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You searched for subject:(Bcl xL). Showing records 1 – 30 of 79 total matches.

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1. Bessou, Margaux. Contribution de la forme mitochondriale de Bcl-xL dans le contrôle de la migration cellulaire : Role of mitochondrial Bcl-xL in the control of cell migration.

Degree: Docteur es, Biologie cellulaire, 2017, Lyon

Les protéines de la famille Bcl-2 sont les principaux régulateurs de la mort cellulaire par apoptose. Au sein de cette famille, la protéine Bcl-xL appartient… (more)

Subjects/Keywords: Protéines Bcl-2; Migration; Mitochondrie; Cancer; Calcium; Bcl-xL; Bcl-2 proteins; Migration; Mitochondria; Cancer; Calcium; Bcl-xL; 571.6

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bessou, M. (2017). Contribution de la forme mitochondriale de Bcl-xL dans le contrôle de la migration cellulaire : Role of mitochondrial Bcl-xL in the control of cell migration. (Doctoral Dissertation). Lyon. Retrieved from http://www.theses.fr/2017LYSE1104

Chicago Manual of Style (16th Edition):

Bessou, Margaux. “Contribution de la forme mitochondriale de Bcl-xL dans le contrôle de la migration cellulaire : Role of mitochondrial Bcl-xL in the control of cell migration.” 2017. Doctoral Dissertation, Lyon. Accessed January 28, 2021. http://www.theses.fr/2017LYSE1104.

MLA Handbook (7th Edition):

Bessou, Margaux. “Contribution de la forme mitochondriale de Bcl-xL dans le contrôle de la migration cellulaire : Role of mitochondrial Bcl-xL in the control of cell migration.” 2017. Web. 28 Jan 2021.

Vancouver:

Bessou M. Contribution de la forme mitochondriale de Bcl-xL dans le contrôle de la migration cellulaire : Role of mitochondrial Bcl-xL in the control of cell migration. [Internet] [Doctoral dissertation]. Lyon; 2017. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2017LYSE1104.

Council of Science Editors:

Bessou M. Contribution de la forme mitochondriale de Bcl-xL dans le contrôle de la migration cellulaire : Role of mitochondrial Bcl-xL in the control of cell migration. [Doctoral Dissertation]. Lyon; 2017. Available from: http://www.theses.fr/2017LYSE1104

2. Oh, Victor Hyun. EXPLORING THE EFFECTS OF SODIUM BUTYRATE ON CELL MORPHOLOGY, CELL GROWTH, AND EXPRESSION OF A RECOMBINANT ANTI-APOPTOTIC VENUS–BCL-XL FUSION PROTEIN IN GREEN MICROALGAE CHLAMYDOMONAS REINHARDTII.

Degree: 2014, Johns Hopkins University

 Green eukaryotic microalgae are an attractive economic, and efficient, alternative to conventional recombinant protein expression systems and have the potential to revolutionize the biotechnology industry.… (more)

Subjects/Keywords: sodium butyrate; chlamydomonas; recombinant; bcl-xL

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APA (6th Edition):

Oh, V. H. (2014). EXPLORING THE EFFECTS OF SODIUM BUTYRATE ON CELL MORPHOLOGY, CELL GROWTH, AND EXPRESSION OF A RECOMBINANT ANTI-APOPTOTIC VENUS–BCL-XL FUSION PROTEIN IN GREEN MICROALGAE CHLAMYDOMONAS REINHARDTII. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/37085

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oh, Victor Hyun. “EXPLORING THE EFFECTS OF SODIUM BUTYRATE ON CELL MORPHOLOGY, CELL GROWTH, AND EXPRESSION OF A RECOMBINANT ANTI-APOPTOTIC VENUS–BCL-XL FUSION PROTEIN IN GREEN MICROALGAE CHLAMYDOMONAS REINHARDTII.” 2014. Thesis, Johns Hopkins University. Accessed January 28, 2021. http://jhir.library.jhu.edu/handle/1774.2/37085.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oh, Victor Hyun. “EXPLORING THE EFFECTS OF SODIUM BUTYRATE ON CELL MORPHOLOGY, CELL GROWTH, AND EXPRESSION OF A RECOMBINANT ANTI-APOPTOTIC VENUS–BCL-XL FUSION PROTEIN IN GREEN MICROALGAE CHLAMYDOMONAS REINHARDTII.” 2014. Web. 28 Jan 2021.

Vancouver:

Oh VH. EXPLORING THE EFFECTS OF SODIUM BUTYRATE ON CELL MORPHOLOGY, CELL GROWTH, AND EXPRESSION OF A RECOMBINANT ANTI-APOPTOTIC VENUS–BCL-XL FUSION PROTEIN IN GREEN MICROALGAE CHLAMYDOMONAS REINHARDTII. [Internet] [Thesis]. Johns Hopkins University; 2014. [cited 2021 Jan 28]. Available from: http://jhir.library.jhu.edu/handle/1774.2/37085.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oh VH. EXPLORING THE EFFECTS OF SODIUM BUTYRATE ON CELL MORPHOLOGY, CELL GROWTH, AND EXPRESSION OF A RECOMBINANT ANTI-APOPTOTIC VENUS–BCL-XL FUSION PROTEIN IN GREEN MICROALGAE CHLAMYDOMONAS REINHARDTII. [Thesis]. Johns Hopkins University; 2014. Available from: http://jhir.library.jhu.edu/handle/1774.2/37085

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Karafin, Teele. Etude de la fonction de Translationally Controlled Tumor Protein (TCTP) dans différents modèles génétiques dans la souris : Functional Study of Translationally Controlled Tumor Protein (TCTP) in Different Murine Genetic Models.

Degree: Docteur es, Aspects moléculaires et cellulaires de la biologie, 2016, Université Paris-Saclay (ComUE)

TCTP est une protéine de 20 kDa que l’on retrouve souvent sous forme de dimère. Elle est fortement conservée dans la phylogénie et on la… (more)

Subjects/Keywords: Tctp; P53; Bcl-XL; Apoptose; Réversion tumorale; Tctp; P53; Bcl-XL; Apoptosis; Tumour reversion

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APA (6th Edition):

Karafin, T. (2016). Etude de la fonction de Translationally Controlled Tumor Protein (TCTP) dans différents modèles génétiques dans la souris : Functional Study of Translationally Controlled Tumor Protein (TCTP) in Different Murine Genetic Models. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2016SACLS211

Chicago Manual of Style (16th Edition):

Karafin, Teele. “Etude de la fonction de Translationally Controlled Tumor Protein (TCTP) dans différents modèles génétiques dans la souris : Functional Study of Translationally Controlled Tumor Protein (TCTP) in Different Murine Genetic Models.” 2016. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 28, 2021. http://www.theses.fr/2016SACLS211.

MLA Handbook (7th Edition):

Karafin, Teele. “Etude de la fonction de Translationally Controlled Tumor Protein (TCTP) dans différents modèles génétiques dans la souris : Functional Study of Translationally Controlled Tumor Protein (TCTP) in Different Murine Genetic Models.” 2016. Web. 28 Jan 2021.

Vancouver:

Karafin T. Etude de la fonction de Translationally Controlled Tumor Protein (TCTP) dans différents modèles génétiques dans la souris : Functional Study of Translationally Controlled Tumor Protein (TCTP) in Different Murine Genetic Models. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2016. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2016SACLS211.

Council of Science Editors:

Karafin T. Etude de la fonction de Translationally Controlled Tumor Protein (TCTP) dans différents modèles génétiques dans la souris : Functional Study of Translationally Controlled Tumor Protein (TCTP) in Different Murine Genetic Models. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2016. Available from: http://www.theses.fr/2016SACLS211

4. Rivière, Étienne. Implication de la protéine Bcl-xL dans la mégacaryopoïèse humaine normale et dans le purpura thrombopénique immunologique chronique : Involvement of Bcl-xL in human normal megakaryopoiesis and in chronic immune thrombocytopenia.

Degree: Docteur es, Biologie cellulaire et physiopathologie, 2015, Bordeaux

La protéine Bcl-xL fait partie de la famille des protéines anti-apoptotiques Bcl-2. Il a été montré que cette protéine avait un rôle majeur dans la… (more)

Subjects/Keywords: Mégacaryopoïèse; Apoptose; Purpura thrombopénique immunologique chronique; Bcl-xL; Megakaryopoiesis; Apoptosis; Chronic immune thrombocytopenia; Bcl-xL

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APA (6th Edition):

Rivière, . (2015). Implication de la protéine Bcl-xL dans la mégacaryopoïèse humaine normale et dans le purpura thrombopénique immunologique chronique : Involvement of Bcl-xL in human normal megakaryopoiesis and in chronic immune thrombocytopenia. (Doctoral Dissertation). Bordeaux. Retrieved from http://www.theses.fr/2015BORD0148

Chicago Manual of Style (16th Edition):

Rivière, Étienne. “Implication de la protéine Bcl-xL dans la mégacaryopoïèse humaine normale et dans le purpura thrombopénique immunologique chronique : Involvement of Bcl-xL in human normal megakaryopoiesis and in chronic immune thrombocytopenia.” 2015. Doctoral Dissertation, Bordeaux. Accessed January 28, 2021. http://www.theses.fr/2015BORD0148.

MLA Handbook (7th Edition):

Rivière, Étienne. “Implication de la protéine Bcl-xL dans la mégacaryopoïèse humaine normale et dans le purpura thrombopénique immunologique chronique : Involvement of Bcl-xL in human normal megakaryopoiesis and in chronic immune thrombocytopenia.” 2015. Web. 28 Jan 2021.

Vancouver:

Rivière . Implication de la protéine Bcl-xL dans la mégacaryopoïèse humaine normale et dans le purpura thrombopénique immunologique chronique : Involvement of Bcl-xL in human normal megakaryopoiesis and in chronic immune thrombocytopenia. [Internet] [Doctoral dissertation]. Bordeaux; 2015. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2015BORD0148.

Council of Science Editors:

Rivière . Implication de la protéine Bcl-xL dans la mégacaryopoïèse humaine normale et dans le purpura thrombopénique immunologique chronique : Involvement of Bcl-xL in human normal megakaryopoiesis and in chronic immune thrombocytopenia. [Doctoral Dissertation]. Bordeaux; 2015. Available from: http://www.theses.fr/2015BORD0148


NSYSU

5. Chang Chien, Jung-Jung. Effect of quinacrine on Bcl-xL and Bcl-2 expression in human leukemia K562 cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

 Quinacrine, also known as mepacrine, is a drug applied on anti-protozoal and anti-rheumatic therapy. Moreover, quinacrine functioned as an intrapleural sclerosing agent as well. Although… (more)

Subjects/Keywords: Quinacrine; Chronic myeloid leukemia; Bcl-xL; Bcl-2; apoptosis

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APA (6th Edition):

Chang Chien, J. (2014). Effect of quinacrine on Bcl-xL and Bcl-2 expression in human leukemia K562 cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617114-144613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chang Chien, Jung-Jung. “Effect of quinacrine on Bcl-xL and Bcl-2 expression in human leukemia K562 cells.” 2014. Thesis, NSYSU. Accessed January 28, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617114-144613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chang Chien, Jung-Jung. “Effect of quinacrine on Bcl-xL and Bcl-2 expression in human leukemia K562 cells.” 2014. Web. 28 Jan 2021.

Vancouver:

Chang Chien J. Effect of quinacrine on Bcl-xL and Bcl-2 expression in human leukemia K562 cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 28]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617114-144613.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chang Chien J. Effect of quinacrine on Bcl-xL and Bcl-2 expression in human leukemia K562 cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0617114-144613

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Vanderbilt University

6. Adams, Clare Marie. The contribution of miRNA biogenesis and Myc-regulated miRNA in apoptosis and tumorigenesis.

Degree: PhD, Pathology, 2015, Vanderbilt University

 microRNA (miRNA) are critical mediators of cellular signaling and regulate most biological processes. Thus, it is not surprising that miRNA dysregulation significantly contributes to tumorigenesis.… (more)

Subjects/Keywords: p53; miRNA; Dicer; Myc; HDAC; Bcl-2; Bcl-xL; apoptosis; tumorigenesis

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APA (6th Edition):

Adams, C. M. (2015). The contribution of miRNA biogenesis and Myc-regulated miRNA in apoptosis and tumorigenesis. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/14501

Chicago Manual of Style (16th Edition):

Adams, Clare Marie. “The contribution of miRNA biogenesis and Myc-regulated miRNA in apoptosis and tumorigenesis.” 2015. Doctoral Dissertation, Vanderbilt University. Accessed January 28, 2021. http://hdl.handle.net/1803/14501.

MLA Handbook (7th Edition):

Adams, Clare Marie. “The contribution of miRNA biogenesis and Myc-regulated miRNA in apoptosis and tumorigenesis.” 2015. Web. 28 Jan 2021.

Vancouver:

Adams CM. The contribution of miRNA biogenesis and Myc-regulated miRNA in apoptosis and tumorigenesis. [Internet] [Doctoral dissertation]. Vanderbilt University; 2015. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/1803/14501.

Council of Science Editors:

Adams CM. The contribution of miRNA biogenesis and Myc-regulated miRNA in apoptosis and tumorigenesis. [Doctoral Dissertation]. Vanderbilt University; 2015. Available from: http://hdl.handle.net/1803/14501


University of Louisville

7. Eno, Colins Ogwandi, 1982-. The role of endogenous Bcl-xL in regulation of apoptotic signaling pathways.

Degree: PhD, 2012, University of Louisville

Bcl-2 proteins are major regulators of cellular responses to various apoptotic stimuli. Among them, overexpression of the anti-apoptotic BcI-2 protein BcI-xL modulates organelle-specific apoptotic pathways.… (more)

Subjects/Keywords: Apoptosis; oxidative stress Bcl-2; outer mitochondria membrane; Bcl-xL

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APA (6th Edition):

Eno, Colins Ogwandi, 1. (2012). The role of endogenous Bcl-xL in regulation of apoptotic signaling pathways. (Doctoral Dissertation). University of Louisville. Retrieved from 10.18297/etd/408 ; https://ir.library.louisville.edu/etd/408

Chicago Manual of Style (16th Edition):

Eno, Colins Ogwandi, 1982-. “The role of endogenous Bcl-xL in regulation of apoptotic signaling pathways.” 2012. Doctoral Dissertation, University of Louisville. Accessed January 28, 2021. 10.18297/etd/408 ; https://ir.library.louisville.edu/etd/408.

MLA Handbook (7th Edition):

Eno, Colins Ogwandi, 1982-. “The role of endogenous Bcl-xL in regulation of apoptotic signaling pathways.” 2012. Web. 28 Jan 2021.

Vancouver:

Eno, Colins Ogwandi 1. The role of endogenous Bcl-xL in regulation of apoptotic signaling pathways. [Internet] [Doctoral dissertation]. University of Louisville; 2012. [cited 2021 Jan 28]. Available from: 10.18297/etd/408 ; https://ir.library.louisville.edu/etd/408.

Council of Science Editors:

Eno, Colins Ogwandi 1. The role of endogenous Bcl-xL in regulation of apoptotic signaling pathways. [Doctoral Dissertation]. University of Louisville; 2012. Available from: 10.18297/etd/408 ; https://ir.library.louisville.edu/etd/408

8. EL Dhaybi, Mohamad. Déterminants moléculaires non-apoptotiques de l'activité oncogénique de Bcl-xL : rôle de la monodéamidation de Bcl-xL : Non apoptotic molecular of oncogenic activity of Bcl-xL : role of Bcl-xL monodeamidation.

Degree: Docteur es, Cancérologie, 2017, Limoges

Bcl-xL est un oncogène surexprimé dans plusieurs types de cancers et qui joue un rôle important dans la survie cellulaire en régulant deux processus: l'apoptose… (more)

Subjects/Keywords: Bcl-xL; Autophagie; Apoptose; Cancer; Modification post-traductionnelle; Bcl-xL; Autophagy; Apoptosis; Cancer; Post-translational modification; 616.994

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APA (6th Edition):

EL Dhaybi, M. (2017). Déterminants moléculaires non-apoptotiques de l'activité oncogénique de Bcl-xL : rôle de la monodéamidation de Bcl-xL : Non apoptotic molecular of oncogenic activity of Bcl-xL : role of Bcl-xL monodeamidation. (Doctoral Dissertation). Limoges. Retrieved from http://www.theses.fr/2017LIMO0034

Chicago Manual of Style (16th Edition):

EL Dhaybi, Mohamad. “Déterminants moléculaires non-apoptotiques de l'activité oncogénique de Bcl-xL : rôle de la monodéamidation de Bcl-xL : Non apoptotic molecular of oncogenic activity of Bcl-xL : role of Bcl-xL monodeamidation.” 2017. Doctoral Dissertation, Limoges. Accessed January 28, 2021. http://www.theses.fr/2017LIMO0034.

MLA Handbook (7th Edition):

EL Dhaybi, Mohamad. “Déterminants moléculaires non-apoptotiques de l'activité oncogénique de Bcl-xL : rôle de la monodéamidation de Bcl-xL : Non apoptotic molecular of oncogenic activity of Bcl-xL : role of Bcl-xL monodeamidation.” 2017. Web. 28 Jan 2021.

Vancouver:

EL Dhaybi M. Déterminants moléculaires non-apoptotiques de l'activité oncogénique de Bcl-xL : rôle de la monodéamidation de Bcl-xL : Non apoptotic molecular of oncogenic activity of Bcl-xL : role of Bcl-xL monodeamidation. [Internet] [Doctoral dissertation]. Limoges; 2017. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2017LIMO0034.

Council of Science Editors:

EL Dhaybi M. Déterminants moléculaires non-apoptotiques de l'activité oncogénique de Bcl-xL : rôle de la monodéamidation de Bcl-xL : Non apoptotic molecular of oncogenic activity of Bcl-xL : role of Bcl-xL monodeamidation. [Doctoral Dissertation]. Limoges; 2017. Available from: http://www.theses.fr/2017LIMO0034

9. Renault, Thibaud. Régulations de la protéine proapoptotique Bax : rôle des kinases Akt et GSK-3β et de la protéine antiapoptotique Bcl-xL : Regulations of proapoptotic protein Bax : role of Akt and GSK-3β kinases and of antiapoptotic protein Bcl-xL.

Degree: Docteur es, Sciences, technologie, santé. Biochimie, 2010, Université de Bordeaux Segalen

La protéine proapoptotique Bax joue un rôle fondamental au cours de la voie intrinsèque de l’apoptose. Elle participe au déclenchement de la mort en permettant… (more)

Subjects/Keywords: Bax; Apoptose; Mitochondrie; Akt; GSK-3β; Bcl-xL; Bax; Apoptosis; Mitochondria; Akt; GSK-3β; Bcl-xL

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APA (6th Edition):

Renault, T. (2010). Régulations de la protéine proapoptotique Bax : rôle des kinases Akt et GSK-3β et de la protéine antiapoptotique Bcl-xL : Regulations of proapoptotic protein Bax : role of Akt and GSK-3β kinases and of antiapoptotic protein Bcl-xL. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2010BOR21794

Chicago Manual of Style (16th Edition):

Renault, Thibaud. “Régulations de la protéine proapoptotique Bax : rôle des kinases Akt et GSK-3β et de la protéine antiapoptotique Bcl-xL : Regulations of proapoptotic protein Bax : role of Akt and GSK-3β kinases and of antiapoptotic protein Bcl-xL.” 2010. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed January 28, 2021. http://www.theses.fr/2010BOR21794.

MLA Handbook (7th Edition):

Renault, Thibaud. “Régulations de la protéine proapoptotique Bax : rôle des kinases Akt et GSK-3β et de la protéine antiapoptotique Bcl-xL : Regulations of proapoptotic protein Bax : role of Akt and GSK-3β kinases and of antiapoptotic protein Bcl-xL.” 2010. Web. 28 Jan 2021.

Vancouver:

Renault T. Régulations de la protéine proapoptotique Bax : rôle des kinases Akt et GSK-3β et de la protéine antiapoptotique Bcl-xL : Regulations of proapoptotic protein Bax : role of Akt and GSK-3β kinases and of antiapoptotic protein Bcl-xL. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2010. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2010BOR21794.

Council of Science Editors:

Renault T. Régulations de la protéine proapoptotique Bax : rôle des kinases Akt et GSK-3β et de la protéine antiapoptotique Bcl-xL : Regulations of proapoptotic protein Bax : role of Akt and GSK-3β kinases and of antiapoptotic protein Bcl-xL. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2010. Available from: http://www.theses.fr/2010BOR21794


Université Paris-Sud – Paris XI

10. Thébault, Stéphanie. Etude des complexes entre TCTP (Translationally Controlled Tumor Protein) et ses partenaires : study of complexes involving TCTP (Translationally Controlled Tumor Protein).

Degree: Docteur es, Cancérologie, 2013, Université Paris-Sud – Paris XI

La thématique du laboratoire de l’équipe d’Adam Telerman porte sur la réversion tumorale, un processus rare au cours duquel les cellules cancéreuses perdent leur phénotype… (more)

Subjects/Keywords: Réversion tumorale; Apoptose; TCTP; Mcl-1 et Bcl-xl; Tumor reversion; Apoptosis; TCTP; Mcl-1 and Bcl-xl

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APA (6th Edition):

Thébault, S. (2013). Etude des complexes entre TCTP (Translationally Controlled Tumor Protein) et ses partenaires : study of complexes involving TCTP (Translationally Controlled Tumor Protein). (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2013PA11T024

Chicago Manual of Style (16th Edition):

Thébault, Stéphanie. “Etude des complexes entre TCTP (Translationally Controlled Tumor Protein) et ses partenaires : study of complexes involving TCTP (Translationally Controlled Tumor Protein).” 2013. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed January 28, 2021. http://www.theses.fr/2013PA11T024.

MLA Handbook (7th Edition):

Thébault, Stéphanie. “Etude des complexes entre TCTP (Translationally Controlled Tumor Protein) et ses partenaires : study of complexes involving TCTP (Translationally Controlled Tumor Protein).” 2013. Web. 28 Jan 2021.

Vancouver:

Thébault S. Etude des complexes entre TCTP (Translationally Controlled Tumor Protein) et ses partenaires : study of complexes involving TCTP (Translationally Controlled Tumor Protein). [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2013. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2013PA11T024.

Council of Science Editors:

Thébault S. Etude des complexes entre TCTP (Translationally Controlled Tumor Protein) et ses partenaires : study of complexes involving TCTP (Translationally Controlled Tumor Protein). [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2013. Available from: http://www.theses.fr/2013PA11T024

11. Faustino, Viviane Dias. Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência.

Degree: Mestrado, Distúrbios do Crescimento Celular, Hemodinâmicos e da Hemostasia, 2012, University of São Paulo

As estatísticas relacionadas aos cânceres hematológicos indicam que a incidência e mortalidade dessas doenças têm aumentado ao longo dos anos. Embora a maioria dos casos… (more)

Subjects/Keywords: Apoptose; Apoptosis; Bcl-2 gene; Gene therapy; Genes Bcl-2; Inibição simultânea; Interferência de RNA; Neoplasias; Neoplasms; Protein Bcl-XL; Proteína Bcl-XL; RNA interference; Simultaneous inhibition; Terapia de genes

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APA (6th Edition):

Faustino, V. D. (2012). Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência. (Masters Thesis). University of São Paulo. Retrieved from http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;

Chicago Manual of Style (16th Edition):

Faustino, Viviane Dias. “Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência.” 2012. Masters Thesis, University of São Paulo. Accessed January 28, 2021. http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;.

MLA Handbook (7th Edition):

Faustino, Viviane Dias. “Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência.” 2012. Web. 28 Jan 2021.

Vancouver:

Faustino VD. Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência. [Internet] [Masters thesis]. University of São Paulo; 2012. [cited 2021 Jan 28]. Available from: http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;.

Council of Science Editors:

Faustino VD. Inibição simultânea dos genes antiapoptóticos Bcl-2 e Bcl-XL em células de leucemia  linfoide aguda e células de linfoma do manto mediante RNA de interferência. [Masters Thesis]. University of São Paulo; 2012. Available from: http://www.teses.usp.br/teses/disponiveis/5/5167/tde-22012013-154327/ ;

12. Beaumatin, Florian. Étude des fonctions de survie de l'oncogène Bcl xL : rôles de la déamidation de Bcl xL et de l'interaction avec la protéine Rab7 : Study of the survival functions of the oncogene Bcl xL : the roles of Bcl xL deamidation and interaction with the protein Rab7.

Degree: Docteur es, Sciences, technologie, santé. Biochimie, 2012, Université de Bordeaux Segalen

La protéine Bcl xL, membre de la famille de Bcl 2, est essentiellement décrite pour son rôle dans l'inhibition de la mort des cellules. Récemment,… (more)

Subjects/Keywords: Autophagie; Apoptose; Endocytose; Cancer; Deamidation; Bcl xL; Rab7; Famille de Bcl-2; Autophagy; Apoptosis; Endocytosis; Cancer; Deamidation; Bcl xL; Rab7; Bcl 2 family

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APA (6th Edition):

Beaumatin, F. (2012). Étude des fonctions de survie de l'oncogène Bcl xL : rôles de la déamidation de Bcl xL et de l'interaction avec la protéine Rab7 : Study of the survival functions of the oncogene Bcl xL : the roles of Bcl xL deamidation and interaction with the protein Rab7. (Doctoral Dissertation). Université de Bordeaux Segalen. Retrieved from http://www.theses.fr/2012BOR22012

Chicago Manual of Style (16th Edition):

Beaumatin, Florian. “Étude des fonctions de survie de l'oncogène Bcl xL : rôles de la déamidation de Bcl xL et de l'interaction avec la protéine Rab7 : Study of the survival functions of the oncogene Bcl xL : the roles of Bcl xL deamidation and interaction with the protein Rab7.” 2012. Doctoral Dissertation, Université de Bordeaux Segalen. Accessed January 28, 2021. http://www.theses.fr/2012BOR22012.

MLA Handbook (7th Edition):

Beaumatin, Florian. “Étude des fonctions de survie de l'oncogène Bcl xL : rôles de la déamidation de Bcl xL et de l'interaction avec la protéine Rab7 : Study of the survival functions of the oncogene Bcl xL : the roles of Bcl xL deamidation and interaction with the protein Rab7.” 2012. Web. 28 Jan 2021.

Vancouver:

Beaumatin F. Étude des fonctions de survie de l'oncogène Bcl xL : rôles de la déamidation de Bcl xL et de l'interaction avec la protéine Rab7 : Study of the survival functions of the oncogene Bcl xL : the roles of Bcl xL deamidation and interaction with the protein Rab7. [Internet] [Doctoral dissertation]. Université de Bordeaux Segalen; 2012. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2012BOR22012.

Council of Science Editors:

Beaumatin F. Étude des fonctions de survie de l'oncogène Bcl xL : rôles de la déamidation de Bcl xL et de l'interaction avec la protéine Rab7 : Study of the survival functions of the oncogene Bcl xL : the roles of Bcl xL deamidation and interaction with the protein Rab7. [Doctoral Dissertation]. Université de Bordeaux Segalen; 2012. Available from: http://www.theses.fr/2012BOR22012

13. Abou samra, Alma. Conception, synthèse et évaluation biologique d’inhibiteurs des protéines anti-apoptotiques de la famille Bcl-2 : Development and biological evaluation of small molecules inhibitors of anti-apoptotic proteins of Bcl-2 family.

Degree: Docteur es, Chimie thérapeutique, 2017, Université Paris-Saclay (ComUE)

La mitochondrie joue un rôle capital dans la mort cellulaire programmée ou apoptose par l’intermédiaire des protéines de la famille Bcl-2. Le dérèglement de l'apoptose… (more)

Subjects/Keywords: Apoptose; Inhibiteurs multiples; Bcl-2; Bcl-XL; Mcl-1; Meiogynine A; Apoptosis; Multiple inhibitors; Bcl-2; Bcl-XL; Mcl-1; Meiogynine A

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APA (6th Edition):

Abou samra, A. (2017). Conception, synthèse et évaluation biologique d’inhibiteurs des protéines anti-apoptotiques de la famille Bcl-2 : Development and biological evaluation of small molecules inhibitors of anti-apoptotic proteins of Bcl-2 family. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2017SACLS390

Chicago Manual of Style (16th Edition):

Abou samra, Alma. “Conception, synthèse et évaluation biologique d’inhibiteurs des protéines anti-apoptotiques de la famille Bcl-2 : Development and biological evaluation of small molecules inhibitors of anti-apoptotic proteins of Bcl-2 family.” 2017. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 28, 2021. http://www.theses.fr/2017SACLS390.

MLA Handbook (7th Edition):

Abou samra, Alma. “Conception, synthèse et évaluation biologique d’inhibiteurs des protéines anti-apoptotiques de la famille Bcl-2 : Development and biological evaluation of small molecules inhibitors of anti-apoptotic proteins of Bcl-2 family.” 2017. Web. 28 Jan 2021.

Vancouver:

Abou samra A. Conception, synthèse et évaluation biologique d’inhibiteurs des protéines anti-apoptotiques de la famille Bcl-2 : Development and biological evaluation of small molecules inhibitors of anti-apoptotic proteins of Bcl-2 family. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2017. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2017SACLS390.

Council of Science Editors:

Abou samra A. Conception, synthèse et évaluation biologique d’inhibiteurs des protéines anti-apoptotiques de la famille Bcl-2 : Development and biological evaluation of small molecules inhibitors of anti-apoptotic proteins of Bcl-2 family. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2017. Available from: http://www.theses.fr/2017SACLS390


University of Alberta

14. Craik, Alison C. BAD-interacting proteins in breast cancer cells.

Degree: MS, Department of Biochemistry, 2012, University of Alberta

 Taxane chemotherapy is widely used in the treatment of breast cancer. Despite its widespread clinical use, the molecular mechanisms of paclitaxel induced apoptosis remain unclear.… (more)

Subjects/Keywords: BCL-XL; BCL-2 antagonist of cell death; apoptosis; BAD; 14-3-3; paclitaxel

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APA (6th Edition):

Craik, A. C. (2012). BAD-interacting proteins in breast cancer cells. (Masters Thesis). University of Alberta. Retrieved from https://era.library.ualberta.ca/files/fq977v49m

Chicago Manual of Style (16th Edition):

Craik, Alison C. “BAD-interacting proteins in breast cancer cells.” 2012. Masters Thesis, University of Alberta. Accessed January 28, 2021. https://era.library.ualberta.ca/files/fq977v49m.

MLA Handbook (7th Edition):

Craik, Alison C. “BAD-interacting proteins in breast cancer cells.” 2012. Web. 28 Jan 2021.

Vancouver:

Craik AC. BAD-interacting proteins in breast cancer cells. [Internet] [Masters thesis]. University of Alberta; 2012. [cited 2021 Jan 28]. Available from: https://era.library.ualberta.ca/files/fq977v49m.

Council of Science Editors:

Craik AC. BAD-interacting proteins in breast cancer cells. [Masters Thesis]. University of Alberta; 2012. Available from: https://era.library.ualberta.ca/files/fq977v49m


McMaster University

15. Kale, Justin. Characterization of the Activation Mechanism of Bax.

Degree: PhD, 2017, McMaster University

Mitochondrial outer membrane permeabilization (MOMP) is regulated by protein-protein and protein-membrane interactions between Bcl-2 family proteins. These interactions are governed by the concentrations and relative… (more)

Subjects/Keywords: Bax; Apoptosis; Bcl-2 proteins; Bcl-XL; Biochemistry; Biophysics; FRET; Fluorescence Spectroscopy; Cancer; AKT

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APA (6th Edition):

Kale, J. (2017). Characterization of the Activation Mechanism of Bax. (Doctoral Dissertation). McMaster University. Retrieved from http://hdl.handle.net/11375/21485

Chicago Manual of Style (16th Edition):

Kale, Justin. “Characterization of the Activation Mechanism of Bax.” 2017. Doctoral Dissertation, McMaster University. Accessed January 28, 2021. http://hdl.handle.net/11375/21485.

MLA Handbook (7th Edition):

Kale, Justin. “Characterization of the Activation Mechanism of Bax.” 2017. Web. 28 Jan 2021.

Vancouver:

Kale J. Characterization of the Activation Mechanism of Bax. [Internet] [Doctoral dissertation]. McMaster University; 2017. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/11375/21485.

Council of Science Editors:

Kale J. Characterization of the Activation Mechanism of Bax. [Doctoral Dissertation]. McMaster University; 2017. Available from: http://hdl.handle.net/11375/21485


Virginia Commonwealth University

16. Nam, Sang-Jin. GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A2 REGULATES BCL-XL PROTEIN LEVELS IN MICE LUNG.

Degree: MS, Biochemistry, 2014, Virginia Commonwealth University

  With previous indication of the Group VIA phospholipase A2 (iPLA2β) enzyme regulating ER-stress induced apoptosis in β-cells by regulating the anti-apoptotic protein Bcl-xL via… (more)

Subjects/Keywords: iPLA2 beta; Bcl-xL; mouse lung; alternative splicing; Biochemistry

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APA (6th Edition):

Nam, S. (2014). GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A2 REGULATES BCL-XL PROTEIN LEVELS IN MICE LUNG. (Thesis). Virginia Commonwealth University. Retrieved from https://doi.org/10.25772/PDH8-KA92 ; https://scholarscompass.vcu.edu/etd/3512

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nam, Sang-Jin. “GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A2 REGULATES BCL-XL PROTEIN LEVELS IN MICE LUNG.” 2014. Thesis, Virginia Commonwealth University. Accessed January 28, 2021. https://doi.org/10.25772/PDH8-KA92 ; https://scholarscompass.vcu.edu/etd/3512.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nam, Sang-Jin. “GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A2 REGULATES BCL-XL PROTEIN LEVELS IN MICE LUNG.” 2014. Web. 28 Jan 2021.

Vancouver:

Nam S. GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A2 REGULATES BCL-XL PROTEIN LEVELS IN MICE LUNG. [Internet] [Thesis]. Virginia Commonwealth University; 2014. [cited 2021 Jan 28]. Available from: https://doi.org/10.25772/PDH8-KA92 ; https://scholarscompass.vcu.edu/etd/3512.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nam S. GROUP VIA CALCIUM-INDEPENDENT PHOSPHOLIPASE A2 REGULATES BCL-XL PROTEIN LEVELS IN MICE LUNG. [Thesis]. Virginia Commonwealth University; 2014. Available from: https://doi.org/10.25772/PDH8-KA92 ; https://scholarscompass.vcu.edu/etd/3512

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

17. Richard, Travis. ARK5 Regulates Subcellular Localization of hnRNP A1 During Hypertonic Stress .

Degree: 2017, University of Ottawa

 During cellular stress, the regulation of protein synthesis is a key adaptive mechanism used by cells to survive. In response to various stresses, heterogeneous nuclear… (more)

Subjects/Keywords: ARK5; hnRNP A1; Bcl-xL; IRES; Hypertonic Stress; Protein Synthesis Regulation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Richard, T. (2017). ARK5 Regulates Subcellular Localization of hnRNP A1 During Hypertonic Stress . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/36212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Richard, Travis. “ARK5 Regulates Subcellular Localization of hnRNP A1 During Hypertonic Stress .” 2017. Thesis, University of Ottawa. Accessed January 28, 2021. http://hdl.handle.net/10393/36212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Richard, Travis. “ARK5 Regulates Subcellular Localization of hnRNP A1 During Hypertonic Stress .” 2017. Web. 28 Jan 2021.

Vancouver:

Richard T. ARK5 Regulates Subcellular Localization of hnRNP A1 During Hypertonic Stress . [Internet] [Thesis]. University of Ottawa; 2017. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/10393/36212.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Richard T. ARK5 Regulates Subcellular Localization of hnRNP A1 During Hypertonic Stress . [Thesis]. University of Ottawa; 2017. Available from: http://hdl.handle.net/10393/36212

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Ottawa

18. Jordan, Lindsay. Signalling Towards IRES .

Degree: 2011, University of Ottawa

 XIAP and Bcl-xL are critical anti-apoptotic molecules that directly inhibit caspases and block mitochondrial membrane permeabilization, respectively. In addition to preventing apoptosis, both XIAP and… (more)

Subjects/Keywords: XIAP; Bcl-xL; PDCD4; Apaf-1; IRES; S6K2; translation; apoptosis

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APA (6th Edition):

Jordan, L. (2011). Signalling Towards IRES . (Thesis). University of Ottawa. Retrieved from http://hdl.handle.net/10393/19946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jordan, Lindsay. “Signalling Towards IRES .” 2011. Thesis, University of Ottawa. Accessed January 28, 2021. http://hdl.handle.net/10393/19946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jordan, Lindsay. “Signalling Towards IRES .” 2011. Web. 28 Jan 2021.

Vancouver:

Jordan L. Signalling Towards IRES . [Internet] [Thesis]. University of Ottawa; 2011. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/10393/19946.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jordan L. Signalling Towards IRES . [Thesis]. University of Ottawa; 2011. Available from: http://hdl.handle.net/10393/19946

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Harvard University

19. Ho-Shing, Olivia E. Cell Type-Specific Analysis of Bcl-xL Genomic Imprinting in the Mouse Brain.

Degree: PhD, 2019, Harvard University

Genomic imprinting is a form of epigenetic inheritance that causes preferential expression of one allele based on its parent of origin. Imprinted expression plays crucial… (more)

Subjects/Keywords: genomic imprinting; Bcl-xL; synaptic plasticity; visual cortex; parent-of-origin

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APA (6th Edition):

Ho-Shing, O. E. (2019). Cell Type-Specific Analysis of Bcl-xL Genomic Imprinting in the Mouse Brain. (Doctoral Dissertation). Harvard University. Retrieved from http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029525

Chicago Manual of Style (16th Edition):

Ho-Shing, Olivia E. “Cell Type-Specific Analysis of Bcl-xL Genomic Imprinting in the Mouse Brain.” 2019. Doctoral Dissertation, Harvard University. Accessed January 28, 2021. http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029525.

MLA Handbook (7th Edition):

Ho-Shing, Olivia E. “Cell Type-Specific Analysis of Bcl-xL Genomic Imprinting in the Mouse Brain.” 2019. Web. 28 Jan 2021.

Vancouver:

Ho-Shing OE. Cell Type-Specific Analysis of Bcl-xL Genomic Imprinting in the Mouse Brain. [Internet] [Doctoral dissertation]. Harvard University; 2019. [cited 2021 Jan 28]. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029525.

Council of Science Editors:

Ho-Shing OE. Cell Type-Specific Analysis of Bcl-xL Genomic Imprinting in the Mouse Brain. [Doctoral Dissertation]. Harvard University; 2019. Available from: http://nrs.harvard.edu/urn-3:HUL.InstRepos:42029525


University of Melbourne

20. Tse, Janson W. T. Investigating the mechanisms by which histone deacetylase inhibitors induce apoptosis in cancer cells.

Degree: 2017, University of Melbourne

 Cancer develops as a multi-step process through the accumulation of abnormal genetic alterations in tumour suppressor genes and oncogenes. Superimposed upon these genetic changes are… (more)

Subjects/Keywords: proteasome inhibitors; cancer; ATF3; BCL-XL; histone deacetylase inhibitors

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APA (6th Edition):

Tse, J. W. T. (2017). Investigating the mechanisms by which histone deacetylase inhibitors induce apoptosis in cancer cells. (Doctoral Dissertation). University of Melbourne. Retrieved from http://hdl.handle.net/11343/192923

Chicago Manual of Style (16th Edition):

Tse, Janson W T. “Investigating the mechanisms by which histone deacetylase inhibitors induce apoptosis in cancer cells.” 2017. Doctoral Dissertation, University of Melbourne. Accessed January 28, 2021. http://hdl.handle.net/11343/192923.

MLA Handbook (7th Edition):

Tse, Janson W T. “Investigating the mechanisms by which histone deacetylase inhibitors induce apoptosis in cancer cells.” 2017. Web. 28 Jan 2021.

Vancouver:

Tse JWT. Investigating the mechanisms by which histone deacetylase inhibitors induce apoptosis in cancer cells. [Internet] [Doctoral dissertation]. University of Melbourne; 2017. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/11343/192923.

Council of Science Editors:

Tse JWT. Investigating the mechanisms by which histone deacetylase inhibitors induce apoptosis in cancer cells. [Doctoral Dissertation]. University of Melbourne; 2017. Available from: http://hdl.handle.net/11343/192923


University of South Florida

21. Timme, Cindy R. Drug Resistance Mechanisms to Gamma-secretase Inhibitors in Human Colon Cancer Cells.

Degree: 2013, University of South Florida

 Colorectal cancer is the third leading cause of cancer-related mortality. Much progress has been achieved in combating this disease with surgical resection and chemotherapy in… (more)

Subjects/Keywords: autophagy; Bcl-xL; GSI; Mcl-1; obatoclax; oxaliplatin; Biology; Cell Biology

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APA (6th Edition):

Timme, C. R. (2013). Drug Resistance Mechanisms to Gamma-secretase Inhibitors in Human Colon Cancer Cells. (Thesis). University of South Florida. Retrieved from https://scholarcommons.usf.edu/etd/4954

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Timme, Cindy R. “Drug Resistance Mechanisms to Gamma-secretase Inhibitors in Human Colon Cancer Cells.” 2013. Thesis, University of South Florida. Accessed January 28, 2021. https://scholarcommons.usf.edu/etd/4954.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Timme, Cindy R. “Drug Resistance Mechanisms to Gamma-secretase Inhibitors in Human Colon Cancer Cells.” 2013. Web. 28 Jan 2021.

Vancouver:

Timme CR. Drug Resistance Mechanisms to Gamma-secretase Inhibitors in Human Colon Cancer Cells. [Internet] [Thesis]. University of South Florida; 2013. [cited 2021 Jan 28]. Available from: https://scholarcommons.usf.edu/etd/4954.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Timme CR. Drug Resistance Mechanisms to Gamma-secretase Inhibitors in Human Colon Cancer Cells. [Thesis]. University of South Florida; 2013. Available from: https://scholarcommons.usf.edu/etd/4954

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Cincinnati

22. Griesinger, Andrea. The role of THPO/MPL signaling in AML1-ETO self-renewal.

Degree: MS, Medicine: Cell and Molecular Biology, 2011, University of Cincinnati

  Chromosomal translocation (8;21)(q22;q22) gives rise to the fusion gene AML1-ETO (AE) which is present in 10-15% of all acute myeloid leukemia and 40% of… (more)

Subjects/Keywords: Cellular Biology; THPO/MPL; AML; Bcl-xL; AML1-ETO

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APA (6th Edition):

Griesinger, A. (2011). The role of THPO/MPL signaling in AML1-ETO self-renewal. (Masters Thesis). University of Cincinnati. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321969049

Chicago Manual of Style (16th Edition):

Griesinger, Andrea. “The role of THPO/MPL signaling in AML1-ETO self-renewal.” 2011. Masters Thesis, University of Cincinnati. Accessed January 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321969049.

MLA Handbook (7th Edition):

Griesinger, Andrea. “The role of THPO/MPL signaling in AML1-ETO self-renewal.” 2011. Web. 28 Jan 2021.

Vancouver:

Griesinger A. The role of THPO/MPL signaling in AML1-ETO self-renewal. [Internet] [Masters thesis]. University of Cincinnati; 2011. [cited 2021 Jan 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321969049.

Council of Science Editors:

Griesinger A. The role of THPO/MPL signaling in AML1-ETO self-renewal. [Masters Thesis]. University of Cincinnati; 2011. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=ucin1321969049


The Ohio State University

23. Shaw, Yeng-Jeng. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.

Degree: PhD, Pharmacy, 2005, The Ohio State University

 Prostate cancer is the most common form of cancer in men and the second leading cause of cancer-related deaths in the United States. So far,… (more)

Subjects/Keywords: Akt; Bcl-xL; DNMT

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APA (6th Edition):

Shaw, Y. (2005). Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982

Chicago Manual of Style (16th Edition):

Shaw, Yeng-Jeng. “Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.” 2005. Doctoral Dissertation, The Ohio State University. Accessed January 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982.

MLA Handbook (7th Edition):

Shaw, Yeng-Jeng. “Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer.” 2005. Web. 28 Jan 2021.

Vancouver:

Shaw Y. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2021 Jan 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982.

Council of Science Editors:

Shaw Y. Small molecule-based drug design of anticancer agents that target protein kinase B/ AKT, Bcl-xL and DNA methyltransferases for the treatment of prostate cancer. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128693982


The Ohio State University

24. Shiau, Chung-Wai. Thiazolidinediones: from peroxisome-proliferator-activated receptor γ(PPARγ) to anticancer agents.

Degree: PhD, Pharmacy, 2005, The Ohio State University

 Thiazolidinediones, which are peroxisome-proliferator-activated receptor γ (PPARγ) agonists that are used as insulin sensitizers for treatment of type 2 diabetes, have been reported to induce… (more)

Subjects/Keywords: Chemistry, Pharmaceutical; Thiazolidinediones; prostate cancer; Bcl-2/Bcl-xL

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APA (6th Edition):

Shiau, C. (2005). Thiazolidinediones: from peroxisome-proliferator-activated receptor γ(PPARγ) to anticancer agents. (Doctoral Dissertation). The Ohio State University. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=osu1128111032

Chicago Manual of Style (16th Edition):

Shiau, Chung-Wai. “Thiazolidinediones: from peroxisome-proliferator-activated receptor γ(PPARγ) to anticancer agents.” 2005. Doctoral Dissertation, The Ohio State University. Accessed January 28, 2021. http://rave.ohiolink.edu/etdc/view?acc_num=osu1128111032.

MLA Handbook (7th Edition):

Shiau, Chung-Wai. “Thiazolidinediones: from peroxisome-proliferator-activated receptor γ(PPARγ) to anticancer agents.” 2005. Web. 28 Jan 2021.

Vancouver:

Shiau C. Thiazolidinediones: from peroxisome-proliferator-activated receptor γ(PPARγ) to anticancer agents. [Internet] [Doctoral dissertation]. The Ohio State University; 2005. [cited 2021 Jan 28]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128111032.

Council of Science Editors:

Shiau C. Thiazolidinediones: from peroxisome-proliferator-activated receptor γ(PPARγ) to anticancer agents. [Doctoral Dissertation]. The Ohio State University; 2005. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=osu1128111032


Université Paris-Sud – Paris XI

25. Garancher, Alexandra. Les facteurs de transcription MAF dans l’oncogenèse : implication de NRL dans le médulloblastome : MAF transcription factors in oncogenesis : involvement of NRL in medulloblastoma.

Degree: Docteur es, Biologie, 2014, Université Paris-Sud – Paris XI

Les facteurs de transcription de la famille MAF sont impliqués d’une part au cours du développement dans des processus de différenciation terminale et d’autre part… (more)

Subjects/Keywords: MAF; NRL; Apoptose; Médulloblastome; Facteur de transcription; Protéine anti-apoptotique BCL-XL; MAF; NRL; Apoptosis; Medulloblastoma; Transcription factor; Anti-apoptotic protein BCL-XL

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APA (6th Edition):

Garancher, A. (2014). Les facteurs de transcription MAF dans l’oncogenèse : implication de NRL dans le médulloblastome : MAF transcription factors in oncogenesis : involvement of NRL in medulloblastoma. (Doctoral Dissertation). Université Paris-Sud – Paris XI. Retrieved from http://www.theses.fr/2014PA112103

Chicago Manual of Style (16th Edition):

Garancher, Alexandra. “Les facteurs de transcription MAF dans l’oncogenèse : implication de NRL dans le médulloblastome : MAF transcription factors in oncogenesis : involvement of NRL in medulloblastoma.” 2014. Doctoral Dissertation, Université Paris-Sud – Paris XI. Accessed January 28, 2021. http://www.theses.fr/2014PA112103.

MLA Handbook (7th Edition):

Garancher, Alexandra. “Les facteurs de transcription MAF dans l’oncogenèse : implication de NRL dans le médulloblastome : MAF transcription factors in oncogenesis : involvement of NRL in medulloblastoma.” 2014. Web. 28 Jan 2021.

Vancouver:

Garancher A. Les facteurs de transcription MAF dans l’oncogenèse : implication de NRL dans le médulloblastome : MAF transcription factors in oncogenesis : involvement of NRL in medulloblastoma. [Internet] [Doctoral dissertation]. Université Paris-Sud – Paris XI; 2014. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2014PA112103.

Council of Science Editors:

Garancher A. Les facteurs de transcription MAF dans l’oncogenèse : implication de NRL dans le médulloblastome : MAF transcription factors in oncogenesis : involvement of NRL in medulloblastoma. [Doctoral Dissertation]. Université Paris-Sud – Paris XI; 2014. Available from: http://www.theses.fr/2014PA112103

26. Iannelli, Antonio. Lésions hépatiques induites par l'ischémie reperfusion et la NAFLD : mécanismes et protection : Liver injuries due to ischemia reperfusion and NAFLD : mechanisms and protection.

Degree: Docteur es, Sciences de l'environnement terrestre, 2010, Aix-Marseille 2

 Le pregnane X receptor (PXR) est un récepteur nucléaire associé à la réponse au stresscellulaire. Des travaux in vitro de notre groupe ont démontré que… (more)

Subjects/Keywords: Pregnane X receptor; Ischémie reperfusion; Foie; Bcl-xL; Stéatose; Stéatohépatite; Obésité; Syndrome métabolique; Bariatrique; Pregnane X receptor; Ischemia reperfusion; Liver; Bcl-xL; Steatosis; Steatohepatitis; Obesity; Metabolic syndrome; Bariatric

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APA (6th Edition):

Iannelli, A. (2010). Lésions hépatiques induites par l'ischémie reperfusion et la NAFLD : mécanismes et protection : Liver injuries due to ischemia reperfusion and NAFLD : mechanisms and protection. (Doctoral Dissertation). Aix-Marseille 2. Retrieved from http://www.theses.fr/2010AIX20659

Chicago Manual of Style (16th Edition):

Iannelli, Antonio. “Lésions hépatiques induites par l'ischémie reperfusion et la NAFLD : mécanismes et protection : Liver injuries due to ischemia reperfusion and NAFLD : mechanisms and protection.” 2010. Doctoral Dissertation, Aix-Marseille 2. Accessed January 28, 2021. http://www.theses.fr/2010AIX20659.

MLA Handbook (7th Edition):

Iannelli, Antonio. “Lésions hépatiques induites par l'ischémie reperfusion et la NAFLD : mécanismes et protection : Liver injuries due to ischemia reperfusion and NAFLD : mechanisms and protection.” 2010. Web. 28 Jan 2021.

Vancouver:

Iannelli A. Lésions hépatiques induites par l'ischémie reperfusion et la NAFLD : mécanismes et protection : Liver injuries due to ischemia reperfusion and NAFLD : mechanisms and protection. [Internet] [Doctoral dissertation]. Aix-Marseille 2; 2010. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2010AIX20659.

Council of Science Editors:

Iannelli A. Lésions hépatiques induites par l'ischémie reperfusion et la NAFLD : mécanismes et protection : Liver injuries due to ischemia reperfusion and NAFLD : mechanisms and protection. [Doctoral Dissertation]. Aix-Marseille 2; 2010. Available from: http://www.theses.fr/2010AIX20659

27. Remeur, Camille. Synthèse et évaluation biologique de dérivés de la meiogynine A, un produit naturel, qui ciblent la restauration de l'apoptose : Synthesis and biological evaluation of meiogynine A derivatives, a natural product, targeting apoptosis.

Degree: Docteur es, Chimie thérapeutique, 2015, Université Paris-Saclay (ComUE)

L'apoptose, appelée aussi mort cellulaire programmée, est un processus physiologique qui permet d'éliminer les cellules endommagées ou superflues lors du développement cellulaire. Le phénomène d'échappement… (more)

Subjects/Keywords: Diels-Alder; Apoptose; Produit naturel; Meiogynine A; Bcl-XL; Mcl-1; Diels-Alder; Apoptosis; Natural product; Meiogynine A; Bcl-XL; Mcl-1

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APA (6th Edition):

Remeur, C. (2015). Synthèse et évaluation biologique de dérivés de la meiogynine A, un produit naturel, qui ciblent la restauration de l'apoptose : Synthesis and biological evaluation of meiogynine A derivatives, a natural product, targeting apoptosis. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2015SACLS158

Chicago Manual of Style (16th Edition):

Remeur, Camille. “Synthèse et évaluation biologique de dérivés de la meiogynine A, un produit naturel, qui ciblent la restauration de l'apoptose : Synthesis and biological evaluation of meiogynine A derivatives, a natural product, targeting apoptosis.” 2015. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed January 28, 2021. http://www.theses.fr/2015SACLS158.

MLA Handbook (7th Edition):

Remeur, Camille. “Synthèse et évaluation biologique de dérivés de la meiogynine A, un produit naturel, qui ciblent la restauration de l'apoptose : Synthesis and biological evaluation of meiogynine A derivatives, a natural product, targeting apoptosis.” 2015. Web. 28 Jan 2021.

Vancouver:

Remeur C. Synthèse et évaluation biologique de dérivés de la meiogynine A, un produit naturel, qui ciblent la restauration de l'apoptose : Synthesis and biological evaluation of meiogynine A derivatives, a natural product, targeting apoptosis. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2015. [cited 2021 Jan 28]. Available from: http://www.theses.fr/2015SACLS158.

Council of Science Editors:

Remeur C. Synthèse et évaluation biologique de dérivés de la meiogynine A, un produit naturel, qui ciblent la restauration de l'apoptose : Synthesis and biological evaluation of meiogynine A derivatives, a natural product, targeting apoptosis. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2015. Available from: http://www.theses.fr/2015SACLS158


University of Michigan

28. Karl, Elisabeta. Role of Bcl-2 and Bcl-xL in angiogenesis.

Degree: PhD, Molecular biology, 2006, University of Michigan

 Tumor progression is dependent on angiogenesis, the process of the new vessel formation from pre-existing capillaries. Vascular endothelial growth factor (VEGF), a key regulator of… (more)

Subjects/Keywords: Angiogenesis; Bcl-2; Role; Vegf; Xl

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APA (6th Edition):

Karl, E. (2006). Role of Bcl-2 and Bcl-xL in angiogenesis. (Doctoral Dissertation). University of Michigan. Retrieved from http://hdl.handle.net/2027.42/126190

Chicago Manual of Style (16th Edition):

Karl, Elisabeta. “Role of Bcl-2 and Bcl-xL in angiogenesis.” 2006. Doctoral Dissertation, University of Michigan. Accessed January 28, 2021. http://hdl.handle.net/2027.42/126190.

MLA Handbook (7th Edition):

Karl, Elisabeta. “Role of Bcl-2 and Bcl-xL in angiogenesis.” 2006. Web. 28 Jan 2021.

Vancouver:

Karl E. Role of Bcl-2 and Bcl-xL in angiogenesis. [Internet] [Doctoral dissertation]. University of Michigan; 2006. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/2027.42/126190.

Council of Science Editors:

Karl E. Role of Bcl-2 and Bcl-xL in angiogenesis. [Doctoral Dissertation]. University of Michigan; 2006. Available from: http://hdl.handle.net/2027.42/126190


Vanderbilt University

29. Gordy, Laura Elizabeth. Repertoire selection and effector differentiation during NKT cell development.

Degree: PhD, Microbiology and Immunology, 2012, Vanderbilt University

 Natural killer T (NKT) cells are innate-like lymphocytes that develop and mature in the thymus. From there, they home to the peripheral lymphoid tissues where,… (more)

Subjects/Keywords: T-bet; IL-15; Bcl-xL; development; survival; NKT cells; Nur77; negative selection

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APA (6th Edition):

Gordy, L. E. (2012). Repertoire selection and effector differentiation during NKT cell development. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/11710

Chicago Manual of Style (16th Edition):

Gordy, Laura Elizabeth. “Repertoire selection and effector differentiation during NKT cell development.” 2012. Doctoral Dissertation, Vanderbilt University. Accessed January 28, 2021. http://hdl.handle.net/1803/11710.

MLA Handbook (7th Edition):

Gordy, Laura Elizabeth. “Repertoire selection and effector differentiation during NKT cell development.” 2012. Web. 28 Jan 2021.

Vancouver:

Gordy LE. Repertoire selection and effector differentiation during NKT cell development. [Internet] [Doctoral dissertation]. Vanderbilt University; 2012. [cited 2021 Jan 28]. Available from: http://hdl.handle.net/1803/11710.

Council of Science Editors:

Gordy LE. Repertoire selection and effector differentiation during NKT cell development. [Doctoral Dissertation]. Vanderbilt University; 2012. Available from: http://hdl.handle.net/1803/11710

30. 박, 호진. Studies on the Mechanisms of Malignant Transformation in Neurofibromatosis Type 1.

Degree: 2009, Ajou University

"신경섬유종증 제1형(Neurofibromatosis Type 1, NF1)은 1882년 Fredrich von Recklin-ghausen이 처음 기술하였으며, 말초신경을 따라 다기관에 다발적으로 종양을 형성하는 상염색체 우성 유전 질환으로 약 3500명중 1명꼴로 발생한다.… (more)

Subjects/Keywords: 신경섬유종증; 세포자멸사; 뉴로파이브로민; Ras; Bcl-xL

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APA (6th Edition):

박, . (2009). Studies on the Mechanisms of Malignant Transformation in Neurofibromatosis Type 1. (Thesis). Ajou University. Retrieved from http://repository.ajou.ac.kr/handle/201003/2279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000009843

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

박, 호진. “Studies on the Mechanisms of Malignant Transformation in Neurofibromatosis Type 1.” 2009. Thesis, Ajou University. Accessed January 28, 2021. http://repository.ajou.ac.kr/handle/201003/2279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000009843.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

박, 호진. “Studies on the Mechanisms of Malignant Transformation in Neurofibromatosis Type 1.” 2009. Web. 28 Jan 2021.

Vancouver:

박 . Studies on the Mechanisms of Malignant Transformation in Neurofibromatosis Type 1. [Internet] [Thesis]. Ajou University; 2009. [cited 2021 Jan 28]. Available from: http://repository.ajou.ac.kr/handle/201003/2279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000009843.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

박 . Studies on the Mechanisms of Malignant Transformation in Neurofibromatosis Type 1. [Thesis]. Ajou University; 2009. Available from: http://repository.ajou.ac.kr/handle/201003/2279 ; http://dcoll.ajou.ac.kr:9080/dcollection/jsp/common/DcLoOrgPer.jsp?sItemId=000000009843

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3]

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