alpha and beta cells. (Doctoral Dissertation). Michigan Technological University. Retrieved from http://digitalcommons.mtu.edu/etdr/241

Chicago Manual of Style (16th Edition):

Mohan, Ramkumar. “Role of miR-483 in pancreatic alpha and beta cells.” 2016. Doctoral Dissertation, Michigan Technological University. Accessed July 22, 2019. http://digitalcommons.mtu.edu/etdr/241.

MLA Handbook (7th Edition):

Mohan, Ramkumar. “Role of miR-483 in pancreatic alpha and beta cells.” 2016. Web. 22 Jul 2019.

Vancouver:

Mohan R. Role of miR-483 in pancreatic alpha and beta cells. [Internet] [Doctoral dissertation]. Michigan Technological University; 2016. [cited 2019 Jul 22]. Available from: http://digitalcommons.mtu.edu/etdr/241.

Council of Science Editors:

Mohan R. Role of miR-483 in pancreatic alpha and beta cells. [Doctoral Dissertation]. Michigan Technological University; 2016. Available from: http://digitalcommons.mtu.edu/etdr/241


University of Western Ontario

28. Watson, Alanna R. Nicotinamide Phosphoribosyltransferase In Smooth Muscle Cells And Aortic Integrity.

Degree: 2017, University of Western Ontario

 The thoracic aortic wall can degenerate over time with catastrophic consequences. Vascular smooth muscle cells (SMCs) can resist and repair artery damage but their capacities… (more)

Subjects/Keywords: Vascular smooth muscle cells; Aortic disease; Nampt; DNA damage; Cell senescence; Extracellular matrix; Nicotinamide riboside; Biochemical Phenomena, Metabolism, and Nutrition

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Watson, A. R. (2017). Nicotinamide Phosphoribosyltransferase In Smooth Muscle Cells And Aortic Integrity. (Thesis). University of Western Ontario. Retrieved from https://ir.lib.uwo.ca/etd/5060

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Watson, Alanna R. “Nicotinamide Phosphoribosyltransferase In Smooth Muscle Cells And Aortic Integrity.” 2017. Thesis, University of Western Ontario. Accessed July 22, 2019. https://ir.lib.uwo.ca/etd/5060.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Watson, Alanna R. “Nicotinamide Phosphoribosyltransferase In Smooth Muscle Cells And Aortic Integrity.” 2017. Web. 22 Jul 2019.

Vancouver:

Watson AR. Nicotinamide Phosphoribosyltransferase In Smooth Muscle Cells And Aortic Integrity. [Internet] [Thesis]. University of Western Ontario; 2017. [cited 2019 Jul 22]. Available from: https://ir.lib.uwo.ca/etd/5060.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Watson AR. Nicotinamide Phosphoribosyltransferase In Smooth Muscle Cells And Aortic Integrity. [Thesis]. University of Western Ontario; 2017. Available from: https://ir.lib.uwo.ca/etd/5060

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Swick, Jennifer C. Effect of the Flavonoid Quercetin on Adipocytes.

Degree: MS, Nutrition, 2011, University of Massachusetts

  Obesity is an urgent global public health concern as prevalence rates continue to increase, especially among children. At the cellular level obesity is defined… (more)

Subjects/Keywords: Obesity quercetin flavonoids adipocytes; Alternative and Complementary Medicine; Biochemical Phenomena, Metabolism, and Nutrition; Nutritional and Metabolic Diseases

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Swick, J. C. (2011). Effect of the Flavonoid Quercetin on Adipocytes. (Masters Thesis). University of Massachusetts. Retrieved from https://scholarworks.umass.edu/theses/724

Chicago Manual of Style (16th Edition):

Swick, Jennifer C. “Effect of the Flavonoid Quercetin on Adipocytes.” 2011. Masters Thesis, University of Massachusetts. Accessed July 22, 2019. https://scholarworks.umass.edu/theses/724.

MLA Handbook (7th Edition):

Swick, Jennifer C. “Effect of the Flavonoid Quercetin on Adipocytes.” 2011. Web. 22 Jul 2019.

Vancouver:

Swick JC. Effect of the Flavonoid Quercetin on Adipocytes. [Internet] [Masters thesis]. University of Massachusetts; 2011. [cited 2019 Jul 22]. Available from: https://scholarworks.umass.edu/theses/724.

Council of Science Editors:

Swick JC. Effect of the Flavonoid Quercetin on Adipocytes. [Masters Thesis]. University of Massachusetts; 2011. Available from: https://scholarworks.umass.edu/theses/724


University of Louisville

30. Lang, Anna L. Vinyl chloride-diet interactions in liver disease : potential roles of autophagy and energy management.

Degree: MS, 2016, University of Louisville

  Vinyl chloride (VC) is a prevalent environmental toxicant that has been shown to cause liver injury at high, occupational exposures. However, most studies have… (more)

Subjects/Keywords: liver disease; vinyl chloride; autophagy; mTOR; Biochemical Phenomena, Metabolism, and Nutrition; Disorders of Environmental Origin; Medicine and Health Sciences

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lang, A. L. (2016). Vinyl chloride-diet interactions in liver disease : potential roles of autophagy and energy management. (Masters Thesis). University of Louisville. Retrieved from 10.18297/etd/2529 ; https://ir.library.louisville.edu/etd/2529

Chicago Manual of Style (16th Edition):

Lang, Anna L. “Vinyl chloride-diet interactions in liver disease : potential roles of autophagy and energy management.” 2016. Masters Thesis, University of Louisville. Accessed July 22, 2019. 10.18297/etd/2529 ; https://ir.library.louisville.edu/etd/2529.

MLA Handbook (7th Edition):

Lang, Anna L. “Vinyl chloride-diet interactions in liver disease : potential roles of autophagy and energy management.” 2016. Web. 22 Jul 2019.

Vancouver:

Lang AL. Vinyl chloride-diet interactions in liver disease : potential roles of autophagy and energy management. [Internet] [Masters thesis]. University of Louisville; 2016. [cited 2019 Jul 22]. Available from: 10.18297/etd/2529 ; https://ir.library.louisville.edu/etd/2529.

Council of Science Editors:

Lang AL. Vinyl chloride-diet interactions in liver disease : potential roles of autophagy and energy management. [Masters Thesis]. University of Louisville; 2016. Available from: 10.18297/etd/2529 ; https://ir.library.louisville.edu/etd/2529

[1] [2] [3] [4] [5] … [18]

.