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You searched for subject:(Beta cells). Showing records 1 – 30 of 478 total matches.

[1] [2] [3] [4] [5] … [16]

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McMaster University

1. Abdulla, Solen. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.

Degree: MSc, 2017, McMaster University

The Wnt/β-catenin pathway is a fundamental regulator of embryonic development and adult tissue homeostasis. The key effector, β-catenin, is a multifunctional protein that occupies dual… (more)

Subjects/Keywords: β-catenin; Stem Cells

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APA (6th Edition):

Abdulla, S. (2017). INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. (Masters Thesis). McMaster University. Retrieved from http://hdl.handle.net/11375/23461

Chicago Manual of Style (16th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Masters Thesis, McMaster University. Accessed April 14, 2021. http://hdl.handle.net/11375/23461.

MLA Handbook (7th Edition):

Abdulla, Solen. “INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL.” 2017. Web. 14 Apr 2021.

Vancouver:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Internet] [Masters thesis]. McMaster University; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/11375/23461.

Council of Science Editors:

Abdulla S. INVESTIGATING NOVEL β-CATENIN SIGNALLING MECHANISMS IN AN EMBRYONIC STEM CELL MODEL. [Masters Thesis]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/23461

2. ZAIATZ BITTENCOURT, VANESSA. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.

Degree: School of Biochemistry & Immunology. Discipline of Biochemistry, 2019, Trinity College Dublin

 It is now known that metabolism, in addition to providing energy and biochemical building blocks, also regulates immune cell function. Over the last few years,… (more)

Subjects/Keywords: immunometabolism; nk cells; tgf beta

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APA (6th Edition):

ZAIATZ BITTENCOURT, V. (2019). Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. (Thesis). Trinity College Dublin. Retrieved from http://hdl.handle.net/2262/85996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

ZAIATZ BITTENCOURT, VANESSA. “Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.” 2019. Thesis, Trinity College Dublin. Accessed April 14, 2021. http://hdl.handle.net/2262/85996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

ZAIATZ BITTENCOURT, VANESSA. “Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function.” 2019. Web. 14 Apr 2021.

Vancouver:

ZAIATZ BITTENCOURT V. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. [Internet] [Thesis]. Trinity College Dublin; 2019. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2262/85996.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

ZAIATZ BITTENCOURT V. Investigating the impact of natural killer (NK) cell metabolism on NK cell effector function. [Thesis]. Trinity College Dublin; 2019. Available from: http://hdl.handle.net/2262/85996

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Kellaway, Sophie. Stem cells from patients with Congenital Hyperinsulinism.

Degree: 2016, University of Manchester

 Diabetes and congenital hyperinsulinism (CHI) are severe diseases affecting the pancreas. Current models for testing drugs to treat these diseases are in vivo in rodents… (more)

Subjects/Keywords: stem cells; pancreas; beta cells; congenital hyperinsulinism

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APA (6th Edition):

Kellaway, S. (2016). Stem cells from patients with Congenital Hyperinsulinism. (Doctoral Dissertation). University of Manchester. Retrieved from http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305721

Chicago Manual of Style (16th Edition):

Kellaway, Sophie. “Stem cells from patients with Congenital Hyperinsulinism.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 14, 2021. http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305721.

MLA Handbook (7th Edition):

Kellaway, Sophie. “Stem cells from patients with Congenital Hyperinsulinism.” 2016. Web. 14 Apr 2021.

Vancouver:

Kellaway S. Stem cells from patients with Congenital Hyperinsulinism. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 14]. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305721.

Council of Science Editors:

Kellaway S. Stem cells from patients with Congenital Hyperinsulinism. [Doctoral Dissertation]. University of Manchester; 2016. Available from: http://www.manchester.ac.uk/escholar/uk-ac-man-scw:305721


University of Minnesota

4. Yang, Ying. Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA.

Degree: MS, Stem cell biology, 2012, University of Minnesota

University of Minnesota M.S. thesis. December 2012. Major: Stem cell biology. Advisor: Dr. Jonathan M.W.Slack. 1 computer file (PDF); vi, 54 pages.

The pancreas and… (more)

Subjects/Keywords: Beta cells; Cell reprogramming; Hepatic progenitor cells

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APA (6th Edition):

Yang, Y. (2012). Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA. (Masters Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/160346

Chicago Manual of Style (16th Edition):

Yang, Ying. “Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA.” 2012. Masters Thesis, University of Minnesota. Accessed April 14, 2021. http://purl.umn.edu/160346.

MLA Handbook (7th Edition):

Yang, Ying. “Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA.” 2012. Web. 14 Apr 2021.

Vancouver:

Yang Y. Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA. [Internet] [Masters thesis]. University of Minnesota; 2012. [cited 2021 Apr 14]. Available from: http://purl.umn.edu/160346.

Council of Science Editors:

Yang Y. Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA. [Masters Thesis]. University of Minnesota; 2012. Available from: http://purl.umn.edu/160346


University of Minnesota

5. Yang, Ying. Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA.

Degree: MS, Stem cell biology, 2012, University of Minnesota

 The pancreas and liver arises from adjacent areas in the anterior endoderm of the developing embryo. This close relatedness underlies the possibility of direct reprogramming… (more)

Subjects/Keywords: Beta cells; Cell reprogramming; Hepatic progenitor cells

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APA (6th Edition):

Yang, Y. (2012). Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA. (Masters Thesis). University of Minnesota. Retrieved from http://purl.umn.edu/160346

Chicago Manual of Style (16th Edition):

Yang, Ying. “Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA.” 2012. Masters Thesis, University of Minnesota. Accessed April 14, 2021. http://purl.umn.edu/160346.

MLA Handbook (7th Edition):

Yang, Ying. “Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA.” 2012. Web. 14 Apr 2021.

Vancouver:

Yang Y. Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA. [Internet] [Masters thesis]. University of Minnesota; 2012. [cited 2021 Apr 14]. Available from: http://purl.umn.edu/160346.

Council of Science Editors:

Yang Y. Reprogramming of hepatic progenitor cells towards a beta-cell character using Pdx1, Ngn3 and MafA. [Masters Thesis]. University of Minnesota; 2012. Available from: http://purl.umn.edu/160346


Vanderbilt University

6. Scoville, David William. Identification and characterization of MAFA coregulators: MLL3/4 and its role in mouse and human islet β-cells.

Degree: PhD, Cell and Developmental Biology, 2015, Vanderbilt University

 CELL AND DEVELOPMENTAL BIOLOGY Identification and characterization of MAFA coregulators: MLL3/4 and its role in mouse and human islet β-cells David William Scoville Dissertation under… (more)

Subjects/Keywords: MafA; MafB; coregulators; islet β-cells; pancreas

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APA (6th Edition):

Scoville, D. W. (2015). Identification and characterization of MAFA coregulators: MLL3/4 and its role in mouse and human islet β-cells. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/13141

Chicago Manual of Style (16th Edition):

Scoville, David William. “Identification and characterization of MAFA coregulators: MLL3/4 and its role in mouse and human islet β-cells.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 14, 2021. http://hdl.handle.net/1803/13141.

MLA Handbook (7th Edition):

Scoville, David William. “Identification and characterization of MAFA coregulators: MLL3/4 and its role in mouse and human islet β-cells.” 2015. Web. 14 Apr 2021.

Vancouver:

Scoville DW. Identification and characterization of MAFA coregulators: MLL3/4 and its role in mouse and human islet β-cells. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1803/13141.

Council of Science Editors:

Scoville DW. Identification and characterization of MAFA coregulators: MLL3/4 and its role in mouse and human islet β-cells. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/13141


University of Rochester

7. Lo, Chi-Wen. The Role of Serpin B13 Antibody in Regeneration of Pancreatic β-Cells.

Degree: PhD, 2020, University of Rochester

 Successful therapy for type 1 diabetes (T1D) requires both suppression of autoimmune inflammation and restoration of β-cell mass. Our previous work demonstrated that exposure to… (more)

Subjects/Keywords: Beta cells (β-cells); Cathepsin l; E-cadherin; Regenerating (reg) genes; Serpin b13; Type 1 diabetes

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APA (6th Edition):

Lo, C. (2020). The Role of Serpin B13 Antibody in Regeneration of Pancreatic β-Cells. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/35811

Chicago Manual of Style (16th Edition):

Lo, Chi-Wen. “The Role of Serpin B13 Antibody in Regeneration of Pancreatic β-Cells.” 2020. Doctoral Dissertation, University of Rochester. Accessed April 14, 2021. http://hdl.handle.net/1802/35811.

MLA Handbook (7th Edition):

Lo, Chi-Wen. “The Role of Serpin B13 Antibody in Regeneration of Pancreatic β-Cells.” 2020. Web. 14 Apr 2021.

Vancouver:

Lo C. The Role of Serpin B13 Antibody in Regeneration of Pancreatic β-Cells. [Internet] [Doctoral dissertation]. University of Rochester; 2020. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1802/35811.

Council of Science Editors:

Lo C. The Role of Serpin B13 Antibody in Regeneration of Pancreatic β-Cells. [Doctoral Dissertation]. University of Rochester; 2020. Available from: http://hdl.handle.net/1802/35811


Indian Institute of Science

8. Chopra, Sunita. Context Dependent Effects of the Transforming Growth Factor-beta Signaling and Role Played by WNT4 in the Activation of Fibroblasts.

Degree: PhD, Faculty of Science, 2018, Indian Institute of Science

 Transforming growth factor-β (TGF-β) superfamily of cytokines comprises of several members, which can broadly be sub-divided into three classes [TGF-βs, Activin/Nodal, and Bone morphogenetic proteins… (more)

Subjects/Keywords: Activation of Fibroblasts; Factor-beta Signaling; Fibroblast Cells; TGF-β; Fibroblasts; WNT4; Molecular Reproduction, Development and Genetics

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APA (6th Edition):

Chopra, S. (2018). Context Dependent Effects of the Transforming Growth Factor-beta Signaling and Role Played by WNT4 in the Activation of Fibroblasts. (Doctoral Dissertation). Indian Institute of Science. Retrieved from http://etd.iisc.ac.in/handle/2005/3882

Chicago Manual of Style (16th Edition):

Chopra, Sunita. “Context Dependent Effects of the Transforming Growth Factor-beta Signaling and Role Played by WNT4 in the Activation of Fibroblasts.” 2018. Doctoral Dissertation, Indian Institute of Science. Accessed April 14, 2021. http://etd.iisc.ac.in/handle/2005/3882.

MLA Handbook (7th Edition):

Chopra, Sunita. “Context Dependent Effects of the Transforming Growth Factor-beta Signaling and Role Played by WNT4 in the Activation of Fibroblasts.” 2018. Web. 14 Apr 2021.

Vancouver:

Chopra S. Context Dependent Effects of the Transforming Growth Factor-beta Signaling and Role Played by WNT4 in the Activation of Fibroblasts. [Internet] [Doctoral dissertation]. Indian Institute of Science; 2018. [cited 2021 Apr 14]. Available from: http://etd.iisc.ac.in/handle/2005/3882.

Council of Science Editors:

Chopra S. Context Dependent Effects of the Transforming Growth Factor-beta Signaling and Role Played by WNT4 in the Activation of Fibroblasts. [Doctoral Dissertation]. Indian Institute of Science; 2018. Available from: http://etd.iisc.ac.in/handle/2005/3882


Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

9. Gargani, Sofia. Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας.

Degree: 2013, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ)

Introduction: Under normal healthy conditions, organisms maintaina dynamic endocrinecell mass throughout life. Pancreatic beta cell massare able to maintain plasma glucose levels increasing insulin secretion… (more)

Subjects/Keywords: Μεταμόσχευση; Ανθρώπινα παγκρεατικά νησίδια; β παγκρεατικά κύτταρα; Παχυσαρκία; Transplantation; Human pancreatic islets; Beta pancreatic cells; Obesity

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APA (6th Edition):

Gargani, S. (2013). Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας. (Thesis). Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Retrieved from http://hdl.handle.net/10442/hedi/38796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gargani, Sofia. “Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας.” 2013. Thesis, Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ). Accessed April 14, 2021. http://hdl.handle.net/10442/hedi/38796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gargani, Sofia. “Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας.” 2013. Web. 14 Apr 2021.

Vancouver:

Gargani S. Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας. [Internet] [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10442/hedi/38796.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gargani S. Προσαρμοστικές αλλαγές μεταμοσχευμένων ανθρώπινων παγκρεατικών νησιδίων σε περιβάλλον παχυσαρκίας. [Thesis]. Aristotle University Of Thessaloniki (AUTH); Αριστοτέλειο Πανεπιστήμιο Θεσσαλονίκης (ΑΠΘ); 2013. Available from: http://hdl.handle.net/10442/hedi/38796

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vilnius Gediminas Technical University

10. Urbanavičiūtė, Laura. Beta-gliukanų poveikis pelių dendritinėms ląstelėms.

Degree: Master, Chemical Engineering, 2011, Vilnius Gediminas Technical University

Baigiamajame magistro darbe buvo ištirtas β-gliukanų poveikis pelių dendritinėms ląstelėms. Tyrimams buvo naudojami skirtingi Saccharomyces cerevisiae 1,3/ 1,6 β- gliukanų bandiniai βG1, βG2, βG3 ir… (more)

Subjects/Keywords: β-gliukanai; Dendritinės ląstelės; Fagocitozė; Proliferacija; Brendimas; β-glucans; Dendritic cells; Phagocytosis; Proliferation; Maturation

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APA (6th Edition):

Urbanavičiūtė, Laura. (2011). Beta-gliukanų poveikis pelių dendritinėms ląstelėms. (Masters Thesis). Vilnius Gediminas Technical University. Retrieved from http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20110621_170301-00534 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

Urbanavičiūtė, Laura. “Beta-gliukanų poveikis pelių dendritinėms ląstelėms.” 2011. Masters Thesis, Vilnius Gediminas Technical University. Accessed April 14, 2021. http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20110621_170301-00534 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

Urbanavičiūtė, Laura. “Beta-gliukanų poveikis pelių dendritinėms ląstelėms.” 2011. Web. 14 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

Urbanavičiūtė, Laura. Beta-gliukanų poveikis pelių dendritinėms ląstelėms. [Internet] [Masters thesis]. Vilnius Gediminas Technical University; 2011. [cited 2021 Apr 14]. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20110621_170301-00534 ;.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

Urbanavičiūtė, Laura. Beta-gliukanų poveikis pelių dendritinėms ląstelėms. [Masters Thesis]. Vilnius Gediminas Technical University; 2011. Available from: http://vddb.laba.lt/obj/LT-eLABa-0001:E.02~2011~D_20110621_170301-00534 ;

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Vanderbilt University

11. Conrad, Elizabeth Elrod. Examining the roles of MafB in the pancreatic islet.

Degree: PhD, Molecular Physiology and Biophysics, 2015, Vanderbilt University

 Analysis of MafB-/- mice suggested that this transcription factor was essential to islet α- and β-cell formation during development, although the postnatal physiological impact could… (more)

Subjects/Keywords: pancreas; islets; diabetes; beta cells; transcription factors

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APA (6th Edition):

Conrad, E. E. (2015). Examining the roles of MafB in the pancreatic islet. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14322

Chicago Manual of Style (16th Edition):

Conrad, Elizabeth Elrod. “Examining the roles of MafB in the pancreatic islet.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed April 14, 2021. http://hdl.handle.net/1803/14322.

MLA Handbook (7th Edition):

Conrad, Elizabeth Elrod. “Examining the roles of MafB in the pancreatic islet.” 2015. Web. 14 Apr 2021.

Vancouver:

Conrad EE. Examining the roles of MafB in the pancreatic islet. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1803/14322.

Council of Science Editors:

Conrad EE. Examining the roles of MafB in the pancreatic islet. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/14322


Penn State University

12. Bai, Yuting. ROLE OF TYROSINE KINASE ITK IN SKIN ALPHA BETA T CELLS .

Degree: 2011, Penn State University

 ABSTRACT Itk, a member of the Tec family of tyrosine kinases, plays an important role in T cell development and differentiation. Itk regulates the development… (more)

Subjects/Keywords: skin; alpha beta T cells; Itk

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APA (6th Edition):

Bai, Y. (2011). ROLE OF TYROSINE KINASE ITK IN SKIN ALPHA BETA T CELLS . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bai, Yuting. “ROLE OF TYROSINE KINASE ITK IN SKIN ALPHA BETA T CELLS .” 2011. Thesis, Penn State University. Accessed April 14, 2021. https://submit-etda.libraries.psu.edu/catalog/11968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bai, Yuting. “ROLE OF TYROSINE KINASE ITK IN SKIN ALPHA BETA T CELLS .” 2011. Web. 14 Apr 2021.

Vancouver:

Bai Y. ROLE OF TYROSINE KINASE ITK IN SKIN ALPHA BETA T CELLS . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Apr 14]. Available from: https://submit-etda.libraries.psu.edu/catalog/11968.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bai Y. ROLE OF TYROSINE KINASE ITK IN SKIN ALPHA BETA T CELLS . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11968

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Toronto

13. Desai, Tejas. Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction.

Degree: 2013, University of Toronto

Within the β-cell, there is evidence that sirtuin 1 (SIRT1), a key regulator of nutrient metabolism, plays a beneficial role on insulin secretion. Excess circulating… (more)

Subjects/Keywords: Lipotoxicity; Pancreatic Beta Cells; Sirtuins; 0719

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APA (6th Edition):

Desai, T. (2013). Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/69993

Chicago Manual of Style (16th Edition):

Desai, Tejas. “Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction.” 2013. Masters Thesis, University of Toronto. Accessed April 14, 2021. http://hdl.handle.net/1807/69993.

MLA Handbook (7th Edition):

Desai, Tejas. “Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction.” 2013. Web. 14 Apr 2021.

Vancouver:

Desai T. Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction. [Internet] [Masters thesis]. University of Toronto; 2013. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1807/69993.

Council of Science Editors:

Desai T. Exploring the Possibility of Changes in SIRT1 Activity in Lipotoxicity-mediated β-cell Dysfunction. [Masters Thesis]. University of Toronto; 2013. Available from: http://hdl.handle.net/1807/69993


University of Houston

14. -8465-3791. Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells.

Degree: PhD, Biology and Biochemistry, 2015, University of Houston

 Despite its slow development and our capacity for early detection using endoscopy, colorectal cancer remains the second leading cause of cancer death in the United… (more)

Subjects/Keywords: Estrogen receptor beta; Colon cancer epithelial cells

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APA (6th Edition):

-8465-3791. (2015). Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells. (Doctoral Dissertation). University of Houston. Retrieved from http://hdl.handle.net/10657/2018

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-8465-3791. “Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells.” 2015. Doctoral Dissertation, University of Houston. Accessed April 14, 2021. http://hdl.handle.net/10657/2018.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-8465-3791. “Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells.” 2015. Web. 14 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-8465-3791. Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells. [Internet] [Doctoral dissertation]. University of Houston; 2015. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/10657/2018.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-8465-3791. Defining Anti-Tumorigenic and Anti-Inflammatory Effects Mediated By Estrogen Receptor Beta In Colon Epithelial Cells. [Doctoral Dissertation]. University of Houston; 2015. Available from: http://hdl.handle.net/10657/2018

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Columbia University

15. Gonzalez, Bryan Jose. HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function.

Degree: 2019, Columbia University

 Mutations in HNF1A cause Maturity Onset Diabetes of the Young type 3, the second most frequent form of diabetes caused by single gene mutation. We… (more)

Subjects/Keywords: Cytology; Insulin; Pancreatic beta cells; Biology; Genes

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APA (6th Edition):

Gonzalez, B. J. (2019). HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/d8-d6v3-ca52

Chicago Manual of Style (16th Edition):

Gonzalez, Bryan Jose. “HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function.” 2019. Doctoral Dissertation, Columbia University. Accessed April 14, 2021. https://doi.org/10.7916/d8-d6v3-ca52.

MLA Handbook (7th Edition):

Gonzalez, Bryan Jose. “HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function.” 2019. Web. 14 Apr 2021.

Vancouver:

Gonzalez BJ. HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function. [Internet] [Doctoral dissertation]. Columbia University; 2019. [cited 2021 Apr 14]. Available from: https://doi.org/10.7916/d8-d6v3-ca52.

Council of Science Editors:

Gonzalez BJ. HNF1A Deficiency Impairs Beta-cell Fate, Granule Maturation and Function. [Doctoral Dissertation]. Columbia University; 2019. Available from: https://doi.org/10.7916/d8-d6v3-ca52


Columbia University

16. Fan, Jason Chen. Markers and Mechanisms of β-cell Dedifferentiation.

Degree: 2018, Columbia University

 Human and murine diabetes is characterized by pancreatic β-cell dedifferentiation, a process in which β-cells lose expression of markers of maturity and gain those of… (more)

Subjects/Keywords: Medicine; Biology; Cytology; Pancreatic beta cells

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APA (6th Edition):

Fan, J. C. (2018). Markers and Mechanisms of β-cell Dedifferentiation. (Doctoral Dissertation). Columbia University. Retrieved from https://doi.org/10.7916/D8RB8GGD

Chicago Manual of Style (16th Edition):

Fan, Jason Chen. “Markers and Mechanisms of β-cell Dedifferentiation.” 2018. Doctoral Dissertation, Columbia University. Accessed April 14, 2021. https://doi.org/10.7916/D8RB8GGD.

MLA Handbook (7th Edition):

Fan, Jason Chen. “Markers and Mechanisms of β-cell Dedifferentiation.” 2018. Web. 14 Apr 2021.

Vancouver:

Fan JC. Markers and Mechanisms of β-cell Dedifferentiation. [Internet] [Doctoral dissertation]. Columbia University; 2018. [cited 2021 Apr 14]. Available from: https://doi.org/10.7916/D8RB8GGD.

Council of Science Editors:

Fan JC. Markers and Mechanisms of β-cell Dedifferentiation. [Doctoral Dissertation]. Columbia University; 2018. Available from: https://doi.org/10.7916/D8RB8GGD


University of Manitoba

17. Lee, Jong Han. Prohibitin expression and function in ethanol treated pancreatic beta-cells.

Degree: Physiology, 2010, University of Manitoba

 Type 2 diabetes is now recognized as a worldwide epidemic. Pancreatic beta-cell decompensation in the presence of insulin resistance is a major mechanism for the… (more)

Subjects/Keywords: prohibitin; ethanol; beta-cells; oxidative stress

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APA (6th Edition):

Lee, J. H. (2010). Prohibitin expression and function in ethanol treated pancreatic beta-cells. (Thesis). University of Manitoba. Retrieved from http://hdl.handle.net/1993/4164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Jong Han. “Prohibitin expression and function in ethanol treated pancreatic beta-cells.” 2010. Thesis, University of Manitoba. Accessed April 14, 2021. http://hdl.handle.net/1993/4164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Jong Han. “Prohibitin expression and function in ethanol treated pancreatic beta-cells.” 2010. Web. 14 Apr 2021.

Vancouver:

Lee JH. Prohibitin expression and function in ethanol treated pancreatic beta-cells. [Internet] [Thesis]. University of Manitoba; 2010. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1993/4164.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee JH. Prohibitin expression and function in ethanol treated pancreatic beta-cells. [Thesis]. University of Manitoba; 2010. Available from: http://hdl.handle.net/1993/4164

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Université Catholique de Louvain

18. Stockis, Julie. Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells.

Degree: 2011, Université Catholique de Louvain

 Regulatory T cells (Tregs) are a subset of CD4+ T cells specialized in the inhibition of immune responses. Tregs are required for the maintenance of… (more)

Subjects/Keywords: Regulatory T cells; TGF-beta; Cancer immunotherapy

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APA (6th Edition):

Stockis, J. (2011). Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells. (Thesis). Université Catholique de Louvain. Retrieved from http://hdl.handle.net/2078.1/76134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Stockis, Julie. “Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells.” 2011. Thesis, Université Catholique de Louvain. Accessed April 14, 2021. http://hdl.handle.net/2078.1/76134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Stockis, Julie. “Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells.” 2011. Web. 14 Apr 2021.

Vancouver:

Stockis J. Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells. [Internet] [Thesis]. Université Catholique de Louvain; 2011. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2078.1/76134.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Stockis J. Activation of the latent form of Transforming Growth Factor-beta occurs at the surface of human regulatory T cells. [Thesis]. Université Catholique de Louvain; 2011. Available from: http://hdl.handle.net/2078.1/76134

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Oregon

19. Hill, Jennifer. Bacterial Regulation of Host Pancreatic Beta Cell Development.

Degree: PhD, Department of Biology, 2018, University of Oregon

 Diabetes is a metabolic disease characterized by the loss of functional pancreatic beta cells. The incidence of diabetes has risen rapidly in recent decades, which… (more)

Subjects/Keywords: BefA; Beta cells; Development; Diabetes; Microbiota; Zebrafish

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APA (6th Edition):

Hill, J. (2018). Bacterial Regulation of Host Pancreatic Beta Cell Development. (Doctoral Dissertation). University of Oregon. Retrieved from http://hdl.handle.net/1794/23140

Chicago Manual of Style (16th Edition):

Hill, Jennifer. “Bacterial Regulation of Host Pancreatic Beta Cell Development.” 2018. Doctoral Dissertation, University of Oregon. Accessed April 14, 2021. http://hdl.handle.net/1794/23140.

MLA Handbook (7th Edition):

Hill, Jennifer. “Bacterial Regulation of Host Pancreatic Beta Cell Development.” 2018. Web. 14 Apr 2021.

Vancouver:

Hill J. Bacterial Regulation of Host Pancreatic Beta Cell Development. [Internet] [Doctoral dissertation]. University of Oregon; 2018. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1794/23140.

Council of Science Editors:

Hill J. Bacterial Regulation of Host Pancreatic Beta Cell Development. [Doctoral Dissertation]. University of Oregon; 2018. Available from: http://hdl.handle.net/1794/23140


University of Minnesota

20. Atchison, Nicole Ann. Nanomaterial solutions for the protection of insulin producing beta cells.

Degree: PhD, Biomedical Engineering, 2013, University of Minnesota

 Islet transplantation is a promising treatment for type 1 diabetes. However, even with the many successes, islet transplantation has yet to reach its full potential.… (more)

Subjects/Keywords: ATP; Beta cells; Liposome; Nanomaterials; Peptides

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APA (6th Edition):

Atchison, N. A. (2013). Nanomaterial solutions for the protection of insulin producing beta cells. (Doctoral Dissertation). University of Minnesota. Retrieved from http://purl.umn.edu/161507

Chicago Manual of Style (16th Edition):

Atchison, Nicole Ann. “Nanomaterial solutions for the protection of insulin producing beta cells.” 2013. Doctoral Dissertation, University of Minnesota. Accessed April 14, 2021. http://purl.umn.edu/161507.

MLA Handbook (7th Edition):

Atchison, Nicole Ann. “Nanomaterial solutions for the protection of insulin producing beta cells.” 2013. Web. 14 Apr 2021.

Vancouver:

Atchison NA. Nanomaterial solutions for the protection of insulin producing beta cells. [Internet] [Doctoral dissertation]. University of Minnesota; 2013. [cited 2021 Apr 14]. Available from: http://purl.umn.edu/161507.

Council of Science Editors:

Atchison NA. Nanomaterial solutions for the protection of insulin producing beta cells. [Doctoral Dissertation]. University of Minnesota; 2013. Available from: http://purl.umn.edu/161507


University of Melbourne

21. Quah, Hong Sheng. Dissecting the interferon response required for triggering autoimmune diabetes.

Degree: 2016, University of Melbourne

 Type 1 diabetes (T1D) is characterized by the autoimmune destruction of β cells in the islets of Langerhans by immune cells. The interferon (IFN) family… (more)

Subjects/Keywords: type 1 diabetes; interferon; granzyme; beta cells

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APA (6th Edition):

Quah, H. S. (2016). Dissecting the interferon response required for triggering autoimmune diabetes. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/95518

Chicago Manual of Style (16th Edition):

Quah, Hong Sheng. “Dissecting the interferon response required for triggering autoimmune diabetes.” 2016. Doctoral Dissertation, University of Melbourne. Accessed April 14, 2021. http://hdl.handle.net/11343/95518.

MLA Handbook (7th Edition):

Quah, Hong Sheng. “Dissecting the interferon response required for triggering autoimmune diabetes.” 2016. Web. 14 Apr 2021.

Vancouver:

Quah HS. Dissecting the interferon response required for triggering autoimmune diabetes. [Internet] [Doctoral dissertation]. University of Melbourne; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/11343/95518.

Council of Science Editors:

Quah HS. Dissecting the interferon response required for triggering autoimmune diabetes. [Doctoral Dissertation]. University of Melbourne; 2016. Available from: http://hdl.handle.net/11343/95518


University of Aberdeen

22. Robertson, Lindsay. Characterisation of the MAL2 proteins and their interaction with TPD52.

Degree: School of Biological Sciences., 2006, University of Aberdeen

Subjects/Keywords: Pancreatic beta cells

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APA (6th Edition):

Robertson, L. (2006). Characterisation of the MAL2 proteins and their interaction with TPD52. (Doctoral Dissertation). University of Aberdeen. Retrieved from http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer

Chicago Manual of Style (16th Edition):

Robertson, Lindsay. “Characterisation of the MAL2 proteins and their interaction with TPD52.” 2006. Doctoral Dissertation, University of Aberdeen. Accessed April 14, 2021. http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer.

MLA Handbook (7th Edition):

Robertson, Lindsay. “Characterisation of the MAL2 proteins and their interaction with TPD52.” 2006. Web. 14 Apr 2021.

Vancouver:

Robertson L. Characterisation of the MAL2 proteins and their interaction with TPD52. [Internet] [Doctoral dissertation]. University of Aberdeen; 2006. [cited 2021 Apr 14]. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer.

Council of Science Editors:

Robertson L. Characterisation of the MAL2 proteins and their interaction with TPD52. [Doctoral Dissertation]. University of Aberdeen; 2006. Available from: http://digitool.abdn.ac.uk:80/webclient/DeliveryManager?application=DIGITOOL-3&owner=resourcediscovery&custom_att_2=simple_viewer&pid=185669 ; http://digitool.abdn.ac.uk:1801/webclient/DeliveryManager?pid=185669&custom_att_2=simple_viewer


Louisiana State University

23. Earpina, Srikanth. Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells.

Degree: MS, Life Sciences, 2013, Louisiana State University

 Diabetes is an inflammatory disease associated with hyperglycemia. Chronic exposure of pancreatic â-cells to glucolipotoxicity stimulates a low-grade inflammation associated with the release of pro-inflammatory… (more)

Subjects/Keywords: glucose; palmitic acid; pancreatic beta cells; inflammation

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APA (6th Edition):

Earpina, S. (2013). Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells. (Masters Thesis). Louisiana State University. Retrieved from etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770

Chicago Manual of Style (16th Edition):

Earpina, Srikanth. “Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells.” 2013. Masters Thesis, Louisiana State University. Accessed April 14, 2021. etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770.

MLA Handbook (7th Edition):

Earpina, Srikanth. “Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells.” 2013. Web. 14 Apr 2021.

Vancouver:

Earpina S. Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells. [Internet] [Masters thesis]. Louisiana State University; 2013. [cited 2021 Apr 14]. Available from: etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770.

Council of Science Editors:

Earpina S. Insulin Secretagogue Activity of Ellagic Acid-Rich Muscadine (Vitis Rotundifolia) and Indian Gooseberry (Emblica Officinalis) Extracts on Pancreatic Beta Cells. [Masters Thesis]. Louisiana State University; 2013. Available from: etd-11102013-230409 ; https://digitalcommons.lsu.edu/gradschool_theses/1770


Vanderbilt University

24. -9253-9449. THE ROLES OF TRANSCRIPTIONAL COREGULATOR SIN3 IN PANCREATIC β- CELL DIFFERENTIATION, FUNCTION, AND SURVIVAL.

Degree: PhD, Cell & Developmental Biology, 2020, Vanderbilt University

 Sin3a and Sin3b are paralogous transcriptional coregulators that direct cellular differentiation, survival, and function. Mouse Sin3a and Sin3b are co-produced in most pancreatic cells during… (more)

Subjects/Keywords: transcriptional coregulator; beta cells; differentiation; function; survival

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APA (6th Edition):

-9253-9449. (2020). THE ROLES OF TRANSCRIPTIONAL COREGULATOR SIN3 IN PANCREATIC β- CELL DIFFERENTIATION, FUNCTION, AND SURVIVAL. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/15942

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

-9253-9449. “THE ROLES OF TRANSCRIPTIONAL COREGULATOR SIN3 IN PANCREATIC β- CELL DIFFERENTIATION, FUNCTION, AND SURVIVAL.” 2020. Doctoral Dissertation, Vanderbilt University. Accessed April 14, 2021. http://hdl.handle.net/1803/15942.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

-9253-9449. “THE ROLES OF TRANSCRIPTIONAL COREGULATOR SIN3 IN PANCREATIC β- CELL DIFFERENTIATION, FUNCTION, AND SURVIVAL.” 2020. Web. 14 Apr 2021.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

-9253-9449. THE ROLES OF TRANSCRIPTIONAL COREGULATOR SIN3 IN PANCREATIC β- CELL DIFFERENTIATION, FUNCTION, AND SURVIVAL. [Internet] [Doctoral dissertation]. Vanderbilt University; 2020. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1803/15942.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

-9253-9449. THE ROLES OF TRANSCRIPTIONAL COREGULATOR SIN3 IN PANCREATIC β- CELL DIFFERENTIATION, FUNCTION, AND SURVIVAL. [Doctoral Dissertation]. Vanderbilt University; 2020. Available from: http://hdl.handle.net/1803/15942

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


University of Sydney

25. Farr, Ryan. Molecular Markers of Pancreatic β-cell Death .

Degree: 2017, University of Sydney

 Abstract Loss of insulin-producing β-cells is central to the development of Type 1 diabetes (T1D). Currently, we lack diagnostic tools to quantitate this β-cell loss.… (more)

Subjects/Keywords: microRNA; biomarkers; Type 1 diabetes; beta cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Farr, R. (2017). Molecular Markers of Pancreatic β-cell Death . (Thesis). University of Sydney. Retrieved from http://hdl.handle.net/2123/17308

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Farr, Ryan. “Molecular Markers of Pancreatic β-cell Death .” 2017. Thesis, University of Sydney. Accessed April 14, 2021. http://hdl.handle.net/2123/17308.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Farr, Ryan. “Molecular Markers of Pancreatic β-cell Death .” 2017. Web. 14 Apr 2021.

Vancouver:

Farr R. Molecular Markers of Pancreatic β-cell Death . [Internet] [Thesis]. University of Sydney; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/2123/17308.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Farr R. Molecular Markers of Pancreatic β-cell Death . [Thesis]. University of Sydney; 2017. Available from: http://hdl.handle.net/2123/17308

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of New South Wales

26. Pearson, Gemma. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.

Degree: Garvan Institute of Medical Research, 2014, University of New South Wales

 Glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells is vital for whole-bodyglucose homeostasis, and loss of β-cell function can result in type 2 diabetes(T2D), a major… (more)

Subjects/Keywords: Lipids; Pancreatic beta cells; Insulin secretion; Lysosomes

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APA (6th Edition):

Pearson, G. (2014). THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. (Doctoral Dissertation). University of New South Wales. Retrieved from http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true

Chicago Manual of Style (16th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Doctoral Dissertation, University of New South Wales. Accessed April 14, 2021. http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

MLA Handbook (7th Edition):

Pearson, Gemma. “THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS.” 2014. Web. 14 Apr 2021.

Vancouver:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Internet] [Doctoral dissertation]. University of New South Wales; 2014. [cited 2021 Apr 14]. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true.

Council of Science Editors:

Pearson G. THE ROLE(S) OF LIPID SPECIES IN GLUCOSE-STIMULATED INSULIN SECRETION FROM PANCREATIC β-­CELLS. [Doctoral Dissertation]. University of New South Wales; 2014. Available from: http://handle.unsw.edu.au/1959.4/53494 ; https://unsworks.unsw.edu.au/fapi/datastream/unsworks:12189/SOURCE02?view=true


Vanderbilt University

27. Clayton, Hannah Worchel. Variables that influence transcription factor-mediated acinar to beta cell reprogramming.

Degree: PhD, Cell and Developmental Biology, 2017, Vanderbilt University

 Reprogramming of pancreatic cells into new beta-like cells is a potential therapy for Type 1 diabetes. Pancreatic acinar cells are an appealing target for cellular… (more)

Subjects/Keywords: acinar-to-ductal metaplasia; Reprogramming; inflammation; beta cells; acinar cells; diabetes

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APA (6th Edition):

Clayton, H. W. (2017). Variables that influence transcription factor-mediated acinar to beta cell reprogramming. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/10597

Chicago Manual of Style (16th Edition):

Clayton, Hannah Worchel. “Variables that influence transcription factor-mediated acinar to beta cell reprogramming.” 2017. Doctoral Dissertation, Vanderbilt University. Accessed April 14, 2021. http://hdl.handle.net/1803/10597.

MLA Handbook (7th Edition):

Clayton, Hannah Worchel. “Variables that influence transcription factor-mediated acinar to beta cell reprogramming.” 2017. Web. 14 Apr 2021.

Vancouver:

Clayton HW. Variables that influence transcription factor-mediated acinar to beta cell reprogramming. [Internet] [Doctoral dissertation]. Vanderbilt University; 2017. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1803/10597.

Council of Science Editors:

Clayton HW. Variables that influence transcription factor-mediated acinar to beta cell reprogramming. [Doctoral Dissertation]. Vanderbilt University; 2017. Available from: http://hdl.handle.net/1803/10597


University of Toronto

28. Korytnikov, Roman. Role of Tankyrase Inhibitors in the Generation of NKX6-1+ Endoderm.

Degree: 2016, University of Toronto

Despite continuous research efforts, there is still no cure for type 1 diabetes (T1D). Generating human β-cells from human pluripotent stem cells holds an unprecedented… (more)

Subjects/Keywords: Beta cells; Diabetes; NKX6-1; Pancreas; Stem cells; Tankyrase; 0719

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APA (6th Edition):

Korytnikov, R. (2016). Role of Tankyrase Inhibitors in the Generation of NKX6-1+ Endoderm. (Masters Thesis). University of Toronto. Retrieved from http://hdl.handle.net/1807/92864

Chicago Manual of Style (16th Edition):

Korytnikov, Roman. “Role of Tankyrase Inhibitors in the Generation of NKX6-1+ Endoderm.” 2016. Masters Thesis, University of Toronto. Accessed April 14, 2021. http://hdl.handle.net/1807/92864.

MLA Handbook (7th Edition):

Korytnikov, Roman. “Role of Tankyrase Inhibitors in the Generation of NKX6-1+ Endoderm.” 2016. Web. 14 Apr 2021.

Vancouver:

Korytnikov R. Role of Tankyrase Inhibitors in the Generation of NKX6-1+ Endoderm. [Internet] [Masters thesis]. University of Toronto; 2016. [cited 2021 Apr 14]. Available from: http://hdl.handle.net/1807/92864.

Council of Science Editors:

Korytnikov R. Role of Tankyrase Inhibitors in the Generation of NKX6-1+ Endoderm. [Masters Thesis]. University of Toronto; 2016. Available from: http://hdl.handle.net/1807/92864


Harvard University

29. Rosado-Olivieri, Edwin. In Vitro Modeling of Human β Cell Differentiation, Regeneration and Function.

Degree: PhD, 2019, Harvard University

Stem cell-derived β cells offer an inexhaustible source of functional β cells for cell therapy and disease modeling for diabetes. We capitalize on the in… (more)

Subjects/Keywords: stem cells; beta cells; diabetes; developmental biology; organ regeneration

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Rosado-Olivieri, E. (2019). In Vitro Modeling of Human β Cell Differentiation, Regeneration and Function. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42013108

Chicago Manual of Style (16th Edition):

Rosado-Olivieri, Edwin. “In Vitro Modeling of Human β Cell Differentiation, Regeneration and Function.” 2019. Doctoral Dissertation, Harvard University. Accessed April 14, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42013108.

MLA Handbook (7th Edition):

Rosado-Olivieri, Edwin. “In Vitro Modeling of Human β Cell Differentiation, Regeneration and Function.” 2019. Web. 14 Apr 2021.

Vancouver:

Rosado-Olivieri E. In Vitro Modeling of Human β Cell Differentiation, Regeneration and Function. [Internet] [Doctoral dissertation]. Harvard University; 2019. [cited 2021 Apr 14]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42013108.

Council of Science Editors:

Rosado-Olivieri E. In Vitro Modeling of Human β Cell Differentiation, Regeneration and Function. [Doctoral Dissertation]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42013108

30. Kellaway, Sophie. Stem cells from patients with congenital hyperinsulinism.

Degree: PhD, 2016, University of Manchester

 Diabetes and congenital hyperinsulinism (CHI) are severe diseases affecting the pancreas. Current models for testing drugs to treat these diseases are in vivo in rodents… (more)

Subjects/Keywords: 616.4; stem cells; pancreas; beta cells; congenital hyperinsulinism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kellaway, S. (2016). Stem cells from patients with congenital hyperinsulinism. (Doctoral Dissertation). University of Manchester. Retrieved from https://www.research.manchester.ac.uk/portal/en/theses/stem-cells-from-patients-with-congenital-hyperinsulinism(24ad993f-9f5d-416d-a336-bf37227cc52a).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697809

Chicago Manual of Style (16th Edition):

Kellaway, Sophie. “Stem cells from patients with congenital hyperinsulinism.” 2016. Doctoral Dissertation, University of Manchester. Accessed April 14, 2021. https://www.research.manchester.ac.uk/portal/en/theses/stem-cells-from-patients-with-congenital-hyperinsulinism(24ad993f-9f5d-416d-a336-bf37227cc52a).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697809.

MLA Handbook (7th Edition):

Kellaway, Sophie. “Stem cells from patients with congenital hyperinsulinism.” 2016. Web. 14 Apr 2021.

Vancouver:

Kellaway S. Stem cells from patients with congenital hyperinsulinism. [Internet] [Doctoral dissertation]. University of Manchester; 2016. [cited 2021 Apr 14]. Available from: https://www.research.manchester.ac.uk/portal/en/theses/stem-cells-from-patients-with-congenital-hyperinsulinism(24ad993f-9f5d-416d-a336-bf37227cc52a).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697809.

Council of Science Editors:

Kellaway S. Stem cells from patients with congenital hyperinsulinism. [Doctoral Dissertation]. University of Manchester; 2016. Available from: https://www.research.manchester.ac.uk/portal/en/theses/stem-cells-from-patients-with-congenital-hyperinsulinism(24ad993f-9f5d-416d-a336-bf37227cc52a).html ; http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.697809

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