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You searched for subject:(Base excision Repair). Showing records 1 – 30 of 78 total matches.

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1. Schermerhorn, Kelly. A Kinetic Perspective on DNA Base Excision Repair.

Degree: PhD, Chemistry, 2014, Brown University

 The DNA base excision repair (BER) pathway is compromised of several enzymes, including DNA glycosylases, apurinic/apyrimidinic (AP) endonuclease 1 (APE1), DNA polymerase β (pol β)… (more)

Subjects/Keywords: Base Excision Repair

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APA (6th Edition):

Schermerhorn, K. (2014). A Kinetic Perspective on DNA Base Excision Repair. (Doctoral Dissertation). Brown University. Retrieved from https://repository.library.brown.edu/studio/item/bdr:386264/

Chicago Manual of Style (16th Edition):

Schermerhorn, Kelly. “A Kinetic Perspective on DNA Base Excision Repair.” 2014. Doctoral Dissertation, Brown University. Accessed November 27, 2020. https://repository.library.brown.edu/studio/item/bdr:386264/.

MLA Handbook (7th Edition):

Schermerhorn, Kelly. “A Kinetic Perspective on DNA Base Excision Repair.” 2014. Web. 27 Nov 2020.

Vancouver:

Schermerhorn K. A Kinetic Perspective on DNA Base Excision Repair. [Internet] [Doctoral dissertation]. Brown University; 2014. [cited 2020 Nov 27]. Available from: https://repository.library.brown.edu/studio/item/bdr:386264/.

Council of Science Editors:

Schermerhorn K. A Kinetic Perspective on DNA Base Excision Repair. [Doctoral Dissertation]. Brown University; 2014. Available from: https://repository.library.brown.edu/studio/item/bdr:386264/


University of Oxford

2. Fletcher, Sally C. Regulation of the base excision repair pathway.

Degree: PhD, 2017, University of Oxford

 Maintenance of genomic stability is paramount for survival of an organism; failure to repair DNA damage ultimately leads to the accumulation of genetically unstable cells… (more)

Subjects/Keywords: Base excision repair; XRCC1; Sp1

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APA (6th Edition):

Fletcher, S. C. (2017). Regulation of the base excision repair pathway. (Doctoral Dissertation). University of Oxford. Retrieved from http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778

Chicago Manual of Style (16th Edition):

Fletcher, Sally C. “Regulation of the base excision repair pathway.” 2017. Doctoral Dissertation, University of Oxford. Accessed November 27, 2020. http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778.

MLA Handbook (7th Edition):

Fletcher, Sally C. “Regulation of the base excision repair pathway.” 2017. Web. 27 Nov 2020.

Vancouver:

Fletcher SC. Regulation of the base excision repair pathway. [Internet] [Doctoral dissertation]. University of Oxford; 2017. [cited 2020 Nov 27]. Available from: http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778.

Council of Science Editors:

Fletcher SC. Regulation of the base excision repair pathway. [Doctoral Dissertation]. University of Oxford; 2017. Available from: http://ora.ox.ac.uk/objects/uuid:e3db2385-f9aa-4289-9547-3e37cbeddec9 ; https://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.757778


Drexel University

3. Atkins, Andrew James. Predicting the effects of intracellular protein variation on base excision repair capacity in human cells.

Degree: 2012, Drexel University

Base excision repair (BER) is one of the primary means by which cells cope with genotoxic stress and DNA damage. BER is carried out by… (more)

Subjects/Keywords: Biomedical engineering; DNA repair; Base excision repair

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APA (6th Edition):

Atkins, A. J. (2012). Predicting the effects of intracellular protein variation on base excision repair capacity in human cells. (Thesis). Drexel University. Retrieved from http://hdl.handle.net/1860/3765

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Atkins, Andrew James. “Predicting the effects of intracellular protein variation on base excision repair capacity in human cells.” 2012. Thesis, Drexel University. Accessed November 27, 2020. http://hdl.handle.net/1860/3765.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Atkins, Andrew James. “Predicting the effects of intracellular protein variation on base excision repair capacity in human cells.” 2012. Web. 27 Nov 2020.

Vancouver:

Atkins AJ. Predicting the effects of intracellular protein variation on base excision repair capacity in human cells. [Internet] [Thesis]. Drexel University; 2012. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1860/3765.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Atkins AJ. Predicting the effects of intracellular protein variation on base excision repair capacity in human cells. [Thesis]. Drexel University; 2012. Available from: http://hdl.handle.net/1860/3765

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Utah

4. Cao, Sheng. Synthesis of fluorinated analogs of oxidative DNA lesions and their use to probe features of recognition and repair by base excision repair glycosylases.

Degree: PhD, Chemistry;, 2010, University of Utah

 Damage to DNA occurs through various sources, both exogenous and endogenous. Base lesions resulting from the oxidation of guanine include 8-oxo-7,8-dihydroguanine (OG), guanidinohydantoin (Gh), and… (more)

Subjects/Keywords: DNA lesions; Base excision repair; Glycosylases

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APA (6th Edition):

Cao, S. (2010). Synthesis of fluorinated analogs of oxidative DNA lesions and their use to probe features of recognition and repair by base excision repair glycosylases. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1138/rec/1125

Chicago Manual of Style (16th Edition):

Cao, Sheng. “Synthesis of fluorinated analogs of oxidative DNA lesions and their use to probe features of recognition and repair by base excision repair glycosylases.” 2010. Doctoral Dissertation, University of Utah. Accessed November 27, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1138/rec/1125.

MLA Handbook (7th Edition):

Cao, Sheng. “Synthesis of fluorinated analogs of oxidative DNA lesions and their use to probe features of recognition and repair by base excision repair glycosylases.” 2010. Web. 27 Nov 2020.

Vancouver:

Cao S. Synthesis of fluorinated analogs of oxidative DNA lesions and their use to probe features of recognition and repair by base excision repair glycosylases. [Internet] [Doctoral dissertation]. University of Utah; 2010. [cited 2020 Nov 27]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1138/rec/1125.

Council of Science Editors:

Cao S. Synthesis of fluorinated analogs of oxidative DNA lesions and their use to probe features of recognition and repair by base excision repair glycosylases. [Doctoral Dissertation]. University of Utah; 2010. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd2/id/1138/rec/1125


Vanderbilt University

5. Shi, Rongxin. Characterization of heat-like repeat superfamily of DNA glycosylases.

Degree: PhD, Biological Sciences, 2018, Vanderbilt University

 DNA glycosylases preserve genome integrity and define the specificity of the base excision repair pathway for discreet, detrimental modifications. Bacterial HEAT-like repeat glycosylases AlkC and… (more)

Subjects/Keywords: DNA glycosylase; Base excision repair; AlkC

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APA (6th Edition):

Shi, R. (2018). Characterization of heat-like repeat superfamily of DNA glycosylases. (Doctoral Dissertation). Vanderbilt University. Retrieved from http://hdl.handle.net/1803/12440

Chicago Manual of Style (16th Edition):

Shi, Rongxin. “Characterization of heat-like repeat superfamily of DNA glycosylases.” 2018. Doctoral Dissertation, Vanderbilt University. Accessed November 27, 2020. http://hdl.handle.net/1803/12440.

MLA Handbook (7th Edition):

Shi, Rongxin. “Characterization of heat-like repeat superfamily of DNA glycosylases.” 2018. Web. 27 Nov 2020.

Vancouver:

Shi R. Characterization of heat-like repeat superfamily of DNA glycosylases. [Internet] [Doctoral dissertation]. Vanderbilt University; 2018. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1803/12440.

Council of Science Editors:

Shi R. Characterization of heat-like repeat superfamily of DNA glycosylases. [Doctoral Dissertation]. Vanderbilt University; 2018. Available from: http://hdl.handle.net/1803/12440


Louisiana State University

6. Li, Mingyang. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.

Degree: PhD, Laboratory and Basic Science Research, 2017, Louisiana State University

  In eukaryotes, DNA repair mechanisms detect and repair damaged DNA. DNA damage is primarily caused by a variety of exogenous and endogenous sources. Several… (more)

Subjects/Keywords: DNA repair; DNA damage; Base excision repair; Double-strand break repair

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APA (6th Edition):

Li, M. (2017). DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. (Doctoral Dissertation). Louisiana State University. Retrieved from https://digitalcommons.lsu.edu/gradschool_dissertations/4186

Chicago Manual of Style (16th Edition):

Li, Mingyang. “DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.” 2017. Doctoral Dissertation, Louisiana State University. Accessed November 27, 2020. https://digitalcommons.lsu.edu/gradschool_dissertations/4186.

MLA Handbook (7th Edition):

Li, Mingyang. “DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast.” 2017. Web. 27 Nov 2020.

Vancouver:

Li M. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. [Internet] [Doctoral dissertation]. Louisiana State University; 2017. [cited 2020 Nov 27]. Available from: https://digitalcommons.lsu.edu/gradschool_dissertations/4186.

Council of Science Editors:

Li M. DNA Base Excision Repair and Double Strand Break Repair in Human Fibroblast. [Doctoral Dissertation]. Louisiana State University; 2017. Available from: https://digitalcommons.lsu.edu/gradschool_dissertations/4186


University of Texas Medical Branch – Galveston

7. [No author]. Essentiality of Mitochondrial Genome Integrity in Cell Survival .

Degree: 2012, University of Texas Medical Branch – Galveston

 The mitochondrial genome encodes 22 tRNAs, 2 rRNAs, and 13 polypeptides that are subunits of the electron transport chain involved in oxidative phosphorylation. Because of… (more)

Subjects/Keywords: Base Excision Repair; Mitochondrial Genome; Mitochondrial DNA Repair; Oxidative Stress; Apoptosis

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APA (6th Edition):

author], [. (2012). Essentiality of Mitochondrial Genome Integrity in Cell Survival . (Thesis). University of Texas Medical Branch – Galveston. Retrieved from http://hdl.handle.net/2152.3/821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

author], [No. “Essentiality of Mitochondrial Genome Integrity in Cell Survival .” 2012. Thesis, University of Texas Medical Branch – Galveston. Accessed November 27, 2020. http://hdl.handle.net/2152.3/821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

author], [No. “Essentiality of Mitochondrial Genome Integrity in Cell Survival .” 2012. Web. 27 Nov 2020.

Vancouver:

author] [. Essentiality of Mitochondrial Genome Integrity in Cell Survival . [Internet] [Thesis]. University of Texas Medical Branch – Galveston; 2012. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/2152.3/821.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

author] [. Essentiality of Mitochondrial Genome Integrity in Cell Survival . [Thesis]. University of Texas Medical Branch – Galveston; 2012. Available from: http://hdl.handle.net/2152.3/821

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


New Jersey Institute of Technology

8. Xing, Qingyu. Triplex dna receptor-gold nanoparticle conjugates for detection of oxidized dna base.

Degree: MSin Chemistry - (M.S.), Chemistry and Environmental Science, 2016, New Jersey Institute of Technology

  Oxidations of DNA nucleobases are believed to be one of the sources of aging and some age-dependent disease, such as cancer. Human body have… (more)

Subjects/Keywords: DNA nucleobase oxidation; Base-excision-repair; Oxidized dna base detection; Chemistry

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APA (6th Edition):

Xing, Q. (2016). Triplex dna receptor-gold nanoparticle conjugates for detection of oxidized dna base. (Thesis). New Jersey Institute of Technology. Retrieved from https://digitalcommons.njit.edu/theses/265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Xing, Qingyu. “Triplex dna receptor-gold nanoparticle conjugates for detection of oxidized dna base.” 2016. Thesis, New Jersey Institute of Technology. Accessed November 27, 2020. https://digitalcommons.njit.edu/theses/265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Xing, Qingyu. “Triplex dna receptor-gold nanoparticle conjugates for detection of oxidized dna base.” 2016. Web. 27 Nov 2020.

Vancouver:

Xing Q. Triplex dna receptor-gold nanoparticle conjugates for detection of oxidized dna base. [Internet] [Thesis]. New Jersey Institute of Technology; 2016. [cited 2020 Nov 27]. Available from: https://digitalcommons.njit.edu/theses/265.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Xing Q. Triplex dna receptor-gold nanoparticle conjugates for detection of oxidized dna base. [Thesis]. New Jersey Institute of Technology; 2016. Available from: https://digitalcommons.njit.edu/theses/265

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


North-West University

9. Steenkamp, Anzaan. Establishing the comet assay to determine the effects of different perturbations on DNA repair capacity / by Anzaan Steenkamp .

Degree: 2011, North-West University

 Single cell gel electrophoresis (SCGE), more commonly known as the Comet assay, is an uncomplicated, affordable and versatile method for investigating DNA damage and repair.… (more)

Subjects/Keywords: Comet assay; DNA repair capacity; Base excision repair (BER); Nucleotide excision repair (NER); Electron transport chain; Metallothionein

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APA (6th Edition):

Steenkamp, A. (2011). Establishing the comet assay to determine the effects of different perturbations on DNA repair capacity / by Anzaan Steenkamp . (Thesis). North-West University. Retrieved from http://hdl.handle.net/10394/4628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Steenkamp, Anzaan. “Establishing the comet assay to determine the effects of different perturbations on DNA repair capacity / by Anzaan Steenkamp .” 2011. Thesis, North-West University. Accessed November 27, 2020. http://hdl.handle.net/10394/4628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Steenkamp, Anzaan. “Establishing the comet assay to determine the effects of different perturbations on DNA repair capacity / by Anzaan Steenkamp .” 2011. Web. 27 Nov 2020.

Vancouver:

Steenkamp A. Establishing the comet assay to determine the effects of different perturbations on DNA repair capacity / by Anzaan Steenkamp . [Internet] [Thesis]. North-West University; 2011. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/10394/4628.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Steenkamp A. Establishing the comet assay to determine the effects of different perturbations on DNA repair capacity / by Anzaan Steenkamp . [Thesis]. North-West University; 2011. Available from: http://hdl.handle.net/10394/4628

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. Lia, Debora. Role of alphaOGG1 in the Maintenance of Mitochondrial Physiology : Fonction de l'alphaOGG1 sur la maintenance de la physiologie mitochondriale.

Degree: Docteur es, Sciences de la vie et de la santé, 2018, Université Paris-Saclay (ComUE)

Les mitochondries sont des structures uniques dans la cellule mammifère. Ces organites portent leur propre génome (ADN mitochondrial, ADNmt) qui se compose d'une petite molécule… (more)

Subjects/Keywords: ADN repair; Stabilite ADNmt; Réparation par Excision de Bases; Stress Oxydant; DNA repair; MtDNA stability; Base excision Repair; Oxidative Stress

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APA (6th Edition):

Lia, D. (2018). Role of alphaOGG1 in the Maintenance of Mitochondrial Physiology : Fonction de l'alphaOGG1 sur la maintenance de la physiologie mitochondriale. (Doctoral Dissertation). Université Paris-Saclay (ComUE). Retrieved from http://www.theses.fr/2018SACLS125

Chicago Manual of Style (16th Edition):

Lia, Debora. “Role of alphaOGG1 in the Maintenance of Mitochondrial Physiology : Fonction de l'alphaOGG1 sur la maintenance de la physiologie mitochondriale.” 2018. Doctoral Dissertation, Université Paris-Saclay (ComUE). Accessed November 27, 2020. http://www.theses.fr/2018SACLS125.

MLA Handbook (7th Edition):

Lia, Debora. “Role of alphaOGG1 in the Maintenance of Mitochondrial Physiology : Fonction de l'alphaOGG1 sur la maintenance de la physiologie mitochondriale.” 2018. Web. 27 Nov 2020.

Vancouver:

Lia D. Role of alphaOGG1 in the Maintenance of Mitochondrial Physiology : Fonction de l'alphaOGG1 sur la maintenance de la physiologie mitochondriale. [Internet] [Doctoral dissertation]. Université Paris-Saclay (ComUE); 2018. [cited 2020 Nov 27]. Available from: http://www.theses.fr/2018SACLS125.

Council of Science Editors:

Lia D. Role of alphaOGG1 in the Maintenance of Mitochondrial Physiology : Fonction de l'alphaOGG1 sur la maintenance de la physiologie mitochondriale. [Doctoral Dissertation]. Université Paris-Saclay (ComUE); 2018. Available from: http://www.theses.fr/2018SACLS125


Georgia Tech

11. Balachander, Sathya. Characterization of ribonucleotides embedded in DNA.

Degree: PhD, Biology, 2018, Georgia Tech

 Ribonucleotides (rNMPs) are the most abundant non-standard nucleotides in genomic DNA. Presence of rNMPs embedded in DNA alters the DNA structure, function and their properties,… (more)

Subjects/Keywords: Ribonucleotides; Base excision repair; Yeast; RNase H2; APE1; Ribose-seq

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APA (6th Edition):

Balachander, S. (2018). Characterization of ribonucleotides embedded in DNA. (Doctoral Dissertation). Georgia Tech. Retrieved from http://hdl.handle.net/1853/62220

Chicago Manual of Style (16th Edition):

Balachander, Sathya. “Characterization of ribonucleotides embedded in DNA.” 2018. Doctoral Dissertation, Georgia Tech. Accessed November 27, 2020. http://hdl.handle.net/1853/62220.

MLA Handbook (7th Edition):

Balachander, Sathya. “Characterization of ribonucleotides embedded in DNA.” 2018. Web. 27 Nov 2020.

Vancouver:

Balachander S. Characterization of ribonucleotides embedded in DNA. [Internet] [Doctoral dissertation]. Georgia Tech; 2018. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1853/62220.

Council of Science Editors:

Balachander S. Characterization of ribonucleotides embedded in DNA. [Doctoral Dissertation]. Georgia Tech; 2018. Available from: http://hdl.handle.net/1853/62220


University of Vermont

12. Guo, Yin. Biochemical Characterization of DNA Glycosylases from Mycobacterium Tuberculosis.

Degree: PhD, Microbiology and Molecular Genetics, 2010, University of Vermont

 The DNA glycosylases function in the first step of the base excision repair (BER) process, that is responsible for removing base lesions resulting from oxidation,… (more)

Subjects/Keywords: Mycobacterium tuberculosis; Base Excision repair; Oxidative DNA glycosylases

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APA (6th Edition):

Guo, Y. (2010). Biochemical Characterization of DNA Glycosylases from Mycobacterium Tuberculosis. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/96

Chicago Manual of Style (16th Edition):

Guo, Yin. “Biochemical Characterization of DNA Glycosylases from Mycobacterium Tuberculosis.” 2010. Doctoral Dissertation, University of Vermont. Accessed November 27, 2020. https://scholarworks.uvm.edu/graddis/96.

MLA Handbook (7th Edition):

Guo, Yin. “Biochemical Characterization of DNA Glycosylases from Mycobacterium Tuberculosis.” 2010. Web. 27 Nov 2020.

Vancouver:

Guo Y. Biochemical Characterization of DNA Glycosylases from Mycobacterium Tuberculosis. [Internet] [Doctoral dissertation]. University of Vermont; 2010. [cited 2020 Nov 27]. Available from: https://scholarworks.uvm.edu/graddis/96.

Council of Science Editors:

Guo Y. Biochemical Characterization of DNA Glycosylases from Mycobacterium Tuberculosis. [Doctoral Dissertation]. University of Vermont; 2010. Available from: https://scholarworks.uvm.edu/graddis/96


Northeastern University

13. Moore, Stephen Paul Gifford. DNA damage and base excision repair: an important role during zebrafish embryogenesis.

Degree: PhD, Department of Biology, 2017, Northeastern University

 Damage to DNA is an unavoidable consequence of life. Unrepaired DNA damage is mutagenic, promotes genomic instability, and leads to the onset of numerous serious… (more)

Subjects/Keywords: base excision repair; DNA damage; DNA methylation; embryogenesis; epigenetic; zebrafish

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APA (6th Edition):

Moore, S. P. G. (2017). DNA damage and base excision repair: an important role during zebrafish embryogenesis. (Doctoral Dissertation). Northeastern University. Retrieved from http://hdl.handle.net/2047/D20261165

Chicago Manual of Style (16th Edition):

Moore, Stephen Paul Gifford. “DNA damage and base excision repair: an important role during zebrafish embryogenesis.” 2017. Doctoral Dissertation, Northeastern University. Accessed November 27, 2020. http://hdl.handle.net/2047/D20261165.

MLA Handbook (7th Edition):

Moore, Stephen Paul Gifford. “DNA damage and base excision repair: an important role during zebrafish embryogenesis.” 2017. Web. 27 Nov 2020.

Vancouver:

Moore SPG. DNA damage and base excision repair: an important role during zebrafish embryogenesis. [Internet] [Doctoral dissertation]. Northeastern University; 2017. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/2047/D20261165.

Council of Science Editors:

Moore SPG. DNA damage and base excision repair: an important role during zebrafish embryogenesis. [Doctoral Dissertation]. Northeastern University; 2017. Available from: http://hdl.handle.net/2047/D20261165


University of Southern California

14. Corral, Roman. Genetic variation in the base excision repair pathway, environmental risk factors and colorectal adenoma risk.

Degree: MS, Molecular Epidemiology, 2012, University of Southern California

 Three inter-related and consistent factors emerging from the epidemiological literature for colorectal adenoma are cigarette smoking, alcohol intake, and dietary folate levels. Oxidative damage caused… (more)

Subjects/Keywords: colorectal adenoma; base excision repair; folate; alcohol; smoking

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APA (6th Edition):

Corral, R. (2012). Genetic variation in the base excision repair pathway, environmental risk factors and colorectal adenoma risk. (Masters Thesis). University of Southern California. Retrieved from http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93708/rec/3013

Chicago Manual of Style (16th Edition):

Corral, Roman. “Genetic variation in the base excision repair pathway, environmental risk factors and colorectal adenoma risk.” 2012. Masters Thesis, University of Southern California. Accessed November 27, 2020. http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93708/rec/3013.

MLA Handbook (7th Edition):

Corral, Roman. “Genetic variation in the base excision repair pathway, environmental risk factors and colorectal adenoma risk.” 2012. Web. 27 Nov 2020.

Vancouver:

Corral R. Genetic variation in the base excision repair pathway, environmental risk factors and colorectal adenoma risk. [Internet] [Masters thesis]. University of Southern California; 2012. [cited 2020 Nov 27]. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93708/rec/3013.

Council of Science Editors:

Corral R. Genetic variation in the base excision repair pathway, environmental risk factors and colorectal adenoma risk. [Masters Thesis]. University of Southern California; 2012. Available from: http://digitallibrary.usc.edu/cdm/compoundobject/collection/p15799coll3/id/93708/rec/3013


Queens University

15. Mulder, Jeanne Elizabeh. DNA Repair Mechanisms, Aflatoxin B1-Induced DNA Damage and Carcinogenesis .

Degree: Pharmacology and Toxicology, 2013, Queens University

 The studies described in this thesis investigated the relationship between DNA repair mechanisms, aflatoxin B1 (AFB1)-induced DNA damage and carcinogenesis. Mice deficient in 8-oxoguanine glycosylase… (more)

Subjects/Keywords: Aflatoxin B1 ; Carcinogenesis ; Base Excision Repair ; 8-oxoguanine glycosylase 1 ; Oxidative DNA Damage ; Nucleotide Excision Repair ; p53

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APA (6th Edition):

Mulder, J. E. (2013). DNA Repair Mechanisms, Aflatoxin B1-Induced DNA Damage and Carcinogenesis . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/8427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mulder, Jeanne Elizabeh. “DNA Repair Mechanisms, Aflatoxin B1-Induced DNA Damage and Carcinogenesis .” 2013. Thesis, Queens University. Accessed November 27, 2020. http://hdl.handle.net/1974/8427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mulder, Jeanne Elizabeh. “DNA Repair Mechanisms, Aflatoxin B1-Induced DNA Damage and Carcinogenesis .” 2013. Web. 27 Nov 2020.

Vancouver:

Mulder JE. DNA Repair Mechanisms, Aflatoxin B1-Induced DNA Damage and Carcinogenesis . [Internet] [Thesis]. Queens University; 2013. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1974/8427.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mulder JE. DNA Repair Mechanisms, Aflatoxin B1-Induced DNA Damage and Carcinogenesis . [Thesis]. Queens University; 2013. Available from: http://hdl.handle.net/1974/8427

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. McLaughlin, Dylan. Characterizing effects of sumoylation on Thymine-DNA Glycosylase base excision repair activity in vivo.

Degree: 2014, Johns Hopkins University

 Thymine-DNA glycosylase (TDG) is an enzyme that recognizes and repairs G/T and G/U mismatches in the base excision repair (BER) pathway. Its BER function is… (more)

Subjects/Keywords: Thymine-DNA Glycosylase; TDG; sumoylation; SUMO; DNA repair; Base excision repair; BER

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APA (6th Edition):

McLaughlin, D. (2014). Characterizing effects of sumoylation on Thymine-DNA Glycosylase base excision repair activity in vivo. (Thesis). Johns Hopkins University. Retrieved from http://jhir.library.jhu.edu/handle/1774.2/37271

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

McLaughlin, Dylan. “Characterizing effects of sumoylation on Thymine-DNA Glycosylase base excision repair activity in vivo.” 2014. Thesis, Johns Hopkins University. Accessed November 27, 2020. http://jhir.library.jhu.edu/handle/1774.2/37271.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

McLaughlin, Dylan. “Characterizing effects of sumoylation on Thymine-DNA Glycosylase base excision repair activity in vivo.” 2014. Web. 27 Nov 2020.

Vancouver:

McLaughlin D. Characterizing effects of sumoylation on Thymine-DNA Glycosylase base excision repair activity in vivo. [Internet] [Thesis]. Johns Hopkins University; 2014. [cited 2020 Nov 27]. Available from: http://jhir.library.jhu.edu/handle/1774.2/37271.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

McLaughlin D. Characterizing effects of sumoylation on Thymine-DNA Glycosylase base excision repair activity in vivo. [Thesis]. Johns Hopkins University; 2014. Available from: http://jhir.library.jhu.edu/handle/1774.2/37271

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Florida International University

17. Beaver, Jill M. Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair.

Degree: PhD, Biochemistry, 2016, Florida International University

  Trinucleotide repeat (TNR) expansions are the cause of over 40 human neurodegenerative diseases, and are linked to DNA damage and base excision repair (BER).… (more)

Subjects/Keywords: DNA repair; trinucleotide repeats; DNA damage; trinucleotide repeat instability; DNA base excision repair; Biochemistry

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APA (6th Edition):

Beaver, J. M. (2016). Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair. (Doctoral Dissertation). Florida International University. Retrieved from https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178

Chicago Manual of Style (16th Edition):

Beaver, Jill M. “Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair.” 2016. Doctoral Dissertation, Florida International University. Accessed November 27, 2020. https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178.

MLA Handbook (7th Edition):

Beaver, Jill M. “Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair.” 2016. Web. 27 Nov 2020.

Vancouver:

Beaver JM. Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair. [Internet] [Doctoral dissertation]. Florida International University; 2016. [cited 2020 Nov 27]. Available from: https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178.

Council of Science Editors:

Beaver JM. Trinucleotide Repeat Instability is Modulated by DNA Base Lesions and DNA Base Excision Repair. [Doctoral Dissertation]. Florida International University; 2016. Available from: https://digitalcommons.fiu.edu/etd/3056 ; 10.25148/etd.FIDC001178 ; FIDC001178

18. Weeks, Lachelle Dawn. Elucidating a role for uracil DNA glycosylase (UNG)-initiated DNA base excision repair in the cellular sensitivity to the antifolate, pemetrexed.

Degree: PhD, Pathology, 2014, Case Western Reserve University School of Graduate Studies

 Antifolates are among the oldest anti-cancer chemotherapeutic agents. Despite decades of research, precise mechanisms of antifolate-mediated cell death remain ill defined. Natural and acquired resistance… (more)

Subjects/Keywords: Pathology; uracil DNA glycosylase; pemetrexed; antifolates; chemotherapy; DNA repair; base excision repair

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APA (6th Edition):

Weeks, L. D. (2014). Elucidating a role for uracil DNA glycosylase (UNG)-initiated DNA base excision repair in the cellular sensitivity to the antifolate, pemetrexed. (Doctoral Dissertation). Case Western Reserve University School of Graduate Studies. Retrieved from http://rave.ohiolink.edu/etdc/view?acc_num=case1386198769

Chicago Manual of Style (16th Edition):

Weeks, Lachelle Dawn. “Elucidating a role for uracil DNA glycosylase (UNG)-initiated DNA base excision repair in the cellular sensitivity to the antifolate, pemetrexed.” 2014. Doctoral Dissertation, Case Western Reserve University School of Graduate Studies. Accessed November 27, 2020. http://rave.ohiolink.edu/etdc/view?acc_num=case1386198769.

MLA Handbook (7th Edition):

Weeks, Lachelle Dawn. “Elucidating a role for uracil DNA glycosylase (UNG)-initiated DNA base excision repair in the cellular sensitivity to the antifolate, pemetrexed.” 2014. Web. 27 Nov 2020.

Vancouver:

Weeks LD. Elucidating a role for uracil DNA glycosylase (UNG)-initiated DNA base excision repair in the cellular sensitivity to the antifolate, pemetrexed. [Internet] [Doctoral dissertation]. Case Western Reserve University School of Graduate Studies; 2014. [cited 2020 Nov 27]. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1386198769.

Council of Science Editors:

Weeks LD. Elucidating a role for uracil DNA glycosylase (UNG)-initiated DNA base excision repair in the cellular sensitivity to the antifolate, pemetrexed. [Doctoral Dissertation]. Case Western Reserve University School of Graduate Studies; 2014. Available from: http://rave.ohiolink.edu/etdc/view?acc_num=case1386198769


University of Vermont

19. Zhou, Jia. Dna Glycosylases Remove Oxidized Base Damages From G-Quadruplex Dna Structures.

Degree: PhD, Microbiology and Molecular Genetics, 2015, University of Vermont

  The G-quadruplex DNA is a four-stranded DNA structure that is highly susceptible to oxidation due to its G-rich sequence and its structure. Oxidative DNA… (more)

Subjects/Keywords: base damage; base excision repair (BER); DNA glycosylase; promoter; quadruplex DNA; telomere; Biochemistry; Molecular Biology

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APA (6th Edition):

Zhou, J. (2015). Dna Glycosylases Remove Oxidized Base Damages From G-Quadruplex Dna Structures. (Doctoral Dissertation). University of Vermont. Retrieved from https://scholarworks.uvm.edu/graddis/529

Chicago Manual of Style (16th Edition):

Zhou, Jia. “Dna Glycosylases Remove Oxidized Base Damages From G-Quadruplex Dna Structures.” 2015. Doctoral Dissertation, University of Vermont. Accessed November 27, 2020. https://scholarworks.uvm.edu/graddis/529.

MLA Handbook (7th Edition):

Zhou, Jia. “Dna Glycosylases Remove Oxidized Base Damages From G-Quadruplex Dna Structures.” 2015. Web. 27 Nov 2020.

Vancouver:

Zhou J. Dna Glycosylases Remove Oxidized Base Damages From G-Quadruplex Dna Structures. [Internet] [Doctoral dissertation]. University of Vermont; 2015. [cited 2020 Nov 27]. Available from: https://scholarworks.uvm.edu/graddis/529.

Council of Science Editors:

Zhou J. Dna Glycosylases Remove Oxidized Base Damages From G-Quadruplex Dna Structures. [Doctoral Dissertation]. University of Vermont; 2015. Available from: https://scholarworks.uvm.edu/graddis/529


University of Lethbridge

20. University of Lethbridge. Faculty of Arts and Science. Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria .

Degree: 2018, University of Lethbridge

 Formation of guanine oxidation products is among the most frequently occurring DNA damaging events, and has been suspected to be related to aging, Alzheimer’s disease… (more)

Subjects/Keywords: DNA repair; DNA repair – Computer simulation; DNA damage; Oxidation; oxidation of guanine; base excision repair; computer modelling; DNA glycosylases

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APA (6th Edition):

Science, U. o. L. F. o. A. a. (2018). Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria . (Thesis). University of Lethbridge. Retrieved from http://hdl.handle.net/10133/5093

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Science, University of Lethbridge. Faculty of Arts and. “Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria .” 2018. Thesis, University of Lethbridge. Accessed November 27, 2020. http://hdl.handle.net/10133/5093.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Science, University of Lethbridge. Faculty of Arts and. “Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria .” 2018. Web. 27 Nov 2020.

Vancouver:

Science UoLFoAa. Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria . [Internet] [Thesis]. University of Lethbridge; 2018. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/10133/5093.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Science UoLFoAa. Computational investigation of oxidative damage to guanine: formation, recognition and removal by DNA repair enzymes in humans and bacteria . [Thesis]. University of Lethbridge; 2018. Available from: http://hdl.handle.net/10133/5093

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Rochester

21. Miller, Adam Scott. Human Telomere DNA Damage by Oxidation and Direct Effect Ionizing Radiation, and the Promotion of Telomere DNA Repair by Telomere Proteins.

Degree: PhD, 2013, University of Rochester

 Eukaryotic chromosomes contain linear DNA that is capped by a tract of specific triplet G-rich sequences called telomeres. Telomeres are distinctive from the rest of… (more)

Subjects/Keywords: Telomere; Long Patch Base Excision Repair; Oxidation; Direct Effect Ionizing Radiation; Shelterin

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APA (6th Edition):

Miller, A. S. (2013). Human Telomere DNA Damage by Oxidation and Direct Effect Ionizing Radiation, and the Promotion of Telomere DNA Repair by Telomere Proteins. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/26792

Chicago Manual of Style (16th Edition):

Miller, Adam Scott. “Human Telomere DNA Damage by Oxidation and Direct Effect Ionizing Radiation, and the Promotion of Telomere DNA Repair by Telomere Proteins.” 2013. Doctoral Dissertation, University of Rochester. Accessed November 27, 2020. http://hdl.handle.net/1802/26792.

MLA Handbook (7th Edition):

Miller, Adam Scott. “Human Telomere DNA Damage by Oxidation and Direct Effect Ionizing Radiation, and the Promotion of Telomere DNA Repair by Telomere Proteins.” 2013. Web. 27 Nov 2020.

Vancouver:

Miller AS. Human Telomere DNA Damage by Oxidation and Direct Effect Ionizing Radiation, and the Promotion of Telomere DNA Repair by Telomere Proteins. [Internet] [Doctoral dissertation]. University of Rochester; 2013. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1802/26792.

Council of Science Editors:

Miller AS. Human Telomere DNA Damage by Oxidation and Direct Effect Ionizing Radiation, and the Promotion of Telomere DNA Repair by Telomere Proteins. [Doctoral Dissertation]. University of Rochester; 2013. Available from: http://hdl.handle.net/1802/26792


University of Utah

22. Alshykhly, Omar R. Formation and enzyme processing of 5-carboxamido- 5-formamido-2-iminohydantoin, a major oxidation product of guanine in dna.

Degree: PhD, Chemistry, 2015, University of Utah

 Oxidative damage to DNA, a factor in cancer, mutation, and aging, is attributed to reactive oxygen species (ROS). ROS are formed exogenously and endogenously and… (more)

Subjects/Keywords: Base excision repair; DNA damage; Fenton reaction; Guanine oxidation; Reactive oxygen species; X-ray

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APA (6th Edition):

Alshykhly, O. R. (2015). Formation and enzyme processing of 5-carboxamido- 5-formamido-2-iminohydantoin, a major oxidation product of guanine in dna. (Doctoral Dissertation). University of Utah. Retrieved from http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/3988/rec/1060

Chicago Manual of Style (16th Edition):

Alshykhly, Omar R. “Formation and enzyme processing of 5-carboxamido- 5-formamido-2-iminohydantoin, a major oxidation product of guanine in dna.” 2015. Doctoral Dissertation, University of Utah. Accessed November 27, 2020. http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/3988/rec/1060.

MLA Handbook (7th Edition):

Alshykhly, Omar R. “Formation and enzyme processing of 5-carboxamido- 5-formamido-2-iminohydantoin, a major oxidation product of guanine in dna.” 2015. Web. 27 Nov 2020.

Vancouver:

Alshykhly OR. Formation and enzyme processing of 5-carboxamido- 5-formamido-2-iminohydantoin, a major oxidation product of guanine in dna. [Internet] [Doctoral dissertation]. University of Utah; 2015. [cited 2020 Nov 27]. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/3988/rec/1060.

Council of Science Editors:

Alshykhly OR. Formation and enzyme processing of 5-carboxamido- 5-formamido-2-iminohydantoin, a major oxidation product of guanine in dna. [Doctoral Dissertation]. University of Utah; 2015. Available from: http://content.lib.utah.edu/cdm/singleitem/collection/etd3/id/3988/rec/1060


Wayne State University

23. Wei, Shanqiao. Development Of A Novel Class Of Chemicals For Labeling Abasic Sites In Cellular Dna And Killing Cancer Cells.

Degree: PhD, Chemistry, 2016, Wayne State University

  Abasic (AP) sites are the most common type of lesions in DNA. Numerous endogenous and exogenous agents and cellular processes can induce the formation… (more)

Subjects/Keywords: abasic site; alkoxyamines; base excision repair; B-cell lymphomas; cytotoxicity; Biochemistry; Molecular Biology

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APA (6th Edition):

Wei, S. (2016). Development Of A Novel Class Of Chemicals For Labeling Abasic Sites In Cellular Dna And Killing Cancer Cells. (Doctoral Dissertation). Wayne State University. Retrieved from https://digitalcommons.wayne.edu/oa_dissertations/1669

Chicago Manual of Style (16th Edition):

Wei, Shanqiao. “Development Of A Novel Class Of Chemicals For Labeling Abasic Sites In Cellular Dna And Killing Cancer Cells.” 2016. Doctoral Dissertation, Wayne State University. Accessed November 27, 2020. https://digitalcommons.wayne.edu/oa_dissertations/1669.

MLA Handbook (7th Edition):

Wei, Shanqiao. “Development Of A Novel Class Of Chemicals For Labeling Abasic Sites In Cellular Dna And Killing Cancer Cells.” 2016. Web. 27 Nov 2020.

Vancouver:

Wei S. Development Of A Novel Class Of Chemicals For Labeling Abasic Sites In Cellular Dna And Killing Cancer Cells. [Internet] [Doctoral dissertation]. Wayne State University; 2016. [cited 2020 Nov 27]. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1669.

Council of Science Editors:

Wei S. Development Of A Novel Class Of Chemicals For Labeling Abasic Sites In Cellular Dna And Killing Cancer Cells. [Doctoral Dissertation]. Wayne State University; 2016. Available from: https://digitalcommons.wayne.edu/oa_dissertations/1669


Queens University

24. Gupta, Neeraj. DNA oxidation and base excision repair in lung and liver of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone treated mice .

Degree: Pharmacology and Toxicology, 2011, Queens University

 4-(Methylnitrosamino)-1-(3-pyridyl)-1-butanone (NNK) is a potent pulmonary carcinogen found in unburned tobacco and tobacco smoke. To exert its carcinogenic effect, NNK is metabolically activated to reactive… (more)

Subjects/Keywords: 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone ; Oxidative DNA Damage ; Base Excision Repair

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APA (6th Edition):

Gupta, N. (2011). DNA oxidation and base excision repair in lung and liver of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone treated mice . (Thesis). Queens University. Retrieved from http://hdl.handle.net/1974/6465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gupta, Neeraj. “DNA oxidation and base excision repair in lung and liver of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone treated mice .” 2011. Thesis, Queens University. Accessed November 27, 2020. http://hdl.handle.net/1974/6465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gupta, Neeraj. “DNA oxidation and base excision repair in lung and liver of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone treated mice .” 2011. Web. 27 Nov 2020.

Vancouver:

Gupta N. DNA oxidation and base excision repair in lung and liver of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone treated mice . [Internet] [Thesis]. Queens University; 2011. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1974/6465.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gupta N. DNA oxidation and base excision repair in lung and liver of 4-(methylnitrosamino)-1-(3-pyridyl)-1-butanone treated mice . [Thesis]. Queens University; 2011. Available from: http://hdl.handle.net/1974/6465

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


University of Kentucky

25. Soman, Sony. OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE.

Degree: 2013, University of Kentucky

 Previous studies from our laboratory and others show a significant increase in levels of both nuclear and mitochondrial DNA and RNA oxidation in vulnerable brain… (more)

Subjects/Keywords: Alzheimer’s disease (AD); oxidative stress; base excision repair; DNA oxidation; Other Chemistry

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APA (6th Edition):

Soman, S. (2013). OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/chemistry_etds/28

Chicago Manual of Style (16th Edition):

Soman, Sony. “OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE.” 2013. Doctoral Dissertation, University of Kentucky. Accessed November 27, 2020. https://uknowledge.uky.edu/chemistry_etds/28.

MLA Handbook (7th Edition):

Soman, Sony. “OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE.” 2013. Web. 27 Nov 2020.

Vancouver:

Soman S. OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE. [Internet] [Doctoral dissertation]. University of Kentucky; 2013. [cited 2020 Nov 27]. Available from: https://uknowledge.uky.edu/chemistry_etds/28.

Council of Science Editors:

Soman S. OXIDATIVE DAMAGE TO DNA IN ALZHEIMER'S DISEASE. [Doctoral Dissertation]. University of Kentucky; 2013. Available from: https://uknowledge.uky.edu/chemistry_etds/28


University of Kentucky

26. Carpenter, Brittany L. INTEGRIN α6β4 PROMOTES PANCREATIC CANCER INVASION BY ALTERING DNA REPAIR-MEDIATED EPIGENETICS.

Degree: 2016, University of Kentucky

 Integrin α6β4 is upregulated in pancreatic carcinoma, where signaling promotes metastatic properties, in part by altering the transcriptome. Such alterations can be accomplished through DNA… (more)

Subjects/Keywords: Integrin α6β4; DNA Methylation; Base Excision Repair; Pancreatic Cancer; EGFR Signaling; Cancer Biology; Molecular Biology

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APA (6th Edition):

Carpenter, B. L. (2016). INTEGRIN α6β4 PROMOTES PANCREATIC CANCER INVASION BY ALTERING DNA REPAIR-MEDIATED EPIGENETICS. (Doctoral Dissertation). University of Kentucky. Retrieved from https://uknowledge.uky.edu/biochem_etds/27

Chicago Manual of Style (16th Edition):

Carpenter, Brittany L. “INTEGRIN α6β4 PROMOTES PANCREATIC CANCER INVASION BY ALTERING DNA REPAIR-MEDIATED EPIGENETICS.” 2016. Doctoral Dissertation, University of Kentucky. Accessed November 27, 2020. https://uknowledge.uky.edu/biochem_etds/27.

MLA Handbook (7th Edition):

Carpenter, Brittany L. “INTEGRIN α6β4 PROMOTES PANCREATIC CANCER INVASION BY ALTERING DNA REPAIR-MEDIATED EPIGENETICS.” 2016. Web. 27 Nov 2020.

Vancouver:

Carpenter BL. INTEGRIN α6β4 PROMOTES PANCREATIC CANCER INVASION BY ALTERING DNA REPAIR-MEDIATED EPIGENETICS. [Internet] [Doctoral dissertation]. University of Kentucky; 2016. [cited 2020 Nov 27]. Available from: https://uknowledge.uky.edu/biochem_etds/27.

Council of Science Editors:

Carpenter BL. INTEGRIN α6β4 PROMOTES PANCREATIC CANCER INVASION BY ALTERING DNA REPAIR-MEDIATED EPIGENETICS. [Doctoral Dissertation]. University of Kentucky; 2016. Available from: https://uknowledge.uky.edu/biochem_etds/27


Universidade do Rio Grande do Norte

27. Oliveira, Andrea de Lima. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar .

Degree: 2009, Universidade do Rio Grande do Norte

 The genome of all organisms is subject to injuries that can be caused by endogenous and environmental factors. If these lesions are not corrected, it… (more)

Subjects/Keywords: Reparo por excisão de base; AP endonuclease; Cana-de-açúcar; Base excision repair; AP endonuclease; Sugarcane

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APA (6th Edition):

Oliveira, A. d. L. (2009). Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar . (Thesis). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/18626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oliveira, Andrea de Lima. “Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar .” 2009. Thesis, Universidade do Rio Grande do Norte. Accessed November 27, 2020. http://repositorio.ufrn.br/handle/123456789/18626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oliveira, Andrea de Lima. “Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar .” 2009. Web. 27 Nov 2020.

Vancouver:

Oliveira AdL. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar . [Internet] [Thesis]. Universidade do Rio Grande do Norte; 2009. [cited 2020 Nov 27]. Available from: http://repositorio.ufrn.br/handle/123456789/18626.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oliveira AdL. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar . [Thesis]. Universidade do Rio Grande do Norte; 2009. Available from: http://repositorio.ufrn.br/handle/123456789/18626

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Andrea de Lima Oliveira. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar.

Degree: 2009, Universidade Federal do Rio Grande do Norte

The genome of all organisms is subject to injuries that can be caused by endogenous and environmental factors. If these lesions are not corrected, it… (more)

Subjects/Keywords: Reparo por excisão de base; AP endonuclease; Cana-de-açúcar; BIOLOGIA MOLECULAR; Base excision repair; AP endonuclease; Sugarcane

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APA (6th Edition):

Oliveira, A. d. L. (2009). Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar. (Thesis). Universidade Federal do Rio Grande do Norte. Retrieved from http://bdtd.bczm.ufrn.br/tedesimplificado//tde_busca/arquivo.php?codArquivo=2363

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Oliveira, Andrea de Lima. “Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar.” 2009. Thesis, Universidade Federal do Rio Grande do Norte. Accessed November 27, 2020. http://bdtd.bczm.ufrn.br/tedesimplificado//tde_busca/arquivo.php?codArquivo=2363.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Oliveira, Andrea de Lima. “Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar.” 2009. Web. 27 Nov 2020.

Vancouver:

Oliveira AdL. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar. [Internet] [Thesis]. Universidade Federal do Rio Grande do Norte; 2009. [cited 2020 Nov 27]. Available from: http://bdtd.bczm.ufrn.br/tedesimplificado//tde_busca/arquivo.php?codArquivo=2363.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Oliveira AdL. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar. [Thesis]. Universidade Federal do Rio Grande do Norte; 2009. Available from: http://bdtd.bczm.ufrn.br/tedesimplificado//tde_busca/arquivo.php?codArquivo=2363

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Universidade do Rio Grande do Norte

29. Oliveira, Andrea de Lima. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar .

Degree: 2009, Universidade do Rio Grande do Norte

 The genome of all organisms is subject to injuries that can be caused by endogenous and environmental factors. If these lesions are not corrected, it… (more)

Subjects/Keywords: Reparo por excisão de base; AP endonuclease; Cana-de-açúcar; Base excision repair; AP endonuclease; Sugarcane

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oliveira, A. d. L. (2009). Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar . (Masters Thesis). Universidade do Rio Grande do Norte. Retrieved from http://repositorio.ufrn.br/handle/123456789/18626

Chicago Manual of Style (16th Edition):

Oliveira, Andrea de Lima. “Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar .” 2009. Masters Thesis, Universidade do Rio Grande do Norte. Accessed November 27, 2020. http://repositorio.ufrn.br/handle/123456789/18626.

MLA Handbook (7th Edition):

Oliveira, Andrea de Lima. “Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar .” 2009. Web. 27 Nov 2020.

Vancouver:

Oliveira AdL. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar . [Internet] [Masters thesis]. Universidade do Rio Grande do Norte; 2009. [cited 2020 Nov 27]. Available from: http://repositorio.ufrn.br/handle/123456789/18626.

Council of Science Editors:

Oliveira AdL. Caracterização de dois cDNAs homológos e uma AP endonuclease em cana-de-açúcar . [Masters Thesis]. Universidade do Rio Grande do Norte; 2009. Available from: http://repositorio.ufrn.br/handle/123456789/18626


University of Rochester

30. Cole, Hope Abigail. Activities of Two Base Excision Repair Enzymes on Nucleosomal Substrates.

Degree: PhD, 2009, University of Rochester

 In eukaryotic cells DNA is assembled into chromatin, the basic repeating unit of which is a nucleosome core particle (NCP), made up of approximately 147… (more)

Subjects/Keywords: APE; Nucleosome; Base Excision Repair (BER); Chromatin; UDG

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cole, H. A. (2009). Activities of Two Base Excision Repair Enzymes on Nucleosomal Substrates. (Doctoral Dissertation). University of Rochester. Retrieved from http://hdl.handle.net/1802/6632

Chicago Manual of Style (16th Edition):

Cole, Hope Abigail. “Activities of Two Base Excision Repair Enzymes on Nucleosomal Substrates.” 2009. Doctoral Dissertation, University of Rochester. Accessed November 27, 2020. http://hdl.handle.net/1802/6632.

MLA Handbook (7th Edition):

Cole, Hope Abigail. “Activities of Two Base Excision Repair Enzymes on Nucleosomal Substrates.” 2009. Web. 27 Nov 2020.

Vancouver:

Cole HA. Activities of Two Base Excision Repair Enzymes on Nucleosomal Substrates. [Internet] [Doctoral dissertation]. University of Rochester; 2009. [cited 2020 Nov 27]. Available from: http://hdl.handle.net/1802/6632.

Council of Science Editors:

Cole HA. Activities of Two Base Excision Repair Enzymes on Nucleosomal Substrates. [Doctoral Dissertation]. University of Rochester; 2009. Available from: http://hdl.handle.net/1802/6632

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