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University of Debrecen

1. Lajtos, Tamás. Az ErbB1-β1 integrin molekuláris kölcsönhatás a PI3K-Akt függő túlélési útvonalon át fokozza a glia tumorok sugárrezisztenciáját .

Degree: DE – TEK – Természettudományi és Technológiai Kar – Biológiai és Ökológiai Intézet, 2010, University of Debrecen

 Az epidermális növekedési faktor receptor (EGFR, ErbB1) a sejtek proliferációjában, differenciálódásában, migrációjában és túlélésében fontos szereppel bíró receptor tirozinkináz. Amplifikációja megfigyelhető a magas (IV.) grádusú… (more)

Subjects/Keywords: EGFR; ErbB1; beta1 integrin; glioblasztóma; sugárrezisztencia; FRET

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lajtos, T. (2010). Az ErbB1-β1 integrin molekuláris kölcsönhatás a PI3K-Akt függő túlélési útvonalon át fokozza a glia tumorok sugárrezisztenciáját . (Thesis). University of Debrecen. Retrieved from http://hdl.handle.net/2437/95571

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lajtos, Tamás. “Az ErbB1-β1 integrin molekuláris kölcsönhatás a PI3K-Akt függő túlélési útvonalon át fokozza a glia tumorok sugárrezisztenciáját .” 2010. Thesis, University of Debrecen. Accessed April 10, 2021. http://hdl.handle.net/2437/95571.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lajtos, Tamás. “Az ErbB1-β1 integrin molekuláris kölcsönhatás a PI3K-Akt függő túlélési útvonalon át fokozza a glia tumorok sugárrezisztenciáját .” 2010. Web. 10 Apr 2021.

Vancouver:

Lajtos T. Az ErbB1-β1 integrin molekuláris kölcsönhatás a PI3K-Akt függő túlélési útvonalon át fokozza a glia tumorok sugárrezisztenciáját . [Internet] [Thesis]. University of Debrecen; 2010. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/2437/95571.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lajtos T. Az ErbB1-β1 integrin molekuláris kölcsönhatás a PI3K-Akt függő túlélési útvonalon át fokozza a glia tumorok sugárrezisztenciáját . [Thesis]. University of Debrecen; 2010. Available from: http://hdl.handle.net/2437/95571

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. Theret, Louis. Etude de l'interface fonctionnelle LRP-1/intégrine beta1 dans le cadre de la progression tumorale. : Role of the functional interface LRP-1/integrin beta1 in tumor progression.

Degree: Docteur es, Biochimie et biologie moléculaire, 2017, Reims

Résumé : LRP-1 est un récepteur d’endocytose qui fut d’abord associé à des propriétés anti-tumorales via l’internalisation et le catabolisme de protéases matricielles. Cependant, malgré… (more)

Subjects/Keywords: Lrp-1; Intégrine beta1; Cancer; Endocytose; Recyclage; Lrp-1; Integrin beta1; Cancer; Endocytosis; Recycling

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APA (6th Edition):

Theret, L. (2017). Etude de l'interface fonctionnelle LRP-1/intégrine beta1 dans le cadre de la progression tumorale. : Role of the functional interface LRP-1/integrin beta1 in tumor progression. (Doctoral Dissertation). Reims. Retrieved from http://www.theses.fr/2017REIMS015

Chicago Manual of Style (16th Edition):

Theret, Louis. “Etude de l'interface fonctionnelle LRP-1/intégrine beta1 dans le cadre de la progression tumorale. : Role of the functional interface LRP-1/integrin beta1 in tumor progression.” 2017. Doctoral Dissertation, Reims. Accessed April 10, 2021. http://www.theses.fr/2017REIMS015.

MLA Handbook (7th Edition):

Theret, Louis. “Etude de l'interface fonctionnelle LRP-1/intégrine beta1 dans le cadre de la progression tumorale. : Role of the functional interface LRP-1/integrin beta1 in tumor progression.” 2017. Web. 10 Apr 2021.

Vancouver:

Theret L. Etude de l'interface fonctionnelle LRP-1/intégrine beta1 dans le cadre de la progression tumorale. : Role of the functional interface LRP-1/integrin beta1 in tumor progression. [Internet] [Doctoral dissertation]. Reims; 2017. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2017REIMS015.

Council of Science Editors:

Theret L. Etude de l'interface fonctionnelle LRP-1/intégrine beta1 dans le cadre de la progression tumorale. : Role of the functional interface LRP-1/integrin beta1 in tumor progression. [Doctoral Dissertation]. Reims; 2017. Available from: http://www.theses.fr/2017REIMS015


Universidade Estadual de Campinas

3. Peres, Karina Colombera, 1992-. Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide.

Degree: Faculdade de Ciências Médicas; Programa de Pós-Graduação em Clínica Médica, 2018, Universidade Estadual de Campinas

Orientadores: Laura Sterian Ward, Natassia Elena Bufalo

Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas

Made available in DSpace on 2018-09-03T07:23:43Z (GMT).… (more)

Subjects/Keywords: Neoplasias da glândula tireoide; Marcadores moleculares; Fator de crescimento transformador beta1; Thyroid gland neoplasms; Molecular markers; Transforming growth factor beta1

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APA (6th Edition):

Peres, Karina Colombera, 1. (2018). Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide. (Masters Thesis). Universidade Estadual de Campinas. Retrieved from PERES, Karina Colombera. Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide. 2018. 1 recurso online (93 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/331055>. Acesso em: 3 set. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/331055

Chicago Manual of Style (16th Edition):

Peres, Karina Colombera, 1992-. “Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide.” 2018. Masters Thesis, Universidade Estadual de Campinas. Accessed April 10, 2021. PERES, Karina Colombera. Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide. 2018. 1 recurso online (93 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/331055>. Acesso em: 3 set. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/331055.

MLA Handbook (7th Edition):

Peres, Karina Colombera, 1992-. “Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide.” 2018. Web. 10 Apr 2021.

Vancouver:

Peres, Karina Colombera 1. Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide. [Internet] [Masters thesis]. Universidade Estadual de Campinas; 2018. [cited 2021 Apr 10]. Available from: PERES, Karina Colombera. Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide. 2018. 1 recurso online (93 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/331055>. Acesso em: 3 set. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/331055.

Council of Science Editors:

Peres, Karina Colombera 1. Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide. [Masters Thesis]. Universidade Estadual de Campinas; 2018. Available from: PERES, Karina Colombera. Estudo do papel de TGF-ß1 no carcinoma diferenciado da tireoide. 2018. 1 recurso online (93 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Ciências Médicas, Campinas, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/331055>. Acesso em: 3 set. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/331055


Universidade Estadual de Campinas

4. Soares, Ciro Dantas, 1990-. Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells.

Degree: Faculdade de Odontologia de Piracicaba; Programa de Pós-Graduação em Estomatopatologia, 2016, Universidade Estadual de Campinas

Orientador: Jacks Jorge Junior

Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba

Made available in DSpace on 2018-08-31T01:39:35Z (GMT). No. of… (more)

Subjects/Keywords: Fator de crescimento transformador beta1; Ciclooxigenase 2; Melanoma; Citotoxicidade; Transforming growth factor beta1; Ciclooxigenase 2; Melanoma; Cytotoxicity

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APA (6th Edition):

Soares, Ciro Dantas, 1. (2016). Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells. (Masters Thesis). Universidade Estadual de Campinas. Retrieved from SOARES, Ciro Dantas. Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells. 2016. 1 recurso online (81 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/325027>. Acesso em: 30 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/325027

Chicago Manual of Style (16th Edition):

Soares, Ciro Dantas, 1990-. “Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells.” 2016. Masters Thesis, Universidade Estadual de Campinas. Accessed April 10, 2021. SOARES, Ciro Dantas. Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells. 2016. 1 recurso online (81 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/325027>. Acesso em: 30 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/325027.

MLA Handbook (7th Edition):

Soares, Ciro Dantas, 1990-. “Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells.” 2016. Web. 10 Apr 2021.

Vancouver:

Soares, Ciro Dantas 1. Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells. [Internet] [Masters thesis]. Universidade Estadual de Campinas; 2016. [cited 2021 Apr 10]. Available from: SOARES, Ciro Dantas. Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells. 2016. 1 recurso online (81 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/325027>. Acesso em: 30 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/325027.

Council of Science Editors:

Soares, Ciro Dantas 1. Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells. [Masters Thesis]. Universidade Estadual de Campinas; 2016. Available from: SOARES, Ciro Dantas. Análise da expressão de COX-2, p-Akt e TGF beta 1 em melanomas cutâneos primários e metastáticos e da citotoxicidade de trans-desidrocrotonina em células de melanoma = Analyses of COX-2, p-Akt and TGF beta 1 in primary and metastatic melanomas and of the citotoxicity of trans-dehydrocrotonin in melanoma cells. 2016. 1 recurso online (81 p.). Dissertação (mestrado) - Universidade Estadual de Campinas, Faculdade de Odontologia de Piracicaba, Piracicaba, SP. Disponível em: <http://www.repositorio.unicamp.br/handle/REPOSIP/325027>. Acesso em: 30 ago. 2018. ; http://repositorio.unicamp.br/jspui/handle/REPOSIP/325027

5. Antonio Carlos Aloise. TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno.

Degree: 2009, Universidade Federal de São Paulo

Introducao: A busca por uma fonte alternativa de celulas para a Engenharia Tecidual ossea, se deve a morbidade da area doadora e maior dificuldade de… (more)

Subjects/Keywords: Fibroblastos; Diferenciacao celular; TGF-Beta1; Colageno; CIRURGIA PLASTICA E RESTAURADORA

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APA (6th Edition):

Aloise, A. C. (2009). TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno. (Thesis). Universidade Federal de São Paulo. Retrieved from http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1890

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aloise, Antonio Carlos. “TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno.” 2009. Thesis, Universidade Federal de São Paulo. Accessed April 10, 2021. http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1890.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aloise, Antonio Carlos. “TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno.” 2009. Web. 10 Apr 2021.

Vancouver:

Aloise AC. TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno. [Internet] [Thesis]. Universidade Federal de São Paulo; 2009. [cited 2021 Apr 10]. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1890.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aloise AC. TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno. [Thesis]. Universidade Federal de São Paulo; 2009. Available from: http://www.bdtd.unifesp.br/tede//tde_busca/arquivo.php?codArquivo=1890

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Adelaide

6. Ingman, Wendy Vanessa. The effect of transforming growth factor beta1 null mutation on murine reproductive function.

Degree: 2002, University of Adelaide

 Transforming growth factor beta 1 (TGFβ1) is a multifunctional cytokine implicated in gonad and secondary sex organ development, spermatogenesis and ovarian function, immunoregulation of pregnancy,… (more)

Subjects/Keywords: beta1; mutation; murine; reproduction

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APA (6th Edition):

Ingman, W. V. (2002). The effect of transforming growth factor beta1 null mutation on murine reproductive function. (Thesis). University of Adelaide. Retrieved from http://hdl.handle.net/2440/79715

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ingman, Wendy Vanessa. “The effect of transforming growth factor beta1 null mutation on murine reproductive function.” 2002. Thesis, University of Adelaide. Accessed April 10, 2021. http://hdl.handle.net/2440/79715.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ingman, Wendy Vanessa. “The effect of transforming growth factor beta1 null mutation on murine reproductive function.” 2002. Web. 10 Apr 2021.

Vancouver:

Ingman WV. The effect of transforming growth factor beta1 null mutation on murine reproductive function. [Internet] [Thesis]. University of Adelaide; 2002. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/2440/79715.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ingman WV. The effect of transforming growth factor beta1 null mutation on murine reproductive function. [Thesis]. University of Adelaide; 2002. Available from: http://hdl.handle.net/2440/79715

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. Van Ry, Pam Marie. Protein Therapy for Muscular Dystrophy and Other Muscle Diseases.

Degree: 2014, University of Nevada – Reno

 Despite the exponential advancements in understanding the underlying cause of muscular diseases, there has been little progress in developing an effective treatment which results in… (more)

Subjects/Keywords: alpha7 beta1 integrin; Duchenne Muscular Dystrophy; MDC1A; Muscular Dystrophy; Protein; Therapy

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APA (6th Edition):

Van Ry, P. M. (2014). Protein Therapy for Muscular Dystrophy and Other Muscle Diseases. (Thesis). University of Nevada – Reno. Retrieved from http://hdl.handle.net/11714/2928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Van Ry, Pam Marie. “Protein Therapy for Muscular Dystrophy and Other Muscle Diseases.” 2014. Thesis, University of Nevada – Reno. Accessed April 10, 2021. http://hdl.handle.net/11714/2928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Van Ry, Pam Marie. “Protein Therapy for Muscular Dystrophy and Other Muscle Diseases.” 2014. Web. 10 Apr 2021.

Vancouver:

Van Ry PM. Protein Therapy for Muscular Dystrophy and Other Muscle Diseases. [Internet] [Thesis]. University of Nevada – Reno; 2014. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11714/2928.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Van Ry PM. Protein Therapy for Muscular Dystrophy and Other Muscle Diseases. [Thesis]. University of Nevada – Reno; 2014. Available from: http://hdl.handle.net/11714/2928

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universitat de Valencia

8. Bolás González-Lliberós, Gema. Caracterización de la disintegrina antiangiogénica jerdostatina. Generación de un modelo transgénico animal .

Degree: 2016, Universitat de Valencia

 Determinados venenos de serpientes contienen antagonistas específicos de integrinas llamados disintegrinas. Dentro de la familia de las disintegrinas encontramos las disintegrinas cortas monoméricas que contienen… (more)

Subjects/Keywords: disintegrina (R/K)TS; jerdostatina; integrina alpha1 beta1; angiogénesis; transgénico

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APA (6th Edition):

Bolás González-Lliberós, G. (2016). Caracterización de la disintegrina antiangiogénica jerdostatina. Generación de un modelo transgénico animal . (Doctoral Dissertation). Universitat de Valencia. Retrieved from http://hdl.handle.net/10550/55697

Chicago Manual of Style (16th Edition):

Bolás González-Lliberós, Gema. “Caracterización de la disintegrina antiangiogénica jerdostatina. Generación de un modelo transgénico animal .” 2016. Doctoral Dissertation, Universitat de Valencia. Accessed April 10, 2021. http://hdl.handle.net/10550/55697.

MLA Handbook (7th Edition):

Bolás González-Lliberós, Gema. “Caracterización de la disintegrina antiangiogénica jerdostatina. Generación de un modelo transgénico animal .” 2016. Web. 10 Apr 2021.

Vancouver:

Bolás González-Lliberós G. Caracterización de la disintegrina antiangiogénica jerdostatina. Generación de un modelo transgénico animal . [Internet] [Doctoral dissertation]. Universitat de Valencia; 2016. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10550/55697.

Council of Science Editors:

Bolás González-Lliberós G. Caracterización de la disintegrina antiangiogénica jerdostatina. Generación de un modelo transgénico animal . [Doctoral Dissertation]. Universitat de Valencia; 2016. Available from: http://hdl.handle.net/10550/55697

9. Moore, Lakisha Dionne. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.

Degree: PhD, 2008, University of Alabama – Birmingham

Overexpresssion of transforming growth factor (TGF)-[beta] has been implicated in promoting immune suppression, tumor angiogenesis, tumor cell migration, and invasion in many cancers including carcinoma… (more)

Subjects/Keywords: Breast Neoplasms  – genetics <; br>; Neoplasm Metastasis <; br>; RNA Interference <; br>; Transforming Growth Factor beta1  – genetics <; br>; Transforming Growth Factor beta1  – metabolism

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APA (6th Edition):

Moore, L. D. (2008). Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,233

Chicago Manual of Style (16th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 10, 2021. http://contentdm.mhsl.uab.edu/u?/etd,233.

MLA Handbook (7th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Web. 10 Apr 2021.

Vancouver:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2021 Apr 10]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233.

Council of Science Editors:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233


University of Helsinki

10. Tsubari, Minna. Construction of mink lung epithelial cell λpCEV27 cDNA library.

Degree: 1994, University of Helsinki

Subjects/Keywords: expression cloning; TGF-beta1; growth regulation; Perinnöllisyystiede; expression cloning; TGF-beta1; growth regulation

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APA (6th Edition):

Tsubari, M. (1994). Construction of mink lung epithelial cell λpCEV27 cDNA library. (Thesis). University of Helsinki. Retrieved from http://hdl.handle.net/10138/157651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsubari, Minna. “Construction of mink lung epithelial cell λpCEV27 cDNA library.” 1994. Thesis, University of Helsinki. Accessed April 10, 2021. http://hdl.handle.net/10138/157651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsubari, Minna. “Construction of mink lung epithelial cell λpCEV27 cDNA library.” 1994. Web. 10 Apr 2021.

Vancouver:

Tsubari M. Construction of mink lung epithelial cell λpCEV27 cDNA library. [Internet] [Thesis]. University of Helsinki; 1994. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10138/157651.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsubari M. Construction of mink lung epithelial cell λpCEV27 cDNA library. [Thesis]. University of Helsinki; 1994. Available from: http://hdl.handle.net/10138/157651

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. Rafael Braga Petito. Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural.

Degree: 2008, Fundação Oswaldo Cruz

Neural fibrosis is one of the causes of deformities during leprosy, which remains a major global health problem. Neural degeneration progresses after the patient healing… (more)

Subjects/Keywords: BIOLOGIA MOLECULAR; Mycobacterium leprae; Fibrose/complicações; Hanseníase; Saúde Pública; Fator Transformador de Crescimento beta1/análise; Mycobacterium leprae; Fibrosis/Complications; Leprosy; Public Health; Transforming Growth Factor beta1/analysis

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APA (6th Edition):

Petito, R. B. (2008). Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural. (Thesis). Fundação Oswaldo Cruz. Retrieved from http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petito, Rafael Braga. “Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural.” 2008. Thesis, Fundação Oswaldo Cruz. Accessed April 10, 2021. http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petito, Rafael Braga. “Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural.” 2008. Web. 10 Apr 2021.

Vancouver:

Petito RB. Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural. [Internet] [Thesis]. Fundação Oswaldo Cruz; 2008. [cited 2021 Apr 10]. Available from: http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petito RB. Study of the effects of Mycobacterium leprae, and their lipid components of the TGF-b1 changes in extracellular matrix neural. [Thesis]. Fundação Oswaldo Cruz; 2008. Available from: http://www.bdtd.cict.fiocruz.br/tedesimplificado/tde_busca/arquivo.php?codArquivo=224

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. Schaff, Mathieu. Etude des mécanismes d'adhérence et d'activation des plaquettes sanguines appliquée à l'identification de nouvelles cibles anti-thrombotiques plus sûres : Study of blood platelet adhesion and activation mechanisms to identify safer antithrombotic targets.

Degree: Docteur es, Hématologie et physiopathologie vasculaire, 2012, Université de Strasbourg

L’adhérence, l’activation et l’agrégation des plaquettes sanguines sont essentielles à l’hémostase mais peuvent également conduire à la thrombose artérielle sur plaque d’athérosclérose, aujourd’hui première cause… (more)

Subjects/Keywords: Plaquettes; Hémostase; Thrombose artérielle; Intégrine alpha6 beta1; Ténascine-C; Beta-arrestine; Platelets; Hemostasis; Arterial thrombosis; Integrin alpha6 beta1; Tenascin-C; Beat-arrestin; 573.1; 612.13; 616.13

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APA (6th Edition):

Schaff, M. (2012). Etude des mécanismes d'adhérence et d'activation des plaquettes sanguines appliquée à l'identification de nouvelles cibles anti-thrombotiques plus sûres : Study of blood platelet adhesion and activation mechanisms to identify safer antithrombotic targets. (Doctoral Dissertation). Université de Strasbourg. Retrieved from http://www.theses.fr/2012STRAJ102

Chicago Manual of Style (16th Edition):

Schaff, Mathieu. “Etude des mécanismes d'adhérence et d'activation des plaquettes sanguines appliquée à l'identification de nouvelles cibles anti-thrombotiques plus sûres : Study of blood platelet adhesion and activation mechanisms to identify safer antithrombotic targets.” 2012. Doctoral Dissertation, Université de Strasbourg. Accessed April 10, 2021. http://www.theses.fr/2012STRAJ102.

MLA Handbook (7th Edition):

Schaff, Mathieu. “Etude des mécanismes d'adhérence et d'activation des plaquettes sanguines appliquée à l'identification de nouvelles cibles anti-thrombotiques plus sûres : Study of blood platelet adhesion and activation mechanisms to identify safer antithrombotic targets.” 2012. Web. 10 Apr 2021.

Vancouver:

Schaff M. Etude des mécanismes d'adhérence et d'activation des plaquettes sanguines appliquée à l'identification de nouvelles cibles anti-thrombotiques plus sûres : Study of blood platelet adhesion and activation mechanisms to identify safer antithrombotic targets. [Internet] [Doctoral dissertation]. Université de Strasbourg; 2012. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2012STRAJ102.

Council of Science Editors:

Schaff M. Etude des mécanismes d'adhérence et d'activation des plaquettes sanguines appliquée à l'identification de nouvelles cibles anti-thrombotiques plus sûres : Study of blood platelet adhesion and activation mechanisms to identify safer antithrombotic targets. [Doctoral Dissertation]. Université de Strasbourg; 2012. Available from: http://www.theses.fr/2012STRAJ102


North-West University

13. Campbell, Berenice. Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell .

Degree: 2010, North-West University

 Pigmentation disorders occur in multiple conditions (Hakozaki et al., 2006:105). Although many modalities of treatments are available, none are completely satisfactory (Briganti et al., 2003:101).… (more)

Subjects/Keywords: Pigmentation; Topical delivery; Pheroid; Transforming growth factor-beta1; Tumour necrosis factor-alpha; Bestatin; Tape stripping; Pigmentasie; Topikale aflewering; Transformerende groei faktor-beta1; Tumor nekrosis faktor-alpha; Bestatien; Bandstroping

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APA (6th Edition):

Campbell, B. (2010). Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell . (Thesis). North-West University. Retrieved from http://hdl.handle.net/10394/4739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Campbell, Berenice. “Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell .” 2010. Thesis, North-West University. Accessed April 10, 2021. http://hdl.handle.net/10394/4739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Campbell, Berenice. “Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell .” 2010. Web. 10 Apr 2021.

Vancouver:

Campbell B. Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell . [Internet] [Thesis]. North-West University; 2010. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10394/4739.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Campbell B. Pheroid technology for the topical delivery of depigmenting agents transforming growth factor–ß1 and tumor necrosis factor–a / Berenice Campbell . [Thesis]. North-West University; 2010. Available from: http://hdl.handle.net/10394/4739

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


UCLA

14. Noel, Onika Doreen Veronica. Beta1 Integrin and the Extracellular Matrix Provide Integrative Signals for Vascular Development.

Degree: Molecular Biology, 2014, UCLA

 Extracellular matrix (ECM) organization is critical for the maintenance of vascular integrity and for regulating biochemical signals that are instructive during the development and maturation… (more)

Subjects/Keywords: Molecular biology; Biomedical engineering; Aneurysm; Beta1 Integrin; ECM Assembly; Extracellular Matrix; Flow Mapping; Vascular Biology

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APA (6th Edition):

Noel, O. D. V. (2014). Beta1 Integrin and the Extracellular Matrix Provide Integrative Signals for Vascular Development. (Thesis). UCLA. Retrieved from http://www.escholarship.org/uc/item/0974f1br

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Noel, Onika Doreen Veronica. “Beta1 Integrin and the Extracellular Matrix Provide Integrative Signals for Vascular Development.” 2014. Thesis, UCLA. Accessed April 10, 2021. http://www.escholarship.org/uc/item/0974f1br.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Noel, Onika Doreen Veronica. “Beta1 Integrin and the Extracellular Matrix Provide Integrative Signals for Vascular Development.” 2014. Web. 10 Apr 2021.

Vancouver:

Noel ODV. Beta1 Integrin and the Extracellular Matrix Provide Integrative Signals for Vascular Development. [Internet] [Thesis]. UCLA; 2014. [cited 2021 Apr 10]. Available from: http://www.escholarship.org/uc/item/0974f1br.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Noel ODV. Beta1 Integrin and the Extracellular Matrix Provide Integrative Signals for Vascular Development. [Thesis]. UCLA; 2014. Available from: http://www.escholarship.org/uc/item/0974f1br

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. Liu, Jun. Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration.

Degree: PhD, Medical Microbiology and Immunology (graduate program), 2013, Creighton University

 Liver fibrosis and cancer metastasis remain significant health problems, with a mortality rate of nine million each year worldwide. Effective therapies for these problems are… (more)

Subjects/Keywords: Epithelial-Mesenchymal Transition – genetics; Cell Movement; Liver Cirrhosis; Lung Neoplasm; Transforming Growth Factor beta1 – physiology

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APA (6th Edition):

Liu, J. (2013). Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration. (Doctoral Dissertation). Creighton University. Retrieved from http://hdl.handle.net/10504/42059

Chicago Manual of Style (16th Edition):

Liu, Jun. “Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration.” 2013. Doctoral Dissertation, Creighton University. Accessed April 10, 2021. http://hdl.handle.net/10504/42059.

MLA Handbook (7th Edition):

Liu, Jun. “Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration.” 2013. Web. 10 Apr 2021.

Vancouver:

Liu J. Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration. [Internet] [Doctoral dissertation]. Creighton University; 2013. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10504/42059.

Council of Science Editors:

Liu J. Snail1-mediated Gene Repression Links EMT to Enhanced Biliary Epithelial Cell Survival and Lung Epithelial Cancer Cell Migration. [Doctoral Dissertation]. Creighton University; 2013. Available from: http://hdl.handle.net/10504/42059


University of Michigan

16. Bouza, Alexandra. Voltage-Gated Sodium Channel B1 Subunit Processing by BACE1 and y-Secretase: Regulatory Mechanisms and Downstream Signaling.

Degree: PhD, Pharmacology, 2020, University of Michigan

 Voltage-gated sodium channels (VGSCs) are heterotrimeric proteins comprised of one pore-forming, alpha subunit and two non-pore forming, beta subunits. Variants in SCN1B, the gene which… (more)

Subjects/Keywords: Voltage-Gated Sodium Channel Beta1 Subunit Processing; Pharmacy and Pharmacology; Health Sciences

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APA (6th Edition):

Bouza, A. (2020). Voltage-Gated Sodium Channel B1 Subunit Processing by BACE1 and y-Secretase: Regulatory Mechanisms and Downstream Signaling. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/155189

Chicago Manual of Style (16th Edition):

Bouza, Alexandra. “Voltage-Gated Sodium Channel B1 Subunit Processing by BACE1 and y-Secretase: Regulatory Mechanisms and Downstream Signaling.” 2020. Doctoral Dissertation, University of Michigan. Accessed April 10, 2021. http://hdl.handle.net/2027.42/155189.

MLA Handbook (7th Edition):

Bouza, Alexandra. “Voltage-Gated Sodium Channel B1 Subunit Processing by BACE1 and y-Secretase: Regulatory Mechanisms and Downstream Signaling.” 2020. Web. 10 Apr 2021.

Vancouver:

Bouza A. Voltage-Gated Sodium Channel B1 Subunit Processing by BACE1 and y-Secretase: Regulatory Mechanisms and Downstream Signaling. [Internet] [Doctoral dissertation]. University of Michigan; 2020. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/2027.42/155189.

Council of Science Editors:

Bouza A. Voltage-Gated Sodium Channel B1 Subunit Processing by BACE1 and y-Secretase: Regulatory Mechanisms and Downstream Signaling. [Doctoral Dissertation]. University of Michigan; 2020. Available from: http://hdl.handle.net/2027.42/155189


Wayne State University

17. White, Jennell. The potential role of innate immunity in the pathogenesis of post-operative adhesions.

Degree: PhD, Physiology, 2011, Wayne State University

  Post-operative adhesion development occurs in the vast majority of patients following abdominal surgery and is a natural occurrence of peritoneal-wound healing. These fibrous bands… (more)

Subjects/Keywords: adhesions, hypoxia, macrophages, TGF-beta1, type I collagen, VEGF; Obstetrics and Gynecology; Physiology; Surgery

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APA (6th Edition):

White, J. (2011). The potential role of innate immunity in the pathogenesis of post-operative adhesions. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/401

Chicago Manual of Style (16th Edition):

White, Jennell. “The potential role of innate immunity in the pathogenesis of post-operative adhesions.” 2011. Doctoral Dissertation, Wayne State University. Accessed April 10, 2021. https://digitalcommons.wayne.edu/oa_dissertations/401.

MLA Handbook (7th Edition):

White, Jennell. “The potential role of innate immunity in the pathogenesis of post-operative adhesions.” 2011. Web. 10 Apr 2021.

Vancouver:

White J. The potential role of innate immunity in the pathogenesis of post-operative adhesions. [Internet] [Doctoral dissertation]. Wayne State University; 2011. [cited 2021 Apr 10]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/401.

Council of Science Editors:

White J. The potential role of innate immunity in the pathogenesis of post-operative adhesions. [Doctoral Dissertation]. Wayne State University; 2011. Available from: https://digitalcommons.wayne.edu/oa_dissertations/401


University of Colorado

18. Wang, Huan. Signaling from Matrix Elasticity and TGF-beta1 to Cells of the Cardiac Valve.

Degree: PhD, 2013, University of Colorado

  Coordinated movement of cardiac valves controls unidirectional flow of the blood with every heart beat. Cardiac valves are composed of thin, pliable leaflets that… (more)

Subjects/Keywords: cadherin; Hydrogel elasticity; Mechanosensing; PI3K signaling; TGF-beta1 signaling; Valvular myofibroblasts; Biology

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APA (6th Edition):

Wang, H. (2013). Signaling from Matrix Elasticity and TGF-beta1 to Cells of the Cardiac Valve. (Doctoral Dissertation). University of Colorado. Retrieved from https://scholar.colorado.edu/mcdb_gradetds/16

Chicago Manual of Style (16th Edition):

Wang, Huan. “Signaling from Matrix Elasticity and TGF-beta1 to Cells of the Cardiac Valve.” 2013. Doctoral Dissertation, University of Colorado. Accessed April 10, 2021. https://scholar.colorado.edu/mcdb_gradetds/16.

MLA Handbook (7th Edition):

Wang, Huan. “Signaling from Matrix Elasticity and TGF-beta1 to Cells of the Cardiac Valve.” 2013. Web. 10 Apr 2021.

Vancouver:

Wang H. Signaling from Matrix Elasticity and TGF-beta1 to Cells of the Cardiac Valve. [Internet] [Doctoral dissertation]. University of Colorado; 2013. [cited 2021 Apr 10]. Available from: https://scholar.colorado.edu/mcdb_gradetds/16.

Council of Science Editors:

Wang H. Signaling from Matrix Elasticity and TGF-beta1 to Cells of the Cardiac Valve. [Doctoral Dissertation]. University of Colorado; 2013. Available from: https://scholar.colorado.edu/mcdb_gradetds/16


University College Cork

19. O'Riordan, Ronan T. Pregnancy-specific glycoprotein function, conservation and receptor investigation.

Degree: 2014, University College Cork

 Pregnancy-specific glycoproteins (PSGs) are highly glycosylated secreted proteins encoded by multi-gene families in some placental mammals. They are carcinoembryonic antigen (CEA) family and immunoglobulin (Ig)… (more)

Subjects/Keywords: Integrin; Evolution; TGF-beta1; Glycosylation; IL-6; TNF-alpha; Secretion; Pregnancy-specific gycoprotein

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APA (6th Edition):

O'Riordan, R. T. (2014). Pregnancy-specific glycoprotein function, conservation and receptor investigation. (Thesis). University College Cork. Retrieved from http://hdl.handle.net/10468/1952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

O'Riordan, Ronan T. “Pregnancy-specific glycoprotein function, conservation and receptor investigation.” 2014. Thesis, University College Cork. Accessed April 10, 2021. http://hdl.handle.net/10468/1952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

O'Riordan, Ronan T. “Pregnancy-specific glycoprotein function, conservation and receptor investigation.” 2014. Web. 10 Apr 2021.

Vancouver:

O'Riordan RT. Pregnancy-specific glycoprotein function, conservation and receptor investigation. [Internet] [Thesis]. University College Cork; 2014. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10468/1952.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

O'Riordan RT. Pregnancy-specific glycoprotein function, conservation and receptor investigation. [Thesis]. University College Cork; 2014. Available from: http://hdl.handle.net/10468/1952

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

20. Baello, Stephanie. The Role of Astrocyte-Derived Factors in the Regulation of Multidrug Resistance at the Developing Blood-Brain Barrier.

Degree: PhD, 2016, University of Toronto

P-glycoprotein (P-gp; encoded by Abcb1) at the blood-brain barrier (BBB) plays an important role in regulating the movement of exogenous and endogenous substrates into the… (more)

Subjects/Keywords: blood-brain barrier; brain endothelium; fetus; neurovascular unit; p-glycoprotein; tgf-beta1; 0719

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APA (6th Edition):

Baello, S. (2016). The Role of Astrocyte-Derived Factors in the Regulation of Multidrug Resistance at the Developing Blood-Brain Barrier. (Doctoral Dissertation). University of Toronto. Retrieved from http://hdl.handle.net/1807/89047

Chicago Manual of Style (16th Edition):

Baello, Stephanie. “The Role of Astrocyte-Derived Factors in the Regulation of Multidrug Resistance at the Developing Blood-Brain Barrier.” 2016. Doctoral Dissertation, University of Toronto. Accessed April 10, 2021. http://hdl.handle.net/1807/89047.

MLA Handbook (7th Edition):

Baello, Stephanie. “The Role of Astrocyte-Derived Factors in the Regulation of Multidrug Resistance at the Developing Blood-Brain Barrier.” 2016. Web. 10 Apr 2021.

Vancouver:

Baello S. The Role of Astrocyte-Derived Factors in the Regulation of Multidrug Resistance at the Developing Blood-Brain Barrier. [Internet] [Doctoral dissertation]. University of Toronto; 2016. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/1807/89047.

Council of Science Editors:

Baello S. The Role of Astrocyte-Derived Factors in the Regulation of Multidrug Resistance at the Developing Blood-Brain Barrier. [Doctoral Dissertation]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/89047


University of Melbourne

21. Li, Meina. A novel non-canonical pathway with potential for safer modulation of TGF-beta1 in steroid-resistant airway diseases.

Degree: 2017, University of Melbourne

 Glucocorticoids remain central to treatment regimens for chronic airway inflammatory diseases, but the therapeutic responses are often limited by impaired responses in severe diseases. Glucocorticoid… (more)

Subjects/Keywords: TGF-beta1; glucocorticoid insensitivity; phospho-cofilin1; LIM kinase; phospholipase D; (lyso)phosphatidic acid

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APA (6th Edition):

Li, M. (2017). A novel non-canonical pathway with potential for safer modulation of TGF-beta1 in steroid-resistant airway diseases. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/208016

Chicago Manual of Style (16th Edition):

Li, Meina. “A novel non-canonical pathway with potential for safer modulation of TGF-beta1 in steroid-resistant airway diseases.” 2017. Doctoral Dissertation, University of Melbourne. Accessed April 10, 2021. http://hdl.handle.net/11343/208016.

MLA Handbook (7th Edition):

Li, Meina. “A novel non-canonical pathway with potential for safer modulation of TGF-beta1 in steroid-resistant airway diseases.” 2017. Web. 10 Apr 2021.

Vancouver:

Li M. A novel non-canonical pathway with potential for safer modulation of TGF-beta1 in steroid-resistant airway diseases. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/11343/208016.

Council of Science Editors:

Li M. A novel non-canonical pathway with potential for safer modulation of TGF-beta1 in steroid-resistant airway diseases. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/208016


University of Georgia

22. Lee, Intaek. Regulation of [beta]1 integrin function by aberrant N-linked glycosylation and Vacuolar H[superscript plus]-ATPase 16-kDa subunit expression.

Degree: 2014, University of Georgia

 Integrins are a family of cell surface glycoproteins that functions as cell adhesion receptors to the extracellular matrix (ECM). The interactions between integrins and ECM… (more)

Subjects/Keywords: N-acetylglucosaminyltransferase V; beta1 integrin; Extracellular matrix; Vacuolar H+-ATPase; 16-kDa subunit; neurite outgrowth.

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APA (6th Edition):

Lee, I. (2014). Regulation of [beta]1 integrin function by aberrant N-linked glycosylation and Vacuolar H[superscript plus]-ATPase 16-kDa subunit expression. (Thesis). University of Georgia. Retrieved from http://hdl.handle.net/10724/22428

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Intaek. “Regulation of [beta]1 integrin function by aberrant N-linked glycosylation and Vacuolar H[superscript plus]-ATPase 16-kDa subunit expression.” 2014. Thesis, University of Georgia. Accessed April 10, 2021. http://hdl.handle.net/10724/22428.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Intaek. “Regulation of [beta]1 integrin function by aberrant N-linked glycosylation and Vacuolar H[superscript plus]-ATPase 16-kDa subunit expression.” 2014. Web. 10 Apr 2021.

Vancouver:

Lee I. Regulation of [beta]1 integrin function by aberrant N-linked glycosylation and Vacuolar H[superscript plus]-ATPase 16-kDa subunit expression. [Internet] [Thesis]. University of Georgia; 2014. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10724/22428.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee I. Regulation of [beta]1 integrin function by aberrant N-linked glycosylation and Vacuolar H[superscript plus]-ATPase 16-kDa subunit expression. [Thesis]. University of Georgia; 2014. Available from: http://hdl.handle.net/10724/22428

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Ferreira, Roberto Rodrigues. Polimorfismo do gene de TGF-ß1 e a correlação com a susceptibilidade e progressão da Doença de Chagas.

Degree: 2014, Brazil

Made available in DSpace on 2015-10-23T12:45:32Z (GMT). No. of bitstreams: 2 robertoferreiraiocmest2014.pdf: 2709079 bytes, checksum: e93a9ead39b05b47e7d6166d6417e363 (MD5) license.txt: 1748 bytes, checksum: 8a4605be74aa9ea9d79846c1fba20a33 (MD5) Previous issue… (more)

Subjects/Keywords: Fator de Crescimento Transformador beta1; Polimorfismo Genético; Doença de Chagas; Cardiopatias; Trypanosoma cruzi

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APA (6th Edition):

Ferreira, R. R. (2014). Polimorfismo do gene de TGF-ß1 e a correlação com a susceptibilidade e progressão da Doença de Chagas. (Masters Thesis). Brazil. Retrieved from https://www.arca.fiocruz.br/handle/icict/12067

Chicago Manual of Style (16th Edition):

Ferreira, Roberto Rodrigues. “Polimorfismo do gene de TGF-ß1 e a correlação com a susceptibilidade e progressão da Doença de Chagas.” 2014. Masters Thesis, Brazil. Accessed April 10, 2021. https://www.arca.fiocruz.br/handle/icict/12067.

MLA Handbook (7th Edition):

Ferreira, Roberto Rodrigues. “Polimorfismo do gene de TGF-ß1 e a correlação com a susceptibilidade e progressão da Doença de Chagas.” 2014. Web. 10 Apr 2021.

Vancouver:

Ferreira RR. Polimorfismo do gene de TGF-ß1 e a correlação com a susceptibilidade e progressão da Doença de Chagas. [Internet] [Masters thesis]. Brazil; 2014. [cited 2021 Apr 10]. Available from: https://www.arca.fiocruz.br/handle/icict/12067.

Council of Science Editors:

Ferreira RR. Polimorfismo do gene de TGF-ß1 e a correlação com a susceptibilidade e progressão da Doença de Chagas. [Masters Thesis]. Brazil; 2014. Available from: https://www.arca.fiocruz.br/handle/icict/12067

24. Aloise, Antonio Carlos [UNIFESP]. TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno.

Degree: 2009, Universidade Federal de São Paulo (UNIFESP)

Made available in DSpace on 2015-07-22T20:50:51Z (GMT). No. of bitstreams: 0 Previous issue date: 2009-09-30

Introducao: A busca por uma fonte alternativa de celulas para… (more)

Subjects/Keywords: Colágeno; Diferenciacão celular; TGF-beta1; Fibroblastos

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Aloise, A. C. [. (2009). TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno. (Doctoral Dissertation). Universidade Federal de São Paulo (UNIFESP). Retrieved from http://repositorio.unifesp.br/handle/11600/10115

Chicago Manual of Style (16th Edition):

Aloise, Antonio Carlos [UNIFESP]. “TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno.” 2009. Doctoral Dissertation, Universidade Federal de São Paulo (UNIFESP). Accessed April 10, 2021. http://repositorio.unifesp.br/handle/11600/10115.

MLA Handbook (7th Edition):

Aloise, Antonio Carlos [UNIFESP]. “TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno.” 2009. Web. 10 Apr 2021.

Vancouver:

Aloise AC[. TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno. [Internet] [Doctoral dissertation]. Universidade Federal de São Paulo (UNIFESP); 2009. [cited 2021 Apr 10]. Available from: http://repositorio.unifesp.br/handle/11600/10115.

Council of Science Editors:

Aloise AC[. TGF-beta1 em fibroblastos dérmicos humanos cultivados em esponja de colágeno. [Doctoral Dissertation]. Universidade Federal de São Paulo (UNIFESP); 2009. Available from: http://repositorio.unifesp.br/handle/11600/10115

25. Jacobo, Andréia. Expressão imuno-histoquímica de TGF Β1 em pacientes com adenomiose.

Degree: 2016, Brazil

Introdução: Proteínas da Superfamília do fator transformador de crescimento β (TGF-β) estão implicadas na regulação de diversas funções biológicas. Embora alguns estudos revelaram a sua… (more)

Subjects/Keywords: Adenomiose; Fator de crescimento transformador beta1; Imuno-histoquímica; Endométrio; Adenomyosis; TGF-β1; Pathogenesis; Immunohistochemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jacobo, A. (2016). Expressão imuno-histoquímica de TGF Β1 em pacientes com adenomiose. (Masters Thesis). Brazil. Retrieved from http://hdl.handle.net/10183/150683

Chicago Manual of Style (16th Edition):

Jacobo, Andréia. “Expressão imuno-histoquímica de TGF Β1 em pacientes com adenomiose.” 2016. Masters Thesis, Brazil. Accessed April 10, 2021. http://hdl.handle.net/10183/150683.

MLA Handbook (7th Edition):

Jacobo, Andréia. “Expressão imuno-histoquímica de TGF Β1 em pacientes com adenomiose.” 2016. Web. 10 Apr 2021.

Vancouver:

Jacobo A. Expressão imuno-histoquímica de TGF Β1 em pacientes com adenomiose. [Internet] [Masters thesis]. Brazil; 2016. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10183/150683.

Council of Science Editors:

Jacobo A. Expressão imuno-histoquímica de TGF Β1 em pacientes com adenomiose. [Masters Thesis]. Brazil; 2016. Available from: http://hdl.handle.net/10183/150683


University of Delaware

26. Wang, Yichen. The mechanisms underlying ocular fibrotic diseases.

Degree: PhD, University of Delaware, Department of Biological Sciences, 2017, University of Delaware

 The normal function of the eye requires all key optical media (cornea, lens and retina) to be transparent. Disruption of any of these highly ordered… (more)

Subjects/Keywords: Biological sciences; Aniridia fibrosis syndrome (AFS); Beta1-integrin; Canonical wnt signaling; Egr1; Lens development; Posterior capsular opacification (PCO)

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APA (6th Edition):

Wang, Y. (2017). The mechanisms underlying ocular fibrotic diseases. (Doctoral Dissertation). University of Delaware. Retrieved from http://udspace.udel.edu/handle/19716/22917

Chicago Manual of Style (16th Edition):

Wang, Yichen. “The mechanisms underlying ocular fibrotic diseases.” 2017. Doctoral Dissertation, University of Delaware. Accessed April 10, 2021. http://udspace.udel.edu/handle/19716/22917.

MLA Handbook (7th Edition):

Wang, Yichen. “The mechanisms underlying ocular fibrotic diseases.” 2017. Web. 10 Apr 2021.

Vancouver:

Wang Y. The mechanisms underlying ocular fibrotic diseases. [Internet] [Doctoral dissertation]. University of Delaware; 2017. [cited 2021 Apr 10]. Available from: http://udspace.udel.edu/handle/19716/22917.

Council of Science Editors:

Wang Y. The mechanisms underlying ocular fibrotic diseases. [Doctoral Dissertation]. University of Delaware; 2017. Available from: http://udspace.udel.edu/handle/19716/22917


University of Southern California

27. Schmid, Daniela. Integrin expression and signaling during palatal fusion.

Degree: PhD, Cranio-Facial Biology, 2008, University of Southern California

 The fusion of the secondary palate is a complex event requiring epithelial and mesenchymal cell differentiation. Palatal fusion and mesenchymal confluence entail adhesion of the… (more)

Subjects/Keywords: palate development; integrin-linked kinase; beta1 integrin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Schmid, D. (2008). Integrin expression and signaling during palatal fusion. (Doctoral Dissertation). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/90105/rec/3557

Chicago Manual of Style (16th Edition):

Schmid, Daniela. “Integrin expression and signaling during palatal fusion.” 2008. Doctoral Dissertation, University of Southern California. Accessed April 10, 2021. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/90105/rec/3557.

MLA Handbook (7th Edition):

Schmid, Daniela. “Integrin expression and signaling during palatal fusion.” 2008. Web. 10 Apr 2021.

Vancouver:

Schmid D. Integrin expression and signaling during palatal fusion. [Internet] [Doctoral dissertation]. University of Southern California; 2008. [cited 2021 Apr 10]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/90105/rec/3557.

Council of Science Editors:

Schmid D. Integrin expression and signaling during palatal fusion. [Doctoral Dissertation]. University of Southern California; 2008. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll127/id/90105/rec/3557

28. Gazquez, Elodie. Études des interactions fonctionnelles entre l'endothéline-3, les intégrines beta1 et les propriétés élastiques du tissu embryonnaire au cours du développement du système nerveux entérique : Functional interactions between endotheline-3, beta1 integrines and the elastic properties of the embryonic gut tissu during enteric nervous system development.

Degree: Docteur es, Biologie Cellulaire et du Développement, 2016, Université Pierre et Marie Curie – Paris VI

Le système nerveux entérique (SNE) provient des cellules de crête neurale entériques (CCNEs) qui migrent au sein de l'intestin embryonnaire, prolifèrent et se différencient en… (more)

Subjects/Keywords: Cellules de crêtes neurales entérique; Endotheline-3; Integrine beta1; Migration; Biomécanique; Élasticité tissulaire; Neural crest cells; Migration; Enteric nervous system; 571.6

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APA (6th Edition):

Gazquez, E. (2016). Études des interactions fonctionnelles entre l'endothéline-3, les intégrines beta1 et les propriétés élastiques du tissu embryonnaire au cours du développement du système nerveux entérique : Functional interactions between endotheline-3, beta1 integrines and the elastic properties of the embryonic gut tissu during enteric nervous system development. (Doctoral Dissertation). Université Pierre et Marie Curie – Paris VI. Retrieved from http://www.theses.fr/2016PA066240

Chicago Manual of Style (16th Edition):

Gazquez, Elodie. “Études des interactions fonctionnelles entre l'endothéline-3, les intégrines beta1 et les propriétés élastiques du tissu embryonnaire au cours du développement du système nerveux entérique : Functional interactions between endotheline-3, beta1 integrines and the elastic properties of the embryonic gut tissu during enteric nervous system development.” 2016. Doctoral Dissertation, Université Pierre et Marie Curie – Paris VI. Accessed April 10, 2021. http://www.theses.fr/2016PA066240.

MLA Handbook (7th Edition):

Gazquez, Elodie. “Études des interactions fonctionnelles entre l'endothéline-3, les intégrines beta1 et les propriétés élastiques du tissu embryonnaire au cours du développement du système nerveux entérique : Functional interactions between endotheline-3, beta1 integrines and the elastic properties of the embryonic gut tissu during enteric nervous system development.” 2016. Web. 10 Apr 2021.

Vancouver:

Gazquez E. Études des interactions fonctionnelles entre l'endothéline-3, les intégrines beta1 et les propriétés élastiques du tissu embryonnaire au cours du développement du système nerveux entérique : Functional interactions between endotheline-3, beta1 integrines and the elastic properties of the embryonic gut tissu during enteric nervous system development. [Internet] [Doctoral dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. [cited 2021 Apr 10]. Available from: http://www.theses.fr/2016PA066240.

Council of Science Editors:

Gazquez E. Études des interactions fonctionnelles entre l'endothéline-3, les intégrines beta1 et les propriétés élastiques du tissu embryonnaire au cours du développement du système nerveux entérique : Functional interactions between endotheline-3, beta1 integrines and the elastic properties of the embryonic gut tissu during enteric nervous system development. [Doctoral Dissertation]. Université Pierre et Marie Curie – Paris VI; 2016. Available from: http://www.theses.fr/2016PA066240

29. Vaz, Emília Rezende. Desenvolvimento de peptideo bioativo modulador da resposta immune.

Degree: 2014, Federal University of Uberlândia

Autoimmune diseases are a group of different diseases which are characterized by an immune disorder leading to decreased tolerance to components of the body itself.… (more)

Subjects/Keywords: TGF-Beta1; Inflamação; Autoimunidade; TNF-α; IL-10; Peptídeos; Inflamação - Aspectos imunológicos; Inflammation; Autoimmunity; CNPQ::CIENCIAS BIOLOGICAS::GENETICA

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Vaz, E. R. (2014). Desenvolvimento de peptideo bioativo modulador da resposta immune. (Masters Thesis). Federal University of Uberlândia. Retrieved from https://repositorio.ufu.br/handle/123456789/15882

Chicago Manual of Style (16th Edition):

Vaz, Emília Rezende. “Desenvolvimento de peptideo bioativo modulador da resposta immune.” 2014. Masters Thesis, Federal University of Uberlândia. Accessed April 10, 2021. https://repositorio.ufu.br/handle/123456789/15882.

MLA Handbook (7th Edition):

Vaz, Emília Rezende. “Desenvolvimento de peptideo bioativo modulador da resposta immune.” 2014. Web. 10 Apr 2021.

Vancouver:

Vaz ER. Desenvolvimento de peptideo bioativo modulador da resposta immune. [Internet] [Masters thesis]. Federal University of Uberlândia; 2014. [cited 2021 Apr 10]. Available from: https://repositorio.ufu.br/handle/123456789/15882.

Council of Science Editors:

Vaz ER. Desenvolvimento de peptideo bioativo modulador da resposta immune. [Masters Thesis]. Federal University of Uberlândia; 2014. Available from: https://repositorio.ufu.br/handle/123456789/15882

30. Oliveira, Fernanda dos Santos de. A contribuição do fator transformador de crescimento beta 1 - TGF-ß1 na fibrose hepática : estudos in vivo e in vitro.

Degree: 2009, Brazil

A fibrose hepática é caracterizada pelo acúmulo de matriz extracelular em resposta à lesão hepática crônica. Importantes causas de lesões hepáticas crônicas são: hepatites virais,… (more)

Subjects/Keywords: Fator transformador de crescimento beta1; Cirrose hepática

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APA (6th Edition):

Oliveira, F. d. S. d. (2009). A contribuição do fator transformador de crescimento beta 1 - TGF-ß1 na fibrose hepática : estudos in vivo e in vitro. (Doctoral Dissertation). Brazil. Retrieved from http://hdl.handle.net/10183/17756

Chicago Manual of Style (16th Edition):

Oliveira, Fernanda dos Santos de. “A contribuição do fator transformador de crescimento beta 1 - TGF-ß1 na fibrose hepática : estudos in vivo e in vitro.” 2009. Doctoral Dissertation, Brazil. Accessed April 10, 2021. http://hdl.handle.net/10183/17756.

MLA Handbook (7th Edition):

Oliveira, Fernanda dos Santos de. “A contribuição do fator transformador de crescimento beta 1 - TGF-ß1 na fibrose hepática : estudos in vivo e in vitro.” 2009. Web. 10 Apr 2021.

Vancouver:

Oliveira FdSd. A contribuição do fator transformador de crescimento beta 1 - TGF-ß1 na fibrose hepática : estudos in vivo e in vitro. [Internet] [Doctoral dissertation]. Brazil; 2009. [cited 2021 Apr 10]. Available from: http://hdl.handle.net/10183/17756.

Council of Science Editors:

Oliveira FdSd. A contribuição do fator transformador de crescimento beta 1 - TGF-ß1 na fibrose hepática : estudos in vivo e in vitro. [Doctoral Dissertation]. Brazil; 2009. Available from: http://hdl.handle.net/10183/17756

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